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You searched for subject:(non homologous end joining NHEJ ). Showing records 1 – 30 of 31012 total matches.

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McMaster University

1. Koechlin, Lucas. Characterization of Prokaryotic Ku DNA Binding Properties.

Degree: MSc, 2020, McMaster University

 DNA damage occurs to all living things; its subsequent repair is a crucial component of life. The most dangerous, and potentially most useful form of… (more)

Subjects/Keywords: DNA repair; Non-Homologous End-Joining; Bacteria; Ku; Protein DNA interactions; NHEJ; DNA damage; AMR

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APA (6th Edition):

Koechlin, L. (2020). Characterization of Prokaryotic Ku DNA Binding Properties. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/25572

Chicago Manual of Style (16th Edition):

Koechlin, Lucas. “Characterization of Prokaryotic Ku DNA Binding Properties.” 2020. Masters Thesis, McMaster University. Accessed April 12, 2021. http://hdl.handle.net/11375/25572.

MLA Handbook (7th Edition):

Koechlin, Lucas. “Characterization of Prokaryotic Ku DNA Binding Properties.” 2020. Web. 12 Apr 2021.

Vancouver:

Koechlin L. Characterization of Prokaryotic Ku DNA Binding Properties. [Internet] [Masters thesis]. McMaster University; 2020. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/11375/25572.

Council of Science Editors:

Koechlin L. Characterization of Prokaryotic Ku DNA Binding Properties. [Masters Thesis]. McMaster University; 2020. Available from: http://hdl.handle.net/11375/25572


University of Illinois – Chicago

2. Li, Jing. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).

Degree: 2017, University of Illinois – Chicago

 The repair of DNA double-strand breaks (DSB) is central to the maintenance of genomic integrity. Major DSB repair pathways in mammalian cells include homologous recombination… (more)

Subjects/Keywords: tyrosyl-DNA phosphodiesterase 1 (TDP1); non-homologous end joining (NHEJ); DNA double-strand breaks (DSB)

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APA (6th Edition):

Li, J. (2017). Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Jing. “Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).” 2017. Thesis, University of Illinois – Chicago. Accessed April 12, 2021. http://hdl.handle.net/10027/22078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Jing. “Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).” 2017. Web. 12 Apr 2021.

Vancouver:

Li J. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10027/22078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li J. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/22078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

3. Vaidya, Amita M. Study of age-associated DNA double strand break repair by non-homologous end joining in novel reporter mouse model.

Degree: PhD, 2014, University of Rochester

 Aging is a combination of progressive decline in function and fitness of an organism. Its complexity arises from numerous genetic and environmental pressures that influence… (more)

Subjects/Keywords: Aging; Cancer; Genome instability; Microhomology-mediated End Joining, Mouse model; Non-homologous End Joining; NHEJ; DNA repair; AAV; I-Scel; Reporter

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APA (6th Edition):

Vaidya, A. M. (2014). Study of age-associated DNA double strand break repair by non-homologous end joining in novel reporter mouse model. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/28787

Chicago Manual of Style (16th Edition):

Vaidya, Amita M. “Study of age-associated DNA double strand break repair by non-homologous end joining in novel reporter mouse model.” 2014. Doctoral Dissertation, University of Rochester. Accessed April 12, 2021. http://hdl.handle.net/1802/28787.

MLA Handbook (7th Edition):

Vaidya, Amita M. “Study of age-associated DNA double strand break repair by non-homologous end joining in novel reporter mouse model.” 2014. Web. 12 Apr 2021.

Vancouver:

Vaidya AM. Study of age-associated DNA double strand break repair by non-homologous end joining in novel reporter mouse model. [Internet] [Doctoral dissertation]. University of Rochester; 2014. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1802/28787.

Council of Science Editors:

Vaidya AM. Study of age-associated DNA double strand break repair by non-homologous end joining in novel reporter mouse model. [Doctoral Dissertation]. University of Rochester; 2014. Available from: http://hdl.handle.net/1802/28787

4. Tsouroula, Aikaterini. Double strand break repair within constitutive heterochromatin : Étude de la réparation des cassures doubles brins de l'ADN dans l'hétérochromatine constitutive.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université de Strasbourg

L'hétérochromatine, de nature compacte et répétitive, limite l’accès à l'ADN et fait de la réparation des DSBs un processus difficile que les cellules doivent surmonter… (more)

Subjects/Keywords: Hétérochromatine; CRISPR/ Cas9; NHEJ; HR; RAD51; SSA; Heterochromatin; CRISPR/ Cas9; Non-Homologous End Joining; Homologous recombination; RAD51; Single Strand Annealing; 572.8

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APA (6th Edition):

Tsouroula, A. (2017). Double strand break repair within constitutive heterochromatin : Étude de la réparation des cassures doubles brins de l'ADN dans l'hétérochromatine constitutive. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ036

Chicago Manual of Style (16th Edition):

Tsouroula, Aikaterini. “Double strand break repair within constitutive heterochromatin : Étude de la réparation des cassures doubles brins de l'ADN dans l'hétérochromatine constitutive.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed April 12, 2021. http://www.theses.fr/2017STRAJ036.

MLA Handbook (7th Edition):

Tsouroula, Aikaterini. “Double strand break repair within constitutive heterochromatin : Étude de la réparation des cassures doubles brins de l'ADN dans l'hétérochromatine constitutive.” 2017. Web. 12 Apr 2021.

Vancouver:

Tsouroula A. Double strand break repair within constitutive heterochromatin : Étude de la réparation des cassures doubles brins de l'ADN dans l'hétérochromatine constitutive. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2021 Apr 12]. Available from: http://www.theses.fr/2017STRAJ036.

