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You searched for subject:(lung cancer). Showing records 1 – 30 of 1124 total matches.

[1] [2] [3] [4] [5] … [38]

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Harvard University

1. Li, Richard. Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer.

Degree: Doctor of Medicine, 2016, Harvard University

Background: Patients with non-small cell lung cancer (NSCLC) are known to be at high risk for venous thromboembolism (VTE), but previous studies have not specifically… (more)

Subjects/Keywords: lung cancer; thromboembolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, R. (2016). Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276

Chicago Manual of Style (16th Edition):

Li, Richard. “Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer.” 2016. Doctoral Dissertation, Harvard University. Accessed February 21, 2020. http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276.

MLA Handbook (7th Edition):

Li, Richard. “Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer.” 2016. Web. 21 Feb 2020.

Vancouver:

Li R. Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer. [Internet] [Doctoral dissertation]. Harvard University; 2016. [cited 2020 Feb 21]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276.

Council of Science Editors:

Li R. Venous Thromboembolism Risk in Patients With Locally Advanced Non-Small Cell Lung Cancer. [Doctoral Dissertation]. Harvard University; 2016. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:40620276


University of Dundee

2. Robertson, Holly. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.

Degree: PhD, 2019, University of Dundee

Lung cancer is the leading cause of cancer related mortality worldwide and since the discovery of the important role that NRF2 plays in regulating the… (more)

Subjects/Keywords: NRF2; Lung Cancer

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APA (6th Edition):

Robertson, H. (2019). Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364

Chicago Manual of Style (16th Edition):

Robertson, Holly. “Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.” 2019. Doctoral Dissertation, University of Dundee. Accessed February 21, 2020. https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364.

MLA Handbook (7th Edition):

Robertson, Holly. “Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression.” 2019. Web. 21 Feb 2020.

Vancouver:

Robertson H. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. [Internet] [Doctoral dissertation]. University of Dundee; 2019. [cited 2020 Feb 21]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364.

Council of Science Editors:

Robertson H. Mechanisms of repression of the transcription factor NRF2 by KEAP1- and B-TrCP-dependent ubiquitin ligases and how the dysregulation of NRF2 contributes to lung cancer progression. [Doctoral Dissertation]. University of Dundee; 2019. Available from: https://discovery.dundee.ac.uk/en/studentTheses/4a7f87db-89f1-4bc4-b273-1bd7eb72ab0e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.775364


University of New South Wales

3. Wong, Sze Wing (Alice). Functions of S100A8 in lung cancer.

Degree: Medical Sciences, 2018, University of New South Wales

 S100A8 is an anti-inflammatory oxidant scavenger protein that induces IL-10 in airway epithelial cells in normal lungs following inhalation. These mechanisms contribute to its ability… (more)

Subjects/Keywords: Lung cancer; S100A8

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APA (6th Edition):

Wong, S. W. (. (2018). Functions of S100A8 in lung cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60230

Chicago Manual of Style (16th Edition):

Wong, Sze Wing (Alice). “Functions of S100A8 in lung cancer.” 2018. Doctoral Dissertation, University of New South Wales. Accessed February 21, 2020. http://handle.unsw.edu.au/1959.4/60230.

MLA Handbook (7th Edition):

Wong, Sze Wing (Alice). “Functions of S100A8 in lung cancer.” 2018. Web. 21 Feb 2020.

Vancouver:

Wong SW(. Functions of S100A8 in lung cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2018. [cited 2020 Feb 21]. Available from: http://handle.unsw.edu.au/1959.4/60230.

Council of Science Editors:

Wong SW(. Functions of S100A8 in lung cancer. [Doctoral Dissertation]. University of New South Wales; 2018. Available from: http://handle.unsw.edu.au/1959.4/60230


University of Southern California

4. Selamat, Suhaida Adura. DNA methylation changes in the development of lung adenocarcinoma.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

Lung cancer accounted for 13% of total cancer cases and 18% of cancer deaths globally in 2008. The combination of increasing smoking prevalence in many… (more)

Subjects/Keywords: DNA methylation; lung adenocarcinoma; lung cancer; epigenetics

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APA (6th Edition):

Selamat, S. A. (2012). DNA methylation changes in the development of lung adenocarcinoma. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2066

Chicago Manual of Style (16th Edition):

Selamat, Suhaida Adura. “DNA methylation changes in the development of lung adenocarcinoma.” 2012. Doctoral Dissertation, University of Southern California. Accessed February 21, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2066.

