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You searched for subject:(ubiquitin). Showing records 1 – 30 of 630 total matches.

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University of Minnesota

1. Randles, Leah Ann. Defining how the 26S proteasome recognizes ubiquitinated substrates.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2012, University of Minnesota

 Regulated protein degradation in eukaryotes is performed predominantly by the ubiquitin-proteasome pathway. Prior to their degradation by the 26S proteasome, protein substrates become covalently modified… (more)

Subjects/Keywords: Proteasome; Ubiquitin; Ubiquitin Receptor

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APA (6th Edition):

Randles, L. A. (2012). Defining how the 26S proteasome recognizes ubiquitinated substrates. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/162241

Chicago Manual of Style (16th Edition):

Randles, Leah Ann. “Defining how the 26S proteasome recognizes ubiquitinated substrates.” 2012. Doctoral Dissertation, University of Minnesota. Accessed March 20, 2019. http://hdl.handle.net/11299/162241.

MLA Handbook (7th Edition):

Randles, Leah Ann. “Defining how the 26S proteasome recognizes ubiquitinated substrates.” 2012. Web. 20 Mar 2019.

Vancouver:

Randles LA. Defining how the 26S proteasome recognizes ubiquitinated substrates. [Internet] [Doctoral dissertation]. University of Minnesota; 2012. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/11299/162241.

Council of Science Editors:

Randles LA. Defining how the 26S proteasome recognizes ubiquitinated substrates. [Doctoral Dissertation]. University of Minnesota; 2012. Available from: http://hdl.handle.net/11299/162241


University of Dundee

2. Leidecker, Orsolya. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.

Degree: PhD, 2012, University of Dundee

 NEDD8 modification of proteins is extensively studied in the recent years, and the ubiquitin-like molecule has been shown to be involved in numerous signalling pathways.… (more)

Subjects/Keywords: nedd8; ubiquitin

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APA (6th Edition):

Leidecker, O. (2012). Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf

Chicago Manual of Style (16th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Doctoral Dissertation, University of Dundee. Accessed March 20, 2019. http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf.

MLA Handbook (7th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Web. 20 Mar 2019.

Vancouver:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf.

Council of Science Editors:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Doctoral Dissertation]. University of Dundee; 2012. Available from: http://hdl.handle.net/10588/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf


Penn State University

3. Davidshofer, Kristine C. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.

Degree: PhD, Physiology, 2008, Penn State University

Ubiquitin is a highly conserved eukaryotic protein of 76 amino acids that is added post-translationally to other proteins or to itself by a hierarchical cascade… (more)

Subjects/Keywords: RING domain; ubiquitin; ubiquitin conjugating enzyme

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APA (6th Edition):

Davidshofer, K. C. (2008). ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8500

Chicago Manual of Style (16th Edition):

Davidshofer, Kristine C. “ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.” 2008. Doctoral Dissertation, Penn State University. Accessed March 20, 2019. https://etda.libraries.psu.edu/catalog/8500.

MLA Handbook (7th Edition):

Davidshofer, Kristine C. “ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.” 2008. Web. 20 Mar 2019.

Vancouver:

Davidshofer KC. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2019 Mar 20]. Available from: https://etda.libraries.psu.edu/catalog/8500.

Council of Science Editors:

Davidshofer KC. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8500


University of Hong Kong

4. 王琳; Wang, Lin. The role of MDM2 in hepatic lipid metabolism.

Degree: Master of Medical Sciences, 2016, University of Hong Kong

 Background and objective: Liver is a core organ in regulation of lipid homeostasis, which is vital for life and health. Obesity is closely associated with… (more)

Subjects/Keywords: Ubiquitin; Lipids - Metabolism

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APA (6th Edition):

王琳; Wang, L. (2016). The role of MDM2 in hepatic lipid metabolism. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/236279

Chicago Manual of Style (16th Edition):

王琳; Wang, Lin. “The role of MDM2 in hepatic lipid metabolism.” 2016. Masters Thesis, University of Hong Kong. Accessed March 20, 2019. http://hdl.handle.net/10722/236279.

MLA Handbook (7th Edition):

王琳; Wang, Lin. “The role of MDM2 in hepatic lipid metabolism.” 2016. Web. 20 Mar 2019.

Vancouver:

王琳; Wang L. The role of MDM2 in hepatic lipid metabolism. [Internet] [Masters thesis]. University of Hong Kong; 2016. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/10722/236279.

Council of Science Editors:

王琳; Wang L. The role of MDM2 in hepatic lipid metabolism. [Masters Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/236279


University of Hong Kong

5. 成云; Cheng, Yun. β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.

