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You searched for subject:(p38). Showing records 1 – 30 of 176 total matches.

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1. Tasker, Alexander John. Processes of hybrid knowledge creation in pastoralist development.

Degree: PhD, 2018, University of Sussex

 This thesis addresses an under-researched disjunction surrounding knowledge creation between, and within, development and pastoralist groups. Many academics increasingly recognise pastoralist populations as creative and… (more)

Subjects/Keywords: SF0140.P38

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APA (6th Edition):

Tasker, A. J. (2018). Processes of hybrid knowledge creation in pastoralist development. (Doctoral Dissertation). University of Sussex. Retrieved from http://sro.sussex.ac.uk/id/eprint/80671/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767025

Chicago Manual of Style (16th Edition):

Tasker, Alexander John. “Processes of hybrid knowledge creation in pastoralist development.” 2018. Doctoral Dissertation, University of Sussex. Accessed April 19, 2019. http://sro.sussex.ac.uk/id/eprint/80671/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767025.

MLA Handbook (7th Edition):

Tasker, Alexander John. “Processes of hybrid knowledge creation in pastoralist development.” 2018. Web. 19 Apr 2019.

Vancouver:

Tasker AJ. Processes of hybrid knowledge creation in pastoralist development. [Internet] [Doctoral dissertation]. University of Sussex; 2018. [cited 2019 Apr 19]. Available from: http://sro.sussex.ac.uk/id/eprint/80671/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767025.

Council of Science Editors:

Tasker AJ. Processes of hybrid knowledge creation in pastoralist development. [Doctoral Dissertation]. University of Sussex; 2018. Available from: http://sro.sussex.ac.uk/id/eprint/80671/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767025


Louisiana State University

2. Mathews, Amber Lynn. Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia.

Degree: MS, 2010, Louisiana State University

 Microsporidia are ubiquitous, obligate intracellular eukaryotes that cause chronic diarrhea and disseminated diseases in humans, especially in immunocompromised individuals. Macrophages, cellular components of the innate… (more)

Subjects/Keywords: interleukin 23; p38 gamma; innate immunity; Encephalitozoon; microsporidia; macrophage; dendritic cells; interleukin 12; immune response; p38 MAPK; p38 gamma; p38 delta

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APA (6th Edition):

Mathews, A. L. (2010). Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia. (Masters Thesis). Louisiana State University. Retrieved from etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366

Chicago Manual of Style (16th Edition):

Mathews, Amber Lynn. “Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia.” 2010. Masters Thesis, Louisiana State University. Accessed April 19, 2019. etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366.

MLA Handbook (7th Edition):

Mathews, Amber Lynn. “Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia.” 2010. Web. 19 Apr 2019.

Vancouver:

Mathews AL. Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia. [Internet] [Masters thesis]. Louisiana State University; 2010. [cited 2019 Apr 19]. Available from: etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366.

Council of Science Editors:

Mathews AL. Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia. [Masters Thesis]. Louisiana State University; 2010. Available from: etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366


University College Cork

3. O'Callaghan, Carol. Novel insights into the expression and function of p38δ MAPK in oesophageal squamous cell carcinoma.

Degree: 2015, University College Cork

 Oesophageal cancer is an aggressive malignancy which is resistant to conventional therapy and has a poor prognosis. A greater understanding of the underlying molecular biology… (more)

Subjects/Keywords: Cancer; Oesophageal; p38; MAPK

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APA (6th Edition):

O'Callaghan, C. (2015). Novel insights into the expression and function of p38δ MAPK in oesophageal squamous cell carcinoma. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/2082

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Callaghan, Carol. “Novel insights into the expression and function of p38δ MAPK in oesophageal squamous cell carcinoma.” 2015. Thesis, University College Cork. Accessed April 19, 2019. http://hdl.handle.net/10468/2082.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Callaghan, Carol. “Novel insights into the expression and function of p38δ MAPK in oesophageal squamous cell carcinoma.” 2015. Web. 19 Apr 2019.

Vancouver:

O'Callaghan C. Novel insights into the expression and function of p38δ MAPK in oesophageal squamous cell carcinoma. [Internet] [Thesis]. University College Cork; 2015. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/10468/2082.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Callaghan C. Novel insights into the expression and function of p38δ MAPK in oesophageal squamous cell carcinoma. [Thesis]. University College Cork; 2015. Available from: http://hdl.handle.net/10468/2082

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. 細山, 有規子. Effects of gallotannin on osteoclastogenesis and the p38 MAP kinase pathway : 破骨細胞形成とp38 MAP kinase経路に対するGallotanninの影響.

Degree: 博士(歯学), 2017, Osaka Dental University / 大阪歯科大学

We examined the effect of gallotannin on osteoclast differentiation in vitro. We found that gallotannin suppressed osteoclast differentiation in a bone marrow culture system stimulated… (more)

Subjects/Keywords: Osteoclast|Gallotannin|Tannin|p38 MAP kinase|NFATc1

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APA (6th Edition):

細山, . (2017). Effects of gallotannin on osteoclastogenesis and the p38 MAP kinase pathway : 破骨細胞形成とp38 MAP kinase経路に対するGallotanninの影響. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000135/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

細山, 有規子. “Effects of gallotannin on osteoclastogenesis and the p38 MAP kinase pathway : 破骨細胞形成とp38 MAP kinase経路に対するGallotanninの影響.” 2017. Thesis, Osaka Dental University / 大阪歯科大学. Accessed April 19, 2019. http://id.nii.ac.jp/1392/00000135/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

細山, 有規子. “Effects of gallotannin on osteoclastogenesis and the p38 MAP kinase pathway : 破骨細胞形成とp38 MAP kinase経路に対するGallotanninの影響.” 2017. Web. 19 Apr 2019.

