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University: University of Cambridge

You searched for subject:(neurodegeneration). Showing records 1 – 18 of 18 total matches.

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University of Cambridge

1. Sohail, Anood. Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila .

Degree: 2019, University of Cambridge

 Axons possess a continuous network of smooth tubular endoplasmic reticulum (ER), extending from the nuclear envelope throughout the neuron to synapses. Mutations affecting proteins with… (more)

Subjects/Keywords: Axonal ER; neurodegeneration; drosophila

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APA (6th Edition):

Sohail, A. (2019). Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/290113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sohail, Anood. “Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila .” 2019. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/290113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sohail, Anood. “Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila .” 2019. Web. 23 Aug 2019.

Vancouver:

Sohail A. Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila . [Internet] [Thesis]. University of Cambridge; 2019. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/290113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sohail A. Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila . [Thesis]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/290113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

2. Levy, Daniel Robert Siegfried. Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system .

Degree: 2018, University of Cambridge

 Leucine rich repeat kinase 2 (LRRK2) is a 286 kDa protein expressed in a variety of tissues and cell types, including neuronal tissue and innate… (more)

Subjects/Keywords: immunology; Parkinson's disease; neurodegeneration

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APA (6th Edition):

Levy, D. R. S. (2018). Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/273736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Levy, Daniel Robert Siegfried. “Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system .” 2018. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/273736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Levy, Daniel Robert Siegfried. “Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system .” 2018. Web. 23 Aug 2019.

Vancouver:

Levy DRS. Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system . [Internet] [Thesis]. University of Cambridge; 2018. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/273736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Levy DRS. Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system . [Thesis]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/273736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Santivanez Perez, Jessica Andrea. Effect of Parkinson's disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations.

Degree: PhD, 2017, University of Cambridge

 Parkinson’s disease (PD) is the second most common age-related neurodegenerative condition. It is neuropathologically characterised by the presence of Lewy pathology and the degeneration of… (more)

Subjects/Keywords: 616.8; Alpha-synuclein; Parkinson's Disease; iPSCs; Neurodegeneration

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APA (6th Edition):

Santivanez Perez, J. A. (2017). Effect of Parkinson's disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/267708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.725516

Chicago Manual of Style (16th Edition):

Santivanez Perez, Jessica Andrea. “Effect of Parkinson's disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations.” 2017. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/267708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.725516.

MLA Handbook (7th Edition):

Santivanez Perez, Jessica Andrea. “Effect of Parkinson's disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations.” 2017. Web. 23 Aug 2019.

Vancouver:

Santivanez Perez JA. Effect of Parkinson's disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations. [Internet] [Doctoral dissertation]. University of Cambridge; 2017. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/267708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.725516.

Council of Science Editors:

Santivanez Perez JA. Effect of Parkinson's disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations. [Doctoral Dissertation]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/267708 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.725516


University of Cambridge

4. Santivanez Perez, Jessica Andrea. Effect of Parkinson’s disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations .

Degree: 2017, University of Cambridge

 Parkinson’s disease (PD) is the second most common age-related neurodegenerative condition. It is neuropathologically characterised by the presence of Lewy pathology and the degeneration of… (more)

Subjects/Keywords: Alpha-synuclein; Parkinson's Disease; iPSCs; Neurodegeneration

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APA (6th Edition):

Santivanez Perez, J. A. (2017). Effect of Parkinson’s disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/267708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santivanez Perez, Jessica Andrea. “Effect of Parkinson’s disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations .” 2017. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/267708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santivanez Perez, Jessica Andrea. “Effect of Parkinson’s disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations .” 2017. Web. 23 Aug 2019.

Vancouver:

Santivanez Perez JA. Effect of Parkinson’s disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations . [Internet] [Thesis]. University of Cambridge; 2017. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/267708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santivanez Perez JA. Effect of Parkinson’s disease-related alpha-synuclein abnormalities on the maturation of distinct iPSC-derived neuronal populations . [Thesis]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/267708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Iljina, Marija. Aggregation of alpha-synuclein using single-molecule spectroscopy .

