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You searched for subject:(tumor suppressor tumor gene). Showing records 1 – 30 of 16755 total matches.

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University of Minnesota

1. Clark, Christopher. Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes.

Degree: PhD, Microbiology, Immunology and Cancer Biology, 2018, University of Minnesota

 The studies performed in this dissertation focused on the characterization of two candidate cancer genes, TM9SF2 and WAC, and their role in colorectal cancer (CRC).… (more)

Subjects/Keywords: Cancer; Driver gene; Genetics; Oncogene; Tumor suppressor

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APA (6th Edition):

Clark, C. (2018). Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/201708

Chicago Manual of Style (16th Edition):

Clark, Christopher. “Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes.” 2018. Doctoral Dissertation, University of Minnesota. Accessed March 08, 2021. http://hdl.handle.net/11299/201708.

MLA Handbook (7th Edition):

Clark, Christopher. “Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes.” 2018. Web. 08 Mar 2021.

Vancouver:

Clark C. Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes. [Internet] [Doctoral dissertation]. University of Minnesota; 2018. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/11299/201708.

Council of Science Editors:

Clark C. Characterization Of TM9SF2 And WAC As Novel Colorectal Cancer Driver Genes. [Doctoral Dissertation]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/201708

2. Mesrobian, Cristina M. The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators.

Degree: PhD, Division of Biology and Medicine. Pathobiology, 2009, Brown University

 Maintenance of an intricate balance between positive and negative regulators of the cell cycle is vital, as altered cell cycle regulation results in the uncontrolled… (more)

Subjects/Keywords: tumor suppressor

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APA (6th Edition):

Mesrobian, C. M. (2009). The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:173/

Chicago Manual of Style (16th Edition):

Mesrobian, Cristina M. “The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators.” 2009. Doctoral Dissertation, Brown University. Accessed March 08, 2021. https://repository.library.brown.edu/studio/item/bdr:173/.

MLA Handbook (7th Edition):

Mesrobian, Cristina M. “The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators.” 2009. Web. 08 Mar 2021.

Vancouver:

Mesrobian CM. The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators. [Internet] [Doctoral dissertation]. Brown University; 2009. [cited 2021 Mar 08]. Available from: https://repository.library.brown.edu/studio/item/bdr:173/.

Council of Science Editors:

Mesrobian CM. The Cul3 E3 Ligase Acts as a Tumor Suppressor by Targeting Ubiquitin-Mediated Proteolysis of Distinct Cell Cycle Regulators. [Doctoral Dissertation]. Brown University; 2009. Available from: https://repository.library.brown.edu/studio/item/bdr:173/


NSYSU

3. Lin, James. The Differential Expression of Bcl10 in the Tumor Cell Lines.

Degree: Master, Biological Sciences, 2004, NSYSU

 Bcl10 is one of the apoptosis regulatory protein. It is located at 1p22,one site harbor tumor suppressor tumor gene. We screen Bcl10 expression in different… (more)

Subjects/Keywords: tumor suppressor tumor gene

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APA (6th Edition):

Lin, J. (2004). The Differential Expression of Bcl10 in the Tumor Cell Lines. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, James. “The Differential Expression of Bcl10 in the Tumor Cell Lines.” 2004. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, James. “The Differential Expression of Bcl10 in the Tumor Cell Lines.” 2004. Web. 08 Mar 2021.

Vancouver:

Lin J. The Differential Expression of Bcl10 in the Tumor Cell Lines. [Internet] [Thesis]. NSYSU; 2004. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin J. The Differential Expression of Bcl10 in the Tumor Cell Lines. [Thesis]. NSYSU; 2004. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

4. Visuttijai, Kittichate. Cellular, Molecular and Functional Characterization of the Tumor Suppressor Candidate MYO1C.

Degree: 2016, University of Gothenburg / Göteborgs Universitet

Tumor suppressor genes play a role as a growth regulator and a gatekeeper of a cell. Their inactivation is often detected in malignant tumors. Identification… (more)

Subjects/Keywords: MYO1C; myosin IC; tumor suppressor gene; cancer; tumor; PI3K/AKT signaling

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APA (6th Edition):

Visuttijai, K. (2016). Cellular, Molecular and Functional Characterization of the Tumor Suppressor Candidate MYO1C. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/41552

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Visuttijai, Kittichate. “Cellular, Molecular and Functional Characterization of the Tumor Suppressor Candidate MYO1C.” 2016. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed March 08, 2021. http://hdl.handle.net/2077/41552.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Visuttijai, Kittichate. “Cellular, Molecular and Functional Characterization of the Tumor Suppressor Candidate MYO1C.” 2016. Web. 08 Mar 2021.

Vancouver:

Visuttijai K. Cellular, Molecular and Functional Characterization of the Tumor Suppressor Candidate MYO1C. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2016. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2077/41552.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Visuttijai K. Cellular, Molecular and Functional Characterization of the Tumor Suppressor Candidate MYO1C. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2016. Available from: http://hdl.handle.net/2077/41552

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Shin, Yi-Li. Part Iï¼Analysis of the tumor suppressor gene p16ï¼p27 and Rb expression in nasopharyngeal carcinoma in Taiwan Part IIï¼Tumor characteristics of two newly established nasopharyngeal carcinoma cell lines.

