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You searched for subject:(potassium channel). Showing records 1 – 30 of 139 total matches.

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University of Manchester

1. Al-Chawishly, Mohammed Fadhil Saleem. PHARMACOLOGY OF Kv7 CHANNELS IN RAT PULMONARY ARTERY.

Degree: 2019, University of Manchester

 the pulmonary artery smooth muscle cells of rat, Kv7 channels are proposed to make an important contribution to the resting membrane potential and the regulation… (more)

Subjects/Keywords: Kv7 potassium channel

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Al-Chawishly, M. F. S. (2019). PHARMACOLOGY OF Kv7 CHANNELS IN RAT PULMONARY ARTERY. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319831

Chicago Manual of Style (16th Edition):

Al-Chawishly, Mohammed Fadhil Saleem. “PHARMACOLOGY OF Kv7 CHANNELS IN RAT PULMONARY ARTERY.” 2019. Doctoral Dissertation, University of Manchester. Accessed January 15, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319831.

MLA Handbook (7th Edition):

Al-Chawishly, Mohammed Fadhil Saleem. “PHARMACOLOGY OF Kv7 CHANNELS IN RAT PULMONARY ARTERY.” 2019. Web. 15 Jan 2021.

Vancouver:

Al-Chawishly MFS. PHARMACOLOGY OF Kv7 CHANNELS IN RAT PULMONARY ARTERY. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2021 Jan 15]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319831.

Council of Science Editors:

Al-Chawishly MFS. PHARMACOLOGY OF Kv7 CHANNELS IN RAT PULMONARY ARTERY. [Doctoral Dissertation]. University of Manchester; 2019. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:319831


University of Edinburgh

2. Pan, Geng. Potassium channel expression and function in the N9 murine microglial cell line.

Degree: PhD, 2012, University of Edinburgh

 Microglia are immunocompetent cells in the central nervous system that have many similarities with macrophages of peripheral tissues. Their activation protects local cells from foreign… (more)

Subjects/Keywords: microglia; potassium channel

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APA (6th Edition):

Pan, G. (2012). Potassium channel expression and function in the N9 murine microglial cell line. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8191

Chicago Manual of Style (16th Edition):

Pan, Geng. “Potassium channel expression and function in the N9 murine microglial cell line.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed January 15, 2021. http://hdl.handle.net/1842/8191.

MLA Handbook (7th Edition):

Pan, Geng. “Potassium channel expression and function in the N9 murine microglial cell line.” 2012. Web. 15 Jan 2021.

Vancouver:

Pan G. Potassium channel expression and function in the N9 murine microglial cell line. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1842/8191.

Council of Science Editors:

Pan G. Potassium channel expression and function in the N9 murine microglial cell line. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/8191


Temple University

3. Thomson, Andrew Shane. Voltage-dependent gating at the selectivity filter of the MthK K+ channel.

Degree: PhD, 2013, Temple University

Biochemistry

Voltage-dependent K+ channels can undergo a gating process known as C-type inactivation. This type of gating consists of entry into a nonconducting state that… (more)

Subjects/Keywords: Biochemistry; Channel; Inactivation; MthK; Potassium

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APA (6th Edition):

Thomson, A. S. (2013). Voltage-dependent gating at the selectivity filter of the MthK K+ channel. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214824

Chicago Manual of Style (16th Edition):

Thomson, Andrew Shane. “Voltage-dependent gating at the selectivity filter of the MthK K+ channel.” 2013. Doctoral Dissertation, Temple University. Accessed January 15, 2021. http://digital.library.temple.edu/u?/p245801coll10,214824.

MLA Handbook (7th Edition):

Thomson, Andrew Shane. “Voltage-dependent gating at the selectivity filter of the MthK K+ channel.” 2013. Web. 15 Jan 2021.

Vancouver:

Thomson AS. Voltage-dependent gating at the selectivity filter of the MthK K+ channel. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2021 Jan 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214824.

Council of Science Editors:

Thomson AS. Voltage-dependent gating at the selectivity filter of the MthK K+ channel. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214824


University of Vermont

4. Stirling, Lee. Dual Roles for Rhoa/Rho-Kinase in the Regulated Trafficking of a Voltage-Sensitive Potassium Channel.

Degree: MS, Cell and Molecular Biology, 2009, University of Vermont

 Kv1.2 is a member of the Shaker family of voltage-sensitive potassium channels and contributes to regulation of membrane excitability. The electrophysiological activity of Kv1.2 undergoes… (more)

Subjects/Keywords: potassium channel

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APA (6th Edition):

Stirling, L. (2009). Dual Roles for Rhoa/Rho-Kinase in the Regulated Trafficking of a Voltage-Sensitive Potassium Channel. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stirling, Lee. “Dual Roles for Rhoa/Rho-Kinase in the Regulated Trafficking of a Voltage-Sensitive Potassium Channel.” 2009. Thesis, University of Vermont. Accessed January 15, 2021. https://scholarworks.uvm.edu/graddis/223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stirling, Lee. “Dual Roles for Rhoa/Rho-Kinase in the Regulated Trafficking of a Voltage-Sensitive Potassium Channel.” 2009. Web. 15 Jan 2021.