MLA Handbook (7th Edition):

Selamat, Suhaida Adura. “DNA methylation changes in the development of lung adenocarcinoma.” 2012. Web. 21 Feb 2020.

Vancouver:

Selamat SA. DNA methylation changes in the development of lung adenocarcinoma. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2020 Feb 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2066.

Council of Science Editors:

Selamat SA. DNA methylation changes in the development of lung adenocarcinoma. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2066


University of Dundee

5. Buchanan, Deans. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.

Degree: Thesis (M.D.), 2010, University of Dundee

Lung cancer has developed from a rare condition into the leading cause of cancerrelated death in the United Kingdom. Lung cancer patients face a disease… (more)

Subjects/Keywords: Lung cancer; Supportive care; Symptoms

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APA (6th Edition):

Buchanan, D. (2010). The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52

Chicago Manual of Style (16th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Doctoral Dissertation, University of Dundee. Accessed February 21, 2020. http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52.

MLA Handbook (7th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Web. 21 Feb 2020.

Vancouver:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Internet] [Doctoral dissertation]. University of Dundee; 2010. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52.

Council of Science Editors:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Doctoral Dissertation]. University of Dundee; 2010. Available from: http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52


University of Alberta

6. Al-Hallak, MHD Kamal. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 In 2002, lung cancer was responsible for 17.6% of the total worldwide deaths from cancer. Beyond the three traditional forms of cancer treatment, surgery, radiation… (more)

Subjects/Keywords: inhalable nanoparticles; Lung cancer; Macrophages

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APA (6th Edition):

Al-Hallak, M. K. (2012). Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/mk61rj53p

Chicago Manual of Style (16th Edition):

Al-Hallak, MHD Kamal. “Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.” 2012. Doctoral Dissertation, University of Alberta. Accessed February 21, 2020. https://era.library.ualberta.ca/files/mk61rj53p.

MLA Handbook (7th Edition):

Al-Hallak, MHD Kamal. “Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.” 2012. Web. 21 Feb 2020.

Vancouver:

Al-Hallak MK. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2020 Feb 21]. Available from: https://era.library.ualberta.ca/files/mk61rj53p.

Council of Science Editors:

Al-Hallak MK. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/mk61rj53p


University of Dundee

7. Buchanan, Deans. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.

Degree: Thesis (M.D.), 2010, University of Dundee

Lung cancer has developed from a rare condition into the leading cause of cancerrelated death in the United Kingdom. Lung cancer patients face a disease… (more)

Subjects/Keywords: Lung cancer; Supportive care; Symptoms

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APA (6th Edition):

Buchanan, D. (2010). The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/1047

Chicago Manual of Style (16th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Doctoral Dissertation, University of Dundee. Accessed February 21, 2020. http://hdl.handle.net/10588/1047.

MLA Handbook (7th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Web. 21 Feb 2020.

Vancouver:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Internet] [Doctoral dissertation]. University of Dundee; 2010. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10588/1047.

Council of Science Editors:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Doctoral Dissertation]. University of Dundee; 2010. Available from: http://hdl.handle.net/10588/1047


Universiteit Utrecht

8. Breeveld, A.R. Shape of the exposure response relation for crystalline silica and risk of lung cancer.

Degree: 2011, Universiteit Utrecht

 Crystalline silica exposure mostly occurs in occupational settings such as mining, construction, several industries (e.g. foundries) and agriculture. Occupational exposure to silica dust is known… (more)

Subjects/Keywords: crystalline silica; lung cancer

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APA (6th Edition):

Breeveld, A. R. (2011). Shape of the exposure response relation for crystalline silica and risk of lung cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/209135

Chicago Manual of Style (16th Edition):

Breeveld, A R. “Shape of the exposure response relation for crystalline silica and risk of lung cancer.” 2011. Masters Thesis, Universiteit Utrecht. Accessed February 21, 2020. http://dspace.library.uu.nl:8080/handle/1874/209135.

MLA Handbook (7th Edition):

Breeveld, A R. “Shape of the exposure response relation for crystalline silica and risk of lung cancer.” 2011. Web. 21 Feb 2020.

Vancouver:

Breeveld AR. Shape of the exposure response relation for crystalline silica and risk of lung cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2020 Feb 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/209135.

