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You searched for subject:(shRNA). Showing records 1 – 30 of 87 total matches.

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University of Cambridge

1. Erard, Nicolas Pascal Jean. Optimization of molecular tools for high-throughput genetic screening.

Degree: PhD, 2018, University of Cambridge

 Forward genetic screening allows for the identification of any genes important for a particular biological process or phenotype. While the power of this approach is… (more)

Subjects/Keywords: Cancer; shRNA; CRISPR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Erard, N. P. J. (2018). Optimization of molecular tools for high-throughput genetic screening. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/271895

Chicago Manual of Style (16th Edition):

Erard, Nicolas Pascal Jean. “Optimization of molecular tools for high-throughput genetic screening.” 2018. Doctoral Dissertation, University of Cambridge. Accessed April 16, 2021. https://www.repository.cam.ac.uk/handle/1810/271895.

MLA Handbook (7th Edition):

Erard, Nicolas Pascal Jean. “Optimization of molecular tools for high-throughput genetic screening.” 2018. Web. 16 Apr 2021.

Vancouver:

Erard NPJ. Optimization of molecular tools for high-throughput genetic screening. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Apr 16]. Available from: https://www.repository.cam.ac.uk/handle/1810/271895.

Council of Science Editors:

Erard NPJ. Optimization of molecular tools for high-throughput genetic screening. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/271895

2. 김, 은영. Regulation of Senescence Phenotypes by TIS21 gene in Huh7 Human Liver Cancer Cells.

Degree: 2011, Ajou University

일반적으로 Cellular senescence란 시간이 지나면 자연적으로 세포 증식이 억제되고 세포내 항상성 유지가 어렵게 되는 것을 말한다. 이러한 cellular senescence는 tumour cell에서도 일어나는데 tumour cell에서의 senescence는… (more)

Subjects/Keywords: TIS21; doxorubicin; 세포노화유도; Huh7; shRNA

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APA (6th Edition):

김, . (2011). Regulation of Senescence Phenotypes by TIS21 gene in Huh7 Human Liver Cancer Cells. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/4405 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000011837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

김, 은영. “Regulation of Senescence Phenotypes by TIS21 gene in Huh7 Human Liver Cancer Cells.” 2011. Thesis, Ajou University. Accessed April 16, 2021. http://repository.ajou.ac.kr/handle/201003/4405 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000011837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

김, 은영. “Regulation of Senescence Phenotypes by TIS21 gene in Huh7 Human Liver Cancer Cells.” 2011. Web. 16 Apr 2021.

Vancouver:

김 . Regulation of Senescence Phenotypes by TIS21 gene in Huh7 Human Liver Cancer Cells. [Internet] [Thesis]. Ajou University; 2011. [cited 2021 Apr 16]. Available from: http://repository.ajou.ac.kr/handle/201003/4405 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000011837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

김 . Regulation of Senescence Phenotypes by TIS21 gene in Huh7 Human Liver Cancer Cells. [Thesis]. Ajou University; 2011. Available from: http://repository.ajou.ac.kr/handle/201003/4405 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000011837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

3. Erard, Nicolas Pascal Jean. Optimization of molecular tools for high-throughput genetic screening.

Degree: PhD, 2018, University of Cambridge

 Forward genetic screening allows for the identification of any genes important for a particular biological process or phenotype. While the power of this approach is… (more)

Subjects/Keywords: 616.99; Cancer; shRNA; CRISPR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Erard, N. P. J. (2018). Optimization of molecular tools for high-throughput genetic screening. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.18903 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744534

Chicago Manual of Style (16th Edition):

Erard, Nicolas Pascal Jean. “Optimization of molecular tools for high-throughput genetic screening.” 2018. Doctoral Dissertation, University of Cambridge. Accessed April 16, 2021. https://doi.org/10.17863/CAM.18903 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744534.

MLA Handbook (7th Edition):

Erard, Nicolas Pascal Jean. “Optimization of molecular tools for high-throughput genetic screening.” 2018. Web. 16 Apr 2021.

Vancouver:

Erard NPJ. Optimization of molecular tools for high-throughput genetic screening. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Apr 16]. Available from: https://doi.org/10.17863/CAM.18903 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744534.

Council of Science Editors:

Erard NPJ. Optimization of molecular tools for high-throughput genetic screening. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.18903 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744534


University of Texas – Austin

4. -8375-338X. Characterization of the biogenesis and function of miRNAs encoded by diverse tumor viruses.

Degree: PhD, Cell and Molecular Biology, 2016, University of Texas – Austin

 Some eukaryotic viruses express small RNAs called microRNAs (miRNAs) to regulate host and viral gene expression. Due to their small genomic footprint, ability to regulate… (more)

Subjects/Keywords: miRNA; Polyomavirus; BLV; shRNA; RNAi

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APA (6th Edition):

-8375-338X. (2016). Characterization of the biogenesis and function of miRNAs encoded by diverse tumor viruses. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/68600

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-8375-338X. “Characterization of the biogenesis and function of miRNAs encoded by diverse tumor viruses.” 2016. Doctoral Dissertation, University of Texas – Austin. Accessed April 16, 2021. http://hdl.handle.net/2152/68600.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-8375-338X. “Characterization of the biogenesis and function of miRNAs encoded by diverse tumor viruses.” 2016. Web. 16 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-8375-338X. Characterization of the biogenesis and function of miRNAs encoded by diverse tumor viruses. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2016. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2152/68600.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-8375-338X. Characterization of the biogenesis and function of miRNAs encoded by diverse tumor viruses. [Doctoral Dissertation]. University of Texas – Austin; 2016. Available from: http://hdl.handle.net/2152/68600

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

5. Jivrajani Mehul. Targeted Delivery of shRNA to Cancer Cell;.

Degree: 2015, Nirma University

newline

Advisors/Committee Members: Nivsarkar Manish.

