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You searched for subject:(mitogen activated protein kinases). Showing records 1 – 30 of 17949 total matches.

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University of Hong Kong

1. Chiu, Tsz-ling. Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries.

Degree: Master of Medical Sciences, 2014, University of Hong Kong

 Background: Regulation of vascular tone is complex. Various complementary signaling pathways causing contraction and relaxation of vascular smooth muscle take place to ensure proper blood… (more)

Subjects/Keywords: Vascular smooth muscle; Mitogen-activated protein kinases

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APA (6th Edition):

Chiu, T. (2014). Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. (Masters Thesis). University of Hong Kong. Retrieved from Chiu, T. [趙芷菱]. (2014). Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5303896 ; http://dx.doi.org/10.5353/th_b5303896 ; http://hdl.handle.net/10722/206497

Chicago Manual of Style (16th Edition):

Chiu, Tsz-ling. “Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries.” 2014. Masters Thesis, University of Hong Kong. Accessed April 20, 2019. Chiu, T. [趙芷菱]. (2014). Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5303896 ; http://dx.doi.org/10.5353/th_b5303896 ; http://hdl.handle.net/10722/206497.

MLA Handbook (7th Edition):

Chiu, Tsz-ling. “Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries.” 2014. Web. 20 Apr 2019.

Vancouver:

Chiu T. Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2019 Apr 20]. Available from: Chiu, T. [趙芷菱]. (2014). Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5303896 ; http://dx.doi.org/10.5353/th_b5303896 ; http://hdl.handle.net/10722/206497.

Council of Science Editors:

Chiu T. Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. [Masters Thesis]. University of Hong Kong; 2014. Available from: Chiu, T. [趙芷菱]. (2014). Role of mitogen-activated protein kinases in vascular relaxation in porcine coronary arteries. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5303896 ; http://dx.doi.org/10.5353/th_b5303896 ; http://hdl.handle.net/10722/206497


University of Texas Southwestern Medical Center

2. Juang, Yu-Chi. Structural Analysis of STE20-Related Proline-Alanine-Rich Kinase (SPAK).

Degree: 2009, University of Texas Southwestern Medical Center

 Ste20-related proline-alanine-rich kinase (SPAK) is a kinase that regulates ion cotransporters including KCC, NKCC1, NKCC2 and NCC. Recently, SPAK was identified as a target regulated… (more)

Subjects/Keywords: Mitogen-Activated Protein Kinases; Transcription Factors; Protein-Serine-Threonine Kinases

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APA (6th Edition):

Juang, Y. (2009). Structural Analysis of STE20-Related Proline-Alanine-Rich Kinase (SPAK). (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Juang, Yu-Chi. “Structural Analysis of STE20-Related Proline-Alanine-Rich Kinase (SPAK).” 2009. Thesis, University of Texas Southwestern Medical Center. Accessed April 20, 2019. http://hdl.handle.net/2152.5/634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Juang, Yu-Chi. “Structural Analysis of STE20-Related Proline-Alanine-Rich Kinase (SPAK).” 2009. Web. 20 Apr 2019.

Vancouver:

Juang Y. Structural Analysis of STE20-Related Proline-Alanine-Rich Kinase (SPAK). [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2009. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/2152.5/634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Juang Y. Structural Analysis of STE20-Related Proline-Alanine-Rich Kinase (SPAK). [Thesis]. University of Texas Southwestern Medical Center; 2009. Available from: http://hdl.handle.net/2152.5/634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

3. Pizzimenti, Natalie Maria. Mitogen activated protein kinase signaling in mouse skeletal muscle.

Degree: 2014, Michigan State University

Thesis M.S. Michigan State University. Physiology - Master of Science 2014.

AMP activated protein kinase (AMPK) and p38 mitogen activated protein kinases (MAPKs) are activated(more)

Subjects/Keywords: Physiology; Mitochondria – Formation; Protein kinases; Mitogen-activated protein kinases

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APA (6th Edition):

Pizzimenti, N. M. (2014). Mitogen activated protein kinase signaling in mouse skeletal muscle. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pizzimenti, Natalie Maria. “Mitogen activated protein kinase signaling in mouse skeletal muscle.” 2014. Thesis, Michigan State University. Accessed April 20, 2019. http://etd.lib.msu.edu/islandora/object/etd:2416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pizzimenti, Natalie Maria. “Mitogen activated protein kinase signaling in mouse skeletal muscle.” 2014. Web. 20 Apr 2019.

Vancouver:

Pizzimenti NM. Mitogen activated protein kinase signaling in mouse skeletal muscle. [Internet] [Thesis]. Michigan State University; 2014. [cited 2019 Apr 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pizzimenti NM. Mitogen activated protein kinase signaling in mouse skeletal muscle. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Taylor, Clinton A., IV. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.

Degree: 2016, University of Texas Southwestern Medical Center

Protein-protein interactions are essential for nearly every cellular process. Within signaling pathways, such interactions carry out numerous functions such as defining substrate specificity, inhibition of… (more)

Subjects/Keywords: Mitogen-Activated Protein Kinase Kinases; Protein-Serine-Threonine Kinases; Signal Transduction; Transcription Factors

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APA (6th Edition):

Taylor, Clinton A., I. (2016). Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Thesis, University of Texas Southwestern Medical Center. Accessed April 20, 2019. http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Taylor, Clinton A., IV. “Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling.” 2016. Web. 20 Apr 2019.

