Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(mouse models). Showing records 1 – 30 of 213 total matches.

[1] [2] [3] [4] [5] [6] [7] [8]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


Universiteit Utrecht

1. Sanders, A. BRCA1 breast cancer: metastasis en mouse models.

Degree: 2012, Universiteit Utrecht

 The BRCA1 gene is a tumour suppressor gene frequently mutated in heritable forms of breast cancers. BRCA1 deficient tumours show a basal-like phenotype. Attempts have… (more)

Subjects/Keywords: BRCA1; breast cancer; metastasis; mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sanders, A. (2012). BRCA1 breast cancer: metastasis en mouse models. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/256356

Chicago Manual of Style (16th Edition):

Sanders, A. “BRCA1 breast cancer: metastasis en mouse models.” 2012. Masters Thesis, Universiteit Utrecht. Accessed August 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/256356.

MLA Handbook (7th Edition):

Sanders, A. “BRCA1 breast cancer: metastasis en mouse models.” 2012. Web. 18 Aug 2019.

Vancouver:

Sanders A. BRCA1 breast cancer: metastasis en mouse models. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2019 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/256356.

Council of Science Editors:

Sanders A. BRCA1 breast cancer: metastasis en mouse models. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/256356


University of Southern California

2. Bhatt, Brinda. Influence of genetic background on the effect of traffic-related air pollution on obesity phenotypes.

Degree: MS, Molecular Microbiology and Immunology, 2013, University of Southern California

 Obesity is defined as the state that occurs when there is insufficient energy expenditure to a high caloric intake, resulting in increased BMI; and it… (more)

Subjects/Keywords: genetics; air pollution; obesity; hmdp; mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bhatt, B. (2013). Influence of genetic background on the effect of traffic-related air pollution on obesity phenotypes. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298459/rec/3479

Chicago Manual of Style (16th Edition):

Bhatt, Brinda. “Influence of genetic background on the effect of traffic-related air pollution on obesity phenotypes.” 2013. Masters Thesis, University of Southern California. Accessed August 18, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298459/rec/3479.

MLA Handbook (7th Edition):

Bhatt, Brinda. “Influence of genetic background on the effect of traffic-related air pollution on obesity phenotypes.” 2013. Web. 18 Aug 2019.

Vancouver:

Bhatt B. Influence of genetic background on the effect of traffic-related air pollution on obesity phenotypes. [Internet] [Masters thesis]. University of Southern California; 2013. [cited 2019 Aug 18]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298459/rec/3479.

Council of Science Editors:

Bhatt B. Influence of genetic background on the effect of traffic-related air pollution on obesity phenotypes. [Masters Thesis]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/298459/rec/3479


Duke University

3. Lopez, Giselle Yvette. Biochemical Characterization and Genetic Modeling of Glioma-Associated Mutations in Isocitrate Dehydrogenases.

Degree: 2014, Duke University

  Gliomas are the most common tumors of the central nervous system. Our lab recently identified mutations in IDH1 and IDH2 as occurring frequently in… (more)

Subjects/Keywords: Pathology; gliomas; isocitrate dehydrogenase; mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lopez, G. Y. (2014). Biochemical Characterization and Genetic Modeling of Glioma-Associated Mutations in Isocitrate Dehydrogenases. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/8628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lopez, Giselle Yvette. “Biochemical Characterization and Genetic Modeling of Glioma-Associated Mutations in Isocitrate Dehydrogenases. ” 2014. Thesis, Duke University. Accessed August 18, 2019. http://hdl.handle.net/10161/8628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lopez, Giselle Yvette. “Biochemical Characterization and Genetic Modeling of Glioma-Associated Mutations in Isocitrate Dehydrogenases. ” 2014. Web. 18 Aug 2019.

Vancouver:

Lopez GY. Biochemical Characterization and Genetic Modeling of Glioma-Associated Mutations in Isocitrate Dehydrogenases. [Internet] [Thesis]. Duke University; 2014. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10161/8628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lopez GY. Biochemical Characterization and Genetic Modeling of Glioma-Associated Mutations in Isocitrate Dehydrogenases. [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/8628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

4. Sparks, Erin Elizabeth. Notch Signaling is Essential to Modulate Intrahepatic Bile Duct Structure.

Degree: PhD, Cell and Developmental Biology, 2011, Vanderbilt University

 Cholangiopathies, or diseases which affect the biliary epithelial cells of the liver, are an important health concern in the United States. Specifically, the diagnosis of… (more)

Subjects/Keywords: Cholangiopathies; Alagille; Resin Casting; Mouse Models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sparks, E. E. (2011). Notch Signaling is Essential to Modulate Intrahepatic Bile Duct Structure. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03102011-141048/ ;

Chicago Manual of Style (16th Edition):

Sparks, Erin Elizabeth. “Notch Signaling is Essential to Modulate Intrahepatic Bile Duct Structure.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed August 18, 2019. http://etd.library.vanderbilt.edu/available/etd-03102011-141048/ ;.

MLA Handbook (7th Edition):

Sparks, Erin Elizabeth. “Notch Signaling is Essential to Modulate Intrahepatic Bile Duct Structure.” 2011. Web. 18 Aug 2019.

Vancouver:

Sparks EE. Notch Signaling is Essential to Modulate Intrahepatic Bile Duct Structure. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Aug 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-03102011-141048/ ;.

