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You searched for subject:(lung cancer). Showing records 1 – 30 of 864 total matches.

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1. Hallqvist, Andreas. Optimization of radiotherapy in locally advanced lung cancer.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

Lung cancer is the leading cause of cancer death worldwide as well as in Sweden, where the incidence is around 3500 new cases per year.… (more)

Subjects/Keywords: radiotherapy; lung cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hallqvist, A. (2011). Optimization of radiotherapy in locally advanced lung cancer. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/27822

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hallqvist, Andreas. “Optimization of radiotherapy in locally advanced lung cancer.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed September 22, 2017. http://hdl.handle.net/2077/27822.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hallqvist, Andreas. “Optimization of radiotherapy in locally advanced lung cancer.” 2011. Web. 22 Sep 2017.

Vancouver:

Hallqvist A. Optimization of radiotherapy in locally advanced lung cancer. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2077/27822.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hallqvist A. Optimization of radiotherapy in locally advanced lung cancer. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/27822

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

2. Selamat, Suhaida Adura. DNA methylation changes in the development of lung adenocarcinoma.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2012, University of Southern California

Lung cancer accounted for 13% of total cancer cases and 18% of cancer deaths globally in 2008. The combination of increasing smoking prevalence in many… (more)

Subjects/Keywords: DNA methylation; lung adenocarcinoma; lung cancer; epigenetics

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APA (6th Edition):

Selamat, S. A. (2012). DNA methylation changes in the development of lung adenocarcinoma. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2060

Chicago Manual of Style (16th Edition):

Selamat, Suhaida Adura. “DNA methylation changes in the development of lung adenocarcinoma.” 2012. Doctoral Dissertation, University of Southern California. Accessed September 22, 2017. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2060.

MLA Handbook (7th Edition):

Selamat, Suhaida Adura. “DNA methylation changes in the development of lung adenocarcinoma.” 2012. Web. 22 Sep 2017.

Vancouver:

Selamat SA. DNA methylation changes in the development of lung adenocarcinoma. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2017 Sep 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2060.

Council of Science Editors:

Selamat SA. DNA methylation changes in the development of lung adenocarcinoma. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/2645/rec/2060


University of Dundee

3. Buchanan, Deans. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.

Degree: Thesis (M.D.), 2010, University of Dundee

Lung cancer has developed from a rare condition into the leading cause of cancerrelated death in the United Kingdom. Lung cancer patients face a disease… (more)

Subjects/Keywords: Lung cancer; Supportive care; Symptoms

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APA (6th Edition):

Buchanan, D. (2010). The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52

Chicago Manual of Style (16th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Doctoral Dissertation, University of Dundee. Accessed September 22, 2017. http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52.

MLA Handbook (7th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Web. 22 Sep 2017.

Vancouver:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Internet] [Doctoral dissertation]. University of Dundee; 2010. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52.

Council of Science Editors:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Doctoral Dissertation]. University of Dundee; 2010. Available from: http://hdl.handle.net/10588/43cf70da-fac1-41d5-be95-213521128c52


Cornell University

4. Jirapatnakul, Artit. Computer Methods For Pulmonary Nodule Characterization From Ct Images .

Degree: 2011, Cornell University

 Computed tomography (CT) scans provide radiologists a non-invasive method of imaging internal structures of the body. Although CT scans have enabled the earlier detection of… (more)

Subjects/Keywords: pulmonary nodule; characterization; lung cancer

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APA (6th Edition):

Jirapatnakul, A. (2011). Computer Methods For Pulmonary Nodule Characterization From Ct Images . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/29192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jirapatnakul, Artit. “Computer Methods For Pulmonary Nodule Characterization From Ct Images .” 2011. Thesis, Cornell University. Accessed September 22, 2017. http://hdl.handle.net/1813/29192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jirapatnakul, Artit. “Computer Methods For Pulmonary Nodule Characterization From Ct Images .” 2011. Web. 22 Sep 2017.

Vancouver:

Jirapatnakul A. Computer Methods For Pulmonary Nodule Characterization From Ct Images . [Internet] [Thesis]. Cornell University; 2011. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1813/29192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jirapatnakul A. Computer Methods For Pulmonary Nodule Characterization From Ct Images . [Thesis]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/29192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

5. Al-Hallak, MHD Kamal. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 In 2002, lung cancer was responsible for 17.6% of the total worldwide deaths from cancer. Beyond the three traditional forms of cancer treatment, surgery, radiation… (more)

Subjects/Keywords: inhalable nanoparticles; Lung cancer; Macrophages

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APA (6th Edition):

Al-Hallak, M. K. (2012). Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/mk61rj53p

Chicago Manual of Style (16th Edition):

Al-Hallak, MHD Kamal. “Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.” 2012. Doctoral Dissertation, University of Alberta. Accessed September 22, 2017. https://era.library.ualberta.ca/files/mk61rj53p.

MLA Handbook (7th Edition):

Al-Hallak, MHD Kamal. “Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions.” 2012. Web. 22 Sep 2017.

Vancouver:

Al-Hallak MK. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2017 Sep 22]. Available from: https://era.library.ualberta.ca/files/mk61rj53p.

