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You searched for subject:(replication fork). Showing records 1 – 30 of 30 total matches.

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Vanderbilt University

1. Dhingra, Nalini. Elucidating the role of Dpb11 in replication initiation.

Degree: PhD, Biological Sciences, 2015, Vanderbilt University

 Initiation of DNA Replication is a highly regulated process and is characterized by the conversion of inactive Mcm2-7 double hexamer around dsDNA to an active… (more)

Subjects/Keywords: initiation; replication fork; kinase; helicase; replication

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APA (6th Edition):

Dhingra, N. (2015). Elucidating the role of Dpb11 in replication initiation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07082015-230321/ ;

Chicago Manual of Style (16th Edition):

Dhingra, Nalini. “Elucidating the role of Dpb11 in replication initiation.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 20, 2020. http://etd.library.vanderbilt.edu/available/etd-07082015-230321/ ;.

MLA Handbook (7th Edition):

Dhingra, Nalini. “Elucidating the role of Dpb11 in replication initiation.” 2015. Web. 20 Jan 2020.

Vancouver:

Dhingra N. Elucidating the role of Dpb11 in replication initiation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Jan 20]. Available from: http://etd.library.vanderbilt.edu/available/etd-07082015-230321/ ;.

Council of Science Editors:

Dhingra N. Elucidating the role of Dpb11 in replication initiation. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-07082015-230321/ ;


University of Southern California

2. Ding, Lin. Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe.

Degree: PhD, Molecular Biology, 2014, University of Southern California

 A healthy cell needs to accurately duplicate its genome and pass one copy to each of its daughter cells. The DNA double helix is accessed… (more)

Subjects/Keywords: replication fork; helicases; MCM; S. pombe; ubiquitin ligase; fork regression; fork recovery; homologous recombination

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APA (6th Edition):

Ding, L. (2014). Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492

Chicago Manual of Style (16th Edition):

Ding, Lin. “Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 20, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492.

MLA Handbook (7th Edition):

Ding, Lin. “Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe.” 2014. Web. 20 Jan 2020.

Vancouver:

Ding L. Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Jan 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492.

Council of Science Editors:

Ding L. Essential and non-essential helicases maintain genome stability in Schizosaccharomyces pombe. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/360582/rec/2492


University of Guelph

3. Dhavarasa, Piriththiv. Mechanistic Understanding Of PCNA-based Interactions: The Interplay Between CAF-I and Rrm3p for the DNA Sliding Clamp PCNA .

Degree: 2017, University of Guelph

 The Proliferating Cell Nuclear Antigen (PCNA) is a homotrimeric ring that encircles DNA at the replication fork and acts as a docking hub for numerous… (more)

Subjects/Keywords: CAF-I; RRM3; PCNA; Epigenetics; Replication Fork

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APA (6th Edition):

Dhavarasa, P. (2017). Mechanistic Understanding Of PCNA-based Interactions: The Interplay Between CAF-I and Rrm3p for the DNA Sliding Clamp PCNA . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dhavarasa, Piriththiv. “Mechanistic Understanding Of PCNA-based Interactions: The Interplay Between CAF-I and Rrm3p for the DNA Sliding Clamp PCNA .” 2017. Thesis, University of Guelph. Accessed January 20, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dhavarasa, Piriththiv. “Mechanistic Understanding Of PCNA-based Interactions: The Interplay Between CAF-I and Rrm3p for the DNA Sliding Clamp PCNA .” 2017. Web. 20 Jan 2020.

Vancouver:

Dhavarasa P. Mechanistic Understanding Of PCNA-based Interactions: The Interplay Between CAF-I and Rrm3p for the DNA Sliding Clamp PCNA . [Internet] [Thesis]. University of Guelph; 2017. [cited 2020 Jan 20]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12089.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dhavarasa P. Mechanistic Understanding Of PCNA-based Interactions: The Interplay Between CAF-I and Rrm3p for the DNA Sliding Clamp PCNA . [Thesis]. University of Guelph; 2017. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/12089

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

4. Zhong, Yuan. The role of Cdc7 in replication fork progression in response to DNA damage.

Degree: PhD, Molecular Biology, 2012, University of Southern California

 Cdc7-Dbf4 is an essential protein kinase complex required for every single origin firing. As a target of the intra-S checkpoint, Cdc7 kinase activity has also… (more)

Subjects/Keywords: Cdc7; replication fork; DNA damage; fork progression; MMS

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APA (6th Edition):

Zhong, Y. (2012). The role of Cdc7 in replication fork progression in response to DNA damage. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199

Chicago Manual of Style (16th Edition):

Zhong, Yuan. “The role of Cdc7 in replication fork progression in response to DNA damage.” 2012. Doctoral Dissertation, University of Southern California. Accessed January 20, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199.

MLA Handbook (7th Edition):

Zhong, Yuan. “The role of Cdc7 in replication fork progression in response to DNA damage.” 2012. Web. 20 Jan 2020.

Vancouver:

Zhong Y. The role of Cdc7 in replication fork progression in response to DNA damage. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Jan 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199.

Council of Science Editors:

Zhong Y. The role of Cdc7 in replication fork progression in response to DNA damage. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/18011/rec/7199


University of Cambridge

5. Can, Geylani. S-phase checkpoint activity and function throughout the cell cycle.

Degree: PhD, 2017, University of Cambridge

 DNA damage or replication stress during S-phase can activate the S-phase checkpoint which executes a variety of responses, such as the inhibition of origin firing… (more)

Subjects/Keywords: Cell cycle; DNA replication; DNA damage; Rad53; Cdc45; replication fork stabilisation

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APA (6th Edition):

Can, G. (2017). S-phase checkpoint activity and function throughout the cell cycle. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/268506 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744275

Chicago Manual of Style (16th Edition):

Can, Geylani. “S-phase checkpoint activity and function throughout the cell cycle.” 2017. Doctoral Dissertation, University of Cambridge. Accessed January 20, 2020. https://www.repository.cam.ac.uk/handle/1810/268506 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744275.

