Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(metabolic dysfunction). Showing records 1 – 30 of 34 total matches.

[1] [2]

Search Limiters

Last 2 Years | English Only

▼ Search Limiters


University of Sydney

1. Gasparini, Sylvia Jane. The Role of Osteoblasts and Sex Hormones in Glucocorticoid-induced Metabolic Dysfunction .

Degree: 2017, University of Sydney

 Glucocorticoids are some of the most widely prescribed medications world-wide, however their use is often limited by unwanted effects. Our team has previously shown that… (more)

Subjects/Keywords: Glucocorticoids; osteocalcin; androgens; metabolic dysfunction; sex differences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gasparini, S. J. (2017). The Role of Osteoblasts and Sex Hormones in Glucocorticoid-induced Metabolic Dysfunction . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gasparini, Sylvia Jane. “The Role of Osteoblasts and Sex Hormones in Glucocorticoid-induced Metabolic Dysfunction .” 2017. Thesis, University of Sydney. Accessed November 30, 2020. http://hdl.handle.net/2123/17083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gasparini, Sylvia Jane. “The Role of Osteoblasts and Sex Hormones in Glucocorticoid-induced Metabolic Dysfunction .” 2017. Web. 30 Nov 2020.

Vancouver:

Gasparini SJ. The Role of Osteoblasts and Sex Hormones in Glucocorticoid-induced Metabolic Dysfunction . [Internet] [Thesis]. University of Sydney; 2017. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/2123/17083.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gasparini SJ. The Role of Osteoblasts and Sex Hormones in Glucocorticoid-induced Metabolic Dysfunction . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17083

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Sydney

2. Ha, Connie Wing Yan. Interplay between diet, gut microbiome and host responses during the development and management of dysbiosis .

Degree: 2018, University of Sydney

 The mammalian gut contains a metabolically active microbial community that lies at the intersection between the host and its nutrient environment. Compositional changes in the… (more)

Subjects/Keywords: gut microbiome; diet; dysbiosis; metabolic dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ha, C. W. Y. (2018). Interplay between diet, gut microbiome and host responses during the development and management of dysbiosis . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/20211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ha, Connie Wing Yan. “Interplay between diet, gut microbiome and host responses during the development and management of dysbiosis .” 2018. Thesis, University of Sydney. Accessed November 30, 2020. http://hdl.handle.net/2123/20211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ha, Connie Wing Yan. “Interplay between diet, gut microbiome and host responses during the development and management of dysbiosis .” 2018. Web. 30 Nov 2020.

Vancouver:

Ha CWY. Interplay between diet, gut microbiome and host responses during the development and management of dysbiosis . [Internet] [Thesis]. University of Sydney; 2018. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/2123/20211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ha CWY. Interplay between diet, gut microbiome and host responses during the development and management of dysbiosis . [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/20211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

3. Xia, Mingcan. Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications.

Degree: 2012, Penn State University

 Atherosclerosis (also known as arteriosclerotic vascular disease or ASVD) is a chronic inflammatory disease and characterized by patchy thickening of the inner lining of arterial… (more)

Subjects/Keywords: atherosclerosis; metabolic dysfunction; NKG2D ligand; NKG2D; Immune activation; diabetes; cardiovascular diseases; liver dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xia, M. (2012). Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xia, Mingcan. “Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications.” 2012. Thesis, Penn State University. Accessed November 30, 2020. https://submit-etda.libraries.psu.edu/catalog/13899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xia, Mingcan. “Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications.” 2012. Web. 30 Nov 2020.

Vancouver:

Xia M. Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications. [Internet] [Thesis]. Penn State University; 2012. [cited 2020 Nov 30]. Available from: https://submit-etda.libraries.psu.edu/catalog/13899.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xia M. Roles of Nkg2d/ligand-mediated Immune activations in progression of metabolic dysfunctions and vascular complications. [Thesis]. Penn State University; 2012. Available from: https://submit-etda.libraries.psu.edu/catalog/13899

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

4. Streeter, Cassandra. A Genome-Wide Association Study Of Equine Metabolic Syndrome And Pituitary Pars Intermedia Dysfunction.

Degree: M.S., Animal Science, Animal Science, 2012, Cornell University

 Laminitis remains a poorly understood form of lameness in the horse despite being heavily researched. Endocrinopathic laminitis is an insidious form of laminitis that often… (more)

Subjects/Keywords: equine metabolic syndrome; pituitary pars intermedia dysfunction; insulin resistance

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Streeter, C. (2012). A Genome-Wide Association Study Of Equine Metabolic Syndrome And Pituitary Pars Intermedia Dysfunction. (Masters Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/29264

Chicago Manual of Style (16th Edition):

Streeter, Cassandra. “A Genome-Wide Association Study Of Equine Metabolic Syndrome And Pituitary Pars Intermedia Dysfunction.” 2012. Masters Thesis, Cornell University. Accessed November 30, 2020. http://hdl.handle.net/1813/29264.

MLA Handbook (7th Edition):

Streeter, Cassandra. “A Genome-Wide Association Study Of Equine Metabolic Syndrome And Pituitary Pars Intermedia Dysfunction.” 2012. Web. 30 Nov 2020.

Vancouver:

Streeter C. A Genome-Wide Association Study Of Equine Metabolic Syndrome And Pituitary Pars Intermedia Dysfunction. [Internet] [Masters thesis]. Cornell University; 2012. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/1813/29264.

Council of Science Editors:

Streeter C. A Genome-Wide Association Study Of Equine Metabolic Syndrome And Pituitary Pars Intermedia Dysfunction. [Masters Thesis]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29264


Texas A&M University

5. Zawieja, Scott D. Macrophage Polarization And Nitric Oxide Mechanisms In Lymphatic Dysfunction In A Rat Model Of Metabolic Syndrome.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

Metabolic syndrome (MetSyn) is the clustering of multiple metabolic disorders that further increase the risk for cardiovascular disease and has been recently linked to poor… (more)

Subjects/Keywords: Lymphatic; Metabolic Syndrome; Macrophage; Macrophage Polarization; Endothelial Dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zawieja, S. D. (2014). Macrophage Polarization And Nitric Oxide Mechanisms In Lymphatic Dysfunction In A Rat Model Of Metabolic Syndrome. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/154145

Chicago Manual of Style (16th Edition):

Zawieja, Scott D. “Macrophage Polarization And Nitric Oxide Mechanisms In Lymphatic Dysfunction In A Rat Model Of Metabolic Syndrome.” 2014. Doctoral Dissertation, Texas A&M University. Accessed November 30, 2020. http://hdl.handle.net/1969.1/154145.

MLA Handbook (7th Edition):

Zawieja, Scott D. “Macrophage Polarization And Nitric Oxide Mechanisms In Lymphatic Dysfunction In A Rat Model Of Metabolic Syndrome.” 2014. Web. 30 Nov 2020.

Vancouver:

Zawieja SD. Macrophage Polarization And Nitric Oxide Mechanisms In Lymphatic Dysfunction In A Rat Model Of Metabolic Syndrome. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/1969.1/154145.