Degree: Master, Biological Sciences, 2000, NSYSU

 第ä¸é¨ä»½ Nasopharyngeal carcinomaï¼NPCï¼ is a malignant tumor which occurs at high incidence in southern China. Several risk factors have now been recognized, but the molecular… (more)

Subjects/Keywords: nasopharyngeal carcinoma; tumor suppressor gene

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APA (6th Edition):

Shin, Y. (2000). Part Iï¼Analysis of the tumor suppressor gene p16ï¼p27 and Rb expression in nasopharyngeal carcinoma in Taiwan Part IIï¼Tumor characteristics of two newly established nasopharyngeal carcinoma cell lines. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0808100-123811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shin, Yi-Li. “Part Iï¼Analysis of the tumor suppressor gene p16ï¼p27 and Rb expression in nasopharyngeal carcinoma in Taiwan Part IIï¼Tumor characteristics of two newly established nasopharyngeal carcinoma cell lines.” 2000. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0808100-123811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shin, Yi-Li. “Part Iï¼Analysis of the tumor suppressor gene p16ï¼p27 and Rb expression in nasopharyngeal carcinoma in Taiwan Part IIï¼Tumor characteristics of two newly established nasopharyngeal carcinoma cell lines.” 2000. Web. 08 Mar 2021.

Vancouver:

Shin Y. Part Iï¼Analysis of the tumor suppressor gene p16ï¼p27 and Rb expression in nasopharyngeal carcinoma in Taiwan Part IIï¼Tumor characteristics of two newly established nasopharyngeal carcinoma cell lines. [Internet] [Thesis]. NSYSU; 2000. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0808100-123811.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shin Y. Part Iï¼Analysis of the tumor suppressor gene p16ï¼p27 and Rb expression in nasopharyngeal carcinoma in Taiwan Part IIï¼Tumor characteristics of two newly established nasopharyngeal carcinoma cell lines. [Thesis]. NSYSU; 2000. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0808100-123811

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Zhao, Weilin. RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma.

Degree: 博士(医学), 2017, Mie University / 三重大学

Background: Nasopharyngeal carcinoma (NPC) is a malignancy of the head and neck that is prevalent in Southeast Asia and southern China. Recent studies in epigenetics… (more)

Subjects/Keywords: RERG; Nasopharyngeal carcinoma; Tumor suppressor gene; DNA methylation; Angiogenesis

Page 1 Page 2 Page 3

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APA (6th Edition):

Zhao, W. (2017). RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma. (Thesis). Mie University / 三重大学. Retrieved from http://hdl.handle.net/10076/00017324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhao, Weilin. “RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma.” 2017. Thesis, Mie University / 三重大学. Accessed March 08, 2021. http://hdl.handle.net/10076/00017324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhao, Weilin. “RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma.” 2017. Web. 08 Mar 2021.

Vancouver:

Zhao W. RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma. [Internet] [Thesis]. Mie University / 三重大学; 2017. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10076/00017324.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhao W. RERG suppresses cell proliferation, migration and angiogenesis through ERK/NF-кB signaling pathway in nasopharyngeal carcinoma. [Thesis]. Mie University / 三重大学; 2017. Available from: http://hdl.handle.net/10076/00017324

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

7. Lu, Wan-Jin. Illuminating the P53 Regulatory Network in Genetic Models.

Degree: 2011, University of Texas Southwestern Medical Center

The file named "thesis.pdf" is the primary dissertation file. Supplemental videos ("Movie1.avi", "Movie2.avi") are also provided (in Audio Video Interleaved format).

The tumor suppressor gene(more)

Subjects/Keywords: Genes, P53; Drosophila melanogaster; Gene Regulatory Networks; Tumor Suppressor Proteins

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APA (6th Edition):

Lu, W. (2011). Illuminating the P53 Regulatory Network in Genetic Models. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lu, Wan-Jin. “Illuminating the P53 Regulatory Network in Genetic Models.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed March 08, 2021. http://hdl.handle.net/2152.5/857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lu, Wan-Jin. “Illuminating the P53 Regulatory Network in Genetic Models.” 2011. Web. 08 Mar 2021.

Vancouver:

Lu W. Illuminating the P53 Regulatory Network in Genetic Models. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2152.5/857.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lu W. Illuminating the P53 Regulatory Network in Genetic Models. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/857

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

8. Weinheimer, Adam S. Investigating the Tumor Suppressor Role of RUNX1 in Human Breast Cancer.

Degree: Biochemistry, 2018, University of Vermont

  Breast cancer (BrCa) remains the leading cause of cancer-related deaths in women worldwide. Current research suggests that the transcription factor RUNX1 functions as a… (more)

Subjects/Keywords: Breast Cancer; RUNX1; Gene Knockdown; CRISPR; Biochemistry; Tumor Suppressor

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APA (6th Edition):

Weinheimer, A. S. (2018). Investigating the Tumor Suppressor Role of RUNX1 in Human Breast Cancer. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weinheimer, Adam S. “Investigating the Tumor Suppressor Role of RUNX1 in Human Breast Cancer.” 2018. Thesis, University of Vermont. Accessed March 08, 2021. https://scholarworks.uvm.edu/hcoltheses/260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weinheimer, Adam S. “Investigating the Tumor Suppressor Role of RUNX1 in Human Breast Cancer.” 2018. Web. 08 Mar 2021.