Council of Science Editors:

Breeveld AR. Shape of the exposure response relation for crystalline silica and risk of lung cancer. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/209135


Penn State University

9. Crampsie, Melissa Ashley. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE.

Degree: PhD, Pharmacology, 2011, Penn State University

 Goals of Dissertation Research Lung cancer is currently the leading cause of cancer deaths among men and women and has a survival rate of only… (more)

Subjects/Keywords: ISC-4; lung cancer; chemoprevention

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APA (6th Edition):

Crampsie, M. A. (2011). INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/12391

Chicago Manual of Style (16th Edition):

Crampsie, Melissa Ashley. “INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE.” 2011. Doctoral Dissertation, Penn State University. Accessed February 21, 2020. https://etda.libraries.psu.edu/catalog/12391.

MLA Handbook (7th Edition):

Crampsie, Melissa Ashley. “INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE.” 2011. Web. 21 Feb 2020.

Vancouver:

Crampsie MA. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE. [Internet] [Doctoral dissertation]. Penn State University; 2011. [cited 2020 Feb 21]. Available from: https://etda.libraries.psu.edu/catalog/12391.

Council of Science Editors:

Crampsie MA. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE. [Doctoral Dissertation]. Penn State University; 2011. Available from: https://etda.libraries.psu.edu/catalog/12391


UCLA

10. Grimes, Brandon Scott. The immune contexture and genomic landscape of lung adenomatous premalignancy.

Degree: Clinical Research, 2017, UCLA

 Rationale/Objective: Over the past 30 years, the five-year survival rate for lung cancer has increased by only 5%. A better understanding of genomic alterations and… (more)

Subjects/Keywords: Biostatistics; Lung Cancer; Neoepitopes; Premalignancy

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APA (6th Edition):

Grimes, B. S. (2017). The immune contexture and genomic landscape of lung adenomatous premalignancy. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5691h7fs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Grimes, Brandon Scott. “The immune contexture and genomic landscape of lung adenomatous premalignancy.” 2017. Thesis, UCLA. Accessed February 21, 2020. http://www.escholarship.org/uc/item/5691h7fs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Grimes, Brandon Scott. “The immune contexture and genomic landscape of lung adenomatous premalignancy.” 2017. Web. 21 Feb 2020.

Vancouver:

Grimes BS. The immune contexture and genomic landscape of lung adenomatous premalignancy. [Internet] [Thesis]. UCLA; 2017. [cited 2020 Feb 21]. Available from: http://www.escholarship.org/uc/item/5691h7fs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Grimes BS. The immune contexture and genomic landscape of lung adenomatous premalignancy. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/5691h7fs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

11. Zeevi, Chen. Chemotherapy and biological treatment of lung cancer .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

Thesis on chemotherapy and biological treatment of lung cancer. Advisors/Committee Members: Pórszász, Róbert (advisor), Debreceni Egyetem::Általános Orvostudományi Kar::Farmakológiai és Farmakoterápiai Intézet (advisor).

Subjects/Keywords: lung cancer

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APA (6th Edition):

Zeevi, C. (n.d.). Chemotherapy and biological treatment of lung cancer . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/247211

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zeevi, Chen. “Chemotherapy and biological treatment of lung cancer .” Thesis, University of Debrecen. Accessed February 21, 2020. http://hdl.handle.net/2437/247211.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zeevi, Chen. “Chemotherapy and biological treatment of lung cancer .” Web. 21 Feb 2020.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Zeevi C. Chemotherapy and biological treatment of lung cancer . [Internet] [Thesis]. University of Debrecen; [cited 2020 Feb 21]. Available from: http://hdl.handle.net/2437/247211.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Zeevi C. Chemotherapy and biological treatment of lung cancer . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/247211

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Boston University

12. Garrison, Carly. Characterizing microRNA regulators of lung disease.

Degree: PhD, Genetics & Genomics, 2016, Boston University

Lung diseases are one of the leading causes of mortality and morbidity worldwide. Understanding these diseases at a molecular level remains a critical component to… (more)

Subjects/Keywords: Genetics; Cancer; Lung; MicroRNA; Therapeutic

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APA (6th Edition):

Garrison, C. (2016). Characterizing microRNA regulators of lung disease. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/14598

Chicago Manual of Style (16th Edition):

Garrison, Carly. “Characterizing microRNA regulators of lung disease.” 2016. Doctoral Dissertation, Boston University. Accessed February 21, 2020. http://hdl.handle.net/2144/14598.

