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You searched for subject:(mutations). Showing records 1 – 30 of 813 total matches.

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1. Magdoud, Hajer. Implication fonctionnelle de la protéine adaptatrice Lnk dans les cellules normales et pathologiques du lignage mégacaryocytaire : Functional application of the Lnk adaptor protein in normal and pathological megakaryopoiesis.

Degree: Docteur es, Biologie moléculaire et cellulaire, 2014, Paris 13

 L’adaptateur Lnk est un inhibiteur clé dans le système hématopoïétique. Son invalidation chez la souris entraîne l’hyperprolifération des lignages myéloïdes (mégacaryocytaire et érythoïde). Les cellules… (more)

Subjects/Keywords: Mutations de Lnk; Lnk mutations

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APA (6th Edition):

Magdoud, H. (2014). Implication fonctionnelle de la protéine adaptatrice Lnk dans les cellules normales et pathologiques du lignage mégacaryocytaire : Functional application of the Lnk adaptor protein in normal and pathological megakaryopoiesis. (Doctoral Dissertation). Paris 13. Retrieved from http://www.theses.fr/2014PA132003

Chicago Manual of Style (16th Edition):

Magdoud, Hajer. “Implication fonctionnelle de la protéine adaptatrice Lnk dans les cellules normales et pathologiques du lignage mégacaryocytaire : Functional application of the Lnk adaptor protein in normal and pathological megakaryopoiesis.” 2014. Doctoral Dissertation, Paris 13. Accessed September 19, 2020. http://www.theses.fr/2014PA132003.

MLA Handbook (7th Edition):

Magdoud, Hajer. “Implication fonctionnelle de la protéine adaptatrice Lnk dans les cellules normales et pathologiques du lignage mégacaryocytaire : Functional application of the Lnk adaptor protein in normal and pathological megakaryopoiesis.” 2014. Web. 19 Sep 2020.

Vancouver:

Magdoud H. Implication fonctionnelle de la protéine adaptatrice Lnk dans les cellules normales et pathologiques du lignage mégacaryocytaire : Functional application of the Lnk adaptor protein in normal and pathological megakaryopoiesis. [Internet] [Doctoral dissertation]. Paris 13; 2014. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2014PA132003.

Council of Science Editors:

Magdoud H. Implication fonctionnelle de la protéine adaptatrice Lnk dans les cellules normales et pathologiques du lignage mégacaryocytaire : Functional application of the Lnk adaptor protein in normal and pathological megakaryopoiesis. [Doctoral Dissertation]. Paris 13; 2014. Available from: http://www.theses.fr/2014PA132003

2. Karkanis, Dimitrios. Mutations économiques et démographiques en Chine : croissance ou développement ? : Economic and Demographic Mutations in China : Growth or Development?.

Degree: Docteur es, Sciences économiques et démographiques, 2016, Université Toulouse I – Capitole

Le processus de croissance économique de la Chine ces dernières décennies, tiré principalement par la dynamique du secteur des exportations, est accompagné par une croissance… (more)

Subjects/Keywords: Mutations démographiques - Chine; Mutations économiques - Chine

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APA (6th Edition):

Karkanis, D. (2016). Mutations économiques et démographiques en Chine : croissance ou développement ? : Economic and Demographic Mutations in China : Growth or Development?. (Doctoral Dissertation). Université Toulouse I – Capitole. Retrieved from http://www.theses.fr/2016TOU10017

Chicago Manual of Style (16th Edition):

Karkanis, Dimitrios. “Mutations économiques et démographiques en Chine : croissance ou développement ? : Economic and Demographic Mutations in China : Growth or Development?.” 2016. Doctoral Dissertation, Université Toulouse I – Capitole. Accessed September 19, 2020. http://www.theses.fr/2016TOU10017.

MLA Handbook (7th Edition):

Karkanis, Dimitrios. “Mutations économiques et démographiques en Chine : croissance ou développement ? : Economic and Demographic Mutations in China : Growth or Development?.” 2016. Web. 19 Sep 2020.

Vancouver:

Karkanis D. Mutations économiques et démographiques en Chine : croissance ou développement ? : Economic and Demographic Mutations in China : Growth or Development?. [Internet] [Doctoral dissertation]. Université Toulouse I – Capitole; 2016. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2016TOU10017.

Council of Science Editors:

Karkanis D. Mutations économiques et démographiques en Chine : croissance ou développement ? : Economic and Demographic Mutations in China : Growth or Development?. [Doctoral Dissertation]. Université Toulouse I – Capitole; 2016. Available from: http://www.theses.fr/2016TOU10017


Wright State University

3. Alsaran, Hadel Mohammed. Functional characterization of cancer-related mutations of ERK3.

Degree: MS, Biochemistry and Molecular Biology, 2016, Wright State University

 Extracellular signal-regulated kinase 3 (ERK3) is an atypical member of the mitogen-activated protein kinase (MAPK) family. Recent studies have shown that ERK3 is highly upregulated… (more)

Subjects/Keywords: Biochemistry; ERK3; mutations

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APA (6th Edition):

Alsaran, H. M. (2016). Functional characterization of cancer-related mutations of ERK3. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1483791134536552

Chicago Manual of Style (16th Edition):

Alsaran, Hadel Mohammed. “Functional characterization of cancer-related mutations of ERK3.” 2016. Masters Thesis, Wright State University. Accessed September 19, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1483791134536552.

MLA Handbook (7th Edition):

Alsaran, Hadel Mohammed. “Functional characterization of cancer-related mutations of ERK3.” 2016. Web. 19 Sep 2020.

Vancouver:

Alsaran HM. Functional characterization of cancer-related mutations of ERK3. [Internet] [Masters thesis]. Wright State University; 2016. [cited 2020 Sep 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1483791134536552.

Council of Science Editors:

Alsaran HM. Functional characterization of cancer-related mutations of ERK3. [Masters Thesis]. Wright State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1483791134536552


University of Saskatchewan

4. MacAusland-Berg, Josh 1993-. FRK cancer-related mutations: Effect on enzymatic activity and cellular processes.

Degree: 2019, University of Saskatchewan

 Cancer-associated FRK mutations have not been thoroughly studied however, a previous study analyzed six cancer related mutations of BRK L16F, R131L, V253M, N317S, L343F, P450L;… (more)

Subjects/Keywords: Cancer associated mutations

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APA (6th Edition):

MacAusland-Berg, J. 1. (2019). FRK cancer-related mutations: Effect on enzymatic activity and cellular processes. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/12357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MacAusland-Berg, Josh 1993-. “FRK cancer-related mutations: Effect on enzymatic activity and cellular processes.” 2019. Thesis, University of Saskatchewan. Accessed September 19, 2020. http://hdl.handle.net/10388/12357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MacAusland-Berg, Josh 1993-. “FRK cancer-related mutations: Effect on enzymatic activity and cellular processes.” 2019. Web. 19 Sep 2020.

