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You searched for subject:(neurodegeneration). Showing records 1 – 30 of 550 total matches.

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University of Rochester

1. Harder, Jeffrey M. The Role of Bcl-2 Family Members in Retinal Ganglion Cell Death.

Degree: PhD, 2013, University of Rochester

 Glaucomas are a group of diseases that often lead to blindness. Glaucomas are unified by the characteristic cell death of the output neuron of the… (more)

Subjects/Keywords: Neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harder, J. M. (2013). The Role of Bcl-2 Family Members in Retinal Ganglion Cell Death. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26812

Chicago Manual of Style (16th Edition):

Harder, Jeffrey M. “The Role of Bcl-2 Family Members in Retinal Ganglion Cell Death.” 2013. Doctoral Dissertation, University of Rochester. Accessed July 22, 2019. http://hdl.handle.net/1802/26812.

MLA Handbook (7th Edition):

Harder, Jeffrey M. “The Role of Bcl-2 Family Members in Retinal Ganglion Cell Death.” 2013. Web. 22 Jul 2019.

Vancouver:

Harder JM. The Role of Bcl-2 Family Members in Retinal Ganglion Cell Death. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/1802/26812.

Council of Science Editors:

Harder JM. The Role of Bcl-2 Family Members in Retinal Ganglion Cell Death. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26812


Cornell University

2. Brady, Owen. Investigating Mechanisms Of Ftld-Tdp Pathogenesis .

Degree: 2014, Cornell University

 Frontotemporal lobar degeneration (FTLD) is a devastating dementia disorder that causes profound changes in personality, behavior, and language abilities. Major breakthroughs in the past decade… (more)

Subjects/Keywords: Neurodegeneration; Lysosomes; TMEM106B

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APA (6th Edition):

Brady, O. (2014). Investigating Mechanisms Of Ftld-Tdp Pathogenesis . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/36146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brady, Owen. “Investigating Mechanisms Of Ftld-Tdp Pathogenesis .” 2014. Thesis, Cornell University. Accessed July 22, 2019. http://hdl.handle.net/1813/36146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brady, Owen. “Investigating Mechanisms Of Ftld-Tdp Pathogenesis .” 2014. Web. 22 Jul 2019.

Vancouver:

Brady O. Investigating Mechanisms Of Ftld-Tdp Pathogenesis . [Internet] [Thesis]. Cornell University; 2014. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/1813/36146.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brady O. Investigating Mechanisms Of Ftld-Tdp Pathogenesis . [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36146

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

3. Tseng, LeinWeih Andrew. Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity.

Degree: PhD, Molecular and Cellular Pharmacology (Medicine), 2010, University of Miami

 Neuronal pentraxin with chromo domain (NPCD) belongs to a family of neuronally-expressed pentraxin proteins thought to be involved in synaptic refinement and plasticity. One isoform… (more)

Subjects/Keywords: Protein Aggregation; Neurodegeneration

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APA (6th Edition):

Tseng, L. A. (2010). Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/662

Chicago Manual of Style (16th Edition):

Tseng, LeinWeih Andrew. “Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity.” 2010. Doctoral Dissertation, University of Miami. Accessed July 22, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/662.

MLA Handbook (7th Edition):

Tseng, LeinWeih Andrew. “Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity.” 2010. Web. 22 Jul 2019.

Vancouver:

Tseng LA. Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2019 Jul 22]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/662.

Council of Science Editors:

Tseng LA. Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/662


University of Miami

4. Tseng, LeinWeih Andrew. Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity.

Degree: PhD, Molecular and Cellular Pharmacology (Medicine), 2010, University of Miami

 Neuronal pentraxin with chromo domain (NPCD) belongs to a family of neuronally-expressed pentraxin proteins thought to be involved in synaptic refinement and plasticity. One isoform… (more)

Subjects/Keywords: Protein Aggregation; Neurodegeneration

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APA (6th Edition):

Tseng, L. A. (2010). Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/928

Chicago Manual of Style (16th Edition):

Tseng, LeinWeih Andrew. “Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity.” 2010. Doctoral Dissertation, University of Miami. Accessed July 22, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/928.

MLA Handbook (7th Edition):

Tseng, LeinWeih Andrew. “Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity.” 2010. Web. 22 Jul 2019.

Vancouver:

Tseng LA. Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2019 Jul 22]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/928.

Council of Science Editors:

Tseng LA. Ubiquitylation of Neuronal Pentraxin with Chromo Domain by the E3 Ubiquitin Ligase Mayven/KLHL2 and Effects on Aggresome Formation and Neuronal Cytotoxicity. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/928


University of Arizona

5. Coyne, Alyssa N. Dysregulation of mRNA Transport and Translation in ALS .

Degree: 2016, University of Arizona

 Amyotrophic Lateral Sclerosis (ALS) is a progressive neurodegenerative disease affecting upper and lower motor neurons. Although many cellular processes such as cytoskeletal maintenance and synaptic… (more)

Subjects/Keywords: Drosophila; neurodegeneration; ALS

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APA (6th Edition):

Coyne, A. N. (2016). Dysregulation of mRNA Transport and Translation in ALS . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/623181

Chicago Manual of Style (16th Edition):

Coyne, Alyssa N. “Dysregulation of mRNA Transport and Translation in ALS .” 2016. Doctoral Dissertation, University of Arizona. Accessed July 22, 2019. http://hdl.handle.net/10150/623181.

