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You searched for subject:(kras). Showing records 1 – 30 of 146 total matches.

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Brno University of Technology

1. Buryška, Petr. Staré Brno - urbanisticko architektonická studie revitalizace města .

Degree: 2009, Brno University of Technology

Revitalizace brownfieldu; Brownfield revitalization Advisors/Committee Members: Františák, Luboš (advisor).

Subjects/Keywords: Kras; Kras

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Buryška, P. (2009). Staré Brno - urbanisticko architektonická studie revitalizace města . (Thesis). Brno University of Technology. Retrieved from http://hdl.handle.net/11012/7815

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Buryška, Petr. “Staré Brno - urbanisticko architektonická studie revitalizace města .” 2009. Thesis, Brno University of Technology. Accessed October 19, 2019. http://hdl.handle.net/11012/7815.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Buryška, Petr. “Staré Brno - urbanisticko architektonická studie revitalizace města .” 2009. Web. 19 Oct 2019.

Vancouver:

Buryška P. Staré Brno - urbanisticko architektonická studie revitalizace města . [Internet] [Thesis]. Brno University of Technology; 2009. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/11012/7815.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Buryška P. Staré Brno - urbanisticko architektonická studie revitalizace města . [Thesis]. Brno University of Technology; 2009. Available from: http://hdl.handle.net/11012/7815

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Minho

2. Carvalho, Patrícia Dias. Exploring galectin-3-KRASmut-p16ink4a interplay in colorectal cancer .

Degree: 2014, Universidade do Minho

KRAS is the most frequently mutated RAS isoform in many cancers, including colorectal cancer (CRC; 30-50% of the cases), which is a leading cause of… (more)

Subjects/Keywords: KRAS; Gal-3; p16; KRAS mutations; Interaction; Colorectal cancer; Mutações KRAS; Interação; Cancro colorretal

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APA (6th Edition):

Carvalho, P. D. (2014). Exploring galectin-3-KRASmut-p16ink4a interplay in colorectal cancer . (Masters Thesis). Universidade do Minho. Retrieved from http://hdl.handle.net/1822/34581

Chicago Manual of Style (16th Edition):

Carvalho, Patrícia Dias. “Exploring galectin-3-KRASmut-p16ink4a interplay in colorectal cancer .” 2014. Masters Thesis, Universidade do Minho. Accessed October 19, 2019. http://hdl.handle.net/1822/34581.

MLA Handbook (7th Edition):

Carvalho, Patrícia Dias. “Exploring galectin-3-KRASmut-p16ink4a interplay in colorectal cancer .” 2014. Web. 19 Oct 2019.

Vancouver:

Carvalho PD. Exploring galectin-3-KRASmut-p16ink4a interplay in colorectal cancer . [Internet] [Masters thesis]. Universidade do Minho; 2014. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/1822/34581.

Council of Science Editors:

Carvalho PD. Exploring galectin-3-KRASmut-p16ink4a interplay in colorectal cancer . [Masters Thesis]. Universidade do Minho; 2014. Available from: http://hdl.handle.net/1822/34581


Vilnius University

3. Džervienė, Elena. Sergančiųjų išplitusiu storosios žarnos vėžiu dažniausių KRAS geno mutacijų nustatymas tikro laiko polimerazės grandininės reakcijos metodu.

Degree: Master, 2014, Vilnius University

SANTRAUKA Darbo autorė: Elena Džervienė Darbo vadovas: prof. dr. Arvydas Laurinavičius Vilnius, 2011 m. Pagrindinės sąvokos: KRAS geno mutacijos, TL-PGR. Tyrimo tikslas - įvertinti dažniausias… (more)

Subjects/Keywords: KRAS geno mutacijos; TL-PGR

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APA (6th Edition):

Džervienė, Elena. (2014). Sergančiųjų išplitusiu storosios žarnos vėžiu dažniausių KRAS geno mutacijų nustatymas tikro laiko polimerazės grandininės reakcijos metodu. (Masters Thesis). Vilnius University. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20140627_165835-85990 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Džervienė, Elena. “Sergančiųjų išplitusiu storosios žarnos vėžiu dažniausių KRAS geno mutacijų nustatymas tikro laiko polimerazės grandininės reakcijos metodu.” 2014. Masters Thesis, Vilnius University. Accessed October 19, 2019. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20140627_165835-85990 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Džervienė, Elena. “Sergančiųjų išplitusiu storosios žarnos vėžiu dažniausių KRAS geno mutacijų nustatymas tikro laiko polimerazės grandininės reakcijos metodu.” 2014. Web. 19 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Džervienė, Elena. Sergančiųjų išplitusiu storosios žarnos vėžiu dažniausių KRAS geno mutacijų nustatymas tikro laiko polimerazės grandininės reakcijos metodu. [Internet] [Masters thesis]. Vilnius University; 2014. [cited 2019 Oct 19]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20140627_165835-85990 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Džervienė, Elena. Sergančiųjų išplitusiu storosios žarnos vėžiu dažniausių KRAS geno mutacijų nustatymas tikro laiko polimerazės grandininės reakcijos metodu. [Masters Thesis]. Vilnius University; 2014. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20140627_165835-85990 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Univerzitet u Beogradu

4. Nikolić, Aleksandra, 1975-. Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka.

Degree: Biološki fakultet, 2014, Univerzitet u Beogradu

Molekularna biologija / Molecular biology

Karcinom pankreasa spada u najinvazivnije i najprogresivnije humane maligne bolesti. Genetske promene karakteristične za ovu bolest su aktivacija onkogena KRAS(more)

Subjects/Keywords: pancreatic cancer; SMAD4; KRAS

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APA (6th Edition):

Nikolić, Aleksandra, 1. (2014). Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nikolić, Aleksandra, 1975-. “Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka.” 2014. Thesis, Univerzitet u Beogradu. Accessed October 19, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nikolić, Aleksandra, 1975-. “Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka.” 2014. Web. 19 Oct 2019.

