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You searched for subject:(toxicology). Showing records 1 – 30 of 4045 total matches.

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1. Bradie, Johanna. Brine-induced mortality of non-indigenous species in ballast water.

Degree: MS, Great Lakes Institute for Environmental Research, 2009, National Library of Canada

 Transoceanic vessels entering the Great Lakes are required to undergo ballast water exchange to reduce the risk of transporting non-indigenous species. Ballast water exchange effectively… (more)

Subjects/Keywords: Toxicology.

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APA (6th Edition):

Bradie, J. (2009). Brine-induced mortality of non-indigenous species in ballast water. (Masters Thesis). National Library of Canada. Retrieved from http://scholar.uwindsor.ca/etd/362

Chicago Manual of Style (16th Edition):

Bradie, Johanna. “Brine-induced mortality of non-indigenous species in ballast water.” 2009. Masters Thesis, National Library of Canada. Accessed September 21, 2020. http://scholar.uwindsor.ca/etd/362.

MLA Handbook (7th Edition):

Bradie, Johanna. “Brine-induced mortality of non-indigenous species in ballast water.” 2009. Web. 21 Sep 2020.

Vancouver:

Bradie J. Brine-induced mortality of non-indigenous species in ballast water. [Internet] [Masters thesis]. National Library of Canada; 2009. [cited 2020 Sep 21]. Available from: http://scholar.uwindsor.ca/etd/362.

Council of Science Editors:

Bradie J. Brine-induced mortality of non-indigenous species in ballast water. [Masters Thesis]. National Library of Canada; 2009. Available from: http://scholar.uwindsor.ca/etd/362

2. Liu, Jian. Factors Affecting Critical Toxicokinetic Parameters of Polychlorinated Biphenyls (PCBS) in Japanese Koi (CYPRINUS CARPIO): Effect of Diet on Chemical Assimilation and Influence of Feces to Whole Body Chemical Elimination.

Degree: MS, Great Lakes Institute for Environmental Research, 2009, National Library of Canada

 This thesis provides enhanced calibration of critical parameters, chemical uptake efficiency (AE), chemical whole body elimination rate coefficient (ktot), and fecal elimination rate coefficients (kex),… (more)

Subjects/Keywords: Toxicology.

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APA (6th Edition):

Liu, J. (2009). Factors Affecting Critical Toxicokinetic Parameters of Polychlorinated Biphenyls (PCBS) in Japanese Koi (CYPRINUS CARPIO): Effect of Diet on Chemical Assimilation and Influence of Feces to Whole Body Chemical Elimination. (Masters Thesis). National Library of Canada. Retrieved from http://scholar.uwindsor.ca/etd/366

Chicago Manual of Style (16th Edition):

Liu, Jian. “Factors Affecting Critical Toxicokinetic Parameters of Polychlorinated Biphenyls (PCBS) in Japanese Koi (CYPRINUS CARPIO): Effect of Diet on Chemical Assimilation and Influence of Feces to Whole Body Chemical Elimination.” 2009. Masters Thesis, National Library of Canada. Accessed September 21, 2020. http://scholar.uwindsor.ca/etd/366.

MLA Handbook (7th Edition):

Liu, Jian. “Factors Affecting Critical Toxicokinetic Parameters of Polychlorinated Biphenyls (PCBS) in Japanese Koi (CYPRINUS CARPIO): Effect of Diet on Chemical Assimilation and Influence of Feces to Whole Body Chemical Elimination.” 2009. Web. 21 Sep 2020.

Vancouver:

Liu J. Factors Affecting Critical Toxicokinetic Parameters of Polychlorinated Biphenyls (PCBS) in Japanese Koi (CYPRINUS CARPIO): Effect of Diet on Chemical Assimilation and Influence of Feces to Whole Body Chemical Elimination. [Internet] [Masters thesis]. National Library of Canada; 2009. [cited 2020 Sep 21]. Available from: http://scholar.uwindsor.ca/etd/366.

Council of Science Editors:

Liu J. Factors Affecting Critical Toxicokinetic Parameters of Polychlorinated Biphenyls (PCBS) in Japanese Koi (CYPRINUS CARPIO): Effect of Diet on Chemical Assimilation and Influence of Feces to Whole Body Chemical Elimination. [Masters Thesis]. National Library of Canada; 2009. Available from: http://scholar.uwindsor.ca/etd/366

3. Angell, Robin. PCB Accumulation and Toxicokinetics in Amphibians: Accumulation Patterns' Regulation by Interspecific Differences and Toxicokinetics in Green Frogs (Rana clamitans) During Hibernation.

Degree: MS, Great Lakes Institute for Environmental Research, 2009, National Library of Canada

 This thesis examined contaminant accumulation in amphibian young of year and elimination kinetics during amphibian hibernation. Chapter 2 evaluated PCB concentrations in four amphibian species… (more)

Subjects/Keywords: Toxicology.

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APA (6th Edition):

Angell, R. (2009). PCB Accumulation and Toxicokinetics in Amphibians: Accumulation Patterns' Regulation by Interspecific Differences and Toxicokinetics in Green Frogs (Rana clamitans) During Hibernation. (Masters Thesis). National Library of Canada. Retrieved from http://scholar.uwindsor.ca/etd/359

Chicago Manual of Style (16th Edition):

Angell, Robin. “PCB Accumulation and Toxicokinetics in Amphibians: Accumulation Patterns' Regulation by Interspecific Differences and Toxicokinetics in Green Frogs (Rana clamitans) During Hibernation.” 2009. Masters Thesis, National Library of Canada. Accessed September 21, 2020. http://scholar.uwindsor.ca/etd/359.

MLA Handbook (7th Edition):

Angell, Robin. “PCB Accumulation and Toxicokinetics in Amphibians: Accumulation Patterns' Regulation by Interspecific Differences and Toxicokinetics in Green Frogs (Rana clamitans) During Hibernation.” 2009. Web. 21 Sep 2020.

