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You searched for subject:(physiology). Showing records 1 – 30 of 16974 total matches.

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1. Carahalios, Adam. An analysis of the bicycle-rider interface forces in stationary road cycling.

Degree: 2015, University of Colorado at Boulder

  Two distinct studies were undertaken in order to examine the effects that both external weight distribution, and internal, bike-rider interface forces had on cyclists.… (more)

Subjects/Keywords: Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carahalios, A. (2015). An analysis of the bicycle-rider interface forces in stationary road cycling. (Thesis). University of Colorado at Boulder. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=1600509

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carahalios, Adam. “An analysis of the bicycle-rider interface forces in stationary road cycling.” 2015. Thesis, University of Colorado at Boulder. Accessed February 27, 2021. http://pqdtopen.proquest.com/#viewpdf?dispub=1600509.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carahalios, Adam. “An analysis of the bicycle-rider interface forces in stationary road cycling.” 2015. Web. 27 Feb 2021.

Vancouver:

Carahalios A. An analysis of the bicycle-rider interface forces in stationary road cycling. [Internet] [Thesis]. University of Colorado at Boulder; 2015. [cited 2021 Feb 27]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=1600509.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carahalios A. An analysis of the bicycle-rider interface forces in stationary road cycling. [Thesis]. University of Colorado at Boulder; 2015. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=1600509

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The University of Utah

2. Barrett-O'Keefe, Zachary. Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction.

Degree: 2016, The University of Utah

  The objective of this dissertation was to systematically investigate the hemodynamic response to exercise in heart failure with reduced ejection fraction (HFrEF) and healthy… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Barrett-O'Keefe, Z. (2016). Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction. (Thesis). The University of Utah. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=10001028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barrett-O'Keefe, Zachary. “Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction.” 2016. Thesis, The University of Utah. Accessed February 27, 2021. http://pqdtopen.proquest.com/#viewpdf?dispub=10001028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barrett-O'Keefe, Zachary. “Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction.” 2016. Web. 27 Feb 2021.

Vancouver:

Barrett-O'Keefe Z. Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction. [Internet] [Thesis]. The University of Utah; 2016. [cited 2021 Feb 27]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10001028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barrett-O'Keefe Z. Cardiovascular control during exercise and the role of the sympathetic nervous system in heart failure with reduced ejection fraction. [Thesis]. The University of Utah; 2016. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10001028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

3. Karbassi, Elaheh. Orchestration of Cardiac Gene Expression Mediated by Global Chromatin Architecture.

Degree: Molec, Cell, & Integ Physiology, 2016, UCLA

 The underlying mechanisms by which cell identity is achieved in a cell type-specific manner during development are unknown. In this project, we examine the mechanisms… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Karbassi, E. (2016). Orchestration of Cardiac Gene Expression Mediated by Global Chromatin Architecture. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/9mh471vk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Karbassi, Elaheh. “Orchestration of Cardiac Gene Expression Mediated by Global Chromatin Architecture.” 2016. Thesis, UCLA. Accessed February 27, 2021. http://www.escholarship.org/uc/item/9mh471vk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Karbassi, Elaheh. “Orchestration of Cardiac Gene Expression Mediated by Global Chromatin Architecture.” 2016. Web. 27 Feb 2021.

Vancouver:

Karbassi E. Orchestration of Cardiac Gene Expression Mediated by Global Chromatin Architecture. [Internet] [Thesis]. UCLA; 2016. [cited 2021 Feb 27]. Available from: http://www.escholarship.org/uc/item/9mh471vk.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Karbassi E. Orchestration of Cardiac Gene Expression Mediated by Global Chromatin Architecture. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/9mh471vk

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

4. Vuong, Helen Elaine. Neuropeptide releasing amacrine cells modulate microcircuits in the inner retina.

Degree: Molec, Cell, & Integ Physiology, 2015, UCLA

 Amacrine cells form the most diverse group of interneurons in the retina. There are at least 30 identified types, which are differentiated based on their… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Vuong, H. E. (2015). Neuropeptide releasing amacrine cells modulate microcircuits in the inner retina. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7141s4ms

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vuong, Helen Elaine. “Neuropeptide releasing amacrine cells modulate microcircuits in the inner retina.” 2015. Thesis, UCLA. Accessed February 27, 2021. http://www.escholarship.org/uc/item/7141s4ms.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vuong, Helen Elaine. “Neuropeptide releasing amacrine cells modulate microcircuits in the inner retina.” 2015. Web. 27 Feb 2021.

Vancouver:

Vuong HE. Neuropeptide releasing amacrine cells modulate microcircuits in the inner retina. [Internet] [Thesis]. UCLA; 2015. [cited 2021 Feb 27]. Available from: http://www.escholarship.org/uc/item/7141s4ms.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vuong HE. Neuropeptide releasing amacrine cells modulate microcircuits in the inner retina. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/7141s4ms

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

5. Lindsay, Catherine Claire. Klotho Enhances Postnatal Skeletal Muscle Growth.

Degree: Physiological Science, 2015, UCLA

 Klotho is a pleiotrophic gene that regulates homeostasis in various tissues. Although Klotho function has been typically viewed in the context of an "anti-aging" molecule,… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Lindsay, C. C. (2015). Klotho Enhances Postnatal Skeletal Muscle Growth. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/8hg0t2p8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lindsay, Catherine Claire. “Klotho Enhances Postnatal Skeletal Muscle Growth.” 2015. Thesis, UCLA. Accessed February 27, 2021. http://www.escholarship.org/uc/item/8hg0t2p8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lindsay, Catherine Claire. “Klotho Enhances Postnatal Skeletal Muscle Growth.” 2015. Web. 27 Feb 2021.