Vancouver:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2016. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/2152.5/6152.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Taylor, Clinton A. I. Regulation and Dysregulation via Docking Interactions in WNK and ERK1/2 MAPK Signaling. [Thesis]. University of Texas Southwestern Medical Center; 2016. Available from: http://hdl.handle.net/2152.5/6152

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

5. Rysä, J. (Jaana). Gene expression profiling in experimental models of cardiac load.

Degree: 2008, University of Oulu

 Abstract Cardiac hypertrophy provides an adaptive mechanism to maintain cardiac output in response to increased workload, and although initially beneficial, hypertrophy eventually leads to heart… (more)

Subjects/Keywords: DNA microarrays; diastolic heart failure; hypertrophy; mitogen-activated protein kinases; stretch

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APA (6th Edition):

Rysä, J. (. (2008). Gene expression profiling in experimental models of cardiac load. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789514287664

Chicago Manual of Style (16th Edition):

Rysä, J (Jaana). “Gene expression profiling in experimental models of cardiac load.” 2008. Doctoral Dissertation, University of Oulu. Accessed April 20, 2019. http://urn.fi/urn:isbn:9789514287664.

MLA Handbook (7th Edition):

Rysä, J (Jaana). “Gene expression profiling in experimental models of cardiac load.” 2008. Web. 20 Apr 2019.

Vancouver:

Rysä J(. Gene expression profiling in experimental models of cardiac load. [Internet] [Doctoral dissertation]. University of Oulu; 2008. [cited 2019 Apr 20]. Available from: http://urn.fi/urn:isbn:9789514287664.

Council of Science Editors:

Rysä J(. Gene expression profiling in experimental models of cardiac load. [Doctoral Dissertation]. University of Oulu; 2008. Available from: http://urn.fi/urn:isbn:9789514287664


University of Hong Kong

6. Zhang, Pingde. TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway.

Degree: PhD, 2011, University of Hong Kong

 Ovarian cancer is the most lethal gynecological malignancy. Most of ovarian cancer patients relapse and subsequently die due to the development of resistance to chemotherapy.… (more)

Subjects/Keywords: JNK mitogen-activated protein kinases.; Ovaries - Cancer.; Cisplatin.; p53 antioncogene.

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APA (6th Edition):

Zhang, P. (2011). TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. (Doctoral Dissertation). University of Hong Kong. Retrieved from Zhang, P. [张萍德]. (2011). TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775294 ; http://dx.doi.org/10.5353/th_b4775294 ; http://hdl.handle.net/10722/174474

Chicago Manual of Style (16th Edition):

Zhang, Pingde. “TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed April 20, 2019. Zhang, P. [张萍德]. (2011). TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775294 ; http://dx.doi.org/10.5353/th_b4775294 ; http://hdl.handle.net/10722/174474.

MLA Handbook (7th Edition):

Zhang, Pingde. “TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway.” 2011. Web. 20 Apr 2019.

Vancouver:

Zhang P. TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2019 Apr 20]. Available from: Zhang, P. [张萍德]. (2011). TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775294 ; http://dx.doi.org/10.5353/th_b4775294 ; http://hdl.handle.net/10722/174474.

Council of Science Editors:

Zhang P. TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: Zhang, P. [张萍德]. (2011). TAp73α enhances the cellular sensitivity to cisplatin in ovarian cancer cells via the JNK signaling pathway. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775294 ; http://dx.doi.org/10.5353/th_b4775294 ; http://hdl.handle.net/10722/174474


University of Texas Southwestern Medical Center

7. An, Zhenyi. The Regulation of Autophagy and Its Role in Mitotic Exit.

Degree: 2014, University of Texas Southwestern Medical Center

 Autophagy is an evolutionarily conserved pathway in which cells enclose cytoplasmic contents in double membrane vesicles and deliver them to the lysosome for degradation. Autophagy… (more)

Subjects/Keywords: Apoptosis Regulatory Proteins; Autophagy; Mitogen-Activated Protein Kinases; Phosphorylation

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APA (6th Edition):

An, Z. (2014). The Regulation of Autophagy and Its Role in Mitotic Exit. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed April 20, 2019. http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

An, Zhenyi. “The Regulation of Autophagy and Its Role in Mitotic Exit.” 2014. Web. 20 Apr 2019.

Vancouver:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/2152.5/3582.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

An Z. The Regulation of Autophagy and Its Role in Mitotic Exit. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3582

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

8. Sun, Bin. Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis.

Degree: PhD, Toxicology, 2010, University of Georgia

 The goal of this work was to identify mechanisms by which inhibition of calcium-independent phospholipase A2 (iPLA2) induces cytostasis in human LNCaP (p53 wild-type and… (more)

Subjects/Keywords: Phospholipase A2; Phospholipids; p53; Mitogen Activated Protein Kinases; Reactive species; Phospholipid Profiles

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APA (6th Edition):

Sun, B. (2010). Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/sun_bin_201008_phd

Chicago Manual of Style (16th Edition):

Sun, Bin. “Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis.” 2010. Doctoral Dissertation, University of Georgia. Accessed April 20, 2019. http://purl.galileo.usg.edu/uga_etd/sun_bin_201008_phd.

MLA Handbook (7th Edition):

Sun, Bin. “Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis.” 2010. Web. 20 Apr 2019.

Vancouver:

Sun B. Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis. [Internet] [Doctoral dissertation]. University of Georgia; 2010. [cited 2019 Apr 20]. Available from: http://purl.galileo.usg.edu/uga_etd/sun_bin_201008_phd.

