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Dates: 2010 – 2014

You searched for subject:(lung cells). Showing records 1 – 30 of 56 total matches.

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University of Alberta

1. Ionescu, Lavinia Iuliana. Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases.

Degree: PhD, Department of Physiology, 2012, University of Alberta

 Recent discoveries in stem cell biology have generated enthusiasm about the possibility of harnessing stem cells for organ repair and regeneration. The ability of pluri-… (more)

Subjects/Keywords: asthma; mesenchymal stem cells; acute lung injury; lung; lipopolysaccharide; ovalbumin

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APA (6th Edition):

Ionescu, L. I. (2012). Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/kh04dq54w

Chicago Manual of Style (16th Edition):

Ionescu, Lavinia Iuliana. “Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases.” 2012. Doctoral Dissertation, University of Alberta. Accessed September 17, 2019. https://era.library.ualberta.ca/files/kh04dq54w.

MLA Handbook (7th Edition):

Ionescu, Lavinia Iuliana. “Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases.” 2012. Web. 17 Sep 2019.

Vancouver:

Ionescu LI. Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2019 Sep 17]. Available from: https://era.library.ualberta.ca/files/kh04dq54w.

Council of Science Editors:

Ionescu LI. Evaluation of Mesenchymal Stem Cell-Based Therapies for Inflammatory Lung Diseases. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/kh04dq54w


University of Manchester

2. Gieschen-Krische, Mary. The role of NKT cells following solid organ transplantation.

Degree: PhD, 2014, University of Manchester

 Introduction: NKT cells are categorised as borderline between NK and T cells, sharing phenotypic and functional characteristics of both cells, demonstrating their capacity to contritube… (more)

Subjects/Keywords: acute lung injury; immunosuppression; NKT cells; lung; transplantation

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APA (6th Edition):

Gieschen-Krische, M. (2014). The role of NKT cells following solid organ transplantation. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287

Chicago Manual of Style (16th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Doctoral Dissertation, University of Manchester. Accessed September 17, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287.

MLA Handbook (7th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Web. 17 Sep 2019.

Vancouver:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Sep 17]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287.

Council of Science Editors:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-nkt-cells-following-solid-organ-transplantation(321a0a4b-336e-44dd-a608-58f7ea58e27e).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.764287


University of Manchester

3. Gieschen-Krische, Mary. The role of NKT cells following solid organ transplantation.

Degree: 2014, University of Manchester

 Introduction: NKT cells are categorised as borderline between NK and T cells, sharing phenotypic and functional characteristics of both cells, demonstrating their capacity to contritube… (more)

Subjects/Keywords: NKT cells; lung; transplantation; immunosuppression; acute lung injury

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APA (6th Edition):

Gieschen-Krische, M. (2014). The role of NKT cells following solid organ transplantation. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866

Chicago Manual of Style (16th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Doctoral Dissertation, University of Manchester. Accessed September 17, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866.

MLA Handbook (7th Edition):

Gieschen-Krische, Mary. “The role of NKT cells following solid organ transplantation.” 2014. Web. 17 Sep 2019.

Vancouver:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Sep 17]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866.

Council of Science Editors:

Gieschen-Krische M. The role of NKT cells following solid organ transplantation. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:242866


University of Toronto

4. Kim, Bo Ram. Investigation of the Oncogenic Role of Sox2 in the Pathogenesis of Lung Squamous Cell Carcinoma using Normal Human Lung Basal Progenitors.

Degree: 2012, University of Toronto

Sox2 is the most frequently amplified oncogene in lung squamous cell carcinoma (SCC). Lung SCC arises in the proximal to central airways and is thought… (more)

Subjects/Keywords: lung squamous cell carcinoma; Sox2; basal cells; lung progenitor; 0379

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APA (6th Edition):

Kim, B. R. (2012). Investigation of the Oncogenic Role of Sox2 in the Pathogenesis of Lung Squamous Cell Carcinoma using Normal Human Lung Basal Progenitors. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/32250

Chicago Manual of Style (16th Edition):

Kim, Bo Ram. “Investigation of the Oncogenic Role of Sox2 in the Pathogenesis of Lung Squamous Cell Carcinoma using Normal Human Lung Basal Progenitors.” 2012. Masters Thesis, University of Toronto. Accessed September 17, 2019. http://hdl.handle.net/1807/32250.

MLA Handbook (7th Edition):

Kim, Bo Ram. “Investigation of the Oncogenic Role of Sox2 in the Pathogenesis of Lung Squamous Cell Carcinoma using Normal Human Lung Basal Progenitors.” 2012. Web. 17 Sep 2019.

Vancouver:

Kim BR. Investigation of the Oncogenic Role of Sox2 in the Pathogenesis of Lung Squamous Cell Carcinoma using Normal Human Lung Basal Progenitors. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/1807/32250.

Council of Science Editors:

Kim BR. Investigation of the Oncogenic Role of Sox2 in the Pathogenesis of Lung Squamous Cell Carcinoma using Normal Human Lung Basal Progenitors. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/32250


University of Kashmir

5. Qazi Danish Mushtaq. A study on crosstalk between lung carcinoma and immune cells;.

Degree: 2013, University of Kashmir

newlineTumor cells are seen to modulate the phenotype of all major immune cells to newlineexpress tumor favouring phenotypes. Inflammation associated with tumors, a result of… (more)

Subjects/Keywords: Cross-Talk; Immune Cells; Lung Carcinoma

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APA (6th Edition):

Mushtaq, Q. D. (2013). A study on crosstalk between lung carcinoma and immune cells;. (Thesis). University of Kashmir. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/14385

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mushtaq, Qazi Danish. “A study on crosstalk between lung carcinoma and immune cells;.” 2013. Thesis, University of Kashmir. Accessed September 17, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/14385.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mushtaq, Qazi Danish. “A study on crosstalk between lung carcinoma and immune cells;.” 2013. Web. 17 Sep 2019.