Vancouver:

細山 . Effects of gallotannin on osteoclastogenesis and the p38 MAP kinase pathway : 破骨細胞形成とp38 MAP kinase経路に対するGallotanninの影響. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. [cited 2019 Apr 19]. Available from: http://id.nii.ac.jp/1392/00000135/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

細山 . Effects of gallotannin on osteoclastogenesis and the p38 MAP kinase pathway : 破骨細胞形成とp38 MAP kinase経路に対するGallotanninの影響. [Thesis]. Osaka Dental University / 大阪歯科大学; 2017. Available from: http://id.nii.ac.jp/1392/00000135/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

5. Leung, Chun Yin Bosco. A role for the p38 MAPK in animal cell size regulation.

Degree: 2018, University of Toronto

Cell size within animal tissues is highly uniform. Previous data has shown that the p38 pathway plays a role in this regulation. The mTOR inhibitor… (more)

Subjects/Keywords: cell; cell size; mTOR; p38; Torin2; 0306

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APA (6th Edition):

Leung, C. Y. B. (2018). A role for the p38 MAPK in animal cell size regulation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91597

Chicago Manual of Style (16th Edition):

Leung, Chun Yin Bosco. “A role for the p38 MAPK in animal cell size regulation.” 2018. Masters Thesis, University of Toronto. Accessed April 19, 2019. http://hdl.handle.net/1807/91597.

MLA Handbook (7th Edition):

Leung, Chun Yin Bosco. “A role for the p38 MAPK in animal cell size regulation.” 2018. Web. 19 Apr 2019.

Vancouver:

Leung CYB. A role for the p38 MAPK in animal cell size regulation. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1807/91597.

Council of Science Editors:

Leung CYB. A role for the p38 MAPK in animal cell size regulation. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91597

6. Amin, Jakia. Regulation of autophagy in mammalian cells by stress activated signaling pathways.

Degree: 2013, Technische Universität Dortmund

 Autophagy is an intracellular bulk degradation process of cytoplasmic proteins and organelles aimed to preserve cellular homeostasis. It involves the formation of double-membrane structures, called… (more)

Subjects/Keywords: Autophagy; Microtubules; p38; Senescence; 540; 570

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APA (6th Edition):

Amin, J. (2013). Regulation of autophagy in mammalian cells by stress activated signaling pathways. (Thesis). Technische Universität Dortmund. Retrieved from http://hdl.handle.net/2003/30125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amin, Jakia. “Regulation of autophagy in mammalian cells by stress activated signaling pathways.” 2013. Thesis, Technische Universität Dortmund. Accessed April 19, 2019. http://hdl.handle.net/2003/30125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amin, Jakia. “Regulation of autophagy in mammalian cells by stress activated signaling pathways.” 2013. Web. 19 Apr 2019.

Vancouver:

Amin J. Regulation of autophagy in mammalian cells by stress activated signaling pathways. [Internet] [Thesis]. Technische Universität Dortmund; 2013. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/2003/30125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amin J. Regulation of autophagy in mammalian cells by stress activated signaling pathways. [Thesis]. Technische Universität Dortmund; 2013. Available from: http://hdl.handle.net/2003/30125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

7. Gaffey, Kate. Glucocorticoid resistance in COPD : the role of p38 MAPK.

Degree: PhD, 2013, University of Manchester

 Chronic Obstructive Pulmonary Disease (COPD) is a chronic, inflammatory condition, characterised by airflow limitation. The use of glucocorticoids (GC) as an anti-inflammatory treatment in COPD… (more)

Subjects/Keywords: 616.24; COPD; inflammation; glucocorticoid resistance; p38 MAPK

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APA (6th Edition):

Gaffey, K. (2013). Glucocorticoid resistance in COPD : the role of p38 MAPK. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/glucocorticoid-resistance-in-copd-the-role-of-p38-mapk(9c60954b-f891-4f8b-8004-825e6d173503).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574385

Chicago Manual of Style (16th Edition):

Gaffey, Kate. “Glucocorticoid resistance in COPD : the role of p38 MAPK.” 2013. Doctoral Dissertation, University of Manchester. Accessed April 19, 2019. https://www.research.manchester.ac.uk/portal/en/theses/glucocorticoid-resistance-in-copd-the-role-of-p38-mapk(9c60954b-f891-4f8b-8004-825e6d173503).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574385.

MLA Handbook (7th Edition):

Gaffey, Kate. “Glucocorticoid resistance in COPD : the role of p38 MAPK.” 2013. Web. 19 Apr 2019.

Vancouver:

Gaffey K. Glucocorticoid resistance in COPD : the role of p38 MAPK. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Apr 19]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/glucocorticoid-resistance-in-copd-the-role-of-p38-mapk(9c60954b-f891-4f8b-8004-825e6d173503).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574385.

Council of Science Editors:

Gaffey K. Glucocorticoid resistance in COPD : the role of p38 MAPK. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/glucocorticoid-resistance-in-copd-the-role-of-p38-mapk(9c60954b-f891-4f8b-8004-825e6d173503).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.574385


Université de Sherbrooke

8. Emond-Boisjoly, Marc-Alexandre. Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes .

Degree: 2016, Université de Sherbrooke

 Résumé: L’autophagie est un processus essentiel au maintien de l’homéostasie cellulaire. Elle permet de dégrader et recycler aussi bien des organelles entières que des composants… (more)

Subjects/Keywords: Autophagie; ERK; DUSP; Cardiomyocyte; Autophagy; p38; Starvation

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APA (6th Edition):

Emond-Boisjoly, M. (2016). Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/8908

Chicago Manual of Style (16th Edition):

Emond-Boisjoly, Marc-Alexandre. “Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes .” 2016. Masters Thesis, Université de Sherbrooke. Accessed April 19, 2019. http://hdl.handle.net/11143/8908.

MLA Handbook (7th Edition):

Emond-Boisjoly, Marc-Alexandre. “Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes .” 2016. Web. 19 Apr 2019.

Vancouver:

Emond-Boisjoly M. Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes . [Internet] [Masters thesis]. Université de Sherbrooke; 2016. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/11143/8908.