Vancouver:

Lindsay CC. Klotho Enhances Postnatal Skeletal Muscle Growth. [Internet] [Thesis]. UCLA; 2015. [cited 2021 Feb 27]. Available from: http://www.escholarship.org/uc/item/8hg0t2p8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lindsay CC. Klotho Enhances Postnatal Skeletal Muscle Growth. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/8hg0t2p8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

6. Ronca, Rich Daniel. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.

Degree: PhD, 2013, Temple University

Pharmacology

Ischemic stroke is the third leading cause of death and the leading cause of morbidity in the United States. Cognitive deficits, specifically with respect… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Ronca, R. D. (2013). The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253154

Chicago Manual of Style (16th Edition):

Ronca, Rich Daniel. “The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.” 2013. Doctoral Dissertation, Temple University. Accessed February 27, 2021. http://digital.library.temple.edu/u?/p245801coll10,253154.

MLA Handbook (7th Edition):

Ronca, Rich Daniel. “The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.” 2013. Web. 27 Feb 2021.

Vancouver:

Ronca RD. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2021 Feb 27]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253154.

Council of Science Editors:

Ronca RD. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,253154


Temple University

7. Bhavanasi, Dheeraj. Role of Protein Kinase C delta in regulating platelet functional responses.

Degree: PhD, 2014, Temple University

Physiology

Platelets upon activation change their shape, aggregate and secrete alpha and dense granule contents among which ADP acts as a feedback activator. Different Protein… (more)

Subjects/Keywords: Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bhavanasi, D. (2014). Role of Protein Kinase C delta in regulating platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,283099

Chicago Manual of Style (16th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Doctoral Dissertation, Temple University. Accessed February 27, 2021. http://digital.library.temple.edu/u?/p245801coll10,283099.

MLA Handbook (7th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Web. 27 Feb 2021.

Vancouver:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2021 Feb 27]. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099.

Council of Science Editors:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099


Temple University

8. Powers, Jeffery. microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure.

Degree: PhD, 2016, Temple University

Physiology

Historical emphasis on the left ventricle (LV) has left clinicians with a lack of efficacious right ventricle (RV)-specific therapies, and classical pharmacological interventions for… (more)

Subjects/Keywords: Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Powers, J. (2016). microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,417461

Chicago Manual of Style (16th Edition):

Powers, Jeffery. “microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure.” 2016. Doctoral Dissertation, Temple University. Accessed February 27, 2021. http://digital.library.temple.edu/u?/p245801coll10,417461.

MLA Handbook (7th Edition):

Powers, Jeffery. “microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure.” 2016. Web. 27 Feb 2021.

Vancouver:

Powers J. microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2021 Feb 27]. Available from: http://digital.library.temple.edu/u?/p245801coll10,417461.

Council of Science Editors:

Powers J. microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,417461


University of California – San Diego

9. Linsmayer, Lauren. Metabolic Physiology of Healthy and Bleached Corals.

Degree: Marine biology, 2018, University of California – San Diego

 Coral reef productivity is based upon the relationship between the coral host and its symbiotic dinoflagellate. While it is well known that the photosynthetic symbionts… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Linsmayer, L. (2018). Metabolic Physiology of Healthy and Bleached Corals. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/2g32s5gv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Linsmayer, Lauren. “Metabolic Physiology of Healthy and Bleached Corals.” 2018. Thesis, University of California – San Diego. Accessed February 27, 2021. http://www.escholarship.org/uc/item/2g32s5gv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Linsmayer, Lauren. “Metabolic Physiology of Healthy and Bleached Corals.” 2018. Web. 27 Feb 2021.

Vancouver:

Linsmayer L. Metabolic Physiology of Healthy and Bleached Corals. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Feb 27]. Available from: http://www.escholarship.org/uc/item/2g32s5gv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Linsmayer L. Metabolic Physiology of Healthy and Bleached Corals. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/2g32s5gv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

10. Sarji, Shervin. Pulmonary Hypertension in Rats is Associated with Vascular Remodeling and Neuro-inflammation in the Brain.

Degree: Physiological Science, 2018, UCLA

 Pulmonary arterial hypertension (PAH) is a chronic pulmonary vascular disease characterized by increased pulmonary vascular resistance. Recent studies implicate sympathetic overactivation in PAH and demonstrate… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Sarji, S. (2018). Pulmonary Hypertension in Rats is Associated with Vascular Remodeling and Neuro-inflammation in the Brain. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/9vq1w873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sarji, Shervin. “Pulmonary Hypertension in Rats is Associated with Vascular Remodeling and Neuro-inflammation in the Brain.” 2018. Thesis, UCLA. Accessed February 27, 2021. http://www.escholarship.org/uc/item/9vq1w873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sarji, Shervin. “Pulmonary Hypertension in Rats is Associated with Vascular Remodeling and Neuro-inflammation in the Brain.” 2018. Web. 27 Feb 2021.

