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You searched for subject:(laminin). Showing records 1 – 30 of 94 total matches.

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Uppsala University

1. Fransson, Sara Willumsen. Cellular localization of the blood-brain barrier in the brainstem: Area postrema and nucleus tractus solitarius.

Degree: Medical Biochemistry and Microbiology, 2008, Uppsala University

  The blood-brain barrier regulates the transport into the brain and protects the central nerve system (CNS) from toxics substances. However some areas of the… (more)

Subjects/Keywords: Laminin; RECA1; EBA; Glia cells; NPY; Immunohistochemistry

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APA (6th Edition):

Fransson, S. W. (2008). Cellular localization of the blood-brain barrier in the brainstem: Area postrema and nucleus tractus solitarius. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fransson, Sara Willumsen. “Cellular localization of the blood-brain barrier in the brainstem: Area postrema and nucleus tractus solitarius.” 2008. Thesis, Uppsala University. Accessed August 23, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fransson, Sara Willumsen. “Cellular localization of the blood-brain barrier in the brainstem: Area postrema and nucleus tractus solitarius.” 2008. Web. 23 Aug 2019.

Vancouver:

Fransson SW. Cellular localization of the blood-brain barrier in the brainstem: Area postrema and nucleus tractus solitarius. [Internet] [Thesis]. Uppsala University; 2008. [cited 2019 Aug 23]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fransson SW. Cellular localization of the blood-brain barrier in the brainstem: Area postrema and nucleus tractus solitarius. [Thesis]. Uppsala University; 2008. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

2. Rahman, Shakib Hafizur. Effects of Hyperthermia and Subsequent Minocycline Treatment in Acute Ischemic Stroke.

Degree: MS, Department of Medicine, 2012, University of Alberta

 Stroke, a reduction in blood flow to the brain, activates various proteins that contribute to neuronal death. The matrix metalloproteinases (MMPs), specifically MMP2 and MMP9,… (more)

Subjects/Keywords: hyperthermia; stroke; laminin; matrix metalloprotease; ischemia; minocycline

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APA (6th Edition):

Rahman, S. H. (2012). Effects of Hyperthermia and Subsequent Minocycline Treatment in Acute Ischemic Stroke. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/9019s270q

Chicago Manual of Style (16th Edition):

Rahman, Shakib Hafizur. “Effects of Hyperthermia and Subsequent Minocycline Treatment in Acute Ischemic Stroke.” 2012. Masters Thesis, University of Alberta. Accessed August 23, 2019. https://era.library.ualberta.ca/files/9019s270q.

MLA Handbook (7th Edition):

Rahman, Shakib Hafizur. “Effects of Hyperthermia and Subsequent Minocycline Treatment in Acute Ischemic Stroke.” 2012. Web. 23 Aug 2019.

Vancouver:

Rahman SH. Effects of Hyperthermia and Subsequent Minocycline Treatment in Acute Ischemic Stroke. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2019 Aug 23]. Available from: https://era.library.ualberta.ca/files/9019s270q.

Council of Science Editors:

Rahman SH. Effects of Hyperthermia and Subsequent Minocycline Treatment in Acute Ischemic Stroke. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/9019s270q


Clemson University

3. Elliott, Kathryn Adrian. A Comprehensive Model and Modulation of Cellular Signaling Involved in Early Mammary Development and Aggressive Cancer Using a Novel Recombinant Protein of the G3 Domain of Laminin-5.

Degree: PhD, Biological Sciences, 2017, Clemson University

 The mammary gland is a unique and specialized epidermal organ; mammary organogenesis begins in the embryo but is not fully complete until puberty. As such,… (more)

Subjects/Keywords: Breast cancer; Development; Integrin; Laminin; Mammary; TNBC

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APA (6th Edition):

Elliott, K. A. (2017). A Comprehensive Model and Modulation of Cellular Signaling Involved in Early Mammary Development and Aggressive Cancer Using a Novel Recombinant Protein of the G3 Domain of Laminin-5. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/2071

Chicago Manual of Style (16th Edition):

Elliott, Kathryn Adrian. “A Comprehensive Model and Modulation of Cellular Signaling Involved in Early Mammary Development and Aggressive Cancer Using a Novel Recombinant Protein of the G3 Domain of Laminin-5.” 2017. Doctoral Dissertation, Clemson University. Accessed August 23, 2019. https://tigerprints.clemson.edu/all_dissertations/2071.

MLA Handbook (7th Edition):

Elliott, Kathryn Adrian. “A Comprehensive Model and Modulation of Cellular Signaling Involved in Early Mammary Development and Aggressive Cancer Using a Novel Recombinant Protein of the G3 Domain of Laminin-5.” 2017. Web. 23 Aug 2019.

Vancouver:

Elliott KA. A Comprehensive Model and Modulation of Cellular Signaling Involved in Early Mammary Development and Aggressive Cancer Using a Novel Recombinant Protein of the G3 Domain of Laminin-5. [Internet] [Doctoral dissertation]. Clemson University; 2017. [cited 2019 Aug 23]. Available from: https://tigerprints.clemson.edu/all_dissertations/2071.

Council of Science Editors:

Elliott KA. A Comprehensive Model and Modulation of Cellular Signaling Involved in Early Mammary Development and Aggressive Cancer Using a Novel Recombinant Protein of the G3 Domain of Laminin-5. [Doctoral Dissertation]. Clemson University; 2017. Available from: https://tigerprints.clemson.edu/all_dissertations/2071


University of Arizona

4. Das, Lipsa. Regulation of Intracellular Trafficking of Laminin Binding Integrins in Prostate Cancer .

Degree: 2017, University of Arizona

Laminin binding integrins (α6β1 and α3β1) are persistently but differentially expressed throughout prostate cancer progression and metastasis. Prostate cancer primarily invades through laminin rich nerve… (more)

Subjects/Keywords: Cancer; Endosomes; Integrin; Laminin; Rab11; Trafficking

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APA (6th Edition):

Das, L. (2017). Regulation of Intracellular Trafficking of Laminin Binding Integrins in Prostate Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/625651

Chicago Manual of Style (16th Edition):

Das, Lipsa. “Regulation of Intracellular Trafficking of Laminin Binding Integrins in Prostate Cancer .” 2017. Doctoral Dissertation, University of Arizona. Accessed August 23, 2019. http://hdl.handle.net/10150/625651.