Council of Science Editors:

Al-Hallak MK. Inhalable nanoparticles in lung cancer treatment; efficacy, safety, distribution and nanoparticle-macrophage interactions. [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/mk61rj53p


Universiteit Utrecht

6. Breeveld, A.R. Shape of the exposure response relation for crystalline silica and risk of lung cancer.

Degree: 2011, Universiteit Utrecht

 Crystalline silica exposure mostly occurs in occupational settings such as mining, construction, several industries (e.g. foundries) and agriculture. Occupational exposure to silica dust is known… (more)

Subjects/Keywords: crystalline silica; lung cancer

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APA (6th Edition):

Breeveld, A. R. (2011). Shape of the exposure response relation for crystalline silica and risk of lung cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/209135

Chicago Manual of Style (16th Edition):

Breeveld, A R. “Shape of the exposure response relation for crystalline silica and risk of lung cancer.” 2011. Masters Thesis, Universiteit Utrecht. Accessed September 22, 2017. http://dspace.library.uu.nl:8080/handle/1874/209135.

MLA Handbook (7th Edition):

Breeveld, A R. “Shape of the exposure response relation for crystalline silica and risk of lung cancer.” 2011. Web. 22 Sep 2017.

Vancouver:

Breeveld AR. Shape of the exposure response relation for crystalline silica and risk of lung cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2017 Sep 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/209135.

Council of Science Editors:

Breeveld AR. Shape of the exposure response relation for crystalline silica and risk of lung cancer. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/209135


Vanderbilt University

7. Samanta, Debangshu. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 Inactivating mutations in TGF-©¬ receptors and Smad signal transducers that contribute to resistance to TGF-©¬, are associated with only very small number of NSCLC. The… (more)

Subjects/Keywords: Smoking; Lung cancer; Smad3; Chemoresistance

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APA (6th Edition):

Samanta, D. (2012). Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;

Chicago Manual of Style (16th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed September 22, 2017. http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

MLA Handbook (7th Edition):

Samanta, Debangshu. “Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer.” 2012. Web. 22 Sep 2017.

Vancouver:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2017 Sep 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;.

Council of Science Editors:

Samanta D. Long-term smoking-mediated downregulation of Smad3 induces tumorigenicity and carboplatin resistance in non-small cell lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu/available/etd-04122012-114031/ ;


University of Miami

8. Milone, Mary Anne. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.

Degree: PhD, Nursing (Nursing), 2010, University of Miami

Lung cancer is the most common cause of cancer deaths worldwide. Hope is considered essential to life and has been positively associated with coping.… (more)

Subjects/Keywords: Cancer; Lung; Palliative; Hope

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APA (6th Edition):

Milone, M. A. (2010). The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. (Doctoral Dissertation). University of Miami. Retrieved from http://scholarlyrepository.miami.edu/oa_dissertations/409 ; etd-05122010-143404

Chicago Manual of Style (16th Edition):

Milone, Mary Anne. “The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.” 2010. Doctoral Dissertation, University of Miami. Accessed September 22, 2017. http://scholarlyrepository.miami.edu/oa_dissertations/409 ; etd-05122010-143404.

MLA Handbook (7th Edition):

Milone, Mary Anne. “The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer.” 2010. Web. 22 Sep 2017.

Vancouver:

Milone MA. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. [Internet] [Doctoral dissertation]. University of Miami; 2010. [cited 2017 Sep 22]. Available from: http://scholarlyrepository.miami.edu/oa_dissertations/409 ; etd-05122010-143404.

Council of Science Editors:

Milone MA. The Level of Hope in Patients Receiving Treatment for the Diagnosis of Lung Cancer. [Doctoral Dissertation]. University of Miami; 2010. Available from: http://scholarlyrepository.miami.edu/oa_dissertations/409 ; etd-05122010-143404


University of Dundee

9. Buchanan, Deans. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.

Degree: Thesis (M.D.), 2010, University of Dundee

Lung cancer has developed from a rare condition into the leading cause of cancerrelated death in the United Kingdom. Lung cancer patients face a disease… (more)

Subjects/Keywords: Lung cancer; Supportive care; Symptoms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buchanan, D. (2010). The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. (Doctoral Dissertation). University of Dundee. Retrieved from http://hdl.handle.net/10588/1047

Chicago Manual of Style (16th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Doctoral Dissertation, University of Dundee. Accessed September 22, 2017. http://hdl.handle.net/10588/1047.

MLA Handbook (7th Edition):

Buchanan, Deans. “The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs.” 2010. Web. 22 Sep 2017.

Vancouver:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Internet] [Doctoral dissertation]. University of Dundee; 2010. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/10588/1047.

