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You searched for subject:(tumorigenicity). Showing records 1 – 15 of 15 total matches.

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1. 尾立, 西市. TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

 Tropomyosin-related kinase B (TrkB) plays an important role in tumor progression in various kinds of cancers; however, little is known about biological significance of TrkB… (more)

Subjects/Keywords: LCNEC; TrkB; BDNF; Invasion; Tumorigenicity; Lung cancer

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APA (6th Edition):

尾立, . (2013). TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

尾立, 西市. “TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である.” 2013. Thesis, Kyushu University / 九州大学. Accessed March 08, 2021. http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

尾立, 西市. “TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である.” 2013. Web. 08 Mar 2021.

Vancouver:

尾立 . TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

尾立 . TrkB/BDNF signaling pathway is a potential therapeutic target for pulmonary large cell neuroendocrine carcinoma : TrkB/BDNFシグナル経路は肺大細胞神経内分泌癌の治療標的である. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/26360 ; http://dx.doi.org/10.15017/26360

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Glasgow

2. Qiu, Jin. Cellular and molecular characterisation of a novel murine erythroleukaemia system.

Degree: PhD, 1997, University of Glasgow

 The ELM erythroleukaemia system is unusual in that the erythroleukaemia cells growing in vivo in the spleen cannot be maintained in vitro except in contact… (more)

Subjects/Keywords: 572.8; Leukaemia; Tumorigenicity

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APA (6th Edition):

Qiu, J. (1997). Cellular and molecular characterisation of a novel murine erythroleukaemia system. (Doctoral Dissertation). University of Glasgow. Retrieved from http://theses.gla.ac.uk/74874/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364085

Chicago Manual of Style (16th Edition):

Qiu, Jin. “Cellular and molecular characterisation of a novel murine erythroleukaemia system.” 1997. Doctoral Dissertation, University of Glasgow. Accessed March 08, 2021. http://theses.gla.ac.uk/74874/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364085.

MLA Handbook (7th Edition):

Qiu, Jin. “Cellular and molecular characterisation of a novel murine erythroleukaemia system.” 1997. Web. 08 Mar 2021.

Vancouver:

Qiu J. Cellular and molecular characterisation of a novel murine erythroleukaemia system. [Internet] [Doctoral dissertation]. University of Glasgow; 1997. [cited 2021 Mar 08]. Available from: http://theses.gla.ac.uk/74874/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364085.

Council of Science Editors:

Qiu J. Cellular and molecular characterisation of a novel murine erythroleukaemia system. [Doctoral Dissertation]. University of Glasgow; 1997. Available from: http://theses.gla.ac.uk/74874/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.364085


Louisiana State University

3. Kong, Haixia. Molecular determinants of Kaposi's sarcoma-associated herpesvirus tumorigenicity.

Degree: MS, Veterinary Pathology and Pathobiology, 2008, Louisiana State University

 A conditional silencing system using anti-gB siRNAs was devised to investigate the structure and function of the KSHV gB. Transient co-transfection of plasmids constitutively expressing… (more)

Subjects/Keywords: tumorigenicity; siRNA; KSHV; glycoprotein B

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APA (6th Edition):

Kong, H. (2008). Molecular determinants of Kaposi's sarcoma-associated herpesvirus tumorigenicity. (Masters Thesis). Louisiana State University. Retrieved from etd-06282008-141328 ; https://digitalcommons.lsu.edu/gradschool_theses/2933

Chicago Manual of Style (16th Edition):

Kong, Haixia. “Molecular determinants of Kaposi's sarcoma-associated herpesvirus tumorigenicity.” 2008. Masters Thesis, Louisiana State University. Accessed March 08, 2021. etd-06282008-141328 ; https://digitalcommons.lsu.edu/gradschool_theses/2933.

MLA Handbook (7th Edition):

Kong, Haixia. “Molecular determinants of Kaposi's sarcoma-associated herpesvirus tumorigenicity.” 2008. Web. 08 Mar 2021.