Vancouver:

MacAusland-Berg J1. FRK cancer-related mutations: Effect on enzymatic activity and cellular processes. [Internet] [Thesis]. University of Saskatchewan; 2019. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10388/12357.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MacAusland-Berg J1. FRK cancer-related mutations: Effect on enzymatic activity and cellular processes. [Thesis]. University of Saskatchewan; 2019. Available from: http://hdl.handle.net/10388/12357

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

5. Chan, Marina. Studies of the Misprocessing Mutations R1202D and E1204K in the Drug and Organic Anion Transporter, MRP1 (ABCC1) in Cultured HEK cells .

Degree: Pharmacology and Toxicology, 2013, Queens University

 Multidrug resistance protein 1 (MRP1) is a drug and organic anion transporter of the ATP-binding cassette superfamily. Previous studies showed that opposite charge substitutions of… (more)

Subjects/Keywords: Misprocessing Mutations ; MRP1

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APA (6th Edition):

Chan, M. (2013). Studies of the Misprocessing Mutations R1202D and E1204K in the Drug and Organic Anion Transporter, MRP1 (ABCC1) in Cultured HEK cells . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chan, Marina. “Studies of the Misprocessing Mutations R1202D and E1204K in the Drug and Organic Anion Transporter, MRP1 (ABCC1) in Cultured HEK cells .” 2013. Thesis, Queens University. Accessed September 19, 2020. http://hdl.handle.net/1974/8470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chan, Marina. “Studies of the Misprocessing Mutations R1202D and E1204K in the Drug and Organic Anion Transporter, MRP1 (ABCC1) in Cultured HEK cells .” 2013. Web. 19 Sep 2020.

Vancouver:

Chan M. Studies of the Misprocessing Mutations R1202D and E1204K in the Drug and Organic Anion Transporter, MRP1 (ABCC1) in Cultured HEK cells . [Internet] [Thesis]. Queens University; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1974/8470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chan M. Studies of the Misprocessing Mutations R1202D and E1204K in the Drug and Organic Anion Transporter, MRP1 (ABCC1) in Cultured HEK cells . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Birgy, André. Paysage adaptatif des bêta-lactamases TEM-1 et CTX-M-15 : Adaptative landscape of beta-lactamases TEM-1 and CTX-M-15.

Degree: Docteur es, Sciences de la vie et de la santé. Microbiologie, 2017, Sorbonne Paris Cité

De part leur importance médicale, leur capacité à évoluer et leur facilité de manipulations, les bêta-lactamases TEM-1 et plus récemment CTX-M-15, se sont imposées comme… (more)

Subjects/Keywords: Mutations compensatrices; Distribution de l'effet des mutations; Compensatory mutations; Distribution of the effect of mutations

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APA (6th Edition):

Birgy, A. (2017). Paysage adaptatif des bêta-lactamases TEM-1 et CTX-M-15 : Adaptative landscape of beta-lactamases TEM-1 and CTX-M-15. (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2017USPCC261

Chicago Manual of Style (16th Edition):

Birgy, André. “Paysage adaptatif des bêta-lactamases TEM-1 et CTX-M-15 : Adaptative landscape of beta-lactamases TEM-1 and CTX-M-15.” 2017. Doctoral Dissertation, Sorbonne Paris Cité. Accessed September 19, 2020. http://www.theses.fr/2017USPCC261.

MLA Handbook (7th Edition):

Birgy, André. “Paysage adaptatif des bêta-lactamases TEM-1 et CTX-M-15 : Adaptative landscape of beta-lactamases TEM-1 and CTX-M-15.” 2017. Web. 19 Sep 2020.

Vancouver:

Birgy A. Paysage adaptatif des bêta-lactamases TEM-1 et CTX-M-15 : Adaptative landscape of beta-lactamases TEM-1 and CTX-M-15. [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2017. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2017USPCC261.

Council of Science Editors:

Birgy A. Paysage adaptatif des bêta-lactamases TEM-1 et CTX-M-15 : Adaptative landscape of beta-lactamases TEM-1 and CTX-M-15. [Doctoral Dissertation]. Sorbonne Paris Cité; 2017. Available from: http://www.theses.fr/2017USPCC261

7. Wells, Owen Spencer. Cellular and biochemical analyses of TDP1 mediated chromosomal break repair.

Degree: PhD, 2014, University of Sussex

 Tyrosyl DNA phosphodiesterase 1 (TDP1) is an end- rocessing enzyme involved in the repair of abortive topoisomerase I (Top1) complexes. Although not essential for survival,… (more)

Subjects/Keywords: 570; QH0460 Mutations

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APA (6th Edition):

Wells, O. S. (2014). Cellular and biochemical analyses of TDP1 mediated chromosomal break repair. (Doctoral Dissertation). University of Sussex. Retrieved from http://sro.sussex.ac.uk/id/eprint/49564/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618903

Chicago Manual of Style (16th Edition):

Wells, Owen Spencer. “Cellular and biochemical analyses of TDP1 mediated chromosomal break repair.” 2014. Doctoral Dissertation, University of Sussex. Accessed September 19, 2020. http://sro.sussex.ac.uk/id/eprint/49564/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618903.

MLA Handbook (7th Edition):

Wells, Owen Spencer. “Cellular and biochemical analyses of TDP1 mediated chromosomal break repair.” 2014. Web. 19 Sep 2020.

Vancouver:

Wells OS. Cellular and biochemical analyses of TDP1 mediated chromosomal break repair. [Internet] [Doctoral dissertation]. University of Sussex; 2014. [cited 2020 Sep 19]. Available from: http://sro.sussex.ac.uk/id/eprint/49564/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618903.