MLA Handbook (7th Edition):

Das, Lipsa. “Regulation of Intracellular Trafficking of Laminin Binding Integrins in Prostate Cancer .” 2017. Web. 23 Aug 2019.

Vancouver:

Das L. Regulation of Intracellular Trafficking of Laminin Binding Integrins in Prostate Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2017. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10150/625651.

Council of Science Editors:

Das L. Regulation of Intracellular Trafficking of Laminin Binding Integrins in Prostate Cancer . [Doctoral Dissertation]. University of Arizona; 2017. Available from: http://hdl.handle.net/10150/625651

5. Michopoulou, Anna. Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes.

Degree: Docteur es, Biologie, 2016, Lyon

 La phase de l'épithélialisation de la réparation cutanée se déroule en impliquant plusieurs processus dynamiques et interactifs pendant lesquels les kératinocytes migrent, prolifèrent et se… (more)

Subjects/Keywords: MMP-9; Syndecan-1; Keratinocyte; Laminin 332; MMP-9; Syndecan-1; Keratinocyte; Laminin 332; 571.6

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APA (6th Edition):

Michopoulou, A. (2016). Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1130

Chicago Manual of Style (16th Edition):

Michopoulou, Anna. “Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes.” 2016. Doctoral Dissertation, Lyon. Accessed August 23, 2019. http://www.theses.fr/2016LYSE1130.

MLA Handbook (7th Edition):

Michopoulou, Anna. “Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes.” 2016. Web. 23 Aug 2019.

Vancouver:

Michopoulou A. Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2019 Aug 23]. Available from: http://www.theses.fr/2016LYSE1130.

Council of Science Editors:

Michopoulou A. Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1130


University of Bradford

6. Berends, Rebecca Fay. The effects of TGF-β on the behaviour of a keratinocyte cell line : implications in wound repair.

Degree: PhD, 2011, University of Bradford

 TGF-β isoforms are important signalling molecules in wound repair in the skin. Transforming growth factor β3 (TGF-β3) has been implicated in scarless healing. In both… (more)

Subjects/Keywords: 570; HaCaT; Keratinocyte; Transforming growth factor ?; ECM; Wound repair; Laminin and integrins; Signalling molecules; Human skin; Scarring; Integrin; Laminin

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APA (6th Edition):

Berends, R. F. (2011). The effects of TGF-β on the behaviour of a keratinocyte cell line : implications in wound repair. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/5393

Chicago Manual of Style (16th Edition):

Berends, Rebecca Fay. “The effects of TGF-β on the behaviour of a keratinocyte cell line : implications in wound repair.” 2011. Doctoral Dissertation, University of Bradford. Accessed August 23, 2019. http://hdl.handle.net/10454/5393.

MLA Handbook (7th Edition):

Berends, Rebecca Fay. “The effects of TGF-β on the behaviour of a keratinocyte cell line : implications in wound repair.” 2011. Web. 23 Aug 2019.

Vancouver:

Berends RF. The effects of TGF-β on the behaviour of a keratinocyte cell line : implications in wound repair. [Internet] [Doctoral dissertation]. University of Bradford; 2011. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10454/5393.

Council of Science Editors:

Berends RF. The effects of TGF-β on the behaviour of a keratinocyte cell line : implications in wound repair. [Doctoral Dissertation]. University of Bradford; 2011. Available from: http://hdl.handle.net/10454/5393


University of Cincinnati

7. Buschmann, Mary McVey. Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium.

Degree: PhD, Medicine : Cell and Molecular Biology, 2010, University of Cincinnati

 Normal epithelial cells rely on spatial cues from the extracellular matrix to proliferate, migrate, and survive. The extracellular matrix protein, laminin-332 (LM-332) seems to be… (more)

Subjects/Keywords: Cellular Biology; Laminin-332; Extracellular Matrix; Epithelia; MDCK; Proliferation

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APA (6th Edition):

Buschmann, M. M. (2010). Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275661166

Chicago Manual of Style (16th Edition):

Buschmann, Mary McVey. “Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium.” 2010. Doctoral Dissertation, University of Cincinnati. Accessed August 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275661166.

MLA Handbook (7th Edition):

Buschmann, Mary McVey. “Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium.” 2010. Web. 23 Aug 2019.

Vancouver:

Buschmann MM. Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium. [Internet] [Doctoral dissertation]. University of Cincinnati; 2010. [cited 2019 Aug 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275661166.

Council of Science Editors:

Buschmann MM. Laminin-332-Mediated Proliferation Control: Mechanisms Regulating Formation of the Epithelium. [Doctoral Dissertation]. University of Cincinnati; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1275661166


Wright State University

8. Jani, Meghna. Transcriptional regulation of LAMB3 by p53.

Degree: MS, Biochemistry and Molecular Biology, 2008, Wright State University

  The p53 tumor-suppressor plays a very important role in the prevention of cancer and it is known that about 50% of all human tumors… (more)

Subjects/Keywords: Molecular Biology; p53; LAMB3; Hdm2; HdmX; transcriptional; Laminin-5

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APA (6th Edition):

Jani, M. (2008). Transcriptional regulation of LAMB3 by p53. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1229958362

Chicago Manual of Style (16th Edition):

Jani, Meghna. “Transcriptional regulation of LAMB3 by p53.” 2008. Masters Thesis, Wright State University. Accessed August 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1229958362.

MLA Handbook (7th Edition):

Jani, Meghna. “Transcriptional regulation of LAMB3 by p53.” 2008. Web. 23 Aug 2019.

Vancouver:

Jani M. Transcriptional regulation of LAMB3 by p53. [Internet] [Masters thesis]. Wright State University; 2008. [cited 2019 Aug 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1229958362.

