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You searched for subject:(jak vnit n ). Showing records 1 – 30 of 8019 total matches.

[1] [2] [3] [4] [5] … [268]

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1. Tardif, Nicolas. Mechanosignaling through Caveolae : A New Role for the Control of JAK-STAT Signaling : Mécano-signalisation par les cavéoles : un rôle nouveau dans le contrôle de la voie de signalisation JAK-STAT.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université Paris-Saclay (ComUE)

Les cavéoles sont des invaginations en forme de coupelle à la membrane plasmique. Ces organelles multifonctionnelles jouent entre autres, un rôle clé dans la mécano-protection… (more)

Subjects/Keywords: Mécanosignalisation; Mécanotransduction; Cavéoles; JAK-STAT; Mechanosignalling; Mechanotransduction; Caveolae; JAK-STAT

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tardif, N. (2018). Mechanosignaling through Caveolae : A New Role for the Control of JAK-STAT Signaling : Mécano-signalisation par les cavéoles : un rôle nouveau dans le contrôle de la voie de signalisation JAK-STAT. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS337

Chicago Manual of Style (16th Edition):

Tardif, Nicolas. “Mechanosignaling through Caveolae : A New Role for the Control of JAK-STAT Signaling : Mécano-signalisation par les cavéoles : un rôle nouveau dans le contrôle de la voie de signalisation JAK-STAT.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 18, 2021. http://www.theses.fr/2018SACLS337.

MLA Handbook (7th Edition):

Tardif, Nicolas. “Mechanosignaling through Caveolae : A New Role for the Control of JAK-STAT Signaling : Mécano-signalisation par les cavéoles : un rôle nouveau dans le contrôle de la voie de signalisation JAK-STAT.” 2018. Web. 18 Jan 2021.

Vancouver:

Tardif N. Mechanosignaling through Caveolae : A New Role for the Control of JAK-STAT Signaling : Mécano-signalisation par les cavéoles : un rôle nouveau dans le contrôle de la voie de signalisation JAK-STAT. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2018SACLS337.

Council of Science Editors:

Tardif N. Mechanosignaling through Caveolae : A New Role for the Control of JAK-STAT Signaling : Mécano-signalisation par les cavéoles : un rôle nouveau dans le contrôle de la voie de signalisation JAK-STAT. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS337

2. de Carvalho Leandro, Danilo. Análise da imunidade de Aedes Aegypti (Diptera: Culicidae) ao vírus dengue em populações de campo com competência vetorial diferenciada .

Degree: 2011, Universidade Federal de Pernambuco

 Um dos determinantes envolvidos no complexo ciclo de transmissão da dengue é o nível de susceptibilidade do Aedes aegypti ao vírus dengue (DENV), ou seja,… (more)

Subjects/Keywords: Interação mosquito-DENV; Toll; JAK-STAT; RNAi.

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APA (6th Edition):

de Carvalho Leandro, D. (2011). Análise da imunidade de Aedes Aegypti (Diptera: Culicidae) ao vírus dengue em populações de campo com competência vetorial diferenciada . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/1020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

de Carvalho Leandro, Danilo. “Análise da imunidade de Aedes Aegypti (Diptera: Culicidae) ao vírus dengue em populações de campo com competência vetorial diferenciada .” 2011. Thesis, Universidade Federal de Pernambuco. Accessed January 18, 2021. http://repositorio.ufpe.br/handle/123456789/1020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

de Carvalho Leandro, Danilo. “Análise da imunidade de Aedes Aegypti (Diptera: Culicidae) ao vírus dengue em populações de campo com competência vetorial diferenciada .” 2011. Web. 18 Jan 2021.

Vancouver:

de Carvalho Leandro D. Análise da imunidade de Aedes Aegypti (Diptera: Culicidae) ao vírus dengue em populações de campo com competência vetorial diferenciada . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2011. [cited 2021 Jan 18]. Available from: http://repositorio.ufpe.br/handle/123456789/1020.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

de Carvalho Leandro D. Análise da imunidade de Aedes Aegypti (Diptera: Culicidae) ao vírus dengue em populações de campo com competência vetorial diferenciada . [Thesis]. Universidade Federal de Pernambuco; 2011. Available from: http://repositorio.ufpe.br/handle/123456789/1020

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Amborn, Anna. JAK Medlemsbank och räntan : Ett räntefritt alternativ.

Degree: Business and Economic Studies, 2012, University of Gävle

Titel: JAK Medlemsbank och räntan – Ett räntefritt alternativ. Nivå: Kandidatnivå. Författare: Anna Amborn och Linda Elgestad. Handledare: Maria Fregidou-Malama. Datum: 2012-06-28. Syfte: Studien… (more)

Subjects/Keywords: Räntefri; JAK Medlemsbank; ränta; avgift; lånekostnad; kooperativ

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APA (6th Edition):

Amborn, A. (2012). JAK Medlemsbank och räntan : Ett räntefritt alternativ. (Thesis). University of Gävle. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:hig:diva-12320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amborn, Anna. “JAK Medlemsbank och räntan : Ett räntefritt alternativ.” 2012. Thesis, University of Gävle. Accessed January 18, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:hig:diva-12320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amborn, Anna. “JAK Medlemsbank och räntan : Ett räntefritt alternativ.” 2012. Web. 18 Jan 2021.

Vancouver:

Amborn A. JAK Medlemsbank och räntan : Ett räntefritt alternativ. [Internet] [Thesis]. University of Gävle; 2012. [cited 2021 Jan 18]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hig:diva-12320.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amborn A. JAK Medlemsbank och räntan : Ett räntefritt alternativ. [Thesis]. University of Gävle; 2012. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hig:diva-12320

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Notre Dame

4. Francis James Raycroft. Characterization of the JAK1 / Stat3 pathway during zebrafish retinal development and light-induced photoreceptor regeneration</h1>.

Degree: Biological Sciences, 2014, University of Notre Dame

  The zebrafish is an excellent model organism to use in the study of molecular pathways important in both retinal development and neuronal regeneration. The… (more)

Subjects/Keywords: jak; stat3; socs; retina; zebrafish; regeneration

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APA (6th Edition):

Raycroft, F. J. (2014). Characterization of the JAK1 / Stat3 pathway during zebrafish retinal development and light-induced photoreceptor regeneration</h1>. (Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/jw827942h2c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raycroft, Francis James. “Characterization of the JAK1 / Stat3 pathway during zebrafish retinal development and light-induced photoreceptor regeneration</h1>.” 2014. Thesis, University of Notre Dame. Accessed January 18, 2021. https://curate.nd.edu/show/jw827942h2c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raycroft, Francis James. “Characterization of the JAK1 / Stat3 pathway during zebrafish retinal development and light-induced photoreceptor regeneration</h1>.” 2014. Web. 18 Jan 2021.