MLA Handbook (7th Edition):

Can, Geylani. “S-phase checkpoint activity and function throughout the cell cycle.” 2017. Web. 20 Jan 2020.

Vancouver:

Can G. S-phase checkpoint activity and function throughout the cell cycle. [Internet] [Doctoral dissertation]. University of Cambridge; 2017. [cited 2020 Jan 20]. Available from: https://www.repository.cam.ac.uk/handle/1810/268506 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744275.

Council of Science Editors:

Can G. S-phase checkpoint activity and function throughout the cell cycle. [Doctoral Dissertation]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/268506 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744275


University of Oxford

6. Nguyen, Michael Ong. Investigating the molecular mechanism of replication restart in fission yeast.

Degree: PhD, 2014, University of Oxford

 Successful replication of the genome during each cell cycle requires that every replication fork merge with its opposing fork. However, lesions in the template DNA… (more)

Subjects/Keywords: 572.8; Biochemistry; DNA replication; homologous recombination; replication fork barrier

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APA (6th Edition):

Nguyen, M. O. (2014). Investigating the molecular mechanism of replication restart in fission yeast. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:b90fff59-d5b7-43b2-b648-61c0bc977ee9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618528

Chicago Manual of Style (16th Edition):

Nguyen, Michael Ong. “Investigating the molecular mechanism of replication restart in fission yeast.” 2014. Doctoral Dissertation, University of Oxford. Accessed January 20, 2020. http://ora.ox.ac.uk/objects/uuid:b90fff59-d5b7-43b2-b648-61c0bc977ee9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618528.

MLA Handbook (7th Edition):

Nguyen, Michael Ong. “Investigating the molecular mechanism of replication restart in fission yeast.” 2014. Web. 20 Jan 2020.

Vancouver:

Nguyen MO. Investigating the molecular mechanism of replication restart in fission yeast. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Jan 20]. Available from: http://ora.ox.ac.uk/objects/uuid:b90fff59-d5b7-43b2-b648-61c0bc977ee9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618528.

Council of Science Editors:

Nguyen MO. Investigating the molecular mechanism of replication restart in fission yeast. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:b90fff59-d5b7-43b2-b648-61c0bc977ee9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618528


University of Cambridge

7. Can, Geylani. S-phase checkpoint activity and function throughout the cell cycle.

Degree: PhD, 2017, University of Cambridge

 DNA damage or replication stress during S-phase can activate the S-phase checkpoint which executes a variety of responses, such as the inhibition of origin firing… (more)

Subjects/Keywords: Cell cycle; DNA replication; DNA damage; Rad53; Cdc45; replication fork stabilisation

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APA (6th Edition):

Can, G. (2017). S-phase checkpoint activity and function throughout the cell cycle. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/268506

Chicago Manual of Style (16th Edition):

Can, Geylani. “S-phase checkpoint activity and function throughout the cell cycle.” 2017. Doctoral Dissertation, University of Cambridge. Accessed January 20, 2020. https://www.repository.cam.ac.uk/handle/1810/268506.

MLA Handbook (7th Edition):

Can, Geylani. “S-phase checkpoint activity and function throughout the cell cycle.” 2017. Web. 20 Jan 2020.

Vancouver:

Can G. S-phase checkpoint activity and function throughout the cell cycle. [Internet] [Doctoral dissertation]. University of Cambridge; 2017. [cited 2020 Jan 20]. Available from: https://www.repository.cam.ac.uk/handle/1810/268506.

Council of Science Editors:

Can G. S-phase checkpoint activity and function throughout the cell cycle. [Doctoral Dissertation]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/268506


University of Minnesota

8. Raghunandan, Maya. Novel roles for the Fanconi Anemia pathway protein FANCD2 in the recovery of stalled replication forks.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2017, University of Minnesota

 Fanconi Anemia (FA) is an inherited cancer predisposition syndrome that is characterized by a cellular hypersensitivity to DNA interstrand crosslinks (ICLs). To repair these DNA… (more)

Subjects/Keywords: Chromatin remodelling; DNA replication fork stalling; FANCD2; BRCA2; ATRX; Fanconi Anemia; Homologous recombination; Replication restart

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APA (6th Edition):

Raghunandan, M. (2017). Novel roles for the Fanconi Anemia pathway protein FANCD2 in the recovery of stalled replication forks. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/198368

Chicago Manual of Style (16th Edition):

Raghunandan, Maya. “Novel roles for the Fanconi Anemia pathway protein FANCD2 in the recovery of stalled replication forks.” 2017. Doctoral Dissertation, University of Minnesota. Accessed January 20, 2020. http://hdl.handle.net/11299/198368.

MLA Handbook (7th Edition):

Raghunandan, Maya. “Novel roles for the Fanconi Anemia pathway protein FANCD2 in the recovery of stalled replication forks.” 2017. Web. 20 Jan 2020.

Vancouver:

Raghunandan M. Novel roles for the Fanconi Anemia pathway protein FANCD2 in the recovery of stalled replication forks. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/11299/198368.