MLA Handbook (7th Edition):

Garrison, Carly. “Characterizing microRNA regulators of lung disease.” 2016. Web. 21 Feb 2020.

Vancouver:

Garrison C. Characterizing microRNA regulators of lung disease. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/2144/14598.

Council of Science Editors:

Garrison C. Characterizing microRNA regulators of lung disease. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/14598


Boston University

13. Perez-Rogers, Joseph. Development of a minimally invasive molecular biomarker for early detection of lung cancer.

Degree: PhD, Bioinformatics GRS, 2017, Boston University

 The diagnostic evaluation of ever smokers with pulmonary nodules represents a growing clinical challenge due to the implementation of lung cancer screening. The high false-positive… (more)

Subjects/Keywords: Bioinformatics; Biomarker; Classifier; Lung cancer

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APA (6th Edition):

Perez-Rogers, J. (2017). Development of a minimally invasive molecular biomarker for early detection of lung cancer. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/21969

Chicago Manual of Style (16th Edition):

Perez-Rogers, Joseph. “Development of a minimally invasive molecular biomarker for early detection of lung cancer.” 2017. Doctoral Dissertation, Boston University. Accessed February 21, 2020. http://hdl.handle.net/2144/21969.

MLA Handbook (7th Edition):

Perez-Rogers, Joseph. “Development of a minimally invasive molecular biomarker for early detection of lung cancer.” 2017. Web. 21 Feb 2020.

Vancouver:

Perez-Rogers J. Development of a minimally invasive molecular biomarker for early detection of lung cancer. [Internet] [Doctoral dissertation]. Boston University; 2017. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/2144/21969.

Council of Science Editors:

Perez-Rogers J. Development of a minimally invasive molecular biomarker for early detection of lung cancer. [Doctoral Dissertation]. Boston University; 2017. Available from: http://hdl.handle.net/2144/21969


Cornell University

14. Jirapatnakul, Artit. Computer Methods For Pulmonary Nodule Characterization From Ct Images .

Degree: 2011, Cornell University

 Computed tomography (CT) scans provide radiologists a non-invasive method of imaging internal structures of the body. Although CT scans have enabled the earlier detection of… (more)

Subjects/Keywords: pulmonary nodule; characterization; lung cancer

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APA (6th Edition):

Jirapatnakul, A. (2011). Computer Methods For Pulmonary Nodule Characterization From Ct Images . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/29192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jirapatnakul, Artit. “Computer Methods For Pulmonary Nodule Characterization From Ct Images .” 2011. Thesis, Cornell University. Accessed February 21, 2020. http://hdl.handle.net/1813/29192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jirapatnakul, Artit. “Computer Methods For Pulmonary Nodule Characterization From Ct Images .” 2011. Web. 21 Feb 2020.

Vancouver:

Jirapatnakul A. Computer Methods For Pulmonary Nodule Characterization From Ct Images . [Internet] [Thesis]. Cornell University; 2011. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/1813/29192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jirapatnakul A. Computer Methods For Pulmonary Nodule Characterization From Ct Images . [Thesis]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/29192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

15. Samanta, Debangshu. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Inactivating mutations in TGF-©¬ receptors and Smad signal transducers that contribute to resistance to TGF-©¬, are associated with only very small number of NSCLC. The… (more)

Subjects/Keywords: Smoking; Lung cancer; Smad3; Chemoresistance

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APA (6th Edition):

Samanta, D. (2012). Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;

Chicago Manual of Style (16th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed February 21, 2020. http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

MLA Handbook (7th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Web. 21 Feb 2020.

Vancouver:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Feb 21]. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

Council of Science Editors:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;


Vanderbilt University

16. Meador, Catherine Belle. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 EGFR-mutant lung cancers are highly sensitive to EGFR tyrosine kinase inhibitors (TKIs; erlotinib/gefitinib/afatinib), but tumors develop drug resistance within 9-16 months. Resistance to gefitinib/erlotinib commonly… (more)

Subjects/Keywords: targeted therapy; EGFR; lung cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Meador, C. B. (2015). Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07202015-172236/ ;

Chicago Manual of Style (16th Edition):

Meador, Catherine Belle. “Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed February 21, 2020. http://etd.library.vanderbilt.edu/available/etd-07202015-172236/ ;.