Vancouver:

Angell R. PCB Accumulation and Toxicokinetics in Amphibians: Accumulation Patterns' Regulation by Interspecific Differences and Toxicokinetics in Green Frogs (Rana clamitans) During Hibernation. [Internet] [Masters thesis]. National Library of Canada; 2009. [cited 2020 Sep 21]. Available from: http://scholar.uwindsor.ca/etd/359.

Council of Science Editors:

Angell R. PCB Accumulation and Toxicokinetics in Amphibians: Accumulation Patterns' Regulation by Interspecific Differences and Toxicokinetics in Green Frogs (Rana clamitans) During Hibernation. [Masters Thesis]. National Library of Canada; 2009. Available from: http://scholar.uwindsor.ca/etd/359


UCLA

4. Malkin, Daniel. Characterizing Previously Unknown Levels of Genotoxicity Found in Lymphocyte-Mediated Pathogeneses.

Degree: Molecular Toxicology, 2016, UCLA

 The genome is constantly subjected to DNA damaging events from the environment in the form of radiation, chemicals, and pollution. Endogenous production of reactive oxygen… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Malkin, D. (2016). Characterizing Previously Unknown Levels of Genotoxicity Found in Lymphocyte-Mediated Pathogeneses. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/6sn029gb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Malkin, Daniel. “Characterizing Previously Unknown Levels of Genotoxicity Found in Lymphocyte-Mediated Pathogeneses.” 2016. Thesis, UCLA. Accessed September 21, 2020. http://www.escholarship.org/uc/item/6sn029gb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Malkin, Daniel. “Characterizing Previously Unknown Levels of Genotoxicity Found in Lymphocyte-Mediated Pathogeneses.” 2016. Web. 21 Sep 2020.

Vancouver:

Malkin D. Characterizing Previously Unknown Levels of Genotoxicity Found in Lymphocyte-Mediated Pathogeneses. [Internet] [Thesis]. UCLA; 2016. [cited 2020 Sep 21]. Available from: http://www.escholarship.org/uc/item/6sn029gb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Malkin D. Characterizing Previously Unknown Levels of Genotoxicity Found in Lymphocyte-Mediated Pathogeneses. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/6sn029gb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

5. Keebaugh, Andrew James. Mechanisms of the Exacerbation of Atherosclerosis by Semi-Volatile Organic Constituents of Inhaled Ambient Particulate Matter in apoE -/- Mice.

Degree: Environmental Toxicology, 2017, University of California – Irvine

 Emissions-related particulate matter exposures are thought to be associated with thousands of excess deaths per year, many of which are related to cardiovascular disease. Ultrafine… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Keebaugh, A. J. (2017). Mechanisms of the Exacerbation of Atherosclerosis by Semi-Volatile Organic Constituents of Inhaled Ambient Particulate Matter in apoE -/- Mice. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/5dd619sk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keebaugh, Andrew James. “Mechanisms of the Exacerbation of Atherosclerosis by Semi-Volatile Organic Constituents of Inhaled Ambient Particulate Matter in apoE -/- Mice.” 2017. Thesis, University of California – Irvine. Accessed September 21, 2020. http://www.escholarship.org/uc/item/5dd619sk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keebaugh, Andrew James. “Mechanisms of the Exacerbation of Atherosclerosis by Semi-Volatile Organic Constituents of Inhaled Ambient Particulate Matter in apoE -/- Mice.” 2017. Web. 21 Sep 2020.

Vancouver:

Keebaugh AJ. Mechanisms of the Exacerbation of Atherosclerosis by Semi-Volatile Organic Constituents of Inhaled Ambient Particulate Matter in apoE -/- Mice. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2020 Sep 21]. Available from: http://www.escholarship.org/uc/item/5dd619sk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keebaugh AJ. Mechanisms of the Exacerbation of Atherosclerosis by Semi-Volatile Organic Constituents of Inhaled Ambient Particulate Matter in apoE -/- Mice. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/5dd619sk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

6. Smith, Latasha L. Environmental toxicant effects on the dopaminergic system in the nematode Caenorhabditis elegans: Strengths and limitations of various approaches to analysis .

Degree: 2019, Duke University

  In order to develop a better understanding of the role environmental toxicants may play in the onset and progression of neurodegenerative diseases, it has… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Smith, L. L. (2019). Environmental toxicant effects on the dopaminergic system in the nematode Caenorhabditis elegans: Strengths and limitations of various approaches to analysis . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/19891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Latasha L. “Environmental toxicant effects on the dopaminergic system in the nematode Caenorhabditis elegans: Strengths and limitations of various approaches to analysis .” 2019. Thesis, Duke University. Accessed September 21, 2020. http://hdl.handle.net/10161/19891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Latasha L. “Environmental toxicant effects on the dopaminergic system in the nematode Caenorhabditis elegans: Strengths and limitations of various approaches to analysis .” 2019. Web. 21 Sep 2020.

Vancouver:

Smith LL. Environmental toxicant effects on the dopaminergic system in the nematode Caenorhabditis elegans: Strengths and limitations of various approaches to analysis . [Internet] [Thesis]. Duke University; 2019. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/10161/19891.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith LL. Environmental toxicant effects on the dopaminergic system in the nematode Caenorhabditis elegans: Strengths and limitations of various approaches to analysis . [Thesis]. Duke University; 2019. Available from: http://hdl.handle.net/10161/19891

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Santa Cruz

7. Murata, Yasuhiko. Tuna Consumption and Tuna Mercury Concentration - Implications for Human Health.

Degree: Microbiology and Environmental Toxicology, 2018, University of California – Santa Cruz

 To our knowledge, no studies have analyzed tuna consumption levels, hair mercury, and knowledge in a population, let alone a high risk population such as… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Murata, Y. (2018). Tuna Consumption and Tuna Mercury Concentration - Implications for Human Health. (Thesis). University of California – Santa Cruz. Retrieved from http://www.escholarship.org/uc/item/0v23k18c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Murata, Yasuhiko. “Tuna Consumption and Tuna Mercury Concentration - Implications for Human Health.” 2018. Thesis, University of California – Santa Cruz. Accessed September 21, 2020. http://www.escholarship.org/uc/item/0v23k18c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Murata, Yasuhiko. “Tuna Consumption and Tuna Mercury Concentration - Implications for Human Health.” 2018. Web. 21 Sep 2020.