Vancouver:

Sarji S. Pulmonary Hypertension in Rats is Associated with Vascular Remodeling and Neuro-inflammation in the Brain. [Internet] [Thesis]. UCLA; 2018. [cited 2021 Feb 27]. Available from: http://www.escholarship.org/uc/item/9vq1w873.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sarji S. Pulmonary Hypertension in Rats is Associated with Vascular Remodeling and Neuro-inflammation in the Brain. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/9vq1w873

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

11. Tu, Lan Ngoc Ly. MOLECULAR FUNCTION OF MAMMALIAN TRANSLOCATOR PROTEIN (TSPO).

Degree: PhD, Molecular and Integrative Physiology, 2017, Cornell University

 Translocator protein (TSPO), previously known as the peripheral benzodiazepine receptor (PBR), is a mitochondrial outer membrane protein highly conserved from bacteria to humans. TSPO is… (more)

Subjects/Keywords: physiology

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APA (6th Edition):

Tu, L. N. L. (2017). MOLECULAR FUNCTION OF MAMMALIAN TRANSLOCATOR PROTEIN (TSPO). (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/56808

Chicago Manual of Style (16th Edition):

Tu, Lan Ngoc Ly. “MOLECULAR FUNCTION OF MAMMALIAN TRANSLOCATOR PROTEIN (TSPO).” 2017. Doctoral Dissertation, Cornell University. Accessed February 27, 2021. http://hdl.handle.net/1813/56808.

MLA Handbook (7th Edition):

Tu, Lan Ngoc Ly. “MOLECULAR FUNCTION OF MAMMALIAN TRANSLOCATOR PROTEIN (TSPO).” 2017. Web. 27 Feb 2021.

Vancouver:

Tu LNL. MOLECULAR FUNCTION OF MAMMALIAN TRANSLOCATOR PROTEIN (TSPO). [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/1813/56808.

Council of Science Editors:

Tu LNL. MOLECULAR FUNCTION OF MAMMALIAN TRANSLOCATOR PROTEIN (TSPO). [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/56808


Boston University

12. Olix, Daniel. The impact of primary motor cortex, spinal cord, and sciatic nerve cooling on spinal reflex activity in the rat: a reversible deactivation study.

Degree: MS, Medical Sciences, 2015, Boston University

 The influence of spinal reflex arcs on lower limb movement cannot be understated, but the individual contribution of various parts of the reflex pathway, namely… (more)

Subjects/Keywords: Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Olix, D. (2015). The impact of primary motor cortex, spinal cord, and sciatic nerve cooling on spinal reflex activity in the rat: a reversible deactivation study. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16280

Chicago Manual of Style (16th Edition):

Olix, Daniel. “The impact of primary motor cortex, spinal cord, and sciatic nerve cooling on spinal reflex activity in the rat: a reversible deactivation study.” 2015. Masters Thesis, Boston University. Accessed February 27, 2021. http://hdl.handle.net/2144/16280.

MLA Handbook (7th Edition):

Olix, Daniel. “The impact of primary motor cortex, spinal cord, and sciatic nerve cooling on spinal reflex activity in the rat: a reversible deactivation study.” 2015. Web. 27 Feb 2021.

Vancouver:

Olix D. The impact of primary motor cortex, spinal cord, and sciatic nerve cooling on spinal reflex activity in the rat: a reversible deactivation study. [Internet] [Masters thesis]. Boston University; 2015. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2144/16280.

Council of Science Editors:

Olix D. The impact of primary motor cortex, spinal cord, and sciatic nerve cooling on spinal reflex activity in the rat: a reversible deactivation study. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16280


Boston University

13. Rhee, Younghwa. Efficacy of losartan and growth hormone combinatorial therapy in DyW mice.

Degree: MS, Sargent College of Health and Rehabilitation Sciences, 2015, Boston University

 Merosin-deficient CMD type 1A (MDC1A) is the second most common form of congenital muscular dystrophy (CMD) that presents at or near birth. MDC1A is caused… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Rhee, Y. (2015). Efficacy of losartan and growth hormone combinatorial therapy in DyW mice. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16344

Chicago Manual of Style (16th Edition):

Rhee, Younghwa. “Efficacy of losartan and growth hormone combinatorial therapy in DyW mice.” 2015. Masters Thesis, Boston University. Accessed February 27, 2021. http://hdl.handle.net/2144/16344.

MLA Handbook (7th Edition):

Rhee, Younghwa. “Efficacy of losartan and growth hormone combinatorial therapy in DyW mice.” 2015. Web. 27 Feb 2021.

Vancouver:

Rhee Y. Efficacy of losartan and growth hormone combinatorial therapy in DyW mice. [Internet] [Masters thesis]. Boston University; 2015. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2144/16344.

Council of Science Editors:

Rhee Y. Efficacy of losartan and growth hormone combinatorial therapy in DyW mice. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16344

14. Kanuri, Sri Harsha. Role of spleen tyrosine kinase in tumor necrosis factor like weak inhibitor of apoptosis induced dopaminergic neuronal cell death and microglial activation in invitro and in vivo models of Parkinson disease.

Degree: 2017, Iowa State University

 Tweak and TNF family members are novel regulators of acute and chronic inflammation. Tweak / Fn14 interaction appears to be involved in angiogenesis, inflammation, proliferation,… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Kanuri, S. H. (2017). Role of spleen tyrosine kinase in tumor necrosis factor like weak inhibitor of apoptosis induced dopaminergic neuronal cell death and microglial activation in invitro and in vivo models of Parkinson disease. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/16389

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanuri, Sri Harsha. “Role of spleen tyrosine kinase in tumor necrosis factor like weak inhibitor of apoptosis induced dopaminergic neuronal cell death and microglial activation in invitro and in vivo models of Parkinson disease.” 2017. Thesis, Iowa State University. Accessed February 27, 2021. https://lib.dr.iastate.edu/etd/16389.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanuri, Sri Harsha. “Role of spleen tyrosine kinase in tumor necrosis factor like weak inhibitor of apoptosis induced dopaminergic neuronal cell death and microglial activation in invitro and in vivo models of Parkinson disease.” 2017. Web. 27 Feb 2021.

