Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

University: University of Florida

You searched for subject:(phosphorylation). Showing records 1 – 30 of 75 total matches.

[1] [2] [3]

Search Limiters

Last 2 Years | English Only

▼ Search Limiters


University of Florida

1. Gokirmak, Tufan. Phosphorylation Mediated Regulation of 14-3-3 Protein Dimerization in Arabidopsis Thaliana and the Effect of Dimerization in 14-3-3/target Interactions.

Degree: PhD, Plant Molecular and Cellular Biology, 2010, University of Florida

 14-3-3s are a family of regulatory proteins that are uniquely eukaryotic, evolutionarily conserved across all eukaryotes, and deeply involved in protein-protein interactions that mediate diverse… (more)

Subjects/Keywords: Amino acids; Cells; Dimerization; Dimers; Fluorescence; Phosphorylation; Protein isoforms; Proteins; Protoplasts; Yeasts; arabidopsis, difopein, dimerization, gbf3, interaction, localization, monomerization, phosphopeptide, phosphorylation, r18, regulation, target, toxicity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gokirmak, T. (2010). Phosphorylation Mediated Regulation of 14-3-3 Protein Dimerization in Arabidopsis Thaliana and the Effect of Dimerization in 14-3-3/target Interactions. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0042062

Chicago Manual of Style (16th Edition):

Gokirmak, Tufan. “Phosphorylation Mediated Regulation of 14-3-3 Protein Dimerization in Arabidopsis Thaliana and the Effect of Dimerization in 14-3-3/target Interactions.” 2010. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0042062.

MLA Handbook (7th Edition):

Gokirmak, Tufan. “Phosphorylation Mediated Regulation of 14-3-3 Protein Dimerization in Arabidopsis Thaliana and the Effect of Dimerization in 14-3-3/target Interactions.” 2010. Web. 22 Oct 2019.

Vancouver:

Gokirmak T. Phosphorylation Mediated Regulation of 14-3-3 Protein Dimerization in Arabidopsis Thaliana and the Effect of Dimerization in 14-3-3/target Interactions. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0042062.

Council of Science Editors:

Gokirmak T. Phosphorylation Mediated Regulation of 14-3-3 Protein Dimerization in Arabidopsis Thaliana and the Effect of Dimerization in 14-3-3/target Interactions. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0042062


University of Florida

2. Anderson, Ethan M. N-Methyl-D-Aspartate Receptor 1 Splicing and Phosphorylation Changes Are Associated with Behavioral Alterations during Morphine Tolerance and Withdrawal.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2012, University of Florida

 Morphine is an opioid drug which is used clinically for thetreatment of pain and recreationally for its rewarding effects. Repeated use ofmorphine can lead to… (more)

Subjects/Keywords: Dosage; Morphine; Motivation; N methyl D aspartate receptors; Neurons; Opioid analgesics; Pain; Phosphorylation; Rats; Splicing; calcineurin  – creb  – dependence  – morphine  – neuron  – neuroscience  – nmda  – nr1  – phosphorylation  – pka  – receptor  – splicing  – tolerance  – withdrawal

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anderson, E. M. (2012). N-Methyl-D-Aspartate Receptor 1 Splicing and Phosphorylation Changes Are Associated with Behavioral Alterations during Morphine Tolerance and Withdrawal. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044849

Chicago Manual of Style (16th Edition):

Anderson, Ethan M. “N-Methyl-D-Aspartate Receptor 1 Splicing and Phosphorylation Changes Are Associated with Behavioral Alterations during Morphine Tolerance and Withdrawal.” 2012. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0044849.

MLA Handbook (7th Edition):

Anderson, Ethan M. “N-Methyl-D-Aspartate Receptor 1 Splicing and Phosphorylation Changes Are Associated with Behavioral Alterations during Morphine Tolerance and Withdrawal.” 2012. Web. 22 Oct 2019.

Vancouver:

Anderson EM. N-Methyl-D-Aspartate Receptor 1 Splicing and Phosphorylation Changes Are Associated with Behavioral Alterations during Morphine Tolerance and Withdrawal. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0044849.

Council of Science Editors:

Anderson EM. N-Methyl-D-Aspartate Receptor 1 Splicing and Phosphorylation Changes Are Associated with Behavioral Alterations during Morphine Tolerance and Withdrawal. [Doctoral Dissertation]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/UFE0044849


University of Florida

3. MONTERO,CINDY. The Effect of Epigallocatechin-Gallate on Adiponectin Expression and the Insulin Signaling Pathway.

Degree: MS, Food Science and Human Nutrition, 2011, University of Florida

 Adiponectin, a hormone secreted from adipocytes, in low circulating levels has been epidemiologically associated with obesity, insulin resistance, type 2 diabetes, and cardiovascular disease; it… (more)

Subjects/Keywords: Adipocytes; Gallates; Insulin; Insulin resistance; Obesity; Phosphorylation; Plasmas; Receptors; Secretion; Type 2 diabetes mellitus

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

MONTERO,CINDY. (2011). The Effect of Epigallocatechin-Gallate on Adiponectin Expression and the Insulin Signaling Pathway. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0042647

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

MONTERO,CINDY. “The Effect of Epigallocatechin-Gallate on Adiponectin Expression and the Insulin Signaling Pathway.” 2011. Masters Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0042647.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

MONTERO,CINDY. “The Effect of Epigallocatechin-Gallate on Adiponectin Expression and the Insulin Signaling Pathway.” 2011. Web. 22 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

MONTERO,CINDY. The Effect of Epigallocatechin-Gallate on Adiponectin Expression and the Insulin Signaling Pathway. [Internet] [Masters thesis]. University of Florida; 2011. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0042647.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

MONTERO,CINDY. The Effect of Epigallocatechin-Gallate on Adiponectin Expression and the Insulin Signaling Pathway. [Masters Thesis]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0042647

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Florida

4. Mwaniki, Anne. Est Discovery in Threatend Tsuga Caroliniana.

Degree: MS, Plant Molecular and Cellular Biology, 2009, University of Florida

 Hemlock Woolly adelgid (HWA) has largely infested Tsuga species across North America. HWA was introduced into the United States from Japan and other parts of… (more)

Subjects/Keywords: Amino acids; Dehydration; Drosophila; Enzymes; Heat stress disorders; Metabolism; Molecules; Nucleic acids; Phosphorylation; RNA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mwaniki, A. (2009). Est Discovery in Threatend Tsuga Caroliniana. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0025099

Chicago Manual of Style (16th Edition):

Mwaniki, Anne. “Est Discovery in Threatend Tsuga Caroliniana.” 2009. Masters Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0025099.

