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You searched for subject:(liver damage). Showing records 1 – 30 of 33 total matches.

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University of Hong Kong

1. Yang, Sitian. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

 Hepatocellular carcinoma (HCC) is highly resistant to chemotherapy and one of the most common causes of cancer related death worldwide. Chemoresistance in HCC leads to… (more)

Subjects/Keywords: Liver - Cancer - Chemotherapy; DNA damage

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APA (6th Edition):

Yang, S. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076

Chicago Manual of Style (16th Edition):

Yang, Sitian. “Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed June 15, 2019. Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076.

MLA Handbook (7th Edition):

Yang, Sitian. “Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.” 2015. Web. 15 Jun 2019.

Vancouver:

Yang S. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jun 15]. Available from: Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076.

Council of Science Editors:

Yang S. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Yang, S. [楊斯恬]. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5435663 ; http://dx.doi.org/10.5353/th_b5435663 ; http://hdl.handle.net/10722/224076


Mississippi State University

2. Chen, Joseph. INVESTIGATION OF HIGH RATE MECHANICAL PROPERTIES AND DAMAGE EVOLUTION IN PORCINE LIVER TISSUE.

Degree: MS, Agricultural and Biological Engineering, 2010, Mississippi State University

  Each year, 6.4 million automobile accidents account for approximately 40,000 deaths in the United States. With increasing requirements for automobile safety, computational models capable… (more)

Subjects/Keywords: Damage; High-Rate Biomechanics; Liver

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APA (6th Edition):

Chen, J. (2010). INVESTIGATION OF HIGH RATE MECHANICAL PROPERTIES AND DAMAGE EVOLUTION IN PORCINE LIVER TISSUE. (Masters Thesis). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-06302010-125836/ ;

Chicago Manual of Style (16th Edition):

Chen, Joseph. “INVESTIGATION OF HIGH RATE MECHANICAL PROPERTIES AND DAMAGE EVOLUTION IN PORCINE LIVER TISSUE.” 2010. Masters Thesis, Mississippi State University. Accessed June 15, 2019. http://sun.library.msstate.edu/ETD-db/theses/available/etd-06302010-125836/ ;.

MLA Handbook (7th Edition):

Chen, Joseph. “INVESTIGATION OF HIGH RATE MECHANICAL PROPERTIES AND DAMAGE EVOLUTION IN PORCINE LIVER TISSUE.” 2010. Web. 15 Jun 2019.

Vancouver:

Chen J. INVESTIGATION OF HIGH RATE MECHANICAL PROPERTIES AND DAMAGE EVOLUTION IN PORCINE LIVER TISSUE. [Internet] [Masters thesis]. Mississippi State University; 2010. [cited 2019 Jun 15]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-06302010-125836/ ;.

Council of Science Editors:

Chen J. INVESTIGATION OF HIGH RATE MECHANICAL PROPERTIES AND DAMAGE EVOLUTION IN PORCINE LIVER TISSUE. [Masters Thesis]. Mississippi State University; 2010. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-06302010-125836/ ;

3. Rasal, Vijaykumar P. Evaluation of hepatoprotective activity by some flavonoids on lysosomal enzymes activity in carbon tetrachloride vapour induced liver damage in rats; -.

Degree: Pharmacy, 2012, KLE University

None

Bibliography p.118-136

Advisors/Committee Members: Purnima Ashok.

Subjects/Keywords: Pharmacy; liver damage

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APA (6th Edition):

Rasal, V. P. (2012). Evaluation of hepatoprotective activity by some flavonoids on lysosomal enzymes activity in carbon tetrachloride vapour induced liver damage in rats; -. (Thesis). KLE University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/5928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rasal, Vijaykumar P. “Evaluation of hepatoprotective activity by some flavonoids on lysosomal enzymes activity in carbon tetrachloride vapour induced liver damage in rats; -.” 2012. Thesis, KLE University. Accessed June 15, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/5928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rasal, Vijaykumar P. “Evaluation of hepatoprotective activity by some flavonoids on lysosomal enzymes activity in carbon tetrachloride vapour induced liver damage in rats; -.” 2012. Web. 15 Jun 2019.

Vancouver:

Rasal VP. Evaluation of hepatoprotective activity by some flavonoids on lysosomal enzymes activity in carbon tetrachloride vapour induced liver damage in rats; -. [Internet] [Thesis]. KLE University; 2012. [cited 2019 Jun 15]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/5928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rasal VP. Evaluation of hepatoprotective activity by some flavonoids on lysosomal enzymes activity in carbon tetrachloride vapour induced liver damage in rats; -. [Thesis]. KLE University; 2012. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/5928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

4. Bona, Silvia Regina. Melatonina protege o fígado em um modelo experimental de cirrose.

Degree: 2014, Universidade do Rio Grande do Sul

Base teórica: As doenças hepáticas representam um grande problema de saúde pública, sendo responsáveis por um considerável número de atendimentos e internações hospitalares, com crescente… (more)

Subjects/Keywords: Antioxidantes; Oxidative damage; Cirrose hepática; Antioxidants; Estresse oxidativo; Liver cirrhosis

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APA (6th Edition):

Bona, S. R. (2014). Melatonina protege o fígado em um modelo experimental de cirrose. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/129635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bona, Silvia Regina. “Melatonina protege o fígado em um modelo experimental de cirrose.” 2014. Thesis, Universidade do Rio Grande do Sul. Accessed June 15, 2019. http://hdl.handle.net/10183/129635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bona, Silvia Regina. “Melatonina protege o fígado em um modelo experimental de cirrose.” 2014. Web. 15 Jun 2019.

Vancouver:

Bona SR. Melatonina protege o fígado em um modelo experimental de cirrose. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2014. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/10183/129635.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bona SR. Melatonina protege o fígado em um modelo experimental de cirrose. [Thesis]. Universidade do Rio Grande do Sul; 2014. Available from: http://hdl.handle.net/10183/129635

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

5. Yang, Sitian. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

abstract

Surgery

Doctoral

Doctor of Philosophy

Subjects/Keywords: Liver - Cancer - Chemotherapy; DNA damage

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, S. (2015). Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/209505

Chicago Manual of Style (16th Edition):

Yang, Sitian. “Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed June 15, 2019. http://hdl.handle.net/10722/209505.

MLA Handbook (7th Edition):

Yang, Sitian. “Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma.” 2015. Web. 15 Jun 2019.

Vancouver:

Yang S. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/10722/209505.