Vancouver:

Raycroft FJ. Characterization of the JAK1 / Stat3 pathway during zebrafish retinal development and light-induced photoreceptor regeneration</h1>. [Internet] [Thesis]. University of Notre Dame; 2014. [cited 2021 Jan 18]. Available from: https://curate.nd.edu/show/jw827942h2c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raycroft FJ. Characterization of the JAK1 / Stat3 pathway during zebrafish retinal development and light-induced photoreceptor regeneration</h1>. [Thesis]. University of Notre Dame; 2014. Available from: https://curate.nd.edu/show/jw827942h2c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

5. Younis, Usir. Inhalational Delivery of a JAK3 Inhibitor for the Novel Treatment of Asthma and the Investigation of Pharmaceutical Salts in HFA Propellant Systems .

Degree: 2018, University of Arizona

 Asthma is a significant lung disease involving chronic inflammation and remodeling of the airways, resulting in reduced quality of life for those who suffer from… (more)

Subjects/Keywords: asthma; HFA; inhalation; JAK-STAT; pMDI; tofacitinib

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APA (6th Edition):

Younis, U. (2018). Inhalational Delivery of a JAK3 Inhibitor for the Novel Treatment of Asthma and the Investigation of Pharmaceutical Salts in HFA Propellant Systems . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/626756

Chicago Manual of Style (16th Edition):

Younis, Usir. “Inhalational Delivery of a JAK3 Inhibitor for the Novel Treatment of Asthma and the Investigation of Pharmaceutical Salts in HFA Propellant Systems .” 2018. Doctoral Dissertation, University of Arizona. Accessed January 18, 2021. http://hdl.handle.net/10150/626756.

MLA Handbook (7th Edition):

Younis, Usir. “Inhalational Delivery of a JAK3 Inhibitor for the Novel Treatment of Asthma and the Investigation of Pharmaceutical Salts in HFA Propellant Systems .” 2018. Web. 18 Jan 2021.

Vancouver:

Younis U. Inhalational Delivery of a JAK3 Inhibitor for the Novel Treatment of Asthma and the Investigation of Pharmaceutical Salts in HFA Propellant Systems . [Internet] [Doctoral dissertation]. University of Arizona; 2018. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10150/626756.

Council of Science Editors:

Younis U. Inhalational Delivery of a JAK3 Inhibitor for the Novel Treatment of Asthma and the Investigation of Pharmaceutical Salts in HFA Propellant Systems . [Doctoral Dissertation]. University of Arizona; 2018. Available from: http://hdl.handle.net/10150/626756

6. SILVA, Juan Luiz Coelho da. Impacto clínico e laboratorial de mutações no gene ASXL1 em pacientes com neoplasias mieloproliferativas .

Degree: 2016, Universidade Federal de Pernambuco

 Algumas evidências destacam mutações no gene ASXL1 como um evento importante na evolução clínica de pacientes com neoplasias hematológicas, particularmente em leucemias mieloides agudas e… (more)

Subjects/Keywords: Neoplasias mieloides; Modificadores epigenéticos; JAK-STAT; Prognóstico; Myeloid neoplasm; Epigenetic modifiers; JAK-STAT; Prognostic

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APA (6th Edition):

SILVA, J. L. C. d. (2016). Impacto clínico e laboratorial de mutações no gene ASXL1 em pacientes com neoplasias mieloproliferativas . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/19535

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SILVA, Juan Luiz Coelho da. “Impacto clínico e laboratorial de mutações no gene ASXL1 em pacientes com neoplasias mieloproliferativas .” 2016. Thesis, Universidade Federal de Pernambuco. Accessed January 18, 2021. http://repositorio.ufpe.br/handle/123456789/19535.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SILVA, Juan Luiz Coelho da. “Impacto clínico e laboratorial de mutações no gene ASXL1 em pacientes com neoplasias mieloproliferativas .” 2016. Web. 18 Jan 2021.

Vancouver:

SILVA JLCd. Impacto clínico e laboratorial de mutações no gene ASXL1 em pacientes com neoplasias mieloproliferativas . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2016. [cited 2021 Jan 18]. Available from: http://repositorio.ufpe.br/handle/123456789/19535.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SILVA JLCd. Impacto clínico e laboratorial de mutações no gene ASXL1 em pacientes com neoplasias mieloproliferativas . [Thesis]. Universidade Federal de Pernambuco; 2016. Available from: http://repositorio.ufpe.br/handle/123456789/19535

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Secardin, Lise. Modélisation des néoplasmes myéloprolifératifs grâce aux cellules souches induites à la pluripotence (IPSC) : Modeling of myeloproliferative neoplasms thanks to an induced pluripotent stem cell model (IPSC).

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie. Hématologie et oncologie, 2016, Sorbonne Paris Cité

Les néoplasmes myéloprolifératifs (NMP) sont hémopathies malignes aboutissant à la surproduction d'une ou plusieurs lignées myéloïdes. Elles sont dues à l'acquisition de mutations sur l'axe… (more)

Subjects/Keywords: Néoplasmes myéloprolifératifs; JAK/STAT; TET2; IPSC; Myeloproliferative neoplasms; JAK/STAT; TET2; IPSC

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APA (6th Edition):

Secardin, L. (2016). Modélisation des néoplasmes myéloprolifératifs grâce aux cellules souches induites à la pluripotence (IPSC) : Modeling of myeloproliferative neoplasms thanks to an induced pluripotent stem cell model (IPSC). (Doctoral Dissertation). Sorbonne Paris Cité. Retrieved from http://www.theses.fr/2016USPCC313

Chicago Manual of Style (16th Edition):

Secardin, Lise. “Modélisation des néoplasmes myéloprolifératifs grâce aux cellules souches induites à la pluripotence (IPSC) : Modeling of myeloproliferative neoplasms thanks to an induced pluripotent stem cell model (IPSC).” 2016. Doctoral Dissertation, Sorbonne Paris Cité. Accessed January 18, 2021. http://www.theses.fr/2016USPCC313.

MLA Handbook (7th Edition):

Secardin, Lise. “Modélisation des néoplasmes myéloprolifératifs grâce aux cellules souches induites à la pluripotence (IPSC) : Modeling of myeloproliferative neoplasms thanks to an induced pluripotent stem cell model (IPSC).” 2016. Web. 18 Jan 2021.

Vancouver:

Secardin L. Modélisation des néoplasmes myéloprolifératifs grâce aux cellules souches induites à la pluripotence (IPSC) : Modeling of myeloproliferative neoplasms thanks to an induced pluripotent stem cell model (IPSC). [Internet] [Doctoral dissertation]. Sorbonne Paris Cité; 2016. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2016USPCC313.