Vancouver:

Kong H. Molecular determinants of Kaposi's sarcoma-associated herpesvirus tumorigenicity. [Internet] [Masters thesis]. Louisiana State University; 2008. [cited 2021 Mar 08]. Available from: etd-06282008-141328 ; https://digitalcommons.lsu.edu/gradschool_theses/2933.

Council of Science Editors:

Kong H. Molecular determinants of Kaposi's sarcoma-associated herpesvirus tumorigenicity. [Masters Thesis]. Louisiana State University; 2008. Available from: etd-06282008-141328 ; https://digitalcommons.lsu.edu/gradschool_theses/2933


NSYSU

4. Chen, Jir-Wen. none.

Degree: Master, Biological Sciences, 2003, NSYSU

 Hepatocellular carcinoma (HCC) is one of the most prevalent cancers worldwide. Hepatocarcinogenesis is considered a multifactorial and mulitstep process that involves the activation of oncogenes… (more)

Subjects/Keywords: hepatocellular carcinoma; cell motility; tumorigenicity; PTEN

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APA (6th Edition):

Chen, J. (2003). none. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0731103-110240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jir-Wen. “none.” 2003. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0731103-110240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jir-Wen. “none.” 2003. Web. 08 Mar 2021.

Vancouver:

Chen J. none. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0731103-110240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen J. none. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0731103-110240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Wu, Chia-Ling. The effect of Dlk overexpression on the tumorigenicity of hepatoma cells.

Degree: Master, Biological Sciences, 2004, NSYSU

 Dlk is a transmembrane protein that possesses six epidermal growth factor-like sequences at the extracellular domain, a single transmembrane domain and an intracellular tail. The… (more)

Subjects/Keywords: overexpression; tumorigenicity; hepatoma cells; Dlk protein

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APA (6th Edition):

Wu, C. (2004). The effect of Dlk overexpression on the tumorigenicity of hepatoma cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904104-203607

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Chia-Ling. “The effect of Dlk overexpression on the tumorigenicity of hepatoma cells.” 2004. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904104-203607.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Chia-Ling. “The effect of Dlk overexpression on the tumorigenicity of hepatoma cells.” 2004. Web. 08 Mar 2021.

Vancouver:

Wu C. The effect of Dlk overexpression on the tumorigenicity of hepatoma cells. [Internet] [Thesis]. NSYSU; 2004. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904104-203607.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu C. The effect of Dlk overexpression on the tumorigenicity of hepatoma cells. [Thesis]. NSYSU; 2004. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904104-203607

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

6. Liang, Qin. Development of Genome Engineering Strategies for Cell Therapy Safety.

Degree: PhD, 2018, University of Toronto

 Cell therapy is the process of introducing healthy, therapeutic cells into human bodies to treat diseases. Cell therapies have great promise as a treatment or… (more)

Subjects/Keywords: Cell division essential gene; Cell therapy; Gene editing; Safety switch; Suicide gene; Tumorigenicity; 0307

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APA (6th Edition):

Liang, Q. (2018). Development of Genome Engineering Strategies for Cell Therapy Safety. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/82985

Chicago Manual of Style (16th Edition):

Liang, Qin. “Development of Genome Engineering Strategies for Cell Therapy Safety.” 2018. Doctoral Dissertation, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/82985.

MLA Handbook (7th Edition):

Liang, Qin. “Development of Genome Engineering Strategies for Cell Therapy Safety.” 2018. Web. 08 Mar 2021.

Vancouver:

Liang Q. Development of Genome Engineering Strategies for Cell Therapy Safety. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/82985.

