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You searched for subject:(ryanodine receptor). Showing records 1 – 30 of 54 total matches.

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University College Cork

1. Zheng, Limian. Ryanodine receptor expression in trophoblasts.

Degree: 2013, University College Cork

 Trophoblasts of the placenta are the frontline cells involved in communication and exchange of materials between the mother and fetus. Within trophoblasts, calcium signalling proteins… (more)

Subjects/Keywords: Calcium; Ryanodine receptor; Trophoblast; Ryanodine – Receptors.; Calcium channels.

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APA (6th Edition):

Zheng, L. (2013). Ryanodine receptor expression in trophoblasts. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/1092

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zheng, Limian. “Ryanodine receptor expression in trophoblasts.” 2013. Thesis, University College Cork. Accessed July 02, 2020. http://hdl.handle.net/10468/1092.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zheng, Limian. “Ryanodine receptor expression in trophoblasts.” 2013. Web. 02 Jul 2020.

Vancouver:

Zheng L. Ryanodine receptor expression in trophoblasts. [Internet] [Thesis]. University College Cork; 2013. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10468/1092.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zheng L. Ryanodine receptor expression in trophoblasts. [Thesis]. University College Cork; 2013. Available from: http://hdl.handle.net/10468/1092

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

2. Oo, Ye Wint. Effects of calmodulin on the function of cardiac and skeletal ryanodine receptors.

Degree: PhD, 2016, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Dantrolene is a muscle relaxant that has been used clinically for the treatment of the malignant hyperthermia (MH)… (more)

Subjects/Keywords: skeletal muscle; myocytes; heart; ryanodine receptor; calmodulin

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APA (6th Edition):

Oo, Y. W. (2016). Effects of calmodulin on the function of cardiac and skeletal ryanodine receptors. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1332736

Chicago Manual of Style (16th Edition):

Oo, Ye Wint. “Effects of calmodulin on the function of cardiac and skeletal ryanodine receptors.” 2016. Doctoral Dissertation, University of Newcastle. Accessed July 02, 2020. http://hdl.handle.net/1959.13/1332736.

MLA Handbook (7th Edition):

Oo, Ye Wint. “Effects of calmodulin on the function of cardiac and skeletal ryanodine receptors.” 2016. Web. 02 Jul 2020.

Vancouver:

Oo YW. Effects of calmodulin on the function of cardiac and skeletal ryanodine receptors. [Internet] [Doctoral dissertation]. University of Newcastle; 2016. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1959.13/1332736.

Council of Science Editors:

Oo YW. Effects of calmodulin on the function of cardiac and skeletal ryanodine receptors. [Doctoral Dissertation]. University of Newcastle; 2016. Available from: http://hdl.handle.net/1959.13/1332736


University of Manchester

3. Handhle, Ahmed. Towards understanding the formation of an SR luminal Ca-sensor.

Degree: PhD, 2012, University of Manchester

 Calcium induced calcium release (CICR) is the process mediating cardiac excitation contraction coupling (ECC). In brief, depolarisation of the plasma membrane of the cardiac myocyte… (more)

Subjects/Keywords: 612.1; Ryanodine Receptor; Luminal Ca-Sensor

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APA (6th Edition):

Handhle, A. (2012). Towards understanding the formation of an SR luminal Ca-sensor. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/towards-understanding-the-formation-of-an-sr-luminal-casensor(1d0b27ac-0f7f-42fe-81bd-cce48dd42b55).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566584

Chicago Manual of Style (16th Edition):

Handhle, Ahmed. “Towards understanding the formation of an SR luminal Ca-sensor.” 2012. Doctoral Dissertation, University of Manchester. Accessed July 02, 2020. https://www.research.manchester.ac.uk/portal/en/theses/towards-understanding-the-formation-of-an-sr-luminal-casensor(1d0b27ac-0f7f-42fe-81bd-cce48dd42b55).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566584.

MLA Handbook (7th Edition):

Handhle, Ahmed. “Towards understanding the formation of an SR luminal Ca-sensor.” 2012. Web. 02 Jul 2020.

Vancouver:

Handhle A. Towards understanding the formation of an SR luminal Ca-sensor. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Jul 02]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/towards-understanding-the-formation-of-an-sr-luminal-casensor(1d0b27ac-0f7f-42fe-81bd-cce48dd42b55).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566584.

Council of Science Editors:

Handhle A. Towards understanding the formation of an SR luminal Ca-sensor. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/towards-understanding-the-formation-of-an-sr-luminal-casensor(1d0b27ac-0f7f-42fe-81bd-cce48dd42b55).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.566584


University of Exeter

4. Clowsley, Alexander Harrington. Optimisation and comparison of dSTORM and DNA-PAINT super-resolution for quantitative cardiac protein imaging.

Degree: PhD, 2017, University of Exeter

 Fluorescence microscopy techniques, restricted by the diffraction limit of light, have seen a remarkable advancement in recent years. An approach called dSTORM (direct stochastic optical… (more)

Subjects/Keywords: 530; DNA-PAINT; Cardiac; Ryanodine Receptor

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APA (6th Edition):

Clowsley, A. H. (2017). Optimisation and comparison of dSTORM and DNA-PAINT super-resolution for quantitative cardiac protein imaging. (Doctoral Dissertation). University of Exeter. Retrieved from http://hdl.handle.net/10871/33021

Chicago Manual of Style (16th Edition):

Clowsley, Alexander Harrington. “Optimisation and comparison of dSTORM and DNA-PAINT super-resolution for quantitative cardiac protein imaging.” 2017. Doctoral Dissertation, University of Exeter. Accessed July 02, 2020. http://hdl.handle.net/10871/33021.

MLA Handbook (7th Edition):

Clowsley, Alexander Harrington. “Optimisation and comparison of dSTORM and DNA-PAINT super-resolution for quantitative cardiac protein imaging.” 2017. Web. 02 Jul 2020.

Vancouver:

Clowsley AH. Optimisation and comparison of dSTORM and DNA-PAINT super-resolution for quantitative cardiac protein imaging. [Internet] [Doctoral dissertation]. University of Exeter; 2017. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10871/33021.

Council of Science Editors:

Clowsley AH. Optimisation and comparison of dSTORM and DNA-PAINT super-resolution for quantitative cardiac protein imaging. [Doctoral Dissertation]. University of Exeter; 2017. Available from: http://hdl.handle.net/10871/33021


University of Minnesota

5. Her, Cheng. Structural Dynamics of the Calmodulin-Ryanodine Receptor Interaction Using Bifunctional Spin Labels and EPR.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2018, University of Minnesota

 Muscle contraction and relaxation are regulated by changes in intracellular calcium levels. To facilitate muscle contraction, calcium is released from the intracellular calcium reservoir into… (more)

Subjects/Keywords: calmodulin; DEER; EPR; ryanodine receptor; spin label

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APA (6th Edition):

Her, C. (2018). Structural Dynamics of the Calmodulin-Ryanodine Receptor Interaction Using Bifunctional Spin Labels and EPR. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/201032

Chicago Manual of Style (16th Edition):

Her, Cheng. “Structural Dynamics of the Calmodulin-Ryanodine Receptor Interaction Using Bifunctional Spin Labels and EPR.” 2018. Doctoral Dissertation, University of Minnesota. Accessed July 02, 2020. http://hdl.handle.net/11299/201032.

