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You searched for subject:(oligodendrocytes). Showing records 1 – 30 of 66 total matches.

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University of Edinburgh

1. Swire, Matthew. Investigating endothelin receptor B signalling during myelination.

Degree: PhD, 2017, University of Edinburgh

 A key process enabling the correct functioning of neural circuits involves the formation of multi‐layered membranous myelin sheaths around axons. Myelin sheaths, made by specialised… (more)

Subjects/Keywords: oligodendrocytes; myelination; EDNRB

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APA (6th Edition):

Swire, M. (2017). Investigating endothelin receptor B signalling during myelination. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28912

Chicago Manual of Style (16th Edition):

Swire, Matthew. “Investigating endothelin receptor B signalling during myelination.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed October 21, 2018. http://hdl.handle.net/1842/28912.

MLA Handbook (7th Edition):

Swire, Matthew. “Investigating endothelin receptor B signalling during myelination.” 2017. Web. 21 Oct 2018.

Vancouver:

Swire M. Investigating endothelin receptor B signalling during myelination. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/1842/28912.

Council of Science Editors:

Swire M. Investigating endothelin receptor B signalling during myelination. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28912


Penn State University

2. Todoric, Bozo. ROLES OF FERRITIN RECEPTOR AND ITS LIGANDS.

Degree: PhD, Cell and Molecular Biology, 2009, Penn State University

 Oligodendrocyte progenitor cells (OPCs) originate in the subventricular zone (SVZ) and populate the developing brain, proliferating and differentiating into myelin producing oligodendrocytes in early post-natal… (more)

Subjects/Keywords: iron; demyelinating disease; oligodendrocytes

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APA (6th Edition):

Todoric, B. (2009). ROLES OF FERRITIN RECEPTOR AND ITS LIGANDS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/10052

Chicago Manual of Style (16th Edition):

Todoric, Bozo. “ROLES OF FERRITIN RECEPTOR AND ITS LIGANDS.” 2009. Doctoral Dissertation, Penn State University. Accessed October 21, 2018. https://etda.libraries.psu.edu/catalog/10052.

MLA Handbook (7th Edition):

Todoric, Bozo. “ROLES OF FERRITIN RECEPTOR AND ITS LIGANDS.” 2009. Web. 21 Oct 2018.

Vancouver:

Todoric B. ROLES OF FERRITIN RECEPTOR AND ITS LIGANDS. [Internet] [Doctoral dissertation]. Penn State University; 2009. [cited 2018 Oct 21]. Available from: https://etda.libraries.psu.edu/catalog/10052.

Council of Science Editors:

Todoric B. ROLES OF FERRITIN RECEPTOR AND ITS LIGANDS. [Doctoral Dissertation]. Penn State University; 2009. Available from: https://etda.libraries.psu.edu/catalog/10052


University of Pennsylvania

3. Feigenson, Keith. The Role of Wnt Signaling in Oligodendrocyte Development.

Degree: 2011, University of Pennsylvania

 Abstract The Role of Wnt Signaling in Oligodendrocyte Development Keith Feigenson Judith Grinspan, Ph.D. The developmental regulation of the central nervous system is established through… (more)

Subjects/Keywords: Wnt; Development; Myelin; Oligodendrocytes; BMP

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APA (6th Edition):

Feigenson, K. (2011). The Role of Wnt Signaling in Oligodendrocyte Development. (Thesis). University of Pennsylvania. Retrieved from http://repository.upenn.edu/edissertations/1544

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feigenson, Keith. “The Role of Wnt Signaling in Oligodendrocyte Development.” 2011. Thesis, University of Pennsylvania. Accessed October 21, 2018. http://repository.upenn.edu/edissertations/1544.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feigenson, Keith. “The Role of Wnt Signaling in Oligodendrocyte Development.” 2011. Web. 21 Oct 2018.

Vancouver:

Feigenson K. The Role of Wnt Signaling in Oligodendrocyte Development. [Internet] [Thesis]. University of Pennsylvania; 2011. [cited 2018 Oct 21]. Available from: http://repository.upenn.edu/edissertations/1544.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feigenson K. The Role of Wnt Signaling in Oligodendrocyte Development. [Thesis]. University of Pennsylvania; 2011. Available from: http://repository.upenn.edu/edissertations/1544

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

4. Zhang, Naibo. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.

Degree: MS, Biochemistry and Molecular Biology, 2015, University of Southern California

 Multiple Sclerosis (MS) is a CNS degenerative disease. It is one of the most common chronic disabling CNS disease in young adults. In MS, oligodendrocytes(OLs)… (more)

Subjects/Keywords: multiple sclerosis; oligodendrocytes; remyelination

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APA (6th Edition):

Zhang, N. (2015). Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6319

Chicago Manual of Style (16th Edition):

Zhang, Naibo. “Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.” 2015. Masters Thesis, University of Southern California. Accessed October 21, 2018. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6319.

MLA Handbook (7th Edition):

Zhang, Naibo. “Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination.” 2015. Web. 21 Oct 2018.

Vancouver:

Zhang N. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2018 Oct 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6319.

Council of Science Editors:

Zhang N. Targeting Ryk-signaling to attenuate multiple sclerosis by promoting oligodendrocyte differentiation and myelination. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/615832/rec/6319

5. Marie, Corentine. The role of Chd7 & Chd8 chromatin remodelers in oligodendrogenesis and (re)myelination : Le rôle de Chd7 & Chd8 facteurs du remodelage de la chromatine dans l'oligodendrogenese et la (re)myélinisation.

Degree: Docteur es, Neurosciences, 2017, Université Pierre et Marie Curie – Paris VI

Les oligodendrocytes (OLs) sont les cellules myélinisantes du système nerveux central, s’enroulant autour des axones et permettant la conduction saltatoire du potentiel d’action. Dans la… (more)

Subjects/Keywords: Oligodendrocytes; Développement; Différenciation; Remodelage; Chromatine; Chd7; Oligodendrocytes; Chromatin; Chd7; 573.86

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APA (6th Edition):

Marie, C. (2017). The role of Chd7 & Chd8 chromatin remodelers in oligodendrogenesis and (re)myelination : Le rôle de Chd7 & Chd8 facteurs du remodelage de la chromatine dans l'oligodendrogenese et la (re)myélinisation. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2017PA066365

Chicago Manual of Style (16th Edition):

Marie, Corentine. “The role of Chd7 & Chd8 chromatin remodelers in oligodendrogenesis and (re)myelination : Le rôle de Chd7 & Chd8 facteurs du remodelage de la chromatine dans l'oligodendrogenese et la (re)myélinisation.” 2017. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 21, 2018. http://www.theses.fr/2017PA066365.

