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You searched for subject:(keratinocyte). Showing records 1 – 30 of 97 total matches.

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1. 신, 재영. Immunohistochemical Evaluation and Gene Expression Profiling of Senile Lentigo.

Degree: 2012, Ajou University

BACKGROUND AND OBJECTIVES: Senile lentigines typically appear as dark brown macules on sun-exposed areas. Histologically, they are characterized by hyperpigmentation in the basal layer of… (more)

Subjects/Keywords: lentigo; immunohistochemistry; melanocyte; keratinocyte; microarray

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APA (6th Edition):

신, . (2012). Immunohistochemical Evaluation and Gene Expression Profiling of Senile Lentigo. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/7582 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

신, 재영. “Immunohistochemical Evaluation and Gene Expression Profiling of Senile Lentigo.” 2012. Thesis, Ajou University. Accessed January 25, 2021. http://repository.ajou.ac.kr/handle/201003/7582 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

신, 재영. “Immunohistochemical Evaluation and Gene Expression Profiling of Senile Lentigo.” 2012. Web. 25 Jan 2021.

Vancouver:

신 . Immunohistochemical Evaluation and Gene Expression Profiling of Senile Lentigo. [Internet] [Thesis]. Ajou University; 2012. [cited 2021 Jan 25]. Available from: http://repository.ajou.ac.kr/handle/201003/7582 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

신 . Immunohistochemical Evaluation and Gene Expression Profiling of Senile Lentigo. [Thesis]. Ajou University; 2012. Available from: http://repository.ajou.ac.kr/handle/201003/7582 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000012356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

2. Kuo, I-Hsin. Activation of Epidermal Toll-like Receptor 2 Enhances Tight Junction Function:Implications for Atopic Dermatitis and Skin Barrier Repair.

Degree: PhD, 2013, University of Rochester

 Atopic dermatitis (AD) is a Th2-skewed, allergen-driven skin disease characterized by epidermal tight junction (TJ) defects and a propensity for Staphylococcus aureus (S. aureus) skin… (more)

Subjects/Keywords: TLR2; Tight Junction; Atopic Dermatitis; Keratinocyte

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APA (6th Edition):

Kuo, I. (2013). Activation of Epidermal Toll-like Receptor 2 Enhances Tight Junction Function:Implications for Atopic Dermatitis and Skin Barrier Repair. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/27274

Chicago Manual of Style (16th Edition):

Kuo, I-Hsin. “Activation of Epidermal Toll-like Receptor 2 Enhances Tight Junction Function:Implications for Atopic Dermatitis and Skin Barrier Repair.” 2013. Doctoral Dissertation, University of Rochester. Accessed January 25, 2021. http://hdl.handle.net/1802/27274.

MLA Handbook (7th Edition):

Kuo, I-Hsin. “Activation of Epidermal Toll-like Receptor 2 Enhances Tight Junction Function:Implications for Atopic Dermatitis and Skin Barrier Repair.” 2013. Web. 25 Jan 2021.

Vancouver:

Kuo I. Activation of Epidermal Toll-like Receptor 2 Enhances Tight Junction Function:Implications for Atopic Dermatitis and Skin Barrier Repair. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/1802/27274.

Council of Science Editors:

Kuo I. Activation of Epidermal Toll-like Receptor 2 Enhances Tight Junction Function:Implications for Atopic Dermatitis and Skin Barrier Repair. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/27274


Penn State University

3. Smith, Kayla Jo. THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY.

Degree: 2017, Penn State University

 Barrier tissues such as the skin and intestine are important for the first line of defense against injury and exposure to potentially harmful toxicants or… (more)

Subjects/Keywords: aryl hydrocarbon receptor; skin; intestine; inflammation; keratinocyte

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APA (6th Edition):

Smith, K. J. (2017). THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13833kjs5049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Kayla Jo. “THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY.” 2017. Thesis, Penn State University. Accessed January 25, 2021. https://submit-etda.libraries.psu.edu/catalog/13833kjs5049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Kayla Jo. “THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY.” 2017. Web. 25 Jan 2021.

Vancouver:

Smith KJ. THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY. [Internet] [Thesis]. Penn State University; 2017. [cited 2021 Jan 25]. Available from: https://submit-etda.libraries.psu.edu/catalog/13833kjs5049.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith KJ. THE ROLE OF THE ARYL HYDROCARBON RECEPTOR IN MODULATING SKIN AND INTESTINAL EPITHELIAL PHYSIOLOGY. [Thesis]. Penn State University; 2017. Available from: https://submit-etda.libraries.psu.edu/catalog/13833kjs5049

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Texas

4. Alsrhani, Abdullah Falleh. Studies in Trypsin as an Alarm Substance in Zebrafish.

Degree: 2018, University of North Texas

 Previous studies have shown that fish release alarming substances into the water to alert their kin to escape from danger. In our laboratory, we found… (more)

Subjects/Keywords: Trypsin; Behavior; Zebrafish; Knockdown; Keratinocyte; and PAR2

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APA (6th Edition):

Alsrhani, A. F. (2018). Studies in Trypsin as an Alarm Substance in Zebrafish. (Thesis). University of North Texas. Retrieved from https://digital.library.unt.edu/ark:/67531/metadc1248500/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alsrhani, Abdullah Falleh. “Studies in Trypsin as an Alarm Substance in Zebrafish.” 2018. Thesis, University of North Texas. Accessed January 25, 2021. https://digital.library.unt.edu/ark:/67531/metadc1248500/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alsrhani, Abdullah Falleh. “Studies in Trypsin as an Alarm Substance in Zebrafish.” 2018. Web. 25 Jan 2021.

Vancouver:

Alsrhani AF. Studies in Trypsin as an Alarm Substance in Zebrafish. [Internet] [Thesis]. University of North Texas; 2018. [cited 2021 Jan 25]. Available from: https://digital.library.unt.edu/ark:/67531/metadc1248500/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alsrhani AF. Studies in Trypsin as an Alarm Substance in Zebrafish. [Thesis]. University of North Texas; 2018. Available from: https://digital.library.unt.edu/ark:/67531/metadc1248500/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Shetty, N. Developing methods of measuring and manipulating melanocyte/keratinocyte ratios to inform potential treatment of vitiligo vulgaris.

