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You searched for subject:(replication stress). Showing records 1 – 30 of 75 total matches.

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University of Toronto

1. Gallo, David. Rad5 Recruitment and Function in the Saccharomyces cerevisiae Replication Stress Response.

Degree: PhD, 2018, University of Toronto

 DNA replication stress poses a significant threat to the genome integrity of actively dividing cells and is observed in many pathological human disorders. Replication stress(more)

Subjects/Keywords: DNA replication; Replication stress; 0487

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APA (6th Edition):

Gallo, D. (2018). Rad5 Recruitment and Function in the Saccharomyces cerevisiae Replication Stress Response. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/91799

Chicago Manual of Style (16th Edition):

Gallo, David. “Rad5 Recruitment and Function in the Saccharomyces cerevisiae Replication Stress Response.” 2018. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/91799.

MLA Handbook (7th Edition):

Gallo, David. “Rad5 Recruitment and Function in the Saccharomyces cerevisiae Replication Stress Response.” 2018. Web. 15 Apr 2021.

Vancouver:

Gallo D. Rad5 Recruitment and Function in the Saccharomyces cerevisiae Replication Stress Response. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/91799.

Council of Science Editors:

Gallo D. Rad5 Recruitment and Function in the Saccharomyces cerevisiae Replication Stress Response. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91799


Vanderbilt University

2. Badu-Nkansah, Akosua Agyeman. Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks.

Degree: PhD, Biochemistry, 2016, Vanderbilt University

 Genomic replication is a highly challenging task. The DNA replication machinery must precisely duplicate billions of base pairs while tolerating a multitude of obstacles including… (more)

Subjects/Keywords: DNA Repair; Replication Stress; Replication Forks; DNA Replication; DNA Damage

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APA (6th Edition):

Badu-Nkansah, A. A. (2016). Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13267

Chicago Manual of Style (16th Edition):

Badu-Nkansah, Akosua Agyeman. “Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/13267.

MLA Handbook (7th Edition):

Badu-Nkansah, Akosua Agyeman. “Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks.” 2016. Web. 15 Apr 2021.

Vancouver:

Badu-Nkansah AA. Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/13267.

Council of Science Editors:

Badu-Nkansah AA. Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13267


Vanderbilt University

3. Guler, Gulfem Dilek. Human DNA helicase B in replication fork surveillance and replication stress recovery.

Degree: PhD, Biological Sciences, 2012, Vanderbilt University

 Correct and faithful genome duplication is crucial for preserving genomic integrity. Genome duplication is, therefore, highly regulated through a complex network of proteins that accomplish… (more)

Subjects/Keywords: Replication protein A; RPA; DNA helicase; DNA replication; DNA repair; replication stress; HelB; HDHB

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APA (6th Edition):

Guler, G. D. (2012). Human DNA helicase B in replication fork surveillance and replication stress recovery. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15350

Chicago Manual of Style (16th Edition):

Guler, Gulfem Dilek. “Human DNA helicase B in replication fork surveillance and replication stress recovery.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/15350.

MLA Handbook (7th Edition):

Guler, Gulfem Dilek. “Human DNA helicase B in replication fork surveillance and replication stress recovery.” 2012. Web. 15 Apr 2021.

Vancouver:

Guler GD. Human DNA helicase B in replication fork surveillance and replication stress recovery. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/15350.

Council of Science Editors:

Guler GD. Human DNA helicase B in replication fork surveillance and replication stress recovery. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/15350

4. R. Choudhary. MECHANISMS CONTROLLING THE INTEGRITY OF CONVERGING FORKS DURING REPLICATION TERMINATION.

Degree: 2018, Università degli Studi di Milano

 During S phase, natural fork pausing elements including replication termination zones (TERs) and transcribed genes, can easily lead to genotoxicity and chromosome fragility at fragile… (more)

Subjects/Keywords: Replication termination; replication; checkpoints; Top2; Genomic instability replication stress; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Choudhary, R. (2018). MECHANISMS CONTROLLING THE INTEGRITY OF CONVERGING FORKS DURING REPLICATION TERMINATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/559540

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choudhary, R.. “MECHANISMS CONTROLLING THE INTEGRITY OF CONVERGING FORKS DURING REPLICATION TERMINATION.” 2018. Thesis, Università degli Studi di Milano. Accessed April 15, 2021. http://hdl.handle.net/2434/559540.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choudhary, R.. “MECHANISMS CONTROLLING THE INTEGRITY OF CONVERGING FORKS DURING REPLICATION TERMINATION.” 2018. Web. 15 Apr 2021.

Vancouver:

Choudhary R. MECHANISMS CONTROLLING THE INTEGRITY OF CONVERGING FORKS DURING REPLICATION TERMINATION. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2434/559540.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choudhary R. MECHANISMS CONTROLLING THE INTEGRITY OF CONVERGING FORKS DURING REPLICATION TERMINATION. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/559540

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

5. Luzwick, Jessica Whitney. Regulation of the ATR Pathway in the Replication Stress Response.

Degree: PhD, Biochemistry, 2016, Vanderbilt University

 Every cell divisions cycle, over 6.8 billion base pairs of DNA must be accurately replicated. To further complicate this process, the DNA is damaged at… (more)

Subjects/Keywords: replication; ATR inhibitor; DNA damage response; replication stress; ATR; ATRIP

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APA (6th Edition):

Luzwick, J. W. (2016). Regulation of the ATR Pathway in the Replication Stress Response. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13041

Chicago Manual of Style (16th Edition):

Luzwick, Jessica Whitney. “Regulation of the ATR Pathway in the Replication Stress Response.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/13041.