MLA Handbook (7th Edition):

Meador, Catherine Belle. “Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma.” 2015. Web. 21 Feb 2020.

Vancouver:

Meador CB. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Feb 21]. Available from: http://etd.library.vanderbilt.edu/available/etd-07202015-172236/ ;.

Council of Science Editors:

Meador CB. Optimizing the sequence of targeted therapy in EGFR-mutant lung adenocarcinoma. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-07202015-172236/ ;


University of New South Wales

17. Luk, Peter Ping-Kit. The influence of the EGFR pathway in modulating the efficacy of gemcitabine.

Degree: Clinical School - St George Hospital, 2011, University of New South Wales

 Multiple anti-cancer drugs are often combined to increase their effectiveness. This study investigates the combination between an epidermal growth factor receptor (EGFR) inhibitor and gemcitabine,… (more)

Subjects/Keywords: Lung cancer; Gemcitabine; EGFR

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APA (6th Edition):

Luk, P. P. (2011). The influence of the EGFR pathway in modulating the efficacy of gemcitabine. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Doctoral Dissertation, University of New South Wales. Accessed February 21, 2020. http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true.

MLA Handbook (7th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Web. 21 Feb 2020.

Vancouver:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2020 Feb 21]. Available from: http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true.

Council of Science Editors:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50384 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9271/SOURCE02?view=true


University of Miami

18. Milone, Mary Anne. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.

Degree: PhD, Nursing (Nursing), 2010, University of Miami

Lung cancer is the most common cause of cancer deaths worldwide. Hope is considered essential to life and has been positively associated with coping.… (more)

Subjects/Keywords: Cancer; Lung; Palliative; Hope

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Milone, M. A. (2010). The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. (Doctoral Dissertation). University of Miami. Retrieved from https://scholarlyrepository.miami.edu/oa_dissertations/409

Chicago Manual of Style (16th Edition):

Milone, Mary Anne. “The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.” 2010. Doctoral Dissertation, University of Miami. Accessed February 21, 2020. https://scholarlyrepository.miami.edu/oa_dissertations/409.

MLA Handbook (7th Edition):

Milone, Mary Anne. “The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.” 2010. Web. 21 Feb 2020.

Vancouver:

Milone MA. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2020 Feb 21]. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/409.

Council of Science Editors:

Milone MA. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. [Doctoral Dissertation]. University of Miami; 2010. Available from: https://scholarlyrepository.miami.edu/oa_dissertations/409


Queens University

19. Ahn, Joseph. Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer .

Degree: Biochemistry, 2012, Queens University

 Fer is a ubiquitously expressed non-receptor protein-tyrosine kinase that regulates normal physiology through signaling in a variety of cell types. Fer signals downstream of growth… (more)

Subjects/Keywords: FER Kinase; Lung Cancer

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APA (6th Edition):

Ahn, J. (2012). Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahn, Joseph. “Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer .” 2012. Thesis, Queens University. Accessed February 21, 2020. http://hdl.handle.net/1974/7441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahn, Joseph. “Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer .” 2012. Web. 21 Feb 2020.

Vancouver:

Ahn J. Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer . [Internet] [Thesis]. Queens University; 2012. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/1974/7441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahn J. Fer Protein-Tyrosine Kinase as a Potential Therapeutic Target in Lung Cancer . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

20. Muench, Edward David, 1987-. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.

Degree: M. Eng., 2012, University of Louisville

 Vascular endothelial growth factor (VEGF), a commonly overexpressed oncogene in a variety of malignancies including non-small cell lung cancer (NSCLC), is a key regulator of… (more)

Subjects/Keywords: VEGF; Lung cancer; Quadruplex; Cancer; Oligonucleotide

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APA (6th Edition):

Muench, Edward David, 1. (2012). The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022

Chicago Manual of Style (16th Edition):

Muench, Edward David, 1987-. “The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.” 2012. Masters Thesis, University of Louisville. Accessed February 21, 2020. 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022.

MLA Handbook (7th Edition):

Muench, Edward David, 1987-. “The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.” 2012. Web. 21 Feb 2020.

Vancouver:

Muench, Edward David 1. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. [Internet] [Masters thesis]. University of Louisville; 2012. [cited 2020 Feb 21]. Available from: 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022.

