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You searched for subject:(microglia). Showing records 1 – 30 of 654 total matches.

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1. Conner, Kelly R. EFFECTS OF AT1R ANTAGONIST L-158,809 ON NORMAL BRAIN TISSUE INJURY FOLLOWING WHOLE-BRAIN IRRADIATION.

Degree: 2009, Wake Forest University

 Primary and metastatic brain cancers are commonly treated with partial or whole-brain irradiation (WBI). Twenty to fifty percent of patients that receive WBI develop progressive… (more)

Subjects/Keywords: microglia

…20 CHAPTER II Figure 1 Markers of microglia, proliferating cells, and immature neurons… …72 Figure 3 Quantitative analysis of proliferating and activated microglia in the DG GCL… …112 Figure 2 Markers of microglia, proliferating cells, activated microglia, and immature… …microglia, and neurons (14,17,31). Numerous animal studies have revealed that these… …increases in activated microglia occurs concomitant with reductions in neurogenesis… 

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APA (6th Edition):

Conner, K. R. (2009). EFFECTS OF AT1R ANTAGONIST L-158,809 ON NORMAL BRAIN TISSUE INJURY FOLLOWING WHOLE-BRAIN IRRADIATION. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/14666

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Conner, Kelly R. “EFFECTS OF AT1R ANTAGONIST L-158,809 ON NORMAL BRAIN TISSUE INJURY FOLLOWING WHOLE-BRAIN IRRADIATION.” 2009. Thesis, Wake Forest University. Accessed November 26, 2020. http://hdl.handle.net/10339/14666.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Conner, Kelly R. “EFFECTS OF AT1R ANTAGONIST L-158,809 ON NORMAL BRAIN TISSUE INJURY FOLLOWING WHOLE-BRAIN IRRADIATION.” 2009. Web. 26 Nov 2020.

Vancouver:

Conner KR. EFFECTS OF AT1R ANTAGONIST L-158,809 ON NORMAL BRAIN TISSUE INJURY FOLLOWING WHOLE-BRAIN IRRADIATION. [Internet] [Thesis]. Wake Forest University; 2009. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10339/14666.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Conner KR. EFFECTS OF AT1R ANTAGONIST L-158,809 ON NORMAL BRAIN TISSUE INJURY FOLLOWING WHOLE-BRAIN IRRADIATION. [Thesis]. Wake Forest University; 2009. Available from: http://hdl.handle.net/10339/14666

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McGill University

2. Williams, Kenneth C. Biology of Adult Human Microglia Studied in vitro and in situ: Immune Accessory and Effector Functions.

Degree: PhD, Department of Neurology and Neurosurgery, 1993, McGill University

This thesis studying the biology of adult human derived microglia in vitro and in situ, assessed putative immune accessory/effector functions of microglia and correlated in… (more)

Subjects/Keywords: Microglia

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APA (6th Edition):

Williams, K. C. (1993). Biology of Adult Human Microglia Studied in vitro and in situ: Immune Accessory and Effector Functions. (Doctoral Dissertation). McGill University. Retrieved from https://escholarship.mcgill.ca/downloads/jm214r524.pdf ; https://escholarship.mcgill.ca/concern/theses/w9505273t

Chicago Manual of Style (16th Edition):

Williams, Kenneth C. “Biology of Adult Human Microglia Studied in vitro and in situ: Immune Accessory and Effector Functions.” 1993. Doctoral Dissertation, McGill University. Accessed November 26, 2020. https://escholarship.mcgill.ca/downloads/jm214r524.pdf ; https://escholarship.mcgill.ca/concern/theses/w9505273t.

MLA Handbook (7th Edition):

Williams, Kenneth C. “Biology of Adult Human Microglia Studied in vitro and in situ: Immune Accessory and Effector Functions.” 1993. Web. 26 Nov 2020.

Vancouver:

Williams KC. Biology of Adult Human Microglia Studied in vitro and in situ: Immune Accessory and Effector Functions. [Internet] [Doctoral dissertation]. McGill University; 1993. [cited 2020 Nov 26]. Available from: https://escholarship.mcgill.ca/downloads/jm214r524.pdf ; https://escholarship.mcgill.ca/concern/theses/w9505273t.

Council of Science Editors:

Williams KC. Biology of Adult Human Microglia Studied in vitro and in situ: Immune Accessory and Effector Functions. [Doctoral Dissertation]. McGill University; 1993. Available from: https://escholarship.mcgill.ca/downloads/jm214r524.pdf ; https://escholarship.mcgill.ca/concern/theses/w9505273t

3. Ritzau, Rachel Nicole. Restoration of microglial transporter expression by NRF2 activation.

Degree: MS, Toxicology, 2017, Rutgers University

 Activation of brain microglia can be both beneficial and detrimental during the pathogenesis of neuroinflammation. Healthy microglia will propagate short-term inflammation as a means of… (more)

Subjects/Keywords: Microglia

…2. mRNA Expression of Nrf2 Target Genes in Activated Microglia …..61 3-3. mRNA Expression… …of Efflux Transporters in Activated Microglia ….62 3-4. Quantification of the Nuclear… …and microglia. While the fourth type of glial cell, Schwann cells are located exclusively in… …microglia are the resident immune cells of the CNS. Therefore, microglia respond to both physical… …trauma and various pathological assaults to the brain (Hof, et al., 2013). Microglia… 

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APA (6th Edition):

Ritzau, R. N. (2017). Restoration of microglial transporter expression by NRF2 activation. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/55671/

Chicago Manual of Style (16th Edition):

Ritzau, Rachel Nicole. “Restoration of microglial transporter expression by NRF2 activation.” 2017. Masters Thesis, Rutgers University. Accessed November 26, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/55671/.