Council of Science Editors:

Sparks EE. Notch Signaling is Essential to Modulate Intrahepatic Bile Duct Structure. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu/available/etd-03102011-141048/ ;


University of Toronto

5. Kusdaya, Nayasta Ademmana. Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation.

Degree: 2015, University of Toronto

Genomic analysis of breast cancer (BC) has revealed the existence of recurrent copy number losses and mutations in GATA3 and RUNX1. Interestingly, these are predominantly… (more)

Subjects/Keywords: Breast Cancer; Gata3; Mouse Models; Runx1; 0369

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kusdaya, N. A. (2015). Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/94523

Chicago Manual of Style (16th Edition):

Kusdaya, Nayasta Ademmana. “Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation.” 2015. Masters Thesis, University of Toronto. Accessed August 18, 2019. http://hdl.handle.net/1807/94523.

MLA Handbook (7th Edition):

Kusdaya, Nayasta Ademmana. “Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation.” 2015. Web. 18 Aug 2019.

Vancouver:

Kusdaya NA. Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1807/94523.

Council of Science Editors:

Kusdaya NA. Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/94523


University of California – San Francisco

6. Westcott, Peter Maxwell Kienitz. Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse.

Degree: Pharmaceutical Sciences and Pharmacogenomics, 2015, University of California – San Francisco

Mouse models have been an essential tool for cancer research for many decades, and have seen application to ever more diverse and complex questions with… (more)

Subjects/Keywords: Biology; Genetics; Cancer; Genomics; Kras; Mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Westcott, P. M. K. (2015). Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/71f4t6xt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Westcott, Peter Maxwell Kienitz. “Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse.” 2015. Thesis, University of California – San Francisco. Accessed August 18, 2019. http://www.escholarship.org/uc/item/71f4t6xt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Westcott, Peter Maxwell Kienitz. “Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse.” 2015. Web. 18 Aug 2019.

Vancouver:

Westcott PMK. Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2019 Aug 18]. Available from: http://www.escholarship.org/uc/item/71f4t6xt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Westcott PMK. Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/71f4t6xt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

7. Pederick, Daniel Tyler. Disruption of Cell-Cell Adhesion Codes Underlies the Unique X-linked Inheritance Pattern of Protocadherin 19 Girls Clustering Epilepsy.

Degree: 2017, University of Adelaide

 Epilepsy is a disease of the central nervous system (CNS) caused by increased neuronal activity resulting in seizures and often loss of consciousness. Epilepsy can… (more)

Subjects/Keywords: Epilepsy; mouse models; cortical development; adhesion codes

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pederick, D. T. (2017). Disruption of Cell-Cell Adhesion Codes Underlies the Unique X-linked Inheritance Pattern of Protocadherin 19 Girls Clustering Epilepsy. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/119088

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pederick, Daniel Tyler. “Disruption of Cell-Cell Adhesion Codes Underlies the Unique X-linked Inheritance Pattern of Protocadherin 19 Girls Clustering Epilepsy.” 2017. Thesis, University of Adelaide. Accessed August 18, 2019. http://hdl.handle.net/2440/119088.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pederick, Daniel Tyler. “Disruption of Cell-Cell Adhesion Codes Underlies the Unique X-linked Inheritance Pattern of Protocadherin 19 Girls Clustering Epilepsy.” 2017. Web. 18 Aug 2019.

Vancouver:

Pederick DT. Disruption of Cell-Cell Adhesion Codes Underlies the Unique X-linked Inheritance Pattern of Protocadherin 19 Girls Clustering Epilepsy. [Internet] [Thesis]. University of Adelaide; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/2440/119088.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pederick DT. Disruption of Cell-Cell Adhesion Codes Underlies the Unique X-linked Inheritance Pattern of Protocadherin 19 Girls Clustering Epilepsy. [Thesis]. University of Adelaide; 2017. Available from: http://hdl.handle.net/2440/119088

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

8. Kusdaya, Nayasta Ademmana. Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation.

Degree: 2015, University of Toronto

Genomic analysis of breast cancer (BC) has revealed the existence of recurrent copy number losses and mutations in GATA3 and RUNX1. Interestingly, these are predominantly… (more)

Subjects/Keywords: Breast Cancer; Gata3; Mouse Models; Runx1; 0369

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kusdaya, N. A. (2015). Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/80292

Chicago Manual of Style (16th Edition):

Kusdaya, Nayasta Ademmana. “Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation.” 2015. Masters Thesis, University of Toronto. Accessed August 18, 2019. http://hdl.handle.net/1807/80292.

MLA Handbook (7th Edition):

Kusdaya, Nayasta Ademmana. “Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation.” 2015. Web. 18 Aug 2019.

Vancouver:

Kusdaya NA. Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1807/80292.

Council of Science Editors:

Kusdaya NA. Genetic Analysis of Gata3 and Runx1 in Mammary Tumor Formation. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/80292


Vanderbilt University

9. Siemann, Justin Kyle. Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications.

Degree: PhD, Neuroscience, 2015, Vanderbilt University

 NEUROSCIENCE Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications Justin Kyle Siemann Dissertation under the direction of Mark T. Wallace Multisensory integration… (more)

Subjects/Keywords: mouse models; serotonin; mouse behavior; visual processing; autism spectrum disorders; multisensory integration; auditory processing

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Siemann, J. K. (2015). Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12272015-153630/ ;

Chicago Manual of Style (16th Edition):

Siemann, Justin Kyle. “Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed August 18, 2019. http://etd.library.vanderbilt.edu/available/etd-12272015-153630/ ;.

