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You searched for subject:(transgenic mouse models). Showing records 1 – 19 of 19 total matches.

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Dalhousie University

1. Fraser, Leanne M. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.

Degree: PhD, Department of Psychology and Neuroscience, 2013, Dalhousie University

 The triple transgenic (3xTg-AD) mouse model of Alzheimer’s disease (AD) possesses three transgenes that lead to the development of amyloid-beta (A?) plaques (APPswe, PS1M146V) and… (more)

Subjects/Keywords: "Transgenic mice"; "Alzheimer's disease"; "Mouse models"; "Behaviour"; "Memory"; "Motor coordination"

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APA (6th Edition):

Fraser, L. M. (2013). Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/28080

Chicago Manual of Style (16th Edition):

Fraser, Leanne M. “Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.” 2013. Doctoral Dissertation, Dalhousie University. Accessed March 03, 2021. http://hdl.handle.net/10222/28080.

MLA Handbook (7th Edition):

Fraser, Leanne M. “Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.” 2013. Web. 03 Mar 2021.

Vancouver:

Fraser LM. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10222/28080.

Council of Science Editors:

Fraser LM. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/28080


University College Cork

2. Heimer-McGinn, Victoria. Development and characterization of novel transgenic techniques for the study of neuronal circuitry.

Degree: 2013, University College Cork

 Modern neuroscience relies heavily on sophisticated tools that allow us to visualize and manipulate cells with precise spatial and temporal control. Transgenic mouse models, for… (more)

Subjects/Keywords: Activity-dependent competition; Neuronal circuits; Tetanus toxin; Dendritic spines; Transgenic mouse models; Transgenic mice; Molecular neurobiology; Microbial toxins

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APA (6th Edition):

Heimer-McGinn, V. (2013). Development and characterization of novel transgenic techniques for the study of neuronal circuitry. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/1251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Heimer-McGinn, Victoria. “Development and characterization of novel transgenic techniques for the study of neuronal circuitry.” 2013. Thesis, University College Cork. Accessed March 03, 2021. http://hdl.handle.net/10468/1251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Heimer-McGinn, Victoria. “Development and characterization of novel transgenic techniques for the study of neuronal circuitry.” 2013. Web. 03 Mar 2021.

Vancouver:

Heimer-McGinn V. Development and characterization of novel transgenic techniques for the study of neuronal circuitry. [Internet] [Thesis]. University College Cork; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10468/1251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Heimer-McGinn V. Development and characterization of novel transgenic techniques for the study of neuronal circuitry. [Thesis]. University College Cork; 2013. Available from: http://hdl.handle.net/10468/1251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

3. Nguyen, Annee. Peripheral Neuropathy and Sexual Dimorphism in Prion Disease Mouse Models.

Degree: Biology, 2018, University of California – San Diego

 In this study, I characterize peripheral neuropathic phenotypes in F35 mice (knock-out model of prion disease) and 93N mice (novel knock-in model of prion disease)… (more)

Subjects/Keywords: Biology; Pathology; Neurosciences; N-terminus; peripheral neuropathy; prion disease; sexual dimorphism; transgenic mouse models

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APA (6th Edition):

Nguyen, A. (2018). Peripheral Neuropathy and Sexual Dimorphism in Prion Disease Mouse Models. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/5hp992p6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Annee. “Peripheral Neuropathy and Sexual Dimorphism in Prion Disease Mouse Models.” 2018. Thesis, University of California – San Diego. Accessed March 03, 2021. http://www.escholarship.org/uc/item/5hp992p6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Annee. “Peripheral Neuropathy and Sexual Dimorphism in Prion Disease Mouse Models.” 2018. Web. 03 Mar 2021.

Vancouver:

Nguyen A. Peripheral Neuropathy and Sexual Dimorphism in Prion Disease Mouse Models. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 03]. Available from: http://www.escholarship.org/uc/item/5hp992p6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen A. Peripheral Neuropathy and Sexual Dimorphism in Prion Disease Mouse Models. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/5hp992p6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Palmer, Daniel. Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease.