Council of Science Editors:

Sun B. Inhibition of calcium-independent phospholipase A2 induces prostate cancer cell cytostasis. [Doctoral Dissertation]. University of Georgia; 2010. Available from: http://purl.galileo.usg.edu/uga_etd/sun_bin_201008_phd

9. Karagiannidis, Ioannis. In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης.

Degree: 2016, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

The impact of a potential autophagy (LC3a/b) deregulation in hyper and in hypo stages during sepsis-induced kidney injury and the temporal profile of phosphorylated extracellular… (more)

Subjects/Keywords: Απόπτωση (προγραμματισμένος κυτταρικός θάνατος); Αυτοφαγία; Πειραματικά μοντέλα σήψης; Mitogen – Activated Protein Kinases (MAPKs)

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APA (6th Edition):

Karagiannidis, I. (2016). In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/37872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Karagiannidis, Ioannis. “In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης.” 2016. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 20, 2019. http://hdl.handle.net/10442/hedi/37872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Karagiannidis, Ioannis. “In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης.” 2016. Web. 20 Apr 2019.

Vancouver:

Karagiannidis I. In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/10442/hedi/37872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Karagiannidis I. In Vivo μελέτη της συμβολής της φωσφορυλίωσης των MAP κινασών στην αποπτωτική διαδικασία της σήψης. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. Available from: http://hdl.handle.net/10442/hedi/37872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

10. Lyashenko, Eugenia. Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway.

Degree: 2015, Columbia University

 Cells process information from their environment, such as the stimuli to grow, divide, or die, via cell signaling. Deregulated processing of extracellular stimuli can lead… (more)

Subjects/Keywords: Mitogen-activated protein kinases; Weber-Fechner law; Cellular control mechanisms; Bioinformatics; Biology

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APA (6th Edition):

Lyashenko, E. (2015). Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8RF5SVZ

Chicago Manual of Style (16th Edition):

Lyashenko, Eugenia. “Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway.” 2015. Doctoral Dissertation, Columbia University. Accessed April 20, 2019. https://doi.org/10.7916/D8RF5SVZ.

MLA Handbook (7th Edition):

Lyashenko, Eugenia. “Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway.” 2015. Web. 20 Apr 2019.

Vancouver:

Lyashenko E. Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2019 Apr 20]. Available from: https://doi.org/10.7916/D8RF5SVZ.

Council of Science Editors:

Lyashenko E. Cell Memory in the Mitogen-Activated Protein Kinase Signaling Pathway. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://doi.org/10.7916/D8RF5SVZ


Johannes Gutenberg Universität Mainz

11. Küppers, Monika. Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene.

Degree: 2008, Johannes Gutenberg Universität Mainz

Die Zellen eines Organismus unterliegen ständig den Einflüssen wachstumsfördernder und –hemmender Signale. Die korrekte Verarbeitung dieser Signale ist essentiell für die Aufrechterhaltung der Gewebehomöostase. Wachstumsfördernde… (more)

Subjects/Keywords: Kontaktinhibition, Zellzyklus, Mitogen-aktivierte Protein Kinasen (MAPK), differentielle Genexpression; contact-inhibition, cell cycle, mitogen-activated protein kinases (MAPK), differential gene expression; Natural sciences and mathematics

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APA (6th Edition):

Küppers, M. (2008). Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/

Chicago Manual of Style (16th Edition):

Küppers, Monika. “Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene.” 2008. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed April 20, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/.

MLA Handbook (7th Edition):

Küppers, Monika. “Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene.” 2008. Web. 20 Apr 2019.

Vancouver:

Küppers M. Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2008. [cited 2019 Apr 20]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/.

Council of Science Editors:

Küppers M. Molekulare Mechanismen der Kontaktinhibition: Identifizierung neuer Tumorsuppressorgene. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2008. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2008/1722/


University of Western Australia

12. Arulpragasam, Ajanthy. The role of 14-3-3 proteins in calcium-sensing receptor cell signalling and expression.

Degree: PhD, 2010, University of Western Australia

[Truncated abstract] The calcium-sensing receptor (CaR), belonging to class C of G protein-coupled receptors (GPCRs), is highly expressed in tissues that govern the CaR’s primary… (more)

Subjects/Keywords: G proteins; Calcium; Cell receptors; Cellular signal transduction; Mitogen-activated protein kinases; Cytology; 14-3-3 proteins; Signalling; Calcium-sensing receptor

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APA (6th Edition):

Arulpragasam, A. (2010). The role of 14-3-3 proteins in calcium-sensing receptor cell signalling and expression. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30648&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Arulpragasam, Ajanthy. “The role of 14-3-3 proteins in calcium-sensing receptor cell signalling and expression.” 2010. Doctoral Dissertation, University of Western Australia. Accessed April 20, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30648&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Arulpragasam, Ajanthy. “The role of 14-3-3 proteins in calcium-sensing receptor cell signalling and expression.” 2010. Web. 20 Apr 2019.

Vancouver:

Arulpragasam A. The role of 14-3-3 proteins in calcium-sensing receptor cell signalling and expression. [Internet] [Doctoral dissertation]. University of Western Australia; 2010. [cited 2019 Apr 20]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30648&local_base=GEN01-INS01.

Council of Science Editors:

Arulpragasam A. The role of 14-3-3 proteins in calcium-sensing receptor cell signalling and expression. [Doctoral Dissertation]. University of Western Australia; 2010. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30648&local_base=GEN01-INS01


University of Hong Kong

13. Lam, King-yin, Andy. Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling.

Degree: M. Phil., 2010, University of Hong Kong

published_or_final_version

Biochemistry

Master

Master of Philosophy

Advisors/Committee Members: Shiu, SYW, Yao, KM.

Subjects/Keywords: Transcription factors.; Cellular signal transduction.; Mitogen-activated protein kinases.