Vancouver:

Nikolić, Aleksandra 1. Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka. [Internet] [Thesis]. Univerzitet u Beogradu; 2014. [cited 2019 Oct 19]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nikolić, Aleksandra 1. Strukturna i funkcionalna analiza 5' regulatornog regiona gena SMAD4 u karcinomu pankreasa čoveka. [Thesis]. Univerzitet u Beogradu; 2014. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

5. Sasine, Joshua P. KRAS Expands and Exhausts Hematopoietic Stem Cells.

Degree: Molec, Cell, & Integ Physiology, 2018, UCLA

 Expressing the oncogenic mutant form of Kras in hematopoietic stem cells (HSCs) induces a rapidly fatal myeloproliferative neoplasm in mice, suggesting Kras signaling plays a… (more)

Subjects/Keywords: Biology; Hematopoietic Stem Cell; KRAS

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APA (6th Edition):

Sasine, J. P. (2018). KRAS Expands and Exhausts Hematopoietic Stem Cells. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/5zr267g2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sasine, Joshua P. “KRAS Expands and Exhausts Hematopoietic Stem Cells.” 2018. Thesis, UCLA. Accessed October 19, 2019. http://www.escholarship.org/uc/item/5zr267g2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sasine, Joshua P. “KRAS Expands and Exhausts Hematopoietic Stem Cells.” 2018. Web. 19 Oct 2019.

Vancouver:

Sasine JP. KRAS Expands and Exhausts Hematopoietic Stem Cells. [Internet] [Thesis]. UCLA; 2018. [cited 2019 Oct 19]. Available from: http://www.escholarship.org/uc/item/5zr267g2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sasine JP. KRAS Expands and Exhausts Hematopoietic Stem Cells. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/5zr267g2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

6. Amato, Katherine Renee. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.

Degree: PhD, Cancer Biology, 2015, Vanderbilt University

 Lung cancer remains the leading cause of cancer related deaths in the United States despite a significant number of advancements in the molecular diagnosis and… (more)

Subjects/Keywords: TKI; EPH; KRAS; EGFR

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APA (6th Edition):

Amato, K. R. (2015). Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03232015-094458/ ;

Chicago Manual of Style (16th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed October 19, 2019. http://etd.library.vanderbilt.edu/available/etd-03232015-094458/ ;.

MLA Handbook (7th Edition):

Amato, Katherine Renee. “Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer.” 2015. Web. 19 Oct 2019.

Vancouver:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2019 Oct 19]. Available from: http://etd.library.vanderbilt.edu/available/etd-03232015-094458/ ;.

Council of Science Editors:

Amato KR. Targeting the EPHA2 receptor tyrosine kinase in KRAS and EGFR mutant lung cancer. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-03232015-094458/ ;


Univerzitet u Beogradu

7. Jakovljević, Ksenija V. 1981-. Analiza učestalosti i tipova mutacija KRAS i BRAF gena u karcinomima kolorektuma u populaciji Srbije.

Degree: Biološki fakultet, 2012, Univerzitet u Beogradu

Molekularna biologija - molekularna genetika / Molecular biology - molecular genetics

Uvod: Kolorektalni kancer (CRC) se po svojoj učestalosti i smrtnosti nalazi u samom vrhu… (more)

Subjects/Keywords: Colorectal carcinoma; KRAS gene; BRAF gene; mutations

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APA (6th Edition):

Jakovljević, K. V. 1. (2012). Analiza učestalosti i tipova mutacija KRAS i BRAF gena u karcinomima kolorektuma u populaciji Srbije. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:2342/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jakovljević, Ksenija V 1981-. “Analiza učestalosti i tipova mutacija KRAS i BRAF gena u karcinomima kolorektuma u populaciji Srbije.” 2012. Thesis, Univerzitet u Beogradu. Accessed October 19, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:2342/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jakovljević, Ksenija V 1981-. “Analiza učestalosti i tipova mutacija KRAS i BRAF gena u karcinomima kolorektuma u populaciji Srbije.” 2012. Web. 19 Oct 2019.

Vancouver:

Jakovljević KV1. Analiza učestalosti i tipova mutacija KRAS i BRAF gena u karcinomima kolorektuma u populaciji Srbije. [Internet] [Thesis]. Univerzitet u Beogradu; 2012. [cited 2019 Oct 19]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:2342/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jakovljević KV1. Analiza učestalosti i tipova mutacija KRAS i BRAF gena u karcinomima kolorektuma u populaciji Srbije. [Thesis]. Univerzitet u Beogradu; 2012. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:2342/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

8. Morris, John Pacheco. Acinar plasticity and Kras dependent specification of pancreatic ductal adenocarcinoma precursors.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 Pancreatic ductal adenocarcinoma (PDA) is characterized by near universal mutations in the Kras proto-oncogene. Targeting constitutively active Kras to the pancreas results in the development… (more)

Subjects/Keywords: Molecular biology; Kras; Pancreatic Cancer; Plasticity

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APA (6th Edition):

Morris, J. P. (2011). Acinar plasticity and Kras dependent specification of pancreatic ductal adenocarcinoma precursors. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/0b87k4k6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Morris, John Pacheco. “Acinar plasticity and Kras dependent specification of pancreatic ductal adenocarcinoma precursors.” 2011. Thesis, University of California – San Francisco. Accessed October 19, 2019. http://www.escholarship.org/uc/item/0b87k4k6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Morris, John Pacheco. “Acinar plasticity and Kras dependent specification of pancreatic ductal adenocarcinoma precursors.” 2011. Web. 19 Oct 2019.

