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You searched for subject:(vascular smooth muscle). Showing records 61 – 90 of 273 total matches.

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University of Hong Kong

61. Xu, Yanchun. Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries.

Degree: PhD, 2007, University of Hong Kong

abstract

published_or_final_version

Pharmacology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Man, RYK.

Subjects/Keywords: Flavonoids.; Coronary arteries.; Vascular smooth muscle

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APA (6th Edition):

Xu, Y. (2007). Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. (Doctoral Dissertation). University of Hong Kong. Retrieved from Xu, Y. [徐艷春]. (2007). Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3879461 ; http://dx.doi.org/10.5353/th_b3879461 ; http://hdl.handle.net/10722/52321

Chicago Manual of Style (16th Edition):

Xu, Yanchun. “Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries.” 2007. Doctoral Dissertation, University of Hong Kong. Accessed October 17, 2019. Xu, Y. [徐艷春]. (2007). Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3879461 ; http://dx.doi.org/10.5353/th_b3879461 ; http://hdl.handle.net/10722/52321.

MLA Handbook (7th Edition):

Xu, Yanchun. “Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries.” 2007. Web. 17 Oct 2019.

Vancouver:

Xu Y. Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. [Internet] [Doctoral dissertation]. University of Hong Kong; 2007. [cited 2019 Oct 17]. Available from: Xu, Y. [徐艷春]. (2007). Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3879461 ; http://dx.doi.org/10.5353/th_b3879461 ; http://hdl.handle.net/10722/52321.

Council of Science Editors:

Xu Y. Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. [Doctoral Dissertation]. University of Hong Kong; 2007. Available from: Xu, Y. [徐艷春]. (2007). Vascular effects and signaling mechanisms of flavonoids in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3879461 ; http://dx.doi.org/10.5353/th_b3879461 ; http://hdl.handle.net/10722/52321


University of Manchester

62. Losa Llabata, Marta. Gene regulation in embryonic development.

Degree: PhD, 2016, University of Manchester

 Branchial arches (BAs) are a series of transient structures that develop on the ventro-lateral surface of the head in vertebrate embryos. BAs initially appear as… (more)

Subjects/Keywords: 612.6; branchial arch; neural crest cells; aortic arch; vascular smooth muscle cells

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APA (6th Edition):

Losa Llabata, M. (2016). Gene regulation in embryonic development. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005

Chicago Manual of Style (16th Edition):

Losa Llabata, Marta. “Gene regulation in embryonic development.” 2016. Doctoral Dissertation, University of Manchester. Accessed October 17, 2019. https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005.

MLA Handbook (7th Edition):

Losa Llabata, Marta. “Gene regulation in embryonic development.” 2016. Web. 17 Oct 2019.

Vancouver:

Losa Llabata M. Gene regulation in embryonic development. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Oct 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005.

Council of Science Editors:

Losa Llabata M. Gene regulation in embryonic development. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.703005


University of Hong Kong

63. Ho, Ka-lai, Cally. Functional properties of aortic smooth muscle in bicuspid aortic valvedisease.

Degree: Master of Medical Sciences, 2011, University of Hong Kong

published_or_final_version

Physiology

Master

Master of Medical Sciences

Subjects/Keywords: Vascular smooth muscle.; Aortic valve - Diseases.

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APA (6th Edition):

Ho, Ka-lai, C. (2011). Functional properties of aortic smooth muscle in bicuspid aortic valvedisease. (Masters Thesis). University of Hong Kong. Retrieved from Ho, K. C. [何嘉麗]. (2011). Functional properties of aortic smooth muscle in bicuspid aortic valve disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4669948 ; http://dx.doi.org/10.5353/th_b4669948 ; http://hdl.handle.net/10722/144022

Chicago Manual of Style (16th Edition):

Ho, Ka-lai, Cally. “Functional properties of aortic smooth muscle in bicuspid aortic valvedisease.” 2011. Masters Thesis, University of Hong Kong. Accessed October 17, 2019. Ho, K. C. [何嘉麗]. (2011). Functional properties of aortic smooth muscle in bicuspid aortic valve disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4669948 ; http://dx.doi.org/10.5353/th_b4669948 ; http://hdl.handle.net/10722/144022.

MLA Handbook (7th Edition):

Ho, Ka-lai, Cally. “Functional properties of aortic smooth muscle in bicuspid aortic valvedisease.” 2011. Web. 17 Oct 2019.

Vancouver:

Ho, Ka-lai C. Functional properties of aortic smooth muscle in bicuspid aortic valvedisease. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Oct 17]. Available from: Ho, K. C. [何嘉麗]. (2011). Functional properties of aortic smooth muscle in bicuspid aortic valve disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4669948 ; http://dx.doi.org/10.5353/th_b4669948 ; http://hdl.handle.net/10722/144022.

Council of Science Editors:

Ho, Ka-lai C. Functional properties of aortic smooth muscle in bicuspid aortic valvedisease. [Masters Thesis]. University of Hong Kong; 2011. Available from: Ho, K. C. [何嘉麗]. (2011). Functional properties of aortic smooth muscle in bicuspid aortic valve disease. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4669948 ; http://dx.doi.org/10.5353/th_b4669948 ; http://hdl.handle.net/10722/144022


Penn State University

64. Houck, Kristy Lee. SPHINGOLIPID AND PHOSPHOLIPID METABOLITES REGULATE THE PHENOTYPE OF VASCULAR SMOOTH MUSCLE CELLS.

Degree: PhD, Pharmacology, 2008, Penn State University

 Cardiovascular disease, particularly atherosclerosis, is a major cause of concern as it is the primary cause of death today. Atherosclerosis is an inflammatory disease that… (more)

Subjects/Keywords: toll-like receptor; diglycerides; ceramide-1-phosphate; vascular smooth muscle; signaling cascades; cytokine

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APA (6th Edition):

Houck, K. L. (2008). SPHINGOLIPID AND PHOSPHOLIPID METABOLITES REGULATE THE PHENOTYPE OF VASCULAR SMOOTH MUSCLE CELLS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8344

Chicago Manual of Style (16th Edition):

Houck, Kristy Lee. “SPHINGOLIPID AND PHOSPHOLIPID METABOLITES REGULATE THE PHENOTYPE OF VASCULAR SMOOTH MUSCLE CELLS.” 2008. Doctoral Dissertation, Penn State University. Accessed October 17, 2019. https://etda.libraries.psu.edu/catalog/8344.