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APA (6th Edition):

Lam, King-yin, A. (2010). Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. (Masters Thesis). University of Hong Kong. Retrieved from Lam, K. A. [林敬賢]. (2010). Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4425101 ; http://dx.doi.org/10.5353/th_b4425101 ; http://hdl.handle.net/10722/65109

Chicago Manual of Style (16th Edition):

Lam, King-yin, Andy. “Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling.” 2010. Masters Thesis, University of Hong Kong. Accessed April 20, 2019. Lam, K. A. [林敬賢]. (2010). Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4425101 ; http://dx.doi.org/10.5353/th_b4425101 ; http://hdl.handle.net/10722/65109.

MLA Handbook (7th Edition):

Lam, King-yin, Andy. “Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling.” 2010. Web. 20 Apr 2019.

Vancouver:

Lam, King-yin A. Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. [Internet] [Masters thesis]. University of Hong Kong; 2010. [cited 2019 Apr 20]. Available from: Lam, K. A. [林敬賢]. (2010). Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4425101 ; http://dx.doi.org/10.5353/th_b4425101 ; http://hdl.handle.net/10722/65109.

Council of Science Editors:

Lam, King-yin A. Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. [Masters Thesis]. University of Hong Kong; 2010. Available from: Lam, K. A. [林敬賢]. (2010). Differential regulation of FOXM1 isoforms by RaF/MEK/ERK signaling. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4425101 ; http://dx.doi.org/10.5353/th_b4425101 ; http://hdl.handle.net/10722/65109


University of Texas Southwestern Medical Center

14. Lo, Miao-Chia. The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos.

Degree: 2005, University of Texas Southwestern Medical Center

 In C. elegans embryos, the combined Wnt/MAPK pathway polarizes the founder cell of mesendoderm, EMS blastomere, such that EMS produces two daughters with distinct developmental… (more)

Subjects/Keywords: Caenorhabditis elegans; DNA-Binding Proteins; Mitogen-Activated Protein Kinases

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APA (6th Edition):

Lo, M. (2005). The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lo, Miao-Chia. “The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos.” 2005. Thesis, University of Texas Southwestern Medical Center. Accessed April 20, 2019. http://hdl.handle.net/2152.5/566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lo, Miao-Chia. “The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos.” 2005. Web. 20 Apr 2019.

Vancouver:

Lo M. The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2005. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/2152.5/566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lo M. The Study of Wnt Signaling Effector POP-1/TCF in C. Elegans Early Embryos. [Thesis]. University of Texas Southwestern Medical Center; 2005. Available from: http://hdl.handle.net/2152.5/566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina – Greensboro

15. Eanes, Lauren A. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.

Degree: 2014, University of North Carolina – Greensboro

 Estrogen receptor (ER) antagonists such as tamoxifen have been used successfully to treat ER+ breast cancers for more than 30 years. Tamoxifen targets the ER… (more)

Subjects/Keywords: Breast – Cancer – Treatment; Tamoxifen; Drug resistance in cancer cells; Estrogen – Receptors – Pathophysiology; Mitogen-activated protein kinases

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APA (6th Edition):

Eanes, L. A. (2014). Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160

Chicago Manual of Style (16th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Masters Thesis, University of North Carolina – Greensboro. Accessed April 20, 2019. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160.

MLA Handbook (7th Edition):

Eanes, Lauren A. “Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells.” 2014. Web. 20 Apr 2019.

Vancouver:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2014. [cited 2019 Apr 20]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160.

Council of Science Editors:

Eanes LA. Analysis of molecular target(s) of naringenin in MCF-7 breast cancer cells. [Masters Thesis]. University of North Carolina – Greensboro; 2014. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=16160


University of Missouri – Columbia

16. Larue, Clayton T., 1977-. Regulation of plant development in Arabidopsis.

Degree: PhD, 2008, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] The development of a complex organism, such as a plant, requires the function of multiple… (more)

Subjects/Keywords: Arabidopsis  – Development; Abscission (Botany); Mitogen-activated protein kinases

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APA (6th Edition):

Larue, Clayton T., 1. (2008). Regulation of plant development in Arabidopsis. (Doctoral Dissertation). University of Missouri – Columbia. Retrieved from https://doi.org/10.32469/10355/6051

Chicago Manual of Style (16th Edition):

Larue, Clayton T., 1977-. “Regulation of plant development in Arabidopsis.” 2008. Doctoral Dissertation, University of Missouri – Columbia. Accessed April 20, 2019. https://doi.org/10.32469/10355/6051.

MLA Handbook (7th Edition):

Larue, Clayton T., 1977-. “Regulation of plant development in Arabidopsis.” 2008. Web. 20 Apr 2019.

Vancouver:

Larue, Clayton T. 1. Regulation of plant development in Arabidopsis. [Internet] [Doctoral dissertation]. University of Missouri – Columbia; 2008. [cited 2019 Apr 20]. Available from: https://doi.org/10.32469/10355/6051.

Council of Science Editors:

Larue, Clayton T. 1. Regulation of plant development in Arabidopsis. [Doctoral Dissertation]. University of Missouri – Columbia; 2008. Available from: https://doi.org/10.32469/10355/6051


Michigan State University

17. Gopallawa, Indiwari. Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2.

Degree: 2015, Michigan State University

Thesis Ph. D. Michigan State University. Biochemistry and Molecular Biology 2015.

Apoptosis is now known to be an important regulator of maintaining normal organ homeostasis.… (more)

Subjects/Keywords: Mitogen-activated protein kinases; Phosphatases; Angiotensin converting enzyme; Angiotensin II – Antagonists; Angiotensins; Apoptosis; Epithelial cells; Cellular biology; Molecular biology; Biochemistry

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APA (6th Edition):

Gopallawa, I. (2015). Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gopallawa, Indiwari. “Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2.” 2015. Thesis, Michigan State University. Accessed April 20, 2019. http://etd.lib.msu.edu/islandora/object/etd:3683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gopallawa, Indiwari. “Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2.” 2015. Web. 20 Apr 2019.

