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You searched for subject:(p38 Mitogen Activated Protein Kinases). Showing records 1 – 30 of 22296 total matches.

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Michigan State University

1. Pizzimenti, Natalie Maria. Mitogen activated protein kinase signaling in mouse skeletal muscle.

Degree: 2014, Michigan State University

Thesis M.S. Michigan State University. Physiology - Master of Science 2014.

AMP activated protein kinase (AMPK) and p38 mitogen activated protein kinases (MAPKs) are activated(more)

Subjects/Keywords: Physiology; Mitochondria – Formation; Protein kinases; Mitogen-activated protein kinases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pizzimenti, N. M. (2014). Mitogen activated protein kinase signaling in mouse skeletal muscle. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pizzimenti, Natalie Maria. “Mitogen activated protein kinase signaling in mouse skeletal muscle.” 2014. Thesis, Michigan State University. Accessed March 07, 2021. http://etd.lib.msu.edu/islandora/object/etd:2416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pizzimenti, Natalie Maria. “Mitogen activated protein kinase signaling in mouse skeletal muscle.” 2014. Web. 07 Mar 2021.

Vancouver:

Pizzimenti NM. Mitogen activated protein kinase signaling in mouse skeletal muscle. [Internet] [Thesis]. Michigan State University; 2014. [cited 2021 Mar 07]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pizzimenti NM. Mitogen activated protein kinase signaling in mouse skeletal muscle. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

2. Kaikkonen, L. (Leena). p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function.

Degree: 2014, University of Oulu

Abstract Cardiovascular diseases are the leading causes of death in the developed countries and their incidence is not expected to decrease in the future. There… (more)

Subjects/Keywords: B-type natriuretic peptide; GATA-4 transcription factor; heart failure; mitogen-activated protein kinases; myocardial contraction; p38 mitogen-activated protein kinase; ventricular remodeling; B-tyypin natriureettinen peptidi; mitogeeniaktivoituvat proteiinikinaasit; p38 mitogeeniaktivoituva proteiinikinaasi; sydämen vajaatoiminta; sydänlihaksen supistuminen; sydänlihaksen uudelleenmuovautuminen; transkriptiotekijä GATA-4

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APA (6th Edition):

Kaikkonen, L. (. (2014). p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526205038

Chicago Manual of Style (16th Edition):

Kaikkonen, L (Leena). “p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function.” 2014. Doctoral Dissertation, University of Oulu. Accessed March 07, 2021. http://urn.fi/urn:isbn:9789526205038.

MLA Handbook (7th Edition):

Kaikkonen, L (Leena). “p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function.” 2014. Web. 07 Mar 2021.

Vancouver:

Kaikkonen L(. p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function. [Internet] [Doctoral dissertation]. University of Oulu; 2014. [cited 2021 Mar 07]. Available from: http://urn.fi/urn:isbn:9789526205038.

Council of Science Editors:

Kaikkonen L(. p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function. [Doctoral Dissertation]. University of Oulu; 2014. Available from: http://urn.fi/urn:isbn:9789526205038

3. Francis, Dana May. The Molecular Basis of Mitogen-Activated Protein Kinase (MAPK) Regulation by Kinases and Phosphatases.

Degree: PhD, Molecular Pharmacology, Physiology, and Biotechnology, 2013, Brown University

Mitogen-activated protein kinases direct the cellular response to environmental and developmental signals. MAPKs are activated through kinase modules resulting in phosphorylation of the MAPK on… (more)

Subjects/Keywords: mitogen-activated protein kinase; p38; protein tyrosine phosphatase; binding specificity; NMR spectroscopy; ITC; SAXS

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APA (6th Edition):

Francis, D. M. (2013). The Molecular Basis of Mitogen-Activated Protein Kinase (MAPK) Regulation by Kinases and Phosphatases. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:320583/

Chicago Manual of Style (16th Edition):

Francis, Dana May. “The Molecular Basis of Mitogen-Activated Protein Kinase (MAPK) Regulation by Kinases and Phosphatases.” 2013. Doctoral Dissertation, Brown University. Accessed March 07, 2021. https://repository.library.brown.edu/studio/item/bdr:320583/.

MLA Handbook (7th Edition):

Francis, Dana May. “The Molecular Basis of Mitogen-Activated Protein Kinase (MAPK) Regulation by Kinases and Phosphatases.” 2013. Web. 07 Mar 2021.

Vancouver:

Francis DM. The Molecular Basis of Mitogen-Activated Protein Kinase (MAPK) Regulation by Kinases and Phosphatases. [Internet] [Doctoral dissertation]. Brown University; 2013. [cited 2021 Mar 07]. Available from: https://repository.library.brown.edu/studio/item/bdr:320583/.