Degree: PhD, 2020, Canterbury Christ Church University

 Introduction My doctoral work focuses on the disease vitiligo vulgaris. Several treatments already exist however, one totally successful remedy is unavailable. I have focused on… (more)

Subjects/Keywords: Vitiligo vulgaris; Melanocyte–keratinocyte transplantation procedure

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APA (6th Edition):

Shetty, N. (2020). Developing methods of measuring and manipulating melanocyte/keratinocyte ratios to inform potential treatment of vitiligo vulgaris. (Doctoral Dissertation). Canterbury Christ Church University. Retrieved from https://repository.canterbury.ac.uk/item/8wq64/developing-methods-of-measuring-and-manipulating-melanocyte-keratinocyte-ratios-to-inform-potential-treatment-of-vitiligo-vulgaris ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.818071

Chicago Manual of Style (16th Edition):

Shetty, N. “Developing methods of measuring and manipulating melanocyte/keratinocyte ratios to inform potential treatment of vitiligo vulgaris.” 2020. Doctoral Dissertation, Canterbury Christ Church University. Accessed January 25, 2021. https://repository.canterbury.ac.uk/item/8wq64/developing-methods-of-measuring-and-manipulating-melanocyte-keratinocyte-ratios-to-inform-potential-treatment-of-vitiligo-vulgaris ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.818071.

MLA Handbook (7th Edition):

Shetty, N. “Developing methods of measuring and manipulating melanocyte/keratinocyte ratios to inform potential treatment of vitiligo vulgaris.” 2020. Web. 25 Jan 2021.

Vancouver:

Shetty N. Developing methods of measuring and manipulating melanocyte/keratinocyte ratios to inform potential treatment of vitiligo vulgaris. [Internet] [Doctoral dissertation]. Canterbury Christ Church University; 2020. [cited 2021 Jan 25]. Available from: https://repository.canterbury.ac.uk/item/8wq64/developing-methods-of-measuring-and-manipulating-melanocyte-keratinocyte-ratios-to-inform-potential-treatment-of-vitiligo-vulgaris ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.818071.

Council of Science Editors:

Shetty N. Developing methods of measuring and manipulating melanocyte/keratinocyte ratios to inform potential treatment of vitiligo vulgaris. [Doctoral Dissertation]. Canterbury Christ Church University; 2020. Available from: https://repository.canterbury.ac.uk/item/8wq64/developing-methods-of-measuring-and-manipulating-melanocyte-keratinocyte-ratios-to-inform-potential-treatment-of-vitiligo-vulgaris ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.818071

6. Michopoulou, Anna. Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes.

Degree: Docteur es, Biologie, 2016, Lyon

 La phase de l'épithélialisation de la réparation cutanée se déroule en impliquant plusieurs processus dynamiques et interactifs pendant lesquels les kératinocytes migrent, prolifèrent et se… (more)

Subjects/Keywords: MMP-9; Syndecan-1; Keratinocyte; Laminin 332; MMP-9; Syndecan-1; Keratinocyte; Laminin 332; 571.6

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APA (6th Edition):

Michopoulou, A. (2016). Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2016LYSE1130

Chicago Manual of Style (16th Edition):

Michopoulou, Anna. “Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes.” 2016. Doctoral Dissertation, Lyon. Accessed January 25, 2021. http://www.theses.fr/2016LYSE1130.

MLA Handbook (7th Edition):

Michopoulou, Anna. “Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes.” 2016. Web. 25 Jan 2021.

Vancouver:

Michopoulou A. Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes. [Internet] [Doctoral dissertation]. Lyon; 2016. [cited 2021 Jan 25]. Available from: http://www.theses.fr/2016LYSE1130.

Council of Science Editors:

Michopoulou A. Receptor syndecan-1 controls MMP-9 expression during keratinocyte migration : Le récepteur syndecan-1 contrôle l'expression de MMP-9 au cours de la migration des kératinocytes. [Doctoral Dissertation]. Lyon; 2016. Available from: http://www.theses.fr/2016LYSE1130


NSYSU

7. You, Huey-Ling. The roles of tumor susceptibility gene 101 in keratinocyte differentiation and chromatin remodeling of p16INK4a promotor.

Degree: PhD, Biological Sciences, 2007, NSYSU

 Tumor Susceptibility Gene 101, TSG101, exhibits multiple biological functions including the regulation of gene transcription, vesicular trafficking, cellular growth and differentiation. However, the signals involve… (more)

Subjects/Keywords: p16INK4a promotor; differentiation; TSG101; keratinocyte

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APA (6th Edition):

You, H. (2007). The roles of tumor susceptibility gene 101 in keratinocyte differentiation and chromatin remodeling of p16INK4a promotor. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110107-114213

Chicago Manual of Style (16th Edition):

You, Huey-Ling. “The roles of tumor susceptibility gene 101 in keratinocyte differentiation and chromatin remodeling of p16INK4a promotor.” 2007. Doctoral Dissertation, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110107-114213.

MLA Handbook (7th Edition):

You, Huey-Ling. “The roles of tumor susceptibility gene 101 in keratinocyte differentiation and chromatin remodeling of p16INK4a promotor.” 2007. Web. 25 Jan 2021.

Vancouver:

You H. The roles of tumor susceptibility gene 101 in keratinocyte differentiation and chromatin remodeling of p16INK4a promotor. [Internet] [Doctoral dissertation]. NSYSU; 2007. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110107-114213.

Council of Science Editors:

You H. The roles of tumor susceptibility gene 101 in keratinocyte differentiation and chromatin remodeling of p16INK4a promotor. [Doctoral Dissertation]. NSYSU; 2007. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110107-114213

8. Daehn, Ilse Sofia. Effect of growth factors on T-lymphocyte induced keratinocyte apoptosis.

Degree: 2007, Flinders University

 Atopic eczema is a T-lymphocyte mediated chronic inflammatory skin disorder. The interaction of CD4+ T-lymphocytes with epidermal keratinocytes results in dysregulated, chronic inflammation and altered barrier function.… (more)

Subjects/Keywords: keratinocyte; apoptosis; eczema; growth factors

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APA (6th Edition):

Daehn, I. S. (2007). Effect of growth factors on T-lymphocyte induced keratinocyte apoptosis. (Thesis). Flinders University. Retrieved from http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20071215.233705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Daehn, Ilse Sofia. “Effect of growth factors on T-lymphocyte induced keratinocyte apoptosis.” 2007. Thesis, Flinders University. Accessed January 25, 2021. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20071215.233705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Daehn, Ilse Sofia. “Effect of growth factors on T-lymphocyte induced keratinocyte apoptosis.” 2007. Web. 25 Jan 2021.

