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You searched for subject:(ras proteins). Showing records 1 – 30 of 48 total matches.

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University of Alberta

1. Mattar, Christine. Variable lipidation of Ras isoforms directs their differential membrane association.

Degree: MS, Department of Biochemistry, 2000, University of Alberta

Subjects/Keywords: Ras proteins.; Ras oncogenes.

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APA (6th Edition):

Mattar, C. (2000). Variable lipidation of Ras isoforms directs their differential membrane association. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/9z9031792

Chicago Manual of Style (16th Edition):

Mattar, Christine. “Variable lipidation of Ras isoforms directs their differential membrane association.” 2000. Masters Thesis, University of Alberta. Accessed January 22, 2020. https://era.library.ualberta.ca/files/9z9031792.

MLA Handbook (7th Edition):

Mattar, Christine. “Variable lipidation of Ras isoforms directs their differential membrane association.” 2000. Web. 22 Jan 2020.

Vancouver:

Mattar C. Variable lipidation of Ras isoforms directs their differential membrane association. [Internet] [Masters thesis]. University of Alberta; 2000. [cited 2020 Jan 22]. Available from: https://era.library.ualberta.ca/files/9z9031792.

Council of Science Editors:

Mattar C. Variable lipidation of Ras isoforms directs their differential membrane association. [Masters Thesis]. University of Alberta; 2000. Available from: https://era.library.ualberta.ca/files/9z9031792

2. Parker, Jillian Ann. The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting.

Degree: PhD, Department of Chemistry and Chemical Biology, 2017, Northeastern University

 The small GTPase Ras is a bimolecular switch protein involved in a multitude of cellular signaling pathways, including those that control cell proliferation, differentiation, and… (more)

Subjects/Keywords: bimolecular switch proteins; guanine nucleotide exchange factors; Ras isoform; K-Ras

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APA (6th Edition):

Parker, J. A. (2017). The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20240308

Chicago Manual of Style (16th Edition):

Parker, Jillian Ann. “The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting.” 2017. Doctoral Dissertation, Northeastern University. Accessed January 22, 2020. http://hdl.handle.net/2047/D20240308.

MLA Handbook (7th Edition):

Parker, Jillian Ann. “The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting.” 2017. Web. 22 Jan 2020.

Vancouver:

Parker JA. The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting. [Internet] [Doctoral dissertation]. Northeastern University; 2017. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2047/D20240308.

Council of Science Editors:

Parker JA. The most oncogenic Ras isoform: specific insights into K-Ras structure, dimerization, and targeting. [Doctoral Dissertation]. Northeastern University; 2017. Available from: http://hdl.handle.net/2047/D20240308


Northeastern University

3. Johnson, Christian William. Allosteric communication in the activity of Ras GTPases.

Degree: PhD, Department of Chemistry and Chemical Biology, 2016, Northeastern University

 H-, N-, K-Ras are closely related (95% conserved) membrane associated hub proteins that act in a variety of signal transduction pathways, including cellular proliferation and… (more)

Subjects/Keywords: cancer; GTPases; protein allostery; protein crystallography; Ras; Allosteric proteins; Ras proteins; Structure; Guanosine triphosphatase; Allosteric regulation; Crystallography; Enzyme kinetics

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APA (6th Edition):

Johnson, C. W. (2016). Allosteric communication in the activity of Ras GTPases. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20199679

Chicago Manual of Style (16th Edition):

Johnson, Christian William. “Allosteric communication in the activity of Ras GTPases.” 2016. Doctoral Dissertation, Northeastern University. Accessed January 22, 2020. http://hdl.handle.net/2047/D20199679.

MLA Handbook (7th Edition):

Johnson, Christian William. “Allosteric communication in the activity of Ras GTPases.” 2016. Web. 22 Jan 2020.

Vancouver:

Johnson CW. Allosteric communication in the activity of Ras GTPases. [Internet] [Doctoral dissertation]. Northeastern University; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2047/D20199679.

Council of Science Editors:

Johnson CW. Allosteric communication in the activity of Ras GTPases. [Doctoral Dissertation]. Northeastern University; 2016. Available from: http://hdl.handle.net/2047/D20199679


Northeastern University

4. Salter, Shores. Structural characterization of NRas Q61L and Q61R mutants and their potential affect on intrinsic hydrolysis.

Degree: MS, Department of Chemistry and Chemical Biology, 2016, Northeastern University

Ras GTPases are a subfamily of a larger family of hydrolase enzymes, which function as key regulators of signal transduction pathways. Site-specific mutations in nucleotide… (more)

Subjects/Keywords: cancer; hydrolysis; melanoma; NRas; oncogenic; Ras; G proteins; Structure; Ras proteins; Structure; Guanosine triphosphatase; Hydrolysis; Hydrolases; Cellular signal transduction

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APA (6th Edition):

Salter, S. (2016). Structural characterization of NRas Q61L and Q61R mutants and their potential affect on intrinsic hydrolysis. (Masters Thesis). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20213052

Chicago Manual of Style (16th Edition):

Salter, Shores. “Structural characterization of NRas Q61L and Q61R mutants and their potential affect on intrinsic hydrolysis.” 2016. Masters Thesis, Northeastern University. Accessed January 22, 2020. http://hdl.handle.net/2047/D20213052.

MLA Handbook (7th Edition):

Salter, Shores. “Structural characterization of NRas Q61L and Q61R mutants and their potential affect on intrinsic hydrolysis.” 2016. Web. 22 Jan 2020.

Vancouver:

Salter S. Structural characterization of NRas Q61L and Q61R mutants and their potential affect on intrinsic hydrolysis. [Internet] [Masters thesis]. Northeastern University; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2047/D20213052.