MLA Handbook (7th Edition):

Coyne, Alyssa N. “Dysregulation of mRNA Transport and Translation in ALS .” 2016. Web. 22 Jul 2019.

Vancouver:

Coyne AN. Dysregulation of mRNA Transport and Translation in ALS . [Internet] [Doctoral dissertation]. University of Arizona; 2016. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/10150/623181.

Council of Science Editors:

Coyne AN. Dysregulation of mRNA Transport and Translation in ALS . [Doctoral Dissertation]. University of Arizona; 2016. Available from: http://hdl.handle.net/10150/623181


University of Edinburgh

6. Vincenti, James Edward. Role of activation of microglia in neurodegenerative prion disease.

Degree: PhD, 2015, University of Edinburgh

 Prion diseases are a group of fatal neurodegenerative protein-misfolding diseases. Microglia, the resident myeloid cells found within the brain, have been shown to demonstrate a… (more)

Subjects/Keywords: 612.8; microglia; neurodegeneration; neuroimmunology; prion

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APA (6th Edition):

Vincenti, J. E. (2015). Role of activation of microglia in neurodegenerative prion disease. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/15928

Chicago Manual of Style (16th Edition):

Vincenti, James Edward. “Role of activation of microglia in neurodegenerative prion disease.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed July 22, 2019. http://hdl.handle.net/1842/15928.

MLA Handbook (7th Edition):

Vincenti, James Edward. “Role of activation of microglia in neurodegenerative prion disease.” 2015. Web. 22 Jul 2019.

Vancouver:

Vincenti JE. Role of activation of microglia in neurodegenerative prion disease. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/1842/15928.

Council of Science Editors:

Vincenti JE. Role of activation of microglia in neurodegenerative prion disease. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/15928


Kent State University

7. Kumar, Varun. Protein Kinase C Signaling in Neurodegeneration.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2016, Kent State University

 Global cerebral ischemia occurs due to cardiac arrest, which results in delayed neurodegeneration. Pharmacological agents such as Protein Kinase C epsilon (PKC epsilon) peptide activators… (more)

Subjects/Keywords: Neurobiology; PKC, Neurodegeneration, ATF2

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APA (6th Edition):

Kumar, V. (2016). Protein Kinase C Signaling in Neurodegeneration. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1455721051

Chicago Manual of Style (16th Edition):

Kumar, Varun. “Protein Kinase C Signaling in Neurodegeneration.” 2016. Doctoral Dissertation, Kent State University. Accessed July 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1455721051.

MLA Handbook (7th Edition):

Kumar, Varun. “Protein Kinase C Signaling in Neurodegeneration.” 2016. Web. 22 Jul 2019.

Vancouver:

Kumar V. Protein Kinase C Signaling in Neurodegeneration. [Internet] [Doctoral dissertation]. Kent State University; 2016. [cited 2019 Jul 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1455721051.

Council of Science Editors:

Kumar V. Protein Kinase C Signaling in Neurodegeneration. [Doctoral Dissertation]. Kent State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1455721051


University of Sydney

8. Cross, Nathan. Obstructive sleep apnoea in older adults: Predictors of cognitive decline and neurodegeneration .

Degree: 2017, University of Sydney

 There is growing evidence that sleep disturbances share associations with neurodegeneration, and poor sleep is emerging as an independent risk factor for dementia. As a… (more)

Subjects/Keywords: sleep; apnoea; ageing; neurodegeneration

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APA (6th Edition):

Cross, N. (2017). Obstructive sleep apnoea in older adults: Predictors of cognitive decline and neurodegeneration . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17763

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cross, Nathan. “Obstructive sleep apnoea in older adults: Predictors of cognitive decline and neurodegeneration .” 2017. Thesis, University of Sydney. Accessed July 22, 2019. http://hdl.handle.net/2123/17763.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cross, Nathan. “Obstructive sleep apnoea in older adults: Predictors of cognitive decline and neurodegeneration .” 2017. Web. 22 Jul 2019.

Vancouver:

Cross N. Obstructive sleep apnoea in older adults: Predictors of cognitive decline and neurodegeneration . [Internet] [Thesis]. University of Sydney; 2017. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/2123/17763.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cross N. Obstructive sleep apnoea in older adults: Predictors of cognitive decline and neurodegeneration . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17763

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

9. Seo, Woo Sung. Role of p53 and PARP-1 in neurodegenerative diseases .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 The thesis presents a general review of major neurodegenerative diseases and also discusses the role of p53 and PARP-1 in neurodegeneration. p53 and PARP-1 are… (more)

Subjects/Keywords: Neurodegeneration; neuroscience

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APA (6th Edition):

Seo, W. S. (n.d.). Role of p53 and PARP-1 in neurodegenerative diseases . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/243137