Degree: PhD, Department of Psychology, 2017, University of Guelph

 In the course of Alzheimer’s Disease (AD) pathology, there is a progressive worsening of cognitive symptoms. One of the important steps in AD research has… (more)

Subjects/Keywords: Alzheimer's; Transgenic Mouse Models; Visual Spatial Learning; Attention; 3xTG; 5xFAD; APP/PS1; Touchscreen Operant Chamber

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APA (6th Edition):

Palmer, D. (2017). Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148

Chicago Manual of Style (16th Edition):

Palmer, Daniel. “Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease.” 2017. Doctoral Dissertation, University of Guelph. Accessed March 03, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148.

MLA Handbook (7th Edition):

Palmer, Daniel. “Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease.” 2017. Web. 03 Mar 2021.

Vancouver:

Palmer D. Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease. [Internet] [Doctoral dissertation]. University of Guelph; 2017. [cited 2021 Mar 03]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148.

Council of Science Editors:

Palmer D. Assessment of Visual Sustained Attention and Visual Spatial Integration in the APP/PS1, 5xFAD, and 3xTG Transgenic mouse models of Alzheimer’s disease. [Doctoral Dissertation]. University of Guelph; 2017. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/10148


Penn State University

5. Yura, Renee E. MEPRIN METALLOPROTEASES MODULATE THE HOST RESPONSE TO ESCHERICHIA COLI .

Degree: 2008, Penn State University

 Meprin metalloproteases, composed of alpha and/or beta subunits, consist of membrane-bound and secreted forms that are abundantly expressed in kidney and intestinal epithelial cells. They… (more)

Subjects/Keywords: meprins; metalloproteases; lipopolysaccharide; urinary tract infection; transgenic mouse models

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APA (6th Edition):

Yura, R. E. (2008). MEPRIN METALLOPROTEASES MODULATE THE HOST RESPONSE TO ESCHERICHIA COLI . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/8602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yura, Renee E. “MEPRIN METALLOPROTEASES MODULATE THE HOST RESPONSE TO ESCHERICHIA COLI .” 2008. Thesis, Penn State University. Accessed March 03, 2021. https://submit-etda.libraries.psu.edu/catalog/8602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yura, Renee E. “MEPRIN METALLOPROTEASES MODULATE THE HOST RESPONSE TO ESCHERICHIA COLI .” 2008. Web. 03 Mar 2021.

Vancouver:

Yura RE. MEPRIN METALLOPROTEASES MODULATE THE HOST RESPONSE TO ESCHERICHIA COLI . [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Mar 03]. Available from: https://submit-etda.libraries.psu.edu/catalog/8602.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yura RE. MEPRIN METALLOPROTEASES MODULATE THE HOST RESPONSE TO ESCHERICHIA COLI . [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/8602

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

6. Anwar, Sabina Zareen. Functional characterisation of synuclein-based novel genetic mouse models.

Degree: PhD, 2011, University of Oxford

 Synucleins are highly conserved presynaptic proteins with unknown function. α-synuclein plays a key role regulating dopamine homeostasis and is intimately involved in Parkinson’s disease (PD)… (more)

Subjects/Keywords: 616.83307; Medical Sciences; Neuroscience; Neurogenetics; Parkinsons disease; synuclein; alpha-synuclein; dopamine; basal ganglia; fast-scan cyclic voltammetry; transgenic mouse models

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APA (6th Edition):

Anwar, S. Z. (2011). Functional characterisation of synuclein-based novel genetic mouse models. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:b14fc29e-2bc8-4a31-865b-f4ec0e0f6f2c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558207

Chicago Manual of Style (16th Edition):

Anwar, Sabina Zareen. “Functional characterisation of synuclein-based novel genetic mouse models.” 2011. Doctoral Dissertation, University of Oxford. Accessed March 03, 2021. http://ora.ox.ac.uk/objects/uuid:b14fc29e-2bc8-4a31-865b-f4ec0e0f6f2c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558207.