Council of Science Editors:

Salter S. Structural characterization of NRas Q61L and Q61R mutants and their potential affect on intrinsic hydrolysis. [Masters Thesis]. Northeastern University; 2016. Available from: http://hdl.handle.net/2047/D20213052


University of Hong Kong

5. Chan, Yuk-shing. Expression of RAs-related Nuclear (RAN) protein in breast cancer.

Degree: Master of Medical Sciences, 2010, University of Hong Kong

published_or_final_version

Pathology

Master

Master of Medical Sciences

Subjects/Keywords: Breast - Cancer.; Ras proteins.

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APA (6th Edition):

Chan, Y. (2010). Expression of RAs-related Nuclear (RAN) protein in breast cancer. (Masters Thesis). University of Hong Kong. Retrieved from Chan, Y. [陳旭勝]. (2010). Expression of RAs-related Nuclear (RAN) protein in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4467100 ; http://dx.doi.org/10.5353/th_b4467100 ; http://hdl.handle.net/10722/130889

Chicago Manual of Style (16th Edition):

Chan, Yuk-shing. “Expression of RAs-related Nuclear (RAN) protein in breast cancer.” 2010. Masters Thesis, University of Hong Kong. Accessed January 22, 2020. Chan, Y. [陳旭勝]. (2010). Expression of RAs-related Nuclear (RAN) protein in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4467100 ; http://dx.doi.org/10.5353/th_b4467100 ; http://hdl.handle.net/10722/130889.

MLA Handbook (7th Edition):

Chan, Yuk-shing. “Expression of RAs-related Nuclear (RAN) protein in breast cancer.” 2010. Web. 22 Jan 2020.

Vancouver:

Chan Y. Expression of RAs-related Nuclear (RAN) protein in breast cancer. [Internet] [Masters thesis]. University of Hong Kong; 2010. [cited 2020 Jan 22]. Available from: Chan, Y. [陳旭勝]. (2010). Expression of RAs-related Nuclear (RAN) protein in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4467100 ; http://dx.doi.org/10.5353/th_b4467100 ; http://hdl.handle.net/10722/130889.

Council of Science Editors:

Chan Y. Expression of RAs-related Nuclear (RAN) protein in breast cancer. [Masters Thesis]. University of Hong Kong; 2010. Available from: Chan, Y. [陳旭勝]. (2010). Expression of RAs-related Nuclear (RAN) protein in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4467100 ; http://dx.doi.org/10.5353/th_b4467100 ; http://hdl.handle.net/10722/130889

6. Shishina, Anna. Nucleotide-Dependent Preferential Localization of Ras in Model Membranes with Lipid Raft Nanodomains.

Degree: 2017, Marquette University

 Membrane proteins constitute a third of all proteins in the cell and more than 50% of drug targets. However, the analysis of membrane proteins has… (more)

Subjects/Keywords: FRET; lipid membranes; lipid raft; Ras proteins; semi-synthetic lipidated Ras; time-resolved fluorescence spectroscopy; Medicinal-Pharmaceutical Chemistry; Oncology; Physical Chemistry

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APA (6th Edition):

Shishina, A. (2017). Nucleotide-Dependent Preferential Localization of Ras in Model Membranes with Lipid Raft Nanodomains. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/dissertations_mu/752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shishina, Anna. “Nucleotide-Dependent Preferential Localization of Ras in Model Membranes with Lipid Raft Nanodomains.” 2017. Thesis, Marquette University. Accessed January 22, 2020. https://epublications.marquette.edu/dissertations_mu/752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shishina, Anna. “Nucleotide-Dependent Preferential Localization of Ras in Model Membranes with Lipid Raft Nanodomains.” 2017. Web. 22 Jan 2020.

Vancouver:

Shishina A. Nucleotide-Dependent Preferential Localization of Ras in Model Membranes with Lipid Raft Nanodomains. [Internet] [Thesis]. Marquette University; 2017. [cited 2020 Jan 22]. Available from: https://epublications.marquette.edu/dissertations_mu/752.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shishina A. Nucleotide-Dependent Preferential Localization of Ras in Model Membranes with Lipid Raft Nanodomains. [Thesis]. Marquette University; 2017. Available from: https://epublications.marquette.edu/dissertations_mu/752

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

7. Keller, Jeffrey Wayne. The contribution of RAS function to transformation of the colonic epithelium: functional differences, similarities, and cooperation between RAS family members.

Degree: PhD, Cell and Developmental Biology, 2006, Vanderbilt University

 Constitutively activating mutations of members of the RAS family of small G proteins provide an important oncogenic contribution to a significant percentage of human cancers.… (more)

Subjects/Keywords: Ras proteins; Colon  – Cancer  – Genetic aspects; transformation; Ras oncogenes

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APA (6th Edition):

Keller, J. W. (2006). The contribution of RAS function to transformation of the colonic epithelium: functional differences, similarities, and cooperation between RAS family members. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-05162006-153153/ ;

Chicago Manual of Style (16th Edition):

Keller, Jeffrey Wayne. “The contribution of RAS function to transformation of the colonic epithelium: functional differences, similarities, and cooperation between RAS family members.” 2006. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu/available/etd-05162006-153153/ ;.

MLA Handbook (7th Edition):

Keller, Jeffrey Wayne. “The contribution of RAS function to transformation of the colonic epithelium: functional differences, similarities, and cooperation between RAS family members.” 2006. Web. 22 Jan 2020.

Vancouver:

Keller JW. The contribution of RAS function to transformation of the colonic epithelium: functional differences, similarities, and cooperation between RAS family members. [Internet] [Doctoral dissertation]. Vanderbilt University; 2006. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-05162006-153153/ ;.