MLA Handbook (7th Edition):

Her, Cheng. “Structural Dynamics of the Calmodulin-Ryanodine Receptor Interaction Using Bifunctional Spin Labels and EPR.” 2018. Web. 02 Jul 2020.

Vancouver:

Her C. Structural Dynamics of the Calmodulin-Ryanodine Receptor Interaction Using Bifunctional Spin Labels and EPR. [Internet] [Doctoral dissertation]. University of Minnesota; 2018. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/11299/201032.

Council of Science Editors:

Her C. Structural Dynamics of the Calmodulin-Ryanodine Receptor Interaction Using Bifunctional Spin Labels and EPR. [Doctoral Dissertation]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/201032


University of Rochester

6. Loy, Ryan Eric. Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease.

Degree: PhD, 2011, University of Rochester

 The type 1 isoform of the ryanodine receptor (RyR1) controls the release of Ca2+ from the sarcoplasmic reticulum (SR) during skeletal muscle excitation-contraction (EC) coupling.… (more)

Subjects/Keywords: Ryanodine Receptor; Myopathy; Central Core Disease; Malignant Hyperthermia; Ca2+

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APA (6th Edition):

Loy, R. E. (2011). Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/13950

Chicago Manual of Style (16th Edition):

Loy, Ryan Eric. “Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease.” 2011. Doctoral Dissertation, University of Rochester. Accessed July 02, 2020. http://hdl.handle.net/1802/13950.

MLA Handbook (7th Edition):

Loy, Ryan Eric. “Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease.” 2011. Web. 02 Jul 2020.

Vancouver:

Loy RE. Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1802/13950.

Council of Science Editors:

Loy RE. Altered Type 1 Ryanodine Receptor Activity and Functional Rescue In a Mouse Model of Central Core Disease. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/13950


NSYSU

7. Ko, Pin-yu. Studies on the role of hydrogen peroxide in the myocyte secretion of IGF-1 at developing neuromuscular synapse.

Degree: Master, Biological Sciences, 2016, NSYSU

 The successful synaptogenesis at neuromuscular junction is an intricate process relies on dynamic interaction of nerve- and muscle-derived molecules. We have previously demonstrated that muscle-derived… (more)

Subjects/Keywords: IGF-1; secretion; H2O2; sarcoplasmic reticulum; ryanodine receptor

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APA (6th Edition):

Ko, P. (2016). Studies on the role of hydrogen peroxide in the myocyte secretion of IGF-1 at developing neuromuscular synapse. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0627116-094848

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ko, Pin-yu. “Studies on the role of hydrogen peroxide in the myocyte secretion of IGF-1 at developing neuromuscular synapse.” 2016. Thesis, NSYSU. Accessed July 02, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0627116-094848.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ko, Pin-yu. “Studies on the role of hydrogen peroxide in the myocyte secretion of IGF-1 at developing neuromuscular synapse.” 2016. Web. 02 Jul 2020.

Vancouver:

Ko P. Studies on the role of hydrogen peroxide in the myocyte secretion of IGF-1 at developing neuromuscular synapse. [Internet] [Thesis]. NSYSU; 2016. [cited 2020 Jul 02]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0627116-094848.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ko P. Studies on the role of hydrogen peroxide in the myocyte secretion of IGF-1 at developing neuromuscular synapse. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0627116-094848

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Massey University

8. Roesl, Cornelia. Functional and structural characterisation of the malignant hyperthermia associated RYR1 mutation R245W : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry .

Degree: 2013, Massey University

 Malignant hyperthermia (MH) is an autosomal dominant pharmacogenetic disorder of skeletal muscle triggered by volatile halogenated anaesthetics. Susceptible individuals can exhibit symptoms including tachycardia, high… (more)

Subjects/Keywords: Malignant hyperthermia; Ryanodine receptors; RYR1; Gene receptor mutation

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APA (6th Edition):

Roesl, C. (2013). Functional and structural characterisation of the malignant hyperthermia associated RYR1 mutation R245W : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/5150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roesl, Cornelia. “Functional and structural characterisation of the malignant hyperthermia associated RYR1 mutation R245W : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry .” 2013. Thesis, Massey University. Accessed July 02, 2020. http://hdl.handle.net/10179/5150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roesl, Cornelia. “Functional and structural characterisation of the malignant hyperthermia associated RYR1 mutation R245W : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry .” 2013. Web. 02 Jul 2020.

Vancouver:

Roesl C. Functional and structural characterisation of the malignant hyperthermia associated RYR1 mutation R245W : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry . [Internet] [Thesis]. Massey University; 2013. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10179/5150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roesl C. Functional and structural characterisation of the malignant hyperthermia associated RYR1 mutation R245W : a thesis presented to Massey University in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry . [Thesis]. Massey University; 2013. Available from: http://hdl.handle.net/10179/5150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of St. Andrews

9. Reilly-O'Donnell, Benedict. Investigating the role of Zn²⁺ in regulating the function of intracellular Ca²⁺-release channels .

Degree: 2018, University of St. Andrews

 The tightly regulated openings of the cardiac ryanodine receptor (RyR2) help to ensure that intracellular Ca²⁺- release from the sarcoplasmic reticulum (SR) can only occur… (more)

Subjects/Keywords: Calcium dynamics; Zinc; Ryanodine receptor; Two pore channel; Electrophysiology

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APA (6th Edition):

Reilly-O'Donnell, B. (2018). Investigating the role of Zn²⁺ in regulating the function of intracellular Ca²⁺-release channels . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/16145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reilly-O'Donnell, Benedict. “Investigating the role of Zn²⁺ in regulating the function of intracellular Ca²⁺-release channels .” 2018. Thesis, University of St. Andrews. Accessed July 02, 2020. http://hdl.handle.net/10023/16145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reilly-O'Donnell, Benedict. “Investigating the role of Zn²⁺ in regulating the function of intracellular Ca²⁺-release channels .” 2018. Web. 02 Jul 2020.

Vancouver:

Reilly-O'Donnell B. Investigating the role of Zn²⁺ in regulating the function of intracellular Ca²⁺-release channels . [Internet] [Thesis]. University of St. Andrews; 2018. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/10023/16145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reilly-O'Donnell B. Investigating the role of Zn²⁺ in regulating the function of intracellular Ca²⁺-release channels . [Thesis]. University of St. Andrews; 2018. Available from: http://hdl.handle.net/10023/16145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Gilbert, Guillaume. Les voies de signalisation calciques impliquées dans la réponse à l’étirement dans les artères intrapulmonaires. Modifications dans l’hypertension pulmonaire : Ca2+ signaling pathways involved in response to stretch in pulmonary arteries. Implication in pulmonary hypertension.

