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You searched for subject:(store operated calcium entry). Showing records 1 – 30 of 6838 total matches.

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University of Alberta

1. Prins, Daniel S. Insights into the Regulation of Store-operated Calcium Entry.

Degree: PhD, Department of Biochemistry, 2015, University of Alberta

Calcium (Ca2+) ions serve a crucial role in numerous intracellular signaling pathways, controlling physiological functions as diverse as cell proliferation, immune system function, and muscular… (more)

Subjects/Keywords: STIM1; store-operated calcium entry

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APA (6th Edition):

Prins, D. S. (2015). Insights into the Regulation of Store-operated Calcium Entry. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/j6731653j

Chicago Manual of Style (16th Edition):

Prins, Daniel S. “Insights into the Regulation of Store-operated Calcium Entry.” 2015. Doctoral Dissertation, University of Alberta. Accessed October 31, 2020. https://era.library.ualberta.ca/files/j6731653j.

MLA Handbook (7th Edition):

Prins, Daniel S. “Insights into the Regulation of Store-operated Calcium Entry.” 2015. Web. 31 Oct 2020.

Vancouver:

Prins DS. Insights into the Regulation of Store-operated Calcium Entry. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2020 Oct 31]. Available from: https://era.library.ualberta.ca/files/j6731653j.

Council of Science Editors:

Prins DS. Insights into the Regulation of Store-operated Calcium Entry. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/j6731653j


University of Rochester

2. Carrell, Ellie M. Short- and Long-term Roles for Orai1 in Skeletal Muscle Structure and Function.

Degree: PhD, 2016, University of Rochester

 Skeletal muscle cells are specialized for rapid and efficient intracellular calcium (Ca2+) release and reuptake. During stimulation, Ca2+ ions are released from the high capacity… (more)

Subjects/Keywords: Myosin; Development; Hypertrophy; Store-operated calcium entry

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APA (6th Edition):

Carrell, E. M. (2016). Short- and Long-term Roles for Orai1 in Skeletal Muscle Structure and Function. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/31494

Chicago Manual of Style (16th Edition):

Carrell, Ellie M. “Short- and Long-term Roles for Orai1 in Skeletal Muscle Structure and Function.” 2016. Doctoral Dissertation, University of Rochester. Accessed October 31, 2020. http://hdl.handle.net/1802/31494.

MLA Handbook (7th Edition):

Carrell, Ellie M. “Short- and Long-term Roles for Orai1 in Skeletal Muscle Structure and Function.” 2016. Web. 31 Oct 2020.

Vancouver:

Carrell EM. Short- and Long-term Roles for Orai1 in Skeletal Muscle Structure and Function. [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/1802/31494.

Council of Science Editors:

Carrell EM. Short- and Long-term Roles for Orai1 in Skeletal Muscle Structure and Function. [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/31494


University of Tasmania

3. Mitchell, CB. STIM1 mediates multiple signalling pathways in neuronal growth cones.

Degree: 2014, University of Tasmania

Calcium is an intracellular second messenger that is vital for normal neuronal function. The maintenance of calcium homeostasis is critical for healthy neuronal function, and… (more)

Subjects/Keywords: STIM1; store-operated calcium entry; neuron; growth cone; calcium signalling

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APA (6th Edition):

Mitchell, C. (2014). STIM1 mediates multiple signalling pathways in neuronal growth cones. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/18740/1/Front-Mitchell-thesis.pdf ; https://eprints.utas.edu.au/18740/2/Whole-Mitchell-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mitchell, CB. “STIM1 mediates multiple signalling pathways in neuronal growth cones.” 2014. Thesis, University of Tasmania. Accessed October 31, 2020. https://eprints.utas.edu.au/18740/1/Front-Mitchell-thesis.pdf ; https://eprints.utas.edu.au/18740/2/Whole-Mitchell-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mitchell, CB. “STIM1 mediates multiple signalling pathways in neuronal growth cones.” 2014. Web. 31 Oct 2020.

Vancouver:

Mitchell C. STIM1 mediates multiple signalling pathways in neuronal growth cones. [Internet] [Thesis]. University of Tasmania; 2014. [cited 2020 Oct 31]. Available from: https://eprints.utas.edu.au/18740/1/Front-Mitchell-thesis.pdf ; https://eprints.utas.edu.au/18740/2/Whole-Mitchell-thesis.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mitchell C. STIM1 mediates multiple signalling pathways in neuronal growth cones. [Thesis]. University of Tasmania; 2014. Available from: https://eprints.utas.edu.au/18740/1/Front-Mitchell-thesis.pdf ; https://eprints.utas.edu.au/18740/2/Whole-Mitchell-thesis.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Thompson, AC. Deciphering the role of STIM1 in nervous system development.

Degree: 2017, University of Tasmania

 The wiring of the nervous system during development requires each neuron to find and connect with a correct synaptic target in a process known as… (more)

Subjects/Keywords: STIM1; store-operated calcium entry; calcium; axon pathfinding; nervous system development

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APA (6th Edition):

Thompson, A. (2017). Deciphering the role of STIM1 in nervous system development. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/27187/1/Thompson_whole_thesis.pdf ; Thompson, AC ORCID: 0000-0003-4041-8439 <https://orcid.org/0000-0003-4041-8439> 2017 , 'Deciphering the role of STIM1 in nervous system development', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thompson, AC. “Deciphering the role of STIM1 in nervous system development.” 2017. Thesis, University of Tasmania. Accessed October 31, 2020. https://eprints.utas.edu.au/27187/1/Thompson_whole_thesis.pdf ; Thompson, AC ORCID: 0000-0003-4041-8439 <https://orcid.org/0000-0003-4041-8439> 2017 , 'Deciphering the role of STIM1 in nervous system development', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thompson, AC. “Deciphering the role of STIM1 in nervous system development.” 2017. Web. 31 Oct 2020.

Vancouver:

Thompson A. Deciphering the role of STIM1 in nervous system development. [Internet] [Thesis]. University of Tasmania; 2017. [cited 2020 Oct 31]. Available from: https://eprints.utas.edu.au/27187/1/Thompson_whole_thesis.pdf ; Thompson, AC ORCID: 0000-0003-4041-8439 <https://orcid.org/0000-0003-4041-8439> 2017 , 'Deciphering the role of STIM1 in nervous system development', PhD thesis, University of Tasmania..

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thompson A. Deciphering the role of STIM1 in nervous system development. [Thesis]. University of Tasmania; 2017. Available from: https://eprints.utas.edu.au/27187/1/Thompson_whole_thesis.pdf ; Thompson, AC ORCID: 0000-0003-4041-8439 <https://orcid.org/0000-0003-4041-8439> 2017 , 'Deciphering the role of STIM1 in nervous system development', PhD thesis, University of Tasmania.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Boutry, Maxime. Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11 : Lysosomal dysfunctions and neurodegenerescence : the example of spastic paraplegia type SPG11.

