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You searched for subject:(senescence). Showing records 1 – 30 of 486 total matches.

[1] [2] [3] [4] [5] … [17]

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1. Cooper, Leroy Leon. Age-Associated Arrhythmogenic Substrate and Trigger in a Rabbit Model of Cardiac Aging.

Degree: PhD, Molecular Pharmacology, Physiology, and Biotechnology, 2013, Brown University

 Aging is associated with an increased risk of arrhythmias and sudden cardiac death (SCD). Yet, much remains unknown about the mechanisms that underlie the age-related,… (more)

Subjects/Keywords: senescence

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cooper, L. L. (2013). Age-Associated Arrhythmogenic Substrate and Trigger in a Rabbit Model of Cardiac Aging. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:320600/

Chicago Manual of Style (16th Edition):

Cooper, Leroy Leon. “Age-Associated Arrhythmogenic Substrate and Trigger in a Rabbit Model of Cardiac Aging.” 2013. Doctoral Dissertation, Brown University. Accessed June 18, 2019. https://repository.library.brown.edu/studio/item/bdr:320600/.

MLA Handbook (7th Edition):

Cooper, Leroy Leon. “Age-Associated Arrhythmogenic Substrate and Trigger in a Rabbit Model of Cardiac Aging.” 2013. Web. 18 Jun 2019.

Vancouver:

Cooper LL. Age-Associated Arrhythmogenic Substrate and Trigger in a Rabbit Model of Cardiac Aging. [Internet] [Doctoral dissertation]. Brown University; 2013. [cited 2019 Jun 18]. Available from: https://repository.library.brown.edu/studio/item/bdr:320600/.

Council of Science Editors:

Cooper LL. Age-Associated Arrhythmogenic Substrate and Trigger in a Rabbit Model of Cardiac Aging. [Doctoral Dissertation]. Brown University; 2013. Available from: https://repository.library.brown.edu/studio/item/bdr:320600/

2. Peckham, Edward Joseph. Changes in Chromatin Conformation During Replicative Senescence of Normal Human Diploid Fibroblasts.

Degree: PhD, Molecular Biology, Cell Biology, and Biochemistry, 2012, Brown University

 In this thesis we provide evidence that the FAIRE method can reveal interesting and relevant changes in genome-wide chromatin organization as cells undergo replicative senescence,… (more)

Subjects/Keywords: senescence

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APA (6th Edition):

Peckham, E. J. (2012). Changes in Chromatin Conformation During Replicative Senescence of Normal Human Diploid Fibroblasts. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:297552/

Chicago Manual of Style (16th Edition):

Peckham, Edward Joseph. “Changes in Chromatin Conformation During Replicative Senescence of Normal Human Diploid Fibroblasts.” 2012. Doctoral Dissertation, Brown University. Accessed June 18, 2019. https://repository.library.brown.edu/studio/item/bdr:297552/.

MLA Handbook (7th Edition):

Peckham, Edward Joseph. “Changes in Chromatin Conformation During Replicative Senescence of Normal Human Diploid Fibroblasts.” 2012. Web. 18 Jun 2019.

Vancouver:

Peckham EJ. Changes in Chromatin Conformation During Replicative Senescence of Normal Human Diploid Fibroblasts. [Internet] [Doctoral dissertation]. Brown University; 2012. [cited 2019 Jun 18]. Available from: https://repository.library.brown.edu/studio/item/bdr:297552/.

Council of Science Editors:

Peckham EJ. Changes in Chromatin Conformation During Replicative Senescence of Normal Human Diploid Fibroblasts. [Doctoral Dissertation]. Brown University; 2012. Available from: https://repository.library.brown.edu/studio/item/bdr:297552/

3. KHAIDIZAR, FIQRI DIZAR. The effect of Nampt overexpression on cellular senescence : 細胞老化に対するNampt高発現の効果; サイボウ ロウカ ニ タイスル Nampt コウハツゲン ノ コウカ.

Degree: 博士(バイオサイエンス), 2017, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: Senescence

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APA (6th Edition):

KHAIDIZAR, F. D. (2017). The effect of Nampt overexpression on cellular senescence : 細胞老化に対するNampt高発現の効果; サイボウ ロウカ ニ タイスル Nampt コウハツゲン ノ コウカ. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/12191

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KHAIDIZAR, FIQRI DIZAR. “The effect of Nampt overexpression on cellular senescence : 細胞老化に対するNampt高発現の効果; サイボウ ロウカ ニ タイスル Nampt コウハツゲン ノ コウカ.” 2017. Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed June 18, 2019. http://hdl.handle.net/10061/12191.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KHAIDIZAR, FIQRI DIZAR. “The effect of Nampt overexpression on cellular senescence : 細胞老化に対するNampt高発現の効果; サイボウ ロウカ ニ タイスル Nampt コウハツゲン ノ コウカ.” 2017. Web. 18 Jun 2019.

Vancouver:

KHAIDIZAR FD. The effect of Nampt overexpression on cellular senescence : 細胞老化に対するNampt高発現の効果; サイボウ ロウカ ニ タイスル Nampt コウハツゲン ノ コウカ. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; 2017. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10061/12191.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KHAIDIZAR FD. The effect of Nampt overexpression on cellular senescence : 細胞老化に対するNampt高発現の効果; サイボウ ロウカ ニ タイスル Nampt コウハツゲン ノ コウカ. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; 2017. Available from: http://hdl.handle.net/10061/12191

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

4. Kumar, Ritesh. Premature Senescence In Primary Glioblastoma Cells .

Degree: 2018, University of Ottawa

 Glioblastoma is the most common and fatal adult primary brain tumour. Despite maximum therapy, median survival time is 14 months after diagnosis. Senescence is a… (more)

Subjects/Keywords: Glioblastoma; Senescence

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APA (6th Edition):

Kumar, R. (2018). Premature Senescence In Primary Glioblastoma Cells . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/37934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kumar, Ritesh. “Premature Senescence In Primary Glioblastoma Cells .” 2018. Thesis, University of Ottawa. Accessed June 18, 2019. http://hdl.handle.net/10393/37934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kumar, Ritesh. “Premature Senescence In Primary Glioblastoma Cells .” 2018. Web. 18 Jun 2019.

