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You searched for subject:(mTOR). Showing records 1 – 30 of 447 total matches.

[1] [2] [3] [4] [5] … [15]

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1. Demirkan, Gokhan. Profiling of mTORC1 Signaling in Rat Liver Using Mass Spectrometry.

Degree: PhD, Molecular Biology, Cell Biology, and Biochemistry, 2011, Brown University

 Our understanding of signaling networks in the physiological context of an organism remains limited, partly due to the technical challenge of identifying serine/threonine phosphorylated peptides… (more)

Subjects/Keywords: mTOR

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APA (6th Edition):

Demirkan, G. (2011). Profiling of mTORC1 Signaling in Rat Liver Using Mass Spectrometry. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:11198/

Chicago Manual of Style (16th Edition):

Demirkan, Gokhan. “Profiling of mTORC1 Signaling in Rat Liver Using Mass Spectrometry.” 2011. Doctoral Dissertation, Brown University. Accessed February 23, 2020. https://repository.library.brown.edu/studio/item/bdr:11198/.

MLA Handbook (7th Edition):

Demirkan, Gokhan. “Profiling of mTORC1 Signaling in Rat Liver Using Mass Spectrometry.” 2011. Web. 23 Feb 2020.

Vancouver:

Demirkan G. Profiling of mTORC1 Signaling in Rat Liver Using Mass Spectrometry. [Internet] [Doctoral dissertation]. Brown University; 2011. [cited 2020 Feb 23]. Available from: https://repository.library.brown.edu/studio/item/bdr:11198/.

Council of Science Editors:

Demirkan G. Profiling of mTORC1 Signaling in Rat Liver Using Mass Spectrometry. [Doctoral Dissertation]. Brown University; 2011. Available from: https://repository.library.brown.edu/studio/item/bdr:11198/

2. Hare, Stephen. The development and characterisation of everolimus resistant breast cancer cells.

Degree: PhD, 2018, Brunel University

 The mTOR inhibitor and rapalogue everolimus was approved use in 2012, in HR+, HER-2-, post-menopausal patients, who had previously failed aromatase inhibitor treatment. mTOR pathway… (more)

Subjects/Keywords: mTOR; Rapalogue

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APA (6th Edition):

Hare, S. (2018). The development and characterisation of everolimus resistant breast cancer cells. (Doctoral Dissertation). Brunel University. Retrieved from http://bura.brunel.ac.uk/handle/2438/17466 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767703

Chicago Manual of Style (16th Edition):

Hare, Stephen. “The development and characterisation of everolimus resistant breast cancer cells.” 2018. Doctoral Dissertation, Brunel University. Accessed February 23, 2020. http://bura.brunel.ac.uk/handle/2438/17466 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767703.

MLA Handbook (7th Edition):

Hare, Stephen. “The development and characterisation of everolimus resistant breast cancer cells.” 2018. Web. 23 Feb 2020.

Vancouver:

Hare S. The development and characterisation of everolimus resistant breast cancer cells. [Internet] [Doctoral dissertation]. Brunel University; 2018. [cited 2020 Feb 23]. Available from: http://bura.brunel.ac.uk/handle/2438/17466 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767703.

Council of Science Editors:

Hare S. The development and characterisation of everolimus resistant breast cancer cells. [Doctoral Dissertation]. Brunel University; 2018. Available from: http://bura.brunel.ac.uk/handle/2438/17466 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767703


Texas A&M University

3. Kim, Hye Mee. Polyphenolics from Mango (Mangifera indica L.) and Pomegranate (Punica granatum L.) Suppress Inflammation in in vivo and in vitro Models for Colitis.

Degree: 2013, Texas A&M University

 Ulcerative colitis is a chronic inflammation of the large intestine, and it may increase risk of human colorectal cancer. Polyphenolics from mango and pomegranate have… (more)

Subjects/Keywords: Mango; Pomegranate; Colitis; Rat; mTOR

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APA (6th Edition):

Kim, H. M. (2013). Polyphenolics from Mango (Mangifera indica L.) and Pomegranate (Punica granatum L.) Suppress Inflammation in in vivo and in vitro Models for Colitis. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Hye Mee. “Polyphenolics from Mango (Mangifera indica L.) and Pomegranate (Punica granatum L.) Suppress Inflammation in in vivo and in vitro Models for Colitis.” 2013. Thesis, Texas A&M University. Accessed February 23, 2020. http://hdl.handle.net/1969.1/151871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Hye Mee. “Polyphenolics from Mango (Mangifera indica L.) and Pomegranate (Punica granatum L.) Suppress Inflammation in in vivo and in vitro Models for Colitis.” 2013. Web. 23 Feb 2020.

Vancouver:

Kim HM. Polyphenolics from Mango (Mangifera indica L.) and Pomegranate (Punica granatum L.) Suppress Inflammation in in vivo and in vitro Models for Colitis. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/1969.1/151871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim HM. Polyphenolics from Mango (Mangifera indica L.) and Pomegranate (Punica granatum L.) Suppress Inflammation in in vivo and in vitro Models for Colitis. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

4. Raut, Anuja. Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism.

Degree: MS, Pharmaceutical Sciences, 2014, University of Southern California

 Rapamycin is a potent immunosuppressive macrolide which binds to FK506 binding protein (FKBP12), the cytosolic receptor to rapamycin. The FKBP12-Rapamycin complex binds to and allosterically… (more)

Subjects/Keywords: rapamycin; mTOR; immunosuppressant; pharmacology

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APA (6th Edition):

Raut, A. (2014). Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4002

Chicago Manual of Style (16th Edition):

Raut, Anuja. “Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism.” 2014. Masters Thesis, University of Southern California. Accessed February 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4002.