Council of Science Editors:

Emond-Boisjoly M. Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes . [Masters Thesis]. Université de Sherbrooke; 2016. Available from: http://hdl.handle.net/11143/8908


Université de Sherbrooke

9. Plamondon, Philippe. La MAP kinase p38γ influence la structure des cardiomyocytes .

Degree: 2014, Université de Sherbrooke

 Le cœur est un organe central au fonctionnement du système cardiovasculaire. Il est physiologiquement compartimenté et est constitué de cellules spécialisées qui régulent les impulsions… (more)

Subjects/Keywords: MAP kinase; p38; Cardiomyocyte; Localisation; Domaine PDZ

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APA (6th Edition):

Plamondon, P. (2014). La MAP kinase p38γ influence la structure des cardiomyocytes . (Masters Thesis). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/5307

Chicago Manual of Style (16th Edition):

Plamondon, Philippe. “La MAP kinase p38γ influence la structure des cardiomyocytes .” 2014. Masters Thesis, Université de Sherbrooke. Accessed April 19, 2019. http://savoirs.usherbrooke.ca/handle/11143/5307.

MLA Handbook (7th Edition):

Plamondon, Philippe. “La MAP kinase p38γ influence la structure des cardiomyocytes .” 2014. Web. 19 Apr 2019.

Vancouver:

Plamondon P. La MAP kinase p38γ influence la structure des cardiomyocytes . [Internet] [Masters thesis]. Université de Sherbrooke; 2014. [cited 2019 Apr 19]. Available from: http://savoirs.usherbrooke.ca/handle/11143/5307.

Council of Science Editors:

Plamondon P. La MAP kinase p38γ influence la structure des cardiomyocytes . [Masters Thesis]. Université de Sherbrooke; 2014. Available from: http://savoirs.usherbrooke.ca/handle/11143/5307


Université Laval

10. Kadiri, Maleck. Rôle des MAPK dans la migration des lymphocytes Th17 à travers le collagène tridimensionnel (3D).

Degree: 2017, Université Laval

 La migration des lymphocytes T à travers la matrice extracellulaire (MEC) est un évènement clé dans la réponse immune adaptative et dans les maladies inflammatoires.… (more)

Subjects/Keywords: MAP kinases p38; Lymphocytes T; Collagène

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APA (6th Edition):

Kadiri, M. (2017). Rôle des MAPK dans la migration des lymphocytes Th17 à travers le collagène tridimensionnel (3D). (Thesis). Université Laval. Retrieved from http://hdl.handle.net/20.500.11794/27767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kadiri, Maleck. “Rôle des MAPK dans la migration des lymphocytes Th17 à travers le collagène tridimensionnel (3D).” 2017. Thesis, Université Laval. Accessed April 19, 2019. http://hdl.handle.net/20.500.11794/27767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kadiri, Maleck. “Rôle des MAPK dans la migration des lymphocytes Th17 à travers le collagène tridimensionnel (3D).” 2017. Web. 19 Apr 2019.

Vancouver:

Kadiri M. Rôle des MAPK dans la migration des lymphocytes Th17 à travers le collagène tridimensionnel (3D). [Internet] [Thesis]. Université Laval; 2017. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/20.500.11794/27767.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kadiri M. Rôle des MAPK dans la migration des lymphocytes Th17 à travers le collagène tridimensionnel (3D). [Thesis]. Université Laval; 2017. Available from: http://hdl.handle.net/20.500.11794/27767

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

11. Ewen, Katherine. Large-scale proteomic screen and signal transduction analysis used to study embryonic gonad and germ cell development.

Degree: PhD, 2010, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

In the fields of sex determination, embryonic gonad development and germ cell differentiation, much effort has been placed… (more)

Subjects/Keywords: gonad; germ cell; proteomic; p38 MAPK

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APA (6th Edition):

Ewen, K. (2010). Large-scale proteomic screen and signal transduction analysis used to study embryonic gonad and germ cell development. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/807499

Chicago Manual of Style (16th Edition):

Ewen, Katherine. “Large-scale proteomic screen and signal transduction analysis used to study embryonic gonad and germ cell development.” 2010. Doctoral Dissertation, University of Newcastle. Accessed April 19, 2019. http://hdl.handle.net/1959.13/807499.

MLA Handbook (7th Edition):

Ewen, Katherine. “Large-scale proteomic screen and signal transduction analysis used to study embryonic gonad and germ cell development.” 2010. Web. 19 Apr 2019.

Vancouver:

Ewen K. Large-scale proteomic screen and signal transduction analysis used to study embryonic gonad and germ cell development. [Internet] [Doctoral dissertation]. University of Newcastle; 2010. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1959.13/807499.

Council of Science Editors:

Ewen K. Large-scale proteomic screen and signal transduction analysis used to study embryonic gonad and germ cell development. [Doctoral Dissertation]. University of Newcastle; 2010. Available from: http://hdl.handle.net/1959.13/807499


University of Vermont

12. Bauman, Brittany Nicole. Role of p38 MAPK inhibitor in conditioned fear.

Degree: Biology, 2015, University of Vermont

 <h1>p38 mitogen activated protein kinase (p38) is a kinase that has been implicated in cellular plasticity, stress, and psychiatric disorders and recently in the process… (more)

Subjects/Keywords: p38; conditioned fear; GSK3; inhibition; freezing

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APA (6th Edition):

Bauman, B. N. (2015). Role of p38 MAPK inhibitor in conditioned fear. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/186

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bauman, Brittany Nicole. “Role of p38 MAPK inhibitor in conditioned fear.” 2015. Thesis, University of Vermont. Accessed April 19, 2019. https://scholarworks.uvm.edu/hcoltheses/186.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bauman, Brittany Nicole. “Role of p38 MAPK inhibitor in conditioned fear.” 2015. Web. 19 Apr 2019.