MLA Handbook (7th Edition):

Siemann, Justin Kyle. “Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications.” 2015. Web. 18 Aug 2019.

Vancouver:

Siemann JK. Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Aug 18]. Available from: http://etd.library.vanderbilt.edu/available/etd-12272015-153630/ ;.

Council of Science Editors:

Siemann JK. Evaluating Multisensory Processing in the Mouse Model: Clinical and Translational Implications. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-12272015-153630/ ;


Dalhousie University

10. Fraser, Leanne M. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.

Degree: PhD, Department of Psychology and Neuroscience, 2013, Dalhousie University

 The triple transgenic (3xTg-AD) mouse model of Alzheimer’s disease (AD) possesses three transgenes that lead to the development of amyloid-beta (A?) plaques (APPswe, PS1M146V) and… (more)

Subjects/Keywords: "Transgenic mice"; "Alzheimer's disease"; "Mouse models"; "Behaviour"; "Memory"; "Motor coordination"

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fraser, L. M. (2013). Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. (Doctoral Dissertation). Dalhousie University. Retrieved from http://hdl.handle.net/10222/28080

Chicago Manual of Style (16th Edition):

Fraser, Leanne M. “Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.” 2013. Doctoral Dissertation, Dalhousie University. Accessed August 18, 2019. http://hdl.handle.net/10222/28080.

MLA Handbook (7th Edition):

Fraser, Leanne M. “Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study.” 2013. Web. 18 Aug 2019.

Vancouver:

Fraser LM. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. [Internet] [Doctoral dissertation]. Dalhousie University; 2013. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10222/28080.

Council of Science Editors:

Fraser LM. Locomotor behaviour, emotionality, and cognition in the 3xTg-AD mouse model of Alzheimer's disease: A cross-sectional study. [Doctoral Dissertation]. Dalhousie University; 2013. Available from: http://hdl.handle.net/10222/28080

11. Benesch, Matthew GK. Autotaxin and Tumor-Promoting Inflammation.

Degree: PhD, Department of Biochemistry, 2015, University of Alberta

 Autotaxin is a secreted enzyme that produces most of the extracellular lysophosphatidate from lysophosphatidylcholine, the most abundant phospholipid in plasma. Lysophosphatidate mediates many physiological and… (more)

Subjects/Keywords: mouse models; autotaxin inhibition; breast cancer; thyroid cancer; lysophosphatidate

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Benesch, M. G. (2015). Autotaxin and Tumor-Promoting Inflammation. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/h128nh66c

Chicago Manual of Style (16th Edition):

Benesch, Matthew GK. “Autotaxin and Tumor-Promoting Inflammation.” 2015. Doctoral Dissertation, University of Alberta. Accessed August 18, 2019. https://era.library.ualberta.ca/files/h128nh66c.

MLA Handbook (7th Edition):

Benesch, Matthew GK. “Autotaxin and Tumor-Promoting Inflammation.” 2015. Web. 18 Aug 2019.

Vancouver:

Benesch MG. Autotaxin and Tumor-Promoting Inflammation. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2019 Aug 18]. Available from: https://era.library.ualberta.ca/files/h128nh66c.

Council of Science Editors:

Benesch MG. Autotaxin and Tumor-Promoting Inflammation. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/h128nh66c


The Ohio State University

12. Berman-Booty, Lisa Danielle. Suppression of Carcinogenesis and Tumor Progression by an Energy Restriction-Mimetic Agent in Murine Models of Prostate Cancer.

Degree: PhD, Veterinary Biosciences, 2013, The Ohio State University

 Cancer cells preferentially utilize glycolysis to generate energy even in the presence of sufficient oxygen for oxidative phosphorylation. This shift in energy metabolism, termed the… (more)

Subjects/Keywords: Molecular Biology; energy restriction-mimetic agent; prostate cancer; mouse models; TRAMP

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Berman-Booty, L. D. (2013). Suppression of Carcinogenesis and Tumor Progression by an Energy Restriction-Mimetic Agent in Murine Models of Prostate Cancer. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1365512650

Chicago Manual of Style (16th Edition):

Berman-Booty, Lisa Danielle. “Suppression of Carcinogenesis and Tumor Progression by an Energy Restriction-Mimetic Agent in Murine Models of Prostate Cancer.” 2013. Doctoral Dissertation, The Ohio State University. Accessed August 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1365512650.

MLA Handbook (7th Edition):

Berman-Booty, Lisa Danielle. “Suppression of Carcinogenesis and Tumor Progression by an Energy Restriction-Mimetic Agent in Murine Models of Prostate Cancer.” 2013. Web. 18 Aug 2019.

Vancouver:

Berman-Booty LD. Suppression of Carcinogenesis and Tumor Progression by an Energy Restriction-Mimetic Agent in Murine Models of Prostate Cancer. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Aug 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1365512650.

Council of Science Editors:

Berman-Booty LD. Suppression of Carcinogenesis and Tumor Progression by an Energy Restriction-Mimetic Agent in Murine Models of Prostate Cancer. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1365512650


The Ohio State University

13. Chaffee, Beth K. Preclinical Modeling of Musculoskeletal Cancer.

Degree: PhD, Comparative and Veterinary Medicine, 2013, The Ohio State University

 Preclinical animal models serve as invaluable tools in the investigation of disease and the development of novel therapeutic strategies to prevent and treat disease. Osteosarcoma… (more)

Subjects/Keywords: Biomedical Research; Oncology; osteosarcoma; metastasis; radiation; mouse models; bone

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chaffee, B. K. (2013). Preclinical Modeling of Musculoskeletal Cancer. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1376844544

Chicago Manual of Style (16th Edition):

Chaffee, Beth K. “Preclinical Modeling of Musculoskeletal Cancer.” 2013. Doctoral Dissertation, The Ohio State University. Accessed August 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1376844544.