Council of Science Editors:

Liang Q. Development of Genome Engineering Strategies for Cell Therapy Safety. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/82985

7. Heddleston, John Michael. The Role of Hypoxia in Modulating Glioma Cell Tumorigenic Potential.

Degree: PhD, Cell Biology, 2011, Case Western Reserve University School of Graduate Studies

 Normal stem cells reside in functional niches critical for self-renewal and maintenance. Neural and hematopoietic stem cell niches, in particular, are characterized by restricted availability… (more)

Subjects/Keywords: Cellular Biology; Molecular Biology; glioma stem cell; hypoxia; HIF; epigenetic; MLL1; tumorigenicity

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APA (6th Edition):

Heddleston, J. M. (2011). The Role of Hypoxia in Modulating Glioma Cell Tumorigenic Potential. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1310043767

Chicago Manual of Style (16th Edition):

Heddleston, John Michael. “The Role of Hypoxia in Modulating Glioma Cell Tumorigenic Potential.” 2011. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed March 08, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1310043767.

MLA Handbook (7th Edition):

Heddleston, John Michael. “The Role of Hypoxia in Modulating Glioma Cell Tumorigenic Potential.” 2011. Web. 08 Mar 2021.

Vancouver:

Heddleston JM. The Role of Hypoxia in Modulating Glioma Cell Tumorigenic Potential. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2011. [cited 2021 Mar 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1310043767.

Council of Science Editors:

Heddleston JM. The Role of Hypoxia in Modulating Glioma Cell Tumorigenic Potential. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1310043767


Université de Montréal

8. Cassim, Shamir. Rôle du métabolisme du glucose dans le phénotype tumoral hépatocytaire.

Degree: 2019, Université de Montréal

Subjects/Keywords: Carcinome Hépatocellulaire; Glucose; Métabolisme; Adaptabilité; Tumorigénicité; Hepatocellular carcinoma; Metabolism; Adaptability; Tumorigenicity; Health Sciences - Oncology / Sciences de la santé - Oncologie (UMI : 0992)

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APA (6th Edition):

Cassim, S. (2019). Rôle du métabolisme du glucose dans le phénotype tumoral hépatocytaire. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/21835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cassim, Shamir. “Rôle du métabolisme du glucose dans le phénotype tumoral hépatocytaire.” 2019. Thesis, Université de Montréal. Accessed March 08, 2021. http://hdl.handle.net/1866/21835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cassim, Shamir. “Rôle du métabolisme du glucose dans le phénotype tumoral hépatocytaire.” 2019. Web. 08 Mar 2021.

Vancouver:

Cassim S. Rôle du métabolisme du glucose dans le phénotype tumoral hépatocytaire. [Internet] [Thesis]. Université de Montréal; 2019. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1866/21835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cassim S. Rôle du métabolisme du glucose dans le phénotype tumoral hépatocytaire. [Thesis]. Université de Montréal; 2019. Available from: http://hdl.handle.net/1866/21835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

9. Clark, Aaron J. The Expression and Function of Wilms' Tumor 1 in Malignant Glioma.

Degree: PhD, Anatomy & Neurobiology, 2006, Virginia Commonwealth University

 The Wilms' tumor 1 gene is overexpressed in many types of cancer and is associated with poor prognosis and resistance to anti-cancer therapies. In vitro… (more)

Subjects/Keywords: apoptosis; gene therapy; p53; ionizing radiation; tumorigenicity; brain tumor; glioblastoma; Anatomy; Medicine and Health Sciences; Nervous System

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APA (6th Edition):

Clark, A. J. (2006). The Expression and Function of Wilms' Tumor 1 in Malignant Glioma. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/M8HC-M620 ; https://scholarscompass.vcu.edu/etd/1006

Chicago Manual of Style (16th Edition):

Clark, Aaron J. “The Expression and Function of Wilms' Tumor 1 in Malignant Glioma.” 2006. Doctoral Dissertation, Virginia Commonwealth University. Accessed March 08, 2021. https://doi.org/10.25772/M8HC-M620 ; https://scholarscompass.vcu.edu/etd/1006.