Vancouver:

Bauman BN. Role of p38 MAPK inhibitor in conditioned fear. [Internet] [Thesis]. University of Vermont; 2015. [cited 2019 Apr 19]. Available from: https://scholarworks.uvm.edu/hcoltheses/186.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bauman BN. Role of p38 MAPK inhibitor in conditioned fear. [Thesis]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/hcoltheses/186

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

13. Wissing, Erin R. Uncovering the complexity of muscular dystrophy pathology through disease signaling.

Degree: PhD, Medicine: Molecular Genetics, Biochemistry, and Microbiology, 2014, University of Cincinnati

 The muscular dystrophies (MD) are a diverse group of inherited disorders best characterized by muscle wasting. Progressive weakening and wasting of skeletal muscle leads to… (more)

Subjects/Keywords: Molecular Biology; p38 MAPK; Muscular dystrophy; cell death; Debio 025; p38 inhibitor; ERK1 2

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APA (6th Edition):

Wissing, E. R. (2014). Uncovering the complexity of muscular dystrophy pathology through disease signaling. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393236115

Chicago Manual of Style (16th Edition):

Wissing, Erin R. “Uncovering the complexity of muscular dystrophy pathology through disease signaling.” 2014. Doctoral Dissertation, University of Cincinnati. Accessed April 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393236115.

MLA Handbook (7th Edition):

Wissing, Erin R. “Uncovering the complexity of muscular dystrophy pathology through disease signaling.” 2014. Web. 19 Apr 2019.

Vancouver:

Wissing ER. Uncovering the complexity of muscular dystrophy pathology through disease signaling. [Internet] [Doctoral dissertation]. University of Cincinnati; 2014. [cited 2019 Apr 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393236115.

Council of Science Editors:

Wissing ER. Uncovering the complexity of muscular dystrophy pathology through disease signaling. [Doctoral Dissertation]. University of Cincinnati; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1393236115


Université Paris-Sud – Paris XI

14. Desterke, Christophe. Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive : Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive.

Degree: Docteur es, Médecine, 2011, Université Paris-Sud – Paris XI

 La myélofibrose primitive (MFP) est un néoplasme myéloprolifératif (NMP) chronique BCR-ABL1-négatif associant une dérégulation de l’hématopoïèse (myéloprolifération, dysmégacaryopoïèse et migration des cellules souches et progéniteurs… (more)

Subjects/Keywords: Myélofibrose primitive; Mégacaryocyte; Flt3; MAPK p38; Inflammation; Primary myelofibrosis; Megakaryocyte; Flt3; P38 MAPKs; Inflammatory process

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APA (6th Edition):

Desterke, C. (2011). Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive : Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA11T020

Chicago Manual of Style (16th Edition):

Desterke, Christophe. “Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive : Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 19, 2019. http://www.theses.fr/2011PA11T020.

MLA Handbook (7th Edition):

Desterke, Christophe. “Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive : Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive.” 2011. Web. 19 Apr 2019.

Vancouver:

Desterke C. Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive : Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2011PA11T020.

Council of Science Editors:

Desterke C. Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive : Rôle du couple Flt3-ligand/Flt3 et de l'activation des "Mitogen-activated protein kinases" p38 dans la dysmégacaryopoïèse des patients atteints de myélofibrose primitive. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA11T020


Université Montpellier II

15. Paillas, Salomé. Étude des mécanismes de résistance à l’Irinotécan dans le cancer colorectal : implication de la MAPK p38 : Study of the resistance's mechanisms to Irinotecan in colorectal cancer : p38 MAPK's involvement.

Degree: Docteur es, Biologie Santé, 2011, Université Montpellier II

Malgré les récentes avancées réalisées dans le traitement du cancer du côlon, la résistance des tumeurs est une cause fréquente de l'échec des chimiothérapies. Cette… (more)

Subjects/Keywords: MAPK p38; Résistance; Autophagie; Cancer; Irinotécan; P38 MAPK; Resistance; Autophagy; Cancer; Irinotecan

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paillas, S. (2011). Étude des mécanismes de résistance à l’Irinotécan dans le cancer colorectal : implication de la MAPK p38 : Study of the resistance's mechanisms to Irinotecan in colorectal cancer : p38 MAPK's involvement. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2011MON20058

Chicago Manual of Style (16th Edition):

Paillas, Salomé. “Étude des mécanismes de résistance à l’Irinotécan dans le cancer colorectal : implication de la MAPK p38 : Study of the resistance's mechanisms to Irinotecan in colorectal cancer : p38 MAPK's involvement.” 2011. Doctoral Dissertation, Université Montpellier II. Accessed April 19, 2019. http://www.theses.fr/2011MON20058.

MLA Handbook (7th Edition):

Paillas, Salomé. “Étude des mécanismes de résistance à l’Irinotécan dans le cancer colorectal : implication de la MAPK p38 : Study of the resistance's mechanisms to Irinotecan in colorectal cancer : p38 MAPK's involvement.” 2011. Web. 19 Apr 2019.

Vancouver:

Paillas S. Étude des mécanismes de résistance à l’Irinotécan dans le cancer colorectal : implication de la MAPK p38 : Study of the resistance's mechanisms to Irinotecan in colorectal cancer : p38 MAPK's involvement. [Internet] [Doctoral dissertation]. Université Montpellier II; 2011. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2011MON20058.

Council of Science Editors:

Paillas S. Étude des mécanismes de résistance à l’Irinotécan dans le cancer colorectal : implication de la MAPK p38 : Study of the resistance's mechanisms to Irinotecan in colorectal cancer : p38 MAPK's involvement. [Doctoral Dissertation]. Université Montpellier II; 2011. Available from: http://www.theses.fr/2011MON20058

16. Boucherit, Nicolas. Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii : Interference of the phagosomal conversion by Coxiella burnetii.