Degree: PhD, 2016, University of Hong Kong

 CREB-H is an endoplasmic reticulum-tethered bZIP transcription factor, which critically regulates lipid homeostasis and gluconeogenesis in the liver. CREB-H is proteolytically activated by regulated intramembrane… (more)

Subjects/Keywords: Transcription factors; Ubiquitin

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APA (6th Edition):

成云; Cheng, Y. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Doctoral Dissertation). University of Hong Kong. Retrieved from Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862

Chicago Manual of Style (16th Edition):

成云; Cheng, Yun. “β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.” 2016. Doctoral Dissertation, University of Hong Kong. Accessed March 20, 2019. Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862.

MLA Handbook (7th Edition):

成云; Cheng, Yun. “β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.” 2016. Web. 20 Mar 2019.

Vancouver:

成云; Cheng Y. β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. [Internet] [Doctoral dissertation]. University of Hong Kong; 2016. [cited 2019 Mar 20]. Available from: Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862.

Council of Science Editors:

成云; Cheng Y. β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. [Doctoral Dissertation]. University of Hong Kong; 2016. Available from: Cheng, Y. [成云]. (2016). β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5816255. ; http://dx.doi.org/10.5353/th_b5816255 ; http://hdl.handle.net/10722/237862


University of Hong Kong

6. Li, Tao. Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1.

Degree: M. Phil., 2016, University of Hong Kong

DNA methylation is a crucial epigenetic modification and functions as a key factor in controlling gene expression and mammalian development. The DNA methyltransferase DNMT1 is… (more)

Subjects/Keywords: Ubiquitin; DNA - Methylation

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APA (6th Edition):

Li, T. (2016). Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1. (Masters Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/240653

Chicago Manual of Style (16th Edition):

Li, Tao. “Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1.” 2016. Masters Thesis, University of Hong Kong. Accessed March 20, 2019. http://hdl.handle.net/10722/240653.

MLA Handbook (7th Edition):

Li, Tao. “Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1.” 2016. Web. 20 Mar 2019.

Vancouver:

Li T. Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1. [Internet] [Masters thesis]. University of Hong Kong; 2016. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/10722/240653.

Council of Science Editors:

Li T. Structural and mechanistic insights into UHRF1-coordinated maintenance DNA methylation by DNMT1. [Masters Thesis]. University of Hong Kong; 2016. Available from: http://hdl.handle.net/10722/240653


University of Hong Kong

7. Ruan, Yafei. Investigating the regulation of UHRF1 in cell cycle.

Degree: Master of Medical Sciences, 2015, University of Hong Kong

UHRF1 is a multi-domain protein with multiple functions in mammalian cells including maintenance of DNA methylation, histone modification, DNA damage et al. UHRF1 itself also… (more)

Subjects/Keywords: Cell cycle; Ubiquitin

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APA (6th Edition):

Ruan, Y. (2015). Investigating the regulation of UHRF1 in cell cycle. (Masters Thesis). University of Hong Kong. Retrieved from Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484

Chicago Manual of Style (16th Edition):

Ruan, Yafei. “Investigating the regulation of UHRF1 in cell cycle.” 2015. Masters Thesis, University of Hong Kong. Accessed March 20, 2019. Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484.

MLA Handbook (7th Edition):

Ruan, Yafei. “Investigating the regulation of UHRF1 in cell cycle.” 2015. Web. 20 Mar 2019.

Vancouver:

Ruan Y. Investigating the regulation of UHRF1 in cell cycle. [Internet] [Masters thesis]. University of Hong Kong; 2015. [cited 2019 Mar 20]. Available from: Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484.

Council of Science Editors:

Ruan Y. Investigating the regulation of UHRF1 in cell cycle. [Masters Thesis]. University of Hong Kong; 2015. Available from: Ruan, Y. [阮雅菲]. (2015). Investigating the regulation of UHRF1 in cell cycle. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5659412 ; http://hdl.handle.net/10722/221484


University of Hong Kong

8. Tucker, Wesley Owen. Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast.

Degree: PhD, 2013, University of Hong Kong

DNA aptamers have been studied since their inception in 1990, but have only targeted membrane and serum proteins in therapeutics. Their potential as inhibitors of… (more)

Subjects/Keywords: Ubiquitin; Osteoblasts; Oligonucleotides

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APA (6th Edition):

Tucker, W. O. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640

Chicago Manual of Style (16th Edition):

Tucker, Wesley Owen. “Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed March 20, 2019. Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640.

MLA Handbook (7th Edition):

Tucker, Wesley Owen. “Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast.” 2013. Web. 20 Mar 2019.