Council of Science Editors:

Zawieja SD. Macrophage Polarization And Nitric Oxide Mechanisms In Lymphatic Dysfunction In A Rat Model Of Metabolic Syndrome. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/154145


University of Sydney

6. Kim, Sarah. The Role of Skeletal Glucocorticoid Signalling in Diet-induced Bone Loss and Metabolic Dysfunction .

Degree: 2017, University of Sydney

 Osteoblasts have recently been identified as important co-regulators of whole-body energy metabolism. Our team has previously demonstrated that the adverse skeletal and metabolic effects of… (more)

Subjects/Keywords: Glucocorticoids; high fat diet; osteoblasts; bone loss; metabolic dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, S. (2017). The Role of Skeletal Glucocorticoid Signalling in Diet-induced Bone Loss and Metabolic Dysfunction . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Sarah. “The Role of Skeletal Glucocorticoid Signalling in Diet-induced Bone Loss and Metabolic Dysfunction .” 2017. Thesis, University of Sydney. Accessed November 30, 2020. http://hdl.handle.net/2123/17966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Sarah. “The Role of Skeletal Glucocorticoid Signalling in Diet-induced Bone Loss and Metabolic Dysfunction .” 2017. Web. 30 Nov 2020.

Vancouver:

Kim S. The Role of Skeletal Glucocorticoid Signalling in Diet-induced Bone Loss and Metabolic Dysfunction . [Internet] [Thesis]. University of Sydney; 2017. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/2123/17966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim S. The Role of Skeletal Glucocorticoid Signalling in Diet-induced Bone Loss and Metabolic Dysfunction . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

7. AKAN, ILHAN. MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATE.

Degree: PhD, 2012, Temple University

Biology

Sialylation, a posttranslational modification of both glycolipids and glycoproteins, is typically found on the terminal positions of glycan chains. Unique among most other sugars,… (more)

Subjects/Keywords: Biology; Neurosciences; Molecular biology; Diabetes; Drosophila; Metabolic dysfunction; Neurodegeneration; Shaker; Sialic acid

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

AKAN, I. (2012). MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATE. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213122

Chicago Manual of Style (16th Edition):

AKAN, ILHAN. “MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATE.” 2012. Doctoral Dissertation, Temple University. Accessed November 30, 2020. http://digital.library.temple.edu/u?/p245801coll10,213122.

MLA Handbook (7th Edition):

AKAN, ILHAN. “MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATE.” 2012. Web. 30 Nov 2020.

Vancouver:

AKAN I. MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATE. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Nov 30]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213122.

Council of Science Editors:

AKAN I. MUTANT DROSOPHILA LACKING SIALIC ACID EXHIBIT NEURODEGENERATION AND METABOLIC DYSFUNCTION LEADING TO A DIABETIC STATE. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,213122


University of Toronto

8. Mansuri, Sudaba. Vitamin D and Type 2 Diabetes Mellitus in an Aboriginal Community.

Degree: 2014, University of Toronto

Both vitamin D deficiency and type 2 diabetes mellitus (T2DM) are highly prevalent among the Aboriginal Canadian population. Emerging evidence suggests that vitamin D may… (more)

Subjects/Keywords: 25(OH)D; Aboriginal; beta cell dysfunction; insulin resistance; metabolic syndrome; type 2 diabetes; 0570

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mansuri, S. (2014). Vitamin D and Type 2 Diabetes Mellitus in an Aboriginal Community. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67979

Chicago Manual of Style (16th Edition):

Mansuri, Sudaba. “Vitamin D and Type 2 Diabetes Mellitus in an Aboriginal Community.” 2014. Masters Thesis, University of Toronto. Accessed November 30, 2020. http://hdl.handle.net/1807/67979.

MLA Handbook (7th Edition):

Mansuri, Sudaba. “Vitamin D and Type 2 Diabetes Mellitus in an Aboriginal Community.” 2014. Web. 30 Nov 2020.

Vancouver:

Mansuri S. Vitamin D and Type 2 Diabetes Mellitus in an Aboriginal Community. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/1807/67979.

Council of Science Editors:

Mansuri S. Vitamin D and Type 2 Diabetes Mellitus in an Aboriginal Community. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67979


University of South Carolina

9. Torrance, Torrance. Sleep, Shiftwork Adaptation, Autonomic Dysfunction, And Metabolic Syndrome.

Degree: PhD, Epidemiology and Biostatistics, 2018, University of South Carolina

  Background: Sleep is considered a physiological necessity and its disruption is associated with a wide variety of adverse mental and physical health outcomes including… (more)

Subjects/Keywords: Epidemiology; Medicine and Health Sciences; Public Health; Sleep; Shiftwork Adaptation; Autonomic Dysfunction; Metabolic Syndrome

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Torrance, T. (2018). Sleep, Shiftwork Adaptation, Autonomic Dysfunction, And Metabolic Syndrome. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/4852

Chicago Manual of Style (16th Edition):

Torrance, Torrance. “Sleep, Shiftwork Adaptation, Autonomic Dysfunction, And Metabolic Syndrome.” 2018. Doctoral Dissertation, University of South Carolina. Accessed November 30, 2020. https://scholarcommons.sc.edu/etd/4852.

MLA Handbook (7th Edition):

Torrance, Torrance. “Sleep, Shiftwork Adaptation, Autonomic Dysfunction, And Metabolic Syndrome.” 2018. Web. 30 Nov 2020.

Vancouver:

Torrance T. Sleep, Shiftwork Adaptation, Autonomic Dysfunction, And Metabolic Syndrome. [Internet] [Doctoral dissertation]. University of South Carolina; 2018. [cited 2020 Nov 30]. Available from: https://scholarcommons.sc.edu/etd/4852.

Council of Science Editors:

Torrance T. Sleep, Shiftwork Adaptation, Autonomic Dysfunction, And Metabolic Syndrome. [Doctoral Dissertation]. University of South Carolina; 2018. Available from: https://scholarcommons.sc.edu/etd/4852

10. Schmitt, Florent. Rôle de la stéaroyl-coenzyme A désaturase 1, une enzyme de synthèse des acides gras mono-insaturés, dans un modèle transgénique d’étude de la Sclérose Latérale Amyotrophique : Role of stearoyl-coenzyme A desaturase 1, an enzyme for the synthesis of mono-unsaturated fatty acids, in a transgenic model for the study of amyotrophic lateral sclerosis.