Vancouver:

Kanuri SH. Role of spleen tyrosine kinase in tumor necrosis factor like weak inhibitor of apoptosis induced dopaminergic neuronal cell death and microglial activation in invitro and in vivo models of Parkinson disease. [Internet] [Thesis]. Iowa State University; 2017. [cited 2021 Feb 27]. Available from: https://lib.dr.iastate.edu/etd/16389.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanuri SH. Role of spleen tyrosine kinase in tumor necrosis factor like weak inhibitor of apoptosis induced dopaminergic neuronal cell death and microglial activation in invitro and in vivo models of Parkinson disease. [Thesis]. Iowa State University; 2017. Available from: https://lib.dr.iastate.edu/etd/16389

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


West Virginia University

15. Porter, Katrina L. The Role of Neurokinin B in the Regulation of the Preovulatory LH Surge in Ewes.

Degree: PhD, Physiology, Pharmacology & Neuroscience, 2013, West Virginia University

 Hypothalamic input is vital to the regulation of reproductive function. Gonadotropin-releasing hormone (GnRH) is secreted by hypothalamic GnRH neurons into the hypophysial portal circulation. When… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Porter, K. L. (2013). The Role of Neurokinin B in the Regulation of the Preovulatory LH Surge in Ewes. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.622 ; https://researchrepository.wvu.edu/etd/622

Chicago Manual of Style (16th Edition):

Porter, Katrina L. “The Role of Neurokinin B in the Regulation of the Preovulatory LH Surge in Ewes.” 2013. Doctoral Dissertation, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.622 ; https://researchrepository.wvu.edu/etd/622.

MLA Handbook (7th Edition):

Porter, Katrina L. “The Role of Neurokinin B in the Regulation of the Preovulatory LH Surge in Ewes.” 2013. Web. 27 Feb 2021.

Vancouver:

Porter KL. The Role of Neurokinin B in the Regulation of the Preovulatory LH Surge in Ewes. [Internet] [Doctoral dissertation]. West Virginia University; 2013. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.622 ; https://researchrepository.wvu.edu/etd/622.

Council of Science Editors:

Porter KL. The Role of Neurokinin B in the Regulation of the Preovulatory LH Surge in Ewes. [Doctoral Dissertation]. West Virginia University; 2013. Available from: https://doi.org/10.33915/etd.622 ; https://researchrepository.wvu.edu/etd/622


West Virginia University

16. Baseler, Walter. Examination of Novel Cardiac Mechanisms Influencing Mitochondrial Proteomes during Diabetes Mellitus.

Degree: PhD, Exercise Physiology, 2012, West Virginia University

 Cardiac complications, including diabetic cardiomyopathy, are the leading cause of morbidity and mortality within diabetes mellitus. Mitochondrial dysfunction has been suggested as an underlying factor… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Baseler, W. (2012). Examination of Novel Cardiac Mechanisms Influencing Mitochondrial Proteomes during Diabetes Mellitus. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.4830 ; https://researchrepository.wvu.edu/etd/4830

Chicago Manual of Style (16th Edition):

Baseler, Walter. “Examination of Novel Cardiac Mechanisms Influencing Mitochondrial Proteomes during Diabetes Mellitus.” 2012. Doctoral Dissertation, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.4830 ; https://researchrepository.wvu.edu/etd/4830.

MLA Handbook (7th Edition):

Baseler, Walter. “Examination of Novel Cardiac Mechanisms Influencing Mitochondrial Proteomes during Diabetes Mellitus.” 2012. Web. 27 Feb 2021.

Vancouver:

Baseler W. Examination of Novel Cardiac Mechanisms Influencing Mitochondrial Proteomes during Diabetes Mellitus. [Internet] [Doctoral dissertation]. West Virginia University; 2012. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.4830 ; https://researchrepository.wvu.edu/etd/4830.

Council of Science Editors:

Baseler W. Examination of Novel Cardiac Mechanisms Influencing Mitochondrial Proteomes during Diabetes Mellitus. [Doctoral Dissertation]. West Virginia University; 2012. Available from: https://doi.org/10.33915/etd.4830 ; https://researchrepository.wvu.edu/etd/4830


West Virginia University

17. Goodwill, Adam G. Vascular Consequences of Metabolic Syndrome Related Shifts in Cyclooxygenase Mediated Arachidonic Acid Metabolism.

Degree: PhD, Physiology, Pharmacology & Neuroscience, 2011, West Virginia University

 The metabolic syndrome is a multi-faceted disease state in which the initial pathology of obesity leads to the development of a cadre of comorbidities including,… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Goodwill, A. G. (2011). Vascular Consequences of Metabolic Syndrome Related Shifts in Cyclooxygenase Mediated Arachidonic Acid Metabolism. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.3424 ; https://researchrepository.wvu.edu/etd/3424

Chicago Manual of Style (16th Edition):

Goodwill, Adam G. “Vascular Consequences of Metabolic Syndrome Related Shifts in Cyclooxygenase Mediated Arachidonic Acid Metabolism.” 2011. Doctoral Dissertation, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.3424 ; https://researchrepository.wvu.edu/etd/3424.

MLA Handbook (7th Edition):

Goodwill, Adam G. “Vascular Consequences of Metabolic Syndrome Related Shifts in Cyclooxygenase Mediated Arachidonic Acid Metabolism.” 2011. Web. 27 Feb 2021.