Subjects/Keywords: Cancer; Cell; shRNA

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APA (6th Edition):

Mehul, J. (2015). Targeted Delivery of shRNA to Cancer Cell;. (Thesis). Nirma University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/44918

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mehul, Jivrajani. “Targeted Delivery of shRNA to Cancer Cell;.” 2015. Thesis, Nirma University. Accessed April 16, 2021. http://shodhganga.inflibnet.ac.in/handle/10603/44918.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mehul, Jivrajani. “Targeted Delivery of shRNA to Cancer Cell;.” 2015. Web. 16 Apr 2021.

Vancouver:

Mehul J. Targeted Delivery of shRNA to Cancer Cell;. [Internet] [Thesis]. Nirma University; 2015. [cited 2021 Apr 16]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/44918.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mehul J. Targeted Delivery of shRNA to Cancer Cell;. [Thesis]. Nirma University; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/44918

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tasmania

6. Casey, NP. The Therapeutic potential of lentivector-delivered RNAi.

Degree: 2010, University of Tasmania

 Many forms of leukaemia are caused by chromosomal translocations, which result in specific and characteristic genomic sequences. Where these sequences are unique to the leukaemic… (more)

Subjects/Keywords: lentivirus; vector; RNAi; shRNA; chrosomal-translocation; leukaemia

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APA (6th Edition):

Casey, N. (2010). The Therapeutic potential of lentivector-delivered RNAi. (Thesis). University of Tasmania. Retrieved from https://eprints.utas.edu.au/10783/1/01_Casey_front.pdf ; https://eprints.utas.edu.au/10783/2/01_Casey_whole.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Casey, NP. “The Therapeutic potential of lentivector-delivered RNAi.” 2010. Thesis, University of Tasmania. Accessed April 16, 2021. https://eprints.utas.edu.au/10783/1/01_Casey_front.pdf ; https://eprints.utas.edu.au/10783/2/01_Casey_whole.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Casey, NP. “The Therapeutic potential of lentivector-delivered RNAi.” 2010. Web. 16 Apr 2021.

Vancouver:

Casey N. The Therapeutic potential of lentivector-delivered RNAi. [Internet] [Thesis]. University of Tasmania; 2010. [cited 2021 Apr 16]. Available from: https://eprints.utas.edu.au/10783/1/01_Casey_front.pdf ; https://eprints.utas.edu.au/10783/2/01_Casey_whole.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Casey N. The Therapeutic potential of lentivector-delivered RNAi. [Thesis]. University of Tasmania; 2010. Available from: https://eprints.utas.edu.au/10783/1/01_Casey_front.pdf ; https://eprints.utas.edu.au/10783/2/01_Casey_whole.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

7. Blaser, Julian. The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia.

Degree: 2012, University of Manchester

 Phosphoinositides (PIs) are pivotal lipid molecules with both scaffolding and signalling functions regulating key aspects of cellular physiology. For example, phosphatidylinositol (3,4,5)-trisphosphate, generated by phosphoinositide… (more)

Subjects/Keywords: RNAi; shRNA; acute myeloid leukaemia; phosphoinositides

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Blaser, J. (2012). The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:182916

Chicago Manual of Style (16th Edition):

Blaser, Julian. “The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia.” 2012. Doctoral Dissertation, University of Manchester. Accessed April 16, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:182916.

MLA Handbook (7th Edition):

Blaser, Julian. “The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia.” 2012. Web. 16 Apr 2021.

Vancouver:

Blaser J. The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2021 Apr 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:182916.

Council of Science Editors:

Blaser J. The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:182916


Queens University

8. Lian, Eric. Characterization of isoform specific RET knockdown in cancer cell lines .

Degree: Pathology and Molecular Medicine, 2013, Queens University

 The REarranged in Transfection (RET) tyrosine kinase is an important signalling protein for the development of neural crest-derived tissues such as the enteric and sympathetic… (more)

Subjects/Keywords: Migration ; RET ; Cancer ; Isoforms ; shRNA ; Invasion

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APA (6th Edition):

Lian, E. (2013). Characterization of isoform specific RET knockdown in cancer cell lines . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8237

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lian, Eric. “Characterization of isoform specific RET knockdown in cancer cell lines .” 2013. Thesis, Queens University. Accessed April 16, 2021. http://hdl.handle.net/1974/8237.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lian, Eric. “Characterization of isoform specific RET knockdown in cancer cell lines .” 2013. Web. 16 Apr 2021.

Vancouver:

Lian E. Characterization of isoform specific RET knockdown in cancer cell lines . [Internet] [Thesis]. Queens University; 2013. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1974/8237.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lian E. Characterization of isoform specific RET knockdown in cancer cell lines . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8237

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

9. Ledger, Scott. Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection.

Degree: Clinical School - St Vincent's Hospital, 2016, University of New South Wales

 HIV currently infects 35 million people worldwide and antiretroviral treatments are expensive, lifelong, and fail to provide a cure. Gene therapy provides an alternate approach… (more)

Subjects/Keywords: shRNA; HIV; Gene therapy; CCR5; C46; lentivirus

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APA (6th Edition):

Ledger, S. (2016). Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Ledger, Scott. “Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection.” 2016. Doctoral Dissertation, University of New South Wales. Accessed April 16, 2021. http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true.

MLA Handbook (7th Edition):

Ledger, Scott. “Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection.” 2016. Web. 16 Apr 2021.

Vancouver:

Ledger S. Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2021 Apr 16]. Available from: http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true.