Degree: Docteur es, Neurosciences, 2013, Université de Strasbourg

La sclérose latérale amyotrophique est une maladie neurodégénérative associée à un dysfonctionnement métabolique. Des altérations du métabolisme des lipides, décrites chez les patients SLA et… (more)

Subjects/Keywords: Sclérose latérale amyotrophique; Métabolisme; SCD1; Acides gras; Amyotrophic lateral sclerosis; Stearoyl-CoA desaturase 1; Metabolic dysfunction; Lipid metabolism; 572.4; 616.8

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schmitt, F. (2013). Rôle de la stéaroyl-coenzyme A désaturase 1, une enzyme de synthèse des acides gras mono-insaturés, dans un modèle transgénique d’étude de la Sclérose Latérale Amyotrophique : Role of stearoyl-coenzyme A desaturase 1, an enzyme for the synthesis of mono-unsaturated fatty acids, in a transgenic model for the study of amyotrophic lateral sclerosis. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2013STRAJ085

Chicago Manual of Style (16th Edition):

Schmitt, Florent. “Rôle de la stéaroyl-coenzyme A désaturase 1, une enzyme de synthèse des acides gras mono-insaturés, dans un modèle transgénique d’étude de la Sclérose Latérale Amyotrophique : Role of stearoyl-coenzyme A desaturase 1, an enzyme for the synthesis of mono-unsaturated fatty acids, in a transgenic model for the study of amyotrophic lateral sclerosis.” 2013. Doctoral Dissertation, Université de Strasbourg. Accessed November 30, 2020. http://www.theses.fr/2013STRAJ085.

MLA Handbook (7th Edition):

Schmitt, Florent. “Rôle de la stéaroyl-coenzyme A désaturase 1, une enzyme de synthèse des acides gras mono-insaturés, dans un modèle transgénique d’étude de la Sclérose Latérale Amyotrophique : Role of stearoyl-coenzyme A desaturase 1, an enzyme for the synthesis of mono-unsaturated fatty acids, in a transgenic model for the study of amyotrophic lateral sclerosis.” 2013. Web. 30 Nov 2020.

Vancouver:

Schmitt F. Rôle de la stéaroyl-coenzyme A désaturase 1, une enzyme de synthèse des acides gras mono-insaturés, dans un modèle transgénique d’étude de la Sclérose Latérale Amyotrophique : Role of stearoyl-coenzyme A desaturase 1, an enzyme for the synthesis of mono-unsaturated fatty acids, in a transgenic model for the study of amyotrophic lateral sclerosis. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2013. [cited 2020 Nov 30]. Available from: http://www.theses.fr/2013STRAJ085.

Council of Science Editors:

Schmitt F. Rôle de la stéaroyl-coenzyme A désaturase 1, une enzyme de synthèse des acides gras mono-insaturés, dans un modèle transgénique d’étude de la Sclérose Latérale Amyotrophique : Role of stearoyl-coenzyme A desaturase 1, an enzyme for the synthesis of mono-unsaturated fatty acids, in a transgenic model for the study of amyotrophic lateral sclerosis. [Doctoral Dissertation]. Université de Strasbourg; 2013. Available from: http://www.theses.fr/2013STRAJ085


Université de Lorraine

11. Chau, Kénora. Dysfonction diastolique, rigidité artérielle aortique et hypertension : facteurs anthropométriques et métaboliques associés et prise en charge en population générale : Diastolic dysfunction, aortic stiffness and hypertension : anthropometric and metabolic risk factors and management in general population.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Université de Lorraine

L’augmentation de l’obésité durant les dernières décennies expose la population à un risque accru de problèmes métaboliques et des maladies cardiovasculaires. Ce travail étudiait, d’une… (more)

Subjects/Keywords: Dysfonction diastolique; Rigidité artérielle; Hypertension artérielle; Obésité; Facteurs métaboliques; Diastolic dysfunction; Aortic stiffness; Hypertension; Obesity; Metabolic factors; 616.398; 616.105

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chau, K. (2018). Dysfonction diastolique, rigidité artérielle aortique et hypertension : facteurs anthropométriques et métaboliques associés et prise en charge en population générale : Diastolic dysfunction, aortic stiffness and hypertension : anthropometric and metabolic risk factors and management in general population. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2018LORR0281

Chicago Manual of Style (16th Edition):

Chau, Kénora. “Dysfonction diastolique, rigidité artérielle aortique et hypertension : facteurs anthropométriques et métaboliques associés et prise en charge en population générale : Diastolic dysfunction, aortic stiffness and hypertension : anthropometric and metabolic risk factors and management in general population.” 2018. Doctoral Dissertation, Université de Lorraine. Accessed November 30, 2020. http://www.theses.fr/2018LORR0281.

MLA Handbook (7th Edition):

Chau, Kénora. “Dysfonction diastolique, rigidité artérielle aortique et hypertension : facteurs anthropométriques et métaboliques associés et prise en charge en population générale : Diastolic dysfunction, aortic stiffness and hypertension : anthropometric and metabolic risk factors and management in general population.” 2018. Web. 30 Nov 2020.

Vancouver:

Chau K. Dysfonction diastolique, rigidité artérielle aortique et hypertension : facteurs anthropométriques et métaboliques associés et prise en charge en population générale : Diastolic dysfunction, aortic stiffness and hypertension : anthropometric and metabolic risk factors and management in general population. [Internet] [Doctoral dissertation]. Université de Lorraine; 2018. [cited 2020 Nov 30]. Available from: http://www.theses.fr/2018LORR0281.

Council of Science Editors:

Chau K. Dysfonction diastolique, rigidité artérielle aortique et hypertension : facteurs anthropométriques et métaboliques associés et prise en charge en population générale : Diastolic dysfunction, aortic stiffness and hypertension : anthropometric and metabolic risk factors and management in general population. [Doctoral Dissertation]. Université de Lorraine; 2018. Available from: http://www.theses.fr/2018LORR0281


York University

12. Abdifarkoosh, Ghoncheh. Sex-Related Differences in Microvascular Adaptations to Obesity.

Degree: MSc -MS, Kinesiology & Health Science, 2018, York University

 Capillaries within skeletal muscle and adipose tissue have a critical role in the maintenance of proper tissue function. A reduction in capillary density is correlated… (more)

Subjects/Keywords: Biology; Angiogenesis; Vascular adaptation; Sex-related differences; Adipose tissue; Skeletal muscle; Obesity; Diet-induced metabolic dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abdifarkoosh, G. (2018). Sex-Related Differences in Microvascular Adaptations to Obesity. (Masters Thesis). York University. Retrieved from http://hdl.handle.net/10315/34389

Chicago Manual of Style (16th Edition):

Abdifarkoosh, Ghoncheh. “Sex-Related Differences in Microvascular Adaptations to Obesity.” 2018. Masters Thesis, York University. Accessed November 30, 2020. http://hdl.handle.net/10315/34389.

MLA Handbook (7th Edition):

Abdifarkoosh, Ghoncheh. “Sex-Related Differences in Microvascular Adaptations to Obesity.” 2018. Web. 30 Nov 2020.

Vancouver:

Abdifarkoosh G. Sex-Related Differences in Microvascular Adaptations to Obesity. [Internet] [Masters thesis]. York University; 2018. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10315/34389.