Council of Science Editors:

Keller JW. The contribution of RAS function to transformation of the colonic epithelium: functional differences, similarities, and cooperation between RAS family members. [Doctoral Dissertation]. Vanderbilt University; 2006. Available from: http://etd.library.vanderbilt.edu/available/etd-05162006-153153/ ;


Northeastern University

8. Knihtila, Ryan Robert. GTP hydrolysis in RAS: how mutations affect hydrogen bonding and catalysis.

Degree: PhD, Department of Chemistry and Chemical Biology, 2016, Northeastern University

RAS GTPases (H, K and NRAS) are closely related membrane associated GTP binding proteins (95% sequence identity) that participate in numerous cell signaling processes, including… (more)

Subjects/Keywords: cancer; enzymology; neutron; RAS; Guanosine triphosphatase; Ras proteins; Mutation (Biology); Hydrolysis; Cellular signal transduction; Oncogenes; Crystallography

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APA (6th Edition):

Knihtila, R. R. (2016). GTP hydrolysis in RAS: how mutations affect hydrogen bonding and catalysis. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20211606

Chicago Manual of Style (16th Edition):

Knihtila, Ryan Robert. “GTP hydrolysis in RAS: how mutations affect hydrogen bonding and catalysis.” 2016. Doctoral Dissertation, Northeastern University. Accessed January 22, 2020. http://hdl.handle.net/2047/D20211606.

MLA Handbook (7th Edition):

Knihtila, Ryan Robert. “GTP hydrolysis in RAS: how mutations affect hydrogen bonding and catalysis.” 2016. Web. 22 Jan 2020.

Vancouver:

Knihtila RR. GTP hydrolysis in RAS: how mutations affect hydrogen bonding and catalysis. [Internet] [Doctoral dissertation]. Northeastern University; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2047/D20211606.

Council of Science Editors:

Knihtila RR. GTP hydrolysis in RAS: how mutations affect hydrogen bonding and catalysis. [Doctoral Dissertation]. Northeastern University; 2016. Available from: http://hdl.handle.net/2047/D20211606


University of Groningen

9. Szymanski, Mariusz Krzysztof. Cardiorenal interaction: experimental and clinical studies.

Degree: PhD, 2012, University of Groningen

 Nieuwe mogelijkheden voor onderzoek naar slecht functioneren van hart en nieren Hartfalen is een complex klinisch syndroom gekarakteriseerd door een verminderde hartfunctie en slechte terugstroom… (more)

Subjects/Keywords: Proefschriften (vorm); Renine angiotensine systeem; Ras-proteins; Positronemissietomografie; Hartzwakte; Nierinsufficiëntie; Diermodellen; Pathologische fysiologie; cardiologie

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APA (6th Edition):

Szymanski, M. K. (2012). Cardiorenal interaction: experimental and clinical studies. (Doctoral Dissertation). University of Groningen. Retrieved from http://hdl.handle.net/11370/397feb17-1760-4483-b16f-ad323eaadfa8

Chicago Manual of Style (16th Edition):

Szymanski, Mariusz Krzysztof. “Cardiorenal interaction: experimental and clinical studies.” 2012. Doctoral Dissertation, University of Groningen. Accessed January 22, 2020. http://hdl.handle.net/11370/397feb17-1760-4483-b16f-ad323eaadfa8.

MLA Handbook (7th Edition):

Szymanski, Mariusz Krzysztof. “Cardiorenal interaction: experimental and clinical studies.” 2012. Web. 22 Jan 2020.

Vancouver:

Szymanski MK. Cardiorenal interaction: experimental and clinical studies. [Internet] [Doctoral dissertation]. University of Groningen; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11370/397feb17-1760-4483-b16f-ad323eaadfa8.

Council of Science Editors:

Szymanski MK. Cardiorenal interaction: experimental and clinical studies. [Doctoral Dissertation]. University of Groningen; 2012. Available from: http://hdl.handle.net/11370/397feb17-1760-4483-b16f-ad323eaadfa8

10. Bender, R. Hugh F. K-Ras, but Not H-Ras or N-Ras, Hyperactivation Regulates Brain Neural Stem Cell Proliferation in a Raf/Rb-dependent Manner.

Degree: PhD, Biology & Biomedical Sciences (Developmental, Regenerative, & Stem Cell Biology), 2015, Washington University in St. Louis

  Neural stem cells (NSCs) give rise to all the major cell types in the brain, including neurons, oligodendrocytes, and astrocytes. However, the intracellular signaling… (more)

Subjects/Keywords: cell proliferation, neural stem cells, raf kinases, ras proteins, retinoblastoma protein; Biology

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APA (6th Edition):

Bender, R. H. F. (2015). K-Ras, but Not H-Ras or N-Ras, Hyperactivation Regulates Brain Neural Stem Cell Proliferation in a Raf/Rb-dependent Manner. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/art_sci_etds/443

Chicago Manual of Style (16th Edition):

Bender, R Hugh F. “K-Ras, but Not H-Ras or N-Ras, Hyperactivation Regulates Brain Neural Stem Cell Proliferation in a Raf/Rb-dependent Manner.” 2015. Doctoral Dissertation, Washington University in St. Louis. Accessed January 22, 2020. https://openscholarship.wustl.edu/art_sci_etds/443.

MLA Handbook (7th Edition):

Bender, R Hugh F. “K-Ras, but Not H-Ras or N-Ras, Hyperactivation Regulates Brain Neural Stem Cell Proliferation in a Raf/Rb-dependent Manner.” 2015. Web. 22 Jan 2020.

Vancouver:

Bender RHF. K-Ras, but Not H-Ras or N-Ras, Hyperactivation Regulates Brain Neural Stem Cell Proliferation in a Raf/Rb-dependent Manner. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2015. [cited 2020 Jan 22]. Available from: https://openscholarship.wustl.edu/art_sci_etds/443.

Council of Science Editors:

Bender RHF. K-Ras, but Not H-Ras or N-Ras, Hyperactivation Regulates Brain Neural Stem Cell Proliferation in a Raf/Rb-dependent Manner. [Doctoral Dissertation]. Washington University in St. Louis; 2015. Available from: https://openscholarship.wustl.edu/art_sci_etds/443

11. Galiakhmetov, Azamat. Dynamics and Interactions of Membrane Proteins.

Degree: 2018, Marquette University

 Membrane proteins are members of the class of proteins that perform their functions while being associated with a lipid bilayer. In the cell, they serve… (more)

Subjects/Keywords: Fluorescence anisotropy; Membrane proteins; Nanodisc; POR; RAS; solution NMR; Biochemistry; Cell Biology

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APA (6th Edition):

Galiakhmetov, A. (2018). Dynamics and Interactions of Membrane Proteins. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/dissertations_mu/801

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Galiakhmetov, Azamat. “Dynamics and Interactions of Membrane Proteins.” 2018. Thesis, Marquette University. Accessed January 22, 2020. https://epublications.marquette.edu/dissertations_mu/801.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Galiakhmetov, Azamat. “Dynamics and Interactions of Membrane Proteins.” 2018. Web. 22 Jan 2020.