Vancouver:

Goodwill AG. Vascular Consequences of Metabolic Syndrome Related Shifts in Cyclooxygenase Mediated Arachidonic Acid Metabolism. [Internet] [Doctoral dissertation]. West Virginia University; 2011. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.3424 ; https://researchrepository.wvu.edu/etd/3424.

Council of Science Editors:

Goodwill AG. Vascular Consequences of Metabolic Syndrome Related Shifts in Cyclooxygenase Mediated Arachidonic Acid Metabolism. [Doctoral Dissertation]. West Virginia University; 2011. Available from: https://doi.org/10.33915/etd.3424 ; https://researchrepository.wvu.edu/etd/3424


West Virginia University

18. Butcher, Joshua Thomas. Altered Hemodynamic Control in the Skeletal Muscle Microcirculation in the Metabolic Syndrome: The Emergence of a new Attractor.

Degree: PhD, Microbiology, Immunology, and Cell Biology, 2013, West Virginia University

 Peripheral vascular disease is a pathological disease state whereby the peripheral vascular system becomes progressively limited in its ability to adequately perfuse extremities despite increases… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Butcher, J. T. (2013). Altered Hemodynamic Control in the Skeletal Muscle Microcirculation in the Metabolic Syndrome: The Emergence of a new Attractor. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.354 ; https://researchrepository.wvu.edu/etd/354

Chicago Manual of Style (16th Edition):

Butcher, Joshua Thomas. “Altered Hemodynamic Control in the Skeletal Muscle Microcirculation in the Metabolic Syndrome: The Emergence of a new Attractor.” 2013. Doctoral Dissertation, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.354 ; https://researchrepository.wvu.edu/etd/354.

MLA Handbook (7th Edition):

Butcher, Joshua Thomas. “Altered Hemodynamic Control in the Skeletal Muscle Microcirculation in the Metabolic Syndrome: The Emergence of a new Attractor.” 2013. Web. 27 Feb 2021.

Vancouver:

Butcher JT. Altered Hemodynamic Control in the Skeletal Muscle Microcirculation in the Metabolic Syndrome: The Emergence of a new Attractor. [Internet] [Doctoral dissertation]. West Virginia University; 2013. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.354 ; https://researchrepository.wvu.edu/etd/354.

Council of Science Editors:

Butcher JT. Altered Hemodynamic Control in the Skeletal Muscle Microcirculation in the Metabolic Syndrome: The Emergence of a new Attractor. [Doctoral Dissertation]. West Virginia University; 2013. Available from: https://doi.org/10.33915/etd.354 ; https://researchrepository.wvu.edu/etd/354


West Virginia University

19. Vejandla, Himani. C-peptide Improves Hyperglycemia-induced Endothelial Redox Balance by Preventing Mitochondrial Oxidative Stress and Enhancing NADPH Synthesis.

Degree: PhD, Physiology, Pharmacology & Neuroscience, 2012, West Virginia University

 C-peptide, a by-product of insulin biosynthesis, has been shown to ameliorate diabetes-induced renal impairment. Yet, the mechanisms underlying this protective benefit remain unclear. Our studies… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Vejandla, H. (2012). C-peptide Improves Hyperglycemia-induced Endothelial Redox Balance by Preventing Mitochondrial Oxidative Stress and Enhancing NADPH Synthesis. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.3565 ; https://researchrepository.wvu.edu/etd/3565

Chicago Manual of Style (16th Edition):

Vejandla, Himani. “C-peptide Improves Hyperglycemia-induced Endothelial Redox Balance by Preventing Mitochondrial Oxidative Stress and Enhancing NADPH Synthesis.” 2012. Doctoral Dissertation, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.3565 ; https://researchrepository.wvu.edu/etd/3565.

MLA Handbook (7th Edition):

Vejandla, Himani. “C-peptide Improves Hyperglycemia-induced Endothelial Redox Balance by Preventing Mitochondrial Oxidative Stress and Enhancing NADPH Synthesis.” 2012. Web. 27 Feb 2021.

Vancouver:

Vejandla H. C-peptide Improves Hyperglycemia-induced Endothelial Redox Balance by Preventing Mitochondrial Oxidative Stress and Enhancing NADPH Synthesis. [Internet] [Doctoral dissertation]. West Virginia University; 2012. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.3565 ; https://researchrepository.wvu.edu/etd/3565.

Council of Science Editors:

Vejandla H. C-peptide Improves Hyperglycemia-induced Endothelial Redox Balance by Preventing Mitochondrial Oxidative Stress and Enhancing NADPH Synthesis. [Doctoral Dissertation]. West Virginia University; 2012. Available from: https://doi.org/10.33915/etd.3565 ; https://researchrepository.wvu.edu/etd/3565


West Virginia University

20. Reger, Brian Lee. The Effects of Aerobic Exercise Training on Arterial Stiffness in the Metabolic Syndrome.

Degree: MS, Exercise Physiology, 2012, West Virginia University

 Background: Arterial dysfunction, due in part to arterial stiffening, is recognized as a surrogate end point for cardiovascular disease (CVD) and predicts the risk of… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Reger, B. L. (2012). The Effects of Aerobic Exercise Training on Arterial Stiffness in the Metabolic Syndrome. (Thesis). West Virginia University. Retrieved from https://doi.org/10.33915/etd.317 ; https://researchrepository.wvu.edu/etd/317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reger, Brian Lee. “The Effects of Aerobic Exercise Training on Arterial Stiffness in the Metabolic Syndrome.” 2012. Thesis, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.317 ; https://researchrepository.wvu.edu/etd/317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reger, Brian Lee. “The Effects of Aerobic Exercise Training on Arterial Stiffness in the Metabolic Syndrome.” 2012. Web. 27 Feb 2021.