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Seo, Woo Sung. “Role of p53 and PARP-1 in neurodegenerative diseases .” Thesis, University of Debrecen. Accessed July 22, 2019. http://hdl.handle.net/2437/243137.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Seo, Woo Sung. “Role of p53 and PARP-1 in neurodegenerative diseases .” Web. 22 Jul 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Seo WS. Role of p53 and PARP-1 in neurodegenerative diseases . [Internet] [Thesis]. University of Debrecen; [cited 2019 Jul 22]. Available from: http://hdl.handle.net/2437/243137.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Seo WS. Role of p53 and PARP-1 in neurodegenerative diseases . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/243137

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Manchester

10. Howard, Daniel. The role of the cytoskeleton in Alzheimer’s disease: a Drosophila perspective.

Degree: 2016, University of Manchester

 Alzheimer’s disease (AD) pathogenesis is likely to be caused by dysfunction of two neuronal proteins, amyloid-beta (Aβ) and tau. Whilst excellent in vivo assays have… (more)

Subjects/Keywords: drosophila; neurodegeneration; amyloid; tau

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APA (6th Edition):

Howard, D. (2016). The role of the cytoskeleton in Alzheimer’s disease: a Drosophila perspective. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300556

Chicago Manual of Style (16th Edition):

Howard, Daniel. “The role of the cytoskeleton in Alzheimer’s disease: a Drosophila perspective.” 2016. Doctoral Dissertation, University of Manchester. Accessed July 22, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300556.

MLA Handbook (7th Edition):

Howard, Daniel. “The role of the cytoskeleton in Alzheimer’s disease: a Drosophila perspective.” 2016. Web. 22 Jul 2019.

Vancouver:

Howard D. The role of the cytoskeleton in Alzheimer’s disease: a Drosophila perspective. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Jul 22]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300556.

Council of Science Editors:

Howard D. The role of the cytoskeleton in Alzheimer’s disease: a Drosophila perspective. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:300556


University of Cambridge

11. Sohail, Anood. Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila .

Degree: 2019, University of Cambridge

 Axons possess a continuous network of smooth tubular endoplasmic reticulum (ER), extending from the nuclear envelope throughout the neuron to synapses. Mutations affecting proteins with… (more)

Subjects/Keywords: Axonal ER; neurodegeneration; drosophila

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APA (6th Edition):

Sohail, A. (2019). Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/290113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sohail, Anood. “Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila .” 2019. Thesis, University of Cambridge. Accessed July 22, 2019. https://www.repository.cam.ac.uk/handle/1810/290113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sohail, Anood. “Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila .” 2019. Web. 22 Jul 2019.

Vancouver:

Sohail A. Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila . [Internet] [Thesis]. University of Cambridge; 2019. [cited 2019 Jul 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/290113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sohail A. Visualizing Roles of Spastic Paraplegia Proteins in Organizing Axonal ER in Live Drosophila . [Thesis]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/290113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

12. Levy, Daniel Robert Siegfried. Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system .

Degree: 2018, University of Cambridge

 Leucine rich repeat kinase 2 (LRRK2) is a 286 kDa protein expressed in a variety of tissues and cell types, including neuronal tissue and innate… (more)

Subjects/Keywords: immunology; Parkinson's disease; neurodegeneration

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APA (6th Edition):

Levy, D. R. S. (2018). Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system . (Thesis). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/273736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Levy, Daniel Robert Siegfried. “Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system .” 2018. Thesis, University of Cambridge. Accessed July 22, 2019. https://www.repository.cam.ac.uk/handle/1810/273736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Levy, Daniel Robert Siegfried. “Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system .” 2018. Web. 22 Jul 2019.

Vancouver:

Levy DRS. Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system . [Internet] [Thesis]. University of Cambridge; 2018. [cited 2019 Jul 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/273736.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Levy DRS. Exploring the role of Leucine Rich Repeat Kinase 2 within the innate immune system . [Thesis]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/273736

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

13. Ward, Nicholas John. The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease.

Degree: PhD, Neuroscience, 2014, Vanderbilt University

 Neuronal responses to stress are an important component of neurodegenerative disease and may represent targets for therapeutic intervention. In normal and pathogenic physiological conditions, members… (more)

Subjects/Keywords: neurodegeneration; glaucoma; TRPV1; TRP channels

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APA (6th Edition):

Ward, N. J. (2014). The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;

Chicago Manual of Style (16th Edition):

Ward, Nicholas John. “The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed July 22, 2019. http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;.

MLA Handbook (7th Edition):

Ward, Nicholas John. “The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease.” 2014. Web. 22 Jul 2019.

Vancouver:

Ward NJ. The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2019 Jul 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;.

Council of Science Editors:

Ward NJ. The Transient Receptor Potential Vanilloid-1 Channel and Neuronal Survival in Degenerative Disease. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-07212014-152449/ ;


University of New South Wales

14. Chiu, Alexander Simon Che-Kean. A toxicological analysis of the cyanotoxin β-methylamino-L-alanine at multiple neural levels and identification of novel modes of activity.

