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You searched for subject:(macrophage). Showing records 1 – 30 of 772 total matches.

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Texas A&M University

1. Chou, Wen-Ko. Basic and Applied Aspects of Chicken Macrophage Biology.

Degree: 2013, Texas A&M University

 The best way to prevent infectious disease is to directly establish mucosal immunity and also induce circulatory immunity to avoid pathogen invasion and spread. In… (more)

Subjects/Keywords: Macrophage; CD40

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APA (6th Edition):

Chou, W. (2013). Basic and Applied Aspects of Chicken Macrophage Biology. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158623

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chou, Wen-Ko. “Basic and Applied Aspects of Chicken Macrophage Biology.” 2013. Thesis, Texas A&M University. Accessed July 20, 2018. http://hdl.handle.net/1969.1/158623.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chou, Wen-Ko. “Basic and Applied Aspects of Chicken Macrophage Biology.” 2013. Web. 20 Jul 2018.

Vancouver:

Chou W. Basic and Applied Aspects of Chicken Macrophage Biology. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1969.1/158623.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chou W. Basic and Applied Aspects of Chicken Macrophage Biology. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/158623

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

2. Hardbower, Dana Michelle. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.

Degree: PhD, Microbiology and Immunology, 2017, Vanderbilt University

 Macrophages represent a dynamic and plastic subset of the innate immune system. Macrophage functions include immune surveillance and clearance of pathogens, but they have also… (more)

Subjects/Keywords: Macrophage; EGFR; ODC; Helicobacter pylori; Macrophage Activation

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APA (6th Edition):

Hardbower, D. M. (2017). Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03142017-111446/ ;

Chicago Manual of Style (16th Edition):

Hardbower, Dana Michelle. “Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed July 20, 2018. http://etd.library.vanderbilt.edu/available/etd-03142017-111446/ ;.

MLA Handbook (7th Edition):

Hardbower, Dana Michelle. “Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis.” 2017. Web. 20 Jul 2018.

Vancouver:

Hardbower DM. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2018 Jul 20]. Available from: http://etd.library.vanderbilt.edu/available/etd-03142017-111446/ ;.

Council of Science Editors:

Hardbower DM. Mechanisms Regulating Macrophage Activation and Function during Bacterial Infection and Carcinogenesis. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-03142017-111446/ ;


Victoria University of Wellington

3. Kharkrang, Marie Clare Lydia. Mechanisms Involved in Type II Macrophage Activation and Effector Functions.

Degree: 2010, Victoria University of Wellington

 Autoimmunities are extremely difficult to treat and involved in their pathogenesis are pro-inflammatory immune responses redirected against one's own tissues. Studies in our lab have… (more)

Subjects/Keywords: Immunology; Macrophage; Imminomodulation

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APA (6th Edition):

Kharkrang, M. C. L. (2010). Mechanisms Involved in Type II Macrophage Activation and Effector Functions. (Masters Thesis). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/1451

Chicago Manual of Style (16th Edition):

Kharkrang, Marie Clare Lydia. “Mechanisms Involved in Type II Macrophage Activation and Effector Functions.” 2010. Masters Thesis, Victoria University of Wellington. Accessed July 20, 2018. http://hdl.handle.net/10063/1451.

MLA Handbook (7th Edition):

Kharkrang, Marie Clare Lydia. “Mechanisms Involved in Type II Macrophage Activation and Effector Functions.” 2010. Web. 20 Jul 2018.

Vancouver:

Kharkrang MCL. Mechanisms Involved in Type II Macrophage Activation and Effector Functions. [Internet] [Masters thesis]. Victoria University of Wellington; 2010. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/10063/1451.

Council of Science Editors:

Kharkrang MCL. Mechanisms Involved in Type II Macrophage Activation and Effector Functions. [Masters Thesis]. Victoria University of Wellington; 2010. Available from: http://hdl.handle.net/10063/1451


Victoria University of Wellington

4. Stone, Sarrabeth Marie. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.

Degree: 2013, Victoria University of Wellington

 Multiple Sclerosis (MS) is an immune-mediated disease of the central nervous system (CNS) which causes demyelination and damage to the neuronal axons. MS is a… (more)

Subjects/Keywords: Immunology; Macrophage; Microglia

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APA (6th Edition):

Stone, S. M. (2013). Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/3072

Chicago Manual of Style (16th Edition):

Stone, Sarrabeth Marie. “Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.” 2013. Doctoral Dissertation, Victoria University of Wellington. Accessed July 20, 2018. http://hdl.handle.net/10063/3072.

MLA Handbook (7th Edition):

Stone, Sarrabeth Marie. “Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis.” 2013. Web. 20 Jul 2018.

Vancouver:

Stone SM. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2013. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/10063/3072.

