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You searched for subject:(protease activated receptor). Showing records 1 – 30 of 32 total matches.

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Vanderbilt University

1. Young, Summer Elizabeth. Modulation of thrombin receptor signaling.

Degree: PhD, Pharmacology, 2013, Vanderbilt University

 The platelet thrombin receptors Protease-activated receptor 1 (PAR1) and Protease activated receptor 4 (PAR4) stand at the intersection of coagulation and platelet activation. Thrombin receptors… (more)

Subjects/Keywords: protease-activated receptor; thrombosis; thrombin receptors

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APA (6th Edition):

Young, S. E. (2013). Modulation of thrombin receptor signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14534

Chicago Manual of Style (16th Edition):

Young, Summer Elizabeth. “Modulation of thrombin receptor signaling.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 28, 2020. http://hdl.handle.net/1803/14534.

MLA Handbook (7th Edition):

Young, Summer Elizabeth. “Modulation of thrombin receptor signaling.” 2013. Web. 28 Oct 2020.

Vancouver:

Young SE. Modulation of thrombin receptor signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1803/14534.

Council of Science Editors:

Young SE. Modulation of thrombin receptor signaling. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/14534


Temple University

2. Mao, Yingying. ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION.

Degree: PhD, 2009, Temple University

Physiology

Platelets act as a fundamental component of the hemostatic process and their activation leads to the formation of a stable clot at the injured… (more)

Subjects/Keywords: Biology, Physiology; ischemic injury; PAR1 agonist; plasmin; platelet; protease-activated receptor

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APA (6th Edition):

Mao, Y. (2009). ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,47279

Chicago Manual of Style (16th Edition):

Mao, Yingying. “ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION.” 2009. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,47279.

MLA Handbook (7th Edition):

Mao, Yingying. “ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION.” 2009. Web. 28 Oct 2020.

Vancouver:

Mao Y. ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,47279.

Council of Science Editors:

Mao Y. ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,47279


Vanderbilt University

3. Schulte, Michael Lawrence. Application of Organocatalysis to the Synthesis of Pharmaceutically Relevant Scaffolds: Chiral Aziridines and β-Fluoroamines; Total Synthesis of Stemaphylline; and Discovery of Selective PAR4 Antagonists.

Degree: PhD, Chemistry, 2013, Vanderbilt University

 Aziridines represent an important class of nitrogen-containing heterocycles with a wide range of synthetic utility. Despite their value, synthetic approaches are limited in terms of… (more)

Subjects/Keywords: organocatalysis; aziridines; fluoroamines; stemaphylline; stemaphylline N-oxide; protease activated receptor

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APA (6th Edition):

Schulte, M. L. (2013). Application of Organocatalysis to the Synthesis of Pharmaceutically Relevant Scaffolds: Chiral Aziridines and β-Fluoroamines; Total Synthesis of Stemaphylline; and Discovery of Selective PAR4 Antagonists. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13263

Chicago Manual of Style (16th Edition):

Schulte, Michael Lawrence. “Application of Organocatalysis to the Synthesis of Pharmaceutically Relevant Scaffolds: Chiral Aziridines and β-Fluoroamines; Total Synthesis of Stemaphylline; and Discovery of Selective PAR4 Antagonists.” 2013. Doctoral Dissertation, Vanderbilt University. Accessed October 28, 2020. http://hdl.handle.net/1803/13263.

MLA Handbook (7th Edition):

Schulte, Michael Lawrence. “Application of Organocatalysis to the Synthesis of Pharmaceutically Relevant Scaffolds: Chiral Aziridines and β-Fluoroamines; Total Synthesis of Stemaphylline; and Discovery of Selective PAR4 Antagonists.” 2013. Web. 28 Oct 2020.

Vancouver:

Schulte ML. Application of Organocatalysis to the Synthesis of Pharmaceutically Relevant Scaffolds: Chiral Aziridines and β-Fluoroamines; Total Synthesis of Stemaphylline; and Discovery of Selective PAR4 Antagonists. [Internet] [Doctoral dissertation]. Vanderbilt University; 2013. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1803/13263.

Council of Science Editors:

Schulte ML. Application of Organocatalysis to the Synthesis of Pharmaceutically Relevant Scaffolds: Chiral Aziridines and β-Fluoroamines; Total Synthesis of Stemaphylline; and Discovery of Selective PAR4 Antagonists. [Doctoral Dissertation]. Vanderbilt University; 2013. Available from: http://hdl.handle.net/1803/13263


University of Melbourne

4. Saeed, Muhammad Azeem. The role of Protease-activated receptor 1 in inflammatory bowel disease.

Degree: 2015, University of Melbourne

 Inflammatory bowel disease (IBD), a group of chronic, debilitating inflammatory disorders of the gastrointestinal tract, is a common cause of morbidity and mortality worldwide. Crohn’s… (more)

Subjects/Keywords: Protease-activated receptor 1; inflammatory bowel disease; colitis; citrobacter rodentium

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APA (6th Edition):

Saeed, M. A. (2015). The role of Protease-activated receptor 1 in inflammatory bowel disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/59618

Chicago Manual of Style (16th Edition):

Saeed, Muhammad Azeem. “The role of Protease-activated receptor 1 in inflammatory bowel disease.” 2015. Doctoral Dissertation, University of Melbourne. Accessed October 28, 2020. http://hdl.handle.net/11343/59618.

MLA Handbook (7th Edition):

Saeed, Muhammad Azeem. “The role of Protease-activated receptor 1 in inflammatory bowel disease.” 2015. Web. 28 Oct 2020.

Vancouver:

Saeed MA. The role of Protease-activated receptor 1 in inflammatory bowel disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/11343/59618.

Council of Science Editors:

Saeed MA. The role of Protease-activated receptor 1 in inflammatory bowel disease. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/59618


University of New South Wales

5. Castano Rodriguez, Natalia. The role of innate immunity in Helicobacter pylori-related gastric cancer.

Degree: Biotechnology & Biomolecular Sciences, 2014, University of New South Wales

 Gastric cancer (GC) is the fifth most common cancer and the third most common cause of cancer-related death worldwide. While Helicobacter pylori infection has been… (more)

Subjects/Keywords: stomach neoplasm; Helicobacter pylori; innate immunity; pattern recognition receptor; protease activated receptor; autophagy

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APA (6th Edition):

Castano Rodriguez, N. (2014). The role of innate immunity in Helicobacter pylori-related gastric cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53964 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12675/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Castano Rodriguez, Natalia. “The role of innate immunity in Helicobacter pylori-related gastric cancer.” 2014. Doctoral Dissertation, University of New South Wales. Accessed October 28, 2020. http://handle.unsw.edu.au/1959.4/53964 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12675/SOURCE02?view=true.

MLA Handbook (7th Edition):

Castano Rodriguez, Natalia. “The role of innate immunity in Helicobacter pylori-related gastric cancer.” 2014. Web. 28 Oct 2020.

Vancouver:

Castano Rodriguez N. The role of innate immunity in Helicobacter pylori-related gastric cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2020 Oct 28]. Available from: http://handle.unsw.edu.au/1959.4/53964 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12675/SOURCE02?view=true.

Council of Science Editors:

Castano Rodriguez N. The role of innate immunity in Helicobacter pylori-related gastric cancer. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53964 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12675/SOURCE02?view=true

6. José Américo Gonçalves Fagundes. Avaliação da expressão do receptor tipo 2 ativado por protease (PAR2) e da atividade proteolítica presente no fluido crevicular de pacientes com periodontite crônica.

