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You searched for subject:(pro apoptotic chemotherapeuitc drugs). Showing records 1 – 30 of 6993 total matches.

[1] [2] [3] [4] [5] … [234]

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1. Buzzy, Christina Antonopoulos. NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES.

Degree: PhD, Pathology, 2015, Case Western Reserve University School of Graduate Studies

 Dysregulated IL-1ß production has been implicated in a host of disease settings, ranging from autoinflammatory syndromes to diabetes and Alzheimer’s disease. The first-identified interleukin-converting enzyme… (more)

Subjects/Keywords: Immunology; Biology; caspase-8; IL-1b processing; pro-apoptotic chemotherapeuitc drugs; NLRP3; inflammasome

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APA (6th Edition):

Buzzy, C. A. (2015). NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855

Chicago Manual of Style (16th Edition):

Buzzy, Christina Antonopoulos. “NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES.” 2015. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed December 01, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855.

MLA Handbook (7th Edition):

Buzzy, Christina Antonopoulos. “NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES.” 2015. Web. 01 Dec 2020.

Vancouver:

Buzzy CA. NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2015. [cited 2020 Dec 01]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855.

Council of Science Editors:

Buzzy CA. NON-CANONICAL IL-1ß PROCESSING VIA CASPASE-8 IN MURINE DENDRITIC CELLS AND MACROPHAGES. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1417718855


University of the Western Cape

2. Emjedi, Zaakiyah Z. Targeted delivery of embelin to cancer cells .

Degree: 2013, University of the Western Cape

 Apoptosis or programmed cell death is vital to the development of organisms as they maintain the balance between cell death and cell growth. Failure to… (more)

Subjects/Keywords: Apoptosis Carcinogenesis Initiator Caspase Effector Caspase Pro–apoptotic Biconjugated nanoparticles Gold nanoparticles Embelin Ceramide

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APA (6th Edition):

Emjedi, Z. Z. (2013). Targeted delivery of embelin to cancer cells . (Thesis). University of the Western Cape. Retrieved from http://hdl.handle.net/11394/4251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Emjedi, Zaakiyah Z. “Targeted delivery of embelin to cancer cells .” 2013. Thesis, University of the Western Cape. Accessed December 01, 2020. http://hdl.handle.net/11394/4251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Emjedi, Zaakiyah Z. “Targeted delivery of embelin to cancer cells .” 2013. Web. 01 Dec 2020.

Vancouver:

Emjedi ZZ. Targeted delivery of embelin to cancer cells . [Internet] [Thesis]. University of the Western Cape; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/11394/4251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Emjedi ZZ. Targeted delivery of embelin to cancer cells . [Thesis]. University of the Western Cape; 2013. Available from: http://hdl.handle.net/11394/4251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

3. Choucair, Hassan. Mechanisms of Cancer Cell Death Mediated by Novel Fatty Acid Metabolite Analogues .

Degree: University of Sydney

 New molecules that kill cancer cells by alternate mechanisms are urgently required to provide additional strategies for the treatment of tumours that are resistant to… (more)

Subjects/Keywords: Cancer; Aryl-urea; Mitochondria; Pro-apoptotic agents

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APA (6th Edition):

Choucair, H. (n.d.). Mechanisms of Cancer Cell Death Mediated by Novel Fatty Acid Metabolite Analogues . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/22128

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choucair, Hassan. “Mechanisms of Cancer Cell Death Mediated by Novel Fatty Acid Metabolite Analogues .” Thesis, University of Sydney. Accessed December 01, 2020. http://hdl.handle.net/2123/22128.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choucair, Hassan. “Mechanisms of Cancer Cell Death Mediated by Novel Fatty Acid Metabolite Analogues .” Web. 01 Dec 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Choucair H. Mechanisms of Cancer Cell Death Mediated by Novel Fatty Acid Metabolite Analogues . [Internet] [Thesis]. University of Sydney; [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2123/22128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Choucair H. Mechanisms of Cancer Cell Death Mediated by Novel Fatty Acid Metabolite Analogues . [Thesis]. University of Sydney; Available from: http://hdl.handle.net/2123/22128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Arizona State University

4. Agarwal, Shubhangi. MRI Guided Analysis of Changes in Tumor Oxygenation in Response to Hypoxia Activated/Targeted Therapeutics.

Degree: Bioengineering, 2017, Arizona State University

 A tumor is a heterogeneous combination of proliferating tumor cells, infiltrating immune cells and stromal components along with a variety of associated host tissue cells,… (more)

Subjects/Keywords: Biomedical engineering; HAP; Hypoxia; Hypoxia Activated Pro-drugs; MRI; NSCLC

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APA (6th Edition):

Agarwal, S. (2017). MRI Guided Analysis of Changes in Tumor Oxygenation in Response to Hypoxia Activated/Targeted Therapeutics. (Doctoral Dissertation). Arizona State University. Retrieved from http://repository.asu.edu/items/44236

Chicago Manual of Style (16th Edition):

Agarwal, Shubhangi. “MRI Guided Analysis of Changes in Tumor Oxygenation in Response to Hypoxia Activated/Targeted Therapeutics.” 2017. Doctoral Dissertation, Arizona State University. Accessed December 01, 2020. http://repository.asu.edu/items/44236.

MLA Handbook (7th Edition):

Agarwal, Shubhangi. “MRI Guided Analysis of Changes in Tumor Oxygenation in Response to Hypoxia Activated/Targeted Therapeutics.” 2017. Web. 01 Dec 2020.

Vancouver:

Agarwal S. MRI Guided Analysis of Changes in Tumor Oxygenation in Response to Hypoxia Activated/Targeted Therapeutics. [Internet] [Doctoral dissertation]. Arizona State University; 2017. [cited 2020 Dec 01]. Available from: http://repository.asu.edu/items/44236.

Council of Science Editors:

Agarwal S. MRI Guided Analysis of Changes in Tumor Oxygenation in Response to Hypoxia Activated/Targeted Therapeutics. [Doctoral Dissertation]. Arizona State University; 2017. Available from: http://repository.asu.edu/items/44236


University of Michigan

5. Blatt, Neal Benjamin. Characterization of a novel pro-apoptotic benzodiazepine.

Degree: PhD, Pure Sciences, 2002, University of Michigan

 The factors that cause the autoimmune disease systemic lupus erythematosus (SLE) remain poorly understood. As such, treatment of this debilitating and potentially fatal disease is… (more)

Subjects/Keywords: Apoptosis; Apoptotic; Benzodiazepine; Characterization; Cytotoxicity; Immunosuppression; Lymphocytes; Novel; Pro; Systemic Lupus Erythematosus

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APA (6th Edition):

Blatt, N. B. (2002). Characterization of a novel pro-apoptotic benzodiazepine. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/129155

Chicago Manual of Style (16th Edition):

Blatt, Neal Benjamin. “Characterization of a novel pro-apoptotic benzodiazepine.” 2002. Doctoral Dissertation, University of Michigan. Accessed December 01, 2020. http://hdl.handle.net/2027.42/129155.