Council of Science Editors:

Tsouroula A. Double strand break repair within constitutive heterochromatin : Étude de la réparation des cassures doubles brins de l'ADN dans l'hétérochromatine constitutive. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ036

5. Bordelet, Hélène. Régulation de la résection aux cassures double-brin par l'hétérochromatine SIR dépendante : Regulation of resection at double strand-breaks by SIR mediated heterochromatin.

Degree: Docteur es, Sciences de la vie et de la santé, 2019, Université Paris-Saclay (ComUE)

L'hétérochromatine est une caractéristique conservée des chromosomes eucaryotes, avec des rôles centraux dans la régulation de l'expression des gènes et le maintien de la stabilité… (more)

Subjects/Keywords: Hétérochromatine; Recombinaison Homologue; NHEJ; Résection; S. cerevisiae; Complexe SIR; Heterochromatin; Homologous Recombination; SIR complex; Resection; Non-Homologous End Joining; S. cerevisiae

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APA (6th Edition):

Bordelet, H. (2019). Régulation de la résection aux cassures double-brin par l'hétérochromatine SIR dépendante : Regulation of resection at double strand-breaks by SIR mediated heterochromatin. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLS300

Chicago Manual of Style (16th Edition):

Bordelet, Hélène. “Régulation de la résection aux cassures double-brin par l'hétérochromatine SIR dépendante : Regulation of resection at double strand-breaks by SIR mediated heterochromatin.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed April 12, 2021. http://www.theses.fr/2019SACLS300.

MLA Handbook (7th Edition):

Bordelet, Hélène. “Régulation de la résection aux cassures double-brin par l'hétérochromatine SIR dépendante : Regulation of resection at double strand-breaks by SIR mediated heterochromatin.” 2019. Web. 12 Apr 2021.

Vancouver:

Bordelet H. Régulation de la résection aux cassures double-brin par l'hétérochromatine SIR dépendante : Regulation of resection at double strand-breaks by SIR mediated heterochromatin. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Apr 12]. Available from: http://www.theses.fr/2019SACLS300.

Council of Science Editors:

Bordelet H. Régulation de la résection aux cassures double-brin par l'hétérochromatine SIR dépendante : Regulation of resection at double strand-breaks by SIR mediated heterochromatin. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLS300


University of Cambridge

6. Liang, Shikang. DNA-PKcs/ Artemis Complex in DNA Double-Strand-Break Repair: Cryo-EM and Biochemical Studies.

Degree: PhD, 2020, University of Cambridge

 DNA is the main carrier of inheritance and DNA damage will thus have serious consequences. DNA double-strand breaks (DSB) are amongst the most lethal forms… (more)

Subjects/Keywords: DNA repair; DNA double-strand break; Non-Homologous End Joining; Cryo-EM; DNA-PKcs/ Artemis complex; NHEJ spatial organisation; NHEJ temporal organisation; Structural Biology; structure-based drug discovery

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APA (6th Edition):

Liang, S. (2020). DNA-PKcs/ Artemis Complex in DNA Double-Strand-Break Repair: Cryo-EM and Biochemical Studies. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/303615

Chicago Manual of Style (16th Edition):

Liang, Shikang. “DNA-PKcs/ Artemis Complex in DNA Double-Strand-Break Repair: Cryo-EM and Biochemical Studies.” 2020. Doctoral Dissertation, University of Cambridge. Accessed April 12, 2021. https://www.repository.cam.ac.uk/handle/1810/303615.

MLA Handbook (7th Edition):

Liang, Shikang. “DNA-PKcs/ Artemis Complex in DNA Double-Strand-Break Repair: Cryo-EM and Biochemical Studies.” 2020. Web. 12 Apr 2021.

Vancouver:

Liang S. DNA-PKcs/ Artemis Complex in DNA Double-Strand-Break Repair: Cryo-EM and Biochemical Studies. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2021 Apr 12]. Available from: https://www.repository.cam.ac.uk/handle/1810/303615.

Council of Science Editors:

Liang S. DNA-PKcs/ Artemis Complex in DNA Double-Strand-Break Repair: Cryo-EM and Biochemical Studies. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/303615


Virginia Tech

7. Puthiyaveetil Abdulkader, Abdul Gafoor. Altered Kinetics of Non-Homologous End Joining Mediated DNA Repair in Mouse Models of Aging and Leukemia.

Degree: PhD, Veterinary Medical Sciences, 2012, Virginia Tech

 DNA encodes the genetic instructions for the development and function of organisms and hence maintaining genomic integrity is essential for the propagation of life. However,… (more)

Subjects/Keywords: alternative end joining; non-homologous end joining; class switch recombination; aging; B lymphocytes; chromosomal translocation

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APA (6th Edition):

Puthiyaveetil Abdulkader, A. G. (2012). Altered Kinetics of Non-Homologous End Joining Mediated DNA Repair in Mouse Models of Aging and Leukemia. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/77233

Chicago Manual of Style (16th Edition):

Puthiyaveetil Abdulkader, Abdul Gafoor. “Altered Kinetics of Non-Homologous End Joining Mediated DNA Repair in Mouse Models of Aging and Leukemia.” 2012. Doctoral Dissertation, Virginia Tech. Accessed April 12, 2021. http://hdl.handle.net/10919/77233.

MLA Handbook (7th Edition):

Puthiyaveetil Abdulkader, Abdul Gafoor. “Altered Kinetics of Non-Homologous End Joining Mediated DNA Repair in Mouse Models of Aging and Leukemia.” 2012. Web. 12 Apr 2021.

Vancouver:

Puthiyaveetil Abdulkader AG. Altered Kinetics of Non-Homologous End Joining Mediated DNA Repair in Mouse Models of Aging and Leukemia. [Internet] [Doctoral dissertation]. Virginia Tech; 2012. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10919/77233.

Council of Science Editors:

Puthiyaveetil Abdulkader AG. Altered Kinetics of Non-Homologous End Joining Mediated DNA Repair in Mouse Models of Aging and Leukemia. [Doctoral Dissertation]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/77233

8. De Melo, Abinadabe Jackson. Molecular basis for the structural role of human DNA ligase IV : Base moléculaire pour le rôle structural de l'ADN humain Ligase IV.