Council of Science Editors:

Jani M. Transcriptional regulation of LAMB3 by p53. [Masters Thesis]. Wright State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1229958362


Duke University

9. Francisco, Aubrey Therese. Laminin-Functionalized Polyethylene Glycol Hydrogels for Nucleus Pulposus Regeneration .

Degree: 2013, Duke University

  Intervertebral disc (IVD) disorders and age-related degeneration are believed to contribute to low back pain. There is significant interest in cell-based strategies for regenerating… (more)

Subjects/Keywords: Biomedical engineering; cell delivery; intervertebral disc; laminin; nucleus pulposus; polyethylene glycol

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APA (6th Edition):

Francisco, A. T. (2013). Laminin-Functionalized Polyethylene Glycol Hydrogels for Nucleus Pulposus Regeneration . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/8204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Francisco, Aubrey Therese. “Laminin-Functionalized Polyethylene Glycol Hydrogels for Nucleus Pulposus Regeneration .” 2013. Thesis, Duke University. Accessed August 23, 2019. http://hdl.handle.net/10161/8204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Francisco, Aubrey Therese. “Laminin-Functionalized Polyethylene Glycol Hydrogels for Nucleus Pulposus Regeneration .” 2013. Web. 23 Aug 2019.

Vancouver:

Francisco AT. Laminin-Functionalized Polyethylene Glycol Hydrogels for Nucleus Pulposus Regeneration . [Internet] [Thesis]. Duke University; 2013. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10161/8204.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Francisco AT. Laminin-Functionalized Polyethylene Glycol Hydrogels for Nucleus Pulposus Regeneration . [Thesis]. Duke University; 2013. Available from: http://hdl.handle.net/10161/8204

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

10. Newsome, Laura Jean. Global Gene Expression Profiles and Proteomic Assessments in Adult Females with Obstructive Sleep Apnea Syndrome.

Degree: PhD, Human Nutrition, Foods, and Exercise, 2012, Virginia Tech

 Obstructive sleep apnea syndrome (OSAS) is a complex disorder characterized by repetitive bouts of upper airway collapse during sleep, causing subsequent intermittent hypoxia, hypercapnia, and… (more)

Subjects/Keywords: Obstructive Sleep Apnea Syndrome; gene expression profiling; Laminin cellular adhesion

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APA (6th Edition):

Newsome, L. J. (2012). Global Gene Expression Profiles and Proteomic Assessments in Adult Females with Obstructive Sleep Apnea Syndrome. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/77306

Chicago Manual of Style (16th Edition):

Newsome, Laura Jean. “Global Gene Expression Profiles and Proteomic Assessments in Adult Females with Obstructive Sleep Apnea Syndrome.” 2012. Doctoral Dissertation, Virginia Tech. Accessed August 23, 2019. http://hdl.handle.net/10919/77306.

MLA Handbook (7th Edition):

Newsome, Laura Jean. “Global Gene Expression Profiles and Proteomic Assessments in Adult Females with Obstructive Sleep Apnea Syndrome.” 2012. Web. 23 Aug 2019.

Vancouver:

Newsome LJ. Global Gene Expression Profiles and Proteomic Assessments in Adult Females with Obstructive Sleep Apnea Syndrome. [Internet] [Doctoral dissertation]. Virginia Tech; 2012. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10919/77306.

Council of Science Editors:

Newsome LJ. Global Gene Expression Profiles and Proteomic Assessments in Adult Females with Obstructive Sleep Apnea Syndrome. [Doctoral Dissertation]. Virginia Tech; 2012. Available from: http://hdl.handle.net/10919/77306


University of British Columbia

11. Viveiros, Ryan. An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegans .

Degree: 2008, University of British Columbia

 During C. elegans embryogenesis, myoblasts initially form two rows along the left and right lateral midlines and at ~290 min of development migrate dorsally and… (more)

Subjects/Keywords: Cell migration; Muscles; Laminin

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APA (6th Edition):

Viveiros, R. (2008). An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegans . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Viveiros, Ryan. “An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegans .” 2008. Thesis, University of British Columbia. Accessed August 23, 2019. http://hdl.handle.net/2429/631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Viveiros, Ryan. “An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegans .” 2008. Web. 23 Aug 2019.

Vancouver:

Viveiros R. An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegans . [Internet] [Thesis]. University of British Columbia; 2008. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/2429/631.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Viveiros R. An investigation into the genes mediating myoblast migration in the nematode : Caenorhabditis elegans . [Thesis]. University of British Columbia; 2008. Available from: http://hdl.handle.net/2429/631

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

12. Körner, Zandra. Congenital muscular dystrophy with laminin α2 chain-deficiency. Initiation of disease and development of treatment.

Degree: 2016, University of Lund

 Congenital muscle dystrophy type 1A (MDC1A) is a muscle disease caused bymutations in the LAMA2 gene, encoding the basement membrane protein lamininα2 chain. MDC1A patients… (more)

Subjects/Keywords: Cell- och molekylärbiologi; muskeldystrofi; Muscular dystrophy; Autophagy; proteasome; Laminin; MDC1A

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APA (6th Edition):

Körner, Z. (2016). Congenital muscular dystrophy with laminin α2 chain-deficiency. Initiation of disease and development of treatment. (Doctoral Dissertation). University of Lund. Retrieved from http://lup.lub.lu.se/record/2610e14d-b29e-4c7d-8285-5c14a32b1e18 ; http://portal.research.lu.se/ws/files/10296340/e_spik_Zandra_.pdf

Chicago Manual of Style (16th Edition):

Körner, Zandra. “Congenital muscular dystrophy with laminin α2 chain-deficiency. Initiation of disease and development of treatment.” 2016. Doctoral Dissertation, University of Lund. Accessed August 23, 2019. http://lup.lub.lu.se/record/2610e14d-b29e-4c7d-8285-5c14a32b1e18 ; http://portal.research.lu.se/ws/files/10296340/e_spik_Zandra_.pdf.

MLA Handbook (7th Edition):

Körner, Zandra. “Congenital muscular dystrophy with laminin α2 chain-deficiency. Initiation of disease and development of treatment.” 2016. Web. 23 Aug 2019.

Vancouver:

Körner Z. Congenital muscular dystrophy with laminin α2 chain-deficiency. Initiation of disease and development of treatment. [Internet] [Doctoral dissertation]. University of Lund; 2016. [cited 2019 Aug 23]. Available from: http://lup.lub.lu.se/record/2610e14d-b29e-4c7d-8285-5c14a32b1e18 ; http://portal.research.lu.se/ws/files/10296340/e_spik_Zandra_.pdf.

