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You searched for subject:(somatic mutations). Showing records 1 – 30 of 32 total matches.

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University of Cambridge

1. GARG, SUMEDHA. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.

Degree: PhD, 2019, University of Cambridge

 Primary aldosteronism (PA) accounts for 5-10% of all hypertension. One of the major causes of PA is sporadic formation of aldosterone-producing adenomas (APAs). These benign… (more)

Subjects/Keywords: aldosterone; adenoma; somatic mutations; hypertension

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

GARG, S. (2019). Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289390

Chicago Manual of Style (16th Edition):

GARG, SUMEDHA. “Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.” 2019. Doctoral Dissertation, University of Cambridge. Accessed January 22, 2021. https://www.repository.cam.ac.uk/handle/1810/289390.

MLA Handbook (7th Edition):

GARG, SUMEDHA. “Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.” 2019. Web. 22 Jan 2021.

Vancouver:

GARG S. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Jan 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/289390.

Council of Science Editors:

GARG S. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289390


University of California – Santa Cruz

2. Radenbaugh, Amie. The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data.

Degree: Biomolecular Engineering and Bioinformatics, 2015, University of California – Santa Cruz

 Much of our current understanding of cancer has come from investigating how normal cells are transformed into malignant cancers through the stepwise acquisition of somatic(more)

Subjects/Keywords: Bioinformatics; editing; mutations; NGS; RNA; somatic; variants

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APA (6th Edition):

Radenbaugh, A. (2015). The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data. (Thesis). University of California – Santa Cruz. Retrieved from http://www.escholarship.org/uc/item/0dt1w1fx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radenbaugh, Amie. “The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data.” 2015. Thesis, University of California – Santa Cruz. Accessed January 22, 2021. http://www.escholarship.org/uc/item/0dt1w1fx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radenbaugh, Amie. “The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data.” 2015. Web. 22 Jan 2021.

Vancouver:

Radenbaugh A. The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data. [Internet] [Thesis]. University of California – Santa Cruz; 2015. [cited 2021 Jan 22]. Available from: http://www.escholarship.org/uc/item/0dt1w1fx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radenbaugh A. The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data. [Thesis]. University of California – Santa Cruz; 2015. Available from: http://www.escholarship.org/uc/item/0dt1w1fx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Maddaly Ravi. Analysis of somatic cell mutations at the Glycophorina locus in human erythrocytes.

Degree: Human Genetics, 2001, Sri Ramachandra University

Introduction: Dose evaluation of exposed humans to radiation becomes necessary after exposure. Such a dose evaluation can be done by biological dosimetric methods of which… (more)

Subjects/Keywords: Glycophorina; Human erythrocytes; Somatic cell mutations

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APA (6th Edition):

Ravi, M. (2001). Analysis of somatic cell mutations at the Glycophorina locus in human erythrocytes. (Thesis). Sri Ramachandra University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/17910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ravi, Maddaly. “Analysis of somatic cell mutations at the Glycophorina locus in human erythrocytes.” 2001. Thesis, Sri Ramachandra University. Accessed January 22, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/17910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ravi, Maddaly. “Analysis of somatic cell mutations at the Glycophorina locus in human erythrocytes.” 2001. Web. 22 Jan 2021.

Vancouver:

Ravi M. Analysis of somatic cell mutations at the Glycophorina locus in human erythrocytes. [Internet] [Thesis]. Sri Ramachandra University; 2001. [cited 2021 Jan 22]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/17910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ravi M. Analysis of somatic cell mutations at the Glycophorina locus in human erythrocytes. [Thesis]. Sri Ramachandra University; 2001. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/17910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

4. Zhang, Yang. Computational approaches for analyzing regulatory regions in the human genome.

Degree: PhD, Bioengineering, 2017, University of Illinois – Urbana-Champaign

 The cis-regulatory elements (CRE) in the human genome play a critical role in transcriptional regulation. Alterations of the CRE have long been considered as the… (more)

Subjects/Keywords: Regulatory elements; Evolution; Somatic mutations; Chromatin organization

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APA (6th Edition):

Zhang, Y. (2017). Computational approaches for analyzing regulatory regions in the human genome. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/99491

Chicago Manual of Style (16th Edition):

Zhang, Yang. “Computational approaches for analyzing regulatory regions in the human genome.” 2017. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 22, 2021. http://hdl.handle.net/2142/99491.

MLA Handbook (7th Edition):

Zhang, Yang. “Computational approaches for analyzing regulatory regions in the human genome.” 2017. Web. 22 Jan 2021.

Vancouver:

Zhang Y. Computational approaches for analyzing regulatory regions in the human genome. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2017. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2142/99491.

Council of Science Editors:

Zhang Y. Computational approaches for analyzing regulatory regions in the human genome. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2017. Available from: http://hdl.handle.net/2142/99491


UCLA

5. Chang, Vivian Y. Whole Exome Sequencing of Pediatric Gastric Adenocarcinoma: A Germline and Somatic Mutation Analysis.

Degree: Bioinformatics, 2012, UCLA

 BackgroundGastric adenocarcinoma is a rare diagnosis in childhood. A 14-year old male patient presented with metastatic gastric adenocarcinoma, and a strong family history of colon… (more)

Subjects/Keywords: Molecular biology; Bioinformatics; Genetics; exome sequencing; germline mutations; pediatric cancer; somatic mutations

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APA (6th Edition):

Chang, V. Y. (2012). Whole Exome Sequencing of Pediatric Gastric Adenocarcinoma: A Germline and Somatic Mutation Analysis. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5bg855cr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang, Vivian Y. “Whole Exome Sequencing of Pediatric Gastric Adenocarcinoma: A Germline and Somatic Mutation Analysis.” 2012. Thesis, UCLA. Accessed January 22, 2021. http://www.escholarship.org/uc/item/5bg855cr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang, Vivian Y. “Whole Exome Sequencing of Pediatric Gastric Adenocarcinoma: A Germline and Somatic Mutation Analysis.” 2012. Web. 22 Jan 2021.

Vancouver:

Chang VY. Whole Exome Sequencing of Pediatric Gastric Adenocarcinoma: A Germline and Somatic Mutation Analysis. [Internet] [Thesis]. UCLA; 2012. [cited 2021 Jan 22]. Available from: http://www.escholarship.org/uc/item/5bg855cr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang VY. Whole Exome Sequencing of Pediatric Gastric Adenocarcinoma: A Germline and Somatic Mutation Analysis. [Thesis]. UCLA; 2012. Available from: http://www.escholarship.org/uc/item/5bg855cr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. G.E.M. Melloni. COMPUTATIONAL FRAMEWORKS FOR THE IDENTIFICATION OF SOMATIC AND GERMLINE VARIANTS CONTRIBUTING TO CANCER PREDISPOSITION AND DEVELOPMENT.