Degree: Biotechnology & Biomolecular Sciences, 2013, University of New South Wales

 The cyanotoxin β-methylamino-L-alanine (BMAA) has long been associated with the elevated incidence of amyotrophic lateral sclerosis/Parkinsonism dementia complex (ALS/PDC). BMAA has been shown to be… (more)

Subjects/Keywords: ALS/PDC; BMAA; Neurodegeneration; Cyanotoxin

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APA (6th Edition):

Chiu, A. S. C. (2013). A toxicological analysis of the cyanotoxin β-methylamino-L-alanine at multiple neural levels and identification of novel modes of activity. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52422 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11095/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Chiu, Alexander Simon Che-Kean. “A toxicological analysis of the cyanotoxin β-methylamino-L-alanine at multiple neural levels and identification of novel modes of activity.” 2013. Doctoral Dissertation, University of New South Wales. Accessed July 22, 2019. http://handle.unsw.edu.au/1959.4/52422 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11095/SOURCE01?view=true.

MLA Handbook (7th Edition):

Chiu, Alexander Simon Che-Kean. “A toxicological analysis of the cyanotoxin β-methylamino-L-alanine at multiple neural levels and identification of novel modes of activity.” 2013. Web. 22 Jul 2019.

Vancouver:

Chiu ASC. A toxicological analysis of the cyanotoxin β-methylamino-L-alanine at multiple neural levels and identification of novel modes of activity. [Internet] [Doctoral dissertation]. University of New South Wales; 2013. [cited 2019 Jul 22]. Available from: http://handle.unsw.edu.au/1959.4/52422 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11095/SOURCE01?view=true.

Council of Science Editors:

Chiu ASC. A toxicological analysis of the cyanotoxin β-methylamino-L-alanine at multiple neural levels and identification of novel modes of activity. [Doctoral Dissertation]. University of New South Wales; 2013. Available from: http://handle.unsw.edu.au/1959.4/52422 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:11095/SOURCE01?view=true


University of Minnesota

15. Amar, Fatou. Cellular And Molecular Mechanism Of Action Of The Amyloid-Beta Oligomer Abeta Star 56.

Degree: PhD, Neuroscience, 2016, University of Minnesota

 Alzheimer’s disease (AD) is a progressive neurodegenerative disorder, with asymptomatic and symptomatic phases. Hallmark lesions of AD include extracellular deposits of fibrillar amyloid-β (A β)… (more)

Subjects/Keywords: Alzheimer's disease; molecular neuroscience; neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Amar, F. (2016). Cellular And Molecular Mechanism Of Action Of The Amyloid-Beta Oligomer Abeta Star 56. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/178973

Chicago Manual of Style (16th Edition):

Amar, Fatou. “Cellular And Molecular Mechanism Of Action Of The Amyloid-Beta Oligomer Abeta Star 56.” 2016. Doctoral Dissertation, University of Minnesota. Accessed July 22, 2019. http://hdl.handle.net/11299/178973.

MLA Handbook (7th Edition):

Amar, Fatou. “Cellular And Molecular Mechanism Of Action Of The Amyloid-Beta Oligomer Abeta Star 56.” 2016. Web. 22 Jul 2019.

Vancouver:

Amar F. Cellular And Molecular Mechanism Of Action Of The Amyloid-Beta Oligomer Abeta Star 56. [Internet] [Doctoral dissertation]. University of Minnesota; 2016. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11299/178973.

Council of Science Editors:

Amar F. Cellular And Molecular Mechanism Of Action Of The Amyloid-Beta Oligomer Abeta Star 56. [Doctoral Dissertation]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/178973


University of Pennsylvania

16. Covy, Jason P. The Pathological and Biochemical Characterization of Leucine-Rich Repeat Kinase 2 in Parkinson’s Disease.

Degree: 2010, University of Pennsylvania

 Parkinson's disease (PD) is a debilitating and progressive neurodegenerative disorder that affects over 6 million people worldwide. Despite being the most common movement disorder in… (more)

Subjects/Keywords: LRRK2; Parkinson's disease; neurodegeneration; Neurosciences

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APA (6th Edition):

Covy, J. P. (2010). The Pathological and Biochemical Characterization of Leucine-Rich Repeat Kinase 2 in Parkinson’s Disease. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1553

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Covy, Jason P. “The Pathological and Biochemical Characterization of Leucine-Rich Repeat Kinase 2 in Parkinson’s Disease.” 2010. Thesis, University of Pennsylvania. Accessed July 22, 2019. https://repository.upenn.edu/edissertations/1553.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Covy, Jason P. “The Pathological and Biochemical Characterization of Leucine-Rich Repeat Kinase 2 in Parkinson’s Disease.” 2010. Web. 22 Jul 2019.