MLA Handbook (7th Edition):

Clark, Aaron J. “The Expression and Function of Wilms' Tumor 1 in Malignant Glioma.” 2006. Web. 08 Mar 2021.

Vancouver:

Clark AJ. The Expression and Function of Wilms' Tumor 1 in Malignant Glioma. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2006. [cited 2021 Mar 08]. Available from: https://doi.org/10.25772/M8HC-M620 ; https://scholarscompass.vcu.edu/etd/1006.

Council of Science Editors:

Clark AJ. The Expression and Function of Wilms' Tumor 1 in Malignant Glioma. [Doctoral Dissertation]. Virginia Commonwealth University; 2006. Available from: https://doi.org/10.25772/M8HC-M620 ; https://scholarscompass.vcu.edu/etd/1006


Virginia Commonwealth University

10. Hall, Charles. Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence.

Degree: MS, Microbiology & Immunology, 2013, Virginia Commonwealth University

 Metastatic breast cancer treatment has seen few advances in recent years, yet treatment resistance continues to rise, causing disease recurrence. A pilot study was performed… (more)

Subjects/Keywords: Metastatic breast cancer; Immunotherapy; Her2/neu; IFN-γ; CD44+CD24-/low stem-like phenotype; Tumorigenicity; Mouse mammary carcinoma; Invasion; Common gamma-chain cytokines; Central memory T cells; Natural Killer cells; Natural Killer T cells; Tumor Microenvironment; Tumor-specific cytotoxicity; Prophylactic adoptive cellular therapy; Medicine and Health Sciences

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APA (6th Edition):

Hall, C. (2013). Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/EXEB-VC80 ; https://scholarscompass.vcu.edu/etd/3176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hall, Charles. “Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence.” 2013. Thesis, Virginia Commonwealth University. Accessed March 08, 2021. https://doi.org/10.25772/EXEB-VC80 ; https://scholarscompass.vcu.edu/etd/3176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hall, Charles. “Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence.” 2013. Web. 08 Mar 2021.

Vancouver:

Hall C. Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence. [Internet] [Thesis]. Virginia Commonwealth University; 2013. [cited 2021 Mar 08]. Available from: https://doi.org/10.25772/EXEB-VC80 ; https://scholarscompass.vcu.edu/etd/3176.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hall C. Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence. [Thesis]. Virginia Commonwealth University; 2013. Available from: https://doi.org/10.25772/EXEB-VC80 ; https://scholarscompass.vcu.edu/etd/3176

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

11. Friedrich, Carsten. Modulation of the biology of malignant peripheral nerve sheath tumor cells by midkine.

Degree: 2007, Freie Universität Berlin

 Malignant peripheral nerve sheath tumors (MPNST) are aggressive soft tissue tumors arising sporadically, but more frequently in patients with Neurofibromatosis type 1 (NF1). Prognosis of… (more)

Subjects/Keywords: MPNST; NF1; Midkine; tumorigenicity; apoptosis; angiogenesis; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Friedrich, C. (2007). Modulation of the biology of malignant peripheral nerve sheath tumor cells by midkine. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-7527

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Friedrich, Carsten. “Modulation of the biology of malignant peripheral nerve sheath tumor cells by midkine.” 2007. Thesis, Freie Universität Berlin. Accessed March 08, 2021. http://dx.doi.org/10.17169/refubium-7527.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Friedrich, Carsten. “Modulation of the biology of malignant peripheral nerve sheath tumor cells by midkine.” 2007. Web. 08 Mar 2021.