Degree: 2010, Universidade de Taubaté

O receptor tipo 2 ativado por protease (PAR2) é um receptor pro-inflamatório que pode ser ativado por tripsina, triptase, protease 3 produzida pelo neutrófilo e… (more)

Subjects/Keywords: doença periodontal; receptor tipo 2 ativado por protease; proteases; inflamação; periodontal disease; protease-activated receptor-2; proteases; inflammation; ODONTOLOGIA

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APA (6th Edition):

Fagundes, J. A. G. (2010). Avaliação da expressão do receptor tipo 2 ativado por protease (PAR2) e da atividade proteolítica presente no fluido crevicular de pacientes com periodontite crônica. (Thesis). Universidade de Taubaté. Retrieved from http://www.bdtd.unitau.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fagundes, José Américo Gonçalves. “Avaliação da expressão do receptor tipo 2 ativado por protease (PAR2) e da atividade proteolítica presente no fluido crevicular de pacientes com periodontite crônica.” 2010. Thesis, Universidade de Taubaté. Accessed October 28, 2020. http://www.bdtd.unitau.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fagundes, José Américo Gonçalves. “Avaliação da expressão do receptor tipo 2 ativado por protease (PAR2) e da atividade proteolítica presente no fluido crevicular de pacientes com periodontite crônica.” 2010. Web. 28 Oct 2020.

Vancouver:

Fagundes JAG. Avaliação da expressão do receptor tipo 2 ativado por protease (PAR2) e da atividade proteolítica presente no fluido crevicular de pacientes com periodontite crônica. [Internet] [Thesis]. Universidade de Taubaté; 2010. [cited 2020 Oct 28]. Available from: http://www.bdtd.unitau.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=429.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fagundes JAG. Avaliação da expressão do receptor tipo 2 ativado por protease (PAR2) e da atividade proteolítica presente no fluido crevicular de pacientes com periodontite crônica. [Thesis]. Universidade de Taubaté; 2010. Available from: http://www.bdtd.unitau.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=429

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

7. Frei, Jan Niclas. Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1.

Degree: PhD, 2020, University of Cambridge

 The research conducted for this thesis was aimed at providing a comprehensive description of the dynamic and conformational landscape of the minimally thermostabilised avian β1-adrenergic… (more)

Subjects/Keywords: G protein-coupled receptor; Nuclear magnetic resonance spectroscopy; β1-adrenergic receptor; Protease-activated receptor 1; Second harmonic generation

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APA (6th Edition):

Frei, J. N. (2020). Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/303160

Chicago Manual of Style (16th Edition):

Frei, Jan Niclas. “Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1.” 2020. Doctoral Dissertation, University of Cambridge. Accessed October 28, 2020. https://www.repository.cam.ac.uk/handle/1810/303160.

MLA Handbook (7th Edition):

Frei, Jan Niclas. “Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1.” 2020. Web. 28 Oct 2020.

Vancouver:

Frei JN. Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2020 Oct 28]. Available from: https://www.repository.cam.ac.uk/handle/1810/303160.

Council of Science Editors:

Frei JN. Characterisation of the conformational landscape of the β1-adrenergic receptor and development of a strategy towards NMR studies of the protease-activated receptor 1. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/303160


University of Cincinnati

8. Hall, David. The effects of protease-activated receptor 2 on atherosclerosis.

Degree: MS, Allied Health Sciences: Nutrition, 2016, University of Cincinnati

 Background: Cardiovascular disease (CVD), including heart attack, stroke, and heart failure, is the leading cause of morbidity and mortality worldwide. Atherosclerosis is a progressive disease… (more)

Subjects/Keywords: Nutrition; Atherosclerosis; Protease-activated receptor 2; High-fat diet; Coagulation cascade; Adipose

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APA (6th Edition):

Hall, D. (2016). The effects of protease-activated receptor 2 on atherosclerosis. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459438552

Chicago Manual of Style (16th Edition):

Hall, David. “The effects of protease-activated receptor 2 on atherosclerosis.” 2016. Masters Thesis, University of Cincinnati. Accessed October 28, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459438552.

MLA Handbook (7th Edition):

Hall, David. “The effects of protease-activated receptor 2 on atherosclerosis.” 2016. Web. 28 Oct 2020.

Vancouver:

Hall D. The effects of protease-activated receptor 2 on atherosclerosis. [Internet] [Masters thesis]. University of Cincinnati; 2016. [cited 2020 Oct 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459438552.

Council of Science Editors:

Hall D. The effects of protease-activated receptor 2 on atherosclerosis. [Masters Thesis]. University of Cincinnati; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459438552


University of Pennsylvania

9. Smith, Jenell Rene. Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma.

Degree: 2015, University of Pennsylvania

 Peripheral neural trauma is known to induce blood-spinal cord barrier (BSCB) breakdown, but if and how BSCB breakdown contributes to pain is unknown. The studies… (more)

Subjects/Keywords: activated protein C; blood-spinal cord barrier; nerve root injury; pain; protease-activated receptor; thrombin; Biomedical

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APA (6th Edition):

Smith, J. R. (2015). Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Jenell Rene. “Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma.” 2015. Thesis, University of Pennsylvania. Accessed October 28, 2020. https://repository.upenn.edu/edissertations/2025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Jenell Rene. “Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma.” 2015. Web. 28 Oct 2020.

Vancouver:

Smith JR. Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2020 Oct 28]. Available from: https://repository.upenn.edu/edissertations/2025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith JR. Defining the Role of Blood-Spinal Cord Barrier Breakdown and Spinal Thrombin Signaling tn the Development of Pain Following Neural Trauma. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/2025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Carvalho, João Henrique de. Análise da expressão e atividade de receptores ativados por proteases em plaquetas de pacientes com hipertensão arterial pulmonar.

Degree: Mestrado, Fisiopatologia Experimental, 2009, University of São Paulo

A hipertensão arterial pulmonar é uma síndrome clínica e hemodinâmica, caracterizada pelo aumento de resistência vascular na microcirculação. A vasoconstrição presente na doença ocorre principalmente… (more)

Subjects/Keywords: Hipertensão pulmonar; P-Selectin; Plaquetas; Platelets; Protease activated receptor 1/antagonists; Pulmonary hypertension; Receptor PAR-1/Antagonistas; Selectina-P; Thrombosis; Trombose

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APA (6th Edition):

Carvalho, J. H. d. (2009). Análise da expressão e atividade de receptores ativados por proteases em plaquetas de pacientes com hipertensão arterial pulmonar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01062009-114009/ ;

Chicago Manual of Style (16th Edition):

Carvalho, João Henrique de. “Análise da expressão e atividade de receptores ativados por proteases em plaquetas de pacientes com hipertensão arterial pulmonar.” 2009. Masters Thesis, University of São Paulo. Accessed October 28, 2020. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01062009-114009/ ;.

MLA Handbook (7th Edition):

Carvalho, João Henrique de. “Análise da expressão e atividade de receptores ativados por proteases em plaquetas de pacientes com hipertensão arterial pulmonar.” 2009. Web. 28 Oct 2020.

Vancouver:

Carvalho JHd. Análise da expressão e atividade de receptores ativados por proteases em plaquetas de pacientes com hipertensão arterial pulmonar. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2020 Oct 28]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01062009-114009/ ;.