Vancouver:

Covy JP. The Pathological and Biochemical Characterization of Leucine-Rich Repeat Kinase 2 in Parkinson’s Disease. [Internet] [Thesis]. University of Pennsylvania; 2010. [cited 2019 Jul 22]. Available from: https://repository.upenn.edu/edissertations/1553.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Covy JP. The Pathological and Biochemical Characterization of Leucine-Rich Repeat Kinase 2 in Parkinson’s Disease. [Thesis]. University of Pennsylvania; 2010. Available from: https://repository.upenn.edu/edissertations/1553

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

17. Braidy, Nady. NAD+ metabolism in neurodegeneration and aging.

Degree: Medical Sciences, 2011, University of New South Wales

 Elevated levels of several kynurenine pathway (KP) metabolites, e.g. quinolinic acid (QUIN), have been implicated in the aetiology of several neurodegenerative diseases such as Alzheimer‟s… (more)

Subjects/Keywords: NAD+; metabolism; neurodegeneration; aging

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APA (6th Edition):

Braidy, N. (2011). NAD+ metabolism in neurodegeneration and aging. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51345 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10026/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Braidy, Nady. “NAD+ metabolism in neurodegeneration and aging.” 2011. Doctoral Dissertation, University of New South Wales. Accessed July 22, 2019. http://handle.unsw.edu.au/1959.4/51345 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10026/SOURCE02?view=true.

MLA Handbook (7th Edition):

Braidy, Nady. “NAD+ metabolism in neurodegeneration and aging.” 2011. Web. 22 Jul 2019.

Vancouver:

Braidy N. NAD+ metabolism in neurodegeneration and aging. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2019 Jul 22]. Available from: http://handle.unsw.edu.au/1959.4/51345 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10026/SOURCE02?view=true.

Council of Science Editors:

Braidy N. NAD+ metabolism in neurodegeneration and aging. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51345 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10026/SOURCE02?view=true


University of Manitoba

18. Acosta, Crystal May R. Antigenic induction of nerve growth factor (NGF) in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS).

Degree: Pharmacy, 2013, University of Manitoba

 Nerve growth factor (NGF) represents a new therapeutic strategy for multiple sclerosis (MS) because of its immunomodulatory and neuroprotective activity. To analyze changes in NGF… (more)

Subjects/Keywords: MS; NGF; Neurodegeneration; Neuroimmunology

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APA (6th Edition):

Acosta, C. M. R. (2013). Antigenic induction of nerve growth factor (NGF) in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/23504

Chicago Manual of Style (16th Edition):

Acosta, Crystal May R. “Antigenic induction of nerve growth factor (NGF) in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS).” 2013. Masters Thesis, University of Manitoba. Accessed July 22, 2019. http://hdl.handle.net/1993/23504.

MLA Handbook (7th Edition):

Acosta, Crystal May R. “Antigenic induction of nerve growth factor (NGF) in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS).” 2013. Web. 22 Jul 2019.

Vancouver:

Acosta CMR. Antigenic induction of nerve growth factor (NGF) in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). [Internet] [Masters thesis]. University of Manitoba; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/1993/23504.

Council of Science Editors:

Acosta CMR. Antigenic induction of nerve growth factor (NGF) in experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). [Masters Thesis]. University of Manitoba; 2013. Available from: http://hdl.handle.net/1993/23504


University of Missouri – Columbia

19. Pinnegar, Abbie Jolene. Investigating canine degenerative myelopathy as a disease model of amyotrophic lateral sclerosis for histopathological evidence of dysphagia.

Degree: 2012, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Unspecified swallowing impairment (dysphagia) was recently reported in dogs with degenerative myelopathy (DM), a proposed… (more)

Subjects/Keywords: dysphagia; brainstem nuclei; neurodegeneration

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APA (6th Edition):

Pinnegar, A. J. (2012). Investigating canine degenerative myelopathy as a disease model of amyotrophic lateral sclerosis for histopathological evidence of dysphagia. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/35437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pinnegar, Abbie Jolene. “Investigating canine degenerative myelopathy as a disease model of amyotrophic lateral sclerosis for histopathological evidence of dysphagia.” 2012. Thesis, University of Missouri – Columbia. Accessed July 22, 2019. http://hdl.handle.net/10355/35437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pinnegar, Abbie Jolene. “Investigating canine degenerative myelopathy as a disease model of amyotrophic lateral sclerosis for histopathological evidence of dysphagia.” 2012. Web. 22 Jul 2019.

Vancouver:

Pinnegar AJ. Investigating canine degenerative myelopathy as a disease model of amyotrophic lateral sclerosis for histopathological evidence of dysphagia. [Internet] [Thesis]. University of Missouri – Columbia; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/10355/35437.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pinnegar AJ. Investigating canine degenerative myelopathy as a disease model of amyotrophic lateral sclerosis for histopathological evidence of dysphagia. [Thesis]. University of Missouri – Columbia; 2012. Available from: http://hdl.handle.net/10355/35437

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

20. Bleiberg, Benjamin Aaron. Evaluating the concordance of N-terminal and full length Huntington's disease modifiers and identifying potential therapeutic targets in Drosophila.

Degree: MS, Medical Sciences, 2016, Boston University

 Huntington’s disease (HD) is one of nine polyglutamine diseases and it is caused by a CAG expansion in the HTT gene. HD is an autosomal,… (more)

Subjects/Keywords: Medicine; Drosophila; Neurodegeneration; Huntington's disease

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APA (6th Edition):

Bleiberg, B. A. (2016). Evaluating the concordance of N-terminal and full length Huntington's disease modifiers and identifying potential therapeutic targets in Drosophila. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16770

Chicago Manual of Style (16th Edition):

Bleiberg, Benjamin Aaron. “Evaluating the concordance of N-terminal and full length Huntington's disease modifiers and identifying potential therapeutic targets in Drosophila.” 2016. Masters Thesis, Boston University. Accessed July 22, 2019. http://hdl.handle.net/2144/16770.