Vancouver:

Gopallawa I. Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2. [Internet] [Thesis]. Michigan State University; 2015. [cited 2019 Apr 20]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3683.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gopallawa I. Angiotensin 1-7/Mas promotes alveolar epithelial cell survival through upregulation of map kinase phosphatase-2. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:3683

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

18. Lee, Jin Suk. Role of phosphatases in controlling arabidopsis mapk signalling cascades .

Degree: 2008, University of British Columbia

 Plants possess integrated signalling networks that mediate the responses to various environmental conditions. Mitogen-activated protein kinases (MAPKs) constitute a highly conserved family of enzymes in… (more)

Subjects/Keywords: Mitogen activated protein kinase phosphatase; Genetics

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APA (6th Edition):

Lee, J. S. (2008). Role of phosphatases in controlling arabidopsis mapk signalling cascades . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Jin Suk. “Role of phosphatases in controlling arabidopsis mapk signalling cascades .” 2008. Thesis, University of British Columbia. Accessed April 20, 2019. http://hdl.handle.net/2429/732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Jin Suk. “Role of phosphatases in controlling arabidopsis mapk signalling cascades .” 2008. Web. 20 Apr 2019.

Vancouver:

Lee JS. Role of phosphatases in controlling arabidopsis mapk signalling cascades . [Internet] [Thesis]. University of British Columbia; 2008. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/2429/732.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee JS. Role of phosphatases in controlling arabidopsis mapk signalling cascades . [Thesis]. University of British Columbia; 2008. Available from: http://hdl.handle.net/2429/732

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

19. Torabi, Mohammad Ali. Effects of deoxynivalenol and deepoxy-deoxynivalenol on bovine ovarian theca cell function .

Degree: 2017, Université de Montréal

 La mycotoxine déoxynivalénol (DON) et son métabolite déépoxy-déoxynivalenol (DOM-1) ont des effets significatifs sur la modification de la fonction des cellules thècales de l’ovaire bovin.… (more)

Subjects/Keywords: Phosphoprotéome; Les cellules de la thèque; Déoxynivalénol; Déépoxy-déoxynivalenol; Protéines kinases activées par des mitogènes; La prolifération; Phosphoproteome; Theca cells; Deoxynivalenol; Deepoxy-deoxynivalenol; Mitogen-activated protein kinases; Proliferation

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APA (6th Edition):

Torabi, M. A. (2017). Effects of deoxynivalenol and deepoxy-deoxynivalenol on bovine ovarian theca cell function . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/19167

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Torabi, Mohammad Ali. “Effects of deoxynivalenol and deepoxy-deoxynivalenol on bovine ovarian theca cell function .” 2017. Thesis, Université de Montréal. Accessed April 20, 2019. http://hdl.handle.net/1866/19167.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Torabi, Mohammad Ali. “Effects of deoxynivalenol and deepoxy-deoxynivalenol on bovine ovarian theca cell function .” 2017. Web. 20 Apr 2019.

Vancouver:

Torabi MA. Effects of deoxynivalenol and deepoxy-deoxynivalenol on bovine ovarian theca cell function . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1866/19167.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Torabi MA. Effects of deoxynivalenol and deepoxy-deoxynivalenol on bovine ovarian theca cell function . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/19167

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

20. 鄭軍偉.; Cheng, Kwan-wai. Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase.

Degree: Master of Medical Sciences, 2005, University of Hong Kong

published_or_final_version

Medical Sciences

Master

Master of Medical Sciences

Subjects/Keywords: Nucleosides.; Mitogen-activated protein kinases.; Genetic regulation.; Protein kinase C.

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APA (6th Edition):

鄭軍偉.; Cheng, K. (2005). Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase. (Masters Thesis). University of Hong Kong. Retrieved from Cheng, K. [鄭軍偉]. (2005). Regulation of equilibrative nucleoside transporter-1 by protein kinase C and mitogen-activating protein kinase. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4500998 ; http://dx.doi.org/10.5353/th_b4500998 ; http://hdl.handle.net/10722/131551

Chicago Manual of Style (16th Edition):

鄭軍偉.; Cheng, Kwan-wai. “Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase.” 2005. Masters Thesis, University of Hong Kong. Accessed April 20, 2019. Cheng, K. [鄭軍偉]. (2005). Regulation of equilibrative nucleoside transporter-1 by protein kinase C and mitogen-activating protein kinase. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4500998 ; http://dx.doi.org/10.5353/th_b4500998 ; http://hdl.handle.net/10722/131551.

MLA Handbook (7th Edition):

鄭軍偉.; Cheng, Kwan-wai. “Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase.” 2005. Web. 20 Apr 2019.

Vancouver:

鄭軍偉.; Cheng K. Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase. [Internet] [Masters thesis]. University of Hong Kong; 2005. [cited 2019 Apr 20]. Available from: Cheng, K. [鄭軍偉]. (2005). Regulation of equilibrative nucleoside transporter-1 by protein kinase C and mitogen-activating protein kinase. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4500998 ; http://dx.doi.org/10.5353/th_b4500998 ; http://hdl.handle.net/10722/131551.

Council of Science Editors:

鄭軍偉.; Cheng K. Regulation of equilibrative nucleoside transporter-1 by protein kinaseC and mitogen-activating protein kinase. [Masters Thesis]. University of Hong Kong; 2005. Available from: Cheng, K. [鄭軍偉]. (2005). Regulation of equilibrative nucleoside transporter-1 by protein kinase C and mitogen-activating protein kinase. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4500998 ; http://dx.doi.org/10.5353/th_b4500998 ; http://hdl.handle.net/10722/131551

21. Varela-Nieto, Isabel. Insulin receptor substrate 2 (IRS2)-deficient mice show sensorineural hearing loss that is delayed by concomitant protein tyrosine phosphatase 1B (PTP1B) loss of function.