Vancouver:

Galiakhmetov A. Dynamics and Interactions of Membrane Proteins. [Internet] [Thesis]. Marquette University; 2018. [cited 2020 Jan 22]. Available from: https://epublications.marquette.edu/dissertations_mu/801.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Galiakhmetov A. Dynamics and Interactions of Membrane Proteins. [Thesis]. Marquette University; 2018. Available from: https://epublications.marquette.edu/dissertations_mu/801

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

12. Katsoulotos, Gregory Peter. The function of the signaling protein Ras guanine releasing protein 4 (RasGRP4) in human mast cells.

Degree: Clinical School - St George Hospital, 2006, University of New South Wales

 Mast cells have been implicated in the pathogenesis of both atopic and non-atopic asthma. Rasguanine nucleotide-releasing protein 4 (RasGRP4) is a mast cell-restricted guanine nucleotideexchange… (more)

Subjects/Keywords: Interleukins; Cytokines; Ras proteins; Mast cells

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APA (6th Edition):

Katsoulotos, G. P. (2006). The function of the signaling protein Ras guanine releasing protein 4 (RasGRP4) in human mast cells. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/27341 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1240/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Katsoulotos, Gregory Peter. “The function of the signaling protein Ras guanine releasing protein 4 (RasGRP4) in human mast cells.” 2006. Doctoral Dissertation, University of New South Wales. Accessed January 22, 2020. http://handle.unsw.edu.au/1959.4/27341 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1240/SOURCE02?view=true.

MLA Handbook (7th Edition):

Katsoulotos, Gregory Peter. “The function of the signaling protein Ras guanine releasing protein 4 (RasGRP4) in human mast cells.” 2006. Web. 22 Jan 2020.

Vancouver:

Katsoulotos GP. The function of the signaling protein Ras guanine releasing protein 4 (RasGRP4) in human mast cells. [Internet] [Doctoral dissertation]. University of New South Wales; 2006. [cited 2020 Jan 22]. Available from: http://handle.unsw.edu.au/1959.4/27341 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1240/SOURCE02?view=true.

Council of Science Editors:

Katsoulotos GP. The function of the signaling protein Ras guanine releasing protein 4 (RasGRP4) in human mast cells. [Doctoral Dissertation]. University of New South Wales; 2006. Available from: http://handle.unsw.edu.au/1959.4/27341 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1240/SOURCE02?view=true


Duke University

13. Fuller, Deirdre Marie. Analysis of TCR Signaling and Erk Activation in T Cell Development and Autoimmunity .

Degree: 2012, Duke University

  LAT is a transmembrane adaptor protein that is critical for the emanation of signals downstream of the TCR. Following TCR engagement, LAT is phosphorylated… (more)

Subjects/Keywords: Immunology; Autoimmunity; Ras signaling; T cell receptor signaling; T cells; Transmembrane adaptor proteins

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APA (6th Edition):

Fuller, D. M. (2012). Analysis of TCR Signaling and Erk Activation in T Cell Development and Autoimmunity . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fuller, Deirdre Marie. “Analysis of TCR Signaling and Erk Activation in T Cell Development and Autoimmunity .” 2012. Thesis, Duke University. Accessed January 22, 2020. http://hdl.handle.net/10161/5772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fuller, Deirdre Marie. “Analysis of TCR Signaling and Erk Activation in T Cell Development and Autoimmunity .” 2012. Web. 22 Jan 2020.

Vancouver:

Fuller DM. Analysis of TCR Signaling and Erk Activation in T Cell Development and Autoimmunity . [Internet] [Thesis]. Duke University; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10161/5772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fuller DM. Analysis of TCR Signaling and Erk Activation in T Cell Development and Autoimmunity . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Appleman, Victoria A. Mechanisms of KRAS-Mediated Pancreatic Tumor Formation and Progression: A Dissertation.

Degree: Interdisciplinary Graduate Program, Molecular, Cell and Cancer Biology, 2012, U of Massachusetts : Med

  Pancreatic cancer is the 4th leading cause of cancer related death in the United States with a median survival time of less than 6… (more)

Subjects/Keywords: Pancreatic Neoplasms; Proto-Oncogene Proteins; ras Proteins; Proto-Oncogene Proteins B-raf; Amino Acids, Peptides, and Proteins; Cancer Biology; Digestive System Diseases; Endocrine System Diseases; Neoplasms

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APA (6th Edition):

Appleman, V. A. (2012). Mechanisms of KRAS-Mediated Pancreatic Tumor Formation and Progression: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/600

Chicago Manual of Style (16th Edition):

Appleman, Victoria A. “Mechanisms of KRAS-Mediated Pancreatic Tumor Formation and Progression: A Dissertation.” 2012. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 22, 2020. https://escholarship.umassmed.edu/gsbs_diss/600.

MLA Handbook (7th Edition):

Appleman, Victoria A. “Mechanisms of KRAS-Mediated Pancreatic Tumor Formation and Progression: A Dissertation.” 2012. Web. 22 Jan 2020.

Vancouver:

Appleman VA. Mechanisms of KRAS-Mediated Pancreatic Tumor Formation and Progression: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2012. [cited 2020 Jan 22]. Available from: https://escholarship.umassmed.edu/gsbs_diss/600.