MLA Handbook (7th Edition):

Ritzau, Rachel Nicole. “Restoration of microglial transporter expression by NRF2 activation.” 2017. Web. 26 Nov 2020.

Vancouver:

Ritzau RN. Restoration of microglial transporter expression by NRF2 activation. [Internet] [Masters thesis]. Rutgers University; 2017. [cited 2020 Nov 26]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55671/.

Council of Science Editors:

Ritzau RN. Restoration of microglial transporter expression by NRF2 activation. [Masters Thesis]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55671/

4. 黄, 宏智. Activation of microglial cells in the trigeminal subnucleus caudalis evoked by inflammatory stimulation of the oral mucosa : 口腔粘膜への侵害刺激により誘発される三叉神経脊髄路核尾側亜核内マイクログリアへの活性化.

Degree: 博士(歯学), 2016, Osaka Dental University / 大阪歯科大学

To study the inflammatory hyperalgesia induced by an acute noxious stimulation of oral mucosa with 5% formalin (5%FOR), we performed an immunohistochemical study on the… (more)

Subjects/Keywords: microglia; Vc; nociception; microglia; Vc; nociception

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APA (6th Edition):

黄, . (2016). Activation of microglial cells in the trigeminal subnucleus caudalis evoked by inflammatory stimulation of the oral mucosa : 口腔粘膜への侵害刺激により誘発される三叉神経脊髄路核尾側亜核内マイクログリアへの活性化. (Thesis). Osaka Dental University / 大阪歯科大学. Retrieved from http://id.nii.ac.jp/1392/00000059/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

黄, 宏智. “Activation of microglial cells in the trigeminal subnucleus caudalis evoked by inflammatory stimulation of the oral mucosa : 口腔粘膜への侵害刺激により誘発される三叉神経脊髄路核尾側亜核内マイクログリアへの活性化.” 2016. Thesis, Osaka Dental University / 大阪歯科大学. Accessed November 26, 2020. http://id.nii.ac.jp/1392/00000059/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

黄, 宏智. “Activation of microglial cells in the trigeminal subnucleus caudalis evoked by inflammatory stimulation of the oral mucosa : 口腔粘膜への侵害刺激により誘発される三叉神経脊髄路核尾側亜核内マイクログリアへの活性化.” 2016. Web. 26 Nov 2020.

Vancouver:

黄 . Activation of microglial cells in the trigeminal subnucleus caudalis evoked by inflammatory stimulation of the oral mucosa : 口腔粘膜への侵害刺激により誘発される三叉神経脊髄路核尾側亜核内マイクログリアへの活性化. [Internet] [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. [cited 2020 Nov 26]. Available from: http://id.nii.ac.jp/1392/00000059/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

黄 . Activation of microglial cells in the trigeminal subnucleus caudalis evoked by inflammatory stimulation of the oral mucosa : 口腔粘膜への侵害刺激により誘発される三叉神経脊髄路核尾側亜核内マイクログリアへの活性化. [Thesis]. Osaka Dental University / 大阪歯科大学; 2016. Available from: http://id.nii.ac.jp/1392/00000059/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Santos, Adriessa Aparecida dos. Melatonina sintetizada por microglias de cerebelo em cultura regula o processo de fagocitose.

Degree: Mestrado, Fisiologia Geral, 2015, University of São Paulo

A melatonina é uma indolamina sintetizada principalmente pela glândula pineal, cuja função está associada à marcação do escuro. Além deste papel cronobiótico, a melatonina também… (more)

Subjects/Keywords: Fagocitose; Melatonin; Melatonina; Microglia; Microglia; Phagocytosis

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APA (6th Edition):

Santos, A. A. d. (2015). Melatonina sintetizada por microglias de cerebelo em cultura regula o processo de fagocitose. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41135/tde-17072015-153623/ ;

Chicago Manual of Style (16th Edition):

Santos, Adriessa Aparecida dos. “Melatonina sintetizada por microglias de cerebelo em cultura regula o processo de fagocitose.” 2015. Masters Thesis, University of São Paulo. Accessed November 26, 2020. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-17072015-153623/ ;.

MLA Handbook (7th Edition):

Santos, Adriessa Aparecida dos. “Melatonina sintetizada por microglias de cerebelo em cultura regula o processo de fagocitose.” 2015. Web. 26 Nov 2020.

Vancouver:

Santos AAd. Melatonina sintetizada por microglias de cerebelo em cultura regula o processo de fagocitose. [Internet] [Masters thesis]. University of São Paulo; 2015. [cited 2020 Nov 26]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-17072015-153623/ ;.

Council of Science Editors:

Santos AAd. Melatonina sintetizada por microglias de cerebelo em cultura regula o processo de fagocitose. [Masters Thesis]. University of São Paulo; 2015. Available from: http://www.teses.usp.br/teses/disponiveis/41/41135/tde-17072015-153623/ ;


University of Rochester

6. McConnell, Sarah Elizabeth Allen; Opanashuk, Lisa A. Methamphetamine-induced Neuroinflammation and Neurotoxicity:a Role for Interleukin-1β.