MLA Handbook (7th Edition):

Blatt, Neal Benjamin. “Characterization of a novel pro-apoptotic benzodiazepine.” 2002. Web. 01 Dec 2020.

Vancouver:

Blatt NB. Characterization of a novel pro-apoptotic benzodiazepine. [Internet] [Doctoral dissertation]. University of Michigan; 2002. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2027.42/129155.

Council of Science Editors:

Blatt NB. Characterization of a novel pro-apoptotic benzodiazepine. [Doctoral Dissertation]. University of Michigan; 2002. Available from: http://hdl.handle.net/2027.42/129155


University of Michigan

6. Bednarski, Jeffrey John, II. Modulation of activation -induced cell death in lymphocytes by a pro-apoptotic benzodiazepine.

Degree: PhD, Pure Sciences, 2003, University of Michigan

 The autoimmune disease systemic lupus erythematosus (SLE) poses formidable therapeutic challenges; neither the exact population of pathogenic lymphocytes nor specific targets for drug development are… (more)

Subjects/Keywords: Activation-induced Cell Death; Aids; Apoptotic; Benzodiazepine; Immune Deficiency; Lymphocytes; Modulation; Pro

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APA (6th Edition):

Bednarski, Jeffrey John, I. (2003). Modulation of activation -induced cell death in lymphocytes by a pro-apoptotic benzodiazepine. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/123346

Chicago Manual of Style (16th Edition):

Bednarski, Jeffrey John, II. “Modulation of activation -induced cell death in lymphocytes by a pro-apoptotic benzodiazepine.” 2003. Doctoral Dissertation, University of Michigan. Accessed December 01, 2020. http://hdl.handle.net/2027.42/123346.

MLA Handbook (7th Edition):

Bednarski, Jeffrey John, II. “Modulation of activation -induced cell death in lymphocytes by a pro-apoptotic benzodiazepine.” 2003. Web. 01 Dec 2020.

Vancouver:

Bednarski, Jeffrey John I. Modulation of activation -induced cell death in lymphocytes by a pro-apoptotic benzodiazepine. [Internet] [Doctoral dissertation]. University of Michigan; 2003. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2027.42/123346.

Council of Science Editors:

Bednarski, Jeffrey John I. Modulation of activation -induced cell death in lymphocytes by a pro-apoptotic benzodiazepine. [Doctoral Dissertation]. University of Michigan; 2003. Available from: http://hdl.handle.net/2027.42/123346


University of Windsor

7. Gueorguieva, Deyzi. Identification and functional characterization of unique single domain antibodies against pro-apoptotic protein Bax.

Degree: MS, Chemistry and Biochemistry, 2006, University of Windsor

Subjects/Keywords: ANTIBODIES; APOPTOTIC; BAX; CHARACTERIZATION; DOMAIN; FUNCTIONAL; IDENTIFICATION; PRO; PROTEIN; SINGLE; UNIQUE

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APA (6th Edition):

Gueorguieva, D. (2006). Identification and functional characterization of unique single domain antibodies against pro-apoptotic protein Bax. (Masters Thesis). University of Windsor. Retrieved from https://scholar.uwindsor.ca/etd/7104

Chicago Manual of Style (16th Edition):

Gueorguieva, Deyzi. “Identification and functional characterization of unique single domain antibodies against pro-apoptotic protein Bax.” 2006. Masters Thesis, University of Windsor. Accessed December 01, 2020. https://scholar.uwindsor.ca/etd/7104.

MLA Handbook (7th Edition):

Gueorguieva, Deyzi. “Identification and functional characterization of unique single domain antibodies against pro-apoptotic protein Bax.” 2006. Web. 01 Dec 2020.

Vancouver:

Gueorguieva D. Identification and functional characterization of unique single domain antibodies against pro-apoptotic protein Bax. [Internet] [Masters thesis]. University of Windsor; 2006. [cited 2020 Dec 01]. Available from: https://scholar.uwindsor.ca/etd/7104.

Council of Science Editors:

Gueorguieva D. Identification and functional characterization of unique single domain antibodies against pro-apoptotic protein Bax. [Masters Thesis]. University of Windsor; 2006. Available from: https://scholar.uwindsor.ca/etd/7104


University of South Florida

8. Desai, Shraddha R. Role of Protein Kinase C-iota in Glioblastoma.

Degree: 2011, University of South Florida

 The focus of this research was to investigate the role of protein kinase C-iota (PKC-é) in the regulation of Bad function, a pro-apoptotic member of… (more)

Subjects/Keywords: Cell cycle progression; Cell survival and proliferation; Cyclin dependent kinase; Phosphatidylinositol (3)-kinase; Pro-apoptotic molecule-Bad; Signal Transduction; American Studies; Arts and Humanities; Biochemistry; Cell Biology

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APA (6th Edition):

Desai, S. R. (2011). Role of Protein Kinase C-iota in Glioblastoma. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/3070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Desai, Shraddha R. “Role of Protein Kinase C-iota in Glioblastoma.” 2011. Thesis, University of South Florida. Accessed December 01, 2020. https://scholarcommons.usf.edu/etd/3070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Desai, Shraddha R. “Role of Protein Kinase C-iota in Glioblastoma.” 2011. Web. 01 Dec 2020.

Vancouver:

Desai SR. Role of Protein Kinase C-iota in Glioblastoma. [Internet] [Thesis]. University of South Florida; 2011. [cited 2020 Dec 01]. Available from: https://scholarcommons.usf.edu/etd/3070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Desai SR. Role of Protein Kinase C-iota in Glioblastoma. [Thesis]. University of South Florida; 2011. Available from: https://scholarcommons.usf.edu/etd/3070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Western Kentucky University

9. Kotadia, Nayna. A Study on the Protein Interaction with Different Platinum Compounds.

Degree: MS, Department of Chemistry, 2008, Western Kentucky University

 Since the discovery of anti-tumor activity of cisplatin in 1960, significant progress has been made in treating metastatic or advanced cancer with cisplatin and platinum… (more)

Subjects/Keywords: cancer treatments; apoptotic program; platinum anticancer drugs; Chemistry; Medicinal-Pharmaceutical Chemistry

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APA (6th Edition):

Kotadia, N. (2008). A Study on the Protein Interaction with Different Platinum Compounds. (Masters Thesis). Western Kentucky University. Retrieved from https://digitalcommons.wku.edu/theses/8

Chicago Manual of Style (16th Edition):

Kotadia, Nayna. “A Study on the Protein Interaction with Different Platinum Compounds.” 2008. Masters Thesis, Western Kentucky University. Accessed December 01, 2020. https://digitalcommons.wku.edu/theses/8.