Vancouver:

Kumar R. Premature Senescence In Primary Glioblastoma Cells . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10393/37934.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kumar R. Premature Senescence In Primary Glioblastoma Cells . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/37934

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Besnard, Emilie. Modifications de l'organisation de la chromatine liées à l’entrée en sénescence et son impact sur la réplication du génome : Impact of chromatin structure modification in senescence on the DNA replication program.

Degree: Docteur es, Biologie cellulaire, 2010, Université Montpellier I

L'entrée en sénescence, considérée comme un arrêt irréversible du cycle, se caractérise par une modification de l'organisation de la chromatine formant de véritables foyers d'… (more)

Subjects/Keywords: Senescence; Réplication; Chromatine; Senescence; Replication; Chromatin

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APA (6th Edition):

Besnard, E. (2010). Modifications de l'organisation de la chromatine liées à l’entrée en sénescence et son impact sur la réplication du génome : Impact of chromatin structure modification in senescence on the DNA replication program. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2010MON1T008

Chicago Manual of Style (16th Edition):

Besnard, Emilie. “Modifications de l'organisation de la chromatine liées à l’entrée en sénescence et son impact sur la réplication du génome : Impact of chromatin structure modification in senescence on the DNA replication program.” 2010. Doctoral Dissertation, Université Montpellier I. Accessed June 18, 2019. http://www.theses.fr/2010MON1T008.

MLA Handbook (7th Edition):

Besnard, Emilie. “Modifications de l'organisation de la chromatine liées à l’entrée en sénescence et son impact sur la réplication du génome : Impact of chromatin structure modification in senescence on the DNA replication program.” 2010. Web. 18 Jun 2019.

Vancouver:

Besnard E. Modifications de l'organisation de la chromatine liées à l’entrée en sénescence et son impact sur la réplication du génome : Impact of chromatin structure modification in senescence on the DNA replication program. [Internet] [Doctoral dissertation]. Université Montpellier I; 2010. [cited 2019 Jun 18]. Available from: http://www.theses.fr/2010MON1T008.

Council of Science Editors:

Besnard E. Modifications de l'organisation de la chromatine liées à l’entrée en sénescence et son impact sur la réplication du génome : Impact of chromatin structure modification in senescence on the DNA replication program. [Doctoral Dissertation]. Université Montpellier I; 2010. Available from: http://www.theses.fr/2010MON1T008


University of Guelph

6. Mahmood, Kashif. Arabidopsis NAC Transcription Factors: Roles in Natural and Stress-Induced Senescence .

Degree: 2015, University of Guelph

 Plant senescence is an important biological phenomenon that involves systematic degradation of plant cells and tissue structures. Whereas, this process is important for the efficient… (more)

Subjects/Keywords: Leaf senescence; stress-induced senescence; abiotic stresses

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APA (6th Edition):

Mahmood, K. (2015). Arabidopsis NAC Transcription Factors: Roles in Natural and Stress-Induced Senescence . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mahmood, Kashif. “Arabidopsis NAC Transcription Factors: Roles in Natural and Stress-Induced Senescence .” 2015. Thesis, University of Guelph. Accessed June 18, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mahmood, Kashif. “Arabidopsis NAC Transcription Factors: Roles in Natural and Stress-Induced Senescence .” 2015. Web. 18 Jun 2019.

Vancouver:

Mahmood K. Arabidopsis NAC Transcription Factors: Roles in Natural and Stress-Induced Senescence . [Internet] [Thesis]. University of Guelph; 2015. [cited 2019 Jun 18]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mahmood K. Arabidopsis NAC Transcription Factors: Roles in Natural and Stress-Induced Senescence . [Thesis]. University of Guelph; 2015. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

7. Zhang, Gao. Induction of Cellular Senescence As A Novel Therapeutic Strategy for Melanoma Treatment.

Degree: 2012, University of Pennsylvania

 Oncogene-induced senescence (OIS) is a well-documented phenomenon both in vitro and in vivo as a tumor-suppression mechanism. It functions as a barrier to tumor initiation… (more)

Subjects/Keywords: Melanoma; Senescence; Therapy-induced senescence; Biomedical

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APA (6th Edition):

Zhang, G. (2012). Induction of Cellular Senescence As A Novel Therapeutic Strategy for Melanoma Treatment. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Gao. “Induction of Cellular Senescence As A Novel Therapeutic Strategy for Melanoma Treatment.” 2012. Thesis, University of Pennsylvania. Accessed June 18, 2019. https://repository.upenn.edu/edissertations/599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Gao. “Induction of Cellular Senescence As A Novel Therapeutic Strategy for Melanoma Treatment.” 2012. Web. 18 Jun 2019.

Vancouver:

Zhang G. Induction of Cellular Senescence As A Novel Therapeutic Strategy for Melanoma Treatment. [Internet] [Thesis]. University of Pennsylvania; 2012. [cited 2019 Jun 18]. Available from: https://repository.upenn.edu/edissertations/599.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang G. Induction of Cellular Senescence As A Novel Therapeutic Strategy for Melanoma Treatment. [Thesis]. University of Pennsylvania; 2012. Available from: https://repository.upenn.edu/edissertations/599

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Dalhousie University

8. Cyr, David P. KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE.

Degree: MS, Department of Microbiology & Immunology, 2013, Dalhousie University

 Autophagy (literally to ‘self-eat’) is an intracellular, catabolic mechanism to degrade and recycle cytoplasmic contents in response to metabolic, oxidative, and genotoxic stresses. Autophagy plays… (more)

Subjects/Keywords: KSHV; vGPCR; autophagy; senescence

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APA (6th Edition):

Cyr, D. P. (2013). KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/36316

Chicago Manual of Style (16th Edition):

Cyr, David P. “KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE.” 2013. Masters Thesis, Dalhousie University. Accessed June 18, 2019. http://hdl.handle.net/10222/36316.

MLA Handbook (7th Edition):

Cyr, David P. “KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE.” 2013. Web. 18 Jun 2019.

Vancouver:

Cyr DP. KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE. [Internet] [Masters thesis]. Dalhousie University; 2013. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10222/36316.