Council of Science Editors:

Stone SM. Type II Activation of Macrophages and Microglia: Roles in T cell biasing and experimental autoimmune encephalomyelitis. [Doctoral Dissertation]. Victoria University of Wellington; 2013. Available from: http://hdl.handle.net/10063/3072


University of Edinburgh

5. Ramachandran, Prakash. Identification and characterisation of the restorative hepatic macrophage.

Degree: PhD, 2014, University of Edinburgh

 Long thought to be irreversible, it is now clear that liver fibrogenesis is a dynamic process, with scar tissue capable of being remodelled as well… (more)

Subjects/Keywords: liver fibrosis; macrophage

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APA (6th Edition):

Ramachandran, P. (2014). Identification and characterisation of the restorative hepatic macrophage. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9555

Chicago Manual of Style (16th Edition):

Ramachandran, Prakash. “Identification and characterisation of the restorative hepatic macrophage.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed July 20, 2018. http://hdl.handle.net/1842/9555.

MLA Handbook (7th Edition):

Ramachandran, Prakash. “Identification and characterisation of the restorative hepatic macrophage.” 2014. Web. 20 Jul 2018.

Vancouver:

Ramachandran P. Identification and characterisation of the restorative hepatic macrophage. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1842/9555.

Council of Science Editors:

Ramachandran P. Identification and characterisation of the restorative hepatic macrophage. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/9555


University of Toledo Health Science Campus

6. Roach, Kylie A. Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>.

Degree: MSBS, College of Graduate Studies, 2007, University of Toledo Health Science Campus

 <i>Francisella tularensis</i>is a gram negative bacteria that is an intracellular pathogen that infects host macrophages. <i>F. ularensis</i>subspecies are able to survive and persist within macrophages… (more)

Subjects/Keywords: Francisella; iglC; Macrophage

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APA (6th Edition):

Roach, K. A. (2007). Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300

Chicago Manual of Style (16th Edition):

Roach, Kylie A. “Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>.” 2007. Masters Thesis, University of Toledo Health Science Campus. Accessed July 20, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300.

MLA Handbook (7th Edition):

Roach, Kylie A. “Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>.” 2007. Web. 20 Jul 2018.

Vancouver:

Roach KA. Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2007. [cited 2018 Jul 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300.

Council of Science Editors:

Roach KA. Mechanisms for Growth and Persistence of <i>Francisella tularensis</i>Within Macrophages: a Role for <i>iglC</i>. [Masters Thesis]. University of Toledo Health Science Campus; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1199283300


University of Edinburgh

7. Stutchfield, Benjamin Mark. Manipulating macrophages to enhance liver regeneration.

Degree: PhD, 2015, University of Edinburgh

 Acute liver failure confers a high risk of death, with liver transplantation offering the only effective therapy for life-threatening cases. Hepatic macrophages are crucial for… (more)

Subjects/Keywords: liver; macrophage; regeneration

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APA (6th Edition):

Stutchfield, B. M. (2015). Manipulating macrophages to enhance liver regeneration. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/21686

Chicago Manual of Style (16th Edition):

Stutchfield, Benjamin Mark. “Manipulating macrophages to enhance liver regeneration.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed July 20, 2018. http://hdl.handle.net/1842/21686.

MLA Handbook (7th Edition):

Stutchfield, Benjamin Mark. “Manipulating macrophages to enhance liver regeneration.” 2015. Web. 20 Jul 2018.

Vancouver:

Stutchfield BM. Manipulating macrophages to enhance liver regeneration. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1842/21686.

Council of Science Editors:

Stutchfield BM. Manipulating macrophages to enhance liver regeneration. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/21686


University of Toronto

8. Britto, Karen Elma. The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production.

Degree: 2010, University of Toronto

Atherosclerosis is an inflammatory disease of the cardiovascular system. Discoidin domain receptor 1 is a receptor tyrosine kinase that binds collagens. Previous work in our… (more)

Subjects/Keywords: DDR1; Macrophage; 0306

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APA (6th Edition):

Britto, K. E. (2010). The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/25438

Chicago Manual of Style (16th Edition):

Britto, Karen Elma. “The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production.” 2010. Masters Thesis, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/25438.

MLA Handbook (7th Edition):

Britto, Karen Elma. “The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production.” 2010. Web. 20 Jul 2018.

Vancouver:

Britto KE. The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/25438.

Council of Science Editors:

Britto KE. The Role of Discoidin Domain Receptor 1 (Ddr1) on Macrophages in Adhesion and Cytokine Production. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/25438


Cornell University

9. Sinha, Siddhartha. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.

Degree: 2014, Cornell University

 Atherosclerosis is a decades-long process whose patients often remain asymptomatic until after a heart attack or stroke. Novel therapeutic approaches focus on tuning the body's… (more)

Subjects/Keywords: Atherosclerosis; Inflammation; Macrophage

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APA (6th Edition):

Sinha, S. (2014). Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/37128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. ” 2014. Thesis, Cornell University. Accessed July 20, 2018. http://hdl.handle.net/1813/37128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. ” 2014. Web. 20 Jul 2018.