Degree: 2017, Università degli Studi di Milano

 The most recent cancer classification from NIH includes ~200 types of tumor that originates from several tissue types (http://www.cancer.gov/types). Although macroscopic and microscopic characteristics varies… (more)

Subjects/Keywords: Somatic Mutations; Germline Mutations; Next Generation Sequencing; Whole Exome Sequencing; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Melloni, G. (2017). COMPUTATIONAL FRAMEWORKS FOR THE IDENTIFICATION OF SOMATIC AND GERMLINE VARIANTS CONTRIBUTING TO CANCER PREDISPOSITION AND DEVELOPMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/462986

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melloni, G.E.M.. “COMPUTATIONAL FRAMEWORKS FOR THE IDENTIFICATION OF SOMATIC AND GERMLINE VARIANTS CONTRIBUTING TO CANCER PREDISPOSITION AND DEVELOPMENT.” 2017. Thesis, Università degli Studi di Milano. Accessed January 22, 2021. http://hdl.handle.net/2434/462986.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melloni, G.E.M.. “COMPUTATIONAL FRAMEWORKS FOR THE IDENTIFICATION OF SOMATIC AND GERMLINE VARIANTS CONTRIBUTING TO CANCER PREDISPOSITION AND DEVELOPMENT.” 2017. Web. 22 Jan 2021.

Vancouver:

Melloni G. COMPUTATIONAL FRAMEWORKS FOR THE IDENTIFICATION OF SOMATIC AND GERMLINE VARIANTS CONTRIBUTING TO CANCER PREDISPOSITION AND DEVELOPMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2434/462986.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melloni G. COMPUTATIONAL FRAMEWORKS FOR THE IDENTIFICATION OF SOMATIC AND GERMLINE VARIANTS CONTRIBUTING TO CANCER PREDISPOSITION AND DEVELOPMENT. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/462986

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

7. Song, Zhuo. Stochastic modeling of mitochondrial polymerase gamma replication and novel algorithms to enrich rare disease alleles and detect tumor somatic mutations in deep sequencing data.

Degree: PhD, Human Genetics, 2012, Vanderbilt University

 The activity of polymerase ã (pol ã) is complicated. To understand how its kinetics values affect the final function of the pol ã, I created… (more)

Subjects/Keywords: tumor somatic mutations; targeted sequencing; NRTI; polymerase gamma; mtDNA replication

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APA (6th Edition):

Song, Z. (2012). Stochastic modeling of mitochondrial polymerase gamma replication and novel algorithms to enrich rare disease alleles and detect tumor somatic mutations in deep sequencing data. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10702

Chicago Manual of Style (16th Edition):

Song, Zhuo. “Stochastic modeling of mitochondrial polymerase gamma replication and novel algorithms to enrich rare disease alleles and detect tumor somatic mutations in deep sequencing data.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2021. http://hdl.handle.net/1803/10702.

MLA Handbook (7th Edition):

Song, Zhuo. “Stochastic modeling of mitochondrial polymerase gamma replication and novel algorithms to enrich rare disease alleles and detect tumor somatic mutations in deep sequencing data.” 2012. Web. 22 Jan 2021.

Vancouver:

Song Z. Stochastic modeling of mitochondrial polymerase gamma replication and novel algorithms to enrich rare disease alleles and detect tumor somatic mutations in deep sequencing data. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1803/10702.

Council of Science Editors:

Song Z. Stochastic modeling of mitochondrial polymerase gamma replication and novel algorithms to enrich rare disease alleles and detect tumor somatic mutations in deep sequencing data. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/10702


Delft University of Technology

8. Dentro, S.C. (author). Interpretable classification of tumours through multiple instance learning and somatic mutations.

Degree: 2013, Delft University of Technology

Next generation sequencing is brought into the clinic. Screening of disease associated genes will aid the diagnosis of disorders with a genetic component. The diagnosis… (more)

Subjects/Keywords: cancer; multiple instance learning; somatic mutations; classification; machine learning

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APA (6th Edition):

Dentro, S. C. (. (2013). Interpretable classification of tumours through multiple instance learning and somatic mutations. (Masters Thesis). Delft University of Technology. Retrieved from http://resolver.tudelft.nl/uuid:e23175cb-d1a8-4cb6-af7b-9909f6412cf5

Chicago Manual of Style (16th Edition):

Dentro, S C (author). “Interpretable classification of tumours through multiple instance learning and somatic mutations.” 2013. Masters Thesis, Delft University of Technology. Accessed January 22, 2021. http://resolver.tudelft.nl/uuid:e23175cb-d1a8-4cb6-af7b-9909f6412cf5.

MLA Handbook (7th Edition):

Dentro, S C (author). “Interpretable classification of tumours through multiple instance learning and somatic mutations.” 2013. Web. 22 Jan 2021.

Vancouver:

Dentro SC(. Interpretable classification of tumours through multiple instance learning and somatic mutations. [Internet] [Masters thesis]. Delft University of Technology; 2013. [cited 2021 Jan 22]. Available from: http://resolver.tudelft.nl/uuid:e23175cb-d1a8-4cb6-af7b-9909f6412cf5.

Council of Science Editors:

Dentro SC(. Interpretable classification of tumours through multiple instance learning and somatic mutations. [Masters Thesis]. Delft University of Technology; 2013. Available from: http://resolver.tudelft.nl/uuid:e23175cb-d1a8-4cb6-af7b-9909f6412cf5


University of Edinburgh

9. Pethick, Joanna Margaret. Detecting selection in the evolution of cancer genomes.

Degree: PhD, 2015, University of Edinburgh

 Cancer is a disease of the genome, requiring mutation or epimutation of specific genes to develop. The subsequent progression of cancer and response to therapies… (more)

Subjects/Keywords: 616.99; somatic cancer mutations; advanced evolutionary analysis framework; localised mutation pattern

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APA (6th Edition):

Pethick, J. M. (2015). Detecting selection in the evolution of cancer genomes. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/19548

Chicago Manual of Style (16th Edition):

Pethick, Joanna Margaret. “Detecting selection in the evolution of cancer genomes.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed January 22, 2021. http://hdl.handle.net/1842/19548.

MLA Handbook (7th Edition):

Pethick, Joanna Margaret. “Detecting selection in the evolution of cancer genomes.” 2015. Web. 22 Jan 2021.

Vancouver:

Pethick JM. Detecting selection in the evolution of cancer genomes. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1842/19548.

Council of Science Editors:

Pethick JM. Detecting selection in the evolution of cancer genomes. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/19548


Vanderbilt University

10. O'Brien, Timothy Daniel. Investigating the Genetic Influences of the Germline and Somatic Genomes in Three Subtypes of Lung Cancer.