Degree: Docteur es, Neurosciences, 2017, Université Pierre et Marie Curie – Paris VI

Les lysosomes sont importants pour la survie et la fonction des cellules du système nerveux central et en particulier des neurones. Le mécanisme de la… (more)

Subjects/Keywords: Lysosome; Reformation des lysosomes; Gangliosides; Cholestérol; Maladies neurodégénératives; Store-Operated Calcium entry (SOCE); Lysosome; Neurodegeneration; Store-Operated Calcium entry (SOCE); 573.8

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APA (6th Edition):

Boutry, M. (2017). Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11 : Lysosomal dysfunctions and neurodegenerescence : the example of spastic paraplegia type SPG11. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066295

Chicago Manual of Style (16th Edition):

Boutry, Maxime. “Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11 : Lysosomal dysfunctions and neurodegenerescence : the example of spastic paraplegia type SPG11.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 31, 2020. http://www.theses.fr/2017PA066295.

MLA Handbook (7th Edition):

Boutry, Maxime. “Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11 : Lysosomal dysfunctions and neurodegenerescence : the example of spastic paraplegia type SPG11.” 2017. Web. 31 Oct 2020.

Vancouver:

Boutry M. Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11 : Lysosomal dysfunctions and neurodegenerescence : the example of spastic paraplegia type SPG11. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2020 Oct 31]. Available from: http://www.theses.fr/2017PA066295.

Council of Science Editors:

Boutry M. Dysfonctions des lysosomes et neurodégénérescence : l'exemple de la paraplégie spastique de type SPG11 : Lysosomal dysfunctions and neurodegenerescence : the example of spastic paraplegia type SPG11. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066295


Cornell University

6. Midthun, Kari. Associations Of Stim1 During Store-Operated Calcium Entry And Development Of A Novel Method For Multi-Biomolecule Patterning.

Degree: PhD, Chemistry and Chemical Biology, 2013, Cornell University

 To understand immune responses, such as asthma and allergies, we must first understand the inter- and intramolecular regulation of the signaling pathways responsible for these… (more)

Subjects/Keywords: STIM1; store-operated calcium entry; imprint lithography; multi-biomolecule patterning

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APA (6th Edition):

Midthun, K. (2013). Associations Of Stim1 During Store-Operated Calcium Entry And Development Of A Novel Method For Multi-Biomolecule Patterning. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/33889

Chicago Manual of Style (16th Edition):

Midthun, Kari. “Associations Of Stim1 During Store-Operated Calcium Entry And Development Of A Novel Method For Multi-Biomolecule Patterning.” 2013. Doctoral Dissertation, Cornell University. Accessed October 31, 2020. http://hdl.handle.net/1813/33889.

MLA Handbook (7th Edition):

Midthun, Kari. “Associations Of Stim1 During Store-Operated Calcium Entry And Development Of A Novel Method For Multi-Biomolecule Patterning.” 2013. Web. 31 Oct 2020.

Vancouver:

Midthun K. Associations Of Stim1 During Store-Operated Calcium Entry And Development Of A Novel Method For Multi-Biomolecule Patterning. [Internet] [Doctoral dissertation]. Cornell University; 2013. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/1813/33889.

Council of Science Editors:

Midthun K. Associations Of Stim1 During Store-Operated Calcium Entry And Development Of A Novel Method For Multi-Biomolecule Patterning. [Doctoral Dissertation]. Cornell University; 2013. Available from: http://hdl.handle.net/1813/33889


University of Pennsylvania

7. Dolan, Andrew Thomas. Systems Modeling of Calcium Homeostasis and Mobilization in Platelets Mediated by Ip3 and Store-Operated Calcium Entry.

Degree: 2014, University of Pennsylvania

 Platelet aggregation is one of the body's first responses to vascular damage to prevent blood loss; upon injury to the endothelium platelets react to the… (more)

Subjects/Keywords: platelets; signal transduction; store-operated calcium entry; systems biology; Chemical Engineering

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APA (6th Edition):

Dolan, A. T. (2014). Systems Modeling of Calcium Homeostasis and Mobilization in Platelets Mediated by Ip3 and Store-Operated Calcium Entry. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1262

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dolan, Andrew Thomas. “Systems Modeling of Calcium Homeostasis and Mobilization in Platelets Mediated by Ip3 and Store-Operated Calcium Entry.” 2014. Thesis, University of Pennsylvania. Accessed October 31, 2020. https://repository.upenn.edu/edissertations/1262.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dolan, Andrew Thomas. “Systems Modeling of Calcium Homeostasis and Mobilization in Platelets Mediated by Ip3 and Store-Operated Calcium Entry.” 2014. Web. 31 Oct 2020.

Vancouver:

Dolan AT. Systems Modeling of Calcium Homeostasis and Mobilization in Platelets Mediated by Ip3 and Store-Operated Calcium Entry. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2020 Oct 31]. Available from: https://repository.upenn.edu/edissertations/1262.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dolan AT. Systems Modeling of Calcium Homeostasis and Mobilization in Platelets Mediated by Ip3 and Store-Operated Calcium Entry. [Thesis]. University of Pennsylvania; 2014. Available from: https://repository.upenn.edu/edissertations/1262

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Extremadura

8. Diez Bello, Raquel. Papel del remodelado de la entrada de calcio en el desarrollo tumoral .

Degree: 2019, Universidad de Extremadura

 El ion calcio y la entrada de éste desde el medio extracelular hasta el citoplasma, a través de canales localizados en la membrana plasmástica, juegan… (more)

Subjects/Keywords: Entrada capacitativa de calcio; Cáncer; Homeostasis de calcio; Store-operated calcium entry; Cancer; Calcium homeostasis

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APA (6th Edition):

Diez Bello, R. (2019). Papel del remodelado de la entrada de calcio en el desarrollo tumoral . (Thesis). Universidad de Extremadura. Retrieved from http://hdl.handle.net/10662/9715

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Diez Bello, Raquel. “Papel del remodelado de la entrada de calcio en el desarrollo tumoral .” 2019. Thesis, Universidad de Extremadura. Accessed October 31, 2020. http://hdl.handle.net/10662/9715.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Diez Bello, Raquel. “Papel del remodelado de la entrada de calcio en el desarrollo tumoral .” 2019. Web. 31 Oct 2020.