Vancouver:

Murata Y. Tuna Consumption and Tuna Mercury Concentration - Implications for Human Health. [Internet] [Thesis]. University of California – Santa Cruz; 2018. [cited 2020 Sep 21]. Available from: http://www.escholarship.org/uc/item/0v23k18c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Murata Y. Tuna Consumption and Tuna Mercury Concentration - Implications for Human Health. [Thesis]. University of California – Santa Cruz; 2018. Available from: http://www.escholarship.org/uc/item/0v23k18c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

8. Haire, Kambria K. Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity.

Degree: 2015, University of South Florida

  Drug toxicity may cause liver injury, resulting in damage to cells and tissues. This damage can lead to cytotoxic events that may result in… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Haire, K. K. (2015). Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity. (Thesis). University of South Florida. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=3739583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haire, Kambria K. “Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity.” 2015. Thesis, University of South Florida. Accessed September 21, 2020. http://pqdtopen.proquest.com/#viewpdf?dispub=3739583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haire, Kambria K. “Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity.” 2015. Web. 21 Sep 2020.

Vancouver:

Haire KK. Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity. [Internet] [Thesis]. University of South Florida; 2015. [cited 2020 Sep 21]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=3739583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haire KK. Elucidation of the Role of Poly(ADP-Ribose) Polymerase in Drug-Induced Toxicity. [Thesis]. University of South Florida; 2015. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=3739583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

9. Chung, Elena, 1995. Characterization of transforming growth factor-β1 in β2 adrenergic receptor dysfunction and signaling mechanisms in human airway smooth muscle.

Degree: MS, Glucocorticoids, 2020, Rutgers University

 Underlying source(s) of airway smooth muscle hypercontractility and hyporesponsiveness to bronchodilatory therapy in the context of inflammatory diseases of the lung remain unclear. In asthma… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Chung, Elena, 1. (2020). Characterization of transforming growth factor-β1 in β2 adrenergic receptor dysfunction and signaling mechanisms in human airway smooth muscle. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64220/

Chicago Manual of Style (16th Edition):

Chung, Elena, 1995. “Characterization of transforming growth factor-β1 in β2 adrenergic receptor dysfunction and signaling mechanisms in human airway smooth muscle.” 2020. Masters Thesis, Rutgers University. Accessed September 21, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/64220/.

MLA Handbook (7th Edition):

Chung, Elena, 1995. “Characterization of transforming growth factor-β1 in β2 adrenergic receptor dysfunction and signaling mechanisms in human airway smooth muscle.” 2020. Web. 21 Sep 2020.

Vancouver:

Chung, Elena 1. Characterization of transforming growth factor-β1 in β2 adrenergic receptor dysfunction and signaling mechanisms in human airway smooth muscle. [Internet] [Masters thesis]. Rutgers University; 2020. [cited 2020 Sep 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64220/.

Council of Science Editors:

Chung, Elena 1. Characterization of transforming growth factor-β1 in β2 adrenergic receptor dysfunction and signaling mechanisms in human airway smooth muscle. [Masters Thesis]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64220/


Boston University

10. Vallier, Ashley. Chiral separation of the enantiomers of ephedrine, amphetamine, methamphetamine, MDA, MDMA, and phentermine in blood using LC-MS/MS.

Degree: MS, Biomedical Forensic Sciences, 2018, Boston University

 Amphetamine-type stimulants are widely abused due to their ability to stimulate the central nervous system and elicit feelings of confidence, wakefulness, mood elevation, and euphoria.… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Vallier, A. (2018). Chiral separation of the enantiomers of ephedrine, amphetamine, methamphetamine, MDA, MDMA, and phentermine in blood using LC-MS/MS. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/31283

Chicago Manual of Style (16th Edition):

Vallier, Ashley. “Chiral separation of the enantiomers of ephedrine, amphetamine, methamphetamine, MDA, MDMA, and phentermine in blood using LC-MS/MS.” 2018. Masters Thesis, Boston University. Accessed September 21, 2020. http://hdl.handle.net/2144/31283.

MLA Handbook (7th Edition):

Vallier, Ashley. “Chiral separation of the enantiomers of ephedrine, amphetamine, methamphetamine, MDA, MDMA, and phentermine in blood using LC-MS/MS.” 2018. Web. 21 Sep 2020.

Vancouver:

Vallier A. Chiral separation of the enantiomers of ephedrine, amphetamine, methamphetamine, MDA, MDMA, and phentermine in blood using LC-MS/MS. [Internet] [Masters thesis]. Boston University; 2018. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/2144/31283.

Council of Science Editors:

Vallier A. Chiral separation of the enantiomers of ephedrine, amphetamine, methamphetamine, MDA, MDMA, and phentermine in blood using LC-MS/MS. [Masters Thesis]. Boston University; 2018. Available from: http://hdl.handle.net/2144/31283


Boston University

11. Kim, Stephanie. The impact of environmental metabolic disruptors on PPARgamma transcriptional regulation of adipocyte differentiation and function.