Degree: Docteur es, Biologie, 2016, Aix Marseille Université

Les défauts dans la réparation des cassures double-brin de l'ADN (DSBs) peuvent avoir d'importantes conséquences pouvant entrainer une instabilité génomique et conduire à la mort… (more)

Subjects/Keywords: Cassures double-Brin de l'ADN (DSBs); Jonction des extrémités Non Homologues (NHEJ); DNA Ligase IV; Xrcc4; Xlf; DNA double-Strand breaks (DSBs); Non-Homologous End Joining (NHEJ); DNA Ligase IV; Xrcc4; Xlf; 570

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APA (6th Edition):

De Melo, A. J. (2016). Molecular basis for the structural role of human DNA ligase IV : Base moléculaire pour le rôle structural de l'ADN humain Ligase IV. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2016AIXM4040

Chicago Manual of Style (16th Edition):

De Melo, Abinadabe Jackson. “Molecular basis for the structural role of human DNA ligase IV : Base moléculaire pour le rôle structural de l'ADN humain Ligase IV.” 2016. Doctoral Dissertation, Aix Marseille Université. Accessed April 12, 2021. http://www.theses.fr/2016AIXM4040.

MLA Handbook (7th Edition):

De Melo, Abinadabe Jackson. “Molecular basis for the structural role of human DNA ligase IV : Base moléculaire pour le rôle structural de l'ADN humain Ligase IV.” 2016. Web. 12 Apr 2021.

Vancouver:

De Melo AJ. Molecular basis for the structural role of human DNA ligase IV : Base moléculaire pour le rôle structural de l'ADN humain Ligase IV. [Internet] [Doctoral dissertation]. Aix Marseille Université 2016. [cited 2021 Apr 12]. Available from: http://www.theses.fr/2016AIXM4040.

Council of Science Editors:

De Melo AJ. Molecular basis for the structural role of human DNA ligase IV : Base moléculaire pour le rôle structural de l'ADN humain Ligase IV. [Doctoral Dissertation]. Aix Marseille Université 2016. Available from: http://www.theses.fr/2016AIXM4040


Indian Institute of Science

9. Sebastian, Robin. Physiological And Exogenous Means of Regulating DNA Damage Response : Insights into Mechanisms of DNA Repair And Genomic Instability.

Degree: PhD, Faculty of Science, 2017, Indian Institute of Science

 Maintenance of genomic integrity with high fidelity is of prime importance to any organism. An insult which may result in compromised genome integrity is prevented… (more)

Subjects/Keywords: DNA Damage; DNA Repair; Genomic Instability; DNA Damage Response (DDR); Endosulfan Induced DNA Damage; Genomic Integrity; Non Homologous DNA End Joining (NHEJ); Microhomology Mediated DNA End Joining (MMEJ); Reactive Oxygen Species; DNA Repair Pathways; Biochemistry

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APA (6th Edition):

Sebastian, R. (2017). Physiological And Exogenous Means of Regulating DNA Damage Response : Insights into Mechanisms of DNA Repair And Genomic Instability. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/2676

Chicago Manual of Style (16th Edition):

Sebastian, Robin. “Physiological And Exogenous Means of Regulating DNA Damage Response : Insights into Mechanisms of DNA Repair And Genomic Instability.” 2017. Doctoral Dissertation, Indian Institute of Science. Accessed April 12, 2021. http://etd.iisc.ac.in/handle/2005/2676.

MLA Handbook (7th Edition):

Sebastian, Robin. “Physiological And Exogenous Means of Regulating DNA Damage Response : Insights into Mechanisms of DNA Repair And Genomic Instability.” 2017. Web. 12 Apr 2021.

Vancouver:

Sebastian R. Physiological And Exogenous Means of Regulating DNA Damage Response : Insights into Mechanisms of DNA Repair And Genomic Instability. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2017. [cited 2021 Apr 12]. Available from: http://etd.iisc.ac.in/handle/2005/2676.

Council of Science Editors:

Sebastian R. Physiological And Exogenous Means of Regulating DNA Damage Response : Insights into Mechanisms of DNA Repair And Genomic Instability. [Doctoral Dissertation]. Indian Institute of Science; 2017. Available from: http://etd.iisc.ac.in/handle/2005/2676

10. Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.

Degree: 博士(医学), 2017, Nara Medical University / 奈良県立医科大学

DNA-dependent protein kinase (DNA-PK) is a serine/threonine kinase that plays an essential role in the repair of DNA double-strand breaks (DSBs) in the non-homologous end-joining(more)

Subjects/Keywords: DNA double-strand breaks; Non-homologous end-joining; Acetylation; DNA-PKcs

Page 1

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APA (6th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, M. (2017). Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. “Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.” 2017. Thesis, Nara Medical University / 奈良県立医科大学. Accessed April 12, 2021. http://hdl.handle.net/10564/3342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. “Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.” 2017. Web. 12 Apr 2021.

Vancouver:

Mori, Eiichiro; Davis, Anthony J.; Hasegawa M. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10564/3342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mori, Eiichiro; Davis, Anthony J.; Hasegawa M. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. Available from: http://hdl.handle.net/10564/3342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

11. Shirodkar, Purnata V. Characterization of APLF in Non-homologous End-joining.

Degree: 2011, University of Toronto

APLF (Aprataxin and Polynucleotide kinase-Like Factor), a novel protein with a forkhead-associated (FHA) domain and two poly(ADP-ribose)-binding zinc fingers (PBZ), interacts with core non-homologous end-joining(more)

Subjects/Keywords: APLF; Non-homologous End-joining; Ku; nuclear localization; 0760; 0307

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APA (6th Edition):

Shirodkar, P. V. (2011). Characterization of APLF in Non-homologous End-joining. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/29632

Chicago Manual of Style (16th Edition):

Shirodkar, Purnata V. “Characterization of APLF in Non-homologous End-joining.” 2011. Masters Thesis, University of Toronto. Accessed April 12, 2021. http://hdl.handle.net/1807/29632.