Vancouver:

Diez Bello R. Papel del remodelado de la entrada de calcio en el desarrollo tumoral . [Internet] [Thesis]. Universidad de Extremadura; 2019. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10662/9715.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Diez Bello R. Papel del remodelado de la entrada de calcio en el desarrollo tumoral . [Thesis]. Universidad de Extremadura; 2019. Available from: http://hdl.handle.net/10662/9715

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

9. Fenech, Melissa. The Secretory Pathway Calcium ATPase 2C promotes increased cytosolic calcium levels.

Degree: 2019, University of Western Ontario

 Pancreatic acinar cell exocytosis requires precise calcium (Ca2+) signals. When cytosolic Ca2+ levels remain high, cellular functions are disrupted, which is associated with initiation of… (more)

Subjects/Keywords: calcium homeostasis; calcium signaling; secretory pathway calcium ATPase 2C; Orai1; store operated calcium entry; store independent calcium entry; STIM1; Cellular and Molecular Physiology

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APA (6th Edition):

Fenech, M. (2019). The Secretory Pathway Calcium ATPase 2C promotes increased cytosolic calcium levels. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fenech, Melissa. “The Secretory Pathway Calcium ATPase 2C promotes increased cytosolic calcium levels.” 2019. Thesis, University of Western Ontario. Accessed October 31, 2020. https://ir.lib.uwo.ca/etd/6689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fenech, Melissa. “The Secretory Pathway Calcium ATPase 2C promotes increased cytosolic calcium levels.” 2019. Web. 31 Oct 2020.

Vancouver:

Fenech M. The Secretory Pathway Calcium ATPase 2C promotes increased cytosolic calcium levels. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2020 Oct 31]. Available from: https://ir.lib.uwo.ca/etd/6689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fenech M. The Secretory Pathway Calcium ATPase 2C promotes increased cytosolic calcium levels. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

10. Deng, Xiaoxiang. Dissecting the mechanism of STIM coupling to Orai.

Degree: PhD, 2011, Temple University

Biochemistry

Store-operated Ca2+ entry (SOCE) triggered by the depletion of endoplasmic reticulum (ER) luminal Ca2+ stores is a major Ca2+ entry pathway in non-excitable cells… (more)

Subjects/Keywords: Biochemistry; Cellular biology; 2-APB; CRAC; orai; stim; store-operated calcium entry

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APA (6th Edition):

Deng, X. (2011). Dissecting the mechanism of STIM coupling to Orai. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213111

Chicago Manual of Style (16th Edition):

Deng, Xiaoxiang. “Dissecting the mechanism of STIM coupling to Orai.” 2011. Doctoral Dissertation, Temple University. Accessed October 31, 2020. http://digital.library.temple.edu/u?/p245801coll10,213111.

MLA Handbook (7th Edition):

Deng, Xiaoxiang. “Dissecting the mechanism of STIM coupling to Orai.” 2011. Web. 31 Oct 2020.

Vancouver:

Deng X. Dissecting the mechanism of STIM coupling to Orai. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Oct 31]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213111.

Council of Science Editors:

Deng X. Dissecting the mechanism of STIM coupling to Orai. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,213111


University of Oxford

11. Batchelor, Helen R. Characterisation of store-operated calcium entry in a vascular endothelial cell line and impact on the production of nitric oxide.

Degree: PhD, 2014, University of Oxford

Store-operated calcium entry (SOCE) is a principal mechanism for extracellular calcium entry in non-excitable cell types, and is primarily facilitated by the calcium- release activated… (more)

Subjects/Keywords: 572; Biochemistry; Cardiovascular science; Physiology; CRAC channel; nitric oxide; STIM; eNOS; blood pressure; store-operated calcium entry; calcium; orai; endothelial cell

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APA (6th Edition):

Batchelor, H. R. (2014). Characterisation of store-operated calcium entry in a vascular endothelial cell line and impact on the production of nitric oxide. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:548a6062-2b2f-46ce-b91a-890561b5edff ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711692

Chicago Manual of Style (16th Edition):

Batchelor, Helen R. “Characterisation of store-operated calcium entry in a vascular endothelial cell line and impact on the production of nitric oxide.” 2014. Doctoral Dissertation, University of Oxford. Accessed October 31, 2020. http://ora.ox.ac.uk/objects/uuid:548a6062-2b2f-46ce-b91a-890561b5edff ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711692.

MLA Handbook (7th Edition):

Batchelor, Helen R. “Characterisation of store-operated calcium entry in a vascular endothelial cell line and impact on the production of nitric oxide.” 2014. Web. 31 Oct 2020.

Vancouver:

Batchelor HR. Characterisation of store-operated calcium entry in a vascular endothelial cell line and impact on the production of nitric oxide. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Oct 31]. Available from: http://ora.ox.ac.uk/objects/uuid:548a6062-2b2f-46ce-b91a-890561b5edff ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711692.

Council of Science Editors:

Batchelor HR. Characterisation of store-operated calcium entry in a vascular endothelial cell line and impact on the production of nitric oxide. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:548a6062-2b2f-46ce-b91a-890561b5edff ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711692


University of Michigan

12. Zhang, Irina. Investigating the Role of ER Stress on Mouse Pancreatic Beta-Cell Function.

Degree: PhD, Pharmacology, 2020, University of Michigan

 Type 2 diabetes mellitus (T2DM) is a chronic disease characterized by impaired glucose-stimulated insulin secretion in the setting of increased insulin resistance, a condition in… (more)

Subjects/Keywords: beta cells; ER stress; insulin secretion; store-operated calcium entry (SOCE); calcium signaling; diabetes; Science (General); Science

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APA (6th Edition):

Zhang, I. (2020). Investigating the Role of ER Stress on Mouse Pancreatic Beta-Cell Function. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/163151

Chicago Manual of Style (16th Edition):

Zhang, Irina. “Investigating the Role of ER Stress on Mouse Pancreatic Beta-Cell Function.” 2020. Doctoral Dissertation, University of Michigan. Accessed October 31, 2020. http://hdl.handle.net/2027.42/163151.

MLA Handbook (7th Edition):

Zhang, Irina. “Investigating the Role of ER Stress on Mouse Pancreatic Beta-Cell Function.” 2020. Web. 31 Oct 2020.

Vancouver:

Zhang I. Investigating the Role of ER Stress on Mouse Pancreatic Beta-Cell Function. [Internet] [Doctoral dissertation]. University of Michigan; 2020. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/2027.42/163151.

Council of Science Editors:

Zhang I. Investigating the Role of ER Stress on Mouse Pancreatic Beta-Cell Function. [Doctoral Dissertation]. University of Michigan; 2020. Available from: http://hdl.handle.net/2027.42/163151


University of Toledo Health Science Campus

13. Goswamee, Priyodarshan. The Role of Orai-Mediated Ca2+ Entry in Migration in a Gastroenteropancreatic Neuroendocrine Tumor Model.