MLA Handbook (7th Edition):

Kotadia, Nayna. “A Study on the Protein Interaction with Different Platinum Compounds.” 2008. Web. 01 Dec 2020.

Vancouver:

Kotadia N. A Study on the Protein Interaction with Different Platinum Compounds. [Internet] [Masters thesis]. Western Kentucky University; 2008. [cited 2020 Dec 01]. Available from: https://digitalcommons.wku.edu/theses/8.

Council of Science Editors:

Kotadia N. A Study on the Protein Interaction with Different Platinum Compounds. [Masters Thesis]. Western Kentucky University; 2008. Available from: https://digitalcommons.wku.edu/theses/8

10. Ξαγοράρης, Ιορδάνης. Ρόλος των προ- και αντι-αποπτωτικών γονιδίων στην παθογένεια του πολλαπλού μυελώματος.

Degree: 2005, University of Patras

Το πολλαπλούν μυέλωμα είναι μια νεοπλασία η οποία, έως και σήμερα, παραμένει ανίατη. Στο ΠΜ, το ανοσοποιητικό σύστημα δε κατορθώνει να καταστρέψει τα κακοήθη πλασμοκύτταρα,… (more)

Subjects/Keywords: Πολλαπλούν μυέλωμα; Απόπτωση; Προ-αποπτωτικά γονίδια; Αντι-αποπτωτικά γονίδια; Θαλιδομίδη; Ζολεδρονικό οξύ; 616.079; Multiple myeloma; Pro-apoptotic genes,; Anti-apoptotic genes; Thalidomide,; Zoledronic acid

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APA (6th Edition):

Ξαγοράρης, . (2005). Ρόλος των προ- και αντι-αποπτωτικών γονιδίων στην παθογένεια του πολλαπλού μυελώματος. (Doctoral Dissertation). University of Patras. Retrieved from http://nemertes.lis.upatras.gr/jspui/handle/10889/380

Chicago Manual of Style (16th Edition):

Ξαγοράρης, Ιορδάνης. “Ρόλος των προ- και αντι-αποπτωτικών γονιδίων στην παθογένεια του πολλαπλού μυελώματος.” 2005. Doctoral Dissertation, University of Patras. Accessed December 01, 2020. http://nemertes.lis.upatras.gr/jspui/handle/10889/380.

MLA Handbook (7th Edition):

Ξαγοράρης, Ιορδάνης. “Ρόλος των προ- και αντι-αποπτωτικών γονιδίων στην παθογένεια του πολλαπλού μυελώματος.” 2005. Web. 01 Dec 2020.

Vancouver:

Ξαγοράρης . Ρόλος των προ- και αντι-αποπτωτικών γονιδίων στην παθογένεια του πολλαπλού μυελώματος. [Internet] [Doctoral dissertation]. University of Patras; 2005. [cited 2020 Dec 01]. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/380.

Council of Science Editors:

Ξαγοράρης . Ρόλος των προ- και αντι-αποπτωτικών γονιδίων στην παθογένεια του πολλαπλού μυελώματος. [Doctoral Dissertation]. University of Patras; 2005. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/380


University of Michigan

11. Boitano, Anthony E. Mechanisms of a pro-apoptotic benzodiazepine.

Degree: PhD, Pure Sciences, 2005, University of Michigan

 Bz-423 is a immunomodulatory 1,4-benzodiazepine that exerts its therapeutic effects in autoimmune mice by selectively inducing apoptosis of pathogenic B and T cells. The overall… (more)

Subjects/Keywords: Apoptotic; Bcl-2; Benzodiazepine; Bz-423; Lupus; Mechanisms; Oligomycin Sensitivity-conferring Protein; Pro; Proapoptosis

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APA (6th Edition):

Boitano, A. E. (2005). Mechanisms of a pro-apoptotic benzodiazepine. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/125029

Chicago Manual of Style (16th Edition):

Boitano, Anthony E. “Mechanisms of a pro-apoptotic benzodiazepine.” 2005. Doctoral Dissertation, University of Michigan. Accessed December 01, 2020. http://hdl.handle.net/2027.42/125029.

MLA Handbook (7th Edition):

Boitano, Anthony E. “Mechanisms of a pro-apoptotic benzodiazepine.” 2005. Web. 01 Dec 2020.

Vancouver:

Boitano AE. Mechanisms of a pro-apoptotic benzodiazepine. [Internet] [Doctoral dissertation]. University of Michigan; 2005. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2027.42/125029.

Council of Science Editors:

Boitano AE. Mechanisms of a pro-apoptotic benzodiazepine. [Doctoral Dissertation]. University of Michigan; 2005. Available from: http://hdl.handle.net/2027.42/125029


University of Michigan

12. Emal, Cory Dene. Design and synthesis of mechanism-based cysteine protease inhibitors and pro-apoptotic 1,4-benzodiazepines.

Degree: PhD, Pure Sciences, 2003, University of Michigan

 Chagas' disease is an incapacitating illness that afflicts 16 – 18 million people, killing 21,000 each year in the Americas. Patients infected with Trypanosoma cruzi who… (more)

Subjects/Keywords: Apoptotic; Based; Benzodiazepines-1,4; Chagas' Disease; Cysteine Protease Inhibitors; Design; Falcipain-2; Mechanism; Plasmodium Falciparum; Pro; Synthesis; Trypanosoma Cruzi

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APA (6th Edition):

Emal, C. D. (2003). Design and synthesis of mechanism-based cysteine protease inhibitors and pro-apoptotic 1,4-benzodiazepines. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/123829

Chicago Manual of Style (16th Edition):

Emal, Cory Dene. “Design and synthesis of mechanism-based cysteine protease inhibitors and pro-apoptotic 1,4-benzodiazepines.” 2003. Doctoral Dissertation, University of Michigan. Accessed December 01, 2020. http://hdl.handle.net/2027.42/123829.