Council of Science Editors:

Buchanan D. The clinical care of patients with lung cancer : identifying and supporting those with unmet care needs. [Doctoral Dissertation]. University of Dundee; 2010. Available from: http://hdl.handle.net/10588/1047

10. Santos, Abigail. Coffee and Tea Consumption and the Risk of Lung Cancer in a Population of Postmenopausal Women.

Degree: 2014, University of Massachusetts

Lung cancer has been the leading cause of cancer death in women for the past three decades. Although smoking is the most important risk… (more)

Subjects/Keywords: lung cancer; women; Women's Health

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APA (6th Edition):

Santos, A. (2014). Coffee and Tea Consumption and the Risk of Lung Cancer in a Population of Postmenopausal Women. (Thesis). University of Massachusetts. Retrieved from http://scholarworks.umass.edu/masters_theses_2/42

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Santos, Abigail. “Coffee and Tea Consumption and the Risk of Lung Cancer in a Population of Postmenopausal Women.” 2014. Thesis, University of Massachusetts. Accessed September 22, 2017. http://scholarworks.umass.edu/masters_theses_2/42.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Santos, Abigail. “Coffee and Tea Consumption and the Risk of Lung Cancer in a Population of Postmenopausal Women.” 2014. Web. 22 Sep 2017.

Vancouver:

Santos A. Coffee and Tea Consumption and the Risk of Lung Cancer in a Population of Postmenopausal Women. [Internet] [Thesis]. University of Massachusetts; 2014. [cited 2017 Sep 22]. Available from: http://scholarworks.umass.edu/masters_theses_2/42.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Santos A. Coffee and Tea Consumption and the Risk of Lung Cancer in a Population of Postmenopausal Women. [Thesis]. University of Massachusetts; 2014. Available from: http://scholarworks.umass.edu/masters_theses_2/42

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

11. Parker, Amelia Louise. The Role of betaIII-Tubulin in Non-Small Cell Lung Cancer.

Degree: Women's & Children's Health, 2016, University of New South Wales

 βIII-Tubulin is a neuronal microtubule protein that is aberrantly expressed in epithelial cancers including Non-small cell lung cancer (NSCLC). NSCLC survival rates are dismal and… (more)

Subjects/Keywords: Lung cancer; Tubulin; Microtubule

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APA (6th Edition):

Parker, A. L. (2016). The Role of betaIII-Tubulin in Non-Small Cell Lung Cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/56692

Chicago Manual of Style (16th Edition):

Parker, Amelia Louise. “The Role of betaIII-Tubulin in Non-Small Cell Lung Cancer.” 2016. Doctoral Dissertation, University of New South Wales. Accessed September 22, 2017. http://handle.unsw.edu.au/1959.4/56692.

MLA Handbook (7th Edition):

Parker, Amelia Louise. “The Role of betaIII-Tubulin in Non-Small Cell Lung Cancer.” 2016. Web. 22 Sep 2017.

Vancouver:

Parker AL. The Role of betaIII-Tubulin in Non-Small Cell Lung Cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2017 Sep 22]. Available from: http://handle.unsw.edu.au/1959.4/56692.

Council of Science Editors:

Parker AL. The Role of betaIII-Tubulin in Non-Small Cell Lung Cancer. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/56692


Penn State University

12. Crampsie, Melissa Ashley. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE.

Degree: PhD, Pharmacology, 2011, Penn State University

 Goals of Dissertation Research Lung cancer is currently the leading cause of cancer deaths among men and women and has a survival rate of only… (more)

Subjects/Keywords: ISC-4; lung cancer; chemoprevention

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APA (6th Edition):

Crampsie, M. A. (2011). INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/12391

Chicago Manual of Style (16th Edition):

Crampsie, Melissa Ashley. “INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE.” 2011. Doctoral Dissertation, Penn State University. Accessed September 22, 2017. https://etda.libraries.psu.edu/catalog/12391.

MLA Handbook (7th Edition):

Crampsie, Melissa Ashley. “INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE.” 2011. Web. 22 Sep 2017.

Vancouver:

Crampsie MA. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE. [Internet] [Doctoral dissertation]. Penn State University; 2011. [cited 2017 Sep 22]. Available from: https://etda.libraries.psu.edu/catalog/12391.

Council of Science Editors:

Crampsie MA. INVESTIGATION OF THE MECHANISM OF ACTION OF ISOTHIOCYANATE. [Doctoral Dissertation]. Penn State University; 2011. Available from: https://etda.libraries.psu.edu/catalog/12391


University of New South Wales

13. Luk, Peter Ping-Kit. The influence of the EGFR pathway in modulating the efficacy of gemcitabine.

Degree: Clinical School - St George Hospital, 2011, University of New South Wales

 Multiple anti-cancer drugs are often combined to increase their effectiveness. This study investigates the combination between an epidermal growth factor receptor (EGFR) inhibitor and gemcitabine,… (more)

Subjects/Keywords: Lung cancer; Gemcitabine; EGFR

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APA (6th Edition):

Luk, P. P. (2011). The influence of the EGFR pathway in modulating the efficacy of gemcitabine. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50384

Chicago Manual of Style (16th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Doctoral Dissertation, University of New South Wales. Accessed September 22, 2017. http://handle.unsw.edu.au/1959.4/50384.

MLA Handbook (7th Edition):

Luk, Peter Ping-Kit. “The influence of the EGFR pathway in modulating the efficacy of gemcitabine.” 2011. Web. 22 Sep 2017.

Vancouver:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2017 Sep 22]. Available from: http://handle.unsw.edu.au/1959.4/50384.