Council of Science Editors:

Carvalho JHd. Análise da expressão e atividade de receptores ativados por proteases em plaquetas de pacientes com hipertensão arterial pulmonar. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-01062009-114009/ ;


Vanderbilt University

11. -3743-692X. DEVELOPMENT OF AN IMPROVED IN VIVO TOOL COMPOUND, VU6001221, FOR NON-COMPETITIVE INHIBITION OF CHT, DISCOVERY AND DEVELOPMENT OF M1 PAMS: BENZOMORPHOLINE AND TETRAHYDROQUINOLINE SERIES, AND DEVELOPMENT OF NOVEL, BRAIN-PENETRANT PAR4 ANTAGONISTS.

Degree: PhD, Chemistry, 2020, Vanderbilt University

 Here in, we describe the optimization of high-affinity, choline up-take transporter (CHT) antagonist, ML352, to improved in vivo tool compound, VU6001221. VU6001221, a potent and… (more)

Subjects/Keywords: Choline transporter; CHT; G-protein coupled receptors; muscarinic acetylcholine receptor 1; M1; protease activated receptor 4; PAR4

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APA (6th Edition):

-3743-692X. (2020). DEVELOPMENT OF AN IMPROVED IN VIVO TOOL COMPOUND, VU6001221, FOR NON-COMPETITIVE INHIBITION OF CHT, DISCOVERY AND DEVELOPMENT OF M1 PAMS: BENZOMORPHOLINE AND TETRAHYDROQUINOLINE SERIES, AND DEVELOPMENT OF NOVEL, BRAIN-PENETRANT PAR4 ANTAGONISTS. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10112

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-3743-692X. “DEVELOPMENT OF AN IMPROVED IN VIVO TOOL COMPOUND, VU6001221, FOR NON-COMPETITIVE INHIBITION OF CHT, DISCOVERY AND DEVELOPMENT OF M1 PAMS: BENZOMORPHOLINE AND TETRAHYDROQUINOLINE SERIES, AND DEVELOPMENT OF NOVEL, BRAIN-PENETRANT PAR4 ANTAGONISTS.” 2020. Doctoral Dissertation, Vanderbilt University. Accessed October 28, 2020. http://hdl.handle.net/1803/10112.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-3743-692X. “DEVELOPMENT OF AN IMPROVED IN VIVO TOOL COMPOUND, VU6001221, FOR NON-COMPETITIVE INHIBITION OF CHT, DISCOVERY AND DEVELOPMENT OF M1 PAMS: BENZOMORPHOLINE AND TETRAHYDROQUINOLINE SERIES, AND DEVELOPMENT OF NOVEL, BRAIN-PENETRANT PAR4 ANTAGONISTS.” 2020. Web. 28 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-3743-692X. DEVELOPMENT OF AN IMPROVED IN VIVO TOOL COMPOUND, VU6001221, FOR NON-COMPETITIVE INHIBITION OF CHT, DISCOVERY AND DEVELOPMENT OF M1 PAMS: BENZOMORPHOLINE AND TETRAHYDROQUINOLINE SERIES, AND DEVELOPMENT OF NOVEL, BRAIN-PENETRANT PAR4 ANTAGONISTS. [Internet] [Doctoral dissertation]. Vanderbilt University; 2020. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1803/10112.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-3743-692X. DEVELOPMENT OF AN IMPROVED IN VIVO TOOL COMPOUND, VU6001221, FOR NON-COMPETITIVE INHIBITION OF CHT, DISCOVERY AND DEVELOPMENT OF M1 PAMS: BENZOMORPHOLINE AND TETRAHYDROQUINOLINE SERIES, AND DEVELOPMENT OF NOVEL, BRAIN-PENETRANT PAR4 ANTAGONISTS. [Doctoral Dissertation]. Vanderbilt University; 2020. Available from: http://hdl.handle.net/1803/10112

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

12. Menou, Awen. Implication des protéases à sérine de la famille des Type II Transmembrane Serine Proteases dans la Fibrose Pulmonaire Idiopathique : Serine proteases of the Type II Transmembrane Serine Proteases family involvement in Idiopathic Pulmonary Fibrosis.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie. Biomolécules et thérapeutiques, 2017, Sorbonne Paris Cité

La Fibrose Pulmonaire Idiopathique (FPI) est une pathologie pulmonaire chronique, progressive, irréversible et mortelle, dont les thérapeutiques sont insuffisantes à ce jour. L'activation de la… (more)

Subjects/Keywords: TTSPs; Matriptase; Human Airway Trypsin-like protease; HAT; Récepteur 2 activé par des protéases; PAR-2; TTSPs; Matriptase; Human Airway Trypsin-like protease; HAT; Protease-Activated Receptor-2; PAR-2

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APA (6th Edition):

Menou, A. (2017). Implication des protéases à sérine de la famille des Type II Transmembrane Serine Proteases dans la Fibrose Pulmonaire Idiopathique : Serine proteases of the Type II Transmembrane Serine Proteases family involvement in Idiopathic Pulmonary Fibrosis. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCC020

Chicago Manual of Style (16th Edition):

Menou, Awen. “Implication des protéases à sérine de la famille des Type II Transmembrane Serine Proteases dans la Fibrose Pulmonaire Idiopathique : Serine proteases of the Type II Transmembrane Serine Proteases family involvement in Idiopathic Pulmonary Fibrosis.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed October 28, 2020. http://www.theses.fr/2017USPCC020.

MLA Handbook (7th Edition):

Menou, Awen. “Implication des protéases à sérine de la famille des Type II Transmembrane Serine Proteases dans la Fibrose Pulmonaire Idiopathique : Serine proteases of the Type II Transmembrane Serine Proteases family involvement in Idiopathic Pulmonary Fibrosis.” 2017. Web. 28 Oct 2020.

Vancouver:

Menou A. Implication des protéases à sérine de la famille des Type II Transmembrane Serine Proteases dans la Fibrose Pulmonaire Idiopathique : Serine proteases of the Type II Transmembrane Serine Proteases family involvement in Idiopathic Pulmonary Fibrosis. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2017USPCC020.

Council of Science Editors:

Menou A. Implication des protéases à sérine de la famille des Type II Transmembrane Serine Proteases dans la Fibrose Pulmonaire Idiopathique : Serine proteases of the Type II Transmembrane Serine Proteases family involvement in Idiopathic Pulmonary Fibrosis. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCC020

13. Alves, Vanessa Tubero Euzébio. Efeito da terapia periodontal sobre a expressão do receptor ativado por protease do tipo 2 (PAR2) em pacientes com periodontite crônica.

Degree: PhD, Periodontia, 2014, University of São Paulo

O receptor ativado por protease do tipo 2 (PAR2) está envolvido na patogênese de doenças inflamatórias crônicas, incluindo a periodontite. O PAR2 pode ser ativado… (more)

Subjects/Keywords: Doença periodontal; Inflamação; Inflammation; Non-cirurgical periodontal treatment; Periodontal diseases; Protease activated receptor-2; Receptor ativado por protease do tipo 2; Tratamento não-cirúrgico periodontal

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APA (6th Edition):

Alves, V. T. E. (2014). Efeito da terapia periodontal sobre a expressão do receptor ativado por protease do tipo 2 (PAR2) em pacientes com periodontite crônica. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23146/tde-22092014-142715/ ;

Chicago Manual of Style (16th Edition):

Alves, Vanessa Tubero Euzébio. “Efeito da terapia periodontal sobre a expressão do receptor ativado por protease do tipo 2 (PAR2) em pacientes com periodontite crônica.” 2014. Doctoral Dissertation, University of São Paulo. Accessed October 28, 2020. http://www.teses.usp.br/teses/disponiveis/23/23146/tde-22092014-142715/ ;.