MLA Handbook (7th Edition):

Houck, Kristy Lee. “SPHINGOLIPID AND PHOSPHOLIPID METABOLITES REGULATE THE PHENOTYPE OF VASCULAR SMOOTH MUSCLE CELLS.” 2008. Web. 17 Oct 2019.

Vancouver:

Houck KL. SPHINGOLIPID AND PHOSPHOLIPID METABOLITES REGULATE THE PHENOTYPE OF VASCULAR SMOOTH MUSCLE CELLS. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2019 Oct 17]. Available from: https://etda.libraries.psu.edu/catalog/8344.

Council of Science Editors:

Houck KL. SPHINGOLIPID AND PHOSPHOLIPID METABOLITES REGULATE THE PHENOTYPE OF VASCULAR SMOOTH MUSCLE CELLS. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8344


Clemson University

65. Mcallister, William. Vascular Smooth Muscle Cells in Response to Gold Nanoparticles.

Degree: MS, Bioengineering, 2011, Clemson University

 In this master's thesis we look at elucidating the interactions between nanoparticles and cells. Specifically, we looked at how the cell mechanics are affected, cytotoxicity… (more)

Subjects/Keywords: Atomic Force Microscopy; Cell Mechanics; Gold Nanoparticles; Vascular Smooth Muscle Cells; Biomedical Engineering and Bioengineering

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APA (6th Edition):

Mcallister, W. (2011). Vascular Smooth Muscle Cells in Response to Gold Nanoparticles. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/1101

Chicago Manual of Style (16th Edition):

Mcallister, William. “Vascular Smooth Muscle Cells in Response to Gold Nanoparticles.” 2011. Masters Thesis, Clemson University. Accessed October 17, 2019. https://tigerprints.clemson.edu/all_theses/1101.

MLA Handbook (7th Edition):

Mcallister, William. “Vascular Smooth Muscle Cells in Response to Gold Nanoparticles.” 2011. Web. 17 Oct 2019.

Vancouver:

Mcallister W. Vascular Smooth Muscle Cells in Response to Gold Nanoparticles. [Internet] [Masters thesis]. Clemson University; 2011. [cited 2019 Oct 17]. Available from: https://tigerprints.clemson.edu/all_theses/1101.

Council of Science Editors:

Mcallister W. Vascular Smooth Muscle Cells in Response to Gold Nanoparticles. [Masters Thesis]. Clemson University; 2011. Available from: https://tigerprints.clemson.edu/all_theses/1101


University of Manchester

66. Pieri, Maria. Regulation of vascular smooth muscle actin cytoskeleton by Hic-5.

Degree: 2016, University of Manchester

Vascular smooth muscle cells (VSMC) constitute an important component of blood vessels and are primarily responsible for vessel contraction. In vascular disorders such as hypertension… (more)

Subjects/Keywords: Hic-5; actin cytoskeleton; vascular smooth muscle; ET-1; NA; focal adhesions

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APA (6th Edition):

Pieri, M. (2016). Regulation of vascular smooth muscle actin cytoskeleton by Hic-5. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294745

Chicago Manual of Style (16th Edition):

Pieri, Maria. “Regulation of vascular smooth muscle actin cytoskeleton by Hic-5.” 2016. Doctoral Dissertation, University of Manchester. Accessed October 17, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294745.

MLA Handbook (7th Edition):

Pieri, Maria. “Regulation of vascular smooth muscle actin cytoskeleton by Hic-5.” 2016. Web. 17 Oct 2019.

Vancouver:

Pieri M. Regulation of vascular smooth muscle actin cytoskeleton by Hic-5. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Oct 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294745.

Council of Science Editors:

Pieri M. Regulation of vascular smooth muscle actin cytoskeleton by Hic-5. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:294745


Clemson University

67. Kieu, Tri. The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching.

Degree: MS, Bioengineering, 2013, Clemson University

  The field of nanotechnology research has seen a large growth in the past few decades due to the great potential of novel nano-size material… (more)

Subjects/Keywords: Cytotoxicity; Gold Nanoparticle; Mechanic Properties; Vascular Smooth Muscle Cells; Biomedical Engineering and Bioengineering

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APA (6th Edition):

Kieu, T. (2013). The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/1821

Chicago Manual of Style (16th Edition):

Kieu, Tri. “The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching.” 2013. Masters Thesis, Clemson University. Accessed October 17, 2019. https://tigerprints.clemson.edu/all_theses/1821.

MLA Handbook (7th Edition):

Kieu, Tri. “The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching.” 2013. Web. 17 Oct 2019.

Vancouver:

Kieu T. The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching. [Internet] [Masters thesis]. Clemson University; 2013. [cited 2019 Oct 17]. Available from: https://tigerprints.clemson.edu/all_theses/1821.

Council of Science Editors:

Kieu T. The Effects of Different Size Gold Nanoparticles on Mechanical Properties of Vascular Smooth Muscle Cells Under Mechanical Stretching. [Masters Thesis]. Clemson University; 2013. Available from: https://tigerprints.clemson.edu/all_theses/1821


Clemson University

68. Cheluvaraju, Chaitra. Characterization of anti-proteolytic and anti-proliferative activities of pentagalloylglucose; its potential application as a therapeutic agent in vascular diseases.