Vancouver:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Internet] [Thesis]. Cornell University; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1813/37128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/37128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

10. McHenry, Allison. Characterizing the functional phenotype of infiltrating macrophages in meningiomas.

Degree: MS, Medical Sciences and Public Health GMS, 2015, Boston University

 Meningiomas are the most common primary brain tumors, yet few successful non-invasive treatment options are available for patients. Immunotherapy has provided new insights into treatments… (more)

Subjects/Keywords: Immunology; Macrophage; Meningioma

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APA (6th Edition):

McHenry, A. (2015). Characterizing the functional phenotype of infiltrating macrophages in meningiomas. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/13987

Chicago Manual of Style (16th Edition):

McHenry, Allison. “Characterizing the functional phenotype of infiltrating macrophages in meningiomas.” 2015. Masters Thesis, Boston University. Accessed July 20, 2018. http://hdl.handle.net/2144/13987.

MLA Handbook (7th Edition):

McHenry, Allison. “Characterizing the functional phenotype of infiltrating macrophages in meningiomas.” 2015. Web. 20 Jul 2018.

Vancouver:

McHenry A. Characterizing the functional phenotype of infiltrating macrophages in meningiomas. [Internet] [Masters thesis]. Boston University; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/2144/13987.

Council of Science Editors:

McHenry A. Characterizing the functional phenotype of infiltrating macrophages in meningiomas. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/13987


University of Edinburgh

11. Thiruchelvam, Uma. The phenotype and role of the endometrial macrophage in regulating angiogenesis.

Degree: PhD, 2013, University of Edinburgh

 Introduction: The uterine endometrium is a dynamic tissue that undergoes cycles of proliferation, differentiation breakdown and repair in response to fluctuations in the ovarian-derived sex… (more)

Subjects/Keywords: 612.6; endometrium; macrophage

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APA (6th Edition):

Thiruchelvam, U. (2013). The phenotype and role of the endometrial macrophage in regulating angiogenesis. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/18013

Chicago Manual of Style (16th Edition):

Thiruchelvam, Uma. “The phenotype and role of the endometrial macrophage in regulating angiogenesis.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed July 20, 2018. http://hdl.handle.net/1842/18013.

MLA Handbook (7th Edition):

Thiruchelvam, Uma. “The phenotype and role of the endometrial macrophage in regulating angiogenesis.” 2013. Web. 20 Jul 2018.

Vancouver:

Thiruchelvam U. The phenotype and role of the endometrial macrophage in regulating angiogenesis. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1842/18013.

Council of Science Editors:

Thiruchelvam U. The phenotype and role of the endometrial macrophage in regulating angiogenesis. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/18013


University of Plymouth

12. Habil, Neama. Probiotic modulation of mucosal immune responses in an in vitro co-culture model.

Degree: PhD, 2013, University of Plymouth

 Probiotics confer health benefits through many mechanisms including modulation of the gut immune system. Gut mucosal macrophages play a pivotal role in driving mucosal immune… (more)

Subjects/Keywords: Mucosal Immunology, Probiotic; macrophage, inflammation

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APA (6th Edition):

Habil, N. (2013). Probiotic modulation of mucosal immune responses in an in vitro co-culture model. (Doctoral Dissertation). University of Plymouth. Retrieved from http://hdl.handle.net/10026.1/3132

Chicago Manual of Style (16th Edition):

Habil, Neama. “Probiotic modulation of mucosal immune responses in an in vitro co-culture model.” 2013. Doctoral Dissertation, University of Plymouth. Accessed July 20, 2018. http://hdl.handle.net/10026.1/3132.

MLA Handbook (7th Edition):

Habil, Neama. “Probiotic modulation of mucosal immune responses in an in vitro co-culture model.” 2013. Web. 20 Jul 2018.

Vancouver:

Habil N. Probiotic modulation of mucosal immune responses in an in vitro co-culture model. [Internet] [Doctoral dissertation]. University of Plymouth; 2013. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/10026.1/3132.

Council of Science Editors:

Habil N. Probiotic modulation of mucosal immune responses in an in vitro co-culture model. [Doctoral Dissertation]. University of Plymouth; 2013. Available from: http://hdl.handle.net/10026.1/3132


University of Plymouth

13. Belfield, Louise Alicia. Interactions between porphyromonas gingivalis and macrophages in oral pathology.

Degree: PhD, 2013, University of Plymouth

 Macrophages play a fundamental role in driving both inflammatory and immunosuppressive conditions of the oral mucosa. Periodontitis, a chronic inflammatory condition affecting the supporting structures… (more)

Subjects/Keywords: 616.07; Immunology, macrophage, dental

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APA (6th Edition):

Belfield, L. A. (2013). Interactions between porphyromonas gingivalis and macrophages in oral pathology. (Doctoral Dissertation). University of Plymouth. Retrieved from http://hdl.handle.net/10026.1/1611

Chicago Manual of Style (16th Edition):

Belfield, Louise Alicia. “Interactions between porphyromonas gingivalis and macrophages in oral pathology.” 2013. Doctoral Dissertation, University of Plymouth. Accessed July 20, 2018. http://hdl.handle.net/10026.1/1611.