Degree: Docteur es, Pathologie humaine. Maladies infectieuses, 2013, Aix Marseille Université

Pour survivre et se multiplier dans leurs cellules hôtes les bactéries intracellulaires ont élaboré divers mécanismes d'échappement à la réponse immunitaire. L’altération du trafic intracellulaire… (more)

Subjects/Keywords: Coxiella burnetii; Phagosome; Lypopolysaccharide; P38; VPS41; Endosome; Coxiella burnetii; Phagosome; Lypopolysaccharide; P38; VPS41; Endosome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Boucherit, N. (2013). Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii : Interference of the phagosomal conversion by Coxiella burnetii. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2013AIXM5061

Chicago Manual of Style (16th Edition):

Boucherit, Nicolas. “Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii : Interference of the phagosomal conversion by Coxiella burnetii.” 2013. Doctoral Dissertation, Aix Marseille Université. Accessed April 19, 2019. http://www.theses.fr/2013AIXM5061.

MLA Handbook (7th Edition):

Boucherit, Nicolas. “Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii : Interference of the phagosomal conversion by Coxiella burnetii.” 2013. Web. 19 Apr 2019.

Vancouver:

Boucherit N. Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii : Interference of the phagosomal conversion by Coxiella burnetii. [Internet] [Doctoral dissertation]. Aix Marseille Université 2013. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2013AIXM5061.

Council of Science Editors:

Boucherit N. Mécanisme d'interférence de la conversion du phagosome par Coxiella burnetii : Interference of the phagosomal conversion by Coxiella burnetii. [Doctoral Dissertation]. Aix Marseille Université 2013. Available from: http://www.theses.fr/2013AIXM5061


Universitat Rovira i Virgili

17. Patraca Fierro, José Iván. Papel de factores endocrinos como melatonina y prolactina, y de adipoquinas como leptina en la modulación de la inflamación asociada a procesos neurodegenerativos.

Degree: Departament de Bioquímica i Biotecnologia, 2015, Universitat Rovira i Virgili

 Glia is the group most abundant cells in the brain nervous system, for a long time it was considered only as an element of support… (more)

Subjects/Keywords: Leptina, Prolactina; cèl·lules glials; iNOS, p38-MAPK; leptina, Prolactina; celulas gliales; iNOS, p38-MAPK; Leptin, Prolactin; Glial cells; iNOS, MAPK-p38; 576; 577; 615; 616.8

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APA (6th Edition):

Patraca Fierro, J. I. (2015). Papel de factores endocrinos como melatonina y prolactina, y de adipoquinas como leptina en la modulación de la inflamación asociada a procesos neurodegenerativos. (Thesis). Universitat Rovira i Virgili. Retrieved from http://hdl.handle.net/10803/348868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patraca Fierro, José Iván. “Papel de factores endocrinos como melatonina y prolactina, y de adipoquinas como leptina en la modulación de la inflamación asociada a procesos neurodegenerativos.” 2015. Thesis, Universitat Rovira i Virgili. Accessed April 19, 2019. http://hdl.handle.net/10803/348868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patraca Fierro, José Iván. “Papel de factores endocrinos como melatonina y prolactina, y de adipoquinas como leptina en la modulación de la inflamación asociada a procesos neurodegenerativos.” 2015. Web. 19 Apr 2019.

Vancouver:

Patraca Fierro JI. Papel de factores endocrinos como melatonina y prolactina, y de adipoquinas como leptina en la modulación de la inflamación asociada a procesos neurodegenerativos. [Internet] [Thesis]. Universitat Rovira i Virgili; 2015. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/10803/348868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patraca Fierro JI. Papel de factores endocrinos como melatonina y prolactina, y de adipoquinas como leptina en la modulación de la inflamación asociada a procesos neurodegenerativos. [Thesis]. Universitat Rovira i Virgili; 2015. Available from: http://hdl.handle.net/10803/348868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Al Mohamad, Hazar. Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat.

Degree: Docteur es, Immunologie, oncologie et infectiologie, 2016, Limoges

Les inhibiteurs des histones désacétylase (HDACi) constituent une classe prometteuse de médicaments anticancéreux. Ils peuvent déclencher la voie apoptotique et sont proposés pour le traitement… (more)

Subjects/Keywords: EBV; Réactivation; Apoptose; Vorinostat; MAP kinase p38; Lymphocytes B; EBV; Reactivation; Apoptosis; Vorinostat; P38 MAP Kinase; B lymphocytes; 615.37

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APA (6th Edition):

Al Mohamad, H. (2016). Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2016LIMO0014

Chicago Manual of Style (16th Edition):

Al Mohamad, Hazar. “Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat.” 2016. Doctoral Dissertation, Limoges. Accessed April 19, 2019. http://www.theses.fr/2016LIMO0014.

MLA Handbook (7th Edition):

Al Mohamad, Hazar. “Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat.” 2016. Web. 19 Apr 2019.

Vancouver:

Al Mohamad H. Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat. [Internet] [Doctoral dissertation]. Limoges; 2016. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2016LIMO0014.

Council of Science Editors:

Al Mohamad H. Etude de la balance réactivation/apoptose des cellules B infectées par l' EBV suite au traitement par un HDACi, le vorinostat : Study of the balance reactivation / apoptosis of B cells infected with EBV following treatment with a HDACi, vorinostat. [Doctoral Dissertation]. Limoges; 2016. Available from: http://www.theses.fr/2016LIMO0014

19. Pinsetta, Flávio Roberto. Quinase p38 alfa como alvo para o planejamento de fármacos em Mal de Alzheimer.

Degree: PhD, Física Biológica, 2013, University of São Paulo

O Mal de Alzheimer (MA) foi caracterizado pela primeira vez em 1907 pelo neuropatologista alemão Alois Alzheimer, tendo como sintomas clínicos disfunções cognitivas, fisiológicas, comportamentais,… (more)

Subjects/Keywords: Alzheimer's Disease; Mal de Alzheimer; p38 alpha MAPK and drug design; Quinase p38 alfa e Modelagem molecular

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APA (6th Edition):

Pinsetta, F. R. (2013). Quinase p38 alfa como alvo para o planejamento de fármacos em Mal de Alzheimer. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/60/60136/tde-06092013-104102/ ;

Chicago Manual of Style (16th Edition):

Pinsetta, Flávio Roberto. “Quinase p38 alfa como alvo para o planejamento de fármacos em Mal de Alzheimer.” 2013. Doctoral Dissertation, University of São Paulo. Accessed April 19, 2019. http://www.teses.usp.br/teses/disponiveis/60/60136/tde-06092013-104102/ ;.