Council of Science Editors:

Francis DM. The Molecular Basis of Mitogen-Activated Protein Kinase (MAPK) Regulation by Kinases and Phosphatases. [Doctoral Dissertation]. Brown University; 2013. Available from: https://repository.library.brown.edu/studio/item/bdr:320583/


University of Texas Southwestern Medical Center

4. Taylor, Clinton A., IV. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.

Degree: 2016, University of Texas Southwestern Medical Center

Protein-protein interactions are essential for nearly every cellular process. Within signaling pathways, such interactions carry out numerous functions such as defining substrate specificity, inhibition of… (more)

Subjects/Keywords: Mitogen-Activated Protein Kinase Kinases; Protein-Serine-Threonine Kinases; Signal Transduction; Transcription Factors

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APA (6th Edition):

Taylor, Clinton A., I. (2016). Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Web. 07 Mar 2021.

Vancouver:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

5. Rysä, J. (Jaana). Gene expression profiling in experimental models of cardiac load.

Degree: 2008, University of Oulu

 Abstract Cardiac hypertrophy provides an adaptive mechanism to maintain cardiac output in response to increased workload, and although initially beneficial, hypertrophy eventually leads to heart… (more)

Subjects/Keywords: DNA microarrays; diastolic heart failure; hypertrophy; mitogen-activated protein kinases; stretch

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APA (6th Edition):

Rysä, J. (. (2008). Gene expression profiling in experimental models of cardiac load. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514287664

Chicago Manual of Style (16th Edition):

Rysä, J (Jaana). “Gene expression profiling in experimental models of cardiac load.” 2008. Doctoral Dissertation, University of Oulu. Accessed March 07, 2021. http://urn.fi/urn:isbn:9789514287664.

MLA Handbook (7th Edition):

Rysä, J (Jaana). “Gene expression profiling in experimental models of cardiac load.” 2008. Web. 07 Mar 2021.

Vancouver:

Rysä J(. Gene expression profiling in experimental models of cardiac load. [Internet] [Doctoral dissertation]. University of Oulu; 2008. [cited 2021 Mar 07]. Available from: http://urn.fi/urn:isbn:9789514287664.

Council of Science Editors:

Rysä J(. Gene expression profiling in experimental models of cardiac load. [Doctoral Dissertation]. University of Oulu; 2008. Available from: http://urn.fi/urn:isbn:9789514287664


University of Texas Southwestern Medical Center

6. An, Zhenyi. The Regulation of Autophagy and Its Role in Mitotic Exit.

Degree: 2014, University of Texas Southwestern Medical Center

 Autophagy is an evolutionarily conserved pathway in which cells enclose cytoplasmic contents in double membrane vesicles and deliver them to the lysosome for degradation. Autophagy… (more)

Subjects/Keywords: Apoptosis Regulatory Proteins; Autophagy; Mitogen-Activated Protein Kinases; Phosphorylation

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APA (6th Edition):

An, Z. (2014). The Regulation of Autophagy and Its Role in Mitotic Exit. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Web. 07 Mar 2021.

Vancouver:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Brancho, Deborah Marie. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2005, U of Massachusetts : Med

  The c-Jun NH2-terminal kinase (JNK) group and the p38 group of mitogen-activated protein kinases (MAPK) are stress-activated protein kinases that regulate cell proliferation, differentiation,… (more)

Subjects/Keywords: p38 Mitogen-Activated Protein Kinases; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinase Kinases; MAP Kinase Signaling System; Cell Differentiation; Adipocytes; Amino Acids, Peptides, and Proteins; Cells; Enzymes and Coenzymes

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APA (6th Edition):

Brancho, D. M. (2005). Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/101

Chicago Manual of Style (16th Edition):

Brancho, Deborah Marie. “Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.” 2005. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. https://escholarship.umassmed.edu/gsbs_diss/101.

MLA Handbook (7th Edition):

Brancho, Deborah Marie. “Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.” 2005. Web. 07 Mar 2021.

Vancouver:

Brancho DM. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2005. [cited 2021 Mar 07]. Available from: https://escholarship.umassmed.edu/gsbs_diss/101.