MLA Handbook (7th Edition):

Anwar, Sabina Zareen. “Functional characterisation of synuclein-based novel genetic mouse models.” 2011. Web. 03 Mar 2021.

Vancouver:

Anwar SZ. Functional characterisation of synuclein-based novel genetic mouse models. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2021 Mar 03]. Available from: http://ora.ox.ac.uk/objects/uuid:b14fc29e-2bc8-4a31-865b-f4ec0e0f6f2c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558207.

Council of Science Editors:

Anwar SZ. Functional characterisation of synuclein-based novel genetic mouse models. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:b14fc29e-2bc8-4a31-865b-f4ec0e0f6f2c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558207


University of New South Wales

7. Jeet, Varinder. Development and characterisation of a transgenic mouse model to investigate the mechanisms and treatment options for Androgen independent metastatic prostate cancer.

Degree: Clinical School - Prince of Wales Hospital, 2009, University of New South Wales

 Currently, there are no preclinical immunocompetent mouse models that adequately represent all stages of prostate cancer (PC) especially, androgen depletion independent (ADI) and bone metastatic… (more)

Subjects/Keywords: Androgen independent PCa; Prostate Cancer; Transgenic Mouse Models

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APA (6th Edition):

Jeet, V. (2009). Development and characterisation of a transgenic mouse model to investigate the mechanisms and treatment options for Androgen independent metastatic prostate cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/44578 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:7875/SOURCE1?view=true

Chicago Manual of Style (16th Edition):

Jeet, Varinder. “Development and characterisation of a transgenic mouse model to investigate the mechanisms and treatment options for Androgen independent metastatic prostate cancer.” 2009. Doctoral Dissertation, University of New South Wales. Accessed March 03, 2021. http://handle.unsw.edu.au/1959.4/44578 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:7875/SOURCE1?view=true.

MLA Handbook (7th Edition):

Jeet, Varinder. “Development and characterisation of a transgenic mouse model to investigate the mechanisms and treatment options for Androgen independent metastatic prostate cancer.” 2009. Web. 03 Mar 2021.

Vancouver:

Jeet V. Development and characterisation of a transgenic mouse model to investigate the mechanisms and treatment options for Androgen independent metastatic prostate cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2009. [cited 2021 Mar 03]. Available from: http://handle.unsw.edu.au/1959.4/44578 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:7875/SOURCE1?view=true.

Council of Science Editors:

Jeet V. Development and characterisation of a transgenic mouse model to investigate the mechanisms and treatment options for Androgen independent metastatic prostate cancer. [Doctoral Dissertation]. University of New South Wales; 2009. Available from: http://handle.unsw.edu.au/1959.4/44578 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:7875/SOURCE1?view=true

8. Jung, Sophie. Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2013, Université de Strasbourg

Les maladies auto-immunes, qui touchent plus de 5% de la population, sont induites par une perte de la tolérance aux antigènes du Soi. Ces pathologies,… (more)

Subjects/Keywords: Auto-immunité; Lymphocyte B; Infection bactérienne; Modèles murins transgéniques; Tolérance lymphocytaire B; Autoimmunity; B lymphocyte; Bacterial infection; Transgenic mouse models; B cell tolerance; 571.96; 572.8; 616.97

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APA (6th Edition):

Jung, S. (2013). Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ067

Chicago Manual of Style (16th Edition):

Jung, Sophie. “Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed March 03, 2021. http://www.theses.fr/2013STRAJ067.

MLA Handbook (7th Edition):

Jung, Sophie. “Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model.” 2013. Web. 03 Mar 2021.

Vancouver:

Jung S. Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2021 Mar 03]. Available from: http://www.theses.fr/2013STRAJ067.

Council of Science Editors:

Jung S. Agents infectieux et rupture de tolérance lymphocytaire B : étude des processus de maturation d'affinité et de différenciation plasmocytaire au cours d'une infection bactérienne dans un nouveau modèle knock-in autoréactif : Infectious agents and B cell tolerance breakdown : study of affinity maturation and plasma-cell differentiation processes during bacterial infection in a new autoreactive knock-in mouse model. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ067

9. Franks, Sarah Elizabeth. The Importance of the Insulin-like Growth Factor Signaling Pathway in Lung Cancer and the Potential of its Components as Therapeutic Targets.