MLA Handbook (7th Edition):

Mwaniki, Anne. “Est Discovery in Threatend Tsuga Caroliniana.” 2009. Web. 22 Oct 2019.

Vancouver:

Mwaniki A. Est Discovery in Threatend Tsuga Caroliniana. [Internet] [Masters thesis]. University of Florida; 2009. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0025099.

Council of Science Editors:

Mwaniki A. Est Discovery in Threatend Tsuga Caroliniana. [Masters Thesis]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0025099


University of Florida

5. Carruthers, Aubrey. The Importance of Interferon-Inducible dsRNA-Activated Protein Kinase, PKR, in Breast Cancer Cell Response to Doxorubicin.

Degree: 2012, University of Florida

 It has been previously reported that primary breast cancer tissue displays increased levels of interferon-inducible, double-stranded-RNA-dependent protein kinase, PKR. We wanted to further investigate these… (more)

Subjects/Keywords: Apoptosis; Breast cancer; Cell lines; Cells; Chemotherapy; Dyes; Phosphorylation; Reagents; Small interfering RNA; Tissue samples; Breast – Cancer; Doxorubicin; Immunohistochemistry; Protein kinases

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carruthers, A. (2012). The Importance of Interferon-Inducible dsRNA-Activated Protein Kinase, PKR, in Breast Cancer Cell Response to Doxorubicin. (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00057744

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Carruthers, Aubrey. “The Importance of Interferon-Inducible dsRNA-Activated Protein Kinase, PKR, in Breast Cancer Cell Response to Doxorubicin.” 2012. Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00057744.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Carruthers, Aubrey. “The Importance of Interferon-Inducible dsRNA-Activated Protein Kinase, PKR, in Breast Cancer Cell Response to Doxorubicin.” 2012. Web. 22 Oct 2019.

Vancouver:

Carruthers A. The Importance of Interferon-Inducible dsRNA-Activated Protein Kinase, PKR, in Breast Cancer Cell Response to Doxorubicin. [Internet] [Thesis]. University of Florida; 2012. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00057744.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Carruthers A. The Importance of Interferon-Inducible dsRNA-Activated Protein Kinase, PKR, in Breast Cancer Cell Response to Doxorubicin. [Thesis]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/AA00057744

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

6. Werden, Steven. The Role of Cell Signaling in Poxvirus Tropism The Case of the M-T5 Host-Range Protein of Myxoma Virus.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2009, University of Florida

 THE ROLE OF CELL SIGNALING IN POXVIRUS TROPISM: THE CASE OF THE M-T5 HOST-RANGE PROTEIN OF MYXOMA VIRUS A productive poxvirus infection is heavily dependent… (more)

Subjects/Keywords: Antibodies; Apoptosis; Infections; Myxoma virus; Phosphorylation; Plasmids; Poxviridae; Proteins; Rabbits; Tropisms; akt, ankyrin, fbox, immunoregulator, oncolytic, pike, pranc, scf, vaccinia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Werden, S. (2009). The Role of Cell Signaling in Poxvirus Tropism The Case of the M-T5 Host-Range Protein of Myxoma Virus. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041174

Chicago Manual of Style (16th Edition):

Werden, Steven. “The Role of Cell Signaling in Poxvirus Tropism The Case of the M-T5 Host-Range Protein of Myxoma Virus.” 2009. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0041174.

MLA Handbook (7th Edition):

Werden, Steven. “The Role of Cell Signaling in Poxvirus Tropism The Case of the M-T5 Host-Range Protein of Myxoma Virus.” 2009. Web. 22 Oct 2019.

Vancouver:

Werden S. The Role of Cell Signaling in Poxvirus Tropism The Case of the M-T5 Host-Range Protein of Myxoma Virus. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0041174.

Council of Science Editors:

Werden S. The Role of Cell Signaling in Poxvirus Tropism The Case of the M-T5 Host-Range Protein of Myxoma Virus. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0041174


University of Florida

7. Jackson, Travis. PHLPP1 Regulates Hippocampal Akt Survival Signaling.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2010, University of Florida

 When faced with new situations or challenges, past knowledge and experience can help guide behavior. However, to do so, we depend upon biochemical mechanisms in… (more)

Subjects/Keywords: Brain; Hippocampus; Memory; Neurons; Phosphatases; Phosphorylation; Proteins; Rats; Receptors; Regional disparities; aging, akt, ca1, erk, foxo3a, hippocampus, neuronal, phlpp1, pkc

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jackson, T. (2010). PHLPP1 Regulates Hippocampal Akt Survival Signaling. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041837

Chicago Manual of Style (16th Edition):

Jackson, Travis. “PHLPP1 Regulates Hippocampal Akt Survival Signaling.” 2010. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0041837.

MLA Handbook (7th Edition):

Jackson, Travis. “PHLPP1 Regulates Hippocampal Akt Survival Signaling.” 2010. Web. 22 Oct 2019.

Vancouver:

Jackson T. PHLPP1 Regulates Hippocampal Akt Survival Signaling. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0041837.

Council of Science Editors:

Jackson T. PHLPP1 Regulates Hippocampal Akt Survival Signaling. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041837


University of Florida

8. Warren, Soni A. Cardiac MLCK (cMLCK) and Its Role in Cardiac Function.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2011, University of Florida

 The main focus of our laboratory is to elucidate transcriptional regulation of homeodomain transcription factor Nkx2.5 in the heart. For this study, we have focused… (more)

Subjects/Keywords: Cardiac output; Genetic loci; Heart; Heart ventricles; Journalism; Messenger RNA; Myocardium; Phosphorylation; Polymerase chain reaction; Promoter regions; cmlck  – nkx25

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warren, S. A. (2011). Cardiac MLCK (cMLCK) and Its Role in Cardiac Function. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043629

Chicago Manual of Style (16th Edition):

Warren, Soni A. “Cardiac MLCK (cMLCK) and Its Role in Cardiac Function.” 2011. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0043629.