MLA Handbook (7th Edition):

Pinsetta, Flávio Roberto. “Quinase p38 alfa como alvo para o planejamento de fármacos em Mal de Alzheimer.” 2013. Web. 19 Apr 2019.

Vancouver:

Pinsetta FR. Quinase p38 alfa como alvo para o planejamento de fármacos em Mal de Alzheimer. [Internet] [Doctoral dissertation]. University of São Paulo; 2013. [cited 2019 Apr 19]. Available from: http://www.teses.usp.br/teses/disponiveis/60/60136/tde-06092013-104102/ ;.

Council of Science Editors:

Pinsetta FR. Quinase p38 alfa como alvo para o planejamento de fármacos em Mal de Alzheimer. [Doctoral Dissertation]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/60/60136/tde-06092013-104102/ ;


Université Paris-Sud – Paris XI

20. Selimoglu, Rasim. A Global Approach of Ral Pathway : Identification of a New Actor : Stk38 : Une approche globale de la Voie Ral : identification d'un nouvel acteur : Stk38.

Degree: Docteur es, Génétique et biologie cellulaire des cancers, 2011, Université Paris-Sud – Paris XI

Les GTPases Ral, RalA et RalB, sont des effecteurs proximaux de l’oncogène Ras.Malgré leur forte homologie, leurs activateurs communs (les RalGEFs) et des effecteurs communs… (more)

Subjects/Keywords: Ral GTPases; Stk38; Map4k4; Cytocinèse; Migration cellulaire; Activation de P38; Evolution; Ral GTPases; Stk38; Map4k4; Cytokinesis; Cell migration; P38 activation; Evolution

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APA (6th Edition):

Selimoglu, R. (2011). A Global Approach of Ral Pathway : Identification of a New Actor : Stk38 : Une approche globale de la Voie Ral : identification d'un nouvel acteur : Stk38. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA11T041

Chicago Manual of Style (16th Edition):

Selimoglu, Rasim. “A Global Approach of Ral Pathway : Identification of a New Actor : Stk38 : Une approche globale de la Voie Ral : identification d'un nouvel acteur : Stk38.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 19, 2019. http://www.theses.fr/2011PA11T041.

MLA Handbook (7th Edition):

Selimoglu, Rasim. “A Global Approach of Ral Pathway : Identification of a New Actor : Stk38 : Une approche globale de la Voie Ral : identification d'un nouvel acteur : Stk38.” 2011. Web. 19 Apr 2019.

Vancouver:

Selimoglu R. A Global Approach of Ral Pathway : Identification of a New Actor : Stk38 : Une approche globale de la Voie Ral : identification d'un nouvel acteur : Stk38. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2011PA11T041.

Council of Science Editors:

Selimoglu R. A Global Approach of Ral Pathway : Identification of a New Actor : Stk38 : Une approche globale de la Voie Ral : identification d'un nouvel acteur : Stk38. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA11T041

21. Rahal, Pamela. Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites.

Degree: Docteur es, Sciences de la Vie, 2015, Lyon, École normale supérieure

MLK3 est une ser/thr MAP3K qui active la voie de signalisation des MAPKs dans différents types cellulaires. GSK3β interagit et active MLK3 en la phosphorylant… (more)

Subjects/Keywords: GSK3; MLK3; P38 MAPK; Régénération musculaire; Cellules satellites; Pax7; GSK3; MLK3; P38 MAPK; Muscle regeneration; Satellite cells; Pax7

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APA (6th Edition):

Rahal, P. (2015). Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites. (Doctoral Dissertation). Lyon, École normale supérieure. Retrieved from http://www.theses.fr/2015ENSL1000

Chicago Manual of Style (16th Edition):

Rahal, Pamela. “Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites.” 2015. Doctoral Dissertation, Lyon, École normale supérieure. Accessed April 19, 2019. http://www.theses.fr/2015ENSL1000.

MLA Handbook (7th Edition):

Rahal, Pamela. “Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites.” 2015. Web. 19 Apr 2019.

Vancouver:

Rahal P. Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites. [Internet] [Doctoral dissertation]. Lyon, École normale supérieure; 2015. [cited 2019 Apr 19]. Available from: http://www.theses.fr/2015ENSL1000.

Council of Science Editors:

Rahal P. Role of GSK3β - MLK3 - p38γ MAPK Signalling in Satellite Cell Proliferation Regulation : Le rôle de la voie de signalisation GSK3β-MLK3-p38γ MAPK dans la régulation de la prolifération des cellules satellites. [Doctoral Dissertation]. Lyon, École normale supérieure; 2015. Available from: http://www.theses.fr/2015ENSL1000


Dalhousie University

22. Jones, Quinton RD. THE EFFECT OF INSULIN ON STRESS-RESPONSE PATHWAYS IN A CELLULAR MODEL OF RAT CARDIOMYOCYTES.

Degree: MS, Department of Anatomy & Neurobiolgy with Neuroscience, 2011, Dalhousie University

Insulin was used on a model on H9c2 myotubes to determine the effect of oxygen-glucose deprivation and reoxygenation on the localization and phosphorylation of Hsp27… (more)

Subjects/Keywords: insulin h9c2 cardiomyocytes OGD anoxia ischemia hsp27 p38 mapk

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APA (6th Edition):

Jones, Q. R. (2011). THE EFFECT OF INSULIN ON STRESS-RESPONSE PATHWAYS IN A CELLULAR MODEL OF RAT CARDIOMYOCYTES. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14058

Chicago Manual of Style (16th Edition):

Jones, Quinton RD. “THE EFFECT OF INSULIN ON STRESS-RESPONSE PATHWAYS IN A CELLULAR MODEL OF RAT CARDIOMYOCYTES.” 2011. Masters Thesis, Dalhousie University. Accessed April 19, 2019. http://hdl.handle.net/10222/14058.