MLA Handbook (7th Edition):

Luzwick, Jessica Whitney. “Regulation of the ATR Pathway in the Replication Stress Response.” 2016. Web. 15 Apr 2021.

Vancouver:

Luzwick JW. Regulation of the ATR Pathway in the Replication Stress Response. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/13041.

Council of Science Editors:

Luzwick JW. Regulation of the ATR Pathway in the Replication Stress Response. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13041


Penn State University

6. Choe, Katherine Naeun. HUWE1 Interacts with PCNA to Alleviate Replication Stress.

Degree: 2016, Penn State University

 The integrity of the genome relies on the accurate and faithful duplication of genetic information from a parental cell to its progeny during each cellular… (more)

Subjects/Keywords: DNA replication; Genomic instability; H2AX; DNA Damage; HUWE1; PCNA; Replication stress

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APA (6th Edition):

Choe, K. N. (2016). HUWE1 Interacts with PCNA to Alleviate Replication Stress. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13492kzc152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choe, Katherine Naeun. “HUWE1 Interacts with PCNA to Alleviate Replication Stress.” 2016. Thesis, Penn State University. Accessed April 15, 2021. https://submit-etda.libraries.psu.edu/catalog/13492kzc152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choe, Katherine Naeun. “HUWE1 Interacts with PCNA to Alleviate Replication Stress.” 2016. Web. 15 Apr 2021.

Vancouver:

Choe KN. HUWE1 Interacts with PCNA to Alleviate Replication Stress. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Apr 15]. Available from: https://submit-etda.libraries.psu.edu/catalog/13492kzc152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choe KN. HUWE1 Interacts with PCNA to Alleviate Replication Stress. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13492kzc152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

7. Robbins, Carol Bansbach. Identifying and defining the genome maintenance functions of SMARCAL1.

Degree: PhD, Biochemistry, 2012, Vanderbilt University

 In this dissertation I identify and define SWI/SNF, matrix-associated, actin-dependent regulator of chromatin, A-like 1 (SMARCAL1) as a genome maintenance protein. First, I introduce a… (more)

Subjects/Keywords: phosphorylation; DNA damage response; replication stress

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APA (6th Edition):

Robbins, C. B. (2012). Identifying and defining the genome maintenance functions of SMARCAL1. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12388

Chicago Manual of Style (16th Edition):

Robbins, Carol Bansbach. “Identifying and defining the genome maintenance functions of SMARCAL1.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/12388.

MLA Handbook (7th Edition):

Robbins, Carol Bansbach. “Identifying and defining the genome maintenance functions of SMARCAL1.” 2012. Web. 15 Apr 2021.

Vancouver:

Robbins CB. Identifying and defining the genome maintenance functions of SMARCAL1. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/12388.

Council of Science Editors:

Robbins CB. Identifying and defining the genome maintenance functions of SMARCAL1. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/12388


Penn State University

8. Barnes, Ryan Patrick. The Roles and Regulation of Specialized DNA Polymerases in Mitigating Replication Stress and Replicating Common Fragile Sites.

Degree: 2017, Penn State University

 Replicative DNA polymerases serve as the essential enzymes that duplicate our genome with high fidelity and efficiency. This function is compromised however, when repetitive DNA… (more)

Subjects/Keywords: DNA polymerase; replication stress; genome stability

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APA (6th Edition):

Barnes, R. P. (2017). The Roles and Regulation of Specialized DNA Polymerases in Mitigating Replication Stress and Replicating Common Fragile Sites. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/14528rpb180

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barnes, Ryan Patrick. “The Roles and Regulation of Specialized DNA Polymerases in Mitigating Replication Stress and Replicating Common Fragile Sites.” 2017. Thesis, Penn State University. Accessed April 15, 2021. https://submit-etda.libraries.psu.edu/catalog/14528rpb180.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barnes, Ryan Patrick. “The Roles and Regulation of Specialized DNA Polymerases in Mitigating Replication Stress and Replicating Common Fragile Sites.” 2017. Web. 15 Apr 2021.

Vancouver:

Barnes RP. The Roles and Regulation of Specialized DNA Polymerases in Mitigating Replication Stress and Replicating Common Fragile Sites. [Internet] [Thesis]. Penn State University; 2017. [cited 2021 Apr 15]. Available from: https://submit-etda.libraries.psu.edu/catalog/14528rpb180.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barnes RP. The Roles and Regulation of Specialized DNA Polymerases in Mitigating Replication Stress and Replicating Common Fragile Sites. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/14528rpb180

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

9. Cox, Kelli. Replication stress and the alternative lengthening of telomeres pathway.

Degree: PhD, Pharmacology, 2016, Boston University

 In an effort to achieve replicative immortality, human cancer cells must avoid the constant telomere attrition that accompanies DNA replication. Cancer cells accomplish this by… (more)

Subjects/Keywords: Cellular biology; ALT; Cancer; Replication stress; Telomere

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APA (6th Edition):

Cox, K. (2016). Replication stress and the alternative lengthening of telomeres pathway. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/16743

Chicago Manual of Style (16th Edition):

Cox, Kelli. “Replication stress and the alternative lengthening of telomeres pathway.” 2016. Doctoral Dissertation, Boston University. Accessed April 15, 2021. http://hdl.handle.net/2144/16743.

MLA Handbook (7th Edition):

Cox, Kelli. “Replication stress and the alternative lengthening of telomeres pathway.” 2016. Web. 15 Apr 2021.

Vancouver:

Cox K. Replication stress and the alternative lengthening of telomeres pathway. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2144/16743.