Degree: PhD, Department of Biomedical Sciences, 2015, University of Guelph

 Lung cancer is the leading cause of cancer-related mortalities worldwide. Despite the introduction of new therapeutics, there has been very little improvement in survival for… (more)

Subjects/Keywords: Lung Cancer; The type I insulin-like growth factor receptor (IGF-IR); Akt Isoforms (Akt1 and Akt2); Transgenic Mouse Models; Kruppel-like factor 5 (KLF5)

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APA (6th Edition):

Franks, S. E. (2015). The Importance of the Insulin-like Growth Factor Signaling Pathway in Lung Cancer and the Potential of its Components as Therapeutic Targets. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8860

Chicago Manual of Style (16th Edition):

Franks, Sarah Elizabeth. “The Importance of the Insulin-like Growth Factor Signaling Pathway in Lung Cancer and the Potential of its Components as Therapeutic Targets.” 2015. Doctoral Dissertation, University of Guelph. Accessed March 03, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8860.

MLA Handbook (7th Edition):

Franks, Sarah Elizabeth. “The Importance of the Insulin-like Growth Factor Signaling Pathway in Lung Cancer and the Potential of its Components as Therapeutic Targets.” 2015. Web. 03 Mar 2021.

Vancouver:

Franks SE. The Importance of the Insulin-like Growth Factor Signaling Pathway in Lung Cancer and the Potential of its Components as Therapeutic Targets. [Internet] [Doctoral dissertation]. University of Guelph; 2015. [cited 2021 Mar 03]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8860.

Council of Science Editors:

Franks SE. The Importance of the Insulin-like Growth Factor Signaling Pathway in Lung Cancer and the Potential of its Components as Therapeutic Targets. [Doctoral Dissertation]. University of Guelph; 2015. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8860


University of Connecticut

10. Perrino, Peter. Reanalysis of the Usher syndrome type 2 carrier as “phenotype-free”.

Degree: MS, Psychological Sciences, 2019, University of Connecticut

Subjects/Keywords: Auditory processing disorder; transgenic mouse models; neurodevelopmental disorders; USH2A; rapid auditory processing

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APA (6th Edition):

Perrino, P. (2019). Reanalysis of the Usher syndrome type 2 carrier as “phenotype-free”. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/1326

Chicago Manual of Style (16th Edition):

Perrino, Peter. “Reanalysis of the Usher syndrome type 2 carrier as “phenotype-free”.” 2019. Masters Thesis, University of Connecticut. Accessed March 03, 2021. https://opencommons.uconn.edu/gs_theses/1326.

MLA Handbook (7th Edition):

Perrino, Peter. “Reanalysis of the Usher syndrome type 2 carrier as “phenotype-free”.” 2019. Web. 03 Mar 2021.

Vancouver:

Perrino P. Reanalysis of the Usher syndrome type 2 carrier as “phenotype-free”. [Internet] [Masters thesis]. University of Connecticut; 2019. [cited 2021 Mar 03]. Available from: https://opencommons.uconn.edu/gs_theses/1326.

Council of Science Editors:

Perrino P. Reanalysis of the Usher syndrome type 2 carrier as “phenotype-free”. [Masters Thesis]. University of Connecticut; 2019. Available from: https://opencommons.uconn.edu/gs_theses/1326


East Carolina University

11. Nopparat, Jongdee. [Delta]-catenin: implications in prostate cancer progression.

Degree: PhD, Anatomy and Cell Biology, 2014, East Carolina University

 Prostate cancer (PCa) is the most commonly diagnosed cancer and the second most common cause of cancer death among men in the US. Due to… (more)

Subjects/Keywords: Biology, Molecular; [Delta]-catenin; Glucose deprivation; Prostate – Cancer; Somatic mutations; Transgenic mouse models; δ-catenin; Molecular biology; Prostatic Neoplasms – diagnosis; Catenins – metabolism; Adenocarcinomas – diagnosis

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APA (6th Edition):

Nopparat, J. (2014). [Delta]-catenin: implications in prostate cancer progression. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/4436

Chicago Manual of Style (16th Edition):

Nopparat, Jongdee. “[Delta]-catenin: implications in prostate cancer progression.” 2014. Doctoral Dissertation, East Carolina University. Accessed March 03, 2021. http://hdl.handle.net/10342/4436.

