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You searched for subject:(interleukin 23). Showing records 1 – 30 of 34 total matches.

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Queens University

1. Che Mat, Nor Fazila. Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection .

Degree: Microbiology and Immunology, 2011, Queens University

 Interluekin-23 (IL-23) is a pro-inflammatory cytokine critical to the regulation of innate and adaptive immune responses. The main role for this cytokine is in the… (more)

Subjects/Keywords: Virus ; Interleukin-23 (IL-23)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Che Mat, N. F. (2011). Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6666

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Che Mat, Nor Fazila. “Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection .” 2011. Thesis, Queens University. Accessed May 10, 2021. http://hdl.handle.net/1974/6666.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Che Mat, Nor Fazila. “Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection .” 2011. Web. 10 May 2021.

Vancouver:

Che Mat NF. Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection . [Internet] [Thesis]. Queens University; 2011. [cited 2021 May 10]. Available from: http://hdl.handle.net/1974/6666.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Che Mat NF. Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection . [Thesis]. Queens University; 2011. Available from: http://hdl.handle.net/1974/6666

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Bechara, Rami. Étude de la coopération entre les cellules dendritiques et les lymphocytes T dans les allergies aux produits chimiques et aux médicaments : Study of dendritic cells and T-lymphocytes cooperation in drug and chemical allergy.

Degree: Docteur es, Immunologie, 2017, Université Paris-Saclay (ComUE); Université Saint-Joseph (Beyrouth)

Les allergies aux produits chimiques et aux médicaments constituent un problème majeur de santé publique. L’objectif de ce travail est de mieux comprendre l’interaction entre… (more)

Subjects/Keywords: Cellules dendritiques; Allergie; Lymphocytes T; Interleukin-23; Interleukine-12; Dendritic cells; Allergy; T lymphocytes; Interleukin-23; Interleukin-12

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APA (6th Edition):

Bechara, R. (2017). Étude de la coopération entre les cellules dendritiques et les lymphocytes T dans les allergies aux produits chimiques et aux médicaments : Study of dendritic cells and T-lymphocytes cooperation in drug and chemical allergy. (Doctoral Dissertation). Université Paris-Saclay (ComUE); Université Saint-Joseph (Beyrouth). Retrieved from http://www.theses.fr/2017SACLS474

Chicago Manual of Style (16th Edition):

Bechara, Rami. “Étude de la coopération entre les cellules dendritiques et les lymphocytes T dans les allergies aux produits chimiques et aux médicaments : Study of dendritic cells and T-lymphocytes cooperation in drug and chemical allergy.” 2017. Doctoral Dissertation, Université Paris-Saclay (ComUE); Université Saint-Joseph (Beyrouth). Accessed May 10, 2021. http://www.theses.fr/2017SACLS474.

MLA Handbook (7th Edition):

Bechara, Rami. “Étude de la coopération entre les cellules dendritiques et les lymphocytes T dans les allergies aux produits chimiques et aux médicaments : Study of dendritic cells and T-lymphocytes cooperation in drug and chemical allergy.” 2017. Web. 10 May 2021.

Vancouver:

Bechara R. Étude de la coopération entre les cellules dendritiques et les lymphocytes T dans les allergies aux produits chimiques et aux médicaments : Study of dendritic cells and T-lymphocytes cooperation in drug and chemical allergy. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); Université Saint-Joseph (Beyrouth); 2017. [cited 2021 May 10]. Available from: http://www.theses.fr/2017SACLS474.

Council of Science Editors:

Bechara R. Étude de la coopération entre les cellules dendritiques et les lymphocytes T dans les allergies aux produits chimiques et aux médicaments : Study of dendritic cells and T-lymphocytes cooperation in drug and chemical allergy. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); Université Saint-Joseph (Beyrouth); 2017. Available from: http://www.theses.fr/2017SACLS474


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

3. Delli, Florentina - Silvia. Ο ρόλος των ιντερλευκινών IL16, IL17 και IL23 στην παθογένεια του πομφολυγώδους πεμφιγοειδούς.