Degree: PhD, Human Genetics, 2017, Vanderbilt University

 Lung cancer is classified into two main types: small cell lung cancer (SCLC) and non-small cell lung cancer (NSCLC). NSCLC has several subtypes, but the… (more)

Subjects/Keywords: RNA-Seq; WES; somatic mutations; differential expression; enhancer; eQTL; lung cancer subtypes; GWAS

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APA (6th Edition):

O'Brien, T. D. (2017). Investigating the Genetic Influences of the Germline and Somatic Genomes in Three Subtypes of Lung Cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12384

Chicago Manual of Style (16th Edition):

O'Brien, Timothy Daniel. “Investigating the Genetic Influences of the Germline and Somatic Genomes in Three Subtypes of Lung Cancer.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2021. http://hdl.handle.net/1803/12384.

MLA Handbook (7th Edition):

O'Brien, Timothy Daniel. “Investigating the Genetic Influences of the Germline and Somatic Genomes in Three Subtypes of Lung Cancer.” 2017. Web. 22 Jan 2021.

Vancouver:

O'Brien TD. Investigating the Genetic Influences of the Germline and Somatic Genomes in Three Subtypes of Lung Cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1803/12384.

Council of Science Editors:

O'Brien TD. Investigating the Genetic Influences of the Germline and Somatic Genomes in Three Subtypes of Lung Cancer. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/12384


University of Toronto

11. Green, Blerta. The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells.

Degree: 2011, University of Toronto

Deficiency in Msh2, a component of the mismatch repair (MMR) system, leads to a ~10-fold increase in the mutation frequency in most tissues. By contrast,… (more)

Subjects/Keywords: mismatch repair; somatic hypermutation; germinal center; activation induced cytidine deaminase; B cells; mutations; 0982

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APA (6th Edition):

Green, B. (2011). The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/30614

Chicago Manual of Style (16th Edition):

Green, Blerta. “The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells.” 2011. Masters Thesis, University of Toronto. Accessed January 22, 2021. http://hdl.handle.net/1807/30614.

MLA Handbook (7th Edition):

Green, Blerta. “The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells.” 2011. Web. 22 Jan 2021.

Vancouver:

Green B. The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1807/30614.

Council of Science Editors:

Green B. The Mismatch Repair Pathway Functions Normally at a non-AID Target in Germinal Center B cells. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/30614


Rice University

12. Chen, Zhongyuan. Association Studies in Human Cancers: Metabolic Expression Subtypes and Somatic Mutations/Germline Variations.

Degree: PhD, Engineering, 2019, Rice University

 Cancer is a highly complex genetic disease caused by certain gene mutations. This thesis focuses on two critical categories of association studies for human cancers:… (more)

Subjects/Keywords: Association studies; statistical tests; human cancers, metabolic expression subtypes; somatic mutations; germline variations

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APA (6th Edition):

Chen, Z. (2019). Association Studies in Human Cancers: Metabolic Expression Subtypes and Somatic Mutations/Germline Variations. (Doctoral Dissertation). Rice University. Retrieved from http://hdl.handle.net/1911/106172

Chicago Manual of Style (16th Edition):

Chen, Zhongyuan. “Association Studies in Human Cancers: Metabolic Expression Subtypes and Somatic Mutations/Germline Variations.” 2019. Doctoral Dissertation, Rice University. Accessed January 22, 2021. http://hdl.handle.net/1911/106172.

MLA Handbook (7th Edition):

Chen, Zhongyuan. “Association Studies in Human Cancers: Metabolic Expression Subtypes and Somatic Mutations/Germline Variations.” 2019. Web. 22 Jan 2021.

Vancouver:

Chen Z. Association Studies in Human Cancers: Metabolic Expression Subtypes and Somatic Mutations/Germline Variations. [Internet] [Doctoral dissertation]. Rice University; 2019. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1911/106172.

Council of Science Editors:

Chen Z. Association Studies in Human Cancers: Metabolic Expression Subtypes and Somatic Mutations/Germline Variations. [Doctoral Dissertation]. Rice University; 2019. Available from: http://hdl.handle.net/1911/106172

13. Garg, Sumedha. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.

Degree: PhD, 2019, University of Cambridge

 Primary aldosteronism (PA) accounts for 5-10% of all hypertension. One of the major causes of PA is sporadic formation of aldosterone-producing adenomas (APAs). These benign… (more)

Subjects/Keywords: aldosterone; adenoma; somatic mutations; hypertension

somatic mutations in APAs led to better understanding of molecular mechanisms causing autonomous… …APAs. Following our lab’s discovery of initial four somatic mutations by whole exome… …greatly enhanced when we ascertained that one of the 'private' somatic mutations found… …157 16 LIST OF TABLES Table 1: Reported somatic mutations in APAs… …31 Table 2: Reported prevalence of somatic mutations in different cohorts… 

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APA (6th Edition):

Garg, S. (2019). Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.36638 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767736

Chicago Manual of Style (16th Edition):

Garg, Sumedha. “Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.” 2019. Doctoral Dissertation, University of Cambridge. Accessed January 22, 2021. https://doi.org/10.17863/CAM.36638 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767736.

MLA Handbook (7th Edition):

Garg, Sumedha. “Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.” 2019. Web. 22 Jan 2021.

Vancouver:

Garg S. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Jan 22]. Available from: https://doi.org/10.17863/CAM.36638 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767736.

Council of Science Editors:

Garg S. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.36638 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767736

14. Bianchi, Joy J. Origin of somatic mutations in lymphoid cancers : role of the V(D)J recombinase : Origine des mutations somatiques dans les cancers lymphoïdes : rôle de la recombinase V(D)J.

Degree: Docteur es, Génétique, 2018, Sorbonne Paris Cité

Les cancers lymphoïdes présentent fréquemment des aberrations chromosomiques. Dans les lymphocytes, cette instabilité génomique peut découler d'une activité anormale de la recombinase V(D)J (i.e. RAG… (more)

Subjects/Keywords: Instabilité génomique; Cancers lymphoïdes; Mutations somatiques; RAG recombinase; DNA damage response; Genomic instability; Lymphoid cancers; Somatic mutations; RAG recombinase; DNA damage response; 616.994

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APA (6th Edition):

Bianchi, J. J. (2018). Origin of somatic mutations in lymphoid cancers : role of the V(D)J recombinase : Origine des mutations somatiques dans les cancers lymphoïdes : rôle de la recombinase V(D)J. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2018USPCB057

Chicago Manual of Style (16th Edition):

Bianchi, Joy J. “Origin of somatic mutations in lymphoid cancers : role of the V(D)J recombinase : Origine des mutations somatiques dans les cancers lymphoïdes : rôle de la recombinase V(D)J.” 2018. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 22, 2021. http://www.theses.fr/2018USPCB057.

MLA Handbook (7th Edition):

Bianchi, Joy J. “Origin of somatic mutations in lymphoid cancers : role of the V(D)J recombinase : Origine des mutations somatiques dans les cancers lymphoïdes : rôle de la recombinase V(D)J.” 2018. Web. 22 Jan 2021.