Council of Science Editors:

Raghunandan M. Novel roles for the Fanconi Anemia pathway protein FANCD2 in the recovery of stalled replication forks. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/198368


University of New Mexico

9. De Haro, Leyma. The role of metnase in DNA replication fork stress response and DNA damage.

Degree: Biomedical Sciences Graduate Program, 2009, University of New Mexico

 Metnase is a recently evolved human protein with methylase (SET) and nuclease domains that is widely expressed, especially in proliferating tissues. Metnase promotes plasmid and… (more)

Subjects/Keywords: Metnase; DNA replication stress; DNA damage; non-homologous end joining; hydroxyurea; replication fork

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APA (6th Edition):

De Haro, L. (2009). The role of metnase in DNA replication fork stress response and DNA damage. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/10

Chicago Manual of Style (16th Edition):

De Haro, Leyma. “The role of metnase in DNA replication fork stress response and DNA damage.” 2009. Doctoral Dissertation, University of New Mexico. Accessed January 20, 2020. https://digitalrepository.unm.edu/biom_etds/10.

MLA Handbook (7th Edition):

De Haro, Leyma. “The role of metnase in DNA replication fork stress response and DNA damage.” 2009. Web. 20 Jan 2020.

Vancouver:

De Haro L. The role of metnase in DNA replication fork stress response and DNA damage. [Internet] [Doctoral dissertation]. University of New Mexico; 2009. [cited 2020 Jan 20]. Available from: https://digitalrepository.unm.edu/biom_etds/10.

Council of Science Editors:

De Haro L. The role of metnase in DNA replication fork stress response and DNA damage. [Doctoral Dissertation]. University of New Mexico; 2009. Available from: https://digitalrepository.unm.edu/biom_etds/10

10. S.E. Rossi. INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY.

Degree: 2017, Università degli Studi di Milano

 Eukaryotic cells have evolved the ATR/hCHK1, MEC1/RAD53 kinase-mediated signal transduction pathway, known as replication checkpoint, to protect and stabilize stalled replication forks in human cells… (more)

Subjects/Keywords: Replication stress; replication fork; checkpoint; Rad53; Rrm3; Pif1; Dna2; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Rossi, S. (2017). INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/471797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rossi, S.E.. “INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY.” 2017. Thesis, Università degli Studi di Milano. Accessed January 20, 2020. http://hdl.handle.net/2434/471797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rossi, S.E.. “INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY.” 2017. Web. 20 Jan 2020.

Vancouver:

Rossi S. INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2434/471797.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rossi S. INTERPLAY BETWEEN THE DNA HELICASES PIF1 AND RRM3, THE NUCLEASE DNA2 AND THE CHECKPOINT PATHWAYS IN THE MAINTENANCE OF THE DNA REPLICATION FORK INTEGRITY. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/471797

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


King Abdullah University of Science and Technology

11. Alhudhali, Lubna F. Encounter of T7 Replisome with Abasic DNA Lesion.

Degree: 2019, King Abdullah University of Science and Technology

 In order to monitor the T7 replisome fate upon encountering abasic lesion, I optimized a single molecule flow stretching assay where the replisome encounters either… (more)

Subjects/Keywords: Abasic Lesion; T7 Replisome; DNA replication; DNA damage; Flow stretching single molecule assay; Replication fork

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APA (6th Edition):

Alhudhali, L. F. (2019). Encounter of T7 Replisome with Abasic DNA Lesion. (Thesis). King Abdullah University of Science and Technology. Retrieved from http://hdl.handle.net/10754/660187

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alhudhali, Lubna F. “Encounter of T7 Replisome with Abasic DNA Lesion.” 2019. Thesis, King Abdullah University of Science and Technology. Accessed January 20, 2020. http://hdl.handle.net/10754/660187.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alhudhali, Lubna F. “Encounter of T7 Replisome with Abasic DNA Lesion.” 2019. Web. 20 Jan 2020.

Vancouver:

Alhudhali LF. Encounter of T7 Replisome with Abasic DNA Lesion. [Internet] [Thesis]. King Abdullah University of Science and Technology; 2019. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10754/660187.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alhudhali LF. Encounter of T7 Replisome with Abasic DNA Lesion. [Thesis]. King Abdullah University of Science and Technology; 2019. Available from: http://hdl.handle.net/10754/660187

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

12. Anastassiadis, Theonie. Dissecting The Functions Of Atr In Replication Fork Stability.

Degree: 2017, University of Pennsylvania

 Genome maintenance is required for cellular viability, and failure to preserve genomic integrity is associated with an increased risk of diseases, such as cancer. To… (more)

Subjects/Keywords: ATR; Replication fork; WEE1; Cell Biology; Genetics; Molecular Biology

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APA (6th Edition):

Anastassiadis, T. (2017). Dissecting The Functions Of Atr In Replication Fork Stability. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/3070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Anastassiadis, Theonie. “Dissecting The Functions Of Atr In Replication Fork Stability.” 2017. Thesis, University of Pennsylvania. Accessed January 20, 2020. https://repository.upenn.edu/edissertations/3070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Anastassiadis, Theonie. “Dissecting The Functions Of Atr In Replication Fork Stability.” 2017. Web. 20 Jan 2020.