Council of Science Editors:

Appleman VA. Mechanisms of KRAS-Mediated Pancreatic Tumor Formation and Progression: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2012. Available from: https://escholarship.umassmed.edu/gsbs_diss/600

15. Schlummer, Stefanie. Synthese und biologische Evaluierung von Lipo-, Glyco- und Phosphopeptiden.

Degree: 2005, Universität Dortmund

 Um Aussagen über komplexe molekularbiologische Vorgänge zu treffen, werden an diesen Prozessen beteiligte Proteine bzw. Partialstrukturen dieser Proteine als wichtige Hilfsmittel eingesetzt. Viele dieser Proteine… (more)

Subjects/Keywords: Glycopeptide; glycopeptides; Kernimport; Lipopeptide; lipopeptides; NLS; NLS; N-Ras; N-Ras; nuclear import; Phosphopeptide; phosphopeptides; semisynthetic Proteins; Semisynthetische Proteine; v-Jun-protein; v-Jun-Protein; 540

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schlummer, S. (2005). Synthese und biologische Evaluierung von Lipo-, Glyco- und Phosphopeptiden. (Thesis). Universität Dortmund. Retrieved from http://hdl.handle.net/2003/21355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schlummer, Stefanie. “Synthese und biologische Evaluierung von Lipo-, Glyco- und Phosphopeptiden.” 2005. Thesis, Universität Dortmund. Accessed January 22, 2020. http://hdl.handle.net/2003/21355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schlummer, Stefanie. “Synthese und biologische Evaluierung von Lipo-, Glyco- und Phosphopeptiden.” 2005. Web. 22 Jan 2020.

Vancouver:

Schlummer S. Synthese und biologische Evaluierung von Lipo-, Glyco- und Phosphopeptiden. [Internet] [Thesis]. Universität Dortmund; 2005. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2003/21355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schlummer S. Synthese und biologische Evaluierung von Lipo-, Glyco- und Phosphopeptiden. [Thesis]. Universität Dortmund; 2005. Available from: http://hdl.handle.net/2003/21355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Salamanca

16. Ginel Picardo, Alicia. Caracterización genómica y funcional de fibroblastos embrionarios derivados de ratones knockout para Sos1 y Sos2 .

Degree: 2012, Universidad de Salamanca

 [ES] Las proteínas de la familia Sos (Son of Sevenless), formada por dos miembros, Sos1 y Sos2, funcionan como activadores de GTPasas Ras catalizando el… (more)

Subjects/Keywords: Tesis y disertaciones académicas; Universidad de Salamanca (España); Tesis Doctoral; Genética molecular; Biología molecular; Citología; Proteínas ras; Academic dissertations; Molecular genetics; Molecular biology; Cytology; Ras proteins

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APA (6th Edition):

Ginel Picardo, A. (2012). Caracterización genómica y funcional de fibroblastos embrionarios derivados de ratones knockout para Sos1 y Sos2 . (Thesis). Universidad de Salamanca. Retrieved from http://hdl.handle.net/10366/121162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ginel Picardo, Alicia. “Caracterización genómica y funcional de fibroblastos embrionarios derivados de ratones knockout para Sos1 y Sos2 .” 2012. Thesis, Universidad de Salamanca. Accessed January 22, 2020. http://hdl.handle.net/10366/121162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ginel Picardo, Alicia. “Caracterización genómica y funcional de fibroblastos embrionarios derivados de ratones knockout para Sos1 y Sos2 .” 2012. Web. 22 Jan 2020.

Vancouver:

Ginel Picardo A. Caracterización genómica y funcional de fibroblastos embrionarios derivados de ratones knockout para Sos1 y Sos2 . [Internet] [Thesis]. Universidad de Salamanca; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10366/121162.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ginel Picardo A. Caracterización genómica y funcional de fibroblastos embrionarios derivados de ratones knockout para Sos1 y Sos2 . [Thesis]. Universidad de Salamanca; 2012. Available from: http://hdl.handle.net/10366/121162

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Estrozi, Bruna. Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos).

Degree: PhD, Patologia, 2015, University of São Paulo

A incidência do melanoma cutâneo em pacientes adultos jovens tem aumentado consideravelmente nos últimos anos. Há, contudo, carência de conhecimentos clinicopatológicos e moleculares sobre os… (more)

Subjects/Keywords: Adulto jovem; Melanoma; Melanoma; Neoplasias cutâneas; Proteínas proto-oncogênicas B-raf; Proteínas proto-oncogênicas c-kit; Proteínas proto-oncogênicas p21(ras); Proto-oncogene proteins B-raf; Proto-oncogene proteins c-kit; Proto-oncogene proteins p21(ras); Skin neoplasms; Younf adult

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Estrozi, B. (2015). Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos). (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;

Chicago Manual of Style (16th Edition):

Estrozi, Bruna. “Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos).” 2015. Doctoral Dissertation, University of São Paulo. Accessed January 22, 2020. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;.

MLA Handbook (7th Edition):

Estrozi, Bruna. “Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos).” 2015. Web. 22 Jan 2020.

Vancouver:

Estrozi B. Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos). [Internet] [Doctoral dissertation]. University of São Paulo; 2015. [cited 2020 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;.

Council of Science Editors:

Estrozi B. Avaliação anatomoclínica e molecular do melanoma cutâneo em pacientes jovens (idade 18-30 anos). [Doctoral Dissertation]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-01042015-144721/ ;

18. Chandra, Anchal. The space-time continuum of ras signal transduction pathway.

Degree: 2011, Technische Universität Dortmund

 Die räumliche Aufteilung periphärer Membran-Proteine, wie GNBP (Ras) moduliert deren Spezifität der Signalverarbeitung innerhalb der Zelle. Reversible Palmitoylierung durch Palmitoylacyltransferasen (PATs der DHHC der DHHC… (more)

Subjects/Keywords: Dictyostelium; FLAP; FRAP; FRET-FLIM; Kras; mPDAC; Palmitoylation; PDEб; RA/RBD domain; Ras family proteins; 540; 570

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APA (6th Edition):

Chandra, A. (2011). The space-time continuum of ras signal transduction pathway. (Thesis). Technische Universität Dortmund. Retrieved from http://hdl.handle.net/2003/29179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chandra, Anchal. “The space-time continuum of ras signal transduction pathway.” 2011. Thesis, Technische Universität Dortmund. Accessed January 22, 2020. http://hdl.handle.net/2003/29179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chandra, Anchal. “The space-time continuum of ras signal transduction pathway.” 2011. Web. 22 Jan 2020.