Degree: 2017, University of Cambridge

 The aggregation of alpha-synuclein (αS) protein from soluble monomer into solid amyloid fibrils in the brain is associated with a range of devastating neurodegenerative disorders… (more)

Subjects/Keywords: alpha-synuclein; single-molecule FRET; protein aggregation; neurodegeneration

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APA (6th Edition):

Iljina, M. (2017). Aggregation of alpha-synuclein using single-molecule spectroscopy . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/263216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Iljina, Marija. “Aggregation of alpha-synuclein using single-molecule spectroscopy .” 2017. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/263216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Iljina, Marija. “Aggregation of alpha-synuclein using single-molecule spectroscopy .” 2017. Web. 23 Aug 2019.

Vancouver:

Iljina M. Aggregation of alpha-synuclein using single-molecule spectroscopy . [Internet] [Thesis]. University of Cambridge; 2017. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/263216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Iljina M. Aggregation of alpha-synuclein using single-molecule spectroscopy . [Thesis]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/263216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

6. Cardova, Alzbeta. Modelling prion-induced neurodegeneration in PrP transgenic Drosophila .

Degree: 2019, University of Cambridge

 The aim of my thesis was to develop and characterise PrP transgenic Drosophila melanogaster of various genotypes to study the process of prion-induced neurodegeneration in… (more)

Subjects/Keywords: prion; Drosophila; protein; misfolding; neurodegeneration; PrP; aging; prion disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cardova, A. (2019). Modelling prion-induced neurodegeneration in PrP transgenic Drosophila . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/290412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cardova, Alzbeta. “Modelling prion-induced neurodegeneration in PrP transgenic Drosophila .” 2019. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/290412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cardova, Alzbeta. “Modelling prion-induced neurodegeneration in PrP transgenic Drosophila .” 2019. Web. 23 Aug 2019.

Vancouver:

Cardova A. Modelling prion-induced neurodegeneration in PrP transgenic Drosophila . [Internet] [Thesis]. University of Cambridge; 2019. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/290412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cardova A. Modelling prion-induced neurodegeneration in PrP transgenic Drosophila . [Thesis]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/290412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

7. Cardova, Alzbeta. Modelling prion-induced neurodegeneration in PrP transgenic Drosophila.

Degree: PhD, 2019, University of Cambridge

 The aim of my thesis was to develop and characterise PrP transgenic Drosophila melanogaster of various genotypes to study the process of prion-induced neurodegeneration in… (more)

Subjects/Keywords: prion; Drosophila; protein; misfolding; neurodegeneration; PrP; aging; prion disease

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cardova, A. (2019). Modelling prion-induced neurodegeneration in PrP transgenic Drosophila. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/290412 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767914

Chicago Manual of Style (16th Edition):

Cardova, Alzbeta. “Modelling prion-induced neurodegeneration in PrP transgenic Drosophila.” 2019. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/290412 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767914.

MLA Handbook (7th Edition):

Cardova, Alzbeta. “Modelling prion-induced neurodegeneration in PrP transgenic Drosophila.” 2019. Web. 23 Aug 2019.

Vancouver:

Cardova A. Modelling prion-induced neurodegeneration in PrP transgenic Drosophila. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/290412 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767914.

Council of Science Editors:

Cardova A. Modelling prion-induced neurodegeneration in PrP transgenic Drosophila. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/290412 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767914


University of Cambridge

8. Iljina, Marija. Aggregation of alpha-synuclein using single-molecule spectroscopy.

Degree: PhD, 2017, University of Cambridge

 The aggregation of alpha-synuclein (αS) protein from soluble monomer into solid amyloid fibrils in the brain is associated with a range of devastating neurodegenerative disorders… (more)

Subjects/Keywords: alpha-synuclein; single-molecule FRET; protein aggregation; neurodegeneration

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Iljina, M. (2017). Aggregation of alpha-synuclein using single-molecule spectroscopy. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/263216 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707793

Chicago Manual of Style (16th Edition):

Iljina, Marija. “Aggregation of alpha-synuclein using single-molecule spectroscopy.” 2017. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/263216 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707793.

MLA Handbook (7th Edition):

Iljina, Marija. “Aggregation of alpha-synuclein using single-molecule spectroscopy.” 2017. Web. 23 Aug 2019.

Vancouver:

Iljina M. Aggregation of alpha-synuclein using single-molecule spectroscopy. [Internet] [Doctoral dissertation]. University of Cambridge; 2017. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/263216 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707793.