MLA Handbook (7th Edition):

Warren, Soni A. “Cardiac MLCK (cMLCK) and Its Role in Cardiac Function.” 2011. Web. 22 Oct 2019.

Vancouver:

Warren SA. Cardiac MLCK (cMLCK) and Its Role in Cardiac Function. [Internet] [Doctoral dissertation]. University of Florida; 2011. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0043629.

Council of Science Editors:

Warren SA. Cardiac MLCK (cMLCK) and Its Role in Cardiac Function. [Doctoral Dissertation]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043629


University of Florida

9. Baskin, Rebekah. Characterization of Jak2 Small Molecule Inhibitors for Cancer Therapy and Examination of Novel Cell Survival Signaling Mechanisms.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2012, University of Florida

 Jak2 is a non-receptor tyrosine kinase that is involved in proliferative signaling through its association with various cytokine receptors. Hyperactive Jak2 signaling has been implicated… (more)

Subjects/Keywords: Apoptosis; Cell growth; Cell lines; Cells; Glioblastoma; In vitro fertilization; Molecules; Phosphorylation; Tumors; Viability; cancer  – g6  – glioblastoma  – jak2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baskin, R. (2012). Characterization of Jak2 Small Molecule Inhibitors for Cancer Therapy and Examination of Novel Cell Survival Signaling Mechanisms. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044869

Chicago Manual of Style (16th Edition):

Baskin, Rebekah. “Characterization of Jak2 Small Molecule Inhibitors for Cancer Therapy and Examination of Novel Cell Survival Signaling Mechanisms.” 2012. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0044869.

MLA Handbook (7th Edition):

Baskin, Rebekah. “Characterization of Jak2 Small Molecule Inhibitors for Cancer Therapy and Examination of Novel Cell Survival Signaling Mechanisms.” 2012. Web. 22 Oct 2019.

Vancouver:

Baskin R. Characterization of Jak2 Small Molecule Inhibitors for Cancer Therapy and Examination of Novel Cell Survival Signaling Mechanisms. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0044869.

Council of Science Editors:

Baskin R. Characterization of Jak2 Small Molecule Inhibitors for Cancer Therapy and Examination of Novel Cell Survival Signaling Mechanisms. [Doctoral Dissertation]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/UFE0044869


University of Florida

10. Dai, Xiaoshuang. The Mechanism by Which Lentinula Edodes Modulates Cytokine Production of Innate Immune Cells.

Degree: PhD, Nutritional Sciences, 2015, University of Florida

 Inflammation is associated with many chronic conditions and diseases, including obesity, atherosclerosis, cancer, neurodegenerative and autoimmune diseases, and is tightly coordinated by cytokines. Lentinula edodes,… (more)

Subjects/Keywords: Antiinflammatories; Cell lines; Cells; Chemokines; Cytokines; Inflammation; Macrophages; Phosphorylation; Receptors; Shiitake Mushrooms; ccl3  – cytokine  – inflammation  – lentinan  – monocyte  – mushroom  – thp-1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dai, X. (2015). The Mechanism by Which Lentinula Edodes Modulates Cytokine Production of Innate Immune Cells. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0048989

Chicago Manual of Style (16th Edition):

Dai, Xiaoshuang. “The Mechanism by Which Lentinula Edodes Modulates Cytokine Production of Innate Immune Cells.” 2015. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0048989.

MLA Handbook (7th Edition):

Dai, Xiaoshuang. “The Mechanism by Which Lentinula Edodes Modulates Cytokine Production of Innate Immune Cells.” 2015. Web. 22 Oct 2019.

Vancouver:

Dai X. The Mechanism by Which Lentinula Edodes Modulates Cytokine Production of Innate Immune Cells. [Internet] [Doctoral dissertation]. University of Florida; 2015. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0048989.

Council of Science Editors:

Dai X. The Mechanism by Which Lentinula Edodes Modulates Cytokine Production of Innate Immune Cells. [Doctoral Dissertation]. University of Florida; 2015. Available from: http://ufdc.ufl.edu/UFE0048989


University of Florida

11. Bailey, Rachel M. Modulators of Tauopathy in the Rtg4510 Mouse Model.

Degree: PhD, Medical Sciences - Neuroscience (IDP), 2013, University of Florida

 Tauopathies are neurodegenerative disorders characterized by the accumulation of intracellular neurofibrillary tangles composed of abnormally phosphorylated tau.  Tau bind sand stabilizes microtubules, a function thought… (more)

Subjects/Keywords: Aggregation; Antibodies; Diseases; Epitopes; Genotypes; In vitro fertilization; Mice; Pathology; Phosphorylation; Tauopathies; disease  – lrrk2  – model  – mouse  – neurodegeneration  – parkinson's  – tau  – tauopathy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bailey, R. M. (2013). Modulators of Tauopathy in the Rtg4510 Mouse Model. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0046079

Chicago Manual of Style (16th Edition):

Bailey, Rachel M. “Modulators of Tauopathy in the Rtg4510 Mouse Model.” 2013. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0046079.

MLA Handbook (7th Edition):

Bailey, Rachel M. “Modulators of Tauopathy in the Rtg4510 Mouse Model.” 2013. Web. 22 Oct 2019.

Vancouver:

Bailey RM. Modulators of Tauopathy in the Rtg4510 Mouse Model. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0046079.

Council of Science Editors:

Bailey RM. Modulators of Tauopathy in the Rtg4510 Mouse Model. [Doctoral Dissertation]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0046079


University of Florida

12. Walters, Jewell. Characterization of Microsomal Prostaglandin E Synthase-1 Gene Regulation by the Pro-Inflammatory Cytokine Interleukin 1-beta.