Vancouver:

Weinheimer AS. Investigating the Tumor Suppressor Role of RUNX1 in Human Breast Cancer. [Internet] [Thesis]. University of Vermont; 2018. [cited 2021 Mar 08]. Available from: https://scholarworks.uvm.edu/hcoltheses/260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weinheimer AS. Investigating the Tumor Suppressor Role of RUNX1 in Human Breast Cancer. [Thesis]. University of Vermont; 2018. Available from: https://scholarworks.uvm.edu/hcoltheses/260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

9. Hooks, Jared Cole. Regulation of the Tumor Suppressor ARF by TGFβ in Human Cancer Cells.

Degree: 2016, University of Texas Southwestern Medical Center

 Since its discovery, p14ARF (p19Arf in mice) has been shown to be an important regulator of the cell cycle, carrying out this function through p53-dependent… (more)

Subjects/Keywords: Gene Expression Regulation; Transforming Growth Factor beta; Tumor Suppressor Protein p14ARF

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APA (6th Edition):

Hooks, J. C. (2016). Regulation of the Tumor Suppressor ARF by TGFβ in Human Cancer Cells. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hooks, Jared Cole. “Regulation of the Tumor Suppressor ARF by TGFβ in Human Cancer Cells.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed March 08, 2021. http://hdl.handle.net/2152.5/6143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hooks, Jared Cole. “Regulation of the Tumor Suppressor ARF by TGFβ in Human Cancer Cells.” 2016. Web. 08 Mar 2021.

Vancouver:

Hooks JC. Regulation of the Tumor Suppressor ARF by TGFβ in Human Cancer Cells. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2152.5/6143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hooks JC. Regulation of the Tumor Suppressor ARF by TGFβ in Human Cancer Cells. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Baylor University

10. Yu, Eun-Jeong. The integrin interacts with cki-1/p27kip1, a tumor suppressor gene, of the nematode Caenorhabditis elegans.

Degree: PhD, Biology., 2014, Baylor University

 Integrin is a heterodimeric cell surface receptor for extracellular matrix (ECM) and plays essential roles in regulating cell behaviors such as cell migration, adhesion, growth… (more)

Subjects/Keywords: Caenorhabditis elegans.; Integrin.; Cell cycle.; Nucleolus.; Tumor suppressor gene.

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APA (6th Edition):

Yu, E. (2014). The integrin interacts with cki-1/p27kip1, a tumor suppressor gene, of the nematode Caenorhabditis elegans. (Doctoral Dissertation). Baylor University. Retrieved from http://hdl.handle.net/2104/9200

Chicago Manual of Style (16th Edition):

Yu, Eun-Jeong. “The integrin interacts with cki-1/p27kip1, a tumor suppressor gene, of the nematode Caenorhabditis elegans.” 2014. Doctoral Dissertation, Baylor University. Accessed March 08, 2021. http://hdl.handle.net/2104/9200.

MLA Handbook (7th Edition):

Yu, Eun-Jeong. “The integrin interacts with cki-1/p27kip1, a tumor suppressor gene, of the nematode Caenorhabditis elegans.” 2014. Web. 08 Mar 2021.

Vancouver:

Yu E. The integrin interacts with cki-1/p27kip1, a tumor suppressor gene, of the nematode Caenorhabditis elegans. [Internet] [Doctoral dissertation]. Baylor University; 2014. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2104/9200.

Council of Science Editors:

Yu E. The integrin interacts with cki-1/p27kip1, a tumor suppressor gene, of the nematode Caenorhabditis elegans. [Doctoral Dissertation]. Baylor University; 2014. Available from: http://hdl.handle.net/2104/9200


University of Texas Southwestern Medical Center

11. Sung, Caroline Yeh-Chien. Establishing a Dual-Reporter Mouse Model to Monitor INK4A/ARF Regulation in Vivo.

Degree: 2015, University of Texas Southwestern Medical Center

The general metadata  – e.g., title, author, abstract, subject headings, etc.  – is publicly available, but access to the submitted files is restricted to UT… (more)

Subjects/Keywords: Cyclin-Dependent Kinase Inhibitor p16; Gene Silencing; Tumor Suppressor Protein p14ARF

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APA (6th Edition):

Sung, C. Y. (2015). Establishing a Dual-Reporter Mouse Model to Monitor INK4A/ARF Regulation in Vivo. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sung, Caroline Yeh-Chien. “Establishing a Dual-Reporter Mouse Model to Monitor INK4A/ARF Regulation in Vivo.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed March 08, 2021. http://hdl.handle.net/2152.5/7068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sung, Caroline Yeh-Chien. “Establishing a Dual-Reporter Mouse Model to Monitor INK4A/ARF Regulation in Vivo.” 2015. Web. 08 Mar 2021.

