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You searched for subject:(p27). Showing records 1 – 30 of 70 total matches.

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1. Nowosad, Ada. Rôle de p27/Kip1 dans l'autophagie induite par le stress métabolique : Role of p27/Kip1 in auphagy under metabolic stress conditions.

Degree: Docteur es, Cancérologie, 2018, Université Toulouse III – Paul Sabatier

Les cancers sont caractérisés par une prolifération anarchique des cellules causée par une dérégulation des mécanismes de contrôle du cycle cellulaire, comme la protéine p27Kip1… (more)

Subjects/Keywords: P27; Autophagie; MTORC1; Trafficking; P27; Autophagy; MTORC1; Trafficking

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APA (6th Edition):

Nowosad, A. (2018). Rôle de p27/Kip1 dans l'autophagie induite par le stress métabolique : Role of p27/Kip1 in auphagy under metabolic stress conditions. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2018TOU30194

Chicago Manual of Style (16th Edition):

Nowosad, Ada. “Rôle de p27/Kip1 dans l'autophagie induite par le stress métabolique : Role of p27/Kip1 in auphagy under metabolic stress conditions.” 2018. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed September 25, 2020. http://www.theses.fr/2018TOU30194.

MLA Handbook (7th Edition):

Nowosad, Ada. “Rôle de p27/Kip1 dans l'autophagie induite par le stress métabolique : Role of p27/Kip1 in auphagy under metabolic stress conditions.” 2018. Web. 25 Sep 2020.

Vancouver:

Nowosad A. Rôle de p27/Kip1 dans l'autophagie induite par le stress métabolique : Role of p27/Kip1 in auphagy under metabolic stress conditions. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2018. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2018TOU30194.

Council of Science Editors:

Nowosad A. Rôle de p27/Kip1 dans l'autophagie induite par le stress métabolique : Role of p27/Kip1 in auphagy under metabolic stress conditions. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2018. Available from: http://www.theses.fr/2018TOU30194

2. Perez Madrigal, Diana. The role of ERK5 in cell proliferation.

Degree: PhD, 2013, University of Manchester

 The extracellular signal-regulated protein kinase 5 (ERK5), also known as big mitogen-activated protein (MAP) kinase 1 (BMK1), is a non-redundant mitogen-activated protein kinase (MAPK) implicated… (more)

Subjects/Keywords: 611; MAPKs; ERK5; p21; p27; Cell cycle

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APA (6th Edition):

Perez Madrigal, D. (2013). The role of ERK5 in cell proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-erk5-in-cell-proliferation(ee569cda-581d-4698-80c0-84f15fe88f53).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607028

Chicago Manual of Style (16th Edition):

Perez Madrigal, Diana. “The role of ERK5 in cell proliferation.” 2013. Doctoral Dissertation, University of Manchester. Accessed September 25, 2020. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-erk5-in-cell-proliferation(ee569cda-581d-4698-80c0-84f15fe88f53).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607028.

MLA Handbook (7th Edition):

Perez Madrigal, Diana. “The role of ERK5 in cell proliferation.” 2013. Web. 25 Sep 2020.

Vancouver:

Perez Madrigal D. The role of ERK5 in cell proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2020 Sep 25]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-erk5-in-cell-proliferation(ee569cda-581d-4698-80c0-84f15fe88f53).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607028.

Council of Science Editors:

Perez Madrigal D. The role of ERK5 in cell proliferation. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-erk5-in-cell-proliferation(ee569cda-581d-4698-80c0-84f15fe88f53).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607028


University of Miami

3. Besser, Alexandra. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.

Degree: PhD, Cancer Biology (Medicine), 2015, University of Miami

  In normal cells, p27 regulates cell cycle and functions as an atypical tumor suppressor. Unlike typical tumor suppressors such as p16 and pRb, p27(more)

Subjects/Keywords: p27; Metastasis; EMT; PI3K; STAT3; TGF-B2

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APA (6th Edition):

Besser, A. (2015). p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1545

Chicago Manual of Style (16th Edition):

Besser, Alexandra. “p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.” 2015. Doctoral Dissertation, University of Miami. Accessed September 25, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/1545.

MLA Handbook (7th Edition):

Besser, Alexandra. “p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program.” 2015. Web. 25 Sep 2020.

Vancouver:

Besser A. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. [Internet] [Doctoral dissertation]. University of Miami; 2015. [cited 2020 Sep 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1545.

Council of Science Editors:

Besser A. p27 Drives PI3K-dependent Cancer Metastasis by Activating EMT Transcription Factors to Induce an EMT Program. [Doctoral Dissertation]. University of Miami; 2015. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1545


University of Western Ontario

4. Cecchini, Matthew J. Dissecting the molecular role of distinct binding interfaces on the retinoblastoma tumor suppressor in growth control and tumorigenesis.

Degree: 2011, University of Western Ontario

 The retinoblastoma tumor suppressor protein (pRB) functions to maintain proliferative control and act as a barrier to tumorigenesis. pRB is capable of regulating E2F transcription… (more)

Subjects/Keywords: pRB; E2F1; p27; retinoblastoma; cancer; apoptosis; Biochemistry

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APA (6th Edition):

Cecchini, M. J. (2011). Dissecting the molecular role of distinct binding interfaces on the retinoblastoma tumor suppressor in growth control and tumorigenesis. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cecchini, Matthew J. “Dissecting the molecular role of distinct binding interfaces on the retinoblastoma tumor suppressor in growth control and tumorigenesis.” 2011. Thesis, University of Western Ontario. Accessed September 25, 2020. https://ir.lib.uwo.ca/etd/170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cecchini, Matthew J. “Dissecting the molecular role of distinct binding interfaces on the retinoblastoma tumor suppressor in growth control and tumorigenesis.” 2011. Web. 25 Sep 2020.

Vancouver:

Cecchini MJ. Dissecting the molecular role of distinct binding interfaces on the retinoblastoma tumor suppressor in growth control and tumorigenesis. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2020 Sep 25]. Available from: https://ir.lib.uwo.ca/etd/170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cecchini MJ. Dissecting the molecular role of distinct binding interfaces on the retinoblastoma tumor suppressor in growth control and tumorigenesis. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Clagnan, Willian Simões. Câncer de mama localmente avançado: ciclina A e proteína p27 para predição de resposta à quimioterapia neoadjuvante.