Vancouver:

Mushtaq QD. A study on crosstalk between lung carcinoma and immune cells;. [Internet] [Thesis]. University of Kashmir; 2013. [cited 2019 Sep 17]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/14385.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mushtaq QD. A study on crosstalk between lung carcinoma and immune cells;. [Thesis]. University of Kashmir; 2013. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/14385

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

6. Clifford, Monica Allison. Identification of Molecular and Functional Heterogeneity of Epithelial Progenitor Cells in the Upper Airway.

Degree: 2013, University of Toronto

Upper airways are lined with a pseudostratified mucociliary epithelium maintained by basal cells. To investigate functional and phenotypic heterogeneity within the human basal cell compartment,… (more)

Subjects/Keywords: airway epithelium; lung progenitor cells; 0379; 0307

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APA (6th Edition):

Clifford, M. A. (2013). Identification of Molecular and Functional Heterogeneity of Epithelial Progenitor Cells in the Upper Airway. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/35590

Chicago Manual of Style (16th Edition):

Clifford, Monica Allison. “Identification of Molecular and Functional Heterogeneity of Epithelial Progenitor Cells in the Upper Airway.” 2013. Masters Thesis, University of Toronto. Accessed September 17, 2019. http://hdl.handle.net/1807/35590.

MLA Handbook (7th Edition):

Clifford, Monica Allison. “Identification of Molecular and Functional Heterogeneity of Epithelial Progenitor Cells in the Upper Airway.” 2013. Web. 17 Sep 2019.

Vancouver:

Clifford MA. Identification of Molecular and Functional Heterogeneity of Epithelial Progenitor Cells in the Upper Airway. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/1807/35590.

Council of Science Editors:

Clifford MA. Identification of Molecular and Functional Heterogeneity of Epithelial Progenitor Cells in the Upper Airway. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/35590

7. Svensson, Susanne. In vitro and in vivo inhibition of Chk1 sensitize lung cancer stem cells to chemotherapy.

Degree: 2011, Università degli Studi di Catania

 Development of resistance to radiation and chemotherapy turns the treatment of solid cancers into a therapeutic challenge. One of the most exciting breakthroughs being explored… (more)

Subjects/Keywords: Non-small cell lung cancer; Lung cancer stem cells; DNA damage; Chk1 inhibitors

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APA (6th Edition):

Svensson, S. (2011). In vitro and in vivo inhibition of Chk1 sensitize lung cancer stem cells to chemotherapy. (Thesis). Università degli Studi di Catania. Retrieved from http://hdl.handle.net/10761/98

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Svensson, Susanne. “In vitro and in vivo inhibition of Chk1 sensitize lung cancer stem cells to chemotherapy.” 2011. Thesis, Università degli Studi di Catania. Accessed September 17, 2019. http://hdl.handle.net/10761/98.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Svensson, Susanne. “In vitro and in vivo inhibition of Chk1 sensitize lung cancer stem cells to chemotherapy.” 2011. Web. 17 Sep 2019.

Vancouver:

Svensson S. In vitro and in vivo inhibition of Chk1 sensitize lung cancer stem cells to chemotherapy. [Internet] [Thesis]. Università degli Studi di Catania; 2011. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/10761/98.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Svensson S. In vitro and in vivo inhibition of Chk1 sensitize lung cancer stem cells to chemotherapy. [Thesis]. Università degli Studi di Catania; 2011. Available from: http://hdl.handle.net/10761/98

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

8. Jimenez, Elizabeth. Generation of DNA Aptamers for Lung Cancer Studies.

Degree: PhD, Chemistry, 2013, University of Florida

 The overall objective of this proposal is to develop a strategy for the detection and isolation of circulating tumor cells(CTC's) from peripheral blood of lung(more)

Subjects/Keywords: Adenocarcinoma; Blood; Cancer; Cell lines; Cells; Lung neoplasms; Lungs; Molecules; Small cell lung carcinoma; Tumors; aptamers  – cancer  – cell-selex  – lung

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APA (6th Edition):

Jimenez, E. (2013). Generation of DNA Aptamers for Lung Cancer Studies. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0045913

Chicago Manual of Style (16th Edition):

Jimenez, Elizabeth. “Generation of DNA Aptamers for Lung Cancer Studies.” 2013. Doctoral Dissertation, University of Florida. Accessed September 17, 2019. http://ufdc.ufl.edu/UFE0045913.

MLA Handbook (7th Edition):

Jimenez, Elizabeth. “Generation of DNA Aptamers for Lung Cancer Studies.” 2013. Web. 17 Sep 2019.

Vancouver:

Jimenez E. Generation of DNA Aptamers for Lung Cancer Studies. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2019 Sep 17]. Available from: http://ufdc.ufl.edu/UFE0045913.

Council of Science Editors:

Jimenez E. Generation of DNA Aptamers for Lung Cancer Studies. [Doctoral Dissertation]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0045913

9. Santos, Angela Batista Gomes dos. Perfil celular do tecido pulmonar em crianças de até dois anos: um estudo em autópsias.

Degree: Mestrado, Fisiopatologia Experimental, 2011, University of São Paulo

Introdução: Doenças pulmonares ou infecções que ocorrem no início da vida podem ter permanente impacto na vida adulta. Pouco se sabe sobre o perfil de… (more)

Subjects/Keywords: Autópsia; Autopsy; Células dendríticas; Dendritic cells; Infants; Lactante; Linfócitos; Lung; Lymphocytes; Mast cells; Mastócitos; Pulmão

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APA (6th Edition):

Santos, A. B. G. d. (2011). Perfil celular do tecido pulmonar em crianças de até dois anos: um estudo em autópsias. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052011-153742/ ;

Chicago Manual of Style (16th Edition):

Santos, Angela Batista Gomes dos. “Perfil celular do tecido pulmonar em crianças de até dois anos: um estudo em autópsias.” 2011. Masters Thesis, University of São Paulo. Accessed September 17, 2019. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052011-153742/ ;.

MLA Handbook (7th Edition):

Santos, Angela Batista Gomes dos. “Perfil celular do tecido pulmonar em crianças de até dois anos: um estudo em autópsias.” 2011. Web. 17 Sep 2019.