Vancouver:

Bianchi JJ. Origin of somatic mutations in lymphoid cancers : role of the V(D)J recombinase : Origine des mutations somatiques dans les cancers lymphoïdes : rôle de la recombinase V(D)J. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2018. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2018USPCB057.

Council of Science Editors:

Bianchi JJ. Origin of somatic mutations in lymphoid cancers : role of the V(D)J recombinase : Origine des mutations somatiques dans les cancers lymphoïdes : rôle de la recombinase V(D)J. [Doctoral Dissertation]. Sorbonne Paris Cité; 2018. Available from: http://www.theses.fr/2018USPCB057


Freie Universität Berlin

15. Lüth, Maria. Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors.

Degree: 2011, Freie Universität Berlin

 To detect somatic mitochondrial DNA mutations in neuroectodermal tumors, mutation analysis in patients with medulloblastoma, pilocytic astrocytoma and neurofibromatosis type 1-associated tumors was performed. MtDNA… (more)

Subjects/Keywords: mtDNA; somatic mutations; neurofibromatosis type 1-associated tumors; pilocytic astrocytoma; medulloblastoma; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Lüth, M. (2011). Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/13747

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lüth, Maria. “Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors.” 2011. Thesis, Freie Universität Berlin. Accessed January 22, 2021. https://refubium.fu-berlin.de/handle/fub188/13747.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lüth, Maria. “Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors.” 2011. Web. 22 Jan 2021.

Vancouver:

Lüth M. Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2021 Jan 22]. Available from: https://refubium.fu-berlin.de/handle/fub188/13747.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lüth M. Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors. [Thesis]. Freie Universität Berlin; 2011. Available from: https://refubium.fu-berlin.de/handle/fub188/13747

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Blokzijl, Francis. Decoding the mutational history of human stem cells : From patterns to mechanisms.

Degree: 2018, University Utrecht

 The extreme variation in cancer risk across tissues was recently proposed to depend on the lifetime number of adult stem cell (ASC) divisions, owing to… (more)

Subjects/Keywords: Whole genome sequencing; Somatic mutations; Stem cells; Organoids; Cancer

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APA (6th Edition):

Blokzijl, F. (2018). Decoding the mutational history of human stem cells : From patterns to mechanisms. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/371910 ; URN:NBN:NL:UI:10-1874-371910 ; urn:isbn:978-94-6375-173-5 ; URN:NBN:NL:UI:10-1874-371910 ; https://dspace.library.uu.nl/handle/1874/371910

Chicago Manual of Style (16th Edition):

Blokzijl, Francis. “Decoding the mutational history of human stem cells : From patterns to mechanisms.” 2018. Doctoral Dissertation, University Utrecht. Accessed January 22, 2021. https://dspace.library.uu.nl/handle/1874/371910 ; URN:NBN:NL:UI:10-1874-371910 ; urn:isbn:978-94-6375-173-5 ; URN:NBN:NL:UI:10-1874-371910 ; https://dspace.library.uu.nl/handle/1874/371910.

MLA Handbook (7th Edition):

Blokzijl, Francis. “Decoding the mutational history of human stem cells : From patterns to mechanisms.” 2018. Web. 22 Jan 2021.

Vancouver:

Blokzijl F. Decoding the mutational history of human stem cells : From patterns to mechanisms. [Internet] [Doctoral dissertation]. University Utrecht; 2018. [cited 2021 Jan 22]. Available from: https://dspace.library.uu.nl/handle/1874/371910 ; URN:NBN:NL:UI:10-1874-371910 ; urn:isbn:978-94-6375-173-5 ; URN:NBN:NL:UI:10-1874-371910 ; https://dspace.library.uu.nl/handle/1874/371910.

Council of Science Editors:

Blokzijl F. Decoding the mutational history of human stem cells : From patterns to mechanisms. [Doctoral Dissertation]. University Utrecht; 2018. Available from: https://dspace.library.uu.nl/handle/1874/371910 ; URN:NBN:NL:UI:10-1874-371910 ; urn:isbn:978-94-6375-173-5 ; URN:NBN:NL:UI:10-1874-371910 ; https://dspace.library.uu.nl/handle/1874/371910

17. Vijayan, Vinaya. Understanding and Improving Identification of Somatic Variants.

Degree: PhD, Genetics, Bioinformatics, and Computational Biology, 2016, Virginia Tech

 It is important to understand the entire spectrum of somatic variants to gain more insight into mutations that occur in different cancers for development of… (more)

Subjects/Keywords: Somatic variants; Somatic variant callers; Somatic point mutations; Short tandem repeat variation; Lung squamous cell carcinoma

…in detecting somatic point mutations in exomes 32 Figure 2.2: Sensitivity, precision and… …F1 score of different pipelines detecting somatic point mutations in genomes 33 Figure 2.3… …Factors affecting the detection of somatic point mutations. 34 Figure 2.4: Sensitivity of… …different pipelines in detecting somatic point mutations using a high quality exome data set 35… …pipelines while detecting somatic point mutations in exomes 36 Figure 2.6: Sensitivity as a… 

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APA (6th Edition):

Vijayan, V. (2016). Understanding and Improving Identification of Somatic Variants. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/72969

Chicago Manual of Style (16th Edition):

Vijayan, Vinaya. “Understanding and Improving Identification of Somatic Variants.” 2016. Doctoral Dissertation, Virginia Tech. Accessed January 22, 2021. http://hdl.handle.net/10919/72969.

MLA Handbook (7th Edition):

Vijayan, Vinaya. “Understanding and Improving Identification of Somatic Variants.” 2016. Web. 22 Jan 2021.

Vancouver:

Vijayan V. Understanding and Improving Identification of Somatic Variants. [Internet] [Doctoral dissertation]. Virginia Tech; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10919/72969.

Council of Science Editors:

Vijayan V. Understanding and Improving Identification of Somatic Variants. [Doctoral Dissertation]. Virginia Tech; 2016. Available from: http://hdl.handle.net/10919/72969


East Carolina University

18. Nopparat, Jongdee. [Delta]-catenin: implications in prostate cancer progression.

Degree: PhD, Anatomy and Cell Biology, 2014, East Carolina University

 Prostate cancer (PCa) is the most commonly diagnosed cancer and the second most common cause of cancer death among men in the US. Due to… (more)

Subjects/Keywords: Biology, Molecular; [Delta]-catenin; Glucose deprivation; Prostate – Cancer; Somatic mutations; Transgenic mouse models; δ-catenin; Molecular biology; Prostatic Neoplasms – diagnosis; Catenins – metabolism; Adenocarcinomas – diagnosis

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APA (6th Edition):

Nopparat, J. (2014). [Delta]-catenin: implications in prostate cancer progression. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/4436

Chicago Manual of Style (16th Edition):

Nopparat, Jongdee. “[Delta]-catenin: implications in prostate cancer progression.” 2014. Doctoral Dissertation, East Carolina University. Accessed January 22, 2021. http://hdl.handle.net/10342/4436.