MLA Handbook (7th Edition):

Marie, Corentine. “The role of Chd7 & Chd8 chromatin remodelers in oligodendrogenesis and (re)myelination : Le rôle de Chd7 & Chd8 facteurs du remodelage de la chromatine dans l'oligodendrogenese et la (re)myélinisation.” 2017. Web. 21 Oct 2018.

Vancouver:

Marie C. The role of Chd7 & Chd8 chromatin remodelers in oligodendrogenesis and (re)myelination : Le rôle de Chd7 & Chd8 facteurs du remodelage de la chromatine dans l'oligodendrogenese et la (re)myélinisation. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. [cited 2018 Oct 21]. Available from: http://www.theses.fr/2017PA066365.

Council of Science Editors:

Marie C. The role of Chd7 & Chd8 chromatin remodelers in oligodendrogenesis and (re)myelination : Le rôle de Chd7 & Chd8 facteurs du remodelage de la chromatine dans l'oligodendrogenese et la (re)myélinisation. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2017. Available from: http://www.theses.fr/2017PA066365


Universiteit Utrecht

6. Lee, M.C. van der. The Role of Glia in Parkinson's Disease.

Degree: 2015, Universiteit Utrecht

 Parkinson’s disease (PD) is a progressive neurodegenerative disorder which is characterized by motor symptoms such as tremor, bradykinesia, rigidity, and postural instability. In addition to… (more)

Subjects/Keywords: Parkinson's disease; Glial cells; Microglia; Astrocytes; Oligodendrocytes

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APA (6th Edition):

Lee, M. C. v. d. (2015). The Role of Glia in Parkinson's Disease. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/310719

Chicago Manual of Style (16th Edition):

Lee, M C van der. “The Role of Glia in Parkinson's Disease.” 2015. Masters Thesis, Universiteit Utrecht. Accessed October 21, 2018. http://dspace.library.uu.nl:8080/handle/1874/310719.

MLA Handbook (7th Edition):

Lee, M C van der. “The Role of Glia in Parkinson's Disease.” 2015. Web. 21 Oct 2018.

Vancouver:

Lee MCvd. The Role of Glia in Parkinson's Disease. [Internet] [Masters thesis]. Universiteit Utrecht; 2015. [cited 2018 Oct 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/310719.

Council of Science Editors:

Lee MCvd. The Role of Glia in Parkinson's Disease. [Masters Thesis]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/310719


University of Miami

7. Hackett, Amber R. The Role of NG2 Cells after Contusive Spinal Cord Injury.

Degree: PhD, Neuroscience (Medicine), 2016, University of Miami

 NG2 cells, also known as oligodendrocyte progenitor cells (OPCs), are tiled throughout the adult central nervous system (CNS) and proliferate and differentiate into oligodendrocytes throughout… (more)

Subjects/Keywords: Spinal Cord Injury; Glial Scar; Oligodendrocytes; Cytokines

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APA (6th Edition):

Hackett, A. R. (2016). The Role of NG2 Cells after Contusive Spinal Cord Injury. (Doctoral Dissertation). University of Miami. Retrieved from http://scholarlyrepository.miami.edu/oa_dissertations/1767

Chicago Manual of Style (16th Edition):

Hackett, Amber R. “The Role of NG2 Cells after Contusive Spinal Cord Injury.” 2016. Doctoral Dissertation, University of Miami. Accessed October 21, 2018. http://scholarlyrepository.miami.edu/oa_dissertations/1767.

MLA Handbook (7th Edition):

Hackett, Amber R. “The Role of NG2 Cells after Contusive Spinal Cord Injury.” 2016. Web. 21 Oct 2018.

Vancouver:

Hackett AR. The Role of NG2 Cells after Contusive Spinal Cord Injury. [Internet] [Doctoral dissertation]. University of Miami; 2016. [cited 2018 Oct 21]. Available from: http://scholarlyrepository.miami.edu/oa_dissertations/1767.

Council of Science Editors:

Hackett AR. The Role of NG2 Cells after Contusive Spinal Cord Injury. [Doctoral Dissertation]. University of Miami; 2016. Available from: http://scholarlyrepository.miami.edu/oa_dissertations/1767


Harvard University

8. Han, Juliette. The Role of TSC in Oligodendrocyte Differentiation and Myelination.

Degree: PhD, Neurobiology, 2012, Harvard University

 Tuberous Sclerosis Complex (TSC) is an autosomal dominant syndrome characterized by epilepsy, intellectual disability, and autism. Recent studies have suggested that white matter abnormalities, including… (more)

Subjects/Keywords: autism; CTGF; myelin; oligodendrocytes; TSC; neurosciences

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APA (6th Edition):

Han, J. (2012). The Role of TSC in Oligodendrocyte Differentiation and Myelination. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128

Chicago Manual of Style (16th Edition):

Han, Juliette. “The Role of TSC in Oligodendrocyte Differentiation and Myelination.” 2012. Doctoral Dissertation, Harvard University. Accessed October 21, 2018. http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128.

MLA Handbook (7th Edition):

Han, Juliette. “The Role of TSC in Oligodendrocyte Differentiation and Myelination.” 2012. Web. 21 Oct 2018.

Vancouver:

Han J. The Role of TSC in Oligodendrocyte Differentiation and Myelination. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2018 Oct 21]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128.

Council of Science Editors:

Han J. The Role of TSC in Oligodendrocyte Differentiation and Myelination. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128


Texas A&M University

9. Thompson, Jeffrey. The Role of Caspase-8 in Oligodendrocyte Development and Mechanisms of Oxidative Injury in Neurons and Glia.

Degree: 2012, Texas A&M University

 Apoptosis is essential not only to the normal development of a multicellular organism but also for the maintenance of tissue homeostasis. This proposal seeks to… (more)

Subjects/Keywords: Necroptosis; RIP-1; Oxidative Stress; Oligodendrocytes; Apoptosis

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APA (6th Edition):

Thompson, J. (2012). The Role of Caspase-8 in Oligodendrocyte Development and Mechanisms of Oxidative Injury in Neurons and Glia. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thompson, Jeffrey. “The Role of Caspase-8 in Oligodendrocyte Development and Mechanisms of Oxidative Injury in Neurons and Glia.” 2012. Thesis, Texas A&M University. Accessed October 21, 2018. http://hdl.handle.net/1969.1/148320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thompson, Jeffrey. “The Role of Caspase-8 in Oligodendrocyte Development and Mechanisms of Oxidative Injury in Neurons and Glia.” 2012. Web. 21 Oct 2018.