Degree: PhD, Medical Sciences - Biochemistry and Molecular Biology (IDP), 2009, University of Florida

 The arachidonic acid (AA) pathway is a major contributor to the inflammatory response, pain production and cellular homeostasis. AA is liberated from membrane phospholipids by… (more)

Subjects/Keywords: Biochemistry; Cytokines; Gene expression; Gene induction; Lungs; Messenger RNA; Phosphorylation; Promoter regions; Prostaglandins; RNA; arachidonic, egr1, il1, mpges1, pge2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Walters, J. (2009). Characterization of Microsomal Prostaglandin E Synthase-1 Gene Regulation by the Pro-Inflammatory Cytokine Interleukin 1-beta. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0024982

Chicago Manual of Style (16th Edition):

Walters, Jewell. “Characterization of Microsomal Prostaglandin E Synthase-1 Gene Regulation by the Pro-Inflammatory Cytokine Interleukin 1-beta.” 2009. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0024982.

MLA Handbook (7th Edition):

Walters, Jewell. “Characterization of Microsomal Prostaglandin E Synthase-1 Gene Regulation by the Pro-Inflammatory Cytokine Interleukin 1-beta.” 2009. Web. 22 Oct 2019.

Vancouver:

Walters J. Characterization of Microsomal Prostaglandin E Synthase-1 Gene Regulation by the Pro-Inflammatory Cytokine Interleukin 1-beta. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0024982.

Council of Science Editors:

Walters J. Characterization of Microsomal Prostaglandin E Synthase-1 Gene Regulation by the Pro-Inflammatory Cytokine Interleukin 1-beta. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0024982


University of Florida

13. Smith, Tyler. A Potential Role of BBS5 Phosphorylation in Light-­ Dependent Translocation of Arrestin.

Degree: 2012, University of Florida

 The light-driven translocation of visual arrestin* plays a critical role in regulating light adaptation in photoreceptor cells; however, the exact mechanism of arrestin translocation in… (more)

Subjects/Keywords: Complementary DNA; DNA; Fluorescence; Immunoprecipitation; Kidney dialysis; Monoclonal antibodies; Phosphorylation; Photoreceptors; Polymerase chain reaction; Retina; Arrestins; Light; Photoreceptors; Protein kinase C

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, T. (2012). A Potential Role of BBS5 Phosphorylation in Light-­ Dependent Translocation of Arrestin. (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00060880

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Tyler. “A Potential Role of BBS5 Phosphorylation in Light-­ Dependent Translocation of Arrestin.” 2012. Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00060880.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Tyler. “A Potential Role of BBS5 Phosphorylation in Light-­ Dependent Translocation of Arrestin.” 2012. Web. 22 Oct 2019.

Vancouver:

Smith T. A Potential Role of BBS5 Phosphorylation in Light-­ Dependent Translocation of Arrestin. [Internet] [Thesis]. University of Florida; 2012. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00060880.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith T. A Potential Role of BBS5 Phosphorylation in Light-­ Dependent Translocation of Arrestin. [Thesis]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/AA00060880

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

14. Smith, Ley C. The Interplay of Genomic and Non-Genomic Estrogen Receptor Signaling Pathways in Mediating Cellular Repsonses to Xenoestrogens.

Degree: MS, Veterinary Medical Sciences - Veterinary Medicine, 2013, University of Florida

 Estrogen can exert cellular effects through both nuclear(ESR1 and ESR2) and membrane-bound receptors (GPER). It is unclear if thesereceptors act independently or engage in crosstalk… (more)

Subjects/Keywords: Agonists; Breast cancer; Cell growth; Cells; Dosage; Estrogen receptors; Estrogens; Ligands; Phosphorylation; Receptors; bisphenol-a  – genistein  – gper  – xenoestrogen

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, L. C. (2013). The Interplay of Genomic and Non-Genomic Estrogen Receptor Signaling Pathways in Mediating Cellular Repsonses to Xenoestrogens. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0045522

Chicago Manual of Style (16th Edition):

Smith, Ley C. “The Interplay of Genomic and Non-Genomic Estrogen Receptor Signaling Pathways in Mediating Cellular Repsonses to Xenoestrogens.” 2013. Masters Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0045522.

MLA Handbook (7th Edition):

Smith, Ley C. “The Interplay of Genomic and Non-Genomic Estrogen Receptor Signaling Pathways in Mediating Cellular Repsonses to Xenoestrogens.” 2013. Web. 22 Oct 2019.

Vancouver:

Smith LC. The Interplay of Genomic and Non-Genomic Estrogen Receptor Signaling Pathways in Mediating Cellular Repsonses to Xenoestrogens. [Internet] [Masters thesis]. University of Florida; 2013. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0045522.

Council of Science Editors:

Smith LC. The Interplay of Genomic and Non-Genomic Estrogen Receptor Signaling Pathways in Mediating Cellular Repsonses to Xenoestrogens. [Masters Thesis]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0045522


University of Florida

15. Gnanasambandan, Kavitha. Molecular Mechanisms for the Constitutive Activation of Jak2 Mutations.

Degree: PhD, Medical Sciences - Biochemistry and Molecular Biology (IDP), 2011, University of Florida

 Jak2 tyrosine kinase plays a critical role in hematopoiesis and is essential for life. Constitutive activation of Jak-STAT signaling through Jak2 mutations have been linked… (more)

Subjects/Keywords: Amino acids; Antibodies; Exons; Genetic mutation; Molecular interactions; Phosphorylation; Plasmids; Proteins; Receptors; Simulations; allosteric  – exon12  – inhibitors  – jak2  – mpn  – mutations  – v617f

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gnanasambandan, K. (2011). Molecular Mechanisms for the Constitutive Activation of Jak2 Mutations. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043569

Chicago Manual of Style (16th Edition):

Gnanasambandan, Kavitha. “Molecular Mechanisms for the Constitutive Activation of Jak2 Mutations.” 2011. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0043569.

MLA Handbook (7th Edition):

Gnanasambandan, Kavitha. “Molecular Mechanisms for the Constitutive Activation of Jak2 Mutations.” 2011. Web. 22 Oct 2019.