Council of Science Editors:

Abdifarkoosh G. Sex-Related Differences in Microvascular Adaptations to Obesity. [Masters Thesis]. York University; 2018. Available from: http://hdl.handle.net/10315/34389

13. Malloci, Marine. Rôle des exosomes dans la dysfonction endothéliale associée au syndrome métabolique : Role of exosomes in endothelial dysfunction associated with metabolic syndrome.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Angers

Le syndrome métabolique (SM) est défini comme l'association de troubles favorisant les risques cardiovasculaires et le développement d'un diabète de type 2. Une personne atteinte… (more)

Subjects/Keywords: Syndrome métabolique; Exosomes; Dysfonction endothéliale; Monoxyde d’azote; Stress oxydant; Mitochondrie; Metabolic syndrome; Exosomes; Endothelial dysfunction; Nitric oxide; Oxidative stress; Mitochondria; 616.39

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Malloci, M. (2017). Rôle des exosomes dans la dysfonction endothéliale associée au syndrome métabolique : Role of exosomes in endothelial dysfunction associated with metabolic syndrome. (Doctoral Dissertation). Angers. Retrieved from http://www.theses.fr/2017ANGE0076

Chicago Manual of Style (16th Edition):

Malloci, Marine. “Rôle des exosomes dans la dysfonction endothéliale associée au syndrome métabolique : Role of exosomes in endothelial dysfunction associated with metabolic syndrome.” 2017. Doctoral Dissertation, Angers. Accessed November 30, 2020. http://www.theses.fr/2017ANGE0076.

MLA Handbook (7th Edition):

Malloci, Marine. “Rôle des exosomes dans la dysfonction endothéliale associée au syndrome métabolique : Role of exosomes in endothelial dysfunction associated with metabolic syndrome.” 2017. Web. 30 Nov 2020.

Vancouver:

Malloci M. Rôle des exosomes dans la dysfonction endothéliale associée au syndrome métabolique : Role of exosomes in endothelial dysfunction associated with metabolic syndrome. [Internet] [Doctoral dissertation]. Angers; 2017. [cited 2020 Nov 30]. Available from: http://www.theses.fr/2017ANGE0076.

Council of Science Editors:

Malloci M. Rôle des exosomes dans la dysfonction endothéliale associée au syndrome métabolique : Role of exosomes in endothelial dysfunction associated with metabolic syndrome. [Doctoral Dissertation]. Angers; 2017. Available from: http://www.theses.fr/2017ANGE0076


Virginia Tech

14. Haynie, Kimberly Rebekah. The Role of Neuropeptide Y Y1R in Skeletal Muscle Lipid Metabolism.

Degree: MS, Human Nutrition, Foods, and Exercise, 2009, Virginia Tech

 The Hulver laboratory has recently found that the neuropeptide Y Y1 receptor (NPY Y1R) mRNA expression is elevated in skeletal muscle of obese humans (Hulver,… (more)

Subjects/Keywords: Neuropeptide Y; diacylglycerol; lipid oxidation; metabolic dysfunction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haynie, K. R. (2009). The Role of Neuropeptide Y Y1R in Skeletal Muscle Lipid Metabolism. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/32270

Chicago Manual of Style (16th Edition):

Haynie, Kimberly Rebekah. “The Role of Neuropeptide Y Y1R in Skeletal Muscle Lipid Metabolism.” 2009. Masters Thesis, Virginia Tech. Accessed November 30, 2020. http://hdl.handle.net/10919/32270.

MLA Handbook (7th Edition):

Haynie, Kimberly Rebekah. “The Role of Neuropeptide Y Y1R in Skeletal Muscle Lipid Metabolism.” 2009. Web. 30 Nov 2020.

Vancouver:

Haynie KR. The Role of Neuropeptide Y Y1R in Skeletal Muscle Lipid Metabolism. [Internet] [Masters thesis]. Virginia Tech; 2009. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10919/32270.

Council of Science Editors:

Haynie KR. The Role of Neuropeptide Y Y1R in Skeletal Muscle Lipid Metabolism. [Masters Thesis]. Virginia Tech; 2009. Available from: http://hdl.handle.net/10919/32270


University of Louisville

15. Zajack, Matthew R. Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in ApoE-/-mice.

Degree: MS, 2010, University of Louisville

 Arsenic exposure in drinking-water is a significant worldwide health problem. It causes adverse human health effects, such as cancer, increases the risks for others such… (more)

Subjects/Keywords: Arsenic; Inflammation; Atherosclerosis; Metabolic syndrome; Mitochondrial dysfunction; ApoE-knockout

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zajack, M. R. (2010). Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in ApoE-/-mice. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/1630 ; https://ir.library.louisville.edu/etd/1630

Chicago Manual of Style (16th Edition):

Zajack, Matthew R. “Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in ApoE-/-mice.” 2010. Masters Thesis, University of Louisville. Accessed November 30, 2020. 10.18297/etd/1630 ; https://ir.library.louisville.edu/etd/1630.

MLA Handbook (7th Edition):

Zajack, Matthew R. “Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in ApoE-/-mice.” 2010. Web. 30 Nov 2020.

Vancouver:

Zajack MR. Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in ApoE-/-mice. [Internet] [Masters thesis]. University of Louisville; 2010. [cited 2020 Nov 30]. Available from: 10.18297/etd/1630 ; https://ir.library.louisville.edu/etd/1630.

Council of Science Editors:

Zajack MR. Arsenic in drinking water causes gene expression changes in the liver related to inflammation and metabolic dysfunction and accelerates atherosclerosis in ApoE-/-mice. [Masters Thesis]. University of Louisville; 2010. Available from: 10.18297/etd/1630 ; https://ir.library.louisville.edu/etd/1630


Unitec New Zealand

16. Barnett, Lacey Ellen. The effect of osteopathic manual therapy with breathing retraining on dysfunctional breathing and exercise economy : a randomised controlled trial.

Degree: 2016, Unitec New Zealand

 BACKGROUND: Dysfunctional breathing patterns during exercise can result in a tachypnoeic breathing pattern, expiratory flow limitation and dynamic hyperinflation. These factors can increase the work… (more)

Subjects/Keywords: breathing dysfunction; metabolic demand; osteopathy; manipulation; respiratory mechanics; lung function; breathing pattern disorders; 110499 Complementary and Alternative Medicine not elsewhere classified

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barnett, L. E. (2016). The effect of osteopathic manual therapy with breathing retraining on dysfunctional breathing and exercise economy : a randomised controlled trial. (Thesis). Unitec New Zealand. Retrieved from http://hdl.handle.net/10652/3769

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barnett, Lacey Ellen. “The effect of osteopathic manual therapy with breathing retraining on dysfunctional breathing and exercise economy : a randomised controlled trial.” 2016. Thesis, Unitec New Zealand. Accessed November 30, 2020. http://hdl.handle.net/10652/3769.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barnett, Lacey Ellen. “The effect of osteopathic manual therapy with breathing retraining on dysfunctional breathing and exercise economy : a randomised controlled trial.” 2016. Web. 30 Nov 2020.