Vancouver:

Chandra A. The space-time continuum of ras signal transduction pathway. [Internet] [Thesis]. Technische Universität Dortmund; 2011. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2003/29179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chandra A. The space-time continuum of ras signal transduction pathway. [Thesis]. Technische Universität Dortmund; 2011. Available from: http://hdl.handle.net/2003/29179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Driscoll, David R. The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation.

Degree: Interdisciplinary Graduate Program, Molecular, Cell and Cancer Biology Department, 2016, U of Massachusetts : Med

  Pancreatic ductal adenocarcinoma (PDAC), the most common form of pancreatic cancer, develops through progression of premalignant pancreatic intraepithelial neoplasias (PanINs). In mouse-models, KRAS-activation in… (more)

Subjects/Keywords: Pancreatic Ductal Carcinoma; Multiprotein Complexes; TOR Serine-Threonine Kinases; Proto-Oncogene Proteins p21(ras); Cancer Biology; Neoplasms

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APA (6th Edition):

Driscoll, D. R. (2016). The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/821

Chicago Manual of Style (16th Edition):

Driscoll, David R. “The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 22, 2020. http://escholarship.umassmed.edu/gsbs_diss/821.

MLA Handbook (7th Edition):

Driscoll, David R. “The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation.” 2016. Web. 22 Jan 2020.

Vancouver:

Driscoll DR. The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2020 Jan 22]. Available from: http://escholarship.umassmed.edu/gsbs_diss/821.

Council of Science Editors:

Driscoll DR. The Impact of mTORC2 Signaling on the Initiation and Progression of KRAS-Driven Pancreatic Neoplasias: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/821


University of Texas Southwestern Medical Center

20. Chen, Chiyuan. Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP.

Degree: 2007, University of Texas Southwestern Medical Center

 The RAF/MEK/MAP Kinase signal transduction cascade is the most extensively studied MAPK pathway that mediates diverse cellular responses to environmental cues, and makes a major… (more)

Subjects/Keywords: ras Proteins; MAP Kinase Signaling System; Ubiquitin-Protein Ligases

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APA (6th Edition):

Chen, C. (2007). Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Chiyuan. “Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP.” 2007. Thesis, University of Texas Southwestern Medical Center. Accessed January 22, 2020. http://hdl.handle.net/2152.5/593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Chiyuan. “Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP.” 2007. Web. 22 Jan 2020.

Vancouver:

Chen C. Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2007. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2152.5/593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen C. Mechanistic Basis of Signal Amplitude Modulation by the Ras Effector IMP. [Thesis]. University of Texas Southwestern Medical Center; 2007. Available from: http://hdl.handle.net/2152.5/593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

21. Kim, Ji Mi. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.

Degree: 2016, University of Texas Southwestern Medical Center

 Activating mutations in KRAS are frequently involved in the pathogenesis of non-small cell lung cancer (NSCLC), the disease responsible for the most cancer-related deaths in… (more)

Subjects/Keywords: Active Transport, Cell Nucleus; Cell Nucleus; Karyopherins; Lung Neoplasms; Proto-Oncogene Proteins p21(ras); Receptors, Cytoplasmic and Nuclear

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APA (6th Edition):

Kim, J. M. (2016). Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/5726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Ji Mi. “Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed January 22, 2020. http://hdl.handle.net/2152.5/5726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Ji Mi. “Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer.” 2016. Web. 22 Jan 2020.

Vancouver:

Kim JM. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2152.5/5726.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim JM. Identification of Oncogenic KRAS-Associated Vulnerabilities in Non-Small Cell Lung Cancer. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/5726

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

22. Garcia-Torres, Desiree. Investigating the Regulation of Ras Protein Prenylation.

Degree: PhD, Chemical Biology, 2019, University of Michigan

Ras family small GTPases undergo prenylation for proper localization to the plasma membrane, where they carry out their signaling function. Prevalent mutations in these proteins(more)

Subjects/Keywords: Ras proteins; prenylation; small GTP-binding protein GDP-dissociation stimulator (SmgGDS); small GTPases; farnesyltransferase (FTase); Biological Chemistry; Science

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APA (6th Edition):

Garcia-Torres, D. (2019). Investigating the Regulation of Ras Protein Prenylation. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/151587

Chicago Manual of Style (16th Edition):

Garcia-Torres, Desiree. “Investigating the Regulation of Ras Protein Prenylation.” 2019. Doctoral Dissertation, University of Michigan. Accessed January 22, 2020. http://hdl.handle.net/2027.42/151587.

MLA Handbook (7th Edition):

Garcia-Torres, Desiree. “Investigating the Regulation of Ras Protein Prenylation.” 2019. Web. 22 Jan 2020.

Vancouver:

Garcia-Torres D. Investigating the Regulation of Ras Protein Prenylation. [Internet] [Doctoral dissertation]. University of Michigan; 2019. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2027.42/151587.

Council of Science Editors:

Garcia-Torres D. Investigating the Regulation of Ras Protein Prenylation. [Doctoral Dissertation]. University of Michigan; 2019. Available from: http://hdl.handle.net/2027.42/151587


University of Gothenburg / Göteborgs Universitet

23. Wahlström, Annika. Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer.

Degree: 2009, University of Gothenburg / Göteborgs Universitet

 CAAX proteins, such as the RAS and RHO proteins, are recognized by a specific CAAX motif at the carboxyl terminus, which undergoes posttranslational modifications. First,… (more)

Subjects/Keywords: RCE1; ICMT; K-RAS; CAAX proteins; MPD; lung cancer

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APA (6th Edition):

Wahlström, A. (2009). Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/19643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wahlström, Annika. “Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer.” 2009. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 22, 2020. http://hdl.handle.net/2077/19643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wahlström, Annika. “Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer.” 2009. Web. 22 Jan 2020.