Council of Science Editors:

Luk PP. The influence of the EGFR pathway in modulating the efficacy of gemcitabine. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/50384


Queens University

14. Ahn, Joseph. FER PROTEIN-TYROSINE KINASE AS A POTENTIAL THERAPEUTIC TARGET IN LUNG CANCER .

Degree: Biochemistry, 2012, Queens University

 Fer is a ubiquitously expressed non-receptor protein-tyrosine kinase that regulates normal physiology through signaling in a variety of cell types. Fer signals downstream of growth… (more)

Subjects/Keywords: FER Kinase; Lung Cancer

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APA (6th Edition):

Ahn, J. (2012). FER PROTEIN-TYROSINE KINASE AS A POTENTIAL THERAPEUTIC TARGET IN LUNG CANCER . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahn, Joseph. “FER PROTEIN-TYROSINE KINASE AS A POTENTIAL THERAPEUTIC TARGET IN LUNG CANCER .” 2012. Thesis, Queens University. Accessed September 22, 2017. http://hdl.handle.net/1974/7441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahn, Joseph. “FER PROTEIN-TYROSINE KINASE AS A POTENTIAL THERAPEUTIC TARGET IN LUNG CANCER .” 2012. Web. 22 Sep 2017.

Vancouver:

Ahn J. FER PROTEIN-TYROSINE KINASE AS A POTENTIAL THERAPEUTIC TARGET IN LUNG CANCER . [Internet] [Thesis]. Queens University; 2012. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/1974/7441.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahn J. FER PROTEIN-TYROSINE KINASE AS A POTENTIAL THERAPEUTIC TARGET IN LUNG CANCER . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7441

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

15. Perez-Rogers, Joseph. Development of a minimally invasive molecular biomarker for early detection of lung cancer.

Degree: PhD, Bioinformatics GRS, 2017, Boston University

 The diagnostic evaluation of ever smokers with pulmonary nodules represents a growing clinical challenge due to the implementation of lung cancer screening. The high false-positive… (more)

Subjects/Keywords: Bioinformatics; Biomarker; Classifier; Lung cancer

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APA (6th Edition):

Perez-Rogers, J. (2017). Development of a minimally invasive molecular biomarker for early detection of lung cancer. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/21969

Chicago Manual of Style (16th Edition):

Perez-Rogers, Joseph. “Development of a minimally invasive molecular biomarker for early detection of lung cancer.” 2017. Doctoral Dissertation, Boston University. Accessed September 22, 2017. http://hdl.handle.net/2144/21969.

MLA Handbook (7th Edition):

Perez-Rogers, Joseph. “Development of a minimally invasive molecular biomarker for early detection of lung cancer.” 2017. Web. 22 Sep 2017.

Vancouver:

Perez-Rogers J. Development of a minimally invasive molecular biomarker for early detection of lung cancer. [Internet] [Doctoral dissertation]. Boston University; 2017. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2144/21969.

Council of Science Editors:

Perez-Rogers J. Development of a minimally invasive molecular biomarker for early detection of lung cancer. [Doctoral Dissertation]. Boston University; 2017. Available from: http://hdl.handle.net/2144/21969


Kansas State University

16. Uppalapati, Lakshmi. PEPTIDES AS THERAPEUTICS AND ACTIVE GENE DELIVERY VEHICLES FOR CANCER TREATMENT.

Degree: PhD, Department of Agronomy, 2015, Kansas State University

 Over the years proteins/peptides have evolved as promising therapeutic agents in the treatment of cancer. Considering the advantages of peptides such as their small size,… (more)

Subjects/Keywords: Cancer therapeutics; Nanoparticle; Intratracheal; Lung cancer

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APA (6th Edition):

Uppalapati, L. (2015). PEPTIDES AS THERAPEUTICS AND ACTIVE GENE DELIVERY VEHICLES FOR CANCER TREATMENT. (Doctoral Dissertation). Kansas State University. Retrieved from http://hdl.handle.net/2097/35231

Chicago Manual of Style (16th Edition):

Uppalapati, Lakshmi. “PEPTIDES AS THERAPEUTICS AND ACTIVE GENE DELIVERY VEHICLES FOR CANCER TREATMENT.” 2015. Doctoral Dissertation, Kansas State University. Accessed September 22, 2017. http://hdl.handle.net/2097/35231.

MLA Handbook (7th Edition):

Uppalapati, Lakshmi. “PEPTIDES AS THERAPEUTICS AND ACTIVE GENE DELIVERY VEHICLES FOR CANCER TREATMENT.” 2015. Web. 22 Sep 2017.

Vancouver:

Uppalapati L. PEPTIDES AS THERAPEUTICS AND ACTIVE GENE DELIVERY VEHICLES FOR CANCER TREATMENT. [Internet] [Doctoral dissertation]. Kansas State University; 2015. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2097/35231.