Degree: Docteur es, Biologie Cellulaire et Physiopathologie, 2014, Bordeaux

L’hypertension pulmonaire (HTP) est la principale pathologie de la circulation pulmonaire. Elle secaractérise par une augmentation maintenue de la pression dans les artères intrapulmonaires (AIP)… (more)

Subjects/Keywords: Hypertension pulmonaire; Stretch; Ca2+; Récepteur à la ryanodine; Caveolae; Pulmonary hypertension; Stretch; Ca2+; Ryanodine receptor; Caveolae

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APA (6th Edition):

Gilbert, G. (2014). Les voies de signalisation calciques impliquées dans la réponse à l’étirement dans les artères intrapulmonaires. Modifications dans l’hypertension pulmonaire : Ca2+ signaling pathways involved in response to stretch in pulmonary arteries. Implication in pulmonary hypertension. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2014BORD0179

Chicago Manual of Style (16th Edition):

Gilbert, Guillaume. “Les voies de signalisation calciques impliquées dans la réponse à l’étirement dans les artères intrapulmonaires. Modifications dans l’hypertension pulmonaire : Ca2+ signaling pathways involved in response to stretch in pulmonary arteries. Implication in pulmonary hypertension.” 2014. Doctoral Dissertation, Bordeaux. Accessed July 02, 2020. http://www.theses.fr/2014BORD0179.

MLA Handbook (7th Edition):

Gilbert, Guillaume. “Les voies de signalisation calciques impliquées dans la réponse à l’étirement dans les artères intrapulmonaires. Modifications dans l’hypertension pulmonaire : Ca2+ signaling pathways involved in response to stretch in pulmonary arteries. Implication in pulmonary hypertension.” 2014. Web. 02 Jul 2020.

Vancouver:

Gilbert G. Les voies de signalisation calciques impliquées dans la réponse à l’étirement dans les artères intrapulmonaires. Modifications dans l’hypertension pulmonaire : Ca2+ signaling pathways involved in response to stretch in pulmonary arteries. Implication in pulmonary hypertension. [Internet] [Doctoral dissertation]. Bordeaux; 2014. [cited 2020 Jul 02]. Available from: http://www.theses.fr/2014BORD0179.

Council of Science Editors:

Gilbert G. Les voies de signalisation calciques impliquées dans la réponse à l’étirement dans les artères intrapulmonaires. Modifications dans l’hypertension pulmonaire : Ca2+ signaling pathways involved in response to stretch in pulmonary arteries. Implication in pulmonary hypertension. [Doctoral Dissertation]. Bordeaux; 2014. Available from: http://www.theses.fr/2014BORD0179

11. Bussiere, Renaud. Dérégulation de l’homéostasie calcique du réticulum endoplasmique dans la maladie d’Alzheimer : rôle du récepteur de la ryanodine et de l’isoforme SERCA1 tronquée : Deregulation of endoplasmic reticulum calcium homeostasis in Alzheimer’s disease : role of ryanodine receptor and of the truncated SERCA1 isoform.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Université Côte d'Azur (ComUE)

Le calcium (Ca2+) joue un rôle prépondérant dans la fonction de nos neurones et du système nerveux central. Différents travaux ont rapporté que la dérégulation… (more)

Subjects/Keywords: Récepteur de la ryanodine; Maladie d’Alzheimer; Réticulum endoplasmique; Dérégulation calcique; S1T; Ryanodine receptor; Alzheimer’s disease; Endoplasmic reticulum; Calcium deregulation; S1T

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APA (6th Edition):

Bussiere, R. (2018). Dérégulation de l’homéostasie calcique du réticulum endoplasmique dans la maladie d’Alzheimer : rôle du récepteur de la ryanodine et de l’isoforme SERCA1 tronquée : Deregulation of endoplasmic reticulum calcium homeostasis in Alzheimer’s disease : role of ryanodine receptor and of the truncated SERCA1 isoform. (Doctoral Dissertation). Université Côte d'Azur (ComUE). Retrieved from http://www.theses.fr/2018AZUR4207

Chicago Manual of Style (16th Edition):

Bussiere, Renaud. “Dérégulation de l’homéostasie calcique du réticulum endoplasmique dans la maladie d’Alzheimer : rôle du récepteur de la ryanodine et de l’isoforme SERCA1 tronquée : Deregulation of endoplasmic reticulum calcium homeostasis in Alzheimer’s disease : role of ryanodine receptor and of the truncated SERCA1 isoform.” 2018. Doctoral Dissertation, Université Côte d'Azur (ComUE). Accessed July 02, 2020. http://www.theses.fr/2018AZUR4207.

MLA Handbook (7th Edition):

Bussiere, Renaud. “Dérégulation de l’homéostasie calcique du réticulum endoplasmique dans la maladie d’Alzheimer : rôle du récepteur de la ryanodine et de l’isoforme SERCA1 tronquée : Deregulation of endoplasmic reticulum calcium homeostasis in Alzheimer’s disease : role of ryanodine receptor and of the truncated SERCA1 isoform.” 2018. Web. 02 Jul 2020.

Vancouver:

Bussiere R. Dérégulation de l’homéostasie calcique du réticulum endoplasmique dans la maladie d’Alzheimer : rôle du récepteur de la ryanodine et de l’isoforme SERCA1 tronquée : Deregulation of endoplasmic reticulum calcium homeostasis in Alzheimer’s disease : role of ryanodine receptor and of the truncated SERCA1 isoform. [Internet] [Doctoral dissertation]. Université Côte d'Azur (ComUE); 2018. [cited 2020 Jul 02]. Available from: http://www.theses.fr/2018AZUR4207.

Council of Science Editors:

Bussiere R. Dérégulation de l’homéostasie calcique du réticulum endoplasmique dans la maladie d’Alzheimer : rôle du récepteur de la ryanodine et de l’isoforme SERCA1 tronquée : Deregulation of endoplasmic reticulum calcium homeostasis in Alzheimer’s disease : role of ryanodine receptor and of the truncated SERCA1 isoform. [Doctoral Dissertation]. Université Côte d'Azur (ComUE); 2018. Available from: http://www.theses.fr/2018AZUR4207


Temple University

12. Zhang, Hongyu. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.

Degree: PhD, 2010, Temple University

Physiology

Myocardial infarction (MI) leads to heart failure (HF) and premature death. The respective roles of myocyte death and depressed myocyte contractility in the induction… (more)

Subjects/Keywords: Biology, Physiology; cardiac function; cell death; heart failure; myocardial infarction; myocyte contractility; ryanodine receptor

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APA (6th Edition):

Zhang, H. (2010). INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,90859

Chicago Manual of Style (16th Edition):

Zhang, Hongyu. “INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.” 2010. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,90859.

MLA Handbook (7th Edition):

Zhang, Hongyu. “INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.” 2010. Web. 02 Jul 2020.

Vancouver:

Zhang H. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,90859.