Vancouver:

Morris JP. Acinar plasticity and Kras dependent specification of pancreatic ductal adenocarcinoma precursors. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2019 Oct 19]. Available from: http://www.escholarship.org/uc/item/0b87k4k6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Morris JP. Acinar plasticity and Kras dependent specification of pancreatic ductal adenocarcinoma precursors. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/0b87k4k6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

9. Westcott, Peter Maxwell Kienitz. Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse.

Degree: Pharmaceutical Sciences and Pharmacogenomics, 2015, University of California – San Francisco

 Mouse models have been an essential tool for cancer research for many decades, and have seen application to ever more diverse and complex questions with… (more)

Subjects/Keywords: Biology; Genetics; Cancer; Genomics; Kras; Mouse models

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APA (6th Edition):

Westcott, P. M. K. (2015). Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/71f4t6xt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Westcott, Peter Maxwell Kienitz. “Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse.” 2015. Thesis, University of California – San Francisco. Accessed October 19, 2019. http://www.escholarship.org/uc/item/71f4t6xt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Westcott, Peter Maxwell Kienitz. “Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse.” 2015. Web. 19 Oct 2019.

Vancouver:

Westcott PMK. Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2019 Oct 19]. Available from: http://www.escholarship.org/uc/item/71f4t6xt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Westcott PMK. Genetic and environmental influences on the mutational landscape of Kras-driven lung cancer in the mouse. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/71f4t6xt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

10. Tremblay, Isabelle. La cascade KRas/Raf/Mek/Erk favorise la transcription-dépendante de la voie de signalisation Notch .

Degree: 2012, Université de Sherbrooke

 La voie de signalisation Notch contient quatre récepteurs transmembranaires qui sont activés de façon ligand dépendant. Les contacts cellulaires permettent l'activation des récepteurs et la… (more)

Subjects/Keywords: Transcription; Phosphorylation; Pancréas; Erk; KRas; Notch

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APA (6th Edition):

Tremblay, I. (2012). La cascade KRas/Raf/Mek/Erk favorise la transcription-dépendante de la voie de signalisation Notch . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/6664

Chicago Manual of Style (16th Edition):

Tremblay, Isabelle. “La cascade KRas/Raf/Mek/Erk favorise la transcription-dépendante de la voie de signalisation Notch .” 2012. Masters Thesis, Université de Sherbrooke. Accessed October 19, 2019. http://hdl.handle.net/11143/6664.

MLA Handbook (7th Edition):

Tremblay, Isabelle. “La cascade KRas/Raf/Mek/Erk favorise la transcription-dépendante de la voie de signalisation Notch .” 2012. Web. 19 Oct 2019.

Vancouver:

Tremblay I. La cascade KRas/Raf/Mek/Erk favorise la transcription-dépendante de la voie de signalisation Notch . [Internet] [Masters thesis]. Université de Sherbrooke; 2012. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/11143/6664.

Council of Science Editors:

Tremblay I. La cascade KRas/Raf/Mek/Erk favorise la transcription-dépendante de la voie de signalisation Notch . [Masters Thesis]. Université de Sherbrooke; 2012. Available from: http://hdl.handle.net/11143/6664

11. Santos, Edmilson Ozorio dos. Investigação das quinases Aurora A e Aurora B como potenciais alvos terapêuticos no câncer de pulmão induzido pelo oncogene KRAS.

Degree: Mestrado, Bioquímica, 2013, University of São Paulo

As alterações genéticas mais frequentes em tumores de pulmão são mutações pontuais que ativam o oncogene KRAS. Apesar destas mutações estarem ligadas à oncogênese de… (more)

Subjects/Keywords: Aurora kinases; Biologia molecular; Câncer de pulmão; KRAS; KRAS; Lung cancer; Molecular biology; Quinases Aurora

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APA (6th Edition):

Santos, E. O. d. (2013). Investigação das quinases Aurora A e Aurora B como potenciais alvos terapêuticos no câncer de pulmão induzido pelo oncogene KRAS. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/46/46131/tde-15012014-143156/ ;

Chicago Manual of Style (16th Edition):

Santos, Edmilson Ozorio dos. “Investigação das quinases Aurora A e Aurora B como potenciais alvos terapêuticos no câncer de pulmão induzido pelo oncogene KRAS.” 2013. Masters Thesis, University of São Paulo. Accessed October 19, 2019. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-15012014-143156/ ;.

MLA Handbook (7th Edition):

Santos, Edmilson Ozorio dos. “Investigação das quinases Aurora A e Aurora B como potenciais alvos terapêuticos no câncer de pulmão induzido pelo oncogene KRAS.” 2013. Web. 19 Oct 2019.

Vancouver:

Santos EOd. Investigação das quinases Aurora A e Aurora B como potenciais alvos terapêuticos no câncer de pulmão induzido pelo oncogene KRAS. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2019 Oct 19]. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-15012014-143156/ ;.

Council of Science Editors:

Santos EOd. Investigação das quinases Aurora A e Aurora B como potenciais alvos terapêuticos no câncer de pulmão induzido pelo oncogene KRAS. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/46/46131/tde-15012014-143156/ ;

12. Νικολούλη, Ειρήνη. Μελέτη του ρόλου του ογκογονιδίου Kras στην ανοσολογική απόκριση στα πλαίσια κακοήθειας.

Degree: 2013, University of Patras

Οι ανθρώπινοι όγκοι συνοδεύονται από κάποιου βαθμού φλεγμονή. Οι παράγοντες του καρκινικού κυττάρου που προκαλούν και ρυθμίζουν τη φλεγμονή, καθώς και οι επιδράσεις της φλεγμονής… (more)

Subjects/Keywords: Ογκογονίδιο Kras; Νεοπλασματική πρόοδος; Παρανεοπλασματική φλεγμονή; 616.994 071; Kras; Neoplastic progression; Paraneoplastic inflammation

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APA (6th Edition):

Νικολούλη, . (2013). Μελέτη του ρόλου του ογκογονιδίου Kras στην ανοσολογική απόκριση στα πλαίσια κακοήθειας. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/6977

Chicago Manual of Style (16th Edition):

Νικολούλη, Ειρήνη. “Μελέτη του ρόλου του ογκογονιδίου Kras στην ανοσολογική απόκριση στα πλαίσια κακοήθειας.” 2013. Masters Thesis, University of Patras. Accessed October 19, 2019. http://hdl.handle.net/10889/6977.

MLA Handbook (7th Edition):

Νικολούλη, Ειρήνη. “Μελέτη του ρόλου του ογκογονιδίου Kras στην ανοσολογική απόκριση στα πλαίσια κακοήθειας.” 2013. Web. 19 Oct 2019.