MLA Handbook (7th Edition):

Nopparat, Jongdee. “[Delta]-catenin: implications in prostate cancer progression.” 2014. Web. 03 Mar 2021.

Vancouver:

Nopparat J. [Delta]-catenin: implications in prostate cancer progression. [Internet] [Doctoral dissertation]. East Carolina University; 2014. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10342/4436.

Council of Science Editors:

Nopparat J. [Delta]-catenin: implications in prostate cancer progression. [Doctoral Dissertation]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4436

12. Πετράκης, Ιωάννης. Μηχανισμοί νεφρικής βλάβης στην οικογενή αμυλοειδική πολυνευροπάθεια.

Degree: 2014, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Introduction: Familial Amyloid Polyneuropathy (FAP) has been first described 60 years ago. Nowadays consists one of the commonest forms of hereditary amyloidosis. It is inherited… (more)

Subjects/Keywords: Τρανσθυρετίνη; Ποδοκύτταρα; Αμυλοείδωση; Διαγονιδιακά μοντέλα νεφρικής νόσου; Περιβαλλοντική επίδραση; Μηχανισμοί νεφρικής βλάβης; Transthyretin; Podocytes; Amyloidosis; Transgenic mouse models of renal disease; Environmental interaction; Mechanisms of renal insult

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APA (6th Edition):

Πετράκης, . . (2014). Μηχανισμοί νεφρικής βλάβης στην οικογενή αμυλοειδική πολυνευροπάθεια. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/38838

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Πετράκης, Ιωάννης. “Μηχανισμοί νεφρικής βλάβης στην οικογενή αμυλοειδική πολυνευροπάθεια.” 2014. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed March 03, 2021. http://hdl.handle.net/10442/hedi/38838.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Πετράκης, Ιωάννης. “Μηχανισμοί νεφρικής βλάβης στην οικογενή αμυλοειδική πολυνευροπάθεια.” 2014. Web. 03 Mar 2021.

Vancouver:

Πετράκης . Μηχανισμοί νεφρικής βλάβης στην οικογενή αμυλοειδική πολυνευροπάθεια. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2014. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10442/hedi/38838.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Πετράκης . Μηχανισμοί νεφρικής βλάβης στην οικογενή αμυλοειδική πολυνευροπάθεια. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2014. Available from: http://hdl.handle.net/10442/hedi/38838

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Ha, Michael Neul. In Vitro and In Vivo Studies with Measles Virus and its Interaction with the Mouse Innate Immune System.

Degree: 2012, University of Toronto

Measles is one of the most contagious diseases known to mankind. Despite the availability of a safe and effective vaccine, approximately 164,000 measles-related deaths were… (more)

Subjects/Keywords: molecular biology; virology; paramyxovirus; measles; innate immunity; mouse models; transgenic mouse; fusion; fusion inhibitor; FIP; IRF3; microbiology; ZfFG; plasmacytoid dendritic cell; mouse embryonic fibroblast; receptor; 0720

…128 Figure II. Transgenic mouse expressing vaccinia virus E3L… …131 Appendix II: Generation of transgenic mouse which… …64 Figure 3.1. Verification of SLAM/IRF3KO mouse… …Figure 4.5. Computer generated models of ZfFG resistant mutants… …Mitochondrial antiviral signaling MCMV Mouse cytomegalovirus MCP Membrane cofactor protein MDA… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ha, M. N. (2012). In Vitro and In Vivo Studies with Measles Virus and its Interaction with the Mouse Innate Immune System. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/32725

Chicago Manual of Style (16th Edition):

Ha, Michael Neul. “In Vitro and In Vivo Studies with Measles Virus and its Interaction with the Mouse Innate Immune System.” 2012. Doctoral Dissertation, University of Toronto. Accessed March 03, 2021. http://hdl.handle.net/1807/32725.