Vancouver:

Daehn IS. Effect of growth factors on T-lymphocyte induced keratinocyte apoptosis. [Internet] [Thesis]. Flinders University; 2007. [cited 2021 Jan 25]. Available from: http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20071215.233705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Daehn IS. Effect of growth factors on T-lymphocyte induced keratinocyte apoptosis. [Thesis]. Flinders University; 2007. Available from: http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20071215.233705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.

Degree: 博士(歯学), 2014, Fukuoka Dental College / 福岡歯科大学

Transforming growth factor-β1 (TGF-β1) reportedly causes the differentiation of fibroblasts to myofibroblasts during wound healing. We investigated the mechanism underlying the activation of latent TGF-β1… (more)

Subjects/Keywords: keratinocyte; fibroblast; transforming growth factor-β1; αv-integrin; matrix metalloproteinase

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APA (6th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, H. (2014). Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed January 25, 2021. http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa, Hiroyuki. “Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin.” 2014. Web. 25 Jan 2021.

Vancouver:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. [cited 2021 Jan 25]. Available from: http://id.nii.ac.jp/1167/00000003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hata, Shozaburo; Okamura, Kazuhiko; Hatta, Mitsutoki; Ishikawa H. Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. : Proteolytic and non-proteolytic activation of keratinocyte-derived latent TGF-β1 induces fibroblast differentiation in a wound-healing model using rat skin. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2014. Available from: http://id.nii.ac.jp/1167/00000003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Hiromatsu, Ryo; Hatta, Mitsutoki; Okamura, Kazuhiko; Sakagami, Ryuji. NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. : NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line.

Degree: 博士(歯学), 2015, Fukuoka Dental College / 福岡歯科大学

BACKGROUND:CLCA was postulated to be a calcium-activated chloride channel accessory protein. Recent reports indicate that CLCA isoforms are likely to be expressed in different layers… (more)

Subjects/Keywords: 転写制御; ケラチノサイト; CLCA; NF-kB; transcriptioal control; keratinocyte

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APA (6th Edition):

Hiromatsu, Ryo; Hatta, Mitsutoki; Okamura, Kazuhiko; Sakagami, R. (2015). NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. : NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000037/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hiromatsu, Ryo; Hatta, Mitsutoki; Okamura, Kazuhiko; Sakagami, Ryuji. “NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. : NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line.” 2015. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed January 25, 2021. http://id.nii.ac.jp/1167/00000037/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hiromatsu, Ryo; Hatta, Mitsutoki; Okamura, Kazuhiko; Sakagami, Ryuji. “NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. : NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line.” 2015. Web. 25 Jan 2021.

Vancouver:

Hiromatsu, Ryo; Hatta, Mitsutoki; Okamura, Kazuhiko; Sakagami R. NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. : NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. [cited 2021 Jan 25]. Available from: http://id.nii.ac.jp/1167/00000037/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hiromatsu, Ryo; Hatta, Mitsutoki; Okamura, Kazuhiko; Sakagami R. NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. : NF-κB-regulated transcriptional control of CLCA in a differentiated mouse keratinocyte line. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. Available from: http://id.nii.ac.jp/1167/00000037/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

11. Woods, Michael Connely. Cyclin-D1 Modulation of Vitamin-D-Induced Gene Expression in Oral Keratinocytes.

Degree: Oral Biology, 2013, UCLA

 Background: Oral cancer is the sixth most frequent cancer worldwide. Ninety percent of the newly diagnosed oral cancers (+400,000/ year) are characterized as squamous cell… (more)

Subjects/Keywords: Oncology; Dentistry; Cancer; Cyclin D1; Keratinocyte; Oral; vitamin D

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APA (6th Edition):

Woods, M. C. (2013). Cyclin-D1 Modulation of Vitamin-D-Induced Gene Expression in Oral Keratinocytes. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/7tq970cn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woods, Michael Connely. “Cyclin-D1 Modulation of Vitamin-D-Induced Gene Expression in Oral Keratinocytes.” 2013. Thesis, UCLA. Accessed January 25, 2021. http://www.escholarship.org/uc/item/7tq970cn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woods, Michael Connely. “Cyclin-D1 Modulation of Vitamin-D-Induced Gene Expression in Oral Keratinocytes.” 2013. Web. 25 Jan 2021.

Vancouver:

Woods MC. Cyclin-D1 Modulation of Vitamin-D-Induced Gene Expression in Oral Keratinocytes. [Internet] [Thesis]. UCLA; 2013. [cited 2021 Jan 25]. Available from: http://www.escholarship.org/uc/item/7tq970cn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woods MC. Cyclin-D1 Modulation of Vitamin-D-Induced Gene Expression in Oral Keratinocytes. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/7tq970cn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

12. Bodily, Jason Matthew. THE DIFFERENTIATION-DEPENDENT P742 PROMOTER AND THE HPV31 LIFE CYCLE .

Degree: 2008, Penn State University

 Human papillomaviruses (HPVs) are small circular DNA viruses that persistently infect stratified squamous epithelia. HPVs are implicated in the etiology of a wide variety of… (more)

Subjects/Keywords: papillomaviruses; transcription; keratinocyte; differentiation; replication

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APA (6th Edition):

Bodily, J. M. (2008). THE DIFFERENTIATION-DEPENDENT P742 PROMOTER AND THE HPV31 LIFE CYCLE . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/6506

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bodily, Jason Matthew. “THE DIFFERENTIATION-DEPENDENT P742 PROMOTER AND THE HPV31 LIFE CYCLE .” 2008. Thesis, Penn State University. Accessed January 25, 2021. https://submit-etda.libraries.psu.edu/catalog/6506.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bodily, Jason Matthew. “THE DIFFERENTIATION-DEPENDENT P742 PROMOTER AND THE HPV31 LIFE CYCLE .” 2008. Web. 25 Jan 2021.

Vancouver:

Bodily JM. THE DIFFERENTIATION-DEPENDENT P742 PROMOTER AND THE HPV31 LIFE CYCLE . [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Jan 25]. Available from: https://submit-etda.libraries.psu.edu/catalog/6506.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bodily JM. THE DIFFERENTIATION-DEPENDENT P742 PROMOTER AND THE HPV31 LIFE CYCLE . [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/6506

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

13. Jadoo, Sasha. The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies.

Degree: 2017, University of KwaZulu-Natal

 A number of different methods to generate stratified keratinocyte layers have been published. These involved the use of normal human epidermal keratinocytes (NHEKs/NEKS), which have… (more)

Subjects/Keywords: Chlamydia trachomatis.; Lymphogranuloma venereum.; Keratinocytes.; Stratified keratinocyte model.