Council of Science Editors:

Cox K. Replication stress and the alternative lengthening of telomeres pathway. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16743


Vanderbilt University

10. Bhat, Kamakoti Prakash. Regulation of replication fork stability by ssDNA binding proteins.

Degree: PhD, Biochemistry, 2018, Vanderbilt University

 The replication stress response (RSR) maintains genome stability and promotes the accurate duplication of the genome. ssDNA binding proteins are integral components of the RSR… (more)

Subjects/Keywords: fork remodeling; fork protection; genome stability; DNA repair; DNA replication; replication stress response

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APA (6th Edition):

Bhat, K. P. (2018). Regulation of replication fork stability by ssDNA binding proteins. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10626

Chicago Manual of Style (16th Edition):

Bhat, Kamakoti Prakash. “Regulation of replication fork stability by ssDNA binding proteins.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/10626.

MLA Handbook (7th Edition):

Bhat, Kamakoti Prakash. “Regulation of replication fork stability by ssDNA binding proteins.” 2018. Web. 15 Apr 2021.

Vancouver:

Bhat KP. Regulation of replication fork stability by ssDNA binding proteins. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/10626.

Council of Science Editors:

Bhat KP. Regulation of replication fork stability by ssDNA binding proteins. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/10626


Vanderbilt University

11. Couch, Frank Benjamin IV. Regulation of Stalled Replication Forks by ATR.

Degree: PhD, Biochemistry, 2014, Vanderbilt University

 Errors during DNA replication lead to mutations which contribute to cancer development. To deal with these challenges, cells contain an innate machinery known as the… (more)

Subjects/Keywords: cell cycle checkpoint; SMARCAL1; replication stress; DNA replication; DNA damage response; ATR

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APA (6th Edition):

Couch, F. B. I. (2014). Regulation of Stalled Replication Forks by ATR. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11323

Chicago Manual of Style (16th Edition):

Couch, Frank Benjamin IV. “Regulation of Stalled Replication Forks by ATR.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/11323.

MLA Handbook (7th Edition):

Couch, Frank Benjamin IV. “Regulation of Stalled Replication Forks by ATR.” 2014. Web. 15 Apr 2021.

Vancouver:

Couch FBI. Regulation of Stalled Replication Forks by ATR. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/11323.

Council of Science Editors:

Couch FBI. Regulation of Stalled Replication Forks by ATR. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/11323

12. S.E. Rossi. INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY.

Degree: 2017, Università degli Studi di Milano

 Eukaryotic cells have evolved the ATR/hCHK1, MEC1/RAD53 kinase-mediated signal transduction pathway, known as replication checkpoint, to protect and stabilize stalled replication forks in human cells… (more)

Subjects/Keywords: Replication stress; replication fork; checkpoint; Rad53; Rrm3; Pif1; Dna2; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Rossi, S. (2017). INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/471797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rossi, S.E.. “INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY.” 2017. Thesis, Università degli Studi di Milano. Accessed April 15, 2021. http://hdl.handle.net/2434/471797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rossi, S.E.. “INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY.” 2017. Web. 15 Apr 2021.

Vancouver:

Rossi S. INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2434/471797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rossi S. INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/471797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Matmati, Samah. "Rôle du complexe Stn1-Ten1 et de la télomérase dans le contrôle du stress réplicatif aux télomères chez la levure Schizosaccharomyces pombe" : Stn1-Ten1 complex and telomerase limit replication stress at telomeres in Schizosaccharomyces pombe.

Degree: Docteur es, Pathologie humaine. Oncologie, 2019, Aix Marseille Université

Les télomères sont des structures nucléoprotéiques qui protègent l’extrémité des chromosomes et assurent leur réplication. Ils sont constitués de séquences répétées, qui raccourcissent progressivement à… (more)

Subjects/Keywords: Télomères; Stress réplicatif; Télomérase; Réplication de l’ADN; Sénescence; Levure fissipare; Telomeres; Replication stress; Telomerase; DNA Replication; Senescence; Fission yeast

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APA (6th Edition):

Matmati, S. (2019). "Rôle du complexe Stn1-Ten1 et de la télomérase dans le contrôle du stress réplicatif aux télomères chez la levure Schizosaccharomyces pombe" : Stn1-Ten1 complex and telomerase limit replication stress at telomeres in Schizosaccharomyces pombe. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2019AIXM0598

Chicago Manual of Style (16th Edition):

Matmati, Samah. “"Rôle du complexe Stn1-Ten1 et de la télomérase dans le contrôle du stress réplicatif aux télomères chez la levure Schizosaccharomyces pombe" : Stn1-Ten1 complex and telomerase limit replication stress at telomeres in Schizosaccharomyces pombe.” 2019. Doctoral Dissertation, Aix Marseille Université. Accessed April 15, 2021. http://www.theses.fr/2019AIXM0598.

MLA Handbook (7th Edition):

Matmati, Samah. “"Rôle du complexe Stn1-Ten1 et de la télomérase dans le contrôle du stress réplicatif aux télomères chez la levure Schizosaccharomyces pombe" : Stn1-Ten1 complex and telomerase limit replication stress at telomeres in Schizosaccharomyces pombe.” 2019. Web. 15 Apr 2021.

Vancouver:

Matmati S. "Rôle du complexe Stn1-Ten1 et de la télomérase dans le contrôle du stress réplicatif aux télomères chez la levure Schizosaccharomyces pombe" : Stn1-Ten1 complex and telomerase limit replication stress at telomeres in Schizosaccharomyces pombe. [Internet] [Doctoral dissertation]. Aix Marseille Université 2019. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2019AIXM0598.