Degree: PhD, Biomedical Sciences (Neurosciences and Neurological Disorders), 2015, University of Toledo Health Science Campus

 Gastroenteropancreatic neuroendocrine tumors (GEPNETs) of the midgut are a heterogeneous group of cancers that typically remain asymptomatic until they metastasize to the liver. However, the… (more)

Subjects/Keywords: Biomedical Research; Calcium signaling; Orai1; Orai3; Arachidonate induced Calcium entry; Store operated Calcium entry; Tumor extravasation; Liver metastasis; Carcinoids; Gastroenteropancreatic neuroendocrine tumors

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APA (6th Edition):

Goswamee, P. (2015). The Role of Orai-Mediated Ca2+ Entry in Migration in a Gastroenteropancreatic Neuroendocrine Tumor Model. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1438280470

Chicago Manual of Style (16th Edition):

Goswamee, Priyodarshan. “The Role of Orai-Mediated Ca2+ Entry in Migration in a Gastroenteropancreatic Neuroendocrine Tumor Model.” 2015. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed October 31, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1438280470.

MLA Handbook (7th Edition):

Goswamee, Priyodarshan. “The Role of Orai-Mediated Ca2+ Entry in Migration in a Gastroenteropancreatic Neuroendocrine Tumor Model.” 2015. Web. 31 Oct 2020.

Vancouver:

Goswamee P. The Role of Orai-Mediated Ca2+ Entry in Migration in a Gastroenteropancreatic Neuroendocrine Tumor Model. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2015. [cited 2020 Oct 31]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1438280470.

Council of Science Editors:

Goswamee P. The Role of Orai-Mediated Ca2+ Entry in Migration in a Gastroenteropancreatic Neuroendocrine Tumor Model. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1438280470


Missouri University of Science and Technology

14. Wang, Hsiu-Jen. Zinc oxide nanoparticle disruption of store-operated calcium entry in a muscarinic receptor signaling pathway.

Degree: M.S. in Applied and Environmental Biology, Applied and Environmental Biology, Missouri University of Science and Technology

 "The influences of ZnO nanoparticles on cellular responses to activation of muscarinic receptors were studied in Chinese hamster ovary cells expressing the human M3 muscarinic… (more)

Subjects/Keywords: Store-operated calcium entry; Biology; Environmental Sciences

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APA (6th Edition):

Wang, H. (n.d.). Zinc oxide nanoparticle disruption of store-operated calcium entry in a muscarinic receptor signaling pathway. (Masters Thesis). Missouri University of Science and Technology. Retrieved from https://scholarsmine.mst.edu/masters_theses/6922

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Wang, Hsiu-Jen. “Zinc oxide nanoparticle disruption of store-operated calcium entry in a muscarinic receptor signaling pathway.” Masters Thesis, Missouri University of Science and Technology. Accessed October 31, 2020. https://scholarsmine.mst.edu/masters_theses/6922.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Wang, Hsiu-Jen. “Zinc oxide nanoparticle disruption of store-operated calcium entry in a muscarinic receptor signaling pathway.” Web. 31 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Wang H. Zinc oxide nanoparticle disruption of store-operated calcium entry in a muscarinic receptor signaling pathway. [Internet] [Masters thesis]. Missouri University of Science and Technology; [cited 2020 Oct 31]. Available from: https://scholarsmine.mst.edu/masters_theses/6922.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Wang H. Zinc oxide nanoparticle disruption of store-operated calcium entry in a muscarinic receptor signaling pathway. [Masters Thesis]. Missouri University of Science and Technology; Available from: https://scholarsmine.mst.edu/masters_theses/6922

Note: this citation may be lacking information needed for this citation format:
No year of publication.


University of Toronto

15. Zheng, Le. Structural and Biophysical Studies of the Role of Stromal Interaction Molecules STIM1 and STIM2 in Initiating Store-operated Calcium Entry.

Degree: 2010, University of Toronto

Store-operated calcium entry (SOCE) is the major Ca2+ entry pathway in most non-excitable cells maintaining prolonged elevation of cytosolic Ca2+ levels required for gene transcription.… (more)

Subjects/Keywords: STIM; store-operated calcium entry; structure; EF hand; sterile alpha-motif; NMR; CRAC; Orai; 0786; 0487

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APA (6th Edition):

Zheng, L. (2010). Structural and Biophysical Studies of the Role of Stromal Interaction Molecules STIM1 and STIM2 in Initiating Store-operated Calcium Entry. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24660

Chicago Manual of Style (16th Edition):

Zheng, Le. “Structural and Biophysical Studies of the Role of Stromal Interaction Molecules STIM1 and STIM2 in Initiating Store-operated Calcium Entry.” 2010. Masters Thesis, University of Toronto. Accessed October 31, 2020. http://hdl.handle.net/1807/24660.

MLA Handbook (7th Edition):

Zheng, Le. “Structural and Biophysical Studies of the Role of Stromal Interaction Molecules STIM1 and STIM2 in Initiating Store-operated Calcium Entry.” 2010. Web. 31 Oct 2020.

Vancouver:

Zheng L. Structural and Biophysical Studies of the Role of Stromal Interaction Molecules STIM1 and STIM2 in Initiating Store-operated Calcium Entry. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/1807/24660.

Council of Science Editors:

Zheng L. Structural and Biophysical Studies of the Role of Stromal Interaction Molecules STIM1 and STIM2 in Initiating Store-operated Calcium Entry. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24660


University of Cambridge

16. Rahman, Md Saifur. Drug repurposing against the store-operated calcium entry (SOCE) pathway and subsequent exploration of SOCE in oligodendrocyte progenitor cells.

Degree: PhD, 2020, University of Cambridge

 The store-operated calcium entry (SOCE) pathway is an important route for generating cytosolic Ca2+ signals that regulate a diverse array of biological processes. Abnormal SOCE… (more)

Subjects/Keywords: Store operated calcium entry; SOCE blockers; Drug repurposing; OPCs; OPCS differentiation; multiple sclerosis; SOCE in OPCs

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APA (6th Edition):

Rahman, M. S. (2020). Drug repurposing against the store-operated calcium entry (SOCE) pathway and subsequent exploration of SOCE in oligodendrocyte progenitor cells. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/304875

Chicago Manual of Style (16th Edition):

Rahman, Md Saifur. “Drug repurposing against the store-operated calcium entry (SOCE) pathway and subsequent exploration of SOCE in oligodendrocyte progenitor cells.” 2020. Doctoral Dissertation, University of Cambridge. Accessed October 31, 2020. https://www.repository.cam.ac.uk/handle/1810/304875.

MLA Handbook (7th Edition):

Rahman, Md Saifur. “Drug repurposing against the store-operated calcium entry (SOCE) pathway and subsequent exploration of SOCE in oligodendrocyte progenitor cells.” 2020. Web. 31 Oct 2020.