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APA (6th Edition):

Jadoo, S. (2017). The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies. (Thesis). University of KwaZulu-Natal. Retrieved from https://researchspace.ukzn.ac.za/handle/10413/18370

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jadoo, Sasha. “The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies.” 2017. Thesis, University of KwaZulu-Natal. Accessed January 25, 2021. https://researchspace.ukzn.ac.za/handle/10413/18370.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jadoo, Sasha. “The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies.” 2017. Web. 25 Jan 2021.

Vancouver:

Jadoo S. The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies. [Internet] [Thesis]. University of KwaZulu-Natal; 2017. [cited 2021 Jan 25]. Available from: https://researchspace.ukzn.ac.za/handle/10413/18370.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jadoo S. The development of a stratified keratinocyte model for chlamydia trachomatis pathogenesis studies. [Thesis]. University of KwaZulu-Natal; 2017. Available from: https://researchspace.ukzn.ac.za/handle/10413/18370

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

14. Chastkofsky, Michael I. The Role of Protein Tyrosine Kinase 6 in UVB-Induced Signaling and Skin Carcinogenesis.

Degree: 2015, University of Illinois – Chicago

 Protein Tyrosine Kinase 6 (PTK6) belongs to a small family of Src-related nonmyristoylated intracellular tyrosine kinases and is involved in normal epithelial homeostasis and cancer.… (more)

Subjects/Keywords: PTK6; SIK; BRK; Skin; UVB; Keratinocyte; SCC; Cancer; STAT3; FAK; BCAR1

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APA (6th Edition):

Chastkofsky, M. I. (2015). The Role of Protein Tyrosine Kinase 6 in UVB-Induced Signaling and Skin Carcinogenesis. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/19403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chastkofsky, Michael I. “The Role of Protein Tyrosine Kinase 6 in UVB-Induced Signaling and Skin Carcinogenesis.” 2015. Thesis, University of Illinois – Chicago. Accessed January 25, 2021. http://hdl.handle.net/10027/19403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chastkofsky, Michael I. “The Role of Protein Tyrosine Kinase 6 in UVB-Induced Signaling and Skin Carcinogenesis.” 2015. Web. 25 Jan 2021.

Vancouver:

Chastkofsky MI. The Role of Protein Tyrosine Kinase 6 in UVB-Induced Signaling and Skin Carcinogenesis. [Internet] [Thesis]. University of Illinois – Chicago; 2015. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10027/19403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chastkofsky MI. The Role of Protein Tyrosine Kinase 6 in UVB-Induced Signaling and Skin Carcinogenesis. [Thesis]. University of Illinois – Chicago; 2015. Available from: http://hdl.handle.net/10027/19403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. 鎌口, 真由美. The differences of collagen XVII between the oral mucosa and the skin discover the pathogenesis of oral lesions in pemphigoid.

Degree: 博士(歯学), 歯学, 2019, Hokkaido University

 The basement membrane zone (BMZ) consists of multiple components, including collagen XVII (COL17), which is the major targeted antigen of pemphigoid diseases such as bullous… (more)

Subjects/Keywords: keratinocyte; collagen XVII; oral mucosa; bullous pemphigoid; depletion

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APA (6th Edition):

鎌口, . (2019). The differences of collagen XVII between the oral mucosa and the skin discover the pathogenesis of oral lesions in pemphigoid. (Doctoral Dissertation). Hokkaido University. Retrieved from http://hdl.handle.net/2115/76953

Chicago Manual of Style (16th Edition):

鎌口, 真由美. “The differences of collagen XVII between the oral mucosa and the skin discover the pathogenesis of oral lesions in pemphigoid.” 2019. Doctoral Dissertation, Hokkaido University. Accessed January 25, 2021. http://hdl.handle.net/2115/76953.

MLA Handbook (7th Edition):

鎌口, 真由美. “The differences of collagen XVII between the oral mucosa and the skin discover the pathogenesis of oral lesions in pemphigoid.” 2019. Web. 25 Jan 2021.

Vancouver:

鎌口 . The differences of collagen XVII between the oral mucosa and the skin discover the pathogenesis of oral lesions in pemphigoid. [Internet] [Doctoral dissertation]. Hokkaido University; 2019. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/2115/76953.

Council of Science Editors:

鎌口 . The differences of collagen XVII between the oral mucosa and the skin discover the pathogenesis of oral lesions in pemphigoid. [Doctoral Dissertation]. Hokkaido University; 2019. Available from: http://hdl.handle.net/2115/76953


University of Bradford

16. Lewis, Christopher John. The role of bone morphogenetic protein signalling in the control of skin repair after wounding : cellular and molecular mechanisms of cutaneous wound healing mediated by bone morphogenetic proteins and their antagonist Noggin.

Degree: PhD, 2013, University of Bradford

 Bone morphogenetic proteins (BMPs) and their receptors (BMPRs) coordinate tissue development and postnatal remodelling by regulating proliferation, differentiation and apoptosis. However, their role in wound… (more)

Subjects/Keywords: 617.1; Wound healing, Bone morphogenetic protein (BMP), Skin, Keratinocyte, Noggin, Smad

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APA (6th Edition):

Lewis, C. J. (2013). The role of bone morphogenetic protein signalling in the control of skin repair after wounding : cellular and molecular mechanisms of cutaneous wound healing mediated by bone morphogenetic proteins and their antagonist Noggin. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/7337

Chicago Manual of Style (16th Edition):

Lewis, Christopher John. “The role of bone morphogenetic protein signalling in the control of skin repair after wounding : cellular and molecular mechanisms of cutaneous wound healing mediated by bone morphogenetic proteins and their antagonist Noggin.” 2013. Doctoral Dissertation, University of Bradford. Accessed January 25, 2021. http://hdl.handle.net/10454/7337.

MLA Handbook (7th Edition):

Lewis, Christopher John. “The role of bone morphogenetic protein signalling in the control of skin repair after wounding : cellular and molecular mechanisms of cutaneous wound healing mediated by bone morphogenetic proteins and their antagonist Noggin.” 2013. Web. 25 Jan 2021.

Vancouver:

Lewis CJ. The role of bone morphogenetic protein signalling in the control of skin repair after wounding : cellular and molecular mechanisms of cutaneous wound healing mediated by bone morphogenetic proteins and their antagonist Noggin. [Internet] [Doctoral dissertation]. University of Bradford; 2013. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10454/7337.