Vancouver:

Sung CY. Establishing a Dual-Reporter Mouse Model to Monitor INK4A/ARF Regulation in Vivo. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2152.5/7068.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sung CY. Establishing a Dual-Reporter Mouse Model to Monitor INK4A/ARF Regulation in Vivo. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/7068

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

12. Chen, Jiamin. MicroRNA-193B Functions as a Tumor Suppressor in Malignant Melanoma .

Degree: Pathology and Molecular Medicine, 2012, Queens University

 Cutaneous melanoma is an increasingly common skin cancer characterized by aggressive metastatic growth and poor prognosis. The mechanisms behind melanoma progression are not fully understood,… (more)

Subjects/Keywords: Melanoma ; miR-193b ; Tumor Suppressor

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APA (6th Edition):

Chen, J. (2012). MicroRNA-193B Functions as a Tumor Suppressor in Malignant Melanoma . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jiamin. “MicroRNA-193B Functions as a Tumor Suppressor in Malignant Melanoma .” 2012. Thesis, Queens University. Accessed March 08, 2021. http://hdl.handle.net/1974/7224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jiamin. “MicroRNA-193B Functions as a Tumor Suppressor in Malignant Melanoma .” 2012. Web. 08 Mar 2021.

Vancouver:

Chen J. MicroRNA-193B Functions as a Tumor Suppressor in Malignant Melanoma . [Internet] [Thesis]. Queens University; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1974/7224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen J. MicroRNA-193B Functions as a Tumor Suppressor in Malignant Melanoma . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

13. Barnoud, Thibaut François. Regulation of the retinoblastoma tumor suppressor by the novel Ras effector NORE1A.

Degree: PhD, 2015, University of Louisville

  Ras is the most frequently mutated oncogene in human cancers. It acts as a critical branch point in signal transduction, regulating numerous downstream effectors… (more)

Subjects/Keywords: Ras; NORE1A; retinoblastoma protein; phosphoprotein phosphatase 1; senescence; tumor suppressor gene; Cancer Biology

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APA (6th Edition):

Barnoud, T. F. (2015). Regulation of the retinoblastoma tumor suppressor by the novel Ras effector NORE1A. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2320 ; https://ir.library.louisville.edu/etd/2320

Chicago Manual of Style (16th Edition):

Barnoud, Thibaut François. “Regulation of the retinoblastoma tumor suppressor by the novel Ras effector NORE1A.” 2015. Doctoral Dissertation, University of Louisville. Accessed March 08, 2021. 10.18297/etd/2320 ; https://ir.library.louisville.edu/etd/2320.

MLA Handbook (7th Edition):

Barnoud, Thibaut François. “Regulation of the retinoblastoma tumor suppressor by the novel Ras effector NORE1A.” 2015. Web. 08 Mar 2021.

Vancouver:

Barnoud TF. Regulation of the retinoblastoma tumor suppressor by the novel Ras effector NORE1A. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2021 Mar 08]. Available from: 10.18297/etd/2320 ; https://ir.library.louisville.edu/etd/2320.

Council of Science Editors:

Barnoud TF. Regulation of the retinoblastoma tumor suppressor by the novel Ras effector NORE1A. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2320 ; https://ir.library.louisville.edu/etd/2320


University of Texas Southwestern Medical Center

14. Kurtz, Paula S. In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network.

Degree: 2017, University of Texas Southwestern Medical Center

 p53 is the most commonly mutated gene in human cancers. Despite decades of p53 studies we do not fully understand how p53 suppresses tumors. Similar… (more)

Subjects/Keywords: Drosophila Proteins; Gene Expression Regulation; Genome-Wide Association Study; Tumor Suppressor Protein p53

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kurtz, P. S. (2017). In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7197

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kurtz, Paula S. “In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed March 08, 2021. http://hdl.handle.net/2152.5/7197.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kurtz, Paula S. “In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network.” 2017. Web. 08 Mar 2021.

Vancouver:

Kurtz PS. In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2152.5/7197.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kurtz PS. In Vivo Genome-Wide Analyses of the Drosophila p53 Transcriptional Network. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7197

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

15. fragou, aikaterini. Μεθυλίωση της κυτοσίνης στην κωδική περιοχή και θέσεις ματίσματος του p53: συσχέτιση με μεταβολές του ματίσματος κατά την καρκινογένεση.

Degree: 2015, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

 The dynamic process of DNA methylation and its catalytic involvement in the development, differentiation and pathological situations such as carcinogenicity has now been established. In… (more)

Subjects/Keywords: Μεθυλίωση DNA; Εναλλακτικό μάτισμα; p53 Ογκογονίδιο; Methylation; Alternative splicing; p53 tumor suppressor gene

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

fragou, a. (2015). Μεθυλίωση της κυτοσίνης στην κωδική περιοχή και θέσεις ματίσματος του p53: συσχέτιση με μεταβολές του ματίσματος κατά την καρκινογένεση. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/36439

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

fragou, aikaterini. “Μεθυλίωση της κυτοσίνης στην κωδική περιοχή και θέσεις ματίσματος του p53: συσχέτιση με μεταβολές του ματίσματος κατά την καρκινογένεση.” 2015. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed March 08, 2021. http://hdl.handle.net/10442/hedi/36439.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

fragou, aikaterini. “Μεθυλίωση της κυτοσίνης στην κωδική περιοχή και θέσεις ματίσματος του p53: συσχέτιση με μεταβολές του ματίσματος κατά την καρκινογένεση.” 2015. Web. 08 Mar 2021.