Degree: PhD, 2013, University of Rochester

 Methamphetamine (MA) is a potent, addictive psychostimulant abused by millions of people worldwide. MA exposure induces neurotoxicity, particularly in the striatum, where it damages dopaminergic… (more)

Subjects/Keywords: Microglia; Astrocytes; Striatum

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APA (6th Edition):

McConnell, Sarah Elizabeth Allen; Opanashuk, L. A. (2013). Methamphetamine-induced Neuroinflammation and Neurotoxicity:a Role for Interleukin-1β. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26781

Chicago Manual of Style (16th Edition):

McConnell, Sarah Elizabeth Allen; Opanashuk, Lisa A. “Methamphetamine-induced Neuroinflammation and Neurotoxicity:a Role for Interleukin-1β.” 2013. Doctoral Dissertation, University of Rochester. Accessed November 26, 2020. http://hdl.handle.net/1802/26781.

MLA Handbook (7th Edition):

McConnell, Sarah Elizabeth Allen; Opanashuk, Lisa A. “Methamphetamine-induced Neuroinflammation and Neurotoxicity:a Role for Interleukin-1β.” 2013. Web. 26 Nov 2020.

Vancouver:

McConnell, Sarah Elizabeth Allen; Opanashuk LA. Methamphetamine-induced Neuroinflammation and Neurotoxicity:a Role for Interleukin-1β. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1802/26781.

Council of Science Editors:

McConnell, Sarah Elizabeth Allen; Opanashuk LA. Methamphetamine-induced Neuroinflammation and Neurotoxicity:a Role for Interleukin-1β. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26781


University of Alberta

7. Kenawy, Sara M. The effects of glucose-induced metabolic injury on microglia activity and survival.

Degree: MS, Centre for Neuroscience, 2013, University of Alberta

 Glucose is the sole fuel for the brain in normal physiological conditions. Absence of glucose or its presence in high concentrations has been shown to… (more)

Subjects/Keywords: Diabetes; Glucose; Microglia

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APA (6th Edition):

Kenawy, S. M. (2013). The effects of glucose-induced metabolic injury on microglia activity and survival. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/12579s530

Chicago Manual of Style (16th Edition):

Kenawy, Sara M. “The effects of glucose-induced metabolic injury on microglia activity and survival.” 2013. Masters Thesis, University of Alberta. Accessed November 26, 2020. https://era.library.ualberta.ca/files/12579s530.

MLA Handbook (7th Edition):

Kenawy, Sara M. “The effects of glucose-induced metabolic injury on microglia activity and survival.” 2013. Web. 26 Nov 2020.

Vancouver:

Kenawy SM. The effects of glucose-induced metabolic injury on microglia activity and survival. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2020 Nov 26]. Available from: https://era.library.ualberta.ca/files/12579s530.

Council of Science Editors:

Kenawy SM. The effects of glucose-induced metabolic injury on microglia activity and survival. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/12579s530


Victoria University of Wellington

8. Stone, Sarrabeth Marie. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.

Degree: 2013, Victoria University of Wellington

 Multiple Sclerosis (MS) is an immune-mediated disease of the central nervous system (CNS) which causes demyelination and damage to the neuronal axons. MS is a… (more)

Subjects/Keywords: Immunology; Macrophage; Microglia

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APA (6th Edition):

Stone, S. M. (2013). Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/3072

Chicago Manual of Style (16th Edition):

Stone, Sarrabeth Marie. “Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.” 2013. Doctoral Dissertation, Victoria University of Wellington. Accessed November 26, 2020. http://hdl.handle.net/10063/3072.

MLA Handbook (7th Edition):

Stone, Sarrabeth Marie. “Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.” 2013. Web. 26 Nov 2020.

Vancouver:

Stone SM. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2013. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10063/3072.

Council of Science Editors:

Stone SM. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. [Doctoral Dissertation]. Victoria University of Wellington; 2013. Available from: http://hdl.handle.net/10063/3072

9. Garcia, Paulo Ricardo Medeiros. Signaling pathways involved in the modulation of microglial reactivity by the action of GDNF.

Degree: 2011, RCAAP

 Os processos inflamatórios, que ocorrem no sistema nervoso central (SNC), mediados pela activação das células da glia desempenham um papel importante na morte neuronal e… (more)

Subjects/Keywords: Microglia; Doença neurodegenerativa

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APA (6th Edition):

Garcia, P. R. M. (2011). Signaling pathways involved in the modulation of microglial reactivity by the action of GDNF. (Thesis). RCAAP. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:ubibliorum.ubi.pt:10400.6/2070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garcia, Paulo Ricardo Medeiros. “Signaling pathways involved in the modulation of microglial reactivity by the action of GDNF.” 2011. Thesis, RCAAP. Accessed November 26, 2020. http://www.rcaap.pt/detail.jsp?id=oai:ubibliorum.ubi.pt:10400.6/2070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garcia, Paulo Ricardo Medeiros. “Signaling pathways involved in the modulation of microglial reactivity by the action of GDNF.” 2011. Web. 26 Nov 2020.

Vancouver:

Garcia PRM. Signaling pathways involved in the modulation of microglial reactivity by the action of GDNF. [Internet] [Thesis]. RCAAP; 2011. [cited 2020 Nov 26]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:ubibliorum.ubi.pt:10400.6/2070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garcia PRM. Signaling pathways involved in the modulation of microglial reactivity by the action of GDNF. [Thesis]. RCAAP; 2011. Available from: http://www.rcaap.pt/detail.jsp?id=oai:ubibliorum.ubi.pt:10400.6/2070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

10. Pan, Geng. Potassium channel expression and function in the N9 murine microglial cell line.

Degree: PhD, 2012, University of Edinburgh

Microglia are immunocompetent cells in the central nervous system that have many similarities with macrophages of peripheral tissues. Their activation protects local cells from foreign… (more)

Subjects/Keywords: microglia; potassium channel

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APA (6th Edition):

Pan, G. (2012). Potassium channel expression and function in the N9 murine microglial cell line. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/8191

Chicago Manual of Style (16th Edition):

Pan, Geng. “Potassium channel expression and function in the N9 murine microglial cell line.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed November 26, 2020. http://hdl.handle.net/1842/8191.