MLA Handbook (7th Edition):

Jones, Quinton RD. “THE EFFECT OF INSULIN ON STRESS-RESPONSE PATHWAYS IN A CELLULAR MODEL OF RAT CARDIOMYOCYTES.” 2011. Web. 19 Apr 2019.

Vancouver:

Jones QR. THE EFFECT OF INSULIN ON STRESS-RESPONSE PATHWAYS IN A CELLULAR MODEL OF RAT CARDIOMYOCYTES. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/10222/14058.

Council of Science Editors:

Jones QR. THE EFFECT OF INSULIN ON STRESS-RESPONSE PATHWAYS IN A CELLULAR MODEL OF RAT CARDIOMYOCYTES. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14058


University of Vermont

23. Hare, Brendan Deegan. Glycogen Synthase Kinase-3β: An Investigation Of The Novel Serine 389 Phosphorylation Site.

Degree: PhD, Psychology, 2015, University of Vermont

  Stress associated psychiatric disorders such as depression, anxiety, and post-traumatic stress disorder affect a large proportion of the population. Reductions in the complexity of… (more)

Subjects/Keywords: Anxiety; Depression; Exercise; Glycogen Synthase Kinase; p38 MAPK; Stress; Neurosciences; Psychology

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APA (6th Edition):

Hare, B. D. (2015). Glycogen Synthase Kinase-3β: An Investigation Of The Novel Serine 389 Phosphorylation Site. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/357

Chicago Manual of Style (16th Edition):

Hare, Brendan Deegan. “Glycogen Synthase Kinase-3β: An Investigation Of The Novel Serine 389 Phosphorylation Site.” 2015. Doctoral Dissertation, University of Vermont. Accessed April 19, 2019. https://scholarworks.uvm.edu/graddis/357.

MLA Handbook (7th Edition):

Hare, Brendan Deegan. “Glycogen Synthase Kinase-3β: An Investigation Of The Novel Serine 389 Phosphorylation Site.” 2015. Web. 19 Apr 2019.

Vancouver:

Hare BD. Glycogen Synthase Kinase-3β: An Investigation Of The Novel Serine 389 Phosphorylation Site. [Internet] [Doctoral dissertation]. University of Vermont; 2015. [cited 2019 Apr 19]. Available from: https://scholarworks.uvm.edu/graddis/357.

Council of Science Editors:

Hare BD. Glycogen Synthase Kinase-3β: An Investigation Of The Novel Serine 389 Phosphorylation Site. [Doctoral Dissertation]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/graddis/357

24. Bakarozi, Maria. Ο ρόλος των ενδοκυττάριων σηματοδοτικών μηχανισμών σε ασθενείς με ιογενείς ηπατίτιδες.

Degree: 2015, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

Innate and adaptive immune responses against HBV have been shown to play a pivotal role in the control of viral replication as well as in… (more)

Subjects/Keywords: Ιογενείς ηπατίτιδες; Ενδοκυττάριοι σηματοδοτικοί μηχανισμοί; Chronic viral hepatitis; p38 MAPK

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APA (6th Edition):

Bakarozi, M. (2015). Ο ρόλος των ενδοκυττάριων σηματοδοτικών μηχανισμών σε ασθενείς με ιογενείς ηπατίτιδες. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/35779

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bakarozi, Maria. “Ο ρόλος των ενδοκυττάριων σηματοδοτικών μηχανισμών σε ασθενείς με ιογενείς ηπατίτιδες.” 2015. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed April 19, 2019. http://hdl.handle.net/10442/hedi/35779.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bakarozi, Maria. “Ο ρόλος των ενδοκυττάριων σηματοδοτικών μηχανισμών σε ασθενείς με ιογενείς ηπατίτιδες.” 2015. Web. 19 Apr 2019.

Vancouver:

Bakarozi M. Ο ρόλος των ενδοκυττάριων σηματοδοτικών μηχανισμών σε ασθενείς με ιογενείς ηπατίτιδες. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2015. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/10442/hedi/35779.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bakarozi M. Ο ρόλος των ενδοκυττάριων σηματοδοτικών μηχανισμών σε ασθενείς με ιογενείς ηπατίτιδες. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2015. Available from: http://hdl.handle.net/10442/hedi/35779

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

25. Hsu, Chia. Substrate-level control of glucose metabolism in C2C12 myotubes.

Degree: PhD, Applied Physiology, 2016, Georgia Tech

 Metabolic flexibility is critical for muscle to maintain proper function and overall health. Muscle adapts to metabolic stress with increasing ATP synthesis by enhancing the… (more)

Subjects/Keywords: AMP; AMPK; NAD; NADH; p38; Glucose metabolism; SIRT1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hsu, C. (2016). Substrate-level control of glucose metabolism in C2C12 myotubes. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/58176

Chicago Manual of Style (16th Edition):

Hsu, Chia. “Substrate-level control of glucose metabolism in C2C12 myotubes.” 2016. Doctoral Dissertation, Georgia Tech. Accessed April 19, 2019. http://hdl.handle.net/1853/58176.

MLA Handbook (7th Edition):

Hsu, Chia. “Substrate-level control of glucose metabolism in C2C12 myotubes.” 2016. Web. 19 Apr 2019.

Vancouver:

Hsu C. Substrate-level control of glucose metabolism in C2C12 myotubes. [Internet] [Doctoral dissertation]. Georgia Tech; 2016. [cited 2019 Apr 19]. Available from: http://hdl.handle.net/1853/58176.

Council of Science Editors:

Hsu C. Substrate-level control of glucose metabolism in C2C12 myotubes. [Doctoral Dissertation]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/58176


University of Toledo Health Science Campus

26. Zhang, Nan. Identification of Receptors and Signaling Pathways Involved in Borrelia burgdorferi-Elicited IL-10 and Potential Therapies for Lyme disease.