Vancouver:

fragou a. Μεθυλίωση της κυτοσίνης στην κωδική περιοχή και θέσεις ματίσματος του p53: συσχέτιση με μεταβολές του ματίσματος κατά την καρκινογένεση. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2015. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10442/hedi/36439.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

fragou a. Μεθυλίωση της κυτοσίνης στην κωδική περιοχή και θέσεις ματίσματος του p53: συσχέτιση με μεταβολές του ματίσματος κατά την καρκινογένεση. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2015. Available from: http://hdl.handle.net/10442/hedi/36439

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

16. Wei, Yiliang. Transcriptional regulation in Drosophila  – from genome to gene.

Degree: 2016, Michigan State University

Thesis Ph. D. Michigan State University. Biochemistry and Molecular Biology 2016

Transcriptional regulation plays a major role in gene expression, and is critical for development… (more)

Subjects/Keywords: Gene regulatory networks; Drosophila melanogaster – Molecular genetics; Tumor suppressor proteins – Genetic aspects; Molecular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wei, Y. (2016). Transcriptional regulation in Drosophila  – from genome to gene. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:4288

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wei, Yiliang. “Transcriptional regulation in Drosophila  – from genome to gene.” 2016. Thesis, Michigan State University. Accessed March 08, 2021. http://etd.lib.msu.edu/islandora/object/etd:4288.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wei, Yiliang. “Transcriptional regulation in Drosophila  – from genome to gene.” 2016. Web. 08 Mar 2021.

Vancouver:

Wei Y. Transcriptional regulation in Drosophila  – from genome to gene. [Internet] [Thesis]. Michigan State University; 2016. [cited 2021 Mar 08]. Available from: http://etd.lib.msu.edu/islandora/object/etd:4288.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wei Y. Transcriptional regulation in Drosophila  – from genome to gene. [Thesis]. Michigan State University; 2016. Available from: http://etd.lib.msu.edu/islandora/object/etd:4288

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Costa, Alexandra Fontes da. Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma.

Degree: Mestrado, Patologia Bucal, 2007, University of São Paulo

Os cistos e tumores odontogênicos sempre tiveram grande importância dentro da Odontologia, seja pela grande prevalência clínica seja pelo grande acometimento do indivíduo afetado pela… (more)

Subjects/Keywords: Apoptose; apoptosis; Cisto; Cyst; Odontogenic tumor; Supressão de tumor; Tumor odontogênico; Tumor suppressor

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APA (6th Edition):

Costa, A. F. d. (2007). Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13082007-162555/ ;

Chicago Manual of Style (16th Edition):

Costa, Alexandra Fontes da. “Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma.” 2007. Masters Thesis, University of São Paulo. Accessed March 08, 2021. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13082007-162555/ ;.

MLA Handbook (7th Edition):

Costa, Alexandra Fontes da. “Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma.” 2007. Web. 08 Mar 2021.

Vancouver:

Costa AFd. Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma. [Internet] [Masters thesis]. University of São Paulo; 2007. [cited 2021 Mar 08]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13082007-162555/ ;.

Council of Science Editors:

Costa AFd. Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma. [Masters Thesis]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13082007-162555/ ;

18. Longuini, Viviane Cristina. Identificação de moduladores genéticos em uma grande família com neoplasia endócrina múltipla (NEM1).

Degree: Mestrado, Endocrinologia, 2011, University of São Paulo

A Neoplasia endócrina múltipla tipo 1 (NEM1; OMIM 131100) é uma síndrome endócrina hereditária, que envolve tumores nas glândulas paratireóides, pâncreas endócrino/duodeno e hipófise. Mutações… (more)

Subjects/Keywords: Gene supressor de tumor p27Kip1; Genes neoplásicos; Genes supressores de tumor; Moduladores de fenótipo; Multiple endocrine neoplasia type 1; Neoplasia endócrina múltipla tipo 1; Neoplasic genes; Phenotypic modifiers; Tumor suppressor gene p27Kip1; Tumor suppressor genes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Longuini, V. C. (2011). Identificação de moduladores genéticos em uma grande família com neoplasia endócrina múltipla (NEM1). (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-05052011-145430/ ;

Chicago Manual of Style (16th Edition):

Longuini, Viviane Cristina. “Identificação de moduladores genéticos em uma grande família com neoplasia endócrina múltipla (NEM1).” 2011. Masters Thesis, University of São Paulo. Accessed March 08, 2021. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-05052011-145430/ ;.

MLA Handbook (7th Edition):

Longuini, Viviane Cristina. “Identificação de moduladores genéticos em uma grande família com neoplasia endócrina múltipla (NEM1).” 2011. Web. 08 Mar 2021.

Vancouver:

Longuini VC. Identificação de moduladores genéticos em uma grande família com neoplasia endócrina múltipla (NEM1). [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2021 Mar 08]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-05052011-145430/ ;.