Degree: PhD, Environmental Health, 2019, Boston University

 Metabolic homeostasis is controlled, in part, by a family of proteins called nuclear receptors through which lipophilic hormones and hormone-like molecules regulate gene expression. One… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Kim, S. (2019). The impact of environmental metabolic disruptors on PPARgamma transcriptional regulation of adipocyte differentiation and function. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/36020

Chicago Manual of Style (16th Edition):

Kim, Stephanie. “The impact of environmental metabolic disruptors on PPARgamma transcriptional regulation of adipocyte differentiation and function.” 2019. Doctoral Dissertation, Boston University. Accessed September 21, 2020. http://hdl.handle.net/2144/36020.

MLA Handbook (7th Edition):

Kim, Stephanie. “The impact of environmental metabolic disruptors on PPARgamma transcriptional regulation of adipocyte differentiation and function.” 2019. Web. 21 Sep 2020.

Vancouver:

Kim S. The impact of environmental metabolic disruptors on PPARgamma transcriptional regulation of adipocyte differentiation and function. [Internet] [Doctoral dissertation]. Boston University; 2019. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/2144/36020.

Council of Science Editors:

Kim S. The impact of environmental metabolic disruptors on PPARgamma transcriptional regulation of adipocyte differentiation and function. [Doctoral Dissertation]. Boston University; 2019. Available from: http://hdl.handle.net/2144/36020


Boston University

12. Robinson, Brianna. Determination of fentanyl qualification and quantification in Phormia regina (Meigen) (Calliphoridae) using LC-MSMS.

Degree: MS, Biomedical Forensic Sciences, 2019, Boston University

 As a primary colonizer of carrion, Phormia regina plays a vital role in both nutrient recycling and ecosystems via carrion decomposition. They are currently used… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Robinson, B. (2019). Determination of fentanyl qualification and quantification in Phormia regina (Meigen) (Calliphoridae) using LC-MSMS. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/38699

Chicago Manual of Style (16th Edition):

Robinson, Brianna. “Determination of fentanyl qualification and quantification in Phormia regina (Meigen) (Calliphoridae) using LC-MSMS.” 2019. Masters Thesis, Boston University. Accessed September 21, 2020. http://hdl.handle.net/2144/38699.

MLA Handbook (7th Edition):

Robinson, Brianna. “Determination of fentanyl qualification and quantification in Phormia regina (Meigen) (Calliphoridae) using LC-MSMS.” 2019. Web. 21 Sep 2020.

Vancouver:

Robinson B. Determination of fentanyl qualification and quantification in Phormia regina (Meigen) (Calliphoridae) using LC-MSMS. [Internet] [Masters thesis]. Boston University; 2019. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/2144/38699.

Council of Science Editors:

Robinson B. Determination of fentanyl qualification and quantification in Phormia regina (Meigen) (Calliphoridae) using LC-MSMS. [Masters Thesis]. Boston University; 2019. Available from: http://hdl.handle.net/2144/38699


University of Iowa

13. Koh, Wen Xin. Polychlorinated biphenyls and their hydroxylated metabolites in human serum from urban and rural cohorts in the United States.

Degree: PhD, Human Toxicology, 2015, University of Iowa

  In this dissertation, concentrations of 209 polychlorinated biphenyl (PCB) congeners and 64 hydroxylated PCB (OH-PCB) congeners were determined in 97 adolescents and 86 mothers… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Koh, W. X. (2015). Polychlorinated biphenyls and their hydroxylated metabolites in human serum from urban and rural cohorts in the United States. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5793

Chicago Manual of Style (16th Edition):

Koh, Wen Xin. “Polychlorinated biphenyls and their hydroxylated metabolites in human serum from urban and rural cohorts in the United States.” 2015. Doctoral Dissertation, University of Iowa. Accessed September 21, 2020. https://ir.uiowa.edu/etd/5793.

MLA Handbook (7th Edition):

Koh, Wen Xin. “Polychlorinated biphenyls and their hydroxylated metabolites in human serum from urban and rural cohorts in the United States.” 2015. Web. 21 Sep 2020.

Vancouver:

Koh WX. Polychlorinated biphenyls and their hydroxylated metabolites in human serum from urban and rural cohorts in the United States. [Internet] [Doctoral dissertation]. University of Iowa; 2015. [cited 2020 Sep 21]. Available from: https://ir.uiowa.edu/etd/5793.

Council of Science Editors:

Koh WX. Polychlorinated biphenyls and their hydroxylated metabolites in human serum from urban and rural cohorts in the United States. [Doctoral Dissertation]. University of Iowa; 2015. Available from: https://ir.uiowa.edu/etd/5793


University of Iowa

14. Chen, Xi. Glutathione suppresses the autoxidation of 2',5'-dihydroxy-4-chlorobiphenyl.

Degree: MS, Human Toxicology, 2011, University of Iowa

  Polychlorinated biphenyls (PCBs) are well known as classic environmental pollutants. Lower halogenated PCBs are mainly metabolized to mono- and di-hydroxyl compounds, and the latter… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Chen, X. (2011). Glutathione suppresses the autoxidation of 2',5'-dihydroxy-4-chlorobiphenyl. (Masters Thesis). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/1212

Chicago Manual of Style (16th Edition):

Chen, Xi. “Glutathione suppresses the autoxidation of 2',5'-dihydroxy-4-chlorobiphenyl.” 2011. Masters Thesis, University of Iowa. Accessed September 21, 2020. https://ir.uiowa.edu/etd/1212.

MLA Handbook (7th Edition):

Chen, Xi. “Glutathione suppresses the autoxidation of 2',5'-dihydroxy-4-chlorobiphenyl.” 2011. Web. 21 Sep 2020.

Vancouver:

Chen X. Glutathione suppresses the autoxidation of 2',5'-dihydroxy-4-chlorobiphenyl. [Internet] [Masters thesis]. University of Iowa; 2011. [cited 2020 Sep 21]. Available from: https://ir.uiowa.edu/etd/1212.