Vancouver:

Tucker WO. Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Mar 20]. Available from: Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640.

Council of Science Editors:

Tucker WO. Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Tucker, W. O.. (2013). Towards specific DNA aptamers which bind and inhibit WWP1 HECT ubiquitin ligase in the osteoblast. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108666 ; http://dx.doi.org/10.5353/th_b5108666 ; http://hdl.handle.net/10722/205640


Penn State University

9. Ellis, Christopher Ross. Simulation studies of the glycosylation code.

Degree: PhD, Chemistry, 2014, Penn State University

 This thesis reports computational studies that investigate the impact of protein- carbohydrate and protein-protein interactions upon protein structure and folding. The core of this thesis,… (more)

Subjects/Keywords: glycosylation; toluene; ubiquitin

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APA (6th Edition):

Ellis, C. R. (2014). Simulation studies of the glycosylation code. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/22562

Chicago Manual of Style (16th Edition):

Ellis, Christopher Ross. “Simulation studies of the glycosylation code.” 2014. Doctoral Dissertation, Penn State University. Accessed March 20, 2019. https://etda.libraries.psu.edu/catalog/22562.

MLA Handbook (7th Edition):

Ellis, Christopher Ross. “Simulation studies of the glycosylation code.” 2014. Web. 20 Mar 2019.

Vancouver:

Ellis CR. Simulation studies of the glycosylation code. [Internet] [Doctoral dissertation]. Penn State University; 2014. [cited 2019 Mar 20]. Available from: https://etda.libraries.psu.edu/catalog/22562.

Council of Science Editors:

Ellis CR. Simulation studies of the glycosylation code. [Doctoral Dissertation]. Penn State University; 2014. Available from: https://etda.libraries.psu.edu/catalog/22562


University of California – San Diego

10. Gonzales, Frankie Robert. Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System.

Degree: Chemistry, 2017, University of California – San Diego

 Protein homeostasis in is critical to maintain cell health and viability. Protein homeostasis can be divided into two major categories: protein synthesis, and protein degradation.… (more)

Subjects/Keywords: Biochemistry; Proteasome; Ubiquitin

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APA (6th Edition):

Gonzales, F. R. (2017). Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/6958j596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonzales, Frankie Robert. “Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System.” 2017. Thesis, University of California – San Diego. Accessed March 20, 2019. http://www.escholarship.org/uc/item/6958j596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonzales, Frankie Robert. “Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System.” 2017. Web. 20 Mar 2019.

Vancouver:

Gonzales FR. Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2019 Mar 20]. Available from: http://www.escholarship.org/uc/item/6958j596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonzales FR. Characterizing Postranslational Regulatory Mechanisms of the Ubiquitin Proteasome System. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/6958j596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

11. Leidecker, Orsolya. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.

Degree: PhD, 2012, University of Dundee

 NEDD8 modification of proteins is extensively studied in the recent years, and the ubiquitin-like molecule has been shown to be involved in numerous signalling pathways.… (more)

Subjects/Keywords: 572; nedd8; ubiquitin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Leidecker, O. (2012). Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873

Chicago Manual of Style (16th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Doctoral Dissertation, University of Dundee. Accessed March 20, 2019. https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873.

MLA Handbook (7th Edition):

Leidecker, Orsolya. “Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions.” 2012. Web. 20 Mar 2019.

Vancouver:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Internet] [Doctoral dissertation]. University of Dundee; 2012. [cited 2019 Mar 20]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873.

Council of Science Editors:

Leidecker O. Investigating the response of the NEDD8 ubiquitin-like molecule to diverse stress conditions. [Doctoral Dissertation]. University of Dundee; 2012. Available from: https://discovery.dundee.ac.uk/en/studentTheses/f19d9441-9b48-448d-aa88-6dd2e7f4b5bf ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.578873


University of Nevada – Las Vegas

12. Ibarra, Rebeca Lea. The San1 Ubiquitin Ligase Functions Preferentially with Ubiquitin-Conjugating Enzyme Ubc1 During Protein Quality Control.

Degree: MS, Chemistry and Biochemistry, 2016, University of Nevada – Las Vegas

  Protein quality control (PQC) is a critical process wherein misfolded or damaged proteins are cleared from the cell to maintain protein homeostasis. In eukaryotic… (more)

Subjects/Keywords: Protein Degradation; Protein Misfolding; Protein Quality Control; Ubiquitin; Ubiquitin-Conjugating Enzyme; Ubiquitin Ligase; Biochemistry; Chemistry

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APA (6th Edition):

Ibarra, R. L. (2016). The San1 Ubiquitin Ligase Functions Preferentially with Ubiquitin-Conjugating Enzyme Ubc1 During Protein Quality Control. (Masters Thesis). University of Nevada – Las Vegas. Retrieved from https://digitalscholarship.unlv.edu/thesesdissertations/2783

Chicago Manual of Style (16th Edition):

Ibarra, Rebeca Lea. “The San1 Ubiquitin Ligase Functions Preferentially with Ubiquitin-Conjugating Enzyme Ubc1 During Protein Quality Control.” 2016. Masters Thesis, University of Nevada – Las Vegas. Accessed March 20, 2019. https://digitalscholarship.unlv.edu/thesesdissertations/2783.