MLA Handbook (7th Edition):

Raut, Anuja. “Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism.” 2014. Web. 23 Feb 2020.

Vancouver:

Raut A. Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2020 Feb 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4002.

Council of Science Editors:

Raut A. Mechanism of action of rapamycin and its applications in aging, cancer therapy and metabolism. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385332/rec/4002


Wright State University

5. Mercer, Carol A. Structural and Signaling Elements Important for the Efficient Degradation of BHMT through Macroautophagy.

Degree: PhD, Biomedical Sciences PhD, 2007, Wright State University

 Healthy cells maintain a dynamic and responsive intracellular environment that is marked by the synthesis and degradation of proteins, complex macromolecules and organelles. Autophagy, literally… (more)

Subjects/Keywords: Autophagy; BHMT; mTOR; S6 kinase

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APA (6th Edition):

Mercer, C. A. (2007). Structural and Signaling Elements Important for the Efficient Degradation of BHMT through Macroautophagy. (Doctoral Dissertation). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1176234559

Chicago Manual of Style (16th Edition):

Mercer, Carol A. “Structural and Signaling Elements Important for the Efficient Degradation of BHMT through Macroautophagy.” 2007. Doctoral Dissertation, Wright State University. Accessed February 23, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=wright1176234559.

MLA Handbook (7th Edition):

Mercer, Carol A. “Structural and Signaling Elements Important for the Efficient Degradation of BHMT through Macroautophagy.” 2007. Web. 23 Feb 2020.

Vancouver:

Mercer CA. Structural and Signaling Elements Important for the Efficient Degradation of BHMT through Macroautophagy. [Internet] [Doctoral dissertation]. Wright State University; 2007. [cited 2020 Feb 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1176234559.

Council of Science Editors:

Mercer CA. Structural and Signaling Elements Important for the Efficient Degradation of BHMT through Macroautophagy. [Doctoral Dissertation]. Wright State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1176234559


Université de Montréal

6. Choudhury, Mayukh. The role of mechanistic target of rapamycin (mTOR) pathway and synaptic protein GABAA-R in cortical GABAergic cell connectivity .

Degree: 2016, Université de Montréal

 Quelque 30 % de la population neuronale du cortex mammalien est composée d’une population très hétérogène d’interneurones GABAergiques. Ces interneurones diffèrent quant à leur morphologie,… (more)

Subjects/Keywords: mTOR; GABA; neurodéveloppement; neurodevelopment

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APA (6th Edition):

Choudhury, M. (2016). The role of mechanistic target of rapamycin (mTOR) pathway and synaptic protein GABAA-R in cortical GABAergic cell connectivity . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choudhury, Mayukh. “The role of mechanistic target of rapamycin (mTOR) pathway and synaptic protein GABAA-R in cortical GABAergic cell connectivity .” 2016. Thesis, Université de Montréal. Accessed February 23, 2020. http://hdl.handle.net/1866/13978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choudhury, Mayukh. “The role of mechanistic target of rapamycin (mTOR) pathway and synaptic protein GABAA-R in cortical GABAergic cell connectivity .” 2016. Web. 23 Feb 2020.

Vancouver:

Choudhury M. The role of mechanistic target of rapamycin (mTOR) pathway and synaptic protein GABAA-R in cortical GABAergic cell connectivity . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/1866/13978.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choudhury M. The role of mechanistic target of rapamycin (mTOR) pathway and synaptic protein GABAA-R in cortical GABAergic cell connectivity . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13978

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. García Martínez, Juan Antonio. Seguridad y eficacia de los inhibidores de mTOR en pacientes trasplantados hepáticos.

Degree: 2016, Universidad de Sevilla

 M-TOR es una quinasa de serina y treonina implicada en los procesos de crecimiento y proliferación celular. Los fármacos inhibidores de esta proteína se comportan… (more)

Subjects/Keywords: cirugía; transplante; hígado; inhibidores; mTOR

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APA (6th Edition):

García Martínez, J. A. (2016). Seguridad y eficacia de los inhibidores de mTOR en pacientes trasplantados hepáticos. (Thesis). Universidad de Sevilla. Retrieved from http://hdl.handle.net/11441/39927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

García Martínez, Juan Antonio. “Seguridad y eficacia de los inhibidores de mTOR en pacientes trasplantados hepáticos.” 2016. Thesis, Universidad de Sevilla. Accessed February 23, 2020. http://hdl.handle.net/11441/39927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

García Martínez, Juan Antonio. “Seguridad y eficacia de los inhibidores de mTOR en pacientes trasplantados hepáticos.” 2016. Web. 23 Feb 2020.

Vancouver:

García Martínez JA. Seguridad y eficacia de los inhibidores de mTOR en pacientes trasplantados hepáticos. [Internet] [Thesis]. Universidad de Sevilla; 2016. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/11441/39927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

García Martínez JA. Seguridad y eficacia de los inhibidores de mTOR en pacientes trasplantados hepáticos. [Thesis]. Universidad de Sevilla; 2016. Available from: http://hdl.handle.net/11441/39927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

8. Hsu, Yu-Tien. Mechanisms underlying BDNF stimulation of MTOR-dependent protein synthesis.

Degree: PhD, Neuroscience, 2011, University of Southern California

 Scientists have long been searching for ways to enhance learning and memory and to repair brain injury. Dendritic protein synthesis is now recognized as being… (more)

Subjects/Keywords: BDNF; mTOR; calpain; protein synthesis

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APA (6th Edition):

Hsu, Y. (2011). Mechanisms underlying BDNF stimulation of MTOR-dependent protein synthesis. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626263/rec/4018

Chicago Manual of Style (16th Edition):

Hsu, Yu-Tien. “Mechanisms underlying BDNF stimulation of MTOR-dependent protein synthesis.” 2011. Doctoral Dissertation, University of Southern California. Accessed February 23, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626263/rec/4018.