Vancouver:

Reger BL. The Effects of Aerobic Exercise Training on Arterial Stiffness in the Metabolic Syndrome. [Internet] [Thesis]. West Virginia University; 2012. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.317 ; https://researchrepository.wvu.edu/etd/317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reger BL. The Effects of Aerobic Exercise Training on Arterial Stiffness in the Metabolic Syndrome. [Thesis]. West Virginia University; 2012. Available from: https://doi.org/10.33915/etd.317 ; https://researchrepository.wvu.edu/etd/317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


West Virginia University

21. Akers, Keli Ann. Changes in Body Condition from Prepartum to Postpartum on Health and Reproduction in Dairy Cattle.

Degree: MS, Applied and Environmental Biology, 2014, West Virginia University

 The aim of this study were to body condition score, 72 Holstein-Friesian dairy cows at prepartum, compare lean, moderate, and over-conditioned categories, and relate any… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Akers, K. A. (2014). Changes in Body Condition from Prepartum to Postpartum on Health and Reproduction in Dairy Cattle. (Thesis). West Virginia University. Retrieved from https://doi.org/10.33915/etd.414 ; https://researchrepository.wvu.edu/etd/414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Akers, Keli Ann. “Changes in Body Condition from Prepartum to Postpartum on Health and Reproduction in Dairy Cattle.” 2014. Thesis, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.414 ; https://researchrepository.wvu.edu/etd/414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Akers, Keli Ann. “Changes in Body Condition from Prepartum to Postpartum on Health and Reproduction in Dairy Cattle.” 2014. Web. 27 Feb 2021.

Vancouver:

Akers KA. Changes in Body Condition from Prepartum to Postpartum on Health and Reproduction in Dairy Cattle. [Internet] [Thesis]. West Virginia University; 2014. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.414 ; https://researchrepository.wvu.edu/etd/414.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Akers KA. Changes in Body Condition from Prepartum to Postpartum on Health and Reproduction in Dairy Cattle. [Thesis]. West Virginia University; 2014. Available from: https://doi.org/10.33915/etd.414 ; https://researchrepository.wvu.edu/etd/414

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


West Virginia University

22. Zellner, Leor C. Effects of Age and Early Life Ozone Exposure on the Developmental Dynamics of Afferent Airway Neurons in Postnatal Rats.

Degree: PhD, Physiology, Pharmacology & Neuroscience, 2011, West Virginia University

 The environmental irritant ozone (O3) plays a role in airway inflammation and is known to cause airway hyperresponisiveness especially in susceptible populations such as asthmatics… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Zellner, L. C. (2011). Effects of Age and Early Life Ozone Exposure on the Developmental Dynamics of Afferent Airway Neurons in Postnatal Rats. (Doctoral Dissertation). West Virginia University. Retrieved from https://doi.org/10.33915/etd.3413 ; https://researchrepository.wvu.edu/etd/3413

Chicago Manual of Style (16th Edition):

Zellner, Leor C. “Effects of Age and Early Life Ozone Exposure on the Developmental Dynamics of Afferent Airway Neurons in Postnatal Rats.” 2011. Doctoral Dissertation, West Virginia University. Accessed February 27, 2021. https://doi.org/10.33915/etd.3413 ; https://researchrepository.wvu.edu/etd/3413.

MLA Handbook (7th Edition):

Zellner, Leor C. “Effects of Age and Early Life Ozone Exposure on the Developmental Dynamics of Afferent Airway Neurons in Postnatal Rats.” 2011. Web. 27 Feb 2021.

Vancouver:

Zellner LC. Effects of Age and Early Life Ozone Exposure on the Developmental Dynamics of Afferent Airway Neurons in Postnatal Rats. [Internet] [Doctoral dissertation]. West Virginia University; 2011. [cited 2021 Feb 27]. Available from: https://doi.org/10.33915/etd.3413 ; https://researchrepository.wvu.edu/etd/3413.

Council of Science Editors:

Zellner LC. Effects of Age and Early Life Ozone Exposure on the Developmental Dynamics of Afferent Airway Neurons in Postnatal Rats. [Doctoral Dissertation]. West Virginia University; 2011. Available from: https://doi.org/10.33915/etd.3413 ; https://researchrepository.wvu.edu/etd/3413


Wayne State University

23. Minic, Zeljka. Mechanisms Mediating Modulation Of The Cardiopulmonary Chemoreflex Control Of Regional Sympathetic Outputs By Adenosine A1 And A2a Receptors In The Nts.

Degree: PhD, Physiology, 2013, Wayne State University

  ABSTRACT by ZELJKA MINIC August 2013 Advisor: Dr. Tadeusz J. Scislo Major: Physiology Degree: Doctor of Philosophy Adenosine is an important neuromodulator of cardiovascular… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Minic, Z. (2013). Mechanisms Mediating Modulation Of The Cardiopulmonary Chemoreflex Control Of Regional Sympathetic Outputs By Adenosine A1 And A2a Receptors In The Nts. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/785

Chicago Manual of Style (16th Edition):

Minic, Zeljka. “Mechanisms Mediating Modulation Of The Cardiopulmonary Chemoreflex Control Of Regional Sympathetic Outputs By Adenosine A1 And A2a Receptors In The Nts.” 2013. Doctoral Dissertation, Wayne State University. Accessed February 27, 2021. https://digitalcommons.wayne.edu/oa_dissertations/785.