Council of Science Editors:

Wells OS. Cellular and biochemical analyses of TDP1 mediated chromosomal break repair. [Doctoral Dissertation]. University of Sussex; 2014. Available from: http://sro.sussex.ac.uk/id/eprint/49564/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.618903


University of Houston

8. Wang, Mu 1988-. MUTAGENIC EFFECTS OF CHROMIUM (IV) ON HUMAN ENDOTHELIAL AND LUNG EPITHELIAL CELLS.

Degree: MS, Chemistry, 2014, University of Houston

 Effects of a chromium (IV) compound,diperoxoaquoethylenediaminechromium (IV) monohydrate (Cr(IV)-DPO), on human umbilical vein endothelial cells (HUVEC) and human bronchial epithelial cells (BEAS-2B) were investigated through… (more)

Subjects/Keywords: Mutations; Chromium(IV)

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APA (6th Edition):

Wang, M. 1. (2014). MUTAGENIC EFFECTS OF CHROMIUM (IV) ON HUMAN ENDOTHELIAL AND LUNG EPITHELIAL CELLS. (Masters Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/1430

Chicago Manual of Style (16th Edition):

Wang, Mu 1988-. “MUTAGENIC EFFECTS OF CHROMIUM (IV) ON HUMAN ENDOTHELIAL AND LUNG EPITHELIAL CELLS.” 2014. Masters Thesis, University of Houston. Accessed September 19, 2020. http://hdl.handle.net/10657/1430.

MLA Handbook (7th Edition):

Wang, Mu 1988-. “MUTAGENIC EFFECTS OF CHROMIUM (IV) ON HUMAN ENDOTHELIAL AND LUNG EPITHELIAL CELLS.” 2014. Web. 19 Sep 2020.

Vancouver:

Wang M1. MUTAGENIC EFFECTS OF CHROMIUM (IV) ON HUMAN ENDOTHELIAL AND LUNG EPITHELIAL CELLS. [Internet] [Masters thesis]. University of Houston; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10657/1430.

Council of Science Editors:

Wang M1. MUTAGENIC EFFECTS OF CHROMIUM (IV) ON HUMAN ENDOTHELIAL AND LUNG EPITHELIAL CELLS. [Masters Thesis]. University of Houston; 2014. Available from: http://hdl.handle.net/10657/1430


University of Utah

9. Szerlong, Heather. The nuclear actin-related proteins, Arp7 and Arp9: connecting ARP hetertodimerization to the regulation of chromatin remodling.

Degree: PhD, Oncological Sciences;, 2004, University of Utah

 Actin-related proteins (ARPs) belong to the actin superfamily of proteins with diverse functions in both the cytoplasm and the nucleus. ARPs share limited sequence similarity… (more)

Subjects/Keywords: Purification; Mutations

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APA (6th Edition):

Szerlong, H. (2004). The nuclear actin-related proteins, Arp7 and Arp9: connecting ARP hetertodimerization to the regulation of chromatin remodling. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1999/rec/1197

Chicago Manual of Style (16th Edition):

Szerlong, Heather. “The nuclear actin-related proteins, Arp7 and Arp9: connecting ARP hetertodimerization to the regulation of chromatin remodling.” 2004. Doctoral Dissertation, University of Utah. Accessed September 19, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1999/rec/1197.

MLA Handbook (7th Edition):

Szerlong, Heather. “The nuclear actin-related proteins, Arp7 and Arp9: connecting ARP hetertodimerization to the regulation of chromatin remodling.” 2004. Web. 19 Sep 2020.

Vancouver:

Szerlong H. The nuclear actin-related proteins, Arp7 and Arp9: connecting ARP hetertodimerization to the regulation of chromatin remodling. [Internet] [Doctoral dissertation]. University of Utah; 2004. [cited 2020 Sep 19]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1999/rec/1197.

Council of Science Editors:

Szerlong H. The nuclear actin-related proteins, Arp7 and Arp9: connecting ARP hetertodimerization to the regulation of chromatin remodling. [Doctoral Dissertation]. University of Utah; 2004. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1999/rec/1197


University of Utah

10. Zhang, Jie. Myotonia congenita-associated chloride channelopathies: mutations, function, and structure.

Degree: PhD, Neurology;, 1999, University of Utah

 Myotonia Congenita (MC) is a genetic muscle disorder manifesting myotonia, a delayed relaxation of muscle after contraction. MC includes two inherited forms: autosomal dominant myotonia… (more)

Subjects/Keywords: Physiology; Mutations

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APA (6th Edition):

Zhang, J. (1999). Myotonia congenita-associated chloride channelopathies: mutations, function, and structure. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1478/rec/799

Chicago Manual of Style (16th Edition):

Zhang, Jie. “Myotonia congenita-associated chloride channelopathies: mutations, function, and structure.” 1999. Doctoral Dissertation, University of Utah. Accessed September 19, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1478/rec/799.

MLA Handbook (7th Edition):

Zhang, Jie. “Myotonia congenita-associated chloride channelopathies: mutations, function, and structure.” 1999. Web. 19 Sep 2020.

Vancouver:

Zhang J. Myotonia congenita-associated chloride channelopathies: mutations, function, and structure. [Internet] [Doctoral dissertation]. University of Utah; 1999. [cited 2020 Sep 19]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1478/rec/799.

Council of Science Editors:

Zhang J. Myotonia congenita-associated chloride channelopathies: mutations, function, and structure. [Doctoral Dissertation]. University of Utah; 1999. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1478/rec/799


Queens University

11. Davis, Jordan. Intragenic Rescue of the Function of Long QT Syndrome-Causing Mutant hERG K+ Channels .

Degree: Biomedical and Molecular Sciences, Queens University

 The human-ether-a-go-go-related gene (hERG) encodes the pore forming subunit of the rapidly activating delayed rectifier K+ channel (IKr), which is important for the repolarization phase… (more)

Subjects/Keywords: hERG ; Mutations

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APA (6th Edition):

Davis, J. (n.d.). Intragenic Rescue of the Function of Long QT Syndrome-Causing Mutant hERG K+ Channels . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/24476

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davis, Jordan. “Intragenic Rescue of the Function of Long QT Syndrome-Causing Mutant hERG K+ Channels .” Thesis, Queens University. Accessed September 19, 2020. http://hdl.handle.net/1974/24476.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davis, Jordan. “Intragenic Rescue of the Function of Long QT Syndrome-Causing Mutant hERG K+ Channels .” Web. 19 Sep 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Davis J. Intragenic Rescue of the Function of Long QT Syndrome-Causing Mutant hERG K+ Channels . [Internet] [Thesis]. Queens University; [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1974/24476.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Davis J. Intragenic Rescue of the Function of Long QT Syndrome-Causing Mutant hERG K+ Channels . [Thesis]. Queens University; Available from: http://hdl.handle.net/1974/24476

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Virginia Commonwealth University