MLA Handbook (7th Edition):

Belfield, Louise Alicia. “Interactions between porphyromonas gingivalis and macrophages in oral pathology.” 2013. Web. 20 Jul 2018.

Vancouver:

Belfield LA. Interactions between porphyromonas gingivalis and macrophages in oral pathology. [Internet] [Doctoral dissertation]. University of Plymouth; 2013. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/10026.1/1611.

Council of Science Editors:

Belfield LA. Interactions between porphyromonas gingivalis and macrophages in oral pathology. [Doctoral Dissertation]. University of Plymouth; 2013. Available from: http://hdl.handle.net/10026.1/1611


University of Minnesota

14. Anderson, Katie. Melanoma displays an evolutionarily conserved resistance to modulation of phagocytic signals.

Degree: PhD, Comparative and Molecular Biosciences, 2017, University of Minnesota

 Therapeutic activation of macrophage phagocytosis has the ability to restrain tumor growth through phagocytic clearance of tumor cells and activation of the adaptive immune response.… (more)

Subjects/Keywords: CD47; doxorubicin; macrophage; melanoma; phagocytosis

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APA (6th Edition):

Anderson, K. (2017). Melanoma displays an evolutionarily conserved resistance to modulation of phagocytic signals. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191409

Chicago Manual of Style (16th Edition):

Anderson, Katie. “Melanoma displays an evolutionarily conserved resistance to modulation of phagocytic signals.” 2017. Doctoral Dissertation, University of Minnesota. Accessed July 20, 2018. http://hdl.handle.net/11299/191409.

MLA Handbook (7th Edition):

Anderson, Katie. “Melanoma displays an evolutionarily conserved resistance to modulation of phagocytic signals.” 2017. Web. 20 Jul 2018.

Vancouver:

Anderson K. Melanoma displays an evolutionarily conserved resistance to modulation of phagocytic signals. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/11299/191409.

Council of Science Editors:

Anderson K. Melanoma displays an evolutionarily conserved resistance to modulation of phagocytic signals. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/191409


McMaster University

15. Novakowski, Kyle E. IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO.

Degree: PhD, 2018, McMaster University

Host defense against pathogenic organisms represents one of the most important and highly-conserved biological processes across the evolutionary timescale. The ability to detect, engulf and… (more)

Subjects/Keywords: MARCO; Macrophage; Scavenger Receptor; Phagocytosis

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APA (6th Edition):

Novakowski, K. E. (2018). IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/22802

Chicago Manual of Style (16th Edition):

Novakowski, Kyle E. “IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO.” 2018. Doctoral Dissertation, McMaster University. Accessed July 20, 2018. http://hdl.handle.net/11375/22802.

MLA Handbook (7th Edition):

Novakowski, Kyle E. “IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO.” 2018. Web. 20 Jul 2018.

Vancouver:

Novakowski KE. IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO. [Internet] [Doctoral dissertation]. McMaster University; 2018. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/11375/22802.

Council of Science Editors:

Novakowski KE. IDENTIFICATION AND FUNCTIONAL CHARACTERIZATION OF CONSERVED RESIDUES AND DOMAINS IN THE MACROPHAGE SCAVENGER RECEPTOR MARCO. [Doctoral Dissertation]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/22802


University of Edinburgh

16. Jubb, Alasdair. Effects of glucocorticoids in macrophages.

Degree: PhD, 2015, University of Edinburgh

 Glucocorticoids (GC) are powerful metabolic hormones with anti-inflammatory actions. Despite major side effects they remain widely prescribed therapies. GC regulates gene expression through an intracellular… (more)

Subjects/Keywords: 616.07; macrophage; glucocorticoids; chromatin; transcription

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APA (6th Edition):

Jubb, A. (2015). Effects of glucocorticoids in macrophages. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/19521

Chicago Manual of Style (16th Edition):

Jubb, Alasdair. “Effects of glucocorticoids in macrophages.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed July 20, 2018. http://hdl.handle.net/1842/19521.

MLA Handbook (7th Edition):

Jubb, Alasdair. “Effects of glucocorticoids in macrophages.” 2015. Web. 20 Jul 2018.

Vancouver:

Jubb A. Effects of glucocorticoids in macrophages. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1842/19521.

Council of Science Editors:

Jubb A. Effects of glucocorticoids in macrophages. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/19521


University of Alberta

17. Diaz Satizabal, Laura P. Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation.

Degree: MS, Department of Biological Sciences, 2013, University of Alberta

 During the antibody affinity maturation process, changes to the immunoglobulin genes are initiated by the B-cell specific enzyme activation-induced cytidine deaminase (AID). These changes are… (more)

Subjects/Keywords: Melano-macrophage; Affinity maturation

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APA (6th Edition):

Diaz Satizabal, L. P. (2013). Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/0v838097f

Chicago Manual of Style (16th Edition):

Diaz Satizabal, Laura P. “Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation.” 2013. Masters Thesis, University of Alberta. Accessed July 20, 2018. https://era.library.ualberta.ca/files/0v838097f.