MLA Handbook (7th Edition):

Shirodkar, Purnata V. “Characterization of APLF in Non-homologous End-joining.” 2011. Web. 12 Apr 2021.

Vancouver:

Shirodkar PV. Characterization of APLF in Non-homologous End-joining. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1807/29632.

Council of Science Editors:

Shirodkar PV. Characterization of APLF in Non-homologous End-joining. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/29632


IUPUI

12. Pawelczak, Katherine S. Determining molecular mechanisms of DNA Non-Homologous End Joining proteins.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

DNA double strand breaks (DSB), particularly those induced by ionizing radiation (IR) are complex lesions and if not repaired, these… (more)

Subjects/Keywords: DNA repair, non-homologous end joining; DNA-protein interactions; DNA repair

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APA (6th Edition):

Pawelczak, K. S. (2011). Determining molecular mechanisms of DNA Non-Homologous End Joining proteins. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pawelczak, Katherine S. “Determining molecular mechanisms of DNA Non-Homologous End Joining proteins.” 2011. Thesis, IUPUI. Accessed April 12, 2021. http://hdl.handle.net/1805/2517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pawelczak, Katherine S. “Determining molecular mechanisms of DNA Non-Homologous End Joining proteins.” 2011. Web. 12 Apr 2021.

Vancouver:

Pawelczak KS. Determining molecular mechanisms of DNA Non-Homologous End Joining proteins. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1805/2517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pawelczak KS. Determining molecular mechanisms of DNA Non-Homologous End Joining proteins. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University College London (University of London)

13. Bryntesson, Fredrik Anders. An investigation of genes involved in double-stranded break repair of DNA.

Degree: PhD, 2001, University College London (University of London)

 Damage to DNA can lead to genomic instability and therefore poses a great threat to the living organism. Living organisms have therefore evolved responses to… (more)

Subjects/Keywords: 572; Non homologous end joining

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APA (6th Edition):

Bryntesson, F. A. (2001). An investigation of genes involved in double-stranded break repair of DNA. (Doctoral Dissertation). University College London (University of London). Retrieved from https://discovery.ucl.ac.uk/id/eprint/10102942/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268453

Chicago Manual of Style (16th Edition):

Bryntesson, Fredrik Anders. “An investigation of genes involved in double-stranded break repair of DNA.” 2001. Doctoral Dissertation, University College London (University of London). Accessed April 12, 2021. https://discovery.ucl.ac.uk/id/eprint/10102942/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268453.

MLA Handbook (7th Edition):

Bryntesson, Fredrik Anders. “An investigation of genes involved in double-stranded break repair of DNA.” 2001. Web. 12 Apr 2021.

Vancouver:

Bryntesson FA. An investigation of genes involved in double-stranded break repair of DNA. [Internet] [Doctoral dissertation]. University College London (University of London); 2001. [cited 2021 Apr 12]. Available from: https://discovery.ucl.ac.uk/id/eprint/10102942/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268453.

Council of Science Editors:

Bryntesson FA. An investigation of genes involved in double-stranded break repair of DNA. [Doctoral Dissertation]. University College London (University of London); 2001. Available from: https://discovery.ucl.ac.uk/id/eprint/10102942/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268453

14. Jia, Qi. DNA repair and gene targeting in plant end-joining mutants.

Degree: 2011, Department of Molecular and Development Genetics, Institute of Biology, Faculty of Science, Leiden University

 DNA double-strand breaks (DSBs) can be repaired by homologous recombination (HR) or by non-homologous end joining (NHEJ). The latter mechanism is the major route for… (more)

Subjects/Keywords: Arabidopsis thaliana; DNA double strand break (DSB); Gene targeting; Micro-homology mediated end joining (MMEJ); Non-homologous end joining (NHEJ); T-DNA integration; Arabidopsis thaliana; DNA double strand break (DSB); Gene targeting; Micro-homology mediated end joining (MMEJ); Non-homologous end joining (NHEJ); T-DNA integration

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APA (6th Edition):

Jia, Q. (2011). DNA repair and gene targeting in plant end-joining mutants. (Doctoral Dissertation). Department of Molecular and Development Genetics, Institute of Biology, Faculty of Science, Leiden University. Retrieved from http://hdl.handle.net/1887/17582

Chicago Manual of Style (16th Edition):

Jia, Qi. “DNA repair and gene targeting in plant end-joining mutants.” 2011. Doctoral Dissertation, Department of Molecular and Development Genetics, Institute of Biology, Faculty of Science, Leiden University. Accessed April 12, 2021. http://hdl.handle.net/1887/17582.

MLA Handbook (7th Edition):

Jia, Qi. “DNA repair and gene targeting in plant end-joining mutants.” 2011. Web. 12 Apr 2021.

Vancouver:

Jia Q. DNA repair and gene targeting in plant end-joining mutants. [Internet] [Doctoral dissertation]. Department of Molecular and Development Genetics, Institute of Biology, Faculty of Science, Leiden University; 2011. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1887/17582.

Council of Science Editors:

Jia Q. DNA repair and gene targeting in plant end-joining mutants. [Doctoral Dissertation]. Department of Molecular and Development Genetics, Institute of Biology, Faculty of Science, Leiden University; 2011. Available from: http://hdl.handle.net/1887/17582


Vanderbilt University

15. Obodo, Udochukwu Chinyere. Not Restricted to the Ends: Yeast Telomere Proteins Rif1 and Cdc13 Function in Double-Strand Break Repair Pathways with Implications for Genome Stability.