Council of Science Editors:

Ledger S. Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true


University of Manchester

10. Blaser, Julian. The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia.

Degree: PhD, 2013, University of Manchester

 Phosphoinositides (PIs) are pivotal lipid molecules with both scaffolding and signalling functions regulating key aspects of cellular physiology. For example, phosphatidylinositol (3,4,5)-trisphosphate, generated by phosphoinositide… (more)

Subjects/Keywords: 616.99; phosphoinositides; acute myeloid leukaemia; RNAi; shRNA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Blaser, J. (2013). The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-utilisation-of-shrna-screens-to-investigate-the-role-of-phosphoinositide-modulator-genes-in-actue-myeloid-leukaemia(87b33955-d4e9-4398-bd1d-58e4c5142eb3).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764253

Chicago Manual of Style (16th Edition):

Blaser, Julian. “The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia.” 2013. Doctoral Dissertation, University of Manchester. Accessed April 16, 2021. https://www.research.manchester.ac.uk/portal/en/theses/the-utilisation-of-shrna-screens-to-investigate-the-role-of-phosphoinositide-modulator-genes-in-actue-myeloid-leukaemia(87b33955-d4e9-4398-bd1d-58e4c5142eb3).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764253.

MLA Handbook (7th Edition):

Blaser, Julian. “The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia.” 2013. Web. 16 Apr 2021.

Vancouver:

Blaser J. The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2021 Apr 16]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-utilisation-of-shrna-screens-to-investigate-the-role-of-phosphoinositide-modulator-genes-in-actue-myeloid-leukaemia(87b33955-d4e9-4398-bd1d-58e4c5142eb3).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764253.

Council of Science Editors:

Blaser J. The utilisation of shRNA screens to investigate the role of phosphoinositide modulator genes in actue myeloid leukaemia. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-utilisation-of-shrna-screens-to-investigate-the-role-of-phosphoinositide-modulator-genes-in-actue-myeloid-leukaemia(87b33955-d4e9-4398-bd1d-58e4c5142eb3).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764253

11. Helena Bacha Lopes. Participação de integrinas na diferenciação osteoblástica induzida por superfícies de titânio com nano e microtopografia.

Degree: 2018, University of São Paulo

As integrinas constituem uma família de receptores de membrana que tem como função primária a adesão de células a proteínas da matriz extracelular e alguns… (more)

Subjects/Keywords: CRISPR/Cas9; Integrina; Nanotopografia; Osteoblasto; ShRNA; Titânio; CRISPR/Cas9; Integrin; Nanotopography; Osteoblast; ShRNA; Titanium

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APA (6th Edition):

Lopes, H. B. (2018). Participação de integrinas na diferenciação osteoblástica induzida por superfícies de titânio com nano e microtopografia. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/58/58136/tde-19032019-163700/

Chicago Manual of Style (16th Edition):

Lopes, Helena Bacha. “Participação de integrinas na diferenciação osteoblástica induzida por superfícies de titânio com nano e microtopografia.” 2018. Doctoral Dissertation, University of São Paulo. Accessed April 16, 2021. http://www.teses.usp.br/teses/disponiveis/58/58136/tde-19032019-163700/.

MLA Handbook (7th Edition):

Lopes, Helena Bacha. “Participação de integrinas na diferenciação osteoblástica induzida por superfícies de titânio com nano e microtopografia.” 2018. Web. 16 Apr 2021.

Vancouver:

Lopes HB. Participação de integrinas na diferenciação osteoblástica induzida por superfícies de titânio com nano e microtopografia. [Internet] [Doctoral dissertation]. University of São Paulo; 2018. [cited 2021 Apr 16]. Available from: http://www.teses.usp.br/teses/disponiveis/58/58136/tde-19032019-163700/.

Council of Science Editors:

Lopes HB. Participação de integrinas na diferenciação osteoblástica induzida por superfícies de titânio com nano e microtopografia. [Doctoral Dissertation]. University of São Paulo; 2018. Available from: http://www.teses.usp.br/teses/disponiveis/58/58136/tde-19032019-163700/

12. Amanda Ikegami. Avaliação in vitro e in vivo do impacto do silenciamento do gene NF-kB1 no ciclo celular, capacidade proliferativa e na radiossensibilidade do adenocarcinoma renal.

Degree: 2019, University of São Paulo

O carcinoma de células renais (CCR) é responsável por, aproximadamente, 1 a 3% das neoplasias malignas humanas e, dentre os tumores urológicos, é o mais… (more)

Subjects/Keywords: carcinoma de células renais; NF-kB1; proliferação; shRNA; NF-kB1; proliferation; renal cell carcinoma; shRNA

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APA (6th Edition):

Ikegami, A. (2019). Avaliação in vitro e in vivo do impacto do silenciamento do gene NF-kB1 no ciclo celular, capacidade proliferativa e na radiossensibilidade do adenocarcinoma renal. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/85/85131/tde-10052019-155321/

Chicago Manual of Style (16th Edition):

Ikegami, Amanda. “Avaliação in vitro e in vivo do impacto do silenciamento do gene NF-kB1 no ciclo celular, capacidade proliferativa e na radiossensibilidade do adenocarcinoma renal.” 2019. Masters Thesis, University of São Paulo. Accessed April 16, 2021. http://www.teses.usp.br/teses/disponiveis/85/85131/tde-10052019-155321/.

MLA Handbook (7th Edition):

Ikegami, Amanda. “Avaliação in vitro e in vivo do impacto do silenciamento do gene NF-kB1 no ciclo celular, capacidade proliferativa e na radiossensibilidade do adenocarcinoma renal.” 2019. Web. 16 Apr 2021.

Vancouver:

Ikegami A. Avaliação in vitro e in vivo do impacto do silenciamento do gene NF-kB1 no ciclo celular, capacidade proliferativa e na radiossensibilidade do adenocarcinoma renal. [Internet] [Masters thesis]. University of São Paulo; 2019. [cited 2021 Apr 16]. Available from: http://www.teses.usp.br/teses/disponiveis/85/85131/tde-10052019-155321/.