MLA Handbook (7th Edition):

Diaz Satizabal, Laura P. “Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation.” 2013. Web. 20 Jul 2018.

Vancouver:

Diaz Satizabal LP. Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation. [Internet] [Masters thesis]. University of Alberta; 2013. [cited 2018 Jul 20]. Available from: https://era.library.ualberta.ca/files/0v838097f.

Council of Science Editors:

Diaz Satizabal LP. Melano-macrophage characterization and their possible role in the goldfish (Carassius auratus) antibody affinity maturation. [Masters Thesis]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/0v838097f


University of Alberta

18. Rieger, Aja M. Mechanisms of Acute Inflammatory Control by Phagocytes.

Degree: PhD, Department of Biological Sciences, 2014, University of Alberta

 Phagocytosis is an important, evolutionarily conserved mechanism integral to immune defense and homeostasis. Phagocytosis is initiated by the interaction of receptors on the surface of… (more)

Subjects/Keywords: Phagocytosis; Goldfish; sCSR-1R; Macrophage

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APA (6th Edition):

Rieger, A. M. (2014). Mechanisms of Acute Inflammatory Control by Phagocytes. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cxw42n799q

Chicago Manual of Style (16th Edition):

Rieger, Aja M. “Mechanisms of Acute Inflammatory Control by Phagocytes.” 2014. Doctoral Dissertation, University of Alberta. Accessed July 20, 2018. https://era.library.ualberta.ca/files/cxw42n799q.

MLA Handbook (7th Edition):

Rieger, Aja M. “Mechanisms of Acute Inflammatory Control by Phagocytes.” 2014. Web. 20 Jul 2018.

Vancouver:

Rieger AM. Mechanisms of Acute Inflammatory Control by Phagocytes. [Internet] [Doctoral dissertation]. University of Alberta; 2014. [cited 2018 Jul 20]. Available from: https://era.library.ualberta.ca/files/cxw42n799q.

Council of Science Editors:

Rieger AM. Mechanisms of Acute Inflammatory Control by Phagocytes. [Doctoral Dissertation]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/cxw42n799q


University of Adelaide

19. Borlace, Glenn Neville. Helicobacter pylori: reduced phagocytic killing and altered phagosome maturation in primary human macrophages.

Degree: 2011, University of Adelaide

 Helicobacter pylori (H. pylori) colonises the human gastric mucosa and is the principal causative agent of gastric and duodenal ulcers. Long term infection with H.… (more)

Subjects/Keywords: Helicobacter pylori; macrophage; phagosome maturation

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APA (6th Edition):

Borlace, G. N. (2011). Helicobacter pylori: reduced phagocytic killing and altered phagosome maturation in primary human macrophages. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/66345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Borlace, Glenn Neville. “Helicobacter pylori: reduced phagocytic killing and altered phagosome maturation in primary human macrophages.” 2011. Thesis, University of Adelaide. Accessed July 20, 2018. http://hdl.handle.net/2440/66345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Borlace, Glenn Neville. “Helicobacter pylori: reduced phagocytic killing and altered phagosome maturation in primary human macrophages.” 2011. Web. 20 Jul 2018.

Vancouver:

Borlace GN. Helicobacter pylori: reduced phagocytic killing and altered phagosome maturation in primary human macrophages. [Internet] [Thesis]. University of Adelaide; 2011. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/2440/66345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Borlace GN. Helicobacter pylori: reduced phagocytic killing and altered phagosome maturation in primary human macrophages. [Thesis]. University of Adelaide; 2011. Available from: http://hdl.handle.net/2440/66345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

20. Mokarram-Dorri, Nassir. Modulating immune response inside biomaterial-based nerve conduits to stimulate endogenous peripheral nerve regeneration.

Degree: PhD, Materials Science and Engineering, 2015, Georgia Tech

 Injuries to the peripheral nervous system (PNS) are major and common source of disability, impairing the ability to move muscles and/or feel normal sensations, or… (more)

Subjects/Keywords: Nerve repair; Immunomodulation; Macrophage

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APA (6th Edition):

Mokarram-Dorri, N. (2015). Modulating immune response inside biomaterial-based nerve conduits to stimulate endogenous peripheral nerve regeneration. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/54860

Chicago Manual of Style (16th Edition):

Mokarram-Dorri, Nassir. “Modulating immune response inside biomaterial-based nerve conduits to stimulate endogenous peripheral nerve regeneration.” 2015. Doctoral Dissertation, Georgia Tech. Accessed July 20, 2018. http://hdl.handle.net/1853/54860.