Degree: Mestrado, Ginecologia e Obstetrícia, 2008, University of São Paulo

 Introdução: a disfunção de proteínas que atuam no controle do ciclo celular está diretamente relacionada ao processo inicial de tumorigênese. A expressão de ciclina A… (more)

Subjects/Keywords: breast cancer; Câncer de mama; ciclina A; cyclin A; neoadjuvant; neoadjuvante; p27 protein; proteína p27

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APA (6th Edition):

Clagnan, W. S. (2008). Câncer de mama localmente avançado: ciclina A e proteína p27 para predição de resposta à quimioterapia neoadjuvante. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17145/tde-27092013-105202/ ;

Chicago Manual of Style (16th Edition):

Clagnan, Willian Simões. “Câncer de mama localmente avançado: ciclina A e proteína p27 para predição de resposta à quimioterapia neoadjuvante.” 2008. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/17/17145/tde-27092013-105202/ ;.

MLA Handbook (7th Edition):

Clagnan, Willian Simões. “Câncer de mama localmente avançado: ciclina A e proteína p27 para predição de resposta à quimioterapia neoadjuvante.” 2008. Web. 25 Sep 2020.

Vancouver:

Clagnan WS. Câncer de mama localmente avançado: ciclina A e proteína p27 para predição de resposta à quimioterapia neoadjuvante. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17145/tde-27092013-105202/ ;.

Council of Science Editors:

Clagnan WS. Câncer de mama localmente avançado: ciclina A e proteína p27 para predição de resposta à quimioterapia neoadjuvante. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/17/17145/tde-27092013-105202/ ;

6. Hassumi, Marcela Kazue. Detecção do HPV e avaliação imunoistoquímica de proteínas reguladoras do ciclo celular em carcinomas invasivos de laringe com e sem metástases.

Degree: Mestrado, Patologia, 2008, University of São Paulo

O mecanismo de oncogênese na laringe pode ser controlado por vários fatores, entre eles fatores envolvidos na regulação do ciclo celular e outros de risco,… (more)

Subjects/Keywords: carcinoma de laringe; laryngeal carcinoma; Mdm2; Mdm2; metástase; metastasis; p27; p27; p53; p53

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APA (6th Edition):

Hassumi, M. K. (2008). Detecção do HPV e avaliação imunoistoquímica de proteínas reguladoras do ciclo celular em carcinomas invasivos de laringe com e sem metástases. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17143/tde-30052012-150445/ ;

Chicago Manual of Style (16th Edition):

Hassumi, Marcela Kazue. “Detecção do HPV e avaliação imunoistoquímica de proteínas reguladoras do ciclo celular em carcinomas invasivos de laringe com e sem metástases.” 2008. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/17/17143/tde-30052012-150445/ ;.

MLA Handbook (7th Edition):

Hassumi, Marcela Kazue. “Detecção do HPV e avaliação imunoistoquímica de proteínas reguladoras do ciclo celular em carcinomas invasivos de laringe com e sem metástases.” 2008. Web. 25 Sep 2020.

Vancouver:

Hassumi MK. Detecção do HPV e avaliação imunoistoquímica de proteínas reguladoras do ciclo celular em carcinomas invasivos de laringe com e sem metástases. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-30052012-150445/ ;.

Council of Science Editors:

Hassumi MK. Detecção do HPV e avaliação imunoistoquímica de proteínas reguladoras do ciclo celular em carcinomas invasivos de laringe com e sem metástases. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-30052012-150445/ ;


Université Montpellier II

7. Lossaint, Gérald. Mécanismes orchestrant la sortie du cycle cellulaire opérant en G2 : Mechanisms orchestrating cell cycle exit operating G2.

Degree: Docteur es, Biologie cellulaire, 2010, Université Montpellier II

La dérégulation du système de surveillance qui bloque la prolifération lorsque l'intégrité du génome est compromise fait partie intégrante de la cancérogenèse. Nous cherchons à… (more)

Subjects/Keywords: Inhibiteurs des CDKs p21 et p27; Sénescence; Quiescence; Atm; Chk1-Chk2; Cycline D1; CDKCDKs p21 et p27 Inhibitors p21 and p27; Senescence; Quiescence; Atm; Chk1-2; Cyclin D1

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APA (6th Edition):

Lossaint, G. (2010). Mécanismes orchestrant la sortie du cycle cellulaire opérant en G2 : Mechanisms orchestrating cell cycle exit operating G2. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2010MON20040

Chicago Manual of Style (16th Edition):

Lossaint, Gérald. “Mécanismes orchestrant la sortie du cycle cellulaire opérant en G2 : Mechanisms orchestrating cell cycle exit operating G2.” 2010. Doctoral Dissertation, Université Montpellier II. Accessed September 25, 2020. http://www.theses.fr/2010MON20040.

MLA Handbook (7th Edition):

Lossaint, Gérald. “Mécanismes orchestrant la sortie du cycle cellulaire opérant en G2 : Mechanisms orchestrating cell cycle exit operating G2.” 2010. Web. 25 Sep 2020.

Vancouver:

Lossaint G. Mécanismes orchestrant la sortie du cycle cellulaire opérant en G2 : Mechanisms orchestrating cell cycle exit operating G2. [Internet] [Doctoral dissertation]. Université Montpellier II; 2010. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2010MON20040.

Council of Science Editors:

Lossaint G. Mécanismes orchestrant la sortie du cycle cellulaire opérant en G2 : Mechanisms orchestrating cell cycle exit operating G2. [Doctoral Dissertation]. Université Montpellier II; 2010. Available from: http://www.theses.fr/2010MON20040

8. Côrtes, Arthur Rodriguez Gonzalez. Comparação imunoistoquímica das expressões das proteínas p27 e c-jun na carcinogênese intra-oral.

Degree: Mestrado, Patologia Bucal, 2009, University of São Paulo

A expressão de diversas proteínas do ciclo celular tem sido estudada em vários tipos de lesões malignas. Entre as principais está a p27, que é… (more)

Subjects/Keywords: Carcinoma epidermóide; Carcinoma epidermóide; Displasia epitelial; Displasia epitelial; Imunoistoquímica; Imunoistoquímica; p27 c-jun; p27 c-jun

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APA (6th Edition):

Côrtes, A. R. G. (2009). Comparação imunoistoquímica das expressões das proteínas p27 e c-jun na carcinogênese intra-oral. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-19122009-111607/ ;

Chicago Manual of Style (16th Edition):

Côrtes, Arthur Rodriguez Gonzalez. “Comparação imunoistoquímica das expressões das proteínas p27 e c-jun na carcinogênese intra-oral.” 2009. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-19122009-111607/ ;.

MLA Handbook (7th Edition):

Côrtes, Arthur Rodriguez Gonzalez. “Comparação imunoistoquímica das expressões das proteínas p27 e c-jun na carcinogênese intra-oral.” 2009. Web. 25 Sep 2020.

Vancouver:

Côrtes ARG. Comparação imunoistoquímica das expressões das proteínas p27 e c-jun na carcinogênese intra-oral. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-19122009-111607/ ;.