Vancouver:

Gnanasambandan K. Molecular Mechanisms for the Constitutive Activation of Jak2 Mutations. [Internet] [Doctoral dissertation]. University of Florida; 2011. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0043569.

Council of Science Editors:

Gnanasambandan K. Molecular Mechanisms for the Constitutive Activation of Jak2 Mutations. [Doctoral Dissertation]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043569


University of Florida

16. Beker Aydemir,Tolunay. Role of Zinc and Zinc Transporters in Liver Regeneration.

Degree: PhD, Medical Sciences - Biochemistry and Molecular Biology (IDP), 2011, University of Florida

 Chronic liver disease and cirrhosis rank twelfth and fifth among the leading causes of death in the America and Europe, respectively. Zinc deficiency has been… (more)

Subjects/Keywords: DNA; Hepatectomy; Hepatocytes; Liver; Liver regeneration; Messenger RNA; Phosphatases; Phosphorylation; RNA; Zinc; hepatocyte  – liver  – phosphatase  – proliferation  – regeneration  – zinc  – zip14

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aydemir,Tolunay, B. (2011). Role of Zinc and Zinc Transporters in Liver Regeneration. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043332

Chicago Manual of Style (16th Edition):

Aydemir,Tolunay, Beker. “Role of Zinc and Zinc Transporters in Liver Regeneration.” 2011. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0043332.

MLA Handbook (7th Edition):

Aydemir,Tolunay, Beker. “Role of Zinc and Zinc Transporters in Liver Regeneration.” 2011. Web. 22 Oct 2019.

Vancouver:

Aydemir,Tolunay B. Role of Zinc and Zinc Transporters in Liver Regeneration. [Internet] [Doctoral dissertation]. University of Florida; 2011. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0043332.

Council of Science Editors:

Aydemir,Tolunay B. Role of Zinc and Zinc Transporters in Liver Regeneration. [Doctoral Dissertation]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043332


University of Florida

17. Dabelic, Rea. The Role of Suppressor of Cytokine Signaling 1 (SOCS-1) Mimetic and Antagonist Peptides in Host Defense.

Degree: PhD, Microbiology and Cell Science, 2010, University of Florida

 Suppressors of cytokine signaling (SOCS) are negative regulators of both innate and adaptive immunity via inhibition of signaling by cytokines and Toll-like receptors. In this… (more)

Subjects/Keywords: Antivirals; Boxes; Cytokines; Encephalomyocarditis virus; Infections; Interferons; Macrophages; Phosphorylation; Receptors; Signals; antiviral, cytokine, interferon, mimetics, socs

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dabelic, R. (2010). The Role of Suppressor of Cytokine Signaling 1 (SOCS-1) Mimetic and Antagonist Peptides in Host Defense. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041579

Chicago Manual of Style (16th Edition):

Dabelic, Rea. “The Role of Suppressor of Cytokine Signaling 1 (SOCS-1) Mimetic and Antagonist Peptides in Host Defense.” 2010. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0041579.

MLA Handbook (7th Edition):

Dabelic, Rea. “The Role of Suppressor of Cytokine Signaling 1 (SOCS-1) Mimetic and Antagonist Peptides in Host Defense.” 2010. Web. 22 Oct 2019.

Vancouver:

Dabelic R. The Role of Suppressor of Cytokine Signaling 1 (SOCS-1) Mimetic and Antagonist Peptides in Host Defense. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0041579.

Council of Science Editors:

Dabelic R. The Role of Suppressor of Cytokine Signaling 1 (SOCS-1) Mimetic and Antagonist Peptides in Host Defense. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041579


University of Florida

18. Silmi, Ronya. Optimization of adeno-associated virus capsid increased transduction of human monocyte-derived dendritic cells (moDCs).

Degree: 2013, University of Florida

 While the probability of developing cancer remains relatively high, patients with a cancer diagnosis are dependent on suboptimal therapies such as chemotherapy, vigorous surgery, transplants… (more)

Subjects/Keywords: Antigens; Cancer; Capsid; Dendritic cells; Gene therapy; Phosphorylation; RNA; Site directed mutagenesis; Tumors; Vaccinations; Cancer – Immunotherapy; Dendritic cells; Parvoviruses; Transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Silmi, R. (2013). Optimization of adeno-associated virus capsid increased transduction of human monocyte-derived dendritic cells (moDCs). (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00060438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silmi, Ronya. “Optimization of adeno-associated virus capsid increased transduction of human monocyte-derived dendritic cells (moDCs).” 2013. Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00060438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silmi, Ronya. “Optimization of adeno-associated virus capsid increased transduction of human monocyte-derived dendritic cells (moDCs).” 2013. Web. 22 Oct 2019.

Vancouver:

Silmi R. Optimization of adeno-associated virus capsid increased transduction of human monocyte-derived dendritic cells (moDCs). [Internet] [Thesis]. University of Florida; 2013. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00060438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silmi R. Optimization of adeno-associated virus capsid increased transduction of human monocyte-derived dendritic cells (moDCs). [Thesis]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/AA00060438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

19. Pope, Arthur. Role of Asymmetric Dimethylarginine(ADMA)in the Regulation of Endothelial Derived Nitric Oxide.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2009, University of Florida

 The endogenous NOS inhibitor Asymmetric Dimethylarginine (ADMA) has been demonstrated to be an independent cardiovascular disease risk factor. However, the mechanisms regarding how ADMA levels… (more)

Subjects/Keywords: Diabetes; Diseases; Endothelial cells; Enzymes; Gene silencing; Oxides; Phosphorylation; Plasmas; Protein isoforms; Superoxides; adma, ddah, endothelial, nitric, oxide, superoxide, tetrahydrobiopterin

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pope, A. (2009). Role of Asymmetric Dimethylarginine(ADMA)in the Regulation of Endothelial Derived Nitric Oxide. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0024902

Chicago Manual of Style (16th Edition):

Pope, Arthur. “Role of Asymmetric Dimethylarginine(ADMA)in the Regulation of Endothelial Derived Nitric Oxide.” 2009. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0024902.