MLA Handbook (7th Edition):

Mokarram-Dorri, Nassir. “Modulating immune response inside biomaterial-based nerve conduits to stimulate endogenous peripheral nerve regeneration.” 2015. Web. 20 Jul 2018.

Vancouver:

Mokarram-Dorri N. Modulating immune response inside biomaterial-based nerve conduits to stimulate endogenous peripheral nerve regeneration. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1853/54860.

Council of Science Editors:

Mokarram-Dorri N. Modulating immune response inside biomaterial-based nerve conduits to stimulate endogenous peripheral nerve regeneration. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/54860


Universiteit Utrecht

21. Brown, M.K. Inception of the metastatic phenotype by macrophage-cancer cell fusion.

Degree: 2011, Universiteit Utrecht

 It is now well-established that patient outcome are good if the primary tumor has not spread; can be surgically removed; treated locally with radiation; or… (more)

Subjects/Keywords: Metastasis; Cancer; Macrophage; Fusion theory

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APA (6th Edition):

Brown, M. K. (2011). Inception of the metastatic phenotype by macrophage-cancer cell fusion. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/204660

Chicago Manual of Style (16th Edition):

Brown, M K. “Inception of the metastatic phenotype by macrophage-cancer cell fusion.” 2011. Masters Thesis, Universiteit Utrecht. Accessed July 20, 2018. http://dspace.library.uu.nl:8080/handle/1874/204660.

MLA Handbook (7th Edition):

Brown, M K. “Inception of the metastatic phenotype by macrophage-cancer cell fusion.” 2011. Web. 20 Jul 2018.

Vancouver:

Brown MK. Inception of the metastatic phenotype by macrophage-cancer cell fusion. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2018 Jul 20]. Available from: http://dspace.library.uu.nl:8080/handle/1874/204660.

Council of Science Editors:

Brown MK. Inception of the metastatic phenotype by macrophage-cancer cell fusion. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/204660


University of Toronto

22. Tawadros, Patrick. The Role of Ceramide in Oxidant-mediated Priming of Macrophages for LPS Signaling.

Degree: 2009, University of Toronto

 Introduction: Civilian trauma remains a significant health care problem in North American society. Hemorrhagic shock and resuscitation (S/R) have been shown to prime the immune… (more)

Subjects/Keywords: oxidative stress; macrophage; ceramide; 0307

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APA (6th Edition):

Tawadros, P. (2009). The Role of Ceramide in Oxidant-mediated Priming of Macrophages for LPS Signaling. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/19301

Chicago Manual of Style (16th Edition):

Tawadros, Patrick. “The Role of Ceramide in Oxidant-mediated Priming of Macrophages for LPS Signaling.” 2009. Doctoral Dissertation, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/19301.

MLA Handbook (7th Edition):

Tawadros, Patrick. “The Role of Ceramide in Oxidant-mediated Priming of Macrophages for LPS Signaling.” 2009. Web. 20 Jul 2018.

Vancouver:

Tawadros P. The Role of Ceramide in Oxidant-mediated Priming of Macrophages for LPS Signaling. [Internet] [Doctoral dissertation]. University of Toronto; 2009. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/19301.

Council of Science Editors:

Tawadros P. The Role of Ceramide in Oxidant-mediated Priming of Macrophages for LPS Signaling. [Doctoral Dissertation]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/19301


North Carolina State University

23. Summers, Caroline Rueda. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.

Degree: MS, Nutrition, 2010, North Carolina State University

 Recently, the World Health Organization estimated that chronic diseases are responsible for 46% of disease occurrence and 59% of all deaths worldwide. Growing evidence suggests… (more)

Subjects/Keywords: LPS; tea; mouse macrophage; inflammation

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APA (6th Edition):

Summers, C. R. (2010). Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/6332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Summers, Caroline Rueda. “Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.” 2010. Thesis, North Carolina State University. Accessed July 20, 2018. http://www.lib.ncsu.edu/resolver/1840.16/6332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Summers, Caroline Rueda. “Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.” 2010. Web. 20 Jul 2018.

Vancouver:

Summers CR. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. [Internet] [Thesis]. North Carolina State University; 2010. [cited 2018 Jul 20]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Summers CR. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. [Thesis]. North Carolina State University; 2010. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. GALVAN-PENA, SILVIA. Malonylation of GAPDH in the inflammatory response in macrophages.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2018, Trinity College Dublin

 Succinylation and malonylation are two recently discovered protein post-translational modifications. They involve the attachment to lysines of metabolism-derived succinyl and malonyl groups respectively, resulting in… (more)

Subjects/Keywords: macrophage; inflammation; GAPDH; malonylation

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APA (6th Edition):

GALVAN-PENA, S. (2018). Malonylation of GAPDH in the inflammatory response in macrophages. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/82728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

GALVAN-PENA, SILVIA. “Malonylation of GAPDH in the inflammatory response in macrophages.” 2018. Thesis, Trinity College Dublin. Accessed July 20, 2018. http://hdl.handle.net/2262/82728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

GALVAN-PENA, SILVIA. “Malonylation of GAPDH in the inflammatory response in macrophages.” 2018. Web. 20 Jul 2018.