12. Bokhari, Yahya. DISCOVERING DRIVER MUTATIONS IN BIOLOGICAL DATA.

Degree: PhD, Computer Science, 2018, Virginia Commonwealth University

 <h3>Background</h3> Somatic mutations accumulate in human cells throughout life. Some may have no adverse consequences, but some of them may lead to cancer. A cancer… (more)

Subjects/Keywords: Bioinformatics; Optimization; Driver mutations; Bioinformatics

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APA (6th Edition):

Bokhari, Y. (2018). DISCOVERING DRIVER MUTATIONS IN BIOLOGICAL DATA. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/807X-D010 ; https://scholarscompass.vcu.edu/etd/5637

Chicago Manual of Style (16th Edition):

Bokhari, Yahya. “DISCOVERING DRIVER MUTATIONS IN BIOLOGICAL DATA.” 2018. Doctoral Dissertation, Virginia Commonwealth University. Accessed September 19, 2020. https://doi.org/10.25772/807X-D010 ; https://scholarscompass.vcu.edu/etd/5637.

MLA Handbook (7th Edition):

Bokhari, Yahya. “DISCOVERING DRIVER MUTATIONS IN BIOLOGICAL DATA.” 2018. Web. 19 Sep 2020.

Vancouver:

Bokhari Y. DISCOVERING DRIVER MUTATIONS IN BIOLOGICAL DATA. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2018. [cited 2020 Sep 19]. Available from: https://doi.org/10.25772/807X-D010 ; https://scholarscompass.vcu.edu/etd/5637.

Council of Science Editors:

Bokhari Y. DISCOVERING DRIVER MUTATIONS IN BIOLOGICAL DATA. [Doctoral Dissertation]. Virginia Commonwealth University; 2018. Available from: https://doi.org/10.25772/807X-D010 ; https://scholarscompass.vcu.edu/etd/5637


University of Cambridge

13. GARG, SUMEDHA. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.

Degree: PhD, 2019, University of Cambridge

 Primary aldosteronism (PA) accounts for 5-10% of all hypertension. One of the major causes of PA is sporadic formation of aldosterone-producing adenomas (APAs). These benign… (more)

Subjects/Keywords: aldosterone; adenoma; somatic mutations; hypertension

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APA (6th Edition):

GARG, S. (2019). Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289390

Chicago Manual of Style (16th Edition):

GARG, SUMEDHA. “Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.” 2019. Doctoral Dissertation, University of Cambridge. Accessed September 19, 2020. https://www.repository.cam.ac.uk/handle/1810/289390.

MLA Handbook (7th Edition):

GARG, SUMEDHA. “Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal.” 2019. Web. 19 Sep 2020.

Vancouver:

GARG S. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Sep 19]. Available from: https://www.repository.cam.ac.uk/handle/1810/289390.

Council of Science Editors:

GARG S. Role of two genes, CACNA1D and CADM1, with common or rare mutations in aldosterone producing adenomas of the adrenal. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289390


University College London (University of London)

14. Ahmed, Suhaib. An approach for the prevention of thalassaemia in Pakistan.

Degree: PhD, 1998, University College London (University of London)

 The basic aim of this thesis was to identify a suitable approach for prevention of thalassaemia in Pakistan. The overall carrier rate for β-thalassaemia was… (more)

Subjects/Keywords: 610; Mutations

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APA (6th Edition):

Ahmed, S. (1998). An approach for the prevention of thalassaemia in Pakistan. (Doctoral Dissertation). University College London (University of London). Retrieved from https://discovery.ucl.ac.uk/id/eprint/1317916/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299931

Chicago Manual of Style (16th Edition):

Ahmed, Suhaib. “An approach for the prevention of thalassaemia in Pakistan.” 1998. Doctoral Dissertation, University College London (University of London). Accessed September 19, 2020. https://discovery.ucl.ac.uk/id/eprint/1317916/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299931.

MLA Handbook (7th Edition):

Ahmed, Suhaib. “An approach for the prevention of thalassaemia in Pakistan.” 1998. Web. 19 Sep 2020.

Vancouver:

Ahmed S. An approach for the prevention of thalassaemia in Pakistan. [Internet] [Doctoral dissertation]. University College London (University of London); 1998. [cited 2020 Sep 19]. Available from: https://discovery.ucl.ac.uk/id/eprint/1317916/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299931.

Council of Science Editors:

Ahmed S. An approach for the prevention of thalassaemia in Pakistan. [Doctoral Dissertation]. University College London (University of London); 1998. Available from: https://discovery.ucl.ac.uk/id/eprint/1317916/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299931

15. Green, Clare Patricia. A molecular analysis of the tissue polarity gene prickle and associated transcripts in Drosophila melanogaster.

Degree: PhD, 1999, Open University

 The distinct patterns of bristles and hairs on the surface of an adult Drosophila indicate a planar polarity and reflect the polarity of the underlying… (more)

Subjects/Keywords: 572.8; Mutations

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APA (6th Edition):

Green, C. P. (1999). A molecular analysis of the tissue polarity gene prickle and associated transcripts in Drosophila melanogaster. (Doctoral Dissertation). Open University. Retrieved from http://oro.open.ac.uk/65356/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298071

Chicago Manual of Style (16th Edition):

Green, Clare Patricia. “A molecular analysis of the tissue polarity gene prickle and associated transcripts in Drosophila melanogaster.” 1999. Doctoral Dissertation, Open University. Accessed September 19, 2020. http://oro.open.ac.uk/65356/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298071.

MLA Handbook (7th Edition):

Green, Clare Patricia. “A molecular analysis of the tissue polarity gene prickle and associated transcripts in Drosophila melanogaster.” 1999. Web. 19 Sep 2020.

Vancouver:

Green CP. A molecular analysis of the tissue polarity gene prickle and associated transcripts in Drosophila melanogaster. [Internet] [Doctoral dissertation]. Open University; 1999. [cited 2020 Sep 19]. Available from: http://oro.open.ac.uk/65356/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298071.

Council of Science Editors:

Green CP. A molecular analysis of the tissue polarity gene prickle and associated transcripts in Drosophila melanogaster. [Doctoral Dissertation]. Open University; 1999. Available from: http://oro.open.ac.uk/65356/ ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.298071


University of Houston

16. Peng, Fen 1988-. Genetic Background and Environment Influence the Effects of Mutations in pykF and Help Reveal Mechanisms Underlying Their Benefit.