MLA Handbook (7th Edition):

Pope, Arthur. “Role of Asymmetric Dimethylarginine(ADMA)in the Regulation of Endothelial Derived Nitric Oxide.” 2009. Web. 22 Oct 2019.

Vancouver:

Pope A. Role of Asymmetric Dimethylarginine(ADMA)in the Regulation of Endothelial Derived Nitric Oxide. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0024902.

Council of Science Editors:

Pope A. Role of Asymmetric Dimethylarginine(ADMA)in the Regulation of Endothelial Derived Nitric Oxide. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0024902


University of Florida

20. Szarowicz, Sarah. Anthrax Edema Toxin Impairs Neutrophil Actin Based Motility By Stimulating VASP Phosphorylation.

Degree: PhD, Medical Sciences - Immunology and Microbiology (IDP), 2010, University of Florida

 Inhalation anthrax can lead to sepsis and death within days. The fulminate nature of illness reveals minimal elevation of peripheral polymorphonuclear (PMNs) counts at time… (more)

Subjects/Keywords: Actins; Anthrax; Chemotaxis; Infections; Listeria; Microfilaments; Neutrophils; Phosphorylation; Pseudopodia; Toxins; actin, anthrax, bacillus, cytoskeleton, edema, neutrophils, vasp

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Szarowicz, S. (2010). Anthrax Edema Toxin Impairs Neutrophil Actin Based Motility By Stimulating VASP Phosphorylation. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041387

Chicago Manual of Style (16th Edition):

Szarowicz, Sarah. “Anthrax Edema Toxin Impairs Neutrophil Actin Based Motility By Stimulating VASP Phosphorylation.” 2010. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0041387.

MLA Handbook (7th Edition):

Szarowicz, Sarah. “Anthrax Edema Toxin Impairs Neutrophil Actin Based Motility By Stimulating VASP Phosphorylation.” 2010. Web. 22 Oct 2019.

Vancouver:

Szarowicz S. Anthrax Edema Toxin Impairs Neutrophil Actin Based Motility By Stimulating VASP Phosphorylation. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0041387.

Council of Science Editors:

Szarowicz S. Anthrax Edema Toxin Impairs Neutrophil Actin Based Motility By Stimulating VASP Phosphorylation. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041387


University of Florida

21. Jermanus, Caroline. Porphyromonas gingivalis Inhibits Mitochondrion-Induced Apoptosis through Akt Pathway.

Degree: MS, Dental Sciences - Dentistry, 2010, University of Florida

 Periodontal disease affects the supporting structures surrounding teeth in the oral cavity. Periodontal diseases are polymicrobial chronic infections of multifactorial etiology. Bacteria in the dental… (more)

Subjects/Keywords: Antibodies; Apoptosis; Bacteria; Cell death; Epithelial cells; Infections; Periodontal diseases; Phosphorylation; Porphyromonas gingivalis; Small interfering RNA; akt, apoptosis, epithelial, pgingivalis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jermanus, C. (2010). Porphyromonas gingivalis Inhibits Mitochondrion-Induced Apoptosis through Akt Pathway. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041799

Chicago Manual of Style (16th Edition):

Jermanus, Caroline. “Porphyromonas gingivalis Inhibits Mitochondrion-Induced Apoptosis through Akt Pathway.” 2010. Masters Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0041799.

MLA Handbook (7th Edition):

Jermanus, Caroline. “Porphyromonas gingivalis Inhibits Mitochondrion-Induced Apoptosis through Akt Pathway.” 2010. Web. 22 Oct 2019.

Vancouver:

Jermanus C. Porphyromonas gingivalis Inhibits Mitochondrion-Induced Apoptosis through Akt Pathway. [Internet] [Masters thesis]. University of Florida; 2010. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0041799.

Council of Science Editors:

Jermanus C. Porphyromonas gingivalis Inhibits Mitochondrion-Induced Apoptosis through Akt Pathway. [Masters Thesis]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041799


University of Florida

22. Guthrie, Gregory J. Function and Regulation of Intestinal Zinc Transporter ZIP14.

Degree: PhD, Nutritional Sciences, 2013, University of Florida

 ZIP14 is a transporter that increases the intracellular labile pool of zinc.   ZIP14 has a high level of expression in the gastrointestinal (gi) tract, yet… (more)

Subjects/Keywords: Acrodermatitis; Caco 2 cells; Dextrans; Inflammation; Intestines; Liver; Phosphorylation; Proteins; Tight junctions; Zinc; claudin1  – intestine  – lps  – occludin  – permeability  – slc39a14  – zinc  – zip14

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guthrie, G. J. (2013). Function and Regulation of Intestinal Zinc Transporter ZIP14. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0046238

Chicago Manual of Style (16th Edition):

Guthrie, Gregory J. “Function and Regulation of Intestinal Zinc Transporter ZIP14.” 2013. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0046238.

MLA Handbook (7th Edition):

Guthrie, Gregory J. “Function and Regulation of Intestinal Zinc Transporter ZIP14.” 2013. Web. 22 Oct 2019.

Vancouver:

Guthrie GJ. Function and Regulation of Intestinal Zinc Transporter ZIP14. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0046238.

Council of Science Editors:

Guthrie GJ. Function and Regulation of Intestinal Zinc Transporter ZIP14. [Doctoral Dissertation]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0046238


University of Florida

23. Jager, Lindsey. The Therapeutic Effects of a Suppressor of Cytokine Signaling 1 Mimetic Peptide in Experimental Allergic Encephalomyelitis, a Murine Model of Multiple Sclerosis.

Degree: PhD, Microbiology and Cell Science, 2010, University of Florida

 Suppressors of cytokine signaling (SOCS) negatively regulate several facets of the immune system by interfering with the Janus kinas/signal transducers and activators of transcription (JAK/STAT)… (more)

Subjects/Keywords: Cells; Cytokines; Diseases; Experimental autoimmune encephalomyelitis; Interferons; Mice; Multiple sclerosis; Phosphorylation; Receptors; Splenocytes; eae, il, il17, il23, socs, th17

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jager, L. (2010). The Therapeutic Effects of a Suppressor of Cytokine Signaling 1 Mimetic Peptide in Experimental Allergic Encephalomyelitis, a Murine Model of Multiple Sclerosis. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041578

Chicago Manual of Style (16th Edition):

Jager, Lindsey. “The Therapeutic Effects of a Suppressor of Cytokine Signaling 1 Mimetic Peptide in Experimental Allergic Encephalomyelitis, a Murine Model of Multiple Sclerosis.” 2010. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0041578.