MLA Handbook (7th Edition):

Hsu, Yu-Tien. “Mechanisms underlying BDNF stimulation of MTOR-dependent protein synthesis.” 2011. Web. 23 Feb 2020.

Vancouver:

Hsu Y. Mechanisms underlying BDNF stimulation of MTOR-dependent protein synthesis. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2020 Feb 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626263/rec/4018.

Council of Science Editors:

Hsu Y. Mechanisms underlying BDNF stimulation of MTOR-dependent protein synthesis. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/626263/rec/4018


University of Western Ontario

9. Damerill, Ian. Regulation of IGFBP-1 Phosphorylation in Hypoxia Via mTOR Signaling.

Degree: 2014, University of Western Ontario

 In this study, we provide novel evidence for a role of fetal liver mTOR signaling in regulating IGF-I bioavailability by modulating IGFBP-1 phosphorylation due to… (more)

Subjects/Keywords: mTOR; Hypoxia; IGFBP-1; Biochemistry

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APA (6th Edition):

Damerill, I. (2014). Regulation of IGFBP-1 Phosphorylation in Hypoxia Via mTOR Signaling. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Damerill, Ian. “Regulation of IGFBP-1 Phosphorylation in Hypoxia Via mTOR Signaling.” 2014. Thesis, University of Western Ontario. Accessed February 23, 2020. https://ir.lib.uwo.ca/etd/2473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Damerill, Ian. “Regulation of IGFBP-1 Phosphorylation in Hypoxia Via mTOR Signaling.” 2014. Web. 23 Feb 2020.

Vancouver:

Damerill I. Regulation of IGFBP-1 Phosphorylation in Hypoxia Via mTOR Signaling. [Internet] [Thesis]. University of Western Ontario; 2014. [cited 2020 Feb 23]. Available from: https://ir.lib.uwo.ca/etd/2473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Damerill I. Regulation of IGFBP-1 Phosphorylation in Hypoxia Via mTOR Signaling. [Thesis]. University of Western Ontario; 2014. Available from: https://ir.lib.uwo.ca/etd/2473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

10. Basalem, Osama Salem. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.

Degree: 2016, University of Manchester

Introduction: Congenital Hyperinsulinism (CHI) is a rare neonatal syndrome associated with continuous inappropriate insulin secretion by the pancreatic β-cell in the presence of recurring hypoglycemia.… (more)

Subjects/Keywords: Insulin secretion; Rapamycin; mTOR

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APA (6th Edition):

Basalem, O. S. (2016). Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154

Chicago Manual of Style (16th Edition):

Basalem, Osama Salem. “Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.” 2016. Doctoral Dissertation, University of Manchester. Accessed February 23, 2020. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154.

MLA Handbook (7th Edition):

Basalem, Osama Salem. “Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line.” 2016. Web. 23 Feb 2020.

Vancouver:

Basalem OS. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2020 Feb 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154.

Council of Science Editors:

Basalem OS. Congenital Hyperinsulinism; Effects of Rapamycin on Min6 Pancreatic β-Cell Line. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305154


Virginia Tech

11. Won, Samantha Gwai Lan. Acute and chronic heat stress alters the metabolic profile of skeletal muscle in growing swine.

Degree: MS, Animal and Poultry Sciences, 2012, Virginia Tech

 Heat stress (HS) causes significant losses to the U.S. swine industry in several production and health areas including efficient lean tissue accretion. Perturbations in skeletal… (more)

Subjects/Keywords: heat stress; mTOR; metabolism; swine

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APA (6th Edition):

Won, S. G. L. (2012). Acute and chronic heat stress alters the metabolic profile of skeletal muscle in growing swine. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/34515

Chicago Manual of Style (16th Edition):

Won, Samantha Gwai Lan. “Acute and chronic heat stress alters the metabolic profile of skeletal muscle in growing swine.” 2012. Masters Thesis, Virginia Tech. Accessed February 23, 2020. http://hdl.handle.net/10919/34515.

MLA Handbook (7th Edition):

Won, Samantha Gwai Lan. “Acute and chronic heat stress alters the metabolic profile of skeletal muscle in growing swine.” 2012. Web. 23 Feb 2020.

Vancouver:

Won SGL. Acute and chronic heat stress alters the metabolic profile of skeletal muscle in growing swine. [Internet] [Masters thesis]. Virginia Tech; 2012. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/10919/34515.

Council of Science Editors:

Won SGL. Acute and chronic heat stress alters the metabolic profile of skeletal muscle in growing swine. [Masters Thesis]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/34515


Boston University

12. Byles, Vanessa A. The role of mammalian target of rapamycin (mTOR) in macrophage polarization.

Degree: MA, Medicine, 2013, Boston University

 Macrophages are key orchestrators of the innate immune response with a dynamic role in the promotion and resolution of inflammation. Macrophage polarization to a pro-inflammatory… (more)

Subjects/Keywords: Macrophage; Polarization; Rapamycin (mTOR); Inflammation

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APA (6th Edition):

Byles, V. A. (2013). The role of mammalian target of rapamycin (mTOR) in macrophage polarization. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/17122

Chicago Manual of Style (16th Edition):

Byles, Vanessa A. “The role of mammalian target of rapamycin (mTOR) in macrophage polarization.” 2013. Masters Thesis, Boston University. Accessed February 23, 2020. http://hdl.handle.net/2144/17122.

MLA Handbook (7th Edition):

Byles, Vanessa A. “The role of mammalian target of rapamycin (mTOR) in macrophage polarization.” 2013. Web. 23 Feb 2020.

Vancouver:

Byles VA. The role of mammalian target of rapamycin (mTOR) in macrophage polarization. [Internet] [Masters thesis]. Boston University; 2013. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/2144/17122.