Council of Science Editors:

Matmati S. "Rôle du complexe Stn1-Ten1 et de la télomérase dans le contrôle du stress réplicatif aux télomères chez la levure Schizosaccharomyces pombe" : Stn1-Ten1 complex and telomerase limit replication stress at telomeres in Schizosaccharomyces pombe. [Doctoral Dissertation]. Aix Marseille Université 2019. Available from: http://www.theses.fr/2019AIXM0598

14. Ait Saada, Anissia. Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Université Paris-Saclay (ComUE)

Des stress réplicatifs sont rencontrés à chaque phase S du cycle cellulaire et différents mécanismes permettent leur prise en charge. La recombinaison homologue (RH) tient… (more)

Subjects/Keywords: Recombinaison homologue; Réplication; Défauts mitotiques; Stress Réplicatif; Protection des fourches de réplication; Homologous recombination; Replication; Mitotic defects; Replication Stress; Fork Protection

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APA (6th Edition):

Ait Saada, A. (2018). Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS167

Chicago Manual of Style (16th Edition):

Ait Saada, Anissia. “Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 15, 2021. http://www.theses.fr/2018SACLS167.

MLA Handbook (7th Edition):

Ait Saada, Anissia. “Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress.” 2018. Web. 15 Apr 2021.

Vancouver:

Ait Saada A. Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2018SACLS167.

Council of Science Editors:

Ait Saada A. Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS167

15. Guitton-Sert, Laure. Identification de nouveaux mécanismes de régulation temporelle des origines de réplication dans les cellules humaines : Identification of new mechanisms of temporal regulation of DNA replication origins in human cells.

Degree: Docteur es, Cancérologie, 2015, Université Toulouse III – Paul Sabatier

La duplication de l'ADN au cours de la phase S est initiée à partir de l'activation de plusieurs dizaines de milliers d'origines de réplication. La… (more)

Subjects/Keywords: Réplication de l'ADN; Timing de réplication; Origines de réplication; ADN polymérase Thêta; Stress réplicatif; DNA replication; Replication timing; DNA replication origins; DNA polymerase Theta; Replicative stress

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APA (6th Edition):

Guitton-Sert, L. (2015). Identification de nouveaux mécanismes de régulation temporelle des origines de réplication dans les cellules humaines : Identification of new mechanisms of temporal regulation of DNA replication origins in human cells. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2015TOU30350

Chicago Manual of Style (16th Edition):

Guitton-Sert, Laure. “Identification de nouveaux mécanismes de régulation temporelle des origines de réplication dans les cellules humaines : Identification of new mechanisms of temporal regulation of DNA replication origins in human cells.” 2015. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed April 15, 2021. http://www.theses.fr/2015TOU30350.

MLA Handbook (7th Edition):

Guitton-Sert, Laure. “Identification de nouveaux mécanismes de régulation temporelle des origines de réplication dans les cellules humaines : Identification of new mechanisms of temporal regulation of DNA replication origins in human cells.” 2015. Web. 15 Apr 2021.

Vancouver:

Guitton-Sert L. Identification de nouveaux mécanismes de régulation temporelle des origines de réplication dans les cellules humaines : Identification of new mechanisms of temporal regulation of DNA replication origins in human cells. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2015. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2015TOU30350.

Council of Science Editors:

Guitton-Sert L. Identification de nouveaux mécanismes de régulation temporelle des origines de réplication dans les cellules humaines : Identification of new mechanisms of temporal regulation of DNA replication origins in human cells. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2015. Available from: http://www.theses.fr/2015TOU30350


Université Paris-Sud – Paris XI

16. Wilhelm, Therese. Homologous recombination protects against mitotic defects and unbalanced chromosome segregation caused by spontaneous replication stress : Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée.

Degree: Docteur es, Biologie, 2011, Université Paris-Sud – Paris XI

 Les cellules déficientes pour la recombinaison homologue (RH) présentent un ralentissement des fourches de réplication, un nombre aberrant de centrosomes et une aneuploïdie même en… (more)

Subjects/Keywords: RECOMBINAISON HOMOLOGUE; STRESS DE REPLICATION; DEFECTS DE CENTROSOME; DEFECTS DE SEGREGATION DE CHROMOSOME; STRESS OXIDATIVE; HOMOLOGOUS RECOMBINATION; REPLICATION STRESS; CENTROSOME DEFECTS; CHROMOSOME SEGREGATION DEFECTS; OXIDATIVE STRESS

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APA (6th Edition):

Wilhelm, T. (2011). Homologous recombination protects against mitotic defects and unbalanced chromosome segregation caused by spontaneous replication stress : Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA112059

Chicago Manual of Style (16th Edition):

Wilhelm, Therese. “Homologous recombination protects against mitotic defects and unbalanced chromosome segregation caused by spontaneous replication stress : Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 15, 2021. http://www.theses.fr/2011PA112059.

MLA Handbook (7th Edition):

Wilhelm, Therese. “Homologous recombination protects against mitotic defects and unbalanced chromosome segregation caused by spontaneous replication stress : Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée.” 2011. Web. 15 Apr 2021.

Vancouver:

Wilhelm T. Homologous recombination protects against mitotic defects and unbalanced chromosome segregation caused by spontaneous replication stress : Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2011PA112059.