Council of Science Editors:

Ikegami A. Avaliação in vitro e in vivo do impacto do silenciamento do gene NF-kB1 no ciclo celular, capacidade proliferativa e na radiossensibilidade do adenocarcinoma renal. [Masters Thesis]. University of São Paulo; 2019. Available from: http://www.teses.usp.br/teses/disponiveis/85/85131/tde-10052019-155321/


Johannes Gutenberg Universität Mainz

13. Wrede, Christine. Transfer von antitumoralen Effektoren mittels viraler Vektoren zur experimentellen Tumortherapie.

Degree: 2011, Johannes Gutenberg Universität Mainz

Retrovirale Vektoren basierend auf dem murinen Leukämievirus (MLV) gehören zu den zurzeit am häufigsten verwendeten Vektoren in der Gentherapie. MLV besitzt einen natürlichen Tropismus für… (more)

Subjects/Keywords: murines Leukemievirus (MLV); shRNA; viraler Vektor; Gentherapie; murine leukaemia virus (MLV); shRNA; viral vector; gentherapy; Life sciences

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APA (6th Edition):

Wrede, C. (2011). Transfer von antitumoralen Effektoren mittels viraler Vektoren zur experimentellen Tumortherapie. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2012/3253/

Chicago Manual of Style (16th Edition):

Wrede, Christine. “Transfer von antitumoralen Effektoren mittels viraler Vektoren zur experimentellen Tumortherapie.” 2011. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed April 16, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2012/3253/.

MLA Handbook (7th Edition):

Wrede, Christine. “Transfer von antitumoralen Effektoren mittels viraler Vektoren zur experimentellen Tumortherapie.” 2011. Web. 16 Apr 2021.

Vancouver:

Wrede C. Transfer von antitumoralen Effektoren mittels viraler Vektoren zur experimentellen Tumortherapie. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2011. [cited 2021 Apr 16]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2012/3253/.

Council of Science Editors:

Wrede C. Transfer von antitumoralen Effektoren mittels viraler Vektoren zur experimentellen Tumortherapie. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2011. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2012/3253/

14. Valencia Garcia, Sara. Décryptage du réseau neuronal responsable de l’atonie musculaire pendant le sommeil paradoxal chez le rat : création d’un modèle rongeur du RBD (REM sleep Behavior Disorder) : Neuronal network of paradoxical sleep muscle atonia : a pre-requirement in the creation of a RBD (REM sleep Behavior Disorder) rodent model.

Degree: Docteur es, Neurosciences, 2014, Université Claude Bernard – Lyon I

Les circuits neuronaux responsables du sommeil paradoxal (SP) et de l'atonie musculaire qui le caractéristique sont l'objet de nombreuses recherches expérimentales, notamment en raison de… (more)

Subjects/Keywords: Tronc cérébral; C-Fos; Glutamate; Glycine; AAV-shRNA; Motoneurones somatiques; Brainstem; C-Fos; Glutamate; Glycine; AAV-shRNA; Somatic motoneurons; 612.8

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APA (6th Edition):

Valencia Garcia, S. (2014). Décryptage du réseau neuronal responsable de l’atonie musculaire pendant le sommeil paradoxal chez le rat : création d’un modèle rongeur du RBD (REM sleep Behavior Disorder) : Neuronal network of paradoxical sleep muscle atonia : a pre-requirement in the creation of a RBD (REM sleep Behavior Disorder) rodent model. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10324

Chicago Manual of Style (16th Edition):

Valencia Garcia, Sara. “Décryptage du réseau neuronal responsable de l’atonie musculaire pendant le sommeil paradoxal chez le rat : création d’un modèle rongeur du RBD (REM sleep Behavior Disorder) : Neuronal network of paradoxical sleep muscle atonia : a pre-requirement in the creation of a RBD (REM sleep Behavior Disorder) rodent model.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed April 16, 2021. http://www.theses.fr/2014LYO10324.

MLA Handbook (7th Edition):

Valencia Garcia, Sara. “Décryptage du réseau neuronal responsable de l’atonie musculaire pendant le sommeil paradoxal chez le rat : création d’un modèle rongeur du RBD (REM sleep Behavior Disorder) : Neuronal network of paradoxical sleep muscle atonia : a pre-requirement in the creation of a RBD (REM sleep Behavior Disorder) rodent model.” 2014. Web. 16 Apr 2021.

Vancouver:

Valencia Garcia S. Décryptage du réseau neuronal responsable de l’atonie musculaire pendant le sommeil paradoxal chez le rat : création d’un modèle rongeur du RBD (REM sleep Behavior Disorder) : Neuronal network of paradoxical sleep muscle atonia : a pre-requirement in the creation of a RBD (REM sleep Behavior Disorder) rodent model. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2014LYO10324.

Council of Science Editors:

Valencia Garcia S. Décryptage du réseau neuronal responsable de l’atonie musculaire pendant le sommeil paradoxal chez le rat : création d’un modèle rongeur du RBD (REM sleep Behavior Disorder) : Neuronal network of paradoxical sleep muscle atonia : a pre-requirement in the creation of a RBD (REM sleep Behavior Disorder) rodent model. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10324

15. Helary, Louise. Validation in vivo de l'implication de nouveaux gènes impliqués dans le développement musculaire des mammifères : In vivo validation of the implication of new genes in mammalian muscle development.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2019, Limoges

Même si les acteurs majeurs du développement musculaire ont été identifiés et les voies de transductions décrites, d’autres régulateurs restent encore à découvrir. Un crible… (more)

Subjects/Keywords: Développement musculaire; Knock-out; ShRNA; CRISPR/Cas9; DNAJC2; Muscle development; Knock-out; ShRNA; CRISPR/Cas9; DNAJC2; 599

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APA (6th Edition):

Helary, L. (2019). Validation in vivo de l'implication de nouveaux gènes impliqués dans le développement musculaire des mammifères : In vivo validation of the implication of new genes in mammalian muscle development. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2019LIMO0053

Chicago Manual of Style (16th Edition):

Helary, Louise. “Validation in vivo de l'implication de nouveaux gènes impliqués dans le développement musculaire des mammifères : In vivo validation of the implication of new genes in mammalian muscle development.” 2019. Doctoral Dissertation, Limoges. Accessed April 16, 2021. http://www.theses.fr/2019LIMO0053.