MLA Handbook (7th Edition):

Minic, Zeljka. “Mechanisms Mediating Modulation Of The Cardiopulmonary Chemoreflex Control Of Regional Sympathetic Outputs By Adenosine A1 And A2a Receptors In The Nts.” 2013. Web. 27 Feb 2021.

Vancouver:

Minic Z. Mechanisms Mediating Modulation Of The Cardiopulmonary Chemoreflex Control Of Regional Sympathetic Outputs By Adenosine A1 And A2a Receptors In The Nts. [Internet] [Doctoral dissertation]. Wayne State University; 2013. [cited 2021 Feb 27]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/785.

Council of Science Editors:

Minic Z. Mechanisms Mediating Modulation Of The Cardiopulmonary Chemoreflex Control Of Regional Sympathetic Outputs By Adenosine A1 And A2a Receptors In The Nts. [Doctoral Dissertation]. Wayne State University; 2013. Available from: https://digitalcommons.wayne.edu/oa_dissertations/785


Wayne State University

24. Reynolds, Christian Andrew. Preclinical Evaluation Of Infrared Light Therapy In A Rat Model Of Neonatal Hypoxic-Ischemic Encephalopathy.

Degree: PhD, Physiology, 2015, Wayne State University

  Hypoxic events encountered during the perinatal period leading to pathologic decreases in fetal cerebral oxygen availability are detrimental resulting in neonatal hypoxic-ischemic encephalopathy (HIE).… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Reynolds, C. A. (2015). Preclinical Evaluation Of Infrared Light Therapy In A Rat Model Of Neonatal Hypoxic-Ischemic Encephalopathy. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1294

Chicago Manual of Style (16th Edition):

Reynolds, Christian Andrew. “Preclinical Evaluation Of Infrared Light Therapy In A Rat Model Of Neonatal Hypoxic-Ischemic Encephalopathy.” 2015. Doctoral Dissertation, Wayne State University. Accessed February 27, 2021. https://digitalcommons.wayne.edu/oa_dissertations/1294.

MLA Handbook (7th Edition):

Reynolds, Christian Andrew. “Preclinical Evaluation Of Infrared Light Therapy In A Rat Model Of Neonatal Hypoxic-Ischemic Encephalopathy.” 2015. Web. 27 Feb 2021.

Vancouver:

Reynolds CA. Preclinical Evaluation Of Infrared Light Therapy In A Rat Model Of Neonatal Hypoxic-Ischemic Encephalopathy. [Internet] [Doctoral dissertation]. Wayne State University; 2015. [cited 2021 Feb 27]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1294.

Council of Science Editors:

Reynolds CA. Preclinical Evaluation Of Infrared Light Therapy In A Rat Model Of Neonatal Hypoxic-Ischemic Encephalopathy. [Doctoral Dissertation]. Wayne State University; 2015. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1294


Washington State University

25. [No author]. A Proposed Mechanism for Transcriptional Regulation of Cardiac Hypertrophy .

Degree: 2014, Washington State University

 Conventional medical therapies for treatment of hypertension center around symptom management, and this is due, in part, to limited understanding of the mechanisms responsible for… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

author], [. (2014). A Proposed Mechanism for Transcriptional Regulation of Cardiac Hypertrophy . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/5430

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “A Proposed Mechanism for Transcriptional Regulation of Cardiac Hypertrophy .” 2014. Thesis, Washington State University. Accessed February 27, 2021. http://hdl.handle.net/2376/5430.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “A Proposed Mechanism for Transcriptional Regulation of Cardiac Hypertrophy .” 2014. Web. 27 Feb 2021.

Vancouver:

author] [. A Proposed Mechanism for Transcriptional Regulation of Cardiac Hypertrophy . [Internet] [Thesis]. Washington State University; 2014. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2376/5430.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. A Proposed Mechanism for Transcriptional Regulation of Cardiac Hypertrophy . [Thesis]. Washington State University; 2014. Available from: http://hdl.handle.net/2376/5430

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington State University

26. [No author]. Diabetic cardiomyopathy: phenotypes, mechanisms, and therapeutic targets .

Degree: 2015, Washington State University

 Diabetic cardiomyopathy (DCM) is a syndrome of cardiac dysfunction, fibrosis, and hypertrophy that is often associated with type 2 diabetes mellitus (T2DM) independent of classical… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

author], [. (2015). Diabetic cardiomyopathy: phenotypes, mechanisms, and therapeutic targets . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/6238

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Diabetic cardiomyopathy: phenotypes, mechanisms, and therapeutic targets .” 2015. Thesis, Washington State University. Accessed February 27, 2021. http://hdl.handle.net/2376/6238.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Diabetic cardiomyopathy: phenotypes, mechanisms, and therapeutic targets .” 2015. Web. 27 Feb 2021.

Vancouver:

author] [. Diabetic cardiomyopathy: phenotypes, mechanisms, and therapeutic targets . [Internet] [Thesis]. Washington State University; 2015. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2376/6238.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Diabetic cardiomyopathy: phenotypes, mechanisms, and therapeutic targets . [Thesis]. Washington State University; 2015. Available from: http://hdl.handle.net/2376/6238

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

27. Smith, Michelle Rei. Mrna Granules And Ischemic Preconditioning.

Degree: MS, Physiology, 2012, Wayne State University

  Brain ischemia and reperfusion that occurs after stroke and cardiac arrest, causes translation arrest (TA) in neurons which is irreversible in neurons that will… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Smith, M. R. (2012). Mrna Granules And Ischemic Preconditioning. (Masters Thesis). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_theses/216

Chicago Manual of Style (16th Edition):

Smith, Michelle Rei. “Mrna Granules And Ischemic Preconditioning.” 2012. Masters Thesis, Wayne State University. Accessed February 27, 2021. https://digitalcommons.wayne.edu/oa_theses/216.