Vancouver:

Anastassiadis T. Dissecting The Functions Of Atr In Replication Fork Stability. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2020 Jan 20]. Available from: https://repository.upenn.edu/edissertations/3070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Anastassiadis T. Dissecting The Functions Of Atr In Replication Fork Stability. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/3070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

13. Chavez, Diana Andrea. Replication Fork Remodeling by Helicase-Like Transcription Factor.

Degree: PhD, Biological Sciences, 2018, Vanderbilt University

 During DNA replication, a moving replication fork can encounter various sources of replication stress that can lead the moving fork to stall. Organisms have developed… (more)

Subjects/Keywords: DNA repair; fork remodeling; HIRAN; replication stress; HLTF; x-ray crystallography

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APA (6th Edition):

Chavez, D. A. (2018). Replication Fork Remodeling by Helicase-Like Transcription Factor. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-06252018-151636/ ;

Chicago Manual of Style (16th Edition):

Chavez, Diana Andrea. “Replication Fork Remodeling by Helicase-Like Transcription Factor.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 20, 2020. http://etd.library.vanderbilt.edu/available/etd-06252018-151636/ ;.

MLA Handbook (7th Edition):

Chavez, Diana Andrea. “Replication Fork Remodeling by Helicase-Like Transcription Factor.” 2018. Web. 20 Jan 2020.

Vancouver:

Chavez DA. Replication Fork Remodeling by Helicase-Like Transcription Factor. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Jan 20]. Available from: http://etd.library.vanderbilt.edu/available/etd-06252018-151636/ ;.

Council of Science Editors:

Chavez DA. Replication Fork Remodeling by Helicase-Like Transcription Factor. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://etd.library.vanderbilt.edu/available/etd-06252018-151636/ ;


University of Guelph

14. Cheng, Ashley. Evaluating the Impact of Replication Fork Pausing on Epigenetic Conversions in Saccharomyces cerevisiae: A Novel Approach and First Analysis .

Degree: 2018, University of Guelph

 The subtelomeric regions of Saccharomyces cerevisiae undergo infrequent conversions in the expression state of a gene. The switch in gene expression is thought to be… (more)

Subjects/Keywords: Saccharomyces cerevisiae; gene expression; cellular fluorescence; replication fork pause

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APA (6th Edition):

Cheng, A. (2018). Evaluating the Impact of Replication Fork Pausing on Epigenetic Conversions in Saccharomyces cerevisiae: A Novel Approach and First Analysis . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13936

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheng, Ashley. “Evaluating the Impact of Replication Fork Pausing on Epigenetic Conversions in Saccharomyces cerevisiae: A Novel Approach and First Analysis .” 2018. Thesis, University of Guelph. Accessed January 20, 2020. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13936.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheng, Ashley. “Evaluating the Impact of Replication Fork Pausing on Epigenetic Conversions in Saccharomyces cerevisiae: A Novel Approach and First Analysis .” 2018. Web. 20 Jan 2020.

Vancouver:

Cheng A. Evaluating the Impact of Replication Fork Pausing on Epigenetic Conversions in Saccharomyces cerevisiae: A Novel Approach and First Analysis . [Internet] [Thesis]. University of Guelph; 2018. [cited 2020 Jan 20]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13936.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheng A. Evaluating the Impact of Replication Fork Pausing on Epigenetic Conversions in Saccharomyces cerevisiae: A Novel Approach and First Analysis . [Thesis]. University of Guelph; 2018. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13936

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rockefeller University

15. Sekedat, Matthew David. A Proteomic and Genomic Investigation Into Replication Fork Dynamics in Saccharomyces Cerevisiae.

Degree: 2011, Rockefeller University

 In eukaryotic organisms, each chromosome must be precisely replicated every time a cell divides so that the genetic material can be passed on to the… (more)

Subjects/Keywords: eukaryotic replication forks; Saccharomyces cerevisiae; yeast polymerases; fork dynamics; Life Sciences

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APA (6th Edition):

Sekedat, M. D. (2011). A Proteomic and Genomic Investigation Into Replication Fork Dynamics in Saccharomyces Cerevisiae. (Masters Thesis). Rockefeller University. Retrieved from https://digitalcommons.rockefeller.edu/student_theses_and_dissertations/97

Chicago Manual of Style (16th Edition):

Sekedat, Matthew David. “A Proteomic and Genomic Investigation Into Replication Fork Dynamics in Saccharomyces Cerevisiae.” 2011. Masters Thesis, Rockefeller University. Accessed January 20, 2020. https://digitalcommons.rockefeller.edu/student_theses_and_dissertations/97.

MLA Handbook (7th Edition):

Sekedat, Matthew David. “A Proteomic and Genomic Investigation Into Replication Fork Dynamics in Saccharomyces Cerevisiae.” 2011. Web. 20 Jan 2020.

Vancouver:

Sekedat MD. A Proteomic and Genomic Investigation Into Replication Fork Dynamics in Saccharomyces Cerevisiae. [Internet] [Masters thesis]. Rockefeller University; 2011. [cited 2020 Jan 20]. Available from: https://digitalcommons.rockefeller.edu/student_theses_and_dissertations/97.

Council of Science Editors:

Sekedat MD. A Proteomic and Genomic Investigation Into Replication Fork Dynamics in Saccharomyces Cerevisiae. [Masters Thesis]. Rockefeller University; 2011. Available from: https://digitalcommons.rockefeller.edu/student_theses_and_dissertations/97


National University of Ireland – Galway

16. McGarry, Edel. USP9X controls DNA replication fork stability and the replication stress checkpoint through Claspin .

Degree: 2016, National University of Ireland – Galway

 Cells possess checkpoint pathways which are important for maintaining genome stability and preventing cancer. These signalling pathways include the ATR and CHK1 kinases which are… (more)

Subjects/Keywords: USP9X controls; DNA replication; Fork stability; Claspin; Medicine

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APA (6th Edition):

McGarry, E. (2016). USP9X controls DNA replication fork stability and the replication stress checkpoint through Claspin . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McGarry, Edel. “USP9X controls DNA replication fork stability and the replication stress checkpoint through Claspin .” 2016. Thesis, National University of Ireland – Galway. Accessed January 20, 2020. http://hdl.handle.net/10379/5537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McGarry, Edel. “USP9X controls DNA replication fork stability and the replication stress checkpoint through Claspin .” 2016. Web. 20 Jan 2020.