MLA Handbook (7th Edition):

Helary, Louise. “Validation in vivo de l'implication de nouveaux gènes impliqués dans le développement musculaire des mammifères : In vivo validation of the implication of new genes in mammalian muscle development.” 2019. Web. 16 Apr 2021.

Vancouver:

Helary L. Validation in vivo de l'implication de nouveaux gènes impliqués dans le développement musculaire des mammifères : In vivo validation of the implication of new genes in mammalian muscle development. [Internet] [Doctoral dissertation]. Limoges; 2019. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2019LIMO0053.

Council of Science Editors:

Helary L. Validation in vivo de l'implication de nouveaux gènes impliqués dans le développement musculaire des mammifères : In vivo validation of the implication of new genes in mammalian muscle development. [Doctoral Dissertation]. Limoges; 2019. Available from: http://www.theses.fr/2019LIMO0053


Loyola University Chicago

16. Case, Alicia Marie. Effects of Neuronal Nogo-A on Properties of Excitatory Synapses of the Sensorimotor Cortex.

Degree: PhD, Neuroscience, 2011, Loyola University Chicago

  Recovery after central nervous system (CNS) injury has long been a challenge for clinical investigators. Blockade of the oligodendrocyte-associated inhibitor Nogo-A has shown great… (more)

Subjects/Keywords: AAV; dendritic spine; Development; Nogo-A; shRNA; synapse; Neuroscience and Neurobiology

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APA (6th Edition):

Case, A. M. (2011). Effects of Neuronal Nogo-A on Properties of Excitatory Synapses of the Sensorimotor Cortex. (Doctoral Dissertation). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_diss/192

Chicago Manual of Style (16th Edition):

Case, Alicia Marie. “Effects of Neuronal Nogo-A on Properties of Excitatory Synapses of the Sensorimotor Cortex.” 2011. Doctoral Dissertation, Loyola University Chicago. Accessed April 16, 2021. https://ecommons.luc.edu/luc_diss/192.

MLA Handbook (7th Edition):

Case, Alicia Marie. “Effects of Neuronal Nogo-A on Properties of Excitatory Synapses of the Sensorimotor Cortex.” 2011. Web. 16 Apr 2021.

Vancouver:

Case AM. Effects of Neuronal Nogo-A on Properties of Excitatory Synapses of the Sensorimotor Cortex. [Internet] [Doctoral dissertation]. Loyola University Chicago; 2011. [cited 2021 Apr 16]. Available from: https://ecommons.luc.edu/luc_diss/192.

Council of Science Editors:

Case AM. Effects of Neuronal Nogo-A on Properties of Excitatory Synapses of the Sensorimotor Cortex. [Doctoral Dissertation]. Loyola University Chicago; 2011. Available from: https://ecommons.luc.edu/luc_diss/192


Penn State University

17. Kazi, Abid A. ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES .

Degree: 2011, Penn State University

 PRAS40 and DEPTOR are mTOR binding proteins that affect cell metabolism. Under catabolic conditions such as sepsis and glucocorticoid excess, there is an increase in… (more)

Subjects/Keywords: protein synthesis; mTOR; knockdown; lentivirus; shRNA; sepsis; disuse atrophy

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APA (6th Edition):

Kazi, A. A. (2011). ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11847

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kazi, Abid A. “ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES .” 2011. Thesis, Penn State University. Accessed April 16, 2021. https://submit-etda.libraries.psu.edu/catalog/11847.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kazi, Abid A. “ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES .” 2011. Web. 16 Apr 2021.

Vancouver:

Kazi AA. ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Apr 16]. Available from: https://submit-etda.libraries.psu.edu/catalog/11847.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kazi AA. ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11847

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. M. Rava'. FUNCTIONAL DISSECTION OF ST18 IN LIVER CANCER.

Degree: 2015, Università degli Studi di Milano

 The molecular mechanisms and pathways responsible for the progression of hepatocellular carcinoma (HCC) remain to be fully characterized. Among the genetic lesions associated with HCC… (more)

Subjects/Keywords: ST18; HCC; shRNA; inflammation; EMT; RNAseq; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Rava', M. (2015). FUNCTIONAL DISSECTION OF ST18 IN LIVER CANCER. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/254403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rava', M.. “FUNCTIONAL DISSECTION OF ST18 IN LIVER CANCER.” 2015. Thesis, Università degli Studi di Milano. Accessed April 16, 2021. http://hdl.handle.net/2434/254403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rava', M.. “FUNCTIONAL DISSECTION OF ST18 IN LIVER CANCER.” 2015. Web. 16 Apr 2021.

Vancouver:

Rava' M. FUNCTIONAL DISSECTION OF ST18 IN LIVER CANCER. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2434/254403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rava' M. FUNCTIONAL DISSECTION OF ST18 IN LIVER CANCER. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/254403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. U.A. Cammarata. CLONAL TRACKING AND HIGH THROUGHPUT SHRNA SCREENING IN AMLS.

Degree: 2016, Università degli Studi di Milano

 Acute myeloid leukemia (AML) is a malignant tumor characterized by the uncontrolled proliferation of immature hematopoietic cells (blasts) that colonize the bone marrow leading to… (more)

Subjects/Keywords: Clonal tracking, shRNA screening, AML, Leukemia; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Cammarata, U. (2016). CLONAL TRACKING AND HIGH THROUGHPUT SHRNA SCREENING IN AMLS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/365771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cammarata, U.A.. “CLONAL TRACKING AND HIGH THROUGHPUT SHRNA SCREENING IN AMLS.” 2016. Thesis, Università degli Studi di Milano. Accessed April 16, 2021. http://hdl.handle.net/2434/365771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cammarata, U.A.. “CLONAL TRACKING AND HIGH THROUGHPUT SHRNA SCREENING IN AMLS.” 2016. Web. 16 Apr 2021.

