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You searched for subject:(neurofibromatosis type 1 associated tumors). Showing records 1 – 30 of 26385 total matches.

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Freie Universität Berlin

1. Lüth, Maria. Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors.

Degree: 2011, Freie Universität Berlin

 To detect somatic mitochondrial DNA mutations in neuroectodermal tumors, mutation analysis in patients with medulloblastoma, pilocytic astrocytoma and neurofibromatosis type 1-associated tumors was performed. MtDNA… (more)

Subjects/Keywords: mtDNA; somatic mutations; neurofibromatosis type 1-associated tumors; pilocytic astrocytoma; medulloblastoma; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lüth, M. (2011). Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/13747

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lüth, Maria. “Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors.” 2011. Thesis, Freie Universität Berlin. Accessed January 22, 2021. https://refubium.fu-berlin.de/handle/fub188/13747.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lüth, Maria. “Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors.” 2011. Web. 22 Jan 2021.

Vancouver:

Lüth M. Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2021 Jan 22]. Available from: https://refubium.fu-berlin.de/handle/fub188/13747.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lüth M. Analysis of mitochondrial DNA mutations in patients with neuroectodermal tumors. [Thesis]. Freie Universität Berlin; 2011. Available from: https://refubium.fu-berlin.de/handle/fub188/13747

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

2. Yap, Yoon-Sim. Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer.

Degree: 2018, University of Adelaide

 Title: Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer Overview: These studies were initiated after seeing a series of women with Neurofibromatosis Type(more)

Subjects/Keywords: Breast cancer; neurofibromatosis type 1; neurofibromin

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APA (6th Edition):

Yap, Y. (2018). Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/119702

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yap, Yoon-Sim. “Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer.” 2018. Thesis, University of Adelaide. Accessed January 22, 2021. http://hdl.handle.net/2440/119702.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yap, Yoon-Sim. “Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer.” 2018. Web. 22 Jan 2021.

Vancouver:

Yap Y. Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer. [Internet] [Thesis]. University of Adelaide; 2018. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2440/119702.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yap Y. Germline and Somatic Neurofibromatosis Type 1 Aberrations in Breast Cancer. [Thesis]. University of Adelaide; 2018. Available from: http://hdl.handle.net/2440/119702

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

3. Garg, Shruti. Autism Spectrum Disorder in Neufibromatosis Type 1: Prevalence and characterisation of the phenotype.

Degree: 2015, University of Manchester

 Autism Spectrum disorder (ASD) is a pervasive developmental disorder with a population prevalence of about 1%. The aetiology of ASD is complex and highly heterogeneous… (more)

Subjects/Keywords: Neurofibromatosis Type 1; Autism Spectrum Disorder

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APA (6th Edition):

Garg, S. (2015). Autism Spectrum Disorder in Neufibromatosis Type 1: Prevalence and characterisation of the phenotype. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269838

Chicago Manual of Style (16th Edition):

Garg, Shruti. “Autism Spectrum Disorder in Neufibromatosis Type 1: Prevalence and characterisation of the phenotype.” 2015. Doctoral Dissertation, University of Manchester. Accessed January 22, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269838.

MLA Handbook (7th Edition):

Garg, Shruti. “Autism Spectrum Disorder in Neufibromatosis Type 1: Prevalence and characterisation of the phenotype.” 2015. Web. 22 Jan 2021.

Vancouver:

Garg S. Autism Spectrum Disorder in Neufibromatosis Type 1: Prevalence and characterisation of the phenotype. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 Jan 22]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269838.

Council of Science Editors:

Garg S. Autism Spectrum Disorder in Neufibromatosis Type 1: Prevalence and characterisation of the phenotype. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269838


University of Southern California

4. Xia, Caihong. Carboplatin and vincristine chemotherapy for progressive low grade gliomas in pediatric patients with or without neurofibromatosis type 1 (NF1).

Degree: MS, Applied Biostatistics and Epidemiology, 2014, University of Southern California

 To evaluate and compare the therapeutic effects and toxicity of chemotherapy for low grade gliomas (LGG) in Neurofibromatosis Type 1 (NF1) and non-NF children, patients… (more)

Subjects/Keywords: chemotherapy; low grade gliomas; neurofibromatosis type 1

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APA (6th Edition):

Xia, C. (2014). Carboplatin and vincristine chemotherapy for progressive low grade gliomas in pediatric patients with or without neurofibromatosis type 1 (NF1). (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/507988/rec/1234

Chicago Manual of Style (16th Edition):

Xia, Caihong. “Carboplatin and vincristine chemotherapy for progressive low grade gliomas in pediatric patients with or without neurofibromatosis type 1 (NF1).” 2014. Masters Thesis, University of Southern California. Accessed January 22, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/507988/rec/1234.

MLA Handbook (7th Edition):

Xia, Caihong. “Carboplatin and vincristine chemotherapy for progressive low grade gliomas in pediatric patients with or without neurofibromatosis type 1 (NF1).” 2014. Web. 22 Jan 2021.

Vancouver:

Xia C. Carboplatin and vincristine chemotherapy for progressive low grade gliomas in pediatric patients with or without neurofibromatosis type 1 (NF1). [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2021 Jan 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/507988/rec/1234.

Council of Science Editors:

Xia C. Carboplatin and vincristine chemotherapy for progressive low grade gliomas in pediatric patients with or without neurofibromatosis type 1 (NF1). [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/507988/rec/1234


University of Manchester

5. Garg, Shruti. Autism Spectrum Disorder in Neufibromatosis Type 1 : prevalence and characterisation of the phenotype.

Degree: PhD, 2015, University of Manchester

 Autism Spectrum disorder (ASD) is a pervasive developmental disorder with a population prevalence of about 1%. The aetiology of ASD is complex and highly heterogeneous… (more)

Subjects/Keywords: Autism Spectrum Disorder; Neurofibromatosis Type 1

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APA (6th Edition):

Garg, S. (2015). Autism Spectrum Disorder in Neufibromatosis Type 1 : prevalence and characterisation of the phenotype. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/autism-spectrum-disorder-in-neufibromatosis-type-1-prevalence-and-characterisation-of-the-phenotype(8c34a30e-5ef7-4c95-a28a-d52af41a66d1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816244

Chicago Manual of Style (16th Edition):

Garg, Shruti. “Autism Spectrum Disorder in Neufibromatosis Type 1 : prevalence and characterisation of the phenotype.” 2015. Doctoral Dissertation, University of Manchester. Accessed January 22, 2021. https://www.research.manchester.ac.uk/portal/en/theses/autism-spectrum-disorder-in-neufibromatosis-type-1-prevalence-and-characterisation-of-the-phenotype(8c34a30e-5ef7-4c95-a28a-d52af41a66d1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816244.