MLA Handbook (7th Edition):

Bleiberg, Benjamin Aaron. “Evaluating the concordance of N-terminal and full length Huntington's disease modifiers and identifying potential therapeutic targets in Drosophila.” 2016. Web. 22 Jul 2019.

Vancouver:

Bleiberg BA. Evaluating the concordance of N-terminal and full length Huntington's disease modifiers and identifying potential therapeutic targets in Drosophila. [Internet] [Masters thesis]. Boston University; 2016. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/2144/16770.

Council of Science Editors:

Bleiberg BA. Evaluating the concordance of N-terminal and full length Huntington's disease modifiers and identifying potential therapeutic targets in Drosophila. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16770

21. Catalano, Carmen. Correlazioni molecolari fra angiogenesi e neurodegenerazione.

Degree: 2013, Università degli Studi di Catania

Studio e valutazione dei potenziali effetti del farmaco immunomodulante lenalidomide nelle patologie neurodegenerative.

Subjects/Keywords: Area 05 - Scienze biologiche; lenalidomide, angiogenesis, neurodegeneration

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APA (6th Edition):

Catalano, C. (2013). Correlazioni molecolari fra angiogenesi e neurodegenerazione. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/1321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Catalano, Carmen. “Correlazioni molecolari fra angiogenesi e neurodegenerazione.” 2013. Thesis, Università degli Studi di Catania. Accessed July 22, 2019. http://hdl.handle.net/10761/1321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Catalano, Carmen. “Correlazioni molecolari fra angiogenesi e neurodegenerazione.” 2013. Web. 22 Jul 2019.

Vancouver:

Catalano C. Correlazioni molecolari fra angiogenesi e neurodegenerazione. [Internet] [Thesis]. Università degli Studi di Catania; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/10761/1321.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Catalano C. Correlazioni molecolari fra angiogenesi e neurodegenerazione. [Thesis]. Università degli Studi di Catania; 2013. Available from: http://hdl.handle.net/10761/1321

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

22. Huang, Pei-San. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.

Degree: 2012, Texas A&M University

 Nicotine, the major psychoactive ingredient of tobacco smoke, underlies numerous effects by activating neuronal nicotinic acetylcholine receptors. Both in vitro and in vivo studies suggest… (more)

Subjects/Keywords: nicotine; neuroprotection; aging; nAChRs; neurodegeneration; caloric restriction

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APA (6th Edition):

Huang, P. (2012). Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Pei-San. “Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.” 2012. Thesis, Texas A&M University. Accessed July 22, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Pei-San. “Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.” 2012. Web. 22 Jul 2019.

Vancouver:

Huang P. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang P. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wright State University

23. Davidson, Molly Elizabeth. Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure.

Degree: MS, Pharmacology and Toxicology, 2007, Wright State University

 Sarin is an organophosphorus (OP) ester chemical warfare agent (CWA) that has been used in past terrorist attacks. It remains a threat today because of… (more)

Subjects/Keywords: Health Sciences, Toxicology; sarin; organophosphate; neuroinflammation; neurodegeneration

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APA (6th Edition):

Davidson, M. E. (2007). Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827

Chicago Manual of Style (16th Edition):

Davidson, Molly Elizabeth. “Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure.” 2007. Masters Thesis, Wright State University. Accessed July 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827.

MLA Handbook (7th Edition):

Davidson, Molly Elizabeth. “Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure.” 2007. Web. 22 Jul 2019.

Vancouver:

Davidson ME. Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure. [Internet] [Masters thesis]. Wright State University; 2007. [cited 2019 Jul 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827.

Council of Science Editors:

Davidson ME. Neurodegeneration and Neuroinflammation in a Mouse Model of Sarin Exposure. [Masters Thesis]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1189715827


University of Alberta

24. Mercer, Robert Corrigan Curtis. The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation.

Degree: PhD, Department of Medicine, 2016, University of Alberta

 Prion diseases are invariably fatal neurodegenerative diseases of humans and other mammals. While they can manifest as sporadic, infectious or genetic etiologies, the central event… (more)

Subjects/Keywords: Gerstmann-Sträussler-Schienker; Potassium Channel; Prion; Neurodegeneration

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APA (6th Edition):

Mercer, R. C. C. (2016). The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cmp48sc82v

Chicago Manual of Style (16th Edition):

Mercer, Robert Corrigan Curtis. “The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation.” 2016. Doctoral Dissertation, University of Alberta. Accessed July 22, 2019. https://era.library.ualberta.ca/files/cmp48sc82v.

MLA Handbook (7th Edition):

Mercer, Robert Corrigan Curtis. “The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation.” 2016. Web. 22 Jul 2019.

Vancouver:

Mercer RCC. The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation. [Internet] [Doctoral dissertation]. University of Alberta; 2016. [cited 2019 Jul 22]. Available from: https://era.library.ualberta.ca/files/cmp48sc82v.