Vancouver:

Stirling L. Dual Roles for Rhoa/Rho-Kinase in the Regulated Trafficking of a Voltage-Sensitive Potassium Channel. [Internet] [Thesis]. University of Vermont; 2009. [cited 2021 Jan 15]. Available from: https://scholarworks.uvm.edu/graddis/223.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stirling L. Dual Roles for Rhoa/Rho-Kinase in the Regulated Trafficking of a Voltage-Sensitive Potassium Channel. [Thesis]. University of Vermont; 2009. Available from: https://scholarworks.uvm.edu/graddis/223

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

5. Al-Chawishly, Mohammed. Pharmacology of Kv7 channels in rat pulmonary artery.

Degree: PhD, 2019, University of Manchester

 The pulmonary artery smooth muscle cells of rat, Kv7 channels, are proposed to make an important contribution to the resting membrane potential and the regulation… (more)

Subjects/Keywords: 610; Kv7 potassium channel

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APA (6th Edition):

Al-Chawishly, M. (2019). Pharmacology of Kv7 channels in rat pulmonary artery. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/pharmacology-of-kv7-channels-in-rat-pulmonary-artery(02a33369-bce3-49f6-b4df-725aadd694ba).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.779688

Chicago Manual of Style (16th Edition):

Al-Chawishly, Mohammed. “Pharmacology of Kv7 channels in rat pulmonary artery.” 2019. Doctoral Dissertation, University of Manchester. Accessed January 15, 2021. https://www.research.manchester.ac.uk/portal/en/theses/pharmacology-of-kv7-channels-in-rat-pulmonary-artery(02a33369-bce3-49f6-b4df-725aadd694ba).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.779688.

MLA Handbook (7th Edition):

Al-Chawishly, Mohammed. “Pharmacology of Kv7 channels in rat pulmonary artery.” 2019. Web. 15 Jan 2021.

Vancouver:

Al-Chawishly M. Pharmacology of Kv7 channels in rat pulmonary artery. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2021 Jan 15]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/pharmacology-of-kv7-channels-in-rat-pulmonary-artery(02a33369-bce3-49f6-b4df-725aadd694ba).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.779688.

Council of Science Editors:

Al-Chawishly M. Pharmacology of Kv7 channels in rat pulmonary artery. [Doctoral Dissertation]. University of Manchester; 2019. Available from: https://www.research.manchester.ac.uk/portal/en/theses/pharmacology-of-kv7-channels-in-rat-pulmonary-artery(02a33369-bce3-49f6-b4df-725aadd694ba).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.779688


Vanderbilt University

6. Carrington, Sheridan Jared S. Differential Glycosylation of the Inwardly Rectifying Potassium Channel Kir7.1 by G protein-coupled Receptors.

Degree: PhD, Molecular Physiology and Biophysics, 2019, Vanderbilt University

 Kir7.1 is an inwardly rectifying potassium channel with important roles in the regulation of the membrane potential in retinal pigment epithelium, uterine smooth muscle, and… (more)

Subjects/Keywords: GPCR; ion channel; kir; glycosylation; ion channel; potassium channel

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APA (6th Edition):

Carrington, S. J. S. (2019). Differential Glycosylation of the Inwardly Rectifying Potassium Channel Kir7.1 by G protein-coupled Receptors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10914

Chicago Manual of Style (16th Edition):

Carrington, Sheridan Jared S. “Differential Glycosylation of the Inwardly Rectifying Potassium Channel Kir7.1 by G protein-coupled Receptors.” 2019. Doctoral Dissertation, Vanderbilt University. Accessed January 15, 2021. http://hdl.handle.net/1803/10914.

MLA Handbook (7th Edition):

Carrington, Sheridan Jared S. “Differential Glycosylation of the Inwardly Rectifying Potassium Channel Kir7.1 by G protein-coupled Receptors.” 2019. Web. 15 Jan 2021.

Vancouver:

Carrington SJS. Differential Glycosylation of the Inwardly Rectifying Potassium Channel Kir7.1 by G protein-coupled Receptors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2019. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1803/10914.

Council of Science Editors:

Carrington SJS. Differential Glycosylation of the Inwardly Rectifying Potassium Channel Kir7.1 by G protein-coupled Receptors. [Doctoral Dissertation]. Vanderbilt University; 2019. Available from: http://hdl.handle.net/1803/10914


University of Alberta

7. Mercer, Robert Corrigan Curtis. The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation.

Degree: PhD, Department of Medicine, 2016, University of Alberta

 Prion diseases are invariably fatal neurodegenerative diseases of humans and other mammals. While they can manifest as sporadic, infectious or genetic etiologies, the central event… (more)

Subjects/Keywords: Gerstmann-Sträussler-Schienker; Potassium Channel; Prion; Neurodegeneration

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APA (6th Edition):

Mercer, R. C. C. (2016). The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cmp48sc82v

Chicago Manual of Style (16th Edition):

Mercer, Robert Corrigan Curtis. “The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation.” 2016. Doctoral Dissertation, University of Alberta. Accessed January 15, 2021. https://era.library.ualberta.ca/files/cmp48sc82v.

MLA Handbook (7th Edition):

Mercer, Robert Corrigan Curtis. “The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation.” 2016. Web. 15 Jan 2021.

Vancouver:

Mercer RCC. The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation. [Internet] [Doctoral dissertation]. University of Alberta; 2016. [cited 2021 Jan 15]. Available from: https://era.library.ualberta.ca/files/cmp48sc82v.

Council of Science Editors:

Mercer RCC. The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation. [Doctoral Dissertation]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cmp48sc82v


Penn State University

8. Liu, Hansi. Evolutionary and functional study of K channels – in search for the origin of Shaker channels and a functional study of an Elk channel.

Degree: 2015, Penn State University

 Voltage-gated potassium channels are important for maintaining normal excitability of nervous system. In this study, I investigated the origin of Shaker family channels in basal… (more)

Subjects/Keywords: Potassium Channel; Shaker; Elk; Cnidarian; Ctenophore

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APA (6th Edition):

Liu, H. (2015). Evolutionary and functional study of K channels – in search for the origin of Shaker channels and a functional study of an Elk channel. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/24902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Hansi. “Evolutionary and functional study of K channels – in search for the origin of Shaker channels and a functional study of an Elk channel.” 2015. Thesis, Penn State University. Accessed January 15, 2021. https://submit-etda.libraries.psu.edu/catalog/24902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Hansi. “Evolutionary and functional study of K channels – in search for the origin of Shaker channels and a functional study of an Elk channel.” 2015. Web. 15 Jan 2021.