Council of Science Editors:

Byles VA. The role of mammalian target of rapamycin (mTOR) in macrophage polarization. [Masters Thesis]. Boston University; 2013. Available from: http://hdl.handle.net/2144/17122


Boston University

13. Zgoda, Molly Flynn. Regulation of bone by the MTORC1 pathway in osteoclasts and osteocytes.

Degree: MS, Medical Sciences, 2019, Boston University

 Bone is a highly dynamic organ system comprised of various cell types that are constantly working to maintain the health and stability of bone. The… (more)

Subjects/Keywords: Medicine; Bone; mTOR; Tuberous sclerosis

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APA (6th Edition):

Zgoda, M. F. (2019). Regulation of bone by the MTORC1 pathway in osteoclasts and osteocytes. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/36741

Chicago Manual of Style (16th Edition):

Zgoda, Molly Flynn. “Regulation of bone by the MTORC1 pathway in osteoclasts and osteocytes.” 2019. Masters Thesis, Boston University. Accessed February 23, 2020. http://hdl.handle.net/2144/36741.

MLA Handbook (7th Edition):

Zgoda, Molly Flynn. “Regulation of bone by the MTORC1 pathway in osteoclasts and osteocytes.” 2019. Web. 23 Feb 2020.

Vancouver:

Zgoda MF. Regulation of bone by the MTORC1 pathway in osteoclasts and osteocytes. [Internet] [Masters thesis]. Boston University; 2019. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/2144/36741.

Council of Science Editors:

Zgoda MF. Regulation of bone by the MTORC1 pathway in osteoclasts and osteocytes. [Masters Thesis]. Boston University; 2019. Available from: http://hdl.handle.net/2144/36741


Cornell University

14. Sikalidis, Angelos. Cellular And Animal Metabolic Responses To Indispensable Amino Acid Limitation .

Degree: 2011, Cornell University

 The current thesis was guided by three principal research objectives. The first objective was to identify target genes responsive to indispensable amino acid deprivation with… (more)

Subjects/Keywords: Integrated Stress Response; mTOR; gcn2

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APA (6th Edition):

Sikalidis, A. (2011). Cellular And Animal Metabolic Responses To Indispensable Amino Acid Limitation . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/33491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sikalidis, Angelos. “Cellular And Animal Metabolic Responses To Indispensable Amino Acid Limitation .” 2011. Thesis, Cornell University. Accessed February 23, 2020. http://hdl.handle.net/1813/33491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sikalidis, Angelos. “Cellular And Animal Metabolic Responses To Indispensable Amino Acid Limitation .” 2011. Web. 23 Feb 2020.

Vancouver:

Sikalidis A. Cellular And Animal Metabolic Responses To Indispensable Amino Acid Limitation . [Internet] [Thesis]. Cornell University; 2011. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/1813/33491.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sikalidis A. Cellular And Animal Metabolic Responses To Indispensable Amino Acid Limitation . [Thesis]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/33491

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

15. Đikić, Dragoslava A., 1975-. Утицај реактивних врста кисеоника на активност mTOR сигналног пута код пацијената са мијелопролиферативним неоплазмама.

Degree: Medicinski fakultet, 2018, Univerzitet u Beogradu

медицина - хематологија / medicine - hematology

Заједничка карактеристика малигнитета је повећана концентрација реактивних врста кисеоника (ROS) и реактивних врста азота (RNS) које могу да… (more)

Subjects/Keywords: MPN; ROS; RNS; mTOR

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APA (6th Edition):

Đikić, Dragoslava A., 1. (2018). Утицај реактивних врста кисеоника на активност mTOR сигналног пута код пацијената са мијелопролиферативним неоплазмама. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:18734/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Đikić, Dragoslava A., 1975-. “Утицај реактивних врста кисеоника на активност mTOR сигналног пута код пацијената са мијелопролиферативним неоплазмама.” 2018. Thesis, Univerzitet u Beogradu. Accessed February 23, 2020. https://fedorabg.bg.ac.rs/fedora/get/o:18734/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Đikić, Dragoslava A., 1975-. “Утицај реактивних врста кисеоника на активност mTOR сигналног пута код пацијената са мијелопролиферативним неоплазмама.” 2018. Web. 23 Feb 2020.

Vancouver:

Đikić, Dragoslava A. 1. Утицај реактивних врста кисеоника на активност mTOR сигналног пута код пацијената са мијелопролиферативним неоплазмама. [Internet] [Thesis]. Univerzitet u Beogradu; 2018. [cited 2020 Feb 23]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18734/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Đikić, Dragoslava A. 1. Утицај реактивних врста кисеоника на активност mTOR сигналног пута код пацијената са мијелопролиферативним неоплазмама. [Thesis]. Univerzitet u Beogradu; 2018. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:18734/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Wayne State University

16. Beydoun, Safa. The Impact Of Folate Restriction On Cancer And Aging: A Mechanistic Analysis.

Degree: PhD, Nutrition and Food Science, 2016, Wayne State University

  Aging is a multifactorial process associated with alterations in several physiological functions. It increases susceptibility to disease and ultimately results in mortality. Since the… (more)

Subjects/Keywords: Aging; cancer; mTOR; Nutrition

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APA (6th Edition):

Beydoun, S. (2016). The Impact Of Folate Restriction On Cancer And Aging: A Mechanistic Analysis. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1428

Chicago Manual of Style (16th Edition):

Beydoun, Safa. “The Impact Of Folate Restriction On Cancer And Aging: A Mechanistic Analysis.” 2016. Doctoral Dissertation, Wayne State University. Accessed February 23, 2020. https://digitalcommons.wayne.edu/oa_dissertations/1428.