Degree: PhD, Biological Sciences, 2016, Vanderbilt University

 DNA double-strand breaks (DSBs) are the most lethal form of DNA damage; a single DSB, if left unrepaired, can cause cell death. Inaccurately repaired DSBs… (more)

Subjects/Keywords: Double-strand breaks; telomeres; de novo telomere addition; Cdc13; non-homologous end joining; Rif1

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APA (6th Edition):

Obodo, U. C. (2016). Not Restricted to the Ends: Yeast Telomere Proteins Rif1 and Cdc13 Function in Double-Strand Break Repair Pathways with Implications for Genome Stability. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13466

Chicago Manual of Style (16th Edition):

Obodo, Udochukwu Chinyere. “Not Restricted to the Ends: Yeast Telomere Proteins Rif1 and Cdc13 Function in Double-Strand Break Repair Pathways with Implications for Genome Stability.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 12, 2021. http://hdl.handle.net/1803/13466.

MLA Handbook (7th Edition):

Obodo, Udochukwu Chinyere. “Not Restricted to the Ends: Yeast Telomere Proteins Rif1 and Cdc13 Function in Double-Strand Break Repair Pathways with Implications for Genome Stability.” 2016. Web. 12 Apr 2021.

Vancouver:

Obodo UC. Not Restricted to the Ends: Yeast Telomere Proteins Rif1 and Cdc13 Function in Double-Strand Break Repair Pathways with Implications for Genome Stability. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1803/13466.

Council of Science Editors:

Obodo UC. Not Restricted to the Ends: Yeast Telomere Proteins Rif1 and Cdc13 Function in Double-Strand Break Repair Pathways with Implications for Genome Stability. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13466


University of Oxford

16. Chitnis, Meenali M. Type 1 insulin-like growth factor receptor inhibition as treatment for urological cancer.

Degree: PhD, 2013, University of Oxford

 The type 1 insulin-like growth factor receptor (IGF-1R) is a receptor tyrosine kinase that mediates diverse cellular functions including growth, differentiation, migration and apoptosis protection.… (more)

Subjects/Keywords: 616.99461; Oncology; DNA damage signalling; urological cancer; DNA repair; non-homologous end-joining

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APA (6th Edition):

Chitnis, M. M. (2013). Type 1 insulin-like growth factor receptor inhibition as treatment for urological cancer. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:21282ce9-ce6b-4d26-b262-a3fca6d9c9fc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581325

Chicago Manual of Style (16th Edition):

Chitnis, Meenali M. “Type 1 insulin-like growth factor receptor inhibition as treatment for urological cancer.” 2013. Doctoral Dissertation, University of Oxford. Accessed April 12, 2021. http://ora.ox.ac.uk/objects/uuid:21282ce9-ce6b-4d26-b262-a3fca6d9c9fc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581325.

MLA Handbook (7th Edition):

Chitnis, Meenali M. “Type 1 insulin-like growth factor receptor inhibition as treatment for urological cancer.” 2013. Web. 12 Apr 2021.

Vancouver:

Chitnis MM. Type 1 insulin-like growth factor receptor inhibition as treatment for urological cancer. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2021 Apr 12]. Available from: http://ora.ox.ac.uk/objects/uuid:21282ce9-ce6b-4d26-b262-a3fca6d9c9fc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581325.

Council of Science Editors:

Chitnis MM. Type 1 insulin-like growth factor receptor inhibition as treatment for urological cancer. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:21282ce9-ce6b-4d26-b262-a3fca6d9c9fc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581325


University of Western Ontario

17. Parmar, Gursimran. Generation of Conditional Ku70 Knockouts in Human Cell Lines Using CRISPR/Cas9 and Dual-Nuclease CRISPR/TevCas9.

Degree: 2020, University of Western Ontario

 The Ku heterodimer, Ku70 and Ku80, plays a key role in DNA repair. Viable Ku70 knockouts exist in mice but not in human cell lines.… (more)

Subjects/Keywords: Ku; Non-homologous end joining; Cas9; Gene editing; Gene knockout; Biochemistry; Genetics

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APA (6th Edition):

Parmar, G. (2020). Generation of Conditional Ku70 Knockouts in Human Cell Lines Using CRISPR/Cas9 and Dual-Nuclease CRISPR/TevCas9. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Parmar, Gursimran. “Generation of Conditional Ku70 Knockouts in Human Cell Lines Using CRISPR/Cas9 and Dual-Nuclease CRISPR/TevCas9.” 2020. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/6917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Parmar, Gursimran. “Generation of Conditional Ku70 Knockouts in Human Cell Lines Using CRISPR/Cas9 and Dual-Nuclease CRISPR/TevCas9.” 2020. Web. 12 Apr 2021.

Vancouver:

Parmar G. Generation of Conditional Ku70 Knockouts in Human Cell Lines Using CRISPR/Cas9 and Dual-Nuclease CRISPR/TevCas9. [Internet] [Thesis]. University of Western Ontario; 2020. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/6917.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Parmar G. Generation of Conditional Ku70 Knockouts in Human Cell Lines Using CRISPR/Cas9 and Dual-Nuclease CRISPR/TevCas9. [Thesis]. University of Western Ontario; 2020. Available from: https://ir.lib.uwo.ca/etd/6917

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

18. Iyer, Shilpa. Understanding the genetic requirements for the loss of end protection in the yeast Kluyveromyces lactis.

Degree: 2014, University of Georgia

 Telomeres are the DNA-protein caps that protect chromosome ends from being repaired by either homologous recombination (HR) or non-homologous end joining (NHEJ). The proper functioning… (more)

Subjects/Keywords: yeast; Kluyveromyces lactis; telomere; ALT; recombination; non-homologous end joining; capping; DNA repair; cancer.