MLA Handbook (7th Edition):

Nopparat, Jongdee. “[Delta]-catenin: implications in prostate cancer progression.” 2014. Web. 22 Jan 2021.

Vancouver:

Nopparat J. [Delta]-catenin: implications in prostate cancer progression. [Internet] [Doctoral dissertation]. East Carolina University; 2014. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10342/4436.

Council of Science Editors:

Nopparat J. [Delta]-catenin: implications in prostate cancer progression. [Doctoral Dissertation]. East Carolina University; 2014. Available from: http://hdl.handle.net/10342/4436


University of Lund

19. Ravi, Naveen. Genetic Characterization and Identification of Novel Treatment Targets in Anaplastic Thyroid Carcinoma.

Degree: 2019, University of Lund

 Anaplastic thyroid cancer (ATC) is a rare and highly aggressive thyroid malignancy, usually resistant to conventional therapeutic strategies. The prognosis of ATC is extremely poor… (more)

Subjects/Keywords: Cancer and Oncology; Cell and Molecular Biology; Anaplastic thyroid cancer; copy number alterations; somatic mutations; mutational signatures; DNA methylation analysis; Gene expression analysis

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APA (6th Edition):

Ravi, N. (2019). Genetic Characterization and Identification of Novel Treatment Targets in Anaplastic Thyroid Carcinoma. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/b2b10075-1aea-4976-92c0-5798c5163b81 ; https://portal.research.lu.se/ws/files/71276156/Naveen_Ravi_ALL.pdf

Chicago Manual of Style (16th Edition):

Ravi, Naveen. “Genetic Characterization and Identification of Novel Treatment Targets in Anaplastic Thyroid Carcinoma.” 2019. Doctoral Dissertation, University of Lund. Accessed January 22, 2021. https://lup.lub.lu.se/record/b2b10075-1aea-4976-92c0-5798c5163b81 ; https://portal.research.lu.se/ws/files/71276156/Naveen_Ravi_ALL.pdf.

MLA Handbook (7th Edition):

Ravi, Naveen. “Genetic Characterization and Identification of Novel Treatment Targets in Anaplastic Thyroid Carcinoma.” 2019. Web. 22 Jan 2021.

Vancouver:

Ravi N. Genetic Characterization and Identification of Novel Treatment Targets in Anaplastic Thyroid Carcinoma. [Internet] [Doctoral dissertation]. University of Lund; 2019. [cited 2021 Jan 22]. Available from: https://lup.lub.lu.se/record/b2b10075-1aea-4976-92c0-5798c5163b81 ; https://portal.research.lu.se/ws/files/71276156/Naveen_Ravi_ALL.pdf.

Council of Science Editors:

Ravi N. Genetic Characterization and Identification of Novel Treatment Targets in Anaplastic Thyroid Carcinoma. [Doctoral Dissertation]. University of Lund; 2019. Available from: https://lup.lub.lu.se/record/b2b10075-1aea-4976-92c0-5798c5163b81 ; https://portal.research.lu.se/ws/files/71276156/Naveen_Ravi_ALL.pdf


Kyoto University / 京都大学

20. Sawai, Yugo. Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations : Activation-induced cytidine deaminaseは腫瘍関連遺伝子に変異を誘導することにより膵腫瘍形成に寄与する.

Degree: 博士(医学), 2016, Kyoto University / 京都大学

新制・課程博士

甲第19567号

医博第4074号

Subjects/Keywords: Activation-induced cytidine deaminase; Pancreatic cancer; Acinar-ductal metaplasia; Pancreatic intraepithelial neoplasia; Somatic mutations

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APA (6th Edition):

Sawai, Y. (2016). Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations : Activation-induced cytidine deaminaseは腫瘍関連遺伝子に変異を誘導することにより膵腫瘍形成に寄与する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215393 ; http://dx.doi.org/10.14989/doctor.k19567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sawai, Yugo. “Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations : Activation-induced cytidine deaminaseは腫瘍関連遺伝子に変異を誘導することにより膵腫瘍形成に寄与する.” 2016. Thesis, Kyoto University / 京都大学. Accessed January 22, 2021. http://hdl.handle.net/2433/215393 ; http://dx.doi.org/10.14989/doctor.k19567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sawai, Yugo. “Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations : Activation-induced cytidine deaminaseは腫瘍関連遺伝子に変異を誘導することにより膵腫瘍形成に寄与する.” 2016. Web. 22 Jan 2021.

Vancouver:

Sawai Y. Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations : Activation-induced cytidine deaminaseは腫瘍関連遺伝子に変異を誘導することにより膵腫瘍形成に寄与する. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2433/215393 ; http://dx.doi.org/10.14989/doctor.k19567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sawai Y. Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations : Activation-induced cytidine deaminaseは腫瘍関連遺伝子に変異を誘導することにより膵腫瘍形成に寄与する. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215393 ; http://dx.doi.org/10.14989/doctor.k19567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Lianos, Georgios. Προγνωστικοί παράγοντες και εξατομικευμένη χειρουργική στον καρκίνο του στομάχου.

Degree: 2015, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

GASTRIC CANCER CONSTITUTES THE FOURTH MOST COMMON TYPE OF CANCER AND THESECOND CAUSE OF DEATH RELATED WITH CANCER WORLDWIDE. THE AIM OF THIS THESISIS TO… (more)

Subjects/Keywords: Καρκίνος στομάχου; Προγνωστικοί παράγοντες; Εξατομικευμένη ιατρική; Βιοδείκτες; Ανάλυση γονιδιώματος; Τεχνολογίες επόμενης γενιάς; Γονίδια; Σωματικές μεταλλάξεις; Gastric cancer; Prognostic factors; Personalized medicine; Biomarkers; Genome analyses; Next generation sequencing analyses; Somatic mutations; Genes

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APA (6th Edition):

Lianos, G. (2015). Προγνωστικοί παράγοντες και εξατομικευμένη χειρουργική στον καρκίνο του στομάχου. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/41927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lianos, Georgios. “Προγνωστικοί παράγοντες και εξατομικευμένη χειρουργική στον καρκίνο του στομάχου.” 2015. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed January 22, 2021. http://hdl.handle.net/10442/hedi/41927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lianos, Georgios. “Προγνωστικοί παράγοντες και εξατομικευμένη χειρουργική στον καρκίνο του στομάχου.” 2015. Web. 22 Jan 2021.

Vancouver:

Lianos G. Προγνωστικοί παράγοντες και εξατομικευμένη χειρουργική στον καρκίνο του στομάχου. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2015. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10442/hedi/41927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lianos G. Προγνωστικοί παράγοντες και εξατομικευμένη χειρουργική στον καρκίνο του στομάχου. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2015. Available from: http://hdl.handle.net/10442/hedi/41927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

22. Theil, Jan. The role of somatic mutations in the non-coding region of the rearranged immunoglobulin gene in Hodgkin Reed-Sternberg cells for silenced immunoglobulin gene expression in classical Hodgkins disease.