Council of Science Editors:

Yang S. Novel role of drug-induced non homologous end joining factor 1 in the chemoresistance of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/209505


Australian National University

6. Arfianti. Mechanisms by which obesity accelerates hepatocarcinogenesis: molecular studies in foz/foz mice .

Degree: 2016, Australian National University

 Hepatocellular carcinoma (HCC) is the second most common fatal cancer worldwide and the fastest increasing cause of cancer death in Australia. Epidemiological data indicate that… (more)

Subjects/Keywords: Diabetes; DNA damage; exercise; JNK; non-alcoholic; fatty liver disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arfianti. (2016). Mechanisms by which obesity accelerates hepatocarcinogenesis: molecular studies in foz/foz mice . (Thesis). Australian National University. Retrieved from http://hdl.handle.net/1885/114516

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arfianti. “Mechanisms by which obesity accelerates hepatocarcinogenesis: molecular studies in foz/foz mice .” 2016. Thesis, Australian National University. Accessed June 15, 2019. http://hdl.handle.net/1885/114516.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arfianti. “Mechanisms by which obesity accelerates hepatocarcinogenesis: molecular studies in foz/foz mice .” 2016. Web. 15 Jun 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Arfianti. Mechanisms by which obesity accelerates hepatocarcinogenesis: molecular studies in foz/foz mice . [Internet] [Thesis]. Australian National University; 2016. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/1885/114516.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arfianti. Mechanisms by which obesity accelerates hepatocarcinogenesis: molecular studies in foz/foz mice . [Thesis]. Australian National University; 2016. Available from: http://hdl.handle.net/1885/114516

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

7. Kheradpezhouh. Ehsan. The role of TRPM2 channels in oxidative stress-induced liver damage.

Degree: 2015, University of Adelaide

 The increased production of highly reactive oxygen and nitrogen species plays a significant role in development of a number of liver disorders associated with hepatocellular… (more)

Subjects/Keywords: TRPM2 channel; Acetaminophen toxicity; liver damage; oxidative stress; calcium; curcumin; chlorpromazine

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APA (6th Edition):

Ehsan, K. (2015). The role of TRPM2 channels in oxidative stress-induced liver damage. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/92813

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ehsan, Kheradpezhouh.. “The role of TRPM2 channels in oxidative stress-induced liver damage.” 2015. Thesis, University of Adelaide. Accessed June 15, 2019. http://hdl.handle.net/2440/92813.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ehsan, Kheradpezhouh.. “The role of TRPM2 channels in oxidative stress-induced liver damage.” 2015. Web. 15 Jun 2019.

Vancouver:

Ehsan K. The role of TRPM2 channels in oxidative stress-induced liver damage. [Internet] [Thesis]. University of Adelaide; 2015. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/2440/92813.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ehsan K. The role of TRPM2 channels in oxidative stress-induced liver damage. [Thesis]. University of Adelaide; 2015. Available from: http://hdl.handle.net/2440/92813

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

8. Magimaidas, Andrew. The Role of the Stress Response Gene Gadd45b in Senescence.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The Gadd45 family of proteins (Gadd45a, Gadd45b, and Gadd45g) has been shown to act as stress sensors in response to various… (more)

Subjects/Keywords: Molecular biology; Genetics;

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APA (6th Edition):

Magimaidas, A. (2015). The Role of the Stress Response Gene Gadd45b in Senescence. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,376546

Chicago Manual of Style (16th Edition):

Magimaidas, Andrew. “The Role of the Stress Response Gene Gadd45b in Senescence.” 2015. Doctoral Dissertation, Temple University. Accessed June 15, 2019. http://digital.library.temple.edu/u?/p245801coll10,376546.

MLA Handbook (7th Edition):

Magimaidas, Andrew. “The Role of the Stress Response Gene Gadd45b in Senescence.” 2015. Web. 15 Jun 2019.

Vancouver:

Magimaidas A. The Role of the Stress Response Gene Gadd45b in Senescence. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Jun 15]. Available from: http://digital.library.temple.edu/u?/p245801coll10,376546.

Council of Science Editors:

Magimaidas A. The Role of the Stress Response Gene Gadd45b in Senescence. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,376546

9. Schaftenaar, F.H. The role of tissue damage in hepatic cancer development and outgrowth.

Degree: 2012, Universiteit Utrecht

 Hepatocellular cancer (HCC) is the most common primary hepatic cancer and is overall the 5th most common cancer with 748,300 new liver cancer cases in… (more)

Subjects/Keywords: liver; hepatic malignancies; liver injury; tissue damage; tumor outgrowth; carcinogenesis

damage to the liver, it is clinically relevant to study acute liver damage and their effects on… …stellate cell correlates with severe liver damage and abnormal liver regeneration in… …needed to develop HCC. This experiment again shows the importance of liver injury to the… …x28;Unpublished results, de Bruijn et al.) Acute liver injury affecting preexisting… …tumors So, acute liver injury is not capable of inducing HCC in healthy wt livers of mice. In… 

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APA (6th Edition):

Schaftenaar, F. H. (2012). The role of tissue damage in hepatic cancer development and outgrowth. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/259118

Chicago Manual of Style (16th Edition):

Schaftenaar, F H. “The role of tissue damage in hepatic cancer development and outgrowth.” 2012. Masters Thesis, Universiteit Utrecht. Accessed June 15, 2019. http://dspace.library.uu.nl:8080/handle/1874/259118.

MLA Handbook (7th Edition):

Schaftenaar, F H. “The role of tissue damage in hepatic cancer development and outgrowth.” 2012. Web. 15 Jun 2019.

Vancouver:

Schaftenaar FH. The role of tissue damage in hepatic cancer development and outgrowth. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2019 Jun 15]. Available from: http://dspace.library.uu.nl:8080/handle/1874/259118.

Council of Science Editors:

Schaftenaar FH. The role of tissue damage in hepatic cancer development and outgrowth. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/259118

10. Usha V. Hepatoprotective activity of desmodium gangeticum in paracetamol induced liver damage;.

Degree: Biochemistry, 2015, Periyar University

newline

References P147-197 with Appendices

Advisors/Committee Members: Suriyavathana M.

Subjects/Keywords: Desmodium Gangeticum; Hepatoprotective Activity; Paracetamol Induced Liver Damage

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APA (6th Edition):

V, U. (2015). Hepatoprotective activity of desmodium gangeticum in paracetamol induced liver damage;. (Thesis). Periyar University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/51143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

V, Usha. “Hepatoprotective activity of desmodium gangeticum in paracetamol induced liver damage;.” 2015. Thesis, Periyar University. Accessed June 15, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/51143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

V, Usha. “Hepatoprotective activity of desmodium gangeticum in paracetamol induced liver damage;.” 2015. Web. 15 Jun 2019.

Vancouver:

V U. Hepatoprotective activity of desmodium gangeticum in paracetamol induced liver damage;. [Internet] [Thesis]. Periyar University; 2015. [cited 2019 Jun 15]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/51143.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

V U. Hepatoprotective activity of desmodium gangeticum in paracetamol induced liver damage;. [Thesis]. Periyar University; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/51143

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Gnanadesigam, M. Studies on the hepatoprotective and antioxidant activity of chosen marine halophytes for the management of liver damage;.

Degree: 2015, Alagappa University

None

Advisors/Committee Members: Ravikumar, S.