Vancouver:

Thompson J. The Role of Caspase-8 in Oligodendrocyte Development and Mechanisms of Oxidative Injury in Neurons and Glia. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/1969.1/148320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thompson J. The Role of Caspase-8 in Oligodendrocyte Development and Mechanisms of Oxidative Injury in Neurons and Glia. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Mozafari, Sabah. Characterization of neural precursors derived from iPSCs in vitro and in vivo after transplantation into the demyelinated central nervous system : Caractérisation des précurseurs neuraux dérivés de cellules pluripotentes induites in vitro et in vivo après transplantation dans le système nerveux central démyélinisé.

Degree: Docteur es, Neurosciences, 2016, Université Pierre et Marie Curie – Paris VI

Les précurseurs neuraux dérivés de cellules souches pluripotentes induites (iPS-NPCs) peuvent représenter la source cellulaire autologue idéale pour la thérapie cellulaire visant à promouvoir la… (more)

Subjects/Keywords: Oligodendrocytes; Remyélinisation; Cellules pluripotentes; Transplantation; Précurseurs neuraux; Souris; Humain; Remyelination; Induced pluripotent stem cells; Oligodendrocytes; 612.8

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APA (6th Edition):

Mozafari, S. (2016). Characterization of neural precursors derived from iPSCs in vitro and in vivo after transplantation into the demyelinated central nervous system : Caractérisation des précurseurs neuraux dérivés de cellules pluripotentes induites in vitro et in vivo après transplantation dans le système nerveux central démyélinisé. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066091

Chicago Manual of Style (16th Edition):

Mozafari, Sabah. “Characterization of neural precursors derived from iPSCs in vitro and in vivo after transplantation into the demyelinated central nervous system : Caractérisation des précurseurs neuraux dérivés de cellules pluripotentes induites in vitro et in vivo après transplantation dans le système nerveux central démyélinisé.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed October 21, 2018. http://www.theses.fr/2016PA066091.

MLA Handbook (7th Edition):

Mozafari, Sabah. “Characterization of neural precursors derived from iPSCs in vitro and in vivo after transplantation into the demyelinated central nervous system : Caractérisation des précurseurs neuraux dérivés de cellules pluripotentes induites in vitro et in vivo après transplantation dans le système nerveux central démyélinisé.” 2016. Web. 21 Oct 2018.

Vancouver:

Mozafari S. Characterization of neural precursors derived from iPSCs in vitro and in vivo after transplantation into the demyelinated central nervous system : Caractérisation des précurseurs neuraux dérivés de cellules pluripotentes induites in vitro et in vivo après transplantation dans le système nerveux central démyélinisé. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2018 Oct 21]. Available from: http://www.theses.fr/2016PA066091.

Council of Science Editors:

Mozafari S. Characterization of neural precursors derived from iPSCs in vitro and in vivo after transplantation into the demyelinated central nervous system : Caractérisation des précurseurs neuraux dérivés de cellules pluripotentes induites in vitro et in vivo après transplantation dans le système nerveux central démyélinisé. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066091

11. Macchi, Magali. Etude du rôle des héparans sulfates protéoglycanes dans la mobilisation post-lesionnelle des progéniteurs oligodendrocytaires chez la souris adulte : Role of heparan sulphate proteoglycans in post-lesional mobilization of oligodendrocyte prgenitor cells in adult mice.

Degree: Docteur es, Biologie du dévéloppement, 2015, Aix Marseille Université

La production physiologique continue de cellules myélinisantes dans le système nerveux (SN) de mammifère offre de nouvelles perspectives thérapeutiques. Lors d’une atteinte de la myéline,… (more)

Subjects/Keywords: Mobilisation; Cntf; Heparan Sulfate; Oligodendrocytes; Régénération de la myéline; Mobilization; Cntf; Heparan Sulphate; Oligodendrocytes; Myelin regeneration; 571

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APA (6th Edition):

Macchi, M. (2015). Etude du rôle des héparans sulfates protéoglycanes dans la mobilisation post-lesionnelle des progéniteurs oligodendrocytaires chez la souris adulte : Role of heparan sulphate proteoglycans in post-lesional mobilization of oligodendrocyte prgenitor cells in adult mice. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2015AIXM4069

Chicago Manual of Style (16th Edition):

Macchi, Magali. “Etude du rôle des héparans sulfates protéoglycanes dans la mobilisation post-lesionnelle des progéniteurs oligodendrocytaires chez la souris adulte : Role of heparan sulphate proteoglycans in post-lesional mobilization of oligodendrocyte prgenitor cells in adult mice.” 2015. Doctoral Dissertation, Aix Marseille Université. Accessed October 21, 2018. http://www.theses.fr/2015AIXM4069.

MLA Handbook (7th Edition):

Macchi, Magali. “Etude du rôle des héparans sulfates protéoglycanes dans la mobilisation post-lesionnelle des progéniteurs oligodendrocytaires chez la souris adulte : Role of heparan sulphate proteoglycans in post-lesional mobilization of oligodendrocyte prgenitor cells in adult mice.” 2015. Web. 21 Oct 2018.

Vancouver:

Macchi M. Etude du rôle des héparans sulfates protéoglycanes dans la mobilisation post-lesionnelle des progéniteurs oligodendrocytaires chez la souris adulte : Role of heparan sulphate proteoglycans in post-lesional mobilization of oligodendrocyte prgenitor cells in adult mice. [Internet] [Doctoral dissertation]. Aix Marseille Université 2015. [cited 2018 Oct 21]. Available from: http://www.theses.fr/2015AIXM4069.

Council of Science Editors:

Macchi M. Etude du rôle des héparans sulfates protéoglycanes dans la mobilisation post-lesionnelle des progéniteurs oligodendrocytaires chez la souris adulte : Role of heparan sulphate proteoglycans in post-lesional mobilization of oligodendrocyte prgenitor cells in adult mice. [Doctoral Dissertation]. Aix Marseille Université 2015. Available from: http://www.theses.fr/2015AIXM4069


Université Paris-Sud – Paris XI

12. Hussain, Rashad. Rôle des androgènes et progestagènes dans la remyélinisation du système nerveux central : Role of androgens and progestagens in remyelination of central nervous system.