Council of Science Editors:

Longuini VC. Identificação de moduladores genéticos em uma grande família com neoplasia endócrina múltipla (NEM1). [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-05052011-145430/ ;


University of Gothenburg / Göteborgs Universitet

19. Fransson, Susanne. From 1p3 to PI3K - Studies of neuroblastoma.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

 Neuroblastoma (NB) is a tumor of the sympathetic nervous system and is the most common extra-cranial tumor of childhood, accounting for 7% of all pediatric… (more)

Subjects/Keywords: cancer; neuroblastoma; PI3K; PIK3CD; 1p36; alternative splicing; tumor; neural crest; mucosa; premalignant; tumor suppressor gene; oncogene; gene expression; epigenetics; splicing; signaling; akt; western blot; TaqMan; PIK3R1; p110H; p37H; p85; RAS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fransson, S. (2011). From 1p3 to PI3K - Studies of neuroblastoma. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/23820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fransson, Susanne. “From 1p3 to PI3K - Studies of neuroblastoma.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed March 08, 2021. http://hdl.handle.net/2077/23820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fransson, Susanne. “From 1p3 to PI3K - Studies of neuroblastoma.” 2011. Web. 08 Mar 2021.

Vancouver:

Fransson S. From 1p3 to PI3K - Studies of neuroblastoma. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2077/23820.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fransson S. From 1p3 to PI3K - Studies of neuroblastoma. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/23820

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

20. Vreede, G.A.M. de. Endocytic control of tumor suppression in Drosophila melanogaster.

Degree: 2011, Universiteit Utrecht

 Mutations in Drosophila melanogaster tumor suppressor genes are able to cause neoplastic overgrowth in various epithelial tissues. Screens in Drosophila showed that a multitude of… (more)

Subjects/Keywords: endocytosis; neoplastic; tumor; suppressor; genes; drosophila; tumorigenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vreede, G. A. M. d. (2011). Endocytic control of tumor suppression in Drosophila melanogaster. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/218766

Chicago Manual of Style (16th Edition):

Vreede, G A M de. “Endocytic control of tumor suppression in Drosophila melanogaster.” 2011. Masters Thesis, Universiteit Utrecht. Accessed March 08, 2021. http://dspace.library.uu.nl:8080/handle/1874/218766.

MLA Handbook (7th Edition):

Vreede, G A M de. “Endocytic control of tumor suppression in Drosophila melanogaster.” 2011. Web. 08 Mar 2021.

Vancouver:

Vreede GAMd. Endocytic control of tumor suppression in Drosophila melanogaster. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2021 Mar 08]. Available from: http://dspace.library.uu.nl:8080/handle/1874/218766.

Council of Science Editors:

Vreede GAMd. Endocytic control of tumor suppression in Drosophila melanogaster. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/218766


University of Rochester

21. Gaur, Kriti; Li, Willis X. A Role for Tumor Suppressor Gene Birt-Hogg-Dubé in Regulating Ribosomal RNA (rRNA) Synthesis.

Degree: PhD, 2011, University of Rochester

 Birt-Hogg-Dubé syndrome (BHD) is a hereditary human cancer disorder characterized by renal carcinoma of diverse histology, benign skin lesions and pulmonary cysts, and is caused… (more)

Subjects/Keywords: Kidney Cancer; rRNA Synthesis; Tumor Suppressor

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APA (6th Edition):

Gaur, Kriti; Li, W. X. (2011). A Role for Tumor Suppressor Gene Birt-Hogg-Dubé in Regulating Ribosomal RNA (rRNA) Synthesis. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/15808

Chicago Manual of Style (16th Edition):

Gaur, Kriti; Li, Willis X. “A Role for Tumor Suppressor Gene Birt-Hogg-Dubé in Regulating Ribosomal RNA (rRNA) Synthesis.” 2011. Doctoral Dissertation, University of Rochester. Accessed March 08, 2021. http://hdl.handle.net/1802/15808.

MLA Handbook (7th Edition):

Gaur, Kriti; Li, Willis X. “A Role for Tumor Suppressor Gene Birt-Hogg-Dubé in Regulating Ribosomal RNA (rRNA) Synthesis.” 2011. Web. 08 Mar 2021.

Vancouver:

Gaur, Kriti; Li WX. A Role for Tumor Suppressor Gene Birt-Hogg-Dubé in Regulating Ribosomal RNA (rRNA) Synthesis. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1802/15808.

Council of Science Editors:

Gaur, Kriti; Li WX. A Role for Tumor Suppressor Gene Birt-Hogg-Dubé in Regulating Ribosomal RNA (rRNA) Synthesis. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/15808


University of Alberta

22. Law, Jennifer. MOAP-1: A Candidate Tumor Suppressor Protein.

Degree: MS, Department of Biochemistry, 2011, University of Alberta

 Modulator of apoptosis 1 (MOAP-1) is a BH3-like protein that plays a key role in death receptor-dependent apoptosis and cooperates with the tumor suppressor protein… (more)

Subjects/Keywords: MOAP-1; Tumor Suppressor; Apoptosis; RACK1

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APA (6th Edition):

Law, J. (2011). MOAP-1: A Candidate Tumor Suppressor Protein. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/dj52w5366

Chicago Manual of Style (16th Edition):

Law, Jennifer. “MOAP-1: A Candidate Tumor Suppressor Protein.” 2011. Masters Thesis, University of Alberta. Accessed March 08, 2021. https://era.library.ualberta.ca/files/dj52w5366.

MLA Handbook (7th Edition):

Law, Jennifer. “MOAP-1: A Candidate Tumor Suppressor Protein.” 2011. Web. 08 Mar 2021.