Vancouver:

Santos ABGd. Perfil celular do tecido pulmonar em crianças de até dois anos: um estudo em autópsias. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2019 Sep 17]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052011-153742/ ;.

Council of Science Editors:

Santos ABGd. Perfil celular do tecido pulmonar em crianças de até dois anos: um estudo em autópsias. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-24052011-153742/ ;


RMIT University

10. Elbaz, A. Investigating the immune and cytotoxic responses of mast and lung epithelial cells to engineered nanoparticles.

Degree: 2012, RMIT University

 The applications for engineered nanoparticles have increased dramatically in recent years. They have been introduced into the industrial, electrical, agricultural, pharmaceutical and medical fields due… (more)

Subjects/Keywords: Fields of Research; nanoparticles; mast cells; lung epithelial cells; immune responses and cytotoxicity

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APA (6th Edition):

Elbaz, A. (2012). Investigating the immune and cytotoxic responses of mast and lung epithelial cells to engineered nanoparticles. (Thesis). RMIT University. Retrieved from http://researchbank.rmit.edu.au/view/rmit:161007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Elbaz, A. “Investigating the immune and cytotoxic responses of mast and lung epithelial cells to engineered nanoparticles.” 2012. Thesis, RMIT University. Accessed September 17, 2019. http://researchbank.rmit.edu.au/view/rmit:161007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Elbaz, A. “Investigating the immune and cytotoxic responses of mast and lung epithelial cells to engineered nanoparticles.” 2012. Web. 17 Sep 2019.

Vancouver:

Elbaz A. Investigating the immune and cytotoxic responses of mast and lung epithelial cells to engineered nanoparticles. [Internet] [Thesis]. RMIT University; 2012. [cited 2019 Sep 17]. Available from: http://researchbank.rmit.edu.au/view/rmit:161007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Elbaz A. Investigating the immune and cytotoxic responses of mast and lung epithelial cells to engineered nanoparticles. [Thesis]. RMIT University; 2012. Available from: http://researchbank.rmit.edu.au/view/rmit:161007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

11. Strueby, Lannae L. Paracrine Effect of Mesenchymal Stromal Cells on Multifactorial Lung Injury in Neonatal Mice.

Degree: MS, Department of Physiology, 2014, University of Alberta

 Bronchopulmonary dysplasia (BPD) is a common complication of extreme prematurity. Preterm infants often require mechanical ventilation and supplemental oxygen for survival. These interventions increase the… (more)

Subjects/Keywords: Paracrine; Conditioned Media; Mesenchymal Stromal Cells; Lung Injury; Bronchopulmonary Dysplasia

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APA (6th Edition):

Strueby, L. L. (2014). Paracrine Effect of Mesenchymal Stromal Cells on Multifactorial Lung Injury in Neonatal Mice. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/1g05ff203

Chicago Manual of Style (16th Edition):

Strueby, Lannae L. “Paracrine Effect of Mesenchymal Stromal Cells on Multifactorial Lung Injury in Neonatal Mice.” 2014. Masters Thesis, University of Alberta. Accessed September 17, 2019. https://era.library.ualberta.ca/files/1g05ff203.

MLA Handbook (7th Edition):

Strueby, Lannae L. “Paracrine Effect of Mesenchymal Stromal Cells on Multifactorial Lung Injury in Neonatal Mice.” 2014. Web. 17 Sep 2019.

Vancouver:

Strueby LL. Paracrine Effect of Mesenchymal Stromal Cells on Multifactorial Lung Injury in Neonatal Mice. [Internet] [Masters thesis]. University of Alberta; 2014. [cited 2019 Sep 17]. Available from: https://era.library.ualberta.ca/files/1g05ff203.

Council of Science Editors:

Strueby LL. Paracrine Effect of Mesenchymal Stromal Cells on Multifactorial Lung Injury in Neonatal Mice. [Masters Thesis]. University of Alberta; 2014. Available from: https://era.library.ualberta.ca/files/1g05ff203


University of Oulu

12. Mäkelä, T. (Tuomas). Systemic transplantation of bone marrow stromal cells:an experimental animal study of biodistribution and tissue targeting.

Degree: 2014, University of Oulu

Abstract Bone marrow mesenchymal stromal cells (MSCs) and mononuclear cells (BM-MNCs) have shown great therapeutic potential in various clinical settings. Although intravascular transplantation of the… (more)

Subjects/Keywords: biodistribution; imaging; lung entrapment; stem cells; kantasolut; keuhkoläpäisevyys; kudosjakautuminen; kuvantaminen

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APA (6th Edition):

Mäkelä, T. (. (2014). Systemic transplantation of bone marrow stromal cells:an experimental animal study of biodistribution and tissue targeting. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526206547

Chicago Manual of Style (16th Edition):

Mäkelä, T (Tuomas). “Systemic transplantation of bone marrow stromal cells:an experimental animal study of biodistribution and tissue targeting.” 2014. Doctoral Dissertation, University of Oulu. Accessed September 17, 2019. http://urn.fi/urn:isbn:9789526206547.

MLA Handbook (7th Edition):

Mäkelä, T (Tuomas). “Systemic transplantation of bone marrow stromal cells:an experimental animal study of biodistribution and tissue targeting.” 2014. Web. 17 Sep 2019.

Vancouver:

Mäkelä T(. Systemic transplantation of bone marrow stromal cells:an experimental animal study of biodistribution and tissue targeting. [Internet] [Doctoral dissertation]. University of Oulu; 2014. [cited 2019 Sep 17]. Available from: http://urn.fi/urn:isbn:9789526206547.