MLA Handbook (7th Edition):

Beydoun, Safa. “The Impact Of Folate Restriction On Cancer And Aging: A Mechanistic Analysis.” 2016. Web. 23 Feb 2020.

Vancouver:

Beydoun S. The Impact Of Folate Restriction On Cancer And Aging: A Mechanistic Analysis. [Internet] [Doctoral dissertation]. Wayne State University; 2016. [cited 2020 Feb 23]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1428.

Council of Science Editors:

Beydoun S. The Impact Of Folate Restriction On Cancer And Aging: A Mechanistic Analysis. [Doctoral Dissertation]. Wayne State University; 2016. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1428


University of Sydney

17. Basnett, Jordan. Characterisation of Resistance to Everolimus in Acute Lymphoblastic Leukemia .

Degree: 2016, University of Sydney

 Acute Lymphoblastic Leukemia (ALL) is the most common childhood cancer. Disease relapse following treatment still occurs in a significant minority of children and the majority… (more)

Subjects/Keywords: Everolimus; MTOR; ALL Leukemia; Resistance

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APA (6th Edition):

Basnett, J. (2016). Characterisation of Resistance to Everolimus in Acute Lymphoblastic Leukemia . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Basnett, Jordan. “Characterisation of Resistance to Everolimus in Acute Lymphoblastic Leukemia .” 2016. Thesis, University of Sydney. Accessed February 23, 2020. http://hdl.handle.net/2123/16332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Basnett, Jordan. “Characterisation of Resistance to Everolimus in Acute Lymphoblastic Leukemia .” 2016. Web. 23 Feb 2020.

Vancouver:

Basnett J. Characterisation of Resistance to Everolimus in Acute Lymphoblastic Leukemia . [Internet] [Thesis]. University of Sydney; 2016. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/2123/16332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Basnett J. Characterisation of Resistance to Everolimus in Acute Lymphoblastic Leukemia . [Thesis]. University of Sydney; 2016. Available from: http://hdl.handle.net/2123/16332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Liang, Ning. Regulation of YAP by mTOR and autophagy reveals a therapeutic target of Tuberous Sclerosis Complex : Régulation de YAP par mTOR et l'autophagie se révèle une cible thérapeutique de la sclérose tubéreuse complexe.

Degree: Docteur es, Biologie cellulaire, 2014, Université Paris Descartes – Paris V

La sclérose tubéreuse complexe (TSC) est une maladie génétique caractérisée par une croissance des hamartomes dans différents organes y compris le cerveau, les reins, les… (more)

Subjects/Keywords: TSC; PEComa; YAP; MTOR; Autophagie; TSC; PEComa; YAP; MTOR; Autophagy; 571.6

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APA (6th Edition):

Liang, N. (2014). Regulation of YAP by mTOR and autophagy reveals a therapeutic target of Tuberous Sclerosis Complex : Régulation de YAP par mTOR et l'autophagie se révèle une cible thérapeutique de la sclérose tubéreuse complexe. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T055

Chicago Manual of Style (16th Edition):

Liang, Ning. “Regulation of YAP by mTOR and autophagy reveals a therapeutic target of Tuberous Sclerosis Complex : Régulation de YAP par mTOR et l'autophagie se révèle une cible thérapeutique de la sclérose tubéreuse complexe.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed February 23, 2020. http://www.theses.fr/2014PA05T055.

MLA Handbook (7th Edition):

Liang, Ning. “Regulation of YAP by mTOR and autophagy reveals a therapeutic target of Tuberous Sclerosis Complex : Régulation de YAP par mTOR et l'autophagie se révèle une cible thérapeutique de la sclérose tubéreuse complexe.” 2014. Web. 23 Feb 2020.

Vancouver:

Liang N. Regulation of YAP by mTOR and autophagy reveals a therapeutic target of Tuberous Sclerosis Complex : Régulation de YAP par mTOR et l'autophagie se révèle une cible thérapeutique de la sclérose tubéreuse complexe. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2020 Feb 23]. Available from: http://www.theses.fr/2014PA05T055.

Council of Science Editors:

Liang N. Regulation of YAP by mTOR and autophagy reveals a therapeutic target of Tuberous Sclerosis Complex : Régulation de YAP par mTOR et l'autophagie se révèle une cible thérapeutique de la sclérose tubéreuse complexe. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T055

19. Machado, Camila de Oliveira Freitas. Estudo do envolvimento da proteína colibistina no controle do início da tradução.

Degree: Mestrado, Biologia (Genética), 2014, University of São Paulo

A proteína colibistina (CB), uma Rho GEF neuro-especifica, apresenta papel importante na formação e funcionamento das sinapses inibitórias do sistema nervoso central por interagir com… (more)

Subjects/Keywords: Colibistina; Collybistin; mTOR signalling; Protein synthesis; Sinalização mTOR; Síntese proteica

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APA (6th Edition):

Machado, C. d. O. F. (2014). Estudo do envolvimento da proteína colibistina no controle do início da tradução. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41131/tde-15122014-142323/ ;

Chicago Manual of Style (16th Edition):

Machado, Camila de Oliveira Freitas. “Estudo do envolvimento da proteína colibistina no controle do início da tradução.” 2014. Masters Thesis, University of São Paulo. Accessed February 23, 2020. http://www.teses.usp.br/teses/disponiveis/41/41131/tde-15122014-142323/ ;.

MLA Handbook (7th Edition):

Machado, Camila de Oliveira Freitas. “Estudo do envolvimento da proteína colibistina no controle do início da tradução.” 2014. Web. 23 Feb 2020.

Vancouver:

Machado CdOF. Estudo do envolvimento da proteína colibistina no controle do início da tradução. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2020 Feb 23]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-15122014-142323/ ;.