Council of Science Editors:

Cyr DP. KSHV vGPCR: A VIRAL ONCOPROTEIN THAT TRIGGERS AUTOPHAGY AND CELLULAR SENESCENCE. [Masters Thesis]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/36316


The Ohio State University

9. Moon, Youyoun. Characterization of <i>Petunia x hybrida</i> ‘Mitchell Diploid’ Metacaspases during Petal Senescence.

Degree: PhD, Horticulture and Crop Science, 2010, The Ohio State University

Senescence is the final stage of plant development. It is a genetically controlled process leading to organ or whole plant death. The death process at… (more)

Subjects/Keywords: Horticulture; metacaspases; petunia; senescence

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APA (6th Edition):

Moon, Y. (2010). Characterization of <i>Petunia x hybrida</i> ‘Mitchell Diploid’ Metacaspases during Petal Senescence. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1285165316

Chicago Manual of Style (16th Edition):

Moon, Youyoun. “Characterization of <i>Petunia x hybrida</i> ‘Mitchell Diploid’ Metacaspases during Petal Senescence.” 2010. Doctoral Dissertation, The Ohio State University. Accessed June 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1285165316.

MLA Handbook (7th Edition):

Moon, Youyoun. “Characterization of <i>Petunia x hybrida</i> ‘Mitchell Diploid’ Metacaspases during Petal Senescence.” 2010. Web. 18 Jun 2019.

Vancouver:

Moon Y. Characterization of <i>Petunia x hybrida</i> ‘Mitchell Diploid’ Metacaspases during Petal Senescence. [Internet] [Doctoral dissertation]. The Ohio State University; 2010. [cited 2019 Jun 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1285165316.

Council of Science Editors:

Moon Y. Characterization of <i>Petunia x hybrida</i> ‘Mitchell Diploid’ Metacaspases during Petal Senescence. [Doctoral Dissertation]. The Ohio State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1285165316


Queens University

10. Wesch, Neil. Modelling Telomere Length Dynamics Subject to the Action of Telomerase .

Degree: Physics, Engineering Physics and Astronomy, 2016, Queens University

 All eukaryotic organisms, single-celled or multi-celled, have linear chromosomes, and all linear chromosomes have telomeres at their ends. All eukaryotes have genes which code for… (more)

Subjects/Keywords: telomeres; chromosome biology; senescence; telomerase

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APA (6th Edition):

Wesch, N. (2016). Modelling Telomere Length Dynamics Subject to the Action of Telomerase . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/14347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wesch, Neil. “Modelling Telomere Length Dynamics Subject to the Action of Telomerase .” 2016. Thesis, Queens University. Accessed June 18, 2019. http://hdl.handle.net/1974/14347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wesch, Neil. “Modelling Telomere Length Dynamics Subject to the Action of Telomerase .” 2016. Web. 18 Jun 2019.

Vancouver:

Wesch N. Modelling Telomere Length Dynamics Subject to the Action of Telomerase . [Internet] [Thesis]. Queens University; 2016. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/1974/14347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wesch N. Modelling Telomere Length Dynamics Subject to the Action of Telomerase . [Thesis]. Queens University; 2016. Available from: http://hdl.handle.net/1974/14347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

11. White, William. Alterations in autophagy and senescence in the pathologically aged uraemic heart.

Degree: PhD, 2017, Queen Mary, University of London

 There is much observational evidence to suggest that patients with chronic kidney disease are biologically 'older' than their unaffected peers. This is most obviously seen… (more)

Subjects/Keywords: chronic kidney disease; autophagy; senescence

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APA (6th Edition):

White, W. (2017). Alterations in autophagy and senescence in the pathologically aged uraemic heart. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/30887 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766054

Chicago Manual of Style (16th Edition):

White, William. “Alterations in autophagy and senescence in the pathologically aged uraemic heart.” 2017. Doctoral Dissertation, Queen Mary, University of London. Accessed June 18, 2019. http://qmro.qmul.ac.uk/xmlui/handle/123456789/30887 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766054.

MLA Handbook (7th Edition):

White, William. “Alterations in autophagy and senescence in the pathologically aged uraemic heart.” 2017. Web. 18 Jun 2019.

Vancouver:

White W. Alterations in autophagy and senescence in the pathologically aged uraemic heart. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2017. [cited 2019 Jun 18]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/30887 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766054.

Council of Science Editors:

White W. Alterations in autophagy and senescence in the pathologically aged uraemic heart. [Doctoral Dissertation]. Queen Mary, University of London; 2017. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/30887 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.766054


Universitat de Valencia

12. Mas Bargues, Cristina. Role of p16ink4a and bmi-1 in oxidative stress-induced premature senescence in human dental pulp stem cells .

Degree: 2017, Universitat de Valencia

 Las células madre son aquellas células que se dividen asimétricamente para producir una copia de sí mismas y una segunda célula que seguirá su camino… (more)

Subjects/Keywords: senescence; stem cells; p16

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APA (6th Edition):

Mas Bargues, C. (2017). Role of p16ink4a and bmi-1 in oxidative stress-induced premature senescence in human dental pulp stem cells . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/59486

Chicago Manual of Style (16th Edition):

Mas Bargues, Cristina. “Role of p16ink4a and bmi-1 in oxidative stress-induced premature senescence in human dental pulp stem cells .” 2017. Doctoral Dissertation, Universitat de Valencia. Accessed June 18, 2019. http://hdl.handle.net/10550/59486.

MLA Handbook (7th Edition):

Mas Bargues, Cristina. “Role of p16ink4a and bmi-1 in oxidative stress-induced premature senescence in human dental pulp stem cells .” 2017. Web. 18 Jun 2019.

Vancouver:

Mas Bargues C. Role of p16ink4a and bmi-1 in oxidative stress-induced premature senescence in human dental pulp stem cells . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2017. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10550/59486.

Council of Science Editors:

Mas Bargues C. Role of p16ink4a and bmi-1 in oxidative stress-induced premature senescence in human dental pulp stem cells . [Doctoral Dissertation]. Universitat de Valencia; 2017. Available from: http://hdl.handle.net/10550/59486


University of Cambridge

13. Hoare, Matthew. T-lymphocyte senescence and hepatitis C virus infection.

Degree: PhD, 2010, University of Cambridge

 Hepatitis C virus (HCV) infection is a leading cause of cirrhosis and hepatocellular carcinoma. The degree of fibrosis progression and treatment-related outcomes are critically dependent… (more)

Subjects/Keywords: 610; Hepatitis; Telomere; Senescence; Interferon

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APA (6th Edition):

Hoare, M. (2010). T-lymphocyte senescence and hepatitis C virus infection. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/226746 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555200

Chicago Manual of Style (16th Edition):

Hoare, Matthew. “T-lymphocyte senescence and hepatitis C virus infection.” 2010. Doctoral Dissertation, University of Cambridge. Accessed June 18, 2019. https://www.repository.cam.ac.uk/handle/1810/226746 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555200.