Degree: PhD, Biology, 2015, University of Houston

 Resolving the relationship between genotypic and their effects remains a central challenge in the study of adaptation. Although parallel mutations, a signature of adaptation, have… (more)

Subjects/Keywords: Parallel mutations; Pyruvate kinase; Epistasis

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APA (6th Edition):

Peng, F. 1. (2015). Genetic Background and Environment Influence the Effects of Mutations in pykF and Help Reveal Mechanisms Underlying Their Benefit. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/1978

Chicago Manual of Style (16th Edition):

Peng, Fen 1988-. “Genetic Background and Environment Influence the Effects of Mutations in pykF and Help Reveal Mechanisms Underlying Their Benefit.” 2015. Doctoral Dissertation, University of Houston. Accessed September 19, 2020. http://hdl.handle.net/10657/1978.

MLA Handbook (7th Edition):

Peng, Fen 1988-. “Genetic Background and Environment Influence the Effects of Mutations in pykF and Help Reveal Mechanisms Underlying Their Benefit.” 2015. Web. 19 Sep 2020.

Vancouver:

Peng F1. Genetic Background and Environment Influence the Effects of Mutations in pykF and Help Reveal Mechanisms Underlying Their Benefit. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10657/1978.

Council of Science Editors:

Peng F1. Genetic Background and Environment Influence the Effects of Mutations in pykF and Help Reveal Mechanisms Underlying Their Benefit. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/1978


University of Houston

17. -6149-7095. Causes and Consequences of the Evolution of Mutation Rate.

Degree: PhD, Biology, 2016, University of Houston

 As the ultimate source of all genetic variation, mutation is required for evolution. The mutation rate measures the rate at which mutations occur over time.… (more)

Subjects/Keywords: Evolution; Mutation rate; Mutations; Simulations

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

-6149-7095. (2016). Causes and Consequences of the Evolution of Mutation Rate. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/5400

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-6149-7095. “Causes and Consequences of the Evolution of Mutation Rate.” 2016. Doctoral Dissertation, University of Houston. Accessed September 19, 2020. http://hdl.handle.net/10657/5400.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-6149-7095. “Causes and Consequences of the Evolution of Mutation Rate.” 2016. Web. 19 Sep 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-6149-7095. Causes and Consequences of the Evolution of Mutation Rate. [Internet] [Doctoral dissertation]. University of Houston; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10657/5400.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-6149-7095. Causes and Consequences of the Evolution of Mutation Rate. [Doctoral Dissertation]. University of Houston; 2016. Available from: http://hdl.handle.net/10657/5400

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Illinois – Urbana-Champaign

18. Hou, Jack Pu. DawnRank: discovering personalized driver genes in cancer.

Degree: MS, Bioengineering, 2016, University of Illinois – Urbana-Champaign

 Large-scale cancer genomic studies have revealed that the genetic heterogeneity of the same type of cancer is greater than previously thought. A key question in… (more)

Subjects/Keywords: Computational Biology; Detection; Cancer; Mutations

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APA (6th Edition):

Hou, J. P. (2016). DawnRank: discovering personalized driver genes in cancer. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/95431

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hou, Jack Pu. “DawnRank: discovering personalized driver genes in cancer.” 2016. Thesis, University of Illinois – Urbana-Champaign. Accessed September 19, 2020. http://hdl.handle.net/2142/95431.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hou, Jack Pu. “DawnRank: discovering personalized driver genes in cancer.” 2016. Web. 19 Sep 2020.

Vancouver:

Hou JP. DawnRank: discovering personalized driver genes in cancer. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/2142/95431.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hou JP. DawnRank: discovering personalized driver genes in cancer. [Thesis]. University of Illinois – Urbana-Champaign; 2016. Available from: http://hdl.handle.net/2142/95431

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

19. Rahman, Sheikh Arafatur. Role of Histone H4 Mutations in DNA Repair Pathways.

Degree: MS, Medicine and Health Sciences, 2016, Louisiana State University

 Histone H3K79 methylation has been shown to play roles in different DNA repair pathways. Histone H4 residues serine 64 to threonine 80 surround histone H3K79… (more)

Subjects/Keywords: Histone H4 Mutations; DNA Repair

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rahman, S. A. (2016). Role of Histone H4 Mutations in DNA Repair Pathways. (Masters Thesis). Louisiana State University. Retrieved from etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565

Chicago Manual of Style (16th Edition):

Rahman, Sheikh Arafatur. “Role of Histone H4 Mutations in DNA Repair Pathways.” 2016. Masters Thesis, Louisiana State University. Accessed September 19, 2020. etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565.

MLA Handbook (7th Edition):

Rahman, Sheikh Arafatur. “Role of Histone H4 Mutations in DNA Repair Pathways.” 2016. Web. 19 Sep 2020.

Vancouver:

Rahman SA. Role of Histone H4 Mutations in DNA Repair Pathways. [Internet] [Masters thesis]. Louisiana State University; 2016. [cited 2020 Sep 19]. Available from: etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565.

Council of Science Editors:

Rahman SA. Role of Histone H4 Mutations in DNA Repair Pathways. [Masters Thesis]. Louisiana State University; 2016. Available from: etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565

20. Amamou, Leila. A la recherche d'une nouvelle identité artistique transnationale dans les spots publicitaires tunisiens (1994-2007) : oscillation entre esthétique de l'image et efficacité de la communication : In search of a new transnational artistic identity in tunisian advertising spots (1994-2007) : oscillation between aesthetic image and effective communication.

Degree: Docteur es, Arts et sciences de l'art, 2013, Paris 1

L'objectif de cette thèse est de montrer comment les créateurs des spots publicitaires en Tunisie de 1994 jusqu'à 2007 ont cherché à créer une nouvelle… (more)

Subjects/Keywords: Identité artistique; Tunisie; Mutations culturelles; Artistic identity; Tunisia; Cultural mutations; 730

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Amamou, L. (2013). A la recherche d'une nouvelle identité artistique transnationale dans les spots publicitaires tunisiens (1994-2007) : oscillation entre esthétique de l'image et efficacité de la communication : In search of a new transnational artistic identity in tunisian advertising spots (1994-2007) : oscillation between aesthetic image and effective communication. (Doctoral Dissertation). Paris 1. Retrieved from http://www.theses.fr/2013PA010625

Chicago Manual of Style (16th Edition):

Amamou, Leila. “A la recherche d'une nouvelle identité artistique transnationale dans les spots publicitaires tunisiens (1994-2007) : oscillation entre esthétique de l'image et efficacité de la communication : In search of a new transnational artistic identity in tunisian advertising spots (1994-2007) : oscillation between aesthetic image and effective communication.” 2013. Doctoral Dissertation, Paris 1. Accessed September 19, 2020. http://www.theses.fr/2013PA010625.