Vancouver:

Barnett LE. The effect of osteopathic manual therapy with breathing retraining on dysfunctional breathing and exercise economy : a randomised controlled trial. [Internet] [Thesis]. Unitec New Zealand; 2016. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10652/3769.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barnett LE. The effect of osteopathic manual therapy with breathing retraining on dysfunctional breathing and exercise economy : a randomised controlled trial. [Thesis]. Unitec New Zealand; 2016. Available from: http://hdl.handle.net/10652/3769

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

17. Johnston, Luke. Association of Leg Length with Metabolic Abnormalities Underlying Type 2 Diabetes Mellitus.

Degree: 2013, University of Toronto

The objective of this thesis was to determine the association of leg length (LL), a marker of early childhood conditions, with metabolic abnormalities underlying type… (more)

Subjects/Keywords: Early childhood conditions; Insulin resistance; Beta-cell dysfunction; Anthropometry; Leg length; Metabolic Syndrome; Continuous Metabolic Syndrome score; Type 2 diabetes; Stature; Height; Developmental origins of health and disease; 0766; 0570

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johnston, L. (2013). Association of Leg Length with Metabolic Abnormalities Underlying Type 2 Diabetes Mellitus. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/42975

Chicago Manual of Style (16th Edition):

Johnston, Luke. “Association of Leg Length with Metabolic Abnormalities Underlying Type 2 Diabetes Mellitus.” 2013. Masters Thesis, University of Toronto. Accessed November 30, 2020. http://hdl.handle.net/1807/42975.

MLA Handbook (7th Edition):

Johnston, Luke. “Association of Leg Length with Metabolic Abnormalities Underlying Type 2 Diabetes Mellitus.” 2013. Web. 30 Nov 2020.

Vancouver:

Johnston L. Association of Leg Length with Metabolic Abnormalities Underlying Type 2 Diabetes Mellitus. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/1807/42975.

Council of Science Editors:

Johnston L. Association of Leg Length with Metabolic Abnormalities Underlying Type 2 Diabetes Mellitus. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/42975

18. Dormishian, Mojdeh. Rôle de récepteur 1 des prokinéticines dans les cellules endothéliales : étude in vivo : Role of Prokineticin Receptor 1 in Endothelial Cells : in vivo study.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Université de Strasbourg

L’endothélium est considéré comme le plus grand organe endocrine, et est impliqué dans plusieurs processus physiologiques tels que l’inflammation et l’angiogenèse. Toute altération fonctionnelle/structurale peut… (more)

Subjects/Keywords: Endothélium; Prokinéticines; PKR1; Angiogenèse; Dysfonctionnement endothélial; Désordres métaboliques; Anomalies rénales; Cardiomyopathie; Endothelium; Prokineticins; PKR1; Angiogenesis; Endothelial dysfunction; Metabolic disorders; Renal anomalies; Cardiomyopathy; 572.8; 616.13

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dormishian, M. (2012). Rôle de récepteur 1 des prokinéticines dans les cellules endothéliales : étude in vivo : Role of Prokineticin Receptor 1 in Endothelial Cells : in vivo study. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ072

Chicago Manual of Style (16th Edition):

Dormishian, Mojdeh. “Rôle de récepteur 1 des prokinéticines dans les cellules endothéliales : étude in vivo : Role of Prokineticin Receptor 1 in Endothelial Cells : in vivo study.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed November 30, 2020. http://www.theses.fr/2012STRAJ072.

MLA Handbook (7th Edition):

Dormishian, Mojdeh. “Rôle de récepteur 1 des prokinéticines dans les cellules endothéliales : étude in vivo : Role of Prokineticin Receptor 1 in Endothelial Cells : in vivo study.” 2012. Web. 30 Nov 2020.

Vancouver:

Dormishian M. Rôle de récepteur 1 des prokinéticines dans les cellules endothéliales : étude in vivo : Role of Prokineticin Receptor 1 in Endothelial Cells : in vivo study. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2020 Nov 30]. Available from: http://www.theses.fr/2012STRAJ072.

Council of Science Editors:

Dormishian M. Rôle de récepteur 1 des prokinéticines dans les cellules endothéliales : étude in vivo : Role of Prokineticin Receptor 1 in Endothelial Cells : in vivo study. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ072


University of Wollongong

19. Green, Katrina. Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects.

Degree: PhD, 2012, University of Wollongong

  Second generation antipsychotics (SGAs) are a key pharmacotherapy for the treatment of schizophrenia but can cause serious metabolic side-effects, including obesity and type II… (more)

Subjects/Keywords: obesity; antipsychotic drugs; diabetes; side-effects; olanzapine; metabolic dysfunction; hypothalamus; brainstem; muscarinic; cannabinoid; GABA; neuropeptide Y; pro-opiometanocortin; receptors; mRNA; animal model; antipsychotic; metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Green, K. (2012). Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects. (Doctoral Dissertation). University of Wollongong. Retrieved from 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579

Chicago Manual of Style (16th Edition):

Green, Katrina. “Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects.” 2012. Doctoral Dissertation, University of Wollongong. Accessed November 30, 2020. 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579.

MLA Handbook (7th Edition):

Green, Katrina. “Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects.” 2012. Web. 30 Nov 2020.

Vancouver:

Green K. Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects. [Internet] [Doctoral dissertation]. University of Wollongong; 2012. [cited 2020 Nov 30]. Available from: 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579.

Council of Science Editors:

Green K. Alterations in Hypothalamic and Brainstem Neurotransmitter Signalling Associated with Olanzapine-Induced Metabolic Side-Effects. [Doctoral Dissertation]. University of Wollongong; 2012. Available from: 1109 NEUROSCIENCES, 1116 MEDICAL PHYSIOLOGY ; https://ro.uow.edu.au/theses/3579

20. Schrover, I.M. Metabolic consequences of adipose tissue dysfunction.

Degree: 2018, University Utrecht

 This thesis focuses on the consequences of adipose tissue dysfunction (ATD), which is defined as the imbalance of pro- and anti-inflammatory adipocytokines produced by adipose… (more)

Subjects/Keywords: adiposity; dysfunction; adipokines; metabolic syndrome; hypertension; obesity; cardiovascular disease; spectroscopy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schrover, I. M. (2018). Metabolic consequences of adipose tissue dysfunction. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; http://dspace.library.uu.nl/handle/1874/372756

Chicago Manual of Style (16th Edition):

Schrover, I M. “Metabolic consequences of adipose tissue dysfunction.” 2018. Doctoral Dissertation, University Utrecht. Accessed November 30, 2020. http://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; http://dspace.library.uu.nl/handle/1874/372756.

MLA Handbook (7th Edition):

Schrover, I M. “Metabolic consequences of adipose tissue dysfunction.” 2018. Web. 30 Nov 2020.

Vancouver:

Schrover IM. Metabolic consequences of adipose tissue dysfunction. [Internet] [Doctoral dissertation]. University Utrecht; 2018. [cited 2020 Nov 30]. Available from: http://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; http://dspace.library.uu.nl/handle/1874/372756.