Council of Science Editors:

Wilhelm T. Homologous recombination protects against mitotic defects and unbalanced chromosome segregation caused by spontaneous replication stress : Recombinaison homologue protège contre les défauts de la mitose et la ségrégation des chromosomes déséquilibre causé par le stress de réplication spontanée. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA112059


Vanderbilt University

17. Chavez, Diana Andrea. Replication Fork Remodeling by Helicase-Like Transcription Factor.

Degree: PhD, Biological Sciences, 2018, Vanderbilt University

 During DNA replication, a moving replication fork can encounter various sources of replication stress that can lead the moving fork to stall. Organisms have developed… (more)

Subjects/Keywords: DNA repair; fork remodeling; HIRAN; replication stress; HLTF; x-ray crystallography

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APA (6th Edition):

Chavez, D. A. (2018). Replication Fork Remodeling by Helicase-Like Transcription Factor. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12697

Chicago Manual of Style (16th Edition):

Chavez, Diana Andrea. “Replication Fork Remodeling by Helicase-Like Transcription Factor.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/12697.

MLA Handbook (7th Edition):

Chavez, Diana Andrea. “Replication Fork Remodeling by Helicase-Like Transcription Factor.” 2018. Web. 15 Apr 2021.

Vancouver:

Chavez DA. Replication Fork Remodeling by Helicase-Like Transcription Factor. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/12697.

Council of Science Editors:

Chavez DA. Replication Fork Remodeling by Helicase-Like Transcription Factor. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/12697


Vanderbilt University

18. Nam, Edward Adam. Phospho-regulation of the DNA Damage Response Kinase ATR.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Understanding how cells maintain genome integrity is necessary to gain insight into the pathology of cancer and to identify therapeutic targets and biomarkers. The DNA… (more)

Subjects/Keywords: replication stress; DNA-PK; ATR; ATM; DNA damage

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APA (6th Edition):

Nam, E. A. (2011). Phospho-regulation of the DNA Damage Response Kinase ATR. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14069

Chicago Manual of Style (16th Edition):

Nam, Edward Adam. “Phospho-regulation of the DNA Damage Response Kinase ATR.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14069.

MLA Handbook (7th Edition):

Nam, Edward Adam. “Phospho-regulation of the DNA Damage Response Kinase ATR.” 2011. Web. 15 Apr 2021.

Vancouver:

Nam EA. Phospho-regulation of the DNA Damage Response Kinase ATR. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14069.

Council of Science Editors:

Nam EA. Phospho-regulation of the DNA Damage Response Kinase ATR. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14069


Vanderbilt University

19. Bass, Thomas Edwin. ETAA1 promotes genome stability through activation of ATR.

Degree: PhD, Biochemistry, 2018, Vanderbilt University

 The ATR kinase controls cell cycle transitions and the DNA damage response. Budding yeast contain three activators of Mec1ATR; however, only TOPBP1 is known to… (more)

Subjects/Keywords: ETAA1; TOPBP1; ATR activator; ATR; DNA damage; Replication stress

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APA (6th Edition):

Bass, T. E. (2018). ETAA1 promotes genome stability through activation of ATR. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14559

Chicago Manual of Style (16th Edition):

Bass, Thomas Edwin. “ETAA1 promotes genome stability through activation of ATR.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 15, 2021. http://hdl.handle.net/1803/14559.

MLA Handbook (7th Edition):

Bass, Thomas Edwin. “ETAA1 promotes genome stability through activation of ATR.” 2018. Web. 15 Apr 2021.

Vancouver:

Bass TE. ETAA1 promotes genome stability through activation of ATR. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1803/14559.

Council of Science Editors:

Bass TE. ETAA1 promotes genome stability through activation of ATR. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/14559


Universiteit Utrecht

20. Slaats, G.G.G. Looking beyond cilia in renal ciliopathies.

Degree: 2015, Universiteit Utrecht

 In this work, I have investigated the group of inherited diseases called “ciliopathies”, involving defects in proteins localizing to the cilium or associated complexes and… (more)

Subjects/Keywords: kidney; cilia; cysts; fibrosis; DNA damage; replication stress; ciliopathies; treatment

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APA (6th Edition):

Slaats, G. G. G. (2015). Looking beyond cilia in renal ciliopathies. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/318641

Chicago Manual of Style (16th Edition):

Slaats, G G G. “Looking beyond cilia in renal ciliopathies.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed April 15, 2021. http://dspace.library.uu.nl:8080/handle/1874/318641.

MLA Handbook (7th Edition):

Slaats, G G G. “Looking beyond cilia in renal ciliopathies.” 2015. Web. 15 Apr 2021.

Vancouver:

Slaats GGG. Looking beyond cilia in renal ciliopathies. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2021 Apr 15]. Available from: http://dspace.library.uu.nl:8080/handle/1874/318641.

Council of Science Editors:

Slaats GGG. Looking beyond cilia in renal ciliopathies. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/318641


University of Tennessee – Knoxville

21. Stow, Emily. Analysis of Drosophila Insulator Protein Function in Replication Timing and the Osmotic Stress Response.

Degree: 2019, University of Tennessee – Knoxville

 Chromatin insulators contribute to the three-dimensional organization of the eukaryotic genome. Insulators and their associated proteins form boundaries between differing chromatin environments, regulate enhancer-promoter interactions,… (more)

Subjects/Keywords: Insulator proteins; genome organization; DNA replication; osmotic stress; Drosophila melanogaster

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APA (6th Edition):

Stow, E. (2019). Analysis of Drosophila Insulator Protein Function in Replication Timing and the Osmotic Stress Response. (Doctoral Dissertation). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_graddiss/5612

Chicago Manual of Style (16th Edition):

Stow, Emily. “Analysis of Drosophila Insulator Protein Function in Replication Timing and the Osmotic Stress Response.” 2019. Doctoral Dissertation, University of Tennessee – Knoxville. Accessed April 15, 2021. https://trace.tennessee.edu/utk_graddiss/5612.

MLA Handbook (7th Edition):

Stow, Emily. “Analysis of Drosophila Insulator Protein Function in Replication Timing and the Osmotic Stress Response.” 2019. Web. 15 Apr 2021.