MLA Handbook (7th Edition):

Hoare, Matthew. “T-lymphocyte senescence and hepatitis C virus infection.” 2010. Web. 18 Jun 2019.

Vancouver:

Hoare M. T-lymphocyte senescence and hepatitis C virus infection. [Internet] [Doctoral dissertation]. University of Cambridge; 2010. [cited 2019 Jun 18]. Available from: https://www.repository.cam.ac.uk/handle/1810/226746 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555200.

Council of Science Editors:

Hoare M. T-lymphocyte senescence and hepatitis C virus infection. [Doctoral Dissertation]. University of Cambridge; 2010. Available from: https://www.repository.cam.ac.uk/handle/1810/226746 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555200

14. Marutani, Akiko; Nakamura, Mitsutoshi; Nishimura, Fumihiko; Nakazawa, Tsutomu; Matsuda, Ryosuke; Hironaka, Yasuo; Nakagawa, Ichiro; Tamura, Kentaro; Takeshima, Yasuhiro; Motoyama, Yasushi; Boku, Eishu; Ouji, Yukiteru; Yoshikawa, Masahide. Tumor-inhibition effect of levetiracetam in combination with temozolomide in glioblastoma cells. : 膠芽腫細胞株におけるテモゾロミド併用によるレベチラセタムの抗腫瘍効果.

Degree: 博士(医学), 2017, Nara Medical University / 奈良県立医科大学

Glioblastoma (GBM) is a malignant brain tumor with a poor prognosis. The standard postoperative chemotherapy is temozolomide (TMZ), which does not greatly improve survival. The… (more)

Subjects/Keywords: temozolomide; levetiracetam; premature senescence; glioblastoma

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APA (6th Edition):

Marutani, Akiko; Nakamura, Mitsutoshi; Nishimura, Fumihiko; Nakazawa, Tsutomu; Matsuda, Ryosuke; Hironaka, Yasuo; Nakagawa, Ichiro; Tamura, Kentaro; Takeshima, Yasuhiro; Motoyama, Yasushi; Boku, Eishu; Ouji, Yukiteru; Yoshikawa, M. (2017). Tumor-inhibition effect of levetiracetam in combination with temozolomide in glioblastoma cells. : 膠芽腫細胞株におけるテモゾロミド併用によるレベチラセタムの抗腫瘍効果. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marutani, Akiko; Nakamura, Mitsutoshi; Nishimura, Fumihiko; Nakazawa, Tsutomu; Matsuda, Ryosuke; Hironaka, Yasuo; Nakagawa, Ichiro; Tamura, Kentaro; Takeshima, Yasuhiro; Motoyama, Yasushi; Boku, Eishu; Ouji, Yukiteru; Yoshikawa, Masahide. “Tumor-inhibition effect of levetiracetam in combination with temozolomide in glioblastoma cells. : 膠芽腫細胞株におけるテモゾロミド併用によるレベチラセタムの抗腫瘍効果.” 2017. Thesis, Nara Medical University / 奈良県立医科大学. Accessed June 18, 2019. http://hdl.handle.net/10564/3320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marutani, Akiko; Nakamura, Mitsutoshi; Nishimura, Fumihiko; Nakazawa, Tsutomu; Matsuda, Ryosuke; Hironaka, Yasuo; Nakagawa, Ichiro; Tamura, Kentaro; Takeshima, Yasuhiro; Motoyama, Yasushi; Boku, Eishu; Ouji, Yukiteru; Yoshikawa, Masahide. “Tumor-inhibition effect of levetiracetam in combination with temozolomide in glioblastoma cells. : 膠芽腫細胞株におけるテモゾロミド併用によるレベチラセタムの抗腫瘍効果.” 2017. Web. 18 Jun 2019.

Vancouver:

Marutani, Akiko; Nakamura, Mitsutoshi; Nishimura, Fumihiko; Nakazawa, Tsutomu; Matsuda, Ryosuke; Hironaka, Yasuo; Nakagawa, Ichiro; Tamura, Kentaro; Takeshima, Yasuhiro; Motoyama, Yasushi; Boku, Eishu; Ouji, Yukiteru; Yoshikawa M. Tumor-inhibition effect of levetiracetam in combination with temozolomide in glioblastoma cells. : 膠芽腫細胞株におけるテモゾロミド併用によるレベチラセタムの抗腫瘍効果. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10564/3320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marutani, Akiko; Nakamura, Mitsutoshi; Nishimura, Fumihiko; Nakazawa, Tsutomu; Matsuda, Ryosuke; Hironaka, Yasuo; Nakagawa, Ichiro; Tamura, Kentaro; Takeshima, Yasuhiro; Motoyama, Yasushi; Boku, Eishu; Ouji, Yukiteru; Yoshikawa M. Tumor-inhibition effect of levetiracetam in combination with temozolomide in glioblastoma cells. : 膠芽腫細胞株におけるテモゾロミド併用によるレベチラセタムの抗腫瘍効果. [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. Available from: http://hdl.handle.net/10564/3320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

15. Ferreira-González, Sofía. Cellular senescence exacerbates injury and impairs regeneration in biliary disease.

Degree: PhD, 2017, University of Edinburgh

Senescence is a highly efficient mechanism that provides an irreversible barrier to cell cycle progression to prevent undesired proliferation. However, under pathological circumstances, senescence can… (more)

Subjects/Keywords: senescence; biliary disease; SASP; cholangiopathies

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APA (6th Edition):

Ferreira-González, S. (2017). Cellular senescence exacerbates injury and impairs regeneration in biliary disease. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28835

Chicago Manual of Style (16th Edition):

Ferreira-González, Sofía. “Cellular senescence exacerbates injury and impairs regeneration in biliary disease.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed June 18, 2019. http://hdl.handle.net/1842/28835.

MLA Handbook (7th Edition):

Ferreira-González, Sofía. “Cellular senescence exacerbates injury and impairs regeneration in biliary disease.” 2017. Web. 18 Jun 2019.

Vancouver:

Ferreira-González S. Cellular senescence exacerbates injury and impairs regeneration in biliary disease. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/1842/28835.