Council of Science Editors:

Iljina M. Aggregation of alpha-synuclein using single-molecule spectroscopy. [Doctoral Dissertation]. University of Cambridge; 2017. Available from: https://www.repository.cam.ac.uk/handle/1810/263216 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707793


University of Cambridge

9. Rittman, Timothy. Connectivity biomarkers in neurodegenerative tauopathies.

Degree: PhD, 2015, University of Cambridge

 The primary tauopathies are a group of neurodegenerative diseases affecting movement and cognition. In this thesis I study Progressive Supranuclear Palsy (PSP) and the Corticobasal… (more)

Subjects/Keywords: 616.8; Neurodegeneration; Neuroimaging; Corticobasal syndrome; Parkinson's disease; Progressive Supranuclear Palsy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rittman, T. (2015). Connectivity biomarkers in neurodegenerative tauopathies. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/248866 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655927

Chicago Manual of Style (16th Edition):

Rittman, Timothy. “Connectivity biomarkers in neurodegenerative tauopathies.” 2015. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/248866 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655927.

MLA Handbook (7th Edition):

Rittman, Timothy. “Connectivity biomarkers in neurodegenerative tauopathies.” 2015. Web. 23 Aug 2019.

Vancouver:

Rittman T. Connectivity biomarkers in neurodegenerative tauopathies. [Internet] [Doctoral dissertation]. University of Cambridge; 2015. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/248866 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655927.

Council of Science Editors:

Rittman T. Connectivity biomarkers in neurodegenerative tauopathies. [Doctoral Dissertation]. University of Cambridge; 2015. Available from: https://www.repository.cam.ac.uk/handle/1810/248866 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655927

10. Rittman, Timothy. Connectivity biomarkers in neurodegenerative tauopathies .

Degree: 2015, University of Cambridge

 The primary tauopathies are a group of neurodegenerative diseases affecting movement and cognition. In this thesis I study Progressive Supranuclear Palsy (PSP) and the Corticobasal… (more)

Subjects/Keywords: Neurodegeneration; Neuroimaging; Corticobasal syndrome; Parkinson's disease; Progressive Supranuclear Palsy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rittman, T. (2015). Connectivity biomarkers in neurodegenerative tauopathies . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/248866

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rittman, Timothy. “Connectivity biomarkers in neurodegenerative tauopathies .” 2015. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/248866.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rittman, Timothy. “Connectivity biomarkers in neurodegenerative tauopathies .” 2015. Web. 23 Aug 2019.

Vancouver:

Rittman T. Connectivity biomarkers in neurodegenerative tauopathies . [Internet] [Thesis]. University of Cambridge; 2015. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/248866.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rittman T. Connectivity biomarkers in neurodegenerative tauopathies . [Thesis]. University of Cambridge; 2015. Available from: https://www.repository.cam.ac.uk/handle/1810/248866

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

11. Fisher, Emily Mary. Development of PET radiotracers for imaging neurodegeneration : targeting alpha-synuclein fibrils and TSPO.

Degree: PhD, 2018, University of Cambridge

 Positron emission tomography (PET) is a non-invasive medical imaging technique that allows visualisation and quantification of biochemical, physiological and pharmacological processes in living subjects. This… (more)

Subjects/Keywords: Positron emission tomography; PET; alpha-synuclein; synuclein; a-synuclein; TSPO; radiolabelling; neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fisher, E. M. (2018). Development of PET radiotracers for imaging neurodegeneration : targeting alpha-synuclein fibrils and TSPO. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/278393 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753468

Chicago Manual of Style (16th Edition):

Fisher, Emily Mary. “Development of PET radiotracers for imaging neurodegeneration : targeting alpha-synuclein fibrils and TSPO.” 2018. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/278393 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753468.

MLA Handbook (7th Edition):

Fisher, Emily Mary. “Development of PET radiotracers for imaging neurodegeneration : targeting alpha-synuclein fibrils and TSPO.” 2018. Web. 23 Aug 2019.

Vancouver:

Fisher EM. Development of PET radiotracers for imaging neurodegeneration : targeting alpha-synuclein fibrils and TSPO. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/278393 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753468.

Council of Science Editors:

Fisher EM. Development of PET radiotracers for imaging neurodegeneration : targeting alpha-synuclein fibrils and TSPO. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/278393 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753468


University of Cambridge

12. Momoh, Ayiba. The Contribution of APP gene dosage to the molecular phenotypes of Alzheimer's disease in human trisomy 21 neurons .