Vancouver:

McGarry E. USP9X controls DNA replication fork stability and the replication stress checkpoint through Claspin . [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10379/5537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McGarry E. USP9X controls DNA replication fork stability and the replication stress checkpoint through Claspin . [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/5537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

17. Nellimoottil, Tittu Thomas. Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae.

Degree: PhD, Computational Biology and Bioinformatics, 2014, University of Southern California

 In the first project, I show that in Saccharomyces cerevisiae, the rate of replication fork progression is inversely related to the number of origins that… (more)

Subjects/Keywords: DNA replication; timing; fork; budding yeast; forkhead; Fkh; Saccharomyces cerevisiae; fork speed; ORC; RNA binding proteins; RIP-chip

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APA (6th Edition):

Nellimoottil, T. T. (2014). Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536

Chicago Manual of Style (16th Edition):

Nellimoottil, Tittu Thomas. “Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 20, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536.

MLA Handbook (7th Edition):

Nellimoottil, Tittu Thomas. “Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae.” 2014. Web. 20 Jan 2020.

Vancouver:

Nellimoottil TT. Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Jan 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536.

Council of Science Editors:

Nellimoottil TT. Computational analysis of DNA replication timing and fork dynamics in S. cerevisiae. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/325648/rec/1536

18. Ait Saada, Anissia. Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Paris Saclay

Des stress réplicatifs sont rencontrés à chaque phase S du cycle cellulaire et différents mécanismes permettent leur prise en charge. La recombinaison homologue (RH) tient… (more)

Subjects/Keywords: Recombinaison homologue; Réplication; Défauts mitotiques; Stress Réplicatif; Protection des fourches de réplication; Homologous recombination; Replication; Mitotic defects; Replication Stress; Fork Protection

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APA (6th Edition):

Ait Saada, A. (2018). Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2018SACLS167

Chicago Manual of Style (16th Edition):

Ait Saada, Anissia. “Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress.” 2018. Doctoral Dissertation, Paris Saclay. Accessed January 20, 2020. http://www.theses.fr/2018SACLS167.

MLA Handbook (7th Edition):

Ait Saada, Anissia. “Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress.” 2018. Web. 20 Jan 2020.

Vancouver:

Ait Saada A. Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress. [Internet] [Doctoral dissertation]. Paris Saclay; 2018. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2018SACLS167.

Council of Science Editors:

Ait Saada A. Mécanismes par lesquels la recombinaison homologue prévient les défauts mitotiques induits par le stress réplicatif : Mechanisms by which homologous recombination prevents mitotic defects in response to replication stress. [Doctoral Dissertation]. Paris Saclay; 2018. Available from: http://www.theses.fr/2018SACLS167


University of Washington

19. Abeyta, Antonio. A High Throughput RNAi Screen Reveals the Protein Kinase NEK8 Regulates Replication Fork Protection via the DNA Repair Protein RAD51.

Degree: PhD, 2016, University of Washington

 Proteins essential for homologous recombination play a pivotal role in the repair of DNA double strand breaks, DNA inter-strand crosslinks and replication fork stability. Defects… (more)

Subjects/Keywords: DNA repair; DNA replication; genome stability; homologous recombination; kinase; replication fork protection; Molecular biology; Cellular biology; Biology; molecular and cellular biology

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APA (6th Edition):

Abeyta, A. (2016). A High Throughput RNAi Screen Reveals the Protein Kinase NEK8 Regulates Replication Fork Protection via the DNA Repair Protein RAD51. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/35618

Chicago Manual of Style (16th Edition):

Abeyta, Antonio. “A High Throughput RNAi Screen Reveals the Protein Kinase NEK8 Regulates Replication Fork Protection via the DNA Repair Protein RAD51.” 2016. Doctoral Dissertation, University of Washington. Accessed January 20, 2020. http://hdl.handle.net/1773/35618.

MLA Handbook (7th Edition):

Abeyta, Antonio. “A High Throughput RNAi Screen Reveals the Protein Kinase NEK8 Regulates Replication Fork Protection via the DNA Repair Protein RAD51.” 2016. Web. 20 Jan 2020.

Vancouver:

Abeyta A. A High Throughput RNAi Screen Reveals the Protein Kinase NEK8 Regulates Replication Fork Protection via the DNA Repair Protein RAD51. [Internet] [Doctoral dissertation]. University of Washington; 2016. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1773/35618.

Council of Science Editors:

Abeyta A. A High Throughput RNAi Screen Reveals the Protein Kinase NEK8 Regulates Replication Fork Protection via the DNA Repair Protein RAD51. [Doctoral Dissertation]. University of Washington; 2016. Available from: http://hdl.handle.net/1773/35618


University of Pennsylvania

20. Shastri, Nishita Kalpendu. Defining Sites Of Replication Fork Collapse Caused By Atr Inhibition.

Degree: 2017, University of Pennsylvania

 DEFINING SITES OF REPLICATION FORK COLLAPSE CAUSED BY ATR INHIBITION Nishita K. Shastri Eric J. Brown Replication stress, characterized by stalling of DNA replication and… (more)

Subjects/Keywords: ATR; DNA damage; DNA replication stress; Fork collapse; RPA; Cell Biology; Molecular Biology; Pharmacology

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APA (6th Edition):

Shastri, N. K. (2017). Defining Sites Of Replication Fork Collapse Caused By Atr Inhibition. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shastri, Nishita Kalpendu. “Defining Sites Of Replication Fork Collapse Caused By Atr Inhibition.” 2017. Thesis, University of Pennsylvania. Accessed January 20, 2020. https://repository.upenn.edu/edissertations/2581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shastri, Nishita Kalpendu. “Defining Sites Of Replication Fork Collapse Caused By Atr Inhibition.” 2017. Web. 20 Jan 2020.