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APA (6th Edition):

Iyer, S. (2014). Understanding the genetic requirements for the loss of end protection in the yeast Kluyveromyces lactis. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/23145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iyer, Shilpa. “Understanding the genetic requirements for the loss of end protection in the yeast Kluyveromyces lactis.” 2014. Thesis, University of Georgia. Accessed April 12, 2021. http://hdl.handle.net/10724/23145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iyer, Shilpa. “Understanding the genetic requirements for the loss of end protection in the yeast Kluyveromyces lactis.” 2014. Web. 12 Apr 2021.

Vancouver:

Iyer S. Understanding the genetic requirements for the loss of end protection in the yeast Kluyveromyces lactis. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10724/23145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iyer S. Understanding the genetic requirements for the loss of end protection in the yeast Kluyveromyces lactis. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/23145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

19. Shenoy, Tanushree. Identifying Novel Molecular Biomarkers and Therapeutic Targets for Prostate Cancer.

Degree: Molecular and Medical Pharmacology, 2017, UCLA

 Prostate cancer is the most common malignancy in males, and the third leading cause of male cancer-related death in the Western world. Although most prostate… (more)

Subjects/Keywords: Molecular biology; Cellular biology; CHD1; DNA damage resposne; homologous recombination; non-homologous end-joining; prostate cancer

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APA (6th Edition):

Shenoy, T. (2017). Identifying Novel Molecular Biomarkers and Therapeutic Targets for Prostate Cancer. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5f99t4h9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shenoy, Tanushree. “Identifying Novel Molecular Biomarkers and Therapeutic Targets for Prostate Cancer.” 2017. Thesis, UCLA. Accessed April 12, 2021. http://www.escholarship.org/uc/item/5f99t4h9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shenoy, Tanushree. “Identifying Novel Molecular Biomarkers and Therapeutic Targets for Prostate Cancer.” 2017. Web. 12 Apr 2021.

Vancouver:

Shenoy T. Identifying Novel Molecular Biomarkers and Therapeutic Targets for Prostate Cancer. [Internet] [Thesis]. UCLA; 2017. [cited 2021 Apr 12]. Available from: http://www.escholarship.org/uc/item/5f99t4h9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shenoy T. Identifying Novel Molecular Biomarkers and Therapeutic Targets for Prostate Cancer. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/5f99t4h9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

20. Cutler, Geoffrey Lloyd. Characterizing Valproic Acid-Induced DNA Double Strand Break Repair .

Degree: Pharmacology and Toxicology, 2012, Queens University

 The teratogenic effects of valproic acid (VPA) are well known, though its teratogenic mechanism remains unknown. VPA induces oxidative stress, which may lead to double… (more)

Subjects/Keywords: pKZ1 ; Valproic Acid ; RAD51 ; XRCC4 ; DNA Double Strand Breaks ; Homologous Recombination ; Non-Homologous End Joining ; Neural Tube Defects

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APA (6th Edition):

Cutler, G. L. (2012). Characterizing Valproic Acid-Induced DNA Double Strand Break Repair . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cutler, Geoffrey Lloyd. “Characterizing Valproic Acid-Induced DNA Double Strand Break Repair .” 2012. Thesis, Queens University. Accessed April 12, 2021. http://hdl.handle.net/1974/7597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cutler, Geoffrey Lloyd. “Characterizing Valproic Acid-Induced DNA Double Strand Break Repair .” 2012. Web. 12 Apr 2021.

Vancouver:

Cutler GL. Characterizing Valproic Acid-Induced DNA Double Strand Break Repair . [Internet] [Thesis]. Queens University; 2012. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/1974/7597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cutler GL. Characterizing Valproic Acid-Induced DNA Double Strand Break Repair . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

21. Odebunmi, Oluwaseun. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .

Degree: 2020, University of Ottawa

 There is substantial evidence on the carcinogenic properties of high doses of Ionizing Radiation (IR), however, whether such risks exist following exposure to low doses… (more)

Subjects/Keywords: Ionizing Radiation; Myoblasts; Myogenicity; DNA damage; DNA repair; Double-strand Breaks; LDR priming; Homologous Recombination; Non-Homologous End Joining; Genomic Instability

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APA (6th Edition):

Odebunmi, O. (2020). Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Odebunmi, Oluwaseun. “Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .” 2020. Thesis, University of Ottawa. Accessed April 12, 2021. http://hdl.handle.net/10393/40413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Odebunmi, Oluwaseun. “Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .” 2020. Web. 12 Apr 2021.

Vancouver:

Odebunmi O. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/10393/40413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Odebunmi O. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

22. Genet, Diane. Impact de la surexpression de la lamine B1 sur la réparation des cassures double-chaîne de l’ADN : Impact of lamin B1 overexpression on DNA double-strand break repair.

Degree: Docteur es, Sciences de la vie et de la santé, 2014, Université Paris-Sud – Paris XI

De nombreuses études montrent un rôle important de l'architecture du noyau sur la stabilité du génome. Les lamines sont les constituants majeurs de l’enveloppe nucléaire… (more)

Subjects/Keywords: Lamine B1; Enveloppe nucléaire; Réparation de l’ADN; Non-Homologous-End-Joining; 53BP1; Lamin B1; Nuclear envelope; DNA repair; Non-homologous and joining; 53BP1

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APA (6th Edition):

Genet, D. (2014). Impact de la surexpression de la lamine B1 sur la réparation des cassures double-chaîne de l’ADN : Impact of lamin B1 overexpression on DNA double-strand break repair. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA112204

Chicago Manual of Style (16th Edition):

Genet, Diane. “Impact de la surexpression de la lamine B1 sur la réparation des cassures double-chaîne de l’ADN : Impact of lamin B1 overexpression on DNA double-strand break repair.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 12, 2021. http://www.theses.fr/2014PA112204.

MLA Handbook (7th Edition):

Genet, Diane. “Impact de la surexpression de la lamine B1 sur la réparation des cassures double-chaîne de l’ADN : Impact of lamin B1 overexpression on DNA double-strand break repair.” 2014. Web. 12 Apr 2021.