MLA Handbook (7th Edition):

Jager, Lindsey. “The Therapeutic Effects of a Suppressor of Cytokine Signaling 1 Mimetic Peptide in Experimental Allergic Encephalomyelitis, a Murine Model of Multiple Sclerosis.” 2010. Web. 22 Oct 2019.

Vancouver:

Jager L. The Therapeutic Effects of a Suppressor of Cytokine Signaling 1 Mimetic Peptide in Experimental Allergic Encephalomyelitis, a Murine Model of Multiple Sclerosis. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0041578.

Council of Science Editors:

Jager L. The Therapeutic Effects of a Suppressor of Cytokine Signaling 1 Mimetic Peptide in Experimental Allergic Encephalomyelitis, a Murine Model of Multiple Sclerosis. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041578


University of Florida

24. Humbard, Matthew. Post-translational Modification and the 20S Proteasome System of the Haloarchaeon Haloferax volcanii.

Degree: PhD, Microbiology and Cell Science, 2009, University of Florida

 While much of the study of proteasome systems in all organisms, eukaryotes and prokaryotes, focuses on substrate recognition, ubiquitin-like modifiers, or protein targeting, the 20S… (more)

Subjects/Keywords: Acetylation; Amino acids; Archaea; Enzymes; Haloferax volcanii; Ions; Mass spectroscopy; Phosphorylation; Proteins; Yeasts; acetylation, acetyltransferase, activating, archaea, atpase, dimensional, electrophoresis, haloarchaea, haloferax, knock, mass, methylation, modification, ms, nucleotidase, out, pan, phosphorylation, posttranslational, protease, proteasome, proteome, samp, spectrometry, two, ubiquitin, volcanii

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Humbard, M. (2009). Post-translational Modification and the 20S Proteasome System of the Haloarchaeon Haloferax volcanii. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0024797

Chicago Manual of Style (16th Edition):

Humbard, Matthew. “Post-translational Modification and the 20S Proteasome System of the Haloarchaeon Haloferax volcanii.” 2009. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0024797.

MLA Handbook (7th Edition):

Humbard, Matthew. “Post-translational Modification and the 20S Proteasome System of the Haloarchaeon Haloferax volcanii.” 2009. Web. 22 Oct 2019.

Vancouver:

Humbard M. Post-translational Modification and the 20S Proteasome System of the Haloarchaeon Haloferax volcanii. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0024797.

Council of Science Editors:

Humbard M. Post-translational Modification and the 20S Proteasome System of the Haloarchaeon Haloferax volcanii. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0024797


University of Florida

25. Aller, Walter W., 1956-. The unique activation status of intestinal intraepithelial lymphocytes.

Degree: PhD, Molecular Genetics and Microbiology, 1997, University of Florida

Subjects/Keywords: Antibodies; Antigens; Calcium; Cells; Lymphocytes; Molecules; Phosphorylation; Receptors; Signal transduction; Splenocytes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aller, Walter W., 1. (1997). The unique activation status of intestinal intraepithelial lymphocytes. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00040814

Chicago Manual of Style (16th Edition):

Aller, Walter W., 1956-. “The unique activation status of intestinal intraepithelial lymphocytes.” 1997. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00040814.

MLA Handbook (7th Edition):

Aller, Walter W., 1956-. “The unique activation status of intestinal intraepithelial lymphocytes.” 1997. Web. 22 Oct 2019.

Vancouver:

Aller, Walter W. 1. The unique activation status of intestinal intraepithelial lymphocytes. [Internet] [Doctoral dissertation]. University of Florida; 1997. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00040814.

Council of Science Editors:

Aller, Walter W. 1. The unique activation status of intestinal intraepithelial lymphocytes. [Doctoral Dissertation]. University of Florida; 1997. Available from: http://ufdc.ufl.edu/AA00040814


University of Florida

26. Smith, Ira J. Effects of the Calpain Proteases on the Ubiquitin-Proteasome system and protein synthesis signaling in rat skeletal muscle.

Degree: PhD, Health and Human Performance, 2005, University of Florida

Subjects/Keywords: Calcium; Incubation; Muscles; Muscular atrophy; Phosphorylation; Protein synthesis; Proteins; Rats; Skeletal muscle; Ubiquitins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith, I. J. (2005). Effects of the Calpain Proteases on the Ubiquitin-Proteasome system and protein synthesis signaling in rat skeletal muscle. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00036988

Chicago Manual of Style (16th Edition):

Smith, Ira J. “Effects of the Calpain Proteases on the Ubiquitin-Proteasome system and protein synthesis signaling in rat skeletal muscle.” 2005. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00036988.

MLA Handbook (7th Edition):

Smith, Ira J. “Effects of the Calpain Proteases on the Ubiquitin-Proteasome system and protein synthesis signaling in rat skeletal muscle.” 2005. Web. 22 Oct 2019.

Vancouver:

Smith IJ. Effects of the Calpain Proteases on the Ubiquitin-Proteasome system and protein synthesis signaling in rat skeletal muscle. [Internet] [Doctoral dissertation]. University of Florida; 2005. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00036988.