Council of Science Editors:

Brancho DM. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2005. Available from: https://escholarship.umassmed.edu/gsbs_diss/101


University of Ottawa

8. Stitt, Erin Maureen. The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease .

Degree: 2011, University of Ottawa

 The incidence of chronic kidney disease (CKD) is increasing. CKD is characterized by a gradual decrease in renal function leading to end stage renal disease… (more)

Subjects/Keywords: Podocyte; Prostaglandin E2; Angiotensin II; Kidney; Chronic kidney disease; p38 Mitogen activated protein kinase

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APA (6th Edition):

Stitt, E. M. (2011). The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/19800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stitt, Erin Maureen. “The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease .” 2011. Thesis, University of Ottawa. Accessed March 07, 2021. http://hdl.handle.net/10393/19800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stitt, Erin Maureen. “The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease .” 2011. Web. 07 Mar 2021.

Vancouver:

Stitt EM. The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10393/19800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stitt EM. The Role of Podocyte Prostaglandin E2 and Angiotensin II Receptors in Glomerular Disease . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/19800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Sama, Reddy Ranjith Kumar. FUS/TLS in Stress Response - Implications for Amyotrophic Lateral Sclerosis: A Dissertation.

Degree: Cell Biology, Neurology, 2014, U of Massachusetts : Med

  Amyotrophic lateral sclerosis (ALS), also known as Lou Gehrig’s disease is a fatal neurodegenerative disease. ALS is typically adult onset and is characterized by… (more)

Subjects/Keywords: Amyotrophic Lateral Sclerosis; DNA Damage; DNA-Binding Proteins; Mutation; RNA-Binding Protein FUS; p38 Mitogen-Activated Protein Kinases; Biochemistry, Biophysics, and Structural Biology; Cellular and Molecular Physiology; Molecular Genetics; Nervous System Diseases

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APA (6th Edition):

Sama, R. R. K. (2014). FUS/TLS in Stress Response - Implications for Amyotrophic Lateral Sclerosis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/704

Chicago Manual of Style (16th Edition):

Sama, Reddy Ranjith Kumar. “FUS/TLS in Stress Response - Implications for Amyotrophic Lateral Sclerosis: A Dissertation.” 2014. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/704.

MLA Handbook (7th Edition):

Sama, Reddy Ranjith Kumar. “FUS/TLS in Stress Response - Implications for Amyotrophic Lateral Sclerosis: A Dissertation.” 2014. Web. 07 Mar 2021.

Vancouver:

Sama RRK. FUS/TLS in Stress Response - Implications for Amyotrophic Lateral Sclerosis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2014. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/704.

Council of Science Editors:

Sama RRK. FUS/TLS in Stress Response - Implications for Amyotrophic Lateral Sclerosis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2014. Available from: http://escholarship.umassmed.edu/gsbs_diss/704


Columbia University

10. Lyashenko, Eugenia. Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway.

Degree: 2015, Columbia University

 Cells process information from their environment, such as the stimuli to grow, divide, or die, via cell signaling. Deregulated processing of extracellular stimuli can lead… (more)

Subjects/Keywords: Mitogen-activated protein kinases; Weber-Fechner law; Cellular control mechanisms; Bioinformatics; Biology

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APA (6th Edition):

Lyashenko, E. (2015). Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8RF5SVZ

Chicago Manual of Style (16th Edition):

Lyashenko, Eugenia. “Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway.” 2015. Doctoral Dissertation, Columbia University. Accessed March 07, 2021. https://doi.org/10.7916/D8RF5SVZ.

MLA Handbook (7th Edition):

Lyashenko, Eugenia. “Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway.” 2015. Web. 07 Mar 2021.

Vancouver:

Lyashenko E. Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2021 Mar 07]. Available from: https://doi.org/10.7916/D8RF5SVZ.

Council of Science Editors:

Lyashenko E. Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8RF5SVZ


University of Georgia

11. Sun, Bin. Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis.

Degree: 2014, University of Georgia

 The goal of this work was to identify mechanisms by which inhibition of calcium-independent phospholipase A2 (iPLA2) induces cytostasis in human LNCaP (p53 wild-type and… (more)

Subjects/Keywords: Phospholipase A2; Phospholipids; p53; Mitogen Activated Protein Kinases; Reactive species; Phospholipid Profiles

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APA (6th Edition):

Sun, B. (2014). Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/26800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sun, Bin. “Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis.” 2014. Thesis, University of Georgia. Accessed March 07, 2021. http://hdl.handle.net/10724/26800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sun, Bin. “Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis.” 2014. Web. 07 Mar 2021.

Vancouver:

Sun B. Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10724/26800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sun B. Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/26800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Karagiannidis, Ioannis. In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης.