Degree: 2018, Feinstein Institute for Medical Research

Subjects/Keywords: Cochlea; Mitogen-Activated Protein Kinases; Protein Tyrosine Phosphatase; Insulin Receptor Substrate Proteins; Medicina

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APA (6th Edition):

Varela-Nieto, I. (2018). Insulin receptor substrate 2 (IRS2)-deficient mice show sensorineural hearing loss that is delayed by concomitant protein tyrosine phosphatase 1B (PTP1B) loss of function. (Thesis). Feinstein Institute for Medical Research. Retrieved from http://hdl.handle.net/10486/663875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Varela-Nieto, Isabel. “Insulin receptor substrate 2 (IRS2)-deficient mice show sensorineural hearing loss that is delayed by concomitant protein tyrosine phosphatase 1B (PTP1B) loss of function.” 2018. Thesis, Feinstein Institute for Medical Research. Accessed April 20, 2019. http://hdl.handle.net/10486/663875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Varela-Nieto, Isabel. “Insulin receptor substrate 2 (IRS2)-deficient mice show sensorineural hearing loss that is delayed by concomitant protein tyrosine phosphatase 1B (PTP1B) loss of function.” 2018. Web. 20 Apr 2019.

Vancouver:

Varela-Nieto I. Insulin receptor substrate 2 (IRS2)-deficient mice show sensorineural hearing loss that is delayed by concomitant protein tyrosine phosphatase 1B (PTP1B) loss of function. [Internet] [Thesis]. Feinstein Institute for Medical Research; 2018. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/10486/663875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Varela-Nieto I. Insulin receptor substrate 2 (IRS2)-deficient mice show sensorineural hearing loss that is delayed by concomitant protein tyrosine phosphatase 1B (PTP1B) loss of function. [Thesis]. Feinstein Institute for Medical Research; 2018. Available from: http://hdl.handle.net/10486/663875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Grenoble

22. Kragelj, Jaka. Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK.

Degree: Docteur es, Biologie structurale et nanobiologie, 2014, Université de Grenoble

Les voies de transduction du signal cellulaire permettent aux cellules de répondre aux signaux de l'environnement et de les traiter. Les voies de transduction de… (more)

Subjects/Keywords: RMN; Couplages dipolaires résiduels; Structure des protéines; Kinases kinases activees par un mitogene; Signalisation des MAP Kinases; Proteines intrinsèquement; RMN; Couplages dipolaires residuels; Protein structure; Mitogen-activated protein kinase kinases; MAP Kinase Signaling; Intrinsically disordered proteins; 570

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APA (6th Edition):

Kragelj, J. (2014). Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2014GRENV060

Chicago Manual of Style (16th Edition):

Kragelj, Jaka. “Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK.” 2014. Doctoral Dissertation, Université de Grenoble. Accessed April 20, 2019. http://www.theses.fr/2014GRENV060.

MLA Handbook (7th Edition):

Kragelj, Jaka. “Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK.” 2014. Web. 20 Apr 2019.

Vancouver:

Kragelj J. Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK. [Internet] [Doctoral dissertation]. Université de Grenoble; 2014. [cited 2019 Apr 20]. Available from: http://www.theses.fr/2014GRENV060.

Council of Science Editors:

Kragelj J. Structure and dynamics of intrinsically disordered regions of MAPK signalling proteins : Structure et dynamique des régions intrinsèquement désordonnées des MAPK. [Doctoral Dissertation]. Université de Grenoble; 2014. Available from: http://www.theses.fr/2014GRENV060


University of Oulu

23. Kaikkonen, L. (Leena). p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function.

Degree: 2014, University of Oulu

Abstract Cardiovascular diseases are the leading causes of death in the developed countries and their incidence is not expected to decrease in the future. There… (more)

Subjects/Keywords: B-type natriuretic peptide; GATA-4 transcription factor; heart failure; mitogen-activated protein kinases; myocardial contraction; p38 mitogen-activated protein kinase; ventricular remodeling; B-tyypin natriureettinen peptidi; mitogeeniaktivoituvat proteiinikinaasit; p38 mitogeeniaktivoituva proteiinikinaasi; sydämen vajaatoiminta; sydänlihaksen supistuminen; sydänlihaksen uudelleenmuovautuminen; transkriptiotekijä GATA-4

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APA (6th Edition):

Kaikkonen, L. (. (2014). p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526205038

Chicago Manual of Style (16th Edition):

Kaikkonen, L (Leena). “p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function.” 2014. Doctoral Dissertation, University of Oulu. Accessed April 20, 2019. http://urn.fi/urn:isbn:9789526205038.

MLA Handbook (7th Edition):

Kaikkonen, L (Leena). “p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function.” 2014. Web. 20 Apr 2019.

Vancouver:

Kaikkonen L(. p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function. [Internet] [Doctoral dissertation]. University of Oulu; 2014. [cited 2019 Apr 20]. Available from: http://urn.fi/urn:isbn:9789526205038.