Council of Science Editors:

Muench, Edward David 1. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. [Masters Thesis]. University of Louisville; 2012. Available from: 10.18297/etd/1022 ; https://ir.library.louisville.edu/etd/1022

21. Michel, Noémie. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2013, Université François-Rabelais de Tours

Les kallicréines tissulaires humaines (KLK) ont récemment émergé comme une famille de protéases à serine pouvant jouer un rôle important dans la tumorigenèse. Le but… (more)

Subjects/Keywords: Kallicréines; Cancer du poumon; Kallikreins; Lung cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Michel, N. (2013). Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2013TOUR3303

Chicago Manual of Style (16th Edition):

Michel, Noémie. “Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.” 2013. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed February 21, 2020. http://www.theses.fr/2013TOUR3303.

MLA Handbook (7th Edition):

Michel, Noémie. “Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.” 2013. Web. 21 Feb 2020.

Vancouver:

Michel N. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2013. [cited 2020 Feb 21]. Available from: http://www.theses.fr/2013TOUR3303.

Council of Science Editors:

Michel N. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2013. Available from: http://www.theses.fr/2013TOUR3303


Kansas State University

22. Uppalapati, Lakshmi. Peptides as therapeutics and active gene delivery vehicles for cancer treatment.

Degree: PhD, Department of Agronomy, 2015, Kansas State University

 Over the years proteins/peptides have evolved as promising therapeutic agents in the treatment of cancer. Considering the advantages of peptides such as their small size,… (more)

Subjects/Keywords: Cancer therapeutics; Nanoparticle; Intratracheal; Lung cancer

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APA (6th Edition):

Uppalapati, L. (2015). Peptides as therapeutics and active gene delivery vehicles for cancer treatment. (Doctoral Dissertation). Kansas State University. Retrieved from http://hdl.handle.net/2097/35231

Chicago Manual of Style (16th Edition):

Uppalapati, Lakshmi. “Peptides as therapeutics and active gene delivery vehicles for cancer treatment.” 2015. Doctoral Dissertation, Kansas State University. Accessed February 21, 2020. http://hdl.handle.net/2097/35231.

MLA Handbook (7th Edition):

Uppalapati, Lakshmi. “Peptides as therapeutics and active gene delivery vehicles for cancer treatment.” 2015. Web. 21 Feb 2020.

Vancouver:

Uppalapati L. Peptides as therapeutics and active gene delivery vehicles for cancer treatment. [Internet] [Doctoral dissertation]. Kansas State University; 2015. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/2097/35231.

Council of Science Editors:

Uppalapati L. Peptides as therapeutics and active gene delivery vehicles for cancer treatment. [Doctoral Dissertation]. Kansas State University; 2015. Available from: http://hdl.handle.net/2097/35231


University of New South Wales

23. Jacob, Susannah Aachi. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.

Degree: Public Health & Community Medicine, 2010, University of New South Wales

 Introduction: Chemotherapy utilisation rates can vary widely between area health services, between states and internationally. Variation in chemotherapy utilisation rates is particularly marked in lung(more)

Subjects/Keywords: Lung cancer; Chemotherapy; Utilisation rates; Colorectal cancer

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APA (6th Edition):

Jacob, S. A. (2010). The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Jacob, Susannah Aachi. “The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.” 2010. Doctoral Dissertation, University of New South Wales. Accessed February 21, 2020. http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true.

MLA Handbook (7th Edition):

Jacob, Susannah Aachi. “The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.” 2010. Web. 21 Feb 2020.

Vancouver:

Jacob SA. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. [Internet] [Doctoral dissertation]. University of New South Wales; 2010. [cited 2020 Feb 21]. Available from: http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true.

Council of Science Editors:

Jacob SA. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. [Doctoral Dissertation]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/50381 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:9268/SOURCE02?view=true


Universiteit Utrecht

24. Waal, L.M. de. Treatment of subtypes in non-small cell lung cancer.

Degree: 2012, Universiteit Utrecht

Lung cancer accounts for the highest number of cancer related deaths among all cancer patients. It has been well established that exposure to tobacco significantly… (more)

Subjects/Keywords: lung cancer; treatment; targeted therapy; non-small cell lung cancer

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APA (6th Edition):

Waal, L. M. d. (2012). Treatment of subtypes in non-small cell lung cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/241663

Chicago Manual of Style (16th Edition):

Waal, L M de. “Treatment of subtypes in non-small cell lung cancer.” 2012. Masters Thesis, Universiteit Utrecht. Accessed February 21, 2020. http://dspace.library.uu.nl:8080/handle/1874/241663.

