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You searched for subject:(nervous system diseases). Showing records 1 – 30 of 373 total matches.

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Nelson Mandela Metropolitan University

1. Visser, Claire. Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA).

Degree: Faculty of Science, 2011, Nelson Mandela Metropolitan University

 β-methylamino-L-alanine (BMAA) is thought to be a contributing factor of Amyotrophic Lateral Sclerosis-Parkinsonism Dementia Complex (ALS/PDC). It has been shown that the levels of toxin… (more)

Subjects/Keywords: Neurotoxic agents; Nervous system  – Diseases

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APA (6th Edition):

Visser, C. (2011). Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA). (Thesis). Nelson Mandela Metropolitan University. Retrieved from http://hdl.handle.net/10948/1451

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Visser, Claire. “Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA).” 2011. Thesis, Nelson Mandela Metropolitan University. Accessed September 24, 2020. http://hdl.handle.net/10948/1451.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Visser, Claire. “Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA).” 2011. Web. 24 Sep 2020.

Vancouver:

Visser C. Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA). [Internet] [Thesis]. Nelson Mandela Metropolitan University; 2011. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/10948/1451.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Visser C. Evaluation of model systems for the study of protein association / incorporation of Beta-Methylamino-L-Alanine (BMAA). [Thesis]. Nelson Mandela Metropolitan University; 2011. Available from: http://hdl.handle.net/10948/1451

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Nelson Mandela Metropolitan University

2. Scott, Laura Louise. β-N-Methylamino-L-Alanine is a developmental neurotoxin.

Degree: 2019, Nelson Mandela Metropolitan University

 β-N-methylamino-L-alanine (BMAA) has been implicated in the development of the neurodegenerative diseases Amyotrophic Lateral Sclerosis/Parkinsonism Dementia Complex (ALS/PDC), Amyotrophic Lateral Sclerosis (ALS) and Alzheimer’s Disease… (more)

Subjects/Keywords: Neurotoxic agents; Nervous system  – Diseases

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APA (6th Edition):

Scott, L. L. (2019). β-N-Methylamino-L-Alanine is a developmental neurotoxin. (Thesis). Nelson Mandela Metropolitan University. Retrieved from http://hdl.handle.net/10948/43633

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Scott, Laura Louise. “β-N-Methylamino-L-Alanine is a developmental neurotoxin.” 2019. Thesis, Nelson Mandela Metropolitan University. Accessed September 24, 2020. http://hdl.handle.net/10948/43633.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Scott, Laura Louise. “β-N-Methylamino-L-Alanine is a developmental neurotoxin.” 2019. Web. 24 Sep 2020.

Vancouver:

Scott LL. β-N-Methylamino-L-Alanine is a developmental neurotoxin. [Internet] [Thesis]. Nelson Mandela Metropolitan University; 2019. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/10948/43633.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Scott LL. β-N-Methylamino-L-Alanine is a developmental neurotoxin. [Thesis]. Nelson Mandela Metropolitan University; 2019. Available from: http://hdl.handle.net/10948/43633

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

3. Williamson, Jennifer. INVESTIGATION OF THE HUNTINGTIN-HAP40 INTERACTION IN HUNTINGTON’S DISEASE.

Degree: MSc, 2013, McMaster University

Huntington’s disease (HD) is a neurodegenerative disorder caused by the expansion of a polyglutamine tract in the huntingtin protein. The huntingtin protein has many… (more)

Subjects/Keywords: HAP40; Huntingtin; Huntington's disease; Nervous System Diseases; Nervous System Diseases

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APA (6th Edition):

Williamson, J. (2013). INVESTIGATION OF THE HUNTINGTIN-HAP40 INTERACTION IN HUNTINGTON’S DISEASE. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/15287

Chicago Manual of Style (16th Edition):

Williamson, Jennifer. “INVESTIGATION OF THE HUNTINGTIN-HAP40 INTERACTION IN HUNTINGTON’S DISEASE.” 2013. Masters Thesis, McMaster University. Accessed September 24, 2020. http://hdl.handle.net/11375/15287.

MLA Handbook (7th Edition):

Williamson, Jennifer. “INVESTIGATION OF THE HUNTINGTIN-HAP40 INTERACTION IN HUNTINGTON’S DISEASE.” 2013. Web. 24 Sep 2020.

Vancouver:

Williamson J. INVESTIGATION OF THE HUNTINGTIN-HAP40 INTERACTION IN HUNTINGTON’S DISEASE. [Internet] [Masters thesis]. McMaster University; 2013. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/11375/15287.

Council of Science Editors:

Williamson J. INVESTIGATION OF THE HUNTINGTIN-HAP40 INTERACTION IN HUNTINGTON’S DISEASE. [Masters Thesis]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15287


McMaster University

4. Egier, David A. Tay-Sachs Disease: Mechanisms of Neuropathology and Potential Therapeutic Strategies Utilizing Human Lysosomal Sialidase.

Degree: MSc, 2012, McMaster University

GM2 gangliosidoses encompass a group of chronic neurodegenerative disorders characterized by metabolic defects in ganglioside catabolism and marked intralysosomal accumulation of GM2 in central… (more)

Subjects/Keywords: Tay-Sachs; sialidase; Sandhoff; therapy; lysosomal storage disorder; macrophage; Nervous System Diseases; Nervous System Diseases

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APA (6th Edition):

Egier, D. A. (2012). Tay-Sachs Disease: Mechanisms of Neuropathology and Potential Therapeutic Strategies Utilizing Human Lysosomal Sialidase. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11785

Chicago Manual of Style (16th Edition):

Egier, David A. “Tay-Sachs Disease: Mechanisms of Neuropathology and Potential Therapeutic Strategies Utilizing Human Lysosomal Sialidase.” 2012. Masters Thesis, McMaster University. Accessed September 24, 2020. http://hdl.handle.net/11375/11785.

MLA Handbook (7th Edition):

Egier, David A. “Tay-Sachs Disease: Mechanisms of Neuropathology and Potential Therapeutic Strategies Utilizing Human Lysosomal Sialidase.” 2012. Web. 24 Sep 2020.