MLA Handbook (7th Edition):

Ha, Michael Neul. “In Vitro and In Vivo Studies with Measles Virus and its Interaction with the Mouse Innate Immune System.” 2012. Web. 03 Mar 2021.

Vancouver:

Ha MN. In Vitro and In Vivo Studies with Measles Virus and its Interaction with the Mouse Innate Immune System. [Internet] [Doctoral dissertation]. University of Toronto; 2012. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1807/32725.

Council of Science Editors:

Ha MN. In Vitro and In Vivo Studies with Measles Virus and its Interaction with the Mouse Innate Immune System. [Doctoral Dissertation]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32725

14. Ito-Smith, Kristine M. Characterization of Caytaxin Protein in Animal Models of Cerebellar Dysfunction.

Degree: PhD, Cellular and Molecular Biology, 2012, University of Michigan

 Over 150,000 Americans have been diagnosed with ataxia, however most are untreatable due to yet-unknown etiologies. The work described within this dissertation aims to expand… (more)

Subjects/Keywords: Ataxia; Cayman Ataxia; Transgenic Mouse Models; Alternative Translation; Caytaxin; Atcay/ATCAY; Molecular, Cellular and Developmental Biology; Science

…heterozygote, wild type hesitant heterozygote, wild type Transgenic mouse, positive for the transgene… …Transgenic mouse, negative for the transgene Transgenic line, ATCAY exon 9 mutation Transgenic line… …Caytaxin remains poorly understood. Four mouse models and one rat model have been identified to… …mAb) specific for the Caytaxin protein as well as transgenic mouse lines expressing… …lines, wild type and Atcay mouse mutants, and transgenic ATCAY mouse lines. These analyses… 

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APA (6th Edition):

Ito-Smith, K. M. (2012). Characterization of Caytaxin Protein in Animal Models of Cerebellar Dysfunction. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/95992

Chicago Manual of Style (16th Edition):

Ito-Smith, Kristine M. “Characterization of Caytaxin Protein in Animal Models of Cerebellar Dysfunction.” 2012. Doctoral Dissertation, University of Michigan. Accessed March 03, 2021. http://hdl.handle.net/2027.42/95992.

MLA Handbook (7th Edition):

Ito-Smith, Kristine M. “Characterization of Caytaxin Protein in Animal Models of Cerebellar Dysfunction.” 2012. Web. 03 Mar 2021.

Vancouver:

Ito-Smith KM. Characterization of Caytaxin Protein in Animal Models of Cerebellar Dysfunction. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2027.42/95992.

Council of Science Editors:

Ito-Smith KM. Characterization of Caytaxin Protein in Animal Models of Cerebellar Dysfunction. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/95992

15. Dey, Joyoti. Understanding the role of the Sonic Hedgehog signaling pathway in cerebellar development and medulloblastoma genesis.

Degree: PhD, 2012, University of Washington

 Medulloblastoma is a developmental cancer of the cerebellum. It continues to be the most common pediatric brain cancer associated with dire survival and impaired quality… (more)

Subjects/Keywords: cerebellar development; medulloblastoma; MyoD; neurooncology; Sonic hedgehog signaling; transgenic mouse models; Molecular biology; Oncology; Developmental biology; Molecular and cellular biology

…through lineage tracing and genetic analyses in mouse models of medulloblastoma… …atlas that has generated more than 600 transgenic mouse lines expressing Enhanced Green… …expression and function can lead to cancer and neuropathological conditions. Mouse models of… …the existing mouse models recapitulating this subtype (Hatten and Roussel, 2011)… …Wechsler-Reya, 2011). These mouse models have provided critical information about… 

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APA (6th Edition):

Dey, J. (2012). Understanding the role of the Sonic Hedgehog signaling pathway in cerebellar development and medulloblastoma genesis. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/20238

Chicago Manual of Style (16th Edition):

Dey, Joyoti. “Understanding the role of the Sonic Hedgehog signaling pathway in cerebellar development and medulloblastoma genesis.” 2012. Doctoral Dissertation, University of Washington. Accessed March 03, 2021. http://hdl.handle.net/1773/20238.