Council of Science Editors:

Kaikkonen L(. p38 mitogen-activated protein kinase and transcription factor GATA-4 in the regulation of cardiomyocyte function. [Doctoral Dissertation]. University of Oulu; 2014. Available from: http://urn.fi/urn:isbn:9789526205038

24. Brancho, Deborah Marie. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2005, U of Massachusetts : Med

  The c-Jun NH2-terminal kinase (JNK) group and the p38 group of mitogen-activated protein kinases (MAPK) are stress-activated protein kinases that regulate cell proliferation, differentiation,… (more)

Subjects/Keywords: p38 Mitogen-Activated Protein Kinases; JNK Mitogen-Activated Protein Kinases; Mitogen-Activated Protein Kinase Kinases; MAP Kinase Signaling System; Cell Differentiation; Adipocytes; Amino Acids, Peptides, and Proteins; Cells; Enzymes and Coenzymes

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APA (6th Edition):

Brancho, D. M. (2005). Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/101

Chicago Manual of Style (16th Edition):

Brancho, Deborah Marie. “Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.” 2005. Doctoral Dissertation, U of Massachusetts : Med. Accessed April 20, 2019. https://escholarship.umassmed.edu/gsbs_diss/101.

MLA Handbook (7th Edition):

Brancho, Deborah Marie. “Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation.” 2005. Web. 20 Apr 2019.

Vancouver:

Brancho DM. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2005. [cited 2019 Apr 20]. Available from: https://escholarship.umassmed.edu/gsbs_diss/101.

Council of Science Editors:

Brancho DM. Regulation and Function of Stress-Activated Protein Kinase Signal Transduction Pathways: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2005. Available from: https://escholarship.umassmed.edu/gsbs_diss/101

25. Hsieh, Ricardo. Melanoma primário da mucosa oral: estudo imunoistoquímico e molecular da via da MAPK.

Degree: PhD, Dermatologia, 2012, University of São Paulo

INTRODUÇÃO: O melanoma primário da cavidade oral é uma neoplasia agressiva, rara e originada a partir da proliferação de melanócitos malignos da mucosa. Ele representa… (more)

Subjects/Keywords: Análise mutacional de DNA; DNA mutational analysis; Immunohistochemistry; Imunoistoquímica; Kinases mitogen-activated protein kinase; Melanoma; Melanoma; Mucosa oral; Oral mucosa; Pirosequenciamento; Pyrosequencing; Quinases de proteína quinase ativadas por mitógeno

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APA (6th Edition):

Hsieh, R. (2012). Melanoma primário da mucosa oral: estudo imunoistoquímico e molecular da via da MAPK. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5133/tde-10092012-135829/ ;

Chicago Manual of Style (16th Edition):

Hsieh, Ricardo. “Melanoma primário da mucosa oral: estudo imunoistoquímico e molecular da via da MAPK.” 2012. Doctoral Dissertation, University of São Paulo. Accessed April 20, 2019. http://www.teses.usp.br/teses/disponiveis/5/5133/tde-10092012-135829/ ;.

MLA Handbook (7th Edition):

Hsieh, Ricardo. “Melanoma primário da mucosa oral: estudo imunoistoquímico e molecular da via da MAPK.” 2012. Web. 20 Apr 2019.

Vancouver:

Hsieh R. Melanoma primário da mucosa oral: estudo imunoistoquímico e molecular da via da MAPK. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Apr 20]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5133/tde-10092012-135829/ ;.

Council of Science Editors:

Hsieh R. Melanoma primário da mucosa oral: estudo imunoistoquímico e molecular da via da MAPK. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5133/tde-10092012-135829/ ;


Université de Montréal

26. Daigle, Caroline. Expansion d'une nouvelle famille de protéines kinases (MAPKKKs) impliquée dans le développement reproductif chez les Solanacées .

Degree: 2017, Université de Montréal

 Les cascades de Mitogen-Activated Protein Kinases (MAPKs) sont présentes chez tous les eucaryotes et permettent la transduction des signaux de l’extérieur vers l’intérieur de la… (more)

Subjects/Keywords: Signalisation; Mitogen-Activated Protein Kinases (MAPKs); Développement; Gamétophyte femelle; Ovule; Gamétophyte mâle; Pollen; Solanacées; Évolution; Signaling; Development; Female gametophyte; Male gametophyte; Solanaceae; Evolution

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Daigle, C. (2017). Expansion d'une nouvelle famille de protéines kinases (MAPKKKs) impliquée dans le développement reproductif chez les Solanacées . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/18509

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Daigle, Caroline. “Expansion d'une nouvelle famille de protéines kinases (MAPKKKs) impliquée dans le développement reproductif chez les Solanacées .” 2017. Thesis, Université de Montréal. Accessed April 20, 2019. http://hdl.handle.net/1866/18509.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Daigle, Caroline. “Expansion d'une nouvelle famille de protéines kinases (MAPKKKs) impliquée dans le développement reproductif chez les Solanacées .” 2017. Web. 20 Apr 2019.

Vancouver:

Daigle C. Expansion d'une nouvelle famille de protéines kinases (MAPKKKs) impliquée dans le développement reproductif chez les Solanacées . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1866/18509.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Daigle C. Expansion d'une nouvelle famille de protéines kinases (MAPKKKs) impliquée dans le développement reproductif chez les Solanacées . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/18509

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oulu

27. Tenhunen, O. (Olli). Mitogen-activated protein kinases and transcription factors during increased cardiac workload and remodelling.

Degree: 2006, University of Oulu

 Abstract Cardiac hypertrophy and remodelling are mechanisms of adaptation to increased workload and acute injuries of the heart. In the long-term, these initially beneficial mechanisms… (more)

Subjects/Keywords: mitogen-activated protein kinases; signal transduction; transcription factors; ventricular remodelling

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APA (6th Edition):

Tenhunen, O. (. (2006). Mitogen-activated protein kinases and transcription factors during increased cardiac workload and remodelling. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9514281934

Chicago Manual of Style (16th Edition):

Tenhunen, O (Olli). “Mitogen-activated protein kinases and transcription factors during increased cardiac workload and remodelling.” 2006. Doctoral Dissertation, University of Oulu. Accessed April 20, 2019. http://urn.fi/urn:isbn:9514281934.