MLA Handbook (7th Edition):

Waal, L M de. “Treatment of subtypes in non-small cell lung cancer.” 2012. Web. 21 Feb 2020.

Vancouver:

Waal LMd. Treatment of subtypes in non-small cell lung cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2020 Feb 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/241663.

Council of Science Editors:

Waal LMd. Treatment of subtypes in non-small cell lung cancer. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/241663


University of Southern California

25. Chopra, Nikhil. CpG methylation profiling in lung cancer cell lines, tumors and non-tumors.

Degree: MS, Biochemistry & Molecular Biology, 2009, University of Southern California

 Cell lines are widely used for studying DNA methylation in cancer. Their main applications are in the development of biomarkers, in pharmacological studies and for… (more)

Subjects/Keywords: DNA methylation; lung cancer; cell lines; adenocarcinoma; epigenetics; methylation; CpG; cancer biomarkers; lung; cancer

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APA (6th Edition):

Chopra, N. (2009). CpG methylation profiling in lung cancer cell lines, tumors and non-tumors. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1681

Chicago Manual of Style (16th Edition):

Chopra, Nikhil. “CpG methylation profiling in lung cancer cell lines, tumors and non-tumors.” 2009. Masters Thesis, University of Southern California. Accessed February 21, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1681.

MLA Handbook (7th Edition):

Chopra, Nikhil. “CpG methylation profiling in lung cancer cell lines, tumors and non-tumors.” 2009. Web. 21 Feb 2020.

Vancouver:

Chopra N. CpG methylation profiling in lung cancer cell lines, tumors and non-tumors. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2020 Feb 21]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1681.

Council of Science Editors:

Chopra N. CpG methylation profiling in lung cancer cell lines, tumors and non-tumors. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1681


Kyoto University / 京都大学

26. Nakaoku, Takashi. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.

Degree: 博士(医学), 2016, Kyoto University / 京都大学

リポジトリの登録にあたっては、Peer reviewされた最終版のみ可能であり、その際には下記の出版社のウェブサイトのアドレスを記載することが求められる。当該論文は2014年6月の出版であり、12ヶ月を経過していることから、公開には差し支えはない。http://clincancerres.aacrjournals.org/content/20/12/3087.full

新制・課程博士

甲第19617号

医博第4124号

Subjects/Keywords: Lung Cancer; Imvasive mucinous lung adenocarcinoma; Gene rearrangement; Targeted therapy; NRG1

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APA (6th Edition):

Nakaoku, T. (2016). Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakaoku, Takashi. “Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.” 2016. Thesis, Kyoto University / 京都大学. Accessed February 21, 2020. http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakaoku, Takashi. “Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.” 2016. Web. 21 Feb 2020.

Vancouver:

Nakaoku T. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakaoku T. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

27. Carter, Cambron N. CAD system for lung nodule analysis.

Degree: M. Eng., 2012, University of Louisville

Lung cancer is the deadliest type of known cancer in the United States, claiming hundreds of thousands of lives each year. However, despite the high… (more)

Subjects/Keywords: Lung cancer; Lung nodule; CAD system; Nodule detection; Medical imaging

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APA (6th Edition):

Carter, C. N. (2012). CAD system for lung nodule analysis. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/218 ; https://ir.library.louisville.edu/etd/218

Chicago Manual of Style (16th Edition):

Carter, Cambron N. “CAD system for lung nodule analysis.” 2012. Masters Thesis, University of Louisville. Accessed February 21, 2020. 10.18297/etd/218 ; https://ir.library.louisville.edu/etd/218.

MLA Handbook (7th Edition):

Carter, Cambron N. “CAD system for lung nodule analysis.” 2012. Web. 21 Feb 2020.

Vancouver:

Carter CN. CAD system for lung nodule analysis. [Internet] [Masters thesis]. University of Louisville; 2012. [cited 2020 Feb 21]. Available from: 10.18297/etd/218 ; https://ir.library.louisville.edu/etd/218.