Degree: PhD, Bioengineering, 2010, Clemson University

 Cardiovascular diseases are the leading causes of mortality in the United States and will cost around $500 billion this year alone. Elevated proteolytic activity, increased… (more)

Subjects/Keywords: aortic smooth muscle cells; cathepsins; pentagalloylglucose; polyphenols; proliferation; vascular calcification; Biomedical Engineering and Bioengineering

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APA (6th Edition):

Cheluvaraju, C. (2010). Characterization of anti-proteolytic and anti-proliferative activities of pentagalloylglucose; its potential application as a therapeutic agent in vascular diseases. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/617

Chicago Manual of Style (16th Edition):

Cheluvaraju, Chaitra. “Characterization of anti-proteolytic and anti-proliferative activities of pentagalloylglucose; its potential application as a therapeutic agent in vascular diseases.” 2010. Doctoral Dissertation, Clemson University. Accessed October 17, 2019. https://tigerprints.clemson.edu/all_dissertations/617.

MLA Handbook (7th Edition):

Cheluvaraju, Chaitra. “Characterization of anti-proteolytic and anti-proliferative activities of pentagalloylglucose; its potential application as a therapeutic agent in vascular diseases.” 2010. Web. 17 Oct 2019.

Vancouver:

Cheluvaraju C. Characterization of anti-proteolytic and anti-proliferative activities of pentagalloylglucose; its potential application as a therapeutic agent in vascular diseases. [Internet] [Doctoral dissertation]. Clemson University; 2010. [cited 2019 Oct 17]. Available from: https://tigerprints.clemson.edu/all_dissertations/617.

Council of Science Editors:

Cheluvaraju C. Characterization of anti-proteolytic and anti-proliferative activities of pentagalloylglucose; its potential application as a therapeutic agent in vascular diseases. [Doctoral Dissertation]. Clemson University; 2010. Available from: https://tigerprints.clemson.edu/all_dissertations/617


University of California – Irvine

69. Albay, Ricardo. Characterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid Angiopathy.

Degree: Biological Sciences, 2018, University of California – Irvine

 ABSTRACT OF THE DISSERTATIONCharacterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid AngiopathyByRicardo Albay IIIDoctor of Philosophy in Biological SciencesUniversity… (more)

Subjects/Keywords: Molecular biology; Biochemistry; Alzheimer's Disease; amyloid; antibody; ; Cerebral Vascular Angiopathy; smooth muscle

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APA (6th Edition):

Albay, R. (2018). Characterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid Angiopathy. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/1xp4f2kt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Albay, Ricardo. “Characterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid Angiopathy.” 2018. Thesis, University of California – Irvine. Accessed October 17, 2019. http://www.escholarship.org/uc/item/1xp4f2kt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Albay, Ricardo. “Characterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid Angiopathy.” 2018. Web. 17 Oct 2019.

Vancouver:

Albay R. Characterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid Angiopathy. [Internet] [Thesis]. University of California – Irvine; 2018. [cited 2019 Oct 17]. Available from: http://www.escholarship.org/uc/item/1xp4f2kt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Albay R. Characterization of a pathogenic conformation of amyloid immunoreactivity in Alzheimer’s Disease and Cerebral Amyloid Angiopathy. [Thesis]. University of California – Irvine; 2018. Available from: http://www.escholarship.org/uc/item/1xp4f2kt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

70. Murugesan, Vignesh. Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis.

Degree: 2017, University of Lund

 Atherosclerosis is the underlying cause for myocardal infarction, stroke and peripheral arterial disease, collectively referred to as cardiovascular diseases. These represent the major cause of… (more)

Subjects/Keywords: Dystrophin glycoprotein complex; beta-sarcoglycan; dystrophin; Plaque; vascular smooth muscle cells; atherosclerosis; restenosis

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APA (6th Edition):

Murugesan, V. (2017). Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; http://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf

Chicago Manual of Style (16th Edition):

Murugesan, Vignesh. “Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis.” 2017. Doctoral Dissertation, University of Lund. Accessed October 17, 2019. http://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; http://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf.

MLA Handbook (7th Edition):

Murugesan, Vignesh. “Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis.” 2017. Web. 17 Oct 2019.

Vancouver:

Murugesan V. Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis. [Internet] [Doctoral dissertation]. University of Lund; 2017. [cited 2019 Oct 17]. Available from: http://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; http://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf.

Council of Science Editors:

Murugesan V. Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis. [Doctoral Dissertation]. University of Lund; 2017. Available from: http://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; http://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf


Dublin City University

71. Killeen, Maria T. Endothelial cell impact on smooth muscle cell properties: role of hemodynamic forces.

Degree: School of Biotechnology, 2009, Dublin City University

 The vascular endothelium is a dynamic cell monolayer located at the interface of the vessel wall and bloodstream, where it regulates the physiological effects of… (more)

Subjects/Keywords: Biotechnology; endothelial; smooth muscle; shear stress; cyclic strain; proliferation; signal transduction; vascular biology

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APA (6th Edition):

Killeen, M. T. (2009). Endothelial cell impact on smooth muscle cell properties: role of hemodynamic forces. (Thesis). Dublin City University. Retrieved from http://doras.dcu.ie/4633/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Killeen, Maria T. “Endothelial cell impact on smooth muscle cell properties: role of hemodynamic forces.” 2009. Thesis, Dublin City University. Accessed October 17, 2019. http://doras.dcu.ie/4633/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Killeen, Maria T. “Endothelial cell impact on smooth muscle cell properties: role of hemodynamic forces.” 2009. Web. 17 Oct 2019.