Council of Science Editors:

Lewis CJ. The role of bone morphogenetic protein signalling in the control of skin repair after wounding : cellular and molecular mechanisms of cutaneous wound healing mediated by bone morphogenetic proteins and their antagonist Noggin. [Doctoral Dissertation]. University of Bradford; 2013. Available from: http://hdl.handle.net/10454/7337


University of Western Ontario

17. Ableser, Mark J. Cx43 Reduces Melanoma Growth Within a Keratinocyte Microenvironment and During Tumorigenesis in vivo.

Degree: 2013, University of Western Ontario

 Connexins have been frequently identified as tumor suppressors in many cancers, however, their role in melanoma tumorigenesis remains controversial. Here, we show that B16-BL6 mouse… (more)

Subjects/Keywords: Connexin43; Connexin26; Melanoma; Tumorigenesis; Keratinocyte; BL6 Mouse Melanoma Cells; Cancer Biology

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APA (6th Edition):

Ableser, M. J. (2013). Cx43 Reduces Melanoma Growth Within a Keratinocyte Microenvironment and During Tumorigenesis in vivo. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ableser, Mark J. “Cx43 Reduces Melanoma Growth Within a Keratinocyte Microenvironment and During Tumorigenesis in vivo.” 2013. Thesis, University of Western Ontario. Accessed January 25, 2021. https://ir.lib.uwo.ca/etd/1473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ableser, Mark J. “Cx43 Reduces Melanoma Growth Within a Keratinocyte Microenvironment and During Tumorigenesis in vivo.” 2013. Web. 25 Jan 2021.

Vancouver:

Ableser MJ. Cx43 Reduces Melanoma Growth Within a Keratinocyte Microenvironment and During Tumorigenesis in vivo. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2021 Jan 25]. Available from: https://ir.lib.uwo.ca/etd/1473.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ableser MJ. Cx43 Reduces Melanoma Growth Within a Keratinocyte Microenvironment and During Tumorigenesis in vivo. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1473

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. KOH MEI LING, ROSITA. EFFECTS OF THE TISSUE CULTURE ENVIRONMENT ON HUMAN PRIMARY KERATINOCYTES.

Degree: 2014, National University of Singapore

Subjects/Keywords: Keratinocyte; Culture; Epigenetic Regulation; Oxygen

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APA (6th Edition):

KOH MEI LING, R. (2014). EFFECTS OF THE TISSUE CULTURE ENVIRONMENT ON HUMAN PRIMARY KERATINOCYTES. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/119453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

KOH MEI LING, ROSITA. “EFFECTS OF THE TISSUE CULTURE ENVIRONMENT ON HUMAN PRIMARY KERATINOCYTES.” 2014. Thesis, National University of Singapore. Accessed January 25, 2021. http://scholarbank.nus.edu.sg/handle/10635/119453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

KOH MEI LING, ROSITA. “EFFECTS OF THE TISSUE CULTURE ENVIRONMENT ON HUMAN PRIMARY KERATINOCYTES.” 2014. Web. 25 Jan 2021.

Vancouver:

KOH MEI LING R. EFFECTS OF THE TISSUE CULTURE ENVIRONMENT ON HUMAN PRIMARY KERATINOCYTES. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2021 Jan 25]. Available from: http://scholarbank.nus.edu.sg/handle/10635/119453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

KOH MEI LING R. EFFECTS OF THE TISSUE CULTURE ENVIRONMENT ON HUMAN PRIMARY KERATINOCYTES. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/119453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Clement, Amanda Lynn. Engineering the Keratinocyte Microenvironment: Harnessing Topography to Direct Cellular Function.

Degree: PhD, 2015, Worcester Polytechnic Institute

 Skin wound healing presents a challenging and expensive clinical problem with nearly 20 million wounds requiring intervention leading to an annual cost of more than… (more)

Subjects/Keywords: microtopography; keratinocyte function; tissue engineered skin; dermal-epidermal junction

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APA (6th Edition):

Clement, A. L. (2015). Engineering the Keratinocyte Microenvironment: Harnessing Topography to Direct Cellular Function. (Doctoral Dissertation). Worcester Polytechnic Institute. Retrieved from etd-011215-151605 ; https://digitalcommons.wpi.edu/etd-dissertations/23

Chicago Manual of Style (16th Edition):

Clement, Amanda Lynn. “Engineering the Keratinocyte Microenvironment: Harnessing Topography to Direct Cellular Function.” 2015. Doctoral Dissertation, Worcester Polytechnic Institute. Accessed January 25, 2021. etd-011215-151605 ; https://digitalcommons.wpi.edu/etd-dissertations/23.

MLA Handbook (7th Edition):

Clement, Amanda Lynn. “Engineering the Keratinocyte Microenvironment: Harnessing Topography to Direct Cellular Function.” 2015. Web. 25 Jan 2021.

Vancouver:

Clement AL. Engineering the Keratinocyte Microenvironment: Harnessing Topography to Direct Cellular Function. [Internet] [Doctoral dissertation]. Worcester Polytechnic Institute; 2015. [cited 2021 Jan 25]. Available from: etd-011215-151605 ; https://digitalcommons.wpi.edu/etd-dissertations/23.

Council of Science Editors:

Clement AL. Engineering the Keratinocyte Microenvironment: Harnessing Topography to Direct Cellular Function. [Doctoral Dissertation]. Worcester Polytechnic Institute; 2015. Available from: etd-011215-151605 ; https://digitalcommons.wpi.edu/etd-dissertations/23


Universitat de Valencia

20. Carceller Zazo, Elena. Analysis of the molecular mechanisms that mediate the therapeutic actions of glucocorticoids in skin .

Degree: 2017, Universitat de Valencia

 Los glucocorticoides (GCs) sintéticos han sido ampliamente utilizados para el tratamiento de las enfermedades autoinmunes e inflamatorias y ejercen sus acciones mediante la unión al… (more)

Subjects/Keywords: gilz; glucocorticoid receptor; zfp36; klf4; keratinocyte; mouse; skin

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APA (6th Edition):

Carceller Zazo, E. (2017). Analysis of the molecular mechanisms that mediate the therapeutic actions of glucocorticoids in skin . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/59278

Chicago Manual of Style (16th Edition):

Carceller Zazo, Elena. “Analysis of the molecular mechanisms that mediate the therapeutic actions of glucocorticoids in skin .” 2017. Doctoral Dissertation, Universitat de Valencia. Accessed January 25, 2021. http://hdl.handle.net/10550/59278.