MLA Handbook (7th Edition):

Garg, Shruti. “Autism Spectrum Disorder in Neufibromatosis Type 1 : prevalence and characterisation of the phenotype.” 2015. Web. 22 Jan 2021.

Vancouver:

Garg S. Autism Spectrum Disorder in Neufibromatosis Type 1 : prevalence and characterisation of the phenotype. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2021 Jan 22]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/autism-spectrum-disorder-in-neufibromatosis-type-1-prevalence-and-characterisation-of-the-phenotype(8c34a30e-5ef7-4c95-a28a-d52af41a66d1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816244.

Council of Science Editors:

Garg S. Autism Spectrum Disorder in Neufibromatosis Type 1 : prevalence and characterisation of the phenotype. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/autism-spectrum-disorder-in-neufibromatosis-type-1-prevalence-and-characterisation-of-the-phenotype(8c34a30e-5ef7-4c95-a28a-d52af41a66d1).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.816244


IUPUI

6. McDaniel, Andrew S. THE ROLE OF PAK1 IN THE CELLULAR AND MOLECULAR COMPONENTS OF PLEXIFORM NEUROFIBROMAS.

Degree: 2008, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Neurofibromatosis type I (NF1) is a common genetic disease that affects over 200,000 patients in North America, Europe, and Japan.… (more)

Subjects/Keywords: Neurofibromatosis type 1; Mast cells; Neurofibromatosis

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APA (6th Edition):

McDaniel, A. S. (2008). THE ROLE OF PAK1 IN THE CELLULAR AND MOLECULAR COMPONENTS OF PLEXIFORM NEUROFIBROMAS. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/1696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McDaniel, Andrew S. “THE ROLE OF PAK1 IN THE CELLULAR AND MOLECULAR COMPONENTS OF PLEXIFORM NEUROFIBROMAS.” 2008. Thesis, IUPUI. Accessed January 22, 2021. http://hdl.handle.net/1805/1696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McDaniel, Andrew S. “THE ROLE OF PAK1 IN THE CELLULAR AND MOLECULAR COMPONENTS OF PLEXIFORM NEUROFIBROMAS.” 2008. Web. 22 Jan 2021.

Vancouver:

McDaniel AS. THE ROLE OF PAK1 IN THE CELLULAR AND MOLECULAR COMPONENTS OF PLEXIFORM NEUROFIBROMAS. [Internet] [Thesis]. IUPUI; 2008. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1805/1696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McDaniel AS. THE ROLE OF PAK1 IN THE CELLULAR AND MOLECULAR COMPONENTS OF PLEXIFORM NEUROFIBROMAS. [Thesis]. IUPUI; 2008. Available from: http://hdl.handle.net/1805/1696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Luscan, Armelle. Identification des évènements génétiques impliqués dans la transformation maligne de la neurofibromatose de type 1 : Identification of genetics events involved in malignant transformation in neurofibromatosis type 1.

Degree: Docteur es, Génétique, 2016, Sorbonne Paris Cité

La neurofibromatose de type 1 (NF1) est un syndrome de prédisposition tumorale causée par une mutation perte-de-fonction du gène suppresseur de tumeurs NF1. Près de… (more)

Subjects/Keywords: Neurofibromatose de type 1; MPNST; PRC2; Neurofibromatosis type 1; MPNST; PRC2; 616.042

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APA (6th Edition):

Luscan, A. (2016). Identification des évènements génétiques impliqués dans la transformation maligne de la neurofibromatose de type 1 : Identification of genetics events involved in malignant transformation in neurofibromatosis type 1. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCB050

Chicago Manual of Style (16th Edition):

Luscan, Armelle. “Identification des évènements génétiques impliqués dans la transformation maligne de la neurofibromatose de type 1 : Identification of genetics events involved in malignant transformation in neurofibromatosis type 1.” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 22, 2021. http://www.theses.fr/2016USPCB050.

MLA Handbook (7th Edition):

Luscan, Armelle. “Identification des évènements génétiques impliqués dans la transformation maligne de la neurofibromatose de type 1 : Identification of genetics events involved in malignant transformation in neurofibromatosis type 1.” 2016. Web. 22 Jan 2021.

Vancouver:

Luscan A. Identification des évènements génétiques impliqués dans la transformation maligne de la neurofibromatose de type 1 : Identification of genetics events involved in malignant transformation in neurofibromatosis type 1. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2016USPCB050.

Council of Science Editors:

Luscan A. Identification des évènements génétiques impliqués dans la transformation maligne de la neurofibromatose de type 1 : Identification of genetics events involved in malignant transformation in neurofibromatosis type 1. [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCB050

8. Aubin, Deborah. Modélisation pathologique pour la neurofibromatose de Type 1 : développement d’un test d’étude et applications pour la découverte de molécules actives : Neurofibromatosis disease modelling : development of a test study and applications for the discovery of active molecules.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2019, Université Paris-Saclay (ComUE)

 La neurofibromatose de type 1 (NF1) représente la maladie génétique autosomale dominante la plus fréquente en France, après la mucovisidose, avec une incidence de 1(more)

Subjects/Keywords: Neurofibromatose type 1; Modélisation pathologique; Ostéoblastes; Type 1 neurofibromatosis; Disease modelling; Osteoblasts

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APA (6th Edition):

Aubin, D. (2019). Modélisation pathologique pour la neurofibromatose de Type 1 : développement d’un test d’étude et applications pour la découverte de molécules actives : Neurofibromatosis disease modelling : development of a test study and applications for the discovery of active molecules. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2019SACLE002

Chicago Manual of Style (16th Edition):

Aubin, Deborah. “Modélisation pathologique pour la neurofibromatose de Type 1 : développement d’un test d’étude et applications pour la découverte de molécules actives : Neurofibromatosis disease modelling : development of a test study and applications for the discovery of active molecules.” 2019. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 22, 2021. http://www.theses.fr/2019SACLE002.

MLA Handbook (7th Edition):

Aubin, Deborah. “Modélisation pathologique pour la neurofibromatose de Type 1 : développement d’un test d’étude et applications pour la découverte de molécules actives : Neurofibromatosis disease modelling : development of a test study and applications for the discovery of active molecules.” 2019. Web. 22 Jan 2021.

Vancouver:

Aubin D. Modélisation pathologique pour la neurofibromatose de Type 1 : développement d’un test d’étude et applications pour la découverte de molécules actives : Neurofibromatosis disease modelling : development of a test study and applications for the discovery of active molecules. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2019. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2019SACLE002.