Council of Science Editors:

Körner Z. Congenital muscular dystrophy with laminin α2 chain-deficiency. Initiation of disease and development of treatment. [Doctoral Dissertation]. University of Lund; 2016. Available from: http://lup.lub.lu.se/record/2610e14d-b29e-4c7d-8285-5c14a32b1e18 ; http://portal.research.lu.se/ws/files/10296340/e_spik_Zandra_.pdf


University of Ottawa

13. Hind, Albadrani. Optimizing the Approach for Maintaining Single Muscle Fibers in Culture .

Degree: 2014, University of Ottawa

 The skeletal muscle is a dynamic tissue that has the ability to change and modify itself to fit the level of required activity; a phenomenon… (more)

Subjects/Keywords: Collagen and laminin; Matrigel; Muscle plasticity; Ca2+; Changing culture medium

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APA (6th Edition):

Hind, A. (2014). Optimizing the Approach for Maintaining Single Muscle Fibers in Culture . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31900

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hind, Albadrani. “Optimizing the Approach for Maintaining Single Muscle Fibers in Culture .” 2014. Thesis, University of Ottawa. Accessed August 23, 2019. http://hdl.handle.net/10393/31900.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hind, Albadrani. “Optimizing the Approach for Maintaining Single Muscle Fibers in Culture .” 2014. Web. 23 Aug 2019.

Vancouver:

Hind A. Optimizing the Approach for Maintaining Single Muscle Fibers in Culture . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10393/31900.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hind A. Optimizing the Approach for Maintaining Single Muscle Fibers in Culture . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31900

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Houston

14. -0356-3541. Investigation on Conducting Polymers-Doped with Laminin.

Degree: Biomedical Engineering, Department of, University of Houston

 Conducting polymers (CPs) have been used in biomedical science and engineering research field with wide applications such as biosensors/ actuators, drug delivery systems, and implantable… (more)

Subjects/Keywords: Conducting polymer; Laminin peptide

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APA (6th Edition):

-0356-3541. (n.d.). Investigation on Conducting Polymers-Doped with Laminin. (Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/2934

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

-0356-3541. “Investigation on Conducting Polymers-Doped with Laminin.” Thesis, University of Houston. Accessed August 23, 2019. http://hdl.handle.net/10657/2934.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

-0356-3541. “Investigation on Conducting Polymers-Doped with Laminin.” Web. 23 Aug 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
No year of publication.

Vancouver:

-0356-3541. Investigation on Conducting Polymers-Doped with Laminin. [Internet] [Thesis]. University of Houston; [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10657/2934.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

-0356-3541. Investigation on Conducting Polymers-Doped with Laminin. [Thesis]. University of Houston; Available from: http://hdl.handle.net/10657/2934

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Minnesota

15. San Miguel-Ruiz, Jose Enrique. The role of the actin-binding protein Arp2/3 in growth cone actin dynamics and guidance.

Degree: PhD, Neuroscience, 2014, University of Minnesota

 Neuronal growth cones are responsible for wiring the immature nervous system. They rely on guidance cues present in the extracellular environment to guide axons to… (more)

Subjects/Keywords: Actin; Actin binding proteins; Arp2/3; Axon guidance; L1; Laminin

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APA (6th Edition):

San Miguel-Ruiz, J. E. (2014). The role of the actin-binding protein Arp2/3 in growth cone actin dynamics and guidance. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/163895

Chicago Manual of Style (16th Edition):

San Miguel-Ruiz, Jose Enrique. “The role of the actin-binding protein Arp2/3 in growth cone actin dynamics and guidance.” 2014. Doctoral Dissertation, University of Minnesota. Accessed August 23, 2019. http://hdl.handle.net/11299/163895.

MLA Handbook (7th Edition):

San Miguel-Ruiz, Jose Enrique. “The role of the actin-binding protein Arp2/3 in growth cone actin dynamics and guidance.” 2014. Web. 23 Aug 2019.

Vancouver:

San Miguel-Ruiz JE. The role of the actin-binding protein Arp2/3 in growth cone actin dynamics and guidance. [Internet] [Doctoral dissertation]. University of Minnesota; 2014. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/11299/163895.

Council of Science Editors:

San Miguel-Ruiz JE. The role of the actin-binding protein Arp2/3 in growth cone actin dynamics and guidance. [Doctoral Dissertation]. University of Minnesota; 2014. Available from: http://hdl.handle.net/11299/163895

16. Lefrançois, Louise. Etude des adhésines HBHA et LBP impliquées dans l'interaction de Mycobacterium avium ssp. paratuberculosis avec les cellules épithéliales intestinales, cibles privilégiées de la bactérie in vivo : Characterization of HBHA and LBP adhesins involved in the interaction of Mycobacterium avium subsp. paratuberculosis with intestinal epithelial cells, the preferential target in vivo.

Degree: Docteur es, Sciences de la vie, 2012, Université François-Rabelais de Tours

Mycobacterium avium ssp. paratuberculosis (Map), agent étiologique de la paratuberculose, a évolué en deuxtypes dénommés, S pour« Sheep » et C pour « Cattle ».… (more)

Subjects/Keywords: Paratuberculose; Mycobacterium avium ssp; Paratuberculosis; Adhésines; Heparin Binding HemAgglutinin (HBHA); Laminin Binding Protein (LBP); Polymorphisme; Paratuberculosis; Mycobacterium avium subsp; Paratuberculosis; Adhesins; Heparin Binding HemAgglutinin (HBHA); Laminin Binding Protein (LBP); Polymorphism

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APA (6th Edition):

Lefrançois, L. (2012). Etude des adhésines HBHA et LBP impliquées dans l'interaction de Mycobacterium avium ssp. paratuberculosis avec les cellules épithéliales intestinales, cibles privilégiées de la bactérie in vivo : Characterization of HBHA and LBP adhesins involved in the interaction of Mycobacterium avium subsp. paratuberculosis with intestinal epithelial cells, the preferential target in vivo. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2012TOUR4020

Chicago Manual of Style (16th Edition):

Lefrançois, Louise. “Etude des adhésines HBHA et LBP impliquées dans l'interaction de Mycobacterium avium ssp. paratuberculosis avec les cellules épithéliales intestinales, cibles privilégiées de la bactérie in vivo : Characterization of HBHA and LBP adhesins involved in the interaction of Mycobacterium avium subsp. paratuberculosis with intestinal epithelial cells, the preferential target in vivo.” 2012. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed August 23, 2019. http://www.theses.fr/2012TOUR4020.