Council of Science Editors:

Schrover IM. Metabolic consequences of adipose tissue dysfunction. [Doctoral Dissertation]. University Utrecht; 2018. Available from: http://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; http://dspace.library.uu.nl/handle/1874/372756

21. Thatcher, Mikayla Orton. HMGB1 and Ceramides: Potential Mediators of Cigarette Smoke-induced Metabolic Dysfunction.

Degree: PhD, 2015, Brigham Young University

 While cigarette smoking is a common-knowledge way to stay lean, it has long been known as a risk factor for diabetes and obesity. Here we… (more)

Subjects/Keywords: ceramide; HMGB1; RAGE; cigarette smoking; metabolic dysfunction; myriocin; Physiology

…an insufficiency, of insulin. Metabolic syndrome is a term used to describe the… …of the metabolic syndrome. Prospective data from the San Antonio Heart Study, for example… …to inhaled toxins and metabolic disruption leading to fat gain (Bray and Champagne… …to metabolic morbidity. Certain trafficrelated pollutants may also contribute to impaired… …Nielsen et al. 2012). Metabolic disturbance from inhaled particles may even be responsible… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thatcher, M. O. (2015). HMGB1 and Ceramides: Potential Mediators of Cigarette Smoke-induced Metabolic Dysfunction. (Doctoral Dissertation). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7040&context=etd

Chicago Manual of Style (16th Edition):

Thatcher, Mikayla Orton. “HMGB1 and Ceramides: Potential Mediators of Cigarette Smoke-induced Metabolic Dysfunction.” 2015. Doctoral Dissertation, Brigham Young University. Accessed November 30, 2020. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7040&context=etd.

MLA Handbook (7th Edition):

Thatcher, Mikayla Orton. “HMGB1 and Ceramides: Potential Mediators of Cigarette Smoke-induced Metabolic Dysfunction.” 2015. Web. 30 Nov 2020.

Vancouver:

Thatcher MO. HMGB1 and Ceramides: Potential Mediators of Cigarette Smoke-induced Metabolic Dysfunction. [Internet] [Doctoral dissertation]. Brigham Young University; 2015. [cited 2020 Nov 30]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7040&context=etd.

Council of Science Editors:

Thatcher MO. HMGB1 and Ceramides: Potential Mediators of Cigarette Smoke-induced Metabolic Dysfunction. [Doctoral Dissertation]. Brigham Young University; 2015. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=7040&context=etd


University of Technology, Sydney

22. Al-Odat, Ibrahim. Effects of maternal cigarette smoke exposure on renal and other health outcomes in mice offspring.

Degree: 2016, University of Technology, Sydney

 Background: The burden of kidney disease is significantly increased worldwide. Developmental programming of non-communicable diseases is an established paradigm. Therefore, there is increasing attention to… (more)

Subjects/Keywords: Kidney disease.; Maternal cigarette smoke exposure (SE).; Intrauterine growth retardation (IUGR).; Glial cell line-derived neurotrophic factor (GDNF).; Adulthood renal dysfunction.; Lipid and glucose metabolic disorders.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Al-Odat, I. (2016). Effects of maternal cigarette smoke exposure on renal and other health outcomes in mice offspring. (Thesis). University of Technology, Sydney. Retrieved from http://hdl.handle.net/10453/43398

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Odat, Ibrahim. “Effects of maternal cigarette smoke exposure on renal and other health outcomes in mice offspring.” 2016. Thesis, University of Technology, Sydney. Accessed November 30, 2020. http://hdl.handle.net/10453/43398.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Odat, Ibrahim. “Effects of maternal cigarette smoke exposure on renal and other health outcomes in mice offspring.” 2016. Web. 30 Nov 2020.

Vancouver:

Al-Odat I. Effects of maternal cigarette smoke exposure on renal and other health outcomes in mice offspring. [Internet] [Thesis]. University of Technology, Sydney; 2016. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10453/43398.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Odat I. Effects of maternal cigarette smoke exposure on renal and other health outcomes in mice offspring. [Thesis]. University of Technology, Sydney; 2016. Available from: http://hdl.handle.net/10453/43398

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Schrover, I.M. Metabolic consequences of adipose tissue dysfunction.

Degree: 2018, University Utrecht

 This thesis focuses on the consequences of adipose tissue dysfunction (ATD), which is defined as the imbalance of pro- and anti-inflammatory adipocytokines produced by adipose… (more)

Subjects/Keywords: adiposity; dysfunction; adipokines; metabolic syndrome; hypertension; obesity; cardiovascular disease; spectroscopy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schrover, I. M. (2018). Metabolic consequences of adipose tissue dysfunction. (Doctoral Dissertation). University Utrecht. Retrieved from https://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; https://dspace.library.uu.nl/handle/1874/372756

Chicago Manual of Style (16th Edition):

Schrover, I M. “Metabolic consequences of adipose tissue dysfunction.” 2018. Doctoral Dissertation, University Utrecht. Accessed November 30, 2020. https://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; https://dspace.library.uu.nl/handle/1874/372756.

MLA Handbook (7th Edition):

Schrover, I M. “Metabolic consequences of adipose tissue dysfunction.” 2018. Web. 30 Nov 2020.

Vancouver:

Schrover IM. Metabolic consequences of adipose tissue dysfunction. [Internet] [Doctoral dissertation]. University Utrecht; 2018. [cited 2020 Nov 30]. Available from: https://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; https://dspace.library.uu.nl/handle/1874/372756.

Council of Science Editors:

Schrover IM. Metabolic consequences of adipose tissue dysfunction. [Doctoral Dissertation]. University Utrecht; 2018. Available from: https://dspace.library.uu.nl/handle/1874/372756 ; URN:NBN:NL:UI:10-1874-372756 ; URN:NBN:NL:UI:10-1874-372756 ; https://dspace.library.uu.nl/handle/1874/372756


University of Arizona

24. Zibadi, Sherma. Metabolic Syndrome-Induced Cardiac Fibrosis .

Degree: 2009, University of Arizona

 Recent studies support the association between metabolic syndrome (MetS), a cluster of cardiovascular risk factors, and diastolic dysfunction. Disproportionate collagen accumulation, particularly cross-linking of collagen,… (more)

Subjects/Keywords: Diastolic dysfunction; Fibrosis; Leptin; Lysyl oxidase; Metabolic syndrome; T-lymphocyte

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zibadi, S. (2009). Metabolic Syndrome-Induced Cardiac Fibrosis . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/195321

Chicago Manual of Style (16th Edition):

Zibadi, Sherma. “Metabolic Syndrome-Induced Cardiac Fibrosis .” 2009. Doctoral Dissertation, University of Arizona. Accessed November 30, 2020. http://hdl.handle.net/10150/195321.

MLA Handbook (7th Edition):

Zibadi, Sherma. “Metabolic Syndrome-Induced Cardiac Fibrosis .” 2009. Web. 30 Nov 2020.

Vancouver:

Zibadi S. Metabolic Syndrome-Induced Cardiac Fibrosis . [Internet] [Doctoral dissertation]. University of Arizona; 2009. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10150/195321.