Vancouver:

Friedrich C. Modulation of the biology of malignant peripheral nerve sheath tumor cells by midkine. [Internet] [Thesis]. Freie Universität Berlin; 2007. [cited 2021 Mar 08]. Available from: http://dx.doi.org/10.17169/refubium-7527.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Friedrich C. Modulation of the biology of malignant peripheral nerve sheath tumor cells by midkine. [Thesis]. Freie Universität Berlin; 2007. Available from: http://dx.doi.org/10.17169/refubium-7527

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Carolina

12. Gu, Yiben. Identification and Characterization of Her2/Neu-Transformed Breast Cancer Stem Cells.

Degree: MS, Biological Sciences, 2010, University of South Carolina

  A variety of human solid tumors, including breast cancer, are considered to embrace a hierarchical organization in which only infrequent tumor-initiating cancer stem cells… (more)

Subjects/Keywords: Life Sciences; Medicine and Health Sciences; Physical Sciences and Mathematics; cancer stem cell; extracellular matrix; mammary tumorsphere; self-renewal; tumorigenicity

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APA (6th Edition):

Gu, Y. (2010). Identification and Characterization of Her2/Neu-Transformed Breast Cancer Stem Cells. (Masters Thesis). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/343

Chicago Manual of Style (16th Edition):

Gu, Yiben. “Identification and Characterization of Her2/Neu-Transformed Breast Cancer Stem Cells.” 2010. Masters Thesis, University of South Carolina. Accessed March 08, 2021. https://scholarcommons.sc.edu/etd/343.

MLA Handbook (7th Edition):

Gu, Yiben. “Identification and Characterization of Her2/Neu-Transformed Breast Cancer Stem Cells.” 2010. Web. 08 Mar 2021.

Vancouver:

Gu Y. Identification and Characterization of Her2/Neu-Transformed Breast Cancer Stem Cells. [Internet] [Masters thesis]. University of South Carolina; 2010. [cited 2021 Mar 08]. Available from: https://scholarcommons.sc.edu/etd/343.

Council of Science Editors:

Gu Y. Identification and Characterization of Her2/Neu-Transformed Breast Cancer Stem Cells. [Masters Thesis]. University of South Carolina; 2010. Available from: https://scholarcommons.sc.edu/etd/343

13. Landis, Melissa D. Elucidating Molecular Mechanisms of ERBB2/Neu-Induced Mammary Tumorigenesis.

Degree: PhD, Pharmacology, 2006, Case Western Reserve University School of Graduate Studies

 The 15-30% of human breast cancers that have upregulated HER2/ErbB2/Neu are highly aggressive and resistant to traditional treatments, resulting in poor prognosis. To identify novel… (more)

Subjects/Keywords: mammary; breast cancer; tumor progression; transgenic mice; erbB2/neu/HER2; transforming growth factor beta; activin; microarray; synchronous tumorigenicity; susceptible cell population; bitransgenic; luteinizing hormone-overexpressing transgenic mice

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APA (6th Edition):

Landis, M. D. (2006). Elucidating Molecular Mechanisms of ERBB2/Neu-Induced Mammary Tumorigenesis. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1134662885

Chicago Manual of Style (16th Edition):

Landis, Melissa D. “Elucidating Molecular Mechanisms of ERBB2/Neu-Induced Mammary Tumorigenesis.” 2006. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed March 08, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1134662885.

MLA Handbook (7th Edition):

Landis, Melissa D. “Elucidating Molecular Mechanisms of ERBB2/Neu-Induced Mammary Tumorigenesis.” 2006. Web. 08 Mar 2021.

Vancouver:

Landis MD. Elucidating Molecular Mechanisms of ERBB2/Neu-Induced Mammary Tumorigenesis. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2006. [cited 2021 Mar 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1134662885.

Council of Science Editors:

Landis MD. Elucidating Molecular Mechanisms of ERBB2/Neu-Induced Mammary Tumorigenesis. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2006. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1134662885

14. Rapino, Francesca, 1982-. Induced transdifferentiation of human B-leukemia/lymphoma cell lines and inhibition of leukemogenicity.