MLA Handbook (7th Edition):

Carceller Zazo, Elena. “Analysis of the molecular mechanisms that mediate the therapeutic actions of glucocorticoids in skin .” 2017. Web. 25 Jan 2021.

Vancouver:

Carceller Zazo E. Analysis of the molecular mechanisms that mediate the therapeutic actions of glucocorticoids in skin . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2017. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10550/59278.

Council of Science Editors:

Carceller Zazo E. Analysis of the molecular mechanisms that mediate the therapeutic actions of glucocorticoids in skin . [Doctoral Dissertation]. Universitat de Valencia; 2017. Available from: http://hdl.handle.net/10550/59278


Universitat de Valencia

21. Sanchis Gandía, Ana. Estudio funcional del receptor de glucocorticoides en desarrollo y reparación epitelial .

Degree: 2013, Universitat de Valencia

 El receptor de glucocorticoides (GR) juega un papel crucial en la morfogénesis epidérmica durante el desarrollo embrionario, como se ha demostrado mediante el análisis de… (more)

Subjects/Keywords: wound healing; keratinocyte; genetically modified mice; eye development skin; glucocorticoid receptor

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APA (6th Edition):

Sanchis Gandía, A. (2013). Estudio funcional del receptor de glucocorticoides en desarrollo y reparación epitelial . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/29048

Chicago Manual of Style (16th Edition):

Sanchis Gandía, Ana. “Estudio funcional del receptor de glucocorticoides en desarrollo y reparación epitelial .” 2013. Doctoral Dissertation, Universitat de Valencia. Accessed January 25, 2021. http://hdl.handle.net/10550/29048.

MLA Handbook (7th Edition):

Sanchis Gandía, Ana. “Estudio funcional del receptor de glucocorticoides en desarrollo y reparación epitelial .” 2013. Web. 25 Jan 2021.

Vancouver:

Sanchis Gandía A. Estudio funcional del receptor de glucocorticoides en desarrollo y reparación epitelial . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2013. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10550/29048.

Council of Science Editors:

Sanchis Gandía A. Estudio funcional del receptor de glucocorticoides en desarrollo y reparación epitelial . [Doctoral Dissertation]. Universitat de Valencia; 2013. Available from: http://hdl.handle.net/10550/29048


Loyola University Chicago

22. Larm, Ashley Lynn. Characterizing the Mechanisms by Which Community Associated Methicillin-Resistant Staphylococcus Aureus Influences Keratinocyte Innate Immune Responses During Recurrent Infection.

Degree: MS, Biological Science, 2014, Loyola University Chicago

  Community associated–methicillin resistant Staphylococcus aureus (CA–MRSA) infection has become a major health concern. In human epidermal keratinocytes, S. aureus is mainly recognized through toll–like… (more)

Subjects/Keywords: Community-associated MRSA; Keratinocyte immune response; Immunology and Infectious Disease

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APA (6th Edition):

Larm, A. L. (2014). Characterizing the Mechanisms by Which Community Associated Methicillin-Resistant Staphylococcus Aureus Influences Keratinocyte Innate Immune Responses During Recurrent Infection. (Thesis). Loyola University Chicago. Retrieved from https://ecommons.luc.edu/luc_theses/2627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Larm, Ashley Lynn. “Characterizing the Mechanisms by Which Community Associated Methicillin-Resistant Staphylococcus Aureus Influences Keratinocyte Innate Immune Responses During Recurrent Infection.” 2014. Thesis, Loyola University Chicago. Accessed January 25, 2021. https://ecommons.luc.edu/luc_theses/2627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Larm, Ashley Lynn. “Characterizing the Mechanisms by Which Community Associated Methicillin-Resistant Staphylococcus Aureus Influences Keratinocyte Innate Immune Responses During Recurrent Infection.” 2014. Web. 25 Jan 2021.

Vancouver:

Larm AL. Characterizing the Mechanisms by Which Community Associated Methicillin-Resistant Staphylococcus Aureus Influences Keratinocyte Innate Immune Responses During Recurrent Infection. [Internet] [Thesis]. Loyola University Chicago; 2014. [cited 2021 Jan 25]. Available from: https://ecommons.luc.edu/luc_theses/2627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Larm AL. Characterizing the Mechanisms by Which Community Associated Methicillin-Resistant Staphylococcus Aureus Influences Keratinocyte Innate Immune Responses During Recurrent Infection. [Thesis]. Loyola University Chicago; 2014. Available from: https://ecommons.luc.edu/luc_theses/2627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

23. Li, Min. KGF-1 and KGF receptor expression in human periodontal disease and in vitro microwounding-associated-ligand-independent KGFR activation.

Degree: PhD, Dental Science, 2007, University of British Columbia

 Objectives: Periodontal disease is a chronic inflammation resulting in periodontal attachment loss. Keratinocyte Growth Factor-1 (KGF-1) is upregulated in chronic inflammation and specifically stimulates epithelial… (more)

Subjects/Keywords: periodontal disease; keratinocyte; KGFR; ligand

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APA (6th Edition):

Li, M. (2007). KGF-1 and KGF receptor expression in human periodontal disease and in vitro microwounding-associated-ligand-independent KGFR activation. (Doctoral Dissertation). University of British Columbia. Retrieved from http://hdl.handle.net/2429/418

Chicago Manual of Style (16th Edition):

Li, Min. “KGF-1 and KGF receptor expression in human periodontal disease and in vitro microwounding-associated-ligand-independent KGFR activation.” 2007. Doctoral Dissertation, University of British Columbia. Accessed January 25, 2021. http://hdl.handle.net/2429/418.

MLA Handbook (7th Edition):

Li, Min. “KGF-1 and KGF receptor expression in human periodontal disease and in vitro microwounding-associated-ligand-independent KGFR activation.” 2007. Web. 25 Jan 2021.

Vancouver:

Li M. KGF-1 and KGF receptor expression in human periodontal disease and in vitro microwounding-associated-ligand-independent KGFR activation. [Internet] [Doctoral dissertation]. University of British Columbia; 2007. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/2429/418.