Council of Science Editors:

Secardin L. Modélisation des néoplasmes myéloprolifératifs grâce aux cellules souches induites à la pluripotence (IPSC) : Modeling of myeloproliferative neoplasms thanks to an induced pluripotent stem cell model (IPSC). [Doctoral Dissertation]. Sorbonne Paris Cité; 2016. Available from: http://www.theses.fr/2016USPCC313


Université Paris-Sud – Paris XI

8. Ruez, Richard. Mécanotransduction par les cavéoles : rôle dans l'activation de stat3 par l'interferon alpha : Mechannotransduction by the caveolae : a role in the activation of stat3 by the interferon alpha.

Degree: Docteur es, Biologie, 2011, Université Paris-Sud – Paris XI

Hypothèse : Notre équipe étudie le rôle, mal connu, du trafic membranaire dans le contrôle de l’activation de la voie de signalisation JAK/STAT par les… (more)

Subjects/Keywords: Signalisation; Interféron; Cavéole; Cavéoline; JAK/STAT; Mecanotransduction; Signaling; Interferon; Caveolae; Caveolin; JAK/STAT; Mechanotransduction

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APA (6th Edition):

Ruez, R. (2011). Mécanotransduction par les cavéoles : rôle dans l'activation de stat3 par l'interferon alpha : Mechannotransduction by the caveolae : a role in the activation of stat3 by the interferon alpha. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA112232

Chicago Manual of Style (16th Edition):

Ruez, Richard. “Mécanotransduction par les cavéoles : rôle dans l'activation de stat3 par l'interferon alpha : Mechannotransduction by the caveolae : a role in the activation of stat3 by the interferon alpha.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed January 18, 2021. http://www.theses.fr/2011PA112232.

MLA Handbook (7th Edition):

Ruez, Richard. “Mécanotransduction par les cavéoles : rôle dans l'activation de stat3 par l'interferon alpha : Mechannotransduction by the caveolae : a role in the activation of stat3 by the interferon alpha.” 2011. Web. 18 Jan 2021.

Vancouver:

Ruez R. Mécanotransduction par les cavéoles : rôle dans l'activation de stat3 par l'interferon alpha : Mechannotransduction by the caveolae : a role in the activation of stat3 by the interferon alpha. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2011PA112232.

Council of Science Editors:

Ruez R. Mécanotransduction par les cavéoles : rôle dans l'activation de stat3 par l'interferon alpha : Mechannotransduction by the caveolae : a role in the activation of stat3 by the interferon alpha. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA112232

9. Jark, Paulo César [UNESP]. Estudo da via jak2/stat3 e de seus inibidores em linfomas multicêntricos difusos de grandes células B caninos.

Degree: 2016, Universidade Estadual Paulista

A via Janus Kinase (JAK) e do transdutor de sinal e ativador de transcrição (STAT) desempenham papéis importantes na patogênese de neoplasias hematopoiéticas. A ativação… (more)

Subjects/Keywords: Oncologia; Neoplasias hematopoiéticas; Cães; Terapias alvo; Inibidores de JAK; Oncology; Hematopoietic neoplasia; Dogs; Target therapy; JAK inhibitors

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APA (6th Edition):

Jark, P. C. [. (2016). Estudo da via jak2/stat3 e de seus inibidores em linfomas multicêntricos difusos de grandes células B caninos. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/146685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jark, Paulo César [UNESP]. “Estudo da via jak2/stat3 e de seus inibidores em linfomas multicêntricos difusos de grandes células B caninos.” 2016. Thesis, Universidade Estadual Paulista. Accessed January 18, 2021. http://hdl.handle.net/11449/146685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jark, Paulo César [UNESP]. “Estudo da via jak2/stat3 e de seus inibidores em linfomas multicêntricos difusos de grandes células B caninos.” 2016. Web. 18 Jan 2021.

Vancouver:

Jark PC[. Estudo da via jak2/stat3 e de seus inibidores em linfomas multicêntricos difusos de grandes células B caninos. [Internet] [Thesis]. Universidade Estadual Paulista; 2016. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/11449/146685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jark PC[. Estudo da via jak2/stat3 e de seus inibidores em linfomas multicêntricos difusos de grandes células B caninos. [Thesis]. Universidade Estadual Paulista; 2016. Available from: http://hdl.handle.net/11449/146685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Torres Espinosa, Alba Yurani. Lien entre signalisation JAK/STAT, remodelage cellulaire et extrusion d’un groupe de cellules épithéliales dans l’ovaire de drosophile : Link Between JAK/STAT signaling, cell remodeling and extrusion from the follicular epithelium in the Drosophila ovary.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université Paris-Saclay (ComUE)

Les cellules épithéliales changent en forme et en nombre au cours de divers processus morphogénétiques pendant le développement. La dynamique du réseau d’acto-myosine en interaction… (more)

Subjects/Keywords: Morphogenèse; Extrusion épithéliale; Signalisation JAK/STAT; Acto-Myosine; Apoptose; Morphogenesis; Epithelial cell extrusion; JAK/STAT signaling; Acto-Myosin; Apoptosis

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APA (6th Edition):

Torres Espinosa, A. Y. (2016). Lien entre signalisation JAK/STAT, remodelage cellulaire et extrusion d’un groupe de cellules épithéliales dans l’ovaire de drosophile : Link Between JAK/STAT signaling, cell remodeling and extrusion from the follicular epithelium in the Drosophila ovary. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2016SACLS558

Chicago Manual of Style (16th Edition):

Torres Espinosa, Alba Yurani. “Lien entre signalisation JAK/STAT, remodelage cellulaire et extrusion d’un groupe de cellules épithéliales dans l’ovaire de drosophile : Link Between JAK/STAT signaling, cell remodeling and extrusion from the follicular epithelium in the Drosophila ovary.” 2016. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 18, 2021. http://www.theses.fr/2016SACLS558.

MLA Handbook (7th Edition):

Torres Espinosa, Alba Yurani. “Lien entre signalisation JAK/STAT, remodelage cellulaire et extrusion d’un groupe de cellules épithéliales dans l’ovaire de drosophile : Link Between JAK/STAT signaling, cell remodeling and extrusion from the follicular epithelium in the Drosophila ovary.” 2016. Web. 18 Jan 2021.

Vancouver:

Torres Espinosa AY. Lien entre signalisation JAK/STAT, remodelage cellulaire et extrusion d’un groupe de cellules épithéliales dans l’ovaire de drosophile : Link Between JAK/STAT signaling, cell remodeling and extrusion from the follicular epithelium in the Drosophila ovary. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2016. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2016SACLS558.