Degree: Departament de Ciències Experimentals i de la Salut, 2013, Universitat Pompeu Fabra

 Las neoplasias malignas de células B abarcan una amplia variedad de enfermedades diferentes, incluyendo el linfoma no Hodgkin (LNH) y leucemia. Actualmente, la quimioterapia, la… (more)

Subjects/Keywords: Burkitt lymphoma; B-cell lymphoblastic leukemia; Transdifferentiation; Tumorigenicity; Compounds; Linfoma de Burkitt; Leucemia linfoblastica de celulas B; Transdiferenciación; Tumorigenicidad; Screening; Compuestos; 616

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rapino, Francesca, 1. (2013). Induced transdifferentiation of human B-leukemia/lymphoma cell lines and inhibition of leukemogenicity. (Thesis). Universitat Pompeu Fabra. Retrieved from http://hdl.handle.net/10803/128575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rapino, Francesca, 1982-. “Induced transdifferentiation of human B-leukemia/lymphoma cell lines and inhibition of leukemogenicity.” 2013. Thesis, Universitat Pompeu Fabra. Accessed March 08, 2021. http://hdl.handle.net/10803/128575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rapino, Francesca, 1982-. “Induced transdifferentiation of human B-leukemia/lymphoma cell lines and inhibition of leukemogenicity.” 2013. Web. 08 Mar 2021.

Vancouver:

Rapino, Francesca 1. Induced transdifferentiation of human B-leukemia/lymphoma cell lines and inhibition of leukemogenicity. [Internet] [Thesis]. Universitat Pompeu Fabra; 2013. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10803/128575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rapino, Francesca 1. Induced transdifferentiation of human B-leukemia/lymphoma cell lines and inhibition of leukemogenicity. [Thesis]. Universitat Pompeu Fabra; 2013. Available from: http://hdl.handle.net/10803/128575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Liu, He. Eukaryotic Initiation Factor 2-associated glycoprotein P67 inhibits the tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) and involves its differentiation and migration.

Degree: PhD, College of Arts and Sciences / Department of Chemistry and Biochemistry, 2019, Kent State University

 Rhabdomyosarcoma (RMS) is a soft tissue tumor that arises from connective tissue predominantly among children and adolescents. RMS may occur anywhere in the body with… (more)

Subjects/Keywords: Biochemistry; Rhabdomyosarcoma; RMS; Alveolar Rhabdomyosarcoma; ARMS; Rh30 cells; eIF2-asscociated glycoprotein P67; Tumorigenicity; Xenograft; insulin; IGF-1; IRS-1; AKT; FAK; SRC; Differentiation; Migration; GTPases; Rac1; Cdc42

…P67 can function as a tumor suppressor which inhibits the tumorigenicity of Rh30 cells ex… …tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) by P67 ex vivo and in vivo 2.1… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liu, H. (2019). Eukaryotic Initiation Factor 2-associated glycoprotein P67 inhibits the tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) and involves its differentiation and migration. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1564585690280704

Chicago Manual of Style (16th Edition):

Liu, He. “Eukaryotic Initiation Factor 2-associated glycoprotein P67 inhibits the tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) and involves its differentiation and migration.” 2019. Doctoral Dissertation, Kent State University. Accessed March 08, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=kent1564585690280704.

MLA Handbook (7th Edition):

Liu, He. “Eukaryotic Initiation Factor 2-associated glycoprotein P67 inhibits the tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) and involves its differentiation and migration.” 2019. Web. 08 Mar 2021.

Vancouver:

Liu H. Eukaryotic Initiation Factor 2-associated glycoprotein P67 inhibits the tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) and involves its differentiation and migration. [Internet] [Doctoral dissertation]. Kent State University; 2019. [cited 2021 Mar 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1564585690280704.

Council of Science Editors:

Liu H. Eukaryotic Initiation Factor 2-associated glycoprotein P67 inhibits the tumorigenicity of Alveolar Rhabdomyosarcoma (ARMS) and involves its differentiation and migration. [Doctoral Dissertation]. Kent State University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1564585690280704

.