Council of Science Editors:

Zhang H. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,90859


University of Newcastle

13. Li, Jiao. Regulation of cardiac calcium release channels during acute beta-adrenergic stimulation.

Degree: PhD, 2013, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

During β-adrenergic stimulation of the heart, ryanodine receptors (RyRs) Ca²⁺ release channels in the SR can be phosphorylated… (more)

Subjects/Keywords: cardiac ryanodine receptor; beta-adrenergic signalling; calcium signalling; phosphorylation; heart rate; PKA; CaMKII

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APA (6th Edition):

Li, J. (2013). Regulation of cardiac calcium release channels during acute beta-adrenergic stimulation. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1036949

Chicago Manual of Style (16th Edition):

Li, Jiao. “Regulation of cardiac calcium release channels during acute beta-adrenergic stimulation.” 2013. Doctoral Dissertation, University of Newcastle. Accessed July 02, 2020. http://hdl.handle.net/1959.13/1036949.

MLA Handbook (7th Edition):

Li, Jiao. “Regulation of cardiac calcium release channels during acute beta-adrenergic stimulation.” 2013. Web. 02 Jul 2020.

Vancouver:

Li J. Regulation of cardiac calcium release channels during acute beta-adrenergic stimulation. [Internet] [Doctoral dissertation]. University of Newcastle; 2013. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1959.13/1036949.

Council of Science Editors:

Li J. Regulation of cardiac calcium release channels during acute beta-adrenergic stimulation. [Doctoral Dissertation]. University of Newcastle; 2013. Available from: http://hdl.handle.net/1959.13/1036949


The Ohio State University

14. Walton, Shane David. Engineering an Anti-arrhythmic Calmodulin.

Degree: PhD, Biophysics, 2016, The Ohio State University

  Calmodulin (CaM) is an integral part of Ca2+ signaling in all cell types, performing diverse functions through its interaction with target proteins such as… (more)

Subjects/Keywords: Biophysics; calmodulin; protein engineering; cardiac arrhythmia; CPVT; LQTS; ryanodine receptor regulation; AAV9

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APA (6th Edition):

Walton, S. D. (2016). Engineering an Anti-arrhythmic Calmodulin. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1461159861

Chicago Manual of Style (16th Edition):

Walton, Shane David. “Engineering an Anti-arrhythmic Calmodulin.” 2016. Doctoral Dissertation, The Ohio State University. Accessed July 02, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1461159861.

MLA Handbook (7th Edition):

Walton, Shane David. “Engineering an Anti-arrhythmic Calmodulin.” 2016. Web. 02 Jul 2020.

Vancouver:

Walton SD. Engineering an Anti-arrhythmic Calmodulin. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2020 Jul 02]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1461159861.

Council of Science Editors:

Walton SD. Engineering an Anti-arrhythmic Calmodulin. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1461159861

15. Handhle, Ahmed. Towards Understanding the Formation of an SR Luminal Ca-Sensor.

Degree: 2012, University of Manchester

 Calcium induced calcium release (CICR) is the process mediating cardiac excitation contraction coupling (ECC). In brief, depolarisation of the plasma membrane of the cardiac myocyte… (more)

Subjects/Keywords: Ryanodine Receptor; Luminal Ca-Sensor

…45 Figure 3.1: Purification of rabbit skeletal ryanodine receptor (RyR1)… …69 Figure 3.2 Purification of canine cardiac ryanodine receptor (RyR2)… …Ryanodine receptor soyabean lecithin Sudden cardiac death Sodium dodecyl sulphate polyacrylamide… …the skeletal muscle ryanodine receptor isoform, RyR1, from sheep skeletal muscle employing… …biochemical and biophysical techniques was next employed to examine whether the ryanodine receptor… 

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APA (6th Edition):

Handhle, A. (2012). Towards Understanding the Formation of an SR Luminal Ca-Sensor. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:163463

Chicago Manual of Style (16th Edition):

Handhle, Ahmed. “Towards Understanding the Formation of an SR Luminal Ca-Sensor.” 2012. Doctoral Dissertation, University of Manchester. Accessed July 02, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:163463.

MLA Handbook (7th Edition):

Handhle, Ahmed. “Towards Understanding the Formation of an SR Luminal Ca-Sensor.” 2012. Web. 02 Jul 2020.

Vancouver:

Handhle A. Towards Understanding the Formation of an SR Luminal Ca-Sensor. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Jul 02]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:163463.

Council of Science Editors:

Handhle A. Towards Understanding the Formation of an SR Luminal Ca-Sensor. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:163463


University of Toronto

16. Ryan, Timothy. Identification of Ryanodine Receptor 1 (RyR1) Interacting Protein Partners Using Liquid Chromatography and Mass Spectrometry.

Degree: 2010, University of Toronto

Ryanodine receptor 1 (RyR1) is a homotetrameric calcium channel located in the sarcoplasmic reticulum (SR) of skeletal muscle. We employed metal affinity chromatography followed by… (more)

Subjects/Keywords: Ryanodine Receptor 1; Metal affinity chromatography; Mass Spectrometry; Protein-protein interactions; 0719

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APA (6th Edition):

Ryan, T. (2010). Identification of Ryanodine Receptor 1 (RyR1) Interacting Protein Partners Using Liquid Chromatography and Mass Spectrometry. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25911

Chicago Manual of Style (16th Edition):

Ryan, Timothy. “Identification of Ryanodine Receptor 1 (RyR1) Interacting Protein Partners Using Liquid Chromatography and Mass Spectrometry.” 2010. Masters Thesis, University of Toronto. Accessed July 02, 2020. http://hdl.handle.net/1807/25911.

MLA Handbook (7th Edition):

Ryan, Timothy. “Identification of Ryanodine Receptor 1 (RyR1) Interacting Protein Partners Using Liquid Chromatography and Mass Spectrometry.” 2010. Web. 02 Jul 2020.

Vancouver:

Ryan T. Identification of Ryanodine Receptor 1 (RyR1) Interacting Protein Partners Using Liquid Chromatography and Mass Spectrometry. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/1807/25911.

Council of Science Editors:

Ryan T. Identification of Ryanodine Receptor 1 (RyR1) Interacting Protein Partners Using Liquid Chromatography and Mass Spectrometry. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25911

17. Talbott, George C. Altered Calcium Handling in Cerebellar Purkinje Neurons with the Malignant Hyperthermia Mutation, RyR1-Y522S/+.

Degree: MS, Biological Sciences, 2011, U of Denver

  To investigate the etiology of malignant hyperthermia and central core disease, mouse models have recently been generated and characterized (Chelu et al., 2006). These… (more)

Subjects/Keywords: Calcium; Cerebellum; Malignant hyperthermia; Purkinje neurons; Ryanodine receptor; Biochemistry, Biophysics, and Structural Biology; Biology

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APA (6th Edition):

Talbott, G. C. (2011). Altered Calcium Handling in Cerebellar Purkinje Neurons with the Malignant Hyperthermia Mutation, RyR1-Y522S/+. (Thesis). U of Denver. Retrieved from https://digitalcommons.du.edu/etd/638

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Talbott, George C. “Altered Calcium Handling in Cerebellar Purkinje Neurons with the Malignant Hyperthermia Mutation, RyR1-Y522S/+.” 2011. Thesis, U of Denver. Accessed July 02, 2020. https://digitalcommons.du.edu/etd/638.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Talbott, George C. “Altered Calcium Handling in Cerebellar Purkinje Neurons with the Malignant Hyperthermia Mutation, RyR1-Y522S/+.” 2011. Web. 02 Jul 2020.