Subjects/Keywords: antioxidant activity; hepatoprotective activity; liver damage; marine halophytes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gnanadesigam, M. (2015). Studies on the hepatoprotective and antioxidant activity of chosen marine halophytes for the management of liver damage;. (Thesis). Alagappa University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/54300

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gnanadesigam, M. “Studies on the hepatoprotective and antioxidant activity of chosen marine halophytes for the management of liver damage;.” 2015. Thesis, Alagappa University. Accessed June 15, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/54300.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gnanadesigam, M. “Studies on the hepatoprotective and antioxidant activity of chosen marine halophytes for the management of liver damage;.” 2015. Web. 15 Jun 2019.

Vancouver:

Gnanadesigam M. Studies on the hepatoprotective and antioxidant activity of chosen marine halophytes for the management of liver damage;. [Internet] [Thesis]. Alagappa University; 2015. [cited 2019 Jun 15]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/54300.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gnanadesigam M. Studies on the hepatoprotective and antioxidant activity of chosen marine halophytes for the management of liver damage;. [Thesis]. Alagappa University; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/54300

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kashmir

12. Nahida Tabassum. Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;.

Degree: 2015, University of Kashmir

newline

Advisors/Committee Members: Shah, M.Y. and Qazi, M.A. and Shah, Azra.

Subjects/Keywords:

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APA (6th Edition):

Tabassum, N. (2015). Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;. (Thesis). University of Kashmir. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/43501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tabassum, Nahida. “Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;.” 2015. Thesis, University of Kashmir. Accessed June 15, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/43501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tabassum, Nahida. “Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;.” 2015. Web. 15 Jun 2019.

Vancouver:

Tabassum N. Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;. [Internet] [Thesis]. University of Kashmir; 2015. [cited 2019 Jun 15]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/43501.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tabassum N. Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;. [Thesis]. University of Kashmir; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/43501

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kashmir

13. Nahida Tabassum. Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;.

Degree: 2015, University of Kashmir

newline

Advisors/Committee Members: Shah, M.Y. and Qazi, M.A. and Shah, Azra.

Subjects/Keywords:

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APA (6th Edition):

Tabassum, N. (2015). Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;. (Thesis). University of Kashmir. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/43505

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tabassum, Nahida. “Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;.” 2015. Thesis, University of Kashmir. Accessed June 15, 2019. http://shodhganga.inflibnet.ac.in/handle/10603/43505.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tabassum, Nahida. “Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;.” 2015. Web. 15 Jun 2019.

Vancouver:

Tabassum N. Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;. [Internet] [Thesis]. University of Kashmir; 2015. [cited 2019 Jun 15]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/43505.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tabassum N. Study of prophylactic and curative effect of cichorium intybus linn and taraxacum officinale webber against Hepatocellular damage induced in experimental animals;. [Thesis]. University of Kashmir; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/43505

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

14. Poole, Brian Douglas. PARVOVIRUS B19 INFECTION OF HEPATOCYTES AND INDUCTION OF APOPTOSIS.

Degree: PhD, Integrative Biosciences, 2004, Penn State University

 ABSTRACT Parvovirus B19 persists in multiple tissues and has been implicated in a variety of diseases including acute fulminant liver failure (AFLF). Despite multiple reports… (more)

Subjects/Keywords: parvovirus B19; Apoptosis; Acute fulminant liver failure; DNA damage

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APA (6th Edition):

Poole, B. D. (2004). PARVOVIRUS B19 INFECTION OF HEPATOCYTES AND INDUCTION OF APOPTOSIS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/6347

Chicago Manual of Style (16th Edition):

Poole, Brian Douglas. “PARVOVIRUS B19 INFECTION OF HEPATOCYTES AND INDUCTION OF APOPTOSIS.” 2004. Doctoral Dissertation, Penn State University. Accessed June 15, 2019. https://etda.libraries.psu.edu/catalog/6347.

MLA Handbook (7th Edition):

Poole, Brian Douglas. “PARVOVIRUS B19 INFECTION OF HEPATOCYTES AND INDUCTION OF APOPTOSIS.” 2004. Web. 15 Jun 2019.

Vancouver:

Poole BD. PARVOVIRUS B19 INFECTION OF HEPATOCYTES AND INDUCTION OF APOPTOSIS. [Internet] [Doctoral dissertation]. Penn State University; 2004. [cited 2019 Jun 15]. Available from: https://etda.libraries.psu.edu/catalog/6347.

Council of Science Editors:

Poole BD. PARVOVIRUS B19 INFECTION OF HEPATOCYTES AND INDUCTION OF APOPTOSIS. [Doctoral Dissertation]. Penn State University; 2004. Available from: https://etda.libraries.psu.edu/catalog/6347


University of Melbourne

15. GAMON, LUKE. Oxidative and chemical modification of amino acids by nitrogen dioxide, ozone and the reactive paracetamol metabolite NAPQI.

Degree: 2016, University of Melbourne

 Oxidative damage has been implicated in a wide range of diseases including cardiovascular disease, diabetes, asthma, cancer and atherosclerosis. While this damage is typically caused… (more)

Subjects/Keywords: radical chemistry; chemistry; science; peptides; pollution; respiratory disease; oxidative damage; drugs; drug metabolites; liver damage; paracetamol; acetaminophen; nitrogen dioxide; no2; no3; o3; ozone, nitrate, nitrate radical, atmospheric chemistry

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APA (6th Edition):

GAMON, L. (2016). Oxidative and chemical modification of amino acids by nitrogen dioxide, ozone and the reactive paracetamol metabolite NAPQI. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/111968

Chicago Manual of Style (16th Edition):

GAMON, LUKE. “Oxidative and chemical modification of amino acids by nitrogen dioxide, ozone and the reactive paracetamol metabolite NAPQI.” 2016. Doctoral Dissertation, University of Melbourne. Accessed June 15, 2019. http://hdl.handle.net/11343/111968.

MLA Handbook (7th Edition):

GAMON, LUKE. “Oxidative and chemical modification of amino acids by nitrogen dioxide, ozone and the reactive paracetamol metabolite NAPQI.” 2016. Web. 15 Jun 2019.

Vancouver:

GAMON L. Oxidative and chemical modification of amino acids by nitrogen dioxide, ozone and the reactive paracetamol metabolite NAPQI. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/11343/111968.

Council of Science Editors:

GAMON L. Oxidative and chemical modification of amino acids by nitrogen dioxide, ozone and the reactive paracetamol metabolite NAPQI. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/111968


Universidade Federal de Santa Maria

16. Lysandro Pinto Borges. EFEITOS DA ADMINISTRAÇÃO DO DISSELENETO DE DIFENILA SOBRE O DANO HEPÁTICO INDUZIDO POR 2-NITROPROPANO, CÁDMIO E TETRACLORETO DE CARBONO.