Vancouver:

Egier DA. Tay-Sachs Disease: Mechanisms of Neuropathology and Potential Therapeutic Strategies Utilizing Human Lysosomal Sialidase. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/11375/11785.

Council of Science Editors:

Egier DA. Tay-Sachs Disease: Mechanisms of Neuropathology and Potential Therapeutic Strategies Utilizing Human Lysosomal Sialidase. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/11785


Ryerson University

5. Ho, Shannon Cheuk Ying. Characterizing the VAC14 multimeric state.

Degree: 2011, Ryerson University

 PtdIns(3,5)₂ is involved in a number of cellular processes, such as the regulation of endolysosome morphology and membrane trafficking, autophagy and ion transport. In mammala,… (more)

Subjects/Keywords: Proteins; Proteins  – Physiological transport; Nervous system  – Degeneration; Nervous system  – Diseases; Molecular biology

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APA (6th Edition):

Ho, S. C. Y. (2011). Characterizing the VAC14 multimeric state. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A1407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ho, Shannon Cheuk Ying. “Characterizing the VAC14 multimeric state.” 2011. Thesis, Ryerson University. Accessed September 24, 2020. https://digital.library.ryerson.ca/islandora/object/RULA%3A1407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ho, Shannon Cheuk Ying. “Characterizing the VAC14 multimeric state.” 2011. Web. 24 Sep 2020.

Vancouver:

Ho SCY. Characterizing the VAC14 multimeric state. [Internet] [Thesis]. Ryerson University; 2011. [cited 2020 Sep 24]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A1407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ho SCY. Characterizing the VAC14 multimeric state. [Thesis]. Ryerson University; 2011. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A1407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

6. Johnson-Kerner, Bethany. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.

Degree: 2013, Columbia University

 Patients with giant axonal neuropathy (GAN) exhibit loss of motor and sensory function and typically live for less than 30 years. GAN is caused by… (more)

Subjects/Keywords: Cytoplasmic filaments; Nervous system – Diseases; Nervous system – Degeneration; Motor neurons; Neuropathy; Neurosciences; Cytology

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APA (6th Edition):

Johnson-Kerner, B. (2013). Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8Z324C3

Chicago Manual of Style (16th Edition):

Johnson-Kerner, Bethany. “Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.” 2013. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/D8Z324C3.

MLA Handbook (7th Edition):

Johnson-Kerner, Bethany. “Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons.” 2013. Web. 24 Sep 2020.

Vancouver:

Johnson-Kerner B. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/D8Z324C3.

Council of Science Editors:

Johnson-Kerner B. Role of Gigaxonin in the Regulation of Intermediate Filaments: a Study Using Giant Axonal Neuropathy Patient-Derived Induced Pluripotent Stem Cell-Motor Neurons. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://doi.org/10.7916/D8Z324C3


Columbia University

7. Shin, William Kihoon. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.

Degree: 2016, Columbia University

 This thesis describes the development of an systems biology method to study transcriptional programs that are activated during early and late phases of cell-fusion mediated… (more)

Subjects/Keywords: Cell hybridization; Central nervous system – Diseases; Nervous system – Degeneration; Genetic transcription; Bioinformatics; Neurosciences; Biology – Classification

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APA (6th Edition):

Shin, W. K. (2016). Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8CR5TV2

Chicago Manual of Style (16th Edition):

Shin, William Kihoon. “Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.” 2016. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/D8CR5TV2.

MLA Handbook (7th Edition):

Shin, William Kihoon. “Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming.” 2016. Web. 24 Sep 2020.

Vancouver:

Shin WK. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/D8CR5TV2.

Council of Science Editors:

Shin WK. Systems Biology Approaches to The Study of Neurological Disorders and Somatic Cell Reprogramming. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8CR5TV2


University of Western Ontario

8. Allen, Matti D. Impacts of Diabetic Neuropathy on the Human Neuromuscular System.

Degree: 2014, University of Western Ontario

 Diabetes mellitus (DM) imparts vascular and metabolic stressors that cause damage and dysfunction to the human nervous system. The disorder associated with this dysfunction is… (more)

Subjects/Keywords: Diabetic polyneuropathy (DPN); human; motor nervous system; muscle; weakness; motor unit; Nervous System Diseases

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APA (6th Edition):

Allen, M. D. (2014). Impacts of Diabetic Neuropathy on the Human Neuromuscular System. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2299

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Allen, Matti D. “Impacts of Diabetic Neuropathy on the Human Neuromuscular System.” 2014. Thesis, University of Western Ontario. Accessed September 24, 2020. https://ir.lib.uwo.ca/etd/2299.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Allen, Matti D. “Impacts of Diabetic Neuropathy on the Human Neuromuscular System.” 2014. Web. 24 Sep 2020.

Vancouver:

Allen MD. Impacts of Diabetic Neuropathy on the Human Neuromuscular System. [Internet] [Thesis]. University of Western Ontario; 2014. [cited 2020 Sep 24]. Available from: https://ir.lib.uwo.ca/etd/2299.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Allen MD. Impacts of Diabetic Neuropathy on the Human Neuromuscular System. [Thesis]. University of Western Ontario; 2014. Available from: https://ir.lib.uwo.ca/etd/2299

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida Atlantic University

9. Morales, George J. A study on neural conduction as in myelinated structure under pathological conditions.

Degree: PhD, 2011, Florida Atlantic University

Summary: A method for modeling and simulating neural action potential (AP) propagation along the length of an axon containing a number of Ranvier nodes is… (more)

Subjects/Keywords: Nervous system – Diseases – Research; Demyelination; Nodes of Ranvier; Neuromuscular diseases – Research

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APA (6th Edition):

Morales, G. J. (2011). A study on neural conduction as in myelinated structure under pathological conditions. (Doctoral Dissertation). Florida Atlantic University. Retrieved from http://purl.flvc.org/FAU/3171723

Chicago Manual of Style (16th Edition):

Morales, George J. “A study on neural conduction as in myelinated structure under pathological conditions.” 2011. Doctoral Dissertation, Florida Atlantic University. Accessed September 24, 2020. http://purl.flvc.org/FAU/3171723.