Degree: 2016, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The impact of a potential autophagy (LC3a/b) deregulation in hyper and in hypo stages during sepsis-induced kidney injury and the temporal profile of phosphorylated extracellular… (more)

Subjects/Keywords: Απόπτωση (προγραμματισμένος κυτταρικός θάνατος); Αυτοφαγία; Πειραματικά μοντέλα σήψης; Mitogen – Activated Protein Kinases (MAPKs)

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APA (6th Edition):

Karagiannidis, I. (2016). In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/37872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Karagiannidis, Ioannis. “In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης.” 2016. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 07, 2021. http://hdl.handle.net/10442/hedi/37872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Karagiannidis, Ioannis. “In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης.” 2016. Web. 07 Mar 2021.

Vancouver:

Karagiannidis I. In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10442/hedi/37872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Karagiannidis I. In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. Available from: http://hdl.handle.net/10442/hedi/37872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

13. Zi, Min. The transition from cardiac remodelling to heart failure: Using mouse models to explore the underlying mechanisms.

Degree: 2017, University of Manchester

Heart failure (HF) is a complex clinical syndrome, in which the heart is incapable of pumping an adequate amount of blood to meet the body's… (more)

Subjects/Keywords: heart failure; cardiac remodelling; animal models; C57BL/6; mitogen-activated protein kinases; signalling

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APA (6th Edition):

Zi, M. (2017). The transition from cardiac remodelling to heart failure: Using mouse models to explore the underlying mechanisms. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311804

Chicago Manual of Style (16th Edition):

Zi, Min. “The transition from cardiac remodelling to heart failure: Using mouse models to explore the underlying mechanisms.” 2017. Doctoral Dissertation, University of Manchester. Accessed March 07, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311804.

MLA Handbook (7th Edition):

Zi, Min. “The transition from cardiac remodelling to heart failure: Using mouse models to explore the underlying mechanisms.” 2017. Web. 07 Mar 2021.

Vancouver:

Zi M. The transition from cardiac remodelling to heart failure: Using mouse models to explore the underlying mechanisms. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2021 Mar 07]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311804.

Council of Science Editors:

Zi M. The transition from cardiac remodelling to heart failure: Using mouse models to explore the underlying mechanisms. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:311804


Rutgers University

14. Gokina, Pradeepa, 1981-. The role of MAPKs in CNS demyelination.

Degree: Biology, 2013, Rutgers University

Subjects/Keywords: Mitogen-activated protein kinases; Demyelination; Oligodendroglia

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APA (6th Edition):

Gokina, Pradeepa, 1. (2013). The role of MAPKs in CNS demyelination. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/41478/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gokina, Pradeepa, 1981-. “The role of MAPKs in CNS demyelination.” 2013. Thesis, Rutgers University. Accessed March 07, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/41478/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gokina, Pradeepa, 1981-. “The role of MAPKs in CNS demyelination.” 2013. Web. 07 Mar 2021.

Vancouver:

Gokina, Pradeepa 1. The role of MAPKs in CNS demyelination. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 Mar 07]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41478/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gokina, Pradeepa 1. The role of MAPKs in CNS demyelination. [Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41478/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

15. Zi, Min. The transition from cardiac remodelling to heart failure : using mouse models to explore the underlying mechanisms.

Degree: PhD, 2017, University of Manchester

 Heart failure (HF) is a complex clinical syndrome, in which the heart is incapable of pumping an adequate amount of blood to meet the body's… (more)

Subjects/Keywords: mitogen-activated protein kinases; C57BL/6; signalling; cardiac remodelling; heart failure; animal models

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APA (6th Edition):

Zi, M. (2017). The transition from cardiac remodelling to heart failure : using mouse models to explore the underlying mechanisms. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-transition-from-cardiac-remodelling-to-heart-failure-using-mouse-models-to-explore-the-underlying-mechanisms(8f4d4cd5-59cc-4718-a63e-e75aef6438b9).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822933

Chicago Manual of Style (16th Edition):

Zi, Min. “The transition from cardiac remodelling to heart failure : using mouse models to explore the underlying mechanisms.” 2017. Doctoral Dissertation, University of Manchester. Accessed March 07, 2021. https://www.research.manchester.ac.uk/portal/en/theses/the-transition-from-cardiac-remodelling-to-heart-failure-using-mouse-models-to-explore-the-underlying-mechanisms(8f4d4cd5-59cc-4718-a63e-e75aef6438b9).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822933.

MLA Handbook (7th Edition):

Zi, Min. “The transition from cardiac remodelling to heart failure : using mouse models to explore the underlying mechanisms.” 2017. Web. 07 Mar 2021.

Vancouver:

Zi M. The transition from cardiac remodelling to heart failure : using mouse models to explore the underlying mechanisms. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2021 Mar 07]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-transition-from-cardiac-remodelling-to-heart-failure-using-mouse-models-to-explore-the-underlying-mechanisms(8f4d4cd5-59cc-4718-a63e-e75aef6438b9).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822933.