MLA Handbook (7th Edition):

Emal, Cory Dene. “Design and synthesis of mechanism-based cysteine protease inhibitors and pro-apoptotic 1,4-benzodiazepines.” 2003. Web. 01 Dec 2020.

Vancouver:

Emal CD. Design and synthesis of mechanism-based cysteine protease inhibitors and pro-apoptotic 1,4-benzodiazepines. [Internet] [Doctoral dissertation]. University of Michigan; 2003. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2027.42/123829.

Council of Science Editors:

Emal CD. Design and synthesis of mechanism-based cysteine protease inhibitors and pro-apoptotic 1,4-benzodiazepines. [Doctoral Dissertation]. University of Michigan; 2003. Available from: http://hdl.handle.net/2027.42/123829


Queens University

13. Arab, Nagla. Analysis of Human Thyroid Cancer-1 and Annexin V as Pro-Survival cDNA Sequences in Saccharomyces Cerevisiae .

Degree: Biology, Queens University

 Overexpressing a mouse Bax cDNA in yeast cells induces cell death. When heterologously expressed in yeast cells overexpression of a mouse cDNA encoding Bax, human… (more)

Subjects/Keywords: Programmed Cell Death (PCD) ; Apoptosis ; Pro-Survival ; Anti-Apoptotic ; Cell Survival ; Yeast ; Thyroid Cancer-1 (TC-1) ; Annexin V (ANXA5)

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APA (6th Edition):

Arab, N. (n.d.). Analysis of Human Thyroid Cancer-1 and Annexin V as Pro-Survival cDNA Sequences in Saccharomyces Cerevisiae . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/24276

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arab, Nagla. “Analysis of Human Thyroid Cancer-1 and Annexin V as Pro-Survival cDNA Sequences in Saccharomyces Cerevisiae .” Thesis, Queens University. Accessed December 01, 2020. http://hdl.handle.net/1974/24276.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arab, Nagla. “Analysis of Human Thyroid Cancer-1 and Annexin V as Pro-Survival cDNA Sequences in Saccharomyces Cerevisiae .” Web. 01 Dec 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Arab N. Analysis of Human Thyroid Cancer-1 and Annexin V as Pro-Survival cDNA Sequences in Saccharomyces Cerevisiae . [Internet] [Thesis]. Queens University; [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1974/24276.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Arab N. Analysis of Human Thyroid Cancer-1 and Annexin V as Pro-Survival cDNA Sequences in Saccharomyces Cerevisiae . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/24276

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Indian Institute of Science

14. Kumar, Krishan. The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine.

Degree: PhD, Faculty of Science, 2018, Indian Institute of Science

 The thesis entitled “The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine”… (more)

Subjects/Keywords: Cancer Nanomedicine; Molecular Drugs; Lipoplex Internalization; α-Tocopherol; Apoptotic Activity; Gemini Tocopheryl Lipids; Pseudoglyceryl Gemini Lipids; Gold Nanoparticles; Gemini Cationic Lipids; Anticancer Drug Delivery; Cationic Gemini Lipid; RNA Aptamer; Doxorubicn; Cationic Pseudoglyceryl Gemini Lipids; Organic Chemistry

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APA (6th Edition):

Kumar, K. (2018). The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/3880

Chicago Manual of Style (16th Edition):

Kumar, Krishan. “The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine.” 2018. Doctoral Dissertation, Indian Institute of Science. Accessed December 01, 2020. http://etd.iisc.ac.in/handle/2005/3880.

MLA Handbook (7th Edition):

Kumar, Krishan. “The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine.” 2018. Web. 01 Dec 2020.

Vancouver:

Kumar K. The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2018. [cited 2020 Dec 01]. Available from: http://etd.iisc.ac.in/handle/2005/3880.

Council of Science Editors:

Kumar K. The Role of Liposomal Hybrids and Gold Nanoparticles in the Efficacious Transport of Nucleic Acids and Small Molecular Drugs for Cancer Nanomedicine. [Doctoral Dissertation]. Indian Institute of Science; 2018. Available from: http://etd.iisc.ac.in/handle/2005/3880


University of Wollongong

15. Green, Katrina. Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects.

Degree: PhD, 2012, University of Wollongong

  Second generation antipsychotics (SGAs) are a key pharmacotherapy for the treatment of schizophrenia but can cause serious metabolic side-effects, including obesity and type II… (more)

Subjects/Keywords: obesity; antipsychotic drugs; diabetes; side-effects; olanzapine; metabolic dysfunction; hypothalamus; brainstem; muscarinic; cannabinoid; GABA; neuropeptide Y; pro-opiometanocortin; receptors; mRNA; animal model; antipsychotic; metabolism

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APA (6th Edition):

Green, K. (2012). Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects. (Doctoral Dissertation). University of Wollongong. Retrieved from 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579

Chicago Manual of Style (16th Edition):

Green, Katrina. “Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects.” 2012. Doctoral Dissertation, University of Wollongong. Accessed December 01, 2020. 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579.

MLA Handbook (7th Edition):

Green, Katrina. “Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects.” 2012. Web. 01 Dec 2020.

Vancouver:

Green K. Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects. [Internet] [Doctoral dissertation]. University of Wollongong; 2012. [cited 2020 Dec 01]. Available from: 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579.

Council of Science Editors:

Green K. Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects. [Doctoral Dissertation]. University of Wollongong; 2012. Available from: 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579


IUPUI

16. Howe, Christopher Ryan. Analogues of Nitrofuran Antibiotics are Potent GroEL/ES Pro-drug Inhibitors with Efficacy against Enterococcus Faecium, Staphylococcus Aureus, and Escherichia Coli.

Degree: 2020, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI) Advisors/Committee Members: Johnson, Steven M., Hoang, Quyen Q., Meroueh, Samy O..

Subjects/Keywords: Pro-Drugs; GroEL/ES; Chaperone Proteins; Nitrofurans; Hydroxyquinolines; Nitroreductases; Inhibitors

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APA (6th Edition):

Howe, C. R. (2020). Analogues of Nitrofuran Antibiotics are Potent GroEL/ES Pro-drug Inhibitors with Efficacy against Enterococcus Faecium, Staphylococcus Aureus, and Escherichia Coli. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/22885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Howe, Christopher Ryan. “Analogues of Nitrofuran Antibiotics are Potent GroEL/ES Pro-drug Inhibitors with Efficacy against Enterococcus Faecium, Staphylococcus Aureus, and Escherichia Coli.” 2020. Thesis, IUPUI. Accessed December 01, 2020. http://hdl.handle.net/1805/22885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Howe, Christopher Ryan. “Analogues of Nitrofuran Antibiotics are Potent GroEL/ES Pro-drug Inhibitors with Efficacy against Enterococcus Faecium, Staphylococcus Aureus, and Escherichia Coli.” 2020. Web. 01 Dec 2020.