MLA Handbook (7th Edition):

Morales, George J. “A study on neural conduction as in myelinated structure under pathological conditions.” 2011. Web. 24 Sep 2020.

Vancouver:

Morales GJ. A study on neural conduction as in myelinated structure under pathological conditions. [Internet] [Doctoral dissertation]. Florida Atlantic University; 2011. [cited 2020 Sep 24]. Available from: http://purl.flvc.org/FAU/3171723.

Council of Science Editors:

Morales GJ. A study on neural conduction as in myelinated structure under pathological conditions. [Doctoral Dissertation]. Florida Atlantic University; 2011. Available from: http://purl.flvc.org/FAU/3171723


University of Maine

10. Willows, Jake. Evaluating the Occurrence of Age-related Peripheral Neuropathy in HET3 Mice and Development of a Whole Tissue Imaging Technique for Analyzing Total Innervation in the Subcutaneous Adipose Depot.

Degree: MS, Zoology, 2019, University of Maine

  Adipose tissue can be characterized as either being a white (energy storing) depot or a brown (energy expending) depot and both have been found… (more)

Subjects/Keywords: Adipose; neurobiology; imaging; aging; peripheral nervous system; Animal Experimentation and Research; Animals; Cardiovascular Diseases; Cardiovascular System; Investigative Techniques; Nervous System; Nervous System Diseases; Nutritional and Metabolic Diseases

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APA (6th Edition):

Willows, J. (2019). Evaluating the Occurrence of Age-related Peripheral Neuropathy in HET3 Mice and Development of a Whole Tissue Imaging Technique for Analyzing Total Innervation in the Subcutaneous Adipose Depot. (Masters Thesis). University of Maine. Retrieved from https://digitalcommons.library.umaine.edu/etd/3121

Chicago Manual of Style (16th Edition):

Willows, Jake. “Evaluating the Occurrence of Age-related Peripheral Neuropathy in HET3 Mice and Development of a Whole Tissue Imaging Technique for Analyzing Total Innervation in the Subcutaneous Adipose Depot.” 2019. Masters Thesis, University of Maine. Accessed September 24, 2020. https://digitalcommons.library.umaine.edu/etd/3121.

MLA Handbook (7th Edition):

Willows, Jake. “Evaluating the Occurrence of Age-related Peripheral Neuropathy in HET3 Mice and Development of a Whole Tissue Imaging Technique for Analyzing Total Innervation in the Subcutaneous Adipose Depot.” 2019. Web. 24 Sep 2020.

Vancouver:

Willows J. Evaluating the Occurrence of Age-related Peripheral Neuropathy in HET3 Mice and Development of a Whole Tissue Imaging Technique for Analyzing Total Innervation in the Subcutaneous Adipose Depot. [Internet] [Masters thesis]. University of Maine; 2019. [cited 2020 Sep 24]. Available from: https://digitalcommons.library.umaine.edu/etd/3121.

Council of Science Editors:

Willows J. Evaluating the Occurrence of Age-related Peripheral Neuropathy in HET3 Mice and Development of a Whole Tissue Imaging Technique for Analyzing Total Innervation in the Subcutaneous Adipose Depot. [Masters Thesis]. University of Maine; 2019. Available from: https://digitalcommons.library.umaine.edu/etd/3121

11. Choudhury, Sourav Roy. Developing an Adeno-Associated Viral Vector (AAV) Toolbox for CNS Gene Therapy: A Dissertation.

Degree: Interdisciplinary Graduate Program, Neurology, 2016, U of Massachusetts : Med

  Neurological disorders – disorders of the brain, spine and associated nerves – are a leading contributor to global disease burden with a sizable economic… (more)

Subjects/Keywords: Genetic Therapy; Genetic Vectors; Huntington Disease; Central Nervous System Diseases; Dependovirus; Genetics; Molecular Genetics; Nervous System Diseases; Neurology; Therapeutics

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APA (6th Edition):

Choudhury, S. R. (2016). Developing an Adeno-Associated Viral Vector (AAV) Toolbox for CNS Gene Therapy: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/809

Chicago Manual of Style (16th Edition):

Choudhury, Sourav Roy. “Developing an Adeno-Associated Viral Vector (AAV) Toolbox for CNS Gene Therapy: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed September 24, 2020. http://escholarship.umassmed.edu/gsbs_diss/809.

MLA Handbook (7th Edition):

Choudhury, Sourav Roy. “Developing an Adeno-Associated Viral Vector (AAV) Toolbox for CNS Gene Therapy: A Dissertation.” 2016. Web. 24 Sep 2020.

Vancouver:

Choudhury SR. Developing an Adeno-Associated Viral Vector (AAV) Toolbox for CNS Gene Therapy: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2020 Sep 24]. Available from: http://escholarship.umassmed.edu/gsbs_diss/809.

Council of Science Editors:

Choudhury SR. Developing an Adeno-Associated Viral Vector (AAV) Toolbox for CNS Gene Therapy: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/809


McMaster University

12. Fallah, Aria. PREDICTORS OF SEIZURE OUTCOMES IN CHILDREN WITH TUBEROUS SCLEROSIS COMPLEX UNDERGOING RESECTION EPILEPSY SURGERY: AN INDIVIDUAL PARTICIPANT DATA META-ANALYSIS.

Degree: MSc, 2013, McMaster University

Objective: To perform a systematic review and individual participant data meta-analysis to identify preoperative factors associated with a good seizure outcome in children with… (more)

Subjects/Keywords: tuberious sclerosis; epilepsy; pediatric; surgery; outcomes; meta-analysis; Nervous System Diseases; Neurology; Pediatrics; Nervous System Diseases

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APA (6th Edition):

Fallah, A. (2013). PREDICTORS OF SEIZURE OUTCOMES IN CHILDREN WITH TUBEROUS SCLEROSIS COMPLEX UNDERGOING RESECTION EPILEPSY SURGERY: AN INDIVIDUAL PARTICIPANT DATA META-ANALYSIS. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/13087

Chicago Manual of Style (16th Edition):

Fallah, Aria. “PREDICTORS OF SEIZURE OUTCOMES IN CHILDREN WITH TUBEROUS SCLEROSIS COMPLEX UNDERGOING RESECTION EPILEPSY SURGERY: AN INDIVIDUAL PARTICIPANT DATA META-ANALYSIS.” 2013. Masters Thesis, McMaster University. Accessed September 24, 2020. http://hdl.handle.net/11375/13087.