Degree: 2020, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Bullous pemphigoid (BP) patients are predominantly above 70 years of age, with limited tolerance to the side effects of the immunosuppressive drugs. Advancements in our… (more)

Subjects/Keywords: Πομφολυγώδες πεμφιγοειδές; IgE; Ιντερλευκίνη 16; Ιντερλευκίνη 17; Ιντερλευκίνη 23; Bullous pemphigoid; IGE; Interleukin 16; Interleukin 17; Interleukin 23

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APA (6th Edition):

Delli, F. -. S. (2020). Ο ρόλος των ιντερλευκινών IL16, IL17 και IL23 στην παθογένεια του πομφολυγώδους πεμφιγοειδούς. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/48559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Delli, Florentina - Silvia. “Ο ρόλος των ιντερλευκινών IL16, IL17 και IL23 στην παθογένεια του πομφολυγώδους πεμφιγοειδούς.” 2020. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed May 10, 2021. http://hdl.handle.net/10442/hedi/48559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Delli, Florentina - Silvia. “Ο ρόλος των ιντερλευκινών IL16, IL17 και IL23 στην παθογένεια του πομφολυγώδους πεμφιγοειδούς.” 2020. Web. 10 May 2021.

Vancouver:

Delli F-S. Ο ρόλος των ιντερλευκινών IL16, IL17 και IL23 στην παθογένεια του πομφολυγώδους πεμφιγοειδούς. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2020. [cited 2021 May 10]. Available from: http://hdl.handle.net/10442/hedi/48559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Delli F-S. Ο ρόλος των ιντερλευκινών IL16, IL17 και IL23 στην παθογένεια του πομφολυγώδους πεμφιγοειδούς. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2020. Available from: http://hdl.handle.net/10442/hedi/48559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Γιαννικόπουλος, Γεώργιος. Μελέτη των μεταβολών της ανοσιακής απάντησης του ασθενούς κατά τη μετάβαση από σήψη σε σοβαρή σήψη και σηπτική καταπληξία: η σημασία των NK-κυττάρων.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Although much information is available for the function of circulating monocytes when signs of sepsis are apparent, little is known on how NK cells respond… (more)

Subjects/Keywords: Σήψη; NK-κύτταρα; Ιντερφερόνη-γ; Ιντερλευκίνη-23; Sepsis; Natural killer cells; Interferon-γ; Interleukin-23

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APA (6th Edition):

Γιαννικόπουλος, . . (2013). Μελέτη των μεταβολών της ανοσιακής απάντησης του ασθενούς κατά τη μετάβαση από σήψη σε σοβαρή σήψη και σηπτική καταπληξία: η σημασία των NK-κυττάρων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/29474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Γιαννικόπουλος, Γεώργιος. “Μελέτη των μεταβολών της ανοσιακής απάντησης του ασθενούς κατά τη μετάβαση από σήψη σε σοβαρή σήψη και σηπτική καταπληξία: η σημασία των NK-κυττάρων.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed May 10, 2021. http://hdl.handle.net/10442/hedi/29474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Γιαννικόπουλος, Γεώργιος. “Μελέτη των μεταβολών της ανοσιακής απάντησης του ασθενούς κατά τη μετάβαση από σήψη σε σοβαρή σήψη και σηπτική καταπληξία: η σημασία των NK-κυττάρων.” 2013. Web. 10 May 2021.

Vancouver:

Γιαννικόπουλος . Μελέτη των μεταβολών της ανοσιακής απάντησης του ασθενούς κατά τη μετάβαση από σήψη σε σοβαρή σήψη και σηπτική καταπληξία: η σημασία των NK-κυττάρων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2021 May 10]. Available from: http://hdl.handle.net/10442/hedi/29474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Γιαννικόπουλος . Μελέτη των μεταβολών της ανοσιακής απάντησης του ασθενούς κατά τη μετάβαση από σήψη σε σοβαρή σήψη και σηπτική καταπληξία: η σημασία των NK-κυττάρων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/29474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Norte

5. Monteiro, Barbara Vanessa de Brito. Análise da resposta Th17 em líquen plano oral .

Degree: 2012, Universidade do Rio Grande do Norte

 Th17 cells have been strongly associated with the pathogenesis of several autoimmune and inflammatory diseases. IL-17 and IL-23 are important cytokines associated with this lineage.… (more)

Subjects/Keywords: Líquen plano bucal; Imunoistoquímica; Células Th17; Interleucina-17; Interleucina-23; Lichen planus; Immunohistochemistry; Th17 cells; Interleukin-17; Interleukin-23

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APA (6th Edition):

Monteiro, B. V. d. B. (2012). Análise da resposta Th17 em líquen plano oral . (Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/17122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Monteiro, Barbara Vanessa de Brito. “Análise da resposta Th17 em líquen plano oral .” 2012. Thesis, Universidade do Rio Grande do Norte. Accessed May 10, 2021. http://repositorio.ufrn.br/handle/123456789/17122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Monteiro, Barbara Vanessa de Brito. “Análise da resposta Th17 em líquen plano oral .” 2012. Web. 10 May 2021.

