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You searched for subject:(miR 203). Showing records 1 – 4 of 4 total matches.

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Universidade Nova

1. Santos, Bruno Filipe da Silva. Immunohistochemistry detection of putative miR-200c and miR-203 targets in breast cancer patients.

Degree: 2014, Universidade Nova

Part of this thesis will be published in the following: Gomes, B.C., Santos, B. 2015. Methods for studying microRNAs expression and their targets in formalin-fixed,… (more)

Subjects/Keywords: ATM; BMI1; Breast cancer; miR-200c; miR-203; SIX1

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APA (6th Edition):

Santos, B. F. d. S. (2014). Immunohistochemistry detection of putative miR-200c and miR-203 targets in breast cancer patients. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, Bruno Filipe da Silva. “Immunohistochemistry detection of putative miR-200c and miR-203 targets in breast cancer patients.” 2014. Thesis, Universidade Nova. Accessed March 30, 2020. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, Bruno Filipe da Silva. “Immunohistochemistry detection of putative miR-200c and miR-203 targets in breast cancer patients.” 2014. Web. 30 Mar 2020.

Vancouver:

Santos BFdS. Immunohistochemistry detection of putative miR-200c and miR-203 targets in breast cancer patients. [Internet] [Thesis]. Universidade Nova; 2014. [cited 2020 Mar 30]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos BFdS. Immunohistochemistry detection of putative miR-200c and miR-203 targets in breast cancer patients. [Thesis]. Universidade Nova; 2014. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/13914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Colorado

2. Zhang, Zhaojie. Genomic Analysis of microRNAs in Mouse Skin: Quantification, Biogenesis, Target Recognition and Regulatory Functions.

Degree: PhD, 2013, University of Colorado

  MiRNAs are a class of small RNAs, approximately 20~22 nucleotides in length. These broadly conserved molecules represent a novel layer of gene regulation that… (more)

Subjects/Keywords: Argonaute; miR-203; miRNA; NGS; Developmental Biology; Molecular Biology

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APA (6th Edition):

Zhang, Z. (2013). Genomic Analysis of microRNAs in Mouse Skin: Quantification, Biogenesis, Target Recognition and Regulatory Functions. (Doctoral Dissertation). University of Colorado. Retrieved from https://scholar.colorado.edu/mcdb_gradetds/23

Chicago Manual of Style (16th Edition):

Zhang, Zhaojie. “Genomic Analysis of microRNAs in Mouse Skin: Quantification, Biogenesis, Target Recognition and Regulatory Functions.” 2013. Doctoral Dissertation, University of Colorado. Accessed March 30, 2020. https://scholar.colorado.edu/mcdb_gradetds/23.

MLA Handbook (7th Edition):

Zhang, Zhaojie. “Genomic Analysis of microRNAs in Mouse Skin: Quantification, Biogenesis, Target Recognition and Regulatory Functions.” 2013. Web. 30 Mar 2020.

Vancouver:

Zhang Z. Genomic Analysis of microRNAs in Mouse Skin: Quantification, Biogenesis, Target Recognition and Regulatory Functions. [Internet] [Doctoral dissertation]. University of Colorado; 2013. [cited 2020 Mar 30]. Available from: https://scholar.colorado.edu/mcdb_gradetds/23.

Council of Science Editors:

Zhang Z. Genomic Analysis of microRNAs in Mouse Skin: Quantification, Biogenesis, Target Recognition and Regulatory Functions. [Doctoral Dissertation]. University of Colorado; 2013. Available from: https://scholar.colorado.edu/mcdb_gradetds/23


Texas Medical Center

3. Gireud, Monica B. STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A.

Degree: MS, 2011, Texas Medical Center

  STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A Monica Gireud, B.S. Thesis Advisor: Vidya Gopalakrishnan, Ph.D. The RE1 Silencing Transcription Factor (REST) is… (more)

Subjects/Keywords: REST; p16; miR-203; Bmi-1; Cell proliferation; Biology; Cancer Biology; Developmental Biology; Molecular and Cellular Neuroscience

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APA (6th Edition):

Gireud, M. B. (2011). STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/174

Chicago Manual of Style (16th Edition):

Gireud, Monica B. “STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A.” 2011. Masters Thesis, Texas Medical Center. Accessed March 30, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/174.

MLA Handbook (7th Edition):

Gireud, Monica B. “STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A.” 2011. Web. 30 Mar 2020.

Vancouver:

Gireud MB. STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A. [Internet] [Masters thesis]. Texas Medical Center; 2011. [cited 2020 Mar 30]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/174.

Council of Science Editors:

Gireud MB. STUDY OF REST AS A NEGATIVE REGULATOR OF P16INK4A. [Masters Thesis]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/174

4. Miller, Taylor Elaine. A novel molecular relationship between PARN and PLD that, when deregulated, contributes to the aggressive phenotype of breast cancer cell lines.

Degree: MS, Biochemistry and Molecular Biology, 2017, Wright State University

 The removal of mRNA transcript poly(A) tails by 3'-5' exonucleases is the rate-limiting step for controlled mRNA decay in eukaryotes. Poly(A)-specific ribonuclease (PARN) is one… (more)

Subjects/Keywords: Biochemistry; Cellular Biology; Molecular Biology; PARN; polyA-specific ribonuclease; deadenylase; phospholipase D; PLD; breast cancer; MDA-MB-231; microRNA; miR; miR-203; mRNA decay; post-transcriptional regulation; miR-dependent deadenylation

…Figure 28. Effect of miR-203 overexpression in MDA-MB-231 on PLD1 expression… …53 Figure 29. Effect of miR-203 overexpression on PLD1 3' UTR luciferase constructs… …breast cancer) and miR-203 can also regulate PLD2 in the U251 glioblastoma cell line (… …TargetScanHuman bioinformatics analysis, miR-203 is predicted to target PLD1, which is currently… …possible AUrich elements (AREs), one of which directly flanks the predicted miR-203… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Miller, T. E. (2017). A novel molecular relationship between PARN and PLD that, when deregulated, contributes to the aggressive phenotype of breast cancer cell lines. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1493581959771992

Chicago Manual of Style (16th Edition):

Miller, Taylor Elaine. “A novel molecular relationship between PARN and PLD that, when deregulated, contributes to the aggressive phenotype of breast cancer cell lines.” 2017. Masters Thesis, Wright State University. Accessed March 30, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1493581959771992.

MLA Handbook (7th Edition):

Miller, Taylor Elaine. “A novel molecular relationship between PARN and PLD that, when deregulated, contributes to the aggressive phenotype of breast cancer cell lines.” 2017. Web. 30 Mar 2020.

Vancouver:

Miller TE. A novel molecular relationship between PARN and PLD that, when deregulated, contributes to the aggressive phenotype of breast cancer cell lines. [Internet] [Masters thesis]. Wright State University; 2017. [cited 2020 Mar 30]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1493581959771992.

Council of Science Editors:

Miller TE. A novel molecular relationship between PARN and PLD that, when deregulated, contributes to the aggressive phenotype of breast cancer cell lines. [Masters Thesis]. Wright State University; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1493581959771992

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