MLA Handbook (7th Edition):

Tenhunen, O (Olli). “Mitogen-activated protein kinases and transcription factors during increased cardiac workload and remodelling.” 2006. Web. 20 Apr 2019.

Vancouver:

Tenhunen O(. Mitogen-activated protein kinases and transcription factors during increased cardiac workload and remodelling. [Internet] [Doctoral dissertation]. University of Oulu; 2006. [cited 2019 Apr 20]. Available from: http://urn.fi/urn:isbn:9514281934.

Council of Science Editors:

Tenhunen O(. Mitogen-activated protein kinases and transcription factors during increased cardiac workload and remodelling. [Doctoral Dissertation]. University of Oulu; 2006. Available from: http://urn.fi/urn:isbn:9514281934

28. Σέρτη, Ελισάβετ. Μελέτη της τροποποίησης της σηματοδότησης των MAPKs-p38 παρουσία των δομικών πρωτεϊνών του ιού της ηπατίτιδας C ( HCV).

Degree: 2010, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Hepatitis C virus (HCV) is a major cause of chronic hepatitis worldwide and often leads to chronic liver disease and hepatocellular carcinoma. The virus typically… (more)

Subjects/Keywords: Ηπατίτιδα C, Ιός (HCV); Καψίδια; Σηματοδότηση; Μιτογόνο, Ενεργοποιούμενες κινάσες; Τ-λεμφοκύτταρα; Hepatitis C, Virus (HCV); Particles; Signalling; Mitogen activated protein kinases (MARKS); T-lymphocytes

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APA (6th Edition):

Σέρτη, . . (2010). Μελέτη της τροποποίησης της σηματοδότησης των MAPKs-p38 παρουσία των δομικών πρωτεϊνών του ιού της ηπατίτιδας C ( HCV). (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/23517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Σέρτη, Ελισάβετ. “Μελέτη της τροποποίησης της σηματοδότησης των MAPKs-p38 παρουσία των δομικών πρωτεϊνών του ιού της ηπατίτιδας C ( HCV).” 2010. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 20, 2019. http://hdl.handle.net/10442/hedi/23517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Σέρτη, Ελισάβετ. “Μελέτη της τροποποίησης της σηματοδότησης των MAPKs-p38 παρουσία των δομικών πρωτεϊνών του ιού της ηπατίτιδας C ( HCV).” 2010. Web. 20 Apr 2019.

Vancouver:

Σέρτη . Μελέτη της τροποποίησης της σηματοδότησης των MAPKs-p38 παρουσία των δομικών πρωτεϊνών του ιού της ηπατίτιδας C ( HCV). [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2010. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/10442/hedi/23517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Σέρτη . Μελέτη της τροποποίησης της σηματοδότησης των MAPKs-p38 παρουσία των δομικών πρωτεϊνών του ιού της ηπατίτιδας C ( HCV). [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2010. Available from: http://hdl.handle.net/10442/hedi/23517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Australia

29. Gebski, Bijanka L. Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy.

Degree: PhD, 2009, University of Western Australia

 [Truncated abstract] The pro-inflammatory cytokine tumour necrosis factor (TNF) has a critical role in skeletal muscle atrophy. The catabolic effect of TNF is partially due… (more)

Subjects/Keywords: JNK mitogen-activated protein kinases; Insulin-like growth factor-binding proteins; Tumor necrosis factor; Duchenne muscular dystrophy; TNF; JNK; IGF-1; Muscle

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gebski, B. L. (2009). Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Gebski, Bijanka L. “Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy.” 2009. Doctoral Dissertation, University of Western Australia. Accessed April 20, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Gebski, Bijanka L. “Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy.” 2009. Web. 20 Apr 2019.

Vancouver:

Gebski BL. Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy. [Internet] [Doctoral dissertation]. University of Western Australia; 2009. [cited 2019 Apr 20]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01.

Council of Science Editors:

Gebski BL. Investigating TNF inhibition of IGF-1 signalling via JNK in cell culture models of skeletal muscle atrophy. [Doctoral Dissertation]. University of Western Australia; 2009. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=12898&local_base=GEN01-INS01

30. Schmitt, John Michael. cAMP regulation of ERKs and cell growth.

Degree: PhD, 2002, Oregon Health Sciences University

Subjects/Keywords: Cyclic AMP; Mitogen-Activated Protein Kinases; Cell Division; rap1 GTP-Binding Proteins

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schmitt, J. M. (2002). cAMP regulation of ERKs and cell growth. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M46971WH ; http://digitalcommons.ohsu.edu/etd/3196

Chicago Manual of Style (16th Edition):

Schmitt, John Michael. “cAMP regulation of ERKs and cell growth.” 2002. Doctoral Dissertation, Oregon Health Sciences University. Accessed April 20, 2019. doi:10.6083/M46971WH ; http://digitalcommons.ohsu.edu/etd/3196.

MLA Handbook (7th Edition):

Schmitt, John Michael. “cAMP regulation of ERKs and cell growth.” 2002. Web. 20 Apr 2019.

Vancouver:

Schmitt JM. cAMP regulation of ERKs and cell growth. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 2002. [cited 2019 Apr 20]. Available from: doi:10.6083/M46971WH ; http://digitalcommons.ohsu.edu/etd/3196.

Council of Science Editors:

Schmitt JM. cAMP regulation of ERKs and cell growth. [Doctoral Dissertation]. Oregon Health Sciences University; 2002. Available from: doi:10.6083/M46971WH ; http://digitalcommons.ohsu.edu/etd/3196

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