Vancouver:

Law J. MOAP-1: A Candidate Tumor Suppressor Protein. [Internet] [Masters thesis]. University of Alberta; 2011. [cited 2021 Mar 08]. Available from: https://era.library.ualberta.ca/files/dj52w5366.

Council of Science Editors:

Law J. MOAP-1: A Candidate Tumor Suppressor Protein. [Masters Thesis]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/dj52w5366


Kyoto University / 京都大学

23. Sanada, Masashi. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.

Degree: 博士(医学), 2014, Kyoto University / 京都大学

This paper was published in Nature 2009 Aug 13;460(7257):904-8. doi: 10.1038/nature08240. http://www.nature.com/nature/journal/v460/n7257/full/nature08240.html

新制・論文博士

乙第12855号

論医博第2085号

Subjects/Keywords: myelodysplasia; LOH; tumor suppressor; ubiquitination; cytokine sensitivity

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APA (6th Edition):

Sanada, M. (2014). Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sanada, Masashi. “Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.” 2014. Thesis, Kyoto University / 京都大学. Accessed March 08, 2021. http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sanada, Masashi. “Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異.” 2014. Web. 08 Mar 2021.

Vancouver:

Sanada M. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sanada M. Gain-of-function of mutated C-CBL tumor suppressor in myeloid neoplasms : 骨髄系腫瘍における腫瘍抑制遺伝子C-CBLの機能獲得型変異. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/192124 ; http://dx.doi.org/10.14989/doctor.r12855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

24. Mikse, Oliver. CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA .

Degree: 2011, Penn State University

 Lung tumor development is believed to occur through a step-wise series of molecular changes that influence cell growth and survival. This process is facilitated by… (more)

Subjects/Keywords: CDC14A; Lung Cancer; FOXO3A; Tumor Suppressor

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APA (6th Edition):

Mikse, O. (2011). CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mikse, Oliver. “CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA .” 2011. Thesis, Penn State University. Accessed March 08, 2021. https://submit-etda.libraries.psu.edu/catalog/11586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mikse, Oliver. “CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA .” 2011. Web. 08 Mar 2021.

Vancouver:

Mikse O. CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Mar 08]. Available from: https://submit-etda.libraries.psu.edu/catalog/11586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mikse O. CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

25. Dean, Ivory. The Tumor-Suppressive Role Of Secreted Maspin Via The Exosomes.

Degree: PhD, Cancer Biology, 2014, Wayne State University

  This dissertation highlights several novel findings. Maspin has been consistently detected in the conditioned media of maspin-expressing cells of normal and tumor breast, prostate… (more)

Subjects/Keywords: Exosomes; Maspin; Microenvironment; Tumor Suppressor; Biology

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APA (6th Edition):

Dean, I. (2014). The Tumor-Suppressive Role Of Secreted Maspin Via The Exosomes. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1089

Chicago Manual of Style (16th Edition):

Dean, Ivory. “The Tumor-Suppressive Role Of Secreted Maspin Via The Exosomes.” 2014. Doctoral Dissertation, Wayne State University. Accessed March 08, 2021. https://digitalcommons.wayne.edu/oa_dissertations/1089.

MLA Handbook (7th Edition):

Dean, Ivory. “The Tumor-Suppressive Role Of Secreted Maspin Via The Exosomes.” 2014. Web. 08 Mar 2021.

Vancouver:

Dean I. The Tumor-Suppressive Role Of Secreted Maspin Via The Exosomes. [Internet] [Doctoral dissertation]. Wayne State University; 2014. [cited 2021 Mar 08]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1089.

Council of Science Editors:

Dean I. The Tumor-Suppressive Role Of Secreted Maspin Via The Exosomes. [Doctoral Dissertation]. Wayne State University; 2014. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1089


University of Texas Southwestern Medical Center

26. Wylie, Annika Dawn. P53 Genes Act to Restrain Mobile Elements.

Degree: 2015, University of Texas Southwestern Medical Center

 Oncogenic stress provokes tumor suppression by p53 but the extent to which this regulatory axis is conserved remains unknown. Using a biosensor to visualize p53… (more)

Subjects/Keywords: Genes, p53; Retroelements; Tumor Suppressor Protein p53

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wylie, A. D. (2015). P53 Genes Act to Restrain Mobile Elements. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/4461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wylie, Annika Dawn. “P53 Genes Act to Restrain Mobile Elements.” 2015. Thesis, University of Texas Southwestern Medical Center. Accessed March 08, 2021. http://hdl.handle.net/2152.5/4461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wylie, Annika Dawn. “P53 Genes Act to Restrain Mobile Elements.” 2015. Web. 08 Mar 2021.

Vancouver:

Wylie AD. P53 Genes Act to Restrain Mobile Elements. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2015. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2152.5/4461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wylie AD. P53 Genes Act to Restrain Mobile Elements. [Thesis]. University of Texas Southwestern Medical Center; 2015. Available from: http://hdl.handle.net/2152.5/4461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

27. Zuo, Hao LIFS. Regulation of the tumor suppressor Fhit by activated Gα subunits.

Degree: 2013, Hong Kong University of Science and Technology

 The FHIT tumor suppressor gene is arguably the most commonly altered gene in cancer since it is inactivated in about 60% of human tumors. Despite… (more)

Subjects/Keywords: Antioncogenes ; Tumor suppressor proteins ; G proteins ; Receptors

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zuo, H. L. (2013). Regulation of the tumor suppressor Fhit by activated Gα subunits. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-92340 ; https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zuo, Hao LIFS. “Regulation of the tumor suppressor Fhit by activated Gα subunits.” 2013. Thesis, Hong Kong University of Science and Technology. Accessed March 08, 2021. http://repository.ust.hk/ir/Record/1783.1-92340 ; https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zuo, Hao LIFS. “Regulation of the tumor suppressor Fhit by activated Gα subunits.” 2013. Web. 08 Mar 2021.