Council of Science Editors:

Mercer RCC. The Prion Protein: Modulation of Potassium Channels and a Novel Mouse Model of a Disease-Causing Hydrophobic Domain Insertion Mutation. [Doctoral Dissertation]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cmp48sc82v

25. O HARA, DARREN. A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2017, Trinity College Dublin

 Summary Mitochondrial dysfunction is recognised as a hallmark of many neurodegenerative diseases. Altered electron transport chain (ETC) complex activities, mitochondrial fusion/fission dynamics, mitochondrial motility kinetics… (more)

Subjects/Keywords: Mitochondria; Mitochondrial Dynamics; Neurodegeneration; Glycosylation; O-GlcNAc

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APA (6th Edition):

O HARA, D. (2017). A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/81959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O HARA, DARREN. “A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons.” 2017. Thesis, Trinity College Dublin. Accessed July 22, 2019. http://hdl.handle.net/2262/81959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O HARA, DARREN. “A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons.” 2017. Web. 22 Jul 2019.

Vancouver:

O HARA D. A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons. [Internet] [Thesis]. Trinity College Dublin; 2017. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/2262/81959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O HARA D. A Study of Mitochondrial Dynamics and Glycosylation Events in PC12 Cells and Neurons. [Thesis]. Trinity College Dublin; 2017. Available from: http://hdl.handle.net/2262/81959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Sundal, Christina. Hereditary diffuse leukoencephalopathy with spheroids: Insights into an adult onset neurodegenerative disease.

Degree: 2013, University of Gothenburg / Göteborgs Universitet

 During the last three decades, the areas of inherited white matter (WM) disorders have expanded. Advances in magnetic resonance imaging (MRI) and genetics have led… (more)

Subjects/Keywords: HDLS; Neurodegeneration

…novel insights into neurodegeneration. 9 "Syftet med studien var att öka kunskapen om… …reasons it is now called pantothenate kinase-associated neurodegeneration (PKAN)… …the term “syndromes of neurodegeneration with brain iron accumulation,” (NBIA)… 

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APA (6th Edition):

Sundal, C. (2013). Hereditary diffuse leukoencephalopathy with spheroids: Insights into an adult onset neurodegenerative disease. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/31995

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sundal, Christina. “Hereditary diffuse leukoencephalopathy with spheroids: Insights into an adult onset neurodegenerative disease.” 2013. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed July 22, 2019. http://hdl.handle.net/2077/31995.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sundal, Christina. “Hereditary diffuse leukoencephalopathy with spheroids: Insights into an adult onset neurodegenerative disease.” 2013. Web. 22 Jul 2019.

Vancouver:

Sundal C. Hereditary diffuse leukoencephalopathy with spheroids: Insights into an adult onset neurodegenerative disease. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/2077/31995.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sundal C. Hereditary diffuse leukoencephalopathy with spheroids: Insights into an adult onset neurodegenerative disease. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2013. Available from: http://hdl.handle.net/2077/31995

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kent State University

27. Renszel, Krystal Marie. USING MUTAGENESIS AND STEM CELLS TO UNDERSTAND RETROVIRAL NEUROVIRULENCE.

Degree: MS, College of Arts and Sciences / School of Biomedical Sciences, 2009, Kent State University

 Certain retroviruses are capable of causing progressive spongiform neurodegeneration which results in paralysis, wasting and death; however, the disease mechanism remains unknown. In this study,… (more)

Subjects/Keywords: Biomedical Research; retrovirus; neurodegeneration; protein glycosylation

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APA (6th Edition):

Renszel, K. M. (2009). USING MUTAGENESIS AND STEM CELLS TO UNDERSTAND RETROVIRAL NEUROVIRULENCE. (Masters Thesis). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1254659655

Chicago Manual of Style (16th Edition):

Renszel, Krystal Marie. “USING MUTAGENESIS AND STEM CELLS TO UNDERSTAND RETROVIRAL NEUROVIRULENCE.” 2009. Masters Thesis, Kent State University. Accessed July 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1254659655.

MLA Handbook (7th Edition):

Renszel, Krystal Marie. “USING MUTAGENESIS AND STEM CELLS TO UNDERSTAND RETROVIRAL NEUROVIRULENCE.” 2009. Web. 22 Jul 2019.

Vancouver:

Renszel KM. USING MUTAGENESIS AND STEM CELLS TO UNDERSTAND RETROVIRAL NEUROVIRULENCE. [Internet] [Masters thesis]. Kent State University; 2009. [cited 2019 Jul 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1254659655.

Council of Science Editors:

Renszel KM. USING MUTAGENESIS AND STEM CELLS TO UNDERSTAND RETROVIRAL NEUROVIRULENCE. [Masters Thesis]. Kent State University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1254659655


Penn State University

28. Robinson, William Foster. Histologic and Electroretinographic Evidence of Diabetes-induced Retinal Neurodegeneration.