Vancouver:

Howe CR. Analogues of Nitrofuran Antibiotics are Potent GroEL/ES Pro-drug Inhibitors with Efficacy against Enterococcus Faecium, Staphylococcus Aureus, and Escherichia Coli. [Internet] [Thesis]. IUPUI; 2020. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1805/22885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Howe CR. Analogues of Nitrofuran Antibiotics are Potent GroEL/ES Pro-drug Inhibitors with Efficacy against Enterococcus Faecium, Staphylococcus Aureus, and Escherichia Coli. [Thesis]. IUPUI; 2020. Available from: http://hdl.handle.net/1805/22885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

17. Buckland, Timothy, W. The Tumor Promoting Role of BAD in Breast Cancer Cells.

Degree: MS, Department of Biochemistry, 2012, University of Alberta

 In Canada, approximately 40% of the population will be diagnosed with cancer and 25% will die of this disease. In order to treat cancer more… (more)

Subjects/Keywords: tumor growth; breast cancer; anti-apoptotic; BAD

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APA (6th Edition):

Buckland, Timothy, W. (2012). The Tumor Promoting Role of BAD in Breast Cancer Cells. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/pr76f4608

Chicago Manual of Style (16th Edition):

Buckland, Timothy, W. “The Tumor Promoting Role of BAD in Breast Cancer Cells.” 2012. Masters Thesis, University of Alberta. Accessed December 01, 2020. https://era.library.ualberta.ca/files/pr76f4608.

MLA Handbook (7th Edition):

Buckland, Timothy, W. “The Tumor Promoting Role of BAD in Breast Cancer Cells.” 2012. Web. 01 Dec 2020.

Vancouver:

Buckland, Timothy W. The Tumor Promoting Role of BAD in Breast Cancer Cells. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2020 Dec 01]. Available from: https://era.library.ualberta.ca/files/pr76f4608.

Council of Science Editors:

Buckland, Timothy W. The Tumor Promoting Role of BAD in Breast Cancer Cells. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/pr76f4608

18. Σιγανάκη, Μαρία-Άννα. Έκφραση αποπτωτικών δεικτών σε δείγματα πνευμονικού ιστού ασθενών με χρόνια αποφρακτική πνευμονοπάθεια με ή χωρίς αστάθεια μικροδορυφορικού DNA.

Degree: 2010, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Abnormal apoptotic events in chronic obstructive pulmonary disease (COPD) subvert cellular homeostasis and may play a primary role in its pathogenesis. However, studies in human… (more)

Subjects/Keywords: Χρόνια αποφρακτική πνευμονοπάθεια (ΧΑΠ); Απόπτωση; Ανοσοϊστοχημεία; Προ-αποπτωτικη πρωτεΐνη P53; Αντι-αποπτωτική πρωτεΐνη BCL2; Πνευμονοκύτταρα τύπου ΙΙ; Κυψελιδικά μακροφάγα; Λεμφοκύτταρα; Chronic obstructive pulmonary disease (COPD); Apoptosis; Immunohistochemistry ( IHC); Pro-apoptotic protein P53; Anti - apoptotic protein BCL2; Pneumocytes type II; Alveolar macrophages; Lymphocytes

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APA (6th Edition):

Σιγανάκη, . . (2010). Έκφραση αποπτωτικών δεικτών σε δείγματα πνευμονικού ιστού ασθενών με χρόνια αποφρακτική πνευμονοπάθεια με ή χωρίς αστάθεια μικροδορυφορικού DNA. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/24601

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Σιγανάκη, Μαρία-Άννα. “Έκφραση αποπτωτικών δεικτών σε δείγματα πνευμονικού ιστού ασθενών με χρόνια αποφρακτική πνευμονοπάθεια με ή χωρίς αστάθεια μικροδορυφορικού DNA.” 2010. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed December 01, 2020. http://hdl.handle.net/10442/hedi/24601.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Σιγανάκη, Μαρία-Άννα. “Έκφραση αποπτωτικών δεικτών σε δείγματα πνευμονικού ιστού ασθενών με χρόνια αποφρακτική πνευμονοπάθεια με ή χωρίς αστάθεια μικροδορυφορικού DNA.” 2010. Web. 01 Dec 2020.

Vancouver:

Σιγανάκη . Έκφραση αποπτωτικών δεικτών σε δείγματα πνευμονικού ιστού ασθενών με χρόνια αποφρακτική πνευμονοπάθεια με ή χωρίς αστάθεια μικροδορυφορικού DNA. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2010. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/10442/hedi/24601.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Σιγανάκη . Έκφραση αποπτωτικών δεικτών σε δείγματα πνευμονικού ιστού ασθενών με χρόνια αποφρακτική πνευμονοπάθεια με ή χωρίς αστάθεια μικροδορυφορικού DNA. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2010. Available from: http://hdl.handle.net/10442/hedi/24601

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Ferreira, Daniela Filipa Santos. Análogos de nucleósidos com atividade antiviral.

Degree: 2017, Universidade Fernando Pessoa

Os vírus são agentes infeciosos de pequenas dimensões. Não possuem metabolismo próprio e são considerados parasitas intracelulares obrigatórios, pois precisam de um hospedeiro para se… (more)

Subjects/Keywords: Atividade antiviral; Análogos de nucleósidos; Toxicidade; Pró-fármacos; Efeitos secundários; HBV; HCV; HHV; Antiviral activity; Nucleoside analogs; Toxicity; Pro-drugs; Side effects; HBV; HCV; HHV; Domínio/Área Científica::Ciências Médicas::Medicina Básica

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APA (6th Edition):

Ferreira, D. F. S. (2017). Análogos de nucleósidos com atividade antiviral. (Thesis). Universidade Fernando Pessoa. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/6567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ferreira, Daniela Filipa Santos. “Análogos de nucleósidos com atividade antiviral.” 2017. Thesis, Universidade Fernando Pessoa. Accessed December 01, 2020. https://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/6567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ferreira, Daniela Filipa Santos. “Análogos de nucleósidos com atividade antiviral.” 2017. Web. 01 Dec 2020.