Degree: Docteur es, Neurosciences, 2011, Université Paris-Sud – Paris XI

La sclérose en plaques (SEP) est une maladie démyélinisante dont les causes ne sont pas encore bien élucidées. Cependant, l’implication des réponses auto-immunes dans la… (more)

Subjects/Keywords: Myéline; Oligodendrocytes; Testostérone; Remyélinisation; Récepteur des androgènes; ARNesCre; Progestérone; Estradiol-17β; Myelin; Oligodendrocytes; Testosterone; Remyelination; Remyelination; ARNesCre; Progesterone; Estradiol-17β

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APA (6th Edition):

Hussain, R. (2011). Rôle des androgènes et progestagènes dans la remyélinisation du système nerveux central : Role of androgens and progestagens in remyelination of central nervous system. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA11T081

Chicago Manual of Style (16th Edition):

Hussain, Rashad. “Rôle des androgènes et progestagènes dans la remyélinisation du système nerveux central : Role of androgens and progestagens in remyelination of central nervous system.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed October 21, 2018. http://www.theses.fr/2011PA11T081.

MLA Handbook (7th Edition):

Hussain, Rashad. “Rôle des androgènes et progestagènes dans la remyélinisation du système nerveux central : Role of androgens and progestagens in remyelination of central nervous system.” 2011. Web. 21 Oct 2018.

Vancouver:

Hussain R. Rôle des androgènes et progestagènes dans la remyélinisation du système nerveux central : Role of androgens and progestagens in remyelination of central nervous system. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2018 Oct 21]. Available from: http://www.theses.fr/2011PA11T081.

Council of Science Editors:

Hussain R. Rôle des androgènes et progestagènes dans la remyélinisation du système nerveux central : Role of androgens and progestagens in remyelination of central nervous system. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA11T081

13. Lu, Puo Yuan. Serum Response Factor Regulates Multiple Aspects of Central Nervous System Development.

Degree: PhD, Biology and Biomedical Sciences: Neurosciences, 2012, Washington University in St. Louis

 SRF is a highly evolutionary conserved activity-dependent transcription factor. Previous studies have shown that neuron-specific deletion of SRF results in deficits in tangential cell migration,… (more)

Subjects/Keywords: Neurosciences; Developmental biology; Astrocytes; Differentiation; Neuronal projection; Oligodendrocytes; SRF; Transcription

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APA (6th Edition):

Lu, P. Y. (2012). Serum Response Factor Regulates Multiple Aspects of Central Nervous System Development. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/etd/714

Chicago Manual of Style (16th Edition):

Lu, Puo Yuan. “Serum Response Factor Regulates Multiple Aspects of Central Nervous System Development.” 2012. Doctoral Dissertation, Washington University in St. Louis. Accessed October 21, 2018. https://openscholarship.wustl.edu/etd/714.

MLA Handbook (7th Edition):

Lu, Puo Yuan. “Serum Response Factor Regulates Multiple Aspects of Central Nervous System Development.” 2012. Web. 21 Oct 2018.

Vancouver:

Lu PY. Serum Response Factor Regulates Multiple Aspects of Central Nervous System Development. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2012. [cited 2018 Oct 21]. Available from: https://openscholarship.wustl.edu/etd/714.

Council of Science Editors:

Lu PY. Serum Response Factor Regulates Multiple Aspects of Central Nervous System Development. [Doctoral Dissertation]. Washington University in St. Louis; 2012. Available from: https://openscholarship.wustl.edu/etd/714

14. Greca, Rossana. Ruolo della PARP nello stress ossidativo.

Degree: 2012, Università degli Studi di Catania

 Abstract Poly(ADP-ribose) polymerases (PARPs) play a crucial role in DNA damage surveillance through their nick sensor functions. Since PARPs over activation leads to an excessive… (more)

Subjects/Keywords: Area 01 - Scienze matematiche e informatiche; Keywords Astrocytes Oligodendrocytes PARP

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APA (6th Edition):

Greca, R. (2012). Ruolo della PARP nello stress ossidativo. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Greca, Rossana. “Ruolo della PARP nello stress ossidativo.” 2012. Thesis, Università degli Studi di Catania. Accessed October 21, 2018. http://hdl.handle.net/10761/1043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Greca, Rossana. “Ruolo della PARP nello stress ossidativo.” 2012. Web. 21 Oct 2018.

Vancouver:

Greca R. Ruolo della PARP nello stress ossidativo. [Internet] [Thesis]. Università degli Studi di Catania; 2012. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/10761/1043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Greca R. Ruolo della PARP nello stress ossidativo. [Thesis]. Università degli Studi di Catania; 2012. Available from: http://hdl.handle.net/10761/1043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Graziano, Adriana Carol Eleonora. Molecular mechanisms involved in krabbe s disease.

Degree: 2014, Università degli Studi di Catania

 Krabbe disease is an autosomal recessive disorder resulting in the deficiency of galactosilcerebrosidase (GALC), a lysosomal enzyme involved in the catabolism of two lipids extremely… (more)

Subjects/Keywords: Area 05 - Scienze biologiche; apoptosis, cell culture, connexin, leukodistrophy, oligodendrocytes, psychosine

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APA (6th Edition):

Graziano, A. C. E. (2014). Molecular mechanisms involved in krabbe s disease. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Graziano, Adriana Carol Eleonora. “Molecular mechanisms involved in krabbe s disease.” 2014. Thesis, Università degli Studi di Catania. Accessed October 21, 2018. http://hdl.handle.net/10761/1507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Graziano, Adriana Carol Eleonora. “Molecular mechanisms involved in krabbe s disease.” 2014. Web. 21 Oct 2018.

Vancouver:

Graziano ACE. Molecular mechanisms involved in krabbe s disease. [Internet] [Thesis]. Università degli Studi di Catania; 2014. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/10761/1507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Graziano ACE. Molecular mechanisms involved in krabbe s disease. [Thesis]. Università degli Studi di Catania; 2014. Available from: http://hdl.handle.net/10761/1507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

16. Ford, Catriona Barbara. CX3CR1/CX3CL1 axis drives the migration and maturation of oligodendroglia in the central nervous system.

Degree: PhD, 2017, University of Edinburgh

 In the central nervous system, the axons of neurons are protected from damage and aided in electrical conductivity by the myelin sheath, a complex of… (more)

Subjects/Keywords: oligodendrocytes; fractalkine; OPC; CX3CL1; CX3CR1; migration; regeneration; stem cells

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APA (6th Edition):

Ford, C. B. (2017). CX3CR1/CX3CL1 axis drives the migration and maturation of oligodendroglia in the central nervous system. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/29533

Chicago Manual of Style (16th Edition):

Ford, Catriona Barbara. “CX3CR1/CX3CL1 axis drives the migration and maturation of oligodendroglia in the central nervous system.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed October 21, 2018. http://hdl.handle.net/1842/29533.