Vancouver:

Liu H. Evolutionary and functional study of K channels – in search for the origin of Shaker channels and a functional study of an Elk channel. [Internet] [Thesis]. Penn State University; 2015. [cited 2021 Jan 15]. Available from: https://submit-etda.libraries.psu.edu/catalog/24902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu H. Evolutionary and functional study of K channels – in search for the origin of Shaker channels and a functional study of an Elk channel. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/24902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

9. Atkinson, Summer. Potassium Channel Allele Modulates Chronic Pain Experience.

Degree: Neuroscience, 2015, University of Vermont

  Chronic musculoskeletal pain is a complex disorder that often causes physical and psychological symptoms. While acute pain promotes healing of damaged tissue, chronic pain… (more)

Subjects/Keywords: KCNS1; chronic musculoskeletal pain; potassium channel

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APA (6th Edition):

Atkinson, S. (2015). Potassium Channel Allele Modulates Chronic Pain Experience. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/castheses/5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Atkinson, Summer. “Potassium Channel Allele Modulates Chronic Pain Experience.” 2015. Thesis, University of Vermont. Accessed January 15, 2021. https://scholarworks.uvm.edu/castheses/5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Atkinson, Summer. “Potassium Channel Allele Modulates Chronic Pain Experience.” 2015. Web. 15 Jan 2021.

Vancouver:

Atkinson S. Potassium Channel Allele Modulates Chronic Pain Experience. [Internet] [Thesis]. University of Vermont; 2015. [cited 2021 Jan 15]. Available from: https://scholarworks.uvm.edu/castheses/5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Atkinson S. Potassium Channel Allele Modulates Chronic Pain Experience. [Thesis]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/castheses/5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

10. Connors, Emilee. Positive Trafficking Pathways of a Voltage Gated Potassium Channel.

Degree: PhD, Pharmacology, 2009, University of Vermont

 ABSTRACT The voltage-gated potassium channel Kv1.2 is a key determinant of cellular excitability in the nervous and cardiovascular systems. In the brain, Kv1.2 is strongly… (more)

Subjects/Keywords: Potassium Channel; Phosphorylation

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APA (6th Edition):

Connors, E. (2009). Positive Trafficking Pathways of a Voltage Gated Potassium Channel. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/52

Chicago Manual of Style (16th Edition):

Connors, Emilee. “Positive Trafficking Pathways of a Voltage Gated Potassium Channel.” 2009. Doctoral Dissertation, University of Vermont. Accessed January 15, 2021. https://scholarworks.uvm.edu/graddis/52.

MLA Handbook (7th Edition):

Connors, Emilee. “Positive Trafficking Pathways of a Voltage Gated Potassium Channel.” 2009. Web. 15 Jan 2021.

Vancouver:

Connors E. Positive Trafficking Pathways of a Voltage Gated Potassium Channel. [Internet] [Doctoral dissertation]. University of Vermont; 2009. [cited 2021 Jan 15]. Available from: https://scholarworks.uvm.edu/graddis/52.

Council of Science Editors:

Connors E. Positive Trafficking Pathways of a Voltage Gated Potassium Channel. [Doctoral Dissertation]. University of Vermont; 2009. Available from: https://scholarworks.uvm.edu/graddis/52


University of Illinois – Chicago

11. Baskaran, Abhinaya. KCNF1 is a Novel Regulator of NSCLC Cell Growth Independent of Its Potassium Ion Channel Function.

Degree: 2017, University of Illinois – Chicago

 Lung cancer is the leading cause of cancer-related deaths worldwide. Non-Small Cell Lung Cancer (NSCLC) is the most common type of lung cancer and is… (more)

Subjects/Keywords: NSCLC; KCNF1; Wnt7a; Potassium ion channel

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APA (6th Edition):

Baskaran, A. (2017). KCNF1 is a Novel Regulator of NSCLC Cell Growth Independent of Its Potassium Ion Channel Function. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baskaran, Abhinaya. “KCNF1 is a Novel Regulator of NSCLC Cell Growth Independent of Its Potassium Ion Channel Function.” 2017. Thesis, University of Illinois – Chicago. Accessed January 15, 2021. http://hdl.handle.net/10027/21955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baskaran, Abhinaya. “KCNF1 is a Novel Regulator of NSCLC Cell Growth Independent of Its Potassium Ion Channel Function.” 2017. Web. 15 Jan 2021.

Vancouver:

Baskaran A. KCNF1 is a Novel Regulator of NSCLC Cell Growth Independent of Its Potassium Ion Channel Function. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/10027/21955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baskaran A. KCNF1 is a Novel Regulator of NSCLC Cell Growth Independent of Its Potassium Ion Channel Function. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

12. Howitt, Lauren. Effect of diet-induced obesity on vascular tone.

Degree: Medical Sciences, 2011, University of New South Wales

 Obesity is an established risk factor for hypertension and associated cardiovascular disease. The mechanisms underlying obesity-induced hypertension are unclear, but may involve functional changes in… (more)

Subjects/Keywords: Potassium channel; Arteriole; Obesity; Myogenic tone

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APA (6th Edition):

Howitt, L. (2011). Effect of diet-induced obesity on vascular tone. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50335 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9216/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Howitt, Lauren. “Effect of diet-induced obesity on vascular tone.” 2011. Doctoral Dissertation, University of New South Wales. Accessed January 15, 2021. http://handle.unsw.edu.au/1959.4/50335 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9216/SOURCE02?view=true.

MLA Handbook (7th Edition):

Howitt, Lauren. “Effect of diet-induced obesity on vascular tone.” 2011. Web. 15 Jan 2021.

Vancouver:

Howitt L. Effect of diet-induced obesity on vascular tone. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2021 Jan 15]. Available from: http://handle.unsw.edu.au/1959.4/50335 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9216/SOURCE02?view=true.