Degree: 2008, Universidade Federal de Santa Maria

 O fígado apresenta extraordinária pluralidade funcional, destacando-se no controle de produção de energia, defesa imunológica e reserva volêmica. No meio ambiente e ocupacionalmente, o ser… (more)

Subjects/Keywords: 2-nitropropano; cádmio; tetracloreto de carbono; BIOQUIMICA; disseleneto de difenila; selênio; dano hepático; carbon tetrachloride; selenium; diphenyl diselenide; liver damage; cadmium; 2-Nitropropane

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APA (6th Edition):

Borges, L. P. (2008). EFEITOS DA ADMINISTRAÇÃO DO DISSELENETO DE DIFENILA SOBRE O DANO HEPÁTICO INDUZIDO POR 2-NITROPROPANO, CÁDMIO E TETRACLORETO DE CARBONO. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=1936

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Borges, Lysandro Pinto. “EFEITOS DA ADMINISTRAÇÃO DO DISSELENETO DE DIFENILA SOBRE O DANO HEPÁTICO INDUZIDO POR 2-NITROPROPANO, CÁDMIO E TETRACLORETO DE CARBONO.” 2008. Thesis, Universidade Federal de Santa Maria. Accessed June 15, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=1936.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Borges, Lysandro Pinto. “EFEITOS DA ADMINISTRAÇÃO DO DISSELENETO DE DIFENILA SOBRE O DANO HEPÁTICO INDUZIDO POR 2-NITROPROPANO, CÁDMIO E TETRACLORETO DE CARBONO.” 2008. Web. 15 Jun 2019.

Vancouver:

Borges LP. EFEITOS DA ADMINISTRAÇÃO DO DISSELENETO DE DIFENILA SOBRE O DANO HEPÁTICO INDUZIDO POR 2-NITROPROPANO, CÁDMIO E TETRACLORETO DE CARBONO. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2008. [cited 2019 Jun 15]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=1936.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Borges LP. EFEITOS DA ADMINISTRAÇÃO DO DISSELENETO DE DIFENILA SOBRE O DANO HEPÁTICO INDUZIDO POR 2-NITROPROPANO, CÁDMIO E TETRACLORETO DE CARBONO. [Thesis]. Universidade Federal de Santa Maria; 2008. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=1936

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Federal de Santa Maria

17. Ethel Antunes Wilhelm. EFEITO HEPATOPROTETOR CAUSADO PELO 3-ALQUINIL SELENOFENO CONTRA O DANO OXIDATIVO INDUZIDO POR AGENTES QUÍMICOS EM RATOS.

Degree: 2009, Universidade Federal de Santa Maria

 O fígado apresenta extraordinária pluralidade funcional, destacando-se no controle de produção de energia, defesa imunológica e reserva volêmica. No meio ambiente e ocupacionalmente, o ser… (more)

Subjects/Keywords: tetracloreto de carbono; 3-alquinil selenofeno; selênio; dano hepático; BIOQUIMICA; selenium; liver damage; 3-alkynyl selenophene; carbon tetrachloride; 2-nitropropane; 2-nitropropano

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APA (6th Edition):

Wilhelm, E. A. (2009). EFEITO HEPATOPROTETOR CAUSADO PELO 3-ALQUINIL SELENOFENO CONTRA O DANO OXIDATIVO INDUZIDO POR AGENTES QUÍMICOS EM RATOS. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=2391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wilhelm, Ethel Antunes. “EFEITO HEPATOPROTETOR CAUSADO PELO 3-ALQUINIL SELENOFENO CONTRA O DANO OXIDATIVO INDUZIDO POR AGENTES QUÍMICOS EM RATOS.” 2009. Thesis, Universidade Federal de Santa Maria. Accessed June 15, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=2391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wilhelm, Ethel Antunes. “EFEITO HEPATOPROTETOR CAUSADO PELO 3-ALQUINIL SELENOFENO CONTRA O DANO OXIDATIVO INDUZIDO POR AGENTES QUÍMICOS EM RATOS.” 2009. Web. 15 Jun 2019.

Vancouver:

Wilhelm EA. EFEITO HEPATOPROTETOR CAUSADO PELO 3-ALQUINIL SELENOFENO CONTRA O DANO OXIDATIVO INDUZIDO POR AGENTES QUÍMICOS EM RATOS. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2009. [cited 2019 Jun 15]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=2391.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wilhelm EA. EFEITO HEPATOPROTETOR CAUSADO PELO 3-ALQUINIL SELENOFENO CONTRA O DANO OXIDATIVO INDUZIDO POR AGENTES QUÍMICOS EM RATOS. [Thesis]. Universidade Federal de Santa Maria; 2009. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=2391

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Conte, Cecile. Comportement mécanique du foie en contexte traumatique : rupture et endommagement des tissus : Posterior urethral injuries associated with motorcycle pelvic injuries in adolescent. : Computerized finite element model of pelvic and urethra and medical applications.

Degree: Docteur es, Sciences du mouvement humain, 2012, Aix Marseille Université

Chaque année en France, on dénombre environ 2000 cas de lésions de la région abdominale lors d'accidents de la route. Ces lésions sont très souvent… (more)

Subjects/Keywords: Biomécanique; Compression; Visco-hyperélasticité; Endommagement; Rupture; Éléments finis; Foie; Tissus mous; Mécanismes lésionnels; Biomechanics; Compression; Visco-hyperelasticity; Damage; Failure; Finite element; Liver; Soft tissues; Injury mechanisms

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APA (6th Edition):

Conte, C. (2012). Comportement mécanique du foie en contexte traumatique : rupture et endommagement des tissus : Posterior urethral injuries associated with motorcycle pelvic injuries in adolescent. : Computerized finite element model of pelvic and urethra and medical applications. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2012AIXM4103

Chicago Manual of Style (16th Edition):

Conte, Cecile. “Comportement mécanique du foie en contexte traumatique : rupture et endommagement des tissus : Posterior urethral injuries associated with motorcycle pelvic injuries in adolescent. : Computerized finite element model of pelvic and urethra and medical applications.” 2012. Doctoral Dissertation, Aix Marseille Université. Accessed June 15, 2019. http://www.theses.fr/2012AIXM4103.

MLA Handbook (7th Edition):

Conte, Cecile. “Comportement mécanique du foie en contexte traumatique : rupture et endommagement des tissus : Posterior urethral injuries associated with motorcycle pelvic injuries in adolescent. : Computerized finite element model of pelvic and urethra and medical applications.” 2012. Web. 15 Jun 2019.

Vancouver:

Conte C. Comportement mécanique du foie en contexte traumatique : rupture et endommagement des tissus : Posterior urethral injuries associated with motorcycle pelvic injuries in adolescent. : Computerized finite element model of pelvic and urethra and medical applications. [Internet] [Doctoral dissertation]. Aix Marseille Université 2012. [cited 2019 Jun 15]. Available from: http://www.theses.fr/2012AIXM4103.