Council of Science Editors:

Torres Espinosa AY. Lien entre signalisation JAK/STAT, remodelage cellulaire et extrusion d’un groupe de cellules épithéliales dans l’ovaire de drosophile : Link Between JAK/STAT signaling, cell remodeling and extrusion from the follicular epithelium in the Drosophila ovary. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2016. Available from: http://www.theses.fr/2016SACLS558

11. Bazzi, Wael. Une nouvelle cascade régulant l'hématopoïèse et la réponse inflammatoire chez la drosophile : A novel cascade controlling hematopoiesis and the inflammatory response in flies.

Degree: Docteur es, Génétique, biologie moléculaire et cellulaire, 2017, Université de Strasbourg

Les cellules immunitaires provenant des deux vagues hématopoïétiques jouent des rôles distincts dans la réponse immunitaire, ce qui pose la question d’une potentielle communication entre… (more)

Subjects/Keywords: Gcm; Inflammation; JAK/STAT; Toll; Tumeurs mélanotiques; Drosophile; Gcm; Inflammation; JAK/STAT; Toll; Melanotic tumors; Drosophila; 572.8; 616.99

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APA (6th Edition):

Bazzi, W. (2017). Une nouvelle cascade régulant l'hématopoïèse et la réponse inflammatoire chez la drosophile : A novel cascade controlling hematopoiesis and the inflammatory response in flies. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ043

Chicago Manual of Style (16th Edition):

Bazzi, Wael. “Une nouvelle cascade régulant l'hématopoïèse et la réponse inflammatoire chez la drosophile : A novel cascade controlling hematopoiesis and the inflammatory response in flies.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed January 18, 2021. http://www.theses.fr/2017STRAJ043.

MLA Handbook (7th Edition):

Bazzi, Wael. “Une nouvelle cascade régulant l'hématopoïèse et la réponse inflammatoire chez la drosophile : A novel cascade controlling hematopoiesis and the inflammatory response in flies.” 2017. Web. 18 Jan 2021.

Vancouver:

Bazzi W. Une nouvelle cascade régulant l'hématopoïèse et la réponse inflammatoire chez la drosophile : A novel cascade controlling hematopoiesis and the inflammatory response in flies. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2021 Jan 18]. Available from: http://www.theses.fr/2017STRAJ043.

Council of Science Editors:

Bazzi W. Une nouvelle cascade régulant l'hématopoïèse et la réponse inflammatoire chez la drosophile : A novel cascade controlling hematopoiesis and the inflammatory response in flies. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ043

12. Nascimento, Augusto Santana [UNESP]. Mecanismos moleculares envolvidos com a resistência química de células tumorais de mama.

Degree: 2017, Universidade Estadual Paulista

 Embora algum progresso tenha sido alcançado nos últimos anos, ainda são necessários estudos capazes de desvendar os mecanismos moleculares envolvidos com o fenótipo de resistência… (more)

Subjects/Keywords: Câncer de mama; Resistência; PepChip; PKC; Jak-Stat

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APA (6th Edition):

Nascimento, A. S. [. (2017). Mecanismos moleculares envolvidos com a resistência química de células tumorais de mama. (Thesis). Universidade Estadual Paulista. Retrieved from http://hdl.handle.net/11449/148592

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nascimento, Augusto Santana [UNESP]. “Mecanismos moleculares envolvidos com a resistência química de células tumorais de mama.” 2017. Thesis, Universidade Estadual Paulista. Accessed January 18, 2021. http://hdl.handle.net/11449/148592.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nascimento, Augusto Santana [UNESP]. “Mecanismos moleculares envolvidos com a resistência química de células tumorais de mama.” 2017. Web. 18 Jan 2021.

Vancouver:

Nascimento AS[. Mecanismos moleculares envolvidos com a resistência química de células tumorais de mama. [Internet] [Thesis]. Universidade Estadual Paulista; 2017. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/11449/148592.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nascimento AS[. Mecanismos moleculares envolvidos com a resistência química de células tumorais de mama. [Thesis]. Universidade Estadual Paulista; 2017. Available from: http://hdl.handle.net/11449/148592

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

13. Davis, Colin. Comparative Study of Colon Cancer Subclones Uncovers Potential Roles for AKAP7 and TP53RK in the Antiviral Response.

Degree: 2016, University of Ottawa

 Tumour heterogeneity is a key hurdle for the effective treatment of cancer using oncolytic viruses (OVs). A better understanding of the pathways involved in delineating… (more)

Subjects/Keywords: Oncolytic Virus; Antiviral; CT26; Clone; Heterogeneity; JAK-STAT; Viral Sensitivity

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APA (6th Edition):

Davis, C. (2016). Comparative Study of Colon Cancer Subclones Uncovers Potential Roles for AKAP7 and TP53RK in the Antiviral Response. (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34183

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davis, Colin. “Comparative Study of Colon Cancer Subclones Uncovers Potential Roles for AKAP7 and TP53RK in the Antiviral Response. ” 2016. Thesis, University of Ottawa. Accessed January 18, 2021. http://hdl.handle.net/10393/34183.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davis, Colin. “Comparative Study of Colon Cancer Subclones Uncovers Potential Roles for AKAP7 and TP53RK in the Antiviral Response. ” 2016. Web. 18 Jan 2021.

Vancouver:

Davis C. Comparative Study of Colon Cancer Subclones Uncovers Potential Roles for AKAP7 and TP53RK in the Antiviral Response. [Internet] [Thesis]. University of Ottawa; 2016. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/10393/34183.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davis C. Comparative Study of Colon Cancer Subclones Uncovers Potential Roles for AKAP7 and TP53RK in the Antiviral Response. [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34183

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Tampere University

14. Hammarén, Henrik. Intramolecular regulation of Janus kinases .

Degree: 2017, Tampere University

 Janus-kinaasit (JAKit) ovat ei-reseptorisia tyrosiinikinaaseja, jotka välittävät yli 60 sytokiinin viestejä soluissa. Nämä viestit säätelevät lukuisia biologisia tapahtumia, kuten immuunijärjestelmän toimintaa, hematopoieesia, aineenvaihduntaa sekä kehitystä.… (more)

Subjects/Keywords: kinaasi ; JAK ; pseudokinaasi ; myeloproliferatiivinen neoplasia ; sytokiini ; kinase ; pseudokinase ; myeloproliferative neoplasm ; cytokine

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APA (6th Edition):

Hammarén, H. (2017). Intramolecular regulation of Janus kinases . (Doctoral Dissertation). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/102330

Chicago Manual of Style (16th Edition):

Hammarén, Henrik. “Intramolecular regulation of Janus kinases .” 2017. Doctoral Dissertation, Tampere University. Accessed January 18, 2021. https://trepo.tuni.fi/handle/10024/102330.