Vancouver:

Rahman MS. Drug repurposing against the store-operated calcium entry (SOCE) pathway and subsequent exploration of SOCE in oligodendrocyte progenitor cells. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Oct 31]. Available from: https://www.repository.cam.ac.uk/handle/1810/304875.

Council of Science Editors:

Rahman MS. Drug repurposing against the store-operated calcium entry (SOCE) pathway and subsequent exploration of SOCE in oligodendrocyte progenitor cells. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/304875


University of Western Ontario

17. Zhu, Jinhui. S-Nitrosylation Suppresses Stromal Interaction Molecule-1 Activation and Ameliorates Cardiomyocyte Hypertrophy.

Degree: 2017, University of Western Ontario

 Stromal interaction molecule 1 (STIM1) is an endo/sarcoplasmic reticulum (ER/SR) calcium (Ca2+) sensor that activates store-operated Ca2+ entry (SOCE) following Ca2+ depletion. SOCE-induced elevation of… (more)

Subjects/Keywords: stromal interaction molecule-1; nitric oxide synthase; S-nitrosylation; cardiomyocyte; hypertrophy; store-operated calcium entry; Cellular and Molecular Physiology

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APA (6th Edition):

Zhu, J. (2017). S-Nitrosylation Suppresses Stromal Interaction Molecule-1 Activation and Ameliorates Cardiomyocyte Hypertrophy. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhu, Jinhui. “S-Nitrosylation Suppresses Stromal Interaction Molecule-1 Activation and Ameliorates Cardiomyocyte Hypertrophy.” 2017. Thesis, University of Western Ontario. Accessed October 31, 2020. https://ir.lib.uwo.ca/etd/4591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhu, Jinhui. “S-Nitrosylation Suppresses Stromal Interaction Molecule-1 Activation and Ameliorates Cardiomyocyte Hypertrophy.” 2017. Web. 31 Oct 2020.

Vancouver:

Zhu J. S-Nitrosylation Suppresses Stromal Interaction Molecule-1 Activation and Ameliorates Cardiomyocyte Hypertrophy. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Oct 31]. Available from: https://ir.lib.uwo.ca/etd/4591.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhu J. S-Nitrosylation Suppresses Stromal Interaction Molecule-1 Activation and Ameliorates Cardiomyocyte Hypertrophy. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4591

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

18. Zhang, Bojun. CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA.

Degree: Biomedical Sciences Graduate Program, 2017, University of New Mexico

  Chronic hypoxia (CH)-induced pulmonary hypertension (PH) is associated with diminished ATP-induced endothelial Ca2+ entry as well as membrane cholesterol in pulmonary arteries. Store-operated Ca2+… (more)

Subjects/Keywords: pulmonary hypertension; T-type calcium channels; ATP; store-operated Ca2+ entry; depolarization-induced Ca2+ entry; STIM1; Cellular and Molecular Physiology; Medicine and Health Sciences

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APA (6th Edition):

Zhang, B. (2017). CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/180

Chicago Manual of Style (16th Edition):

Zhang, Bojun. “CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA.” 2017. Doctoral Dissertation, University of New Mexico. Accessed October 31, 2020. https://digitalrepository.unm.edu/biom_etds/180.

MLA Handbook (7th Edition):

Zhang, Bojun. “CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA.” 2017. Web. 31 Oct 2020.

Vancouver:

Zhang B. CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA. [Internet] [Doctoral dissertation]. University of New Mexico; 2017. [cited 2020 Oct 31]. Available from: https://digitalrepository.unm.edu/biom_etds/180.

Council of Science Editors:

Zhang B. CHOLESTEROL REGULATION OF PULMONARY ENDOTHELIAL CALCIUM ENTRY FOLLOWING CHRONIC HYPOXIA. [Doctoral Dissertation]. University of New Mexico; 2017. Available from: https://digitalrepository.unm.edu/biom_etds/180


University of Oxford

19. Douglas, Sophie Georgina. Regulation of CRAC channels and agonist-induced Ca2+ signals.

Degree: PhD, 2012, University of Oxford

Calcium ions (Ca2+) are extremely important intracellular messengers, activating a plethora of cellular processes. Growing evidence now points to a major role for the local… (more)

Subjects/Keywords: 572.696; Physiology and anatomy; Calcium ions; Store-operated Calcium Channels

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APA (6th Edition):

Douglas, S. G. (2012). Regulation of CRAC channels and agonist-induced Ca2+ signals. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:ae94ca14-ac95-4ea6-b14a-14f9f7bafd63 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581188

Chicago Manual of Style (16th Edition):

Douglas, Sophie Georgina. “Regulation of CRAC channels and agonist-induced Ca2+ signals.” 2012. Doctoral Dissertation, University of Oxford. Accessed October 31, 2020. http://ora.ox.ac.uk/objects/uuid:ae94ca14-ac95-4ea6-b14a-14f9f7bafd63 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581188.

MLA Handbook (7th Edition):

Douglas, Sophie Georgina. “Regulation of CRAC channels and agonist-induced Ca2+ signals.” 2012. Web. 31 Oct 2020.

Vancouver:

Douglas SG. Regulation of CRAC channels and agonist-induced Ca2+ signals. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Oct 31]. Available from: http://ora.ox.ac.uk/objects/uuid:ae94ca14-ac95-4ea6-b14a-14f9f7bafd63 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581188.

Council of Science Editors:

Douglas SG. Regulation of CRAC channels and agonist-induced Ca2+ signals. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:ae94ca14-ac95-4ea6-b14a-14f9f7bafd63 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581188


McMaster University

20. Humber, Brent T. INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING.

Degree: MSc, 2011, McMaster University

Introduction Asthma is a chronic respiratory disease that is becoming more prevalent. Airway hyperresponsivness, a key feature of asthma, involves increased narrowing of the… (more)

Subjects/Keywords: asthma; airway smooth muscle; contraction; airway hyperresponsiveness; src kinase; store-operated calcium entry; Circulatory and Respiratory Physiology; Medical Molecular Biology; Circulatory and Respiratory Physiology

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APA (6th Edition):

Humber, B. T. (2011). INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11744

Chicago Manual of Style (16th Edition):

Humber, Brent T. “INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING.” 2011. Masters Thesis, McMaster University. Accessed October 31, 2020. http://hdl.handle.net/11375/11744.

MLA Handbook (7th Edition):

Humber, Brent T. “INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING.” 2011. Web. 31 Oct 2020.

Vancouver:

Humber BT. INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/11375/11744.