MLA Handbook (7th Edition):

Dey, Joyoti. “Understanding the role of the Sonic Hedgehog signaling pathway in cerebellar development and medulloblastoma genesis.” 2012. Web. 03 Mar 2021.

Vancouver:

Dey J. Understanding the role of the Sonic Hedgehog signaling pathway in cerebellar development and medulloblastoma genesis. [Internet] [Doctoral dissertation]. University of Washington; 2012. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1773/20238.

Council of Science Editors:

Dey J. Understanding the role of the Sonic Hedgehog signaling pathway in cerebellar development and medulloblastoma genesis. [Doctoral Dissertation]. University of Washington; 2012. Available from: http://hdl.handle.net/1773/20238

16. Λέκκα, Ευτυχία. Πειραματικά μοντέλα ανοσοθεραπείας HER-2/neu θετικών όγκων.

Degree: 2009, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Overexpression of the HER-2/neu oncogene by a significant number of human carcinomas has been associated with more aggressive disease. Despite its high oncogenic potential, HER2/neu… (more)

Subjects/Keywords: Ογκοπρωτεΐνη HER/2-neu; Πεπτιδικά εμβόλια; Διαγονιδιακά πειραματόζωα; Κυτταροτοξικά Τ λεμφοκύτταρα; Καρκίνος μαστού; HER-2/neu oncoprotein; Peptide vaccines; Transgenic mouse models; Cytotoxic T lymphocytes; Breast cancer

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APA (6th Edition):

Λέκκα, . . (2009). Πειραματικά μοντέλα ανοσοθεραπείας HER-2/neu θετικών όγκων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/23771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Λέκκα, Ευτυχία. “Πειραματικά μοντέλα ανοσοθεραπείας HER-2/neu θετικών όγκων.” 2009. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 03, 2021. http://hdl.handle.net/10442/hedi/23771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Λέκκα, Ευτυχία. “Πειραματικά μοντέλα ανοσοθεραπείας HER-2/neu θετικών όγκων.” 2009. Web. 03 Mar 2021.

Vancouver:

Λέκκα . Πειραματικά μοντέλα ανοσοθεραπείας HER-2/neu θετικών όγκων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10442/hedi/23771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Λέκκα . Πειραματικά μοντέλα ανοσοθεραπείας HER-2/neu θετικών όγκων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2009. Available from: http://hdl.handle.net/10442/hedi/23771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Zhong, Jia. Exploring the Three-Dimensional Regional Myocardial Function in Transgenic Mouse Models of Cardiac Diseases using Novel MR Tissue Tracking Techniques.

Degree: PhD, Biomedical Engineering, 2009, Case Western Reserve University School of Graduate Studies

  The advent of the genomic age is revolutionizing the experimental cardiovascular research irreversibly. Dissecting molecular switches that control the changes of cardiac physiology with… (more)

Subjects/Keywords: Biomedical Research; Engineering; transgenic mouse models; cardiac diseases; spatial modulation of magnetization (SPAMM) tagging; harmonic phase (HARP); displacement encoding with stimulated echoes (DENSE); strain; regional myocardial function

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APA (6th Edition):

Zhong, J. (2009). Exploring the Three-Dimensional Regional Myocardial Function in Transgenic Mouse Models of Cardiac Diseases using Novel MR Tissue Tracking Techniques. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1247260314

Chicago Manual of Style (16th Edition):

Zhong, Jia. “Exploring the Three-Dimensional Regional Myocardial Function in Transgenic Mouse Models of Cardiac Diseases using Novel MR Tissue Tracking Techniques.” 2009. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed March 03, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1247260314.