MLA Handbook (7th Edition):

Pan, Geng. “Potassium channel expression and function in the N9 murine microglial cell line.” 2012. Web. 26 Nov 2020.

Vancouver:

Pan G. Potassium channel expression and function in the N9 murine microglial cell line. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1842/8191.

Council of Science Editors:

Pan G. Potassium channel expression and function in the N9 murine microglial cell line. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/8191


University of California – Irvine

11. Najafi, Allison Rachel. Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor Inhibition.

Degree: Biological Sciences, 2017, University of California – Irvine

 Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor InhibitionByAllison Rachel NajafiDoctor of Philosophy in Biological Sciences University of California, Irvine, 2017Associate Professor… (more)

Subjects/Keywords: Neurosciences; csf1r; depletion; microglia; repopulation

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APA (6th Edition):

Najafi, A. R. (2017). Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor Inhibition. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/9d2828h1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Najafi, Allison Rachel. “Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor Inhibition.” 2017. Thesis, University of California – Irvine. Accessed November 26, 2020. http://www.escholarship.org/uc/item/9d2828h1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Najafi, Allison Rachel. “Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor Inhibition.” 2017. Web. 26 Nov 2020.

Vancouver:

Najafi AR. Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor Inhibition. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2020 Nov 26]. Available from: http://www.escholarship.org/uc/item/9d2828h1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Najafi AR. Exploring Microglial Homeostasis and Repopulation Dynamics Using Colony-Stimulating Factor 1 Receptor Inhibition. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/9d2828h1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

12. Gofman, Larisa. Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses.

Degree: PhD, 2015, Temple University

Pathology

Ethanol (EtOH) abuse is the third leading cause of preventable death in the United States. Mounting evidence indicates that EtOH-induced neuropathology may result from… (more)

Subjects/Keywords: Neurosciences;

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APA (6th Edition):

Gofman, L. (2015). Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,315641

Chicago Manual of Style (16th Edition):

Gofman, Larisa. “Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses.” 2015. Doctoral Dissertation, Temple University. Accessed November 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,315641.

MLA Handbook (7th Edition):

Gofman, Larisa. “Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses.” 2015. Web. 26 Nov 2020.

Vancouver:

Gofman L. Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Nov 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,315641.

Council of Science Editors:

Gofman L. Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,315641


University of Alberta

13. Lai, Aaron. Determining factors in the differential activation of microglia.

Degree: PhD, Centre for Neuroscience, 2010, University of Alberta

Microglia, the resident immune cells of the central nervous system (CNS), become activated in response to danger signals given out by other cells when homeostasis… (more)

Subjects/Keywords: ATP; hypoxia; neuroinflammation; neuroscience; microglia

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APA (6th Edition):

Lai, A. (2010). Determining factors in the differential activation of microglia. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/tb09j6275

Chicago Manual of Style (16th Edition):

Lai, Aaron. “Determining factors in the differential activation of microglia.” 2010. Doctoral Dissertation, University of Alberta. Accessed November 26, 2020. https://era.library.ualberta.ca/files/tb09j6275.

MLA Handbook (7th Edition):

Lai, Aaron. “Determining factors in the differential activation of microglia.” 2010. Web. 26 Nov 2020.

Vancouver:

Lai A. Determining factors in the differential activation of microglia. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2020 Nov 26]. Available from: https://era.library.ualberta.ca/files/tb09j6275.

Council of Science Editors:

Lai A. Determining factors in the differential activation of microglia. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/tb09j6275


University of Alberta

14. Dhami, Kamaldeep S. The effects of antidepressants on the phenotype of activated microglia and ischemia-injured cortical neurons.

Degree: PhD, Centre for Neuroscience, 2013, University of Alberta

 Depression is one of the most common disorders appearing following a stroke and is also a major factor limiting recovery and rehabilitation in stroke patients.… (more)

Subjects/Keywords: Inflammation; Microglia; Excitotoxicity; Ischemia; Antidepressants

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APA (6th Edition):

Dhami, K. S. (2013). The effects of antidepressants on the phenotype of activated microglia and ischemia-injured cortical neurons. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/5x21tf82p

Chicago Manual of Style (16th Edition):

Dhami, Kamaldeep S. “The effects of antidepressants on the phenotype of activated microglia and ischemia-injured cortical neurons.” 2013. Doctoral Dissertation, University of Alberta. Accessed November 26, 2020. https://era.library.ualberta.ca/files/5x21tf82p.

MLA Handbook (7th Edition):

Dhami, Kamaldeep S. “The effects of antidepressants on the phenotype of activated microglia and ischemia-injured cortical neurons.” 2013. Web. 26 Nov 2020.

Vancouver:

Dhami KS. The effects of antidepressants on the phenotype of activated microglia and ischemia-injured cortical neurons. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2020 Nov 26]. Available from: https://era.library.ualberta.ca/files/5x21tf82p.

Council of Science Editors:

Dhami KS. The effects of antidepressants on the phenotype of activated microglia and ischemia-injured cortical neurons. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/5x21tf82p


University of Newcastle

15. Kongsui, Ratchaniporn. An examination of how environmental disturbances alter microglia morphology and function within the CNS.