Council of Science Editors:

Howitt L. Effect of diet-induced obesity on vascular tone. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50335 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9216/SOURCE02?view=true


NSYSU

13. Lee, Li-ya. Mutations on EF-hands of potassium channel-interacting protein2.2 affect its interaction with Kv channel.

Degree: Master, Institute of Biomedical Sciences, 2006, NSYSU

 Mutagenesis studies on the four EF-hands of KChIP2.2 (Potassium channel-interacting protein 2.2) were carried out to explore the conformational transition upon the binding of Ca2+… (more)

Subjects/Keywords: potassium channel; KChIP2.2; Kv channel

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APA (6th Edition):

Lee, L. (2006). Mutations on EF-hands of potassium channel-interacting protein2.2 affect its interaction with Kv channel. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0728106-024055

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Li-ya. “Mutations on EF-hands of potassium channel-interacting protein2.2 affect its interaction with Kv channel.” 2006. Thesis, NSYSU. Accessed January 15, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0728106-024055.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Li-ya. “Mutations on EF-hands of potassium channel-interacting protein2.2 affect its interaction with Kv channel.” 2006. Web. 15 Jan 2021.

Vancouver:

Lee L. Mutations on EF-hands of potassium channel-interacting protein2.2 affect its interaction with Kv channel. [Internet] [Thesis]. NSYSU; 2006. [cited 2021 Jan 15]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0728106-024055.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee L. Mutations on EF-hands of potassium channel-interacting protein2.2 affect its interaction with Kv channel. [Thesis]. NSYSU; 2006. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0728106-024055

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

14. Li, Xiaofan. Evolution origins and Pip2 modulation of voltage-gated K+ channels.

Degree: 2015, Penn State University

 Voltage-gated K+ channels are important regulators of neuronal excitability. Bilaterians have eight functionally distinct Voltage-gated K+ channel subfamilies: Shaker, Shab, Shaw, Shal, KCNQ, Eag, Erg… (more)

Subjects/Keywords: potassium channel; ion channel; evolution; phosphoinositide; PIP2; excitability; ctenophore; nematostella

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APA (6th Edition):

Li, X. (2015). Evolution origins and Pip2 modulation of voltage-gated K+ channels. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/26772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Xiaofan. “Evolution origins and Pip2 modulation of voltage-gated K+ channels.” 2015. Thesis, Penn State University. Accessed January 15, 2021. https://submit-etda.libraries.psu.edu/catalog/26772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Xiaofan. “Evolution origins and Pip2 modulation of voltage-gated K+ channels.” 2015. Web. 15 Jan 2021.

Vancouver:

Li X. Evolution origins and Pip2 modulation of voltage-gated K+ channels. [Internet] [Thesis]. Penn State University; 2015. [cited 2021 Jan 15]. Available from: https://submit-etda.libraries.psu.edu/catalog/26772.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li X. Evolution origins and Pip2 modulation of voltage-gated K+ channels. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/26772

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

15. Lee, Soomin. The KCNE2 potassium channel subunit in metabolic syndrome.

Degree: Pharmacology and Toxicology, 2016, University of California – Irvine

 Coronary artery disease (CAD) is the number 1 cause of death in the U.S and globally. The traditional risk factors for CAD are hypercholesterolemia, hypertriglyceridemia,… (more)

Subjects/Keywords: Pharmacology; Molecular biology; Atherosclerosis; CAD; KCNE2; NAFLD; Potassium Channel; T2DM

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APA (6th Edition):

Lee, S. (2016). The KCNE2 potassium channel subunit in metabolic syndrome. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/2t30h8pp

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Soomin. “The KCNE2 potassium channel subunit in metabolic syndrome.” 2016. Thesis, University of California – Irvine. Accessed January 15, 2021. http://www.escholarship.org/uc/item/2t30h8pp.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Soomin. “The KCNE2 potassium channel subunit in metabolic syndrome.” 2016. Web. 15 Jan 2021.

Vancouver:

Lee S. The KCNE2 potassium channel subunit in metabolic syndrome. [Internet] [Thesis]. University of California – Irvine; 2016. [cited 2021 Jan 15]. Available from: http://www.escholarship.org/uc/item/2t30h8pp.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee S. The KCNE2 potassium channel subunit in metabolic syndrome. [Thesis]. University of California – Irvine; 2016. Available from: http://www.escholarship.org/uc/item/2t30h8pp

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

16. Lang, Yiqiao Veronica. Pharmacogenomics of Sulfonylureas and Glinides on ATP-Sensitive Potassium Channel.

Degree: MS, Department of Pharmacology, 2012, University of Alberta

 The common ATP-sensitive (KATP) channel variants E23K and S1369A, found in the KCNJ11 and ABCC8 genes respectively, form a haplotype that is associated with an… (more)

Subjects/Keywords: Type 2 Diabetes; ATP-Sensitive Potassium Channel; Sulfonylureas and Glinides; Pharmacogenomics

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APA (6th Edition):

Lang, Y. V. (2012). Pharmacogenomics of Sulfonylureas and Glinides on ATP-Sensitive Potassium Channel. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/08612p55c

Chicago Manual of Style (16th Edition):

Lang, Yiqiao Veronica. “Pharmacogenomics of Sulfonylureas and Glinides on ATP-Sensitive Potassium Channel.” 2012. Masters Thesis, University of Alberta. Accessed January 15, 2021. https://era.library.ualberta.ca/files/08612p55c.

MLA Handbook (7th Edition):

Lang, Yiqiao Veronica. “Pharmacogenomics of Sulfonylureas and Glinides on ATP-Sensitive Potassium Channel.” 2012. Web. 15 Jan 2021.

Vancouver:

Lang YV. Pharmacogenomics of Sulfonylureas and Glinides on ATP-Sensitive Potassium Channel. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2021 Jan 15]. Available from: https://era.library.ualberta.ca/files/08612p55c.