MLA Handbook (7th Edition):

Ford, Catriona Barbara. “CX3CR1/CX3CL1 axis drives the migration and maturation of oligodendroglia in the central nervous system.” 2017. Web. 21 Oct 2018.

Vancouver:

Ford CB. CX3CR1/CX3CL1 axis drives the migration and maturation of oligodendroglia in the central nervous system. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/1842/29533.

Council of Science Editors:

Ford CB. CX3CR1/CX3CL1 axis drives the migration and maturation of oligodendroglia in the central nervous system. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/29533


University of Edinburgh

17. Cleary, Elaine Marie. Effect of C9orf72 hexanucleotide repeat expansions on human induced pluripotent stem cell derived oligodendrocytes.

Degree: PhD, 2017, University of Edinburgh

 A hexanucleotide repeat expansion in the C9orf72 gene is the most common cause of familial amyotrophic lateral sclerosis and frontotemporal dementia. Genetic testing for this… (more)

Subjects/Keywords: C9orf72 gene; amyotrophic lateral sclerosis; frontotemporal dementia; screening; PCR assays; oligodendrocytes

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APA (6th Edition):

Cleary, E. M. (2017). Effect of C9orf72 hexanucleotide repeat expansions on human induced pluripotent stem cell derived oligodendrocytes. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/28816

Chicago Manual of Style (16th Edition):

Cleary, Elaine Marie. “Effect of C9orf72 hexanucleotide repeat expansions on human induced pluripotent stem cell derived oligodendrocytes.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed October 21, 2018. http://hdl.handle.net/1842/28816.

MLA Handbook (7th Edition):

Cleary, Elaine Marie. “Effect of C9orf72 hexanucleotide repeat expansions on human induced pluripotent stem cell derived oligodendrocytes.” 2017. Web. 21 Oct 2018.

Vancouver:

Cleary EM. Effect of C9orf72 hexanucleotide repeat expansions on human induced pluripotent stem cell derived oligodendrocytes. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/1842/28816.

Council of Science Editors:

Cleary EM. Effect of C9orf72 hexanucleotide repeat expansions on human induced pluripotent stem cell derived oligodendrocytes. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/28816


University of Edinburgh

18. Burns, Alasdair Philip. Influence of regulatory T cells on remyelination in the central nervous system.

Degree: 2013, University of Edinburgh

 The aim of this project is to study whether Regulatory T cells (Treg) influence remyelination following a demyelinating insult of the central nervous system (CNS).… (more)

Subjects/Keywords: Remyelination; TREG; Regulatory T cells; Oligodendrocytes; Leukaemia inhibitory factor; LIF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Burns, A. P. (2013). Influence of regulatory T cells on remyelination in the central nervous system. (Thesis). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Burns, Alasdair Philip. “Influence of regulatory T cells on remyelination in the central nervous system.” 2013. Thesis, University of Edinburgh. Accessed October 21, 2018. http://hdl.handle.net/1842/8831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Burns, Alasdair Philip. “Influence of regulatory T cells on remyelination in the central nervous system.” 2013. Web. 21 Oct 2018.

Vancouver:

Burns AP. Influence of regulatory T cells on remyelination in the central nervous system. [Internet] [Thesis]. University of Edinburgh; 2013. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/1842/8831.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Burns AP. Influence of regulatory T cells on remyelination in the central nervous system. [Thesis]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/8831

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

19. Feldmann, Anke. Funktionelle Rolle von SNARE-Proteinen bei der Myelinisierung.

Degree: 2009, Johannes Gutenberg Universität Mainz

 Die Hauptfunktion der Oligodendrozyten im zentralen Nervensystem ist die Myelinisierung. Dabei umwickelt die Zelle mit Ausläufern ihrer Plasmamembran mehrmals die Axone. In den Phasen der… (more)

Subjects/Keywords: SNARE, PLP, Oligodendrozyten, VAMP; SNARE, PLP, oligodendrocytes, VAMP; Life sciences

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APA (6th Edition):

Feldmann, A. (2009). Funktionelle Rolle von SNARE-Proteinen bei der Myelinisierung. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2009/1937/

Chicago Manual of Style (16th Edition):

Feldmann, Anke. “Funktionelle Rolle von SNARE-Proteinen bei der Myelinisierung.” 2009. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed October 21, 2018. http://ubm.opus.hbz-nrw.de/volltexte/2009/1937/.

MLA Handbook (7th Edition):

Feldmann, Anke. “Funktionelle Rolle von SNARE-Proteinen bei der Myelinisierung.” 2009. Web. 21 Oct 2018.

Vancouver:

Feldmann A. Funktionelle Rolle von SNARE-Proteinen bei der Myelinisierung. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2009. [cited 2018 Oct 21]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2009/1937/.

Council of Science Editors:

Feldmann A. Funktionelle Rolle von SNARE-Proteinen bei der Myelinisierung. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2009. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2009/1937/


University of Louisville

20. Dincman, Toros Ali, 1982-. Histone deacetylase inhibition-mediated cytotoxicity of oligodendrocytes.

Degree: PhD, 2011, University of Louisville

 Recent studies indicate post-translational deacetylation by members of the superfamily of histone deacetylase complexes (HDACs) is necessary for oligodendrocyte precursor cell (OPC) differentiation into mature… (more)

Subjects/Keywords: Oligodendrocytes; OPC; Histone deacetylase; HDAC inhibition; Cell culture; DNA damage

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APA (6th Edition):

Dincman, Toros Ali, 1. (2011). Histone deacetylase inhibition-mediated cytotoxicity of oligodendrocytes. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/351 ; https://ir.library.louisville.edu/etd/351

Chicago Manual of Style (16th Edition):

Dincman, Toros Ali, 1982-. “Histone deacetylase inhibition-mediated cytotoxicity of oligodendrocytes.” 2011. Doctoral Dissertation, University of Louisville. Accessed October 21, 2018. 10.18297/etd/351 ; https://ir.library.louisville.edu/etd/351.

MLA Handbook (7th Edition):

Dincman, Toros Ali, 1982-. “Histone deacetylase inhibition-mediated cytotoxicity of oligodendrocytes.” 2011. Web. 21 Oct 2018.