MLA Handbook (7th Edition):

Ibarra, Rebeca Lea. “The San1 Ubiquitin Ligase Functions Preferentially with Ubiquitin-Conjugating Enzyme Ubc1 During Protein Quality Control.” 2016. Web. 20 Mar 2019.

Vancouver:

Ibarra RL. The San1 Ubiquitin Ligase Functions Preferentially with Ubiquitin-Conjugating Enzyme Ubc1 During Protein Quality Control. [Internet] [Masters thesis]. University of Nevada – Las Vegas; 2016. [cited 2019 Mar 20]. Available from: https://digitalscholarship.unlv.edu/thesesdissertations/2783.

Council of Science Editors:

Ibarra RL. The San1 Ubiquitin Ligase Functions Preferentially with Ubiquitin-Conjugating Enzyme Ubc1 During Protein Quality Control. [Masters Thesis]. University of Nevada – Las Vegas; 2016. Available from: https://digitalscholarship.unlv.edu/thesesdissertations/2783


University of Alberta

13. Mottet, Kelly. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.

Degree: MS, Department of Medical Microbiology and Immunology, 2010, University of Alberta

 The significance of poxvirus manipulation of the host ubiquitin proteasome system has become increasingly apparent. Ubiquitin is post-translationally added to target proteins by a highly… (more)

Subjects/Keywords: ligase; ubiquitination; proteasome; ubiquitin; poxvirus

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APA (6th Edition):

Mottet, K. (2010). The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/zp38wf105

Chicago Manual of Style (16th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Masters Thesis, University of Alberta. Accessed March 20, 2019. https://era.library.ualberta.ca/files/zp38wf105.

MLA Handbook (7th Edition):

Mottet, Kelly. “The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system.” 2010. Web. 20 Mar 2019.

Vancouver:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2019 Mar 20]. Available from: https://era.library.ualberta.ca/files/zp38wf105.

Council of Science Editors:

Mottet K. The poxvirus ubiquitin ligase p28 manipulates the ubiquitin proteasome system. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/zp38wf105


University of Adelaide

14. Jolly, Lachlan. The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells.

Degree: 2010, University of Adelaide

 Neural Progenitor Cells (NPCs) are the primordial cells of central nervous system (CNS). Understanding how they are regulated benefits our knowledge of normal development, the… (more)

Subjects/Keywords: neurogenesis; ubiquitin; embryonic stem cell

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APA (6th Edition):

Jolly, L. (2010). The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/65477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jolly, Lachlan. “The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells.” 2010. Thesis, University of Adelaide. Accessed March 20, 2019. http://hdl.handle.net/2440/65477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jolly, Lachlan. “The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells.” 2010. Web. 20 Mar 2019.

Vancouver:

Jolly L. The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells. [Internet] [Thesis]. University of Adelaide; 2010. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/2440/65477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jolly L. The deubiquitylating enzyme USP9X promotes the polarity and self-renewal of neural progenitor cells. [Thesis]. University of Adelaide; 2010. Available from: http://hdl.handle.net/2440/65477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

15. Khut, Poon-Yu. Structure function analysis of the deubiquitylating enzyme Fam.

Degree: 2006, University of Adelaide

 The ubiquitin pathway is a highly conserved post-translational modification system best characterised for its roles in protein degradation and intracellular trafficking and is involved in… (more)

Subjects/Keywords: ubiquitin; proteins

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APA (6th Edition):

Khut, P. (2006). Structure function analysis of the deubiquitylating enzyme Fam. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/59436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Khut, Poon-Yu. “Structure function analysis of the deubiquitylating enzyme Fam.” 2006. Thesis, University of Adelaide. Accessed March 20, 2019. http://hdl.handle.net/2440/59436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Khut, Poon-Yu. “Structure function analysis of the deubiquitylating enzyme Fam.” 2006. Web. 20 Mar 2019.