Council of Science Editors:

Zi M. The transition from cardiac remodelling to heart failure : using mouse models to explore the underlying mechanisms. [Doctoral Dissertation]. University of Manchester; 2017. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-transition-from-cardiac-remodelling-to-heart-failure-using-mouse-models-to-explore-the-underlying-mechanisms(8f4d4cd5-59cc-4718-a63e-e75aef6438b9).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.822933

16. Hirata, Junko. Oxidative stress regulates expression of claudin-1 in human RPE cells : 酸化ストレスによる網膜色素上皮細胞内の接着蛋白質claudin-1の発現変化.

Degree: 博士(医学), 2013, Hiroshima University / 広島大学

 Age-related macular degeneration (AMD) is a neurodegenerative disease associated with irreversible loss of central vision in the elderly. Disruption of the homeostatic function of the… (more)

Subjects/Keywords: Age-related macular degeneration; Retinal pigment epithelium; Oxidative stress; Claudin-1; Tight junction; p38; Mitogen-activated protein kinase

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APA (6th Edition):

Hirata, J. (2013). Oxidative stress regulates expression of claudin-1 in human RPE cells : 酸化ストレスによる網膜色素上皮細胞内の接着蛋白質claudin-1の発現変化. (Thesis). Hiroshima University / 広島大学. Retrieved from http://ir.lib.hiroshima-u.ac.jp/00040614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hirata, Junko. “Oxidative stress regulates expression of claudin-1 in human RPE cells : 酸化ストレスによる網膜色素上皮細胞内の接着蛋白質claudin-1の発現変化.” 2013. Thesis, Hiroshima University / 広島大学. Accessed March 07, 2021. http://ir.lib.hiroshima-u.ac.jp/00040614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hirata, Junko. “Oxidative stress regulates expression of claudin-1 in human RPE cells : 酸化ストレスによる網膜色素上皮細胞内の接着蛋白質claudin-1の発現変化.” 2013. Web. 07 Mar 2021.

Vancouver:

Hirata J. Oxidative stress regulates expression of claudin-1 in human RPE cells : 酸化ストレスによる網膜色素上皮細胞内の接着蛋白質claudin-1の発現変化. [Internet] [Thesis]. Hiroshima University / 広島大学; 2013. [cited 2021 Mar 07]. Available from: http://ir.lib.hiroshima-u.ac.jp/00040614.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hirata J. Oxidative stress regulates expression of claudin-1 in human RPE cells : 酸化ストレスによる網膜色素上皮細胞内の接着蛋白質claudin-1の発現変化. [Thesis]. Hiroshima University / 広島大学; 2013. Available from: http://ir.lib.hiroshima-u.ac.jp/00040614

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kyoto University

17. Sakurai, Kenji. Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis .

Degree: 2020, Kyoto University

Subjects/Keywords: Psoriasis; p38 mitogen-activated protein kinase; Anisomycin; IL-17

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sakurai, K. (2020). Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/245835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sakurai, Kenji. “Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis .” 2020. Thesis, Kyoto University. Accessed March 07, 2021. http://hdl.handle.net/2433/245835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sakurai, Kenji. “Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis .” 2020. Web. 07 Mar 2021.

Vancouver:

Sakurai K. Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis . [Internet] [Thesis]. Kyoto University; 2020. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2433/245835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sakurai K. Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis . [Thesis]. Kyoto University; 2020. Available from: http://hdl.handle.net/2433/245835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. 山本, 梓司. Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Multiple sclerosis is a neuroinflammatory demyelinating and neurodegenerative disease of the central nervous system characterized by recurrent and progressive demyelination/remyelination cycles, neuroinflammation, oligodendrocyte loss, demyelination,… (more)

Subjects/Keywords: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Differentiation; Cell Line, Transformed; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Gene Expression Regulation; Humans; MAP Kinase Signaling System; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Sheath; NLR Family, Pyrin Domain-Containing 3 Protein; Phosphatidic Acids; Proto-Oncogene Proteins c-bcl-2; p38 Mitogen-Activated Protein Kinases

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APA (6th Edition):

山本, . (2018). Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed March 07, 2021. http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Web. 07 Mar 2021.