Council of Science Editors:

Côrtes ARG. Comparação imunoistoquímica das expressões das proteínas p27 e c-jun na carcinogênese intra-oral. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-19122009-111607/ ;


The Ohio State University

9. Nicholas, Courtney. The Protein Arginine Methyltransferase PRMT5 Regulates Proliferation and the Expression of MITF and p27Kip1 in Human Melanoma.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2012, The Ohio State University

 The protein arginine methyltransferase-5 (PRMT5) enzyme is a Type II arginine methyltransferase that can regulate a variety of cellular functions. We hypothesized that PRMT5 plays… (more)

Subjects/Keywords: Molecular Biology; Oncology; melanoma; methyltransferase; PRMT; MITF; p27; proliferation; skin cancer

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APA (6th Edition):

Nicholas, C. (2012). The Protein Arginine Methyltransferase PRMT5 Regulates Proliferation and the Expression of MITF and p27Kip1 in Human Melanoma. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1343078125

Chicago Manual of Style (16th Edition):

Nicholas, Courtney. “The Protein Arginine Methyltransferase PRMT5 Regulates Proliferation and the Expression of MITF and p27Kip1 in Human Melanoma.” 2012. Doctoral Dissertation, The Ohio State University. Accessed September 25, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1343078125.

MLA Handbook (7th Edition):

Nicholas, Courtney. “The Protein Arginine Methyltransferase PRMT5 Regulates Proliferation and the Expression of MITF and p27Kip1 in Human Melanoma.” 2012. Web. 25 Sep 2020.

Vancouver:

Nicholas C. The Protein Arginine Methyltransferase PRMT5 Regulates Proliferation and the Expression of MITF and p27Kip1 in Human Melanoma. [Internet] [Doctoral dissertation]. The Ohio State University; 2012. [cited 2020 Sep 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1343078125.

Council of Science Editors:

Nicholas C. The Protein Arginine Methyltransferase PRMT5 Regulates Proliferation and the Expression of MITF and p27Kip1 in Human Melanoma. [Doctoral Dissertation]. The Ohio State University; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1343078125


University of Otago

10. Guan, Guangzhao. Expression of cyclin D1 and its correlation with p27KIP1 in non-neoplastic and non-dysplastic mucosa, oral epithelial dysplasia and oral squamous cell carcinoma .

Degree: University of Otago

 Background Oral and pharyngeal cancer accounts for one-sixth of the total cancers worldwide. An estimated 263,900 new cases and 128,000 deaths from oral cavity cancer… (more)

Subjects/Keywords: CyclinD1; P27; oral; cancer

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APA (6th Edition):

Guan, G. (n.d.). Expression of cyclin D1 and its correlation with p27KIP1 in non-neoplastic and non-dysplastic mucosa, oral epithelial dysplasia and oral squamous cell carcinoma . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/5125

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Guan, Guangzhao. “Expression of cyclin D1 and its correlation with p27KIP1 in non-neoplastic and non-dysplastic mucosa, oral epithelial dysplasia and oral squamous cell carcinoma .” Doctoral Dissertation, University of Otago. Accessed September 25, 2020. http://hdl.handle.net/10523/5125.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Guan, Guangzhao. “Expression of cyclin D1 and its correlation with p27KIP1 in non-neoplastic and non-dysplastic mucosa, oral epithelial dysplasia and oral squamous cell carcinoma .” Web. 25 Sep 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Guan G. Expression of cyclin D1 and its correlation with p27KIP1 in non-neoplastic and non-dysplastic mucosa, oral epithelial dysplasia and oral squamous cell carcinoma . [Internet] [Doctoral dissertation]. University of Otago; [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10523/5125.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Guan G. Expression of cyclin D1 and its correlation with p27KIP1 in non-neoplastic and non-dysplastic mucosa, oral epithelial dysplasia and oral squamous cell carcinoma . [Doctoral Dissertation]. University of Otago; Available from: http://hdl.handle.net/10523/5125

Note: this citation may be lacking information needed for this citation format:
No year of publication.

11. Σιρινιάν, Χάιδω. Μελέτη ρυθμιστών του κυτταρικού κύκλου και παραγόντων που εμπλέκονται στη διεργασία αποδόμησης των P21cip1 και P27kip1 σε λεμφώματα Β-κυτταρικής αρχής. Συσχέτιση με κλινικές παραμέτρους.

Degree: 2012, University of Patras

 Κατά τη διάρκεια του κυτταρικού κύκλου ένας από τους σημαντικότερους μηχανισμούς ρύθμισης της ομοιόστασης των πρωτεϊνικών μορίων είναι η ελεγχόμενη στο χώρο και στο χρόνο… (more)

Subjects/Keywords: Β-λέμφωμα; Προτεώλυση; Επιβίωση; 571.84; B-lymphoma; Proteolysis; Survival; p27; p21

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APA (6th Edition):

Σιρινιάν, . (2012). Μελέτη ρυθμιστών του κυτταρικού κύκλου και παραγόντων που εμπλέκονται στη διεργασία αποδόμησης των P21cip1 και P27kip1 σε λεμφώματα Β-κυτταρικής αρχής. Συσχέτιση με κλινικές παραμέτρους. (Doctoral Dissertation). University of Patras. Retrieved from http://hdl.handle.net/10889/5264

Chicago Manual of Style (16th Edition):

Σιρινιάν, Χάιδω. “Μελέτη ρυθμιστών του κυτταρικού κύκλου και παραγόντων που εμπλέκονται στη διεργασία αποδόμησης των P21cip1 και P27kip1 σε λεμφώματα Β-κυτταρικής αρχής. Συσχέτιση με κλινικές παραμέτρους.” 2012. Doctoral Dissertation, University of Patras. Accessed September 25, 2020. http://hdl.handle.net/10889/5264.

MLA Handbook (7th Edition):

Σιρινιάν, Χάιδω. “Μελέτη ρυθμιστών του κυτταρικού κύκλου και παραγόντων που εμπλέκονται στη διεργασία αποδόμησης των P21cip1 και P27kip1 σε λεμφώματα Β-κυτταρικής αρχής. Συσχέτιση με κλινικές παραμέτρους.” 2012. Web. 25 Sep 2020.

Vancouver:

Σιρινιάν . Μελέτη ρυθμιστών του κυτταρικού κύκλου και παραγόντων που εμπλέκονται στη διεργασία αποδόμησης των P21cip1 και P27kip1 σε λεμφώματα Β-κυτταρικής αρχής. Συσχέτιση με κλινικές παραμέτρους. [Internet] [Doctoral dissertation]. University of Patras; 2012. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10889/5264.