Council of Science Editors:

Mäkelä T(. Systemic transplantation of bone marrow stromal cells:an experimental animal study of biodistribution and tissue targeting. [Doctoral Dissertation]. University of Oulu; 2014. Available from: http://urn.fi/urn:isbn:9789526206547


University of Cincinnati

13. Akunuru, Shailaja. Regulation of cancer stem cell activity and epithelial mesenchymal transition by Rac1 in Human lung adenocarcinoma cells.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2011, University of Cincinnati

 The cancer stem cell (CSC) theory predicts that a small fraction of cancer cells possesses unique self-renewal, expansion and differentiation activities in tumorigenesis. While this… (more)

Subjects/Keywords: Cellular Biology; Cancer stem cells; EMT; Rac1; Metastasis; Lung cancer; NSCLA

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APA (6th Edition):

Akunuru, S. (2011). Regulation of cancer stem cell activity and epithelial mesenchymal transition by Rac1 in Human lung adenocarcinoma cells. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1314301864

Chicago Manual of Style (16th Edition):

Akunuru, Shailaja. “Regulation of cancer stem cell activity and epithelial mesenchymal transition by Rac1 in Human lung adenocarcinoma cells.” 2011. Doctoral Dissertation, University of Cincinnati. Accessed September 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1314301864.

MLA Handbook (7th Edition):

Akunuru, Shailaja. “Regulation of cancer stem cell activity and epithelial mesenchymal transition by Rac1 in Human lung adenocarcinoma cells.” 2011. Web. 17 Sep 2019.

Vancouver:

Akunuru S. Regulation of cancer stem cell activity and epithelial mesenchymal transition by Rac1 in Human lung adenocarcinoma cells. [Internet] [Doctoral dissertation]. University of Cincinnati; 2011. [cited 2019 Sep 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1314301864.

Council of Science Editors:

Akunuru S. Regulation of cancer stem cell activity and epithelial mesenchymal transition by Rac1 in Human lung adenocarcinoma cells. [Doctoral Dissertation]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1314301864


University of Western Australia

14. McCoy, Melanie Jane. The effect of chemotherapy on the anti-tumour immune response in patients with thoracic malignancies.

Degree: PhD, 2011, University of Western Australia

[Truncated abstract] Chemotherapy and immunotherapy have historically been considered antagonistic treatment options due to the variable lymphopaenia experienced as a side effect of most cytotoxic… (more)

Subjects/Keywords: Mesothelioma; Lung cancer; Chemotherapy; T Cells; Flow cytometry; Anti-tumour immunity

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APA (6th Edition):

McCoy, M. J. (2011). The effect of chemotherapy on the anti-tumour immune response in patients with thoracic malignancies. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30870&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

McCoy, Melanie Jane. “The effect of chemotherapy on the anti-tumour immune response in patients with thoracic malignancies.” 2011. Doctoral Dissertation, University of Western Australia. Accessed September 17, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30870&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

McCoy, Melanie Jane. “The effect of chemotherapy on the anti-tumour immune response in patients with thoracic malignancies.” 2011. Web. 17 Sep 2019.

Vancouver:

McCoy MJ. The effect of chemotherapy on the anti-tumour immune response in patients with thoracic malignancies. [Internet] [Doctoral dissertation]. University of Western Australia; 2011. [cited 2019 Sep 17]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30870&local_base=GEN01-INS01.

Council of Science Editors:

McCoy MJ. The effect of chemotherapy on the anti-tumour immune response in patients with thoracic malignancies. [Doctoral Dissertation]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=30870&local_base=GEN01-INS01


University of Sydney

15. McKnight, Lauren Anne. The role of natural regulatory T cells in the regulation of asthma .

Degree: 2014, University of Sydney

 Allergic asthma is a chronic inflammatory disease of the airways characterised by Th2 sensitisation and inflammation, which represents a failure of tolerance to environmental antigens.… (more)

Subjects/Keywords: Asthma; Flow Cytometry; Lung; Mice; Transgenic; T-Lymphocytes; Regulatory; Th2 Cells

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APA (6th Edition):

McKnight, L. A. (2014). The role of natural regulatory T cells in the regulation of asthma . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/13593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McKnight, Lauren Anne. “The role of natural regulatory T cells in the regulation of asthma .” 2014. Thesis, University of Sydney. Accessed September 17, 2019. http://hdl.handle.net/2123/13593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McKnight, Lauren Anne. “The role of natural regulatory T cells in the regulation of asthma .” 2014. Web. 17 Sep 2019.

Vancouver:

McKnight LA. The role of natural regulatory T cells in the regulation of asthma . [Internet] [Thesis]. University of Sydney; 2014. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/2123/13593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McKnight LA. The role of natural regulatory T cells in the regulation of asthma . [Thesis]. University of Sydney; 2014. Available from: http://hdl.handle.net/2123/13593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

16. Lau, Allison Nicole. Characterization of lung tumor-propagating cells reveals a role for CD24 and Yap/Taz in lung cancer progression and metastasis.

Degree: PhD, Biology: Medical Sciences, Division of, 2014, Harvard University

Lung cancer is the leading cause of cancer deaths worldwide. A large part of this high mortality rate is due to the onset of metastatic… (more)

Subjects/Keywords: Genetics; CD24; lung cancer; metastasis; Taz; tumor propagating cells; Yap

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APA (6th Edition):

Lau, A. N. (2014). Characterization of lung tumor-propagating cells reveals a role for CD24 and Yap/Taz in lung cancer progression and metastasis. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269822

Chicago Manual of Style (16th Edition):

Lau, Allison Nicole. “Characterization of lung tumor-propagating cells reveals a role for CD24 and Yap/Taz in lung cancer progression and metastasis.” 2014. Doctoral Dissertation, Harvard University. Accessed September 17, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269822.

MLA Handbook (7th Edition):

Lau, Allison Nicole. “Characterization of lung tumor-propagating cells reveals a role for CD24 and Yap/Taz in lung cancer progression and metastasis.” 2014. Web. 17 Sep 2019.

Vancouver:

Lau AN. Characterization of lung tumor-propagating cells reveals a role for CD24 and Yap/Taz in lung cancer progression and metastasis. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2019 Sep 17]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269822.