Vancouver:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Mar 07]. Available from: http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

19. Küppers, Monika. Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene.

Degree: 2008, Johannes Gutenberg Universität Mainz

Die Zellen eines Organismus unterliegen ständig den Einflüssen wachstumsfördernder und –hemmender Signale. Die korrekte Verarbeitung dieser Signale ist essentiell für die Aufrechterhaltung der Gewebehomöostase. Wachstumsfördernde… (more)

Subjects/Keywords: Kontaktinhibition, Zellzyklus, Mitogen-aktivierte Protein Kinasen (MAPK), differentielle Genexpression; contact-inhibition, cell cycle, mitogen-activated protein kinases (MAPK), differential gene expression; Natural sciences and mathematics

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APA (6th Edition):

Küppers, M. (2008). Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/

Chicago Manual of Style (16th Edition):

Küppers, Monika. “Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene.” 2008. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed March 07, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/.

MLA Handbook (7th Edition):

Küppers, Monika. “Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene.” 2008. Web. 07 Mar 2021.

Vancouver:

Küppers M. Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2008. [cited 2021 Mar 07]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/.

Council of Science Editors:

Küppers M. Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2008. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/


University of Hong Kong

20. 林敬賢. Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling.

Degree: 2010, University of Hong Kong

Subjects/Keywords: Transcription factors.; Cellular signal transduction.; Mitogen-activated protein kinases.

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APA (6th Edition):

林敬賢.. (2010). Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/65109

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

林敬賢.. “Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling.” 2010. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/65109.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

林敬賢.. “Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling.” 2010. Web. 07 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

林敬賢.. Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. [Internet] [Thesis]. University of Hong Kong; 2010. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/65109.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

林敬賢.. Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. [Thesis]. University of Hong Kong; 2010. Available from: http://hdl.handle.net/10722/65109

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

21. Minkenberg, Bastian. MULTIPLEX TARGETED MUTATION AND ANALYSIS OF RICE MAP KINASE GENES WITH CRISPR/CAS9.

Degree: 2017, Penn State University

 Traditionally, it was very cumbersome to achieve specific changes in the genome of an organism. Recent advances in site-specific nucleases revived the field of genome-editing… (more)

Subjects/Keywords: genome-editing; CRISPR/Cas9; rice; Oryza sativa; off-target prediction; mitogen-activated protein kinases; essential genes; multi-gene family; multiplex

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APA (6th Edition):

Minkenberg, B. (2017). MULTIPLEX TARGETED MUTATION AND ANALYSIS OF RICE MAP KINASE GENES WITH CRISPR/CAS9. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13692bum179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Minkenberg, Bastian. “MULTIPLEX TARGETED MUTATION AND ANALYSIS OF RICE MAP KINASE GENES WITH CRISPR/CAS9.” 2017. Thesis, Penn State University. Accessed March 07, 2021. https://submit-etda.libraries.psu.edu/catalog/13692bum179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Minkenberg, Bastian. “MULTIPLEX TARGETED MUTATION AND ANALYSIS OF RICE MAP KINASE GENES WITH CRISPR/CAS9.” 2017. Web. 07 Mar 2021.

Vancouver:

Minkenberg B. MULTIPLEX TARGETED MUTATION AND ANALYSIS OF RICE MAP KINASE GENES WITH CRISPR/CAS9. [Internet] [Thesis]. Penn State University; 2017. [cited 2021 Mar 07]. Available from: https://submit-etda.libraries.psu.edu/catalog/13692bum179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Minkenberg B. MULTIPLEX TARGETED MUTATION AND ANALYSIS OF RICE MAP KINASE GENES WITH CRISPR/CAS9. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/13692bum179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

22. Lo, Miao-Chia. The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos.

Degree: 2005, University of Texas Southwestern Medical Center

 In C. elegans embryos, the combined Wnt/MAPK pathway polarizes the founder cell of mesendoderm, EMS blastomere, such that EMS produces two daughters with distinct developmental… (more)

Subjects/Keywords: Caenorhabditis elegans; DNA-Binding Proteins; Mitogen-Activated Protein Kinases

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APA (6th Edition):

Lo, M. (2005). The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lo, Miao-Chia. “The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos.” 2005. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lo, Miao-Chia. “The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos.” 2005. Web. 07 Mar 2021.

Vancouver:

Lo M. The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2005. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lo M. The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos. [Thesis]. University of Texas Southwestern Medical Center; 2005. Available from: http://hdl.handle.net/2152.5/566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

23. Grobe, Theresa. Mechanisms of cell activation by Chlamydia trachomatis Serovar K.

Degree: 2014, Freie Universität Berlin

 Chlamydia trachomatis is the most common cause of sexually transmitted disease. They typically induce asymptomatic lower genital tract infections, such as cervicitis, endometritis and salpingitis.… (more)

Subjects/Keywords: chlamydia trachomatis; mitogen-activated protein kinases; nuclear factor-kappa B; interleukin-8; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Grobe, T. (2014). Mechanisms of cell activation by Chlamydia trachomatis Serovar K. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-11204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grobe, Theresa. “Mechanisms of cell activation by Chlamydia trachomatis Serovar K.” 2014. Thesis, Freie Universität Berlin. Accessed March 07, 2021. http://dx.doi.org/10.17169/refubium-11204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grobe, Theresa. “Mechanisms of cell activation by Chlamydia trachomatis Serovar K.” 2014. Web. 07 Mar 2021.