MLA Handbook (7th Edition):

Zhong, Jia. “Exploring the Three-Dimensional Regional Myocardial Function in Transgenic Mouse Models of Cardiac Diseases using Novel MR Tissue Tracking Techniques.” 2009. Web. 03 Mar 2021.

Vancouver:

Zhong J. Exploring the Three-Dimensional Regional Myocardial Function in Transgenic Mouse Models of Cardiac Diseases using Novel MR Tissue Tracking Techniques. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2009. [cited 2021 Mar 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1247260314.

Council of Science Editors:

Zhong J. Exploring the Three-Dimensional Regional Myocardial Function in Transgenic Mouse Models of Cardiac Diseases using Novel MR Tissue Tracking Techniques. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1247260314


Leiden University

18. Kara, F. Monitoring Alzheimer's disease in transgenic mice with ultra high field magnetic resonance imaging.

Degree: 2013, Leiden University

  While aging remains one of the most significant risk factors for development of Alzheimer disease (AD), increasing evidence strongly points to the potential roles… (more)

Subjects/Keywords: 17.6 T; Alzheimer's disease; Transverse relaxation time; Blood flow alterations; Transgenic mouse models of AD; Corpus callosum; Ultra high magnetic field; Magnetic resonance imaging

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APA (6th Edition):

Kara, F. (2013). Monitoring Alzheimer's disease in transgenic mice with ultra high field magnetic resonance imaging. (Doctoral Dissertation). Leiden University. Retrieved from http://hdl.handle.net/1887/22736

Chicago Manual of Style (16th Edition):

Kara, F. “Monitoring Alzheimer's disease in transgenic mice with ultra high field magnetic resonance imaging.” 2013. Doctoral Dissertation, Leiden University. Accessed March 03, 2021. http://hdl.handle.net/1887/22736.

MLA Handbook (7th Edition):

Kara, F. “Monitoring Alzheimer's disease in transgenic mice with ultra high field magnetic resonance imaging.” 2013. Web. 03 Mar 2021.

Vancouver:

Kara F. Monitoring Alzheimer's disease in transgenic mice with ultra high field magnetic resonance imaging. [Internet] [Doctoral dissertation]. Leiden University; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1887/22736.

Council of Science Editors:

Kara F. Monitoring Alzheimer's disease in transgenic mice with ultra high field magnetic resonance imaging. [Doctoral Dissertation]. Leiden University; 2013. Available from: http://hdl.handle.net/1887/22736


Johannes Gutenberg Universität Mainz

19. Steiner, Michel-Alexander. Transgenic mice as a tool for depression research: examples from the endocannabinoid and the corticotropin-releasing hormone system.

Degree: 2007, Johannes Gutenberg Universität Mainz

Major depression belongs to the most serious and widespread psychiatric disorders in today’s society. There is a great need for the delineation of the underlying… (more)

Subjects/Keywords: Transgene Mäuse, Endocannabinoid-System, CRH, Depression, Angst, Tiermodell, Stress, HPA Achse; Transgenic mice, endocannabinoid system, CRH, CRF, depression, anxiety, mouse models, stress, HPA axis; Chemistry and allied sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Steiner, M. (2007). Transgenic mice as a tool for depression research: examples from the endocannabinoid and the corticotropin-releasing hormone system. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2008/1583/

Chicago Manual of Style (16th Edition):

Steiner, Michel-Alexander. “Transgenic mice as a tool for depression research: examples from the endocannabinoid and the corticotropin-releasing hormone system.” 2007. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 03, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2008/1583/.

MLA Handbook (7th Edition):

Steiner, Michel-Alexander. “Transgenic mice as a tool for depression research: examples from the endocannabinoid and the corticotropin-releasing hormone system.” 2007. Web. 03 Mar 2021.

Vancouver:

Steiner M. Transgenic mice as a tool for depression research: examples from the endocannabinoid and the corticotropin-releasing hormone system. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2007. [cited 2021 Mar 03]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1583/.

Council of Science Editors:

Steiner M. Transgenic mice as a tool for depression research: examples from the endocannabinoid and the corticotropin-releasing hormone system. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2007. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1583/

.