Council of Science Editors:

Chen X. Glutathione suppresses the autoxidation of 2',5'-dihydroxy-4-chlorobiphenyl. [Masters Thesis]. University of Iowa; 2011. Available from: https://ir.uiowa.edu/etd/1212


University of Iowa

15. Roh, Taehyun. Exposure to arsenic and atrazine from drinking water and risk of cancer.

Degree: PhD, Human Toxicology, 2016, University of Iowa

  Arsenic and atrazine are two water contaminants of high public health concern in Iowa. Arsenic is released into drinking water from soil and atrazine… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Roh, T. (2016). Exposure to arsenic and atrazine from drinking water and risk of cancer. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2265

Chicago Manual of Style (16th Edition):

Roh, Taehyun. “Exposure to arsenic and atrazine from drinking water and risk of cancer.” 2016. Doctoral Dissertation, University of Iowa. Accessed September 21, 2020. https://ir.uiowa.edu/etd/2265.

MLA Handbook (7th Edition):

Roh, Taehyun. “Exposure to arsenic and atrazine from drinking water and risk of cancer.” 2016. Web. 21 Sep 2020.

Vancouver:

Roh T. Exposure to arsenic and atrazine from drinking water and risk of cancer. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2020 Sep 21]. Available from: https://ir.uiowa.edu/etd/2265.

Council of Science Editors:

Roh T. Exposure to arsenic and atrazine from drinking water and risk of cancer. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/2265

16. Anantharam, Poojya Vellareddy. Development of mouse models of hydrogen sulfide-induced neurotoxicity for the evaluation of neuroprotective strategies.

Degree: 2017, Iowa State University

 H2S is a colorless gas with a strong “rotten egg” odor. While at physiological concentrations, H2S is a vital gasotransmitter; exposure to high concentrations can… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Anantharam, P. V. (2017). Development of mouse models of hydrogen sulfide-induced neurotoxicity for the evaluation of neuroprotective strategies. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/16704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anantharam, Poojya Vellareddy. “Development of mouse models of hydrogen sulfide-induced neurotoxicity for the evaluation of neuroprotective strategies.” 2017. Thesis, Iowa State University. Accessed September 21, 2020. https://lib.dr.iastate.edu/etd/16704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anantharam, Poojya Vellareddy. “Development of mouse models of hydrogen sulfide-induced neurotoxicity for the evaluation of neuroprotective strategies.” 2017. Web. 21 Sep 2020.

Vancouver:

Anantharam PV. Development of mouse models of hydrogen sulfide-induced neurotoxicity for the evaluation of neuroprotective strategies. [Internet] [Thesis]. Iowa State University; 2017. [cited 2020 Sep 21]. Available from: https://lib.dr.iastate.edu/etd/16704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anantharam PV. Development of mouse models of hydrogen sulfide-induced neurotoxicity for the evaluation of neuroprotective strategies. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/16704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Langley, Monica. Characterization of the MitoPark mouse model of Parkinson’s disease for neurotoxicity and neuroprotection studies.

Degree: 2017, Iowa State University

 Parkinson’s disease (PD) is a chronic, progressive neurodegenerative disorder affecting about one million people in the United States and an estimated 10 million people worldwide.… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Langley, M. (2017). Characterization of the MitoPark mouse model of Parkinson’s disease for neurotoxicity and neuroprotection studies. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/16930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Langley, Monica. “Characterization of the MitoPark mouse model of Parkinson’s disease for neurotoxicity and neuroprotection studies.” 2017. Thesis, Iowa State University. Accessed September 21, 2020. https://lib.dr.iastate.edu/etd/16930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Langley, Monica. “Characterization of the MitoPark mouse model of Parkinson’s disease for neurotoxicity and neuroprotection studies.” 2017. Web. 21 Sep 2020.

Vancouver:

Langley M. Characterization of the MitoPark mouse model of Parkinson’s disease for neurotoxicity and neuroprotection studies. [Internet] [Thesis]. Iowa State University; 2017. [cited 2020 Sep 21]. Available from: https://lib.dr.iastate.edu/etd/16930.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Langley M. Characterization of the MitoPark mouse model of Parkinson’s disease for neurotoxicity and neuroprotection studies. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/16930

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

18. Luo, Jie. Prokineticin 2 signaling: Genetic regulation and preclinical assessment in rodent models of Parkinsonism.

Degree: 2018, Iowa State University

 Parkinson’s disease (PD) is the second most common neurodegenerative disorder after Alzheimer’s disease. The etiology of PD is currently not fully understood, but strong evidence… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Luo, J. (2018). Prokineticin 2 signaling: Genetic regulation and preclinical assessment in rodent models of Parkinsonism. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/17254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luo, Jie. “Prokineticin 2 signaling: Genetic regulation and preclinical assessment in rodent models of Parkinsonism.” 2018. Thesis, Iowa State University. Accessed September 21, 2020. https://lib.dr.iastate.edu/etd/17254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luo, Jie. “Prokineticin 2 signaling: Genetic regulation and preclinical assessment in rodent models of Parkinsonism.” 2018. Web. 21 Sep 2020.

Vancouver:

Luo J. Prokineticin 2 signaling: Genetic regulation and preclinical assessment in rodent models of Parkinsonism. [Internet] [Thesis]. Iowa State University; 2018. [cited 2020 Sep 21]. Available from: https://lib.dr.iastate.edu/etd/17254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luo J. Prokineticin 2 signaling: Genetic regulation and preclinical assessment in rodent models of Parkinsonism. [Thesis]. Iowa State University; 2018. Available from: https://lib.dr.iastate.edu/etd/17254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Abongwa, Melanie. Potential new drug targets and therapeutic approaches for parasitic nematode infections.