MLA Handbook (7th Edition):

Alves, Vanessa Tubero Euzébio. “Efeito da terapia periodontal sobre a expressão do receptor ativado por protease do tipo 2 (PAR2) em pacientes com periodontite crônica.” 2014. Web. 28 Oct 2020.

Vancouver:

Alves VTE. Efeito da terapia periodontal sobre a expressão do receptor ativado por protease do tipo 2 (PAR2) em pacientes com periodontite crônica. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Oct 28]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23146/tde-22092014-142715/ ;.

Council of Science Editors:

Alves VTE. Efeito da terapia periodontal sobre a expressão do receptor ativado por protease do tipo 2 (PAR2) em pacientes com periodontite crônica. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/23/23146/tde-22092014-142715/ ;


Queensland University of Technology

14. Ramsay, Andrew John. Kallikrein-related peptidase 4 activation of protease-activated receptor family members and association with prostate cancer.

Degree: 2008, Queensland University of Technology

 Two areas of particular importance in prostate cancer progression are primary tumour development and metastasis. These processes involve a number of physiological events, the mediators… (more)

Subjects/Keywords: prostate cancer; prostate cancer progression; bone metastasis; protease activated receptor (PAR); g protein coupled receptor (GPCR); kallikrein-related peptidase (KLK); serine protease; signal transduction; internalisation

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APA (6th Edition):

Ramsay, A. J. (2008). Kallikrein-related peptidase 4 activation of protease-activated receptor family members and association with prostate cancer. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/29886/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramsay, Andrew John. “Kallikrein-related peptidase 4 activation of protease-activated receptor family members and association with prostate cancer.” 2008. Thesis, Queensland University of Technology. Accessed October 28, 2020. https://eprints.qut.edu.au/29886/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramsay, Andrew John. “Kallikrein-related peptidase 4 activation of protease-activated receptor family members and association with prostate cancer.” 2008. Web. 28 Oct 2020.

Vancouver:

Ramsay AJ. Kallikrein-related peptidase 4 activation of protease-activated receptor family members and association with prostate cancer. [Internet] [Thesis]. Queensland University of Technology; 2008. [cited 2020 Oct 28]. Available from: https://eprints.qut.edu.au/29886/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramsay AJ. Kallikrein-related peptidase 4 activation of protease-activated receptor family members and association with prostate cancer. [Thesis]. Queensland University of Technology; 2008. Available from: https://eprints.qut.edu.au/29886/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. 大槻, 忠良. Thrombin conducts epithelial-mesenchymal transition via protease-activated receptor-1 in human gastric cancer : ヒト胃癌においてトロンビンはPAR1を介して上皮間葉転換を誘導する.

Degree: 博士(医学), University of Fukui / 福井大学

以下に掲載:International Journal of Oncology 45(6) pp.2287-2294 2014. Spandidos Publications 共著者:Tadayoshi Otsuki, Daisuke Fujimoto, Yasuno Hirono, Takanori Goi, Akio Yamaguchi

Subjects/Keywords: protease-activated receptor-1; gastric cancer; epithelial-mesenchynal transition

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APA (6th Edition):

大槻, . (n.d.). Thrombin conducts epithelial-mesenchymal transition via protease-activated receptor-1 in human gastric cancer : ヒト胃癌においてトロンビンはPAR1を介して上皮間葉転換を誘導する. (Thesis). University of Fukui / 福井大学. Retrieved from http://hdl.handle.net/10098/8840

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

大槻, 忠良. “Thrombin conducts epithelial-mesenchymal transition via protease-activated receptor-1 in human gastric cancer : ヒト胃癌においてトロンビンはPAR1を介して上皮間葉転換を誘導する.” Thesis, University of Fukui / 福井大学. Accessed October 28, 2020. http://hdl.handle.net/10098/8840.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

大槻, 忠良. “Thrombin conducts epithelial-mesenchymal transition via protease-activated receptor-1 in human gastric cancer : ヒト胃癌においてトロンビンはPAR1を介して上皮間葉転換を誘導する.” Web. 28 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

大槻 . Thrombin conducts epithelial-mesenchymal transition via protease-activated receptor-1 in human gastric cancer : ヒト胃癌においてトロンビンはPAR1を介して上皮間葉転換を誘導する. [Internet] [Thesis]. University of Fukui / 福井大学; [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10098/8840.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

大槻 . Thrombin conducts epithelial-mesenchymal transition via protease-activated receptor-1 in human gastric cancer : ヒト胃癌においてトロンビンはPAR1を介して上皮間葉転換を誘導する. [Thesis]. University of Fukui / 福井大学; Available from: http://hdl.handle.net/10098/8840

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of California – Riverside

16. Yee, Michael Christopher. Dissection of Alternaria-induced Asthma: Role of the PAR2-beta-arrestin Signaling Axis.

Degree: Biomedical Sciences, 2016, University of California – Riverside

 The goal of this dissertation project was to understand how activation of Protease-activated receptor-2 (PAR2)-beta-arrestin-2-dependent signaling leads to promotion of inflammation in the allergic response… (more)

Subjects/Keywords: Biology; Biochemistry; Immunology; allergic asthma; Alternaria alternata; beta-arrestin-2; biased antagonist; inflammation; Protease-activated receptor-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yee, M. C. (2016). Dissection of Alternaria-induced Asthma: Role of the PAR2-beta-arrestin Signaling Axis. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/9p88q6c8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yee, Michael Christopher. “Dissection of Alternaria-induced Asthma: Role of the PAR2-beta-arrestin Signaling Axis.” 2016. Thesis, University of California – Riverside. Accessed October 28, 2020. http://www.escholarship.org/uc/item/9p88q6c8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yee, Michael Christopher. “Dissection of Alternaria-induced Asthma: Role of the PAR2-beta-arrestin Signaling Axis.” 2016. Web. 28 Oct 2020.

Vancouver:

Yee MC. Dissection of Alternaria-induced Asthma: Role of the PAR2-beta-arrestin Signaling Axis. [Internet] [Thesis]. University of California – Riverside; 2016. [cited 2020 Oct 28]. Available from: http://www.escholarship.org/uc/item/9p88q6c8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yee MC. Dissection of Alternaria-induced Asthma: Role of the PAR2-beta-arrestin Signaling Axis. [Thesis]. University of California – Riverside; 2016. Available from: http://www.escholarship.org/uc/item/9p88q6c8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

17. CHIONH, YOK TENG. The regulatory effects of protease activated receptor 1 on Helicobacter pylori-induced inflammation.

Degree: 2010, University of Melbourne

 Helicobacter pylori, one of the world’s most prevalent pathogens, infects the gastric mucosa of approximately half the human population. These infections cause a wide range… (more)

Subjects/Keywords: Helicobacter pylori; protease activated receptor 1; gastric inflammation

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APA (6th Edition):

CHIONH, Y. T. (2010). The regulatory effects of protease activated receptor 1 on Helicobacter pylori-induced inflammation. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/36258

Chicago Manual of Style (16th Edition):

CHIONH, YOK TENG. “The regulatory effects of protease activated receptor 1 on Helicobacter pylori-induced inflammation.” 2010. Doctoral Dissertation, University of Melbourne. Accessed October 28, 2020. http://hdl.handle.net/11343/36258.