MLA Handbook (7th Edition):

Chaffee, Beth K. “Preclinical Modeling of Musculoskeletal Cancer.” 2013. Web. 18 Aug 2019.

Vancouver:

Chaffee BK. Preclinical Modeling of Musculoskeletal Cancer. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Aug 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1376844544.

Council of Science Editors:

Chaffee BK. Preclinical Modeling of Musculoskeletal Cancer. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1376844544


The Ohio State University

14. Pringle, Daphne R. Mechanisms of Follicular Thyroid Cancer Development and Progression in the Context of Dysregulated PKA.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2013, The Ohio State University

 Thyroid cancer is the most commone endocrine malignancy in the population and incidence rates continue to rise. The two most common types of thyroid cancer,… (more)

Subjects/Keywords: Molecular Biology; Oncology; Genetics; Thyroid cancer, PKA, mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pringle, D. R. (2013). Mechanisms of Follicular Thyroid Cancer Development and Progression in the Context of Dysregulated PKA. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1373732842

Chicago Manual of Style (16th Edition):

Pringle, Daphne R. “Mechanisms of Follicular Thyroid Cancer Development and Progression in the Context of Dysregulated PKA.” 2013. Doctoral Dissertation, The Ohio State University. Accessed August 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1373732842.

MLA Handbook (7th Edition):

Pringle, Daphne R. “Mechanisms of Follicular Thyroid Cancer Development and Progression in the Context of Dysregulated PKA.” 2013. Web. 18 Aug 2019.

Vancouver:

Pringle DR. Mechanisms of Follicular Thyroid Cancer Development and Progression in the Context of Dysregulated PKA. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Aug 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1373732842.

Council of Science Editors:

Pringle DR. Mechanisms of Follicular Thyroid Cancer Development and Progression in the Context of Dysregulated PKA. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1373732842


NSYSU

15. Su, Chuan-Siang. APC Haploinsufficiency Cooperates With Kras and p53 Loss to Accelerate High Grade Glioblastoma Formation in Mice.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

 Glioblastoma multiforme (GBM) is the most common and deadest of brain tumor arising from glial cells. In contrast to other major cancers, decades of clinical… (more)

Subjects/Keywords: biomarkers; Mouse models of glioblastoma multiforme; APC haploinsufficiency; giant cells; malignancy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Su, C. (2015). APC Haploinsufficiency Cooperates With Kras and p53 Loss to Accelerate High Grade Glioblastoma Formation in Mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0630115-114124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Chuan-Siang. “APC Haploinsufficiency Cooperates With Kras and p53 Loss to Accelerate High Grade Glioblastoma Formation in Mice.” 2015. Thesis, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0630115-114124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Chuan-Siang. “APC Haploinsufficiency Cooperates With Kras and p53 Loss to Accelerate High Grade Glioblastoma Formation in Mice.” 2015. Web. 18 Aug 2019.

Vancouver:

Su C. APC Haploinsufficiency Cooperates With Kras and p53 Loss to Accelerate High Grade Glioblastoma Formation in Mice. [Internet] [Thesis]. NSYSU; 2015. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0630115-114124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su C. APC Haploinsufficiency Cooperates With Kras and p53 Loss to Accelerate High Grade Glioblastoma Formation in Mice. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0630115-114124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

16. Liu, Yu-Hsuan. Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

 Prostate cancer (PCa) is one of the most common malignant tumors in men and the second leading cause of cancer deaths in male worldwide. P53… (more)

Subjects/Keywords: Braf; mGluR1; P53; Kras; Mouse models of prostate cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liu, Y. (2015). Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Yu-Hsuan. “Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice.” 2015. Thesis, NSYSU. Accessed August 18, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Yu-Hsuan. “Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice.” 2015. Web. 18 Aug 2019.

Vancouver:

Liu Y. Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice. [Internet] [Thesis]. NSYSU; 2015. [cited 2019 Aug 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu Y. Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Australia

17. Ali Rahman, Ireni Sufinah. Characterisation of the akimba mouse : a model of retinal neovascularisation on a hyperglycaemic background and the in vivo study of endothelin-2 in the development of neovascularisation.

Degree: PhD, 2011, University of Western Australia

[Truncated abstract] Research on diabetic retinopathy (DR) has been hampered by the lack of an animal model that exhibits not only the early stages of… (more)

Subjects/Keywords: VEGF; Diabetic retinopathy; Endothelin; Mouse models; Retinal vasculature; Retinal neovascularisation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ali Rahman, I. S. (2011). Characterisation of the akimba mouse : a model of retinal neovascularisation on a hyperglycaemic background and the in vivo study of endothelin-2 in the development of neovascularisation. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33072&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Ali Rahman, Ireni Sufinah. “Characterisation of the akimba mouse : a model of retinal neovascularisation on a hyperglycaemic background and the in vivo study of endothelin-2 in the development of neovascularisation.” 2011. Doctoral Dissertation, University of Western Australia. Accessed August 18, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33072&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Ali Rahman, Ireni Sufinah. “Characterisation of the akimba mouse : a model of retinal neovascularisation on a hyperglycaemic background and the in vivo study of endothelin-2 in the development of neovascularisation.” 2011. Web. 18 Aug 2019.