Degree: 2019, University of Cambridge

 Down syndrome is a condition caused by trisomy 21 and occurs in 1 out of 700 births. People with trisomy 21 are high risk for… (more)

Subjects/Keywords: Alzheimer's disease; Down syndrome; Down's syndrome; Trisomy 21; Dementia; Stem cells; Neurodegeneration; Neuroscience; APP; Tau

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APA (6th Edition):

Momoh, A. (2019). The Contribution of APP gene dosage to the molecular phenotypes of Alzheimer's disease in human trisomy 21 neurons . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/294433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Momoh, Ayiba. “The Contribution of APP gene dosage to the molecular phenotypes of Alzheimer's disease in human trisomy 21 neurons .” 2019. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/294433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Momoh, Ayiba. “The Contribution of APP gene dosage to the molecular phenotypes of Alzheimer's disease in human trisomy 21 neurons .” 2019. Web. 23 Aug 2019.

Vancouver:

Momoh A. The Contribution of APP gene dosage to the molecular phenotypes of Alzheimer's disease in human trisomy 21 neurons . [Internet] [Thesis]. University of Cambridge; 2019. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/294433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Momoh A. The Contribution of APP gene dosage to the molecular phenotypes of Alzheimer's disease in human trisomy 21 neurons . [Thesis]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/294433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

13. Curry, Nathan. Development and application of correlative STED and AFM to investigate neuronal cells.

Degree: PhD, 2018, University of Cambridge

 Over the past three decades in cellular neuroscience there has been a shift towards the view of the 'tripartite synapse', where, astrocytes  – as well… (more)

Subjects/Keywords: Super-resolution; superresolution; Microscopy; STED; Stimulated emission depletion microscopy; RESOLFT; AFM; Atomic force microscopy; Correlative imaging; Fluorescence; astrocytes; connexin 30; astroglia; alpha-synuclein; neurodegeneration

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APA (6th Edition):

Curry, N. (2018). Development and application of correlative STED and AFM to investigate neuronal cells. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274579 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738329

Chicago Manual of Style (16th Edition):

Curry, Nathan. “Development and application of correlative STED and AFM to investigate neuronal cells.” 2018. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/274579 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738329.

MLA Handbook (7th Edition):

Curry, Nathan. “Development and application of correlative STED and AFM to investigate neuronal cells.” 2018. Web. 23 Aug 2019.

Vancouver:

Curry N. Development and application of correlative STED and AFM to investigate neuronal cells. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/274579 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738329.

Council of Science Editors:

Curry N. Development and application of correlative STED and AFM to investigate neuronal cells. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/274579 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738329


University of Cambridge

14. Khabirova, Eleonora. Models of neurodegeneration using computational approaches.

Degree: PhD, 2016, University of Cambridge

 Alzheimer's disease (AD), as one of the most common neurodegenerative diseases, is characterized by the loss of neuronal dysfunction and death resulting in progressive cognitive… (more)

Subjects/Keywords: Alzheimer's disease; circadian rhythms; computational modeling; gene network analysis; neurodegeneration; abeta-toxicity; spatial orientation; behavioural rhythms; circadian disruption; monte carlo simulations; time series analysis; period estimation methods

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khabirova, E. (2016). Models of neurodegeneration using computational approaches. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274157 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744244

Chicago Manual of Style (16th Edition):

Khabirova, Eleonora. “Models of neurodegeneration using computational approaches.” 2016. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/274157 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744244.

MLA Handbook (7th Edition):

Khabirova, Eleonora. “Models of neurodegeneration using computational approaches.” 2016. Web. 23 Aug 2019.

Vancouver:

Khabirova E. Models of neurodegeneration using computational approaches. [Internet] [Doctoral dissertation]. University of Cambridge; 2016. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/274157 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744244.

Council of Science Editors:

Khabirova E. Models of neurodegeneration using computational approaches. [Doctoral Dissertation]. University of Cambridge; 2016. Available from: https://www.repository.cam.ac.uk/handle/1810/274157 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744244


University of Cambridge

15. Vecchi, Giulia. Proteomics studies of protein homeostasis and aggregation in ageing and neurodegeneration .

Degree: 2018, University of Cambridge

 Upon ageing, a progressive disruption of protein homeostasis often leads to extensive protein aggregation and neurodegeneration. It is therefore important to study at the proteome… (more)

Subjects/Keywords: protein aggregation; ageing; oligomers; proteostasis; neurodegeneration; data analysis; sequence-based predictions; c. elegans; MS-based proteomics; protein interactions; functional annotation analysis; prediction of physico-chemical principles; misfolding diseases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vecchi, G. (2018). Proteomics studies of protein homeostasis and aggregation in ageing and neurodegeneration . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/273348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vecchi, Giulia. “Proteomics studies of protein homeostasis and aggregation in ageing and neurodegeneration .” 2018. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/273348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vecchi, Giulia. “Proteomics studies of protein homeostasis and aggregation in ageing and neurodegeneration .” 2018. Web. 23 Aug 2019.