Vancouver:

Stow E. Analysis of Drosophila Insulator Protein Function in Replication Timing and the Osmotic Stress Response. [Internet] [Doctoral dissertation]. University of Tennessee – Knoxville; 2019. [cited 2021 Apr 15]. Available from: https://trace.tennessee.edu/utk_graddiss/5612.

Council of Science Editors:

Stow E. Analysis of Drosophila Insulator Protein Function in Replication Timing and the Osmotic Stress Response. [Doctoral Dissertation]. University of Tennessee – Knoxville; 2019. Available from: https://trace.tennessee.edu/utk_graddiss/5612

22. A. Ajazi. ATG6/BECLIN 1 COUPLES THE REPLICATION STRESS RESPONSE TO AMINO ACID METABOLISM.

Degree: 2017, Università degli Studi di Milano

 In the budding yeast Saccharomices Cerevisiae, Atg6 is a non-catalytic component of the phosphatidylinositol (PtdIns) kinase complex Vps34-Vps15-Atg6, which phosphorylates PtdIns to produce phosphatidylinositol 3-phosphate… (more)

Subjects/Keywords: DNA replication stress; metabolism; amino acids; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Ajazi, A. (2017). ATG6/BECLIN 1 COUPLES THE REPLICATION STRESS RESPONSE TO AMINO ACID METABOLISM. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/471447

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ajazi, A.. “ATG6/BECLIN 1 COUPLES THE REPLICATION STRESS RESPONSE TO AMINO ACID METABOLISM.” 2017. Thesis, Università degli Studi di Milano. Accessed April 15, 2021. http://hdl.handle.net/2434/471447.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ajazi, A.. “ATG6/BECLIN 1 COUPLES THE REPLICATION STRESS RESPONSE TO AMINO ACID METABOLISM.” 2017. Web. 15 Apr 2021.

Vancouver:

Ajazi A. ATG6/BECLIN 1 COUPLES THE REPLICATION STRESS RESPONSE TO AMINO ACID METABOLISM. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2434/471447.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ajazi A. ATG6/BECLIN 1 COUPLES THE REPLICATION STRESS RESPONSE TO AMINO ACID METABOLISM. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/471447

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

23. Torres, Nikko Paulo. Investigating the Cellular Response to DNA Damaging Agents through High-Resolution Phenotyping in Saccharomyces cerevisiae.

Degree: PhD, 2017, University of Toronto

 Genome maintenance is paramount for cell viability. Cells have at their disposal an intricate and versatile set of pathways that repair DNA damage and maintain… (more)

Subjects/Keywords: DNA Damage; Genome maintenance; Protein relocalization; Replication Stress; Synergy; 0487

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APA (6th Edition):

Torres, N. P. (2017). Investigating the Cellular Response to DNA Damaging Agents through High-Resolution Phenotyping in Saccharomyces cerevisiae. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/79520

Chicago Manual of Style (16th Edition):

Torres, Nikko Paulo. “Investigating the Cellular Response to DNA Damaging Agents through High-Resolution Phenotyping in Saccharomyces cerevisiae.” 2017. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/79520.

MLA Handbook (7th Edition):

Torres, Nikko Paulo. “Investigating the Cellular Response to DNA Damaging Agents through High-Resolution Phenotyping in Saccharomyces cerevisiae.” 2017. Web. 15 Apr 2021.

Vancouver:

Torres NP. Investigating the Cellular Response to DNA Damaging Agents through High-Resolution Phenotyping in Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/79520.

Council of Science Editors:

Torres NP. Investigating the Cellular Response to DNA Damaging Agents through High-Resolution Phenotyping in Saccharomyces cerevisiae. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/79520


University of Toronto

24. Balint, Attila. Assembly of Slx4 Checkpoint Signaling Complexes Behind Stressed DNA Replication Forks.

Degree: PhD, 2016, University of Toronto

 Obstructions to replication fork progression, referred to collectively as DNA replication stress, challenge genome stability. In S. cerevisiae, cells lacking RTT107 or SLX4 show genome… (more)

Subjects/Keywords: Checkpoint; ChIP-seq; Replication; Signaling; Slx4; Stress; 0487

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APA (6th Edition):

Balint, A. (2016). Assembly of Slx4 Checkpoint Signaling Complexes Behind Stressed DNA Replication Forks. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/72950

Chicago Manual of Style (16th Edition):

Balint, Attila. “Assembly of Slx4 Checkpoint Signaling Complexes Behind Stressed DNA Replication Forks.” 2016. Doctoral Dissertation, University of Toronto. Accessed April 15, 2021. http://hdl.handle.net/1807/72950.

MLA Handbook (7th Edition):

Balint, Attila. “Assembly of Slx4 Checkpoint Signaling Complexes Behind Stressed DNA Replication Forks.” 2016. Web. 15 Apr 2021.

Vancouver:

Balint A. Assembly of Slx4 Checkpoint Signaling Complexes Behind Stressed DNA Replication Forks. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1807/72950.