MLA Handbook (7th Edition):

Hammarén, Henrik. “Intramolecular regulation of Janus kinases .” 2017. Web. 18 Jan 2021.

Vancouver:

Hammarén H. Intramolecular regulation of Janus kinases . [Internet] [Doctoral dissertation]. Tampere University; 2017. [cited 2021 Jan 18]. Available from: https://trepo.tuni.fi/handle/10024/102330.

Council of Science Editors:

Hammarén H. Intramolecular regulation of Janus kinases . [Doctoral Dissertation]. Tampere University; 2017. Available from: https://trepo.tuni.fi/handle/10024/102330

15. ウベツ, ギザチョー イスモー. High STAT4 Expression is a Better Prognostic Indicatorin Patients with Hepatocellular Carcinoma After Hepatectomy : STAT4高発現は、肝細胞癌切除症例において予後良好因子である.

Degree: 博士(医学), 2016, Tokushima University / 徳島大学

Subjects/Keywords: 肝臓癌; STAT4; JAK

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APA (6th Edition):

ウベツ, . . (2016). High STAT4 Expression is a Better Prognostic Indicatorin Patients with Hepatocellular Carcinoma After Hepatectomy : STAT4高発現は、肝細胞癌切除症例において予後良好因子である. (Thesis). Tokushima University / 徳島大学. Retrieved from http://repo.lib.tokushima-u.ac.jp/109697

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ウベツ, ギザチョー イスモー. “High STAT4 Expression is a Better Prognostic Indicatorin Patients with Hepatocellular Carcinoma After Hepatectomy : STAT4高発現は、肝細胞癌切除症例において予後良好因子である.” 2016. Thesis, Tokushima University / 徳島大学. Accessed January 18, 2021. http://repo.lib.tokushima-u.ac.jp/109697.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ウベツ, ギザチョー イスモー. “High STAT4 Expression is a Better Prognostic Indicatorin Patients with Hepatocellular Carcinoma After Hepatectomy : STAT4高発現は、肝細胞癌切除症例において予後良好因子である.” 2016. Web. 18 Jan 2021.

Vancouver:

ウベツ . High STAT4 Expression is a Better Prognostic Indicatorin Patients with Hepatocellular Carcinoma After Hepatectomy : STAT4高発現は、肝細胞癌切除症例において予後良好因子である. [Internet] [Thesis]. Tokushima University / 徳島大学; 2016. [cited 2021 Jan 18]. Available from: http://repo.lib.tokushima-u.ac.jp/109697.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ウベツ . High STAT4 Expression is a Better Prognostic Indicatorin Patients with Hepatocellular Carcinoma After Hepatectomy : STAT4高発現は、肝細胞癌切除症例において予後良好因子である. [Thesis]. Tokushima University / 徳島大学; 2016. Available from: http://repo.lib.tokushima-u.ac.jp/109697

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

16. Hokenson, Kristen Elizabeth. BDNF signaling in epilepsy: TRKB-induced JAK/STAT pathway and phosphorylation of LSF in neurons.

Degree: PhD, Pharmacology, 2016, Boston University

 Epilepsy is a neurological disorder that causes recurrent and unprovoked seizures due to imbalances in synaptic transmission in distinct regions of the brain. In both… (more)

Subjects/Keywords: Neurosciences; BDNF; Epilepsy; GABA; JAK/STAT; LSF; Neurogenesis

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APA (6th Edition):

Hokenson, K. E. (2016). BDNF signaling in epilepsy: TRKB-induced JAK/STAT pathway and phosphorylation of LSF in neurons. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/16740

Chicago Manual of Style (16th Edition):

Hokenson, Kristen Elizabeth. “BDNF signaling in epilepsy: TRKB-induced JAK/STAT pathway and phosphorylation of LSF in neurons.” 2016. Doctoral Dissertation, Boston University. Accessed January 18, 2021. http://hdl.handle.net/2144/16740.

MLA Handbook (7th Edition):

Hokenson, Kristen Elizabeth. “BDNF signaling in epilepsy: TRKB-induced JAK/STAT pathway and phosphorylation of LSF in neurons.” 2016. Web. 18 Jan 2021.

Vancouver:

Hokenson KE. BDNF signaling in epilepsy: TRKB-induced JAK/STAT pathway and phosphorylation of LSF in neurons. [Internet] [Doctoral dissertation]. Boston University; 2016. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2144/16740.

Council of Science Editors:

Hokenson KE. BDNF signaling in epilepsy: TRKB-induced JAK/STAT pathway and phosphorylation of LSF in neurons. [Doctoral Dissertation]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16740


University of Toronto

17. Gupta, Shaan. MicroRNA-17 Targets JAK1 and STAT3 to Inhibit Osteoblast Differentiation.

Degree: 2014, University of Toronto

MicroRNA-17 (miR-17) was hypothesized to target janus kinase 1 (JAK1) and signal transducer and activator of transcription 3 (STAT3) to inhibit osteogenesis in pre-osteoblast cells.… (more)

Subjects/Keywords: bone formation; JAK-STAT; microRNA; Osteoblast Differentiation; Osteogenesis; osteoporosis; 0307

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APA (6th Edition):

Gupta, S. (2014). MicroRNA-17 Targets JAK1 and STAT3 to Inhibit Osteoblast Differentiation. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/67894

Chicago Manual of Style (16th Edition):

Gupta, Shaan. “MicroRNA-17 Targets JAK1 and STAT3 to Inhibit Osteoblast Differentiation.” 2014. Masters Thesis, University of Toronto. Accessed January 18, 2021. http://hdl.handle.net/1807/67894.

MLA Handbook (7th Edition):

Gupta, Shaan. “MicroRNA-17 Targets JAK1 and STAT3 to Inhibit Osteoblast Differentiation.” 2014. Web. 18 Jan 2021.

Vancouver:

Gupta S. MicroRNA-17 Targets JAK1 and STAT3 to Inhibit Osteoblast Differentiation. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1807/67894.

Council of Science Editors:

Gupta S. MicroRNA-17 Targets JAK1 and STAT3 to Inhibit Osteoblast Differentiation. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/67894

18. Fernandez, David Jaime. Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes.

Degree: PhD, Biology, 2012, The Catholic University of America

Degree awarded: Ph.D. Biology. The Catholic University of America

The molecular basis for alcohol-induced hepatotoxicity is not well-understood. We sought to further explore a known… (more)

Subjects/Keywords: Cellular biology; Acetylation; Alcohol; Hepatocytes; Jak-STAT; Microtubules; Smad

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APA (6th Edition):

Fernandez, D. J. (2012). Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes. (Doctoral Dissertation). The Catholic University of America. Retrieved from http://hdl.handle.net/1961/etd:388

Chicago Manual of Style (16th Edition):

Fernandez, David Jaime. “Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes.” 2012. Doctoral Dissertation, The Catholic University of America. Accessed January 18, 2021. http://hdl.handle.net/1961/etd:388.