Council of Science Editors:

Carter CN. CAD system for lung nodule analysis. [Masters Thesis]. University of Louisville; 2012. Available from: 10.18297/etd/218 ; https://ir.library.louisville.edu/etd/218

28. Yamazaki, Motohiko. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.

Degree: 博士(医学), 2015, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3976号. 学位記番号: 新大院博(医)甲第622号. 学位授与年月日: 平成27年3月23日

European Journal of Radiology. 2015, 84(1), 178-184.

Objectives: To assess the prognostic value of combined evaluation of… (more)

Subjects/Keywords: Chest CT imaging; lung adenocarcinoma; lung cancer; serum CEA levels; prognosis

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APA (6th Edition):

Yamazaki, M. (2015). A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/32260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamazaki, Motohiko. “A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.” 2015. Thesis, Niigata University / 新潟大学. Accessed February 21, 2020. http://hdl.handle.net/10191/32260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamazaki, Motohiko. “A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.” 2015. Web. 21 Feb 2020.

Vancouver:

Yamazaki M. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. [Internet] [Thesis]. Niigata University / 新潟大学; 2015. [cited 2020 Feb 21]. Available from: http://hdl.handle.net/10191/32260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamazaki M. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. [Thesis]. Niigata University / 新潟大学; 2015. Available from: http://hdl.handle.net/10191/32260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

29. Miller, Joshua Christopher. ADENYLOSUCCINATE SYNTHETASE 1:.

Degree: PhD, Pharmacology, 2010, Penn State University

 Tobacco smoke consists of numerous carcinogens whose effect on lung tumor development includes the induction of mutations in key metabolic genes as well as the… (more)

Subjects/Keywords: lung cancer; lung adenocarcinoma; metabolic reprogramming; adenylosuccinate synthetase; , novel deletion

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Miller, J. C. (2010). ADENYLOSUCCINATE SYNTHETASE 1:. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11282

Chicago Manual of Style (16th Edition):

Miller, Joshua Christopher. “ADENYLOSUCCINATE SYNTHETASE 1:.” 2010. Doctoral Dissertation, Penn State University. Accessed February 21, 2020. https://etda.libraries.psu.edu/catalog/11282.

MLA Handbook (7th Edition):

Miller, Joshua Christopher. “ADENYLOSUCCINATE SYNTHETASE 1:.” 2010. Web. 21 Feb 2020.

Vancouver:

Miller JC. ADENYLOSUCCINATE SYNTHETASE 1:. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Feb 21]. Available from: https://etda.libraries.psu.edu/catalog/11282.

Council of Science Editors:

Miller JC. ADENYLOSUCCINATE SYNTHETASE 1:. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11282


Louisiana State University

30. Williams, Kendrick J. Assessment of detection and characterization of simulated lung nodules with low-dose CT.

Degree: MS, Physical Sciences and Mathematics, 2013, Louisiana State University

 Purpose: The quality of a computed tomography (CT) image and the dose delivered depend upon the acquisition parameters used to acquire the CT scan. Current,… (more)

Subjects/Keywords: Low-Dose CT; Lung Cancer Screening; Computed Tomography; Simulated lung nodules

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, K. J. (2013). Assessment of detection and characterization of simulated lung nodules with low-dose CT. (Masters Thesis). Louisiana State University. Retrieved from etd-09122013-070549 ; https://digitalcommons.lsu.edu/gradschool_theses/3320

Chicago Manual of Style (16th Edition):

Williams, Kendrick J. “Assessment of detection and characterization of simulated lung nodules with low-dose CT.” 2013. Masters Thesis, Louisiana State University. Accessed February 21, 2020. etd-09122013-070549 ; https://digitalcommons.lsu.edu/gradschool_theses/3320.

MLA Handbook (7th Edition):

Williams, Kendrick J. “Assessment of detection and characterization of simulated lung nodules with low-dose CT.” 2013. Web. 21 Feb 2020.

Vancouver:

Williams KJ. Assessment of detection and characterization of simulated lung nodules with low-dose CT. [Internet] [Masters thesis]. Louisiana State University; 2013. [cited 2020 Feb 21]. Available from: etd-09122013-070549 ; https://digitalcommons.lsu.edu/gradschool_theses/3320.

Council of Science Editors:

Williams KJ. Assessment of detection and characterization of simulated lung nodules with low-dose CT. [Masters Thesis]. Louisiana State University; 2013. Available from: etd-09122013-070549 ; https://digitalcommons.lsu.edu/gradschool_theses/3320

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