Council of Science Editors:

Balint A. Assembly of Slx4 Checkpoint Signaling Complexes Behind Stressed DNA Replication Forks. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72950


Cornell University

25. Shah, Pragya. CONFINED CANCER CELL MIGRATION AND ITS IMPACT ON DNA DAMAGE AND GENOMIC INSTABILITY.

Degree: PhD, Biomedical and Biological Sciences, 2020, Cornell University

 Cancer metastasis is the process by which cells from the primary tumor invade into the surrounding extracellular matrix and neighboring tissue and spread to distant… (more)

Subjects/Keywords: ATM; Cancer; Confined Migration; DNA damage; Lamins; Replication stress

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APA (6th Edition):

Shah, P. (2020). CONFINED CANCER CELL MIGRATION AND ITS IMPACT ON DNA DAMAGE AND GENOMIC INSTABILITY. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/103439

Chicago Manual of Style (16th Edition):

Shah, Pragya. “CONFINED CANCER CELL MIGRATION AND ITS IMPACT ON DNA DAMAGE AND GENOMIC INSTABILITY.” 2020. Doctoral Dissertation, Cornell University. Accessed April 15, 2021. http://hdl.handle.net/1813/103439.

MLA Handbook (7th Edition):

Shah, Pragya. “CONFINED CANCER CELL MIGRATION AND ITS IMPACT ON DNA DAMAGE AND GENOMIC INSTABILITY.” 2020. Web. 15 Apr 2021.

Vancouver:

Shah P. CONFINED CANCER CELL MIGRATION AND ITS IMPACT ON DNA DAMAGE AND GENOMIC INSTABILITY. [Internet] [Doctoral dissertation]. Cornell University; 2020. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/1813/103439.

Council of Science Editors:

Shah P. CONFINED CANCER CELL MIGRATION AND ITS IMPACT ON DNA DAMAGE AND GENOMIC INSTABILITY. [Doctoral Dissertation]. Cornell University; 2020. Available from: http://hdl.handle.net/1813/103439


Université Paris-Sud – Paris XI

26. Bellini, Angela. Rôle des voies de réponse au stress dans le maintien de la stabilité génomique chez la levure Schizosaccharomyces pombe : Role of the stress response pathway in genome stability maintenance in Schizosaccharomyces pombe yeast.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2012, Université Paris-Sud – Paris XI

Le génome est sans cesse menacé dans sa structure par des stress génotoxiques d’origine endogène (stress oxydant, blocage de la réplication…) ou exogène (irradiations, produits… (more)

Subjects/Keywords: Stress oxydant; Recombinaison; Réplication; MAPK; SAPK; Rad52; Oxidative stress; Recombination; Replication; MAPK; SAPK; Rad52

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APA (6th Edition):

Bellini, A. (2012). Rôle des voies de réponse au stress dans le maintien de la stabilité génomique chez la levure Schizosaccharomyces pombe : Role of the stress response pathway in genome stability maintenance in Schizosaccharomyces pombe yeast. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA11T048

Chicago Manual of Style (16th Edition):

Bellini, Angela. “Rôle des voies de réponse au stress dans le maintien de la stabilité génomique chez la levure Schizosaccharomyces pombe : Role of the stress response pathway in genome stability maintenance in Schizosaccharomyces pombe yeast.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 15, 2021. http://www.theses.fr/2012PA11T048.

MLA Handbook (7th Edition):

Bellini, Angela. “Rôle des voies de réponse au stress dans le maintien de la stabilité génomique chez la levure Schizosaccharomyces pombe : Role of the stress response pathway in genome stability maintenance in Schizosaccharomyces pombe yeast.” 2012. Web. 15 Apr 2021.

Vancouver:

Bellini A. Rôle des voies de réponse au stress dans le maintien de la stabilité génomique chez la levure Schizosaccharomyces pombe : Role of the stress response pathway in genome stability maintenance in Schizosaccharomyces pombe yeast. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2012PA11T048.

Council of Science Editors:

Bellini A. Rôle des voies de réponse au stress dans le maintien de la stabilité génomique chez la levure Schizosaccharomyces pombe : Role of the stress response pathway in genome stability maintenance in Schizosaccharomyces pombe yeast. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA11T048

27. Courtot, Lilas. Conséquences d'un faible stress réplicatif sur le programme de réplication des cellules normales et cancéreuses : Impact of low replication stress on the replication program of cancer and non-tumor cells.

Degree: Docteur es, Cancérologie, 2020, Université Toulouse III – Paul Sabatier

La réplication de l'ADN est un processus finement orchestré dans les cellules eucaryotes grâce à la régulation temporelle de l'activation des origines de réplication, phénomène… (more)

Subjects/Keywords: Timing de réplication; Stress réplicatif; Cancer; Transmission; Épigénétique et structure de la chromatine; Replication timing; DNA replication stress; Cancer; Transmission; Epigenetics and chromatin

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APA (6th Edition):

Courtot, L. (2020). Conséquences d'un faible stress réplicatif sur le programme de réplication des cellules normales et cancéreuses : Impact of low replication stress on the replication program of cancer and non-tumor cells. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2020TOU30150

Chicago Manual of Style (16th Edition):

Courtot, Lilas. “Conséquences d'un faible stress réplicatif sur le programme de réplication des cellules normales et cancéreuses : Impact of low replication stress on the replication program of cancer and non-tumor cells.” 2020. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed April 15, 2021. http://www.theses.fr/2020TOU30150.

MLA Handbook (7th Edition):

Courtot, Lilas. “Conséquences d'un faible stress réplicatif sur le programme de réplication des cellules normales et cancéreuses : Impact of low replication stress on the replication program of cancer and non-tumor cells.” 2020. Web. 15 Apr 2021.

Vancouver:

Courtot L. Conséquences d'un faible stress réplicatif sur le programme de réplication des cellules normales et cancéreuses : Impact of low replication stress on the replication program of cancer and non-tumor cells. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2020. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2020TOU30150.