Degree: PhD, 2017, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

Almost daily the role number of roles that microglia have been identified as playing a role in expands.… (more)

Subjects/Keywords: microglia; chronic stress; neuroinflammation; Iba1

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APA (6th Edition):

Kongsui, R. (2017). An examination of how environmental disturbances alter microglia morphology and function within the CNS. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1336107

Chicago Manual of Style (16th Edition):

Kongsui, Ratchaniporn. “An examination of how environmental disturbances alter microglia morphology and function within the CNS.” 2017. Doctoral Dissertation, University of Newcastle. Accessed November 26, 2020. http://hdl.handle.net/1959.13/1336107.

MLA Handbook (7th Edition):

Kongsui, Ratchaniporn. “An examination of how environmental disturbances alter microglia morphology and function within the CNS.” 2017. Web. 26 Nov 2020.

Vancouver:

Kongsui R. An examination of how environmental disturbances alter microglia morphology and function within the CNS. [Internet] [Doctoral dissertation]. University of Newcastle; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1959.13/1336107.

Council of Science Editors:

Kongsui R. An examination of how environmental disturbances alter microglia morphology and function within the CNS. [Doctoral Dissertation]. University of Newcastle; 2017. Available from: http://hdl.handle.net/1959.13/1336107


University of Toronto

16. Vincent, Catherine. Expression of Podosomes and Small-conductance Ca2+-activated K+ Channels in Cultured Microglia.

Degree: 2012, University of Toronto

The presence of microglia at sites of CNS injury can potentially shift the balance between neuronal survival and death; however, the mechanisms regulating their mobilization… (more)

Subjects/Keywords: Microglia; podosomes; SK3; SK; 0379

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APA (6th Edition):

Vincent, C. (2012). Expression of Podosomes and Small-conductance Ca2+-activated K+ Channels in Cultured Microglia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35155

Chicago Manual of Style (16th Edition):

Vincent, Catherine. “Expression of Podosomes and Small-conductance Ca2+-activated K+ Channels in Cultured Microglia.” 2012. Masters Thesis, University of Toronto. Accessed November 26, 2020. http://hdl.handle.net/1807/35155.

MLA Handbook (7th Edition):

Vincent, Catherine. “Expression of Podosomes and Small-conductance Ca2+-activated K+ Channels in Cultured Microglia.” 2012. Web. 26 Nov 2020.

Vancouver:

Vincent C. Expression of Podosomes and Small-conductance Ca2+-activated K+ Channels in Cultured Microglia. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1807/35155.

Council of Science Editors:

Vincent C. Expression of Podosomes and Small-conductance Ca2+-activated K+ Channels in Cultured Microglia. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/35155

17. Silva, Cátia Liliana Marques da. Dissecting the role of Profilin-1 in microglial cell function: the impact on ROS production .

Degree: 2015, Universidade de Aveiro

 Microglial cells are the resident immune cells of central nervous system (CNS) and the major players in neuroinflammation. These cells are also responsible for surveilling… (more)

Subjects/Keywords: Biologia molecular; Microglia; Bacteriófagos; Neurónios

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APA (6th Edition):

Silva, C. L. M. d. (2015). Dissecting the role of Profilin-1 in microglial cell function: the impact on ROS production . (Thesis). Universidade de Aveiro. Retrieved from http://hdl.handle.net/10773/15607

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Cátia Liliana Marques da. “Dissecting the role of Profilin-1 in microglial cell function: the impact on ROS production .” 2015. Thesis, Universidade de Aveiro. Accessed November 26, 2020. http://hdl.handle.net/10773/15607.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Cátia Liliana Marques da. “Dissecting the role of Profilin-1 in microglial cell function: the impact on ROS production .” 2015. Web. 26 Nov 2020.

Vancouver:

Silva CLMd. Dissecting the role of Profilin-1 in microglial cell function: the impact on ROS production . [Internet] [Thesis]. Universidade de Aveiro; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/10773/15607.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva CLMd. Dissecting the role of Profilin-1 in microglial cell function: the impact on ROS production . [Thesis]. Universidade de Aveiro; 2015. Available from: http://hdl.handle.net/10773/15607

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

18. Rezaeian Mehrabadi, Abbas. The role of microglial PARP-1 in Alzheimer’s disease.

Degree: Pharmacology and Therapeutics, 2017, University of Manitoba

 Alzheimer’s disease (AD) is a neurodegenerative disorder with profound chronic neuroinflammation. Our previous studies have identified poly(ADP-ribose) polymerase-1 (PARP-1) as a key driver of microglial… (more)

Subjects/Keywords: Alzheimer' Disease; Microglia; PARP-1

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APA (6th Edition):

Rezaeian Mehrabadi, A. (2017). The role of microglial PARP-1 in Alzheimer’s disease. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/32727

Chicago Manual of Style (16th Edition):

Rezaeian Mehrabadi, Abbas. “The role of microglial PARP-1 in Alzheimer’s disease.” 2017. Masters Thesis, University of Manitoba. Accessed November 26, 2020. http://hdl.handle.net/1993/32727.

MLA Handbook (7th Edition):

Rezaeian Mehrabadi, Abbas. “The role of microglial PARP-1 in Alzheimer’s disease.” 2017. Web. 26 Nov 2020.

Vancouver:

Rezaeian Mehrabadi A. The role of microglial PARP-1 in Alzheimer’s disease. [Internet] [Masters thesis]. University of Manitoba; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1993/32727.