Council of Science Editors:

Ferreira-González S. Cellular senescence exacerbates injury and impairs regeneration in biliary disease. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28835


University of Cambridge

16. Hoare, Matthew. T-lymphocyte senescence and hepatitis C virus infection .

Degree: 2010, University of Cambridge

 Hepatitis C virus (HCV) infection is a leading cause of cirrhosis and hepatocellular carcinoma. The degree of fibrosis progression and treatment-related outcomes are critically dependent… (more)

Subjects/Keywords: Hepatitis; Telomere; Senescence; Interferon

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APA (6th Edition):

Hoare, M. (2010). T-lymphocyte senescence and hepatitis C virus infection . (Thesis). University of Cambridge. Retrieved from http://www.dspace.cam.ac.uk/handle/1810/226746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoare, Matthew. “T-lymphocyte senescence and hepatitis C virus infection .” 2010. Thesis, University of Cambridge. Accessed June 18, 2019. http://www.dspace.cam.ac.uk/handle/1810/226746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoare, Matthew. “T-lymphocyte senescence and hepatitis C virus infection .” 2010. Web. 18 Jun 2019.

Vancouver:

Hoare M. T-lymphocyte senescence and hepatitis C virus infection . [Internet] [Thesis]. University of Cambridge; 2010. [cited 2019 Jun 18]. Available from: http://www.dspace.cam.ac.uk/handle/1810/226746.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoare M. T-lymphocyte senescence and hepatitis C virus infection . [Thesis]. University of Cambridge; 2010. Available from: http://www.dspace.cam.ac.uk/handle/1810/226746

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Philipot, Didier. Implication du miR-24 et du miR-199a-5p dans le vieillissement prématuré du chondrocyte au cours de l'arthrose : Implication of miR-24 et du miR-199a-5p in cartilage premature aging during osteoarthritis.

Degree: Docteur es, Biochimie, biologie cellulaire et moléculaire, physiologie et nutrition, 2012, Université Montpellier I

L'arthrose tardive est la plus répandue des maladies ostéo-articulaires dont la prévalence augmente avec l'âge. Dans le cartilage arthrosique, des changements spécifiques des chondrocytes s'opèrent.… (more)

Subjects/Keywords: Arthrose; Senescence; Hypertrophie; Chondrocyte; MicroARNs; Osteoarthritis; Senescence; Hypertrophy; Chondrocytes; MicroRNAs

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APA (6th Edition):

Philipot, D. (2012). Implication du miR-24 et du miR-199a-5p dans le vieillissement prématuré du chondrocyte au cours de l'arthrose : Implication of miR-24 et du miR-199a-5p in cartilage premature aging during osteoarthritis. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2012MON1T015

Chicago Manual of Style (16th Edition):

Philipot, Didier. “Implication du miR-24 et du miR-199a-5p dans le vieillissement prématuré du chondrocyte au cours de l'arthrose : Implication of miR-24 et du miR-199a-5p in cartilage premature aging during osteoarthritis.” 2012. Doctoral Dissertation, Université Montpellier I. Accessed June 18, 2019. http://www.theses.fr/2012MON1T015.

MLA Handbook (7th Edition):

Philipot, Didier. “Implication du miR-24 et du miR-199a-5p dans le vieillissement prématuré du chondrocyte au cours de l'arthrose : Implication of miR-24 et du miR-199a-5p in cartilage premature aging during osteoarthritis.” 2012. Web. 18 Jun 2019.

Vancouver:

Philipot D. Implication du miR-24 et du miR-199a-5p dans le vieillissement prématuré du chondrocyte au cours de l'arthrose : Implication of miR-24 et du miR-199a-5p in cartilage premature aging during osteoarthritis. [Internet] [Doctoral dissertation]. Université Montpellier I; 2012. [cited 2019 Jun 18]. Available from: http://www.theses.fr/2012MON1T015.

Council of Science Editors:

Philipot D. Implication du miR-24 et du miR-199a-5p dans le vieillissement prématuré du chondrocyte au cours de l'arthrose : Implication of miR-24 et du miR-199a-5p in cartilage premature aging during osteoarthritis. [Doctoral Dissertation]. Université Montpellier I; 2012. Available from: http://www.theses.fr/2012MON1T015


Université Montpellier II

18. Kahli, Malik. Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire : HMGA proteins modify the replication program during senescence.

Degree: Docteur es, Biologie Santé, 2011, Université Montpellier II

La sénescence, considérée comme étant un arrêt irréversible du cycle cellulaire, se caractérise par des changements drastiques de l'expression génique et de l'organisation de la… (more)

Subjects/Keywords: Hmga; Senescence; Replication; Chromatine; Origines; Hmga; Senescence; Replication; Chromatin; Origins

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APA (6th Edition):

Kahli, M. (2011). Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire : HMGA proteins modify the replication program during senescence. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2011MON20059

Chicago Manual of Style (16th Edition):

Kahli, Malik. “Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire : HMGA proteins modify the replication program during senescence.” 2011. Doctoral Dissertation, Université Montpellier II. Accessed June 18, 2019. http://www.theses.fr/2011MON20059.

MLA Handbook (7th Edition):

Kahli, Malik. “Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire : HMGA proteins modify the replication program during senescence.” 2011. Web. 18 Jun 2019.

Vancouver:

Kahli M. Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire : HMGA proteins modify the replication program during senescence. [Internet] [Doctoral dissertation]. Université Montpellier II; 2011. [cited 2019 Jun 18]. Available from: http://www.theses.fr/2011MON20059.

Council of Science Editors:

Kahli M. Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire : HMGA proteins modify the replication program during senescence. [Doctoral Dissertation]. Université Montpellier II; 2011. Available from: http://www.theses.fr/2011MON20059


The Ohio State University

19. Bai, Shuangyi. Identification And Characterization Of Senescence-Associated Proteins In Petunia Corollas.

Degree: PhD, Food, Agricultural, and Biological Engineering, 2008, The Ohio State University

Senescence is a degenerate process that leads to the death of plant cells, organs or whole plants. Senescence is not a passive process, but is… (more)

Subjects/Keywords: Horticulture; flower senescence; proteomics; 2-DE; senescence-associated gene

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APA (6th Edition):

Bai, S. (2008). Identification And Characterization Of Senescence-Associated Proteins In Petunia Corollas. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1206041398

Chicago Manual of Style (16th Edition):

Bai, Shuangyi. “Identification And Characterization Of Senescence-Associated Proteins In Petunia Corollas.” 2008. Doctoral Dissertation, The Ohio State University. Accessed June 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1206041398.