Vancouver:

Ferreira DFS. Análogos de nucleósidos com atividade antiviral. [Internet] [Thesis]. Universidade Fernando Pessoa; 2017. [cited 2020 Dec 01]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/6567.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ferreira DFS. Análogos de nucleósidos com atividade antiviral. [Thesis]. Universidade Fernando Pessoa; 2017. Available from: https://www.rcaap.pt/detail.jsp?id=oai:bdigital.ufp.pt:10284/6567

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Bahmed, Amina. The design and synthesis of novel pro-drugs for the treatment of nephropathic cystinosis.

Degree: PhD, 2015, Robert Gordon University

 Cystinosis is a metabolic disorder characterised by the abnormal accumulation of the amino acid cystine in cells leading to a slow destruction of all major… (more)

Subjects/Keywords: 610; Cystagon; Cysteamine; Cystamine; Pro-drugs; Multicomponent crystals; Nephropathic Cystinosis; Peptide synthesis

…Synthesis of Cystamine Pro-drugs Derivatives [1-4] 67 3.1.3.1.1. Synthesis of… …Dihydrate (3) 123 123 4.2. Synthesis of novel cystamine Pro-drugs derivative 125… …synthetic pro-drugs on levels of cystine 152 CHAPTER 5 Conclusions 156 5.1. Conclusion and… …1.4.2. Problems associated with cysteamine treatment 37 1.5. Pro-drug approach 39 1.6… …Crystallography 62 CHAPTER 3 64 Experimental 3.1. Synthesis of Novel Cystamine Pro-drug Derivatives… 

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APA (6th Edition):

Bahmed, A. (2015). The design and synthesis of novel pro-drugs for the treatment of nephropathic cystinosis. (Doctoral Dissertation). Robert Gordon University. Retrieved from http://hdl.handle.net/10059/1227

Chicago Manual of Style (16th Edition):

Bahmed, Amina. “The design and synthesis of novel pro-drugs for the treatment of nephropathic cystinosis.” 2015. Doctoral Dissertation, Robert Gordon University. Accessed December 01, 2020. http://hdl.handle.net/10059/1227.

MLA Handbook (7th Edition):

Bahmed, Amina. “The design and synthesis of novel pro-drugs for the treatment of nephropathic cystinosis.” 2015. Web. 01 Dec 2020.

Vancouver:

Bahmed A. The design and synthesis of novel pro-drugs for the treatment of nephropathic cystinosis. [Internet] [Doctoral dissertation]. Robert Gordon University; 2015. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/10059/1227.

Council of Science Editors:

Bahmed A. The design and synthesis of novel pro-drugs for the treatment of nephropathic cystinosis. [Doctoral Dissertation]. Robert Gordon University; 2015. Available from: http://hdl.handle.net/10059/1227


Jawaharlal Nehru University

21. Pal, Ranjana. Genotypic and exoression status of apoptotic pathway genes in sporadic breast cancer; -.

Degree: Life Sciences, 2010, Jawaharlal Nehru University

None

References p .63-79

Advisors/Committee Members: Bamezai, R N K.

Subjects/Keywords: Life Science; Genotypic; cancer; apoptotic

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APA (6th Edition):

Pal, R. (2010). Genotypic and exoression status of apoptotic pathway genes in sporadic breast cancer; -. (Thesis). Jawaharlal Nehru University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/14169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pal, Ranjana. “Genotypic and exoression status of apoptotic pathway genes in sporadic breast cancer; -.” 2010. Thesis, Jawaharlal Nehru University. Accessed December 01, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/14169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pal, Ranjana. “Genotypic and exoression status of apoptotic pathway genes in sporadic breast cancer; -.” 2010. Web. 01 Dec 2020.

Vancouver:

Pal R. Genotypic and exoression status of apoptotic pathway genes in sporadic breast cancer; -. [Internet] [Thesis]. Jawaharlal Nehru University; 2010. [cited 2020 Dec 01]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/14169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pal R. Genotypic and exoression status of apoptotic pathway genes in sporadic breast cancer; -. [Thesis]. Jawaharlal Nehru University; 2010. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/14169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ghana

22. Agyapong, S.N.A. Antioxidant, Cytotoxic and Apoptotic Properties of Aqueous Extracts of Fleurya Aestuans and Momordica Charantia on Prostate Cancer Cell Lines .

Degree: 2018, University of Ghana

 Background: Prostate cancer is the second highest cancer type among men in Africa. It brings financial and healthcare burden to a number of patients in… (more)

Subjects/Keywords: Antioxidant; Cytotoxic; Apoptotic; Fleurya Aestuans; Momordica Charantia; Prostate Cancer Cell

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APA (6th Edition):

Agyapong, S. N. A. (2018). Antioxidant, Cytotoxic and Apoptotic Properties of Aqueous Extracts of Fleurya Aestuans and Momordica Charantia on Prostate Cancer Cell Lines . (Masters Thesis). University of Ghana. Retrieved from http://ugspace.ug.edu.gh/handle/123456789/28359

Chicago Manual of Style (16th Edition):

Agyapong, S N A. “Antioxidant, Cytotoxic and Apoptotic Properties of Aqueous Extracts of Fleurya Aestuans and Momordica Charantia on Prostate Cancer Cell Lines .” 2018. Masters Thesis, University of Ghana. Accessed December 01, 2020. http://ugspace.ug.edu.gh/handle/123456789/28359.

MLA Handbook (7th Edition):

Agyapong, S N A. “Antioxidant, Cytotoxic and Apoptotic Properties of Aqueous Extracts of Fleurya Aestuans and Momordica Charantia on Prostate Cancer Cell Lines .” 2018. Web. 01 Dec 2020.

Vancouver:

Agyapong SNA. Antioxidant, Cytotoxic and Apoptotic Properties of Aqueous Extracts of Fleurya Aestuans and Momordica Charantia on Prostate Cancer Cell Lines . [Internet] [Masters thesis]. University of Ghana; 2018. [cited 2020 Dec 01]. Available from: http://ugspace.ug.edu.gh/handle/123456789/28359.

Council of Science Editors:

Agyapong SNA. Antioxidant, Cytotoxic and Apoptotic Properties of Aqueous Extracts of Fleurya Aestuans and Momordica Charantia on Prostate Cancer Cell Lines . [Masters Thesis]. University of Ghana; 2018. Available from: http://ugspace.ug.edu.gh/handle/123456789/28359


University of Adelaide

23. Alanazi, Ibrahim Oqla. Understanding the apoptotic signaling pathways in breast cancer using microarrays, proteomics and bioinformatics.