Council of Science Editors:

Machado CdOF. Estudo do envolvimento da proteína colibistina no controle do início da tradução. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-15122014-142323/ ;

20. Silva, Hamilton Augusto Roschel da. Efeito agudo de diferentes velocidades de exercício excêntrico na sinalização da hipertrofia muscular.

Degree: PhD, Biodinâmica do Movimento Humano, 2009, University of São Paulo

Atualmente, alguns pesquisadores tem se dedicado ao estudo do efeito da manipulação do treinamento de força sobre a ativação das vias de sinalização intracelular para… (more)

Subjects/Keywords: Isocinético; Isokinetic; mTOR; mTOR; Strength training; Treinamento de força

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APA (6th Edition):

Silva, H. A. R. d. (2009). Efeito agudo de diferentes velocidades de exercício excêntrico na sinalização da hipertrofia muscular. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-22062012-103944/ ;

Chicago Manual of Style (16th Edition):

Silva, Hamilton Augusto Roschel da. “Efeito agudo de diferentes velocidades de exercício excêntrico na sinalização da hipertrofia muscular.” 2009. Doctoral Dissertation, University of São Paulo. Accessed February 23, 2020. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-22062012-103944/ ;.

MLA Handbook (7th Edition):

Silva, Hamilton Augusto Roschel da. “Efeito agudo de diferentes velocidades de exercício excêntrico na sinalização da hipertrofia muscular.” 2009. Web. 23 Feb 2020.

Vancouver:

Silva HARd. Efeito agudo de diferentes velocidades de exercício excêntrico na sinalização da hipertrofia muscular. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Feb 23]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-22062012-103944/ ;.

Council of Science Editors:

Silva HARd. Efeito agudo de diferentes velocidades de exercício excêntrico na sinalização da hipertrofia muscular. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-22062012-103944/ ;

21. Boudra, Rafik. Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology.

Degree: Docteur es, Physiologie et Génétique Moléculaires, 2015, Université Blaise-Pascale, Clermont-Ferrand II

La Nucléophosmine (NPM1/B23) est une petite chaperonne moléculaire impliquée dans de nombreux processus cellulaires, tels que la régulation de l’expression génique ou le contrôle du… (more)

Subjects/Keywords: Prostate; Cancer; Nucléophosmine; NPM1; PTEN; MTOR; Nucleophosmin; NPM1; PTEN; MTOR

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APA (6th Edition):

Boudra, R. (2015). Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology. (Doctoral Dissertation). Université Blaise-Pascale, Clermont-Ferrand II. Retrieved from http://www.theses.fr/2015CLF22598

Chicago Manual of Style (16th Edition):

Boudra, Rafik. “Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology.” 2015. Doctoral Dissertation, Université Blaise-Pascale, Clermont-Ferrand II. Accessed February 23, 2020. http://www.theses.fr/2015CLF22598.

MLA Handbook (7th Edition):

Boudra, Rafik. “Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology.” 2015. Web. 23 Feb 2020.

Vancouver:

Boudra R. Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology. [Internet] [Doctoral dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2015. [cited 2020 Feb 23]. Available from: http://www.theses.fr/2015CLF22598.

Council of Science Editors:

Boudra R. Rôle de la Nucléophosmine (NPM1) dans la physiopathologie prostatique : Role of Nucleophosmin (NPM1) in prostate physiopathology. [Doctoral Dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2015. Available from: http://www.theses.fr/2015CLF22598


University of Vienna

22. Sinkovics, Benjamin. Investigating mSin1 as the potential targeting subunit of mTORC2.

Degree: 2017, University of Vienna

Obwohl vorangegangene Studien den mechanistic target of ramapamycin complex 2 (mTORC2) mit Zellulären Wachstums-signaltransduktionswegen und Überleben in Verbindung gebracht haben steht die Untersuchung seiner Regulation… (more)

Subjects/Keywords: 42.13 Molekularbiologie; Sin1 / MAPKAP1 / mTOR / mTORC2; Sin1 / MAPKAP1 / mTOR / mTORC2

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APA (6th Edition):

Sinkovics, B. (2017). Investigating mSin1 as the potential targeting subunit of mTORC2. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/45955/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sinkovics, Benjamin. “Investigating mSin1 as the potential targeting subunit of mTORC2.” 2017. Thesis, University of Vienna. Accessed February 23, 2020. http://othes.univie.ac.at/45955/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sinkovics, Benjamin. “Investigating mSin1 as the potential targeting subunit of mTORC2.” 2017. Web. 23 Feb 2020.

Vancouver:

Sinkovics B. Investigating mSin1 as the potential targeting subunit of mTORC2. [Internet] [Thesis]. University of Vienna; 2017. [cited 2020 Feb 23]. Available from: http://othes.univie.ac.at/45955/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sinkovics B. Investigating mSin1 as the potential targeting subunit of mTORC2. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/45955/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Houssaini, Amal. La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension.

Degree: Docteur es, Physiopathologie, 2012, Université Paris-Est

 Les travaux de la thèse portent sur l'implication de la voie de signalisation Akt (sérine/thréonine kinase Akt) et mTOR (mammalian target of rapamycin) dans la… (more)

Subjects/Keywords: Akt; Mtor; Htap; Proliferation; Akt; Mtor; Pah; Proliferation

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APA (6th Edition):

Houssaini, A. (2012). La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0069

Chicago Manual of Style (16th Edition):

Houssaini, Amal. “La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed February 23, 2020. http://www.theses.fr/2012PEST0069.

MLA Handbook (7th Edition):

Houssaini, Amal. “La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension.” 2012. Web. 23 Feb 2020.

Vancouver:

Houssaini A. La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2020 Feb 23]. Available from: http://www.theses.fr/2012PEST0069.