Vancouver:

Grobe T. Mechanisms of cell activation by Chlamydia trachomatis Serovar K. [Internet] [Thesis]. Freie Universität Berlin; 2014. [cited 2021 Mar 07]. Available from: http://dx.doi.org/10.17169/refubium-11204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grobe T. Mechanisms of cell activation by Chlamydia trachomatis Serovar K. [Thesis]. Freie Universität Berlin; 2014. Available from: http://dx.doi.org/10.17169/refubium-11204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina – Greensboro

24. Eanes, Lauren A. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.

Degree: 2014, University of North Carolina – Greensboro

 Estrogen receptor (ER) antagonists such as tamoxifen have been used successfully to treat ER+ breast cancers for more than 30 years. Tamoxifen targets the ER… (more)

Subjects/Keywords: Breast – Cancer – Treatment; Tamoxifen; Drug resistance in cancer cells; Estrogen – Receptors – Pathophysiology; Mitogen-activated protein kinases

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APA (6th Edition):

Eanes, L. A. (2014). Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160

Chicago Manual of Style (16th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Masters Thesis, University of North Carolina – Greensboro. Accessed March 07, 2021. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160.

MLA Handbook (7th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Web. 07 Mar 2021.

Vancouver:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2014. [cited 2021 Mar 07]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160.

Council of Science Editors:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Masters Thesis]. University of North Carolina – Greensboro; 2014. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160


University of Missouri – Columbia

25. Larue, Clayton T., 1977-. Regulation of plant development in Arabidopsis.

Degree: PhD, 2008, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT REQUEST OF AUTHOR.] The development of a complex organism, such as a plant, requires the function of… (more)

Subjects/Keywords: Arabidopsis  – Development; Abscission (Botany); Mitogen-activated protein kinases

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APA (6th Edition):

Larue, Clayton T., 1. (2008). Regulation of plant development in Arabidopsis. (Doctoral Dissertation). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/6051

Chicago Manual of Style (16th Edition):

Larue, Clayton T., 1977-. “Regulation of plant development in Arabidopsis.” 2008. Doctoral Dissertation, University of Missouri – Columbia. Accessed March 07, 2021. http://hdl.handle.net/10355/6051.

MLA Handbook (7th Edition):

Larue, Clayton T., 1977-. “Regulation of plant development in Arabidopsis.” 2008. Web. 07 Mar 2021.

Vancouver:

Larue, Clayton T. 1. Regulation of plant development in Arabidopsis. [Internet] [Doctoral dissertation]. University of Missouri – Columbia; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10355/6051.

Council of Science Editors:

Larue, Clayton T. 1. Regulation of plant development in Arabidopsis. [Doctoral Dissertation]. University of Missouri – Columbia; 2008. Available from: http://hdl.handle.net/10355/6051


University of Hong Kong

26. 趙芷菱. Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries.

Degree: 2014, University of Hong Kong

 Background: Regulation of vascular tone is complex. Various complementary signaling pathways causing contraction and relaxation of vascular smooth muscle take place to ensure proper blood… (more)

Subjects/Keywords: Vascular smooth muscle; Mitogen-activated protein kinases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

趙芷菱. (2014). Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/206497

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

趙芷菱. “Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries.” 2014. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/206497.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

趙芷菱. “Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries.” 2014. Web. 07 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

趙芷菱. Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. [Internet] [Thesis]. University of Hong Kong; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/206497.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

趙芷菱. Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. [Thesis]. University of Hong Kong; 2014. Available from: http://hdl.handle.net/10722/206497

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

27. Gopallawa, Indiwari. Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2.

Degree: 2015, Michigan State University

Thesis Ph. D. Michigan State University. Biochemistry and Molecular Biology 2015.

Apoptosis is now known to be an important regulator of maintaining normal organ homeostasis.… (more)

Subjects/Keywords: Mitogen-activated protein kinases; Phosphatases; Angiotensin converting enzyme; Angiotensin II – Antagonists; Angiotensins; Apoptosis; Epithelial cells; Cellular biology; Molecular biology; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gopallawa, I. (2015). Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gopallawa, Indiwari. “Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2.” 2015. Thesis, Michigan State University. Accessed March 07, 2021. http://etd.lib.msu.edu/islandora/object/etd:3683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gopallawa, Indiwari. “Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2.” 2015. Web. 07 Mar 2021.