MLA Handbook (7th Edition):

Fallah, Aria. “PREDICTORS OF SEIZURE OUTCOMES IN CHILDREN WITH TUBEROUS SCLEROSIS COMPLEX UNDERGOING RESECTION EPILEPSY SURGERY: AN INDIVIDUAL PARTICIPANT DATA META-ANALYSIS.” 2013. Web. 24 Sep 2020.

Vancouver:

Fallah A. PREDICTORS OF SEIZURE OUTCOMES IN CHILDREN WITH TUBEROUS SCLEROSIS COMPLEX UNDERGOING RESECTION EPILEPSY SURGERY: AN INDIVIDUAL PARTICIPANT DATA META-ANALYSIS. [Internet] [Masters thesis]. McMaster University; 2013. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/11375/13087.

Council of Science Editors:

Fallah A. PREDICTORS OF SEIZURE OUTCOMES IN CHILDREN WITH TUBEROUS SCLEROSIS COMPLEX UNDERGOING RESECTION EPILEPSY SURGERY: AN INDIVIDUAL PARTICIPANT DATA META-ANALYSIS. [Masters Thesis]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/13087


Columbia University

13. Colvin, Leigh Elizabeth. The Psychological Factors and Neural Substrates Associated with Metacognition among Community-Dwelling and Neurologic Cohorts of Older Adults.

Degree: 2019, Columbia University

 This project consists of three distinct, but sequential studies that explore the psychological factors and neuropathological substrates of metacognition or self-awareness among older adults. Study… (more)

Subjects/Keywords: Clinical psychology; Neurosciences; Personality – Psychological aspects; Metacognition; Older people; Nervous system – Diseases; Nervous system – Diseases – Etiology

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APA (6th Edition):

Colvin, L. E. (2019). The Psychological Factors and Neural Substrates Associated with Metacognition among Community-Dwelling and Neurologic Cohorts of Older Adults. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-zjyr-6m63

Chicago Manual of Style (16th Edition):

Colvin, Leigh Elizabeth. “The Psychological Factors and Neural Substrates Associated with Metacognition among Community-Dwelling and Neurologic Cohorts of Older Adults.” 2019. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/d8-zjyr-6m63.

MLA Handbook (7th Edition):

Colvin, Leigh Elizabeth. “The Psychological Factors and Neural Substrates Associated with Metacognition among Community-Dwelling and Neurologic Cohorts of Older Adults.” 2019. Web. 24 Sep 2020.

Vancouver:

Colvin LE. The Psychological Factors and Neural Substrates Associated with Metacognition among Community-Dwelling and Neurologic Cohorts of Older Adults. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/d8-zjyr-6m63.

Council of Science Editors:

Colvin LE. The Psychological Factors and Neural Substrates Associated with Metacognition among Community-Dwelling and Neurologic Cohorts of Older Adults. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-zjyr-6m63


University of British Columbia

14. Biro, Andrew J. Specific aspects of neurodegenerative disease .

Degree: 1989, University of British Columbia

 This thesis is broken into four chapters. The first two chapters summarize two separate lines of investigation into the role of a putative neurotoxin in… (more)

Subjects/Keywords: Nervous system  – Degeneration; Nervous system  – Diseases  – Etiology; Nerve Degeneration; Nervous System Diseases  – etiology

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APA (6th Edition):

Biro, A. J. (1989). Specific aspects of neurodegenerative disease . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/28919

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Biro, Andrew J. “Specific aspects of neurodegenerative disease .” 1989. Thesis, University of British Columbia. Accessed September 24, 2020. http://hdl.handle.net/2429/28919.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Biro, Andrew J. “Specific aspects of neurodegenerative disease .” 1989. Web. 24 Sep 2020.

Vancouver:

Biro AJ. Specific aspects of neurodegenerative disease . [Internet] [Thesis]. University of British Columbia; 1989. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/2429/28919.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Biro AJ. Specific aspects of neurodegenerative disease . [Thesis]. University of British Columbia; 1989. Available from: http://hdl.handle.net/2429/28919

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Armstrong, Deborah K. Transitioning Through Middle Age with an Incomplete Spinal Cord Injury: A Qualitative Description of Changes in Physical Function: A Dissertation.

Degree: 2012, U of Massachusetts : Nursing

  Over 260,000 Americans are living with a traumatic spinal cord injury (SCI). Medical advances have increased the longevity of individuals living with SCI into… (more)

Subjects/Keywords: Spinal Cord Injuries; Aging; Middle Aged; Nervous System Diseases; Nursing

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APA (6th Edition):

Armstrong, D. K. (2012). Transitioning Through Middle Age with an Incomplete Spinal Cord Injury: A Qualitative Description of Changes in Physical Function: A Dissertation. (Thesis). U of Massachusetts : Nursing. Retrieved from 10.13028/khcf-bt94 ; https://escholarship.umassmed.edu/gsn_diss/28

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Armstrong, Deborah K. “Transitioning Through Middle Age with an Incomplete Spinal Cord Injury: A Qualitative Description of Changes in Physical Function: A Dissertation.” 2012. Thesis, U of Massachusetts : Nursing. Accessed September 24, 2020. 10.13028/khcf-bt94 ; https://escholarship.umassmed.edu/gsn_diss/28.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Armstrong, Deborah K. “Transitioning Through Middle Age with an Incomplete Spinal Cord Injury: A Qualitative Description of Changes in Physical Function: A Dissertation.” 2012. Web. 24 Sep 2020.