MLA Handbook (7th Edition):

Lefrançois, Louise. “Etude des adhésines HBHA et LBP impliquées dans l'interaction de Mycobacterium avium ssp. paratuberculosis avec les cellules épithéliales intestinales, cibles privilégiées de la bactérie in vivo : Characterization of HBHA and LBP adhesins involved in the interaction of Mycobacterium avium subsp. paratuberculosis with intestinal epithelial cells, the preferential target in vivo.” 2012. Web. 23 Aug 2019.

Vancouver:

Lefrançois L. Etude des adhésines HBHA et LBP impliquées dans l'interaction de Mycobacterium avium ssp. paratuberculosis avec les cellules épithéliales intestinales, cibles privilégiées de la bactérie in vivo : Characterization of HBHA and LBP adhesins involved in the interaction of Mycobacterium avium subsp. paratuberculosis with intestinal epithelial cells, the preferential target in vivo. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2012. [cited 2019 Aug 23]. Available from: http://www.theses.fr/2012TOUR4020.

Council of Science Editors:

Lefrançois L. Etude des adhésines HBHA et LBP impliquées dans l'interaction de Mycobacterium avium ssp. paratuberculosis avec les cellules épithéliales intestinales, cibles privilégiées de la bactérie in vivo : Characterization of HBHA and LBP adhesins involved in the interaction of Mycobacterium avium subsp. paratuberculosis with intestinal epithelial cells, the preferential target in vivo. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2012. Available from: http://www.theses.fr/2012TOUR4020


Ruhr Universität Bochum

17. Radacz, Yvonne. Rekonstitution epithelialer Eigenschaften und Funktionen von Kolonkarzinomzellen nach Reexpression des Tumorsuppressors Smad4.

Degree: 2005, Ruhr Universität Bochum

 Smad4 ist ein Tumorsuppressor der häufig in Tumoren des Gastrointestinaltraktes inaktiviert ist. Seine stabile Transfektion in Smad4-defiziente Kolonkarzinomzellen bewirkte eine Modulation des Wachstums sowie des… (more)

Subjects/Keywords: Dickdarmkrebs; Zellmigration; Invasives Wachstum; Zelladhäsion; Laminin

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APA (6th Edition):

Radacz, Y. (2005). Rekonstitution epithelialer Eigenschaften und Funktionen von Kolonkarzinomzellen nach Reexpression des Tumorsuppressors Smad4. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-13636

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radacz, Yvonne. “Rekonstitution epithelialer Eigenschaften und Funktionen von Kolonkarzinomzellen nach Reexpression des Tumorsuppressors Smad4.” 2005. Thesis, Ruhr Universität Bochum. Accessed August 23, 2019. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-13636.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radacz, Yvonne. “Rekonstitution epithelialer Eigenschaften und Funktionen von Kolonkarzinomzellen nach Reexpression des Tumorsuppressors Smad4.” 2005. Web. 23 Aug 2019.

Vancouver:

Radacz Y. Rekonstitution epithelialer Eigenschaften und Funktionen von Kolonkarzinomzellen nach Reexpression des Tumorsuppressors Smad4. [Internet] [Thesis]. Ruhr Universität Bochum; 2005. [cited 2019 Aug 23]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-13636.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radacz Y. Rekonstitution epithelialer Eigenschaften und Funktionen von Kolonkarzinomzellen nach Reexpression des Tumorsuppressors Smad4. [Thesis]. Ruhr Universität Bochum; 2005. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-13636

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Ruhr Universität Bochum

18. Zboralski, Dirk. Zur Rolle des Tumorsuppressors Smad4 un der Expressionsregulation von Laminin-332 als Inhibitor und Aktivator der Tumorinvasion.

Degree: 2008, Ruhr Universität Bochum

 Die Basalmembran (BM)-Komponente Laminin-332, die sich aus einer α3-, β3- und γ2-Kette zusammensetzt, spielt physiologisch und pathophysiologisch eine wichtige Rolle. Im Rahmen dieser Arbeit wurde… (more)

Subjects/Keywords: Tumorsuppressor-Gen; Basalmembran; Laminin; Transforming Growth Factor beta; Tumor-Nekrose-Faktor (alpha)

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APA (6th Edition):

Zboralski, D. (2008). Zur Rolle des Tumorsuppressors Smad4 un der Expressionsregulation von Laminin-332 als Inhibitor und Aktivator der Tumorinvasion. (Thesis). Ruhr Universität Bochum. Retrieved from http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-24364

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zboralski, Dirk. “Zur Rolle des Tumorsuppressors Smad4 un der Expressionsregulation von Laminin-332 als Inhibitor und Aktivator der Tumorinvasion.” 2008. Thesis, Ruhr Universität Bochum. Accessed August 23, 2019. http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-24364.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zboralski, Dirk. “Zur Rolle des Tumorsuppressors Smad4 un der Expressionsregulation von Laminin-332 als Inhibitor und Aktivator der Tumorinvasion.” 2008. Web. 23 Aug 2019.

Vancouver:

Zboralski D. Zur Rolle des Tumorsuppressors Smad4 un der Expressionsregulation von Laminin-332 als Inhibitor und Aktivator der Tumorinvasion. [Internet] [Thesis]. Ruhr Universität Bochum; 2008. [cited 2019 Aug 23]. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-24364.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zboralski D. Zur Rolle des Tumorsuppressors Smad4 un der Expressionsregulation von Laminin-332 als Inhibitor und Aktivator der Tumorinvasion. [Thesis]. Ruhr Universität Bochum; 2008. Available from: http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:hbz:294-24364

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Aline Rossi da Silveira. Análise por modelagem e dinâmica molecular da interação entre a integrina α6β1 e a laminina 111 humana.