Council of Science Editors:

Zibadi S. Metabolic Syndrome-Induced Cardiac Fibrosis . [Doctoral Dissertation]. University of Arizona; 2009. Available from: http://hdl.handle.net/10150/195321

25. Lachaux, Marianne. Nouvelles cibles pharmacologiques du traitement de la dysfonction cardiovasculaire associée au syndrome métabolique : New pharmacological treatment of Metabolic Syndrome associated cardiovascular dysfunctions.

Degree: Docteur es, Sciences de la vie et de la sante, 2019, Normandie

Le syndrome métabolique (SM) est associé avec une augmentation du risque de survenue d’évènements cardiovasculaires et plus particulièrement d’insuffisance cardiaque à fraction d’éjection préservée (ICFEp).… (more)

Subjects/Keywords: Syndrome métabolique; Insuffisance cardiaque à fraction d'éjection préservée; Macitentan; Finérénone; Iméglimine; Dysfonction diastolique; Dysfonction endothéliale; Metabolic syndrome; Heart failure with reserved ejection fraction; Macitentan; Finérénone; Imeglimine; Diastolic dysfunction; Endothelial dysfunction; 616.39

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lachaux, M. (2019). Nouvelles cibles pharmacologiques du traitement de la dysfonction cardiovasculaire associée au syndrome métabolique : New pharmacological treatment of Metabolic Syndrome associated cardiovascular dysfunctions. (Doctoral Dissertation). Normandie. Retrieved from http://www.theses.fr/2019NORMR010

Chicago Manual of Style (16th Edition):

Lachaux, Marianne. “Nouvelles cibles pharmacologiques du traitement de la dysfonction cardiovasculaire associée au syndrome métabolique : New pharmacological treatment of Metabolic Syndrome associated cardiovascular dysfunctions.” 2019. Doctoral Dissertation, Normandie. Accessed November 30, 2020. http://www.theses.fr/2019NORMR010.

MLA Handbook (7th Edition):

Lachaux, Marianne. “Nouvelles cibles pharmacologiques du traitement de la dysfonction cardiovasculaire associée au syndrome métabolique : New pharmacological treatment of Metabolic Syndrome associated cardiovascular dysfunctions.” 2019. Web. 30 Nov 2020.

Vancouver:

Lachaux M. Nouvelles cibles pharmacologiques du traitement de la dysfonction cardiovasculaire associée au syndrome métabolique : New pharmacological treatment of Metabolic Syndrome associated cardiovascular dysfunctions. [Internet] [Doctoral dissertation]. Normandie; 2019. [cited 2020 Nov 30]. Available from: http://www.theses.fr/2019NORMR010.

Council of Science Editors:

Lachaux M. Nouvelles cibles pharmacologiques du traitement de la dysfonction cardiovasculaire associée au syndrome métabolique : New pharmacological treatment of Metabolic Syndrome associated cardiovascular dysfunctions. [Doctoral Dissertation]. Normandie; 2019. Available from: http://www.theses.fr/2019NORMR010

26. Earl, Nathan R. Microvascular Function in Metabolically Healthy Groups Differing in BMI and Waist Circumference.

Degree: MS, 2014, Brigham Young University

  BACKGROUND: Microvascular dysfunction (MD: impaired performance of blood flow, tissue perfusion, blood pressure, etc.) is one of the earliest stages in the progression of… (more)

Subjects/Keywords: microvascular dysfunction; abdominal obesity; overweight; obese; metabolically healthy; metabolic syndrome; Exercise Science

…Macrovascular and microvascular dysfunction in the metabolic syndrome. Hypertension Research. Apr… …Guillherme L, de Aguiar K, et al. Vascular dysfunction in metabolic disorders: evaluation of some… …differ in BMI and waist circumference but lack any other indicator of metabolic syndrome. While… …any other indicator of metabolic syndrome. We hypothesized that the microvascular function… …without any clinical symptoms associated with metabolic syndrome. The comparison group (… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Earl, N. R. (2014). Microvascular Function in Metabolically Healthy Groups Differing in BMI and Waist Circumference. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5336&context=etd

Chicago Manual of Style (16th Edition):

Earl, Nathan R. “Microvascular Function in Metabolically Healthy Groups Differing in BMI and Waist Circumference.” 2014. Masters Thesis, Brigham Young University. Accessed November 30, 2020. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5336&context=etd.

MLA Handbook (7th Edition):

Earl, Nathan R. “Microvascular Function in Metabolically Healthy Groups Differing in BMI and Waist Circumference.” 2014. Web. 30 Nov 2020.

Vancouver:

Earl NR. Microvascular Function in Metabolically Healthy Groups Differing in BMI and Waist Circumference. [Internet] [Masters thesis]. Brigham Young University; 2014. [cited 2020 Nov 30]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5336&context=etd.

Council of Science Editors:

Earl NR. Microvascular Function in Metabolically Healthy Groups Differing in BMI and Waist Circumference. [Masters Thesis]. Brigham Young University; 2014. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5336&context=etd


North-West University

27. Fourie, Catharina Maria Theresia. The influence of HIV infection on vascular function in an African population / Catharina Maria Theresia Fourie .

Degree: 2010, North-West University

Subjects/Keywords: HIV-1 subtype C; Metabolic syndrome; Dyslipidemia; Lipodystrophy; Inflammation; Endothelial dysfunction; Vascular aging; suPAR; Antiretroviral therapy; Black South Africans

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fourie, C. M. T. (2010). The influence of HIV infection on vascular function in an African population / Catharina Maria Theresia Fourie . (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/4356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fourie, Catharina Maria Theresia. “The influence of HIV infection on vascular function in an African population / Catharina Maria Theresia Fourie .” 2010. Thesis, North-West University. Accessed November 30, 2020. http://hdl.handle.net/10394/4356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fourie, Catharina Maria Theresia. “The influence of HIV infection on vascular function in an African population / Catharina Maria Theresia Fourie .” 2010. Web. 30 Nov 2020.

Vancouver:

Fourie CMT. The influence of HIV infection on vascular function in an African population / Catharina Maria Theresia Fourie . [Internet] [Thesis]. North-West University; 2010. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10394/4356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fourie CMT. The influence of HIV infection on vascular function in an African population / Catharina Maria Theresia Fourie . [Thesis]. North-West University; 2010. Available from: http://hdl.handle.net/10394/4356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Safiedeen, Zainab. Rôle de l'interaction entre le réticulum endoplasmique et les mitochondries dans la dysfonction endothéliale induite par des microparticules humaines : Role of the interaction between endoplasmic reticulum and mitochondria in endothelial dysfunction induced by human microparticles.