Council of Science Editors:

Humber BT. INVOLVEMENT OF SRC TYROSINE KINASE AND CALCIUM-HANDLING IN AIRWAY SMOOTH MUSCLE EXCITATION-CONTRACTION COUPLING. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11744


Université Paris-Sud – Paris XI

21. Djillani, Alaeddine. Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species.

Degree: Docteur es, Pharmacologie, Biologie cellulaire et moléculaire, 2013, Université Paris-Sud – Paris XI

 Les polynucléaires neutrophiles représentent 50-70% des leucocytes sanguins et possèdent un rôle majeur dans la défense de l’organisme contre les pathogènes. Le Ca2+ est un… (more)

Subjects/Keywords: Calcium; Influx capacitif; Polynucléaire neutrophile; Lymphocyte; Courant CRAC; Réticulum endoplasmique; Canaux calciques; SOC; SOCE; Thapsigargine (TG); SERCA; Phagocytose; Production de formes réactives de l'oxygène (FRO); Orai; STIM; 2-APB; Calcium; Capacitive calcium entry; Polymorphonuclear neutrophil; Lymphocyte; CRAC courant; Endoplasmic reticulum; Calcium channel; Store-operated calcium channel (SOC); Store-operated calcium entry (SOCE); Thapsigargin (TG); Sarco-endoplasmic calcium ATPase (SERCA); Phagocytosis; Reactive oxygen species (ROS) production; Orai; STIM; 2-APB

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APA (6th Edition):

Djillani, A. (2013). Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T056

Chicago Manual of Style (16th Edition):

Djillani, Alaeddine. “Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed October 31, 2020. http://www.theses.fr/2013PA11T056.

MLA Handbook (7th Edition):

Djillani, Alaeddine. “Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species.” 2013. Web. 31 Oct 2020.

Vancouver:

Djillani A. Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Oct 31]. Available from: http://www.theses.fr/2013PA11T056.

Council of Science Editors:

Djillani A. Caractérisation des canaux calciques dans les polynucléaires neutrophiles : rôle dans la phagocytose et la production des radicaux libres oxygénés : Characterization of calcium channels in polymorphonuclear neutrophils : role in phagocytosis and reactive oxygen species. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T056


Penn State University

22. Nwokonko, Robert. UNDERSTANDING THE MECHANISM OF UNIMOLECULAR COUPLING BETWEEN STIM AND ORAI.

Degree: 2019, Penn State University

Store-operated calcium (Ca2+) entry (SOCE) is a ubiquitous signaling mechanism in eukaryotic cells crucial for mediating longer–term Ca2+ signals and restoring endoplasmic/sarcoplasmic (ER/SR) reticulum Ca2+… (more)

Subjects/Keywords: Store operated calcium channels; Electrophysiology; Membrane proteins; Orai calcium channel; Endoplasmic reticulum; Calcium signalling

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APA (6th Edition):

Nwokonko, R. (2019). UNDERSTANDING THE MECHANISM OF UNIMOLECULAR COUPLING BETWEEN STIM AND ORAI. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/17055rmn152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nwokonko, Robert. “UNDERSTANDING THE MECHANISM OF UNIMOLECULAR COUPLING BETWEEN STIM AND ORAI.” 2019. Thesis, Penn State University. Accessed October 31, 2020. https://submit-etda.libraries.psu.edu/catalog/17055rmn152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nwokonko, Robert. “UNDERSTANDING THE MECHANISM OF UNIMOLECULAR COUPLING BETWEEN STIM AND ORAI.” 2019. Web. 31 Oct 2020.

Vancouver:

Nwokonko R. UNDERSTANDING THE MECHANISM OF UNIMOLECULAR COUPLING BETWEEN STIM AND ORAI. [Internet] [Thesis]. Penn State University; 2019. [cited 2020 Oct 31]. Available from: https://submit-etda.libraries.psu.edu/catalog/17055rmn152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nwokonko R. UNDERSTANDING THE MECHANISM OF UNIMOLECULAR COUPLING BETWEEN STIM AND ORAI. [Thesis]. Penn State University; 2019. Available from: https://submit-etda.libraries.psu.edu/catalog/17055rmn152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Bonnefond, Marie-Laure. Rôle de la signalisation calcique dans la sensibilisation des cancers ovariens chimiorésistants aux stratégies anti-Bcl-xL : Role of calcium signaling in sensitization of chemoresistant ovarian cancer to anti-Bcl-xL strategies.

Degree: Docteur es, Aspects moleculaires et cellulaires de la biologie, 2017, Normandie

Les cancers de l’ovaire représentent la première cause de décès par cancer gynécologique. L’échec des traitements, lié à l’apparition d’une chimiorésistance, implique de trouver de… (more)

Subjects/Keywords: Cancer de l’ovaire; Ovarian cancer; Apoptosis; Store operated calcium channels; Autosis

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APA (6th Edition):

Bonnefond, M. (2017). Rôle de la signalisation calcique dans la sensibilisation des cancers ovariens chimiorésistants aux stratégies anti-Bcl-xL : Role of calcium signaling in sensitization of chemoresistant ovarian cancer to anti-Bcl-xL strategies. (Doctoral Dissertation). Normandie. Retrieved from http://www.theses.fr/2017NORMC424

Chicago Manual of Style (16th Edition):

Bonnefond, Marie-Laure. “Rôle de la signalisation calcique dans la sensibilisation des cancers ovariens chimiorésistants aux stratégies anti-Bcl-xL : Role of calcium signaling in sensitization of chemoresistant ovarian cancer to anti-Bcl-xL strategies.” 2017. Doctoral Dissertation, Normandie. Accessed October 31, 2020. http://www.theses.fr/2017NORMC424.

MLA Handbook (7th Edition):

Bonnefond, Marie-Laure. “Rôle de la signalisation calcique dans la sensibilisation des cancers ovariens chimiorésistants aux stratégies anti-Bcl-xL : Role of calcium signaling in sensitization of chemoresistant ovarian cancer to anti-Bcl-xL strategies.” 2017. Web. 31 Oct 2020.

Vancouver:

Bonnefond M. Rôle de la signalisation calcique dans la sensibilisation des cancers ovariens chimiorésistants aux stratégies anti-Bcl-xL : Role of calcium signaling in sensitization of chemoresistant ovarian cancer to anti-Bcl-xL strategies. [Internet] [Doctoral dissertation]. Normandie; 2017. [cited 2020 Oct 31]. Available from: http://www.theses.fr/2017NORMC424.

Council of Science Editors:

Bonnefond M. Rôle de la signalisation calcique dans la sensibilisation des cancers ovariens chimiorésistants aux stratégies anti-Bcl-xL : Role of calcium signaling in sensitization of chemoresistant ovarian cancer to anti-Bcl-xL strategies. [Doctoral Dissertation]. Normandie; 2017. Available from: http://www.theses.fr/2017NORMC424


University of Western Ontario

24. Novello, Matthew. Excess NO stabilizes the luminal domain of STIM2 in a Cys-specific manner thereby regulating basal calcium homeostasis and store-operated calcium entry.