Council of Science Editors:

Conte C. Comportement mécanique du foie en contexte traumatique : rupture et endommagement des tissus : Posterior urethral injuries associated with motorcycle pelvic injuries in adolescent. : Computerized finite element model of pelvic and urethra and medical applications. [Doctoral Dissertation]. Aix Marseille Université 2012. Available from: http://www.theses.fr/2012AIXM4103


University of Houston

19. Mallick, Pankajini. Molecular Mechanisms of Irinotecan Induced Fatty Liver Disease and Drug-Drug Interaction.

Degree: Pharmacological and Pharmaceutical Sciences, Department of, 2017, University of Houston

 Chemotherapy drugs due to their inherent cytotoxicity, inevitably causes negative consequences. Liver being the primary site of metabolism for many chemotherapy drugs, hepatotoxicity is collateral… (more)

Subjects/Keywords: Drug-drug interaction; Drug metabolizing enzymes; Irinotecan; Taxol; Fatty liver disease (FLD); diarrhea; Toll-like receptor 4 (TLR4); Cytochrome P450; Ugt1a1; MyD88; Pharmacokinetics; SN-38; Chemotherapy; intestinal damage

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APA (6th Edition):

Mallick, P. (2017). Molecular Mechanisms of Irinotecan Induced Fatty Liver Disease and Drug-Drug Interaction. (Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/3538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mallick, Pankajini. “Molecular Mechanisms of Irinotecan Induced Fatty Liver Disease and Drug-Drug Interaction.” 2017. Thesis, University of Houston. Accessed June 15, 2019. http://hdl.handle.net/10657/3538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mallick, Pankajini. “Molecular Mechanisms of Irinotecan Induced Fatty Liver Disease and Drug-Drug Interaction.” 2017. Web. 15 Jun 2019.

Vancouver:

Mallick P. Molecular Mechanisms of Irinotecan Induced Fatty Liver Disease and Drug-Drug Interaction. [Internet] [Thesis]. University of Houston; 2017. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/10657/3538.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mallick P. Molecular Mechanisms of Irinotecan Induced Fatty Liver Disease and Drug-Drug Interaction. [Thesis]. University of Houston; 2017. Available from: http://hdl.handle.net/10657/3538

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Brunon, Aline. Caractérisation et modélisation de la rupture des tissus hépatiques : Characterization and modeling of the hepatic tissues failure.

Degree: Docteur es, Biomécanique, 2011, INSA Lyon

Dans le cadre de la sécurité routière, les essais de choc ne permettent pas la prédiction des blessures internes, notamment aux organes abdominaux. La simulation… (more)

Subjects/Keywords: Biosciences; Biomécanique; Foie; Tissu hépatique; Rupture; Modélisation; Modèle d'endommagement; Correlation d'images; Hyperélasticité; Biosciences; Biomechanics; Liver; Hepatic tissus; Failure; Modeling; Damage model; Image correlation; Hyperelasticity; 571.407 2

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APA (6th Edition):

Brunon, A. (2011). Caractérisation et modélisation de la rupture des tissus hépatiques : Characterization and modeling of the hepatic tissues failure. (Doctoral Dissertation). INSA Lyon. Retrieved from http://www.theses.fr/2011ISAL0105

Chicago Manual of Style (16th Edition):

Brunon, Aline. “Caractérisation et modélisation de la rupture des tissus hépatiques : Characterization and modeling of the hepatic tissues failure.” 2011. Doctoral Dissertation, INSA Lyon. Accessed June 15, 2019. http://www.theses.fr/2011ISAL0105.

MLA Handbook (7th Edition):

Brunon, Aline. “Caractérisation et modélisation de la rupture des tissus hépatiques : Characterization and modeling of the hepatic tissues failure.” 2011. Web. 15 Jun 2019.

Vancouver:

Brunon A. Caractérisation et modélisation de la rupture des tissus hépatiques : Characterization and modeling of the hepatic tissues failure. [Internet] [Doctoral dissertation]. INSA Lyon; 2011. [cited 2019 Jun 15]. Available from: http://www.theses.fr/2011ISAL0105.

Council of Science Editors:

Brunon A. Caractérisation et modélisation de la rupture des tissus hépatiques : Characterization and modeling of the hepatic tissues failure. [Doctoral Dissertation]. INSA Lyon; 2011. Available from: http://www.theses.fr/2011ISAL0105


Vrije Universiteit Amsterdam

21. Gul, N. Surgery-induced liver metastasis: how it begins and how to end it .

Degree: 2012, Vrije Universiteit Amsterdam

Subjects/Keywords: surgery; inflammation; macrophages; endothelial damage; tumor cell adhesion; liver metastases

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APA (6th Edition):

Gul, N. (2012). Surgery-induced liver metastasis: how it begins and how to end it . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/38333

Chicago Manual of Style (16th Edition):

Gul, N. “Surgery-induced liver metastasis: how it begins and how to end it .” 2012. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed June 15, 2019. http://hdl.handle.net/1871/38333.

MLA Handbook (7th Edition):

Gul, N. “Surgery-induced liver metastasis: how it begins and how to end it .” 2012. Web. 15 Jun 2019.

Vancouver:

Gul N. Surgery-induced liver metastasis: how it begins and how to end it . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2012. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/1871/38333.

Council of Science Editors:

Gul N. Surgery-induced liver metastasis: how it begins and how to end it . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2012. Available from: http://hdl.handle.net/1871/38333


IUPUI

22. Gao, Yong. NONINVASIVE MONITORING OF CCl4 INDUCED ACUTE AND CHRONIC LIVER DAMAGE IN RAT BY SINGLE QUANTUM AND TRIPLE QUANTUM FILTERED 23Na MAGNETIC RESONANCE IMAGING.

Degree: 2008, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

In present study, single quantum (SQ) and triple quantum filtered (TQF) 23Na magnetic resonance imaging (MRI) was used to monitor… (more)

Subjects/Keywords: sodium imaging; liver damage; carbon tetrachloride; rat model; Magnetic resonance imaging; Liver  – Diseases  – Diagnosis; Carbon tetrachloride

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APA (6th Edition):

Gao, Y. (2008). NONINVASIVE MONITORING OF CCl4 INDUCED ACUTE AND CHRONIC LIVER DAMAGE IN RAT BY SINGLE QUANTUM AND TRIPLE QUANTUM FILTERED 23Na MAGNETIC RESONANCE IMAGING. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gao, Yong. “NONINVASIVE MONITORING OF CCl4 INDUCED ACUTE AND CHRONIC LIVER DAMAGE IN RAT BY SINGLE QUANTUM AND TRIPLE QUANTUM FILTERED 23Na MAGNETIC RESONANCE IMAGING.” 2008. Thesis, IUPUI. Accessed June 15, 2019. http://hdl.handle.net/1805/2045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gao, Yong. “NONINVASIVE MONITORING OF CCl4 INDUCED ACUTE AND CHRONIC LIVER DAMAGE IN RAT BY SINGLE QUANTUM AND TRIPLE QUANTUM FILTERED 23Na MAGNETIC RESONANCE IMAGING.” 2008. Web. 15 Jun 2019.