MLA Handbook (7th Edition):

Bai, Shuangyi. “Identification And Characterization Of Senescence-Associated Proteins In Petunia Corollas.” 2008. Web. 18 Jun 2019.

Vancouver:

Bai S. Identification And Characterization Of Senescence-Associated Proteins In Petunia Corollas. [Internet] [Doctoral dissertation]. The Ohio State University; 2008. [cited 2019 Jun 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1206041398.

Council of Science Editors:

Bai S. Identification And Characterization Of Senescence-Associated Proteins In Petunia Corollas. [Doctoral Dissertation]. The Ohio State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1206041398


Dalhousie University

20. Dawson, Paul WH. Hepatitis B Virus X Protein Induces Cellular Senescence and Autophagy.

Degree: MS, Department of Microbiology & Immunology, 2011, Dalhousie University

 Hepatitis B virus (HBV) is a significant global threat to human health due to its ability to cause chronic infections that can lead to hepatocellular… (more)

Subjects/Keywords: Hepatitis B; Autophagy; Cellular Senescence; HBx

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APA (6th Edition):

Dawson, P. W. (2011). Hepatitis B Virus X Protein Induces Cellular Senescence and Autophagy. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14156

Chicago Manual of Style (16th Edition):

Dawson, Paul WH. “Hepatitis B Virus X Protein Induces Cellular Senescence and Autophagy.” 2011. Masters Thesis, Dalhousie University. Accessed June 18, 2019. http://hdl.handle.net/10222/14156.

MLA Handbook (7th Edition):

Dawson, Paul WH. “Hepatitis B Virus X Protein Induces Cellular Senescence and Autophagy.” 2011. Web. 18 Jun 2019.

Vancouver:

Dawson PW. Hepatitis B Virus X Protein Induces Cellular Senescence and Autophagy. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10222/14156.

Council of Science Editors:

Dawson PW. Hepatitis B Virus X Protein Induces Cellular Senescence and Autophagy. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14156


University of Georgia

21. Smith, Maren. Senescence and the Y chromosome.

Degree: BS, Biology, 2009, University of Georgia

 The Y chromosome is a non-recombining, patrilineal chromosome that is comprised of a few male specific genes, repetitive sequences, and transposable elements (Graves 2006). It… (more)

Subjects/Keywords: Senescence; Y chromosome; Drosophila melanogaster; aging

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APA (6th Edition):

Smith, M. (2009). Senescence and the Y chromosome. (Thesis). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/smith_maren_200912_bs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Maren. “Senescence and the Y chromosome.” 2009. Thesis, University of Georgia. Accessed June 18, 2019. http://purl.galileo.usg.edu/uga_etd/smith_maren_200912_bs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Maren. “Senescence and the Y chromosome.” 2009. Web. 18 Jun 2019.

Vancouver:

Smith M. Senescence and the Y chromosome. [Internet] [Thesis]. University of Georgia; 2009. [cited 2019 Jun 18]. Available from: http://purl.galileo.usg.edu/uga_etd/smith_maren_200912_bs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith M. Senescence and the Y chromosome. [Thesis]. University of Georgia; 2009. Available from: http://purl.galileo.usg.edu/uga_etd/smith_maren_200912_bs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Tran, Michaellong. Senescence of Native Perennial Warm Season Grasses Senescence Associated Switchgrass Transcriptome.

Degree: MS, Biology and Microbiology, 2016, South Dakota State University

Senescence of perennial crops enable continuous harvests after one sowing event. Perennials senesce at adapted rates of their native environments; however, early senescencing crops… (more)

Subjects/Keywords: senescence; switchgrass; transcriptome; Agricultural Science; Microbiology

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APA (6th Edition):

Tran, M. (2016). Senescence of Native Perennial Warm Season Grasses Senescence Associated Switchgrass Transcriptome. (Masters Thesis). South Dakota State University. Retrieved from http://openprairie.sdstate.edu/etd/978

Chicago Manual of Style (16th Edition):

Tran, Michaellong. “Senescence of Native Perennial Warm Season Grasses Senescence Associated Switchgrass Transcriptome.” 2016. Masters Thesis, South Dakota State University. Accessed June 18, 2019. http://openprairie.sdstate.edu/etd/978.

MLA Handbook (7th Edition):

Tran, Michaellong. “Senescence of Native Perennial Warm Season Grasses Senescence Associated Switchgrass Transcriptome.” 2016. Web. 18 Jun 2019.

Vancouver:

Tran M. Senescence of Native Perennial Warm Season Grasses Senescence Associated Switchgrass Transcriptome. [Internet] [Masters thesis]. South Dakota State University; 2016. [cited 2019 Jun 18]. Available from: http://openprairie.sdstate.edu/etd/978.

Council of Science Editors:

Tran M. Senescence of Native Perennial Warm Season Grasses Senescence Associated Switchgrass Transcriptome. [Masters Thesis]. South Dakota State University; 2016. Available from: http://openprairie.sdstate.edu/etd/978

23. Amin, Jakia. Regulation of autophagy in mammalian cells by stress activated signaling pathways.

Degree: 2013, Technische Universität Dortmund

 Autophagy is an intracellular bulk degradation process of cytoplasmic proteins and organelles aimed to preserve cellular homeostasis. It involves the formation of double-membrane structures, called… (more)

Subjects/Keywords: Autophagy; Microtubules; p38; Senescence; 540; 570

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APA (6th Edition):

Amin, J. (2013). Regulation of autophagy in mammalian cells by stress activated signaling pathways. (Thesis). Technische Universität Dortmund. Retrieved from http://hdl.handle.net/2003/30125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amin, Jakia. “Regulation of autophagy in mammalian cells by stress activated signaling pathways.” 2013. Thesis, Technische Universität Dortmund. Accessed June 18, 2019. http://hdl.handle.net/2003/30125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amin, Jakia. “Regulation of autophagy in mammalian cells by stress activated signaling pathways.” 2013. Web. 18 Jun 2019.

Vancouver:

Amin J. Regulation of autophagy in mammalian cells by stress activated signaling pathways. [Internet] [Thesis]. Technische Universität Dortmund; 2013. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/2003/30125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amin J. Regulation of autophagy in mammalian cells by stress activated signaling pathways. [Thesis]. Technische Universität Dortmund; 2013. Available from: http://hdl.handle.net/2003/30125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

24. Bertin, David. POTENTIAL NEW AGING MODEL SCHMIDTEA MEDITERRANEA UNDERGOES GRADUAL SENESCENCE, EXPERIENCES ACCELERATED SENESCENCE AT ELEVATED TEMPERATURE .