MLA Handbook (7th Edition):

Smith, Michelle Rei. “Mrna Granules And Ischemic Preconditioning.” 2012. Web. 27 Feb 2021.

Vancouver:

Smith MR. Mrna Granules And Ischemic Preconditioning. [Internet] [Masters thesis]. Wayne State University; 2012. [cited 2021 Feb 27]. Available from: https://digitalcommons.wayne.edu/oa_theses/216.

Council of Science Editors:

Smith MR. Mrna Granules And Ischemic Preconditioning. [Masters Thesis]. Wayne State University; 2012. Available from: https://digitalcommons.wayne.edu/oa_theses/216


University of KwaZulu-Natal

28. Sibiya, Siyabonga G. Evaluating a new drug to combat alzheimer's disease.

Degree: 2014, University of KwaZulu-Natal

 Alzheimer’s disease (AD), a progressive neurodegenerative disorder that affects mostly the limbic system and the neocortical areas of the brain, is the most prevalent form… (more)

Subjects/Keywords: Physiology.

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APA (6th Edition):

Sibiya, S. G. (2014). Evaluating a new drug to combat alzheimer's disease. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/12339

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sibiya, Siyabonga G. “Evaluating a new drug to combat alzheimer's disease.” 2014. Thesis, University of KwaZulu-Natal. Accessed February 27, 2021. http://hdl.handle.net/10413/12339.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sibiya, Siyabonga G. “Evaluating a new drug to combat alzheimer's disease.” 2014. Web. 27 Feb 2021.

Vancouver:

Sibiya SG. Evaluating a new drug to combat alzheimer's disease. [Internet] [Thesis]. University of KwaZulu-Natal; 2014. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/10413/12339.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sibiya SG. Evaluating a new drug to combat alzheimer's disease. [Thesis]. University of KwaZulu-Natal; 2014. Available from: http://hdl.handle.net/10413/12339

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

29. Kim, Hanna. Effect of Roux-en-Y gastric bypass surgery on controlling type 2 diabetes mellitus.

Degree: MS, Medical Sciences, 2020, Boston University

 Diabetes Mellitus is a form of metabolic disease with loss or dysfunction in glycemic control. Type II Diabetes Mellitus (T2DM) is characterized by the loss… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Kim, H. (2020). Effect of Roux-en-Y gastric bypass surgery on controlling type 2 diabetes mellitus. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/41282

Chicago Manual of Style (16th Edition):

Kim, Hanna. “Effect of Roux-en-Y gastric bypass surgery on controlling type 2 diabetes mellitus.” 2020. Masters Thesis, Boston University. Accessed February 27, 2021. http://hdl.handle.net/2144/41282.

MLA Handbook (7th Edition):

Kim, Hanna. “Effect of Roux-en-Y gastric bypass surgery on controlling type 2 diabetes mellitus.” 2020. Web. 27 Feb 2021.

Vancouver:

Kim H. Effect of Roux-en-Y gastric bypass surgery on controlling type 2 diabetes mellitus. [Internet] [Masters thesis]. Boston University; 2020. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/2144/41282.

Council of Science Editors:

Kim H. Effect of Roux-en-Y gastric bypass surgery on controlling type 2 diabetes mellitus. [Masters Thesis]. Boston University; 2020. Available from: http://hdl.handle.net/2144/41282


University of Cape Town

30. Henning, Franclo. Mechanisms underlying the development of weakness in idiopathic inflammatory myopathies: an in vitro single muscle fibre contractility study.

Degree: PhD, Division of Exercise Science and Sports Medicine, 2018, University of Cape Town

 Introduction: Polymyositis (PM), dermatomyositis (DM) and necrotising autoimmune myopathy (NAM) form part of the spectrum of idiopathic inflammatory myopathies (IIMs). Although the pathogenic mechanisms are… (more)

Subjects/Keywords: Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Henning, F. (2018). Mechanisms underlying the development of weakness in idiopathic inflammatory myopathies: an in vitro single muscle fibre contractility study. (Doctoral Dissertation). University of Cape Town. Retrieved from http://hdl.handle.net/11427/29317

Chicago Manual of Style (16th Edition):

Henning, Franclo. “Mechanisms underlying the development of weakness in idiopathic inflammatory myopathies: an in vitro single muscle fibre contractility study.” 2018. Doctoral Dissertation, University of Cape Town. Accessed February 27, 2021. http://hdl.handle.net/11427/29317.

MLA Handbook (7th Edition):

Henning, Franclo. “Mechanisms underlying the development of weakness in idiopathic inflammatory myopathies: an in vitro single muscle fibre contractility study.” 2018. Web. 27 Feb 2021.

Vancouver:

Henning F. Mechanisms underlying the development of weakness in idiopathic inflammatory myopathies: an in vitro single muscle fibre contractility study. [Internet] [Doctoral dissertation]. University of Cape Town; 2018. [cited 2021 Feb 27]. Available from: http://hdl.handle.net/11427/29317.

Council of Science Editors:

Henning F. Mechanisms underlying the development of weakness in idiopathic inflammatory myopathies: an in vitro single muscle fibre contractility study. [Doctoral Dissertation]. University of Cape Town; 2018. Available from: http://hdl.handle.net/11427/29317

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