Vancouver:

Monteiro BVdB. Análise da resposta Th17 em líquen plano oral . [Internet] [Thesis]. Universidade do Rio Grande do Norte; 2012. [cited 2021 May 10]. Available from: http://repositorio.ufrn.br/handle/123456789/17122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Monteiro BVdB. Análise da resposta Th17 em líquen plano oral . [Thesis]. Universidade do Rio Grande do Norte; 2012. Available from: http://repositorio.ufrn.br/handle/123456789/17122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

6. Semple, Catlin. Health research with Manitoba First Nations. An investigation of gene variants affecting the Th17 immune pathway and the P2RX7 receptor.

Degree: Medical Microbiology, 2016, University of Manitoba

 Introduction: Canadian First Nations experience a significantly higher rate of Mycobacterium tuberculosis (MTB) infection than non-Indigenous Canadians. Th17 cells are a subset of CD4+ T… (more)

Subjects/Keywords: First Nations; Indigenous; Interleukin-17; Genetics; P2RX7; IFN-gamma receptor; Interleukin-23 receptor; Immunogenetics; Tuberculosis; Knowledge translation; Interleukin-17 receptor

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APA (6th Edition):

Semple, C. (2016). Health research with Manitoba First Nations. An investigation of gene variants affecting the Th17 immune pathway and the P2RX7 receptor. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31848

Chicago Manual of Style (16th Edition):

Semple, Catlin. “Health research with Manitoba First Nations. An investigation of gene variants affecting the Th17 immune pathway and the P2RX7 receptor.” 2016. Masters Thesis, University of Manitoba. Accessed May 10, 2021. http://hdl.handle.net/1993/31848.

MLA Handbook (7th Edition):

Semple, Catlin. “Health research with Manitoba First Nations. An investigation of gene variants affecting the Th17 immune pathway and the P2RX7 receptor.” 2016. Web. 10 May 2021.

Vancouver:

Semple C. Health research with Manitoba First Nations. An investigation of gene variants affecting the Th17 immune pathway and the P2RX7 receptor. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2021 May 10]. Available from: http://hdl.handle.net/1993/31848.

Council of Science Editors:

Semple C. Health research with Manitoba First Nations. An investigation of gene variants affecting the Th17 immune pathway and the P2RX7 receptor. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31848


University of Oxford

7. Mathisen, Stephanie Jane. Mononuclear phagocytes in intestinal homeostasis and inflammation.

Degree: PhD, 2015, University of Oxford

 Changes to the composition and function of the gut mononuclear phagocyte (MNP) compartment are associated with the development of intestinal inflammation. Much work has focused… (more)

Subjects/Keywords: 616.07; Mucosal Immunology; Mononuclear Phagocytes; Immunology; Macrophages; Monocytes; Inflammatory Bowel Disease; Colitis; Interleukin-23 (IL-23); Intestinal Immunology; Intestinal Inflammation

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APA (6th Edition):

Mathisen, S. J. (2015). Mononuclear phagocytes in intestinal homeostasis and inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from https://ora.ox.ac.uk/objects/uuid:dfa5b8b5-668f-45f8-8a3b-bf18a7b0703a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711750

Chicago Manual of Style (16th Edition):

Mathisen, Stephanie Jane. “Mononuclear phagocytes in intestinal homeostasis and inflammation.” 2015. Doctoral Dissertation, University of Oxford. Accessed May 10, 2021. https://ora.ox.ac.uk/objects/uuid:dfa5b8b5-668f-45f8-8a3b-bf18a7b0703a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711750.

MLA Handbook (7th Edition):

Mathisen, Stephanie Jane. “Mononuclear phagocytes in intestinal homeostasis and inflammation.” 2015. Web. 10 May 2021.

Vancouver:

Mathisen SJ. Mononuclear phagocytes in intestinal homeostasis and inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2015. [cited 2021 May 10]. Available from: https://ora.ox.ac.uk/objects/uuid:dfa5b8b5-668f-45f8-8a3b-bf18a7b0703a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711750.