Vancouver:

Shastri NK. Defining Sites Of Replication Fork Collapse Caused By Atr Inhibition. [Internet] [Thesis]. University of Pennsylvania; 2017. [cited 2020 Jan 20]. Available from: https://repository.upenn.edu/edissertations/2581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shastri NK. Defining Sites Of Replication Fork Collapse Caused By Atr Inhibition. [Thesis]. University of Pennsylvania; 2017. Available from: https://repository.upenn.edu/edissertations/2581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

21. Jackson, Adam. Effect of helicases on the instability of CTG・CAG trinucleotide repeat arrays in the escherichia coli chromosome.

Degree: PhD, 2010, University of Edinburgh

 A trinucleotide repeat (TNR) is a 3 base pair (bp) DNA sequence tandemly repeated in an array. In humans, TNR sequences have been found to… (more)

Subjects/Keywords: 572.8; trinucleotide repeat; TNR; genetic instability; pseudo-hairpins; genetic assay; UvrD; arrested replication fork; helicase

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APA (6th Edition):

Jackson, A. (2010). Effect of helicases on the instability of CTG・CAG trinucleotide repeat arrays in the escherichia coli chromosome. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/4782

Chicago Manual of Style (16th Edition):

Jackson, Adam. “Effect of helicases on the instability of CTG・CAG trinucleotide repeat arrays in the escherichia coli chromosome.” 2010. Doctoral Dissertation, University of Edinburgh. Accessed January 20, 2020. http://hdl.handle.net/1842/4782.

MLA Handbook (7th Edition):

Jackson, Adam. “Effect of helicases on the instability of CTG・CAG trinucleotide repeat arrays in the escherichia coli chromosome.” 2010. Web. 20 Jan 2020.

Vancouver:

Jackson A. Effect of helicases on the instability of CTG・CAG trinucleotide repeat arrays in the escherichia coli chromosome. [Internet] [Doctoral dissertation]. University of Edinburgh; 2010. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1842/4782.

Council of Science Editors:

Jackson A. Effect of helicases on the instability of CTG・CAG trinucleotide repeat arrays in the escherichia coli chromosome. [Doctoral Dissertation]. University of Edinburgh; 2010. Available from: http://hdl.handle.net/1842/4782

22. Iyer, Divya Ramalingam. Single-Molecule Studies of Replication Kinetics in Response to DNA Damage.

Degree: Interdisciplinary Graduate Program, Department of Biochemistry and Molecular Pharmacology, 2017, U of Massachusetts : Med

  In response to DNA damage during S phase, cells slow DNA replication. This slowing is orchestrated by the intra-S checkpoint and involves inhibition of… (more)

Subjects/Keywords: single-molecule replication kinetics; DNA combing; fork rate; fork stalling; origin firing rate; DNA damage; intra-S checkpoint; fission yeast; Biochemistry; Cell Biology; Genetics; Molecular Biology

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APA (6th Edition):

Iyer, D. R. (2017). Single-Molecule Studies of Replication Kinetics in Response to DNA Damage. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/906

Chicago Manual of Style (16th Edition):

Iyer, Divya Ramalingam. “Single-Molecule Studies of Replication Kinetics in Response to DNA Damage.” 2017. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 20, 2020. http://escholarship.umassmed.edu/gsbs_diss/906.

MLA Handbook (7th Edition):

Iyer, Divya Ramalingam. “Single-Molecule Studies of Replication Kinetics in Response to DNA Damage.” 2017. Web. 20 Jan 2020.

Vancouver:

Iyer DR. Single-Molecule Studies of Replication Kinetics in Response to DNA Damage. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2017. [cited 2020 Jan 20]. Available from: http://escholarship.umassmed.edu/gsbs_diss/906.

Council of Science Editors:

Iyer DR. Single-Molecule Studies of Replication Kinetics in Response to DNA Damage. [Doctoral Dissertation]. U of Massachusetts : Med; 2017. Available from: http://escholarship.umassmed.edu/gsbs_diss/906


University of Oxford

23. Latusek, Robert. The role of LATS1 in DNA damage signalling.

Degree: PhD, 2012, University of Oxford

 Genomic DNA is constantly exposed to assaults, which if not dealt with, can lead to genomic instability and carcinogenesis. In response to stress including either… (more)

Subjects/Keywords: 572.86; DNA damage signalling; Oncology; Radiation; Tumours; Biochemistry; Cancer Research; Tumour Suppressor; DNA Damage Repair; Replication Fork Stabilisation

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APA (6th Edition):

Latusek, R. (2012). The role of LATS1 in DNA damage signalling. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:7f21ecaf-20a6-45a9-8403-c8255955920e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589598

Chicago Manual of Style (16th Edition):

Latusek, Robert. “The role of LATS1 in DNA damage signalling.” 2012. Doctoral Dissertation, University of Oxford. Accessed January 20, 2020. http://ora.ox.ac.uk/objects/uuid:7f21ecaf-20a6-45a9-8403-c8255955920e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589598.