Council of Science Editors:

Uppalapati L. PEPTIDES AS THERAPEUTICS AND ACTIVE GENE DELIVERY VEHICLES FOR CANCER TREATMENT. [Doctoral Dissertation]. Kansas State University; 2015. Available from: http://hdl.handle.net/2097/35231


University of Louisville

17. Muench, Edward David, 1987-. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.

Degree: M. Eng., 2012, University of Louisville

 Vascular endothelial growth factor (VEGF), a commonly overexpressed oncogene in a variety of malignancies including non-small cell lung cancer (NSCLC), is a key regulator of… (more)

Subjects/Keywords: VEGF; Lung cancer; Quadruplex; Cancer; Oligonucleotide

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APA (6th Edition):

Muench, Edward David, 1. (2012). The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/1022 ; http://ir.library.louisville.edu/etd/1022

Chicago Manual of Style (16th Edition):

Muench, Edward David, 1987-. “The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.” 2012. Masters Thesis, University of Louisville. Accessed September 22, 2017. 10.18297/etd/1022 ; http://ir.library.louisville.edu/etd/1022.

MLA Handbook (7th Edition):

Muench, Edward David, 1987-. “The VEGF quadruplex-forming sequence inhibits lung cancer cell growth.” 2012. Web. 22 Sep 2017.

Vancouver:

Muench, Edward David 1. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. [Internet] [Masters thesis]. University of Louisville; 2012. [cited 2017 Sep 22]. Available from: 10.18297/etd/1022 ; http://ir.library.louisville.edu/etd/1022.

Council of Science Editors:

Muench, Edward David 1. The VEGF quadruplex-forming sequence inhibits lung cancer cell growth. [Masters Thesis]. University of Louisville; 2012. Available from: 10.18297/etd/1022 ; http://ir.library.louisville.edu/etd/1022


University of New South Wales

18. Jacob, Susannah Aachi. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.

Degree: Public Health & Community Medicine, 2010, University of New South Wales

 Introduction: Chemotherapy utilisation rates can vary widely between area health services, between states and internationally. Variation in chemotherapy utilisation rates is particularly marked in lung(more)

Subjects/Keywords: Lung cancer; Chemotherapy; Utilisation rates; Colorectal cancer

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APA (6th Edition):

Jacob, S. A. (2010). The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/50381

Chicago Manual of Style (16th Edition):

Jacob, Susannah Aachi. “The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.” 2010. Doctoral Dissertation, University of New South Wales. Accessed September 22, 2017. http://handle.unsw.edu.au/1959.4/50381.

MLA Handbook (7th Edition):

Jacob, Susannah Aachi. “The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery.” 2010. Web. 22 Sep 2017.

Vancouver:

Jacob SA. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. [Internet] [Doctoral dissertation]. University of New South Wales; 2010. [cited 2017 Sep 22]. Available from: http://handle.unsw.edu.au/1959.4/50381.

Council of Science Editors:

Jacob SA. The role of chemotherapy in the treatment of cancers of the lung, colon, rectum, brain, prostate, testes and leukaemia: estimation of the optimal chemotherapy utilisation rates as benchmarks for assessment of chemotherapy service delivery. [Doctoral Dissertation]. University of New South Wales; 2010. Available from: http://handle.unsw.edu.au/1959.4/50381

19. Michel, Noémie. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2013, Université François-Rabelais de Tours

Les kallicréines tissulaires humaines (KLK) ont récemment émergé comme une famille de protéases à serine pouvant jouer un rôle important dans la tumorigenèse. Le but… (more)

Subjects/Keywords: Kallicréines; Cancer du poumon; Kallikreins; Lung cancer

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APA (6th Edition):

Michel, N. (2013). Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2013TOUR3303

Chicago Manual of Style (16th Edition):

Michel, Noémie. “Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.” 2013. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed September 22, 2017. http://www.theses.fr/2013TOUR3303.

MLA Handbook (7th Edition):

Michel, Noémie. “Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology.” 2013. Web. 22 Sep 2017.

Vancouver:

Michel N. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2013. [cited 2017 Sep 22]. Available from: http://www.theses.fr/2013TOUR3303.

Council of Science Editors:

Michel N. Analyse de la contribution des kallicréines tissulaires 6 et 12 à la physiopathologie pulmonaire : Analysis of the contribution of tissue kallikreins 6 and 12 to lung pathophysiology. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2013. Available from: http://www.theses.fr/2013TOUR3303


Universiteit Utrecht

20. Waal, L.M. de. Treatment of subtypes in non-small cell lung cancer.

Degree: 2012, Universiteit Utrecht

Lung cancer accounts for the highest number of cancer related deaths among all cancer patients. It has been well established that exposure to tobacco significantly… (more)

Subjects/Keywords: lung cancer; treatment; targeted therapy; non-small cell lung cancer

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APA (6th Edition):

Waal, L. M. d. (2012). Treatment of subtypes in non-small cell lung cancer. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/241663

Chicago Manual of Style (16th Edition):

Waal, L M de. “Treatment of subtypes in non-small cell lung cancer.” 2012. Masters Thesis, Universiteit Utrecht. Accessed September 22, 2017. http://dspace.library.uu.nl:8080/handle/1874/241663.

MLA Handbook (7th Edition):

Waal, L M de. “Treatment of subtypes in non-small cell lung cancer.” 2012. Web. 22 Sep 2017.

Vancouver:

Waal LMd. Treatment of subtypes in non-small cell lung cancer. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2017 Sep 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/241663.