Council of Science Editors:

Lang YV. Pharmacogenomics of Sulfonylureas and Glinides on ATP-Sensitive Potassium Channel. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/08612p55c


Vanderbilt University

17. Jorge, Benjamin S. Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 Epilepsy is a common neurological disease characterized by an enduring predisposition to generate seizures. Although multiple factors contribute to epilepsy, the majority of cases are… (more)

Subjects/Keywords: potassium channel; epileptic encephalopathy; mouse model; genetics; whole-exome sequencing; epilepsy

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APA (6th Edition):

Jorge, B. S. (2014). Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14387

Chicago Manual of Style (16th Edition):

Jorge, Benjamin S. “Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed January 15, 2021. http://hdl.handle.net/1803/14387.

MLA Handbook (7th Edition):

Jorge, Benjamin S. “Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility.” 2014. Web. 15 Jan 2021.

Vancouver:

Jorge BS. Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1803/14387.

Council of Science Editors:

Jorge BS. Genetic Variation in the Voltage-gated Potassium Channel Genes KCNV2 and KCNB1 Contributes to Epilepsy Susceptibility. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/14387

18. Qile, Muge. Targeting Potassium Channel Trafficking in Cardiac Arrhythmia.

Degree: 2020, University Utrecht

 Ion channel trafficking, this is the transport of ion channel proteins within the heart cell, is an underestimated cause of severe cardiac arrhythmias. Importantly, it… (more)

Subjects/Keywords: potassium channel; arrhythmia; trafficking pathway

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APA (6th Edition):

Qile, M. (2020). Targeting Potassium Channel Trafficking in Cardiac Arrhythmia. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/398506 ; URN:NBN:NL:UI:10-1874-398506 ; 10.33540/55 ; 1874/398506 ; urn:isbn:9789464160505 ; URN:NBN:NL:UI:10-1874-398506 ; https://dspace.library.uu.nl/handle/1874/398506

Chicago Manual of Style (16th Edition):

Qile, Muge. “Targeting Potassium Channel Trafficking in Cardiac Arrhythmia.” 2020. Doctoral Dissertation, University Utrecht. Accessed January 15, 2021. https://dspace.library.uu.nl/handle/1874/398506 ; URN:NBN:NL:UI:10-1874-398506 ; 10.33540/55 ; 1874/398506 ; urn:isbn:9789464160505 ; URN:NBN:NL:UI:10-1874-398506 ; https://dspace.library.uu.nl/handle/1874/398506.

MLA Handbook (7th Edition):

Qile, Muge. “Targeting Potassium Channel Trafficking in Cardiac Arrhythmia.” 2020. Web. 15 Jan 2021.

Vancouver:

Qile M. Targeting Potassium Channel Trafficking in Cardiac Arrhythmia. [Internet] [Doctoral dissertation]. University Utrecht; 2020. [cited 2021 Jan 15]. Available from: https://dspace.library.uu.nl/handle/1874/398506 ; URN:NBN:NL:UI:10-1874-398506 ; 10.33540/55 ; 1874/398506 ; urn:isbn:9789464160505 ; URN:NBN:NL:UI:10-1874-398506 ; https://dspace.library.uu.nl/handle/1874/398506.

Council of Science Editors:

Qile M. Targeting Potassium Channel Trafficking in Cardiac Arrhythmia. [Doctoral Dissertation]. University Utrecht; 2020. Available from: https://dspace.library.uu.nl/handle/1874/398506 ; URN:NBN:NL:UI:10-1874-398506 ; 10.33540/55 ; 1874/398506 ; urn:isbn:9789464160505 ; URN:NBN:NL:UI:10-1874-398506 ; https://dspace.library.uu.nl/handle/1874/398506


Queens University

19. Chen, Jeffery. Identification of Retrieval-Trafficking of the Human Ether A-Go-Go-Related Gene Channel .

Degree: Physiology, 2014, Queens University

 The human ether-a-go-go related gene (hERG) encodes the pore-forming subunit of the rapidly activating delayed rectifier potassium channel (IKr). A reduction in the hERG current… (more)

Subjects/Keywords: Arrhythmia ; Protein Trafficking ; Cardiac Electrophysiology ; Potassium Ion Channel

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APA (6th Edition):

Chen, J. (2014). Identification of Retrieval-Trafficking of the Human Ether A-Go-Go-Related Gene Channel . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/12365

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Jeffery. “Identification of Retrieval-Trafficking of the Human Ether A-Go-Go-Related Gene Channel .” 2014. Thesis, Queens University. Accessed January 15, 2021. http://hdl.handle.net/1974/12365.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Jeffery. “Identification of Retrieval-Trafficking of the Human Ether A-Go-Go-Related Gene Channel .” 2014. Web. 15 Jan 2021.

Vancouver:

Chen J. Identification of Retrieval-Trafficking of the Human Ether A-Go-Go-Related Gene Channel . [Internet] [Thesis]. Queens University; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1974/12365.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen J. Identification of Retrieval-Trafficking of the Human Ether A-Go-Go-Related Gene Channel . [Thesis]. Queens University; 2014. Available from: http://hdl.handle.net/1974/12365

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

20. Hogan-Cann, Andrew. Proteolytic Cleavage of the Kv1.5 Channel in the S1-S2 Linker Does Not Affect Channel Function .

Degree: Physiology, 2015, Queens University

 Atrial fibrillation (AF), the most prevalent human cardiac arrhythmia, is characterized by rapid and disorderly electrical activity in the atria of the heart. Kv1.5 channel(more)

Subjects/Keywords: electrophysiology ; Voltage-gated potassium channel ; Cell-surface protein ; Trafficking ; Proteolysis ; Kv1.5

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APA (6th Edition):

Hogan-Cann, A. (2015). Proteolytic Cleavage of the Kv1.5 Channel in the S1-S2 Linker Does Not Affect Channel Function . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hogan-Cann, Andrew. “Proteolytic Cleavage of the Kv1.5 Channel in the S1-S2 Linker Does Not Affect Channel Function .” 2015. Thesis, Queens University. Accessed January 15, 2021. http://hdl.handle.net/1974/13500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hogan-Cann, Andrew. “Proteolytic Cleavage of the Kv1.5 Channel in the S1-S2 Linker Does Not Affect Channel Function .” 2015. Web. 15 Jan 2021.