Vancouver:

Dincman, Toros Ali 1. Histone deacetylase inhibition-mediated cytotoxicity of oligodendrocytes. [Internet] [Doctoral dissertation]. University of Louisville; 2011. [cited 2018 Oct 21]. Available from: 10.18297/etd/351 ; https://ir.library.louisville.edu/etd/351.

Council of Science Editors:

Dincman, Toros Ali 1. Histone deacetylase inhibition-mediated cytotoxicity of oligodendrocytes. [Doctoral Dissertation]. University of Louisville; 2011. Available from: 10.18297/etd/351 ; https://ir.library.louisville.edu/etd/351


University of Toronto

21. Rowland, James W. Generation and Characterization of Neural Stem Cells Derived from Embryonic Stem Cells using the Default Mechanism.

Degree: 2011, University of Toronto

In embryonic stem cells (ESCs) neural differentiation is elicited in the absence of extrinsic signaling in minimal conditions. This ‘default mechanism’ in ESCs produces neural… (more)

Subjects/Keywords: Neural Stem Cells; Oligodendrocytes; Myelin; Differentiation; 0317; 0379

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APA (6th Edition):

Rowland, J. W. (2011). Generation and Characterization of Neural Stem Cells Derived from Embryonic Stem Cells using the Default Mechanism. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/31415

Chicago Manual of Style (16th Edition):

Rowland, James W. “Generation and Characterization of Neural Stem Cells Derived from Embryonic Stem Cells using the Default Mechanism.” 2011. Masters Thesis, University of Toronto. Accessed October 21, 2018. http://hdl.handle.net/1807/31415.

MLA Handbook (7th Edition):

Rowland, James W. “Generation and Characterization of Neural Stem Cells Derived from Embryonic Stem Cells using the Default Mechanism.” 2011. Web. 21 Oct 2018.

Vancouver:

Rowland JW. Generation and Characterization of Neural Stem Cells Derived from Embryonic Stem Cells using the Default Mechanism. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/1807/31415.

Council of Science Editors:

Rowland JW. Generation and Characterization of Neural Stem Cells Derived from Embryonic Stem Cells using the Default Mechanism. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31415

22. Wheeler, Natalie A. Autotaxin in Central Nervous System Development and Disease.

Degree: PhD, Neuroscience, 2016, Virginia Commonwealth University

  During development, oligodendrocytes (OLGs), the myelinating cells of the central nervous system (CNS), undergo a stepwise progression during which OLG progenitors, specified from neural… (more)

Subjects/Keywords: Oligodendrocytes; Myelin; Autotaxin; LPA; Tat; HDAC; Multiple Sclerosis; HIV-1; Neurosciences

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APA (6th Edition):

Wheeler, N. A. (2016). Autotaxin in Central Nervous System Development and Disease. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/4104

Chicago Manual of Style (16th Edition):

Wheeler, Natalie A. “Autotaxin in Central Nervous System Development and Disease.” 2016. Doctoral Dissertation, Virginia Commonwealth University. Accessed October 21, 2018. https://scholarscompass.vcu.edu/etd/4104.

MLA Handbook (7th Edition):

Wheeler, Natalie A. “Autotaxin in Central Nervous System Development and Disease.” 2016. Web. 21 Oct 2018.

Vancouver:

Wheeler NA. Autotaxin in Central Nervous System Development and Disease. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2016. [cited 2018 Oct 21]. Available from: https://scholarscompass.vcu.edu/etd/4104.

Council of Science Editors:

Wheeler NA. Autotaxin in Central Nervous System Development and Disease. [Doctoral Dissertation]. Virginia Commonwealth University; 2016. Available from: https://scholarscompass.vcu.edu/etd/4104


Boston University

23. Townsend-Shobin, Eli. Myelin and glial pathology in aging and congnitive decline: evidence for faulty myelin clearance in the rhesus monkey.

Degree: PhD, Neuroscience, 2018, Boston University

 Aging is associated with a loss of cognitive function related to learning, memory, and executive function with varying severity. Although there is no age-related loss… (more)

Subjects/Keywords: Aging; Immunology; Macrophage; Microglia; Myelin; Oligodendrocytes; White matter

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APA (6th Edition):

Townsend-Shobin, E. (2018). Myelin and glial pathology in aging and congnitive decline: evidence for faulty myelin clearance in the rhesus monkey. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/29985

Chicago Manual of Style (16th Edition):

Townsend-Shobin, Eli. “Myelin and glial pathology in aging and congnitive decline: evidence for faulty myelin clearance in the rhesus monkey.” 2018. Doctoral Dissertation, Boston University. Accessed October 21, 2018. http://hdl.handle.net/2144/29985.

MLA Handbook (7th Edition):

Townsend-Shobin, Eli. “Myelin and glial pathology in aging and congnitive decline: evidence for faulty myelin clearance in the rhesus monkey.” 2018. Web. 21 Oct 2018.

Vancouver:

Townsend-Shobin E. Myelin and glial pathology in aging and congnitive decline: evidence for faulty myelin clearance in the rhesus monkey. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/2144/29985.

Council of Science Editors:

Townsend-Shobin E. Myelin and glial pathology in aging and congnitive decline: evidence for faulty myelin clearance in the rhesus monkey. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/29985

24. Wilding, Anne-Sophie. Etude des mécanismes impliqués dans la mort oligodendrocytaire induite par la protéolipide-protéine mutée : rôle du stress du réticulum endoplasmique et identification des modulateurs à fort potentiel pour le traitement des pathologies dysmyélinisantes : Mechanisms of proteolipid protein mutation-induced oligodendrocyte death : role of endoplasmic reticulum stress and identification of modulatory compounds with high potential for the treatment of dysmyelinating disorders.

Degree: Docteur es, Neurosciences, 2017, Université de Strasbourg

Les mutations de la protéolipide-protéine (PLP) entraînent la mort des oligodendrocytes (OL) et les pathologies de la myéline. Pour contribuer à l'élucidation des mécanismes impliqués,… (more)

Subjects/Keywords: Apoptose; Maladies de la myéline; Neuroprotection; Oligodendrocytes; Pelizaeus- Merzbacher; Stress du réticulum endoplasmique; 4-Phénylbutyrate; Apoptosis; Endoplasmic reticulum stress; Neuroprotection; Myelin diseases; 4-Phenylbutyrate; Oligodendrocytes; Pelizaeus-Merzbacher; 573.8; 616.8

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APA (6th Edition):

Wilding, A. (2017). Etude des mécanismes impliqués dans la mort oligodendrocytaire induite par la protéolipide-protéine mutée : rôle du stress du réticulum endoplasmique et identification des modulateurs à fort potentiel pour le traitement des pathologies dysmyélinisantes : Mechanisms of proteolipid protein mutation-induced oligodendrocyte death : role of endoplasmic reticulum stress and identification of modulatory compounds with high potential for the treatment of dysmyelinating disorders. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ073

Chicago Manual of Style (16th Edition):

Wilding, Anne-Sophie. “Etude des mécanismes impliqués dans la mort oligodendrocytaire induite par la protéolipide-protéine mutée : rôle du stress du réticulum endoplasmique et identification des modulateurs à fort potentiel pour le traitement des pathologies dysmyélinisantes : Mechanisms of proteolipid protein mutation-induced oligodendrocyte death : role of endoplasmic reticulum stress and identification of modulatory compounds with high potential for the treatment of dysmyelinating disorders.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed October 21, 2018. http://www.theses.fr/2017STRAJ073.