MLA Handbook (7th Edition):

Fernandez, David Jaime. “Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes.” 2012. Web. 18 Jan 2021.

Vancouver:

Fernandez DJ. Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes. [Internet] [Doctoral dissertation]. The Catholic University of America; 2012. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1961/etd:388.

Council of Science Editors:

Fernandez DJ. Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes. [Doctoral Dissertation]. The Catholic University of America; 2012. Available from: http://hdl.handle.net/1961/etd:388


University of Debrecen

19. Hamar, Attila. A JAK gátlás lehetőségei az arthritisek kezelésében .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 A JAK-inhibitorok klinikai hatékonyságának és biztonságosságának irodalmi összefoglalója rheumatoid arthritis megbetegedésekben, fókuszpontban a rheumatoid arthritis terápiájában elsőként elfogadott JAK-gátló, a tofacitinib (Xeljanz) készítménnyel. A rheumatoid… (more)

Subjects/Keywords: JAK; rheumatoid arthritis; tofacitinib

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APA (6th Edition):

Hamar, A. (n.d.). A JAK gátlás lehetőségei az arthritisek kezelésében . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/218475

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamar, Attila. “A JAK gátlás lehetőségei az arthritisek kezelésében .” Thesis, University of Debrecen. Accessed January 18, 2021. http://hdl.handle.net/2437/218475.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamar, Attila. “A JAK gátlás lehetőségei az arthritisek kezelésében .” Web. 18 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Hamar A. A JAK gátlás lehetőségei az arthritisek kezelésében . [Internet] [Thesis]. University of Debrecen; [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2437/218475.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Hamar A. A JAK gátlás lehetőségei az arthritisek kezelésében . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/218475

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

20. Fernandez, David Jaime. Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes.

Degree: PhD, Biology, 2012, The Catholic University of America

Degree awarded: Ph.D. Biology. The Catholic University of America

The molecular basis for alcohol-induced hepatotoxicity is not well-understood. We sought to further explore a known… (more)

Subjects/Keywords: Cellular biology; Acetylation; Alcohol; Hepatocytes; Jak-STAT; Microtubules; Smad

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APA (6th Edition):

Fernandez, D. J. (2012). Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes. (Doctoral Dissertation). The Catholic University of America. Retrieved from http://hdl.handle.net/1961/13181

Chicago Manual of Style (16th Edition):

Fernandez, David Jaime. “Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes.” 2012. Doctoral Dissertation, The Catholic University of America. Accessed January 18, 2021. http://hdl.handle.net/1961/13181.

MLA Handbook (7th Edition):

Fernandez, David Jaime. “Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes.” 2012. Web. 18 Jan 2021.

Vancouver:

Fernandez DJ. Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes. [Internet] [Doctoral dissertation]. The Catholic University of America; 2012. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1961/13181.

Council of Science Editors:

Fernandez DJ. Chronic Alcohol Consumption Impairs Clathrin-Mediated Endocytosis and Microtubule-Dependent Nuclear Translocation in Hepatocytes. [Doctoral Dissertation]. The Catholic University of America; 2012. Available from: http://hdl.handle.net/1961/13181


Columbia University

21. Wang, Etienne Cho Ee. Hair Growth Is Induced by Blockade of Macrophage-derived Oncostatin M and Downstream Jak-stat5 Signaling in Hair Follicle Stem Cells.

Degree: 2018, Columbia University

 Our lab recently described a role for JAK-STAT signaling in the maintenance of quiescence during the murine hair cycle. Research into signaling pathways and cytokines/growth… (more)

Subjects/Keywords: Cytology; Immunology; Biomedical engineering; JAK-STAT pathway; Hair – Growth

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APA (6th Edition):

Wang, E. C. E. (2018). Hair Growth Is Induced by Blockade of Macrophage-derived Oncostatin M and Downstream Jak-stat5 Signaling in Hair Follicle Stem Cells. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8SX7RQN

Chicago Manual of Style (16th Edition):

Wang, Etienne Cho Ee. “Hair Growth Is Induced by Blockade of Macrophage-derived Oncostatin M and Downstream Jak-stat5 Signaling in Hair Follicle Stem Cells.” 2018. Doctoral Dissertation, Columbia University. Accessed January 18, 2021. https://doi.org/10.7916/D8SX7RQN.

MLA Handbook (7th Edition):

Wang, Etienne Cho Ee. “Hair Growth Is Induced by Blockade of Macrophage-derived Oncostatin M and Downstream Jak-stat5 Signaling in Hair Follicle Stem Cells.” 2018. Web. 18 Jan 2021.

Vancouver:

Wang ECE. Hair Growth Is Induced by Blockade of Macrophage-derived Oncostatin M and Downstream Jak-stat5 Signaling in Hair Follicle Stem Cells. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2021 Jan 18]. Available from: https://doi.org/10.7916/D8SX7RQN.

Council of Science Editors:

Wang ECE. Hair Growth Is Induced by Blockade of Macrophage-derived Oncostatin M and Downstream Jak-stat5 Signaling in Hair Follicle Stem Cells. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8SX7RQN


University of Manitoba

22. Mahood, Thomas. Differential innate immunity responses to West Nile virus and bacterial infections in mosquitoes.

Degree: Biological Sciences, 2013, University of Manitoba

 Identifying the molecular interactions of pathogens in different mosquito species is critical for understanding how mosquitoes transmit diseases. In this study, the role of the… (more)

Subjects/Keywords: Molecular Biology; Jak-STAT; Aedes aegypti; Culex quinquefasciatus; LPS; Bioinformatics

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APA (6th Edition):

Mahood, T. (2013). Differential innate immunity responses to West Nile virus and bacterial infections in mosquitoes. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/16594

Chicago Manual of Style (16th Edition):

Mahood, Thomas. “Differential innate immunity responses to West Nile virus and bacterial infections in mosquitoes.” 2013. Masters Thesis, University of Manitoba. Accessed January 18, 2021. http://hdl.handle.net/1993/16594.

MLA Handbook (7th Edition):

Mahood, Thomas. “Differential innate immunity responses to West Nile virus and bacterial infections in mosquitoes.” 2013. Web. 18 Jan 2021.