Council of Science Editors:

Rezaeian Mehrabadi A. The role of microglial PARP-1 in Alzheimer’s disease. [Masters Thesis]. University of Manitoba; 2017. Available from: http://hdl.handle.net/1993/32727


University of Oxford

19. Lam Haces Gil, Karla G. Investigation into the epigenetic mechanisms involved in microglial activation in the animal model of multiple sclerosis.

Degree: PhD, 2013, University of Oxford

 In patients with multiple sclerosis (MS), microglia become activated due to the autoimmune inflammatory response which is directed against the central nervous system (CNS). Following… (more)

Subjects/Keywords: 616.8; Multiple Sclerosis; Epigenetics; Microglia

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APA (6th Edition):

Lam Haces Gil, K. G. (2013). Investigation into the epigenetic mechanisms involved in microglial activation in the animal model of multiple sclerosis. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:b4de0ef7-7588-4381-b099-9c8567805546 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655019

Chicago Manual of Style (16th Edition):

Lam Haces Gil, Karla G. “Investigation into the epigenetic mechanisms involved in microglial activation in the animal model of multiple sclerosis.” 2013. Doctoral Dissertation, University of Oxford. Accessed November 26, 2020. http://ora.ox.ac.uk/objects/uuid:b4de0ef7-7588-4381-b099-9c8567805546 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655019.

MLA Handbook (7th Edition):

Lam Haces Gil, Karla G. “Investigation into the epigenetic mechanisms involved in microglial activation in the animal model of multiple sclerosis.” 2013. Web. 26 Nov 2020.

Vancouver:

Lam Haces Gil KG. Investigation into the epigenetic mechanisms involved in microglial activation in the animal model of multiple sclerosis. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Nov 26]. Available from: http://ora.ox.ac.uk/objects/uuid:b4de0ef7-7588-4381-b099-9c8567805546 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655019.

Council of Science Editors:

Lam Haces Gil KG. Investigation into the epigenetic mechanisms involved in microglial activation in the animal model of multiple sclerosis. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:b4de0ef7-7588-4381-b099-9c8567805546 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.655019


University of Edinburgh

20. Vincenti, James Edward. Role of activation of microglia in neurodegenerative prion disease.

Degree: PhD, 2015, University of Edinburgh

 Prion diseases are a group of fatal neurodegenerative protein-misfolding diseases. Microglia, the resident myeloid cells found within the brain, have been shown to demonstrate a… (more)

Subjects/Keywords: 612.8; microglia; neurodegeneration; neuroimmunology; prion

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APA (6th Edition):

Vincenti, J. E. (2015). Role of activation of microglia in neurodegenerative prion disease. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/15928

Chicago Manual of Style (16th Edition):

Vincenti, James Edward. “Role of activation of microglia in neurodegenerative prion disease.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed November 26, 2020. http://hdl.handle.net/1842/15928.

MLA Handbook (7th Edition):

Vincenti, James Edward. “Role of activation of microglia in neurodegenerative prion disease.” 2015. Web. 26 Nov 2020.

Vancouver:

Vincenti JE. Role of activation of microglia in neurodegenerative prion disease. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1842/15928.

Council of Science Editors:

Vincenti JE. Role of activation of microglia in neurodegenerative prion disease. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/15928


University of Gothenburg / Göteborgs Universitet

21. Rymo, Simin. On the stimulatory effect of microglial cells on angiogenesis.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

 ABSTRACT Angiogenesis, the process by which new vessels sprout from pre-existing vessels, is fundamental to development, tissue growth and repair. The main aim of this… (more)

Subjects/Keywords: microglia; angiogenesis; aortic ring

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APA (6th Edition):

Rymo, S. (2011). On the stimulatory effect of microglial cells on angiogenesis. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/25576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rymo, Simin. “On the stimulatory effect of microglial cells on angiogenesis.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed November 26, 2020. http://hdl.handle.net/2077/25576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rymo, Simin. “On the stimulatory effect of microglial cells on angiogenesis.” 2011. Web. 26 Nov 2020.

Vancouver:

Rymo S. On the stimulatory effect of microglial cells on angiogenesis. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/2077/25576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rymo S. On the stimulatory effect of microglial cells on angiogenesis. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/25576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

22. Dharmarajan, Subramanian. Regulation of gliosis in the mouse retina.

Degree: 2017, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

The glial cells of the retina aid in function and maintenance of the retina. The macroglia, Muller cells and the… (more)

Subjects/Keywords: Gliosis; Retina; Microglia; BMP7; YAP

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APA (6th Edition):

Dharmarajan, S. (2017). Regulation of gliosis in the mouse retina. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/13556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dharmarajan, Subramanian. “Regulation of gliosis in the mouse retina.” 2017. Thesis, IUPUI. Accessed November 26, 2020. http://hdl.handle.net/1805/13556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dharmarajan, Subramanian. “Regulation of gliosis in the mouse retina.” 2017. Web. 26 Nov 2020.