Vancouver:

Killeen MT. Endothelial cell impact on smooth muscle cell properties: role of hemodynamic forces. [Internet] [Thesis]. Dublin City University; 2009. [cited 2019 Oct 17]. Available from: http://doras.dcu.ie/4633/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Killeen MT. Endothelial cell impact on smooth muscle cell properties: role of hemodynamic forces. [Thesis]. Dublin City University; 2009. Available from: http://doras.dcu.ie/4633/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

72. Smith, Christopher Lacey. Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors.

Degree: 2013, University of Texas Southwestern Medical Center

 Coronary vascular smooth muscle cells (cVSMC) and cardiac fibroblasts are essential for coronary artery development and are important mediators of myocardial pathogenesis. These cells form… (more)

Subjects/Keywords: Epithelial-Mesenchymal Transition; Muscle, Smooth, Vascular; Platelet-Derived Growth Factor; SOX9 Transcription Factor

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APA (6th Edition):

Smith, C. L. (2013). Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Christopher Lacey. “Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed October 17, 2019. http://hdl.handle.net/2152.5/1707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Christopher Lacey. “Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors.” 2013. Web. 17 Oct 2019.

Vancouver:

Smith CL. Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2152.5/1707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith CL. Regulation of Cardiac Fibroblast and Coronary Vascular Smooth Muscle Development by Platelet Derived Growth Factor Receptors. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

73. Sattar, Joobin. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.

Degree: 2015, University of Toronto

RGS5 within vascular smooth muscle cells (VSMCs) is capable of inhibiting G-protein signaling involved in VSMC recruitment. Based on the suggested ability of statin to… (more)

Subjects/Keywords: Atherosclerosis; G-Protein Signaling; Neointimal Hyperplasia; Pleiotropic; Statins; Vascular Smooth Muscle Cell; 0719

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APA (6th Edition):

Sattar, J. (2015). Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70645

Chicago Manual of Style (16th Edition):

Sattar, Joobin. “Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.” 2015. Masters Thesis, University of Toronto. Accessed October 17, 2019. http://hdl.handle.net/1807/70645.

MLA Handbook (7th Edition):

Sattar, Joobin. “Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment.” 2015. Web. 17 Oct 2019.

Vancouver:

Sattar J. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/1807/70645.

Council of Science Editors:

Sattar J. Investigating the Putative Mechanism and Functional Role of RGS5 Upregulation in Vascular Smooth Muscle Cells Following Statin Treatment. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70645


University of Toronto

74. Dinh, Danny Duy. Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout.

Degree: 2015, University of Toronto

Resistance arteries possess an intrinsic ability, termed the myogenic response, which enables them to adapt to changes in blood pressure, allowing for the regulation of… (more)

Subjects/Keywords: Ca2+ sensitivity; compensation; resistance artery; sphingosine-1-phosphate; tumor necrosis factor; vascular smooth muscle; 0719

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APA (6th Edition):

Dinh, D. D. (2015). Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74688

Chicago Manual of Style (16th Edition):

Dinh, Danny Duy. “Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout.” 2015. Masters Thesis, University of Toronto. Accessed October 17, 2019. http://hdl.handle.net/1807/74688.

MLA Handbook (7th Edition):

Dinh, Danny Duy. “Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout.” 2015. Web. 17 Oct 2019.

Vancouver:

Dinh DD. Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/1807/74688.

Council of Science Editors:

Dinh DD. Recovery of Skeletal Muscle Microvascular Myogenic Reactivity After Smooth Muscle Cell-TNF Knockout. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74688


University of Toronto

75. Fares, Jessica. Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage.

Degree: 2015, University of Toronto

Subarachnoid hemorrhage (SAH) is a devastating type of stroke consisting of an initial intracranial bleed followed by delayed cerebral ischemia (DCI). Current therapeutic strategies are… (more)

Subjects/Keywords: Ischemia; Myogenic response; Neuronal injury; Resistance arteries; Sphingosine-1-phosphate; Vascular smooth muscle; 0719

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APA (6th Edition):

Fares, J. (2015). Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74718

Chicago Manual of Style (16th Edition):

Fares, Jessica. “Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage.” 2015. Masters Thesis, University of Toronto. Accessed October 17, 2019. http://hdl.handle.net/1807/74718.

MLA Handbook (7th Edition):

Fares, Jessica. “Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage.” 2015. Web. 17 Oct 2019.

Vancouver:

Fares J. Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/1807/74718.

Council of Science Editors:

Fares J. Therapeutically Targeting the Cystic Fibrosis Transmembrane Conductance Regulator in Cerebrovascular Dysfunction associated with Subarachnoid Hemorrhage. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/74718


University of Kentucky

76. Lutshumba, Jenny. PROTECTION FROM AORTIC ANEURYSM BY BMAL1 DELETION FROM SMOOTH MUSCLE CELLS.

Degree: 2017, University of Kentucky

 Abdominal aortic aneurysm (AAA) is a devastating condition that occurs primarily among older people with high mortality when a rupture occurs. Currently there is no… (more)

Subjects/Keywords: Aortic Aneurysm; Bmal1; Vascular Smooth Muscle Cells; Matrix Metalloproteinases; Tissue Inhibitor of Metalloproteinases; Cardiovascular Diseases

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APA (6th Edition):

Lutshumba, J. (2017). PROTECTION FROM AORTIC ANEURYSM BY BMAL1 DELETION FROM SMOOTH MUSCLE CELLS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/physiology_etds/32

Chicago Manual of Style (16th Edition):

Lutshumba, Jenny. “PROTECTION FROM AORTIC ANEURYSM BY BMAL1 DELETION FROM SMOOTH MUSCLE CELLS.” 2017. Doctoral Dissertation, University of Kentucky. Accessed October 17, 2019. https://uknowledge.uky.edu/physiology_etds/32.