Council of Science Editors:

Mathisen SJ. Mononuclear phagocytes in intestinal homeostasis and inflammation. [Doctoral Dissertation]. University of Oxford; 2015. Available from: https://ora.ox.ac.uk/objects/uuid:dfa5b8b5-668f-45f8-8a3b-bf18a7b0703a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711750

8. Vinicius Silva Costa. Associação do gene IL23A com a proteção ao diabetes mellitus tipo 1 autoimune.

Degree: 2012, University of São Paulo

 Introdução: Diabetes tipo 1A(DM1A) é uma doença causada pela destruição autoimune das células beta. Em adição aos linfócitos T helper 1(Th1) e Th2, um subtipo… (more)

Subjects/Keywords: Diabetes mellitus tipo 1; Interleucina-23; Sistema imunológico; Immunologic system; Interleukin-23; Type 1 diabetes mellitus

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APA (6th Edition):

Costa, V. S. (2012). Associação do gene IL23A com a proteção ao diabetes mellitus tipo 1 autoimune. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26102012-163557/

Chicago Manual of Style (16th Edition):

Costa, Vinicius Silva. “Associação do gene IL23A com a proteção ao diabetes mellitus tipo 1 autoimune.” 2012. Doctoral Dissertation, University of São Paulo. Accessed May 10, 2021. http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26102012-163557/.

MLA Handbook (7th Edition):

Costa, Vinicius Silva. “Associação do gene IL23A com a proteção ao diabetes mellitus tipo 1 autoimune.” 2012. Web. 10 May 2021.

Vancouver:

Costa VS. Associação do gene IL23A com a proteção ao diabetes mellitus tipo 1 autoimune. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2021 May 10]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26102012-163557/.

Council of Science Editors:

Costa VS. Associação do gene IL23A com a proteção ao diabetes mellitus tipo 1 autoimune. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5135/tde-26102012-163557/

9. Guerra, Evelyn V. Santos. Eosinophils as Drivers of the IL-23/IL-17 Axis: Implications for Acute Aspergillosis and Allergic Asthma: A Dissertation.

Degree: Immunology and Microbiology, Medicine, 2016, U of Massachusetts : Med

  Aspergillus fumigatus is an opportunistic fungal pathogen that causes lethal invasive pulmonary disease in immunocompromised hosts and allergic asthma in sensitized individuals. This dissertation… (more)

Subjects/Keywords: Aspergillus fumigatus; Asthma; Eosinophils; Interleukin-17; Interleukin-23; Pulmonary Aspergillosis; Immunology of Infectious Disease; Immunopathology; Pathogenic Microbiology; Respiratory Tract Diseases

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APA (6th Edition):

Guerra, E. V. S. (2016). Eosinophils as Drivers of the IL-23/IL-17 Axis: Implications for Acute Aspergillosis and Allergic Asthma: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/831

Chicago Manual of Style (16th Edition):

Guerra, Evelyn V Santos. “Eosinophils as Drivers of the IL-23/IL-17 Axis: Implications for Acute Aspergillosis and Allergic Asthma: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed May 10, 2021. https://escholarship.umassmed.edu/gsbs_diss/831.

MLA Handbook (7th Edition):

Guerra, Evelyn V Santos. “Eosinophils as Drivers of the IL-23/IL-17 Axis: Implications for Acute Aspergillosis and Allergic Asthma: A Dissertation.” 2016. Web. 10 May 2021.

Vancouver:

Guerra EVS. Eosinophils as Drivers of the IL-23/IL-17 Axis: Implications for Acute Aspergillosis and Allergic Asthma: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2021 May 10]. Available from: https://escholarship.umassmed.edu/gsbs_diss/831.

Council of Science Editors:

Guerra EVS. Eosinophils as Drivers of the IL-23/IL-17 Axis: Implications for Acute Aspergillosis and Allergic Asthma: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: https://escholarship.umassmed.edu/gsbs_diss/831

10. Conway, Richard. Giant cell arteritis: diagnostic tools, treatment targets, and pathogenic pathways.

Degree: 2017, University College Dublin. School of Medicine  

 Giant cell arteritis (GCA) is the most common form of systemic vasculitis. Up to 25% of patients develop cranial ischaemic complications (CIC). The diagnosis of… (more)

Subjects/Keywords: Giant cell arteritis; Interleukin 12; Interleukin 23; Ultrasound; Ustekinumab; Vasculitis; 0|aGiant cell arteritis; #0|aInterleukins; #0|aUstekinumab