Degree: 2019, University of Western Ontario

 Stromal-interaction molecule 2 (STIM2) is an endoplasmic reticulum (ER) membrane-inserted Ca2+-sensing protein which, together with the plasma membrane Ca2+ channel Orai1, regulates basal Ca2+ homeostasis… (more)

Subjects/Keywords: Stromal interaction molecule 2; nitric oxide; post-translational modifications; S-nitrosylation; store-operated calcium entry; basal calcium homeostasis; Biochemistry; Biophysics; Cellular and Molecular Physiology; Laboratory and Basic Science Research; Molecular Biology; Pharmacology; Structural Biology

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APA (6th Edition):

Novello, M. (2019). Excess NO stabilizes the luminal domain of STIM2 in a Cys-specific manner thereby regulating basal calcium homeostasis and store-operated calcium entry. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Novello, Matthew. “Excess NO stabilizes the luminal domain of STIM2 in a Cys-specific manner thereby regulating basal calcium homeostasis and store-operated calcium entry.” 2019. Thesis, University of Western Ontario. Accessed October 31, 2020. https://ir.lib.uwo.ca/etd/6570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Novello, Matthew. “Excess NO stabilizes the luminal domain of STIM2 in a Cys-specific manner thereby regulating basal calcium homeostasis and store-operated calcium entry.” 2019. Web. 31 Oct 2020.

Vancouver:

Novello M. Excess NO stabilizes the luminal domain of STIM2 in a Cys-specific manner thereby regulating basal calcium homeostasis and store-operated calcium entry. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2020 Oct 31]. Available from: https://ir.lib.uwo.ca/etd/6570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Novello M. Excess NO stabilizes the luminal domain of STIM2 in a Cys-specific manner thereby regulating basal calcium homeostasis and store-operated calcium entry. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

25. Nassrallah, Wissam. Store-Operated Response in CA1 Pyramidal Neurons Exhibits Features of Homeostatic Synaptic Plasticity .

Degree: 2015, University of Ottawa

 Homeostatic synaptic plasticity (HSP) regulates synaptic strength in response to changing neuronal firing patterns. This form of plasticity is defined by neurons’ ability to sense… (more)

Subjects/Keywords: Store-Operated Responese; Homeostatic Synaptic Plasticity; AMPA Receptors; Endoplasmic Reticulum Calcium Stores; Endoplasmic Reticulum

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APA (6th Edition):

Nassrallah, W. (2015). Store-Operated Response in CA1 Pyramidal Neurons Exhibits Features of Homeostatic Synaptic Plasticity . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/33357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nassrallah, Wissam. “Store-Operated Response in CA1 Pyramidal Neurons Exhibits Features of Homeostatic Synaptic Plasticity .” 2015. Thesis, University of Ottawa. Accessed October 31, 2020. http://hdl.handle.net/10393/33357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nassrallah, Wissam. “Store-Operated Response in CA1 Pyramidal Neurons Exhibits Features of Homeostatic Synaptic Plasticity .” 2015. Web. 31 Oct 2020.

Vancouver:

Nassrallah W. Store-Operated Response in CA1 Pyramidal Neurons Exhibits Features of Homeostatic Synaptic Plasticity . [Internet] [Thesis]. University of Ottawa; 2015. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/10393/33357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nassrallah W. Store-Operated Response in CA1 Pyramidal Neurons Exhibits Features of Homeostatic Synaptic Plasticity . [Thesis]. University of Ottawa; 2015. Available from: http://hdl.handle.net/10393/33357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Lin, Ying. Distinct Roles of GABA(A)R Signaling in the Regulation of Two Human T lymphocyte Lines.

Degree: 2017, University of Western Ontario

 In response to antigen presentation, helper T lymphocytes (TH cells) initiate store-operated Ca2+ entry (SOCE) and differentiate into effector subtypes such as TH1 and TH2… (more)

Subjects/Keywords: GABA; T helper cell; Differentiation; Store-operated Calcium Entry; CRTh2; NFAT; Cellular and Molecular Physiology

…5 Figure 1.2 Store-operated calcium entry in T lymphocytes… …ATPase SOCE Store operated calcium entry STIM1 Stromal interaction molecule 1 T1D Type 1… …Operated Calcium Entry (SOCE) Calcium ions (Ca2+) are an important secondary… …messenger in all eukaryotic cells. In T cells, store-operated Ca2+ entry (SOCE) is the… …Ca2+ levels at concentrations in the micromolar range60. 11 Figure 1.2 Store-operated… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, Y. (2017). Distinct Roles of GABA(A)R Signaling in the Regulation of Two Human T lymphocyte Lines. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Ying. “Distinct Roles of GABA(A)R Signaling in the Regulation of Two Human T lymphocyte Lines.” 2017. Thesis, University of Western Ontario. Accessed October 31, 2020. https://ir.lib.uwo.ca/etd/4742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Ying. “Distinct Roles of GABA(A)R Signaling in the Regulation of Two Human T lymphocyte Lines.” 2017. Web. 31 Oct 2020.

Vancouver:

Lin Y. Distinct Roles of GABA(A)R Signaling in the Regulation of Two Human T lymphocyte Lines. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2020 Oct 31]. Available from: https://ir.lib.uwo.ca/etd/4742.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin Y. Distinct Roles of GABA(A)R Signaling in the Regulation of Two Human T lymphocyte Lines. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4742

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Zheng, Shuhua. Targeting NEDD8 Conjugation for Therapeutic Gain in Acute Lymphoblastic Leukemia.

Degree: PhD, Cancer Biology (Medicine), 2017, University of Miami

 Development of resistance and relapse remains a major challenge for acute lymphoblastic leukemia (ALL) treatment. Results from our lab and others showed that ALL cells… (more)

Subjects/Keywords: NEDDylation; Pevonedistat; Acute Lymphoblastic Leukemia; ER stress; Store Operated Calcium Entry; MEK/ERK signaling

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zheng, S. (2017). Targeting NEDD8 Conjugation for Therapeutic Gain in Acute Lymphoblastic Leukemia. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1901

Chicago Manual of Style (16th Edition):

Zheng, Shuhua. “Targeting NEDD8 Conjugation for Therapeutic Gain in Acute Lymphoblastic Leukemia.” 2017. Doctoral Dissertation, University of Miami. Accessed October 31, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/1901.

MLA Handbook (7th Edition):

Zheng, Shuhua. “Targeting NEDD8 Conjugation for Therapeutic Gain in Acute Lymphoblastic Leukemia.” 2017. Web. 31 Oct 2020.