Vancouver:

Gopallawa I. Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2. [Internet] [Thesis]. Michigan State University; 2015. [cited 2021 Mar 07]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gopallawa I. Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:3683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

28. Rattanasinchai, Chotirat. Dissecting the role of mixed lineage kinase 3 (MLK3) in breast cancer invasion and metastasis.

Degree: 2017, Michigan State University

 "MLK3 is a mitogen-activated protein kinase kinase kinase (MAP3K) protein which can activate multiple MAPK pathways. MLK3 has been recently shown to be critical for… (more)

Subjects/Keywords: Breast – Cancer – Cytopathology; Breast – Cancer – Molecular aspects; Cancer invasiveness; Metastasis; Mitogen-activated protein kinases; Molecular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rattanasinchai, C. (2017). Dissecting the role of mixed lineage kinase 3 (MLK3) in breast cancer invasion and metastasis. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:4619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rattanasinchai, Chotirat. “Dissecting the role of mixed lineage kinase 3 (MLK3) in breast cancer invasion and metastasis.” 2017. Thesis, Michigan State University. Accessed March 07, 2021. http://etd.lib.msu.edu/islandora/object/etd:4619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rattanasinchai, Chotirat. “Dissecting the role of mixed lineage kinase 3 (MLK3) in breast cancer invasion and metastasis.” 2017. Web. 07 Mar 2021.

Vancouver:

Rattanasinchai C. Dissecting the role of mixed lineage kinase 3 (MLK3) in breast cancer invasion and metastasis. [Internet] [Thesis]. Michigan State University; 2017. [cited 2021 Mar 07]. Available from: http://etd.lib.msu.edu/islandora/object/etd:4619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rattanasinchai C. Dissecting the role of mixed lineage kinase 3 (MLK3) in breast cancer invasion and metastasis. [Thesis]. Michigan State University; 2017. Available from: http://etd.lib.msu.edu/islandora/object/etd:4619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Eanes, Lauren A. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.

Degree: 2014, NC Docks

 Estrogen receptor (ER) antagonists such as tamoxifen have been used successfully to treat ER+ breast cancers for more than 30 years. Tamoxifen targets the ER… (more)

Subjects/Keywords: Breast $x Cancer $x Treatment; Tamoxifen; Drug resistance in cancer cells; Estrogen $x Receptors $x Pathophysiology; Mitogen-activated protein kinases

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APA (6th Edition):

Eanes, L. A. (2014). Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. (Thesis). NC Docks. Retrieved from http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Thesis, NC Docks. Accessed March 07, 2021. http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Web. 07 Mar 2021.

Vancouver:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Internet] [Thesis]. NC Docks; 2014. [cited 2021 Mar 07]. Available from: http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Thesis]. NC Docks; 2014. Available from: http://libres.uncg.edu/ir/uncg/f/Eanes_uncg_0154M_11469.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Καρκαλή, Αικατερίνη. Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 MAPK (Mitogen Activated Protein Kinases) signaling pathways have been extensively studied and are known to control multiple biological processes, such as proliferation, apoptosis, differentiation and… (more)

Subjects/Keywords: Φωσφατάσες διπλής εξειδίκευσης της Drosophila melanogaster; Ενεργοποιούµενες από µιτογόνα ερεθίσµατα κινάσες; Οξειδωτικό στρες; ΦΩΣΦΟΡΥΛΙΩΣΗ ΠΡΩΤΕΙΝΩΝ; Εµβρυϊκό Νευρικό Σύστηµα της Drosophila melanogaster; Φωσφατάση Puckered; Κινάση του N-τελικού άκρου του µεταγραφικού παράγοντα c-Jun; Κινάση p38; Dual Specificity Phosphatase’s of Drosophila melanogaster; Mitogen – Activated Protein Kinases (MAPKs); Oxidative stress; PROTEIN PHOSPHORYLATION; Embryonic Nervous System of Drosophila melanogaster; Puckered phosphatase; c-Jun N-terminal kinase (JNK); p38 kinase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Καρκαλή, . . (2012). Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Καρκαλή, Αικατερίνη. “Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 07, 2021. http://hdl.handle.net/10442/hedi/36150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Καρκαλή, Αικατερίνη. “Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.” 2012. Web. 07 Mar 2021.

Vancouver:

Καρκαλή . Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10442/hedi/36150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Καρκαλή . Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/36150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [744]

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