Council of Science Editors:

Houssaini A. La voie de signalisation Akt/mTOR : rôle physiopathologique etcible thérapeutique dans l’hypertension artérielle pulmonaire expérimentale : Akt/mTOR pathways : Therapeutic target in experimental pulmonary arterial hypertension. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0069


University of Cincinnati

24. Gulati, Ruhi. Developing Viral Strategies to Study mTOR and its Regulators as Mediators of Epileptogenesis.

Degree: MS, Medicine: Biomedical Research Technology, 2019, University of Cincinnati

 Epilepsy is a crippling neurological condition that is characterized by episodes of unprovoked seizures. Current antiepileptic drugs are effective at controlling seizures, but do not… (more)

Subjects/Keywords: Neurology; Epilepsy; mTOR hyperactivation; mTOR; stereotaxic surgery; PTEN

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APA (6th Edition):

Gulati, R. (2019). Developing Viral Strategies to Study mTOR and its Regulators as Mediators of Epileptogenesis. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563273811946353

Chicago Manual of Style (16th Edition):

Gulati, Ruhi. “Developing Viral Strategies to Study mTOR and its Regulators as Mediators of Epileptogenesis.” 2019. Masters Thesis, University of Cincinnati. Accessed February 23, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563273811946353.

MLA Handbook (7th Edition):

Gulati, Ruhi. “Developing Viral Strategies to Study mTOR and its Regulators as Mediators of Epileptogenesis.” 2019. Web. 23 Feb 2020.

Vancouver:

Gulati R. Developing Viral Strategies to Study mTOR and its Regulators as Mediators of Epileptogenesis. [Internet] [Masters thesis]. University of Cincinnati; 2019. [cited 2020 Feb 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563273811946353.

Council of Science Editors:

Gulati R. Developing Viral Strategies to Study mTOR and its Regulators as Mediators of Epileptogenesis. [Masters Thesis]. University of Cincinnati; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1563273811946353

25. Vicier, Cecile. Implication de la pseudokinase dans la réponse aux inhibiteurs de mTor : Implication of pseudokinase in the response of mTor inhibitors.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université Paris-Saclay (ComUE)

MTor est une protéine centrale de la voie de signalisation PI3K/AKT/mTor et est impliquée dans la croissance, la prolifération et la survie cellulaire. Cette protéine… (more)

Subjects/Keywords: MTor; Rapamycine; Cancer du sein; MTor; Rapamycin; Breast cancer

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APA (6th Edition):

Vicier, C. (2017). Implication de la pseudokinase dans la réponse aux inhibiteurs de mTor : Implication of pseudokinase in the response of mTor inhibitors. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2017SACLS320

Chicago Manual of Style (16th Edition):

Vicier, Cecile. “Implication de la pseudokinase dans la réponse aux inhibiteurs de mTor : Implication of pseudokinase in the response of mTor inhibitors.” 2017. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed February 23, 2020. http://www.theses.fr/2017SACLS320.

MLA Handbook (7th Edition):

Vicier, Cecile. “Implication de la pseudokinase dans la réponse aux inhibiteurs de mTor : Implication of pseudokinase in the response of mTor inhibitors.” 2017. Web. 23 Feb 2020.

Vancouver:

Vicier C. Implication de la pseudokinase dans la réponse aux inhibiteurs de mTor : Implication of pseudokinase in the response of mTor inhibitors. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2017. [cited 2020 Feb 23]. Available from: http://www.theses.fr/2017SACLS320.

Council of Science Editors:

Vicier C. Implication de la pseudokinase dans la réponse aux inhibiteurs de mTor : Implication of pseudokinase in the response of mTor inhibitors. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2017. Available from: http://www.theses.fr/2017SACLS320


University of New Mexico

26. Lanphere, Kathryn. The early time course of autophagy in human peripheral blood mononuclear cells following endurance exercise.

Degree: Health, Exercise, and Sports Sciences, 2014, University of New Mexico

 Exercise disrupts homeostasis and leads to the induction of an important catabolic system called autophagy. Autophagy is a beneficial cell survival process that is induced… (more)

Subjects/Keywords: autophagy; mTOR; endurance exercise; heat shock protein

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APA (6th Edition):

Lanphere, K. (2014). The early time course of autophagy in human peripheral blood mononuclear cells following endurance exercise. (Doctoral Dissertation). University of New Mexico. Retrieved from http://hdl.handle.net/1928/23558

Chicago Manual of Style (16th Edition):

Lanphere, Kathryn. “The early time course of autophagy in human peripheral blood mononuclear cells following endurance exercise.” 2014. Doctoral Dissertation, University of New Mexico. Accessed February 23, 2020. http://hdl.handle.net/1928/23558.

MLA Handbook (7th Edition):

Lanphere, Kathryn. “The early time course of autophagy in human peripheral blood mononuclear cells following endurance exercise.” 2014. Web. 23 Feb 2020.

Vancouver:

Lanphere K. The early time course of autophagy in human peripheral blood mononuclear cells following endurance exercise. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/1928/23558.

Council of Science Editors:

Lanphere K. The early time course of autophagy in human peripheral blood mononuclear cells following endurance exercise. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: http://hdl.handle.net/1928/23558


University of Minnesota

27. Fretham, Stephanie. The Impact of Iron Deficiency During Development on Mammalian Target of Rapamycin Signaling, Neuronal Structure, and Learning and Memory Behavior.