MLA Handbook (7th Edition):

Latusek, Robert. “The role of LATS1 in DNA damage signalling.” 2012. Web. 20 Jan 2020.

Vancouver:

Latusek R. The role of LATS1 in DNA damage signalling. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2020 Jan 20]. Available from: http://ora.ox.ac.uk/objects/uuid:7f21ecaf-20a6-45a9-8403-c8255955920e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589598.

Council of Science Editors:

Latusek R. The role of LATS1 in DNA damage signalling. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:7f21ecaf-20a6-45a9-8403-c8255955920e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589598


Cornell University

24. Patel, Darshil R. MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS .

Degree: 2018, Cornell University

 DNA damage checkpoint pathways are part of the broader cellular DNA damage response (DDR) that promotes genome maintenance when cells experience DNA damage. The RAD9A-HUS1-RAD1… (more)

Subjects/Keywords: Biochemistry; Cellular biology; Molecular biology; Checkpoint; DNA Damage Response; DNA Repair; Genomic Stability; Replication Fork Stability; Tumorigenesis

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APA (6th Edition):

Patel, D. R. (2018). MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/59785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patel, Darshil R. “MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS .” 2018. Thesis, Cornell University. Accessed January 20, 2020. http://hdl.handle.net/1813/59785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patel, Darshil R. “MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS .” 2018. Web. 20 Jan 2020.

Vancouver:

Patel DR. MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS . [Internet] [Thesis]. Cornell University; 2018. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1813/59785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patel DR. MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS . [Thesis]. Cornell University; 2018. Available from: http://hdl.handle.net/1813/59785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

25. Peace, Jared Michael. Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program.

Degree: PhD, Molecular Biology, 2014, University of Southern California

 Eukaryotic cells initiate DNA replication from hundreds to thousands of origins genome wide. The coordinated firing of these origins across a range of times throughout… (more)

Subjects/Keywords: Forkhead; Fkh1; Fkh2; Cdc45; Rif1; Rap1; Pfa4; Mec1; Cdc7; Dbf4; Dbf4 Dependent Kinase (DDK); Orc1; replication origin timing; replication fork rate; chromatin; centromere; telomere; epigenetics; transcription; nuclear architecture; S. cerevisiae

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APA (6th Edition):

Peace, J. M. (2014). Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869

Chicago Manual of Style (16th Edition):

Peace, Jared Michael. “Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program.” 2014. Doctoral Dissertation, University of Southern California. Accessed January 20, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869.

MLA Handbook (7th Edition):

Peace, Jared Michael. “Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program.” 2014. Web. 20 Jan 2020.

Vancouver:

Peace JM. Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2020 Jan 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869.

Council of Science Editors:

Peace JM. Forkhead transcription factors control genome wide dynamics of the S. cerevisiae replication timing program. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/514469/rec/2869


University of Southern California

26. Szyjka, Shawn Joseph. The intra-S phase checkpoint and its effect on replication fork dynamics in saccharomyces cerevisiae.

Degree: PhD, Molecular Biology, 2008, University of Southern California

 Duplication of a cell's genetic material is of paramount importance. This task must be completed accurately, efficiently and occur once and only once within any… (more)

Subjects/Keywords: DNA replication; DNA damage; replication fork; DNA repair; cell cycle checkpoint; phosphatase; Rad53; Mrc1; BrdU; microarray; Claspin; replication fork progression; Rrm3; Pph3; 2-D gel electro

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APA (6th Edition):

Szyjka, S. J. (2008). The intra-S phase checkpoint and its effect on replication fork dynamics in saccharomyces cerevisiae. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/119913/rec/6914

Chicago Manual of Style (16th Edition):

Szyjka, Shawn Joseph. “The intra-S phase checkpoint and its effect on replication fork dynamics in saccharomyces cerevisiae.” 2008. Doctoral Dissertation, University of Southern California. Accessed January 20, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/119913/rec/6914.

MLA Handbook (7th Edition):

Szyjka, Shawn Joseph. “The intra-S phase checkpoint and its effect on replication fork dynamics in saccharomyces cerevisiae.” 2008. Web. 20 Jan 2020.

Vancouver:

Szyjka SJ. The intra-S phase checkpoint and its effect on replication fork dynamics in saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2020 Jan 20]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/119913/rec/6914.

Council of Science Editors:

Szyjka SJ. The intra-S phase checkpoint and its effect on replication fork dynamics in saccharomyces cerevisiae. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/119913/rec/6914


University of Illinois – Urbana-Champaign

27. Kuong, Ka Wai. The role of recombinational repair in the mechanism of killing during thymine starvation in Escherichia coli.

Degree: PhD, 0322, 2012, University of Illinois – Urbana-Champaign

 Thymineless death (TLD) describes a universal phenomenon in which cells cannot recover after starvation for one of the four DNA precursors, dTTP. The mechanism of… (more)

Subjects/Keywords: Thymineless death; recombinational repair; thymine starvation; Hydroxyurea; RecA; double strand breaks; chromosomal fragmentation; replication fork breakage; epistatic analysis; pulse field gel electrophoresis

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APA (6th Edition):

Kuong, K. W. (2012). The role of recombinational repair in the mechanism of killing during thymine starvation in Escherichia coli. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29515

Chicago Manual of Style (16th Edition):

Kuong, Ka Wai. “The role of recombinational repair in the mechanism of killing during thymine starvation in Escherichia coli.” 2012. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed January 20, 2020. http://hdl.handle.net/2142/29515.

MLA Handbook (7th Edition):

Kuong, Ka Wai. “The role of recombinational repair in the mechanism of killing during thymine starvation in Escherichia coli.” 2012. Web. 20 Jan 2020.