Council of Science Editors:

Li M. KGF-1 and KGF receptor expression in human periodontal disease and in vitro microwounding-associated-ligand-independent KGFR activation. [Doctoral Dissertation]. University of British Columbia; 2007. Available from: http://hdl.handle.net/2429/418

24. Sturniolo, Michael Thomas. Tazarotene-Induced Gene 3: A Novel Regulator of Keratinocyte Differentiation.

Degree: PhD, Molecular Virology, 2005, Case Western Reserve University School of Graduate Studies

 Tazarotene-induced gene 3 (TIG3) is a novel regulatory protein expressed in the human epidermis. TIG3 expression in keratinocytes results in a decrease in both proliferation… (more)

Subjects/Keywords: Biology, Cell; TIG3; Transglutaminase; Keratinocyte

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APA (6th Edition):

Sturniolo, M. T. (2005). Tazarotene-Induced Gene 3: A Novel Regulator of Keratinocyte Differentiation. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1100296278

Chicago Manual of Style (16th Edition):

Sturniolo, Michael Thomas. “Tazarotene-Induced Gene 3: A Novel Regulator of Keratinocyte Differentiation.” 2005. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed January 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1100296278.

MLA Handbook (7th Edition):

Sturniolo, Michael Thomas. “Tazarotene-Induced Gene 3: A Novel Regulator of Keratinocyte Differentiation.” 2005. Web. 25 Jan 2021.

Vancouver:

Sturniolo MT. Tazarotene-Induced Gene 3: A Novel Regulator of Keratinocyte Differentiation. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2005. [cited 2021 Jan 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1100296278.

Council of Science Editors:

Sturniolo MT. Tazarotene-Induced Gene 3: A Novel Regulator of Keratinocyte Differentiation. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1100296278


Wright State University

25. Abbas, Asma A. HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS.

Degree: MS, Microbiology and Immunology, 2011, Wright State University

 The hypothesis for this research was: Herpes simplex virus type 1 (HSV-1) infection of murine keratinocyte cell lines (HEL-30 and PAM-212) treated with mitotic inhibitors… (more)

Subjects/Keywords: Immunology; HSV-1 infection; keratinocyte cell lines; mitotic inhibitors

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APA (6th Edition):

Abbas, A. A. (2011). HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668

Chicago Manual of Style (16th Edition):

Abbas, Asma A. “HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS.” 2011. Masters Thesis, Wright State University. Accessed January 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668.

MLA Handbook (7th Edition):

Abbas, Asma A. “HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS.” 2011. Web. 25 Jan 2021.

Vancouver:

Abbas AA. HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS. [Internet] [Masters thesis]. Wright State University; 2011. [cited 2021 Jan 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668.

Council of Science Editors:

Abbas AA. HSV-1 INFECTION IN KERATINOCYTE CELL LINES TREATED WITH MITOTIC INHIBITORS. [Masters Thesis]. Wright State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1303839668


The Ohio State University

26. Johnson, Kelly Elizabeth. Direct Effects of VEGF on Keratinocyte Function During Skin Carcinogenesis and Wound Healing.

Degree: PhD, Integrated Biomedical Science Graduate Program, 2013, The Ohio State University

 Epidermal keratinocytes, the predominant cell type in the epidermis, play a crucial role in two processes in the skin: skin carcinogenesis and cutaneous wound healing.… (more)

Subjects/Keywords: Biomedical Research; skin; keratinocyte; VEGF; skin cancer; wound healing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johnson, K. E. (2013). Direct Effects of VEGF on Keratinocyte Function During Skin Carcinogenesis and Wound Healing. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1376662806

Chicago Manual of Style (16th Edition):

Johnson, Kelly Elizabeth. “Direct Effects of VEGF on Keratinocyte Function During Skin Carcinogenesis and Wound Healing.” 2013. Doctoral Dissertation, The Ohio State University. Accessed January 25, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1376662806.

MLA Handbook (7th Edition):

Johnson, Kelly Elizabeth. “Direct Effects of VEGF on Keratinocyte Function During Skin Carcinogenesis and Wound Healing.” 2013. Web. 25 Jan 2021.

Vancouver:

Johnson KE. Direct Effects of VEGF on Keratinocyte Function During Skin Carcinogenesis and Wound Healing. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2021 Jan 25]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1376662806.

Council of Science Editors:

Johnson KE. Direct Effects of VEGF on Keratinocyte Function During Skin Carcinogenesis and Wound Healing. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1376662806


University of Cambridge

27. Saunders-Wood, Taylor. Investigating Early Lesion Formation Following Papillomavirus Infection Using a Mouse Model and Cell Culture.

Degree: PhD, 2020, University of Cambridge

 Papillomaviruses (PV) are small non-enveloped double-stranded DNA tumour viruses, which are able to infect more than 80 different host species. They are a diverse group… (more)

Subjects/Keywords: virology; papillomavirus; human papillomavirus; mouse model; homeostasis; cell biology; keratinocyte

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APA (6th Edition):

Saunders-Wood, T. (2020). Investigating Early Lesion Formation Following Papillomavirus Infection Using a Mouse Model and Cell Culture. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/307401

Chicago Manual of Style (16th Edition):

Saunders-Wood, Taylor. “Investigating Early Lesion Formation Following Papillomavirus Infection Using a Mouse Model and Cell Culture.” 2020. Doctoral Dissertation, University of Cambridge. Accessed January 25, 2021. https://www.repository.cam.ac.uk/handle/1810/307401.

MLA Handbook (7th Edition):

Saunders-Wood, Taylor. “Investigating Early Lesion Formation Following Papillomavirus Infection Using a Mouse Model and Cell Culture.” 2020. Web. 25 Jan 2021.

Vancouver:

Saunders-Wood T. Investigating Early Lesion Formation Following Papillomavirus Infection Using a Mouse Model and Cell Culture. [Internet] [Doctoral dissertation]. University of Cambridge; 2020. [cited 2021 Jan 25]. Available from: https://www.repository.cam.ac.uk/handle/1810/307401.

Council of Science Editors:

Saunders-Wood T. Investigating Early Lesion Formation Following Papillomavirus Infection Using a Mouse Model and Cell Culture. [Doctoral Dissertation]. University of Cambridge; 2020. Available from: https://www.repository.cam.ac.uk/handle/1810/307401


Penn State University

28. Chapman, Sandra Elizabeth. ANALYZING THE REGULATION OF HPV REPLICATION AND TRANSCRIPTION IN ITS NATURAL HOST, THE KERATINOCYTE .