Vancouver:

Khut P. Structure function analysis of the deubiquitylating enzyme Fam. [Internet] [Thesis]. University of Adelaide; 2006. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/2440/59436.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Khut P. Structure function analysis of the deubiquitylating enzyme Fam. [Thesis]. University of Adelaide; 2006. Available from: http://hdl.handle.net/2440/59436

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ruhr Universität Bochum

16. Nöpel-Dünnebacke, Stefanie. Identifikation und Lokalisation peroxisomaler Proteine des Menschen.

Degree: 2010, Ruhr Universität Bochum

 Problem Weitere Aufklärung der in Teilschritten ungeklärten Peroxisomenbiogenese durch Lokalisation von vierputativ peroxisomalen Proteinen in humanen Fibroblasten. Methode Immunfluoreszenzanalysen der GF-Fusionsproteine in humanen Fibroblasten und… (more)

Subjects/Keywords: Peroxisom; Peroxisom / Biogenese; Ubiquitin; Fibroblast

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APA (6th Edition):

Nöpel-Dünnebacke, S. (2010). Identifikation und Lokalisation peroxisomaler Proteine des Menschen. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nöpel-Dünnebacke, Stefanie. “Identifikation und Lokalisation peroxisomaler Proteine des Menschen.” 2010. Thesis, Ruhr Universität Bochum. Accessed March 20, 2019. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nöpel-Dünnebacke, Stefanie. “Identifikation und Lokalisation peroxisomaler Proteine des Menschen.” 2010. Web. 20 Mar 2019.

Vancouver:

Nöpel-Dünnebacke S. Identifikation und Lokalisation peroxisomaler Proteine des Menschen. [Internet] [Thesis]. Ruhr Universität Bochum; 2010. [cited 2019 Mar 20]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nöpel-Dünnebacke S. Identifikation und Lokalisation peroxisomaler Proteine des Menschen. [Thesis]. Ruhr Universität Bochum; 2010. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-29514

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Nakamura, Kasumi. Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果.

Degree: 博士(医学), 2016, Hamamatsu University School of Medicine / 浜松医科大学

The ubiquitin–proteasome pathway plays an important role in regulating apoptosis and the cell cycle. Recently, proteasome inhibitors have been shown to have antitumor effects and… (more)

Subjects/Keywords: ubiquitin; flavonoid; proteasome; inhibitor

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APA (6th Edition):

Nakamura, K. (2016). Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果. (Thesis). Hamamatsu University School of Medicine / 浜松医科大学. Retrieved from http://hdl.handle.net/10271/3143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakamura, Kasumi. “Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果.” 2016. Thesis, Hamamatsu University School of Medicine / 浜松医科大学. Accessed March 20, 2019. http://hdl.handle.net/10271/3143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakamura, Kasumi. “Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果.” 2016. Web. 20 Mar 2019.

Vancouver:

Nakamura K. Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果. [Internet] [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2016. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/10271/3143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakamura K. Effects of hydroxy groups in the A-ring on the anti-proteasome activity of flavone : フラボンの抗プロテアソーム活性におけるA環のヒドロキシル基の効果. [Thesis]. Hamamatsu University School of Medicine / 浜松医科大学; 2016. Available from: http://hdl.handle.net/10271/3143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

18. 胡晓斌; Hu, Xiaobin. The role of Uhrf1 in development and tumorigenesis.

Degree: PhD, 2014, University of Hong Kong

published_or_final_version

Biochemistry

Doctoral

Doctor of Philosophy

Subjects/Keywords: Ubiquitin; Carcinogenesis

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APA (6th Edition):

胡晓斌; Hu, X. (2014). The role of Uhrf1 in development and tumorigenesis. (Doctoral Dissertation). University of Hong Kong. Retrieved from Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205

Chicago Manual of Style (16th Edition):

胡晓斌; Hu, Xiaobin. “The role of Uhrf1 in development and tumorigenesis.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed March 20, 2019. Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205.

MLA Handbook (7th Edition):

胡晓斌; Hu, Xiaobin. “The role of Uhrf1 in development and tumorigenesis.” 2014. Web. 20 Mar 2019.

Vancouver:

胡晓斌; Hu X. The role of Uhrf1 in development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2019 Mar 20]. Available from: Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205.