Council of Science Editors:

Courtot L. Conséquences d'un faible stress réplicatif sur le programme de réplication des cellules normales et cancéreuses : Impact of low replication stress on the replication program of cancer and non-tumor cells. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2020. Available from: http://www.theses.fr/2020TOU30150

28. Abderahmane, Nina Farida. Rôle de l'interférence à ARN (RNAi) dans l'intégrité du génome chez la levure S. pombe : Role of interference RNA (RNAi) in DNA integrity in the yeast S. pombe.

Degree: Docteur es, Biologie cellulaire, 2020, Université Grenoble Alpes

 Préserver l’intégrité du matériel génétique est crucial pour assurer la survie cellulaire et prévenir le développement tumoral. L’ARN à interférence (RNAi) joue un rôle important… (more)

Subjects/Keywords: Interférence à ARN; Instabilité génomique; Réplication de l'ADN; Réparation de l'ADN; ADN ribosomaux; Levure; Stress réplicatif; RNA Interference; Genomic instability; DNA replication; DNA repair; Ribosomal DNA; Yeast; Replication stress; 570

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abderahmane, N. F. (2020). Rôle de l'interférence à ARN (RNAi) dans l'intégrité du génome chez la levure S. pombe : Role of interference RNA (RNAi) in DNA integrity in the yeast S. pombe. (Doctoral Dissertation). Université Grenoble Alpes. Retrieved from http://www.theses.fr/2020GRALV022

Chicago Manual of Style (16th Edition):

Abderahmane, Nina Farida. “Rôle de l'interférence à ARN (RNAi) dans l'intégrité du génome chez la levure S. pombe : Role of interference RNA (RNAi) in DNA integrity in the yeast S. pombe.” 2020. Doctoral Dissertation, Université Grenoble Alpes. Accessed April 15, 2021. http://www.theses.fr/2020GRALV022.

MLA Handbook (7th Edition):

Abderahmane, Nina Farida. “Rôle de l'interférence à ARN (RNAi) dans l'intégrité du génome chez la levure S. pombe : Role of interference RNA (RNAi) in DNA integrity in the yeast S. pombe.” 2020. Web. 15 Apr 2021.

Vancouver:

Abderahmane NF. Rôle de l'interférence à ARN (RNAi) dans l'intégrité du génome chez la levure S. pombe : Role of interference RNA (RNAi) in DNA integrity in the yeast S. pombe. [Internet] [Doctoral dissertation]. Université Grenoble Alpes; 2020. [cited 2021 Apr 15]. Available from: http://www.theses.fr/2020GRALV022.

Council of Science Editors:

Abderahmane NF. Rôle de l'interférence à ARN (RNAi) dans l'intégrité du génome chez la levure S. pombe : Role of interference RNA (RNAi) in DNA integrity in the yeast S. pombe. [Doctoral Dissertation]. Université Grenoble Alpes; 2020. Available from: http://www.theses.fr/2020GRALV022

29. Smak, Jordann. Cell Cycle-Specific Functions and Regulation of the DNA Nuclease SNM1B.

Degree: PhD, Cellular & Molecular Biology, 2019, University of Michigan

 DNA replication is a fundamental cellular process that ensures the accurate duplication and transmission of genetic information. The replication machinery is frequently challenged by endogenous… (more)

Subjects/Keywords: DNA Repair; DNA Replication Stress; SNM1B Nuclease; Molecular, Cellular and Developmental Biology; Science

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smak, J. (2019). Cell Cycle-Specific Functions and Regulation of the DNA Nuclease SNM1B. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/149866

Chicago Manual of Style (16th Edition):

Smak, Jordann. “Cell Cycle-Specific Functions and Regulation of the DNA Nuclease SNM1B.” 2019. Doctoral Dissertation, University of Michigan. Accessed April 15, 2021. http://hdl.handle.net/2027.42/149866.

MLA Handbook (7th Edition):

Smak, Jordann. “Cell Cycle-Specific Functions and Regulation of the DNA Nuclease SNM1B.” 2019. Web. 15 Apr 2021.

Vancouver:

Smak J. Cell Cycle-Specific Functions and Regulation of the DNA Nuclease SNM1B. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2021 Apr 15]. Available from: http://hdl.handle.net/2027.42/149866.

Council of Science Editors:

Smak J. Cell Cycle-Specific Functions and Regulation of the DNA Nuclease SNM1B. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/149866


University of California – San Francisco

30. Jay, Kyle Aaron. Phenotypes Occuring in Saccharomyces cerevisiae Early after Telomerase Inactivation.

Degree: Biochemistry and Molecular Biology, 2015, University of California – San Francisco

 Telomeres are DNA and protein complexes that cap the ends of linear chromosomes. These caps serve two primary functions: to buffer against the loss of… (more)

Subjects/Keywords: Biology; Molecular biology; Genetics; DNA Damage Response; Mother Cell Aging; Replication Stress; Telomerase; Telomere; TOR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jay, K. A. (2015). Phenotypes Occuring in Saccharomyces cerevisiae Early after Telomerase Inactivation. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/68q699zq

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jay, Kyle Aaron. “Phenotypes Occuring in Saccharomyces cerevisiae Early after Telomerase Inactivation.” 2015. Thesis, University of California – San Francisco. Accessed April 15, 2021. http://www.escholarship.org/uc/item/68q699zq.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jay, Kyle Aaron. “Phenotypes Occuring in Saccharomyces cerevisiae Early after Telomerase Inactivation.” 2015. Web. 15 Apr 2021.

Vancouver:

Jay KA. Phenotypes Occuring in Saccharomyces cerevisiae Early after Telomerase Inactivation. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2021 Apr 15]. Available from: http://www.escholarship.org/uc/item/68q699zq.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jay KA. Phenotypes Occuring in Saccharomyces cerevisiae Early after Telomerase Inactivation. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/68q699zq

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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