Council of Science Editors:

Waal LMd. Treatment of subtypes in non-small cell lung cancer. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/241663


University of Southern California

21. Chopra, Nikhil. CpG methylation profiling in lung cancer cell lines, tumors and non-tumors.

Degree: MS, Biochemistry & Molecular Biology, 2009, University of Southern California

 Cell lines are widely used for studying DNA methylation in cancer. Their main applications are in the development of biomarkers, in pharmacological studies and for… (more)

Subjects/Keywords: DNA methylation; lung cancer; cell lines; adenocarcinoma; epigenetics; methylation; CpG; cancer biomarkers; lung; cancer

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APA (6th Edition):

Chopra, N. (2009). CpG methylation profiling in lung cancer cell lines, tumors and non-tumors. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1677

Chicago Manual of Style (16th Edition):

Chopra, Nikhil. “CpG methylation profiling in lung cancer cell lines, tumors and non-tumors.” 2009. Masters Thesis, University of Southern California. Accessed September 22, 2017. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1677.

MLA Handbook (7th Edition):

Chopra, Nikhil. “CpG methylation profiling in lung cancer cell lines, tumors and non-tumors.” 2009. Web. 22 Sep 2017.

Vancouver:

Chopra N. CpG methylation profiling in lung cancer cell lines, tumors and non-tumors. [Internet] [Masters thesis]. University of Southern California; 2009. [cited 2017 Sep 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1677.

Council of Science Editors:

Chopra N. CpG methylation profiling in lung cancer cell lines, tumors and non-tumors. [Masters Thesis]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/221753/rec/1677


University of New South Wales

22. Nordman, Ina IC. Surrogate endpoints of survival in metastatic carcinoma.

Degree: Clinical School - St Vincent's Hospital, 2008, University of New South Wales

 In most randomised controlled trials (RCTs), a large number of patients need to be followed over many years, for the clinical benefit of the drug… (more)

Subjects/Keywords: metastatic cancer; surrogate endpoints; metastatic carcinoma; endpoints; survival; colorectal cancer; lung cancer; breast cancer; surrogate; non-small cell lung cancer

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APA (6th Edition):

Nordman, I. I. (2008). Surrogate endpoints of survival in metastatic carcinoma. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/42791

Chicago Manual of Style (16th Edition):

Nordman, Ina IC. “Surrogate endpoints of survival in metastatic carcinoma.” 2008. Masters Thesis, University of New South Wales. Accessed September 22, 2017. http://handle.unsw.edu.au/1959.4/42791.

MLA Handbook (7th Edition):

Nordman, Ina IC. “Surrogate endpoints of survival in metastatic carcinoma.” 2008. Web. 22 Sep 2017.

Vancouver:

Nordman II. Surrogate endpoints of survival in metastatic carcinoma. [Internet] [Masters thesis]. University of New South Wales; 2008. [cited 2017 Sep 22]. Available from: http://handle.unsw.edu.au/1959.4/42791.

Council of Science Editors:

Nordman II. Surrogate endpoints of survival in metastatic carcinoma. [Masters Thesis]. University of New South Wales; 2008. Available from: http://handle.unsw.edu.au/1959.4/42791

23. Gilbert, Jennifer L. The Role of Bone Morphogenetic Protein (BMP) Signalling in Lung Cancer and Lung Regeneration.

Degree: 2009, RIAN

 Bone morphogenetic proteins (BMPs) are members of the transforming growth factor beta (TGFβ) super-family of signalling molecules. BMPs play crucial roles in developing and adult… (more)

Subjects/Keywords: Biology; Bone Morphogenetic Protein; BMP; Lung Cancer; Lung Regeneration

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APA (6th Edition):

Gilbert, J. L. (2009). The Role of Bone Morphogenetic Protein (BMP) Signalling in Lung Cancer and Lung Regeneration. (Thesis). RIAN. Retrieved from http://eprints.maynoothuniversity.ie/2407/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gilbert, Jennifer L. “The Role of Bone Morphogenetic Protein (BMP) Signalling in Lung Cancer and Lung Regeneration.” 2009. Thesis, RIAN. Accessed September 22, 2017. http://eprints.maynoothuniversity.ie/2407/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gilbert, Jennifer L. “The Role of Bone Morphogenetic Protein (BMP) Signalling in Lung Cancer and Lung Regeneration.” 2009. Web. 22 Sep 2017.

Vancouver:

Gilbert JL. The Role of Bone Morphogenetic Protein (BMP) Signalling in Lung Cancer and Lung Regeneration. [Internet] [Thesis]. RIAN; 2009. [cited 2017 Sep 22]. Available from: http://eprints.maynoothuniversity.ie/2407/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gilbert JL. The Role of Bone Morphogenetic Protein (BMP) Signalling in Lung Cancer and Lung Regeneration. [Thesis]. RIAN; 2009. Available from: http://eprints.maynoothuniversity.ie/2407/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Yamazaki, Motohiko. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.

Degree: 博士(医学), 2015, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3976号. 学位記番号: 新大院博(医)甲第622号. 学位授与年月日: 平成27年3月23日

European Journal of Radiology. 2015, 84(1), 178-184.