Degree: 2007, Laboratório Nacional de Computação Científica

A matriz extracelular (ECM) é definida como um complexo de proteínas e glicoproteínas que envolve as células nos mais diversos tecidos. A laminina é uma… (more)

Subjects/Keywords: Integrina; Estrutura molecular; Laminina; Molecular structure; Integrin; Laminin; BIOLOGIA MOLECULAR; BIOLOGIA MOLECULAR

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APA (6th Edition):

Silveira, A. R. d. (2007). Análise por modelagem e dinâmica molecular da interação entre a integrina α6β1 e a laminina 111 humana. (Masters Thesis). Laboratório Nacional de Computação Científica. Retrieved from http://www.lncc.br/tdmc/tde_busca/arquivo.php?codArquivo=130

Chicago Manual of Style (16th Edition):

Silveira, Aline Rossi da. “Análise por modelagem e dinâmica molecular da interação entre a integrina α6β1 e a laminina 111 humana.” 2007. Masters Thesis, Laboratório Nacional de Computação Científica. Accessed August 23, 2019. http://www.lncc.br/tdmc/tde_busca/arquivo.php?codArquivo=130.

MLA Handbook (7th Edition):

Silveira, Aline Rossi da. “Análise por modelagem e dinâmica molecular da interação entre a integrina α6β1 e a laminina 111 humana.” 2007. Web. 23 Aug 2019.

Vancouver:

Silveira ARd. Análise por modelagem e dinâmica molecular da interação entre a integrina α6β1 e a laminina 111 humana. [Internet] [Masters thesis]. Laboratório Nacional de Computação Científica; 2007. [cited 2019 Aug 23]. Available from: http://www.lncc.br/tdmc/tde_busca/arquivo.php?codArquivo=130.

Council of Science Editors:

Silveira ARd. Análise por modelagem e dinâmica molecular da interação entre a integrina α6β1 e a laminina 111 humana. [Masters Thesis]. Laboratório Nacional de Computação Científica; 2007. Available from: http://www.lncc.br/tdmc/tde_busca/arquivo.php?codArquivo=130


University of Washington

20. Rasmussen, Jeffrey Philip. Epithelial morphogenesis of the Caenorhabditis elegans pharynx.

Degree: PhD, 2012, University of Washington

 The assembly of cells into functional organs requires the coordination of cell shape and polarity with organ architecture. Although defects in cell shape and polarity… (more)

Subjects/Keywords: Caenorhabditis elegans; epithelial polarity; fusogen; laminin; mesenchymal to epithelial transition; tubulogenesis; Molecular and cellular biology

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APA (6th Edition):

Rasmussen, J. P. (2012). Epithelial morphogenesis of the Caenorhabditis elegans pharynx. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/19742

Chicago Manual of Style (16th Edition):

Rasmussen, Jeffrey Philip. “Epithelial morphogenesis of the Caenorhabditis elegans pharynx.” 2012. Doctoral Dissertation, University of Washington. Accessed August 23, 2019. http://hdl.handle.net/1773/19742.

MLA Handbook (7th Edition):

Rasmussen, Jeffrey Philip. “Epithelial morphogenesis of the Caenorhabditis elegans pharynx.” 2012. Web. 23 Aug 2019.

Vancouver:

Rasmussen JP. Epithelial morphogenesis of the Caenorhabditis elegans pharynx. [Internet] [Doctoral dissertation]. University of Washington; 2012. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1773/19742.

Council of Science Editors:

Rasmussen JP. Epithelial morphogenesis of the Caenorhabditis elegans pharynx. [Doctoral Dissertation]. University of Washington; 2012. Available from: http://hdl.handle.net/1773/19742


Duke University

21. Hwang, Priscilla Y. Cadherin-Mediated Cell-Cell Interactions Regulates Phenotype And Morphology of Nucleus Pulposus Cells Of The Intervertebral Disc .

Degree: 2015, Duke University

  Juvenile nucleus pulposus (NP) cells of the intervertebral disc (IVD) are large, vacuolated cells that form cell clusters with numerous cell-cell interactions. With maturation… (more)

Subjects/Keywords: Biomedical engineering; Biomechanics; intervertebral disc; laminin; microenvironmental cues; N-cadherin; nucleus pulposus; polyethylene glycol (PEG)

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APA (6th Edition):

Hwang, P. Y. (2015). Cadherin-Mediated Cell-Cell Interactions Regulates Phenotype And Morphology of Nucleus Pulposus Cells Of The Intervertebral Disc . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hwang, Priscilla Y. “Cadherin-Mediated Cell-Cell Interactions Regulates Phenotype And Morphology of Nucleus Pulposus Cells Of The Intervertebral Disc .” 2015. Thesis, Duke University. Accessed August 23, 2019. http://hdl.handle.net/10161/9870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hwang, Priscilla Y. “Cadherin-Mediated Cell-Cell Interactions Regulates Phenotype And Morphology of Nucleus Pulposus Cells Of The Intervertebral Disc .” 2015. Web. 23 Aug 2019.

Vancouver:

Hwang PY. Cadherin-Mediated Cell-Cell Interactions Regulates Phenotype And Morphology of Nucleus Pulposus Cells Of The Intervertebral Disc . [Internet] [Thesis]. Duke University; 2015. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10161/9870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hwang PY. Cadherin-Mediated Cell-Cell Interactions Regulates Phenotype And Morphology of Nucleus Pulposus Cells Of The Intervertebral Disc . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/9870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

22. Bridgen, Devin. Regulation of Human Nucleus Pulposus Cell Phenotype and Behavior by Laminin-Mimetic Peptide Substrates .

Degree: 2015, Duke University

  Intervertebral disc (IVD) disorders can cause pain and disability for affected individuals. A subset of IVD disorders are thought to originate in the nucleus… (more)

Subjects/Keywords: Biomedical engineering; Integrin; Intervertebral disc; Laminin peptide; Nucleus pulposus; Polyacrylamide; Substrate stiffness

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APA (6th Edition):

Bridgen, D. (2015). Regulation of Human Nucleus Pulposus Cell Phenotype and Behavior by Laminin-Mimetic Peptide Substrates . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/10513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bridgen, Devin. “Regulation of Human Nucleus Pulposus Cell Phenotype and Behavior by Laminin-Mimetic Peptide Substrates .” 2015. Thesis, Duke University. Accessed August 23, 2019. http://hdl.handle.net/10161/10513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bridgen, Devin. “Regulation of Human Nucleus Pulposus Cell Phenotype and Behavior by Laminin-Mimetic Peptide Substrates .” 2015. Web. 23 Aug 2019.