MLA Handbook (7th Edition):

Wilding, Anne-Sophie. “Etude des mécanismes impliqués dans la mort oligodendrocytaire induite par la protéolipide-protéine mutée : rôle du stress du réticulum endoplasmique et identification des modulateurs à fort potentiel pour le traitement des pathologies dysmyélinisantes : Mechanisms of proteolipid protein mutation-induced oligodendrocyte death : role of endoplasmic reticulum stress and identification of modulatory compounds with high potential for the treatment of dysmyelinating disorders.” 2017. Web. 21 Oct 2018.

Vancouver:

Wilding A. Etude des mécanismes impliqués dans la mort oligodendrocytaire induite par la protéolipide-protéine mutée : rôle du stress du réticulum endoplasmique et identification des modulateurs à fort potentiel pour le traitement des pathologies dysmyélinisantes : Mechanisms of proteolipid protein mutation-induced oligodendrocyte death : role of endoplasmic reticulum stress and identification of modulatory compounds with high potential for the treatment of dysmyelinating disorders. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2018 Oct 21]. Available from: http://www.theses.fr/2017STRAJ073.

Council of Science Editors:

Wilding A. Etude des mécanismes impliqués dans la mort oligodendrocytaire induite par la protéolipide-protéine mutée : rôle du stress du réticulum endoplasmique et identification des modulateurs à fort potentiel pour le traitement des pathologies dysmyélinisantes : Mechanisms of proteolipid protein mutation-induced oligodendrocyte death : role of endoplasmic reticulum stress and identification of modulatory compounds with high potential for the treatment of dysmyelinating disorders. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ073


Case Western Reserve University

25. Lager, Angela Marie. Cell Reprogramming Technologies for Treatment and Understanding of Genetic Disorders of Myelin.

Degree: PhD, Genetics, 2015, Case Western Reserve University

 The oligodendrocyte lineage is essential for high-fidelity information transfer in neural circuits of the central nervous system. Oligodendrocytes arise from a pool of migratory progenitor… (more)

Subjects/Keywords: Developmental Biology; Genetics; myelin; oligodendrocytes; cell reprogramming; regenerative therapy; direct reprogramming; myelin repair

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APA (6th Edition):

Lager, A. M. (2015). Cell Reprogramming Technologies for Treatment and Understanding of Genetic Disorders of Myelin. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1427898199

Chicago Manual of Style (16th Edition):

Lager, Angela Marie. “Cell Reprogramming Technologies for Treatment and Understanding of Genetic Disorders of Myelin.” 2015. Doctoral Dissertation, Case Western Reserve University. Accessed October 21, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1427898199.

MLA Handbook (7th Edition):

Lager, Angela Marie. “Cell Reprogramming Technologies for Treatment and Understanding of Genetic Disorders of Myelin.” 2015. Web. 21 Oct 2018.

Vancouver:

Lager AM. Cell Reprogramming Technologies for Treatment and Understanding of Genetic Disorders of Myelin. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2015. [cited 2018 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1427898199.

Council of Science Editors:

Lager AM. Cell Reprogramming Technologies for Treatment and Understanding of Genetic Disorders of Myelin. [Doctoral Dissertation]. Case Western Reserve University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1427898199

26. Shabbir, Asghar. Le programme d’induction de la mort cellulaire des oligodendrocytes détermine le potentiel de réparation de la myéline au cours du développement : The timing of oligodendrocyte cell death determines the potential of myelin recovery during brain postnatal development.

Degree: Docteur es, Neurosciences, 2013, Université de Strasbourg

Dans cette étude, nous avons utilisé un modèle de souris permettant d’induire des lésions de sévérité variable afin de mimer différentes anomalies de la myéline… (more)

Subjects/Keywords: Régénération des oligodendrocytes; Myélinisation; Développement postnatal; Oligodendrocyte regeneration; Myelination; Postnatal development; 573.8; 611.8

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APA (6th Edition):

Shabbir, A. (2013). Le programme d’induction de la mort cellulaire des oligodendrocytes détermine le potentiel de réparation de la myéline au cours du développement : The timing of oligodendrocyte cell death determines the potential of myelin recovery during brain postnatal development. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ089

Chicago Manual of Style (16th Edition):

Shabbir, Asghar. “Le programme d’induction de la mort cellulaire des oligodendrocytes détermine le potentiel de réparation de la myéline au cours du développement : The timing of oligodendrocyte cell death determines the potential of myelin recovery during brain postnatal development.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed October 21, 2018. http://www.theses.fr/2013STRAJ089.

MLA Handbook (7th Edition):

Shabbir, Asghar. “Le programme d’induction de la mort cellulaire des oligodendrocytes détermine le potentiel de réparation de la myéline au cours du développement : The timing of oligodendrocyte cell death determines the potential of myelin recovery during brain postnatal development.” 2013. Web. 21 Oct 2018.

Vancouver:

Shabbir A. Le programme d’induction de la mort cellulaire des oligodendrocytes détermine le potentiel de réparation de la myéline au cours du développement : The timing of oligodendrocyte cell death determines the potential of myelin recovery during brain postnatal development. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2018 Oct 21]. Available from: http://www.theses.fr/2013STRAJ089.

Council of Science Editors:

Shabbir A. Le programme d’induction de la mort cellulaire des oligodendrocytes détermine le potentiel de réparation de la myéline au cours du développement : The timing of oligodendrocyte cell death determines the potential of myelin recovery during brain postnatal development. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ089


University of California – San Diego

27. Hanson, Kari. Neural substrates of impaired behavioral inhibition in Williams syndrome, a disorder of social cognition.