Council of Science Editors:

Lau AN. Characterization of lung tumor-propagating cells reveals a role for CD24 and Yap/Taz in lung cancer progression and metastasis. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12269822


Utah State University

17. Watterson, Todd L. Effects of Cache Valley Particulate Matter on Human Lung Cells.

Degree: PhD, Animal, Dairy, and Veterinary Sciences, 2012, Utah State University

  During wintertime temperature inversion episodes the concentrations of particulate air pollution, also defined as particulate matter (PM), in Utah’s Cache Valley have often been… (more)

Subjects/Keywords: Cache Valley; particulate matter; lung cells; inversion; pollution; Animal Sciences

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APA (6th Edition):

Watterson, T. L. (2012). Effects of Cache Valley Particulate Matter on Human Lung Cells. (Doctoral Dissertation). Utah State University. Retrieved from https://digitalcommons.usu.edu/etd/1341

Chicago Manual of Style (16th Edition):

Watterson, Todd L. “Effects of Cache Valley Particulate Matter on Human Lung Cells.” 2012. Doctoral Dissertation, Utah State University. Accessed September 17, 2019. https://digitalcommons.usu.edu/etd/1341.

MLA Handbook (7th Edition):

Watterson, Todd L. “Effects of Cache Valley Particulate Matter on Human Lung Cells.” 2012. Web. 17 Sep 2019.

Vancouver:

Watterson TL. Effects of Cache Valley Particulate Matter on Human Lung Cells. [Internet] [Doctoral dissertation]. Utah State University; 2012. [cited 2019 Sep 17]. Available from: https://digitalcommons.usu.edu/etd/1341.

Council of Science Editors:

Watterson TL. Effects of Cache Valley Particulate Matter on Human Lung Cells. [Doctoral Dissertation]. Utah State University; 2012. Available from: https://digitalcommons.usu.edu/etd/1341


University of Rochester

18. Bello-Irizarry, Sheila Ninoshka. MyD88-Dependent Signaling Pathways Contribute to both Host Defense and Immunopathogenesis during Pneumocystis Infection.

Degree: PhD, 2013, University of Rochester

 Pneumocystis Pneumonia (PcP) is a common respiratory infection of HIV patients and other immunocompromised individuals. A functional immune system is critical for host defense against… (more)

Subjects/Keywords: Pneumocystis; Inflammation; Lung Injury; Opportunistic Pathogen; Alveolar Epithelial Cells; MyD88

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APA (6th Edition):

Bello-Irizarry, S. N. (2013). MyD88-Dependent Signaling Pathways Contribute to both Host Defense and Immunopathogenesis during Pneumocystis Infection. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/27245

Chicago Manual of Style (16th Edition):

Bello-Irizarry, Sheila Ninoshka. “MyD88-Dependent Signaling Pathways Contribute to both Host Defense and Immunopathogenesis during Pneumocystis Infection.” 2013. Doctoral Dissertation, University of Rochester. Accessed September 17, 2019. http://hdl.handle.net/1802/27245.

MLA Handbook (7th Edition):

Bello-Irizarry, Sheila Ninoshka. “MyD88-Dependent Signaling Pathways Contribute to both Host Defense and Immunopathogenesis during Pneumocystis Infection.” 2013. Web. 17 Sep 2019.

Vancouver:

Bello-Irizarry SN. MyD88-Dependent Signaling Pathways Contribute to both Host Defense and Immunopathogenesis during Pneumocystis Infection. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/1802/27245.

Council of Science Editors:

Bello-Irizarry SN. MyD88-Dependent Signaling Pathways Contribute to both Host Defense and Immunopathogenesis during Pneumocystis Infection. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/27245


University of Toronto

19. Barker, Elizabeth Joy. Role of Rho small GTPases in the invasion of Lung Epithelial Cells by Legionella pneumophila.

Degree: 2014, University of Toronto

Legionella pneumophila is the main cause of Legionnaires' Disease, a fatal pneumonia. The filamentous form of Legionella (FLp) is capable of invading human lung epithelial… (more)

Subjects/Keywords: Cdc42; invasion; Legionella pneumophila; lung epithelial cells; Rac1; RhoA; 0379

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APA (6th Edition):

Barker, E. J. (2014). Role of Rho small GTPases in the invasion of Lung Epithelial Cells by Legionella pneumophila. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/82625

Chicago Manual of Style (16th Edition):

Barker, Elizabeth Joy. “Role of Rho small GTPases in the invasion of Lung Epithelial Cells by Legionella pneumophila.” 2014. Masters Thesis, University of Toronto. Accessed September 17, 2019. http://hdl.handle.net/1807/82625.

MLA Handbook (7th Edition):

Barker, Elizabeth Joy. “Role of Rho small GTPases in the invasion of Lung Epithelial Cells by Legionella pneumophila.” 2014. Web. 17 Sep 2019.

Vancouver:

Barker EJ. Role of Rho small GTPases in the invasion of Lung Epithelial Cells by Legionella pneumophila. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/1807/82625.

Council of Science Editors:

Barker EJ. Role of Rho small GTPases in the invasion of Lung Epithelial Cells by Legionella pneumophila. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/82625

20. Grabarz, Felipe. Células NKT, macrófagos M2 e o desenvolvimento da fibrose pulmonar.

Degree: Mestrado, Imunologia, 2014, University of São Paulo

A fibrose pulmonar é uma via comum de várias doenças agudas e crônicas do interstício pulmonar que pode resultar na cicatrização anormal do pulmão. Há… (more)

Subjects/Keywords: Bleomicina; Bleomycin; Células NKT; Fibrose pulmonar; Lung fibrosis; M2 macrophages; Macrófagos M2; NKT cells

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APA (6th Edition):

Grabarz, F. (2014). Células NKT, macrófagos M2 e o desenvolvimento da fibrose pulmonar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42133/tde-20022015-171616/ ;

Chicago Manual of Style (16th Edition):

Grabarz, Felipe. “Células NKT, macrófagos M2 e o desenvolvimento da fibrose pulmonar.” 2014. Masters Thesis, University of São Paulo. Accessed September 17, 2019. http://www.teses.usp.br/teses/disponiveis/42/42133/tde-20022015-171616/ ;.