Degree: PhD, Anatomy, 2010, Penn State University

 Diabetic retinopathy is a vision-threatening condition, currently affecting more than 5 million Americans. It is the leading cause of new cases of blindness in working-aged… (more)

Subjects/Keywords: diabetes; retinopathy; neurodegeneration; photoreceptor; electroretinography; ERG; immunohistochemistry

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APA (6th Edition):

Robinson, W. F. (2010). Histologic and Electroretinographic Evidence of Diabetes-induced Retinal Neurodegeneration. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11412

Chicago Manual of Style (16th Edition):

Robinson, William Foster. “Histologic and Electroretinographic Evidence of Diabetes-induced Retinal Neurodegeneration.” 2010. Doctoral Dissertation, Penn State University. Accessed July 22, 2019. https://etda.libraries.psu.edu/catalog/11412.

MLA Handbook (7th Edition):

Robinson, William Foster. “Histologic and Electroretinographic Evidence of Diabetes-induced Retinal Neurodegeneration.” 2010. Web. 22 Jul 2019.

Vancouver:

Robinson WF. Histologic and Electroretinographic Evidence of Diabetes-induced Retinal Neurodegeneration. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2019 Jul 22]. Available from: https://etda.libraries.psu.edu/catalog/11412.

Council of Science Editors:

Robinson WF. Histologic and Electroretinographic Evidence of Diabetes-induced Retinal Neurodegeneration. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11412


Penn State University

29. Hall II, Eric Christopher. The contribution of an iron genetic modifier, HFE, to Alzheimer's disease.

Degree: PhD, Neuroscience, 2009, Penn State University

 Alzheimer’s disease (AD) is a neurodegenerative disorder of the human central nervous system characterized by loss of memory that leads to dementia. The pathological characteristics… (more)

Subjects/Keywords: amyloid; neurodegeneration; HFE; iron; tau; pin1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hall II, E. C. (2009). The contribution of an iron genetic modifier, HFE, to Alzheimer's disease. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/10189

Chicago Manual of Style (16th Edition):

Hall II, Eric Christopher. “The contribution of an iron genetic modifier, HFE, to Alzheimer's disease.” 2009. Doctoral Dissertation, Penn State University. Accessed July 22, 2019. https://etda.libraries.psu.edu/catalog/10189.

MLA Handbook (7th Edition):

Hall II, Eric Christopher. “The contribution of an iron genetic modifier, HFE, to Alzheimer's disease.” 2009. Web. 22 Jul 2019.

Vancouver:

Hall II EC. The contribution of an iron genetic modifier, HFE, to Alzheimer's disease. [Internet] [Doctoral dissertation]. Penn State University; 2009. [cited 2019 Jul 22]. Available from: https://etda.libraries.psu.edu/catalog/10189.

Council of Science Editors:

Hall II EC. The contribution of an iron genetic modifier, HFE, to Alzheimer's disease. [Doctoral Dissertation]. Penn State University; 2009. Available from: https://etda.libraries.psu.edu/catalog/10189


Kent State University

30. Vignos, Megan C. A HISTOPATHOLOGICAL AND MAGNETIC RESONANCE IMAGING ASSESSMENT OF MYELOCORTICAL MULTIPLE SCLEROSIS: A NEW PATHOLOGICAL VARIANT.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2016, Kent State University

 Multiple sclerosis (MS) is an inflammatory-mediated demyelinating, neurodegenerative disease characterized by a diverse clinical course and varying patterns of disease progression. It affects millions of… (more)

Subjects/Keywords: Biomedical Research; Multiple Sclerosis, Histopathology, MRI, Neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vignos, M. C. (2016). A HISTOPATHOLOGICAL AND MAGNETIC RESONANCE IMAGING ASSESSMENT OF MYELOCORTICAL MULTIPLE SCLEROSIS: A NEW PATHOLOGICAL VARIANT. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1461528682

Chicago Manual of Style (16th Edition):

Vignos, Megan C. “A HISTOPATHOLOGICAL AND MAGNETIC RESONANCE IMAGING ASSESSMENT OF MYELOCORTICAL MULTIPLE SCLEROSIS: A NEW PATHOLOGICAL VARIANT.” 2016. Doctoral Dissertation, Kent State University. Accessed July 22, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=kent1461528682.

MLA Handbook (7th Edition):

Vignos, Megan C. “A HISTOPATHOLOGICAL AND MAGNETIC RESONANCE IMAGING ASSESSMENT OF MYELOCORTICAL MULTIPLE SCLEROSIS: A NEW PATHOLOGICAL VARIANT.” 2016. Web. 22 Jul 2019.

Vancouver:

Vignos MC. A HISTOPATHOLOGICAL AND MAGNETIC RESONANCE IMAGING ASSESSMENT OF MYELOCORTICAL MULTIPLE SCLEROSIS: A NEW PATHOLOGICAL VARIANT. [Internet] [Doctoral dissertation]. Kent State University; 2016. [cited 2019 Jul 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1461528682.

Council of Science Editors:

Vignos MC. A HISTOPATHOLOGICAL AND MAGNETIC RESONANCE IMAGING ASSESSMENT OF MYELOCORTICAL MULTIPLE SCLEROSIS: A NEW PATHOLOGICAL VARIANT. [Doctoral Dissertation]. Kent State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1461528682

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