Degree: 2003, Freie Universität Berlin

 The absence of immunoglobulin (Ig) expression in B-cell derived Hodgkin Reed- Sternberg cells of classical Hodgkins disease, has been suggested to be caused by crippling… (more)

Subjects/Keywords: hodgkins disease; immunoglobulin; promoter; somatic mutations; transcription factor; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Theil, J. (2003). The role of somatic mutations in the non-coding region of the rearranged immunoglobulin gene in Hodgkin Reed-Sternberg cells for silenced immunoglobulin gene expression in classical Hodgkins disease. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8062

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Theil, Jan. “The role of somatic mutations in the non-coding region of the rearranged immunoglobulin gene in Hodgkin Reed-Sternberg cells for silenced immunoglobulin gene expression in classical Hodgkins disease.” 2003. Thesis, Freie Universität Berlin. Accessed January 22, 2021. http://dx.doi.org/10.17169/refubium-8062.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Theil, Jan. “The role of somatic mutations in the non-coding region of the rearranged immunoglobulin gene in Hodgkin Reed-Sternberg cells for silenced immunoglobulin gene expression in classical Hodgkins disease.” 2003. Web. 22 Jan 2021.

Vancouver:

Theil J. The role of somatic mutations in the non-coding region of the rearranged immunoglobulin gene in Hodgkin Reed-Sternberg cells for silenced immunoglobulin gene expression in classical Hodgkins disease. [Internet] [Thesis]. Freie Universität Berlin; 2003. [cited 2021 Jan 22]. Available from: http://dx.doi.org/10.17169/refubium-8062.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Theil J. The role of somatic mutations in the non-coding region of the rearranged immunoglobulin gene in Hodgkin Reed-Sternberg cells for silenced immunoglobulin gene expression in classical Hodgkins disease. [Thesis]. Freie Universität Berlin; 2003. Available from: http://dx.doi.org/10.17169/refubium-8062

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Queensland

23. Guo, Zeng. Histopathological, biochemical and genetic characterization of different types of primary aldosteronism, including validation of a new angiotensin assay.

Degree: Faculty of Medicine, 2020, University of Queensland

Subjects/Keywords: Primary aldosteronism; Renin-angiotensin system; Mass spectrometry; Hybrid steroids; Somatic gene mutations; 1101 Medical Biochemistry and Metabolomics; 1103 Clinical Sciences

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APA (6th Edition):

Guo, Z. (2020). Histopathological, biochemical and genetic characterization of different types of primary aldosteronism, including validation of a new angiotensin assay. (Thesis). University of Queensland. Retrieved from https://espace.library.uq.edu.au/view/UQ:d835d46/thumbnail_s44091301_final_thesis_t.jpg ; https://espace.library.uq.edu.au/view/UQ:d835d46/s44091301_final_thesis.pdf ; https://espace.library.uq.edu.au/view/UQ:d835d46

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Zeng. “Histopathological, biochemical and genetic characterization of different types of primary aldosteronism, including validation of a new angiotensin assay.” 2020. Thesis, University of Queensland. Accessed January 22, 2021. https://espace.library.uq.edu.au/view/UQ:d835d46/thumbnail_s44091301_final_thesis_t.jpg ; https://espace.library.uq.edu.au/view/UQ:d835d46/s44091301_final_thesis.pdf ; https://espace.library.uq.edu.au/view/UQ:d835d46.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Zeng. “Histopathological, biochemical and genetic characterization of different types of primary aldosteronism, including validation of a new angiotensin assay.” 2020. Web. 22 Jan 2021.

Vancouver:

Guo Z. Histopathological, biochemical and genetic characterization of different types of primary aldosteronism, including validation of a new angiotensin assay. [Internet] [Thesis]. University of Queensland; 2020. [cited 2021 Jan 22]. Available from: https://espace.library.uq.edu.au/view/UQ:d835d46/thumbnail_s44091301_final_thesis_t.jpg ; https://espace.library.uq.edu.au/view/UQ:d835d46/s44091301_final_thesis.pdf ; https://espace.library.uq.edu.au/view/UQ:d835d46.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo Z. Histopathological, biochemical and genetic characterization of different types of primary aldosteronism, including validation of a new angiotensin assay. [Thesis]. University of Queensland; 2020. Available from: https://espace.library.uq.edu.au/view/UQ:d835d46/thumbnail_s44091301_final_thesis_t.jpg ; https://espace.library.uq.edu.au/view/UQ:d835d46/s44091301_final_thesis.pdf ; https://espace.library.uq.edu.au/view/UQ:d835d46

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Bos, C.J. Induced mutation and somatic recombination as tools for genetic analysis and breeding of imperfect fungi.

Degree: 1986, Landbouwhogeschool Wageningen

 <p/>Many fungi which are important in Agriculture as plant pathogens or in Biotechnology as producers of organic acids, antibiotics or enzymes, are imperfect fungi. These… (more)

Subjects/Keywords: deuteromycotina; somatische hybridisatie; verwijderde hybridisatie; geïnduceerde mutaties; Anamorfe schimmels; deuteromycotina; somatic hybridization; wide hybridization; induced mutations; Anamorphic Fungi

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APA (6th Edition):

Bos, C. J. (1986). Induced mutation and somatic recombination as tools for genetic analysis and breeding of imperfect fungi. (Doctoral Dissertation). Landbouwhogeschool Wageningen. Retrieved from http://library.wur.nl/WebQuery/wurpubs/1091 ; urn:nbn:nl:ui:32-1091 ; urn:nbn:nl:ui:32-1091 ; http://library.wur.nl/WebQuery/wurpubs/1091

Chicago Manual of Style (16th Edition):

Bos, C J. “Induced mutation and somatic recombination as tools for genetic analysis and breeding of imperfect fungi.” 1986. Doctoral Dissertation, Landbouwhogeschool Wageningen. Accessed January 22, 2021. http://library.wur.nl/WebQuery/wurpubs/1091 ; urn:nbn:nl:ui:32-1091 ; urn:nbn:nl:ui:32-1091 ; http://library.wur.nl/WebQuery/wurpubs/1091.

MLA Handbook (7th Edition):

Bos, C J. “Induced mutation and somatic recombination as tools for genetic analysis and breeding of imperfect fungi.” 1986. Web. 22 Jan 2021.

Vancouver:

Bos CJ. Induced mutation and somatic recombination as tools for genetic analysis and breeding of imperfect fungi. [Internet] [Doctoral dissertation]. Landbouwhogeschool Wageningen; 1986. [cited 2021 Jan 22]. Available from: http://library.wur.nl/WebQuery/wurpubs/1091 ; urn:nbn:nl:ui:32-1091 ; urn:nbn:nl:ui:32-1091 ; http://library.wur.nl/WebQuery/wurpubs/1091.