Vancouver:

Νικολούλη . Μελέτη του ρόλου του ογκογονιδίου Kras στην ανοσολογική απόκριση στα πλαίσια κακοήθειας. [Internet] [Masters thesis]. University of Patras; 2013. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/10889/6977.

Council of Science Editors:

Νικολούλη . Μελέτη του ρόλου του ογκογονιδίου Kras στην ανοσολογική απόκριση στα πλαίσια κακοήθειας. [Masters Thesis]. University of Patras; 2013. Available from: http://hdl.handle.net/10889/6977


University of Debrecen

13. Gazdag, Annamária. A KRAS gén mutáció analízise nem kissejtes tüdőrákban és colorectális carcinómában .

Degree: DE – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, University of Debrecen

 A daganatos megbetegedések és halálozások igen magas gyakorisága komoly népegészségügyi probléma világszerte, így hazánkban is. A legtöbb áldozatot követelő daganatféleségek közül diplomadolgozatom elkészítése során a… (more)

Subjects/Keywords: Kras; mutáció; KRAS-MASI; NSCLC; CRC

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gazdag, A. (n.d.). A KRAS gén mutáció analízise nem kissejtes tüdőrákban és colorectális carcinómában . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/233374

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gazdag, Annamária. “A KRAS gén mutáció analízise nem kissejtes tüdőrákban és colorectális carcinómában .” Thesis, University of Debrecen. Accessed October 19, 2019. http://hdl.handle.net/2437/233374.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gazdag, Annamária. “A KRAS gén mutáció analízise nem kissejtes tüdőrákban és colorectális carcinómában .” Web. 19 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Gazdag A. A KRAS gén mutáció analízise nem kissejtes tüdőrákban és colorectális carcinómában . [Internet] [Thesis]. University of Debrecen; [cited 2019 Oct 19]. Available from: http://hdl.handle.net/2437/233374.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Gazdag A. A KRAS gén mutáció analízise nem kissejtes tüdőrákban és colorectális carcinómában . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/233374

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Université Montpellier II

14. Moulière, Florent. Etude de la structure et de l'origine des ADN circulants : application à la mise au point d'un test de détection des mutations KRAS et BRAF dans le cancer colorectal : Study of the form and the origin of the circulating DNA. : application to the conception of a diagnosis assay of the KRAS gene mutation by blood sampling in theragnostic objective.

Degree: Docteur es, Biologie Santé, 2012, Université Montpellier II

Les ADN circulants extracellulaires (ADNcf) sont considérés comme des biomarqueurs potentiels non invasifs de la progression tumorale. Ils présentent l'avantage d'être porteurs des altérations génétiques… (more)

Subjects/Keywords: ADN circulant extracellulaire; Biomarqueur; Cancer colorectal; Mutation KRAS; Q-pcr; Extracellular circulating DNA; Biomarker; Colorectal cancer; KRAS mutation; Q-pcr

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moulière, F. (2012). Etude de la structure et de l'origine des ADN circulants : application à la mise au point d'un test de détection des mutations KRAS et BRAF dans le cancer colorectal : Study of the form and the origin of the circulating DNA. : application to the conception of a diagnosis assay of the KRAS gene mutation by blood sampling in theragnostic objective. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2012MON20245

Chicago Manual of Style (16th Edition):

Moulière, Florent. “Etude de la structure et de l'origine des ADN circulants : application à la mise au point d'un test de détection des mutations KRAS et BRAF dans le cancer colorectal : Study of the form and the origin of the circulating DNA. : application to the conception of a diagnosis assay of the KRAS gene mutation by blood sampling in theragnostic objective.” 2012. Doctoral Dissertation, Université Montpellier II. Accessed October 19, 2019. http://www.theses.fr/2012MON20245.

MLA Handbook (7th Edition):

Moulière, Florent. “Etude de la structure et de l'origine des ADN circulants : application à la mise au point d'un test de détection des mutations KRAS et BRAF dans le cancer colorectal : Study of the form and the origin of the circulating DNA. : application to the conception of a diagnosis assay of the KRAS gene mutation by blood sampling in theragnostic objective.” 2012. Web. 19 Oct 2019.

Vancouver:

Moulière F. Etude de la structure et de l'origine des ADN circulants : application à la mise au point d'un test de détection des mutations KRAS et BRAF dans le cancer colorectal : Study of the form and the origin of the circulating DNA. : application to the conception of a diagnosis assay of the KRAS gene mutation by blood sampling in theragnostic objective. [Internet] [Doctoral dissertation]. Université Montpellier II; 2012. [cited 2019 Oct 19]. Available from: http://www.theses.fr/2012MON20245.

Council of Science Editors:

Moulière F. Etude de la structure et de l'origine des ADN circulants : application à la mise au point d'un test de détection des mutations KRAS et BRAF dans le cancer colorectal : Study of the form and the origin of the circulating DNA. : application to the conception of a diagnosis assay of the KRAS gene mutation by blood sampling in theragnostic objective. [Doctoral Dissertation]. Université Montpellier II; 2012. Available from: http://www.theses.fr/2012MON20245

15. PEK MI XUE MICHELLE. INVESTIGATION OF RAS-DEPENDENT GENE EXPRESSION & CELL SURVIVAL SIGNALLING IN COLORECTAL CANCER.

Degree: 2016, National University of Singapore

Subjects/Keywords: Colorectal cancer; KRAS; KRAS dependency; MEK; CDK4/6; FOXM1

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APA (6th Edition):

MICHELLE, P. M. X. (2016). INVESTIGATION OF RAS-DEPENDENT GENE EXPRESSION & CELL SURVIVAL SIGNALLING IN COLORECTAL CANCER. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/124088

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MICHELLE, PEK MI XUE. “INVESTIGATION OF RAS-DEPENDENT GENE EXPRESSION & CELL SURVIVAL SIGNALLING IN COLORECTAL CANCER.” 2016. Thesis, National University of Singapore. Accessed October 19, 2019. http://scholarbank.nus.edu.sg/handle/10635/124088.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MICHELLE, PEK MI XUE. “INVESTIGATION OF RAS-DEPENDENT GENE EXPRESSION & CELL SURVIVAL SIGNALLING IN COLORECTAL CANCER.” 2016. Web. 19 Oct 2019.