Vancouver:

Talbott GC. Altered Calcium Handling in Cerebellar Purkinje Neurons with the Malignant Hyperthermia Mutation, RyR1-Y522S/+. [Internet] [Thesis]. U of Denver; 2011. [cited 2020 Jul 02]. Available from: https://digitalcommons.du.edu/etd/638.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Talbott GC. Altered Calcium Handling in Cerebellar Purkinje Neurons with the Malignant Hyperthermia Mutation, RyR1-Y522S/+. [Thesis]. U of Denver; 2011. Available from: https://digitalcommons.du.edu/etd/638

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington University in St. Louis

18. Gaur, Namit. Calcium cycling disturbances and arrhythmogenesis.

Degree: PhD, Biomedical Engineering, 2012, Washington University in St. Louis

  In this work, a detailed multiscale computational model of Ca cycling and action potential in a ventricular myocyte is developed and used to study… (more)

Subjects/Keywords: Arrhythmia; Calcium Cycling; Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT); Hypoxia; Ryanodine Receptor; Engineering

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APA (6th Edition):

Gaur, N. (2012). Calcium cycling disturbances and arrhythmogenesis. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/eng_etds/25

Chicago Manual of Style (16th Edition):

Gaur, Namit. “Calcium cycling disturbances and arrhythmogenesis.” 2012. Doctoral Dissertation, Washington University in St. Louis. Accessed July 02, 2020. https://openscholarship.wustl.edu/eng_etds/25.

MLA Handbook (7th Edition):

Gaur, Namit. “Calcium cycling disturbances and arrhythmogenesis.” 2012. Web. 02 Jul 2020.

Vancouver:

Gaur N. Calcium cycling disturbances and arrhythmogenesis. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2012. [cited 2020 Jul 02]. Available from: https://openscholarship.wustl.edu/eng_etds/25.

Council of Science Editors:

Gaur N. Calcium cycling disturbances and arrhythmogenesis. [Doctoral Dissertation]. Washington University in St. Louis; 2012. Available from: https://openscholarship.wustl.edu/eng_etds/25


University of Oulu

19. Anttila, K. (Katja). Swimming muscles of wild, trained and reared fish:aspects of contraction machinery and energy metabolism.

Degree: 2009, University of Oulu

Abstract Billions of reared fish are released to the wild to compensate e.g. for the loss of natural populations. However, the efficiency of the releases… (more)

Subjects/Keywords: dihydropyridine receptor (DHPR); ryanodine receptor (RyR); survival; swimming capacity; dihydropyridiini reseptori (DHPR); ryanodiini reseptori (RyR); selviytyminen; uintikapasiteetti

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APA (6th Edition):

Anttila, K. (. (2009). Swimming muscles of wild, trained and reared fish:aspects of contraction machinery and energy metabolism. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514290770

Chicago Manual of Style (16th Edition):

Anttila, K (Katja). “Swimming muscles of wild, trained and reared fish:aspects of contraction machinery and energy metabolism.” 2009. Doctoral Dissertation, University of Oulu. Accessed July 02, 2020. http://urn.fi/urn:isbn:9789514290770.

MLA Handbook (7th Edition):

Anttila, K (Katja). “Swimming muscles of wild, trained and reared fish:aspects of contraction machinery and energy metabolism.” 2009. Web. 02 Jul 2020.

Vancouver:

Anttila K(. Swimming muscles of wild, trained and reared fish:aspects of contraction machinery and energy metabolism. [Internet] [Doctoral dissertation]. University of Oulu; 2009. [cited 2020 Jul 02]. Available from: http://urn.fi/urn:isbn:9789514290770.

Council of Science Editors:

Anttila K(. Swimming muscles of wild, trained and reared fish:aspects of contraction machinery and energy metabolism. [Doctoral Dissertation]. University of Oulu; 2009. Available from: http://urn.fi/urn:isbn:9789514290770


Kyoto University

20. Kato, Kenta. Characterization of bioactive molecules using genetically engineered ion channels .

Degree: 2010, Kyoto University

Subjects/Keywords: Ca2+ channel; ryanodine receptor; transient receptor potential; STIM1

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APA (6th Edition):

Kato, K. (2010). Characterization of bioactive molecules using genetically engineered ion channels . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/120897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kato, Kenta. “Characterization of bioactive molecules using genetically engineered ion channels .” 2010. Thesis, Kyoto University. Accessed July 02, 2020. http://hdl.handle.net/2433/120897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kato, Kenta. “Characterization of bioactive molecules using genetically engineered ion channels .” 2010. Web. 02 Jul 2020.

Vancouver:

Kato K. Characterization of bioactive molecules using genetically engineered ion channels . [Internet] [Thesis]. Kyoto University; 2010. [cited 2020 Jul 02]. Available from: http://hdl.handle.net/2433/120897.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kato K. Characterization of bioactive molecules using genetically engineered ion channels . [Thesis]. Kyoto University; 2010. Available from: http://hdl.handle.net/2433/120897

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Lefebvre, Romain. Perturbations de l'efflux calcique du réticulum dans la fibre musculaire squelettique de mammifère par l'expression de récepteurs de la ryanodine pathologiques et par certains phophoinositides : Alterations of sarcoplasmic reticulum calcium release by expression of pathological mutant ryanodine receptors and by phophoinositides in mammalian skeletal muscle fibers.

Degree: Docteur es, Physiologie, 2012, Université Claude Bernard – Lyon I

Les ions Ca2+ responsables de la contraction musculaire sont extrudés du réticulum sarcoplasmique (RS) via le récepteur de la ryanodine de type 1 (RyR1). Des… (more)

Subjects/Keywords: Muscle squelettique; Récepteur de la ryanodine; Calcium intracellulaire; Hyperthermie maligne; Myopathie à cores centraux; Phosphoinositides; Skeletal striated muscle; Ryanodine receptor; Intracellular calcium; Malignant hyperthermia; Central core disease; Phosphoinositides; 612.74

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APA (6th Edition):

Lefebvre, R. (2012). Perturbations de l'efflux calcique du réticulum dans la fibre musculaire squelettique de mammifère par l'expression de récepteurs de la ryanodine pathologiques et par certains phophoinositides : Alterations of sarcoplasmic reticulum calcium release by expression of pathological mutant ryanodine receptors and by phophoinositides in mammalian skeletal muscle fibers. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2012LYO10151

Chicago Manual of Style (16th Edition):

Lefebvre, Romain. “Perturbations de l'efflux calcique du réticulum dans la fibre musculaire squelettique de mammifère par l'expression de récepteurs de la ryanodine pathologiques et par certains phophoinositides : Alterations of sarcoplasmic reticulum calcium release by expression of pathological mutant ryanodine receptors and by phophoinositides in mammalian skeletal muscle fibers.” 2012. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed July 02, 2020. http://www.theses.fr/2012LYO10151.