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APA (6th Edition):

Conway, R. (2017). Giant cell arteritis: diagnostic tools, treatment targets, and pathogenic pathways. (Thesis). University College Dublin. School of Medicine  . Retrieved from http://hdl.handle.net/10197/9091

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Conway, Richard. “Giant cell arteritis: diagnostic tools, treatment targets, and pathogenic pathways.” 2017. Thesis, University College Dublin. School of Medicine  . Accessed May 10, 2021. http://hdl.handle.net/10197/9091.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Conway, Richard. “Giant cell arteritis: diagnostic tools, treatment targets, and pathogenic pathways.” 2017. Web. 10 May 2021.

Vancouver:

Conway R. Giant cell arteritis: diagnostic tools, treatment targets, and pathogenic pathways. [Internet] [Thesis]. University College Dublin. School of Medicine  ; 2017. [cited 2021 May 10]. Available from: http://hdl.handle.net/10197/9091.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Conway R. Giant cell arteritis: diagnostic tools, treatment targets, and pathogenic pathways. [Thesis]. University College Dublin. School of Medicine  ; 2017. Available from: http://hdl.handle.net/10197/9091

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. LOW PEY YNG. Regulation of interleukin-12 and interleukin-23 production by tristetraprolin (TTP).

Degree: 2012, National University of Singapore

Subjects/Keywords: Interleukin-12; Interleukin-23; Tristetraprolin; mRNA stability; TH17; TH1

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APA (6th Edition):

YNG, L. P. (2012). Regulation of interleukin-12 and interleukin-23 production by tristetraprolin (TTP). (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/35560

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

YNG, LOW PEY. “Regulation of interleukin-12 and interleukin-23 production by tristetraprolin (TTP).” 2012. Thesis, National University of Singapore. Accessed May 10, 2021. http://scholarbank.nus.edu.sg/handle/10635/35560.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

YNG, LOW PEY. “Regulation of interleukin-12 and interleukin-23 production by tristetraprolin (TTP).” 2012. Web. 10 May 2021.

Vancouver:

YNG LP. Regulation of interleukin-12 and interleukin-23 production by tristetraprolin (TTP). [Internet] [Thesis]. National University of Singapore; 2012. [cited 2021 May 10]. Available from: http://scholarbank.nus.edu.sg/handle/10635/35560.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

YNG LP. Regulation of interleukin-12 and interleukin-23 production by tristetraprolin (TTP). [Thesis]. National University of Singapore; 2012. Available from: http://scholarbank.nus.edu.sg/handle/10635/35560

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Truchetet, Marie-Élise. Etude de l’implication de l’axe IL-23/Th17 dans deux modèles physiopathologiques humains : la réponse à Mycoplasma hominis et la sclérodermie systémique : Study of the IL-23/Th17 axis involvement in two physiopathological human models : response to Mycoplasma hominis and systemic sclerosis.

Degree: Docteur es, Sciences, technologie, santé. Microbiologie-Immunologie, 2012, Université de Bordeaux Segalen

La nature du lien qui unit les deux aspects du système immunitaire, que sont la défense de l’hôte contre les agressions extérieures et la genèse… (more)

Subjects/Keywords: Interleukine-17; Interleukine-23; Lymphocytes T helper 17; Mycoplasma hominis; Sclérodermie systémique; Interleukin-17; Interleukin-23; T helper 17 lymphocytes; Mycoplasma hominis; Systemic sclerosis

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APA (6th Edition):

Truchetet, M. (2012). Etude de l’implication de l’axe IL-23/Th17 dans deux modèles physiopathologiques humains : la réponse à Mycoplasma hominis et la sclérodermie systémique : Study of the IL-23/Th17 axis involvement in two physiopathological human models : response to Mycoplasma hominis and systemic sclerosis. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2012BOR21943

Chicago Manual of Style (16th Edition):

Truchetet, Marie-Élise. “Etude de l’implication de l’axe IL-23/Th17 dans deux modèles physiopathologiques humains : la réponse à Mycoplasma hominis et la sclérodermie systémique : Study of the IL-23/Th17 axis involvement in two physiopathological human models : response to Mycoplasma hominis and systemic sclerosis.” 2012. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed May 10, 2021. http://www.theses.fr/2012BOR21943.