MLA Handbook (7th Edition):

Lutshumba, Jenny. “PROTECTION FROM AORTIC ANEURYSM BY BMAL1 DELETION FROM SMOOTH MUSCLE CELLS.” 2017. Web. 17 Oct 2019.

Vancouver:

Lutshumba J. PROTECTION FROM AORTIC ANEURYSM BY BMAL1 DELETION FROM SMOOTH MUSCLE CELLS. [Internet] [Doctoral dissertation]. University of Kentucky; 2017. [cited 2019 Oct 17]. Available from: https://uknowledge.uky.edu/physiology_etds/32.

Council of Science Editors:

Lutshumba J. PROTECTION FROM AORTIC ANEURYSM BY BMAL1 DELETION FROM SMOOTH MUSCLE CELLS. [Doctoral Dissertation]. University of Kentucky; 2017. Available from: https://uknowledge.uky.edu/physiology_etds/32


Boston University

77. Rim, Nae Gyune. Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application.

Degree: PhD, Biomedical Engineering, 2018, Boston University

 To successfully develop a functional tissue-engineered vascular patch, recapitulating the hierarchical structure of vessel is critical to mimic mechanical properties. Here, we use a cell… (more)

Subjects/Keywords: Biomedical engineering; Finite element modeling; Hydrogel; Tensile testing; Vascular smooth muscle cell

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APA (6th Edition):

Rim, N. G. (2018). Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/30707

Chicago Manual of Style (16th Edition):

Rim, Nae Gyune. “Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application.” 2018. Doctoral Dissertation, Boston University. Accessed October 17, 2019. http://hdl.handle.net/2144/30707.

MLA Handbook (7th Edition):

Rim, Nae Gyune. “Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application.” 2018. Web. 17 Oct 2019.

Vancouver:

Rim NG. Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2144/30707.

Council of Science Editors:

Rim NG. Micropatterned cell sheets as structural building blocks for biomimetic vascular patch application. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/30707


Boston University

78. Gao, Yuan Zhao. Vascular smooth muscle: a target for treatment of aging-induced aortic stiffness.

Degree: PhD, Biomedical Engineering, 2015, Boston University

 Cardiovascular disease is the leading cause of human death worldwide. Currently, the prevalence of cardiovascular disease and health care costs associated with its onset continue… (more)

Subjects/Keywords: Biomedical engineering; Src; Actin cytoskeleton; Aging; Aortic stiffness; Focal adhesions; Vascular smooth muscle

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APA (6th Edition):

Gao, Y. Z. (2015). Vascular smooth muscle: a target for treatment of aging-induced aortic stiffness. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13678

Chicago Manual of Style (16th Edition):

Gao, Yuan Zhao. “Vascular smooth muscle: a target for treatment of aging-induced aortic stiffness.” 2015. Doctoral Dissertation, Boston University. Accessed October 17, 2019. http://hdl.handle.net/2144/13678.

MLA Handbook (7th Edition):

Gao, Yuan Zhao. “Vascular smooth muscle: a target for treatment of aging-induced aortic stiffness.” 2015. Web. 17 Oct 2019.

Vancouver:

Gao YZ. Vascular smooth muscle: a target for treatment of aging-induced aortic stiffness. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2144/13678.

Council of Science Editors:

Gao YZ. Vascular smooth muscle: a target for treatment of aging-induced aortic stiffness. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13678


Dublin City University

79. Morrow, David. The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth.

Degree: School of Biotechnology, 2006, Dublin City University

Vascular Smooth Muscle Cell (SMC) fate decisions are fundamental features in the pathogenesis of vascular disease. We investigated the role of Notch 1 and 3… (more)

Subjects/Keywords: Biotechnology; Vascular Smooth Muscle Cell; Vascular Disease; Cell Growth

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APA (6th Edition):

Morrow, D. (2006). The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth. (Thesis). Dublin City University. Retrieved from http://doras.dcu.ie/17705/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morrow, David. “The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth.” 2006. Thesis, Dublin City University. Accessed October 17, 2019. http://doras.dcu.ie/17705/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morrow, David. “The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth.” 2006. Web. 17 Oct 2019.

Vancouver:

Morrow D. The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth. [Internet] [Thesis]. Dublin City University; 2006. [cited 2019 Oct 17]. Available from: http://doras.dcu.ie/17705/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morrow D. The role of the notch and hedgehog signalling pathways in vascular smooth muscle cell growth. [Thesis]. Dublin City University; 2006. Available from: http://doras.dcu.ie/17705/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

80. 姚惠琪.; Yiu, Wai-ki. The effects of supercooling and re-warming on vascular cells survival and proliferation.

Degree: PhD, 2010, University of Hong Kong

published_or_final_version

Surgery

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Lui, VCH, Cheng, SWK.

Subjects/Keywords: Cell proliferation.; Vascular smooth muscle.; Vascular endothelium.

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APA (6th Edition):

姚惠琪.; Yiu, W. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312

Chicago Manual of Style (16th Edition):

姚惠琪.; Yiu, Wai-ki. “The effects of supercooling and re-warming on vascular cells survival and proliferation.” 2010. Doctoral Dissertation, University of Hong Kong. Accessed October 17, 2019. Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312.

MLA Handbook (7th Edition):

姚惠琪.; Yiu, Wai-ki. “The effects of supercooling and re-warming on vascular cells survival and proliferation.” 2010. Web. 17 Oct 2019.

Vancouver:

姚惠琪.; Yiu W. The effects of supercooling and re-warming on vascular cells survival and proliferation. [Internet] [Doctoral dissertation]. University of Hong Kong; 2010. [cited 2019 Oct 17]. Available from: Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312.