Vancouver:

Cammarata U. CLONAL TRACKING AND HIGH THROUGHPUT SHRNA SCREENING IN AMLS. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/2434/365771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cammarata U. CLONAL TRACKING AND HIGH THROUGHPUT SHRNA SCREENING IN AMLS. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/365771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

20. Bertero, Alessandro. Activin/Nodal Signalling Controls the Epigenome and Epitranscriptome of Human Pluripotent Stem Cells.

Degree: PhD, 2016, University of Cambridge

 Human pluripotent stem cells (hPSCs) are an invaluable model for cellular and developmental biology, and hold great potential for translational applications. While great progress has… (more)

Subjects/Keywords: stem cells; smad; DPY30; TGF beta; m6A; epigenetics; epitranscriptome; shRNA; knockdown

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APA (6th Edition):

Bertero, A. (2016). Activin/Nodal Signalling Controls the Epigenome and Epitranscriptome of Human Pluripotent Stem Cells. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274120

Chicago Manual of Style (16th Edition):

Bertero, Alessandro. “Activin/Nodal Signalling Controls the Epigenome and Epitranscriptome of Human Pluripotent Stem Cells.” 2016. Doctoral Dissertation, University of Cambridge. Accessed April 16, 2021. https://www.repository.cam.ac.uk/handle/1810/274120.

MLA Handbook (7th Edition):

Bertero, Alessandro. “Activin/Nodal Signalling Controls the Epigenome and Epitranscriptome of Human Pluripotent Stem Cells.” 2016. Web. 16 Apr 2021.

Vancouver:

Bertero A. Activin/Nodal Signalling Controls the Epigenome and Epitranscriptome of Human Pluripotent Stem Cells. [Internet] [Doctoral dissertation]. University of Cambridge; 2016. [cited 2021 Apr 16]. Available from: https://www.repository.cam.ac.uk/handle/1810/274120.

Council of Science Editors:

Bertero A. Activin/Nodal Signalling Controls the Epigenome and Epitranscriptome of Human Pluripotent Stem Cells. [Doctoral Dissertation]. University of Cambridge; 2016. Available from: https://www.repository.cam.ac.uk/handle/1810/274120


University of California – San Francisco

21. Blau, James Alan. Pooled screening to reveal the primary effectors of miR-142.

Degree: Pharmaceutical Sciences and Pharmacogenomics, 2015, University of California – San Francisco

 Although microRNAs are key regulators of gene expression, few studies have thoroughly evaluated the upstream regulators of microRNA activity. We seek to understand the upstream… (more)

Subjects/Keywords: Molecular biology; CRISPR-Cas9; miR-142; pooled screen; RNAi; shRNA

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APA (6th Edition):

Blau, J. A. (2015). Pooled screening to reveal the primary effectors of miR-142. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/59b4f61r

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Blau, James Alan. “Pooled screening to reveal the primary effectors of miR-142.” 2015. Thesis, University of California – San Francisco. Accessed April 16, 2021. http://www.escholarship.org/uc/item/59b4f61r.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Blau, James Alan. “Pooled screening to reveal the primary effectors of miR-142.” 2015. Web. 16 Apr 2021.

Vancouver:

Blau JA. Pooled screening to reveal the primary effectors of miR-142. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2021 Apr 16]. Available from: http://www.escholarship.org/uc/item/59b4f61r.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Blau JA. Pooled screening to reveal the primary effectors of miR-142. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/59b4f61r

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

22. Pierce, Thomas Patrick. Use of RNAi in the mouse: inducible and reversible inhibition of Stat3.

Degree: 2012, University of Melbourne

 The signal transducer and activator of transcription 3 (Stat3) is a latent transcription factor that is activated during tissue inflammation and in a wide range… (more)

Subjects/Keywords: RNAi; RNA interference; shRNA; Stat3; miR-21; mouse models; gene knockdown

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APA (6th Edition):

Pierce, T. P. (2012). Use of RNAi in the mouse: inducible and reversible inhibition of Stat3. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37193

Chicago Manual of Style (16th Edition):

Pierce, Thomas Patrick. “Use of RNAi in the mouse: inducible and reversible inhibition of Stat3.” 2012. Doctoral Dissertation, University of Melbourne. Accessed April 16, 2021. http://hdl.handle.net/11343/37193.

MLA Handbook (7th Edition):

Pierce, Thomas Patrick. “Use of RNAi in the mouse: inducible and reversible inhibition of Stat3.” 2012. Web. 16 Apr 2021.

Vancouver:

Pierce TP. Use of RNAi in the mouse: inducible and reversible inhibition of Stat3. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/11343/37193.

Council of Science Editors:

Pierce TP. Use of RNAi in the mouse: inducible and reversible inhibition of Stat3. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37193


University of Edinburgh

23. Brightwell, Sara. Identifying novel regulators of reprogramming using RNA interference.

Degree: PhD, 2015, University of Edinburgh

 Since Yamanaka and Takahashi first described the isolation of induced pluripotent stem cells (iPSCs) in 2006, researchers have invested a vast amount of time and… (more)

Subjects/Keywords: 572.8; reprogramming; iPSC; induced pluripotent stem cells; shRNA

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APA (6th Edition):

Brightwell, S. (2015). Identifying novel regulators of reprogramming using RNA interference. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/16156

Chicago Manual of Style (16th Edition):

Brightwell, Sara. “Identifying novel regulators of reprogramming using RNA interference.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed April 16, 2021. http://hdl.handle.net/1842/16156.

MLA Handbook (7th Edition):

Brightwell, Sara. “Identifying novel regulators of reprogramming using RNA interference.” 2015. Web. 16 Apr 2021.

Vancouver:

Brightwell S. Identifying novel regulators of reprogramming using RNA interference. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1842/16156.