Degree: 2010, Penn State University

 Each papillomavirus is species specific and replicates persistently in a specific type of cutaneous or mucosal epithelium. The keratinocytes of the basal layer of the… (more)

Subjects/Keywords: transcription; papillomaviruses; keratinocyte; immortalization; differentiation; replication

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chapman, S. E. (2010). ANALYZING THE REGULATION OF HPV REPLICATION AND TRANSCRIPTION IN ITS NATURAL HOST, THE KERATINOCYTE . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11273

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chapman, Sandra Elizabeth. “ANALYZING THE REGULATION OF HPV REPLICATION AND TRANSCRIPTION IN ITS NATURAL HOST, THE KERATINOCYTE .” 2010. Thesis, Penn State University. Accessed January 25, 2021. https://submit-etda.libraries.psu.edu/catalog/11273.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chapman, Sandra Elizabeth. “ANALYZING THE REGULATION OF HPV REPLICATION AND TRANSCRIPTION IN ITS NATURAL HOST, THE KERATINOCYTE .” 2010. Web. 25 Jan 2021.

Vancouver:

Chapman SE. ANALYZING THE REGULATION OF HPV REPLICATION AND TRANSCRIPTION IN ITS NATURAL HOST, THE KERATINOCYTE . [Internet] [Thesis]. Penn State University; 2010. [cited 2021 Jan 25]. Available from: https://submit-etda.libraries.psu.edu/catalog/11273.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chapman SE. ANALYZING THE REGULATION OF HPV REPLICATION AND TRANSCRIPTION IN ITS NATURAL HOST, THE KERATINOCYTE . [Thesis]. Penn State University; 2010. Available from: https://submit-etda.libraries.psu.edu/catalog/11273

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. O'Neill, Adrian Thomas. A Microfluidic Platform for Human Epidermal Keratinocyte Cytotoxicity Assays.

Degree: PhD, Biomedical Engineering, 2009, North Carolina State University

 Linear dilution is a method to create linearly varying concentrations of a solution. Linear dilutions are commonly used in biological studies where the threshold concentration… (more)

Subjects/Keywords: keratinocyte; cytotoxicity; dilution; microfluidics; PDMS

keratinocyte irritation studies. Exogenous noxes may also induce the release of IL-1α, IL-1β, IL-6… …differentiation factor and stimulates keratinocyte proliferation. IL-8 release is a good indicator of… …irradiated human keratinocyte cell line: potential role of nitric oxide. FASEB J. 13, 1817-1824… …keratinocyte-derived cytokines in chemical toxicity. Toxicol. Lett. 82-83, 471-476. Monteiro-Riviere… …Assessment of Nanotube Cytotoxicity using Human Keratinocyte Cells. J. Toxicol. Environ. Health… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

O'Neill, A. T. (2009). A Microfluidic Platform for Human Epidermal Keratinocyte Cytotoxicity Assays. (Doctoral Dissertation). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/5477

Chicago Manual of Style (16th Edition):

O'Neill, Adrian Thomas. “A Microfluidic Platform for Human Epidermal Keratinocyte Cytotoxicity Assays.” 2009. Doctoral Dissertation, North Carolina State University. Accessed January 25, 2021. http://www.lib.ncsu.edu/resolver/1840.16/5477.

MLA Handbook (7th Edition):

O'Neill, Adrian Thomas. “A Microfluidic Platform for Human Epidermal Keratinocyte Cytotoxicity Assays.” 2009. Web. 25 Jan 2021.

Vancouver:

O'Neill AT. A Microfluidic Platform for Human Epidermal Keratinocyte Cytotoxicity Assays. [Internet] [Doctoral dissertation]. North Carolina State University; 2009. [cited 2021 Jan 25]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/5477.

Council of Science Editors:

O'Neill AT. A Microfluidic Platform for Human Epidermal Keratinocyte Cytotoxicity Assays. [Doctoral Dissertation]. North Carolina State University; 2009. Available from: http://www.lib.ncsu.edu/resolver/1840.16/5477

30. Rabeony, Hanitriniaina. Étude de l'implication des cytokines dans l'inflammation cutanée et application à l'identification de cibles thérapeutiques pertinentes : Study of involvment of cytokines in skin inflammation and application to the identification of relevant therapeutic targets.

Degree: Docteur es, Biologie, médecine, santé, 2014, Poitiers

Un réseau de cytokine complexe a été décrit dans le psoriasis mettant en évidence le rôle central des cytokines proinflammatoires dans la physiopathologie de cette… (more)

Subjects/Keywords: Psoriasis; Kératinocyte; Inflammation; Cytokine; Différenciation; Imiquimod; Psoriasis; Keratinocyte; Inflammation; Cytokine; Differentiation; Imiquimod; 616.526

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rabeony, H. (2014). Étude de l'implication des cytokines dans l'inflammation cutanée et application à l'identification de cibles thérapeutiques pertinentes : Study of involvment of cytokines in skin inflammation and application to the identification of relevant therapeutic targets. (Doctoral Dissertation). Poitiers. Retrieved from http://www.theses.fr/2014POIT1404

Chicago Manual of Style (16th Edition):

Rabeony, Hanitriniaina. “Étude de l'implication des cytokines dans l'inflammation cutanée et application à l'identification de cibles thérapeutiques pertinentes : Study of involvment of cytokines in skin inflammation and application to the identification of relevant therapeutic targets.” 2014. Doctoral Dissertation, Poitiers. Accessed January 25, 2021. http://www.theses.fr/2014POIT1404.

MLA Handbook (7th Edition):

Rabeony, Hanitriniaina. “Étude de l'implication des cytokines dans l'inflammation cutanée et application à l'identification de cibles thérapeutiques pertinentes : Study of involvment of cytokines in skin inflammation and application to the identification of relevant therapeutic targets.” 2014. Web. 25 Jan 2021.

Vancouver:

Rabeony H. Étude de l'implication des cytokines dans l'inflammation cutanée et application à l'identification de cibles thérapeutiques pertinentes : Study of involvment of cytokines in skin inflammation and application to the identification of relevant therapeutic targets. [Internet] [Doctoral dissertation]. Poitiers; 2014. [cited 2021 Jan 25]. Available from: http://www.theses.fr/2014POIT1404.

Council of Science Editors:

Rabeony H. Étude de l'implication des cytokines dans l'inflammation cutanée et application à l'identification de cibles thérapeutiques pertinentes : Study of involvment of cytokines in skin inflammation and application to the identification of relevant therapeutic targets. [Doctoral Dissertation]. Poitiers; 2014. Available from: http://www.theses.fr/2014POIT1404

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