Vancouver:

Bridgen D. Regulation of Human Nucleus Pulposus Cell Phenotype and Behavior by Laminin-Mimetic Peptide Substrates . [Internet] [Thesis]. Duke University; 2015. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10161/10513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bridgen D. Regulation of Human Nucleus Pulposus Cell Phenotype and Behavior by Laminin-Mimetic Peptide Substrates . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/10513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waterloo

23. Williams, Stacey. α-Dystroglycan is essential for the induction of Egr3, a transcription factor important in muscle spindle formation.

Degree: 2009, University of Waterloo

 Muscle spindle fibers are specialized stretch receptors that allow the perception and coordination of limb movement. Differentiation of muscle spindles is initiated by signals derived… (more)

Subjects/Keywords: muscle spindle; agrin; laminin; α-Dystroglycan

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APA (6th Edition):

Williams, S. (2009). α-Dystroglycan is essential for the induction of Egr3, a transcription factor important in muscle spindle formation. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/4814

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Stacey. “α-Dystroglycan is essential for the induction of Egr3, a transcription factor important in muscle spindle formation.” 2009. Thesis, University of Waterloo. Accessed August 23, 2019. http://hdl.handle.net/10012/4814.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Stacey. “α-Dystroglycan is essential for the induction of Egr3, a transcription factor important in muscle spindle formation.” 2009. Web. 23 Aug 2019.

Vancouver:

Williams S. α-Dystroglycan is essential for the induction of Egr3, a transcription factor important in muscle spindle formation. [Internet] [Thesis]. University of Waterloo; 2009. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/10012/4814.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams S. α-Dystroglycan is essential for the induction of Egr3, a transcription factor important in muscle spindle formation. [Thesis]. University of Waterloo; 2009. Available from: http://hdl.handle.net/10012/4814

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

24. Anderson, Jennifer Louise. Cerebellar synaptic plasticity in two animal models of muscular dystrophy.

Degree: Medical Sciences, 2008, University of New South Wales

 Duchenne muscular dystrophy (DMD) and congenital muscular dystrophy 1A (MDC1A) are the two most common forms of muscular dystrophy in humans, caused by mutations in… (more)

Subjects/Keywords: laminin alpha2; muscular dystrophy; dystrophin; cerebellum

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APA (6th Edition):

Anderson, J. L. (2008). Cerebellar synaptic plasticity in two animal models of muscular dystrophy. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Anderson, Jennifer Louise. “Cerebellar synaptic plasticity in two animal models of muscular dystrophy.” 2008. Doctoral Dissertation, University of New South Wales. Accessed August 23, 2019. http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true.

MLA Handbook (7th Edition):

Anderson, Jennifer Louise. “Cerebellar synaptic plasticity in two animal models of muscular dystrophy.” 2008. Web. 23 Aug 2019.

Vancouver:

Anderson JL. Cerebellar synaptic plasticity in two animal models of muscular dystrophy. [Internet] [Doctoral dissertation]. University of New South Wales; 2008. [cited 2019 Aug 23]. Available from: http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true.

Council of Science Editors:

Anderson JL. Cerebellar synaptic plasticity in two animal models of muscular dystrophy. [Doctoral Dissertation]. University of New South Wales; 2008. Available from: http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true

25. Smuczek, Basilio. Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama.

Degree: Mestrado, Biologia Celular e Tecidual, 2014, University of São Paulo

O câncer de mama é importante problema de saúde pública. O microambiente onde as células cancerígenas se encontram possui moléculas como a laminina e seus… (more)

Subjects/Keywords: Breast cancer; C16 peptide; Câncer de mama; Extracellular matrix; GPNMB; GPNMB; Laminin; Laminina; Matriz extracelular; Peptídeo C16; SPOCK; SPOCK

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APA (6th Edition):

Smuczek, B. (2014). Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;

Chicago Manual of Style (16th Edition):

Smuczek, Basilio. “Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama.” 2014. Masters Thesis, University of São Paulo. Accessed August 23, 2019. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;.

MLA Handbook (7th Edition):

Smuczek, Basilio. “Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama.” 2014. Web. 23 Aug 2019.

Vancouver:

Smuczek B. Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2019 Aug 23]. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;.

Council of Science Editors:

Smuczek B. Peptídeo C16, derivado da laminina, regulando a expressão de potenciais biomarcadorers do câncer de mama. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/42/42134/tde-20022015-090950/ ;


University of Kentucky

26. Grohs, Gillian. THE EFFECTS OF EXERCISE PRECONDITIONING ON FOCAL ISCHEMIC STROKE.

Degree: 2017, University of Kentucky

 Cleaved fragments of the extracellular matrix protein perlecan have been shown to promote neuroprotection and repair after ischemic stroke. The cysteine proteases cathepsin B and… (more)

Subjects/Keywords: Ischemic Stroke; Exercise Preconditioning; Neurovascular Unit; Perlecan Laminin-like Globular Domain 3; Skeletal Muscle; Neurosciences; Physiological Processes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Grohs, G. (2017). THE EFFECTS OF EXERCISE PRECONDITIONING ON FOCAL ISCHEMIC STROKE. (Masters Thesis). University of Kentucky. Retrieved from http://uknowledge.uky.edu/medsci_etds/8

Chicago Manual of Style (16th Edition):

Grohs, Gillian. “THE EFFECTS OF EXERCISE PRECONDITIONING ON FOCAL ISCHEMIC STROKE.” 2017. Masters Thesis, University of Kentucky. Accessed August 23, 2019. http://uknowledge.uky.edu/medsci_etds/8.

MLA Handbook (7th Edition):

Grohs, Gillian. “THE EFFECTS OF EXERCISE PRECONDITIONING ON FOCAL ISCHEMIC STROKE.” 2017. Web. 23 Aug 2019.