Vancouver:

Genet D. Impact de la surexpression de la lamine B1 sur la réparation des cassures double-chaîne de l’ADN : Impact of lamin B1 overexpression on DNA double-strand break repair. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2021 Apr 12]. Available from: http://www.theses.fr/2014PA112204.

Council of Science Editors:

Genet D. Impact de la surexpression de la lamine B1 sur la réparation des cassures double-chaîne de l’ADN : Impact of lamin B1 overexpression on DNA double-strand break repair. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA112204


Kyoto University / 京都大学

23. Kobayashi, Shunsuke. Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining : Rad18とRnf8は、2つの異なった機構(Rad51のフォーカス形成の促進及び非相同末端結合の毒性効果の抑制)によって相同組換えを促進する.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

新制・課程博士

甲第18879号

医博第3990号

Subjects/Keywords: Rad18; RNF8; Double strand break(DSB); Homologous recombination(HR); Nonhomologous end-joining(NHEJ)

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APA (6th Edition):

Kobayashi, S. (2015). Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining : Rad18とRnf8は、2つの異なった機構(Rad51のフォーカス形成の促進及び非相同末端結合の毒性効果の抑制)によって相同組換えを促進する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/199188 ; http://dx.doi.org/10.14989/doctor.k18879

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kobayashi, Shunsuke. “Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining : Rad18とRnf8は、2つの異なった機構(Rad51のフォーカス形成の促進及び非相同末端結合の毒性効果の抑制)によって相同組換えを促進する.” 2015. Thesis, Kyoto University / 京都大学. Accessed April 12, 2021. http://hdl.handle.net/2433/199188 ; http://dx.doi.org/10.14989/doctor.k18879.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kobayashi, Shunsuke. “Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining : Rad18とRnf8は、2つの異なった機構(Rad51のフォーカス形成の促進及び非相同末端結合の毒性効果の抑制)によって相同組換えを促進する.” 2015. Web. 12 Apr 2021.

Vancouver:

Kobayashi S. Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining : Rad18とRnf8は、2つの異なった機構(Rad51のフォーカス形成の促進及び非相同末端結合の毒性効果の抑制)によって相同組換えを促進する. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/2433/199188 ; http://dx.doi.org/10.14989/doctor.k18879.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kobayashi S. Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining : Rad18とRnf8は、2つの異なった機構(Rad51のフォーカス形成の促進及び非相同末端結合の毒性効果の抑制)によって相同組換えを促進する. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/199188 ; http://dx.doi.org/10.14989/doctor.k18879

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Kobayashi, Shunsuke. Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining .

Degree: 2015, Kyoto University

Subjects/Keywords: Rad18; RNF8; Double strand break(DSB); Homologous recombination(HR); Nonhomologous end-joining(NHEJ)

Page 1 Page 2

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APA (6th Edition):

Kobayashi, S. (2015). Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/199188

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kobayashi, Shunsuke. “Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining .” 2015. Thesis, Kyoto University. Accessed April 12, 2021. http://hdl.handle.net/2433/199188.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kobayashi, Shunsuke. “Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining .” 2015. Web. 12 Apr 2021.

Vancouver:

Kobayashi S. Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining . [Internet] [Thesis]. Kyoto University; 2015. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/2433/199188.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kobayashi S. Rad18 and Rnf8 facilitate homologous recombination by two distinct mechanisms, promoting Rad51 focus formation and suppressing the toxic effect of nonhomologous end-joining . [Thesis]. Kyoto University; 2015. Available from: http://hdl.handle.net/2433/199188

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

25. Warmenhoven, John William. Towards the Development of Double Strand Break Repair Simulation in the Biological Stage of Geant4-DNA.

Degree: 2018, University of Manchester

 Radiotherapy is used to deliver a lethal dose of radiation to a target tumour volume whilst sparing healthy tissue as much as possible. The physical… (more)

Subjects/Keywords: Proton Therapy; Radiotherapy; DNA Repair; Non-homologous End Joining; Monte Carlo Simulation; Geant4-DNA; Double Strand Break Repair

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warmenhoven, J. W. (2018). Towards the Development of Double Strand Break Repair Simulation in the Biological Stage of Geant4-DNA. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315267

Chicago Manual of Style (16th Edition):

Warmenhoven, John William. “Towards the Development of Double Strand Break Repair Simulation in the Biological Stage of Geant4-DNA.” 2018. Doctoral Dissertation, University of Manchester. Accessed April 12, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315267.

MLA Handbook (7th Edition):

Warmenhoven, John William. “Towards the Development of Double Strand Break Repair Simulation in the Biological Stage of Geant4-DNA.” 2018. Web. 12 Apr 2021.

Vancouver:

Warmenhoven JW. Towards the Development of Double Strand Break Repair Simulation in the Biological Stage of Geant4-DNA. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2021 Apr 12]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315267.

Council of Science Editors:

Warmenhoven JW. Towards the Development of Double Strand Break Repair Simulation in the Biological Stage of Geant4-DNA. [Doctoral Dissertation]. University of Manchester; 2018. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:315267


McMaster University

26. Andres, Sara N. Functional Structures: The Role of XRCC4 and XLF in Mammalian DNA Double-Strand Break Repair.

Degree: PhD, 2011, McMaster University

DNA double-strand breaks pose a serious threat to genomic integrity. Double-strand breaks can cause chromosomal rearrangement, leading to uncontrolled cell proliferation, or even cell… (more)

Subjects/Keywords: XRCC4; XLF; Non-homologous end-joining; DNA repair; X-ray crystallography; Biochemistry, Biophysics, and Structural Biology; Biochemistry, Biophysics, and Structural Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andres, S. N. (2011). Functional Structures: The Role of XRCC4 and XLF in Mammalian DNA Double-Strand Break Repair. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/11265

Chicago Manual of Style (16th Edition):

Andres, Sara N. “Functional Structures: The Role of XRCC4 and XLF in Mammalian DNA Double-Strand Break Repair.” 2011. Doctoral Dissertation, McMaster University. Accessed April 12, 2021. http://hdl.handle.net/11375/11265.