Vancouver:

Vecchi G. Proteomics studies of protein homeostasis and aggregation in ageing and neurodegeneration . [Internet] [Thesis]. University of Cambridge; 2018. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/273348.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vecchi G. Proteomics studies of protein homeostasis and aggregation in ageing and neurodegeneration . [Thesis]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/273348

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

16. Khabirova, Eleonora. Models of neurodegeneration using computational approaches .

Degree: 2016, University of Cambridge

 Alzheimer's disease (AD), as one of the most common neurodegenerative diseases, is characterized by the loss of neuronal dysfunction and death resulting in progressive cognitive… (more)

Subjects/Keywords: Alzheimer's disease; circadian rhythms; computational modeling; gene network analysis; neurodegeneration; abeta-toxicity; spatial orientation; behavioural rhythms; circadian disruption; monte carlo simulations; time series analysis; period estimation methods

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Khabirova, E. (2016). Models of neurodegeneration using computational approaches . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274157

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Khabirova, Eleonora. “Models of neurodegeneration using computational approaches .” 2016. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/274157.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Khabirova, Eleonora. “Models of neurodegeneration using computational approaches .” 2016. Web. 23 Aug 2019.

Vancouver:

Khabirova E. Models of neurodegeneration using computational approaches . [Internet] [Thesis]. University of Cambridge; 2016. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/274157.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Khabirova E. Models of neurodegeneration using computational approaches . [Thesis]. University of Cambridge; 2016. Available from: https://www.repository.cam.ac.uk/handle/1810/274157

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

17. Curry, Nathan. Development and application of correlative STED and AFM to investigate neuronal cells .

Degree: 2018, University of Cambridge

 Over the past three decades in cellular neuroscience there has been a shift towards the view of the ``tripartite synapse'', where, astrocytes  – as well… (more)

Subjects/Keywords: Super-resolution; superresolution; Microscopy; STED; Stimulated emission depletion microscopy; RESOLFT; AFM; Atomic force microscopy; Correlative imaging; Fluorescence; astrocytes; connexin 30; astroglia; alpha-synuclein; neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Curry, N. (2018). Development and application of correlative STED and AFM to investigate neuronal cells . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/274579

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Curry, Nathan. “Development and application of correlative STED and AFM to investigate neuronal cells .” 2018. Thesis, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/274579.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Curry, Nathan. “Development and application of correlative STED and AFM to investigate neuronal cells .” 2018. Web. 23 Aug 2019.

Vancouver:

Curry N. Development and application of correlative STED and AFM to investigate neuronal cells . [Internet] [Thesis]. University of Cambridge; 2018. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/274579.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Curry N. Development and application of correlative STED and AFM to investigate neuronal cells . [Thesis]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/274579

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

18. Martynyuk, Nataly. Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration.

Degree: PhD, 2019, University of Cambridge

 Rho GTPases and their regulators such as guanosine exchange factors (GEFs) and GTPase activating proteins (GAPs) represent an important class of molecules controlling dendritic spine… (more)

Subjects/Keywords: Chimaerin; Alpha-chimaerin; Dendritic spines; Neurodegeneration; Neuroplasticity; RhoGTPases; Rac; Rho; GTPase activating proteins; GAP; RacGAP; Cdc42; Cytoskeleton; Endocytosis; HEK293; HeLa; Chimerin; CHIN; ROCK; EphA4; C1 domain

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Martynyuk, N. (2019). Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874

Chicago Manual of Style (16th Edition):

Martynyuk, Nataly. “Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration.” 2019. Doctoral Dissertation, University of Cambridge. Accessed August 23, 2019. https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874.

MLA Handbook (7th Edition):

Martynyuk, Nataly. “Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration.” 2019. Web. 23 Aug 2019.

Vancouver:

Martynyuk N. Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2019 Aug 23]. Available from: https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874.

Council of Science Editors:

Martynyuk N. Actions of alpha-chimaerins in mechanisms relevant to dendritic spine formation and neurodegeneration. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/287938 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.763874

.