Vancouver:

Armstrong DK. Transitioning Through Middle Age with an Incomplete Spinal Cord Injury: A Qualitative Description of Changes in Physical Function: A Dissertation. [Internet] [Thesis]. U of Massachusetts : Nursing; 2012. [cited 2020 Sep 24]. Available from: 10.13028/khcf-bt94 ; https://escholarship.umassmed.edu/gsn_diss/28.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Armstrong DK. Transitioning Through Middle Age with an Incomplete Spinal Cord Injury: A Qualitative Description of Changes in Physical Function: A Dissertation. [Thesis]. U of Massachusetts : Nursing; 2012. Available from: 10.13028/khcf-bt94 ; https://escholarship.umassmed.edu/gsn_diss/28

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Fridman, Leticia. Histopathological Characterization of the Dystrophic Phenotype and Development of Therapeutic Candidates for a Gene Therapy Pre-Clinical Study in Dysferlin Deficient Mice.

Degree: Translational Science, Neurology, 2016, U of Massachusetts : Med

  Dysferlin deficient muscular dystrophy is a devastating disease that leads to loss of mobility and quality of life in patients. Dysferlin is a 230… (more)

Subjects/Keywords: dysferlin; muscular dystrophy; membrane resealing; Cell Biology; Nervous System Diseases; Neurology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fridman, L. (2016). Histopathological Characterization of the Dystrophic Phenotype and Development of Therapeutic Candidates for a Gene Therapy Pre-Clinical Study in Dysferlin Deficient Mice. (Masters Thesis). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/881

Chicago Manual of Style (16th Edition):

Fridman, Leticia. “Histopathological Characterization of the Dystrophic Phenotype and Development of Therapeutic Candidates for a Gene Therapy Pre-Clinical Study in Dysferlin Deficient Mice.” 2016. Masters Thesis, U of Massachusetts : Med. Accessed September 24, 2020. http://escholarship.umassmed.edu/gsbs_diss/881.

MLA Handbook (7th Edition):

Fridman, Leticia. “Histopathological Characterization of the Dystrophic Phenotype and Development of Therapeutic Candidates for a Gene Therapy Pre-Clinical Study in Dysferlin Deficient Mice.” 2016. Web. 24 Sep 2020.

Vancouver:

Fridman L. Histopathological Characterization of the Dystrophic Phenotype and Development of Therapeutic Candidates for a Gene Therapy Pre-Clinical Study in Dysferlin Deficient Mice. [Internet] [Masters thesis]. U of Massachusetts : Med; 2016. [cited 2020 Sep 24]. Available from: http://escholarship.umassmed.edu/gsbs_diss/881.

Council of Science Editors:

Fridman L. Histopathological Characterization of the Dystrophic Phenotype and Development of Therapeutic Candidates for a Gene Therapy Pre-Clinical Study in Dysferlin Deficient Mice. [Masters Thesis]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/881


Michigan Technological University

17. Larson, Robert. CENTRAL NEURAL MECHANISMS OF SALT-SENSITIVE HYPERTENSION.

Degree: PhD, Department of Biological Sciences, 2016, Michigan Technological University

  Hypertension (HTN) is a major risk factor for the development of cardiovascular disease, and it’s estimated that over 80 million adults in the United… (more)

Subjects/Keywords: hypertension; cardiovascular; sympathetic nervous system; paraventricular nucleus; Cardiovascular Diseases

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APA (6th Edition):

Larson, R. (2016). CENTRAL NEURAL MECHANISMS OF SALT-SENSITIVE HYPERTENSION. (Doctoral Dissertation). Michigan Technological University. Retrieved from http://digitalcommons.mtu.edu/etdr/188

Chicago Manual of Style (16th Edition):

Larson, Robert. “CENTRAL NEURAL MECHANISMS OF SALT-SENSITIVE HYPERTENSION.” 2016. Doctoral Dissertation, Michigan Technological University. Accessed September 24, 2020. http://digitalcommons.mtu.edu/etdr/188.

MLA Handbook (7th Edition):

Larson, Robert. “CENTRAL NEURAL MECHANISMS OF SALT-SENSITIVE HYPERTENSION.” 2016. Web. 24 Sep 2020.

Vancouver:

Larson R. CENTRAL NEURAL MECHANISMS OF SALT-SENSITIVE HYPERTENSION. [Internet] [Doctoral dissertation]. Michigan Technological University; 2016. [cited 2020 Sep 24]. Available from: http://digitalcommons.mtu.edu/etdr/188.

Council of Science Editors:

Larson R. CENTRAL NEURAL MECHANISMS OF SALT-SENSITIVE HYPERTENSION. [Doctoral Dissertation]. Michigan Technological University; 2016. Available from: http://digitalcommons.mtu.edu/etdr/188


Stellenbosch University

18. Kriel, Saleema. Patients’ perceptions of barriers and facilitators influencing ability to return to work post-stroke.

Degree: MScPhysio, Health, 2019, Stellenbosch University

 ENGLISH SUMMARY : Introduction: Stroke is a neurological condition that impacts on the functional ability of the individual and affects participation in everyday activities, including… (more)

Subjects/Keywords: Cerebrovascular disease  – Patients  – Rehabilitation  – Attitudes; Nervous system  – Diseases  – Rehabilitation; UCTD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kriel, S. (2019). Patients’ perceptions of barriers and facilitators influencing ability to return to work post-stroke. (Masters Thesis). Stellenbosch University. Retrieved from http://hdl.handle.net/10019.1/106157

Chicago Manual of Style (16th Edition):

Kriel, Saleema. “Patients’ perceptions of barriers and facilitators influencing ability to return to work post-stroke.” 2019. Masters Thesis, Stellenbosch University. Accessed September 24, 2020. http://hdl.handle.net/10019.1/106157.

MLA Handbook (7th Edition):

Kriel, Saleema. “Patients’ perceptions of barriers and facilitators influencing ability to return to work post-stroke.” 2019. Web. 24 Sep 2020.

Vancouver:

Kriel S. Patients’ perceptions of barriers and facilitators influencing ability to return to work post-stroke. [Internet] [Masters thesis]. Stellenbosch University; 2019. [cited 2020 Sep 24]. Available from: http://hdl.handle.net/10019.1/106157.