MLA Handbook (7th Edition):

Grabarz, Felipe. “Células NKT, macrófagos M2 e o desenvolvimento da fibrose pulmonar.” 2014. Web. 17 Sep 2019.

Vancouver:

Grabarz F. Células NKT, macrófagos M2 e o desenvolvimento da fibrose pulmonar. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2019 Sep 17]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-20022015-171616/ ;.

Council of Science Editors:

Grabarz F. Células NKT, macrófagos M2 e o desenvolvimento da fibrose pulmonar. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-20022015-171616/ ;


Johannes Gutenberg Universität Mainz

21. Schäfer, Marina. Etablierung eines Triple-Kultur-Modells der oberen Atemwege unter Einbeziehung immunkompetenter Zellen.

Degree: 2014, Johannes Gutenberg Universität Mainz

Organophosphate können chronische Lungenerkrankungen und Vergiftungen hervorrufen. Bei der Vergiftung erfolgt eine Immunreaktion, welche noch nicht erforscht ist. In dieser Arbeit wurden Toxizitätsstudien an dendritischen… (more)

Subjects/Keywords: Triple-Kultur Modell; Dendritische Zellen; Lunge; triple-culture model; dendritic cells; lung; Life sciences

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APA (6th Edition):

Schäfer, M. (2014). Etablierung eines Triple-Kultur-Modells der oberen Atemwege unter Einbeziehung immunkompetenter Zellen. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2014/3843/

Chicago Manual of Style (16th Edition):

Schäfer, Marina. “Etablierung eines Triple-Kultur-Modells der oberen Atemwege unter Einbeziehung immunkompetenter Zellen.” 2014. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed September 17, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2014/3843/.

MLA Handbook (7th Edition):

Schäfer, Marina. “Etablierung eines Triple-Kultur-Modells der oberen Atemwege unter Einbeziehung immunkompetenter Zellen.” 2014. Web. 17 Sep 2019.

Vancouver:

Schäfer M. Etablierung eines Triple-Kultur-Modells der oberen Atemwege unter Einbeziehung immunkompetenter Zellen. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2014. [cited 2019 Sep 17]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2014/3843/.

Council of Science Editors:

Schäfer M. Etablierung eines Triple-Kultur-Modells der oberen Atemwege unter Einbeziehung immunkompetenter Zellen. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2014. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2014/3843/


University of Gothenburg / Göteborgs Universitet

22. Silverpil, Elin. Modulatory role of IL-17 in airway inflammation.

Degree: 2010, University of Gothenburg / Göteborgs Universitet

 IL-17 orchestrates the accumulation of neutrophils to sites of infection and the release of microbicidal substances, and therefore plays a critical role in the innate… (more)

Subjects/Keywords: Respiratory medicine; Lung; IL-17; IL-23; Macrophages; Neutrophils; T cells; Apoptosis; Phagocytosis; Airways

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APA (6th Edition):

Silverpil, E. (2010). Modulatory role of IL-17 in airway inflammation. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/21944

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silverpil, Elin. “Modulatory role of IL-17 in airway inflammation.” 2010. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed September 17, 2019. http://hdl.handle.net/2077/21944.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silverpil, Elin. “Modulatory role of IL-17 in airway inflammation.” 2010. Web. 17 Sep 2019.

Vancouver:

Silverpil E. Modulatory role of IL-17 in airway inflammation. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2010. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/2077/21944.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silverpil E. Modulatory role of IL-17 in airway inflammation. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2010. Available from: http://hdl.handle.net/2077/21944

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

23. DebRoy, Auditi. Stromal Interacting Molecule 1 Signaling and Lung Vascular Barrier Function.

Degree: 2014, University of Illinois – Chicago

 Stromal interacting molecule 1 (STIM1) regulates store-operated Ca2+ entry (SOCE) in endothelial cells (ECs). Here, we show that STIM1 expression in ECs is increased during… (more)

Subjects/Keywords: STIM1; Transcription; Calcium signaling; Sepsis; Inflammation; Lung endothelial cells; NF-κB signaling; P38 MAPK signaling

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APA (6th Edition):

DebRoy, A. (2014). Stromal Interacting Molecule 1 Signaling and Lung Vascular Barrier Function. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/19143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DebRoy, Auditi. “Stromal Interacting Molecule 1 Signaling and Lung Vascular Barrier Function.” 2014. Thesis, University of Illinois – Chicago. Accessed September 17, 2019. http://hdl.handle.net/10027/19143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DebRoy, Auditi. “Stromal Interacting Molecule 1 Signaling and Lung Vascular Barrier Function.” 2014. Web. 17 Sep 2019.

Vancouver:

DebRoy A. Stromal Interacting Molecule 1 Signaling and Lung Vascular Barrier Function. [Internet] [Thesis]. University of Illinois – Chicago; 2014. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/10027/19143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DebRoy A. Stromal Interacting Molecule 1 Signaling and Lung Vascular Barrier Function. [Thesis]. University of Illinois – Chicago; 2014. Available from: http://hdl.handle.net/10027/19143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

24. Sadhukha, Tanmoy. Targeted magnetic hyperthermia for lung cancer.

Degree: PhD, 2013, University of Minnesota

Lung cancer (specifically, non-small cell lung cancer; NSCLC) is the leading cause of cancer-related deaths in the United States. Poor response rates and survival with… (more)

Subjects/Keywords: Cancer stem cells; Lung cancer; Magnetic hyperthermia; Superparamagnetic iron oxide; Targeted drug delivery; Pharmaceutics

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APA (6th Edition):

Sadhukha, T. (2013). Targeted magnetic hyperthermia for lung cancer. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/168301

Chicago Manual of Style (16th Edition):

Sadhukha, Tanmoy. “Targeted magnetic hyperthermia for lung cancer.” 2013. Doctoral Dissertation, University of Minnesota. Accessed September 17, 2019. http://hdl.handle.net/11299/168301.

MLA Handbook (7th Edition):

Sadhukha, Tanmoy. “Targeted magnetic hyperthermia for lung cancer.” 2013. Web. 17 Sep 2019.