Objectives: To assess the prognostic value of combined evaluation of… (more)

Subjects/Keywords: Chest CT imaging; lung adenocarcinoma; lung cancer; serum CEA levels; prognosis

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APA (6th Edition):

Yamazaki, M. (2015). A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/32260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamazaki, Motohiko. “A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.” 2015. Thesis, Niigata University / 新潟大学. Accessed September 22, 2017. http://hdl.handle.net/10191/32260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamazaki, Motohiko. “A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する.” 2015. Web. 22 Sep 2017.

Vancouver:

Yamazaki M. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. [Internet] [Thesis]. Niigata University / 新潟大学; 2015. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/10191/32260.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamazaki M. A combination of preoperative CT findings and postoperative serum CEA levels improves recurrence prediction for stage I lung adenocarcinoma : 術前CT所見と術後血清癌胎児性抗原 (CEA) の値を組み合わせると病理病期 I 期肺腺癌の再発予測能が向上する. [Thesis]. Niigata University / 新潟大学; 2015. Available from: http://hdl.handle.net/10191/32260

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

25. Carter, Cambron N. CAD system for lung nodule analysis.

Degree: M. Eng., 2012, University of Louisville

Lung cancer is the deadliest type of known cancer in the United States, claiming hundreds of thousands of lives each year. However, despite the high… (more)

Subjects/Keywords: Lung cancer; Lung nodule; CAD system; Nodule detection; Medical imaging

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APA (6th Edition):

Carter, C. N. (2012). CAD system for lung nodule analysis. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/218 ; http://ir.library.louisville.edu/etd/218

Chicago Manual of Style (16th Edition):

Carter, Cambron N. “CAD system for lung nodule analysis.” 2012. Masters Thesis, University of Louisville. Accessed September 22, 2017. 10.18297/etd/218 ; http://ir.library.louisville.edu/etd/218.

MLA Handbook (7th Edition):

Carter, Cambron N. “CAD system for lung nodule analysis.” 2012. Web. 22 Sep 2017.

Vancouver:

Carter CN. CAD system for lung nodule analysis. [Internet] [Masters thesis]. University of Louisville; 2012. [cited 2017 Sep 22]. Available from: 10.18297/etd/218 ; http://ir.library.louisville.edu/etd/218.

Council of Science Editors:

Carter CN. CAD system for lung nodule analysis. [Masters Thesis]. University of Louisville; 2012. Available from: 10.18297/etd/218 ; http://ir.library.louisville.edu/etd/218


University of Cincinnati

26. Zandvakili, Inuk. RhoA as a Potential Target in Lung Cancer.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2015, University of Cincinnati

 Many cancers are driven by oncogenic K-Ras, yet K-Ras has remained largely undruggable. In this dissertation we explore inhibiting K-Ras signaling by targeting downstream signaling… (more)

Subjects/Keywords: Surgery; RhoA; KRas; Rho GTPases; Lung Cancer; RhoC; Lung Adenocarcinoma

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APA (6th Edition):

Zandvakili, I. (2015). RhoA as a Potential Target in Lung Cancer. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196

Chicago Manual of Style (16th Edition):

Zandvakili, Inuk. “RhoA as a Potential Target in Lung Cancer.” 2015. Doctoral Dissertation, University of Cincinnati. Accessed September 22, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196.

MLA Handbook (7th Edition):

Zandvakili, Inuk. “RhoA as a Potential Target in Lung Cancer.” 2015. Web. 22 Sep 2017.

Vancouver:

Zandvakili I. RhoA as a Potential Target in Lung Cancer. [Internet] [Doctoral dissertation]. University of Cincinnati; 2015. [cited 2017 Sep 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196.

Council of Science Editors:

Zandvakili I. RhoA as a Potential Target in Lung Cancer. [Doctoral Dissertation]. University of Cincinnati; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196


Penn State University

27. Miller, Joshua Christopher. ADENYLOSUCCINATE SYNTHETASE 1:.

Degree: PhD, Pharmacology, 2010, Penn State University

 Tobacco smoke consists of numerous carcinogens whose effect on lung tumor development includes the induction of mutations in key metabolic genes as well as the… (more)

Subjects/Keywords: lung cancer; lung adenocarcinoma; metabolic reprogramming; adenylosuccinate synthetase; , novel deletion

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APA (6th Edition):

Miller, J. C. (2010). ADENYLOSUCCINATE SYNTHETASE 1:. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11282

Chicago Manual of Style (16th Edition):

Miller, Joshua Christopher. “ADENYLOSUCCINATE SYNTHETASE 1:.” 2010. Doctoral Dissertation, Penn State University. Accessed September 22, 2017. https://etda.libraries.psu.edu/catalog/11282.

MLA Handbook (7th Edition):

Miller, Joshua Christopher. “ADENYLOSUCCINATE SYNTHETASE 1:.” 2010. Web. 22 Sep 2017.

Vancouver:

Miller JC. ADENYLOSUCCINATE SYNTHETASE 1:. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2017 Sep 22]. Available from: https://etda.libraries.psu.edu/catalog/11282.