Vancouver:

MICHELLE PMX. INVESTIGATION OF RAS-DEPENDENT GENE EXPRESSION & CELL SURVIVAL SIGNALLING IN COLORECTAL CANCER. [Internet] [Thesis]. National University of Singapore; 2016. [cited 2019 Oct 19]. Available from: http://scholarbank.nus.edu.sg/handle/10635/124088.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MICHELLE PMX. INVESTIGATION OF RAS-DEPENDENT GENE EXPRESSION & CELL SURVIVAL SIGNALLING IN COLORECTAL CANCER. [Thesis]. National University of Singapore; 2016. Available from: http://scholarbank.nus.edu.sg/handle/10635/124088

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brno University of Technology

16. Švantnerová, Jana. Dětská léčebna se speleoterapií Ostrov u Macochy .

Degree: 2015, Brno University of Technology

 Předmětem diplomové práce je návrh architektonické studie zabývající se otázkou zřízení nového areálu dětské léčebny. Řešené území se nachází ve chráněné krajinné oblasti Moravský kras,… (more)

Subjects/Keywords: Léčebna; speleoterapie; ubytování; penzion; Ostrov u Macochy; Moravský kras; Sanatorium; speleotherapy; accommodation; guest house; Ostrov u Macochy; Moravský kras.

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APA (6th Edition):

Švantnerová, J. (2015). Dětská léčebna se speleoterapií Ostrov u Macochy . (Thesis). Brno University of Technology. Retrieved from http://hdl.handle.net/11012/138341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Švantnerová, Jana. “Dětská léčebna se speleoterapií Ostrov u Macochy .” 2015. Thesis, Brno University of Technology. Accessed October 19, 2019. http://hdl.handle.net/11012/138341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Švantnerová, Jana. “Dětská léčebna se speleoterapií Ostrov u Macochy .” 2015. Web. 19 Oct 2019.

Vancouver:

Švantnerová J. Dětská léčebna se speleoterapií Ostrov u Macochy . [Internet] [Thesis]. Brno University of Technology; 2015. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/11012/138341.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Švantnerová J. Dětská léčebna se speleoterapií Ostrov u Macochy . [Thesis]. Brno University of Technology; 2015. Available from: http://hdl.handle.net/11012/138341

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

17. Huang, Shaoyi. Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy.

Degree: PhD, 2013, Texas Medical Center

  Lung cancer is the leading cause of cancer death in both men and women in the United States and worldwide. Despite improvement in treatment… (more)

Subjects/Keywords: NSCLC; KRAS; Synthetic lethality; Chemoprevention; Medicine and Health Sciences

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APA (6th Edition):

Huang, S. (2013). Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/396

Chicago Manual of Style (16th Edition):

Huang, Shaoyi. “Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy.” 2013. Doctoral Dissertation, Texas Medical Center. Accessed October 19, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/396.

MLA Handbook (7th Edition):

Huang, Shaoyi. “Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy.” 2013. Web. 19 Oct 2019.

Vancouver:

Huang S. Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2013. [cited 2019 Oct 19]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/396.

Council of Science Editors:

Huang S. Induction of synthetic lethality in mutant KRAS cells for non-small cell lung cancers chemoprevention and therapy. [Doctoral Dissertation]. Texas Medical Center; 2013. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/396


NSYSU

18. Ding, Pei-ya. CD24 induces epithelial-mesenchymal transition and bone metastasis in prostate cancer through the upregulation of Wnt signaling pathway.

Degree: Master, Institute of Biomedical Sciences, 2017, NSYSU

 Prostate cancer is usually a very slow growing cancer, often causing no symptoms until it is in an advanced stage. The development is due to… (more)

Subjects/Keywords: Bone metastasis; P53; BRAF; Prostate cancer; KRAS; CD24

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APA (6th Edition):

Ding, P. (2017). CD24 induces epithelial-mesenchymal transition and bone metastasis in prostate cancer through the upregulation of Wnt signaling pathway. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605117-225731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ding, Pei-ya. “CD24 induces epithelial-mesenchymal transition and bone metastasis in prostate cancer through the upregulation of Wnt signaling pathway.” 2017. Thesis, NSYSU. Accessed October 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605117-225731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ding, Pei-ya. “CD24 induces epithelial-mesenchymal transition and bone metastasis in prostate cancer through the upregulation of Wnt signaling pathway.” 2017. Web. 19 Oct 2019.

Vancouver:

Ding P. CD24 induces epithelial-mesenchymal transition and bone metastasis in prostate cancer through the upregulation of Wnt signaling pathway. [Internet] [Thesis]. NSYSU; 2017. [cited 2019 Oct 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605117-225731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ding P. CD24 induces epithelial-mesenchymal transition and bone metastasis in prostate cancer through the upregulation of Wnt signaling pathway. [Thesis]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0605117-225731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Miami University

19. Fenton, Benjamin A. GAP Engineering to Restore GTP Hydrolysis to Oncogenic Kras Mutants.

Degree: MS, Chemistry, 2014, Miami University

 The Kras gene encodes a small GTPase that is involved in pathways regulating cell growth and proliferation. Bound to GTP, Kras sends growth signals until… (more)

Subjects/Keywords: Biochemistry; Kras; rasGAP; active site redesign; GTP hydrolysis; cancer signaling

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APA (6th Edition):

Fenton, B. A. (2014). GAP Engineering to Restore GTP Hydrolysis to Oncogenic Kras Mutants. (Masters Thesis). Miami University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=miami1398812548

Chicago Manual of Style (16th Edition):

Fenton, Benjamin A. “GAP Engineering to Restore GTP Hydrolysis to Oncogenic Kras Mutants.” 2014. Masters Thesis, Miami University. Accessed October 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=miami1398812548.