Vancouver:

Zuo HL. Regulation of the tumor suppressor Fhit by activated Gα subunits. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2013. [cited 2021 Mar 08]. Available from: http://repository.ust.hk/ir/Record/1783.1-92340 ; https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zuo HL. Regulation of the tumor suppressor Fhit by activated Gα subunits. [Thesis]. Hong Kong University of Science and Technology; 2013. Available from: http://repository.ust.hk/ir/Record/1783.1-92340 ; https://doi.org/10.14711/thesis-b1254497 ; http://repository.ust.hk/ir/bitstream/1783.1-92340/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

28. Bassi, Christian. Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress.

Degree: PhD, 2015, University of Toronto

 Loss of function of the phosphatase and tensin homolog (PTEN) tumor suppressor is frequently found in many human malignancies. PTEN antagonizes the Phosphatidylinositide 3-kinase (PI3K)… (more)

Subjects/Keywords: Cancer; DNA damage; PTEN; tumor suppressor; 0307

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bassi, C. (2015). Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69214

Chicago Manual of Style (16th Edition):

Bassi, Christian. “Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress.” 2015. Doctoral Dissertation, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/69214.

MLA Handbook (7th Edition):

Bassi, Christian. “Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress.” 2015. Web. 08 Mar 2021.

Vancouver:

Bassi C. Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/69214.

Council of Science Editors:

Bassi C. Nuclear PTEN Controls DNA Repair and Sensitivity to Genotoxic Stress. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/69214


Michigan State University

29. Gjidoda, Alison Marie. Transcriptional regulation of RNA polymerase III-transcribed genes by the retinoblastoma tumor suppressor protein.

Degree: 2012, Michigan State University

Thesis M.S. Michigan State University. Biochemistry and Molecular Biology 2012.

The Retinoblastoma Tumor Suppressor Protein (Rb) is a critical regulator of cellular proliferation. In the… (more)

Subjects/Keywords: RNA polymerases; Tumor suppressor proteins; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gjidoda, A. M. (2012). Transcriptional regulation of RNA polymerase III-transcribed genes by the retinoblastoma tumor suppressor protein. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:1541

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gjidoda, Alison Marie. “Transcriptional regulation of RNA polymerase III-transcribed genes by the retinoblastoma tumor suppressor protein.” 2012. Thesis, Michigan State University. Accessed March 08, 2021. http://etd.lib.msu.edu/islandora/object/etd:1541.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gjidoda, Alison Marie. “Transcriptional regulation of RNA polymerase III-transcribed genes by the retinoblastoma tumor suppressor protein.” 2012. Web. 08 Mar 2021.

Vancouver:

Gjidoda AM. Transcriptional regulation of RNA polymerase III-transcribed genes by the retinoblastoma tumor suppressor protein. [Internet] [Thesis]. Michigan State University; 2012. [cited 2021 Mar 08]. Available from: http://etd.lib.msu.edu/islandora/object/etd:1541.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gjidoda AM. Transcriptional regulation of RNA polymerase III-transcribed genes by the retinoblastoma tumor suppressor protein. [Thesis]. Michigan State University; 2012. Available from: http://etd.lib.msu.edu/islandora/object/etd:1541

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

30. Smith, Laura Taylor. An Epigenetic approach for identifying novel tumor associated genes from regions of Loss of Heterozygosity in human neoplasias.

Degree: PhD, Medical Microbiology and Immunology, 2005, The Ohio State University

 The incidence of cancer is expected to be 1 in every 3 individuals. Onset of the disease in the population has been attributed to multiple… (more)

Subjects/Keywords: Biology, Genetics; epigenetics; DNA methylation; tumor suppressor gene; metastasis suppressor gene

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, L. T. (2005). An Epigenetic approach for identifying novel tumor associated genes from regions of Loss of Heterozygosity in human neoplasias. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1116959843

Chicago Manual of Style (16th Edition):

Smith, Laura Taylor. “An Epigenetic approach for identifying novel tumor associated genes from regions of Loss of Heterozygosity in human neoplasias.” 2005. Doctoral Dissertation, The Ohio State University. Accessed March 08, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1116959843.

MLA Handbook (7th Edition):

Smith, Laura Taylor. “An Epigenetic approach for identifying novel tumor associated genes from regions of Loss of Heterozygosity in human neoplasias.” 2005. Web. 08 Mar 2021.

Vancouver:

Smith LT. An Epigenetic approach for identifying novel tumor associated genes from regions of Loss of Heterozygosity in human neoplasias. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2021 Mar 08]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1116959843.

Council of Science Editors:

Smith LT. An Epigenetic approach for identifying novel tumor associated genes from regions of Loss of Heterozygosity in human neoplasias. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1116959843

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