Vancouver:

Gao Y. NONINVASIVE MONITORING OF CCl4 INDUCED ACUTE AND CHRONIC LIVER DAMAGE IN RAT BY SINGLE QUANTUM AND TRIPLE QUANTUM FILTERED 23Na MAGNETIC RESONANCE IMAGING. [Internet] [Thesis]. IUPUI; 2008. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/1805/2045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gao Y. NONINVASIVE MONITORING OF CCl4 INDUCED ACUTE AND CHRONIC LIVER DAMAGE IN RAT BY SINGLE QUANTUM AND TRIPLE QUANTUM FILTERED 23Na MAGNETIC RESONANCE IMAGING. [Thesis]. IUPUI; 2008. Available from: http://hdl.handle.net/1805/2045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Texas

23. Barst, Benjamin Daniel. Hepatotoxicity of Mercury to Fish.

Degree: 2010, University of North Texas

 Tissue samples from spotted gar (Lepisosteus oculatus) and largemouth bass (Micropterus salmoides) were collected from Caddo Lake. Gar and bass livers were subjected to histological… (more)

Subjects/Keywords: Liver damage; liver color; mercury; fish; Hepatotoxicology.; Fishes  – Effect of heavy metals on  – Caddo Lake (La. and Tex.); Fishes  – Mercury content  – Caddo Lake (La. and Tex.); Mercury  – Toxicology  – Caddo Lake (La. and Tex.); Mercury  – Environmental aspects  – Caddo Lake (La. and Tex.)

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APA (6th Edition):

Barst, B. D. (2010). Hepatotoxicity of Mercury to Fish. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc31525/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barst, Benjamin Daniel. “Hepatotoxicity of Mercury to Fish.” 2010. Thesis, University of North Texas. Accessed June 15, 2019. https://digital.library.unt.edu/ark:/67531/metadc31525/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barst, Benjamin Daniel. “Hepatotoxicity of Mercury to Fish.” 2010. Web. 15 Jun 2019.

Vancouver:

Barst BD. Hepatotoxicity of Mercury to Fish. [Internet] [Thesis]. University of North Texas; 2010. [cited 2019 Jun 15]. Available from: https://digital.library.unt.edu/ark:/67531/metadc31525/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barst BD. Hepatotoxicity of Mercury to Fish. [Thesis]. University of North Texas; 2010. Available from: https://digital.library.unt.edu/ark:/67531/metadc31525/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

24. Souto, Kátia Elisabete Pires. Doença hepática gordurosa não alcoólica (DHGNA) em pacientes morbidamente obesos submetidos à cirurgia bariátrica : correlação entre os achados histopatológicos das biópsias hepáticas intraoperatórias e estado glicêmico basal.

Degree: 2017, Universidade do Rio Grande do Sul

Introdução: A Doença Hepática Gordurosa Não Alcoólica (DHGNA) tem como causa principal a obesidade. Atualmente não existe tratamento medicamentoso específico para DHGNA. A cirurgia bariátrica… (more)

Subjects/Keywords: Hepatopatia gordurosa não alcoólica; Liver damage is more severe in obese and diabetic patients; Cirurgia bariátrica; Liver biopsy should be considered during bariatric surgery in patients with metabolic syndrome; Diabetes mellitus tipo 2; Early surgery in diabetic patients could prevent progression of NAFLD and promote diabetes remission; Obesidade mórbida

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APA (6th Edition):

Souto, K. E. P. (2017). Doença hepática gordurosa não alcoólica (DHGNA) em pacientes morbidamente obesos submetidos à cirurgia bariátrica : correlação entre os achados histopatológicos das biópsias hepáticas intraoperatórias e estado glicêmico basal. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/170935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Souto, Kátia Elisabete Pires. “Doença hepática gordurosa não alcoólica (DHGNA) em pacientes morbidamente obesos submetidos à cirurgia bariátrica : correlação entre os achados histopatológicos das biópsias hepáticas intraoperatórias e estado glicêmico basal.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed June 15, 2019. http://hdl.handle.net/10183/170935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Souto, Kátia Elisabete Pires. “Doença hepática gordurosa não alcoólica (DHGNA) em pacientes morbidamente obesos submetidos à cirurgia bariátrica : correlação entre os achados histopatológicos das biópsias hepáticas intraoperatórias e estado glicêmico basal.” 2017. Web. 15 Jun 2019.

Vancouver:

Souto KEP. Doença hepática gordurosa não alcoólica (DHGNA) em pacientes morbidamente obesos submetidos à cirurgia bariátrica : correlação entre os achados histopatológicos das biópsias hepáticas intraoperatórias e estado glicêmico basal. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/10183/170935.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Souto KEP. Doença hepática gordurosa não alcoólica (DHGNA) em pacientes morbidamente obesos submetidos à cirurgia bariátrica : correlação entre os achados histopatológicos das biópsias hepáticas intraoperatórias e estado glicêmico basal. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/170935

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Erasmus University Rotterdam

25. Vougioukalaki, Maria. Impact of Endogenous Genomic Damage on Adult Stem Cells and Tissue Homeostasis.

Degree: 2019, Erasmus University Rotterdam

Subjects/Keywords: DNA damage; DNA repair; Adult Stem Cells; small intestine; liver; organoids; ERCC1; Nucleotide Excision Repair; cellular lifespan; anti-ageing strategy; cancer; mutational signature; DNA damage tolerance; REV1

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APA (6th Edition):

Vougioukalaki, M. (2019). Impact of Endogenous Genomic Damage on Adult Stem Cells and Tissue Homeostasis. (Doctoral Dissertation). Erasmus University Rotterdam. Retrieved from http://hdl.handle.net/1765/115132

Chicago Manual of Style (16th Edition):

Vougioukalaki, Maria. “Impact of Endogenous Genomic Damage on Adult Stem Cells and Tissue Homeostasis.” 2019. Doctoral Dissertation, Erasmus University Rotterdam. Accessed June 15, 2019. http://hdl.handle.net/1765/115132.

MLA Handbook (7th Edition):

Vougioukalaki, Maria. “Impact of Endogenous Genomic Damage on Adult Stem Cells and Tissue Homeostasis.” 2019. Web. 15 Jun 2019.

Vancouver:

Vougioukalaki M. Impact of Endogenous Genomic Damage on Adult Stem Cells and Tissue Homeostasis. [Internet] [Doctoral dissertation]. Erasmus University Rotterdam; 2019. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/1765/115132.