Vancouver:

GALVAN-PENA S. Malonylation of GAPDH in the inflammatory response in macrophages. [Internet] [Thesis]. Trinity College Dublin; 2018. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/2262/82728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

GALVAN-PENA S. Malonylation of GAPDH in the inflammatory response in macrophages. [Thesis]. Trinity College Dublin; 2018. Available from: http://hdl.handle.net/2262/82728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

25. Renaud, Stephen. ROLE OF ACTIVATED MACROPHAGES AND PRO-INFLAMMATORY CYTOKINES IN PLACENTAL TROPHOBLAST INVASION AND FETAL OUTCOME .

Degree: Anatomy & Cell Biology, 2008, Queens University

 The invasion of trophoblast cells into the uterine wall is an essential component of normal human pregnancy. These trophoblast cells transform the uterine spiral arterioles… (more)

Subjects/Keywords: Trophoblast; Macrophage

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APA (6th Edition):

Renaud, S. (2008). ROLE OF ACTIVATED MACROPHAGES AND PRO-INFLAMMATORY CYTOKINES IN PLACENTAL TROPHOBLAST INVASION AND FETAL OUTCOME . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/1537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Renaud, Stephen. “ROLE OF ACTIVATED MACROPHAGES AND PRO-INFLAMMATORY CYTOKINES IN PLACENTAL TROPHOBLAST INVASION AND FETAL OUTCOME .” 2008. Thesis, Queens University. Accessed July 20, 2018. http://hdl.handle.net/1974/1537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Renaud, Stephen. “ROLE OF ACTIVATED MACROPHAGES AND PRO-INFLAMMATORY CYTOKINES IN PLACENTAL TROPHOBLAST INVASION AND FETAL OUTCOME .” 2008. Web. 20 Jul 2018.

Vancouver:

Renaud S. ROLE OF ACTIVATED MACROPHAGES AND PRO-INFLAMMATORY CYTOKINES IN PLACENTAL TROPHOBLAST INVASION AND FETAL OUTCOME . [Internet] [Thesis]. Queens University; 2008. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1974/1537.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Renaud S. ROLE OF ACTIVATED MACROPHAGES AND PRO-INFLAMMATORY CYTOKINES IN PLACENTAL TROPHOBLAST INVASION AND FETAL OUTCOME . [Thesis]. Queens University; 2008. Available from: http://hdl.handle.net/1974/1537

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

26. Morris, Matthew. Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide.

Degree: PhD, Animal and Poultry Sciences, 2014, Virginia Tech

 The innate immune system depends for its effectiveness on the function of specialized pattern recognition receptors which enable it to target pathogens for destruction on… (more)

Subjects/Keywords: Immunology; lipopolysaccharide; signaling; monocyte; macrophage

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APA (6th Edition):

Morris, M. (2014). Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/64253

Chicago Manual of Style (16th Edition):

Morris, Matthew. “Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide.” 2014. Doctoral Dissertation, Virginia Tech. Accessed July 20, 2018. http://hdl.handle.net/10919/64253.

MLA Handbook (7th Edition):

Morris, Matthew. “Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide.” 2014. Web. 20 Jul 2018.

Vancouver:

Morris M. Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide. [Internet] [Doctoral dissertation]. Virginia Tech; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/10919/64253.

Council of Science Editors:

Morris M. Molecular mechanisms responsible for the dynamic modulation of macrophage responses to varying dosages of lipopolysaccharide. [Doctoral Dissertation]. Virginia Tech; 2014. Available from: http://hdl.handle.net/10919/64253


University of Edinburgh

27. Zhang, Zhenguang. Role of macrophage 11β-HSD1 in inflammation mediated angiogenesis, arthritis and obesity.

Degree: PhD, 2014, University of Edinburgh

 11β-hydroxysteroid dehydrogenase type 1 (11β-HSD1, encoded by Hsd11b1) is an enzyme that predominantly converts inactive glucocorticoids (cortisone in human and most mammals, 11dehydro-corticosterone in mice… (more)

Subjects/Keywords: 616.07; 11ß-HSD1; macrophage; inflammation

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APA (6th Edition):

Zhang, Z. (2014). Role of macrophage 11β-HSD1 in inflammation mediated angiogenesis, arthritis and obesity. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/9553

Chicago Manual of Style (16th Edition):

Zhang, Zhenguang. “Role of macrophage 11β-HSD1 in inflammation mediated angiogenesis, arthritis and obesity.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed July 20, 2018. http://hdl.handle.net/1842/9553.

MLA Handbook (7th Edition):

Zhang, Zhenguang. “Role of macrophage 11β-HSD1 in inflammation mediated angiogenesis, arthritis and obesity.” 2014. Web. 20 Jul 2018.