Council of Science Editors:

Σιρινιάν . Μελέτη ρυθμιστών του κυτταρικού κύκλου και παραγόντων που εμπλέκονται στη διεργασία αποδόμησης των P21cip1 και P27kip1 σε λεμφώματα Β-κυτταρικής αρχής. Συσχέτιση με κλινικές παραμέτρους. [Doctoral Dissertation]. University of Patras; 2012. Available from: http://hdl.handle.net/10889/5264


University of Miami

12. Darr, Andrew J. FGF2 Maintains the Proliferation of Neural Progenitors by Actively Suppressing the CKI p27Kip1 through Regulation of Cks1b Transcription.

Degree: PhD, Neuroscience (Medicine), 2009, University of Miami

  Identifying the mechanisms that regulate neural precursor cell (NPC) proliferation and differentiation is important for understanding CNS development among different vertebrates. My work has… (more)

Subjects/Keywords: CNS; P27; Stem Cells; FGF

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APA (6th Edition):

Darr, A. J. (2009). FGF2 Maintains the Proliferation of Neural Progenitors by Actively Suppressing the CKI p27Kip1 through Regulation of Cks1b Transcription. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/356

Chicago Manual of Style (16th Edition):

Darr, Andrew J. “FGF2 Maintains the Proliferation of Neural Progenitors by Actively Suppressing the CKI p27Kip1 through Regulation of Cks1b Transcription.” 2009. Doctoral Dissertation, University of Miami. Accessed September 25, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/356.

MLA Handbook (7th Edition):

Darr, Andrew J. “FGF2 Maintains the Proliferation of Neural Progenitors by Actively Suppressing the CKI p27Kip1 through Regulation of Cks1b Transcription.” 2009. Web. 25 Sep 2020.

Vancouver:

Darr AJ. FGF2 Maintains the Proliferation of Neural Progenitors by Actively Suppressing the CKI p27Kip1 through Regulation of Cks1b Transcription. [Internet] [Doctoral dissertation]. University of Miami; 2009. [cited 2020 Sep 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/356.

Council of Science Editors:

Darr AJ. FGF2 Maintains the Proliferation of Neural Progenitors by Actively Suppressing the CKI p27Kip1 through Regulation of Cks1b Transcription. [Doctoral Dissertation]. University of Miami; 2009. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/356


University of Melbourne

13. Raghu, Dinesh. Restoration of tumour suppression in prostate cancer.

Degree: 2018, University of Melbourne

 Aberration in signalling pathways due to the overexpression of oncogenes, and/or loss or mutations in tumour suppressors are key events during carcinogenesis. Hence, novel approaches… (more)

Subjects/Keywords: prostate cancer; tumour suppression; E6AP; MDM4; p53; PML; p27

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APA (6th Edition):

Raghu, D. (2018). Restoration of tumour suppression in prostate cancer. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/212460

Chicago Manual of Style (16th Edition):

Raghu, Dinesh. “Restoration of tumour suppression in prostate cancer.” 2018. Doctoral Dissertation, University of Melbourne. Accessed September 25, 2020. http://hdl.handle.net/11343/212460.

MLA Handbook (7th Edition):

Raghu, Dinesh. “Restoration of tumour suppression in prostate cancer.” 2018. Web. 25 Sep 2020.

Vancouver:

Raghu D. Restoration of tumour suppression in prostate cancer. [Internet] [Doctoral dissertation]. University of Melbourne; 2018. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/11343/212460.

Council of Science Editors:

Raghu D. Restoration of tumour suppression in prostate cancer. [Doctoral Dissertation]. University of Melbourne; 2018. Available from: http://hdl.handle.net/11343/212460


Universitat de Valencia

14. Molina Sánchez, Pedro. Papel de la fosforilación de p27 en la serina 10 en la función endotelial y el remodelado vascular patológico .

Degree: 2015, Universitat de Valencia

 El supresor tumoral p27 ha sido ampliamente implicado en multitud de procesos patológicos donde ejerce un papel protector debido a su acción antiproliferativa. Particularmente, a… (more)

Subjects/Keywords: p27; endotelio; aterosclerosis; cki; aneurisma aórtico abdominal; reactividad vascular; modelos animales

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APA (6th Edition):

Molina Sánchez, P. (2015). Papel de la fosforilación de p27 en la serina 10 en la función endotelial y el remodelado vascular patológico . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/46805

Chicago Manual of Style (16th Edition):

Molina Sánchez, Pedro. “Papel de la fosforilación de p27 en la serina 10 en la función endotelial y el remodelado vascular patológico .” 2015. Doctoral Dissertation, Universitat de Valencia. Accessed September 25, 2020. http://hdl.handle.net/10550/46805.

MLA Handbook (7th Edition):

Molina Sánchez, Pedro. “Papel de la fosforilación de p27 en la serina 10 en la función endotelial y el remodelado vascular patológico .” 2015. Web. 25 Sep 2020.

Vancouver:

Molina Sánchez P. Papel de la fosforilación de p27 en la serina 10 en la función endotelial y el remodelado vascular patológico . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2015. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10550/46805.

Council of Science Editors:

Molina Sánchez P. Papel de la fosforilación de p27 en la serina 10 en la función endotelial y el remodelado vascular patológico . [Doctoral Dissertation]. Universitat de Valencia; 2015. Available from: http://hdl.handle.net/10550/46805

15. Lima, Joelma Sousa de. Expressão imunohistoquímica das proteínas c-Jun não fosforilada/fosforilada e p27 em leucoplasias de pacientes fumantes e não fumantes.

Degree: Mestrado, Patologia Bucal, 2012, University of São Paulo

Em diversos casos, o câncer bucal é precedido por lesões pré-malignas, como por exemplo, a leucoplasia, sendo que o tabaco é um fator de risco.… (more)

Subjects/Keywords: c-Jun protein; Displasia epitelial; Epithelial dysplasia; Leucoplasia Oral; Oral leukoplakia; p27 protein; pc-Jun protein; Proteína c-Jun; Proteína p-c-Jun; Proteína p27; Tabagismo; Tobacco

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APA (6th Edition):

Lima, J. S. d. (2012). Expressão imunohistoquímica das proteínas c-Jun não fosforilada/fosforilada e p27 em leucoplasias de pacientes fumantes e não fumantes. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-113357/ ;

Chicago Manual of Style (16th Edition):

Lima, Joelma Sousa de. “Expressão imunohistoquímica das proteínas c-Jun não fosforilada/fosforilada e p27 em leucoplasias de pacientes fumantes e não fumantes.” 2012. Masters Thesis, University of São Paulo. Accessed September 25, 2020. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-113357/ ;.

MLA Handbook (7th Edition):

Lima, Joelma Sousa de. “Expressão imunohistoquímica das proteínas c-Jun não fosforilada/fosforilada e p27 em leucoplasias de pacientes fumantes e não fumantes.” 2012. Web. 25 Sep 2020.