MLA Handbook (7th Edition):

Truchetet, Marie-Élise. “Etude de l’implication de l’axe IL-23/Th17 dans deux modèles physiopathologiques humains : la réponse à Mycoplasma hominis et la sclérodermie systémique : Study of the IL-23/Th17 axis involvement in two physiopathological human models : response to Mycoplasma hominis and systemic sclerosis.” 2012. Web. 10 May 2021.

Vancouver:

Truchetet M. Etude de l’implication de l’axe IL-23/Th17 dans deux modèles physiopathologiques humains : la réponse à Mycoplasma hominis et la sclérodermie systémique : Study of the IL-23/Th17 axis involvement in two physiopathological human models : response to Mycoplasma hominis and systemic sclerosis. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2012. [cited 2021 May 10]. Available from: http://www.theses.fr/2012BOR21943.

Council of Science Editors:

Truchetet M. Etude de l’implication de l’axe IL-23/Th17 dans deux modèles physiopathologiques humains : la réponse à Mycoplasma hominis et la sclérodermie systémique : Study of the IL-23/Th17 axis involvement in two physiopathological human models : response to Mycoplasma hominis and systemic sclerosis. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2012. Available from: http://www.theses.fr/2012BOR21943

13. Joyce Prieto Bezerra de Menezes. Expressão de Foxp3, IL-17 e IL-23 na Leishmaniose Tegumentar Americana causada por Leishmania (Leishmania) amazonensis e Leishmania (Viannia) braziliensis.

Degree: 2013, University of São Paulo

A leishmaniose tegumentar americana (LTA) apresenta um amplo espectro de manifestações clínicas e imunopatológicas resultante da interação entre as diferentes espécies de Leishmania e os… (more)

Subjects/Keywords: Foxp3; Interleucina-17; Interleucina-23; L amazonensis; Leishmania braziliensis; Leishmaniose cutânea; Cutaneous leishmaniasis; Foxp3; Interleukin-17; Interleukin-23; Leishmania amazonensis; Leishmania braziliensis

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APA (6th Edition):

Menezes, J. P. B. d. (2013). Expressão de Foxp3, IL-17 e IL-23 na Leishmaniose Tegumentar Americana causada por Leishmania (Leishmania) amazonensis e Leishmania (Viannia) braziliensis. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5160/tde-25112013-105911/

Chicago Manual of Style (16th Edition):

Menezes, Joyce Prieto Bezerra de. “Expressão de Foxp3, IL-17 e IL-23 na Leishmaniose Tegumentar Americana causada por Leishmania (Leishmania) amazonensis e Leishmania (Viannia) braziliensis.” 2013. Masters Thesis, University of São Paulo. Accessed May 10, 2021. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-25112013-105911/.

MLA Handbook (7th Edition):

Menezes, Joyce Prieto Bezerra de. “Expressão de Foxp3, IL-17 e IL-23 na Leishmaniose Tegumentar Americana causada por Leishmania (Leishmania) amazonensis e Leishmania (Viannia) braziliensis.” 2013. Web. 10 May 2021.

Vancouver:

Menezes JPBd. Expressão de Foxp3, IL-17 e IL-23 na Leishmaniose Tegumentar Americana causada por Leishmania (Leishmania) amazonensis e Leishmania (Viannia) braziliensis. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 May 10]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-25112013-105911/.

Council of Science Editors:

Menezes JPBd. Expressão de Foxp3, IL-17 e IL-23 na Leishmaniose Tegumentar Americana causada por Leishmania (Leishmania) amazonensis e Leishmania (Viannia) braziliensis. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/5/5160/tde-25112013-105911/

14. Monteiro, Barbara Vanessa de Brito. Análise da resposta Th17 em líquen plano oral.

Degree: 2012, Federal University of Rio Grande do Norte

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Th17 cells have been strongly associated with the pathogenesis of several autoimmune and inflammatory diseases. IL-17 and IL-23(more)

Subjects/Keywords: Líquen plano bucal; Imunoistoquímica; Células Th17; Interleucina-17; Interleucina-23; Lichen planus; Immunohistochemistry; Th17 cells; Interleukin-17; Interleukin-23; CNPQ::CIENCIAS DA SAUDE::ODONTOLOGIA

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APA (6th Edition):

Monteiro, B. V. d. B. (2012). Análise da resposta Th17 em líquen plano oral. (Masters Thesis). Federal University of Rio Grande do Norte. Retrieved from https://repositorio.ufrn.br/jspui/handle/123456789/17122

Chicago Manual of Style (16th Edition):

Monteiro, Barbara Vanessa de Brito. “Análise da resposta Th17 em líquen plano oral.” 2012. Masters Thesis, Federal University of Rio Grande do Norte. Accessed May 10, 2021. https://repositorio.ufrn.br/jspui/handle/123456789/17122.