MLA Handbook (7th Edition):

CHIONH, YOK TENG. “The regulatory effects of protease activated receptor 1 on Helicobacter pylori-induced inflammation.” 2010. Web. 28 Oct 2020.

Vancouver:

CHIONH YT. The regulatory effects of protease activated receptor 1 on Helicobacter pylori-induced inflammation. [Internet] [Doctoral dissertation]. University of Melbourne; 2010. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/11343/36258.

Council of Science Editors:

CHIONH YT. The regulatory effects of protease activated receptor 1 on Helicobacter pylori-induced inflammation. [Doctoral Dissertation]. University of Melbourne; 2010. Available from: http://hdl.handle.net/11343/36258

18. Puech, Clémentine. Développement d’un modèle in vitro de Barrière Hémato-Encéphalique humaine pour des études pharmacologiques : Interactions avec les anticoagulants oraux directs : Development of an in vitro model of a human Blood Brain Barrier for pharmacological studies : Interactions with directs oral anticoagulants.

Degree: Docteur es, Sciences de la vie et de la sante, 2018, Lyon

La barrière hémato-encéphalique (BHE) contrôle le passage des médicaments, en partie par la présence d’ATP Binding Cassette (ABC) transporteurs. Dans de nombreuses pathologies cérébrales, la… (more)

Subjects/Keywords: Barrière hémato-encéphalique; ABC transporteurs; Anticoagulants oraux directs; Protease activated receptor-1; Hémorragies intracérébrales; Modèle in vitro; Blood-brain barrier; ABC transporters; Direct Oral Anticoagulants; Protease activated receptor-1; Intracerebral haemorrhages; In vitro model

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APA (6th Edition):

Puech, C. (2018). Développement d’un modèle in vitro de Barrière Hémato-Encéphalique humaine pour des études pharmacologiques : Interactions avec les anticoagulants oraux directs : Development of an in vitro model of a human Blood Brain Barrier for pharmacological studies : Interactions with directs oral anticoagulants. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2018LYSES060

Chicago Manual of Style (16th Edition):

Puech, Clémentine. “Développement d’un modèle in vitro de Barrière Hémato-Encéphalique humaine pour des études pharmacologiques : Interactions avec les anticoagulants oraux directs : Development of an in vitro model of a human Blood Brain Barrier for pharmacological studies : Interactions with directs oral anticoagulants.” 2018. Doctoral Dissertation, Lyon. Accessed October 28, 2020. http://www.theses.fr/2018LYSES060.

MLA Handbook (7th Edition):

Puech, Clémentine. “Développement d’un modèle in vitro de Barrière Hémato-Encéphalique humaine pour des études pharmacologiques : Interactions avec les anticoagulants oraux directs : Development of an in vitro model of a human Blood Brain Barrier for pharmacological studies : Interactions with directs oral anticoagulants.” 2018. Web. 28 Oct 2020.

Vancouver:

Puech C. Développement d’un modèle in vitro de Barrière Hémato-Encéphalique humaine pour des études pharmacologiques : Interactions avec les anticoagulants oraux directs : Development of an in vitro model of a human Blood Brain Barrier for pharmacological studies : Interactions with directs oral anticoagulants. [Internet] [Doctoral dissertation]. Lyon; 2018. [cited 2020 Oct 28]. Available from: http://www.theses.fr/2018LYSES060.

Council of Science Editors:

Puech C. Développement d’un modèle in vitro de Barrière Hémato-Encéphalique humaine pour des études pharmacologiques : Interactions avec les anticoagulants oraux directs : Development of an in vitro model of a human Blood Brain Barrier for pharmacological studies : Interactions with directs oral anticoagulants. [Doctoral Dissertation]. Lyon; 2018. Available from: http://www.theses.fr/2018LYSES060


Queensland University of Technology

19. Adams, Mark N. In vitro and in vivo studies on protease-activated receptor-2.

Degree: 2012, Queensland University of Technology

Protease-activated receptor-2 (PAR2) is a G protein coupled receptor (GPCR) that is activated by proteolytic cleavage of its amino terminal domain by trypsin-like serine proteases.… (more)

Subjects/Keywords: protease-activated receptor-2 (par2); g-protein coupled receptor (gpcr); signal transduction; trafficking; palmitoylation; agonist; antagonist; prostate cancer; bone metastasis; scid mice; intra-tibial injection

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APA (6th Edition):

Adams, M. N. (2012). In vitro and in vivo studies on protease-activated receptor-2. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/54338/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Adams, Mark N. “In vitro and in vivo studies on protease-activated receptor-2.” 2012. Thesis, Queensland University of Technology. Accessed October 28, 2020. https://eprints.qut.edu.au/54338/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Adams, Mark N. “In vitro and in vivo studies on protease-activated receptor-2.” 2012. Web. 28 Oct 2020.

Vancouver:

Adams MN. In vitro and in vivo studies on protease-activated receptor-2. [Internet] [Thesis]. Queensland University of Technology; 2012. [cited 2020 Oct 28]. Available from: https://eprints.qut.edu.au/54338/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Adams MN. In vitro and in vivo studies on protease-activated receptor-2. [Thesis]. Queensland University of Technology; 2012. Available from: https://eprints.qut.edu.au/54338/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Michigan

20. Delekta, Phillip Charles. GPCR-dependent NF-kB Signaling in Endothelial Dysfunction: A Critical Role for the CARMA3/BcI10/MALT1 Signaling Complex.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 Atherosclerosis is a complex inflammatory disease. Atherosclerotic lesions are characterized by the accumulation of lipid particles and immune cells in the subendothelial space, resulting in… (more)

Subjects/Keywords: CARMA3 Bcl10 MALT1; Atherosclerosis Inflammatory Disease; Endothelial Dysfunction; NF-κB; Protease Activated Receptor-1 PAR-1 GPCR; Endothelial Cell; Microbiology and Immunology; Molecular, Cellular and Developmental Biology; Science (General); Science

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APA (6th Edition):

Delekta, P. C. (2011). GPCR-dependent NF-kB Signaling in Endothelial Dysfunction: A Critical Role for the CARMA3/BcI10/MALT1 Signaling Complex. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/89853

Chicago Manual of Style (16th Edition):

Delekta, Phillip Charles. “GPCR-dependent NF-kB Signaling in Endothelial Dysfunction: A Critical Role for the CARMA3/BcI10/MALT1 Signaling Complex.” 2011. Doctoral Dissertation, University of Michigan. Accessed October 28, 2020. http://hdl.handle.net/2027.42/89853.

MLA Handbook (7th Edition):

Delekta, Phillip Charles. “GPCR-dependent NF-kB Signaling in Endothelial Dysfunction: A Critical Role for the CARMA3/BcI10/MALT1 Signaling Complex.” 2011. Web. 28 Oct 2020.

Vancouver:

Delekta PC. GPCR-dependent NF-kB Signaling in Endothelial Dysfunction: A Critical Role for the CARMA3/BcI10/MALT1 Signaling Complex. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/2027.42/89853.