Degree: 2014, University of Adelaide

 Breast cancer is one of the most common causes of cancer death to women worldwide. In this study A431 cells, derived from epidermoid carcinoma, are… (more)

Subjects/Keywords: breast cancer; apoptotic signaling pathway; microarray; proteomics; bioinformatics

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APA (6th Edition):

Alanazi, I. O. (2014). Understanding the apoptotic signaling pathways in breast cancer using microarrays, proteomics and bioinformatics. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/92350

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alanazi, Ibrahim Oqla. “Understanding the apoptotic signaling pathways in breast cancer using microarrays, proteomics and bioinformatics.” 2014. Thesis, University of Adelaide. Accessed December 01, 2020. http://hdl.handle.net/2440/92350.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alanazi, Ibrahim Oqla. “Understanding the apoptotic signaling pathways in breast cancer using microarrays, proteomics and bioinformatics.” 2014. Web. 01 Dec 2020.

Vancouver:

Alanazi IO. Understanding the apoptotic signaling pathways in breast cancer using microarrays, proteomics and bioinformatics. [Internet] [Thesis]. University of Adelaide; 2014. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2440/92350.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alanazi IO. Understanding the apoptotic signaling pathways in breast cancer using microarrays, proteomics and bioinformatics. [Thesis]. University of Adelaide; 2014. Available from: http://hdl.handle.net/2440/92350

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

24. Miller, Jonathan. Modulation of dendritic cells and autoimmunity by apoptotic and necrotic cells.

Degree: PhD, 2011, University of Manchester

 As the principal antigen-presenting cells to T cells, dendritic cells (DCs) have a key role in the balance of immunity and autoimmunity. They are essential… (more)

Subjects/Keywords: 571.96; Dendritic cell; Autoimmunity; Immune tolerance; Apoptotic cell; Necrotic cell

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APA (6th Edition):

Miller, J. (2011). Modulation of dendritic cells and autoimmunity by apoptotic and necrotic cells. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-dendritic-cells-and-autoimmunity-by-apoptotic-and-necrotic-cells(eab00223-e5a2-4fdd-8baf-aa5966c87ede).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538416

Chicago Manual of Style (16th Edition):

Miller, Jonathan. “Modulation of dendritic cells and autoimmunity by apoptotic and necrotic cells.” 2011. Doctoral Dissertation, University of Manchester. Accessed December 01, 2020. https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-dendritic-cells-and-autoimmunity-by-apoptotic-and-necrotic-cells(eab00223-e5a2-4fdd-8baf-aa5966c87ede).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538416.

MLA Handbook (7th Edition):

Miller, Jonathan. “Modulation of dendritic cells and autoimmunity by apoptotic and necrotic cells.” 2011. Web. 01 Dec 2020.

Vancouver:

Miller J. Modulation of dendritic cells and autoimmunity by apoptotic and necrotic cells. [Internet] [Doctoral dissertation]. University of Manchester; 2011. [cited 2020 Dec 01]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-dendritic-cells-and-autoimmunity-by-apoptotic-and-necrotic-cells(eab00223-e5a2-4fdd-8baf-aa5966c87ede).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538416.

Council of Science Editors:

Miller J. Modulation of dendritic cells and autoimmunity by apoptotic and necrotic cells. [Doctoral Dissertation]. University of Manchester; 2011. Available from: https://www.research.manchester.ac.uk/portal/en/theses/modulation-of-dendritic-cells-and-autoimmunity-by-apoptotic-and-necrotic-cells(eab00223-e5a2-4fdd-8baf-aa5966c87ede).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.538416


University of Edinburgh

25. Chang, Ziyuan. Zebrafish modelling of apoptosis and inflammation in aggressive B-cell lymphoma.

Degree: PhD, 2019, University of Edinburgh

 Apoptosis is a well-orchestrated programmed cell death. In cancer biology the evasion of apoptosis has been considered as one of the key events for tumour… (more)

Subjects/Keywords: zebrafish; apoptosis; Burkitt's lymphoma; B cell lymphoma; apoptotic tumour cells

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APA (6th Edition):

Chang, Z. (2019). Zebrafish modelling of apoptosis and inflammation in aggressive B-cell lymphoma. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/35786

Chicago Manual of Style (16th Edition):

Chang, Ziyuan. “Zebrafish modelling of apoptosis and inflammation in aggressive B-cell lymphoma.” 2019. Doctoral Dissertation, University of Edinburgh. Accessed December 01, 2020. http://hdl.handle.net/1842/35786.

MLA Handbook (7th Edition):

Chang, Ziyuan. “Zebrafish modelling of apoptosis and inflammation in aggressive B-cell lymphoma.” 2019. Web. 01 Dec 2020.

Vancouver:

Chang Z. Zebrafish modelling of apoptosis and inflammation in aggressive B-cell lymphoma. [Internet] [Doctoral dissertation]. University of Edinburgh; 2019. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1842/35786.

Council of Science Editors:

Chang Z. Zebrafish modelling of apoptosis and inflammation in aggressive B-cell lymphoma. [Doctoral Dissertation]. University of Edinburgh; 2019. Available from: http://hdl.handle.net/1842/35786


University of Gothenburg / Göteborgs Universitet

26. Lunavat, Taral. Extracellular Vesicles and RNA interference in tumors.

Degree: 2016, University of Gothenburg / Göteborgs Universitet

 Extracellular vesicles (EVs) including apoptotic bodies (ABs), microvesicles (MVs), exosomes (EXOs) and cell derived artificial nanovesicles (NVs) are important mediators of cell-to-cell communication, in part… (more)

Subjects/Keywords: Melanoma; Therapeutics; Exosome-mimetic nanovesicles; Apoptotic bodies; Microvesicles; Exosomes

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APA (6th Edition):

Lunavat, T. (2016). Extracellular Vesicles and RNA interference in tumors. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/42338

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lunavat, Taral. “Extracellular Vesicles and RNA interference in tumors.” 2016. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed December 01, 2020. http://hdl.handle.net/2077/42338.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lunavat, Taral. “Extracellular Vesicles and RNA interference in tumors.” 2016. Web. 01 Dec 2020.

Vancouver:

Lunavat T. Extracellular Vesicles and RNA interference in tumors. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2016. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2077/42338.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lunavat T. Extracellular Vesicles and RNA interference in tumors. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2016. Available from: http://hdl.handle.net/2077/42338

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

27. Driscoll, Will. Macrophage ADAM17 deficiency augments CD36-dependent apoptotic cell uptake and the linked anti-inflammatory phenotype.