Council of Science Editors:

Bos CJ. Induced mutation and somatic recombination as tools for genetic analysis and breeding of imperfect fungi. [Doctoral Dissertation]. Landbouwhogeschool Wageningen; 1986. Available from: http://library.wur.nl/WebQuery/wurpubs/1091 ; urn:nbn:nl:ui:32-1091 ; urn:nbn:nl:ui:32-1091 ; http://library.wur.nl/WebQuery/wurpubs/1091


Erasmus University Rotterdam

25. MacKenzie, Katherine. Hirschsprung Disease: development & treatment avenues: De ziekte van Hirschsprung: ontwikkeling & behandelingsmogelijkheden.

Degree: 2019, Erasmus University Rotterdam

 textabstractThe work of this thesis investigates the development of, and treatment options for, Hirschsprung disease (HSCR). We explore the missing heritability that is seen in… (more)

Subjects/Keywords: Hirschsprung disease; enteric nervous system; neural crest; somatic mutations; copy number variation; Goldberg-Shprintzen syndrome; KIF1BP; missense variations; cell replacement therapy

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APA (6th Edition):

MacKenzie, K. (2019). Hirschsprung Disease: development & treatment avenues: De ziekte van Hirschsprung: ontwikkeling & behandelingsmogelijkheden. (Doctoral Dissertation). Erasmus University Rotterdam. Retrieved from http://hdl.handle.net/1765/116709

Chicago Manual of Style (16th Edition):

MacKenzie, Katherine. “Hirschsprung Disease: development & treatment avenues: De ziekte van Hirschsprung: ontwikkeling & behandelingsmogelijkheden.” 2019. Doctoral Dissertation, Erasmus University Rotterdam. Accessed January 22, 2021. http://hdl.handle.net/1765/116709.

MLA Handbook (7th Edition):

MacKenzie, Katherine. “Hirschsprung Disease: development & treatment avenues: De ziekte van Hirschsprung: ontwikkeling & behandelingsmogelijkheden.” 2019. Web. 22 Jan 2021.

Vancouver:

MacKenzie K. Hirschsprung Disease: development & treatment avenues: De ziekte van Hirschsprung: ontwikkeling & behandelingsmogelijkheden. [Internet] [Doctoral dissertation]. Erasmus University Rotterdam; 2019. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1765/116709.

Council of Science Editors:

MacKenzie K. Hirschsprung Disease: development & treatment avenues: De ziekte van Hirschsprung: ontwikkeling & behandelingsmogelijkheden. [Doctoral Dissertation]. Erasmus University Rotterdam; 2019. Available from: http://hdl.handle.net/1765/116709

26. Akker, Peter Christiaan van den. Dystrophic epidermolysis bullosa: novel insights into the genotype-phenotype correlation and somatic mosaicism.

Degree: Faculteit Medische Wetenschappen/UMCG, 2013, NARCIS

 Dystrofische epidermolysis bullosa (DEB) is een erfelijke huidaandoening, veroorzaakt door mutaties in het COL7A1 gen. Bij DEB ontstaan reeds door geringe wrijving blaren van de… (more)

Subjects/Keywords: Proefschriften (vorm); Somatic mutations; Populatiegenetica; Databanken; Fenotype; Genotype; Dystrofie; Epidermolysis bullosa; aangeboren afwijkingen; dermatologie (geneeskunde)

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APA (6th Edition):

Akker, P. C. v. d. (2013). Dystrophic epidermolysis bullosa: novel insights into the genotype-phenotype correlation and somatic mosaicism. (Doctoral Dissertation). NARCIS. Retrieved from https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; 3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; 11370/3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; urn:isbn:9789036764308 ; urn:isbn:9789036764292 ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html

Chicago Manual of Style (16th Edition):

Akker, Peter Christiaan van den. “Dystrophic epidermolysis bullosa: novel insights into the genotype-phenotype correlation and somatic mosaicism.” 2013. Doctoral Dissertation, NARCIS. Accessed January 22, 2021. https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; 3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; 11370/3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; urn:isbn:9789036764308 ; urn:isbn:9789036764292 ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html.

MLA Handbook (7th Edition):

Akker, Peter Christiaan van den. “Dystrophic epidermolysis bullosa: novel insights into the genotype-phenotype correlation and somatic mosaicism.” 2013. Web. 22 Jan 2021.

Vancouver:

Akker PCvd. Dystrophic epidermolysis bullosa: novel insights into the genotype-phenotype correlation and somatic mosaicism. [Internet] [Doctoral dissertation]. NARCIS; 2013. [cited 2021 Jan 22]. Available from: https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; 3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; 11370/3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; urn:isbn:9789036764308 ; urn:isbn:9789036764292 ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html.

Council of Science Editors:

Akker PCvd. Dystrophic epidermolysis bullosa: novel insights into the genotype-phenotype correlation and somatic mosaicism. [Doctoral Dissertation]. NARCIS; 2013. Available from: https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; 3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; 11370/3acb2fda-cd7d-4bc4-9b9b-eec911c20d37 ; urn:isbn:9789036764308 ; urn:isbn:9789036764292 ; urn:nbn:nl:ui:11-dbi/525e96a7b771a ; https://www.rug.nl/research/portal/en/publications/dystrophic-epidermolysis-bullosa(3acb2fda-cd7d-4bc4-9b9b-eec911c20d37).html

27. Sawai, Yugo. Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations .

Degree: 2016, Kyoto University

Subjects/Keywords: Activation-induced cytidine deaminase; Pancreatic cancer; Acinar-ductal metaplasia; Pancreatic intraepithelial neoplasia; Somatic mutations

Page 1 Page 2

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APA (6th Edition):

Sawai, Y. (2016). Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/215393

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sawai, Yugo. “Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations .” 2016. Thesis, Kyoto University. Accessed January 22, 2021. http://hdl.handle.net/2433/215393.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sawai, Yugo. “Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations .” 2016. Web. 22 Jan 2021.

Vancouver:

Sawai Y. Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations . [Internet] [Thesis]. Kyoto University; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2433/215393.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sawai Y. Activation-Induced Cytidine Deaminase Contributes to Pancreatic Tumorigenesis by Inducing Tumor-Related Gene Mutations . [Thesis]. Kyoto University; 2016. Available from: http://hdl.handle.net/2433/215393

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Youssef, Sarah Hisham Abdelaziz. Somatic Mutation Detection in Leukemia-Derived Circulating DNA: Utility in Monitoring Clonal Dynamics and Disease Response in Pediatric Acute Lymphoblastic Leukemia.