Vancouver:

Hogan-Cann A. Proteolytic Cleavage of the Kv1.5 Channel in the S1-S2 Linker Does Not Affect Channel Function . [Internet] [Thesis]. Queens University; 2015. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/1974/13500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hogan-Cann A. Proteolytic Cleavage of the Kv1.5 Channel in the S1-S2 Linker Does Not Affect Channel Function . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

21. Adney, Scott. Protein Kinase C Dependent Inhibition of Kir3.2 (GIRK2) Channel Activity and Its Molecular Determinants.

Degree: PhD, Physiology, 2013, Virginia Commonwealth University

 Inwardly rectifying potassium (Kir) channels are critically important for regulating resting membrane potential in excitable cells, a job underscored by the severe pathophysiology associated with… (more)

Subjects/Keywords: PKC; GIRK; PIP2; Kir3; potassium channel; Life Sciences; Physiology

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APA (6th Edition):

Adney, S. (2013). Protein Kinase C Dependent Inhibition of Kir3.2 (GIRK2) Channel Activity and Its Molecular Determinants. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/6457-0944 ; https://scholarscompass.vcu.edu/etd/3214

Chicago Manual of Style (16th Edition):

Adney, Scott. “Protein Kinase C Dependent Inhibition of Kir3.2 (GIRK2) Channel Activity and Its Molecular Determinants.” 2013. Doctoral Dissertation, Virginia Commonwealth University. Accessed January 15, 2021. https://doi.org/10.25772/6457-0944 ; https://scholarscompass.vcu.edu/etd/3214.

MLA Handbook (7th Edition):

Adney, Scott. “Protein Kinase C Dependent Inhibition of Kir3.2 (GIRK2) Channel Activity and Its Molecular Determinants.” 2013. Web. 15 Jan 2021.

Vancouver:

Adney S. Protein Kinase C Dependent Inhibition of Kir3.2 (GIRK2) Channel Activity and Its Molecular Determinants. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2013. [cited 2021 Jan 15]. Available from: https://doi.org/10.25772/6457-0944 ; https://scholarscompass.vcu.edu/etd/3214.

Council of Science Editors:

Adney S. Protein Kinase C Dependent Inhibition of Kir3.2 (GIRK2) Channel Activity and Its Molecular Determinants. [Doctoral Dissertation]. Virginia Commonwealth University; 2013. Available from: https://doi.org/10.25772/6457-0944 ; https://scholarscompass.vcu.edu/etd/3214

22. Tomczyńska, Magdelena. Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2009, Orléans; Académie polonaise des sciences

GIRK2 est situé sur le chromosome 21, dont la trisomie cause le syndrome de Down (DS). Les proportionss des sous-populations de lymphocytes T sont altérées,… (more)

Subjects/Keywords: Girk2; G-protein inwardly rectifying potassium channel 2

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APA (6th Edition):

Tomczyńska, M. (2009). Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome. (Doctoral Dissertation). Orléans; Académie polonaise des sciences. Retrieved from http://www.theses.fr/2009ORLE2067

Chicago Manual of Style (16th Edition):

Tomczyńska, Magdelena. “Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome.” 2009. Doctoral Dissertation, Orléans; Académie polonaise des sciences. Accessed January 15, 2021. http://www.theses.fr/2009ORLE2067.

MLA Handbook (7th Edition):

Tomczyńska, Magdelena. “Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome.” 2009. Web. 15 Jan 2021.

Vancouver:

Tomczyńska M. Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome. [Internet] [Doctoral dissertation]. Orléans; Académie polonaise des sciences; 2009. [cited 2021 Jan 15]. Available from: http://www.theses.fr/2009ORLE2067.

Council of Science Editors:

Tomczyńska M. Biologie de l'endothélium vasculaire isolé de souris transgéniques YAC67 et YAC84- modèles murins du syndrome de Down : Biology of vascular endothelium isolated from transgenic mice YAC67 and YAC84 -mouse models for Down syndrome. [Doctoral Dissertation]. Orléans; Académie polonaise des sciences; 2009. Available from: http://www.theses.fr/2009ORLE2067


University of Manchester

23. Smith, Keith. Molecular Pathophysiology of Cloned KATP Channels and Implications for Cardiovascular Disease.

Degree: 2014, University of Manchester

 ATP-sensitive potassium (KATP) channels are a class of ion channels involved in a multitude of cellular roles. The hallmark feature of these ion channels is… (more)

Subjects/Keywords: ATP-sensitive potassium channel; Kir6.x; SUR2; Coronary spasm; Myocardial infarction

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APA (6th Edition):

Smith, K. (2014). Molecular Pathophysiology of Cloned KATP Channels and Implications for Cardiovascular Disease. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:227797

Chicago Manual of Style (16th Edition):

Smith, Keith. “Molecular Pathophysiology of Cloned KATP Channels and Implications for Cardiovascular Disease.” 2014. Doctoral Dissertation, University of Manchester. Accessed January 15, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:227797.

MLA Handbook (7th Edition):

Smith, Keith. “Molecular Pathophysiology of Cloned KATP Channels and Implications for Cardiovascular Disease.” 2014. Web. 15 Jan 2021.

Vancouver:

Smith K. Molecular Pathophysiology of Cloned KATP Channels and Implications for Cardiovascular Disease. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2021 Jan 15]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:227797.