Degree: PhD, 2013, University of Washington

 Rationale: Phagocytosis of apoptotic cells (efferocytosis) is mediated by apoptotic cell receptors and is essential for resolution of inflammation. In chronic inflammation, apoptotic cell clearance… (more)

Subjects/Keywords: ADAM17; apoptotic; CD36; efferocytosis; phagocytosis; sCD36; Pathology; pathology

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APA (6th Edition):

Driscoll, W. (2013). Macrophage ADAM17 deficiency augments CD36-dependent apoptotic cell uptake and the linked anti-inflammatory phenotype. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/22772

Chicago Manual of Style (16th Edition):

Driscoll, Will. “Macrophage ADAM17 deficiency augments CD36-dependent apoptotic cell uptake and the linked anti-inflammatory phenotype.” 2013. Doctoral Dissertation, University of Washington. Accessed December 01, 2020. http://hdl.handle.net/1773/22772.

MLA Handbook (7th Edition):

Driscoll, Will. “Macrophage ADAM17 deficiency augments CD36-dependent apoptotic cell uptake and the linked anti-inflammatory phenotype.” 2013. Web. 01 Dec 2020.

Vancouver:

Driscoll W. Macrophage ADAM17 deficiency augments CD36-dependent apoptotic cell uptake and the linked anti-inflammatory phenotype. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/1773/22772.

Council of Science Editors:

Driscoll W. Macrophage ADAM17 deficiency augments CD36-dependent apoptotic cell uptake and the linked anti-inflammatory phenotype. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/22772


Boston University

28. Yalonetskaya, Alla. Characterization of nuclear degradation dynamics during cell death and modeling laminopathies using CRISPR/Cas9 in Drosophila melanogaster.

Degree: PhD, Biology, 2019, Boston University

 Characterization of apoptosis, a caspase-dependent cell death program, has led to diverse insights into development, tissue homeostasis, and numerous diseases. About a dozen other distinct… (more)

Subjects/Keywords: Cellular biology; Cell death; Drosophila; Lamin; Non-apoptotic; Nucleus; Nurse cell

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APA (6th Edition):

Yalonetskaya, A. (2019). Characterization of nuclear degradation dynamics during cell death and modeling laminopathies using CRISPR/Cas9 in Drosophila melanogaster. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/39597

Chicago Manual of Style (16th Edition):

Yalonetskaya, Alla. “Characterization of nuclear degradation dynamics during cell death and modeling laminopathies using CRISPR/Cas9 in Drosophila melanogaster.” 2019. Doctoral Dissertation, Boston University. Accessed December 01, 2020. http://hdl.handle.net/2144/39597.

MLA Handbook (7th Edition):

Yalonetskaya, Alla. “Characterization of nuclear degradation dynamics during cell death and modeling laminopathies using CRISPR/Cas9 in Drosophila melanogaster.” 2019. Web. 01 Dec 2020.

Vancouver:

Yalonetskaya A. Characterization of nuclear degradation dynamics during cell death and modeling laminopathies using CRISPR/Cas9 in Drosophila melanogaster. [Internet] [Doctoral dissertation]. Boston University; 2019. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/2144/39597.

Council of Science Editors:

Yalonetskaya A. Characterization of nuclear degradation dynamics during cell death and modeling laminopathies using CRISPR/Cas9 in Drosophila melanogaster. [Doctoral Dissertation]. Boston University; 2019. Available from: http://hdl.handle.net/2144/39597


University of Melbourne

29. IYER, SWETA. Bak (and Bax) activation and apoptotic pore formation: roles for the N and C terminus.

Degree: 2015, University of Melbourne

 Bak and Bax are members of the Bcl-2 family that regulate the mitochondrial pathway of apoptotic cell death. This type of cell death requires either… (more)

Subjects/Keywords: apoptosis; Bak; Bax; apoptotic pore formation; mitochondrial outer membrane permeabilisation

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APA (6th Edition):

IYER, S. (2015). Bak (and Bax) activation and apoptotic pore formation: roles for the N and C terminus. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/55466

Chicago Manual of Style (16th Edition):

IYER, SWETA. “Bak (and Bax) activation and apoptotic pore formation: roles for the N and C terminus.” 2015. Doctoral Dissertation, University of Melbourne. Accessed December 01, 2020. http://hdl.handle.net/11343/55466.

MLA Handbook (7th Edition):

IYER, SWETA. “Bak (and Bax) activation and apoptotic pore formation: roles for the N and C terminus.” 2015. Web. 01 Dec 2020.

Vancouver:

IYER S. Bak (and Bax) activation and apoptotic pore formation: roles for the N and C terminus. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2020 Dec 01]. Available from: http://hdl.handle.net/11343/55466.

Council of Science Editors:

IYER S. Bak (and Bax) activation and apoptotic pore formation: roles for the N and C terminus. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/55466


University of Southern California

30. Kay, Brittany P. Modeling anti-tumoral effects of drug-induced activation of the cell-extrinsic apoptotic pathway.

Degree: PhD, Biomedical Engineering, 2012, University of Southern California

 RshApo2L/TRAIL and Conatumumab bind to transmembrane death receptors and trigger the extrinsic cellular apoptotic pathway through a caspase-signaling cascade resulting in cell death. Tumor size… (more)

Subjects/Keywords: apoptosis; PKPD; PARA; extrinsic apoptotic pathway; cancer; modeling; caspase

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APA (6th Edition):

Kay, B. P. (2012). Modeling anti-tumoral effects of drug-induced activation of the cell-extrinsic apoptotic pathway. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57385/rec/4129

Chicago Manual of Style (16th Edition):

Kay, Brittany P. “Modeling anti-tumoral effects of drug-induced activation of the cell-extrinsic apoptotic pathway.” 2012. Doctoral Dissertation, University of Southern California. Accessed December 01, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57385/rec/4129.

MLA Handbook (7th Edition):

Kay, Brittany P. “Modeling anti-tumoral effects of drug-induced activation of the cell-extrinsic apoptotic pathway.” 2012. Web. 01 Dec 2020.

Vancouver:

Kay BP. Modeling anti-tumoral effects of drug-induced activation of the cell-extrinsic apoptotic pathway. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Dec 01]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57385/rec/4129.

Council of Science Editors:

Kay BP. Modeling anti-tumoral effects of drug-induced activation of the cell-extrinsic apoptotic pathway. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/57385/rec/4129

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