Council of Science Editors:

Aubin D. Modélisation pathologique pour la neurofibromatose de Type 1 : développement d’un test d’étude et applications pour la découverte de molécules actives : Neurofibromatosis disease modelling : development of a test study and applications for the discovery of active molecules. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2019. Available from: http://www.theses.fr/2019SACLE002


Leiden University

9. Zonneveld, Lisette van. Neuronal activity during working memory performance in the developing brain of children and adolescents with Neurofibromatosis Type 1.

Degree: 2011, Leiden University

 This study investigated an aspect of cognitive functioning or more specifically of executive functioning, that appears to be strongly affected in NF1: working memory. The… (more)

Subjects/Keywords: Neurofibromatosis Type 1; Cognitive functioning; Working memory; fMRI; Neuronal activity

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APA (6th Edition):

Zonneveld, L. v. (2011). Neuronal activity during working memory performance in the developing brain of children and adolescents with Neurofibromatosis Type 1. (Masters Thesis). Leiden University. Retrieved from http://hdl.handle.net/1887/18331

Chicago Manual of Style (16th Edition):

Zonneveld, Lisette van. “Neuronal activity during working memory performance in the developing brain of children and adolescents with Neurofibromatosis Type 1.” 2011. Masters Thesis, Leiden University. Accessed January 22, 2021. http://hdl.handle.net/1887/18331.

MLA Handbook (7th Edition):

Zonneveld, Lisette van. “Neuronal activity during working memory performance in the developing brain of children and adolescents with Neurofibromatosis Type 1.” 2011. Web. 22 Jan 2021.

Vancouver:

Zonneveld Lv. Neuronal activity during working memory performance in the developing brain of children and adolescents with Neurofibromatosis Type 1. [Internet] [Masters thesis]. Leiden University; 2011. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1887/18331.

Council of Science Editors:

Zonneveld Lv. Neuronal activity during working memory performance in the developing brain of children and adolescents with Neurofibromatosis Type 1. [Masters Thesis]. Leiden University; 2011. Available from: http://hdl.handle.net/1887/18331


University of Wisconsin – Milwaukee

10. Casnar, Christina. Autism Spectrum Disorder Symptomatology in Children with Neurofibromatosis Type 1.

Degree: PhD, Psychology, 2017, University of Wisconsin – Milwaukee

  Social problems are a common concern of parents of children with Neurofibromatosis type 1 (NF1). There has been a recent surge of research examining… (more)

Subjects/Keywords: Autism Spectrum Disorder; Children; Neurofibromatosis Type 1; Social Skills; Clinical Psychology

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APA (6th Edition):

Casnar, C. (2017). Autism Spectrum Disorder Symptomatology in Children with Neurofibromatosis Type 1. (Doctoral Dissertation). University of Wisconsin – Milwaukee. Retrieved from https://dc.uwm.edu/etd/1591

Chicago Manual of Style (16th Edition):

Casnar, Christina. “Autism Spectrum Disorder Symptomatology in Children with Neurofibromatosis Type 1.” 2017. Doctoral Dissertation, University of Wisconsin – Milwaukee. Accessed January 22, 2021. https://dc.uwm.edu/etd/1591.

MLA Handbook (7th Edition):

Casnar, Christina. “Autism Spectrum Disorder Symptomatology in Children with Neurofibromatosis Type 1.” 2017. Web. 22 Jan 2021.

Vancouver:

Casnar C. Autism Spectrum Disorder Symptomatology in Children with Neurofibromatosis Type 1. [Internet] [Doctoral dissertation]. University of Wisconsin – Milwaukee; 2017. [cited 2021 Jan 22]. Available from: https://dc.uwm.edu/etd/1591.

Council of Science Editors:

Casnar C. Autism Spectrum Disorder Symptomatology in Children with Neurofibromatosis Type 1. [Doctoral Dissertation]. University of Wisconsin – Milwaukee; 2017. Available from: https://dc.uwm.edu/etd/1591


University of Sydney

11. Crawford, Hilda Annette. Neurofibromatosis Type 1 in Adulthood: Quality-of-Life, Life Experiences and Uptake of Health Monitoring .

Degree: 2016, University of Sydney

Neurofibromatosis type 1 (NF1) is a common genetic disorder characterised by skin stigmata, benign and malignant tumours, neurological deficit, cognitive dysfunction and disfigurement. Disease expression… (more)

Subjects/Keywords: Neurofibromatosis type 1; NF1; Quality-of-life; Health monitoring; Disease impact

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APA (6th Edition):

Crawford, H. A. (2016). Neurofibromatosis Type 1 in Adulthood: Quality-of-Life, Life Experiences and Uptake of Health Monitoring . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17214

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crawford, Hilda Annette. “Neurofibromatosis Type 1 in Adulthood: Quality-of-Life, Life Experiences and Uptake of Health Monitoring .” 2016. Thesis, University of Sydney. Accessed January 22, 2021. http://hdl.handle.net/2123/17214.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crawford, Hilda Annette. “Neurofibromatosis Type 1 in Adulthood: Quality-of-Life, Life Experiences and Uptake of Health Monitoring .” 2016. Web. 22 Jan 2021.

Vancouver:

Crawford HA. Neurofibromatosis Type 1 in Adulthood: Quality-of-Life, Life Experiences and Uptake of Health Monitoring . [Internet] [Thesis]. University of Sydney; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/2123/17214.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crawford HA. Neurofibromatosis Type 1 in Adulthood: Quality-of-Life, Life Experiences and Uptake of Health Monitoring . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/17214

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

12. Spence, John Paul. Nf1-DEFICIENT MICE DISPLAY SOCIAL LEARNING DEFICITS THAT ARE RESCUED BY THE DELETION OF PAK1 GENE.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Neurofibromatosis type 1 (NF1) is a neurocutaneous disorder that affects roughly 1 in 3500 individuals. In addition to physical features… (more)

Subjects/Keywords: Neurofibromatosis type 1 (NF1), social learning, p21-activated kinase 1 (PAK1), MAPK, prefrontal cortex, basolateral amygdala; Neurofibromatosis; Social learning; Prefrontal cortex

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APA (6th Edition):

Spence, J. P. (2011). Nf1-DEFICIENT MICE DISPLAY SOCIAL LEARNING DEFICITS THAT ARE RESCUED BY THE DELETION OF PAK1 GENE. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2521

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spence, John Paul. “Nf1-DEFICIENT MICE DISPLAY SOCIAL LEARNING DEFICITS THAT ARE RESCUED BY THE DELETION OF PAK1 GENE.” 2011. Thesis, IUPUI. Accessed January 22, 2021. http://hdl.handle.net/1805/2521.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spence, John Paul. “Nf1-DEFICIENT MICE DISPLAY SOCIAL LEARNING DEFICITS THAT ARE RESCUED BY THE DELETION OF PAK1 GENE.” 2011. Web. 22 Jan 2021.