Council of Science Editors:

Smith K. Molecular Pathophysiology of Cloned KATP Channels and Implications for Cardiovascular Disease. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:227797


University of Illinois – Chicago

24. Dookeran, Keith A. Two‐pore Domain Potassium Channel Genes and Breast Cancer in The Cancer Genome Atlas.

Degree: 2016, University of Illinois – Chicago

 The overarching goal of this dissertation was to systematically evaluate molecular markers (DNA methylation, gene expression, and copy number changes) of K2P channel genes in… (more)

Subjects/Keywords: Two-pore domain potassium channel genes; breast cancer subtype

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APA (6th Edition):

Dookeran, K. A. (2016). Two‐pore Domain Potassium Channel Genes and Breast Cancer in The Cancer Genome Atlas. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21277

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dookeran, Keith A. “Two‐pore Domain Potassium Channel Genes and Breast Cancer in The Cancer Genome Atlas.” 2016. Thesis, University of Illinois – Chicago. Accessed January 15, 2021. http://hdl.handle.net/10027/21277.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dookeran, Keith A. “Two‐pore Domain Potassium Channel Genes and Breast Cancer in The Cancer Genome Atlas.” 2016. Web. 15 Jan 2021.

Vancouver:

Dookeran KA. Two‐pore Domain Potassium Channel Genes and Breast Cancer in The Cancer Genome Atlas. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/10027/21277.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dookeran KA. Two‐pore Domain Potassium Channel Genes and Breast Cancer in The Cancer Genome Atlas. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21277

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

25. Riddle, Melissa. Role of endothelial BKCa channel activity in diminished vasoconstrictor reactivity following chronic hypoxia.

Degree: Biomedical Sciences Graduate Program, 2011, University of New Mexico

 Vasoconstrictor responsiveness of the systemic circulation is attenuated following prolonged exposure to hypoxia. Previous work from our laboratory has demonstrated vasoconstrictor reactivity is diminished due… (more)

Subjects/Keywords: Caveolin-1; Calcium-activated large conductance potassium channel

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APA (6th Edition):

Riddle, M. (2011). Role of endothelial BKCa channel activity in diminished vasoconstrictor reactivity following chronic hypoxia. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/142

Chicago Manual of Style (16th Edition):

Riddle, Melissa. “Role of endothelial BKCa channel activity in diminished vasoconstrictor reactivity following chronic hypoxia.” 2011. Doctoral Dissertation, University of New Mexico. Accessed January 15, 2021. https://digitalrepository.unm.edu/biom_etds/142.

MLA Handbook (7th Edition):

Riddle, Melissa. “Role of endothelial BKCa channel activity in diminished vasoconstrictor reactivity following chronic hypoxia.” 2011. Web. 15 Jan 2021.

Vancouver:

Riddle M. Role of endothelial BKCa channel activity in diminished vasoconstrictor reactivity following chronic hypoxia. [Internet] [Doctoral dissertation]. University of New Mexico; 2011. [cited 2021 Jan 15]. Available from: https://digitalrepository.unm.edu/biom_etds/142.

Council of Science Editors:

Riddle M. Role of endothelial BKCa channel activity in diminished vasoconstrictor reactivity following chronic hypoxia. [Doctoral Dissertation]. University of New Mexico; 2011. Available from: https://digitalrepository.unm.edu/biom_etds/142


University of Melbourne

26. Luna Ramirez, Karen Sofia. Urodacus yaschenkoi, Australian scorpion: phylogeny, venom characterization and pharmacology.

Degree: 2014, University of Melbourne

 Australia has over 40 species of scorpions organised in four families: Buthidae, Bothriuridae, Urodacidae and Liochelidae (old name Ischnuridae). Unlike their overseas counterparts, Australian scorpions… (more)

Subjects/Keywords: toxinology; venom; scorpion; proteomics; cDNA library; antimicrobial; potassium channel blocker

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APA (6th Edition):

Luna Ramirez, K. S. (2014). Urodacus yaschenkoi, Australian scorpion: phylogeny, venom characterization and pharmacology. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/39600

Chicago Manual of Style (16th Edition):

Luna Ramirez, Karen Sofia. “Urodacus yaschenkoi, Australian scorpion: phylogeny, venom characterization and pharmacology.” 2014. Doctoral Dissertation, University of Melbourne. Accessed January 15, 2021. http://hdl.handle.net/11343/39600.

MLA Handbook (7th Edition):

Luna Ramirez, Karen Sofia. “Urodacus yaschenkoi, Australian scorpion: phylogeny, venom characterization and pharmacology.” 2014. Web. 15 Jan 2021.

Vancouver:

Luna Ramirez KS. Urodacus yaschenkoi, Australian scorpion: phylogeny, venom characterization and pharmacology. [Internet] [Doctoral dissertation]. University of Melbourne; 2014. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/11343/39600.

Council of Science Editors:

Luna Ramirez KS. Urodacus yaschenkoi, Australian scorpion: phylogeny, venom characterization and pharmacology. [Doctoral Dissertation]. University of Melbourne; 2014. Available from: http://hdl.handle.net/11343/39600


University of Southern California

27. Tabbakhian, Melia A. KcsA: mutational analysis of ion selectivity with molecular dynamics.

Degree: MS, Physiology and Biophysics, 2013, University of Southern California

 The mechanism of ion selectivity in K⁺-selective ion channels has not yet been determined. In previous studies it was found that K⁺ ions were allowed… (more)

Subjects/Keywords: ion channel; KcsA; ions; potassium; sodium; molecular dynamics; step-wise pulling

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APA (6th Edition):

Tabbakhian, M. A. (2013). KcsA: mutational analysis of ion selectivity with molecular dynamics. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316791/rec/3705

Chicago Manual of Style (16th Edition):

Tabbakhian, Melia A. “KcsA: mutational analysis of ion selectivity with molecular dynamics.” 2013. Masters Thesis, University of Southern California. Accessed January 15, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316791/rec/3705.

MLA Handbook (7th Edition):

Tabbakhian, Melia A. “KcsA: mutational analysis of ion selectivity with molecular dynamics.” 2013. Web. 15 Jan 2021.