MLA Handbook (7th Edition):

Amamou, Leila. “A la recherche d'une nouvelle identité artistique transnationale dans les spots publicitaires tunisiens (1994-2007) : oscillation entre esthétique de l'image et efficacité de la communication : In search of a new transnational artistic identity in tunisian advertising spots (1994-2007) : oscillation between aesthetic image and effective communication.” 2013. Web. 19 Sep 2020.

Vancouver:

Amamou L. A la recherche d'une nouvelle identité artistique transnationale dans les spots publicitaires tunisiens (1994-2007) : oscillation entre esthétique de l'image et efficacité de la communication : In search of a new transnational artistic identity in tunisian advertising spots (1994-2007) : oscillation between aesthetic image and effective communication. [Internet] [Doctoral dissertation]. Paris 1; 2013. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2013PA010625.

Council of Science Editors:

Amamou L. A la recherche d'une nouvelle identité artistique transnationale dans les spots publicitaires tunisiens (1994-2007) : oscillation entre esthétique de l'image et efficacité de la communication : In search of a new transnational artistic identity in tunisian advertising spots (1994-2007) : oscillation between aesthetic image and effective communication. [Doctoral Dissertation]. Paris 1; 2013. Available from: http://www.theses.fr/2013PA010625

21. Bousard, Aurélie. Etude génétique et épigénétique de l'adénocarcinome du rein à cellules claires : Genetic and epigenetic study of clear cell renal cell carcinoma.

Degree: Docteur es, Génétique et Epigénétique, 2015, Evry-Val d'Essonne

Ce travail de thèse comporte trois projets visant à approfondir la caractérisation moléculaire des adénocarcinomes du rein à cellules claires (ccRCC) et à en améliorer… (more)

Subjects/Keywords: Mutations de l'ADN non-codant; Mutations de l'ADN non-codant

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APA (6th Edition):

Bousard, A. (2015). Etude génétique et épigénétique de l'adénocarcinome du rein à cellules claires : Genetic and epigenetic study of clear cell renal cell carcinoma. (Doctoral Dissertation). Evry-Val d'Essonne. Retrieved from http://www.theses.fr/2015EVRY0004

Chicago Manual of Style (16th Edition):

Bousard, Aurélie. “Etude génétique et épigénétique de l'adénocarcinome du rein à cellules claires : Genetic and epigenetic study of clear cell renal cell carcinoma.” 2015. Doctoral Dissertation, Evry-Val d'Essonne. Accessed September 19, 2020. http://www.theses.fr/2015EVRY0004.

MLA Handbook (7th Edition):

Bousard, Aurélie. “Etude génétique et épigénétique de l'adénocarcinome du rein à cellules claires : Genetic and epigenetic study of clear cell renal cell carcinoma.” 2015. Web. 19 Sep 2020.

Vancouver:

Bousard A. Etude génétique et épigénétique de l'adénocarcinome du rein à cellules claires : Genetic and epigenetic study of clear cell renal cell carcinoma. [Internet] [Doctoral dissertation]. Evry-Val d'Essonne; 2015. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2015EVRY0004.

Council of Science Editors:

Bousard A. Etude génétique et épigénétique de l'adénocarcinome du rein à cellules claires : Genetic and epigenetic study of clear cell renal cell carcinoma. [Doctoral Dissertation]. Evry-Val d'Essonne; 2015. Available from: http://www.theses.fr/2015EVRY0004

22. Crahé, Maxime-Morvan. Le breton de Languidic : étude phonétique, morphologique et syntaxique d'un sous-dialecte du breton vannetais : The Breton Micro-dialect of Languidic : a phonetical, morphlogical and syntaxic study of a variety of the Breton Vannetais.

Degree: Docteur es, Breton et celtique, 2013, Rennes 2

Cette étude apporte un nouveau corpus à la description du paysage linguistique de Basse-Bretagne et participe de ce fait à une meilleure connaissance de la… (more)

Subjects/Keywords: Oralite; Dialectologie; Mutations consonantiques; Orality; Dialectology; Consonant mutations; 491.6

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APA (6th Edition):

Crahé, M. (2013). Le breton de Languidic : étude phonétique, morphologique et syntaxique d'un sous-dialecte du breton vannetais : The Breton Micro-dialect of Languidic : a phonetical, morphlogical and syntaxic study of a variety of the Breton Vannetais. (Doctoral Dissertation). Rennes 2. Retrieved from http://www.theses.fr/2013REN20062

Chicago Manual of Style (16th Edition):

Crahé, Maxime-Morvan. “Le breton de Languidic : étude phonétique, morphologique et syntaxique d'un sous-dialecte du breton vannetais : The Breton Micro-dialect of Languidic : a phonetical, morphlogical and syntaxic study of a variety of the Breton Vannetais.” 2013. Doctoral Dissertation, Rennes 2. Accessed September 19, 2020. http://www.theses.fr/2013REN20062.

MLA Handbook (7th Edition):

Crahé, Maxime-Morvan. “Le breton de Languidic : étude phonétique, morphologique et syntaxique d'un sous-dialecte du breton vannetais : The Breton Micro-dialect of Languidic : a phonetical, morphlogical and syntaxic study of a variety of the Breton Vannetais.” 2013. Web. 19 Sep 2020.

Vancouver:

Crahé M. Le breton de Languidic : étude phonétique, morphologique et syntaxique d'un sous-dialecte du breton vannetais : The Breton Micro-dialect of Languidic : a phonetical, morphlogical and syntaxic study of a variety of the Breton Vannetais. [Internet] [Doctoral dissertation]. Rennes 2; 2013. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2013REN20062.

Council of Science Editors:

Crahé M. Le breton de Languidic : étude phonétique, morphologique et syntaxique d'un sous-dialecte du breton vannetais : The Breton Micro-dialect of Languidic : a phonetical, morphlogical and syntaxic study of a variety of the Breton Vannetais. [Doctoral Dissertation]. Rennes 2; 2013. Available from: http://www.theses.fr/2013REN20062


University of Edinburgh

23. Novo, Sonia Marisa. Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244.

Degree: PhD, 2013, University of Edinburgh

 Colorectal cancer is the second most common cause of cancer death in Western Europe and North America. Current therapies are largely ineffective and are associated… (more)

Subjects/Keywords: 616.99; MEK1/2; MAPK pathway; KRAS mutations; BRAF mutations; proliferation; apoptosis

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APA (6th Edition):

Novo, S. M. (2013). Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/11778

Chicago Manual of Style (16th Edition):

Novo, Sonia Marisa. “Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed September 19, 2020. http://hdl.handle.net/1842/11778.