Council of Science Editors:

Kriel S. Patients’ perceptions of barriers and facilitators influencing ability to return to work post-stroke. [Masters Thesis]. Stellenbosch University; 2019. Available from: http://hdl.handle.net/10019.1/106157


Michigan State University

19. Abdu, Mohamed-Tewfik Fawi. Pathology of experimental Leptospira pomona infection in hamsters.

Degree: PhD, 1962, Michigan State University

Subjects/Keywords: Weil's disease; Nervous system – Diseases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abdu, M. F. (1962). Pathology of experimental Leptospira pomona infection in hamsters. (Doctoral Dissertation). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:34318

Chicago Manual of Style (16th Edition):

Abdu, Mohamed-Tewfik Fawi. “Pathology of experimental Leptospira pomona infection in hamsters.” 1962. Doctoral Dissertation, Michigan State University. Accessed September 24, 2020. http://etd.lib.msu.edu/islandora/object/etd:34318.

MLA Handbook (7th Edition):

Abdu, Mohamed-Tewfik Fawi. “Pathology of experimental Leptospira pomona infection in hamsters.” 1962. Web. 24 Sep 2020.

Vancouver:

Abdu MF. Pathology of experimental Leptospira pomona infection in hamsters. [Internet] [Doctoral dissertation]. Michigan State University; 1962. [cited 2020 Sep 24]. Available from: http://etd.lib.msu.edu/islandora/object/etd:34318.

Council of Science Editors:

Abdu MF. Pathology of experimental Leptospira pomona infection in hamsters. [Doctoral Dissertation]. Michigan State University; 1962. Available from: http://etd.lib.msu.edu/islandora/object/etd:34318

20. De Jonge-Moore, Kwame M. Journey with Autism: Successful Educational and Social Interventions from a Single Case Study.

Degree: PhD, Curriculum and Instruction PhD, 2019, Andrews University

  Problem Statistics indicate that Autism Spectrum Disorder (ASD) is on the increase in America and worldwide (Xu, Strathearn, Liu, & Bao, 2018; Baio et… (more)

Subjects/Keywords: Autism; Intervention; Education; Social; Mental and Social Health; Nervous System Diseases

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APA (6th Edition):

De Jonge-Moore, K. M. (2019). Journey with Autism: Successful Educational and Social Interventions from a Single Case Study. (Doctoral Dissertation). Andrews University. Retrieved from https://digitalcommons.andrews.edu/dissertations/1734

Chicago Manual of Style (16th Edition):

De Jonge-Moore, Kwame M. “Journey with Autism: Successful Educational and Social Interventions from a Single Case Study.” 2019. Doctoral Dissertation, Andrews University. Accessed September 24, 2020. https://digitalcommons.andrews.edu/dissertations/1734.

MLA Handbook (7th Edition):

De Jonge-Moore, Kwame M. “Journey with Autism: Successful Educational and Social Interventions from a Single Case Study.” 2019. Web. 24 Sep 2020.

Vancouver:

De Jonge-Moore KM. Journey with Autism: Successful Educational and Social Interventions from a Single Case Study. [Internet] [Doctoral dissertation]. Andrews University; 2019. [cited 2020 Sep 24]. Available from: https://digitalcommons.andrews.edu/dissertations/1734.

Council of Science Editors:

De Jonge-Moore KM. Journey with Autism: Successful Educational and Social Interventions from a Single Case Study. [Doctoral Dissertation]. Andrews University; 2019. Available from: https://digitalcommons.andrews.edu/dissertations/1734


Montana State University

21. Ohlen, Sarah Beth. Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia.

Degree: College of Letters & Science, 2017, Montana State University

 Familial Dysautonomia (FD) is a recessive genetic disorder that leads to devastation of the peripheral nervous system and is the result of incomplete neurodevelopment and… (more)

Subjects/Keywords: Nervous system.; Degeneration (Pathology).; Diseases.; Genetics.; Protein kinases.; Therapeutics.

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APA (6th Edition):

Ohlen, S. B. (2017). Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia. (Thesis). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/15067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ohlen, Sarah Beth. “Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia.” 2017. Thesis, Montana State University. Accessed September 24, 2020. https://scholarworks.montana.edu/xmlui/handle/1/15067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ohlen, Sarah Beth. “Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia.” 2017. Web. 24 Sep 2020.

Vancouver:

Ohlen SB. Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia. [Internet] [Thesis]. Montana State University; 2017. [cited 2020 Sep 24]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/15067.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ohlen SB. Determining function of the IKAP protein in the peripheral nervous system for targeted therapeutic intervention in familial dysautonomia. [Thesis]. Montana State University; 2017. Available from: https://scholarworks.montana.edu/xmlui/handle/1/15067

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

22. Lee, Justin. Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy.

Degree: 2015, Columbia University

 Selective neuronal loss in response to loss or dysfunction of a ubiquitously expressed protein is a hallmark of neurodegenerative disease. Proximal spinal muscular atrophy (SMA)… (more)

Subjects/Keywords: Spinal muscular atrophy; Nervous system – Degeneration; Neuromuscular diseases; Neurosciences; Pathology; Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, J. (2015). Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8513XBQ

Chicago Manual of Style (16th Edition):

Lee, Justin. “Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy.” 2015. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/D8513XBQ.

MLA Handbook (7th Edition):

Lee, Justin. “Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy.” 2015. Web. 24 Sep 2020.

Vancouver:

Lee J. Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/D8513XBQ.

Council of Science Editors:

Lee J. Characterization of motor pool selectivity of neuromuscular degeneration and identification of molecular correlates of disease resistance in Type I spinal muscular atrophy. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8513XBQ


Columbia University

23. Effgen, Gwen Brink. Improving Outcomes after Repetitive Mild Traumatic Brain Injury from Shock Wave Exposure or Stretch Injury.

Degree: 2016, Columbia University

 The prevalence of injuries from improvised explosive devices (IEDs) in recent U.S. military conflicts has highlighted the lack of knowledge of the acute and long-term… (more)

Subjects/Keywords: Brain – Concussion; Improvised explosive devices; Nervous system – Diseases; Biomedical engineering

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APA (6th Edition):

Effgen, G. B. (2016). Improving Outcomes after Repetitive Mild Traumatic Brain Injury from Shock Wave Exposure or Stretch Injury. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8D21XQD

Chicago Manual of Style (16th Edition):

Effgen, Gwen Brink. “Improving Outcomes after Repetitive Mild Traumatic Brain Injury from Shock Wave Exposure or Stretch Injury.” 2016. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/D8D21XQD.