Vancouver:

Mahood T. Differential innate immunity responses to West Nile virus and bacterial infections in mosquitoes. [Internet] [Masters thesis]. University of Manitoba; 2013. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/1993/16594.

Council of Science Editors:

Mahood T. Differential innate immunity responses to West Nile virus and bacterial infections in mosquitoes. [Masters Thesis]. University of Manitoba; 2013. Available from: http://hdl.handle.net/1993/16594


Université Catholique de Louvain

23. Springuel, Lorraine. Loss of MLH1 expression promotes the acquisition of oncogenic JAK1 and JAK3 mutations that cooperatively increase resistance to JAK inhibitors.

Degree: 2015, Université Catholique de Louvain

Malignant transformation evolves as the outcome of 2 connected processes operating within a population of competing cells: on the one hand, the continuous accumulation of… (more)

Subjects/Keywords: JAK; Mismatch repair; MLH1; Mutations; Resistance to TKI

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APA (6th Edition):

Springuel, L. (2015). Loss of MLH1 expression promotes the acquisition of oncogenic JAK1 and JAK3 mutations that cooperatively increase resistance to JAK inhibitors. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/165779

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Springuel, Lorraine. “Loss of MLH1 expression promotes the acquisition of oncogenic JAK1 and JAK3 mutations that cooperatively increase resistance to JAK inhibitors.” 2015. Thesis, Université Catholique de Louvain. Accessed January 18, 2021. http://hdl.handle.net/2078.1/165779.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Springuel, Lorraine. “Loss of MLH1 expression promotes the acquisition of oncogenic JAK1 and JAK3 mutations that cooperatively increase resistance to JAK inhibitors.” 2015. Web. 18 Jan 2021.

Vancouver:

Springuel L. Loss of MLH1 expression promotes the acquisition of oncogenic JAK1 and JAK3 mutations that cooperatively increase resistance to JAK inhibitors. [Internet] [Thesis]. Université Catholique de Louvain; 2015. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2078.1/165779.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Springuel L. Loss of MLH1 expression promotes the acquisition of oncogenic JAK1 and JAK3 mutations that cooperatively increase resistance to JAK inhibitors. [Thesis]. Université Catholique de Louvain; 2015. Available from: http://hdl.handle.net/2078.1/165779

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Dundee

24. Rollings, Christina. The role of JAK1 and JAK3 in CD8⁺ effector T cells.

Degree: PhD, 2016, University of Dundee

 The aim of this project was to explore the role of the tyrosine kinases JAK1 and JAK3 in cytokine signalling, focusing on interleukin-2 signalling in… (more)

Subjects/Keywords: IL-2 signalling; CD8 T cells; jak kinases; tofacitinib; NFIL3

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APA (6th Edition):

Rollings, C. (2016). The role of JAK1 and JAK3 in CD8⁺ effector T cells. (Doctoral Dissertation). University of Dundee. Retrieved from https://discovery.dundee.ac.uk/en/studentTheses/03bc71f9-1291-4e6f-9478-11b9b6227f3f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738560

Chicago Manual of Style (16th Edition):

Rollings, Christina. “The role of JAK1 and JAK3 in CD8⁺ effector T cells.” 2016. Doctoral Dissertation, University of Dundee. Accessed January 18, 2021. https://discovery.dundee.ac.uk/en/studentTheses/03bc71f9-1291-4e6f-9478-11b9b6227f3f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738560.

MLA Handbook (7th Edition):

Rollings, Christina. “The role of JAK1 and JAK3 in CD8⁺ effector T cells.” 2016. Web. 18 Jan 2021.

Vancouver:

Rollings C. The role of JAK1 and JAK3 in CD8⁺ effector T cells. [Internet] [Doctoral dissertation]. University of Dundee; 2016. [cited 2021 Jan 18]. Available from: https://discovery.dundee.ac.uk/en/studentTheses/03bc71f9-1291-4e6f-9478-11b9b6227f3f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738560.

Council of Science Editors:

Rollings C. The role of JAK1 and JAK3 in CD8⁺ effector T cells. [Doctoral Dissertation]. University of Dundee; 2016. Available from: https://discovery.dundee.ac.uk/en/studentTheses/03bc71f9-1291-4e6f-9478-11b9b6227f3f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.738560

25. Dolatabadi, Soheila. The role of fusion oncogenes and cancer stem cells in myxoid liposarcoma.

Degree: 2017, University of Gothenburg / Göteborgs Universitet

 Myxoid liposarcoma (MLS) is characterised by the FUS-DDIT3, or the less common EWSR1-DDIT3 fusion oncogene and is the second most common type of liposarcoma. The… (more)

Subjects/Keywords: FUS; FUS-DDIT3; myxoid liposarcoma; cancer stem cells; JAK-STAT

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APA (6th Edition):

Dolatabadi, S. (2017). The role of fusion oncogenes and cancer stem cells in myxoid liposarcoma. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/52409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dolatabadi, Soheila. “The role of fusion oncogenes and cancer stem cells in myxoid liposarcoma.” 2017. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 18, 2021. http://hdl.handle.net/2077/52409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dolatabadi, Soheila. “The role of fusion oncogenes and cancer stem cells in myxoid liposarcoma.” 2017. Web. 18 Jan 2021.

Vancouver:

Dolatabadi S. The role of fusion oncogenes and cancer stem cells in myxoid liposarcoma. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2017. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2077/52409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dolatabadi S. The role of fusion oncogenes and cancer stem cells in myxoid liposarcoma. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2017. Available from: http://hdl.handle.net/2077/52409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Princeton University

26. Wittes, Julia Sarah. A gene expression screen for JAK/STAT and EGFR target genes during Drosophila melanogaster oogenesis .

Degree: PhD, 2018, Princeton University

 The Janus kinase/Signal transducer and activator of transcription (JAK/STAT) and Epidermal growth factor receptor (EGFR) signaling pathways are conserved regulators of tissue patterning, morphogenesis, and… (more)

Subjects/Keywords: Drosophila melanogaster; EGFR; JAK/STAT; morphogenesis; oogenesis; patterning

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APA (6th Edition):

Wittes, J. S. (2018). A gene expression screen for JAK/STAT and EGFR target genes during Drosophila melanogaster oogenesis . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01w95053238

Chicago Manual of Style (16th Edition):

Wittes, Julia Sarah. “A gene expression screen for JAK/STAT and EGFR target genes during Drosophila melanogaster oogenesis .” 2018. Doctoral Dissertation, Princeton University. Accessed January 18, 2021. http://arks.princeton.edu/ark:/88435/dsp01w95053238.