Vancouver:

Sadhukha T. Targeted magnetic hyperthermia for lung cancer. [Internet] [Doctoral dissertation]. University of Minnesota; 2013. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/11299/168301.

Council of Science Editors:

Sadhukha T. Targeted magnetic hyperthermia for lung cancer. [Doctoral Dissertation]. University of Minnesota; 2013. Available from: http://hdl.handle.net/11299/168301


University of Lund

25. Hasan, Zirak. SIGNALING MECHANISMS IN SEPSIS-INDUCED IMMUNE DYSFUNCTION.

Degree: 2013, University of Lund

 Sepsis and subsequent organ failure remain the major cause of mortality in intensive care units in spite of significant research efforts. The lung is the… (more)

Subjects/Keywords: Kirurgi; abdominal sepsis; neutrophil; lung; chemokines; infection; T-cells; CD44; Rho-kinase; isoprenylation

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APA (6th Edition):

Hasan, Z. (2013). SIGNALING MECHANISMS IN SEPSIS-INDUCED IMMUNE DYSFUNCTION. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/3460779 ; http://portal.research.lu.se/ws/files/4262151/3460784.pdf

Chicago Manual of Style (16th Edition):

Hasan, Zirak. “SIGNALING MECHANISMS IN SEPSIS-INDUCED IMMUNE DYSFUNCTION.” 2013. Doctoral Dissertation, University of Lund. Accessed September 17, 2019. http://lup.lub.lu.se/record/3460779 ; http://portal.research.lu.se/ws/files/4262151/3460784.pdf.

MLA Handbook (7th Edition):

Hasan, Zirak. “SIGNALING MECHANISMS IN SEPSIS-INDUCED IMMUNE DYSFUNCTION.” 2013. Web. 17 Sep 2019.

Vancouver:

Hasan Z. SIGNALING MECHANISMS IN SEPSIS-INDUCED IMMUNE DYSFUNCTION. [Internet] [Doctoral dissertation]. University of Lund; 2013. [cited 2019 Sep 17]. Available from: http://lup.lub.lu.se/record/3460779 ; http://portal.research.lu.se/ws/files/4262151/3460784.pdf.

Council of Science Editors:

Hasan Z. SIGNALING MECHANISMS IN SEPSIS-INDUCED IMMUNE DYSFUNCTION. [Doctoral Dissertation]. University of Lund; 2013. Available from: http://lup.lub.lu.se/record/3460779 ; http://portal.research.lu.se/ws/files/4262151/3460784.pdf


University of Lund

26. Zhang, Su. Adhesive and signaling mechanisms in abdominal sepsis.

Degree: 2012, University of Lund

 Sepsis is a major cause of mortality in intensive care units despite decades of scientific efforts. The lung is recognized as the most sensitive and… (more)

Subjects/Keywords: Klinisk medicin; abdominal sepsis; lung; infection; neutrophil; chemokines; T-cells; HMG-CoA reductase; NFAT

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APA (6th Edition):

Zhang, S. (2012). Adhesive and signaling mechanisms in abdominal sepsis. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/2517719 ; http://portal.research.lu.se/ws/files/3187970/2517763.pdf

Chicago Manual of Style (16th Edition):

Zhang, Su. “Adhesive and signaling mechanisms in abdominal sepsis.” 2012. Doctoral Dissertation, University of Lund. Accessed September 17, 2019. http://lup.lub.lu.se/record/2517719 ; http://portal.research.lu.se/ws/files/3187970/2517763.pdf.

MLA Handbook (7th Edition):

Zhang, Su. “Adhesive and signaling mechanisms in abdominal sepsis.” 2012. Web. 17 Sep 2019.

Vancouver:

Zhang S. Adhesive and signaling mechanisms in abdominal sepsis. [Internet] [Doctoral dissertation]. University of Lund; 2012. [cited 2019 Sep 17]. Available from: http://lup.lub.lu.se/record/2517719 ; http://portal.research.lu.se/ws/files/3187970/2517763.pdf.

Council of Science Editors:

Zhang S. Adhesive and signaling mechanisms in abdominal sepsis. [Doctoral Dissertation]. University of Lund; 2012. Available from: http://lup.lub.lu.se/record/2517719 ; http://portal.research.lu.se/ws/files/3187970/2517763.pdf


McMaster University

27. Lysov, Zakhar. Procoagulant effects of lung cancer chemotherapy on HUVEC, A549 cells, and monocytes.

Degree: MSc, 2011, McMaster University

Cancer patients undergoing chemotherapy have an elevated risk for thrombosis. Although thrombosis is a common complication in cancer patients, the mechanisms of chemotherapy-induced thrombosis… (more)

Subjects/Keywords: Lung Cancer; Thrombosis; Chemotherapy; A549 cells; Coagulation; Endothelial Cells; Monocytes.; Circulatory and Respiratory Physiology; Circulatory and Respiratory Physiology

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APA (6th Edition):

Lysov, Z. (2011). Procoagulant effects of lung cancer chemotherapy on HUVEC, A549 cells, and monocytes. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11601

Chicago Manual of Style (16th Edition):

Lysov, Zakhar. “Procoagulant effects of lung cancer chemotherapy on HUVEC, A549 cells, and monocytes.” 2011. Masters Thesis, McMaster University. Accessed September 17, 2019. http://hdl.handle.net/11375/11601.

MLA Handbook (7th Edition):

Lysov, Zakhar. “Procoagulant effects of lung cancer chemotherapy on HUVEC, A549 cells, and monocytes.” 2011. Web. 17 Sep 2019.

Vancouver:

Lysov Z. Procoagulant effects of lung cancer chemotherapy on HUVEC, A549 cells, and monocytes. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2019 Sep 17]. Available from: http://hdl.handle.net/11375/11601.

Council of Science Editors:

Lysov Z. Procoagulant effects of lung cancer chemotherapy on HUVEC, A549 cells, and monocytes. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11601

28. Faria, Carolina Arruda de. Terapêutica experimental com células mononucleares da medula óssea em modelo animal de enfisema pulmonar.