Degree: PhD, Neuroscience, 2010, University of Minnesota

 Iron deficiency (ID) is the most common micronutrient deficiency, affecting an estimated 2 billion people world wide including 20-30% of pregnant women and their offspring.… (more)

Subjects/Keywords: development; genetic models; hippocampus; iron; mTOR

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APA (6th Edition):

Fretham, S. (2010). The Impact of Iron Deficiency During Development on Mammalian Target of Rapamycin Signaling, Neuronal Structure, and Learning and Memory Behavior. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/191359

Chicago Manual of Style (16th Edition):

Fretham, Stephanie. “The Impact of Iron Deficiency During Development on Mammalian Target of Rapamycin Signaling, Neuronal Structure, and Learning and Memory Behavior.” 2010. Doctoral Dissertation, University of Minnesota. Accessed February 23, 2020. http://hdl.handle.net/11299/191359.

MLA Handbook (7th Edition):

Fretham, Stephanie. “The Impact of Iron Deficiency During Development on Mammalian Target of Rapamycin Signaling, Neuronal Structure, and Learning and Memory Behavior.” 2010. Web. 23 Feb 2020.

Vancouver:

Fretham S. The Impact of Iron Deficiency During Development on Mammalian Target of Rapamycin Signaling, Neuronal Structure, and Learning and Memory Behavior. [Internet] [Doctoral dissertation]. University of Minnesota; 2010. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/11299/191359.

Council of Science Editors:

Fretham S. The Impact of Iron Deficiency During Development on Mammalian Target of Rapamycin Signaling, Neuronal Structure, and Learning and Memory Behavior. [Doctoral Dissertation]. University of Minnesota; 2010. Available from: http://hdl.handle.net/11299/191359


University of Illinois – Urbana-Champaign

28. Arauz Diaz, Edwin Xavier. Biochemical and single-molecule analysis of signaling molecules.

Degree: PhD, Cell and Developmental Biology, 2015, University of Illinois – Urbana-Champaign

 Mammalian cells use a variety of molecules to receive, process, and transmit information from the extracellular environment. Proteins receive and transmit signals through direct protein-protein… (more)

Subjects/Keywords: Single-molecule; mTOR; stoichiometry; phosphatidic acid; interaction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arauz Diaz, E. X. (2015). Biochemical and single-molecule analysis of signaling molecules. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/88266

Chicago Manual of Style (16th Edition):

Arauz Diaz, Edwin Xavier. “Biochemical and single-molecule analysis of signaling molecules.” 2015. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed February 23, 2020. http://hdl.handle.net/2142/88266.

MLA Handbook (7th Edition):

Arauz Diaz, Edwin Xavier. “Biochemical and single-molecule analysis of signaling molecules.” 2015. Web. 23 Feb 2020.

Vancouver:

Arauz Diaz EX. Biochemical and single-molecule analysis of signaling molecules. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2015. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/2142/88266.

Council of Science Editors:

Arauz Diaz EX. Biochemical and single-molecule analysis of signaling molecules. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2015. Available from: http://hdl.handle.net/2142/88266


University of Rochester

29. Prizant, Hen. mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM).

Degree: PhD, 2016, University of Rochester

 Lymphangioleiomyomatosis (LAM) is a devastating rare lung disease in which adenomas consisting of abnormal smooth-muscle cells grow within the lungs and progressively lead to loss… (more)

Subjects/Keywords: Estrogen; GPNMB; Lympangioleiomyomatosis; metalloproteinase; mTOR; Uterus

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APA (6th Edition):

Prizant, H. (2016). mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM). (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30560

Chicago Manual of Style (16th Edition):

Prizant, Hen. “mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM).” 2016. Doctoral Dissertation, University of Rochester. Accessed February 23, 2020. http://hdl.handle.net/1802/30560.

MLA Handbook (7th Edition):

Prizant, Hen. “mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM).” 2016. Web. 23 Feb 2020.

Vancouver:

Prizant H. mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM). [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2020 Feb 23]. Available from: http://hdl.handle.net/1802/30560.

Council of Science Editors:

Prizant H. mTOR-mediated Leiomyoma Growth in Uterine-specific Tsc2-null Mice is Exclusively Dependent on Estradiol Signaling - Implications in the Treatment of Lymphangioleiomyomatosis (LAM). [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/30560


The Ohio State University

30. Alexander, Jessica K. STRESS HORMONE INFLUENCES ON NEURAL AND IMMUNE MECHANISMS OF NEUROPATHIC PAIN.

Degree: PhD, Neuroscience Graduate Studies Program, 2010, The Ohio State University

  Chronic pain is a leading world health problem. It retards personal and societal productivity, and engenders despair. One person in five suffers the multidimensional… (more)

Subjects/Keywords: Neurology; Pain; Stress; Glucocorticoids; MIF; Microglia; mTOR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alexander, J. K. (2010). STRESS HORMONE INFLUENCES ON NEURAL AND IMMUNE MECHANISMS OF NEUROPATHIC PAIN. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1275580267

Chicago Manual of Style (16th Edition):

Alexander, Jessica K. “STRESS HORMONE INFLUENCES ON NEURAL AND IMMUNE MECHANISMS OF NEUROPATHIC PAIN.” 2010. Doctoral Dissertation, The Ohio State University. Accessed February 23, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1275580267.

MLA Handbook (7th Edition):

Alexander, Jessica K. “STRESS HORMONE INFLUENCES ON NEURAL AND IMMUNE MECHANISMS OF NEUROPATHIC PAIN.” 2010. Web. 23 Feb 2020.

Vancouver:

Alexander JK. STRESS HORMONE INFLUENCES ON NEURAL AND IMMUNE MECHANISMS OF NEUROPATHIC PAIN. [Internet] [Doctoral dissertation]. The Ohio State University; 2010. [cited 2020 Feb 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1275580267.

Council of Science Editors:

Alexander JK. STRESS HORMONE INFLUENCES ON NEURAL AND IMMUNE MECHANISMS OF NEUROPATHIC PAIN. [Doctoral Dissertation]. The Ohio State University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1275580267

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