Council of Science Editors:

Smith IJ. Effects of the Calpain Proteases on the Ubiquitin-Proteasome system and protein synthesis signaling in rat skeletal muscle. [Doctoral Dissertation]. University of Florida; 2005. Available from: http://ufdc.ufl.edu/AA00036988


University of Florida

27. Reist, Caroline. Inhibition of renal cell carcinoma growth by HIV-1 protease inhibitor nelfinavir.

Degree: 2013, University of Florida

 To improve cancer therapeutic options, drugs are being investigated to inhibit signaling pathways that promote cell proliferation or survival. A common pathway that promotes cell… (more)

Subjects/Keywords: Apoptosis; Cell growth; Cell lines; Cultured cells; Dosage; Irradiation; Phosphorylation; Reism; Renal cell carcinoma; Tumors; Cancer – Treatment; Protease inhibitors; Renal cell carcinoma

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Reist, C. (2013). Inhibition of renal cell carcinoma growth by HIV-1 protease inhibitor nelfinavir. (Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00061004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Reist, Caroline. “Inhibition of renal cell carcinoma growth by HIV-1 protease inhibitor nelfinavir.” 2013. Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00061004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Reist, Caroline. “Inhibition of renal cell carcinoma growth by HIV-1 protease inhibitor nelfinavir.” 2013. Web. 22 Oct 2019.

Vancouver:

Reist C. Inhibition of renal cell carcinoma growth by HIV-1 protease inhibitor nelfinavir. [Internet] [Thesis]. University of Florida; 2013. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00061004.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Reist C. Inhibition of renal cell carcinoma growth by HIV-1 protease inhibitor nelfinavir. [Thesis]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/AA00061004

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

28. Abner, Clint W., 1971-. Biochemical analysis of human base excision repair.

Degree: PhD, Biochemistry and Molecular Biology, 2002, University of Florida

Subjects/Keywords: Biochemistry; DNA; DNA damage; DNA repair; Enzymes; Gels; Nucleotides; Phosphorylation; Proteins; Yeasts

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abner, Clint W., 1. (2002). Biochemical analysis of human base excision repair. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/AA00039669

Chicago Manual of Style (16th Edition):

Abner, Clint W., 1971-. “Biochemical analysis of human base excision repair.” 2002. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/AA00039669.

MLA Handbook (7th Edition):

Abner, Clint W., 1971-. “Biochemical analysis of human base excision repair.” 2002. Web. 22 Oct 2019.

Vancouver:

Abner, Clint W. 1. Biochemical analysis of human base excision repair. [Internet] [Doctoral dissertation]. University of Florida; 2002. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/AA00039669.

Council of Science Editors:

Abner, Clint W. 1. Biochemical analysis of human base excision repair. [Doctoral Dissertation]. University of Florida; 2002. Available from: http://ufdc.ufl.edu/AA00039669


University of Florida

29. Skehan, Ryan. Mechanism Controlling Mmr-Dependent Apoptosis in Response to Sn1-Methylators.

Degree: MS, Medical Sciences - Medicine, 2011, University of Florida

 In this report we examine the role of the DNA mismatch repair protein MutS? in the activation of MNNG-induced apoptosis and uncover the signaling responsible… (more)

Subjects/Keywords: Annexins; Apoptosis; ATMs; Cell lines; Cells; DNA; DNA damage; DNA mismatch repair; Molecules; Phosphorylation; alkylator  – apoptosis  – atm  – cancer  – lymphoblast  – mismatch  – mnng  – msh6  – p53  – sn1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Skehan, R. (2011). Mechanism Controlling Mmr-Dependent Apoptosis in Response to Sn1-Methylators. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043084

Chicago Manual of Style (16th Edition):

Skehan, Ryan. “Mechanism Controlling Mmr-Dependent Apoptosis in Response to Sn1-Methylators.” 2011. Masters Thesis, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0043084.

MLA Handbook (7th Edition):

Skehan, Ryan. “Mechanism Controlling Mmr-Dependent Apoptosis in Response to Sn1-Methylators.” 2011. Web. 22 Oct 2019.

Vancouver:

Skehan R. Mechanism Controlling Mmr-Dependent Apoptosis in Response to Sn1-Methylators. [Internet] [Masters thesis]. University of Florida; 2011. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0043084.

Council of Science Editors:

Skehan R. Mechanism Controlling Mmr-Dependent Apoptosis in Response to Sn1-Methylators. [Masters Thesis]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043084


University of Florida

30. Zhang, Jiejin. Leptin Antagonist and Soluble Leptin Receptor Block Leptin Action in Vivo Evidence for an Essential Role of Leptin in Homeostatic Energy Regulation.

Degree: PhD, Medical Sciences - Physiology and Pharmacology (IDP), 2009, University of Florida

 One-third of the US adults are obese, contributing to serious health issues. Leptin is an adipocyte-derived hormone that acts on the satiety center in the… (more)

Subjects/Keywords: Body weight; Energy intake; Food intake; Gene therapy; Hypothalamus; Obesity; Phosphorylation; Rats; Receptors; Transgenes; feeding, gene, highfat, leptin, neutralization, obesity, receptor, resistance, signaling, soluble, stat3, therapy, ucp1

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, J. (2009). Leptin Antagonist and Soluble Leptin Receptor Block Leptin Action in Vivo Evidence for an Essential Role of Leptin in Homeostatic Energy Regulation. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0024220

Chicago Manual of Style (16th Edition):

Zhang, Jiejin. “Leptin Antagonist and Soluble Leptin Receptor Block Leptin Action in Vivo Evidence for an Essential Role of Leptin in Homeostatic Energy Regulation.” 2009. Doctoral Dissertation, University of Florida. Accessed October 22, 2019. http://ufdc.ufl.edu/UFE0024220.

MLA Handbook (7th Edition):

Zhang, Jiejin. “Leptin Antagonist and Soluble Leptin Receptor Block Leptin Action in Vivo Evidence for an Essential Role of Leptin in Homeostatic Energy Regulation.” 2009. Web. 22 Oct 2019.

Vancouver:

Zhang J. Leptin Antagonist and Soluble Leptin Receptor Block Leptin Action in Vivo Evidence for an Essential Role of Leptin in Homeostatic Energy Regulation. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2019 Oct 22]. Available from: http://ufdc.ufl.edu/UFE0024220.

Council of Science Editors:

Zhang J. Leptin Antagonist and Soluble Leptin Receptor Block Leptin Action in Vivo Evidence for an Essential Role of Leptin in Homeostatic Energy Regulation. [Doctoral Dissertation]. University of Florida; 2009. Available from: http://ufdc.ufl.edu/UFE0024220

[1] [2] [3]

.