Council of Science Editors:

Vougioukalaki M. Impact of Endogenous Genomic Damage on Adult Stem Cells and Tissue Homeostasis. [Doctoral Dissertation]. Erasmus University Rotterdam; 2019. Available from: http://hdl.handle.net/1765/115132


University of Florida

26. Robbins, Sean W. Investigating the Role of Haptoglobin in Epicatechin-Mediated Protection against Intracerebral Hemorrhage.

Degree: MS, Medical Sciences - Medicine, 2013, University of Florida

Subjects/Keywords: Antioxidants; Blood; Brain damage; Cerebral hemorrhage; Dosage; Hemoglobins; Lesions; Liver; Mice; Strokes; epicatechin; haptoglobin; hemoglobin; ich; stroke

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APA (6th Edition):

Robbins, S. W. (2013). Investigating the Role of Haptoglobin in Epicatechin-Mediated Protection against Intracerebral Hemorrhage. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0046033

Chicago Manual of Style (16th Edition):

Robbins, Sean W. “Investigating the Role of Haptoglobin in Epicatechin-Mediated Protection against Intracerebral Hemorrhage.” 2013. Masters Thesis, University of Florida. Accessed June 15, 2019. http://ufdc.ufl.edu/UFE0046033.

MLA Handbook (7th Edition):

Robbins, Sean W. “Investigating the Role of Haptoglobin in Epicatechin-Mediated Protection against Intracerebral Hemorrhage.” 2013. Web. 15 Jun 2019.

Vancouver:

Robbins SW. Investigating the Role of Haptoglobin in Epicatechin-Mediated Protection against Intracerebral Hemorrhage. [Internet] [Masters thesis]. University of Florida; 2013. [cited 2019 Jun 15]. Available from: http://ufdc.ufl.edu/UFE0046033.

Council of Science Editors:

Robbins SW. Investigating the Role of Haptoglobin in Epicatechin-Mediated Protection against Intracerebral Hemorrhage. [Masters Thesis]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0046033

27. Νέζη, Βασιλική. Συσχέτιση μεταξύ πολυμορφισμού των πολυσακχαριτιδικών υποδοχέων TLR-4 και CD14 και ευπάθειας στην ανάπτυξη αλκοολικής νόσου του ήπατος.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Background: Ethanol abuse is a major social and clinical problem with an increasing percent of drinkers developing alcoholic liver disease (ALD). The incidence and severity… (more)

Subjects/Keywords: Αλκοολική νόσος του ήπατος; Κατάχρηση αλκοόλ; Έλληνες χρήστες; Πολυμορφισμοί; Γονίδια CD14 και Toll-Like-4; Ατομική ευπάθεια; Ενδοτοξιναιμία; Κυτοκίνες; Χρόνια φλεγμονή; Ηπατοκυτταρική βλάβη; Alcoholic liver disease; Alcohol abuse; Greek drinkers; Polymorphisms; Genes CD14 and Toll-Like-4; Susceptibility; Endotoxemia; Cytokines; Chronic inflammation; Hepatocellular damage

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APA (6th Edition):

Νέζη, . . (2012). Συσχέτιση μεταξύ πολυμορφισμού των πολυσακχαριτιδικών υποδοχέων TLR-4 και CD14 και ευπάθειας στην ανάπτυξη αλκοολικής νόσου του ήπατος. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/29547

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Νέζη, Βασιλική. “Συσχέτιση μεταξύ πολυμορφισμού των πολυσακχαριτιδικών υποδοχέων TLR-4 και CD14 και ευπάθειας στην ανάπτυξη αλκοολικής νόσου του ήπατος.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed June 15, 2019. http://hdl.handle.net/10442/hedi/29547.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Νέζη, Βασιλική. “Συσχέτιση μεταξύ πολυμορφισμού των πολυσακχαριτιδικών υποδοχέων TLR-4 και CD14 και ευπάθειας στην ανάπτυξη αλκοολικής νόσου του ήπατος.” 2012. Web. 15 Jun 2019.

Vancouver:

Νέζη . Συσχέτιση μεταξύ πολυμορφισμού των πολυσακχαριτιδικών υποδοχέων TLR-4 και CD14 και ευπάθειας στην ανάπτυξη αλκοολικής νόσου του ήπατος. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/10442/hedi/29547.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Νέζη . Συσχέτιση μεταξύ πολυμορφισμού των πολυσακχαριτιδικών υποδοχέων TLR-4 και CD14 και ευπάθειας στην ανάπτυξη αλκοολικής νόσου του ήπατος. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/29547

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lethbridge

28. Walz, Paul S. Influence of pathogenic bacterial determinants on genome stability of exposed intestinal cells and of distal liver and spleen cells .

Degree: 2011, University of Lethbridge

 Most bacterial infections can be correlated to contamination of consumables such as food and water. Upon contamination, boil water advisories have been ordered to ensure… (more)

Subjects/Keywords: Pathogenic bacteria  – Inactivation  – Research; Escherichia coli O157:H7  – Research; Microbial carcinogenesis  – Research; DNA damage  – Research; Intestines  – Infections  – Pathogenesis; Liver cells; Spleen  – Pathogenesis  – Research; Endotoxins  – Research; Mice as laboratory animals; Dissertations, Academic

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APA (6th Edition):

Walz, P. S. (2011). Influence of pathogenic bacterial determinants on genome stability of exposed intestinal cells and of distal liver and spleen cells . (Thesis). University of Lethbridge. Retrieved from http://hdl.handle.net/10133/3405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walz, Paul S. “Influence of pathogenic bacterial determinants on genome stability of exposed intestinal cells and of distal liver and spleen cells .” 2011. Thesis, University of Lethbridge. Accessed June 15, 2019. http://hdl.handle.net/10133/3405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walz, Paul S. “Influence of pathogenic bacterial determinants on genome stability of exposed intestinal cells and of distal liver and spleen cells .” 2011. Web. 15 Jun 2019.

Vancouver:

Walz PS. Influence of pathogenic bacterial determinants on genome stability of exposed intestinal cells and of distal liver and spleen cells . [Internet] [Thesis]. University of Lethbridge; 2011. [cited 2019 Jun 15]. Available from: http://hdl.handle.net/10133/3405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walz PS. Influence of pathogenic bacterial determinants on genome stability of exposed intestinal cells and of distal liver and spleen cells . [Thesis]. University of Lethbridge; 2011. Available from: http://hdl.handle.net/10133/3405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Flores, Cristina. The Cellular and Molecular Mechanism of Third Hand Cigarette Smoke-Induced Hepatic Steatosis.