Vancouver:

Wahlström A. Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2077/19643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wahlström A. Defining RCE1 and ICMT as Therapeutic Targets in K-RAS-induced Cancer. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. Available from: http://hdl.handle.net/2077/19643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Oliveira Filho, João Bosco de. Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana.

Degree: PhD, Patologia, 2008, University of São Paulo

A subfamília p21 RAS de pequenas GTPases, incluindo KRAS, HRAS e NRAS, participa de muitas redes de sinalização, incluindo proliferação celular, organização do citoesqueleto e… (more)

Subjects/Keywords: Apoptose; Apoptosis; Autoimmunity; Autoimunidade; BCL-2 -like protein 11; BIM; Mitogen activated protein kinase kinases; Proteínas proto-oncogênicas p21 (ras); Proto-oncogene proteins p21 (ras); Quinases de proteína quinase ativadas por mitógeno

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APA (6th Edition):

Oliveira Filho, J. B. d. (2008). Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;

Chicago Manual of Style (16th Edition):

Oliveira Filho, João Bosco de. “Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana.” 2008. Doctoral Dissertation, University of São Paulo. Accessed January 22, 2020. http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;.

MLA Handbook (7th Edition):

Oliveira Filho, João Bosco de. “Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana.” 2008. Web. 22 Jan 2020.

Vancouver:

Oliveira Filho JBd. Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana. [Internet] [Doctoral dissertation]. University of São Paulo; 2008. [cited 2020 Jan 22]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;.

Council of Science Editors:

Oliveira Filho JBd. Mutação em NRAS causa uma síndrome autoimune linfoproliferativa humana. [Doctoral Dissertation]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/5/5144/tde-04112008-174252/ ;


Universidad de Salamanca

25. Grande Rodríguez, María Teresa. La vía de Ras en un modelo de fibrosis renal experimental .

Degree: 2009, Universidad de Salamanca

 [ES] La fibrosis renal es una característica común de la enfermedad renal crónica independientemente de su etiología. Numerosas citoquinas involucradas en el desarrollo de la… (more)

Subjects/Keywords: Tesis y disertaciones académicas; Universidad de Salamanca (España); Academic dissertations; Riñones (Enfermedades); Kidneys-Diseases; Proteínas ras; Ras proteins

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APA (6th Edition):

Grande Rodríguez, M. T. (2009). La vía de Ras en un modelo de fibrosis renal experimental . (Thesis). Universidad de Salamanca. Retrieved from http://hdl.handle.net/10366/76272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grande Rodríguez, María Teresa. “La vía de Ras en un modelo de fibrosis renal experimental .” 2009. Thesis, Universidad de Salamanca. Accessed January 22, 2020. http://hdl.handle.net/10366/76272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grande Rodríguez, María Teresa. “La vía de Ras en un modelo de fibrosis renal experimental .” 2009. Web. 22 Jan 2020.

Vancouver:

Grande Rodríguez MT. La vía de Ras en un modelo de fibrosis renal experimental . [Internet] [Thesis]. Universidad de Salamanca; 2009. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10366/76272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grande Rodríguez MT. La vía de Ras en un modelo de fibrosis renal experimental . [Thesis]. Universidad de Salamanca; 2009. Available from: http://hdl.handle.net/10366/76272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Quattrochi, Brian J. Subtle Controllers: MicroRNAs Drive Pancreatic Tumorigenesis and Progression: A Dissertation.

Degree: PhD, Molecular, Cell and Cancer Biology Department, 2015, U of Massachusetts : Med

  Pancreatic ductal adenocarcinoma (PDAC) is among the most lethal malignancies in the United States, with an average five-year survival rate of just 6.7%. One… (more)

Subjects/Keywords: Pancreatic Ductal Carcinoma; Carcinogenesis; Neoplastic Cell Transformation; ras Genes; MicroRNAs; Oncogenes; Proto-Oncogene Proteins; Cancer Biology; Genetics and Genomics; Neoplasms; Nucleic Acids, Nucleotides, and Nucleosides; Oncology

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APA (6th Edition):

Quattrochi, B. J. (2015). Subtle Controllers: MicroRNAs Drive Pancreatic Tumorigenesis and Progression: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/776

Chicago Manual of Style (16th Edition):

Quattrochi, Brian J. “Subtle Controllers: MicroRNAs Drive Pancreatic Tumorigenesis and Progression: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 22, 2020. http://escholarship.umassmed.edu/gsbs_diss/776.

MLA Handbook (7th Edition):

Quattrochi, Brian J. “Subtle Controllers: MicroRNAs Drive Pancreatic Tumorigenesis and Progression: A Dissertation.” 2015. Web. 22 Jan 2020.

Vancouver:

Quattrochi BJ. Subtle Controllers: MicroRNAs Drive Pancreatic Tumorigenesis and Progression: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2020 Jan 22]. Available from: http://escholarship.umassmed.edu/gsbs_diss/776.

Council of Science Editors:

Quattrochi BJ. Subtle Controllers: MicroRNAs Drive Pancreatic Tumorigenesis and Progression: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/776


Ryerson University

27. Tucholska, Monika. RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages.

Degree: 2008, Ryerson University

 The Fcγ receptor is a cell surface protein essential in the immune response that binds IgG-opsonized particles resulting in phagocytosis. Phagocytosis is a process used… (more)

Subjects/Keywords: Ras proteins; Guanosine triphosphatase genes; GTPase-activating protein; Fc receptors; Cellular immunity; Phagocytosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tucholska, M. (2008). RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A970

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tucholska, Monika. “RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages.” 2008. Thesis, Ryerson University. Accessed January 22, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A970.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tucholska, Monika. “RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages.” 2008. Web. 22 Jan 2020.