Vancouver:

Zhang Z. Role of macrophage 11β-HSD1 in inflammation mediated angiogenesis, arthritis and obesity. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1842/9553.

Council of Science Editors:

Zhang Z. Role of macrophage 11β-HSD1 in inflammation mediated angiogenesis, arthritis and obesity. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/9553


University of Toronto

28. McSheffrey, Gordon Gerald. Host Cellular Responses to Neisseria gonorrhoeae.

Degree: 2016, University of Toronto

Neisseria gonorrhoeae is a human-restricted pathogen associated with increasing morbidity as antibiotic-resistant strains spread globally. N. gonorrhoeae possesses sophisticated mechanisms to survive the host immune… (more)

Subjects/Keywords: gonorrhea; macrophage; phosphoinositides; 0410

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APA (6th Edition):

McSheffrey, G. G. (2016). Host Cellular Responses to Neisseria gonorrhoeae. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/71686

Chicago Manual of Style (16th Edition):

McSheffrey, Gordon Gerald. “Host Cellular Responses to Neisseria gonorrhoeae.” 2016. Masters Thesis, University of Toronto. Accessed July 20, 2018. http://hdl.handle.net/1807/71686.

MLA Handbook (7th Edition):

McSheffrey, Gordon Gerald. “Host Cellular Responses to Neisseria gonorrhoeae.” 2016. Web. 20 Jul 2018.

Vancouver:

McSheffrey GG. Host Cellular Responses to Neisseria gonorrhoeae. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2018 Jul 20]. Available from: http://hdl.handle.net/1807/71686.

Council of Science Editors:

McSheffrey GG. Host Cellular Responses to Neisseria gonorrhoeae. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/71686


University of Notre Dame

29. Cristina Ricardo Carter. Host-Pathogen Interactions:.

Degree: MS, Biological Sciences, 2008, University of Notre Dame

  Leishmania species infect approximately 2 million people each year and 350 million people live in areas where they are at risk for infection. This… (more)

Subjects/Keywords: macrophage; complement receptor 3; leishmaniasis

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APA (6th Edition):

Carter, C. R. (2008). Host-Pathogen Interactions:. (Masters Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/4b29b566244

Chicago Manual of Style (16th Edition):

Carter, Cristina Ricardo. “Host-Pathogen Interactions:.” 2008. Masters Thesis, University of Notre Dame. Accessed July 20, 2018. https://curate.nd.edu/show/4b29b566244.

MLA Handbook (7th Edition):

Carter, Cristina Ricardo. “Host-Pathogen Interactions:.” 2008. Web. 20 Jul 2018.

Vancouver:

Carter CR. Host-Pathogen Interactions:. [Internet] [Masters thesis]. University of Notre Dame; 2008. [cited 2018 Jul 20]. Available from: https://curate.nd.edu/show/4b29b566244.

Council of Science Editors:

Carter CR. Host-Pathogen Interactions:. [Masters Thesis]. University of Notre Dame; 2008. Available from: https://curate.nd.edu/show/4b29b566244


University of Notre Dame

30. Rachel Elizabeth Kasuboski-Polando. The role of pattern recognition receptors in host cell signaling and phagosome maturation during infection with Leishmania.

Degree: PhD, Biological Sciences, 2009, University of Notre Dame

  Leishmaniasis is a devastating group of diseases causing substantial mortality and morbidity throughout the world. Leishmania are obligate intracellular organisms infecting and replicating inside… (more)

Subjects/Keywords: Macrophage; Leishmania; pattern recognition

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APA (6th Edition):

Kasuboski-Polando, R. E. (2009). The role of pattern recognition receptors in host cell signaling and phagosome maturation during infection with Leishmania. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/x059c536z9s

Chicago Manual of Style (16th Edition):

Kasuboski-Polando, Rachel Elizabeth. “The role of pattern recognition receptors in host cell signaling and phagosome maturation during infection with Leishmania.” 2009. Doctoral Dissertation, University of Notre Dame. Accessed July 20, 2018. https://curate.nd.edu/show/x059c536z9s.

MLA Handbook (7th Edition):

Kasuboski-Polando, Rachel Elizabeth. “The role of pattern recognition receptors in host cell signaling and phagosome maturation during infection with Leishmania.” 2009. Web. 20 Jul 2018.

Vancouver:

Kasuboski-Polando RE. The role of pattern recognition receptors in host cell signaling and phagosome maturation during infection with Leishmania. [Internet] [Doctoral dissertation]. University of Notre Dame; 2009. [cited 2018 Jul 20]. Available from: https://curate.nd.edu/show/x059c536z9s.

Council of Science Editors:

Kasuboski-Polando RE. The role of pattern recognition receptors in host cell signaling and phagosome maturation during infection with Leishmania. [Doctoral Dissertation]. University of Notre Dame; 2009. Available from: https://curate.nd.edu/show/x059c536z9s

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