Council of Science Editors:

胡晓斌; Hu X. The role of Uhrf1 in development and tumorigenesis. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: Hu, X. [胡晓斌]. (2014). The role of Uhrf1 in development and tumorigenesis. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5736666 ; http://hdl.handle.net/10722/225205


University of Oxford

19. Tan, Ka-Liong. Translational relevance of AIPL1 and NUB1 in cancer.

Degree: PhD, 2017, University of Oxford

 <b>Background:</b> Aryl Hydrocarbon Receptor Interacting Protein-Like 1 (AIPL1) interacts with NUB1 and restricts the entry of NUB1 protein into the nucleus. The interferon-induced NEDD8 ultimate… (more)

Subjects/Keywords: Breast – Cancer; ubiquitin; Neddylation; chaperone

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APA (6th Edition):

Tan, K. (2017). Translational relevance of AIPL1 and NUB1 in cancer. (Doctoral Dissertation). University of Oxford. Retrieved from https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085

Chicago Manual of Style (16th Edition):

Tan, Ka-Liong. “Translational relevance of AIPL1 and NUB1 in cancer.” 2017. Doctoral Dissertation, University of Oxford. Accessed March 20, 2019. https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085.

MLA Handbook (7th Edition):

Tan, Ka-Liong. “Translational relevance of AIPL1 and NUB1 in cancer.” 2017. Web. 20 Mar 2019.

Vancouver:

Tan K. Translational relevance of AIPL1 and NUB1 in cancer. [Internet] [Doctoral dissertation]. University of Oxford; 2017. [cited 2019 Mar 20]. Available from: https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085.

Council of Science Editors:

Tan K. Translational relevance of AIPL1 and NUB1 in cancer. [Doctoral Dissertation]. University of Oxford; 2017. Available from: https://ora.ox.ac.uk/objects/uuid:9b0e5061-bf81-4006-af9e-5018f113cb97 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.736085


University of Hong Kong

20. Tse, Ho-sum. Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1.

Degree: PhD, 2013, University of Hong Kong

Ubiquitin carboxy-terminal hydrolase L1 (UCH-L1), a protein of 223 amino acids, is a member of deubiquitinating enzymes and it is one of the most abundant… (more)

Subjects/Keywords: Parkinson's disease.; Ubiquitin.; Hydrolases.

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APA (6th Edition):

Tse, H. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505

Chicago Manual of Style (16th Edition):

Tse, Ho-sum. “Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed March 20, 2019. Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505.

MLA Handbook (7th Edition):

Tse, Ho-sum. “Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1.” 2013. Web. 20 Mar 2019.

Vancouver:

Tse H. Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Mar 20]. Available from: Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505.

Council of Science Editors:

Tse H. Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitincarboxyl-terminal hydrolase L1. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Tse, H. [謝灝森]. (2013). Investigation of the impacts of Parkinson's-disease-associated mutations (193M and S18Y) on the structure of human ubiquitin carboxyl-terminal hydrolase L1. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4979938 ; http://dx.doi.org/10.5353/th_b4979938 ; http://hdl.handle.net/10722/181505


University of Toronto

21. Gabrielli, Lisa Marie. Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases.

Degree: 2009, University of Toronto

3BP2 has been previously described as the protein mutated in the osteoporotic disorder, Cherubism. The gain of function mutation that characterizes Cherubism is the result… (more)

Subjects/Keywords: 3BP2; Ubiquitin; Nedd4; HECT; 0760

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APA (6th Edition):

Gabrielli, L. M. (2009). Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18292

Chicago Manual of Style (16th Edition):

Gabrielli, Lisa Marie. “Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases.” 2009. Masters Thesis, University of Toronto. Accessed March 20, 2019. http://hdl.handle.net/1807/18292.

MLA Handbook (7th Edition):

Gabrielli, Lisa Marie. “Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases.” 2009. Web. 20 Mar 2019.

Vancouver:

Gabrielli LM. Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1807/18292.

Council of Science Editors:

Gabrielli LM. Regulation of the 3BP2 Adaptor Protein by the Nedd4 Family of HECT E3 Ubiquitin Ligases. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18292


University of Toronto

22. Wu, Edwin. The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence.

Degree: 2013, University of Toronto

The ubiquitin-proteasome system regulates protein degradation. Although proteasomes localize in the nucleus of proliferating Saccharomyces cerevisiae, they are sequestered into cytoplasmic proteasome storage granules (PSG)… (more)

Subjects/Keywords: proteasome; ubiquitin; quiescence; 0487

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APA (6th Edition):

Wu, E. (2013). The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/43342

Chicago Manual of Style (16th Edition):

Wu, Edwin. “The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence.” 2013. Masters Thesis, University of Toronto. Accessed March 20, 2019. http://hdl.handle.net/1807/43342.

MLA Handbook (7th Edition):

Wu, Edwin. “The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence.” 2013. Web. 20 Mar 2019.

Vancouver:

Wu E. The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1807/43342.