Vancouver:

Lima JSd. Expressão imunohistoquímica das proteínas c-Jun não fosforilada/fosforilada e p27 em leucoplasias de pacientes fumantes e não fumantes. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2020 Sep 25]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-113357/ ;.

Council of Science Editors:

Lima JSd. Expressão imunohistoquímica das proteínas c-Jun não fosforilada/fosforilada e p27 em leucoplasias de pacientes fumantes e não fumantes. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13042013-113357/ ;


KTH

16. Lorenzo, Ximena López. Mapping the Expression of Cyclin Dependent Kinase Inhibitors in High-Risk Neuroblastoma Cell Lines : Dynamics on Cell-Fate Decisions on Proliferation/Cell Cycle Arrest.

Degree: Biotechnology and Health (CBH), 2020, KTH

Poor prognosis for high-risk neuroblastoma patients makes it necessary to find novel treatmentstrategies. This work aims to understand the cell cycle behavior of various… (more)

Subjects/Keywords: Neuroblastoma; NMYC; p53; cell cycle checkpoint; inhibition; regrowth; p21; p27; SKP2; CHK1/2; Neuroblastoma; NMYC; p53; cellcykel checkpoint; hämning; återväxt; p21; p27; SKP2; CHK1/2; Medical Biotechnology; Medicinsk bioteknologi

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APA (6th Edition):

Lorenzo, X. L. (2020). Mapping the Expression of Cyclin Dependent Kinase Inhibitors in High-Risk Neuroblastoma Cell Lines : Dynamics on Cell-Fate Decisions on Proliferation/Cell Cycle Arrest. (Thesis). KTH. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-278705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lorenzo, Ximena López. “Mapping the Expression of Cyclin Dependent Kinase Inhibitors in High-Risk Neuroblastoma Cell Lines : Dynamics on Cell-Fate Decisions on Proliferation/Cell Cycle Arrest.” 2020. Thesis, KTH. Accessed September 25, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-278705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lorenzo, Ximena López. “Mapping the Expression of Cyclin Dependent Kinase Inhibitors in High-Risk Neuroblastoma Cell Lines : Dynamics on Cell-Fate Decisions on Proliferation/Cell Cycle Arrest.” 2020. Web. 25 Sep 2020.

Vancouver:

Lorenzo XL. Mapping the Expression of Cyclin Dependent Kinase Inhibitors in High-Risk Neuroblastoma Cell Lines : Dynamics on Cell-Fate Decisions on Proliferation/Cell Cycle Arrest. [Internet] [Thesis]. KTH; 2020. [cited 2020 Sep 25]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-278705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lorenzo XL. Mapping the Expression of Cyclin Dependent Kinase Inhibitors in High-Risk Neuroblastoma Cell Lines : Dynamics on Cell-Fate Decisions on Proliferation/Cell Cycle Arrest. [Thesis]. KTH; 2020. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-278705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Jeannot, Pauline. Etude de nouvelles fonctions de p27 dans l'oncogenèse et l'invasion cellulaire : Investigation of new functions of p27 in oncogenesis and cell invasion.

Degree: Docteur es, Biologie cellulaire, 2016, Université Toulouse III – Paul Sabatier

 Le cycle cellulaire est un processus finement régulé à différents niveaux. p27 est un inhibiteur des complexes cyclines/CDK et cette fonction lui confère un rôle… (more)

Subjects/Keywords: P27; Pancréas; Oncogènese; Invasion cellulaire; Suppression de tumeur; Invadopode; Invadopode; Migration cellulaire

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APA (6th Edition):

Jeannot, P. (2016). Etude de nouvelles fonctions de p27 dans l'oncogenèse et l'invasion cellulaire : Investigation of new functions of p27 in oncogenesis and cell invasion. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2016TOU30092

Chicago Manual of Style (16th Edition):

Jeannot, Pauline. “Etude de nouvelles fonctions de p27 dans l'oncogenèse et l'invasion cellulaire : Investigation of new functions of p27 in oncogenesis and cell invasion.” 2016. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed September 25, 2020. http://www.theses.fr/2016TOU30092.

MLA Handbook (7th Edition):

Jeannot, Pauline. “Etude de nouvelles fonctions de p27 dans l'oncogenèse et l'invasion cellulaire : Investigation of new functions of p27 in oncogenesis and cell invasion.” 2016. Web. 25 Sep 2020.

Vancouver:

Jeannot P. Etude de nouvelles fonctions de p27 dans l'oncogenèse et l'invasion cellulaire : Investigation of new functions of p27 in oncogenesis and cell invasion. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2016. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2016TOU30092.

Council of Science Editors:

Jeannot P. Etude de nouvelles fonctions de p27 dans l'oncogenèse et l'invasion cellulaire : Investigation of new functions of p27 in oncogenesis and cell invasion. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2016. Available from: http://www.theses.fr/2016TOU30092

18. Guitart, Amélie Valérie. Régulation du compartiment des progéniteurs hématopoïétiques par les faibles concentrations en oxygène : analyse de la survie, de la prolifération et de la différenciation du modèle FDCP-Mix : Hematopoietic progenitor compartment regulation by low oxygen concentration : survival, proliferation and differentiation analysis of the FDCP-Mix model.

Degree: Docteur es, Sciences, technologie, santé. Biologie cellulaire et physiopathologie, 2009, Université de Bordeaux Segalen

Les concentrations d’oxygène (O2) dans la moelle osseuse hématopoïétique, sont très inférieures à celle de l’air (20% d’O2) puisqu’elles vont de 4% dans les zones… (more)

Subjects/Keywords: Hématopoïèse; Cycle cellulaire; Hypoxie; Progéniteur; Quiescence; Physioxie; Différenciation granulocytaire; P27; FDCP-Mix; PRb

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APA (6th Edition):

Guitart, A. V. (2009). Régulation du compartiment des progéniteurs hématopoïétiques par les faibles concentrations en oxygène : analyse de la survie, de la prolifération et de la différenciation du modèle FDCP-Mix : Hematopoietic progenitor compartment regulation by low oxygen concentration : survival, proliferation and differentiation analysis of the FDCP-Mix model. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2009BOR21672

Chicago Manual of Style (16th Edition):

Guitart, Amélie Valérie. “Régulation du compartiment des progéniteurs hématopoïétiques par les faibles concentrations en oxygène : analyse de la survie, de la prolifération et de la différenciation du modèle FDCP-Mix : Hematopoietic progenitor compartment regulation by low oxygen concentration : survival, proliferation and differentiation analysis of the FDCP-Mix model.” 2009. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed September 25, 2020. http://www.theses.fr/2009BOR21672.