Degree: 2015, University of Guelph

 Current model organisms for aging research, commonly C. elegans and D. melanogaster, possess significant limitations such as non-aging life stages, loss of 10-15% of genes… (more)

Subjects/Keywords: Aging; Senescence; Flatworm; Demographic; Lifespan; Biomarker

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APA (6th Edition):

Bertin, D. (2015). POTENTIAL NEW AGING MODEL SCHMIDTEA MEDITERRANEA UNDERGOES GRADUAL SENESCENCE, EXPERIENCES ACCELERATED SENESCENCE AT ELEVATED TEMPERATURE . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8842

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bertin, David. “POTENTIAL NEW AGING MODEL SCHMIDTEA MEDITERRANEA UNDERGOES GRADUAL SENESCENCE, EXPERIENCES ACCELERATED SENESCENCE AT ELEVATED TEMPERATURE .” 2015. Thesis, University of Guelph. Accessed June 18, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8842.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bertin, David. “POTENTIAL NEW AGING MODEL SCHMIDTEA MEDITERRANEA UNDERGOES GRADUAL SENESCENCE, EXPERIENCES ACCELERATED SENESCENCE AT ELEVATED TEMPERATURE .” 2015. Web. 18 Jun 2019.

Vancouver:

Bertin D. POTENTIAL NEW AGING MODEL SCHMIDTEA MEDITERRANEA UNDERGOES GRADUAL SENESCENCE, EXPERIENCES ACCELERATED SENESCENCE AT ELEVATED TEMPERATURE . [Internet] [Thesis]. University of Guelph; 2015. [cited 2019 Jun 18]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8842.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bertin D. POTENTIAL NEW AGING MODEL SCHMIDTEA MEDITERRANEA UNDERGOES GRADUAL SENESCENCE, EXPERIENCES ACCELERATED SENESCENCE AT ELEVATED TEMPERATURE . [Thesis]. University of Guelph; 2015. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/8842

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

25. Manojlović-Gačić, Emilija M., 1979-. Imunohistohemijska analiza ekspresije regulatornih proteina ćelijskog ciklusa i proliferativnog Ki-67 (MIB-1) indeksa u adenomima hipofize.

Degree: Medicinski fakultet, 2016, Univerzitet u Beogradu

Molekularna medicina; patologija - Patologija; neuropatologija; endokrina patologija / Molecular medicine; pathology - Pathology; neuropathology; endocrine pathology

Adenomi hipofize su najčešći primarni tumori hipofize. Predstavljaju… (more)

Subjects/Keywords: Pituitary adenoma; cell-cycle; p16; 21; senescence

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APA (6th Edition):

Manojlović-Gačić, Emilija M., 1. (2016). Imunohistohemijska analiza ekspresije regulatornih proteina ćelijskog ciklusa i proliferativnog Ki-67 (MIB-1) indeksa u adenomima hipofize. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:12601/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manojlović-Gačić, Emilija M., 1979-. “Imunohistohemijska analiza ekspresije regulatornih proteina ćelijskog ciklusa i proliferativnog Ki-67 (MIB-1) indeksa u adenomima hipofize.” 2016. Thesis, Univerzitet u Beogradu. Accessed June 18, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:12601/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manojlović-Gačić, Emilija M., 1979-. “Imunohistohemijska analiza ekspresije regulatornih proteina ćelijskog ciklusa i proliferativnog Ki-67 (MIB-1) indeksa u adenomima hipofize.” 2016. Web. 18 Jun 2019.

Vancouver:

Manojlović-Gačić, Emilija M. 1. Imunohistohemijska analiza ekspresije regulatornih proteina ćelijskog ciklusa i proliferativnog Ki-67 (MIB-1) indeksa u adenomima hipofize. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2019 Jun 18]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12601/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manojlović-Gačić, Emilija M. 1. Imunohistohemijska analiza ekspresije regulatornih proteina ćelijskog ciklusa i proliferativnog Ki-67 (MIB-1) indeksa u adenomima hipofize. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:12601/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

26. Huang, Yu-hsuan. Cloning and characterization of an ethephon-inducible aspartic protease SPAP and its association with promotion of leaf senescence in sweet potato.

Degree: Master, Biological Sciences, 2013, NSYSU

 A full-length cDNA was isolated from sweet potato senescent leaves with PCR-selective subtractive hybridization and RACE PCR. Blast analysis showed that it exhibited high nucleotide… (more)

Subjects/Keywords: Aspartic protease; Ethephon; Leaf senescence; Sweet potato

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, Y. (2013). Cloning and characterization of an ethephon-inducible aspartic protease SPAP and its association with promotion of leaf senescence in sweet potato. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0608113-091307

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yu-hsuan. “Cloning and characterization of an ethephon-inducible aspartic protease SPAP and its association with promotion of leaf senescence in sweet potato.” 2013. Thesis, NSYSU. Accessed June 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0608113-091307.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yu-hsuan. “Cloning and characterization of an ethephon-inducible aspartic protease SPAP and its association with promotion of leaf senescence in sweet potato.” 2013. Web. 18 Jun 2019.

Vancouver:

Huang Y. Cloning and characterization of an ethephon-inducible aspartic protease SPAP and its association with promotion of leaf senescence in sweet potato. [Internet] [Thesis]. NSYSU; 2013. [cited 2019 Jun 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0608113-091307.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang Y. Cloning and characterization of an ethephon-inducible aspartic protease SPAP and its association with promotion of leaf senescence in sweet potato. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0608113-091307

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Yamada, Shuichi; Matsuda, Ryosuke; Nishimura, Fumihiko; Nakagawa, Ichiro; Motoyama, Yasushi; Park, Young-Su; Nakamura, Mitsutoshi; Nakase, Hiroyuki; Ouji, Yukiteru. Carnitine-induced senescence in glioblastoma cells. : 神経膠芽腫細胞におけるカルニチンによるセネッセンスの誘導.