MLA Handbook (7th Edition):

Andres, Sara N. “Functional Structures: The Role of XRCC4 and XLF in Mammalian DNA Double-Strand Break Repair.” 2011. Web. 12 Apr 2021.

Vancouver:

Andres SN. Functional Structures: The Role of XRCC4 and XLF in Mammalian DNA Double-Strand Break Repair. [Internet] [Doctoral dissertation]. McMaster University; 2011. [cited 2021 Apr 12]. Available from: http://hdl.handle.net/11375/11265.

Council of Science Editors:

Andres SN. Functional Structures: The Role of XRCC4 and XLF in Mammalian DNA Double-Strand Break Repair. [Doctoral Dissertation]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11265


University of Western Ontario

27. Hoffer, Sarah M. Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining.

Degree: 2015, University of Western Ontario

 DNA double strand breaks (DSBs) are a toxic and dangerous form of DNA damage repaired primarily by non-homologous end joining (NHEJ) in mammals. The Ku70/80… (more)

Subjects/Keywords: Ku70/80; non-homologous end joining; DNA double strand breaks; microirradiation; protein-protein interactions; lysine signaling; ionizing radiation; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hoffer, S. M. (2015). Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3015

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoffer, Sarah M. “Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining.” 2015. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/3015.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoffer, Sarah M. “Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining.” 2015. Web. 12 Apr 2021.

Vancouver:

Hoffer SM. Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/3015.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoffer SM. Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/3015

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

28. Walden, Elizabeth A. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.

Degree: 2017, University of Western Ontario

 Ku is a key component of the Non-Homologous End Joining DNA repair pathway. Recently, a function for Ku in DNA damage response (DDR) signalling was… (more)

Subjects/Keywords: DNA double strand breaks; Non-homologous end joining; DNA damage response; Cell cycle arrest; Ku; Aurora B; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Walden, E. A. (2017). Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walden, Elizabeth A. “Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.” 2017. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walden, Elizabeth A. “Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.” 2017. Web. 12 Apr 2021.

Vancouver:

Walden EA. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walden EA. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

29. Wang, Huasheng. Structural and Functional Characterization of Non-Homologous End Joining Factors.

Degree: 2017, University of Western Ontario

 DNA double strand breaks represent the most toxic form of DNA damage. In mammals, non-homologous end-joining (NHEJ) is the primary DNA repair pathway for such… (more)

Subjects/Keywords: DNA double strand breaks; Non-homologous end joining; Protein expression; Protein purification; Protein structure; X-ray crystallography; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, H. (2017). Structural and Functional Characterization of Non-Homologous End Joining Factors. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Huasheng. “Structural and Functional Characterization of Non-Homologous End Joining Factors.” 2017. Thesis, University of Western Ontario. Accessed April 12, 2021. https://ir.lib.uwo.ca/etd/4887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Huasheng. “Structural and Functional Characterization of Non-Homologous End Joining Factors.” 2017. Web. 12 Apr 2021.

Vancouver:

Wang H. Structural and Functional Characterization of Non-Homologous End Joining Factors. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2021 Apr 12]. Available from: https://ir.lib.uwo.ca/etd/4887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang H. Structural and Functional Characterization of Non-Homologous End Joining Factors. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

30. Warmenhoven, John. Towards the development of double strand break repair simulation in the biological stage of Geant4-DNA.

Degree: PhD, 2018, University of Manchester

 Radiotherapy is used to deliver a lethal dose of radiation to a target tumour volume whilst sparing healthy tissue as much as possible. The physical… (more)

Subjects/Keywords: Geant4-DNA; Double Strand Break Repair; Monte Carlo Simulation; DNA Repair; Non-homologous End Joining; Radiotherapy; Proton Therapy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warmenhoven, J. (2018). Towards the development of double strand break repair simulation in the biological stage of Geant4-DNA. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/towards-the-development-of-double-strand-break-repair-simulation-in-the-biological-stage-of-geant4dna(fa31e136-61fd-4c12-ab46-7525408ceedb).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816251

Chicago Manual of Style (16th Edition):

Warmenhoven, John. “Towards the development of double strand break repair simulation in the biological stage of Geant4-DNA.” 2018. Doctoral Dissertation, University of Manchester. Accessed April 12, 2021. https://www.research.manchester.ac.uk/portal/en/theses/towards-the-development-of-double-strand-break-repair-simulation-in-the-biological-stage-of-geant4dna(fa31e136-61fd-4c12-ab46-7525408ceedb).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816251.

MLA Handbook (7th Edition):

Warmenhoven, John. “Towards the development of double strand break repair simulation in the biological stage of Geant4-DNA.” 2018. Web. 12 Apr 2021.

Vancouver:

Warmenhoven J. Towards the development of double strand break repair simulation in the biological stage of Geant4-DNA. [Internet] [Doctoral dissertation]. University of Manchester; 2018. [cited 2021 Apr 12]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/towards-the-development-of-double-strand-break-repair-simulation-in-the-biological-stage-of-geant4dna(fa31e136-61fd-4c12-ab46-7525408ceedb).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816251.

Council of Science Editors:

Warmenhoven J. Towards the development of double strand break repair simulation in the biological stage of Geant4-DNA. [Doctoral Dissertation]. University of Manchester; 2018. Available from: https://www.research.manchester.ac.uk/portal/en/theses/towards-the-development-of-double-strand-break-repair-simulation-in-the-biological-stage-of-geant4dna(fa31e136-61fd-4c12-ab46-7525408ceedb).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816251

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