MLA Handbook (7th Edition):

Wittes, Julia Sarah. “A gene expression screen for JAK/STAT and EGFR target genes during Drosophila melanogaster oogenesis .” 2018. Web. 18 Jan 2021.

Vancouver:

Wittes JS. A gene expression screen for JAK/STAT and EGFR target genes during Drosophila melanogaster oogenesis . [Internet] [Doctoral dissertation]. Princeton University; 2018. [cited 2021 Jan 18]. Available from: http://arks.princeton.edu/ark:/88435/dsp01w95053238.

Council of Science Editors:

Wittes JS. A gene expression screen for JAK/STAT and EGFR target genes during Drosophila melanogaster oogenesis . [Doctoral Dissertation]. Princeton University; 2018. Available from: http://arks.princeton.edu/ark:/88435/dsp01w95053238

27. Amet, Rebecca. Evaluation of the anti-cancer effects of a novel guanidinium-based compound, VP79s, in multiple myeloma.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2020, Trinity College Dublin

 Multiple myeloma (MM) is an incurable haematological malignancy accounting for 300-350 cancer diagnoses in Ireland each year. Mainly a disease of the elderly, MM is… (more)

Subjects/Keywords: Multiple myeloma; Cancer; Drug discovery; JAK/STAT3; Cell signalling

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APA (6th Edition):

Amet, R. (2020). Evaluation of the anti-cancer effects of a novel guanidinium-based compound, VP79s, in multiple myeloma. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/93627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Amet, Rebecca. “Evaluation of the anti-cancer effects of a novel guanidinium-based compound, VP79s, in multiple myeloma.” 2020. Thesis, Trinity College Dublin. Accessed January 18, 2021. http://hdl.handle.net/2262/93627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Amet, Rebecca. “Evaluation of the anti-cancer effects of a novel guanidinium-based compound, VP79s, in multiple myeloma.” 2020. Web. 18 Jan 2021.

Vancouver:

Amet R. Evaluation of the anti-cancer effects of a novel guanidinium-based compound, VP79s, in multiple myeloma. [Internet] [Thesis]. Trinity College Dublin; 2020. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/2262/93627.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Amet R. Evaluation of the anti-cancer effects of a novel guanidinium-based compound, VP79s, in multiple myeloma. [Thesis]. Trinity College Dublin; 2020. Available from: http://hdl.handle.net/2262/93627

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

28. Varghese, Leila Mattea Noozhumurry. Molecular mechanisms of Janus kinase signalling in blood cell development and disease.

Degree: 2014, University of Melbourne

 The haematopoietic and immune systems are largely maintained by cytokines, a family of circulating secreted proteins. The binding of cytokines to transmembrane receptors at the… (more)

Subjects/Keywords: JAK-STAT signalling; myeloproliferative neoplasms; thrombopoietin; MPL; SOCS3; JAK2 V617F

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Varghese, L. M. N. (2014). Molecular mechanisms of Janus kinase signalling in blood cell development and disease. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/41768

Chicago Manual of Style (16th Edition):

Varghese, Leila Mattea Noozhumurry. “Molecular mechanisms of Janus kinase signalling in blood cell development and disease.” 2014. Doctoral Dissertation, University of Melbourne. Accessed January 18, 2021. http://hdl.handle.net/11343/41768.

MLA Handbook (7th Edition):

Varghese, Leila Mattea Noozhumurry. “Molecular mechanisms of Janus kinase signalling in blood cell development and disease.” 2014. Web. 18 Jan 2021.

Vancouver:

Varghese LMN. Molecular mechanisms of Janus kinase signalling in blood cell development and disease. [Internet] [Doctoral dissertation]. University of Melbourne; 2014. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/11343/41768.

Council of Science Editors:

Varghese LMN. Molecular mechanisms of Janus kinase signalling in blood cell development and disease. [Doctoral Dissertation]. University of Melbourne; 2014. Available from: http://hdl.handle.net/11343/41768


University of Melbourne

29. Scott, Nicholas. Mechanisms and inhibition of CD4+ T cell migration in pre-clinical and humanised mouse models of type 1 diabetes.

Degree: 2017, University of Melbourne

 Type 1 diabetes (T1D) is an autoimmune disease that develops when the insulin-secreting beta cells in the pancreas are destroyed. This destruction is primarily mediated… (more)

Subjects/Keywords: Type 1 diabetes; autoimmunity; NOD; JAK inhibitors; humanised mice

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Scott, N. (2017). Mechanisms and inhibition of CD4+ T cell migration in pre-clinical and humanised mouse models of type 1 diabetes. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/194111

Chicago Manual of Style (16th Edition):

Scott, Nicholas. “Mechanisms and inhibition of CD4+ T cell migration in pre-clinical and humanised mouse models of type 1 diabetes.” 2017. Doctoral Dissertation, University of Melbourne. Accessed January 18, 2021. http://hdl.handle.net/11343/194111.

MLA Handbook (7th Edition):

Scott, Nicholas. “Mechanisms and inhibition of CD4+ T cell migration in pre-clinical and humanised mouse models of type 1 diabetes.” 2017. Web. 18 Jan 2021.

Vancouver:

Scott N. Mechanisms and inhibition of CD4+ T cell migration in pre-clinical and humanised mouse models of type 1 diabetes. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/11343/194111.

Council of Science Editors:

Scott N. Mechanisms and inhibition of CD4+ T cell migration in pre-clinical and humanised mouse models of type 1 diabetes. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/194111


University of Melbourne

30. Liau, Nicholas. Structural and biochemical characterisation of the regulation of Janus Kinase signalling.

Degree: 2017, University of Melbourne

 The cytokines are a group of extracellular signalling proteins which stimulate a large variety of cellular processes, including the proliferation and differentiation of blood cells,… (more)

Subjects/Keywords: Janus Kinase; JAK; Suppressor of Cytokine Signalling 1; SOCS1; structural biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liau, N. (2017). Structural and biochemical characterisation of the regulation of Janus Kinase signalling. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/197686

Chicago Manual of Style (16th Edition):

Liau, Nicholas. “Structural and biochemical characterisation of the regulation of Janus Kinase signalling.” 2017. Doctoral Dissertation, University of Melbourne. Accessed January 18, 2021. http://hdl.handle.net/11343/197686.

MLA Handbook (7th Edition):

Liau, Nicholas. “Structural and biochemical characterisation of the regulation of Janus Kinase signalling.” 2017. Web. 18 Jan 2021.

Vancouver:

Liau N. Structural and biochemical characterisation of the regulation of Janus Kinase signalling. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2021 Jan 18]. Available from: http://hdl.handle.net/11343/197686.

Council of Science Editors:

Liau N. Structural and biochemical characterisation of the regulation of Janus Kinase signalling. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/197686

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