Council of Science Editors:

姚惠琪.; Yiu W. The effects of supercooling and re-warming on vascular cells survival and proliferation. [Doctoral Dissertation]. University of Hong Kong; 2010. Available from: Yiu, W. [姚惠琪]. (2010). The effects of supercooling and re-warming on vascular cells survival and proliferation. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4423688 ; http://dx.doi.org/10.5353/th_b4423688 ; http://hdl.handle.net/10722/65312

81. Alsabeelah, Nimer Fehaid N. Vascular Calcification in Rat Cultured Smooth Muscle Cells: a Role for Nitric Oxide .

Degree: 2016, University of Hertfordshire

 The underlying inflammatory storm in renal or diabetic disease may induce expression of inducible nitric oxide synthase (iNOS). Similarly, expression of iNOS or nitric oxide… (more)

Subjects/Keywords: Vascular calcification; Nitric oxide; Inducible nitric oxide; Vascular smooth muscle cells; Runt-related transcription factor 2

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APA (6th Edition):

Alsabeelah, N. F. N. (2016). Vascular Calcification in Rat Cultured Smooth Muscle Cells: a Role for Nitric Oxide . (Thesis). University of Hertfordshire. Retrieved from http://hdl.handle.net/2299/17217

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alsabeelah, Nimer Fehaid N. “Vascular Calcification in Rat Cultured Smooth Muscle Cells: a Role for Nitric Oxide .” 2016. Thesis, University of Hertfordshire. Accessed October 17, 2019. http://hdl.handle.net/2299/17217.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alsabeelah, Nimer Fehaid N. “Vascular Calcification in Rat Cultured Smooth Muscle Cells: a Role for Nitric Oxide .” 2016. Web. 17 Oct 2019.

Vancouver:

Alsabeelah NFN. Vascular Calcification in Rat Cultured Smooth Muscle Cells: a Role for Nitric Oxide . [Internet] [Thesis]. University of Hertfordshire; 2016. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2299/17217.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alsabeelah NFN. Vascular Calcification in Rat Cultured Smooth Muscle Cells: a Role for Nitric Oxide . [Thesis]. University of Hertfordshire; 2016. Available from: http://hdl.handle.net/2299/17217

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

82. Bhattacharyya, Aparna. Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering.

Degree: 2012, University of Western Ontario

 One of the challenges in the designing of clinically-relevant vascular substitutes is our lack of understanding on how vascular smooth muscle cells (VSMCs) and vascular(more)

Subjects/Keywords: tissue engineering; vascular smooth muscle cell; vascular endothelial cell; phenotype modulation; polyurethane scaffolds; Notch signaling; Molecular, Cellular, and Tissue Engineering

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APA (6th Edition):

Bhattacharyya, A. (2012). Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bhattacharyya, Aparna. “Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering.” 2012. Thesis, University of Western Ontario. Accessed October 17, 2019. https://ir.lib.uwo.ca/etd/501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bhattacharyya, Aparna. “Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering.” 2012. Web. 17 Oct 2019.

Vancouver:

Bhattacharyya A. Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering. [Internet] [Thesis]. University of Western Ontario; 2012. [cited 2019 Oct 17]. Available from: https://ir.lib.uwo.ca/etd/501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bhattacharyya A. Co-culture of Smooth Muscle Cells and Endothelial Cells on Porous 3D Polyurethane Scaffolds for Vascular Tissue Engineering. [Thesis]. University of Western Ontario; 2012. Available from: https://ir.lib.uwo.ca/etd/501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

83. Chavkin, Nicholas Walter. Elevated Phosphate-Induced Cell Signaling through Phosphate Transporter PiT-1 in Vascular Smooth Muscle Cells.

Degree: PhD, 2017, University of Washington

Vascular calcification (VC) is prevalent in chronic kidney disease and elevated serum inorganic phosphate (Pi) is a recognized risk factor. The type III sodium-dependent phosphate… (more)

Subjects/Keywords: Chronic Kidney Disease; Inorganic Phosphate; RapGEF1; SLC20A1; Vascular Calcification; Vascular Smooth Muscle Cells; Biomedical engineering; Cellular biology; bioengineering

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APA (6th Edition):

Chavkin, N. W. (2017). Elevated Phosphate-Induced Cell Signaling through Phosphate Transporter PiT-1 in Vascular Smooth Muscle Cells. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/38067

Chicago Manual of Style (16th Edition):

Chavkin, Nicholas Walter. “Elevated Phosphate-Induced Cell Signaling through Phosphate Transporter PiT-1 in Vascular Smooth Muscle Cells.” 2017. Doctoral Dissertation, University of Washington. Accessed October 17, 2019. http://hdl.handle.net/1773/38067.

MLA Handbook (7th Edition):

Chavkin, Nicholas Walter. “Elevated Phosphate-Induced Cell Signaling through Phosphate Transporter PiT-1 in Vascular Smooth Muscle Cells.” 2017. Web. 17 Oct 2019.

Vancouver:

Chavkin NW. Elevated Phosphate-Induced Cell Signaling through Phosphate Transporter PiT-1 in Vascular Smooth Muscle Cells. [Internet] [Doctoral dissertation]. University of Washington; 2017. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/1773/38067.

Council of Science Editors:

Chavkin NW. Elevated Phosphate-Induced Cell Signaling through Phosphate Transporter PiT-1 in Vascular Smooth Muscle Cells. [Doctoral Dissertation]. University of Washington; 2017. Available from: http://hdl.handle.net/1773/38067

84. Padget, Rachel Lee. The Effect of Hemodynamic Force on the Maturation of Blood Vessels during Embryogenesis.

Degree: MSin Biology, Biology, 2017, Missouri State University

  Throughout embryonic development, blood vessels are derived from endothelial cells by way of vasculogenesis. During angiogenesis, vessels remodel to form a hierarchy of large-diameter… (more)

Subjects/Keywords: Vascular smooth muscle cells; vessel maturation; vascular development; angiogenesis; hemodynamic force; Biophysics; Developmental Biology; Laboratory and Basic Science Research

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APA (6th Edition):

Padget, R. L. (2017). The Effect of Hemodynamic Force on the Maturation of Blood Vessels during Embryogenesis. (Masters Thesis). Missouri State University. Retrieved from https://bearworks.missouristate.edu/theses/3116

Chicago Manual of Style (16th Edition):

Padget, Rachel Lee. “The Effect of Hemodynamic Force on the Maturation of Blood Vessels during Embryogenesis.” 2017. Masters Thesis, Missouri State University. Accessed October 17, 2019. https://bearworks.missouristate.edu/theses/3116.