Vancouver:

Zheng S. Targeting NEDD8 Conjugation for Therapeutic Gain in Acute Lymphoblastic Leukemia. [Internet] [Doctoral dissertation]. University of Miami; 2017. [cited 2020 Oct 31]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1901.

Council of Science Editors:

Zheng S. Targeting NEDD8 Conjugation for Therapeutic Gain in Acute Lymphoblastic Leukemia. [Doctoral Dissertation]. University of Miami; 2017. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1901

28. Beesetty, Pavani. Consequences of TRPM7 kinase inactivation in immune cells.

Degree: PhD, Biomedical Sciences PhD, 2018, Wright State University

 The characteristic feature of ubiquitously expressed transient receptor potentialmelastatin 7 (TRPM7) protein is the presence of a C-terminal atypical kinase domainfused to a cation channel… (more)

Subjects/Keywords: Immunology; Physiology; blastogenesis; TRPM7; TRPM6; store-operated calcium entry; splenomegaly; interleukin 2

…involved in the upregulation of store-operated calcium entry upon T-cell stimulation… …activity is involved in the upregulation of store-operated calcium entry upon T-cell stimulation… …TRPM7 protein and its domains …... …. …... 4 Figure 2. Store-operated Ca2+ entry and Ca2… …immunodeficiency SERCA - Sarcoplasmic endoplasmic reticulum calcium ATPase SOCE - Store-operated Ca2… …not known. Store-operated Ca2+ entry (SOCE) in T cells As shown in Figure 2… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beesetty, P. (2018). Consequences of TRPM7 kinase inactivation in immune cells. (Doctoral Dissertation). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1526406780596661

Chicago Manual of Style (16th Edition):

Beesetty, Pavani. “Consequences of TRPM7 kinase inactivation in immune cells.” 2018. Doctoral Dissertation, Wright State University. Accessed October 31, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1526406780596661.

MLA Handbook (7th Edition):

Beesetty, Pavani. “Consequences of TRPM7 kinase inactivation in immune cells.” 2018. Web. 31 Oct 2020.

Vancouver:

Beesetty P. Consequences of TRPM7 kinase inactivation in immune cells. [Internet] [Doctoral dissertation]. Wright State University; 2018. [cited 2020 Oct 31]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1526406780596661.

Council of Science Editors:

Beesetty P. Consequences of TRPM7 kinase inactivation in immune cells. [Doctoral Dissertation]. Wright State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1526406780596661

29. Pham, Elizabeth. Development of Protein-based Tools to Image and Modulate Ca2+ Signaling.

Degree: 2011, University of Toronto

Optogenetics has emerged as a branch of biotechnology that combines genetic engineering with optics to observe intracellular changes as well as control cellular function. Despite… (more)

Subjects/Keywords: Calcium Signaling; FRET Biosensors; Store Operated Calcium Entry; Stim1; Orai1; Optogenetics; Photo-activated; LOVS1K; Fusion Proteins; Protein Engineering; 0541

…C Plasma membrane Sarco/endoplasmic reticulum Ca2+-ATPase Store-operated calcium entry… …and replenish ER Ca2+ stores. In particular, influxes through store-operated calcium entry… …Figures Figure 2-1. Store-operated Ca2+ entry… …store-operated Ca2+ entry (SOCE). SOCE-related diseases are also discussed, as well… …cardiac excitation-contraction coupling [1-5]. 2.1.1 Store-Operated Ca2+ Entry… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pham, E. (2011). Development of Protein-based Tools to Image and Modulate Ca2+ Signaling. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/31898

Chicago Manual of Style (16th Edition):

Pham, Elizabeth. “Development of Protein-based Tools to Image and Modulate Ca2+ Signaling.” 2011. Doctoral Dissertation, University of Toronto. Accessed October 31, 2020. http://hdl.handle.net/1807/31898.

MLA Handbook (7th Edition):

Pham, Elizabeth. “Development of Protein-based Tools to Image and Modulate Ca2+ Signaling.” 2011. Web. 31 Oct 2020.

Vancouver:

Pham E. Development of Protein-based Tools to Image and Modulate Ca2+ Signaling. [Internet] [Doctoral dissertation]. University of Toronto; 2011. [cited 2020 Oct 31]. Available from: http://hdl.handle.net/1807/31898.

Council of Science Editors:

Pham E. Development of Protein-based Tools to Image and Modulate Ca2+ Signaling. [Doctoral Dissertation]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31898


Penn State University

30. Mercer, Jason Chance. 3,5-BISTRIFLUOROMETHYL PYRAZOLE (BTP) COMPOUNDS AND REGULATION OF STORE-OPERATED CALCIUM CHANNELS BY THE ACTIN-BINDING PROTEIN DREBRIN.

Degree: 2008, Penn State University

 Stimulation of tyrosine kinase coupled receptors, such as the T cell receptor in T lymphocytes, results in activation of phospholipase-Cγ (PLCγ) which hydrolyzes phosphoinositol 4,5,… (more)

Subjects/Keywords: calcium; store-operated channel; CRAC; BTP; actin; cytoskeleton; drug

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mercer, J. C. (2008). 3,5-BISTRIFLUOROMETHYL PYRAZOLE (BTP) COMPOUNDS AND REGULATION OF STORE-OPERATED CALCIUM CHANNELS BY THE ACTIN-BINDING PROTEIN DREBRIN. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/6526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mercer, Jason Chance. “3,5-BISTRIFLUOROMETHYL PYRAZOLE (BTP) COMPOUNDS AND REGULATION OF STORE-OPERATED CALCIUM CHANNELS BY THE ACTIN-BINDING PROTEIN DREBRIN.” 2008. Thesis, Penn State University. Accessed October 31, 2020. https://submit-etda.libraries.psu.edu/catalog/6526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mercer, Jason Chance. “3,5-BISTRIFLUOROMETHYL PYRAZOLE (BTP) COMPOUNDS AND REGULATION OF STORE-OPERATED CALCIUM CHANNELS BY THE ACTIN-BINDING PROTEIN DREBRIN.” 2008. Web. 31 Oct 2020.

Vancouver:

Mercer JC. 3,5-BISTRIFLUOROMETHYL PYRAZOLE (BTP) COMPOUNDS AND REGULATION OF STORE-OPERATED CALCIUM CHANNELS BY THE ACTIN-BINDING PROTEIN DREBRIN. [Internet] [Thesis]. Penn State University; 2008. [cited 2020 Oct 31]. Available from: https://submit-etda.libraries.psu.edu/catalog/6526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mercer JC. 3,5-BISTRIFLUOROMETHYL PYRAZOLE (BTP) COMPOUNDS AND REGULATION OF STORE-OPERATED CALCIUM CHANNELS BY THE ACTIN-BINDING PROTEIN DREBRIN. [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/6526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [228]

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