Vancouver:

Dharmarajan S. Regulation of gliosis in the mouse retina. [Internet] [Thesis]. IUPUI; 2017. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1805/13556.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dharmarajan S. Regulation of gliosis in the mouse retina. [Thesis]. IUPUI; 2017. Available from: http://hdl.handle.net/1805/13556

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


New Jersey Institute of Technology

23. Padala, Nischal K. Astrocytes modulate microglial phenotype and dendritic cell-like properties.

Degree: PhD, Federated Department of Biological Sciences, 2013, New Jersey Institute of Technology

Microglia are the resident immune cells of the CNS. In the healthy CNS, they express negligible levels of MHC II molecules as well as… (more)

Subjects/Keywords: Microglia; Astrocytes; CNS; Biology

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APA (6th Edition):

Padala, N. K. (2013). Astrocytes modulate microglial phenotype and dendritic cell-like properties. (Doctoral Dissertation). New Jersey Institute of Technology. Retrieved from https://digitalcommons.njit.edu/dissertations/347

Chicago Manual of Style (16th Edition):

Padala, Nischal K. “Astrocytes modulate microglial phenotype and dendritic cell-like properties.” 2013. Doctoral Dissertation, New Jersey Institute of Technology. Accessed November 26, 2020. https://digitalcommons.njit.edu/dissertations/347.

MLA Handbook (7th Edition):

Padala, Nischal K. “Astrocytes modulate microglial phenotype and dendritic cell-like properties.” 2013. Web. 26 Nov 2020.

Vancouver:

Padala NK. Astrocytes modulate microglial phenotype and dendritic cell-like properties. [Internet] [Doctoral dissertation]. New Jersey Institute of Technology; 2013. [cited 2020 Nov 26]. Available from: https://digitalcommons.njit.edu/dissertations/347.

Council of Science Editors:

Padala NK. Astrocytes modulate microglial phenotype and dendritic cell-like properties. [Doctoral Dissertation]. New Jersey Institute of Technology; 2013. Available from: https://digitalcommons.njit.edu/dissertations/347


University of Minnesota

24. Cook, Jennifer. Complement-3a receptor involvement in peripheral and central neuropathic pain.

Degree: PhD, Neuroscience, 2019, University of Minnesota

 VGF (non-acronymic), a neuropeptide precursor protein related to the chromogranin family, has been implicated in neuroplasticity associated with depression, learning and memory, and chronic pain.… (more)

Subjects/Keywords: C3aR1; microglia; pain; SCI

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APA (6th Edition):

Cook, J. (2019). Complement-3a receptor involvement in peripheral and central neuropathic pain. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/209045

Chicago Manual of Style (16th Edition):

Cook, Jennifer. “Complement-3a receptor involvement in peripheral and central neuropathic pain.” 2019. Doctoral Dissertation, University of Minnesota. Accessed November 26, 2020. http://hdl.handle.net/11299/209045.

MLA Handbook (7th Edition):

Cook, Jennifer. “Complement-3a receptor involvement in peripheral and central neuropathic pain.” 2019. Web. 26 Nov 2020.

Vancouver:

Cook J. Complement-3a receptor involvement in peripheral and central neuropathic pain. [Internet] [Doctoral dissertation]. University of Minnesota; 2019. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/11299/209045.

Council of Science Editors:

Cook J. Complement-3a receptor involvement in peripheral and central neuropathic pain. [Doctoral Dissertation]. University of Minnesota; 2019. Available from: http://hdl.handle.net/11299/209045


University of Edinburgh

25. Davies, Claire Linzi. Myeloid cell involvement during the resolution of acute brain inflammation.

Degree: PhD, 2016, University of Edinburgh

 Excessive tissue-damaging inflammation can exacerbate acute brain injury, and non-resolving inflammation is implicated in chronic neurodegeneration. Understanding the mechanisms that resolve deleterious inflammation in the… (more)

Subjects/Keywords: 616.07; inflammation; brain; macrophages; microglia

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APA (6th Edition):

Davies, C. L. (2016). Myeloid cell involvement during the resolution of acute brain inflammation. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/25902

Chicago Manual of Style (16th Edition):

Davies, Claire Linzi. “Myeloid cell involvement during the resolution of acute brain inflammation.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed November 26, 2020. http://hdl.handle.net/1842/25902.

MLA Handbook (7th Edition):

Davies, Claire Linzi. “Myeloid cell involvement during the resolution of acute brain inflammation.” 2016. Web. 26 Nov 2020.

Vancouver:

Davies CL. Myeloid cell involvement during the resolution of acute brain inflammation. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1842/25902.

Council of Science Editors:

Davies CL. Myeloid cell involvement during the resolution of acute brain inflammation. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/25902

26. Grabert, Kathleen. Defining microglial phenotypic diversity and the impact of ageing.

Degree: PhD, 2015, University of Edinburgh

Microglia are the resident macrophages of the central nervous system (CNS) and, as key immune effector cells, form the first line of defence. Microglial cells… (more)

Subjects/Keywords: 612.8; microglia; ageing; phenotype

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APA (6th Edition):

Grabert, K. (2015). Defining microglial phenotypic diversity and the impact of ageing. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17615

Chicago Manual of Style (16th Edition):

Grabert, Kathleen. “Defining microglial phenotypic diversity and the impact of ageing.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed November 26, 2020. http://hdl.handle.net/1842/17615.

MLA Handbook (7th Edition):

Grabert, Kathleen. “Defining microglial phenotypic diversity and the impact of ageing.” 2015. Web. 26 Nov 2020.

Vancouver:

Grabert K. Defining microglial phenotypic diversity and the impact of ageing. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1842/17615.