MLA Handbook (7th Edition):

Guitart, Amélie Valérie. “Régulation du compartiment des progéniteurs hématopoïétiques par les faibles concentrations en oxygène : analyse de la survie, de la prolifération et de la différenciation du modèle FDCP-Mix : Hematopoietic progenitor compartment regulation by low oxygen concentration : survival, proliferation and differentiation analysis of the FDCP-Mix model.” 2009. Web. 25 Sep 2020.

Vancouver:

Guitart AV. Régulation du compartiment des progéniteurs hématopoïétiques par les faibles concentrations en oxygène : analyse de la survie, de la prolifération et de la différenciation du modèle FDCP-Mix : Hematopoietic progenitor compartment regulation by low oxygen concentration : survival, proliferation and differentiation analysis of the FDCP-Mix model. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2009. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2009BOR21672.

Council of Science Editors:

Guitart AV. Régulation du compartiment des progéniteurs hématopoïétiques par les faibles concentrations en oxygène : analyse de la survie, de la prolifération et de la différenciation du modèle FDCP-Mix : Hematopoietic progenitor compartment regulation by low oxygen concentration : survival, proliferation and differentiation analysis of the FDCP-Mix model. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2009. Available from: http://www.theses.fr/2009BOR21672


University of Kansas

19. Paul, Binu M. Regulation of cell proliferation in autosomal dominant polycystic kidney disease.

Degree: PhD, Anatomy & Cell Biology, 2011, University of Kansas

 Autosomal dominant polycystic kidney disease (ADPKD) is a life-threatening genetic disorder characterized by the presence of fluid-filled cysts primarily in the kidneys. Mutations in either… (more)

Subjects/Keywords: Genetics; Molecular biology; Developmental biology; Cell proliferation; Cux1; Kidney development; P27; Polycystic kidney disease

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APA (6th Edition):

Paul, B. M. (2011). Regulation of cell proliferation in autosomal dominant polycystic kidney disease. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/8384

Chicago Manual of Style (16th Edition):

Paul, Binu M. “Regulation of cell proliferation in autosomal dominant polycystic kidney disease.” 2011. Doctoral Dissertation, University of Kansas. Accessed September 25, 2020. http://hdl.handle.net/1808/8384.

MLA Handbook (7th Edition):

Paul, Binu M. “Regulation of cell proliferation in autosomal dominant polycystic kidney disease.” 2011. Web. 25 Sep 2020.

Vancouver:

Paul BM. Regulation of cell proliferation in autosomal dominant polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1808/8384.

Council of Science Editors:

Paul BM. Regulation of cell proliferation in autosomal dominant polycystic kidney disease. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/8384

20. Δούκα, Ευαγγελία. Έλεγχος μεταλλάξεων του γονιδίου p27 σε ασθενείς με χρόνια λεμφοκυτταρική λευχαιμία.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

B-CLL is characterized by the accumulation of resting lymphocytes. The cyclin-dependent kinase (cdk) inhibitor p27, inhibiting all cdk complexes, contributes to cell cycle arrest. High… (more)

Subjects/Keywords: Λευχαιμία, Χρόνια λεμφοκυτταρική; Κυκλίνες; Κυκλινοεξαρτώμενες κινάσες; Αναστολείς κυκλινοεξαρτωμένων κινασών; B - CLL; p27; KIP1; CDKN1B

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APA (6th Edition):

Δούκα, . . (2013). Έλεγχος μεταλλάξεων του γονιδίου p27 σε ασθενείς με χρόνια λεμφοκυτταρική λευχαιμία. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/28515

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Δούκα, Ευαγγελία. “Έλεγχος μεταλλάξεων του γονιδίου p27 σε ασθενείς με χρόνια λεμφοκυτταρική λευχαιμία.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed September 25, 2020. http://hdl.handle.net/10442/hedi/28515.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Δούκα, Ευαγγελία. “Έλεγχος μεταλλάξεων του γονιδίου p27 σε ασθενείς με χρόνια λεμφοκυτταρική λευχαιμία.” 2013. Web. 25 Sep 2020.

Vancouver:

Δούκα . Έλεγχος μεταλλάξεων του γονιδίου p27 σε ασθενείς με χρόνια λεμφοκυτταρική λευχαιμία. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/10442/hedi/28515.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Δούκα . Έλεγχος μεταλλάξεων του γονιδίου p27 σε ασθενείς με χρόνια λεμφοκυτταρική λευχαιμία. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/28515

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Central Florida

21. Ritchey, Lisa. The Role of LIM Kinase 1 and its Substrates in Cell Cycle Progression.

Degree: 2014, University of Central Florida

  LIM Kinase 1 (LIMK1), a modulator of actin and microtubule dynamics, has been shown to be involved in cell cycle progression. In this study… (more)

Subjects/Keywords: Cell cycle; mitosis; lim kinase 1; limk1; aurora a; p27; spindle; Medical Sciences

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APA (6th Edition):

Ritchey, L. (2014). The Role of LIM Kinase 1 and its Substrates in Cell Cycle Progression. (Doctoral Dissertation). University of Central Florida. Retrieved from https://stars.library.ucf.edu/etd/1300

Chicago Manual of Style (16th Edition):

Ritchey, Lisa. “The Role of LIM Kinase 1 and its Substrates in Cell Cycle Progression.” 2014. Doctoral Dissertation, University of Central Florida. Accessed September 25, 2020. https://stars.library.ucf.edu/etd/1300.

MLA Handbook (7th Edition):

Ritchey, Lisa. “The Role of LIM Kinase 1 and its Substrates in Cell Cycle Progression.” 2014. Web. 25 Sep 2020.

Vancouver:

Ritchey L. The Role of LIM Kinase 1 and its Substrates in Cell Cycle Progression. [Internet] [Doctoral dissertation]. University of Central Florida; 2014. [cited 2020 Sep 25]. Available from: https://stars.library.ucf.edu/etd/1300.

Council of Science Editors:

Ritchey L. The Role of LIM Kinase 1 and its Substrates in Cell Cycle Progression. [Doctoral Dissertation]. University of Central Florida; 2014. Available from: https://stars.library.ucf.edu/etd/1300

22. Angela Rosa AndrÃ. AssociaÃÃo da presenÃa de Helicobacter pylori e dos genÃtipos caga e vaca com as alteraÃÃes moleculares dos supressores tumorais P53 e P27 nos adenocarcinomas gÃstricos.