Degree: 2017, Iowa State University

 Parasitic nematode infections remain a serious global public health threat to humans and animals. These infections cause debilitating conditions in humans and significant economic losses… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Abongwa, M. (2017). Potential new drug targets and therapeutic approaches for parasitic nematode infections. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/15240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abongwa, Melanie. “Potential new drug targets and therapeutic approaches for parasitic nematode infections.” 2017. Thesis, Iowa State University. Accessed September 21, 2020. https://lib.dr.iastate.edu/etd/15240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abongwa, Melanie. “Potential new drug targets and therapeutic approaches for parasitic nematode infections.” 2017. Web. 21 Sep 2020.

Vancouver:

Abongwa M. Potential new drug targets and therapeutic approaches for parasitic nematode infections. [Internet] [Thesis]. Iowa State University; 2017. [cited 2020 Sep 21]. Available from: https://lib.dr.iastate.edu/etd/15240.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abongwa M. Potential new drug targets and therapeutic approaches for parasitic nematode infections. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/15240

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Iowa

20. Krishnan, Preethi. Spectral binding of multiple ortho substituted polychlorinated biphenyls and their 5-hydroxy metabolites to microsomal cytochrome P450 enzymes.

Degree: MS, Human Toxicology, 2011, University of Iowa

  Polychlorinated biphenyls (PCBs) are persistent organic pollutants that are carcinogenic and cause a variety of adverse health effects to the nervous, reproductive and endocrine… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Krishnan, P. (2011). Spectral binding of multiple ortho substituted polychlorinated biphenyls and their 5-hydroxy metabolites to microsomal cytochrome P450 enzymes. (Masters Thesis). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2733

Chicago Manual of Style (16th Edition):

Krishnan, Preethi. “Spectral binding of multiple ortho substituted polychlorinated biphenyls and their 5-hydroxy metabolites to microsomal cytochrome P450 enzymes.” 2011. Masters Thesis, University of Iowa. Accessed September 21, 2020. https://ir.uiowa.edu/etd/2733.

MLA Handbook (7th Edition):

Krishnan, Preethi. “Spectral binding of multiple ortho substituted polychlorinated biphenyls and their 5-hydroxy metabolites to microsomal cytochrome P450 enzymes.” 2011. Web. 21 Sep 2020.

Vancouver:

Krishnan P. Spectral binding of multiple ortho substituted polychlorinated biphenyls and their 5-hydroxy metabolites to microsomal cytochrome P450 enzymes. [Internet] [Masters thesis]. University of Iowa; 2011. [cited 2020 Sep 21]. Available from: https://ir.uiowa.edu/etd/2733.

Council of Science Editors:

Krishnan P. Spectral binding of multiple ortho substituted polychlorinated biphenyls and their 5-hydroxy metabolites to microsomal cytochrome P450 enzymes. [Masters Thesis]. University of Iowa; 2011. Available from: https://ir.uiowa.edu/etd/2733


University of Iowa

21. Mapuskar, Kranti Ashok. The role Of sirtuin3 in regulating mitochondrial superoxide metabolism in response to ionizing radiation in mouse embryonic fibroblasts.

Degree: PhD, Human Toxicology, 2013, University of Iowa

  Exposure of mammalian cells to ionizing radiation is believed to affect mitochondrial metabolism, which can lead to increased levels of superoxide (O2*-). Sirtuin3 (Sirt3)… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Mapuskar, K. A. (2013). The role Of sirtuin3 in regulating mitochondrial superoxide metabolism in response to ionizing radiation in mouse embryonic fibroblasts. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/4688

Chicago Manual of Style (16th Edition):

Mapuskar, Kranti Ashok. “The role Of sirtuin3 in regulating mitochondrial superoxide metabolism in response to ionizing radiation in mouse embryonic fibroblasts.” 2013. Doctoral Dissertation, University of Iowa. Accessed September 21, 2020. https://ir.uiowa.edu/etd/4688.

MLA Handbook (7th Edition):

Mapuskar, Kranti Ashok. “The role Of sirtuin3 in regulating mitochondrial superoxide metabolism in response to ionizing radiation in mouse embryonic fibroblasts.” 2013. Web. 21 Sep 2020.

Vancouver:

Mapuskar KA. The role Of sirtuin3 in regulating mitochondrial superoxide metabolism in response to ionizing radiation in mouse embryonic fibroblasts. [Internet] [Doctoral dissertation]. University of Iowa; 2013. [cited 2020 Sep 21]. Available from: https://ir.uiowa.edu/etd/4688.

Council of Science Editors:

Mapuskar KA. The role Of sirtuin3 in regulating mitochondrial superoxide metabolism in response to ionizing radiation in mouse embryonic fibroblasts. [Doctoral Dissertation]. University of Iowa; 2013. Available from: https://ir.uiowa.edu/etd/4688


University of Iowa

22. Doskey, Claire Marie. Toward absolute quantitation in cell culture: expression of dose of xenobiotics and capacity of cells to remove hydrogen peroxide with implications to pharmacological ascorbate in cancer therapy.

Degree: PhD, Human Toxicology, 2016, University of Iowa

  Basic health science research, which includes cell culture, typically underpins clinical and toxicological research. The results are used to predict biological effects of xenobiotics,… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Doskey, C. M. (2016). Toward absolute quantitation in cell culture: expression of dose of xenobiotics and capacity of cells to remove hydrogen peroxide with implications to pharmacological ascorbate in cancer therapy. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/5459

Chicago Manual of Style (16th Edition):

Doskey, Claire Marie. “Toward absolute quantitation in cell culture: expression of dose of xenobiotics and capacity of cells to remove hydrogen peroxide with implications to pharmacological ascorbate in cancer therapy.” 2016. Doctoral Dissertation, University of Iowa. Accessed September 21, 2020. https://ir.uiowa.edu/etd/5459.

MLA Handbook (7th Edition):

Doskey, Claire Marie. “Toward absolute quantitation in cell culture: expression of dose of xenobiotics and capacity of cells to remove hydrogen peroxide with implications to pharmacological ascorbate in cancer therapy.” 2016. Web. 21 Sep 2020.