MLA Handbook (7th Edition):

Fenton, Benjamin A. “GAP Engineering to Restore GTP Hydrolysis to Oncogenic Kras Mutants.” 2014. Web. 19 Oct 2019.

Vancouver:

Fenton BA. GAP Engineering to Restore GTP Hydrolysis to Oncogenic Kras Mutants. [Internet] [Masters thesis]. Miami University; 2014. [cited 2019 Oct 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1398812548.

Council of Science Editors:

Fenton BA. GAP Engineering to Restore GTP Hydrolysis to Oncogenic Kras Mutants. [Masters Thesis]. Miami University; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1398812548


NSYSU

20. Liu, Yu-Hsuan. Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

 Prostate cancer (PCa) is one of the most common malignant tumors in men and the second leading cause of cancer deaths in male worldwide. P53… (more)

Subjects/Keywords: Braf; mGluR1; P53; Kras; Mouse models of prostate cancer

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APA (6th Edition):

Liu, Y. (2015). Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Yu-Hsuan. “Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice.” 2015. Thesis, NSYSU. Accessed October 19, 2019. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Yu-Hsuan. “Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice.” 2015. Web. 19 Oct 2019.

Vancouver:

Liu Y. Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice. [Internet] [Thesis]. NSYSU; 2015. [cited 2019 Oct 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu Y. Characterization and Comparison Two Genetic Mouse Models of Prostate Cancer: K-Ras Signaling and P53 Loss Accelerate Prostate Cancer Cell Dissemination in the Mice. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712115-123250

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. 川原, 大輔. The significance of combined measurement of p53 antibody and other tumor markers for colorectal cancer after curative resection. : 大腸癌術後のp53抗体とその他の腫瘍マーカー測定の意義.

Degree: 博士(医学), 2015, Nagasaki University / 長崎大学

 Background/Aims: Though serum p53 antibody has been widely used, it is mentioned that it is not related to clinical parameters of colorectal cancer (CRC) and… (more)

Subjects/Keywords: p53 antibody; colorectal cancer; CEA; CA19-9; KRAS mutation

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APA (6th Edition):

川原, . (2015). The significance of combined measurement of p53 antibody and other tumor markers for colorectal cancer after curative resection. : 大腸癌術後のp53抗体とその他の腫瘍マーカー測定の意義. (Thesis). Nagasaki University / 長崎大学. Retrieved from http://hdl.handle.net/10069/36361

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

川原, 大輔. “The significance of combined measurement of p53 antibody and other tumor markers for colorectal cancer after curative resection. : 大腸癌術後のp53抗体とその他の腫瘍マーカー測定の意義.” 2015. Thesis, Nagasaki University / 長崎大学. Accessed October 19, 2019. http://hdl.handle.net/10069/36361.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

川原, 大輔. “The significance of combined measurement of p53 antibody and other tumor markers for colorectal cancer after curative resection. : 大腸癌術後のp53抗体とその他の腫瘍マーカー測定の意義.” 2015. Web. 19 Oct 2019.

Vancouver:

川原 . The significance of combined measurement of p53 antibody and other tumor markers for colorectal cancer after curative resection. : 大腸癌術後のp53抗体とその他の腫瘍マーカー測定の意義. [Internet] [Thesis]. Nagasaki University / 長崎大学; 2015. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/10069/36361.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

川原 . The significance of combined measurement of p53 antibody and other tumor markers for colorectal cancer after curative resection. : 大腸癌術後のp53抗体とその他の腫瘍マーカー測定の意義. [Thesis]. Nagasaki University / 長崎大学; 2015. Available from: http://hdl.handle.net/10069/36361

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

22. Usher, Joshua. Use of cell-free nucleic acids in associating PD-L1 gene expression with presence of driver mutations in DNA and demographics across different cancers.

Degree: MS, Biostatistics, 2016, University of Southern California

 Introduction: Measuring allele frequencies of somatic gene mutations in DNA and levels of relative gene expressions in RNA can now be measured using cell-free nucleic… (more)

Subjects/Keywords: PD-L1; BRAF; KRAS; EGFR; cell-free; DNA; RNA

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APA (6th Edition):

Usher, J. (2016). Use of cell-free nucleic acids in associating PD-L1 gene expression with presence of driver mutations in DNA and demographics across different cancers. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/640124/rec/7721

Chicago Manual of Style (16th Edition):

Usher, Joshua. “Use of cell-free nucleic acids in associating PD-L1 gene expression with presence of driver mutations in DNA and demographics across different cancers.” 2016. Masters Thesis, University of Southern California. Accessed October 19, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/640124/rec/7721.

MLA Handbook (7th Edition):

Usher, Joshua. “Use of cell-free nucleic acids in associating PD-L1 gene expression with presence of driver mutations in DNA and demographics across different cancers.” 2016. Web. 19 Oct 2019.

Vancouver:

Usher J. Use of cell-free nucleic acids in associating PD-L1 gene expression with presence of driver mutations in DNA and demographics across different cancers. [Internet] [Masters thesis]. University of Southern California; 2016. [cited 2019 Oct 19]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/640124/rec/7721.

Council of Science Editors:

Usher J. Use of cell-free nucleic acids in associating PD-L1 gene expression with presence of driver mutations in DNA and demographics across different cancers. [Masters Thesis]. University of Southern California; 2016. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/640124/rec/7721


University of Edinburgh

23. Novo, Sonia Marisa. Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244.

Degree: PhD, 2013, University of Edinburgh

 Colorectal cancer is the second most common cause of cancer death in Western Europe and North America. Current therapies are largely ineffective and are associated… (more)

Subjects/Keywords: 616.99; MEK1/2; MAPK pathway; KRAS mutations; BRAF mutations; proliferation; apoptosis

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APA (6th Edition):

Novo, S. M. (2013). Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/11778

Chicago Manual of Style (16th Edition):

Novo, Sonia Marisa. “Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244.” 2013. Doctoral Dissertation, University of Edinburgh. Accessed October 19, 2019. http://hdl.handle.net/1842/11778.