MLA Handbook (7th Edition):

Novo, Sonia Marisa. “Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244.” 2013. Web. 19 Sep 2020.

Vancouver:

Novo SM. Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1842/11778.

Council of Science Editors:

Novo SM. Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/11778

24. Michot, Pauline. Utilisation des séquences de génome complet pour l'identification de mutations délétères responsables d'anomalies génétiques récessives chez le bovin. : Use of whole genome sequence data to identify new deleterious mutations associated to genetic defects in French dairy and beef cattle breeds.

Degree: Docteur es, Génétique animale, 2017, Université Paris-Saclay (ComUE)

L’effectif génétique réduit des races bovines entraîne une augmentation de consanguinité de l’ordre de 1% par génération et une forte dérive génétique. Cette évolution favorise… (more)

Subjects/Keywords: Génomique; Mutations délétères; Anomalies; Bovins; Genomic; Deleterious mutations; Anomalies; Bovine; 576

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APA (6th Edition):

Michot, P. (2017). Utilisation des séquences de génome complet pour l'identification de mutations délétères responsables d'anomalies génétiques récessives chez le bovin. : Use of whole genome sequence data to identify new deleterious mutations associated to genetic defects in French dairy and beef cattle breeds. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2017SACLA011

Chicago Manual of Style (16th Edition):

Michot, Pauline. “Utilisation des séquences de génome complet pour l'identification de mutations délétères responsables d'anomalies génétiques récessives chez le bovin. : Use of whole genome sequence data to identify new deleterious mutations associated to genetic defects in French dairy and beef cattle breeds.” 2017. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed September 19, 2020. http://www.theses.fr/2017SACLA011.

MLA Handbook (7th Edition):

Michot, Pauline. “Utilisation des séquences de génome complet pour l'identification de mutations délétères responsables d'anomalies génétiques récessives chez le bovin. : Use of whole genome sequence data to identify new deleterious mutations associated to genetic defects in French dairy and beef cattle breeds.” 2017. Web. 19 Sep 2020.

Vancouver:

Michot P. Utilisation des séquences de génome complet pour l'identification de mutations délétères responsables d'anomalies génétiques récessives chez le bovin. : Use of whole genome sequence data to identify new deleterious mutations associated to genetic defects in French dairy and beef cattle breeds. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2017. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2017SACLA011.

Council of Science Editors:

Michot P. Utilisation des séquences de génome complet pour l'identification de mutations délétères responsables d'anomalies génétiques récessives chez le bovin. : Use of whole genome sequence data to identify new deleterious mutations associated to genetic defects in French dairy and beef cattle breeds. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2017. Available from: http://www.theses.fr/2017SACLA011

25. Krasovec, Marc. Estimation des taux de mutation : implications pour la diversification et l'évolution du phytoplancton eucaryote : Estimation of mutation rates : implications for diversification and evolution of eukaryotic phytoplankton.

Degree: Docteur es, Evolution moléculaire, 2016, Université Pierre et Marie Curie – Paris VI

Les mutations sont la principale source de diversité sur laquelle agit la sélection pour permettre aux espèces de s'adapter. Les études de l'effet des mutations(more)

Subjects/Keywords: Taux de mutation; Phytoplancton; Fitness; Mutations spontanées; Accumulation de mutations; Evolution eucaryote; Mutation rate; Spontaneous mutations; Eucaryote evolution; 579.8

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APA (6th Edition):

Krasovec, M. (2016). Estimation des taux de mutation : implications pour la diversification et l'évolution du phytoplancton eucaryote : Estimation of mutation rates : implications for diversification and evolution of eukaryotic phytoplankton. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066371

Chicago Manual of Style (16th Edition):

Krasovec, Marc. “Estimation des taux de mutation : implications pour la diversification et l'évolution du phytoplancton eucaryote : Estimation of mutation rates : implications for diversification and evolution of eukaryotic phytoplankton.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed September 19, 2020. http://www.theses.fr/2016PA066371.

MLA Handbook (7th Edition):

Krasovec, Marc. “Estimation des taux de mutation : implications pour la diversification et l'évolution du phytoplancton eucaryote : Estimation of mutation rates : implications for diversification and evolution of eukaryotic phytoplankton.” 2016. Web. 19 Sep 2020.

Vancouver:

Krasovec M. Estimation des taux de mutation : implications pour la diversification et l'évolution du phytoplancton eucaryote : Estimation of mutation rates : implications for diversification and evolution of eukaryotic phytoplankton. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2016PA066371.

Council of Science Editors:

Krasovec M. Estimation des taux de mutation : implications pour la diversification et l'évolution du phytoplancton eucaryote : Estimation of mutation rates : implications for diversification and evolution of eukaryotic phytoplankton. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066371


University of California – Santa Cruz

26. Radenbaugh, Amie. The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data.

Degree: Biomolecular Engineering and Bioinformatics, 2015, University of California – Santa Cruz

 Much of our current understanding of cancer has come from investigating how normal cells are transformed into malignant cancers through the stepwise acquisition of somatic… (more)

Subjects/Keywords: Bioinformatics; editing; mutations; NGS; RNA; somatic; variants

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APA (6th Edition):

Radenbaugh, A. (2015). The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data. (Thesis). University of California – Santa Cruz. Retrieved from http://www.escholarship.org/uc/item/0dt1w1fx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radenbaugh, Amie. “The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data.” 2015. Thesis, University of California – Santa Cruz. Accessed September 19, 2020. http://www.escholarship.org/uc/item/0dt1w1fx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radenbaugh, Amie. “The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data.” 2015. Web. 19 Sep 2020.

Vancouver:

Radenbaugh A. The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data. [Internet] [Thesis]. University of California – Santa Cruz; 2015. [cited 2020 Sep 19]. Available from: http://www.escholarship.org/uc/item/0dt1w1fx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radenbaugh A. The Identification and Characterization of Alterations to DNA and RNA in Cancer Using Next-Generation Sequencing Data. [Thesis]. University of California – Santa Cruz; 2015. Available from: http://www.escholarship.org/uc/item/0dt1w1fx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

27. Pyne, Michael Terry. Characterization of intronic variants in BRCA1 and BRCA2;.

Degree: MS;, Pathology;, 2000, University of Utah

 Approximately 10% of American women develop breast cancer during their lifetimes. Of the cases of breast cancer, about 10% are attributed to a genetic trait;… (more)

Subjects/Keywords: Breast Cancer; Mutations

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APA (6th Edition):

Pyne, M. T. (2000). Characterization of intronic variants in BRCA1 and BRCA2;. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/880/rec/144

Chicago Manual of Style (16th Edition):

Pyne, Michael Terry. “Characterization of intronic variants in BRCA1 and BRCA2;.” 2000. Masters Thesis, University of Utah. Accessed September 19, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/880/rec/144.