Vancouver:

Spence JP. Nf1-DEFICIENT MICE DISPLAY SOCIAL LEARNING DEFICITS THAT ARE RESCUED BY THE DELETION OF PAK1 GENE. [Internet] [Thesis]. IUPUI; 2011. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1805/2521.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spence JP. Nf1-DEFICIENT MICE DISPLAY SOCIAL LEARNING DEFICITS THAT ARE RESCUED BY THE DELETION OF PAK1 GENE. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2521

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Cuberos, Hélène. Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2016, Université François-Rabelais de Tours

LIMK1 et LIMK2 sont des sérines/thréonine kinases capables de phosphoryler et d’inactiver la cofiline, un facteur de dépolymérisation de l’actine. Elles sont régulées négativement par… (more)

Subjects/Keywords: Neurofibromatose de type 1; Déficience intellectuelle; LIMK2; LIMK2‐1; Cofiline; Actine; Neurofibromatosis type 1; Intellectual disability; LIMK2‐1; Cofilin; Actin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cuberos, H. (2016). Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2016TOUR3306

Chicago Manual of Style (16th Edition):

Cuberos, Hélène. “Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability.” 2016. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed January 22, 2021. http://www.theses.fr/2016TOUR3306.

MLA Handbook (7th Edition):

Cuberos, Hélène. “Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability.” 2016. Web. 22 Jan 2021.

Vancouver:

Cuberos H. Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2016. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2016TOUR3306.

Council of Science Editors:

Cuberos H. Les LIM kinases dans la neurofibromatose de type 1 : caractérisation cellulaire et moléculaire de LIMK2-1, une isoforme associée à la déficience intellectuelle : LIM kinases in neurofibromatosis type 1 : cellular and molecular characterization of LIMK2-1, an isoform associated with intellectual disability. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2016. Available from: http://www.theses.fr/2016TOUR3306


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

14. Batzios, Spyros. Διερεύνηση της έκφρασης μορίων του εξωκυττάριου χώρου σε ασθενείς με βλεννοπολυσακχαριδώσεις και νευροϊνωμάτωση τύπου Ι.

Degree: 2015, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

 The aim of the present study was the assessment of the expression of various molecules of the Extracellular Matrix (ECM), such as matrix metalloproteinases (MMPs)… (more)

Subjects/Keywords: Βλεννοπολυσακχαριδώσεις; Νευροϊνωμάτωση τύπου 1; Εξωκυττάριος χώρος; Βιοδείκτες; Mucopolysaccharidoses; Neurofibromatosis type 1; Extracellular matrix; Biomarkers

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APA (6th Edition):

Batzios, S. (2015). Διερεύνηση της έκφρασης μορίων του εξωκυττάριου χώρου σε ασθενείς με βλεννοπολυσακχαριδώσεις και νευροϊνωμάτωση τύπου Ι. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/35697

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Batzios, Spyros. “Διερεύνηση της έκφρασης μορίων του εξωκυττάριου χώρου σε ασθενείς με βλεννοπολυσακχαριδώσεις και νευροϊνωμάτωση τύπου Ι.” 2015. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed January 22, 2021. http://hdl.handle.net/10442/hedi/35697.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Batzios, Spyros. “Διερεύνηση της έκφρασης μορίων του εξωκυττάριου χώρου σε ασθενείς με βλεννοπολυσακχαριδώσεις και νευροϊνωμάτωση τύπου Ι.” 2015. Web. 22 Jan 2021.

Vancouver:

Batzios S. Διερεύνηση της έκφρασης μορίων του εξωκυττάριου χώρου σε ασθενείς με βλεννοπολυσακχαριδώσεις και νευροϊνωμάτωση τύπου Ι. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2015. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10442/hedi/35697.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Batzios S. Διερεύνηση της έκφρασης μορίων του εξωκυττάριου χώρου σε ασθενείς με βλεννοπολυσακχαριδώσεις και νευροϊνωμάτωση τύπου Ι. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2015. Available from: http://hdl.handle.net/10442/hedi/35697

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Tastet, Julie. Etude des gènes LIMK2 et RNF135, impliqués dans les mécanismes moléculaires de la neurofibromatose de type 1, dans l'autisme et la déficience mentale : Study of LIMK2 and RNF135, involved in neurofibromatosis type 1, in autism and mental deficiency.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2012, Université François-Rabelais de Tours

L'autisme et la déficience mentale (DM) sont des pathologies neurodéveloppementales fréquentes qui partagent des facteurs génétiques communs. Afin de mieux comprendre leur étiologie, nous avons… (more)

Subjects/Keywords: Austisme; Déficience mentale; Neurofibromatose de type 1; LIMK2; Isoformes; Neuritogenèse; RNF135; Autism; Mental deficiency; Neurofibromatosis type 1; LIMK2; Isoforms; Neuritogenesis; RNF135

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APA (6th Edition):

Tastet, J. (2012). Etude des gènes LIMK2 et RNF135, impliqués dans les mécanismes moléculaires de la neurofibromatose de type 1, dans l'autisme et la déficience mentale : Study of LIMK2 and RNF135, involved in neurofibromatosis type 1, in autism and mental deficiency. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2012TOUR3311

Chicago Manual of Style (16th Edition):

Tastet, Julie. “Etude des gènes LIMK2 et RNF135, impliqués dans les mécanismes moléculaires de la neurofibromatose de type 1, dans l'autisme et la déficience mentale : Study of LIMK2 and RNF135, involved in neurofibromatosis type 1, in autism and mental deficiency.” 2012. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed January 22, 2021. http://www.theses.fr/2012TOUR3311.

MLA Handbook (7th Edition):

Tastet, Julie. “Etude des gènes LIMK2 et RNF135, impliqués dans les mécanismes moléculaires de la neurofibromatose de type 1, dans l'autisme et la déficience mentale : Study of LIMK2 and RNF135, involved in neurofibromatosis type 1, in autism and mental deficiency.” 2012. Web. 22 Jan 2021.

Vancouver:

Tastet J. Etude des gènes LIMK2 et RNF135, impliqués dans les mécanismes moléculaires de la neurofibromatose de type 1, dans l'autisme et la déficience mentale : Study of LIMK2 and RNF135, involved in neurofibromatosis type 1, in autism and mental deficiency. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2012. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2012TOUR3311.

Council of Science Editors:

Tastet J. Etude des gènes LIMK2 et RNF135, impliqués dans les mécanismes moléculaires de la neurofibromatose de type 1, dans l'autisme et la déficience mentale : Study of LIMK2 and RNF135, involved in neurofibromatosis type 1, in autism and mental deficiency. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2012. Available from: http://www.theses.fr/2012TOUR3311

16. Coutinho, Virginie. Difficultés motrices, cognitives et comportementales chez les enfants et adolescents atteints de neurofibromatose de type 1 (maladie de von Recklinghausen) : Motor, cognitive, and behavioral difficulties in children and adolescents with neurofibromatosis type 1 (von Recklinghausen disease).