Vancouver:

Doskey CM. Toward absolute quantitation in cell culture: expression of dose of xenobiotics and capacity of cells to remove hydrogen peroxide with implications to pharmacological ascorbate in cancer therapy. [Internet] [Doctoral dissertation]. University of Iowa; 2016. [cited 2020 Sep 21]. Available from: https://ir.uiowa.edu/etd/5459.

Council of Science Editors:

Doskey CM. Toward absolute quantitation in cell culture: expression of dose of xenobiotics and capacity of cells to remove hydrogen peroxide with implications to pharmacological ascorbate in cancer therapy. [Doctoral Dissertation]. University of Iowa; 2016. Available from: https://ir.uiowa.edu/etd/5459


Queens University

23. Stokes, Samantha. Examining Altered NF-κB Signalling Following 1,4- Benzoquinone Exposure in HD3 Cells .

Degree: Pharmacology and Toxicology, 2013, Queens University

 In utero exposure to benzene, a known environmental contaminant, is associated with increased risk of childhood leukemia. We have previously shown that in utero benzene… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Stokes, S. (2013). Examining Altered NF-κB Signalling Following 1,4- Benzoquinone Exposure in HD3 Cells . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stokes, Samantha. “Examining Altered NF-κB Signalling Following 1,4- Benzoquinone Exposure in HD3 Cells .” 2013. Thesis, Queens University. Accessed September 21, 2020. http://hdl.handle.net/1974/8228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stokes, Samantha. “Examining Altered NF-κB Signalling Following 1,4- Benzoquinone Exposure in HD3 Cells .” 2013. Web. 21 Sep 2020.

Vancouver:

Stokes S. Examining Altered NF-κB Signalling Following 1,4- Benzoquinone Exposure in HD3 Cells . [Internet] [Thesis]. Queens University; 2013. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/1974/8228.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stokes S. Examining Altered NF-κB Signalling Following 1,4- Benzoquinone Exposure in HD3 Cells . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8228

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

24. Xie, Yuchao. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen is the most prevalent cause of acute liver failure (ALF) and drug-induced liver injury (DILI) in western countries. Extensive studies have revealed important intracellular… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Xie, Y. (2016). ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/22502

Chicago Manual of Style (16th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Doctoral Dissertation, University of Kansas. Accessed September 21, 2020. http://hdl.handle.net/1808/22502.

MLA Handbook (7th Edition):

Xie, Yuchao. “ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE.” 2016. Web. 21 Sep 2020.

Vancouver:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Internet] [Doctoral dissertation]. University of Kansas; 2016. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/1808/22502.

Council of Science Editors:

Xie Y. ACETAMINOPHEN HEPATOTOXICITY IN PRIMARY HUMAN HEPATOCYTES AND MICE. [Doctoral Dissertation]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/22502


University of Kansas

25. Poudel, Samikshya. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.

Degree: MS, Pharmacology, Toxicology & Therapeutics, 2016, University of Kansas

 Acetaminophen (APAP) overdose is the major cause of Acute Liver Failure (ALF) in the Western world. Treatment options for APAP-induced ALF are limited. Studies have… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Poudel, S. (2016). Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. (Masters Thesis). University of Kansas. Retrieved from http://hdl.handle.net/1808/24805

Chicago Manual of Style (16th Edition):

Poudel, Samikshya. “Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.” 2016. Masters Thesis, University of Kansas. Accessed September 21, 2020. http://hdl.handle.net/1808/24805.

MLA Handbook (7th Edition):

Poudel, Samikshya. “Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose.” 2016. Web. 21 Sep 2020.

Vancouver:

Poudel S. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. [Internet] [Masters thesis]. University of Kansas; 2016. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/1808/24805.

Council of Science Editors:

Poudel S. Role of Yes-Associated Protein (YAP) in Liver Injury and Regeneration following Acetaminophen Overdose. [Masters Thesis]. University of Kansas; 2016. Available from: http://hdl.handle.net/1808/24805


University of Kansas

26. Huck, Ian. Role of HNF4a in Liver Regeneration, Liver Cancer Pathogenesis and Global Metabolism.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2018, University of Kansas

 Liver is the central metabolic organ and performs many functions necessary for survival. Hepatocyte Nuclear Factor 4 alpha (HNF4a) is a nuclear receptor well characterized… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Huck, I. (2018). Role of HNF4a in Liver Regeneration, Liver Cancer Pathogenesis and Global Metabolism. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/30113

Chicago Manual of Style (16th Edition):

Huck, Ian. “Role of HNF4a in Liver Regeneration, Liver Cancer Pathogenesis and Global Metabolism.” 2018. Doctoral Dissertation, University of Kansas. Accessed September 21, 2020. http://hdl.handle.net/1808/30113.

MLA Handbook (7th Edition):

Huck, Ian. “Role of HNF4a in Liver Regeneration, Liver Cancer Pathogenesis and Global Metabolism.” 2018. Web. 21 Sep 2020.

Vancouver:

Huck I. Role of HNF4a in Liver Regeneration, Liver Cancer Pathogenesis and Global Metabolism. [Internet] [Doctoral dissertation]. University of Kansas; 2018. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/1808/30113.

Council of Science Editors:

Huck I. Role of HNF4a in Liver Regeneration, Liver Cancer Pathogenesis and Global Metabolism. [Doctoral Dissertation]. University of Kansas; 2018. Available from: http://hdl.handle.net/1808/30113


Loyola University Chicago

27. Reynolds, Erik Joseph. Chronic Effects of Lead Exposure on Atherinops Affinis (topsmelt): Influence of Salinity and Organism Age.