Vancouver:

Tucholska M. RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages. [Internet] [Thesis]. Ryerson University; 2008. [cited 2020 Jan 22]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A970.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tucholska M. RAP, a member of the Ras superfamily of small GTPases and its putative GTPase activating proteins and guanine nucleotide exchange factors in raw 264.7 macrophages. [Thesis]. Ryerson University; 2008. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A970

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McGill University

28. Kalantari, Fariba. A cellular and molecular approach to investigate pathological calcification in liver.

Degree: PhD, Department of Anatomy and Cell Biology., 2008, McGill University

The liver is a vital organ, playing numerous critical roles in the body. The liver's ability to perform essential functions is disturbed by injuries that… (more)

Subjects/Keywords: Calcinosis  – etiology.; Liver Diseases  – pathology.; Liver Transplantation  – pathology.; ras GTPase-Activating Proteins  – metabolism.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kalantari, F. (2008). A cellular and molecular approach to investigate pathological calcification in liver. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile111907.pdf

Chicago Manual of Style (16th Edition):

Kalantari, Fariba. “A cellular and molecular approach to investigate pathological calcification in liver.” 2008. Doctoral Dissertation, McGill University. Accessed January 22, 2020. http://digitool.library.mcgill.ca/thesisfile111907.pdf.

MLA Handbook (7th Edition):

Kalantari, Fariba. “A cellular and molecular approach to investigate pathological calcification in liver.” 2008. Web. 22 Jan 2020.

Vancouver:

Kalantari F. A cellular and molecular approach to investigate pathological calcification in liver. [Internet] [Doctoral dissertation]. McGill University; 2008. [cited 2020 Jan 22]. Available from: http://digitool.library.mcgill.ca/thesisfile111907.pdf.

Council of Science Editors:

Kalantari F. A cellular and molecular approach to investigate pathological calcification in liver. [Doctoral Dissertation]. McGill University; 2008. Available from: http://digitool.library.mcgill.ca/thesisfile111907.pdf


University of Groningen

29. Fatrai, Szabolcs. The role of STAT5 and KRAS in hematopoiesis and acute myeoloid leukemia.

Degree: Faculty of Medical Sciences, 2010, University of Groningen

Subjects/Keywords: Proefschriften (vorm); Transcriptiefactoren; Ras-proteins; Stamcellen; Myeloïde leukemie; Leukemie; Acute aandoeningen; Bloedvorming; hematologie

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fatrai, S. (2010). The role of STAT5 and KRAS in hematopoiesis and acute myeoloid leukemia. (Doctoral Dissertation). University of Groningen. Retrieved from https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; a89b51c1-2041-4b13-b531-4083bd41bb47 ; 11370/a89b51c1-2041-4b13-b531-4083bd41bb47 ; urn:isbn:9789036745390 ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html

Chicago Manual of Style (16th Edition):

Fatrai, Szabolcs. “The role of STAT5 and KRAS in hematopoiesis and acute myeoloid leukemia.” 2010. Doctoral Dissertation, University of Groningen. Accessed January 22, 2020. https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; a89b51c1-2041-4b13-b531-4083bd41bb47 ; 11370/a89b51c1-2041-4b13-b531-4083bd41bb47 ; urn:isbn:9789036745390 ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html.

MLA Handbook (7th Edition):

Fatrai, Szabolcs. “The role of STAT5 and KRAS in hematopoiesis and acute myeoloid leukemia.” 2010. Web. 22 Jan 2020.

Vancouver:

Fatrai S. The role of STAT5 and KRAS in hematopoiesis and acute myeoloid leukemia. [Internet] [Doctoral dissertation]. University of Groningen; 2010. [cited 2020 Jan 22]. Available from: https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; a89b51c1-2041-4b13-b531-4083bd41bb47 ; 11370/a89b51c1-2041-4b13-b531-4083bd41bb47 ; urn:isbn:9789036745390 ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html.

Council of Science Editors:

Fatrai S. The role of STAT5 and KRAS in hematopoiesis and acute myeoloid leukemia. [Doctoral Dissertation]. University of Groningen; 2010. Available from: https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; a89b51c1-2041-4b13-b531-4083bd41bb47 ; 11370/a89b51c1-2041-4b13-b531-4083bd41bb47 ; urn:isbn:9789036745390 ; urn:nbn:nl:ui:11-dbi/4cb42a6c6885b ; https://www.rug.nl/research/portal/en/publications/the-role-of-stat5-and-kras-in-hematopoiesis-and-acute-myeoloid-leukemia(a89b51c1-2041-4b13-b531-4083bd41bb47).html


University of Gothenburg / Göteborgs Universitet

30. Sjögren, Anna-Karin. The Importance of Isoprenylation and Nf1 Deficiency in K-RAS-induced Cancer.

Degree: 2009, University of Gothenburg / Göteborgs Universitet

 The RAS and RHO family proteins contribute to tumorigenesis and metastasis and belong to a family of so called CAAX proteins. The membrane targeting and… (more)

Subjects/Keywords: FTase; GGTase-I; isoprenylation; K-RAS; NF1; CAAX proteins; lung cancer; AML

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sjögren, A. (2009). The Importance of Isoprenylation and Nf1 Deficiency in K-RAS-induced Cancer. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/21218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sjögren, Anna-Karin. “The Importance of Isoprenylation and Nf1 Deficiency in K-RAS-induced Cancer.” 2009. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 22, 2020. http://hdl.handle.net/2077/21218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sjögren, Anna-Karin. “The Importance of Isoprenylation and Nf1 Deficiency in K-RAS-induced Cancer.” 2009. Web. 22 Jan 2020.

Vancouver:

Sjögren A. The Importance of Isoprenylation and Nf1 Deficiency in K-RAS-induced Cancer. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2077/21218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sjögren A. The Importance of Isoprenylation and Nf1 Deficiency in K-RAS-induced Cancer. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2009. Available from: http://hdl.handle.net/2077/21218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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