Degree: Docteur es, Psychologie, 2015, Sorbonne Paris Cité

Les données de la littérature concernant les difficultés cognitives et comportementales dans la Neurofibromatose de type 1 (NF1) sont nombreuses avec des résultats parfois contradictoires.… (more)

Subjects/Keywords: Neurofibromatose de type 1; Enfant; Neuropsychologie; Comportement; Questionnaires aux parents; Neurofibromatosis type 1; Child; Neuropsychology; Behavior; Parental questionnaires; 150

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APA (6th Edition):

Coutinho, V. (2015). Difficultés motrices, cognitives et comportementales chez les enfants et adolescents atteints de neurofibromatose de type 1 (maladie de von Recklinghausen) : Motor, cognitive, and behavioral difficulties in children and adolescents with neurofibromatosis type 1 (von Recklinghausen disease). (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2015USPCB185

Chicago Manual of Style (16th Edition):

Coutinho, Virginie. “Difficultés motrices, cognitives et comportementales chez les enfants et adolescents atteints de neurofibromatose de type 1 (maladie de von Recklinghausen) : Motor, cognitive, and behavioral difficulties in children and adolescents with neurofibromatosis type 1 (von Recklinghausen disease).” 2015. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 22, 2021. http://www.theses.fr/2015USPCB185.

MLA Handbook (7th Edition):

Coutinho, Virginie. “Difficultés motrices, cognitives et comportementales chez les enfants et adolescents atteints de neurofibromatose de type 1 (maladie de von Recklinghausen) : Motor, cognitive, and behavioral difficulties in children and adolescents with neurofibromatosis type 1 (von Recklinghausen disease).” 2015. Web. 22 Jan 2021.

Vancouver:

Coutinho V. Difficultés motrices, cognitives et comportementales chez les enfants et adolescents atteints de neurofibromatose de type 1 (maladie de von Recklinghausen) : Motor, cognitive, and behavioral difficulties in children and adolescents with neurofibromatosis type 1 (von Recklinghausen disease). [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2015. [cited 2021 Jan 22]. Available from: http://www.theses.fr/2015USPCB185.

Council of Science Editors:

Coutinho V. Difficultés motrices, cognitives et comportementales chez les enfants et adolescents atteints de neurofibromatose de type 1 (maladie de von Recklinghausen) : Motor, cognitive, and behavioral difficulties in children and adolescents with neurofibromatosis type 1 (von Recklinghausen disease). [Doctoral Dissertation]. Sorbonne Paris Cité; 2015. Available from: http://www.theses.fr/2015USPCB185


Vanderbilt University

17. Tahaei, Seyedmohammad Ebrahim. Molecular Bases of the Reduced Osteogenic Differentiation Potential in Nf1 Deficient Osteoprogenitors.

Degree: PhD, Pharmacology, 2018, Vanderbilt University

Neurofibromatosis type 1 results from mutations in NF1, a gene that encodes Neurofibromin. This common genetic condition is associated with tibial pseudarthrosis (PA), whose etiology… (more)

Subjects/Keywords: RNA-Seq; Neurofibromatosis type 1; Bone marrow stroll cells; Differentiation; MAPK signaling; Epiregulin

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APA (6th Edition):

Tahaei, S. E. (2018). Molecular Bases of the Reduced Osteogenic Differentiation Potential in Nf1 Deficient Osteoprogenitors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10471

Chicago Manual of Style (16th Edition):

Tahaei, Seyedmohammad Ebrahim. “Molecular Bases of the Reduced Osteogenic Differentiation Potential in Nf1 Deficient Osteoprogenitors.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2021. http://hdl.handle.net/1803/10471.

MLA Handbook (7th Edition):

Tahaei, Seyedmohammad Ebrahim. “Molecular Bases of the Reduced Osteogenic Differentiation Potential in Nf1 Deficient Osteoprogenitors.” 2018. Web. 22 Jan 2021.

Vancouver:

Tahaei SE. Molecular Bases of the Reduced Osteogenic Differentiation Potential in Nf1 Deficient Osteoprogenitors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1803/10471.

Council of Science Editors:

Tahaei SE. Molecular Bases of the Reduced Osteogenic Differentiation Potential in Nf1 Deficient Osteoprogenitors. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/10471


Vanderbilt University

18. Karolak, Matthew Ross. Neurofibromin Regulated Signaling Pathways in Endochondral Ossification.

Degree: PhD, Pharmacology, 2015, Vanderbilt University

Neurofibromatosis type 1 (NF1) is the most common autosomal dominant genetic disorder occurring in 1 of every 3500 live births. NF1 is caused by loss-of-function… (more)

Subjects/Keywords: Dwarfism; FGFR1; NF1; Neurofibromatosis Type 1; Growth Plate; Chondrocyte; Fracture Healing; Neurofibromin

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APA (6th Edition):

Karolak, M. R. (2015). Neurofibromin Regulated Signaling Pathways in Endochondral Ossification. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12844

Chicago Manual of Style (16th Edition):

Karolak, Matthew Ross. “Neurofibromin Regulated Signaling Pathways in Endochondral Ossification.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2021. http://hdl.handle.net/1803/12844.

MLA Handbook (7th Edition):

Karolak, Matthew Ross. “Neurofibromin Regulated Signaling Pathways in Endochondral Ossification.” 2015. Web. 22 Jan 2021.

Vancouver:

Karolak MR. Neurofibromin Regulated Signaling Pathways in Endochondral Ossification. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/1803/12844.

Council of Science Editors:

Karolak MR. Neurofibromin Regulated Signaling Pathways in Endochondral Ossification. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/12844


University of Cincinnati

19. Azage, Meron Y., B.S. Fracture Rates in Adults with Neurofibromatosis Type 1.

Degree: MS, Medicine: Genetic Counseling, 2012, University of Cincinnati

 About 30-50% of patients with Neurofibromatosis Type 1 (NF1) have disease involving the skeletal system. Osteoporosis and low bone density are common findings; however increased… (more)

Subjects/Keywords: Genetics; Neurofibromatosis Type 1 (NF1); Adult; Fractures; Bone; Bone Mineral Density (BMD); Calcium

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APA (6th Edition):

Azage, Meron Y., B. S. (2012). Fracture Rates in Adults with Neurofibromatosis Type 1. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337352142

Chicago Manual of Style (16th Edition):

Azage, Meron Y., B S. “Fracture Rates in Adults with Neurofibromatosis Type 1.” 2012. Masters Thesis, University of Cincinnati. Accessed January 22, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337352142.