Council of Science Editors:

Brightwell S. Identifying novel regulators of reprogramming using RNA interference. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/16156


University of New South Wales

24. Ramadas, Radhika. Identification of modulators of chemotherapeutic resistance using a random shRNA library.

Degree: Biotechnology & Biomolecular Sciences, 2012, University of New South Wales

 High-throughput gene silencing using RNA interference (RNAi) libraries is a rapidly expanding strategy to study the molecular pathways underlying human diseases. A majority of RNAi… (more)

Subjects/Keywords: Chemotherapeutic drug resistance; RNA interference; Random shRNA-encoding library

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APA (6th Edition):

Ramadas, R. (2012). Identification of modulators of chemotherapeutic resistance using a random shRNA library. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52276 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10948/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Ramadas, Radhika. “Identification of modulators of chemotherapeutic resistance using a random shRNA library.” 2012. Doctoral Dissertation, University of New South Wales. Accessed April 16, 2021. http://handle.unsw.edu.au/1959.4/52276 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10948/SOURCE01?view=true.

MLA Handbook (7th Edition):

Ramadas, Radhika. “Identification of modulators of chemotherapeutic resistance using a random shRNA library.” 2012. Web. 16 Apr 2021.

Vancouver:

Ramadas R. Identification of modulators of chemotherapeutic resistance using a random shRNA library. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2021 Apr 16]. Available from: http://handle.unsw.edu.au/1959.4/52276 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10948/SOURCE01?view=true.

Council of Science Editors:

Ramadas R. Identification of modulators of chemotherapeutic resistance using a random shRNA library. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52276 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10948/SOURCE01?view=true

25. Lahmar, Qods. Analyse du potentiel des macrophages double-déficients en MafB et c-Maf en tant qu'agent de thérapie cellulaire : Analyse of the potential of MafB/c-Maf double deficient macrophages as cellular therapeutic agent.

Degree: Docteur es, Immunologie, 2013, Aix Marseille Université

Chez les métazoaires, les cellules spécialisées se caractérisent par la sortie du cycle cellulaire alors que les cellules souches et progénitrices se caractérisent par un… (more)

Subjects/Keywords: Macrophages; Cancer; Maf; Facteurs de transcription; Applications therapeutiques; ShRNA; Lentivirus; Talen; Macrophages; Cancer; Maf; Transcription factors; Therapeutic applications; ShRNA; Lentivirus; Talen; 571

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APA (6th Edition):

Lahmar, Q. (2013). Analyse du potentiel des macrophages double-déficients en MafB et c-Maf en tant qu'agent de thérapie cellulaire : Analyse of the potential of MafB/c-Maf double deficient macrophages as cellular therapeutic agent. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2013AIXM4029

Chicago Manual of Style (16th Edition):

Lahmar, Qods. “Analyse du potentiel des macrophages double-déficients en MafB et c-Maf en tant qu'agent de thérapie cellulaire : Analyse of the potential of MafB/c-Maf double deficient macrophages as cellular therapeutic agent.” 2013. Doctoral Dissertation, Aix Marseille Université. Accessed April 16, 2021. http://www.theses.fr/2013AIXM4029.

MLA Handbook (7th Edition):

Lahmar, Qods. “Analyse du potentiel des macrophages double-déficients en MafB et c-Maf en tant qu'agent de thérapie cellulaire : Analyse of the potential of MafB/c-Maf double deficient macrophages as cellular therapeutic agent.” 2013. Web. 16 Apr 2021.

Vancouver:

Lahmar Q. Analyse du potentiel des macrophages double-déficients en MafB et c-Maf en tant qu'agent de thérapie cellulaire : Analyse of the potential of MafB/c-Maf double deficient macrophages as cellular therapeutic agent. [Internet] [Doctoral dissertation]. Aix Marseille Université 2013. [cited 2021 Apr 16]. Available from: http://www.theses.fr/2013AIXM4029.

Council of Science Editors:

Lahmar Q. Analyse du potentiel des macrophages double-déficients en MafB et c-Maf en tant qu'agent de thérapie cellulaire : Analyse of the potential of MafB/c-Maf double deficient macrophages as cellular therapeutic agent. [Doctoral Dissertation]. Aix Marseille Université 2013. Available from: http://www.theses.fr/2013AIXM4029

26. Luís Bruno da Cruz e Alves de Moraes. Desenvolvimento de xenotransplantes de tumores pancreáticos humanos para varredura genética de alvos moleculares com potencial terapêutico.

Degree: 2018, University of São Paulo

 O adenocarcinoma ductal pancreático (PDAC, pancreatic ductal adenocarcinoma), o tipo mais prevalente de câncer do pâncreas, é uma neoplasia extremamente agressiva e com elevado índice… (more)

Subjects/Keywords: Câncer de pâncreas; Epigenética; Linhagem celular tumoral; RNA de interferência; shRNA; Xenoenxerto; Cancer cell line; Epigenetics; Pancreatic cancer; RNA interference; shRNA; Xenograft

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moraes, L. B. d. C. e. A. d. (2018). Desenvolvimento de xenotransplantes de tumores pancreáticos humanos para varredura genética de alvos moleculares com potencial terapêutico. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/46/46131/tde-07052019-110949/

Chicago Manual of Style (16th Edition):

Moraes, Luís Bruno da Cruz e Alves de. “Desenvolvimento de xenotransplantes de tumores pancreáticos humanos para varredura genética de alvos moleculares com potencial terapêutico.” 2018. Doctoral Dissertation, University of São Paulo. Accessed April 16, 2021. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-07052019-110949/.

MLA Handbook (7th Edition):

Moraes, Luís Bruno da Cruz e Alves de. “Desenvolvimento de xenotransplantes de tumores pancreáticos humanos para varredura genética de alvos moleculares com potencial terapêutico.” 2018. Web. 16 Apr 2021.

Vancouver:

Moraes LBdCeAd. Desenvolvimento de xenotransplantes de tumores pancreáticos humanos para varredura genética de alvos moleculares com potencial terapêutico. [Internet] [Doctoral dissertation]. University of São Paulo; 2018. [cited 2021 Apr 16]. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-07052019-110949/.