Degree: Master, 2008, Universidade Federal do Ceará

O carcinoma gÃstrico à a segunda causa de morte por cÃncer no mundo. No Cearà à o segundo mais freqÃente entre os homens e o… (more)

Subjects/Keywords: CANCEROLOGIA; Neoplasias GÃstricas; CarcinogÃnese gÃstrica; Adenocarcinoma gÃstrico; Helicobacter pylori; Supressores tumorais; Gene p53; ProteÃna p53; Inibidor de Quinase Dependente de Ciclina p27; ProteÃna p27; p27Kip1; Helicobacter pylori; Gastric adenocarcinoma; Tumor suppressors; Gastric carcinogenesis; p53 gene; p27 protein; Adenocarcinoma

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APA (6th Edition):

AndrÃ, A. R. (2008). AssociaÃÃo da presenÃa de Helicobacter pylori e dos genÃtipos caga e vaca com as alteraÃÃes moleculares dos supressores tumorais P53 e P27 nos adenocarcinomas gÃstricos. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1819 ;

Chicago Manual of Style (16th Edition):

AndrÃ, Angela Rosa. “AssociaÃÃo da presenÃa de Helicobacter pylori e dos genÃtipos caga e vaca com as alteraÃÃes moleculares dos supressores tumorais P53 e P27 nos adenocarcinomas gÃstricos.” 2008. Masters Thesis, Universidade Federal do Ceará. Accessed September 25, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1819 ;.

MLA Handbook (7th Edition):

AndrÃ, Angela Rosa. “AssociaÃÃo da presenÃa de Helicobacter pylori e dos genÃtipos caga e vaca com as alteraÃÃes moleculares dos supressores tumorais P53 e P27 nos adenocarcinomas gÃstricos.” 2008. Web. 25 Sep 2020.

Vancouver:

Andrà AR. AssociaÃÃo da presenÃa de Helicobacter pylori e dos genÃtipos caga e vaca com as alteraÃÃes moleculares dos supressores tumorais P53 e P27 nos adenocarcinomas gÃstricos. [Internet] [Masters thesis]. Universidade Federal do Ceará 2008. [cited 2020 Sep 25]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1819 ;.

Council of Science Editors:

Andrà AR. AssociaÃÃo da presenÃa de Helicobacter pylori e dos genÃtipos caga e vaca com as alteraÃÃes moleculares dos supressores tumorais P53 e P27 nos adenocarcinomas gÃstricos. [Masters Thesis]. Universidade Federal do Ceará 2008. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=1819 ;


Temple University

23. Radu, Maria. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.

Degree: 2008, Temple University

Microbiology and Immunology

Ph.D.;

All trans retinoic acid (atRA) has been shown to inhibit the growth of CAOV3 ovarian carcinoma cells. This results from arrest… (more)

Subjects/Keywords: Health Sciences, Immunology; Biology, Cell; Health Sciences, Oncology; retinoic acid; p27; phosphorylation; cell cycle; growth arrest

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APA (6th Edition):

Radu, M. (2008). The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Thesis, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Web. 25 Sep 2020.

Vancouver:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Cíntia Meirelles de Camargo Kosugi. Avaliação dos polimorfismos nos genes TP53, P27 e FAS em mulheres com endometriose.

Degree: 2009, Universidade Federal de São Paulo

Introdução: Endometriose é uma afecção ginecológica benigna e, em sua fisiopatologia, encontramos os distúrbios do ciclo celular e apoptose. Os genes TP53, P27 e FAS,… (more)

Subjects/Keywords: Endometriose; Polimorfismo genético; Gene TP53; Inibidor de Quinase Dependente de Ciclina P27; FAS; GINECOLOGIA E OBSTETRICIA

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APA (6th Edition):

Kosugi, C. M. d. C. (2009). Avaliação dos polimorfismos nos genes TP53, P27 e FAS em mulheres com endometriose. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1911 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1912

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kosugi, Cíntia Meirelles de Camargo. “Avaliação dos polimorfismos nos genes TP53, P27 e FAS em mulheres com endometriose.” 2009. Thesis, Universidade Federal de São Paulo. Accessed September 25, 2020. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1911 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1912.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kosugi, Cíntia Meirelles de Camargo. “Avaliação dos polimorfismos nos genes TP53, P27 e FAS em mulheres com endometriose.” 2009. Web. 25 Sep 2020.

Vancouver:

Kosugi CMdC. Avaliação dos polimorfismos nos genes TP53, P27 e FAS em mulheres com endometriose. [Internet] [Thesis]. Universidade Federal de São Paulo; 2009. [cited 2020 Sep 25]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1911 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1912.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kosugi CMdC. Avaliação dos polimorfismos nos genes TP53, P27 e FAS em mulheres com endometriose. [Thesis]. Universidade Federal de São Paulo; 2009. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1911 ; http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1912

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

25. Overvelde, Sébastien. Détermination immunohistochimique de l'expression de p27Kip1 dans les tumeurs mammaires canines.

Degree: 2004, Université de Montréal

Subjects/Keywords: P27; Tumeurs mammaires; Canin; Immunohistochimie; Ki67; Cycle cellulaire

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APA (6th Edition):

Overvelde, S. (2004). Détermination immunohistochimique de l'expression de p27Kip1 dans les tumeurs mammaires canines. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/14310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Overvelde, Sébastien. “Détermination immunohistochimique de l'expression de p27Kip1 dans les tumeurs mammaires canines.” 2004. Thesis, Université de Montréal. Accessed September 25, 2020. http://hdl.handle.net/1866/14310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Overvelde, Sébastien. “Détermination immunohistochimique de l'expression de p27Kip1 dans les tumeurs mammaires canines.” 2004. Web. 25 Sep 2020.

Vancouver:

Overvelde S. Détermination immunohistochimique de l'expression de p27Kip1 dans les tumeurs mammaires canines. [Internet] [Thesis]. Université de Montréal; 2004. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1866/14310.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Overvelde S. Détermination immunohistochimique de l'expression de p27Kip1 dans les tumeurs mammaires canines. [Thesis]. Université de Montréal; 2004. Available from: http://hdl.handle.net/1866/14310

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

26. Calvé, Annie. PKCbêta1 intervient dans l'action d'une concentration élevée en glucose sur l'expression de l'angiotensinogène et l'hypertrophie des IRPTC.

Degree: 2005, Université de Montréal

Subjects/Keywords: IRPTC; PKC[bêta]1; Angiotensinogène; Hypertrophie; P27���¹

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APA (6th Edition):

Calvé, A. (2005). PKCbêta1 intervient dans l'action d'une concentration élevée en glucose sur l'expression de l'angiotensinogène et l'hypertrophie des IRPTC. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/15300

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Calvé, Annie. “PKCbêta1 intervient dans l'action d'une concentration élevée en glucose sur l'expression de l'angiotensinogène et l'hypertrophie des IRPTC.” 2005. Thesis, Université de Montréal. Accessed September 25, 2020. http://hdl.handle.net/1866/15300.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Calvé, Annie. “PKCbêta1 intervient dans l'action d'une concentration élevée en glucose sur l'expression de l'angiotensinogène et l'hypertrophie des IRPTC.” 2005. Web. 25 Sep 2020.