MLA Handbook (7th Edition):

Pyne, Michael Terry. “Characterization of intronic variants in BRCA1 and BRCA2;.” 2000. Web. 19 Sep 2020.

Vancouver:

Pyne MT. Characterization of intronic variants in BRCA1 and BRCA2;. [Internet] [Masters thesis]. University of Utah; 2000. [cited 2020 Sep 19]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/880/rec/144.

Council of Science Editors:

Pyne MT. Characterization of intronic variants in BRCA1 and BRCA2;. [Masters Thesis]. University of Utah; 2000. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/880/rec/144


University of Utah

28. Li, Xinghai. The role of protein phosphatase 2A in regulating Wnt signaling and apoptosis;.

Degree: PhD, Oncological Sciences;, 2001, University of Utah

 Protein phosphatase 2A (PP2A) is a major serine/threonine-specific phosphatase and regulates a significant array of cellular events. This dissertation primarily describes the novel role of… (more)

Subjects/Keywords: Gene Mutations; Neuroblastoma

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APA (6th Edition):

Li, X. (2001). The role of protein phosphatase 2A in regulating Wnt signaling and apoptosis;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1157/rec/1387

Chicago Manual of Style (16th Edition):

Li, Xinghai. “The role of protein phosphatase 2A in regulating Wnt signaling and apoptosis;.” 2001. Doctoral Dissertation, University of Utah. Accessed September 19, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1157/rec/1387.

MLA Handbook (7th Edition):

Li, Xinghai. “The role of protein phosphatase 2A in regulating Wnt signaling and apoptosis;.” 2001. Web. 19 Sep 2020.

Vancouver:

Li X. The role of protein phosphatase 2A in regulating Wnt signaling and apoptosis;. [Internet] [Doctoral dissertation]. University of Utah; 2001. [cited 2020 Sep 19]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1157/rec/1387.

Council of Science Editors:

Li X. The role of protein phosphatase 2A in regulating Wnt signaling and apoptosis;. [Doctoral Dissertation]. University of Utah; 2001. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1157/rec/1387


University of Utah

29. Dohrmann, Paul Richard. Transcriptional regulation by the ACE2 and SWI5 activators in yeast;.

Degree: PhD, Pathology;, 1994, University of Utah

 Two independent transcriptional pathways regulated by the ACE2 and SWI5 genes have been identified in the yeast Saccharomyces cerevisiae. The CTSl and the HO pathways… (more)

Subjects/Keywords: Genetics; Mutations; RNA

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APA (6th Edition):

Dohrmann, P. R. (1994). Transcriptional regulation by the ACE2 and SWI5 activators in yeast;. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1343/rec/1416

Chicago Manual of Style (16th Edition):

Dohrmann, Paul Richard. “Transcriptional regulation by the ACE2 and SWI5 activators in yeast;.” 1994. Doctoral Dissertation, University of Utah. Accessed September 19, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1343/rec/1416.

MLA Handbook (7th Edition):

Dohrmann, Paul Richard. “Transcriptional regulation by the ACE2 and SWI5 activators in yeast;.” 1994. Web. 19 Sep 2020.

Vancouver:

Dohrmann PR. Transcriptional regulation by the ACE2 and SWI5 activators in yeast;. [Internet] [Doctoral dissertation]. University of Utah; 1994. [cited 2020 Sep 19]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1343/rec/1416.

Council of Science Editors:

Dohrmann PR. Transcriptional regulation by the ACE2 and SWI5 activators in yeast;. [Doctoral Dissertation]. University of Utah; 1994. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd1/id/1343/rec/1416

30. Agalianou, Niki. Επιπολασμός γονιδιακών μεταλλάξεων σε ασθενείς με θηλώδες καρκίνωμα του θυρεοειδούς και έκβαση της νόσου σε ελληνικό πληθυσμό.

Degree: 2019, University of Patras; Πανεπιστήμιο Πατρών

The incidence of BRAF V600E mutation in patients with papillary thyroid carcinoma (PTC) ranges between 38-91% (mean 45% ) and correlates with aggressive behavior. There… (more)

Subjects/Keywords: Θηλώδες καρκίνωμα; Θυρεοειδής; Μεταλλάξεις; BRAF mutations

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APA (6th Edition):

Agalianou, N. (2019). Επιπολασμός γονιδιακών μεταλλάξεων σε ασθενείς με θηλώδες καρκίνωμα του θυρεοειδούς και έκβαση της νόσου σε ελληνικό πληθυσμό. (Thesis). University of Patras; Πανεπιστήμιο Πατρών. Retrieved from http://hdl.handle.net/10442/hedi/45825

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Agalianou, Niki. “Επιπολασμός γονιδιακών μεταλλάξεων σε ασθενείς με θηλώδες καρκίνωμα του θυρεοειδούς και έκβαση της νόσου σε ελληνικό πληθυσμό.” 2019. Thesis, University of Patras; Πανεπιστήμιο Πατρών. Accessed September 19, 2020. http://hdl.handle.net/10442/hedi/45825.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Agalianou, Niki. “Επιπολασμός γονιδιακών μεταλλάξεων σε ασθενείς με θηλώδες καρκίνωμα του θυρεοειδούς και έκβαση της νόσου σε ελληνικό πληθυσμό.” 2019. Web. 19 Sep 2020.

Vancouver:

Agalianou N. Επιπολασμός γονιδιακών μεταλλάξεων σε ασθενείς με θηλώδες καρκίνωμα του θυρεοειδούς και έκβαση της νόσου σε ελληνικό πληθυσμό. [Internet] [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2019. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10442/hedi/45825.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Agalianou N. Επιπολασμός γονιδιακών μεταλλάξεων σε ασθενείς με θηλώδες καρκίνωμα του θυρεοειδούς και έκβαση της νόσου σε ελληνικό πληθυσμό. [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2019. Available from: http://hdl.handle.net/10442/hedi/45825

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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