Vancouver:

Tabbakhian MA. KcsA: mutational analysis of ion selectivity with molecular dynamics. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2021 Jan 15]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316791/rec/3705.

Council of Science Editors:

Tabbakhian MA. KcsA: mutational analysis of ion selectivity with molecular dynamics. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/316791/rec/3705

28. Corsaro, Veronica Carmen. Cooperation between potassium channels and gap junctions: interaction between Kv1.1 channel and Pannexin 1.

Degree: 2012, Università degli Studi di Catania

 The beta 3 subunit of voltage gated has been recently identified as a modulatory macromolecule of pannexin 1. Our interest has focused on the possible… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; potassium channel, pannexin, channelopathies

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Corsaro, V. C. (2012). Cooperation between potassium channels and gap junctions: interaction between Kv1.1 channel and Pannexin 1. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Corsaro, Veronica Carmen. “Cooperation between potassium channels and gap junctions: interaction between Kv1.1 channel and Pannexin 1.” 2012. Thesis, Università degli Studi di Catania. Accessed January 15, 2021. http://hdl.handle.net/10761/1037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Corsaro, Veronica Carmen. “Cooperation between potassium channels and gap junctions: interaction between Kv1.1 channel and Pannexin 1.” 2012. Web. 15 Jan 2021.

Vancouver:

Corsaro VC. Cooperation between potassium channels and gap junctions: interaction between Kv1.1 channel and Pannexin 1. [Internet] [Thesis]. Università degli Studi di Catania; 2012. [cited 2021 Jan 15]. Available from: http://hdl.handle.net/10761/1037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Corsaro VC. Cooperation between potassium channels and gap junctions: interaction between Kv1.1 channel and Pannexin 1. [Thesis]. Università degli Studi di Catania; 2012. Available from: http://hdl.handle.net/10761/1037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

29. FELLOWS, CHRISTOPHER J. The Role of ATP-Sensitive Inward Rectifier Potassium Channels In The Regulation of Reactive Oxygen Species In The Western Honey Bee, APIS Mellifera L.

Degree: MS, Entomology, 2019, Louisiana State University

  Colonies of managed honey bees are annually being lost at an unsustainable rate, partly due to reduced immunocompetence that leads to acute viral outbreaks… (more)

Subjects/Keywords: Honey Bee; ROS; Reactive Oxygen Species; Inward Rectifier Potassium Channel

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

FELLOWS, C. J. (2019). The Role of ATP-Sensitive Inward Rectifier Potassium Channels In The Regulation of Reactive Oxygen Species In The Western Honey Bee, APIS Mellifera L. (Masters Thesis). Louisiana State University. Retrieved from https://digitalcommons.lsu.edu/gradschool_theses/4988

Chicago Manual of Style (16th Edition):

FELLOWS, CHRISTOPHER J. “The Role of ATP-Sensitive Inward Rectifier Potassium Channels In The Regulation of Reactive Oxygen Species In The Western Honey Bee, APIS Mellifera L.” 2019. Masters Thesis, Louisiana State University. Accessed January 15, 2021. https://digitalcommons.lsu.edu/gradschool_theses/4988.

MLA Handbook (7th Edition):

FELLOWS, CHRISTOPHER J. “The Role of ATP-Sensitive Inward Rectifier Potassium Channels In The Regulation of Reactive Oxygen Species In The Western Honey Bee, APIS Mellifera L.” 2019. Web. 15 Jan 2021.

Vancouver:

FELLOWS CJ. The Role of ATP-Sensitive Inward Rectifier Potassium Channels In The Regulation of Reactive Oxygen Species In The Western Honey Bee, APIS Mellifera L. [Internet] [Masters thesis]. Louisiana State University; 2019. [cited 2021 Jan 15]. Available from: https://digitalcommons.lsu.edu/gradschool_theses/4988.

Council of Science Editors:

FELLOWS CJ. The Role of ATP-Sensitive Inward Rectifier Potassium Channels In The Regulation of Reactive Oxygen Species In The Western Honey Bee, APIS Mellifera L. [Masters Thesis]. Louisiana State University; 2019. Available from: https://digitalcommons.lsu.edu/gradschool_theses/4988


University of New South Wales

30. Heide, Juliane. Role of KCNH2 potassium channels in the pathogenesis of schizophrenia.

Degree: Victor Chang Cardiac Research Institute, 2013, University of New South Wales

 Schizophrenia is a devastating mental disorder characterized by hallucinations, delusions, cognitive and behavioural issues. Single nucleotide polymorphisms in the second intron of the KCNH2 gene,… (more)

Subjects/Keywords: Antipsychotics; hERG/Kv11.1 potassium channel; Schizophrenia; CATIE trail; DLPFC; TRC cohort

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Heide, J. (2013). Role of KCNH2 potassium channels in the pathogenesis of schizophrenia. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52857 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11530/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Heide, Juliane. “Role of KCNH2 potassium channels in the pathogenesis of schizophrenia.” 2013. Doctoral Dissertation, University of New South Wales. Accessed January 15, 2021. http://handle.unsw.edu.au/1959.4/52857 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11530/SOURCE01?view=true.

MLA Handbook (7th Edition):

Heide, Juliane. “Role of KCNH2 potassium channels in the pathogenesis of schizophrenia.” 2013. Web. 15 Jan 2021.

Vancouver:

Heide J. Role of KCNH2 potassium channels in the pathogenesis of schizophrenia. [Internet] [Doctoral dissertation]. University of New South Wales; 2013. [cited 2021 Jan 15]. Available from: http://handle.unsw.edu.au/1959.4/52857 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11530/SOURCE01?view=true.

Council of Science Editors:

Heide J. Role of KCNH2 potassium channels in the pathogenesis of schizophrenia. [Doctoral Dissertation]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/52857 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11530/SOURCE01?view=true

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