MLA Handbook (7th Edition):

Effgen, Gwen Brink. “Improving Outcomes after Repetitive Mild Traumatic Brain Injury from Shock Wave Exposure or Stretch Injury.” 2016. Web. 24 Sep 2020.

Vancouver:

Effgen GB. Improving Outcomes after Repetitive Mild Traumatic Brain Injury from Shock Wave Exposure or Stretch Injury. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/D8D21XQD.

Council of Science Editors:

Effgen GB. Improving Outcomes after Repetitive Mild Traumatic Brain Injury from Shock Wave Exposure or Stretch Injury. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8D21XQD


Columbia University

24. Feng, Qiao. Study of Short Forms of P/Q-Type Voltage-Gated Calcium Channels.

Degree: 2017, Columbia University

 P/Q-type voltage-gated calcium channels (CaV2.1) are expressed in both central and peripheral nervous systems, where they play a critical role in neurotransmitter release. Mutations in… (more)

Subjects/Keywords: Central nervous system; Neurotransmitters; Brain – Diseases; Calcium channels; Biology

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APA (6th Edition):

Feng, Q. (2017). Study of Short Forms of P/Q-Type Voltage-Gated Calcium Channels. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D82Z163H

Chicago Manual of Style (16th Edition):

Feng, Qiao. “Study of Short Forms of P/Q-Type Voltage-Gated Calcium Channels.” 2017. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/D82Z163H.

MLA Handbook (7th Edition):

Feng, Qiao. “Study of Short Forms of P/Q-Type Voltage-Gated Calcium Channels.” 2017. Web. 24 Sep 2020.

Vancouver:

Feng Q. Study of Short Forms of P/Q-Type Voltage-Gated Calcium Channels. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/D82Z163H.

Council of Science Editors:

Feng Q. Study of Short Forms of P/Q-Type Voltage-Gated Calcium Channels. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D82Z163H


University of Colorado

25. Mariani, Andrea. Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease.

Degree: MS, 2018, University of Colorado

 One of the most complex and intriguing biological problems is perhaps the development of neurodegeneration. Neurodegenerative diseases are many, and many seem to be their… (more)

Subjects/Keywords: neurodegenerative diseases; nervous system; neurodegeneration; neural dysfunction; Neurosciences

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APA (6th Edition):

Mariani, A. (2018). Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease. (Masters Thesis). University of Colorado. Retrieved from https://scholar.colorado.edu/iphy_gradetds/87

Chicago Manual of Style (16th Edition):

Mariani, Andrea. “Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease.” 2018. Masters Thesis, University of Colorado. Accessed September 24, 2020. https://scholar.colorado.edu/iphy_gradetds/87.

MLA Handbook (7th Edition):

Mariani, Andrea. “Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease.” 2018. Web. 24 Sep 2020.

Vancouver:

Mariani A. Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease. [Internet] [Masters thesis]. University of Colorado; 2018. [cited 2020 Sep 24]. Available from: https://scholar.colorado.edu/iphy_gradetds/87.

Council of Science Editors:

Mariani A. Amyotrophic Lateral Sclerosis: a Proposed Molecular Model of Disease. [Masters Thesis]. University of Colorado; 2018. Available from: https://scholar.colorado.edu/iphy_gradetds/87


Columbia University

26. Ebstein, Sarah Yehudit. TDP-43 and FUS in Amyotrophic Lateral Sclerosis: From Animal Models to Disease Mechanisms.

Degree: 2017, Columbia University

 Amyotrophic lateral sclerosis (ALS) is an aggressive neurodegenerative disease in which motor neurons selectively degenerate, leading to paralysis and death. Rare causal mutations in FUS… (more)

Subjects/Keywords: Neurosciences; Amyotrophic lateral sclerosis; Mutation (Biology); Nervous system – Diseases – Animal models

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APA (6th Edition):

Ebstein, S. Y. (2017). TDP-43 and FUS in Amyotrophic Lateral Sclerosis: From Animal Models to Disease Mechanisms. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8P84Q5F

Chicago Manual of Style (16th Edition):

Ebstein, Sarah Yehudit. “TDP-43 and FUS in Amyotrophic Lateral Sclerosis: From Animal Models to Disease Mechanisms.” 2017. Doctoral Dissertation, Columbia University. Accessed September 24, 2020. https://doi.org/10.7916/D8P84Q5F.

MLA Handbook (7th Edition):

Ebstein, Sarah Yehudit. “TDP-43 and FUS in Amyotrophic Lateral Sclerosis: From Animal Models to Disease Mechanisms.” 2017. Web. 24 Sep 2020.

Vancouver:

Ebstein SY. TDP-43 and FUS in Amyotrophic Lateral Sclerosis: From Animal Models to Disease Mechanisms. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2020 Sep 24]. Available from: https://doi.org/10.7916/D8P84Q5F.

Council of Science Editors:

Ebstein SY. TDP-43 and FUS in Amyotrophic Lateral Sclerosis: From Animal Models to Disease Mechanisms. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D8P84Q5F


California State University – San Bernardino

27. Ubina, Teresa Marie. AN ISOGENIC STEM CELL MODEL OF ALZHEIMER'S DISEASE: DIRECT EXPRESSION OF AMYLOID-BETA.

Degree: MSin Biology, Biology, 2017, California State University – San Bernardino

  Alzheimer’s disease (AD), identified over 100 years ago and intensively studied since the 1970s, has no effective treatments or mechanistic understanding of the underlying… (more)

Subjects/Keywords: Amyloid-beta; Alzheimer's Disease; Stem Cell; Neurodegeneration; Nervous System Diseases

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APA (6th Edition):

Ubina, T. M. (2017). AN ISOGENIC STEM CELL MODEL OF ALZHEIMER'S DISEASE: DIRECT EXPRESSION OF AMYLOID-BETA. (Thesis). California State University – San Bernardino. Retrieved from https://scholarworks.lib.csusb.edu/etd/523

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ubina, Teresa Marie. “AN ISOGENIC STEM CELL MODEL OF ALZHEIMER'S DISEASE: DIRECT EXPRESSION OF AMYLOID-BETA.” 2017. Thesis, California State University – San Bernardino. Accessed September 24, 2020. https://scholarworks.lib.csusb.edu/etd/523.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ubina, Teresa Marie. “AN ISOGENIC STEM CELL MODEL OF ALZHEIMER'S DISEASE: DIRECT EXPRESSION OF AMYLOID-BETA.” 2017. Web. 24 Sep 2020.