Degree: Nara Medical University / 奈良県立医科大学

Carnitine is essential for lipid metabolism in cells and is known to possess antioxidant properties. Previous reports have suggested that antioxidants are able to induce… (more)

Subjects/Keywords: carnitine; glioblastoma; senescence

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APA (6th Edition):

Yamada, Shuichi; Matsuda, Ryosuke; Nishimura, Fumihiko; Nakagawa, Ichiro; Motoyama, Yasushi; Park, Young-Su; Nakamura, Mitsutoshi; Nakase, Hiroyuki; Ouji, Y. (n.d.). Carnitine-induced senescence in glioblastoma cells. : 神経膠芽腫細胞におけるカルニチンによるセネッセンスの誘導. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2782

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamada, Shuichi; Matsuda, Ryosuke; Nishimura, Fumihiko; Nakagawa, Ichiro; Motoyama, Yasushi; Park, Young-Su; Nakamura, Mitsutoshi; Nakase, Hiroyuki; Ouji, Yukiteru. “Carnitine-induced senescence in glioblastoma cells. : 神経膠芽腫細胞におけるカルニチンによるセネッセンスの誘導.” Thesis, Nara Medical University / 奈良県立医科大学. Accessed June 18, 2019. http://hdl.handle.net/10564/2782.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamada, Shuichi; Matsuda, Ryosuke; Nishimura, Fumihiko; Nakagawa, Ichiro; Motoyama, Yasushi; Park, Young-Su; Nakamura, Mitsutoshi; Nakase, Hiroyuki; Ouji, Yukiteru. “Carnitine-induced senescence in glioblastoma cells. : 神経膠芽腫細胞におけるカルニチンによるセネッセンスの誘導.” Web. 18 Jun 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Yamada, Shuichi; Matsuda, Ryosuke; Nishimura, Fumihiko; Nakagawa, Ichiro; Motoyama, Yasushi; Park, Young-Su; Nakamura, Mitsutoshi; Nakase, Hiroyuki; Ouji Y. Carnitine-induced senescence in glioblastoma cells. : 神経膠芽腫細胞におけるカルニチンによるセネッセンスの誘導. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10564/2782.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Yamada, Shuichi; Matsuda, Ryosuke; Nishimura, Fumihiko; Nakagawa, Ichiro; Motoyama, Yasushi; Park, Young-Su; Nakamura, Mitsutoshi; Nakase, Hiroyuki; Ouji Y. Carnitine-induced senescence in glioblastoma cells. : 神経膠芽腫細胞におけるカルニチンによるセネッセンスの誘導. [Thesis]. Nara Medical University / 奈良県立医科大学; Available from: http://hdl.handle.net/10564/2782

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Queens University

28. Han, Xu. The evolutionary consequences of sperm senescence in Drosophila melanogaster .

Degree: Biology, 2014, Queens University

 Sperm senescence, a decline in sperm quality caused by male ageing and by sperm ageing before or after copulation, may have fitness costs manifested as… (more)

Subjects/Keywords: evolutionary consequences; Drosophila melanogaster; nutrition; sperm senescence

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Han, X. (2014). The evolutionary consequences of sperm senescence in Drosophila melanogaster . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8657

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Han, Xu. “The evolutionary consequences of sperm senescence in Drosophila melanogaster .” 2014. Thesis, Queens University. Accessed June 18, 2019. http://hdl.handle.net/1974/8657.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Han, Xu. “The evolutionary consequences of sperm senescence in Drosophila melanogaster .” 2014. Web. 18 Jun 2019.

Vancouver:

Han X. The evolutionary consequences of sperm senescence in Drosophila melanogaster . [Internet] [Thesis]. Queens University; 2014. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/1974/8657.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Han X. The evolutionary consequences of sperm senescence in Drosophila melanogaster . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/8657

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hawaii – Manoa

29. Perez, Pierriden Azucena. Molecular genetic studies of senescence in anthurium.

Degree: 2016, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2012.

Senescence is a complex physiological process and has become an attractive area of research in plant molecular biology.… (more)

Subjects/Keywords: anthurium; Anthurium senescence; Agrobacterium-mediated transformation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Perez, P. A. (2016). Molecular genetic studies of senescence in anthurium. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/101012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perez, Pierriden Azucena. “Molecular genetic studies of senescence in anthurium.” 2016. Thesis, University of Hawaii – Manoa. Accessed June 18, 2019. http://hdl.handle.net/10125/101012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perez, Pierriden Azucena. “Molecular genetic studies of senescence in anthurium.” 2016. Web. 18 Jun 2019.

Vancouver:

Perez PA. Molecular genetic studies of senescence in anthurium. [Internet] [Thesis]. University of Hawaii – Manoa; 2016. [cited 2019 Jun 18]. Available from: http://hdl.handle.net/10125/101012.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perez PA. Molecular genetic studies of senescence in anthurium. [Thesis]. University of Hawaii – Manoa; 2016. Available from: http://hdl.handle.net/10125/101012

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

30. Zhu, Bokai. Modulation Of Skin Cancer By Peroxisome Proliferator-activated Receptor Beta/delta.

Degree: PhD, Molecular Medicine, 2012, Penn State University

 Ligand activation of peroxisome proliferator–activated receptor-β/δ (PPARβ/δ) inhibits chemically-induced skin tumorigenesis and Pparβ/δ-null mice exhibit enhanced chemically-induced skin tumorigenesis compared to wild-type mice. Since over… (more)

Subjects/Keywords: PPARb/d; HRAS; Mitosis; Senescence; ER stress

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhu, B. (2012). Modulation Of Skin Cancer By Peroxisome Proliferator-activated Receptor Beta/delta. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/15143

Chicago Manual of Style (16th Edition):

Zhu, Bokai. “Modulation Of Skin Cancer By Peroxisome Proliferator-activated Receptor Beta/delta.” 2012. Doctoral Dissertation, Penn State University. Accessed June 18, 2019. https://etda.libraries.psu.edu/catalog/15143.

MLA Handbook (7th Edition):

Zhu, Bokai. “Modulation Of Skin Cancer By Peroxisome Proliferator-activated Receptor Beta/delta.” 2012. Web. 18 Jun 2019.

Vancouver:

Zhu B. Modulation Of Skin Cancer By Peroxisome Proliferator-activated Receptor Beta/delta. [Internet] [Doctoral dissertation]. Penn State University; 2012. [cited 2019 Jun 18]. Available from: https://etda.libraries.psu.edu/catalog/15143.

Council of Science Editors:

Zhu B. Modulation Of Skin Cancer By Peroxisome Proliferator-activated Receptor Beta/delta. [Doctoral Dissertation]. Penn State University; 2012. Available from: https://etda.libraries.psu.edu/catalog/15143

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