Degree: Anthropology, 2017, University of California – San Diego

 The evolution of the human brain has yielded advanced cognitive capacities supporting the development of language, technologically advanced material culture, and highly complex social behavior… (more)

Subjects/Keywords: Physical anthropology; Neurosciences; Autism; brain evolution; cholinergic interneurons; genetic disorders; oligodendrocytes; Williams syndrome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hanson, K. (2017). Neural substrates of impaired behavioral inhibition in Williams syndrome, a disorder of social cognition. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/0gp183qr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hanson, Kari. “Neural substrates of impaired behavioral inhibition in Williams syndrome, a disorder of social cognition.” 2017. Thesis, University of California – San Diego. Accessed October 21, 2018. http://www.escholarship.org/uc/item/0gp183qr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hanson, Kari. “Neural substrates of impaired behavioral inhibition in Williams syndrome, a disorder of social cognition.” 2017. Web. 21 Oct 2018.

Vancouver:

Hanson K. Neural substrates of impaired behavioral inhibition in Williams syndrome, a disorder of social cognition. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2018 Oct 21]. Available from: http://www.escholarship.org/uc/item/0gp183qr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hanson K. Neural substrates of impaired behavioral inhibition in Williams syndrome, a disorder of social cognition. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/0gp183qr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tampere University

28. Sundberg, Maria. Differentiation and Purification of Human Pluripotent Stem Cell-derived Neuronal and Glial Cells - graft designing for spinal cord injury repair .

Degree: 2011, Tampere University

 Ihmisen alkion kantasolut ovat pluripotentteja soluja jotka voidaan erilaistaa kaikiksi kolmeksi eri alkiosolukerroksen solutyypiksi. Alkion kantasolujen tehokas kyky jakaantua ja erilaistua hermosoluiksi ja hermotukisoluiksi tekee… (more)

Subjects/Keywords: alkion kantasolut; oligodendrosyytit; selkäydinvaurio; solulajittelu; human embryonic stem cells; oligodendrocytes; spinal cord injury; cell sorting

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sundberg, M. (2011). Differentiation and Purification of Human Pluripotent Stem Cell-derived Neuronal and Glial Cells - graft designing for spinal cord injury repair . (Doctoral Dissertation). Tampere University. Retrieved from http://tampub.uta.fi/handle/10024/66716

Chicago Manual of Style (16th Edition):

Sundberg, Maria. “Differentiation and Purification of Human Pluripotent Stem Cell-derived Neuronal and Glial Cells - graft designing for spinal cord injury repair .” 2011. Doctoral Dissertation, Tampere University. Accessed October 21, 2018. http://tampub.uta.fi/handle/10024/66716.

MLA Handbook (7th Edition):

Sundberg, Maria. “Differentiation and Purification of Human Pluripotent Stem Cell-derived Neuronal and Glial Cells - graft designing for spinal cord injury repair .” 2011. Web. 21 Oct 2018.

Vancouver:

Sundberg M. Differentiation and Purification of Human Pluripotent Stem Cell-derived Neuronal and Glial Cells - graft designing for spinal cord injury repair . [Internet] [Doctoral dissertation]. Tampere University; 2011. [cited 2018 Oct 21]. Available from: http://tampub.uta.fi/handle/10024/66716.

Council of Science Editors:

Sundberg M. Differentiation and Purification of Human Pluripotent Stem Cell-derived Neuronal and Glial Cells - graft designing for spinal cord injury repair . [Doctoral Dissertation]. Tampere University; 2011. Available from: http://tampub.uta.fi/handle/10024/66716


University of Minnesota

29. Zhou, Jie. Mitigating oxidative stress in childhood cerebral adrenoleukodystrophy -an investigation of N-acetylcysteine pharmacology.

Degree: PhD, Experimental & Clinical Pharmacology, 2014, University of Minnesota

 Adrenoleukodystrophy (ALD) is an X-linked genetic disorder which affects the adrenal glands, peripheral neuronal system, the spinal cord and white matter of central nervous system… (more)

Subjects/Keywords: Adrenoleukodystrophy; N-acetylcysteine; Neurodegeneration; Oligodendrocytes; Oxidative stress; Pharmacokinetics and Pharmacodynamics; Experimental & clinical pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhou, J. (2014). Mitigating oxidative stress in childhood cerebral adrenoleukodystrophy -an investigation of N-acetylcysteine pharmacology. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/172111

Chicago Manual of Style (16th Edition):

Zhou, Jie. “Mitigating oxidative stress in childhood cerebral adrenoleukodystrophy -an investigation of N-acetylcysteine pharmacology.” 2014. Doctoral Dissertation, University of Minnesota. Accessed October 21, 2018. http://hdl.handle.net/11299/172111.

MLA Handbook (7th Edition):

Zhou, Jie. “Mitigating oxidative stress in childhood cerebral adrenoleukodystrophy -an investigation of N-acetylcysteine pharmacology.” 2014. Web. 21 Oct 2018.

Vancouver:

Zhou J. Mitigating oxidative stress in childhood cerebral adrenoleukodystrophy -an investigation of N-acetylcysteine pharmacology. [Internet] [Doctoral dissertation]. University of Minnesota; 2014. [cited 2018 Oct 21]. Available from: http://hdl.handle.net/11299/172111.

Council of Science Editors:

Zhou J. Mitigating oxidative stress in childhood cerebral adrenoleukodystrophy -an investigation of N-acetylcysteine pharmacology. [Doctoral Dissertation]. University of Minnesota; 2014. Available from: http://hdl.handle.net/11299/172111


UCLA

30. Kim, Youn-Jung Roy. Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands.

Degree: Molec, Cell, & Integ Physiology, 2018, UCLA

 Currently, available drugs for multiple sclerosis (MS) are predominantly immune-modulatory. They are effective in reducing relapses, but not in slowing down or stopping progressive disease… (more)

Subjects/Keywords: Neurosciences; Physiology; Cuprizone; Estrogen Receptor Beta Ligand; Experimental Autoimmune Encephalomyelitis; Multiple Sclerosis; Myeloid cells; Oligodendrocytes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, Y. R. (2018). Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/09j2798f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Youn-Jung Roy. “Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands.” 2018. Thesis, UCLA. Accessed October 21, 2018. http://www.escholarship.org/uc/item/09j2798f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Youn-Jung Roy. “Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands.” 2018. Web. 21 Oct 2018.

Vancouver:

Kim YR. Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands. [Internet] [Thesis]. UCLA; 2018. [cited 2018 Oct 21]. Available from: http://www.escholarship.org/uc/item/09j2798f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim YR. Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/09j2798f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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