Vancouver:

Ali Rahman IS. Characterisation of the akimba mouse : a model of retinal neovascularisation on a hyperglycaemic background and the in vivo study of endothelin-2 in the development of neovascularisation. [Internet] [Doctoral dissertation]. University of Western Australia; 2011. [cited 2019 Aug 18]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33072&local_base=GEN01-INS01.

Council of Science Editors:

Ali Rahman IS. Characterisation of the akimba mouse : a model of retinal neovascularisation on a hyperglycaemic background and the in vivo study of endothelin-2 in the development of neovascularisation. [Doctoral Dissertation]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33072&local_base=GEN01-INS01


Universiteit Utrecht

18. Tetteh, P.W. Plasticity of intestinal epithelial cells in regeneration and cancer.

Degree: 2015, Universiteit Utrecht

 Cellular plasticity refers to the ability of a cell to change its fate or identity in response to external or intrinsic factors. Regeneration of the… (more)

Subjects/Keywords: plasticity; intestine; epithelium; stem cells; differentiated cells; mouse models; cancer; niche

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tetteh, P. W. (2015). Plasticity of intestinal epithelial cells in regeneration and cancer. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/313330

Chicago Manual of Style (16th Edition):

Tetteh, P W. “Plasticity of intestinal epithelial cells in regeneration and cancer.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed August 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/313330.

MLA Handbook (7th Edition):

Tetteh, P W. “Plasticity of intestinal epithelial cells in regeneration and cancer.” 2015. Web. 18 Aug 2019.

Vancouver:

Tetteh PW. Plasticity of intestinal epithelial cells in regeneration and cancer. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2019 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/313330.

Council of Science Editors:

Tetteh PW. Plasticity of intestinal epithelial cells in regeneration and cancer. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/313330


University of Louisville

19. Barve, Aditya. Establishing a clinically relevant mouse model of human AML to test novel transmethylation inhibitors.

Degree: MS, 2016, University of Louisville

  Acute myeloid leukemia (AML) is a highly heterogeneous clonal disorder characterized by an accumulation of malignant immature myeloid progenitors in the bone marrow (BM)… (more)

Subjects/Keywords: Leukemia; Cancer; Epigenetics; Mouse models; Immune System Diseases; Neoplasms

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barve, A. (2016). Establishing a clinically relevant mouse model of human AML to test novel transmethylation inhibitors. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/2615 ; https://ir.library.louisville.edu/etd/2615

Chicago Manual of Style (16th Edition):

Barve, Aditya. “Establishing a clinically relevant mouse model of human AML to test novel transmethylation inhibitors.” 2016. Masters Thesis, University of Louisville. Accessed August 18, 2019. 10.18297/etd/2615 ; https://ir.library.louisville.edu/etd/2615.

MLA Handbook (7th Edition):

Barve, Aditya. “Establishing a clinically relevant mouse model of human AML to test novel transmethylation inhibitors.” 2016. Web. 18 Aug 2019.

Vancouver:

Barve A. Establishing a clinically relevant mouse model of human AML to test novel transmethylation inhibitors. [Internet] [Masters thesis]. University of Louisville; 2016. [cited 2019 Aug 18]. Available from: 10.18297/etd/2615 ; https://ir.library.louisville.edu/etd/2615.

Council of Science Editors:

Barve A. Establishing a clinically relevant mouse model of human AML to test novel transmethylation inhibitors. [Masters Thesis]. University of Louisville; 2016. Available from: 10.18297/etd/2615 ; https://ir.library.louisville.edu/etd/2615


Case Western Reserve University

20. Komuro, Amanda Katherine. Altered Social Behavior and Neuroinflammation in a Mouse Model of Pten Mislocalization.

Degree: PhD, Molecular Medicine, 2015, Case Western Reserve University

 Autism Spectrum Disorder (ASD) is a neurodevelopmental disorder affecting 1 in 68 children within the United States, whose high heritability is complicated by genetic heterogeneity.… (more)

Subjects/Keywords: Neurosciences; Genetics; autism, mouse, pten, glia, neuroinflammation, social behavior, animal models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Komuro, A. K. (2015). Altered Social Behavior and Neuroinflammation in a Mouse Model of Pten Mislocalization. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1409766264

Chicago Manual of Style (16th Edition):

Komuro, Amanda Katherine. “Altered Social Behavior and Neuroinflammation in a Mouse Model of Pten Mislocalization.” 2015. Doctoral Dissertation, Case Western Reserve University. Accessed August 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1409766264.

MLA Handbook (7th Edition):

Komuro, Amanda Katherine. “Altered Social Behavior and Neuroinflammation in a Mouse Model of Pten Mislocalization.” 2015. Web. 18 Aug 2019.

Vancouver:

Komuro AK. Altered Social Behavior and Neuroinflammation in a Mouse Model of Pten Mislocalization. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2015. [cited 2019 Aug 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1409766264.

Council of Science Editors:

Komuro AK. Altered Social Behavior and Neuroinflammation in a Mouse Model of Pten Mislocalization. [Doctoral Dissertation]. Case Western Reserve University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1409766264


University of Manchester

21. Toulson, Gregory. The role of Secretory Leukocyte Protease Inhibitor (SLPI) in progranulin regulation and neurodegeneration.