Vancouver:

Grohs G. THE EFFECTS OF EXERCISE PRECONDITIONING ON FOCAL ISCHEMIC STROKE. [Internet] [Masters thesis]. University of Kentucky; 2017. [cited 2019 Aug 23]. Available from: http://uknowledge.uky.edu/medsci_etds/8.

Council of Science Editors:

Grohs G. THE EFFECTS OF EXERCISE PRECONDITIONING ON FOCAL ISCHEMIC STROKE. [Masters Thesis]. University of Kentucky; 2017. Available from: http://uknowledge.uky.edu/medsci_etds/8


Université de Montréal

27. Viegas Costa, Luiz Claudio. Étude du rôle de la membrane basale spécialisée dans la maturation de l'émail .

Degree: 2016, Université de Montréal

 Les cellules épithéliales qui produisent l’émail, les améloblastes, sont séparées de l'émail au niveau de la zone de maturation par une membrane basale spécialisée (MBS)… (more)

Subjects/Keywords: Amélogénèse; Membrane basale spécialisée; Maturation de l’émail; Souris humanisées; Laminine γ2; Amelogenesis; Basement membrane; Enamel maturation; Humanized mouse model; Laminin γ2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Viegas Costa, L. C. (2016). Étude du rôle de la membrane basale spécialisée dans la maturation de l'émail . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/16272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Viegas Costa, Luiz Claudio. “Étude du rôle de la membrane basale spécialisée dans la maturation de l'émail .” 2016. Thesis, Université de Montréal. Accessed August 23, 2019. http://hdl.handle.net/1866/16272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Viegas Costa, Luiz Claudio. “Étude du rôle de la membrane basale spécialisée dans la maturation de l'émail .” 2016. Web. 23 Aug 2019.

Vancouver:

Viegas Costa LC. Étude du rôle de la membrane basale spécialisée dans la maturation de l'émail . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1866/16272.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Viegas Costa LC. Étude du rôle de la membrane basale spécialisée dans la maturation de l'émail . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/16272

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rockefeller University

28. Yu, Wei-Ming. Role of Laminins in the Development of the Peripheral Nervous System.

Degree: 2007, Rockefeller University

  To investigate the function of laminins in peripheral nerve development, the laminin γ1 gene was specifically disrupted in Schwann cells. Disruption of laminin γ1… (more)

Subjects/Keywords: Schwann cells; laminin y1 gene; nerve development; Life Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, W. (2007). Role of Laminins in the Development of the Peripheral Nervous System. (Masters Thesis). Rockefeller University. Retrieved from http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/30

Chicago Manual of Style (16th Edition):

Yu, Wei-Ming. “Role of Laminins in the Development of the Peripheral Nervous System.” 2007. Masters Thesis, Rockefeller University. Accessed August 23, 2019. http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/30.

MLA Handbook (7th Edition):

Yu, Wei-Ming. “Role of Laminins in the Development of the Peripheral Nervous System.” 2007. Web. 23 Aug 2019.

Vancouver:

Yu W. Role of Laminins in the Development of the Peripheral Nervous System. [Internet] [Masters thesis]. Rockefeller University; 2007. [cited 2019 Aug 23]. Available from: http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/30.

Council of Science Editors:

Yu W. Role of Laminins in the Development of the Peripheral Nervous System. [Masters Thesis]. Rockefeller University; 2007. Available from: http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/30


Rockefeller University

29. Haegeli, Veronique. Role of Laminin gamma 1 in Cortical Development.

Degree: 2006, Rockefeller University

  Laminins are a major component of basement membranes, including those in the central nervous system (CNS). To explore the role of laminin \l, a… (more)

Subjects/Keywords: laminins; laminin gamma 1; cerebral cortex development; Life Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haegeli, V. (2006). Role of Laminin gamma 1 in Cortical Development. (Masters Thesis). Rockefeller University. Retrieved from http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/52

Chicago Manual of Style (16th Edition):

Haegeli, Veronique. “Role of Laminin gamma 1 in Cortical Development.” 2006. Masters Thesis, Rockefeller University. Accessed August 23, 2019. http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/52.

MLA Handbook (7th Edition):

Haegeli, Veronique. “Role of Laminin gamma 1 in Cortical Development.” 2006. Web. 23 Aug 2019.

Vancouver:

Haegeli V. Role of Laminin gamma 1 in Cortical Development. [Internet] [Masters thesis]. Rockefeller University; 2006. [cited 2019 Aug 23]. Available from: http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/52.

Council of Science Editors:

Haegeli V. Role of Laminin gamma 1 in Cortical Development. [Masters Thesis]. Rockefeller University; 2006. Available from: http://digitalcommons.rockefeller.edu/student_theses_and_dissertations/52


Georgia Tech

30. Dodla, Mahesh Chandra. Bioengineered Scaffolds for Peripheral Nerve Regeneration.

Degree: PhD, Biomedical Engineering, 2007, Georgia Tech

 Nerve autografts are widely used clinically to repair nerve grafts. However, nerve grafts have many limitations, such as, availability of donor nerve grafts, and loss… (more)

Subjects/Keywords: Agarose hydrogels; Laminin; Nerve growth factor; Nerve regeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dodla, M. C. (2007). Bioengineered Scaffolds for Peripheral Nerve Regeneration. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/14504

Chicago Manual of Style (16th Edition):

Dodla, Mahesh Chandra. “Bioengineered Scaffolds for Peripheral Nerve Regeneration.” 2007. Doctoral Dissertation, Georgia Tech. Accessed August 23, 2019. http://hdl.handle.net/1853/14504.

MLA Handbook (7th Edition):

Dodla, Mahesh Chandra. “Bioengineered Scaffolds for Peripheral Nerve Regeneration.” 2007. Web. 23 Aug 2019.

Vancouver:

Dodla MC. Bioengineered Scaffolds for Peripheral Nerve Regeneration. [Internet] [Doctoral dissertation]. Georgia Tech; 2007. [cited 2019 Aug 23]. Available from: http://hdl.handle.net/1853/14504.

Council of Science Editors:

Dodla MC. Bioengineered Scaffolds for Peripheral Nerve Regeneration. [Doctoral Dissertation]. Georgia Tech; 2007. Available from: http://hdl.handle.net/1853/14504

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