MLA Handbook (7th Edition):

Azage, Meron Y., B S. “Fracture Rates in Adults with Neurofibromatosis Type 1.” 2012. Web. 22 Jan 2021.

Vancouver:

Azage, Meron Y. BS. Fracture Rates in Adults with Neurofibromatosis Type 1. [Internet] [Masters thesis]. University of Cincinnati; 2012. [cited 2021 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337352142.

Council of Science Editors:

Azage, Meron Y. BS. Fracture Rates in Adults with Neurofibromatosis Type 1. [Masters Thesis]. University of Cincinnati; 2012. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1337352142


University of Cincinnati

20. Grandine, Hayley. Patient and Parent Experiences of Dual Genetic Diagnoses: Neurofibromatosis Type 1 and an Additional Genetic Disease.

Degree: MS, Medicine: Genetic Counseling, 2016, University of Cincinnati

 Dual or multiple genetic diagnoses, although uncommon, are anecdotally evident from clinical geneticists and genetic counselors, and are found in 1% of individuals who undergo… (more)

Subjects/Keywords: Genetics; Neurofibromatosis Type 1; Dual Diagnosis; Genetic Disease; Emotional Impact; Life Experiences

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APA (6th Edition):

Grandine, H. (2016). Patient and Parent Experiences of Dual Genetic Diagnoses: Neurofibromatosis Type 1 and an Additional Genetic Disease. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459528629

Chicago Manual of Style (16th Edition):

Grandine, Hayley. “Patient and Parent Experiences of Dual Genetic Diagnoses: Neurofibromatosis Type 1 and an Additional Genetic Disease.” 2016. Masters Thesis, University of Cincinnati. Accessed January 22, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459528629.

MLA Handbook (7th Edition):

Grandine, Hayley. “Patient and Parent Experiences of Dual Genetic Diagnoses: Neurofibromatosis Type 1 and an Additional Genetic Disease.” 2016. Web. 22 Jan 2021.

Vancouver:

Grandine H. Patient and Parent Experiences of Dual Genetic Diagnoses: Neurofibromatosis Type 1 and an Additional Genetic Disease. [Internet] [Masters thesis]. University of Cincinnati; 2016. [cited 2021 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459528629.

Council of Science Editors:

Grandine H. Patient and Parent Experiences of Dual Genetic Diagnoses: Neurofibromatosis Type 1 and an Additional Genetic Disease. [Masters Thesis]. University of Cincinnati; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1459528629


Freie Universität Berlin

21. Titze, Sabrina. Impact of epigenetic modifications on the phenotypic variability of neurofibromatosis type 1.

Degree: 2011, Freie Universität Berlin

Neurofibromatosis type 1 (NF1) is a common autosomal dominant disorder caused by heterozygotic inactivation of the NF1 tumor suppressor gene at 17q11.2. The associated phenotypes… (more)

Subjects/Keywords: neurofibromatosis type 1 (NF1); mismatch repair; promoter; methylation; modifier; monozygotic twins; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Titze, S. (2011). Impact of epigenetic modifications on the phenotypic variability of neurofibromatosis type 1. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-5510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Titze, Sabrina. “Impact of epigenetic modifications on the phenotypic variability of neurofibromatosis type 1.” 2011. Thesis, Freie Universität Berlin. Accessed January 22, 2021. http://dx.doi.org/10.17169/refubium-5510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Titze, Sabrina. “Impact of epigenetic modifications on the phenotypic variability of neurofibromatosis type 1.” 2011. Web. 22 Jan 2021.

Vancouver:

Titze S. Impact of epigenetic modifications on the phenotypic variability of neurofibromatosis type 1. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2021 Jan 22]. Available from: http://dx.doi.org/10.17169/refubium-5510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Titze S. Impact of epigenetic modifications on the phenotypic variability of neurofibromatosis type 1. [Thesis]. Freie Universität Berlin; 2011. Available from: http://dx.doi.org/10.17169/refubium-5510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brandeis University

22. Robart, Sarah. Investigating Genetic Counselors' Experiences with Legius Syndrome.

Degree: 2012, Brandeis University

 Legius syndrome (LS), caused by mutations in SPRED1, is a recently identified condition characterized primarily by multiple café-au-lait macules and skin fold freckling, findings that… (more)

Subjects/Keywords: Legius syndrome; SPRED1; Neurofibromatosis type 1; NF1; genetic counselors; skin fold freckling; café-au-lait macules

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APA (6th Edition):

Robart, S. (2012). Investigating Genetic Counselors' Experiences with Legius Syndrome. (Thesis). Brandeis University. Retrieved from http://hdl.handle.net/10192/68

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robart, Sarah. “Investigating Genetic Counselors' Experiences with Legius Syndrome.” 2012. Thesis, Brandeis University. Accessed January 22, 2021. http://hdl.handle.net/10192/68.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robart, Sarah. “Investigating Genetic Counselors' Experiences with Legius Syndrome.” 2012. Web. 22 Jan 2021.

Vancouver:

Robart S. Investigating Genetic Counselors' Experiences with Legius Syndrome. [Internet] [Thesis]. Brandeis University; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10192/68.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robart S. Investigating Genetic Counselors' Experiences with Legius Syndrome. [Thesis]. Brandeis University; 2012. Available from: http://hdl.handle.net/10192/68

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

23. Johansson, L. Gunnar. Identification of Targeted Therapeutics for Malignant Peripheral Nerve Sheath Tumors.

Degree: PhD, Medicine : Cell and Molecular Biology, 2008, University of Cincinnati

Neurofibromatosis type 1 (NF1) is one of the most common autosomal dominant disorders, affecting 1 in 3,500 worldwide. The hallmark of NF1 is the expression… (more)

Subjects/Keywords: Cellular Biology; Neurofibromatosis type 1; NF1; MPNST; mTOR; Cancer

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APA (6th Edition):

Johansson, L. G. (2008). Identification of Targeted Therapeutics for Malignant Peripheral Nerve Sheath Tumors. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1216841242

Chicago Manual of Style (16th Edition):

Johansson, L Gunnar. “Identification of Targeted Therapeutics for Malignant Peripheral Nerve Sheath Tumors.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed January 22, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1216841242.

MLA Handbook (7th Edition):

Johansson, L Gunnar. “Identification of Targeted Therapeutics for Malignant Peripheral Nerve Sheath Tumors.” 2008. Web. 22 Jan 2021.

Vancouver:

Johansson LG. Identification of Targeted Therapeutics for Malignant Peripheral Nerve Sheath Tumors. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2021 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1216841242.