Degree: PhD, 2013, University of Manchester

 Frontotemporal lobar degeneration (FTLD) is an early onset neurodegenerative disorder which selectively destroys frontal and temporal cortical neurones. The resulting damage leads to a range… (more)

Subjects/Keywords: 616.89; SLPI, Progranulin, FTLD, Mouse models, Behavioral testing, Dementia.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Toulson, G. (2013). The role of Secretory Leukocyte Protease Inhibitor (SLPI) in progranulin regulation and neurodegeneration. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-secretory-leukocyte-protease-inhibitor-slpi-in-progranulin-regulation-and-neurodegeneration(bded6e44-c72e-4d21-8133-a16652204a9e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607031

Chicago Manual of Style (16th Edition):

Toulson, Gregory. “The role of Secretory Leukocyte Protease Inhibitor (SLPI) in progranulin regulation and neurodegeneration.” 2013. Doctoral Dissertation, University of Manchester. Accessed August 18, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-secretory-leukocyte-protease-inhibitor-slpi-in-progranulin-regulation-and-neurodegeneration(bded6e44-c72e-4d21-8133-a16652204a9e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607031.

MLA Handbook (7th Edition):

Toulson, Gregory. “The role of Secretory Leukocyte Protease Inhibitor (SLPI) in progranulin regulation and neurodegeneration.” 2013. Web. 18 Aug 2019.

Vancouver:

Toulson G. The role of Secretory Leukocyte Protease Inhibitor (SLPI) in progranulin regulation and neurodegeneration. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Aug 18]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-secretory-leukocyte-protease-inhibitor-slpi-in-progranulin-regulation-and-neurodegeneration(bded6e44-c72e-4d21-8133-a16652204a9e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607031.

Council of Science Editors:

Toulson G. The role of Secretory Leukocyte Protease Inhibitor (SLPI) in progranulin regulation and neurodegeneration. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-secretory-leukocyte-protease-inhibitor-slpi-in-progranulin-regulation-and-neurodegeneration(bded6e44-c72e-4d21-8133-a16652204a9e).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607031


University of Southern California

22. Kong, Seogkyoung. Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice.

Degree: PhD, Biochemistry & Molecular Biology, 2009, University of Southern California

 Glutamate excitotoxicity plays a role in neuronal death in diverse neurodegenerative diseases. Inbred strains of mice differ in their susceptibility to excitotoxin-induced cell death, but… (more)

Subjects/Keywords: glutamate excitotoxicity; genetic variation; Galr1; inbred and congenic mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kong, S. (2009). Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7802

Chicago Manual of Style (16th Edition):

Kong, Seogkyoung. “Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice.” 2009. Doctoral Dissertation, University of Southern California. Accessed August 18, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7802.

MLA Handbook (7th Edition):

Kong, Seogkyoung. “Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice.” 2009. Web. 18 Aug 2019.

Vancouver:

Kong S. Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2019 Aug 18]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7802.

Council of Science Editors:

Kong S. Variation in Galr1 expression determines the susceptibility to excitotoxin-induced neuronal death in mice. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/322194/rec/7802


University of Sydney

23. Caldwell, Aimee Sarah Lee. Unravelling The Role Of Androgens In Polycystic Ovary Syndrome .

Degree: 2017, University of Sydney

 Polycystic Ovary Syndrome (PCOS) is a multifaceted hormonal disorder which affects 5-15% of reproductive-aged women worldwide. While classically recognised as an ovarian disorder, PCOS is… (more)

Subjects/Keywords: Polycystic Ovary Syndrome (PCOS); Androgens; Ovary; Reproduction; Mouse Models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Caldwell, A. S. L. (2017). Unravelling The Role Of Androgens In Polycystic Ovary Syndrome . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/18129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Caldwell, Aimee Sarah Lee. “Unravelling The Role Of Androgens In Polycystic Ovary Syndrome .” 2017. Thesis, University of Sydney. Accessed August 18, 2019. http://hdl.handle.net/2123/18129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Caldwell, Aimee Sarah Lee. “Unravelling The Role Of Androgens In Polycystic Ovary Syndrome .” 2017. Web. 18 Aug 2019.

Vancouver:

Caldwell ASL. Unravelling The Role Of Androgens In Polycystic Ovary Syndrome . [Internet] [Thesis]. University of Sydney; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/2123/18129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Caldwell ASL. Unravelling The Role Of Androgens In Polycystic Ovary Syndrome . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/18129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

24. Bey, Alexandra Lyndon. Using Shank3 Model Mice to Probe the Neuroanatomic Basis of Autism .

Degree: 2017, Duke University

  Autism spectrum disorders (ASDs) are increasingly prevalent, and the costs associated with caring for affected patients across the lifespan are immense. However, the pathophysiology… (more)

Subjects/Keywords: Neurosciences; Genetics; Mental health; autism; behavior; genetics; mouse models; Shank3

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bey, A. L. (2017). Using Shank3 Model Mice to Probe the Neuroanatomic Basis of Autism . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/14354

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bey, Alexandra Lyndon. “Using Shank3 Model Mice to Probe the Neuroanatomic Basis of Autism .” 2017. Thesis, Duke University. Accessed August 18, 2019. http://hdl.handle.net/10161/14354.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bey, Alexandra Lyndon. “Using Shank3 Model Mice to Probe the Neuroanatomic Basis of Autism .” 2017. Web. 18 Aug 2019.