MLA Handbook (7th Edition):

Padget, Rachel Lee. “The Effect of Hemodynamic Force on the Maturation of Blood Vessels during Embryogenesis.” 2017. Web. 17 Oct 2019.

Vancouver:

Padget RL. The Effect of Hemodynamic Force on the Maturation of Blood Vessels during Embryogenesis. [Internet] [Masters thesis]. Missouri State University; 2017. [cited 2019 Oct 17]. Available from: https://bearworks.missouristate.edu/theses/3116.

Council of Science Editors:

Padget RL. The Effect of Hemodynamic Force on the Maturation of Blood Vessels during Embryogenesis. [Masters Thesis]. Missouri State University; 2017. Available from: https://bearworks.missouristate.edu/theses/3116


Boston University

85. Glazyrine, Vassili. The role of vascular endothelial growth factor in heart failure with preserved ejection fraction.

Degree: MS, Medical Sciences, 2015, Boston University

 To this day heart failure with preserved ejection fraction (HFpEF) remains a poorly understood malady. Half of all heart failure (HF) cases are HFpEF, and… (more)

Subjects/Keywords: Medicine; HFpEF; Adult rat ventricular myocyte; Heart failure; Preserved ejection fraction; Vascular endothelial growth factor; Vascular smooth muscle cells

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APA (6th Edition):

Glazyrine, V. (2015). The role of vascular endothelial growth factor in heart failure with preserved ejection fraction. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16220

Chicago Manual of Style (16th Edition):

Glazyrine, Vassili. “The role of vascular endothelial growth factor in heart failure with preserved ejection fraction.” 2015. Masters Thesis, Boston University. Accessed October 17, 2019. http://hdl.handle.net/2144/16220.

MLA Handbook (7th Edition):

Glazyrine, Vassili. “The role of vascular endothelial growth factor in heart failure with preserved ejection fraction.” 2015. Web. 17 Oct 2019.

Vancouver:

Glazyrine V. The role of vascular endothelial growth factor in heart failure with preserved ejection fraction. [Internet] [Masters thesis]. Boston University; 2015. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/2144/16220.

Council of Science Editors:

Glazyrine V. The role of vascular endothelial growth factor in heart failure with preserved ejection fraction. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16220

86. Zeadin, Melec. INVESTIGATING THE ROLE OF LEPTIN AND GSK-3 IN THE OSTEOGENIC DIFFERENTIATION OF VASCULAR SMOOTH MUSCLE CELLS.

Degree: PhD, 2014, McMaster University

Obesity is a major risk factor for insulin resistance, type 2 diabetes, cardiovascular disease (CVD), and vascular calcification. Vascular calcification is correlated with advanced CVD… (more)

Subjects/Keywords: leptin; vascular calcification; vascular smooth muscle cells

…90 4.3.2 Isolation of Vascular Smooth Muscle Cells… …VSMC(s) vascular smooth muscle cell(s) WOSCOPS West of Scotland Coronary… …of elastic tissue and vascular smooth muscle cells (VSMCs) which can synthesize… …Smooth Muscle Cells ................................................................ 73 3.4.5… …Promotes Osteoblast Differentiation and Mineralization of Primary Cultures of Vascular Smooth… 

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APA (6th Edition):

Zeadin, M. (2014). INVESTIGATING THE ROLE OF LEPTIN AND GSK-3 IN THE OSTEOGENIC DIFFERENTIATION OF VASCULAR SMOOTH MUSCLE CELLS. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/16509

Chicago Manual of Style (16th Edition):

Zeadin, Melec. “INVESTIGATING THE ROLE OF LEPTIN AND GSK-3 IN THE OSTEOGENIC DIFFERENTIATION OF VASCULAR SMOOTH MUSCLE CELLS.” 2014. Doctoral Dissertation, McMaster University. Accessed October 17, 2019. http://hdl.handle.net/11375/16509.

MLA Handbook (7th Edition):

Zeadin, Melec. “INVESTIGATING THE ROLE OF LEPTIN AND GSK-3 IN THE OSTEOGENIC DIFFERENTIATION OF VASCULAR SMOOTH MUSCLE CELLS.” 2014. Web. 17 Oct 2019.

Vancouver:

Zeadin M. INVESTIGATING THE ROLE OF LEPTIN AND GSK-3 IN THE OSTEOGENIC DIFFERENTIATION OF VASCULAR SMOOTH MUSCLE CELLS. [Internet] [Doctoral dissertation]. McMaster University; 2014. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/11375/16509.

Council of Science Editors:

Zeadin M. INVESTIGATING THE ROLE OF LEPTIN AND GSK-3 IN THE OSTEOGENIC DIFFERENTIATION OF VASCULAR SMOOTH MUSCLE CELLS. [Doctoral Dissertation]. McMaster University; 2014. Available from: http://hdl.handle.net/11375/16509


University of Minnesota

87. Hald, Eric. Microfabrication Approaches for Understanding the Role of Vascular Mechanics in Progressive Diseases.

Degree: PhD, Biomedical Engineering, 2016, University of Minnesota

Vascular disease is a common cause of death that typically results from long-term alteration of vessel structure and function. The underlying mechanisms that lead to… (more)

Subjects/Keywords: Alzheimer's disease; growth and remodeling; in vitro disease model; microfabrication; vascular mechanics; vascular smooth muscle cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hald, E. (2016). Microfabrication Approaches for Understanding the Role of Vascular Mechanics in Progressive Diseases. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191352

Chicago Manual of Style (16th Edition):

Hald, Eric. “Microfabrication Approaches for Understanding the Role of Vascular Mechanics in Progressive Diseases.” 2016. Doctoral Dissertation, University of Minnesota. Accessed October 17, 2019. http://hdl.handle.net/11299/191352.