Council of Science Editors:

Wu E. The Role of Ubiquitin on Yeast Proteasome Dynamics in Quiescence. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/43342


University of California – Berkeley

23. Worden, Evan Josiah. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.

Degree: Molecular & Cell Biology, 2016, University of California – Berkeley

 The 26S proteasome is responsible for selective protein degradation in eukaryotic cells. Polyubiquitin chains mark proteins for degradation by the proteasome, but before degradation can… (more)

Subjects/Keywords: Biochemistry; deubiquitinase; proteasome; ubiquitin

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APA (6th Edition):

Worden, E. J. (2016). Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/2138s3gn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Worden, Evan Josiah. “Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.” 2016. Thesis, University of California – Berkeley. Accessed March 20, 2019. http://www.escholarship.org/uc/item/2138s3gn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Worden, Evan Josiah. “Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11.” 2016. Web. 20 Mar 2019.

Vancouver:

Worden EJ. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. [Internet] [Thesis]. University of California – Berkeley; 2016. [cited 2019 Mar 20]. Available from: http://www.escholarship.org/uc/item/2138s3gn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Worden EJ. Structural and Functional Characterization of the Proteasomal Deubiquitinase Rpn11. [Thesis]. University of California – Berkeley; 2016. Available from: http://www.escholarship.org/uc/item/2138s3gn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

24. Staren, Daniel M. Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin.

Degree: MS, Molecular Biology, 2012, Loyola University Chicago

Ubiquitin has previously been identified as another natural agonist of CXC chemokine receptor 4 (CXCR4). In addition, recent evidence suggests that ubiquitin may activate… (more)

Subjects/Keywords: CXCR4; Ubiquitin; Molecular Biology

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APA (6th Edition):

Staren, D. M. (2012). Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Staren, Daniel M. “Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin.” 2012. Thesis, Loyola University Chicago. Accessed March 20, 2019. http://ecommons.luc.edu/luc_theses/840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Staren, Daniel M. “Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin.” 2012. Web. 20 Mar 2019.

Vancouver:

Staren DM. Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin. [Internet] [Thesis]. Loyola University Chicago; 2012. [cited 2019 Mar 20]. Available from: http://ecommons.luc.edu/luc_theses/840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Staren DM. Characterization of the Coca Chemokine Receptor Four Agonist Activity of Ubiquitin. [Thesis]. Loyola University Chicago; 2012. Available from: http://ecommons.luc.edu/luc_theses/840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

25. Shrestha, Amit. The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 .

Degree: 2017, University of Ottawa

 In addition to apoptosis, metacapases function to regulate various other processes that promote and sustain life. For example, the Saccharomyces cerevisiae metacaspase Yca1 promotes cellular… (more)

Subjects/Keywords: Proteostasis; Metacaspase; Ubiquitin; Rsp5; Yca1

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APA (6th Edition):

Shrestha, A. (2017). The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/36661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shrestha, Amit. “The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 .” 2017. Thesis, University of Ottawa. Accessed March 20, 2019. http://hdl.handle.net/10393/36661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shrestha, Amit. “The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 .” 2017. Web. 20 Mar 2019.

Vancouver:

Shrestha A. The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/10393/36661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shrestha A. The Proteostasis Function of the Saccharomyces Cerevisiae Metacaspase Yca1 . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/36661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

26. Parvatiyar, Kislay. Ubiquitin Dependent Regulation of Innate Antiviral Signaling.

Degree: PhD, Microbiology and Immunology (Medicine), 2010, University of Miami

 Induction of type I interferons by the transcription factors IRF3 and IRF7 is essential in the initiation of antiviral innate immunity. Activation of IRF3/7 requires… (more)

Subjects/Keywords: Ubiquitin Editing Complex; Signal Transduction

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APA (6th Edition):

Parvatiyar, K. (2010). Ubiquitin Dependent Regulation of Innate Antiviral Signaling. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/651

Chicago Manual of Style (16th Edition):

Parvatiyar, Kislay. “Ubiquitin Dependent Regulation of Innate Antiviral Signaling.” 2010. Doctoral Dissertation, University of Miami. Accessed March 20, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/651.

MLA Handbook (7th Edition):

Parvatiyar, Kislay. “Ubiquitin Dependent Regulation of Innate Antiviral Signaling.” 2010. Web. 20 Mar 2019.

Vancouver:

Parvatiyar K. Ubiquitin Dependent Regulation of Innate Antiviral Signaling. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2019 Mar 20]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/651.