Degree: Docteur es, Medecine, 2016, Angers; Université libannaise de Beyrouth

Le syndrome métabolique est constitué d'une constellation d'anomalies métaboliques telles que l'obésité centrale, une altération de la glycémie à jeun, une hypertriglycéridémie, un faible taux… (more)

Subjects/Keywords: Syndrome métabolique; Microparticules; Maladies cardiovasculaires; Dysfonction endothéliale; Monoxyde d’azote; Stress du réticulum endoplasmique; Mitochondrie; Stress oxydative; Metabolic syndrome; Microparticles; Cardiovascular diseases; Endothelial dysfunction; Nitric oxide; Endoplasmic reticulum stress; Mitochondria; Oxidative stress; 571.6; 616.39

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Safiedeen, Z. (2016). Rôle de l'interaction entre le réticulum endoplasmique et les mitochondries dans la dysfonction endothéliale induite par des microparticules humaines : Role of the interaction between endoplasmic reticulum and mitochondria in endothelial dysfunction induced by human microparticles. (Doctoral Dissertation). Angers; Université libannaise de Beyrouth. Retrieved from http://www.theses.fr/2016ANGE0063

Chicago Manual of Style (16th Edition):

Safiedeen, Zainab. “Rôle de l'interaction entre le réticulum endoplasmique et les mitochondries dans la dysfonction endothéliale induite par des microparticules humaines : Role of the interaction between endoplasmic reticulum and mitochondria in endothelial dysfunction induced by human microparticles.” 2016. Doctoral Dissertation, Angers; Université libannaise de Beyrouth. Accessed November 30, 2020. http://www.theses.fr/2016ANGE0063.

MLA Handbook (7th Edition):

Safiedeen, Zainab. “Rôle de l'interaction entre le réticulum endoplasmique et les mitochondries dans la dysfonction endothéliale induite par des microparticules humaines : Role of the interaction between endoplasmic reticulum and mitochondria in endothelial dysfunction induced by human microparticles.” 2016. Web. 30 Nov 2020.

Vancouver:

Safiedeen Z. Rôle de l'interaction entre le réticulum endoplasmique et les mitochondries dans la dysfonction endothéliale induite par des microparticules humaines : Role of the interaction between endoplasmic reticulum and mitochondria in endothelial dysfunction induced by human microparticles. [Internet] [Doctoral dissertation]. Angers; Université libannaise de Beyrouth; 2016. [cited 2020 Nov 30]. Available from: http://www.theses.fr/2016ANGE0063.

Council of Science Editors:

Safiedeen Z. Rôle de l'interaction entre le réticulum endoplasmique et les mitochondries dans la dysfonction endothéliale induite par des microparticules humaines : Role of the interaction between endoplasmic reticulum and mitochondria in endothelial dysfunction induced by human microparticles. [Doctoral Dissertation]. Angers; Université libannaise de Beyrouth; 2016. Available from: http://www.theses.fr/2016ANGE0063


Virginia Tech

29. Guill, Stephen Gregory. The Influence of Obstructive Sleep Apnea Syndrome on Insulin Resistance, Metabolic Syndrome, and Endothelial Dysfunction in Young Men.

Degree: PhD, Human Nutrition, Foods, and Exercise, 2007, Virginia Tech

 Obstructive sleep apnea syndrome (OSAS), a chronic respiratory disorder affecting as many as 1 in 5 adults, is associated with repetitive collapse of the upper… (more)

Subjects/Keywords: Obstructive Sleep Apnea Syndrome; Insulin Resistance; Metabolic Syndrome; Endothelial Dysfunction; Adipokine; Central Abdominal Fat

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guill, S. G. (2007). The Influence of Obstructive Sleep Apnea Syndrome on Insulin Resistance, Metabolic Syndrome, and Endothelial Dysfunction in Young Men. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/27384

Chicago Manual of Style (16th Edition):

Guill, Stephen Gregory. “The Influence of Obstructive Sleep Apnea Syndrome on Insulin Resistance, Metabolic Syndrome, and Endothelial Dysfunction in Young Men.” 2007. Doctoral Dissertation, Virginia Tech. Accessed November 30, 2020. http://hdl.handle.net/10919/27384.

MLA Handbook (7th Edition):

Guill, Stephen Gregory. “The Influence of Obstructive Sleep Apnea Syndrome on Insulin Resistance, Metabolic Syndrome, and Endothelial Dysfunction in Young Men.” 2007. Web. 30 Nov 2020.

Vancouver:

Guill SG. The Influence of Obstructive Sleep Apnea Syndrome on Insulin Resistance, Metabolic Syndrome, and Endothelial Dysfunction in Young Men. [Internet] [Doctoral dissertation]. Virginia Tech; 2007. [cited 2020 Nov 30]. Available from: http://hdl.handle.net/10919/27384.

Council of Science Editors:

Guill SG. The Influence of Obstructive Sleep Apnea Syndrome on Insulin Resistance, Metabolic Syndrome, and Endothelial Dysfunction in Young Men. [Doctoral Dissertation]. Virginia Tech; 2007. Available from: http://hdl.handle.net/10919/27384


University of Dundee

30. Smirni, Salvatore. Nonlinear dynamics of microcirculation and energy metabolism for the prediction of cardiovascular risk.

Degree: PhD, 2018, University of Dundee

 The peripheral skin microcirculation reflects the overall health status of the cardiovascular system and can be examined non-invasively by laser methods to assess early cardiovascular… (more)

Subjects/Keywords: Skin microcirculation; Energy metabolism; Nonlinear dynamics; Continuous wavelet transform; Vasomotion; Metabolic oscillations; Microvascular oscillations; Attractors; Cardiovascular disease risk; Oxidative stress; Endothelial Dysfunction; Biomedical Optics; Biophotonics; Bioenergetics; Nonlinear phenomena; Biophysics; Physiology; Blood flow; Fluorescence Spectroscopy; Laser Doppler Flowmetry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smirni, S. (2018). Nonlinear dynamics of microcirculation and energy metabolism for the prediction of cardiovascular risk. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/c551cbef-6f00-48ef-b753-ad76ac93daf4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.762962

Chicago Manual of Style (16th Edition):

Smirni, Salvatore. “Nonlinear dynamics of microcirculation and energy metabolism for the prediction of cardiovascular risk.” 2018. Doctoral Dissertation, University of Dundee. Accessed November 30, 2020. https://discovery.dundee.ac.uk/en/studentTheses/c551cbef-6f00-48ef-b753-ad76ac93daf4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.762962.

MLA Handbook (7th Edition):

Smirni, Salvatore. “Nonlinear dynamics of microcirculation and energy metabolism for the prediction of cardiovascular risk.” 2018. Web. 30 Nov 2020.

Vancouver:

Smirni S. Nonlinear dynamics of microcirculation and energy metabolism for the prediction of cardiovascular risk. [Internet] [Doctoral dissertation]. University of Dundee; 2018. [cited 2020 Nov 30]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/c551cbef-6f00-48ef-b753-ad76ac93daf4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.762962.

Council of Science Editors:

Smirni S. Nonlinear dynamics of microcirculation and energy metabolism for the prediction of cardiovascular risk. [Doctoral Dissertation]. University of Dundee; 2018. Available from: https://discovery.dundee.ac.uk/en/studentTheses/c551cbef-6f00-48ef-b753-ad76ac93daf4 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.762962

[1] [2]

.