Council of Science Editors:

Delekta PC. GPCR-dependent NF-kB Signaling in Endothelial Dysfunction: A Critical Role for the CARMA3/BcI10/MALT1 Signaling Complex. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/89853


University of Melbourne

21. Francis, Nidhish. The role of protease-activated receptor-2 in periodontal disease.

Degree: 2017, University of Melbourne

Abstract withheld

Subjects/Keywords: protease-activated receptor-2; periodontal disease; tissue-specific PAR2 knockout mice

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Francis, N. (2017). The role of protease-activated receptor-2 in periodontal disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/178362

Chicago Manual of Style (16th Edition):

Francis, Nidhish. “The role of protease-activated receptor-2 in periodontal disease.” 2017. Doctoral Dissertation, University of Melbourne. Accessed October 28, 2020. http://hdl.handle.net/11343/178362.

MLA Handbook (7th Edition):

Francis, Nidhish. “The role of protease-activated receptor-2 in periodontal disease.” 2017. Web. 28 Oct 2020.

Vancouver:

Francis N. The role of protease-activated receptor-2 in periodontal disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/11343/178362.

Council of Science Editors:

Francis N. The role of protease-activated receptor-2 in periodontal disease. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/178362


Queensland University of Technology

22. Reid, Janet C. Identification and characterization of novel proteolytic interactions of prostate cancer-expressed kallikrein-related peptidases, type II transmembrane serine proteases and matrix metalloproteinases.

Degree: 2015, Queensland University of Technology

 This study investigated interactions of protein-cleaving enzymes (or proteases) that promote prostate cancer progression. It provides the first evidence of a novel regulatory network of… (more)

Subjects/Keywords: Kallikrein-Related Peptidase (KLK); Type II Transmembrane Serine Protease (TTSP); Matrix Metalloproteinase (MMP); Protease-Activated Receptor (PAR); Hepatocyte Growth Factor Activator Inhibitor-1; Hepatocyte Growth Factor (HGF); Calcium Signalling; Activity Based Probe (ABP); Cell Surface; Proteolytic Cascade

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APA (6th Edition):

Reid, J. C. (2015). Identification and characterization of novel proteolytic interactions of prostate cancer-expressed kallikrein-related peptidases, type II transmembrane serine proteases and matrix metalloproteinases. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/81594/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reid, Janet C. “Identification and characterization of novel proteolytic interactions of prostate cancer-expressed kallikrein-related peptidases, type II transmembrane serine proteases and matrix metalloproteinases.” 2015. Thesis, Queensland University of Technology. Accessed October 28, 2020. https://eprints.qut.edu.au/81594/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reid, Janet C. “Identification and characterization of novel proteolytic interactions of prostate cancer-expressed kallikrein-related peptidases, type II transmembrane serine proteases and matrix metalloproteinases.” 2015. Web. 28 Oct 2020.

Vancouver:

Reid JC. Identification and characterization of novel proteolytic interactions of prostate cancer-expressed kallikrein-related peptidases, type II transmembrane serine proteases and matrix metalloproteinases. [Internet] [Thesis]. Queensland University of Technology; 2015. [cited 2020 Oct 28]. Available from: https://eprints.qut.edu.au/81594/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reid JC. Identification and characterization of novel proteolytic interactions of prostate cancer-expressed kallikrein-related peptidases, type II transmembrane serine proteases and matrix metalloproteinases. [Thesis]. Queensland University of Technology; 2015. Available from: https://eprints.qut.edu.au/81594/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Queensland

23. Yau, Mei-Kwan (Annika). Novel agonists & antagonists for protease-activated receptor 2 and C3a receptor.

Degree: Institute for Molecular Bioscience, 2014, University of Queensland

Subjects/Keywords: GPRC; Protease-activated receptor; PAR2; Complement C3a; Agonist; Antagonist; Inflammation; 0304 Medicinal and Biomolecular Chemistry; 1115 Pharmacology and Pharmaceutical Sciences

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APA (6th Edition):

Yau, M. (. (2014). Novel agonists & antagonists for protease-activated receptor 2 and C3a receptor. (Thesis). University of Queensland. Retrieved from http://espace.library.uq.edu.au/view/UQ:327624

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yau, Mei-Kwan (Annika). “Novel agonists & antagonists for protease-activated receptor 2 and C3a receptor.” 2014. Thesis, University of Queensland. Accessed October 28, 2020. http://espace.library.uq.edu.au/view/UQ:327624.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yau, Mei-Kwan (Annika). “Novel agonists & antagonists for protease-activated receptor 2 and C3a receptor.” 2014. Web. 28 Oct 2020.

Vancouver:

Yau M(. Novel agonists & antagonists for protease-activated receptor 2 and C3a receptor. [Internet] [Thesis]. University of Queensland; 2014. [cited 2020 Oct 28]. Available from: http://espace.library.uq.edu.au/view/UQ:327624.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yau M(. Novel agonists & antagonists for protease-activated receptor 2 and C3a receptor. [Thesis]. University of Queensland; 2014. Available from: http://espace.library.uq.edu.au/view/UQ:327624

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

24. Bagher, Mariam. Mast cells and its crosstalk with mesenchymal cells in chronic lung diseases.

Degree: 2019, University of Lund

Subjects/Keywords: Medical and Health Sciences; Mast cells; Fibroblasts; IPF; COPD; Extracellular matrix; Remodelling; Inflammation; Tryptase; Chymase; Protease-activated receptor 2; Migration

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APA (6th Edition):

Bagher, M. (2019). Mast cells and its crosstalk with mesenchymal cells in chronic lung diseases. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/546ffcd1-75fa-42c3-9d52-fea2ad39a233 ; https://portal.research.lu.se/ws/files/63306265/Mariam_Bagher_web_2.pdf

Chicago Manual of Style (16th Edition):

Bagher, Mariam. “Mast cells and its crosstalk with mesenchymal cells in chronic lung diseases.” 2019. Doctoral Dissertation, University of Lund. Accessed October 28, 2020. https://lup.lub.lu.se/record/546ffcd1-75fa-42c3-9d52-fea2ad39a233 ; https://portal.research.lu.se/ws/files/63306265/Mariam_Bagher_web_2.pdf.

MLA Handbook (7th Edition):

Bagher, Mariam. “Mast cells and its crosstalk with mesenchymal cells in chronic lung diseases.” 2019. Web. 28 Oct 2020.

Vancouver:

Bagher M. Mast cells and its crosstalk with mesenchymal cells in chronic lung diseases. [Internet] [Doctoral dissertation]. University of Lund; 2019. [cited 2020 Oct 28]. Available from: https://lup.lub.lu.se/record/546ffcd1-75fa-42c3-9d52-fea2ad39a233 ; https://portal.research.lu.se/ws/files/63306265/Mariam_Bagher_web_2.pdf.