Degree: PhD, Biomedical Sciences (Infection, Immunity, and Transplantation), 2014, University of Toledo Health Science Campus

 B. burgdorferi (Bb) is an extracellular spirochetal bacterium that causes Lyme disease, a significant vector-borne illness in North America and Eurasia. Bb is transmitted into… (more)

Subjects/Keywords: Biomedical Research; Immunology; Borrelia burgdorferi, IL-10, p38 MAPK, neutrophil

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APA (6th Edition):

Zhang, N. (2014). Identification of Receptors and Signaling Pathways Involved in Borrelia burgdorferi-Elicited IL-10 and Potential Therapies for Lyme disease. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1405591577

Chicago Manual of Style (16th Edition):

Zhang, Nan. “Identification of Receptors and Signaling Pathways Involved in Borrelia burgdorferi-Elicited IL-10 and Potential Therapies for Lyme disease.” 2014. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed April 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1405591577.

MLA Handbook (7th Edition):

Zhang, Nan. “Identification of Receptors and Signaling Pathways Involved in Borrelia burgdorferi-Elicited IL-10 and Potential Therapies for Lyme disease.” 2014. Web. 19 Apr 2019.

Vancouver:

Zhang N. Identification of Receptors and Signaling Pathways Involved in Borrelia burgdorferi-Elicited IL-10 and Potential Therapies for Lyme disease. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2014. [cited 2019 Apr 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1405591577.

Council of Science Editors:

Zhang N. Identification of Receptors and Signaling Pathways Involved in Borrelia burgdorferi-Elicited IL-10 and Potential Therapies for Lyme disease. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1405591577


RMIT University

27. Muthusamy, V. The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells.

Degree: 2010, RMIT University

 According to the WHO, three million non-melanoma skin cancers (NMSC) occur annually with one in every three cancers diagnosed is a skin cancer. In most… (more)

Subjects/Keywords: Fields of Research; Ultraviolet radiation; p38 MAPK; JNK; NFkB; TFN[alpha]

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APA (6th Edition):

Muthusamy, V. (2010). The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:6407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muthusamy, V. “The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells.” 2010. Thesis, RMIT University. Accessed April 19, 2019. http://researchbank.rmit.edu.au/view/rmit:6407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muthusamy, V. “The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells.” 2010. Web. 19 Apr 2019.

Vancouver:

Muthusamy V. The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells. [Internet] [Thesis]. RMIT University; 2010. [cited 2019 Apr 19]. Available from: http://researchbank.rmit.edu.au/view/rmit:6407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muthusamy V. The role cell signalling pathways play in tnf[alpha] release from uv-irradiated skin cells. [Thesis]. RMIT University; 2010. Available from: http://researchbank.rmit.edu.au/view/rmit:6407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

28. Chen, Te-Chung. The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

 Arsenic trioxide (ATO), an anti-cancer drug for acute promyelocytic leukemia (APL), was able to induce apoptosis in several cancer cell lines including solid tumors, myeloma… (more)

Subjects/Keywords: ATO; Arsenic trioxide; Helicobacter pylori (Hp); Apoptosis; SUMO; p38

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APA (6th Edition):

Chen, T. (2013). The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Te-Chung. “The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis.” 2013. Thesis, NSYSU. Accessed April 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Te-Chung. “The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis.” 2013. Web. 19 Apr 2019.

Vancouver:

Chen T. The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis. [Internet] [Thesis]. NSYSU; 2013. [cited 2019 Apr 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen T. The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

29. Wang, Pin-Yao. SUMOs mediate the nuclear transfer of p38 and p-p38 during Helicobacter pylori infection.

Degree: PhD, Institute of Biomedical Sciences, 2016, NSYSU

p38 mitogen-activated protein kinase (MAPK) is of the essence in the cellâs response to environmental stresses, cytokines and DNA damage. p38 is distributed both in… (more)

Subjects/Keywords: SUMO-1; SIM; Helicobacter pylori; p38; nuclear translocalization; SUMO-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, P. (2016). SUMOs mediate the nuclear transfer of p38 and p-p38 during Helicobacter pylori infection. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801116-023742

Chicago Manual of Style (16th Edition):

Wang, Pin-Yao. “SUMOs mediate the nuclear transfer of p38 and p-p38 during Helicobacter pylori infection.” 2016. Doctoral Dissertation, NSYSU. Accessed April 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801116-023742.

MLA Handbook (7th Edition):

Wang, Pin-Yao. “SUMOs mediate the nuclear transfer of p38 and p-p38 during Helicobacter pylori infection.” 2016. Web. 19 Apr 2019.

Vancouver:

Wang P. SUMOs mediate the nuclear transfer of p38 and p-p38 during Helicobacter pylori infection. [Internet] [Doctoral dissertation]. NSYSU; 2016. [cited 2019 Apr 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801116-023742.

Council of Science Editors:

Wang P. SUMOs mediate the nuclear transfer of p38 and p-p38 during Helicobacter pylori infection. [Doctoral Dissertation]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801116-023742


NSYSU

30. Lin, Tse-yu. Investigation of the SUMO-interacting motif of p38.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

 SUMOylation is a posttranslational modification by the small ubiquitin-related modifier (SUMO), SUMOylation plays important roles in the regulation of diverse cellular processes. SUMO can interact… (more)

Subjects/Keywords: p38; SUMO-interacting motif; non-covalent interaction; SUMO-1; SUMO-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, T. (2015). Investigation of the SUMO-interacting motif of p38. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Tse-yu. “Investigation of the SUMO-interacting motif of p38.” 2015. Thesis, NSYSU. Accessed April 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Tse-yu. “Investigation of the SUMO-interacting motif of p38.” 2015. Web. 19 Apr 2019.

Vancouver:

Lin T. Investigation of the SUMO-interacting motif of p38. [Internet] [Thesis]. NSYSU; 2015. [cited 2019 Apr 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin T. Investigation of the SUMO-interacting motif of p38. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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