Degree: MS, Pharmaceutical Sciences, 2018, University of Tennessee Health Science Center

  Despite the improved outcome associated with current treatment strategies ofpediatric acute lymphoblastic leukemia (ALL), relapse still represents a major challenge. Pediatric ALL demonstrates branched… (more)

Subjects/Keywords: Acute Lymphoblastic Leukemia; Circulating-tumor DNA (Ct-DNA); Clonal Dynamics; Leukemia-derived Circulating DNA; Pediatrics; Somatic Mutations; Medical Genetics; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences; Pharmacy and Pharmaceutical Sciences

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APA (6th Edition):

Youssef, S. H. A. (2018). Somatic Mutation Detection in Leukemia-Derived Circulating DNA: Utility in Monitoring Clonal Dynamics and Disease Response in Pediatric Acute Lymphoblastic Leukemia. (Thesis). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Youssef, Sarah Hisham Abdelaziz. “Somatic Mutation Detection in Leukemia-Derived Circulating DNA: Utility in Monitoring Clonal Dynamics and Disease Response in Pediatric Acute Lymphoblastic Leukemia.” 2018. Thesis, University of Tennessee Health Science Center. Accessed January 22, 2021. https://dc.uthsc.edu/dissertations/458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Youssef, Sarah Hisham Abdelaziz. “Somatic Mutation Detection in Leukemia-Derived Circulating DNA: Utility in Monitoring Clonal Dynamics and Disease Response in Pediatric Acute Lymphoblastic Leukemia.” 2018. Web. 22 Jan 2021.

Vancouver:

Youssef SHA. Somatic Mutation Detection in Leukemia-Derived Circulating DNA: Utility in Monitoring Clonal Dynamics and Disease Response in Pediatric Acute Lymphoblastic Leukemia. [Internet] [Thesis]. University of Tennessee Health Science Center; 2018. [cited 2021 Jan 22]. Available from: https://dc.uthsc.edu/dissertations/458.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Youssef SHA. Somatic Mutation Detection in Leukemia-Derived Circulating DNA: Utility in Monitoring Clonal Dynamics and Disease Response in Pediatric Acute Lymphoblastic Leukemia. [Thesis]. University of Tennessee Health Science Center; 2018. Available from: https://dc.uthsc.edu/dissertations/458

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Θώμου, Χριστίνα. Επίκτητες σωματικές μεταλλάξεις στο μικροδορυφορικό DNA σε παιδιά με βρογχικό άσθμα.

Degree: 2010, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Objectives High incidence of genetic alterations at the microsatellite (MS) DNA level has been reported in asthmatic adults. Working Hypothesis The aim of this study… (more)

Subjects/Keywords: Παιδικό άσθμα; Γενετική ευαισθησία; Μικροδορυφορική αστάθεια; Απώλεια της ετεροζυγωτίας; Προκλητά πτύελα; Σωματικές μεταλλάξεις; Childhood asthma; Genetic susceptibility; Microsatellite instability ( MSI ); Loss of heterozygosity; Induced sputum; Somatic mutations

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APA (6th Edition):

Θώμου, . . (2010). Επίκτητες σωματικές μεταλλάξεις στο μικροδορυφορικό DNA σε παιδιά με βρογχικό άσθμα. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/24661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Θώμου, Χριστίνα. “Επίκτητες σωματικές μεταλλάξεις στο μικροδορυφορικό DNA σε παιδιά με βρογχικό άσθμα.” 2010. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed January 22, 2021. http://hdl.handle.net/10442/hedi/24661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Θώμου, Χριστίνα. “Επίκτητες σωματικές μεταλλάξεις στο μικροδορυφορικό DNA σε παιδιά με βρογχικό άσθμα.” 2010. Web. 22 Jan 2021.

Vancouver:

Θώμου . Επίκτητες σωματικές μεταλλάξεις στο μικροδορυφορικό DNA σε παιδιά με βρογχικό άσθμα. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2010. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10442/hedi/24661.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Θώμου . Επίκτητες σωματικές μεταλλάξεις στο μικροδορυφορικό DNA σε παιδιά με βρογχικό άσθμα. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2010. Available from: http://hdl.handle.net/10442/hedi/24661

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Uijtewaal, B.A. The production and evaluation of monohaploid potatoes (2n=x=12) for breeding research on cell and plant level.

Degree: 1987, Agricultural University

  The use of monohaploid potato clones in research in theory has many potential advantages in comparison with the use of heterozygous di- and tetraploids.… (more)

Subjects/Keywords: genomen; haploïdie; hybriden; soortkruising; mutaties; plantenveredeling; polyploïdie; aardappelen; solanum tuberosum; somatische hybridisatie; Plantenveredeling, selectiemethoden; genomes; haploidy; hybrids; interspecific hybridization; mutations; plant breeding; polyploidy; potatoes; solanum tuberosum; somatic hybridization; Plant Breeding and Selection Methods

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APA (6th Edition):

Uijtewaal, B. A. (1987). The production and evaluation of monohaploid potatoes (2n=x=12) for breeding research on cell and plant level. (Doctoral Dissertation). Agricultural University. Retrieved from http://library.wur.nl/WebQuery/wurpubs/3806 ; urn:nbn:nl:ui:32-3806 ; urn:nbn:nl:ui:32-3806 ; http://library.wur.nl/WebQuery/wurpubs/3806

Chicago Manual of Style (16th Edition):

Uijtewaal, B A. “The production and evaluation of monohaploid potatoes (2n=x=12) for breeding research on cell and plant level.” 1987. Doctoral Dissertation, Agricultural University. Accessed January 22, 2021. http://library.wur.nl/WebQuery/wurpubs/3806 ; urn:nbn:nl:ui:32-3806 ; urn:nbn:nl:ui:32-3806 ; http://library.wur.nl/WebQuery/wurpubs/3806.

MLA Handbook (7th Edition):

Uijtewaal, B A. “The production and evaluation of monohaploid potatoes (2n=x=12) for breeding research on cell and plant level.” 1987. Web. 22 Jan 2021.

Vancouver:

Uijtewaal BA. The production and evaluation of monohaploid potatoes (2n=x=12) for breeding research on cell and plant level. [Internet] [Doctoral dissertation]. Agricultural University; 1987. [cited 2021 Jan 22]. Available from: http://library.wur.nl/WebQuery/wurpubs/3806 ; urn:nbn:nl:ui:32-3806 ; urn:nbn:nl:ui:32-3806 ; http://library.wur.nl/WebQuery/wurpubs/3806.

Council of Science Editors:

Uijtewaal BA. The production and evaluation of monohaploid potatoes (2n=x=12) for breeding research on cell and plant level. [Doctoral Dissertation]. Agricultural University; 1987. Available from: http://library.wur.nl/WebQuery/wurpubs/3806 ; urn:nbn:nl:ui:32-3806 ; urn:nbn:nl:ui:32-3806 ; http://library.wur.nl/WebQuery/wurpubs/3806

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