Vancouver:

Calvé A. PKCbêta1 intervient dans l'action d'une concentration élevée en glucose sur l'expression de l'angiotensinogène et l'hypertrophie des IRPTC. [Internet] [Thesis]. Université de Montréal; 2005. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/1866/15300.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Calvé A. PKCbêta1 intervient dans l'action d'une concentration élevée en glucose sur l'expression de l'angiotensinogène et l'hypertrophie des IRPTC. [Thesis]. Université de Montréal; 2005. Available from: http://hdl.handle.net/1866/15300

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

27. Chen, Tzu-Ping. Role of Skp2 in epithelial dysplasia and carcinoma of the cervix.

Degree: Master, Biological Sciences, 2003, NSYSU

 The F-box protein Skp2 (S-phase kinase associated protein 2) positively regulates the G1-S transition by controlling the cell cycle inhibitor p27Kip1. The p42/p44 mitogen-activated protein… (more)

Subjects/Keywords: cervical carcinoma; Skp2; p27 Kip1; MAP kinase

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APA (6th Edition):

Chen, T. (2003). Role of Skp2 in epithelial dysplasia and carcinoma of the cervix. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0909103-105834

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Tzu-Ping. “Role of Skp2 in epithelial dysplasia and carcinoma of the cervix.” 2003. Thesis, NSYSU. Accessed September 25, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0909103-105834.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Tzu-Ping. “Role of Skp2 in epithelial dysplasia and carcinoma of the cervix.” 2003. Web. 25 Sep 2020.

Vancouver:

Chen T. Role of Skp2 in epithelial dysplasia and carcinoma of the cervix. [Internet] [Thesis]. NSYSU; 2003. [cited 2020 Sep 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0909103-105834.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen T. Role of Skp2 in epithelial dysplasia and carcinoma of the cervix. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0909103-105834

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

28. Tsai, Feng-Lin. The role of p27kip1 in human malignant brain tumors.

Degree: Master, Biological Sciences, 2003, NSYSU

 Gliomas are the most common human brain tumors and are divided into four stages by WHO classification scheme. Benign gliomas are defined as grades I… (more)

Subjects/Keywords: GBM; astrocytomas; brain tumor; p-Akt; p27

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APA (6th Edition):

Tsai, F. (2003). The role of p27kip1 in human malignant brain tumors. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908103-235600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsai, Feng-Lin. “The role of p27kip1 in human malignant brain tumors.” 2003. Thesis, NSYSU. Accessed September 25, 2020. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908103-235600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsai, Feng-Lin. “The role of p27kip1 in human malignant brain tumors.” 2003. Web. 25 Sep 2020.

Vancouver:

Tsai F. The role of p27kip1 in human malignant brain tumors. [Internet] [Thesis]. NSYSU; 2003. [cited 2020 Sep 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908103-235600.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsai F. The role of p27kip1 in human malignant brain tumors. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0908103-235600

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Perez Madrigal, Diana. The Role of ERK5 in Cell Proliferation.

Degree: 2013, University of Manchester

 The extracellular signal-regulated protein kinase 5 (ERK5), also known as big mitogen-activated protein (MAP) kinase 1 (BMK1), is a non-redundant mitogen-activated protein kinase (MAPK) implicated… (more)

Subjects/Keywords: MAPKs; ERK5; p21; p27; Cell cycle

…dependent protein kinase (CDK) inhibitors, p21 and p27. As a result, low-level CDK2… …regulation of the miR-17-92 cluster. Concerning p27, ERK5 decreases p27 protein stability. The… …stabilisation of p27 in the absence of ERK5 resulted in the accumulation of the protein in the nucleus… …regulates p21 and p27 expression. Together with evidence that cancer patients with poor prognosis… …associated with increased p21 and p27 expression, two cell cycle inhibitors. On the one hand, ERK5… 

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APA (6th Edition):

Perez Madrigal, D. (2013). The Role of ERK5 in Cell Proliferation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:194097

Chicago Manual of Style (16th Edition):

Perez Madrigal, Diana. “The Role of ERK5 in Cell Proliferation.” 2013. Doctoral Dissertation, University of Manchester. Accessed September 25, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:194097.

MLA Handbook (7th Edition):

Perez Madrigal, Diana. “The Role of ERK5 in Cell Proliferation.” 2013. Web. 25 Sep 2020.

Vancouver:

Perez Madrigal D. The Role of ERK5 in Cell Proliferation. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2020 Sep 25]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:194097.

Council of Science Editors:

Perez Madrigal D. The Role of ERK5 in Cell Proliferation. [Doctoral Dissertation]. University of Manchester; 2013. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:194097

30. Wang, Ying. Mouse Models of Menopause and Ovarian Cancer Risks.

Degree: PhD, Molecular Cell and Developmental Biology (Medicine), 2011, University of Miami

 Epithelial ovarian cancer (EOC) is the leading cause of death from gynecological malignancy in Western countries. A better understanding of the etiology and risk factors… (more)

Subjects/Keywords: Ovarian Cancer; Mouse Model; p27; p53

…Wv:p53 double mutant mice ... . ….73 5.5 Metaplastic potential of p27-/- and p53… …MOSE cells ...…77 5.6 Compensation of CKI proteins associated with p27 and p53… …tubular adenoma in Wv/Wv ovaries ...............…..13 Figure 2 IHC staining of p27 in human… …ovarian tumor samples ….…. …..17 Figure 3 Pre-neoplastic changes on the surface of aged p27… …ovaries …...…...….19 Figure 4 Pre-neoplastic changes in the p27-/- ovaries… 

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APA (6th Edition):

Wang, Y. (2011). Mouse Models of Menopause and Ovarian Cancer Risks. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/681

Chicago Manual of Style (16th Edition):

Wang, Ying. “Mouse Models of Menopause and Ovarian Cancer Risks.” 2011. Doctoral Dissertation, University of Miami. Accessed September 25, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/681.

MLA Handbook (7th Edition):

Wang, Ying. “Mouse Models of Menopause and Ovarian Cancer Risks.” 2011. Web. 25 Sep 2020.

Vancouver:

Wang Y. Mouse Models of Menopause and Ovarian Cancer Risks. [Internet] [Doctoral dissertation]. University of Miami; 2011. [cited 2020 Sep 25]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/681.

Council of Science Editors:

Wang Y. Mouse Models of Menopause and Ovarian Cancer Risks. [Doctoral Dissertation]. University of Miami; 2011. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/681

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