Vancouver:

Bey AL. Using Shank3 Model Mice to Probe the Neuroanatomic Basis of Autism . [Internet] [Thesis]. Duke University; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10161/14354.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bey AL. Using Shank3 Model Mice to Probe the Neuroanatomic Basis of Autism . [Thesis]. Duke University; 2017. Available from: http://hdl.handle.net/10161/14354

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

25. Venegas Pino, Daniel. INVESTIGATING THE EFFECTS OF HYPERGLYCEMIA ON THE VASA VASORUM IN THE DEVELOPMENT OF ATHEROSCLEROSIS AND ESTABLISHMENT OF NOVEL MOUSE MODELS OF DIABETES.

Degree: PhD, 2016, McMaster University

The prevalence of diabetes is increasing rapidly around the world. People with diabetes are 2–4 times more likely to die from cerebro and cardio-vascular causes… (more)

Subjects/Keywords: Diabetes; Mouse Models; Atherosclerosis; Microvascular complications; Macrovascular complications; Sex Effects

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Venegas Pino, D. (2016). INVESTIGATING THE EFFECTS OF HYPERGLYCEMIA ON THE VASA VASORUM IN THE DEVELOPMENT OF ATHEROSCLEROSIS AND ESTABLISHMENT OF NOVEL MOUSE MODELS OF DIABETES. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/18778

Chicago Manual of Style (16th Edition):

Venegas Pino, Daniel. “INVESTIGATING THE EFFECTS OF HYPERGLYCEMIA ON THE VASA VASORUM IN THE DEVELOPMENT OF ATHEROSCLEROSIS AND ESTABLISHMENT OF NOVEL MOUSE MODELS OF DIABETES.” 2016. Doctoral Dissertation, McMaster University. Accessed August 18, 2019. http://hdl.handle.net/11375/18778.

MLA Handbook (7th Edition):

Venegas Pino, Daniel. “INVESTIGATING THE EFFECTS OF HYPERGLYCEMIA ON THE VASA VASORUM IN THE DEVELOPMENT OF ATHEROSCLEROSIS AND ESTABLISHMENT OF NOVEL MOUSE MODELS OF DIABETES.” 2016. Web. 18 Aug 2019.

Vancouver:

Venegas Pino D. INVESTIGATING THE EFFECTS OF HYPERGLYCEMIA ON THE VASA VASORUM IN THE DEVELOPMENT OF ATHEROSCLEROSIS AND ESTABLISHMENT OF NOVEL MOUSE MODELS OF DIABETES. [Internet] [Doctoral dissertation]. McMaster University; 2016. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/11375/18778.

Council of Science Editors:

Venegas Pino D. INVESTIGATING THE EFFECTS OF HYPERGLYCEMIA ON THE VASA VASORUM IN THE DEVELOPMENT OF ATHEROSCLEROSIS AND ESTABLISHMENT OF NOVEL MOUSE MODELS OF DIABETES. [Doctoral Dissertation]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/18778


Duke University

26. Lee, Chang-Lung. Mechanisms by which p53 Regulates Radiation-induced Carcinogenesis and Myocardial Injury .

Degree: 2012, Duke University

  Radiation therapy can cause acute toxicity and long-term side effects in normal tissues. Because part of the acute toxicity of radiation is due to… (more)

Subjects/Keywords: Cellular biology; Endothelial cells; Lymphoma; Mouse models; Myocardial injury; p53; Radiation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, C. (2012). Mechanisms by which p53 Regulates Radiation-induced Carcinogenesis and Myocardial Injury . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Chang-Lung. “Mechanisms by which p53 Regulates Radiation-induced Carcinogenesis and Myocardial Injury .” 2012. Thesis, Duke University. Accessed August 18, 2019. http://hdl.handle.net/10161/5605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Chang-Lung. “Mechanisms by which p53 Regulates Radiation-induced Carcinogenesis and Myocardial Injury .” 2012. Web. 18 Aug 2019.

Vancouver:

Lee C. Mechanisms by which p53 Regulates Radiation-induced Carcinogenesis and Myocardial Injury . [Internet] [Thesis]. Duke University; 2012. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10161/5605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee C. Mechanisms by which p53 Regulates Radiation-induced Carcinogenesis and Myocardial Injury . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Case Western Reserve University

27. Vitko, Megan Sue. Intestinal Dysfunction in Cystic Fibrosis.

Degree: PhD, Genetics, 2016, Case Western Reserve University

 Intestinal Dysfunction in Cystic FibrosisAbstract By MEGAN VITKOCystic Fibrosis (CF) is an autosomal recessive genetic disease that is caused by mutations in the cystic fibrosis… (more)

Subjects/Keywords: Genetics; CFTR; Intestinal Obstruction; Cystic Fibrosis; mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vitko, M. S. (2016). Intestinal Dysfunction in Cystic Fibrosis. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266

Chicago Manual of Style (16th Edition):

Vitko, Megan Sue. “Intestinal Dysfunction in Cystic Fibrosis.” 2016. Doctoral Dissertation, Case Western Reserve University. Accessed August 18, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266.

MLA Handbook (7th Edition):

Vitko, Megan Sue. “Intestinal Dysfunction in Cystic Fibrosis.” 2016. Web. 18 Aug 2019.