Council of Science Editors:

Bagher M. Mast cells and its crosstalk with mesenchymal cells in chronic lung diseases. [Doctoral Dissertation]. University of Lund; 2019. Available from: https://lup.lub.lu.se/record/546ffcd1-75fa-42c3-9d52-fea2ad39a233 ; https://portal.research.lu.se/ws/files/63306265/Mariam_Bagher_web_2.pdf

25. Pal, Kasturi. Role of C-Terminal Tails of G Protein Coupled Receptors on Beta-Arrestin 1/2 Dependent Signaling.

Degree: Cell, Molecular and Developmental Biology, 2013, University of California – Riverside

Protease activated receptor 2 (PAR2) and Neurokinin 1 receptor (NK1R) are 7 transmembrane receptors (7TMRs), which signal by G alpha q leading to Ca2+ release… (more)

Subjects/Keywords: Pharmacology; Cellular biology; Molecular biology; Beta arrestin; Cofilin; G protein coupled receptors; Neurokinin 1 Receptor; Phosphorylation; Protease Activated Receptor 2

Protease Activated Receptor 2 (PAR2) C-terminal Tail Direct β-arrestin 1/2 Recruitment… …adrenergic receptor, human cannabinoid receptor (CB1), Protease Activated Receptor 1 and… …of California, Riverside, June 2013 Dr. Kathryn A DeFea, Chairperson Protease activated… …essential for activated receptor recognition and the C-terminal domain for secondary receptor… …coupling to Gq. The mutant receptor also activated ERK1/2 through a β-arrestin1/2 dependent… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pal, K. (2013). Role of C-Terminal Tails of G Protein Coupled Receptors on Beta-Arrestin 1/2 Dependent Signaling. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/5h15c3k0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pal, Kasturi. “Role of C-Terminal Tails of G Protein Coupled Receptors on Beta-Arrestin 1/2 Dependent Signaling.” 2013. Thesis, University of California – Riverside. Accessed October 28, 2020. http://www.escholarship.org/uc/item/5h15c3k0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pal, Kasturi. “Role of C-Terminal Tails of G Protein Coupled Receptors on Beta-Arrestin 1/2 Dependent Signaling.” 2013. Web. 28 Oct 2020.

Vancouver:

Pal K. Role of C-Terminal Tails of G Protein Coupled Receptors on Beta-Arrestin 1/2 Dependent Signaling. [Internet] [Thesis]. University of California – Riverside; 2013. [cited 2020 Oct 28]. Available from: http://www.escholarship.org/uc/item/5h15c3k0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pal K. Role of C-Terminal Tails of G Protein Coupled Receptors on Beta-Arrestin 1/2 Dependent Signaling. [Thesis]. University of California – Riverside; 2013. Available from: http://www.escholarship.org/uc/item/5h15c3k0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Li, Xiuling. MOLECULAR MECHANISMS OF THROMBOXANE A2 RECEPTOR-MEDIATED INVASION IN LUNG CANCER CELLS.

Degree: 2012, University of Kentucky

 Thromboxane A2 receptor (TP) has been shown to play important roles in multiple aspects of cancer development including regulation of tumor growth, survival and metastasis.… (more)

Subjects/Keywords: Thromboxane A2 Receptor; Invasion; Matrix Metalloproteinases; Monocyte Chemoattractant Protein-1; Protease-Activated Receptor; Pharmacy and Pharmaceutical Sciences

…thromboxane synthase PAR= protease-activated receptor VEGF= vascular endothelial growth factor xv… …Most recently, protease-activated receptor 1(PAR1) was identified as a novel… …19 1.6 Protease-activated receptors (PARs) and cancer progression and invasion… …3 1.2 Thromboxane A2 receptor (TP) and its signal transduction… …2 Figure 2. Structures of thromboxane A2 receptor α and β proteins… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, X. (2012). MOLECULAR MECHANISMS OF THROMBOXANE A2 RECEPTOR-MEDIATED INVASION IN LUNG CANCER CELLS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacy_etds/6

Chicago Manual of Style (16th Edition):

Li, Xiuling. “MOLECULAR MECHANISMS OF THROMBOXANE A2 RECEPTOR-MEDIATED INVASION IN LUNG CANCER CELLS.” 2012. Doctoral Dissertation, University of Kentucky. Accessed October 28, 2020. https://uknowledge.uky.edu/pharmacy_etds/6.

MLA Handbook (7th Edition):

Li, Xiuling. “MOLECULAR MECHANISMS OF THROMBOXANE A2 RECEPTOR-MEDIATED INVASION IN LUNG CANCER CELLS.” 2012. Web. 28 Oct 2020.

Vancouver:

Li X. MOLECULAR MECHANISMS OF THROMBOXANE A2 RECEPTOR-MEDIATED INVASION IN LUNG CANCER CELLS. [Internet] [Doctoral dissertation]. University of Kentucky; 2012. [cited 2020 Oct 28]. Available from: https://uknowledge.uky.edu/pharmacy_etds/6.

Council of Science Editors:

Li X. MOLECULAR MECHANISMS OF THROMBOXANE A2 RECEPTOR-MEDIATED INVASION IN LUNG CANCER CELLS. [Doctoral Dissertation]. University of Kentucky; 2012. Available from: https://uknowledge.uky.edu/pharmacy_etds/6


Université de Montréal

27. Babin, Judith. Évaluation pré-clinique et clinique de deux nouvelles stratégies dans le traitement des maladies cardiovasculaires : activité anti-thrombotique de l'inhibition du récepteur P2Y1 et activité anti-inflammatoire de l'inhibition du récepteur PAR-1.

Degree: 2010, Université de Montréal

Subjects/Keywords: P2Y12; P2Y1; protease-activated receptor-1; PAR-1; SCH 530348; plaquette; thrombose; imflammation; leucocyte; platelet; thrombosis; leukocyte; Health Sciences - Pharmacology / Sciences de la santé - Pharmacologie (UMI : 0419)

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APA (6th Edition):

Babin, J. (2010). Évaluation pré-clinique et clinique de deux nouvelles stratégies dans le traitement des maladies cardiovasculaires : activité anti-thrombotique de l'inhibition du récepteur P2Y1 et activité anti-inflammatoire de l'inhibition du récepteur PAR-1. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/6130

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Babin, Judith. “Évaluation pré-clinique et clinique de deux nouvelles stratégies dans le traitement des maladies cardiovasculaires : activité anti-thrombotique de l'inhibition du récepteur P2Y1 et activité anti-inflammatoire de l'inhibition du récepteur PAR-1.” 2010. Thesis, Université de Montréal. Accessed October 28, 2020. http://hdl.handle.net/1866/6130.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Babin, Judith. “Évaluation pré-clinique et clinique de deux nouvelles stratégies dans le traitement des maladies cardiovasculaires : activité anti-thrombotique de l'inhibition du récepteur P2Y1 et activité anti-inflammatoire de l'inhibition du récepteur PAR-1.” 2010. Web. 28 Oct 2020.

Vancouver:

Babin J. Évaluation pré-clinique et clinique de deux nouvelles stratégies dans le traitement des maladies cardiovasculaires : activité anti-thrombotique de l'inhibition du récepteur P2Y1 et activité anti-inflammatoire de l'inhibition du récepteur PAR-1. [Internet] [Thesis]. Université de Montréal; 2010. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/1866/6130.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Babin J. Évaluation pré-clinique et clinique de deux nouvelles stratégies dans le traitement des maladies cardiovasculaires : activité anti-thrombotique de l'inhibition du récepteur P2Y1 et activité anti-inflammatoire de l'inhibition du récepteur PAR-1. [Thesis]. Université de Montréal; 2010. Available from: http://hdl.handle.net/1866/6130

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. LeSarge, Jordan C. Design, Synthesis, and Evaluation of Novel Protease-Activated Receptor 2 (PAR2)-Targeting Imaging Agents for Cancer.