Degree: MS, Biology, 2017, Loyola University Chicago

  Pb (lead) appears in the environment as a consequence of both natural and anthropogenic processes. Mining, smelting, coal burning, lead acid batteries, and cement… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Reynolds, E. J. (2017). Chronic Effects of Lead Exposure on Atherinops Affinis (topsmelt): Influence of Salinity and Organism Age. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/3702

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reynolds, Erik Joseph. “Chronic Effects of Lead Exposure on Atherinops Affinis (topsmelt): Influence of Salinity and Organism Age.” 2017. Thesis, Loyola University Chicago. Accessed September 21, 2020. https://ecommons.luc.edu/luc_theses/3702.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reynolds, Erik Joseph. “Chronic Effects of Lead Exposure on Atherinops Affinis (topsmelt): Influence of Salinity and Organism Age.” 2017. Web. 21 Sep 2020.

Vancouver:

Reynolds EJ. Chronic Effects of Lead Exposure on Atherinops Affinis (topsmelt): Influence of Salinity and Organism Age. [Internet] [Thesis]. Loyola University Chicago; 2017. [cited 2020 Sep 21]. Available from: https://ecommons.luc.edu/luc_theses/3702.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reynolds EJ. Chronic Effects of Lead Exposure on Atherinops Affinis (topsmelt): Influence of Salinity and Organism Age. [Thesis]. Loyola University Chicago; 2017. Available from: https://ecommons.luc.edu/luc_theses/3702

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

28. Saitta, Kyle S., 1989-. The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion.

Degree: PhD, BDNF, 2020, Rutgers University

Demyelinating diseases or demyelination in response to toxic agents can be debilitating to patients and can greatly impair the quality of life. Therefore, new therapeutic… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Saitta, Kyle S., 1. (2020). The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64381/

Chicago Manual of Style (16th Edition):

Saitta, Kyle S., 1989-. “The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion.” 2020. Doctoral Dissertation, Rutgers University. Accessed September 21, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/64381/.

MLA Handbook (7th Edition):

Saitta, Kyle S., 1989-. “The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion.” 2020. Web. 21 Sep 2020.

Vancouver:

Saitta, Kyle S. 1. The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2020 Sep 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64381/.

Council of Science Editors:

Saitta, Kyle S. 1. The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64381/


Rutgers University

29. Shah, Raj D. Role of GRM1 in altering glutamate metabolism and bioavailability.

Degree: PhD, Glutamate, 2020, Rutgers University

 Altered metabolic activity has been implicated in several types of cancer including malignant melanoma. Previously, we have illustrated the role of a neuronal receptor, metabotropic… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Shah, R. D. (2020). Role of GRM1 in altering glutamate metabolism and bioavailability. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64382/

Chicago Manual of Style (16th Edition):

Shah, Raj D. “Role of GRM1 in altering glutamate metabolism and bioavailability.” 2020. Doctoral Dissertation, Rutgers University. Accessed September 21, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/64382/.

MLA Handbook (7th Edition):

Shah, Raj D. “Role of GRM1 in altering glutamate metabolism and bioavailability.” 2020. Web. 21 Sep 2020.

Vancouver:

Shah RD. Role of GRM1 in altering glutamate metabolism and bioavailability. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2020 Sep 21]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64382/.

Council of Science Editors:

Shah RD. Role of GRM1 in altering glutamate metabolism and bioavailability. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64382/

30. Omoruyi, Iyekhoetin Matthew. MUTAGENIC AND OESTROGENIC ACTIVITIES OF COMMERCIALLY PROCESSED FOOD ITEMS AND WATER SAMPLES: A COMPARISON BETWEEN FINLAND AND NIGERIA.

Degree: Faculty of Veterinary Medicine, Food Hygiene and Environmental Health, 2015, University of Helsinki

 Commercially processed food, drinking-water sources and effluent waters discharged into bodies of water from wastewater treatment plants are putative but yet poorly delineated sources of… (more)

Subjects/Keywords: toxicology; toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Omoruyi, I. M. (2015). MUTAGENIC AND OESTROGENIC ACTIVITIES OF COMMERCIALLY PROCESSED FOOD ITEMS AND WATER SAMPLES: A COMPARISON BETWEEN FINLAND AND NIGERIA. (Doctoral Dissertation). University of Helsinki. Retrieved from http://hdl.handle.net/10138/157405

Chicago Manual of Style (16th Edition):

Omoruyi, Iyekhoetin Matthew. “MUTAGENIC AND OESTROGENIC ACTIVITIES OF COMMERCIALLY PROCESSED FOOD ITEMS AND WATER SAMPLES: A COMPARISON BETWEEN FINLAND AND NIGERIA.” 2015. Doctoral Dissertation, University of Helsinki. Accessed September 21, 2020. http://hdl.handle.net/10138/157405.

MLA Handbook (7th Edition):

Omoruyi, Iyekhoetin Matthew. “MUTAGENIC AND OESTROGENIC ACTIVITIES OF COMMERCIALLY PROCESSED FOOD ITEMS AND WATER SAMPLES: A COMPARISON BETWEEN FINLAND AND NIGERIA.” 2015. Web. 21 Sep 2020.

Vancouver:

Omoruyi IM. MUTAGENIC AND OESTROGENIC ACTIVITIES OF COMMERCIALLY PROCESSED FOOD ITEMS AND WATER SAMPLES: A COMPARISON BETWEEN FINLAND AND NIGERIA. [Internet] [Doctoral dissertation]. University of Helsinki; 2015. [cited 2020 Sep 21]. Available from: http://hdl.handle.net/10138/157405.

Council of Science Editors:

Omoruyi IM. MUTAGENIC AND OESTROGENIC ACTIVITIES OF COMMERCIALLY PROCESSED FOOD ITEMS AND WATER SAMPLES: A COMPARISON BETWEEN FINLAND AND NIGERIA. [Doctoral Dissertation]. University of Helsinki; 2015. Available from: http://hdl.handle.net/10138/157405

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