Council of Science Editors:

Johansson LG. Identification of Targeted Therapeutics for Malignant Peripheral Nerve Sheath Tumors. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1216841242

24. Hinman, Melissa N. The Function and Regulation of Neurofibromatosis Type 1 Exon 23a Alternative Splicing.

Degree: PhD, Genetics, 2014, Case Western Reserve University School of Graduate Studies

Neurofibromatosis type 1 (NF1), a common human genetic disease affecting the nervous system, is characterized by phenotypes ranging from increased tumor susceptibility to learning… (more)

Subjects/Keywords: Genetics; Biology; Biomedical Research; Molecular Biology; Neurosciences; Neurofibromatosis type 1; NF1; Alternative splicing; Hu proteins; HuC; ELAVL3; ELAVL proteins

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APA (6th Edition):

Hinman, M. N. (2014). The Function and Regulation of Neurofibromatosis Type 1 Exon 23a Alternative Splicing. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1390500738

Chicago Manual of Style (16th Edition):

Hinman, Melissa N. “The Function and Regulation of Neurofibromatosis Type 1 Exon 23a Alternative Splicing.” 2014. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed January 22, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1390500738.

MLA Handbook (7th Edition):

Hinman, Melissa N. “The Function and Regulation of Neurofibromatosis Type 1 Exon 23a Alternative Splicing.” 2014. Web. 22 Jan 2021.

Vancouver:

Hinman MN. The Function and Regulation of Neurofibromatosis Type 1 Exon 23a Alternative Splicing. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2014. [cited 2021 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1390500738.

Council of Science Editors:

Hinman MN. The Function and Regulation of Neurofibromatosis Type 1 Exon 23a Alternative Splicing. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1390500738


University of Cincinnati

25. RANGWALA, FATIMA ABDULLA. RAS SIGNALING IN SCHWANN CELL TUMOR FORMATION: NEUROFIBROMATOSIS TYPE 1.

Degree: PhD, Medicine : Interdisciplinary (Medical Science Scholars, Neuroscience), 2003, University of Cincinnati

 Patients with Neurofibromatosis Type 1 (NF1) carry a germline mutation in the <i>NF1</i> gene and have an increased risk for developing a variety of benign… (more)

Subjects/Keywords: neurofibromatosis Type 1; Schwann Cell; RAS; MPNST; microarray

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

RANGWALA, F. A. (2003). RAS SIGNALING IN SCHWANN CELL TUMOR FORMATION: NEUROFIBROMATOSIS TYPE 1. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1069774794

Chicago Manual of Style (16th Edition):

RANGWALA, FATIMA ABDULLA. “RAS SIGNALING IN SCHWANN CELL TUMOR FORMATION: NEUROFIBROMATOSIS TYPE 1.” 2003. Doctoral Dissertation, University of Cincinnati. Accessed January 22, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1069774794.

MLA Handbook (7th Edition):

RANGWALA, FATIMA ABDULLA. “RAS SIGNALING IN SCHWANN CELL TUMOR FORMATION: NEUROFIBROMATOSIS TYPE 1.” 2003. Web. 22 Jan 2021.

Vancouver:

RANGWALA FA. RAS SIGNALING IN SCHWANN CELL TUMOR FORMATION: NEUROFIBROMATOSIS TYPE 1. [Internet] [Doctoral dissertation]. University of Cincinnati; 2003. [cited 2021 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1069774794.

Council of Science Editors:

RANGWALA FA. RAS SIGNALING IN SCHWANN CELL TUMOR FORMATION: NEUROFIBROMATOSIS TYPE 1. [Doctoral Dissertation]. University of Cincinnati; 2003. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1069774794


University of Florida

26. Loda, Rebecca. Analysis of NF1 Mutation Mechanisms.

Degree: PhD, Medical Sciences - Genetics (IDP), 2009, University of Florida

 ANALYSIS OF NF1 MUTATION MECHANISMS By Rebecca L. Loda-Hutchinson May 2009 Chair: Margaret R. Wallace Major: Medical Sciences?Genetics Neurofibromin is a large protein encoded by… (more)

Subjects/Keywords: Exons; Genetic mutation; Hum; Messenger RNA; Neurofibroma; Neurofibromatosis 1; Neurofibromatosis 1 genes; Pathos; Schwann cells; Tumors

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APA (6th Edition):

Loda, R. (2009). Analysis of NF1 Mutation Mechanisms. (Doctoral Dissertation). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0024364

Chicago Manual of Style (16th Edition):

Loda, Rebecca. “Analysis of NF1 Mutation Mechanisms.” 2009. Doctoral Dissertation, University of Florida. Accessed January 22, 2021. https://ufdc.ufl.edu/UFE0024364.

MLA Handbook (7th Edition):

Loda, Rebecca. “Analysis of NF1 Mutation Mechanisms.” 2009. Web. 22 Jan 2021.

Vancouver:

Loda R. Analysis of NF1 Mutation Mechanisms. [Internet] [Doctoral dissertation]. University of Florida; 2009. [cited 2021 Jan 22]. Available from: https://ufdc.ufl.edu/UFE0024364.

Council of Science Editors:

Loda R. Analysis of NF1 Mutation Mechanisms. [Doctoral Dissertation]. University of Florida; 2009. Available from: https://ufdc.ufl.edu/UFE0024364


Universidade do Rio Grande do Sul

27. Bueno, Larissa Souza Mario. Vitamina D, polimorfismos do gene VDR e neurofibromatose 1.

Degree: 2012, Universidade do Rio Grande do Sul

Introdução: A Neurofibromatose tipo 1 (NF1) é uma doença genética autossômica dominante causada por mutação no gene NF1 (17q11.2; proteína neurofibromina). A incidência é de… (more)

Subjects/Keywords: Vitamina D; Neurofibromatosis type 1; Polimorfismo genético; Vitamin D; Neurofibromatose 1; Neurofibromas; Hypovitaminosis D; Deficiência de vitamina D; BsmI e FokI Polymorphism; VDR gene; Human; Brazil

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APA (6th Edition):

Bueno, L. S. M. (2012). Vitamina D, polimorfismos do gene VDR e neurofibromatose 1. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/52955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bueno, Larissa Souza Mario. “Vitamina D, polimorfismos do gene VDR e neurofibromatose 1.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed January 22, 2021. http://hdl.handle.net/10183/52955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bueno, Larissa Souza Mario. “Vitamina D, polimorfismos do gene VDR e neurofibromatose 1.” 2012. Web. 22 Jan 2021.