Council of Science Editors:

Parvatiyar K. Ubiquitin Dependent Regulation of Innate Antiviral Signaling. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/651


University of Toronto

27. Heir, Pardeep. Regulation of Cellular Oxygen Sensing Pathways by VHL.

Degree: PhD, 2015, University of Toronto

Erythropoiesis represents a vital physiologic process that can be adjusted to combat compromised oxygen availability, otherwise known as hypoxia. The canonical response to adapt to… (more)

Subjects/Keywords: Erythropoiesis; Hypoxia; Ubiquitin; 0992

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Heir, P. (2015). Regulation of Cellular Oxygen Sensing Pathways by VHL. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/76677

Chicago Manual of Style (16th Edition):

Heir, Pardeep. “Regulation of Cellular Oxygen Sensing Pathways by VHL.” 2015. Doctoral Dissertation, University of Toronto. Accessed March 20, 2019. http://hdl.handle.net/1807/76677.

MLA Handbook (7th Edition):

Heir, Pardeep. “Regulation of Cellular Oxygen Sensing Pathways by VHL.” 2015. Web. 20 Mar 2019.

Vancouver:

Heir P. Regulation of Cellular Oxygen Sensing Pathways by VHL. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1807/76677.

Council of Science Editors:

Heir P. Regulation of Cellular Oxygen Sensing Pathways by VHL. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/76677


University of Toronto

28. Pascoe, Natasha. Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases.

Degree: PhD, 2018, University of Toronto

 Deubiquitinating enzymes represent attractive therapeutic targets that can be leveraged towards the treatment of a variety of devastating human health disorders. This doctoral thesis recounts… (more)

Subjects/Keywords: Biologics; DUBs; Ubiquitin; Yeast; 0307

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pascoe, N. (2018). Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89877

Chicago Manual of Style (16th Edition):

Pascoe, Natasha. “Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases.” 2018. Doctoral Dissertation, University of Toronto. Accessed March 20, 2019. http://hdl.handle.net/1807/89877.

MLA Handbook (7th Edition):

Pascoe, Natasha. “Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases.” 2018. Web. 20 Mar 2019.

Vancouver:

Pascoe N. Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2019 Mar 20]. Available from: http://hdl.handle.net/1807/89877.

Council of Science Editors:

Pascoe N. Yeast Two-Hybrid is an Effective Platform for the Discovery of Novel Inhibitors of Human Deubiquitinases. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89877


University of New South Wales

29. Shearer, Robert. Defining the role of the E3 ubiquitin ligase UBR5 in cancer.

Degree: Clinical School - St Vincent's Hospital, 2016, University of New South Wales

 Despite recent advances, breast cancer remains a major burden on the healthcare system in Australia and abroad. Of particular concern are cancer subtypes that currently… (more)

Subjects/Keywords: Ubiquitin; Cancer; Cilia; UBR5

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shearer, R. (2016). Defining the role of the E3 ubiquitin ligase UBR5 in cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/57120

Chicago Manual of Style (16th Edition):

Shearer, Robert. “Defining the role of the E3 ubiquitin ligase UBR5 in cancer.” 2016. Doctoral Dissertation, University of New South Wales. Accessed March 20, 2019. http://handle.unsw.edu.au/1959.4/57120.

MLA Handbook (7th Edition):

Shearer, Robert. “Defining the role of the E3 ubiquitin ligase UBR5 in cancer.” 2016. Web. 20 Mar 2019.

Vancouver:

Shearer R. Defining the role of the E3 ubiquitin ligase UBR5 in cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2019 Mar 20]. Available from: http://handle.unsw.edu.au/1959.4/57120.

Council of Science Editors:

Shearer R. Defining the role of the E3 ubiquitin ligase UBR5 in cancer. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/57120


Purdue University

30. Wade, Cameron. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.

Degree: MS, PULSe, 2016, Purdue University

 ElaD is a cysteine protease found in Escherichia coli (E. coli) and has been shown to function as a deubiquitinating enzyme (DUB). However, ubiquitin and… (more)

Subjects/Keywords: Bacteria; Deubiquitinase; Escherichia Coli; Ubiquitin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wade, C. (2016). Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. (Thesis). Purdue University. Retrieved from https://docs.lib.purdue.edu/open_access_theses/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wade, Cameron. “Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.” 2016. Thesis, Purdue University. Accessed March 20, 2019. https://docs.lib.purdue.edu/open_access_theses/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wade, Cameron. “Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli.” 2016. Web. 20 Mar 2019.

Vancouver:

Wade C. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. [Internet] [Thesis]. Purdue University; 2016. [cited 2019 Mar 20]. Available from: https://docs.lib.purdue.edu/open_access_theses/1233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wade C. Biochemical Analysis of a Prokaryotic Deubiquitinase from Escherichia Coli. [Thesis]. Purdue University; 2016. Available from: https://docs.lib.purdue.edu/open_access_theses/1233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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