Vancouver:

Kuong KW. The role of recombinational repair in the mechanism of killing during thymine starvation in Escherichia coli. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2012. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2142/29515.

Council of Science Editors:

Kuong KW. The role of recombinational repair in the mechanism of killing during thymine starvation in Escherichia coli. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29515


Wright State University

28. Gadgil, Rujuta Yashodhan. Instability at Trinucleotide Repeat DNAs.

Degree: MS, Biochemistry and Molecular Biology, 2016, Wright State University

 Trinucleotide repeats (TNRs) are sequences prone to formation of non-B DNAstructures and mutations; undergo expansions in vivo to cause various inheritedneurodegenerative diseases. Hairpin structures formed… (more)

Subjects/Keywords: Biochemistry; Molecular Biology; Tri-nucleotide; repeats; non-B DNA; structures; inherited; neurodegenerative; replication; fork; stalling; single; double; strand; DNA; breaks; color; marker; gene; assay; detect; DNA; breaks

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gadgil, R. Y. (2016). Instability at Trinucleotide Repeat DNAs. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204

Chicago Manual of Style (16th Edition):

Gadgil, Rujuta Yashodhan. “Instability at Trinucleotide Repeat DNAs.” 2016. Masters Thesis, Wright State University. Accessed January 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

MLA Handbook (7th Edition):

Gadgil, Rujuta Yashodhan. “Instability at Trinucleotide Repeat DNAs.” 2016. Web. 20 Jan 2020.

Vancouver:

Gadgil RY. Instability at Trinucleotide Repeat DNAs. [Internet] [Masters thesis]. Wright State University; 2016. [cited 2020 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

Council of Science Editors:

Gadgil RY. Instability at Trinucleotide Repeat DNAs. [Masters Thesis]. Wright State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204


University of St. Andrews

29. Richards, Jodi D. Helicases and DNA dependent ATPases of Sulfolobus solfataricus .

Degree: 2008, University of St. Andrews

 DNA is susceptible to various types of damage as a result of normal cellular metabolism or from environmental sources. In order to maintain genome stability… (more)

Subjects/Keywords: Archaea; Helicase; ATPase; Transcription; Stalled replication fork restart; DNA repair

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Richards, J. D. (2008). Helicases and DNA dependent ATPases of Sulfolobus solfataricus . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richards, Jodi D. “Helicases and DNA dependent ATPases of Sulfolobus solfataricus .” 2008. Thesis, University of St. Andrews. Accessed January 20, 2020. http://hdl.handle.net/10023/474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richards, Jodi D. “Helicases and DNA dependent ATPases of Sulfolobus solfataricus .” 2008. Web. 20 Jan 2020.

Vancouver:

Richards JD. Helicases and DNA dependent ATPases of Sulfolobus solfataricus . [Internet] [Thesis]. University of St. Andrews; 2008. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/10023/474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richards JD. Helicases and DNA dependent ATPases of Sulfolobus solfataricus . [Thesis]. University of St. Andrews; 2008. Available from: http://hdl.handle.net/10023/474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

30. Raoof, Mustafa. CHEMOSENSITIZATION OF HEPATOCELLULAR CARCINOMA TO GEMCITABINE BY NON-INVASIVE RADIOFREQUENCY FIELD-INDUCED HYPERTHERMIA.

Degree: MS, 2012, Texas Medical Center

  Gemcitabine is a potent nucleoside analogue against solid tumors however drug resistance rapidly emerges. Removal of gemcitabine incorporated in the DNA by repair mechanisms… (more)

Subjects/Keywords: gemcitabine; stalled replication fork; homologous recombination; hyperthermia; radiofrequency; hepatocellular carcinoma; Genetic Processes; Hepatology; Laboratory and Basic Science Research; Medical Pharmacology; Medicine and Health Sciences; Molecular Biology; Neoplasms; Nucleic Acids, Nucleotides, and Nucleosides; Pharmacology; Surgery

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raoof, M. (2012). CHEMOSENSITIZATION OF HEPATOCELLULAR CARCINOMA TO GEMCITABINE BY NON-INVASIVE RADIOFREQUENCY FIELD-INDUCED HYPERTHERMIA. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/244

Chicago Manual of Style (16th Edition):

Raoof, Mustafa. “CHEMOSENSITIZATION OF HEPATOCELLULAR CARCINOMA TO GEMCITABINE BY NON-INVASIVE RADIOFREQUENCY FIELD-INDUCED HYPERTHERMIA.” 2012. Masters Thesis, Texas Medical Center. Accessed January 20, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/244.

MLA Handbook (7th Edition):

Raoof, Mustafa. “CHEMOSENSITIZATION OF HEPATOCELLULAR CARCINOMA TO GEMCITABINE BY NON-INVASIVE RADIOFREQUENCY FIELD-INDUCED HYPERTHERMIA.” 2012. Web. 20 Jan 2020.

Vancouver:

Raoof M. CHEMOSENSITIZATION OF HEPATOCELLULAR CARCINOMA TO GEMCITABINE BY NON-INVASIVE RADIOFREQUENCY FIELD-INDUCED HYPERTHERMIA. [Internet] [Masters thesis]. Texas Medical Center; 2012. [cited 2020 Jan 20]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/244.

Council of Science Editors:

Raoof M. CHEMOSENSITIZATION OF HEPATOCELLULAR CARCINOMA TO GEMCITABINE BY NON-INVASIVE RADIOFREQUENCY FIELD-INDUCED HYPERTHERMIA. [Masters Thesis]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/244

.