MLA Handbook (7th Edition):

Lefebvre, Romain. “Perturbations de l'efflux calcique du réticulum dans la fibre musculaire squelettique de mammifère par l'expression de récepteurs de la ryanodine pathologiques et par certains phophoinositides : Alterations of sarcoplasmic reticulum calcium release by expression of pathological mutant ryanodine receptors and by phophoinositides in mammalian skeletal muscle fibers.” 2012. Web. 02 Jul 2020.

Vancouver:

Lefebvre R. Perturbations de l'efflux calcique du réticulum dans la fibre musculaire squelettique de mammifère par l'expression de récepteurs de la ryanodine pathologiques et par certains phophoinositides : Alterations of sarcoplasmic reticulum calcium release by expression of pathological mutant ryanodine receptors and by phophoinositides in mammalian skeletal muscle fibers. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2012. [cited 2020 Jul 02]. Available from: http://www.theses.fr/2012LYO10151.

Council of Science Editors:

Lefebvre R. Perturbations de l'efflux calcique du réticulum dans la fibre musculaire squelettique de mammifère par l'expression de récepteurs de la ryanodine pathologiques et par certains phophoinositides : Alterations of sarcoplasmic reticulum calcium release by expression of pathological mutant ryanodine receptors and by phophoinositides in mammalian skeletal muscle fibers. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2012. Available from: http://www.theses.fr/2012LYO10151

22. Le Gall, Marion. Pathophysiologie des escarres dans le muscle squelettique : Pathophysiology of pressure ulcer in skeletal muscle.

Degree: Docteur es, Biologie Santé, 2018, Montpellier

L’escarre est une pathologie liée à l’immobilité des patients, accidentelle ou associée à des comorbidités. Les premières lésions apparaissent dans le muscle avant de se… (more)

Subjects/Keywords: Escarre; Récepteur de la Ryanodine; Paraplégie; Lésion tissulaire; Cicatrisation; Biomarqueurs; Pressure ulcer; Ryanodine receptor; Spinal cord injury; Tissue injurie; Wound healing; Biomarker

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APA (6th Edition):

Le Gall, M. (2018). Pathophysiologie des escarres dans le muscle squelettique : Pathophysiology of pressure ulcer in skeletal muscle. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2018MONTT058

Chicago Manual of Style (16th Edition):

Le Gall, Marion. “Pathophysiologie des escarres dans le muscle squelettique : Pathophysiology of pressure ulcer in skeletal muscle.” 2018. Doctoral Dissertation, Montpellier. Accessed July 02, 2020. http://www.theses.fr/2018MONTT058.

MLA Handbook (7th Edition):

Le Gall, Marion. “Pathophysiologie des escarres dans le muscle squelettique : Pathophysiology of pressure ulcer in skeletal muscle.” 2018. Web. 02 Jul 2020.

Vancouver:

Le Gall M. Pathophysiologie des escarres dans le muscle squelettique : Pathophysiology of pressure ulcer in skeletal muscle. [Internet] [Doctoral dissertation]. Montpellier; 2018. [cited 2020 Jul 02]. Available from: http://www.theses.fr/2018MONTT058.

Council of Science Editors:

Le Gall M. Pathophysiologie des escarres dans le muscle squelettique : Pathophysiology of pressure ulcer in skeletal muscle. [Doctoral Dissertation]. Montpellier; 2018. Available from: http://www.theses.fr/2018MONTT058

23. Lefkowitz, Jason J. Role of Internal Calcium Stores in Exocytosis and Neurotransmission: A Dissertation.

Degree: Neuroscience, Department of Physiology, 2010, U of Massachusetts : Med

  A central concept in the physiology of neurosecretion is that a rise in cytosolic [Ca2+] in the vicinity of plasmalemmal Ca2+ channels due to… (more)

Subjects/Keywords: Calcium Channels; Exocytosis; Chromaffin Cells; Ryanodine Receptor Calcium Release Channel; Neurosecretion; Synaptic Transmission; Biological Factors; Cells; Inorganic Chemicals; Neuroscience and Neurobiology

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APA (6th Edition):

Lefkowitz, J. J. (2010). Role of Internal Calcium Stores in Exocytosis and Neurotransmission: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/467

Chicago Manual of Style (16th Edition):

Lefkowitz, Jason J. “Role of Internal Calcium Stores in Exocytosis and Neurotransmission: A Dissertation.” 2010. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 02, 2020. https://escholarship.umassmed.edu/gsbs_diss/467.

MLA Handbook (7th Edition):

Lefkowitz, Jason J. “Role of Internal Calcium Stores in Exocytosis and Neurotransmission: A Dissertation.” 2010. Web. 02 Jul 2020.

Vancouver:

Lefkowitz JJ. Role of Internal Calcium Stores in Exocytosis and Neurotransmission: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2010. [cited 2020 Jul 02]. Available from: https://escholarship.umassmed.edu/gsbs_diss/467.

Council of Science Editors:

Lefkowitz JJ. Role of Internal Calcium Stores in Exocytosis and Neurotransmission: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2010. Available from: https://escholarship.umassmed.edu/gsbs_diss/467


University of Manchester

24. Whiteley, Gareth. Molecular architecture of Caveolin-3 and the investigation of an interaction with the Ryanodine receptor.

Degree: 2012, University of Manchester

The muscle-specific membrane protein, Caveolin-3, is a building block of caveolae a type of specialised lipid raft. Caveolin-3 is proposed to play a central role… (more)

Subjects/Keywords: Cav3; Cav-3; Caveolin-3; Caveolin3; RyR; RyR1; Ryanodine Receptor; TEM; Transmission Electron Microscopy; Cardiac; Protein; Structure

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APA (6th Edition):

Whiteley, G. (2012). Molecular architecture of Caveolin-3 and the investigation of an interaction with the Ryanodine receptor. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:175631

Chicago Manual of Style (16th Edition):

Whiteley, Gareth. “Molecular architecture of Caveolin-3 and the investigation of an interaction with the Ryanodine receptor.” 2012. Doctoral Dissertation, University of Manchester. Accessed July 02, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:175631.

MLA Handbook (7th Edition):

Whiteley, Gareth. “Molecular architecture of Caveolin-3 and the investigation of an interaction with the Ryanodine receptor.” 2012. Web. 02 Jul 2020.

Vancouver:

Whiteley G. Molecular architecture of Caveolin-3 and the investigation of an interaction with the Ryanodine receptor. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Jul 02]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:175631.

Council of Science Editors:

Whiteley G. Molecular architecture of Caveolin-3 and the investigation of an interaction with the Ryanodine receptor. [Doctoral Dissertation]. University of Manchester; 2012. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:175631


Freie Universität Berlin

25. Theis, Anne-Kathrin. Ryanodinrezeptoraktivierung induziert Langzeitplastizität von Kalziumsignalen in Dornfortsätzen.