MLA Handbook (7th Edition):

Hald, Eric. “Microfabrication Approaches for Understanding the Role of Vascular Mechanics in Progressive Diseases.” 2016. Web. 17 Oct 2019.

Vancouver:

Hald E. Microfabrication Approaches for Understanding the Role of Vascular Mechanics in Progressive Diseases. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2019 Oct 17]. Available from: http://hdl.handle.net/11299/191352.

Council of Science Editors:

Hald E. Microfabrication Approaches for Understanding the Role of Vascular Mechanics in Progressive Diseases. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/191352


University of Pennsylvania

88. Yu, Christopher. Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness.

Degree: 2017, University of Pennsylvania

 Arterial stiffness is a risk factor for several cardiometabolic diseases and is caused by pathological remodeling of the vascular extracellular matrix (ECM). Vascular smooth muscle(more)

Subjects/Keywords: Extracellular matrix stiffness; Long non-coding RNAs; Transcriptome; Vascular smooth muscle cells; Vascular stiffness; Cell Biology; Genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, C. (2017). Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Christopher. “Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness.” 2017. Thesis, University of Pennsylvania. Accessed October 17, 2019. https://repository.upenn.edu/edissertations/2710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Christopher. “Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness.” 2017. Web. 17 Oct 2019.

Vancouver:

Yu C. Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2019 Oct 17]. Available from: https://repository.upenn.edu/edissertations/2710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu C. Regulation Of The Human Vascular Smooth Muscle Cell Transcriptome By Extracellular Matrix Stiffness. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

89. Roszkowska, Monika. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.

Degree: Docteur es, Biochimie, 2018, Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne)

Chez les patients atteints d'athérosclérose, les calcifications vasculaires sont une caracteristique des plaques d'athérome. Elles résultent de la trans-différenciation des cellules musculaires lisses (CMLs) en… (more)

Subjects/Keywords: Cellule musculaire lisse; Phosphatase alcaline; Calcification vasculaire; Chondrocyte; Vascular smooth muscle cell; Tissue-nonspecific alkaline phosphatase; Vascular calcification; Chondrocyte; 572

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roszkowska, M. (2018). Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. (Doctoral Dissertation). Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne). Retrieved from http://www.theses.fr/2018LYSE1059

Chicago Manual of Style (16th Edition):

Roszkowska, Monika. “Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.” 2018. Doctoral Dissertation, Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne). Accessed October 17, 2019. http://www.theses.fr/2018LYSE1059.

MLA Handbook (7th Edition):

Roszkowska, Monika. “Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.” 2018. Web. 17 Oct 2019.

Vancouver:

Roszkowska M. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. [Internet] [Doctoral dissertation]. Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne); 2018. [cited 2019 Oct 17]. Available from: http://www.theses.fr/2018LYSE1059.

Council of Science Editors:

Roszkowska M. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. [Doctoral Dissertation]. Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne); 2018. Available from: http://www.theses.fr/2018LYSE1059


Loma Linda University

90. Hubbell, Margaret C. The Role of VEGF and Smooth Muscle Phenotype in Hypoxic Remodeling of Ovine Carotid and Cerebral Arteries.

Degree: PhD, Basic Sciences, 2017, Loma Linda University

  Arteries are a dynamic tissue with multiple cell types that incorporate systemic and local factors to maintain homeostasis. Hypoxia stimulates capillary angiogenesis to effectively… (more)

Subjects/Keywords: Medical Biochemistry; Medical Sciences; Medicine and Health Sciences; Hypoxia-Ischemia, Brain - Physiopathology; Endothelium, Vascular; Muscle, Smooth, Vascular; Cardiovascular System - Physiopathology; Fetal Hypoxia - Physiopathology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hubbell, M. C. (2017). The Role of VEGF and Smooth Muscle Phenotype in Hypoxic Remodeling of Ovine Carotid and Cerebral Arteries. (Doctoral Dissertation). Loma Linda University. Retrieved from https://scholarsrepository.llu.edu/etd/464

Chicago Manual of Style (16th Edition):

Hubbell, Margaret C. “The Role of VEGF and Smooth Muscle Phenotype in Hypoxic Remodeling of Ovine Carotid and Cerebral Arteries.” 2017. Doctoral Dissertation, Loma Linda University. Accessed October 17, 2019. https://scholarsrepository.llu.edu/etd/464.

MLA Handbook (7th Edition):

Hubbell, Margaret C. “The Role of VEGF and Smooth Muscle Phenotype in Hypoxic Remodeling of Ovine Carotid and Cerebral Arteries.” 2017. Web. 17 Oct 2019.

Vancouver:

Hubbell MC. The Role of VEGF and Smooth Muscle Phenotype in Hypoxic Remodeling of Ovine Carotid and Cerebral Arteries. [Internet] [Doctoral dissertation]. Loma Linda University; 2017. [cited 2019 Oct 17]. Available from: https://scholarsrepository.llu.edu/etd/464.

Council of Science Editors:

Hubbell MC. The Role of VEGF and Smooth Muscle Phenotype in Hypoxic Remodeling of Ovine Carotid and Cerebral Arteries. [Doctoral Dissertation]. Loma Linda University; 2017. Available from: https://scholarsrepository.llu.edu/etd/464

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