Vancouver:

Ubina TM. AN ISOGENIC STEM CELL MODEL OF ALZHEIMER'S DISEASE: DIRECT EXPRESSION OF AMYLOID-BETA. [Internet] [Thesis]. California State University – San Bernardino; 2017. [cited 2020 Sep 24]. Available from: https://scholarworks.lib.csusb.edu/etd/523.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ubina TM. AN ISOGENIC STEM CELL MODEL OF ALZHEIMER'S DISEASE: DIRECT EXPRESSION OF AMYLOID-BETA. [Thesis]. California State University – San Bernardino; 2017. Available from: https://scholarworks.lib.csusb.edu/etd/523

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

28. Lukić, Stevo R., 1968-. Предикција клинички значајних исхода код неуролошких болести применом метода вештачких неуронских мрежа и регресионе анализе.

Degree: 2018, Univerzitet u Beogradu

Неуронауке - Неурологија / Neuroscience - Neurology

Развој и праћење модела регресионе анализе и вештачких неуронских мрежа (БНМ) за предикцију (1) преживљавања након спонтаног можданог… (more)

Subjects/Keywords: nervous system diseases; clinical outcome; prognosis; artificial neural networks; logistic regression

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APA (6th Edition):

Lukić, Stevo R., 1. (2018). Предикција клинички значајних исхода код неуролошких болести применом метода вештачких неуронских мрежа и регресионе анализе. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:19082/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lukić, Stevo R., 1968-. “Предикција клинички значајних исхода код неуролошких болести применом метода вештачких неуронских мрежа и регресионе анализе.” 2018. Thesis, Univerzitet u Beogradu. Accessed September 24, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:19082/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lukić, Stevo R., 1968-. “Предикција клинички значајних исхода код неуролошких болести применом метода вештачких неуронских мрежа и регресионе анализе.” 2018. Web. 24 Sep 2020.

Vancouver:

Lukić, Stevo R. 1. Предикција клинички значајних исхода код неуролошких болести применом метода вештачких неуронских мрежа и регресионе анализе. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2020 Sep 24]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19082/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lukić, Stevo R. 1. Предикција клинички значајних исхода код неуролошких болести применом метода вештачких неуронских мрежа и регресионе анализе. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:19082/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

29. Gray, James Andrew Russell. Modulating the heat-shock response : a potential therapy for lysosomal storage disorders.

Degree: PhD, 2014, University of Oxford

 Lysosomal storage disorders (LSDs) comprise a broad disease group of inherited metabolic disorders, the majority of which are associated with CNS pathology, significant disability and… (more)

Subjects/Keywords: 616.3; Heat shock proteins; Lysosomal storage diseases; Central nervous system – Abnormalities

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gray, J. A. R. (2014). Modulating the heat-shock response : a potential therapy for lysosomal storage disorders. (Doctoral Dissertation). University of Oxford. Retrieved from https://ora.ox.ac.uk/objects/uuid:d9b746c9-9026-4a6e-97b5-00bb848100d7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711726

Chicago Manual of Style (16th Edition):

Gray, James Andrew Russell. “Modulating the heat-shock response : a potential therapy for lysosomal storage disorders.” 2014. Doctoral Dissertation, University of Oxford. Accessed September 24, 2020. https://ora.ox.ac.uk/objects/uuid:d9b746c9-9026-4a6e-97b5-00bb848100d7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711726.

MLA Handbook (7th Edition):

Gray, James Andrew Russell. “Modulating the heat-shock response : a potential therapy for lysosomal storage disorders.” 2014. Web. 24 Sep 2020.

Vancouver:

Gray JAR. Modulating the heat-shock response : a potential therapy for lysosomal storage disorders. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2020 Sep 24]. Available from: https://ora.ox.ac.uk/objects/uuid:d9b746c9-9026-4a6e-97b5-00bb848100d7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711726.

Council of Science Editors:

Gray JAR. Modulating the heat-shock response : a potential therapy for lysosomal storage disorders. [Doctoral Dissertation]. University of Oxford; 2014. Available from: https://ora.ox.ac.uk/objects/uuid:d9b746c9-9026-4a6e-97b5-00bb848100d7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711726


Rutgers University

30. Banga, Karansingh, 1991-. Development of an adaptive serious game for assessing cognitive engagement.

Degree: MS, Computer Science, 2019, Rutgers University

 Cognitive Engagement is defined as "The act of beginning and carrying on of activity with a sense of emotional involvement or commitment and the deliberate… (more)

Subjects/Keywords: Nervous system  – Diseases  – Patients  – Rehabilitation  – Technological innovations; Games  – Therapeutic use

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Banga, Karansingh, 1. (2019). Development of an adaptive serious game for assessing cognitive engagement. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60588/

Chicago Manual of Style (16th Edition):

Banga, Karansingh, 1991-. “Development of an adaptive serious game for assessing cognitive engagement.” 2019. Masters Thesis, Rutgers University. Accessed September 24, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/60588/.

MLA Handbook (7th Edition):

Banga, Karansingh, 1991-. “Development of an adaptive serious game for assessing cognitive engagement.” 2019. Web. 24 Sep 2020.

Vancouver:

Banga, Karansingh 1. Development of an adaptive serious game for assessing cognitive engagement. [Internet] [Masters thesis]. Rutgers University; 2019. [cited 2020 Sep 24]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60588/.

Council of Science Editors:

Banga, Karansingh 1. Development of an adaptive serious game for assessing cognitive engagement. [Masters Thesis]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60588/

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