Council of Science Editors:

Moraes LBdCeAd. Desenvolvimento de xenotransplantes de tumores pancreáticos humanos para varredura genética de alvos moleculares com potencial terapêutico. [Doctoral Dissertation]. University of São Paulo; 2018. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-07052019-110949/

27. Quick-Cleveland, Jen. Primary microRNA processing in humans: biology, biotechnology and disease.

Degree: Biological Chemistry, 2017, UCLA

 MicroRNAs (miRNAs) are a class of non-coding RNAs that tune gene expression by negatively regulating at least 60% of protein-coding genes. In animals, they are… (more)

Subjects/Keywords: Molecular biology; Biochemistry; DGCR8; Fe(III) heme; microRNA; miRNA and cancer; shRNA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Quick-Cleveland, J. (2017). Primary microRNA processing in humans: biology, biotechnology and disease. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/4sh028f3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quick-Cleveland, Jen. “Primary microRNA processing in humans: biology, biotechnology and disease.” 2017. Thesis, UCLA. Accessed April 16, 2021. http://www.escholarship.org/uc/item/4sh028f3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quick-Cleveland, Jen. “Primary microRNA processing in humans: biology, biotechnology and disease.” 2017. Web. 16 Apr 2021.

Vancouver:

Quick-Cleveland J. Primary microRNA processing in humans: biology, biotechnology and disease. [Internet] [Thesis]. UCLA; 2017. [cited 2021 Apr 16]. Available from: http://www.escholarship.org/uc/item/4sh028f3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quick-Cleveland J. Primary microRNA processing in humans: biology, biotechnology and disease. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/4sh028f3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade de Lisboa

28. Brito, Francisco Maria de Aboim Borges Fialho de. Transcriptome profiling of spinal muscular atrophy models using RNA-Seq.

Degree: 2014, Universidade de Lisboa

Tese de mestrado, Bioinformática e Biologia Computacional, Universidade de Lisboa, Faculdade de Ciências, 2014

Spinal Muscular Atrophy (SMA) is a neurodegenerative disorder that represents the… (more)

Subjects/Keywords: RNA-Seq; Spinal muscular antrophy; shRNA; D. melanogaster; H. sapiens; Teses de mestrado - 2014

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brito, F. M. d. A. B. F. d. (2014). Transcriptome profiling of spinal muscular atrophy models using RNA-Seq. (Thesis). Universidade de Lisboa. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brito, Francisco Maria de Aboim Borges Fialho de. “Transcriptome profiling of spinal muscular atrophy models using RNA-Seq.” 2014. Thesis, Universidade de Lisboa. Accessed April 16, 2021. https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brito, Francisco Maria de Aboim Borges Fialho de. “Transcriptome profiling of spinal muscular atrophy models using RNA-Seq.” 2014. Web. 16 Apr 2021.

Vancouver:

Brito FMdABFd. Transcriptome profiling of spinal muscular atrophy models using RNA-Seq. [Internet] [Thesis]. Universidade de Lisboa; 2014. [cited 2021 Apr 16]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15998.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brito FMdABFd. Transcriptome profiling of spinal muscular atrophy models using RNA-Seq. [Thesis]. Universidade de Lisboa; 2014. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/15998

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

29. Ma, Xiangyuan. Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation.

Degree: 2014, University of Toronto

N-glycosylation is a co-translational modification that covalently attaches oligosaccharide to and regulates proper folding, trafficking, and surface residency of secreted proteins. MGAT5 encodes a glycosyltransferase… (more)

Subjects/Keywords: Cell Signaling; Metabolism; Mgat5; N-glycosylation; Pooled Lentiviral shRNA Drop-out Screen; 0307

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ma, X. (2014). Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67885

Chicago Manual of Style (16th Edition):

Ma, Xiangyuan. “Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation.” 2014. Masters Thesis, University of Toronto. Accessed April 16, 2021. http://hdl.handle.net/1807/67885.

MLA Handbook (7th Edition):

Ma, Xiangyuan. “Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation.” 2014. Web. 16 Apr 2021.

Vancouver:

Ma X. Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1807/67885.

Council of Science Editors:

Ma X. Identification of Genes and Pathways Linking Cancer Metabolism to Cell Surface Dynamics through Protein N-glycosylation. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67885


University of Toronto

30. Ramakrishnan, Ashwin. Targeting the Mitochondrial Processing Peptidase as a Therapeutic Strategy for Acute Myeloid Leukemia.

Degree: 2014, University of Toronto

Acute myeloid leukemia (AML) is a highly heterogeneous disease characterized by a wide variety of cytogenetic abnormalities. Treatment has been largely unchanged for the past… (more)

Subjects/Keywords: Acute Myeloid Leukemia; Mitochondrial Processing Peptidase; Mitochondrial protein import; NanoString; shRNA; 0992

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ramakrishnan, A. (2014). Targeting the Mitochondrial Processing Peptidase as a Therapeutic Strategy for Acute Myeloid Leukemia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68527

Chicago Manual of Style (16th Edition):

Ramakrishnan, Ashwin. “Targeting the Mitochondrial Processing Peptidase as a Therapeutic Strategy for Acute Myeloid Leukemia.” 2014. Masters Thesis, University of Toronto. Accessed April 16, 2021. http://hdl.handle.net/1807/68527.

MLA Handbook (7th Edition):

Ramakrishnan, Ashwin. “Targeting the Mitochondrial Processing Peptidase as a Therapeutic Strategy for Acute Myeloid Leukemia.” 2014. Web. 16 Apr 2021.

Vancouver:

Ramakrishnan A. Targeting the Mitochondrial Processing Peptidase as a Therapeutic Strategy for Acute Myeloid Leukemia. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 Apr 16]. Available from: http://hdl.handle.net/1807/68527.

Council of Science Editors:

Ramakrishnan A. Targeting the Mitochondrial Processing Peptidase as a Therapeutic Strategy for Acute Myeloid Leukemia. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68527

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