Degree: 2016, Freie Universität Berlin

 Die meisten erregenden Synapsen befinden sich auf den sogenannten Dornfortsätzen, kleinen Vorstülpungen der neuronalen Membran [1, 2]. Neben den synaptischen Eingängen erhalten diese Dornfortsätze auch… (more)

Subjects/Keywords: ryanodine receptor; calcium signaling; backpropagating action potential; dendritic spines; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Theis, A. (2016). Ryanodinrezeptoraktivierung induziert Langzeitplastizität von Kalziumsignalen in Dornfortsätzen. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-7611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Theis, Anne-Kathrin. “Ryanodinrezeptoraktivierung induziert Langzeitplastizität von Kalziumsignalen in Dornfortsätzen.” 2016. Thesis, Freie Universität Berlin. Accessed July 02, 2020. http://dx.doi.org/10.17169/refubium-7611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Theis, Anne-Kathrin. “Ryanodinrezeptoraktivierung induziert Langzeitplastizität von Kalziumsignalen in Dornfortsätzen.” 2016. Web. 02 Jul 2020.

Vancouver:

Theis A. Ryanodinrezeptoraktivierung induziert Langzeitplastizität von Kalziumsignalen in Dornfortsätzen. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2020 Jul 02]. Available from: http://dx.doi.org/10.17169/refubium-7611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Theis A. Ryanodinrezeptoraktivierung induziert Langzeitplastizität von Kalziumsignalen in Dornfortsätzen. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-7611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

26. Saravanan, Palaniappan. Effect of omega-3 fatty acids on atrial fibrillation following coronary artery bypass surgery and cardiac calcium handling in humans.

Degree: PhD, 2011, University of Manchester

 Omega 3 poly unsaturated fatty acids (n-3 PUFA) have been shown to protect against sudden cardiac death following myocardial infarction and reduce the risk of… (more)

Subjects/Keywords: 616.1; Omega-3 fatty acids; Atrial fibrillation; Coronary by-pass surgery; Calcium handling; Ryanodine receptor; Phospholamban; Sudden cardiac death

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APA (6th Edition):

Saravanan, P. (2011). Effect of omega-3 fatty acids on atrial fibrillation following coronary artery bypass surgery and cardiac calcium handling in humans. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/effect-of-omega3-fatty-acids-on-atrial-fibrillation-following-coronary-artery-bypass-surgery-and-cardiac-calcium-handling-in-humans(660e7236-f91d-4512-b3a4-822360b02378).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.549047

Chicago Manual of Style (16th Edition):

Saravanan, Palaniappan. “Effect of omega-3 fatty acids on atrial fibrillation following coronary artery bypass surgery and cardiac calcium handling in humans.” 2011. Doctoral Dissertation, University of Manchester. Accessed July 02, 2020. https://www.research.manchester.ac.uk/portal/en/theses/effect-of-omega3-fatty-acids-on-atrial-fibrillation-following-coronary-artery-bypass-surgery-and-cardiac-calcium-handling-in-humans(660e7236-f91d-4512-b3a4-822360b02378).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.549047.

MLA Handbook (7th Edition):

Saravanan, Palaniappan. “Effect of omega-3 fatty acids on atrial fibrillation following coronary artery bypass surgery and cardiac calcium handling in humans.” 2011. Web. 02 Jul 2020.

Vancouver:

Saravanan P. Effect of omega-3 fatty acids on atrial fibrillation following coronary artery bypass surgery and cardiac calcium handling in humans. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Jul 02]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/effect-of-omega3-fatty-acids-on-atrial-fibrillation-following-coronary-artery-bypass-surgery-and-cardiac-calcium-handling-in-humans(660e7236-f91d-4512-b3a4-822360b02378).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.549047.

Council of Science Editors:

Saravanan P. Effect of omega-3 fatty acids on atrial fibrillation following coronary artery bypass surgery and cardiac calcium handling in humans. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/effect-of-omega3-fatty-acids-on-atrial-fibrillation-following-coronary-artery-bypass-surgery-and-cardiac-calcium-handling-in-humans(660e7236-f91d-4512-b3a4-822360b02378).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.549047


University of Manchester

27. Whiteley, Gareth. Molecular architecture of Caveolin-3 and the investigation of an interaction with the ryanodine receptor.

Degree: PhD, 2012, University of Manchester

 The muscle-specific membrane protein, Caveolin-3, is a building block of caveolae a type of specialised lipid raft. Caveolin-3 is proposed to play a central role… (more)

Subjects/Keywords: 612.3; TEM; Structure; Cardiac; Transmission Electron Microscopy; Ryanodine Receptor; Protein; RyR; Caveolin3; Caveolin-3; Cav-3; RyR1; Cav3

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Whiteley, G. (2012). Molecular architecture of Caveolin-3 and the investigation of an interaction with the ryanodine receptor. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/molecular-architecture-of-caveolin3-and-the-investigation-of-an-interaction-with-the-ryanodine-receptor(d5d4e1f1-88c5-4619-b208-7742d0cd81f5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713533

Chicago Manual of Style (16th Edition):

Whiteley, Gareth. “Molecular architecture of Caveolin-3 and the investigation of an interaction with the ryanodine receptor.” 2012. Doctoral Dissertation, University of Manchester. Accessed July 02, 2020. https://www.research.manchester.ac.uk/portal/en/theses/molecular-architecture-of-caveolin3-and-the-investigation-of-an-interaction-with-the-ryanodine-receptor(d5d4e1f1-88c5-4619-b208-7742d0cd81f5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713533.

MLA Handbook (7th Edition):

Whiteley, Gareth. “Molecular architecture of Caveolin-3 and the investigation of an interaction with the ryanodine receptor.” 2012. Web. 02 Jul 2020.

Vancouver:

Whiteley G. Molecular architecture of Caveolin-3 and the investigation of an interaction with the ryanodine receptor. [Internet] [Doctoral dissertation]. University of Manchester; 2012. [cited 2020 Jul 02]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/molecular-architecture-of-caveolin3-and-the-investigation-of-an-interaction-with-the-ryanodine-receptor(d5d4e1f1-88c5-4619-b208-7742d0cd81f5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713533.

Council of Science Editors:

Whiteley G. Molecular architecture of Caveolin-3 and the investigation of an interaction with the ryanodine receptor. [Doctoral Dissertation]. University of Manchester; 2012. Available from: https://www.research.manchester.ac.uk/portal/en/theses/molecular-architecture-of-caveolin3-and-the-investigation-of-an-interaction-with-the-ryanodine-receptor(d5d4e1f1-88c5-4619-b208-7742d0cd81f5).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.713533


Virginia Commonwealth University

28. Nguyen, Minh. The Role of CaMK-II in Skeletal Muscle Function and Swimming Behavior in Zebrafish.

Degree: MS, Biology, 2013, Virginia Commonwealth University

 Previous research showed mutations in muscle sarcoplasmic reticulum-bound calcium handler proteins cause swimming defects in embryonic zebrafish. CaMK-II is a highly conserved Ca2+/calmodulin-dependent protein kinase… (more)

Subjects/Keywords: skeletal muscle; CaMK-II; zebrafish development; ryanodine receptor; phospholamban; developmental biology; zebrafish swimming; Biology; Life Sciences

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APA (6th Edition):

Nguyen, M. (2013). The Role of CaMK-II in Skeletal Muscle Function and Swimming Behavior in Zebrafish. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/2HR2-SD83 ; https://scholarscompass.vcu.edu/etd/3046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Minh. “The Role of CaMK-II in Skeletal Muscle Function and Swimming Behavior in Zebrafish.” 2013. Thesis, Virginia Commonwealth University. Accessed July 02, 2020. https://doi.org/10.25772/2HR2-SD83 ; https://scholarscompass.vcu.edu/etd/3046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Minh. “The Role of CaMK-II in Skeletal Muscle Function and Swimming Behavior in Zebrafish.” 2013. Web. 02 Jul 2020.