Council of Science Editors:

Grabert K. Defining microglial phenotypic diversity and the impact of ageing. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/17615


Freie Universität Berlin

27. Schwendinger, Nina. Der Einfluss von dem Wirt enstammenden Osteopontin auf Gliome.

Degree: 2020, Freie Universität Berlin

 Das Glioblastom ist der häufigste maligne Tumor des Gehirns. Als WHO Grad IV Tumor zeichnet sich dieser Hirntumor durch einen schnellen prä- und postoperativen Progress… (more)

Subjects/Keywords: osteopontin; glioma; microglia; ddc:610

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APA (6th Edition):

Schwendinger, N. (2020). Der Einfluss von dem Wirt enstammenden Osteopontin auf Gliome. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-27533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schwendinger, Nina. “Der Einfluss von dem Wirt enstammenden Osteopontin auf Gliome.” 2020. Thesis, Freie Universität Berlin. Accessed November 26, 2020. http://dx.doi.org/10.17169/refubium-27533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schwendinger, Nina. “Der Einfluss von dem Wirt enstammenden Osteopontin auf Gliome.” 2020. Web. 26 Nov 2020.

Vancouver:

Schwendinger N. Der Einfluss von dem Wirt enstammenden Osteopontin auf Gliome. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2020 Nov 26]. Available from: http://dx.doi.org/10.17169/refubium-27533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schwendinger N. Der Einfluss von dem Wirt enstammenden Osteopontin auf Gliome. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-27533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

28. Sawicki, Caroline. Repeated Social Defeat Stress Promotes Reactive Brain Endothelium and Microglia-Dependent Pain Sensitivity.

Degree: PhD, Oral Biology, 2020, The Ohio State University

 Exposure to chronic stress disrupts the homeostatic communication pathways between the central nervous system (CNS) and peripheral immune system, leading to dysregulated and heightened neuroinflammation… (more)

Subjects/Keywords: Neurosciences; stress; pain; anxiety; microglia

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APA (6th Edition):

Sawicki, C. (2020). Repeated Social Defeat Stress Promotes Reactive Brain Endothelium and Microglia-Dependent Pain Sensitivity. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1586338402925441

Chicago Manual of Style (16th Edition):

Sawicki, Caroline. “Repeated Social Defeat Stress Promotes Reactive Brain Endothelium and Microglia-Dependent Pain Sensitivity.” 2020. Doctoral Dissertation, The Ohio State University. Accessed November 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1586338402925441.

MLA Handbook (7th Edition):

Sawicki, Caroline. “Repeated Social Defeat Stress Promotes Reactive Brain Endothelium and Microglia-Dependent Pain Sensitivity.” 2020. Web. 26 Nov 2020.

Vancouver:

Sawicki C. Repeated Social Defeat Stress Promotes Reactive Brain Endothelium and Microglia-Dependent Pain Sensitivity. [Internet] [Doctoral dissertation]. The Ohio State University; 2020. [cited 2020 Nov 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1586338402925441.

Council of Science Editors:

Sawicki C. Repeated Social Defeat Stress Promotes Reactive Brain Endothelium and Microglia-Dependent Pain Sensitivity. [Doctoral Dissertation]. The Ohio State University; 2020. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1586338402925441

29. Mandrekar-Colucci, Shweta Dilip. PPARɣ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease.

Degree: PhD, Neurosciences, 2011, Case Western Reserve University School of Graduate Studies

  Alzheimer’s disease (AD) is a chronic neurodegenerative disease characterized by the progressive loss of cognition and memory. The pathological hallmarks of AD include extracellular… (more)

Subjects/Keywords: Neurosciences; Alzheimer's Disease; Microglia; PPARgamma

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APA (6th Edition):

Mandrekar-Colucci, S. D. (2011). PPARɣ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357

Chicago Manual of Style (16th Edition):

Mandrekar-Colucci, Shweta Dilip. “PPARɣ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease.” 2011. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed November 26, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357.

MLA Handbook (7th Edition):

Mandrekar-Colucci, Shweta Dilip. “PPARɣ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease.” 2011. Web. 26 Nov 2020.

Vancouver:

Mandrekar-Colucci SD. PPARɣ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2011. [cited 2020 Nov 26]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357.

Council of Science Editors:

Mandrekar-Colucci SD. PPARɣ Activation Rapidly Ameliorates Amyloid Pathology and Restores Cognition in a Mouse Model of Alzheimer’s Disease. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1307730357


University of Edinburgh

30. Lloyd, Amy Frances. Cellular and molecular mechanisms underpinning microglial activation during remyelination.

Degree: PhD, 2019, University of Edinburgh

 Failed regeneration of myelin in the central nervous system (CNS) contributes to axon loss/dysfunction in prevalent neurodegenerative disorders, for which there is an unmet need… (more)

Subjects/Keywords: remyelination; microglia; Multiple Sclerosis; inflammatory microglia; inflammatory microglial death

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lloyd, A. F. (2019). Cellular and molecular mechanisms underpinning microglial activation during remyelination. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/35746

Chicago Manual of Style (16th Edition):

Lloyd, Amy Frances. “Cellular and molecular mechanisms underpinning microglial activation during remyelination.” 2019. Doctoral Dissertation, University of Edinburgh. Accessed November 26, 2020. http://hdl.handle.net/1842/35746.

MLA Handbook (7th Edition):

Lloyd, Amy Frances. “Cellular and molecular mechanisms underpinning microglial activation during remyelination.” 2019. Web. 26 Nov 2020.

Vancouver:

Lloyd AF. Cellular and molecular mechanisms underpinning microglial activation during remyelination. [Internet] [Doctoral dissertation]. University of Edinburgh; 2019. [cited 2020 Nov 26]. Available from: http://hdl.handle.net/1842/35746.

Council of Science Editors:

Lloyd AF. Cellular and molecular mechanisms underpinning microglial activation during remyelination. [Doctoral Dissertation]. University of Edinburgh; 2019. Available from: http://hdl.handle.net/1842/35746

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