Council of Science Editors:

Miller JC. ADENYLOSUCCINATE SYNTHETASE 1:. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11282


Kyoto University / 京都大学

28. Nakaoku, Takashi. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.

Degree: 博士(医学), 2016, Kyoto University / 京都大学

リポジトリの登録にあたっては、Peer reviewされた最終版のみ可能であり、その際には下記の出版社のウェブサイトのアドレスを記載することが求められる。当該論文は2014年6月の出版であり、12ヶ月を経過していることから、公開には差し支えはない。http://clincancerres.aacrjournals.org/content/20/12/3087.full

新制・課程博士

甲第19617号

医博第4124号

Subjects/Keywords: Lung Cancer; Imvasive mucinous lung adenocarcinoma; Gene rearrangement; Targeted therapy; NRG1

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APA (6th Edition):

Nakaoku, T. (2016). Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nakaoku, Takashi. “Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.” 2016. Thesis, Kyoto University / 京都大学. Accessed September 22, 2017. http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nakaoku, Takashi. “Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常.” 2016. Web. 22 Sep 2017.

Vancouver:

Nakaoku T. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. [Internet] [Thesis]. Kyoto University / 京都大学; 2016. [cited 2017 Sep 22]. Available from: http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nakaoku T. Druggable Oncogene Fusions in Invasive Mucinous Lung Adenocarcinoma : 浸潤性粘液肺腺がんの遺伝子異常. [Thesis]. Kyoto University / 京都大学; 2016. Available from: http://hdl.handle.net/2433/215443 ; http://dx.doi.org/10.14989/doctor.k19617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Utah State University

29. Ward, Sherissa A. The Effect of Polydimethylsiloxane Substrate Modification on A549 Human Epithelial Lung Cancer Cell Morphology and Biomechanics.

Degree: MS, Biological and Irrigation Engineering, 2015, Utah State University

  In this thesis the effect of mechanical stimuli on A549 lung cancer cells is studied. Modifications of polydimethylsiloxane (PDMS) surfaces are employed to alter… (more)

Subjects/Keywords: Polydimethylsiloxane substrate; modification; A549; Lung Cancer; Engineering

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APA (6th Edition):

Ward, S. A. (2015). The Effect of Polydimethylsiloxane Substrate Modification on A549 Human Epithelial Lung Cancer Cell Morphology and Biomechanics. (Masters Thesis). Utah State University. Retrieved from http://digitalcommons.usu.edu/etd/4161

Chicago Manual of Style (16th Edition):

Ward, Sherissa A. “The Effect of Polydimethylsiloxane Substrate Modification on A549 Human Epithelial Lung Cancer Cell Morphology and Biomechanics.” 2015. Masters Thesis, Utah State University. Accessed September 22, 2017. http://digitalcommons.usu.edu/etd/4161.

MLA Handbook (7th Edition):

Ward, Sherissa A. “The Effect of Polydimethylsiloxane Substrate Modification on A549 Human Epithelial Lung Cancer Cell Morphology and Biomechanics.” 2015. Web. 22 Sep 2017.

Vancouver:

Ward SA. The Effect of Polydimethylsiloxane Substrate Modification on A549 Human Epithelial Lung Cancer Cell Morphology and Biomechanics. [Internet] [Masters thesis]. Utah State University; 2015. [cited 2017 Sep 22]. Available from: http://digitalcommons.usu.edu/etd/4161.

Council of Science Editors:

Ward SA. The Effect of Polydimethylsiloxane Substrate Modification on A549 Human Epithelial Lung Cancer Cell Morphology and Biomechanics. [Masters Thesis]. Utah State University; 2015. Available from: http://digitalcommons.usu.edu/etd/4161


University of Alberta

30. Sarfraz, Muhammad K. Nano-carrier Macrophage Interaction: Role of Secondary Toxicity.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2016, University of Alberta

 Macrophages are the primary cells of innate immunity that provide the first line of defence against any external stimuli. Their phagocytic properties enable them to… (more)

Subjects/Keywords: Secondary Toxicity; inflammation; nanoparticles; micelles; lung cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sarfraz, M. K. (2016). Nano-carrier Macrophage Interaction: Role of Secondary Toxicity. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/cfx719m630

Chicago Manual of Style (16th Edition):

Sarfraz, Muhammad K. “Nano-carrier Macrophage Interaction: Role of Secondary Toxicity.” 2016. Doctoral Dissertation, University of Alberta. Accessed September 22, 2017. https://era.library.ualberta.ca/files/cfx719m630.

MLA Handbook (7th Edition):

Sarfraz, Muhammad K. “Nano-carrier Macrophage Interaction: Role of Secondary Toxicity.” 2016. Web. 22 Sep 2017.

Vancouver:

Sarfraz MK. Nano-carrier Macrophage Interaction: Role of Secondary Toxicity. [Internet] [Doctoral dissertation]. University of Alberta; 2016. [cited 2017 Sep 22]. Available from: https://era.library.ualberta.ca/files/cfx719m630.

Council of Science Editors:

Sarfraz MK. Nano-carrier Macrophage Interaction: Role of Secondary Toxicity. [Doctoral Dissertation]. University of Alberta; 2016. Available from: https://era.library.ualberta.ca/files/cfx719m630

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