Degree: 2018, University of Western Ontario

 Aberrant function and over-expression of protease-activated receptor 2 (PAR2), a GPCR, is associated with various cancers and inflammatory diseases. PAR2-targeting ligands have been developed with… (more)

Subjects/Keywords: Molecular imaging; protease-activated receptor 2 (PAR2); imaging agents; positron emission tomography (PET); fluorescence; peptides.; Medicinal-Pharmaceutical Chemistry

…nuclear magnetic resonance P: proton PAR: protease-activated receptor PBS: phosphate buffered… …1.2 Protease-activated Receptor 2 There are four known PARs, designated PAR1, PAR2, PAR3… …PARs are activated through a specific protease that cleaves the N-terminus of the receptor to… …Receptor 2 2.1 Introduction Protease-activated receptors (PARs) are a class of G… …xix 1 Chapter 1 1 Introduction 1.1 Protease-activated Receptors Protease-activated… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

LeSarge, J. C. (2018). Design, Synthesis, and Evaluation of Novel Protease-Activated Receptor 2 (PAR2)-Targeting Imaging Agents for Cancer. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LeSarge, Jordan C. “Design, Synthesis, and Evaluation of Novel Protease-Activated Receptor 2 (PAR2)-Targeting Imaging Agents for Cancer.” 2018. Thesis, University of Western Ontario. Accessed October 28, 2020. https://ir.lib.uwo.ca/etd/5533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LeSarge, Jordan C. “Design, Synthesis, and Evaluation of Novel Protease-Activated Receptor 2 (PAR2)-Targeting Imaging Agents for Cancer.” 2018. Web. 28 Oct 2020.

Vancouver:

LeSarge JC. Design, Synthesis, and Evaluation of Novel Protease-Activated Receptor 2 (PAR2)-Targeting Imaging Agents for Cancer. [Internet] [Thesis]. University of Western Ontario; 2018. [cited 2020 Oct 28]. Available from: https://ir.lib.uwo.ca/etd/5533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LeSarge JC. Design, Synthesis, and Evaluation of Novel Protease-Activated Receptor 2 (PAR2)-Targeting Imaging Agents for Cancer. [Thesis]. University of Western Ontario; 2018. Available from: https://ir.lib.uwo.ca/etd/5533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. 葉山,朋美. ヒト歯髄培養細胞におけるplasma kallikreinによるProtease Activated Receptor-1を介した炎症促進とplasminによるcalcineurinを介したCOX-2発現 : Plasma kallikrein promotes inflammation via PAR-1 activation and plasmin-induced COX-2 expression via calcineurin activation in human dental pulp.

Degree: 博士(歯学), 2016, Nihon University / 日本大学

Subjects/Keywords: 血漿カリクレイン; plasma kallikirein; プラスミン; plasmin; プロテアーゼ活性化型受容体1; protease activated receptor-1; 炎症; inflammation; ヒト歯髄細胞; human dental pulp cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

葉山,朋美. (2016). ヒト歯髄培養細胞におけるplasma kallikreinによるProtease Activated Receptor-1を介した炎症促進とplasminによるcalcineurinを介したCOX-2発現 : Plasma kallikrein promotes inflammation via PAR-1 activation and plasmin-induced COX-2 expression via calcineurin activation in human dental pulp. (Thesis). Nihon University / 日本大学. Retrieved from http://repository.nihon-u.ac.jp/xmlui/handle/11263/759 ; http://dx.doi.org/10.15006/32665A5109

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

葉山,朋美. “ヒト歯髄培養細胞におけるplasma kallikreinによるProtease Activated Receptor-1を介した炎症促進とplasminによるcalcineurinを介したCOX-2発現 : Plasma kallikrein promotes inflammation via PAR-1 activation and plasmin-induced COX-2 expression via calcineurin activation in human dental pulp.” 2016. Thesis, Nihon University / 日本大学. Accessed October 28, 2020. http://repository.nihon-u.ac.jp/xmlui/handle/11263/759 ; http://dx.doi.org/10.15006/32665A5109.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

葉山,朋美. “ヒト歯髄培養細胞におけるplasma kallikreinによるProtease Activated Receptor-1を介した炎症促進とplasminによるcalcineurinを介したCOX-2発現 : Plasma kallikrein promotes inflammation via PAR-1 activation and plasmin-induced COX-2 expression via calcineurin activation in human dental pulp.” 2016. Web. 28 Oct 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

葉山,朋美. ヒト歯髄培養細胞におけるplasma kallikreinによるProtease Activated Receptor-1を介した炎症促進とplasminによるcalcineurinを介したCOX-2発現 : Plasma kallikrein promotes inflammation via PAR-1 activation and plasmin-induced COX-2 expression via calcineurin activation in human dental pulp. [Internet] [Thesis]. Nihon University / 日本大学; 2016. [cited 2020 Oct 28]. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/759 ; http://dx.doi.org/10.15006/32665A5109.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

葉山,朋美. ヒト歯髄培養細胞におけるplasma kallikreinによるProtease Activated Receptor-1を介した炎症促進とplasminによるcalcineurinを介したCOX-2発現 : Plasma kallikrein promotes inflammation via PAR-1 activation and plasmin-induced COX-2 expression via calcineurin activation in human dental pulp. [Thesis]. Nihon University / 日本大学; 2016. Available from: http://repository.nihon-u.ac.jp/xmlui/handle/11263/759 ; http://dx.doi.org/10.15006/32665A5109

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Universitetet i Tromsø

30. Larsen, Anett Kristin. Proteases from seafood as activators of protease-activated receptor-2 in airway epithelial cells .

Degree: 2010, Universitetet i Tromsø

 The work is part of a larger thematic research initiative, “Airway reactions to biological particles derived from seafood”, and was initiated in 2005 after a… (more)

Subjects/Keywords: VDP::Medisinske fag: 700::Basale medisinske, odontologiske og veterinærmedisinske fag: 710::Medisinsk molekylærbiologi: 711; VDP::Medical disciplines: 700::Basic medical, dental and veterinary science disciplines: 710::Medical molecular biology: 711; laksetrypsin; protease-aktivert reseptor-2; nuclear factor-κB; interleukin-8; A549; salmon trypsin; protease-activated receptor-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Larsen, A. K. (2010). Proteases from seafood as activators of protease-activated receptor-2 in airway epithelial cells . (Doctoral Dissertation). Universitetet i Tromsø. Retrieved from http://hdl.handle.net/10037/2892

Chicago Manual of Style (16th Edition):

Larsen, Anett Kristin. “Proteases from seafood as activators of protease-activated receptor-2 in airway epithelial cells .” 2010. Doctoral Dissertation, Universitetet i Tromsø. Accessed October 28, 2020. http://hdl.handle.net/10037/2892.

MLA Handbook (7th Edition):

Larsen, Anett Kristin. “Proteases from seafood as activators of protease-activated receptor-2 in airway epithelial cells .” 2010. Web. 28 Oct 2020.

Vancouver:

Larsen AK. Proteases from seafood as activators of protease-activated receptor-2 in airway epithelial cells . [Internet] [Doctoral dissertation]. Universitetet i Tromsø 2010. [cited 2020 Oct 28]. Available from: http://hdl.handle.net/10037/2892.

Council of Science Editors:

Larsen AK. Proteases from seafood as activators of protease-activated receptor-2 in airway epithelial cells . [Doctoral Dissertation]. Universitetet i Tromsø 2010. Available from: http://hdl.handle.net/10037/2892

[1] [2]

.