MLA Handbook (7th Edition):

Novo, Sonia Marisa. “Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244.” 2013. Web. 19 Oct 2019.

Vancouver:

Novo SM. Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244. [Internet] [Doctoral dissertation]. University of Edinburgh; 2013. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/1842/11778.

Council of Science Editors:

Novo SM. Use of an ex vivo model of human colorectal tumours to study response to the MEK1/2 inhibitor AZD6244. [Doctoral Dissertation]. University of Edinburgh; 2013. Available from: http://hdl.handle.net/1842/11778


Duke University

24. Lampson, Benjamin Logan. Oncogenic KRAS Expression and Signaling .

Degree: 2012, Duke University

  RAS is a small GTPase that helps to convert extracellular cues into intracellular actions. It is the most commonly mutated oncogene and is found… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Medicine; cancer; codon; eNOS; KRAS; pancreatic; translation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lampson, B. L. (2012). Oncogenic KRAS Expression and Signaling . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lampson, Benjamin Logan. “Oncogenic KRAS Expression and Signaling .” 2012. Thesis, Duke University. Accessed October 19, 2019. http://hdl.handle.net/10161/5394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lampson, Benjamin Logan. “Oncogenic KRAS Expression and Signaling .” 2012. Web. 19 Oct 2019.

Vancouver:

Lampson BL. Oncogenic KRAS Expression and Signaling . [Internet] [Thesis]. Duke University; 2012. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/10161/5394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lampson BL. Oncogenic KRAS Expression and Signaling . [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

25. Pershing, Nicole L. Evaluation of Altered Kras Codon Bias and NOS Inhibition During Lung Tumorigenesis .

Degree: 2014, Duke University

  The small GTPases HRAS, NRAS and KRAS are mutated in approximately one-third of all human cancers, rendering the proteins constitutively active and oncogenic. Lung… (more)

Subjects/Keywords: Oncology; Molecular biology; Codon bias; KRAS; Lung Cancer; NOS

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APA (6th Edition):

Pershing, N. L. (2014). Evaluation of Altered Kras Codon Bias and NOS Inhibition During Lung Tumorigenesis . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9040

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pershing, Nicole L. “Evaluation of Altered Kras Codon Bias and NOS Inhibition During Lung Tumorigenesis .” 2014. Thesis, Duke University. Accessed October 19, 2019. http://hdl.handle.net/10161/9040.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pershing, Nicole L. “Evaluation of Altered Kras Codon Bias and NOS Inhibition During Lung Tumorigenesis .” 2014. Web. 19 Oct 2019.

Vancouver:

Pershing NL. Evaluation of Altered Kras Codon Bias and NOS Inhibition During Lung Tumorigenesis . [Internet] [Thesis]. Duke University; 2014. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/10161/9040.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pershing NL. Evaluation of Altered Kras Codon Bias and NOS Inhibition During Lung Tumorigenesis . [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/9040

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

26. Zandvakili, Inuk. RhoA as a Potential Target in Lung Cancer.

Degree: PhD, Medicine: Molecular and Developmental Biology, 2015, University of Cincinnati

 Many cancers are driven by oncogenic K-Ras, yet K-Ras has remained largely undruggable. In this dissertation we explore inhibiting K-Ras signaling by targeting downstream signaling… (more)

Subjects/Keywords: Surgery; RhoA; KRas; Rho GTPases; Lung Cancer; RhoC; Lung Adenocarcinoma

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zandvakili, I. (2015). RhoA as a Potential Target in Lung Cancer. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196

Chicago Manual of Style (16th Edition):

Zandvakili, Inuk. “RhoA as a Potential Target in Lung Cancer.” 2015. Doctoral Dissertation, University of Cincinnati. Accessed October 19, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196.

MLA Handbook (7th Edition):

Zandvakili, Inuk. “RhoA as a Potential Target in Lung Cancer.” 2015. Web. 19 Oct 2019.

Vancouver:

Zandvakili I. RhoA as a Potential Target in Lung Cancer. [Internet] [Doctoral dissertation]. University of Cincinnati; 2015. [cited 2019 Oct 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196.

Council of Science Editors:

Zandvakili I. RhoA as a Potential Target in Lung Cancer. [Doctoral Dissertation]. University of Cincinnati; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1445342196


Université de Sherbrooke

27. Placet, Morgane. CDK8 : une cible de la voie KRAS/MAP Kinase dans la carcinogénèse colorectale .

Degree: 2014, Université de Sherbrooke

 La voie KRAS/BRAF/MEK/ERK MAP Kinase joue un rôle clé dans le contrôle de la prolifération des cellules épithéliales intestinales normales et cancéreuses. En effet, on… (more)

Subjects/Keywords: CDK8; Voie KRAS/MAP Kinase; Transformation cellulaire; Cancer colorectal

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APA (6th Edition):

Placet, M. (2014). CDK8 : une cible de la voie KRAS/MAP Kinase dans la carcinogénèse colorectale . (Masters Thesis). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/572

Chicago Manual of Style (16th Edition):

Placet, Morgane. “CDK8 : une cible de la voie KRAS/MAP Kinase dans la carcinogénèse colorectale .” 2014. Masters Thesis, Université de Sherbrooke. Accessed October 19, 2019. http://savoirs.usherbrooke.ca/handle/11143/572.

MLA Handbook (7th Edition):

Placet, Morgane. “CDK8 : une cible de la voie KRAS/MAP Kinase dans la carcinogénèse colorectale .” 2014. Web. 19 Oct 2019.

Vancouver:

Placet M. CDK8 : une cible de la voie KRAS/MAP Kinase dans la carcinogénèse colorectale . [Internet] [Masters thesis]. Université de Sherbrooke; 2014. [cited 2019 Oct 19]. Available from: http://savoirs.usherbrooke.ca/handle/11143/572.