Vancouver:

Bueno LSM. Vitamina D, polimorfismos do gene VDR e neurofibromatose 1. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10183/52955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bueno LSM. Vitamina D, polimorfismos do gene VDR e neurofibromatose 1. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/52955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

28. Protas, Júlia Schneider. Avaliação da qualidade de vida em pacientes adultos com neurofibromatose tipo 1.

Degree: 2016, Universidade do Rio Grande do Sul

Base teórica: A qualidade de vida é uma variável amplamente estudada nas ciências da saúde e tem se tornado, cada vez mais, um indicativo importante… (more)

Subjects/Keywords: Qualidade de vida; Quality of life; Neurofibromatosis type 1; Neurofibromatose 1; Depressão; Coping strategies; Perception of family support; Depressive symptoms; Anxiety symptoms

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APA (6th Edition):

Protas, J. S. (2016). Avaliação da qualidade de vida em pacientes adultos com neurofibromatose tipo 1. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/149459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Protas, Júlia Schneider. “Avaliação da qualidade de vida em pacientes adultos com neurofibromatose tipo 1.” 2016. Thesis, Universidade do Rio Grande do Sul. Accessed January 22, 2021. http://hdl.handle.net/10183/149459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Protas, Júlia Schneider. “Avaliação da qualidade de vida em pacientes adultos com neurofibromatose tipo 1.” 2016. Web. 22 Jan 2021.

Vancouver:

Protas JS. Avaliação da qualidade de vida em pacientes adultos com neurofibromatose tipo 1. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2016. [cited 2021 Jan 22]. Available from: http://hdl.handle.net/10183/149459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Protas JS. Avaliação da qualidade de vida em pacientes adultos com neurofibromatose tipo 1. [Thesis]. Universidade do Rio Grande do Sul; 2016. Available from: http://hdl.handle.net/10183/149459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

29. BENNETT, MICHAEL R. PERTURBATIONS IN OLIGODENDROCYTE PROGENITOR GROWTH AND DIFFERENTIATION: NEUROFIBROMIN AND FGF2 SIGNALING.

Degree: PhD, Medicine : Neuroscience/Medical Science Scholars Interdisiplinary, 2004, University of Cincinnati

 The study of oligodendrogenesis and of stem cells in general has become a burgeoning field in recent years. The ability to replace specific cells of… (more)

Subjects/Keywords: Oligodendrocyte; Neurofibromin; FGF2 Signaling; Ras Signaling; Neurofibromatosis Type 1; Notch-1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

BENNETT, M. R. (2004). PERTURBATIONS IN OLIGODENDROCYTE PROGENITOR GROWTH AND DIFFERENTIATION: NEUROFIBROMIN AND FGF2 SIGNALING. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1100015367

Chicago Manual of Style (16th Edition):

BENNETT, MICHAEL R. “PERTURBATIONS IN OLIGODENDROCYTE PROGENITOR GROWTH AND DIFFERENTIATION: NEUROFIBROMIN AND FGF2 SIGNALING.” 2004. Doctoral Dissertation, University of Cincinnati. Accessed January 22, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1100015367.

MLA Handbook (7th Edition):

BENNETT, MICHAEL R. “PERTURBATIONS IN OLIGODENDROCYTE PROGENITOR GROWTH AND DIFFERENTIATION: NEUROFIBROMIN AND FGF2 SIGNALING.” 2004. Web. 22 Jan 2021.

Vancouver:

BENNETT MR. PERTURBATIONS IN OLIGODENDROCYTE PROGENITOR GROWTH AND DIFFERENTIATION: NEUROFIBROMIN AND FGF2 SIGNALING. [Internet] [Doctoral dissertation]. University of Cincinnati; 2004. [cited 2021 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1100015367.

Council of Science Editors:

BENNETT MR. PERTURBATIONS IN OLIGODENDROCYTE PROGENITOR GROWTH AND DIFFERENTIATION: NEUROFIBROMIN AND FGF2 SIGNALING. [Doctoral Dissertation]. University of Cincinnati; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1100015367

30. José Roberto Lopes Ferraz Filho. Ressonância magnética com imagem de tensor de difusão e espectroscopia na avaliação dos focos de hipersinal na ponderação T2 no encéfalo em crianças e adolescentes com neurofibromatose tipo 1.

Degree: 2011, Faculdade de Medicina de São José do Rio Preto

A neurofibromatose tipo 1 (NF1) em crianças e adolescentes está frequentemente associada com o aparecimento ou desaparecimento de lesões focais de hipersinal no encéfalo na… (more)

Subjects/Keywords: Ressonância magnética; Unidentified bright objects; Imagem de tensor de difusão; Neurofibromatose tipo 1; Espectroscopia de Ressonância Magnética; RADIOLOGIA MEDICA; Neurofibromatosis type 1; Magnetic resonance imaging; Unidentified Bright Objects; Diffusion tensor imaging; Magnetic resonance spectroscopy.; Neurofibromatosis type 1; Espectroscopía de Resonancia Magnética; Magnetic Resonance Spectroscopy; Neurofibromatose tipo 1; Espectroscopia por ressonância magnética

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Filho, J. R. L. F. (2011). Ressonância magnética com imagem de tensor de difusão e espectroscopia na avaliação dos focos de hipersinal na ponderação T2 no encéfalo em crianças e adolescentes com neurofibromatose tipo 1. (Thesis). Faculdade de Medicina de São José do Rio Preto. Retrieved from http://bdtd.famerp.br//tde_busca/arquivo.php?codArquivo=336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Filho, José Roberto Lopes Ferraz. “Ressonância magnética com imagem de tensor de difusão e espectroscopia na avaliação dos focos de hipersinal na ponderação T2 no encéfalo em crianças e adolescentes com neurofibromatose tipo 1.” 2011. Thesis, Faculdade de Medicina de São José do Rio Preto. Accessed January 22, 2021. http://bdtd.famerp.br//tde_busca/arquivo.php?codArquivo=336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Filho, José Roberto Lopes Ferraz. “Ressonância magnética com imagem de tensor de difusão e espectroscopia na avaliação dos focos de hipersinal na ponderação T2 no encéfalo em crianças e adolescentes com neurofibromatose tipo 1.” 2011. Web. 22 Jan 2021.

Vancouver:

Filho JRLF. Ressonância magnética com imagem de tensor de difusão e espectroscopia na avaliação dos focos de hipersinal na ponderação T2 no encéfalo em crianças e adolescentes com neurofibromatose tipo 1. [Internet] [Thesis]. Faculdade de Medicina de São José do Rio Preto; 2011. [cited 2021 Jan 22]. Available from: http://bdtd.famerp.br//tde_busca/arquivo.php?codArquivo=336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Filho JRLF. Ressonância magnética com imagem de tensor de difusão e espectroscopia na avaliação dos focos de hipersinal na ponderação T2 no encéfalo em crianças e adolescentes com neurofibromatose tipo 1. [Thesis]. Faculdade de Medicina de São José do Rio Preto; 2011. Available from: http://bdtd.famerp.br//tde_busca/arquivo.php?codArquivo=336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [880]

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