Vancouver:

Vitko MS. Intestinal Dysfunction in Cystic Fibrosis. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2016. [cited 2019 Aug 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266.

Council of Science Editors:

Vitko MS. Intestinal Dysfunction in Cystic Fibrosis. [Doctoral Dissertation]. Case Western Reserve University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266


Boston University

28. Belghasem, Mostafa. Pathological and molecular profiling in hypertension-induced glomerular injury.

Degree: PhD, Pathology & Laboratory Medicine, 2015, Boston University

 The increased prevalence of chronic kidney disease (CKD) has become a major global health burden. This increase in CKD burden parallels the increase in hypertension… (more)

Subjects/Keywords: Pathology; Chronic kidney disease; Glomerulosclerosis; Hypertension; Podocyte; Mouse models

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Belghasem, M. (2015). Pathological and molecular profiling in hypertension-induced glomerular injury. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/13940

Chicago Manual of Style (16th Edition):

Belghasem, Mostafa. “Pathological and molecular profiling in hypertension-induced glomerular injury.” 2015. Doctoral Dissertation, Boston University. Accessed August 18, 2019. http://hdl.handle.net/2144/13940.

MLA Handbook (7th Edition):

Belghasem, Mostafa. “Pathological and molecular profiling in hypertension-induced glomerular injury.” 2015. Web. 18 Aug 2019.

Vancouver:

Belghasem M. Pathological and molecular profiling in hypertension-induced glomerular injury. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/2144/13940.

Council of Science Editors:

Belghasem M. Pathological and molecular profiling in hypertension-induced glomerular injury. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13940

29. Zhang, Yiming. Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering : 骨再生研究モデルとしての免疫正常および免疫不全動物の比較について.

Degree: 博士(歯学), 2015, Matsumoto Dental University / 松本歯科大学

Objectives To understand the differences and similarities between immunocompetent and immunodeficient mice as ectopic transplantation animal models for bone tissue engineering. Materials and Methods Osteogenic… (more)

Subjects/Keywords: bone tissue engineering; animal models; ectopic transplantation; immunological reaction; nude mouse

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, . Y. (2015). Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering : 骨再生研究モデルとしての免疫正常および免疫不全動物の比較について. (Thesis). Matsumoto Dental University / 松本歯科大学. Retrieved from http://id.nii.ac.jp/1070/00002430/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Yiming. “Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering : 骨再生研究モデルとしての免疫正常および免疫不全動物の比較について.” 2015. Thesis, Matsumoto Dental University / 松本歯科大学. Accessed August 18, 2019. http://id.nii.ac.jp/1070/00002430/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Yiming. “Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering : 骨再生研究モデルとしての免疫正常および免疫不全動物の比較について.” 2015. Web. 18 Aug 2019.

Vancouver:

Zhang Y. Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering : 骨再生研究モデルとしての免疫正常および免疫不全動物の比較について. [Internet] [Thesis]. Matsumoto Dental University / 松本歯科大学; 2015. [cited 2019 Aug 18]. Available from: http://id.nii.ac.jp/1070/00002430/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang Y. Comparing immunocompetent and immunodeficient mice as animal models for bone tissue engineering : 骨再生研究モデルとしての免疫正常および免疫不全動物の比較について. [Thesis]. Matsumoto Dental University / 松本歯科大学; 2015. Available from: http://id.nii.ac.jp/1070/00002430/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Miami

30. Puplampu-Dove, Yvonne A. Potentiating Tumor Immunity Using Aptamer-Targeted RNAi to Render CD8+ T Cells Resistant to TGFβ Inhibition.

Degree: PhD, Microbiology and Immunology (Medicine), 2017, University of Miami

 Cancer is one of the leading causes of death in the world. Current treatments for cancer include chemotherapy, radiation therapy, surgery, immunotherapy and transplantation. While… (more)

Subjects/Keywords: TGFbeta; cancer immunotherapy; preclinical mouse models; tumor targeting; aptamers; siRNA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Puplampu-Dove, Y. A. (2017). Potentiating Tumor Immunity Using Aptamer-Targeted RNAi to Render CD8+ T Cells Resistant to TGFβ Inhibition. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/1970

Chicago Manual of Style (16th Edition):

Puplampu-Dove, Yvonne A. “Potentiating Tumor Immunity Using Aptamer-Targeted RNAi to Render CD8+ T Cells Resistant to TGFβ Inhibition.” 2017. Doctoral Dissertation, University of Miami. Accessed August 18, 2019. https://scholarlyrepository.miami.edu/oa_dissertations/1970.

MLA Handbook (7th Edition):

Puplampu-Dove, Yvonne A. “Potentiating Tumor Immunity Using Aptamer-Targeted RNAi to Render CD8+ T Cells Resistant to TGFβ Inhibition.” 2017. Web. 18 Aug 2019.

Vancouver:

Puplampu-Dove YA. Potentiating Tumor Immunity Using Aptamer-Targeted RNAi to Render CD8+ T Cells Resistant to TGFβ Inhibition. [Internet] [Doctoral dissertation]. University of Miami; 2017. [cited 2019 Aug 18]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1970.

Council of Science Editors:

Puplampu-Dove YA. Potentiating Tumor Immunity Using Aptamer-Targeted RNAi to Render CD8+ T Cells Resistant to TGFβ Inhibition. [Doctoral Dissertation]. University of Miami; 2017. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/1970

[1] [2] [3] [4] [5] [6] [7] [8]

.