Council of Science Editors:

Placet M. CDK8 : une cible de la voie KRAS/MAP Kinase dans la carcinogénèse colorectale . [Masters Thesis]. Université de Sherbrooke; 2014. Available from: http://savoirs.usherbrooke.ca/handle/11143/572

28. Nikolić Aleksandra. Structural and functional analysis of smad4 gene 5' regulatory region in human pancreatic cancer.

Degree: PhD, Biology, 2010, University of Belgrade

Pancreatic carcinoma is one of the most invasive and progressive human malignant neoplasms. This malignancy is characterized by activation of KRAS oncogene and inactivation of… (more)

Subjects/Keywords: karcinom pankreasa; SMAD4; KRAS; pancreatic cancer; SMAD4; AS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aleksandra, N. (2010). Structural and functional analysis of smad4 gene 5' regulatory region in human pancreatic cancer. (Doctoral Dissertation). University of Belgrade. Retrieved from http://dx.doi.org/10.2298/BG20100416NIKOLIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=551 ; https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=36854287

Chicago Manual of Style (16th Edition):

Aleksandra, Nikolić. “Structural and functional analysis of smad4 gene 5' regulatory region in human pancreatic cancer.” 2010. Doctoral Dissertation, University of Belgrade. Accessed October 19, 2019. http://dx.doi.org/10.2298/BG20100416NIKOLIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=551 ; https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=36854287.

MLA Handbook (7th Edition):

Aleksandra, Nikolić. “Structural and functional analysis of smad4 gene 5' regulatory region in human pancreatic cancer.” 2010. Web. 19 Oct 2019.

Vancouver:

Aleksandra N. Structural and functional analysis of smad4 gene 5' regulatory region in human pancreatic cancer. [Internet] [Doctoral dissertation]. University of Belgrade; 2010. [cited 2019 Oct 19]. Available from: http://dx.doi.org/10.2298/BG20100416NIKOLIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=551 ; https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=36854287.

Council of Science Editors:

Aleksandra N. Structural and functional analysis of smad4 gene 5' regulatory region in human pancreatic cancer. [Doctoral Dissertation]. University of Belgrade; 2010. Available from: http://dx.doi.org/10.2298/BG20100416NIKOLIC ; http://eteze.bg.ac.rs/application/showtheses?thesesId=551 ; https://fedorabg.bg.ac.rs/fedora/get/o:6695/bdef:Content/get ; http://vbs.rs/scripts/cobiss?command=SEARCH&base=99999&select=ID=36854287


Uppsala University

29. Mattsson, Johanna. Genetic heterogeneity of EGFR and KRAS mutations in primary tumor tissue from non-small cell lung cancer patients.

Degree: Medical Biochemistry and Microbiology, 2011, Uppsala University

  Activated epidermal growth factor receptor (EGFR) and Kirsten rat sarcoma viral oncogene homolog (KRAS) mutations characterize molecular subgroups of non-small cell lung cancer (NSCLC)… (more)

Subjects/Keywords: Non-small cell lung cancer; KRAS; EGFR; heterogeneity; mutation

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APA (6th Edition):

Mattsson, J. (2011). Genetic heterogeneity of EGFR and KRAS mutations in primary tumor tissue from non-small cell lung cancer patients. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-159359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mattsson, Johanna. “Genetic heterogeneity of EGFR and KRAS mutations in primary tumor tissue from non-small cell lung cancer patients.” 2011. Thesis, Uppsala University. Accessed October 19, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-159359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mattsson, Johanna. “Genetic heterogeneity of EGFR and KRAS mutations in primary tumor tissue from non-small cell lung cancer patients.” 2011. Web. 19 Oct 2019.

Vancouver:

Mattsson J. Genetic heterogeneity of EGFR and KRAS mutations in primary tumor tissue from non-small cell lung cancer patients. [Internet] [Thesis]. Uppsala University; 2011. [cited 2019 Oct 19]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-159359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mattsson J. Genetic heterogeneity of EGFR and KRAS mutations in primary tumor tissue from non-small cell lung cancer patients. [Thesis]. Uppsala University; 2011. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-159359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Waikato

30. Law, Tania Alice. Detection of somatic mutations in EGFR and KRAS genes in formalin-fixed paraffin-embedded specimens from Non-Small Cell Lung Cancer Patients .

Degree: 2012, University of Waikato

 Non-small-cell lung cancer (NSCLC) accounts for 80% of lung cancer and has a 5 year survival rate of just 15%. Recently, tyrosine kinase inhibitors (TKIs)… (more)

Subjects/Keywords: EGFR; KRAS; NSCLC patients; FFPE specimens; mutation detection

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APA (6th Edition):

Law, T. A. (2012). Detection of somatic mutations in EGFR and KRAS genes in formalin-fixed paraffin-embedded specimens from Non-Small Cell Lung Cancer Patients . (Masters Thesis). University of Waikato. Retrieved from http://hdl.handle.net/10289/6601

Chicago Manual of Style (16th Edition):

Law, Tania Alice. “Detection of somatic mutations in EGFR and KRAS genes in formalin-fixed paraffin-embedded specimens from Non-Small Cell Lung Cancer Patients .” 2012. Masters Thesis, University of Waikato. Accessed October 19, 2019. http://hdl.handle.net/10289/6601.

MLA Handbook (7th Edition):

Law, Tania Alice. “Detection of somatic mutations in EGFR and KRAS genes in formalin-fixed paraffin-embedded specimens from Non-Small Cell Lung Cancer Patients .” 2012. Web. 19 Oct 2019.

Vancouver:

Law TA. Detection of somatic mutations in EGFR and KRAS genes in formalin-fixed paraffin-embedded specimens from Non-Small Cell Lung Cancer Patients . [Internet] [Masters thesis]. University of Waikato; 2012. [cited 2019 Oct 19]. Available from: http://hdl.handle.net/10289/6601.

Council of Science Editors:

Law TA. Detection of somatic mutations in EGFR and KRAS genes in formalin-fixed paraffin-embedded specimens from Non-Small Cell Lung Cancer Patients . [Masters Thesis]. University of Waikato; 2012. Available from: http://hdl.handle.net/10289/6601

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