Vancouver:

Nguyen M. The Role of CaMK-II in Skeletal Muscle Function and Swimming Behavior in Zebrafish. [Internet] [Thesis]. Virginia Commonwealth University; 2013. [cited 2020 Jul 02]. Available from: https://doi.org/10.25772/2HR2-SD83 ; https://scholarscompass.vcu.edu/etd/3046.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen M. The Role of CaMK-II in Skeletal Muscle Function and Swimming Behavior in Zebrafish. [Thesis]. Virginia Commonwealth University; 2013. Available from: https://doi.org/10.25772/2HR2-SD83 ; https://scholarscompass.vcu.edu/etd/3046

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Sanchez, Colline. Biosenseurs fluorescents appliqués à l’étude de la fonction du réticulum sarcoplasmique dans le couplage excitation-contraction du muscle squelettique : Investigating sarcoplasmic reticulum function during skeletal muscle excitation-contraction coupling using fluorescent biosensors.

Degree: Docteur es, Physiologie, 2019, Lyon

 La cascade d’évènements permettant la contraction de la fibre musculaire striée squelettique en réponse à l’activité électrique de sa membrane plasmique est regroupée sous le… (more)

Subjects/Keywords: Muscle squelettique; Couplage excitation-contraction; Réticulum sarcoplasmique; Récepteur de la ryanodine; Biosenseurs fluorescents; Potentiel de membrane; Calcium intracellulaire; Skeletal muscle; Excitation-contraction coupling; Sarcoplasmic reticulum; Ryanodine receptor; Fluorescent biosensors; Membrane voltage; Intracellular calcium; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sanchez, C. (2019). Biosenseurs fluorescents appliqués à l’étude de la fonction du réticulum sarcoplasmique dans le couplage excitation-contraction du muscle squelettique : Investigating sarcoplasmic reticulum function during skeletal muscle excitation-contraction coupling using fluorescent biosensors. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2019LYSE1164

Chicago Manual of Style (16th Edition):

Sanchez, Colline. “Biosenseurs fluorescents appliqués à l’étude de la fonction du réticulum sarcoplasmique dans le couplage excitation-contraction du muscle squelettique : Investigating sarcoplasmic reticulum function during skeletal muscle excitation-contraction coupling using fluorescent biosensors.” 2019. Doctoral Dissertation, Lyon. Accessed July 02, 2020. http://www.theses.fr/2019LYSE1164.

MLA Handbook (7th Edition):

Sanchez, Colline. “Biosenseurs fluorescents appliqués à l’étude de la fonction du réticulum sarcoplasmique dans le couplage excitation-contraction du muscle squelettique : Investigating sarcoplasmic reticulum function during skeletal muscle excitation-contraction coupling using fluorescent biosensors.” 2019. Web. 02 Jul 2020.

Vancouver:

Sanchez C. Biosenseurs fluorescents appliqués à l’étude de la fonction du réticulum sarcoplasmique dans le couplage excitation-contraction du muscle squelettique : Investigating sarcoplasmic reticulum function during skeletal muscle excitation-contraction coupling using fluorescent biosensors. [Internet] [Doctoral dissertation]. Lyon; 2019. [cited 2020 Jul 02]. Available from: http://www.theses.fr/2019LYSE1164.

Council of Science Editors:

Sanchez C. Biosenseurs fluorescents appliqués à l’étude de la fonction du réticulum sarcoplasmique dans le couplage excitation-contraction du muscle squelettique : Investigating sarcoplasmic reticulum function during skeletal muscle excitation-contraction coupling using fluorescent biosensors. [Doctoral Dissertation]. Lyon; 2019. Available from: http://www.theses.fr/2019LYSE1164


Université de Grenoble

30. Cacheux, Marine. Effets fonctionnels de mutations de gènes codant des protéines du complexe de relâchement du calcium impliqués dans les pathologies du muscle strié : Mutations of calcium release complex proteins in squeletal and cardiac muscles.

Degree: Docteur es, Biologie cellulaire, 2012, Université de Grenoble

La contraction des muscles striés est sous la dépendance du Complexe de Relâchement du Calcium (CRC). Ce complexe protéique est constitué principalement de deux canaux… (more)

Subjects/Keywords: Récepteur de la ryanodine; Triadine; Complexe de mobilisation du calcium; Contraction musculaire; Myopathies à cores; Tachycardie ventriculaire catecholergique; Ryanodine receptor; Triadin; Calcium release complex; Muscle contraction; Structuralmyopathies with cores; Catecholergic ventricular tachycardy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cacheux, M. (2012). Effets fonctionnels de mutations de gènes codant des protéines du complexe de relâchement du calcium impliqués dans les pathologies du muscle strié : Mutations of calcium release complex proteins in squeletal and cardiac muscles. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2012GRENV075

Chicago Manual of Style (16th Edition):

Cacheux, Marine. “Effets fonctionnels de mutations de gènes codant des protéines du complexe de relâchement du calcium impliqués dans les pathologies du muscle strié : Mutations of calcium release complex proteins in squeletal and cardiac muscles.” 2012. Doctoral Dissertation, Université de Grenoble. Accessed July 02, 2020. http://www.theses.fr/2012GRENV075.

MLA Handbook (7th Edition):

Cacheux, Marine. “Effets fonctionnels de mutations de gènes codant des protéines du complexe de relâchement du calcium impliqués dans les pathologies du muscle strié : Mutations of calcium release complex proteins in squeletal and cardiac muscles.” 2012. Web. 02 Jul 2020.

Vancouver:

Cacheux M. Effets fonctionnels de mutations de gènes codant des protéines du complexe de relâchement du calcium impliqués dans les pathologies du muscle strié : Mutations of calcium release complex proteins in squeletal and cardiac muscles. [Internet] [Doctoral dissertation]. Université de Grenoble; 2012. [cited 2020 Jul 02]. Available from: http://www.theses.fr/2012GRENV075.

Council of Science Editors:

Cacheux M. Effets fonctionnels de mutations de gènes codant des protéines du complexe de relâchement du calcium impliqués dans les pathologies du muscle strié : Mutations of calcium release complex proteins in squeletal and cardiac muscles. [Doctoral Dissertation]. Université de Grenoble; 2012. Available from: http://www.theses.fr/2012GRENV075

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