Degree: Cell, Molecular and Developmental Biology, 2017, University of California – Riverside

 Recent evidence indicates that third hand smoke (THS) is present mainly in places where smoking happens regularly but also in places where smoking has been… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Toxicology; Fatty acids; Lipid metabolism; Liver; Oxidative stress; Oxidative stress mediated damage; THS toxins

…in behavior, lung damage, lipid accumulation in the liver and altered glucose and lipid… …induced liver damage. Our results show that THS is a new risk factor contributing to the… …increase oxidative-stress-mediated damage (protein damage, DNA damage and functional damage… …contributing to accumulation of lipids in the liver. THS-exposed mice have high levels of TG and… …results in less lipid accumulation in the liver. These findings provide evidence of the cellular… 

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APA (6th Edition):

Flores, C. (2017). The Cellular and Molecular Mechanism of Third Hand Cigarette Smoke-Induced Hepatic Steatosis. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/9wg116sj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Flores, Cristina. “The Cellular and Molecular Mechanism of Third Hand Cigarette Smoke-Induced Hepatic Steatosis.” 2017. Thesis, University of California – Riverside. Accessed June 15, 2019. http://www.escholarship.org/uc/item/9wg116sj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Flores, Cristina. “The Cellular and Molecular Mechanism of Third Hand Cigarette Smoke-Induced Hepatic Steatosis.” 2017. Web. 15 Jun 2019.

Vancouver:

Flores C. The Cellular and Molecular Mechanism of Third Hand Cigarette Smoke-Induced Hepatic Steatosis. [Internet] [Thesis]. University of California – Riverside; 2017. [cited 2019 Jun 15]. Available from: http://www.escholarship.org/uc/item/9wg116sj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Flores C. The Cellular and Molecular Mechanism of Third Hand Cigarette Smoke-Induced Hepatic Steatosis. [Thesis]. University of California – Riverside; 2017. Available from: http://www.escholarship.org/uc/item/9wg116sj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. 佐貫, 直子. Threshold Doses for Focal Liver Reaction Following Stereotactic Ablative Body Radiotherapy for Small Hepatocellular Carcinoma Depend on Liver Function : Evaluation on Magnetic Resonance Imaging with Gd-EOB-DTPA : 肝細胞癌に対する体幹部定位放射線治療後の肝臓の局所反応は肝機能に依存する:Gd-EOB-DTPA造影MRIによる評価; カンサイボウガン ニ タイスル タイカンブ テイイ ホウシャセン チリョウゴ ノ カンゾウ ノ キョクショ ハンノウ ワ カンキノウ ニ イゾン スル : Gd-EOB-DTPA ゾウエイ MRI ニヨル ヒョウカ.

Degree: 博士(医学), 2014, Tokai University Repository / 東海大学

Subjects/Keywords: Gd-EOB-DTPA, Hepatocellular carcinoma, MRI, Radiation-induced liver damage, Stereotactic body radiation therapy; Gd-EOB-DTPA, 肝細胞癌, MRI, 放射線誘発性肝障害, 体幹部定位放射線治療

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APA (6th Edition):

佐貫, . (2014). Threshold Doses for Focal Liver Reaction Following Stereotactic Ablative Body Radiotherapy for Small Hepatocellular Carcinoma Depend on Liver Function : Evaluation on Magnetic Resonance Imaging with Gd-EOB-DTPA : 肝細胞癌に対する体幹部定位放射線治療後の肝臓の局所反応は肝機能に依存する:Gd-EOB-DTPA造影MRIによる評価; カンサイボウガン ニ タイスル タイカンブ テイイ ホウシャセン チリョウゴ ノ カンゾウ ノ キョクショ ハンノウ ワ カンキノウ ニ イゾン スル : Gd-EOB-DTPA ゾウエイ MRI ニヨル ヒョウカ. (Thesis). Tokai University Repository / 東海大学. Retrieved from https://opac.time.u-tokai.ac.jp/webopac/TD00000124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

佐貫, 直子. “Threshold Doses for Focal Liver Reaction Following Stereotactic Ablative Body Radiotherapy for Small Hepatocellular Carcinoma Depend on Liver Function : Evaluation on Magnetic Resonance Imaging with Gd-EOB-DTPA : 肝細胞癌に対する体幹部定位放射線治療後の肝臓の局所反応は肝機能に依存する:Gd-EOB-DTPA造影MRIによる評価; カンサイボウガン ニ タイスル タイカンブ テイイ ホウシャセン チリョウゴ ノ カンゾウ ノ キョクショ ハンノウ ワ カンキノウ ニ イゾン スル : Gd-EOB-DTPA ゾウエイ MRI ニヨル ヒョウカ.” 2014. Thesis, Tokai University Repository / 東海大学. Accessed June 15, 2019. https://opac.time.u-tokai.ac.jp/webopac/TD00000124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

佐貫, 直子. “Threshold Doses for Focal Liver Reaction Following Stereotactic Ablative Body Radiotherapy for Small Hepatocellular Carcinoma Depend on Liver Function : Evaluation on Magnetic Resonance Imaging with Gd-EOB-DTPA : 肝細胞癌に対する体幹部定位放射線治療後の肝臓の局所反応は肝機能に依存する:Gd-EOB-DTPA造影MRIによる評価; カンサイボウガン ニ タイスル タイカンブ テイイ ホウシャセン チリョウゴ ノ カンゾウ ノ キョクショ ハンノウ ワ カンキノウ ニ イゾン スル : Gd-EOB-DTPA ゾウエイ MRI ニヨル ヒョウカ.” 2014. Web. 15 Jun 2019.

Vancouver:

佐貫 . Threshold Doses for Focal Liver Reaction Following Stereotactic Ablative Body Radiotherapy for Small Hepatocellular Carcinoma Depend on Liver Function : Evaluation on Magnetic Resonance Imaging with Gd-EOB-DTPA : 肝細胞癌に対する体幹部定位放射線治療後の肝臓の局所反応は肝機能に依存する:Gd-EOB-DTPA造影MRIによる評価; カンサイボウガン ニ タイスル タイカンブ テイイ ホウシャセン チリョウゴ ノ カンゾウ ノ キョクショ ハンノウ ワ カンキノウ ニ イゾン スル : Gd-EOB-DTPA ゾウエイ MRI ニヨル ヒョウカ. [Internet] [Thesis]. Tokai University Repository / 東海大学; 2014. [cited 2019 Jun 15]. Available from: https://opac.time.u-tokai.ac.jp/webopac/TD00000124.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

佐貫 . Threshold Doses for Focal Liver Reaction Following Stereotactic Ablative Body Radiotherapy for Small Hepatocellular Carcinoma Depend on Liver Function : Evaluation on Magnetic Resonance Imaging with Gd-EOB-DTPA : 肝細胞癌に対する体幹部定位放射線治療後の肝臓の局所反応は肝機能に依存する:Gd-EOB-DTPA造影MRIによる評価; カンサイボウガン ニ タイスル タイカンブ テイイ ホウシャセン チリョウゴ ノ カンゾウ ノ キョクショ ハンノウ ワ カンキノウ ニ イゾン スル : Gd-EOB-DTPA ゾウエイ MRI ニヨル ヒョウカ. [Thesis]. Tokai University Repository / 東海大学; 2014. Available from: https://opac.time.u-tokai.ac.jp/webopac/TD00000124

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.