Degree: Mestrado, Biotecnologia, 2011, University of São Paulo

O enfisema pulmonar define-se como a destruição das paredes alveolares e consequente dispneia progressiva. Este trabalho objetivou a adequação de um modelo de transplante celular… (more)

Subjects/Keywords: Animal models; Bone marrow cells; Células da medula óssea; Células mononucleares; Células tronco; Enfisema pulmonar animal; Lung; Modelos animais; Mononuclear cells; Pulmão; Pulmonary emphysema animal; Stem cells

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APA (6th Edition):

Faria, C. A. d. (2011). Terapêutica experimental com células mononucleares da medula óssea em modelo animal de enfisema pulmonar. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/87/87131/tde-30052012-082259/ ;

Chicago Manual of Style (16th Edition):

Faria, Carolina Arruda de. “Terapêutica experimental com células mononucleares da medula óssea em modelo animal de enfisema pulmonar.” 2011. Masters Thesis, University of São Paulo. Accessed September 17, 2019. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-30052012-082259/ ;.

MLA Handbook (7th Edition):

Faria, Carolina Arruda de. “Terapêutica experimental com células mononucleares da medula óssea em modelo animal de enfisema pulmonar.” 2011. Web. 17 Sep 2019.

Vancouver:

Faria CAd. Terapêutica experimental com células mononucleares da medula óssea em modelo animal de enfisema pulmonar. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2019 Sep 17]. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-30052012-082259/ ;.

Council of Science Editors:

Faria CAd. Terapêutica experimental com células mononucleares da medula óssea em modelo animal de enfisema pulmonar. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/87/87131/tde-30052012-082259/ ;

29. Vasconcelos, Michelle. O papel da sinalização Notch na diferenciação do epitélio pulmonar.

Degree: PhD, Biologia Celular e Tecidual, 2012, University of São Paulo

O epitélio pulmonar é formado por uma grande diversidade celular, que incluí: células secretoras, ciliadas, basais e neuroendócrinas (NE). A distribuição balanceada destes tipos celulares… (more)

Subjects/Keywords: Cell differentiation; Células ciliadas; Diferenciação celular; Epitélio; Epithelium; Expressão gênica; Gene expression; Hair cells; Lung; Notch signaling; Pulmão; Sinalização Notch

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vasconcelos, M. (2012). O papel da sinalização Notch na diferenciação do epitélio pulmonar. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42134/tde-16052012-100050/ ;

Chicago Manual of Style (16th Edition):

Vasconcelos, Michelle. “O papel da sinalização Notch na diferenciação do epitélio pulmonar.” 2012. Doctoral Dissertation, University of São Paulo. Accessed September 17, 2019. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-16052012-100050/ ;.

MLA Handbook (7th Edition):

Vasconcelos, Michelle. “O papel da sinalização Notch na diferenciação do epitélio pulmonar.” 2012. Web. 17 Sep 2019.

Vancouver:

Vasconcelos M. O papel da sinalização Notch na diferenciação do epitélio pulmonar. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2019 Sep 17]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-16052012-100050/ ;.

Council of Science Editors:

Vasconcelos M. O papel da sinalização Notch na diferenciação do epitélio pulmonar. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-16052012-100050/ ;


Université Paris-Sud – Paris XI

30. Grabowski, Nadège. Toxicologie pulmonaire de nanoparticules biodégradables : effets cytotoxiques et inflammatoires sur cellules épithéliales et macrophages : Pulmonary toxicology of biodegradable nanoparticles : cytotoxic and inflammatory effects towards epithelial cells and macrophages.

Degree: Docteur es, Pharmacotechnie et Biopharmacie, 2013, Université Paris-Sud – Paris XI

 Ce projet de thèse se propose d’évaluer le devenir, la cytotoxicité et la réponse inflammatoire pulmonaire in vitro suite à l’exposition aux nanoparticules, et plus… (more)

Subjects/Keywords: Nanoparticules; Toxicologie; Poumon; Co-culture; Cellules épithéliales; Macrophages; Nanoparticles; Toxicology; Lung; Co-culture; Epithelilal cells; Macrophages

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Grabowski, N. (2013). Toxicologie pulmonaire de nanoparticules biodégradables : effets cytotoxiques et inflammatoires sur cellules épithéliales et macrophages : Pulmonary toxicology of biodegradable nanoparticles : cytotoxic and inflammatory effects towards epithelial cells and macrophages. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA114845

Chicago Manual of Style (16th Edition):

Grabowski, Nadège. “Toxicologie pulmonaire de nanoparticules biodégradables : effets cytotoxiques et inflammatoires sur cellules épithéliales et macrophages : Pulmonary toxicology of biodegradable nanoparticles : cytotoxic and inflammatory effects towards epithelial cells and macrophages.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 17, 2019. http://www.theses.fr/2013PA114845.

MLA Handbook (7th Edition):

Grabowski, Nadège. “Toxicologie pulmonaire de nanoparticules biodégradables : effets cytotoxiques et inflammatoires sur cellules épithéliales et macrophages : Pulmonary toxicology of biodegradable nanoparticles : cytotoxic and inflammatory effects towards epithelial cells and macrophages.” 2013. Web. 17 Sep 2019.

Vancouver:

Grabowski N. Toxicologie pulmonaire de nanoparticules biodégradables : effets cytotoxiques et inflammatoires sur cellules épithéliales et macrophages : Pulmonary toxicology of biodegradable nanoparticles : cytotoxic and inflammatory effects towards epithelial cells and macrophages. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2019 Sep 17]. Available from: http://www.theses.fr/2013PA114845.

Council of Science Editors:

Grabowski N. Toxicologie pulmonaire de nanoparticules biodégradables : effets cytotoxiques et inflammatoires sur cellules épithéliales et macrophages : Pulmonary toxicology of biodegradable nanoparticles : cytotoxic and inflammatory effects towards epithelial cells and macrophages. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA114845

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