MLA Handbook (7th Edition):

Monteiro, Barbara Vanessa de Brito. “Análise da resposta Th17 em líquen plano oral.” 2012. Web. 10 May 2021.

Vancouver:

Monteiro BVdB. Análise da resposta Th17 em líquen plano oral. [Internet] [Masters thesis]. Federal University of Rio Grande do Norte; 2012. [cited 2021 May 10]. Available from: https://repositorio.ufrn.br/jspui/handle/123456789/17122.

Council of Science Editors:

Monteiro BVdB. Análise da resposta Th17 em líquen plano oral. [Masters Thesis]. Federal University of Rio Grande do Norte; 2012. Available from: https://repositorio.ufrn.br/jspui/handle/123456789/17122


Univerzitet u Beogradu

15. Barjaktarević, Igor Ž. Uloga Interleukina-23 u prevremenom razvoju emfizema pluća među HIV-1 + pušačima.

Degree: Medicinski fakultet, 2017, Univerzitet u Beogradu

Pulmologija, Interna Medicina - Imunologija / Pulmonology - Immunology

Genska ekspresija matriks metaloproteinaze-9 (MMP-9) je pojačana u alveolarnim makrofagima (AM) HIV-1 pozitivnih pušača obolelih od… (more)

Subjects/Keywords: HIV; alveolar macrophage; Interleukin IL-23; matrix metalloproteinase MMP-9; emphysema; T lymphocytes; Th-17

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APA (6th Edition):

Barjaktarević, I. . (2017). Uloga Interleukina-23 u prevremenom razvoju emfizema pluća među HIV-1 + pušačima. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:14289/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barjaktarević, Igor Ž. “Uloga Interleukina-23 u prevremenom razvoju emfizema pluća među HIV-1 + pušačima.” 2017. Thesis, Univerzitet u Beogradu. Accessed May 10, 2021. https://fedorabg.bg.ac.rs/fedora/get/o:14289/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barjaktarević, Igor Ž. “Uloga Interleukina-23 u prevremenom razvoju emfizema pluća među HIV-1 + pušačima.” 2017. Web. 10 May 2021.

Vancouver:

Barjaktarević I. Uloga Interleukina-23 u prevremenom razvoju emfizema pluća među HIV-1 + pušačima. [Internet] [Thesis]. Univerzitet u Beogradu; 2017. [cited 2021 May 10]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:14289/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barjaktarević I. Uloga Interleukina-23 u prevremenom razvoju emfizema pluća među HIV-1 + pušačima. [Thesis]. Univerzitet u Beogradu; 2017. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:14289/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

16. Hall, Gregory. Dextran sulfate sodium colitis facilitates murine colonization by Shiga toxin-producing E. coli: a novel model for the study of Shiga toxicosis.

Degree: PhD, Pathology, 2018, Boston University

 Shiga toxin-producing E. coli (STEC) are globally relevant bacterial pathogens responsible for epidemic outbreaks of hemorrhagic diarrhea with variable progression to potentially fatal systemic Shiga… (more)

Subjects/Keywords: Pathology; EHEC; Interleukin 23; Shiga toxin-producing E. coli; Shiga toxins; STEC

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APA (6th Edition):

Hall, G. (2018). Dextran sulfate sodium colitis facilitates murine colonization by Shiga toxin-producing E. coli: a novel model for the study of Shiga toxicosis. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/33001

Chicago Manual of Style (16th Edition):

Hall, Gregory. “Dextran sulfate sodium colitis facilitates murine colonization by Shiga toxin-producing E. coli: a novel model for the study of Shiga toxicosis.” 2018. Doctoral Dissertation, Boston University. Accessed May 10, 2021. http://hdl.handle.net/2144/33001.

MLA Handbook (7th Edition):

Hall, Gregory. “Dextran sulfate sodium colitis facilitates murine colonization by Shiga toxin-producing E. coli: a novel model for the study of Shiga toxicosis.” 2018. Web. 10 May 2021.

Vancouver:

Hall G. Dextran sulfate sodium colitis facilitates murine colonization by Shiga toxin-producing E. coli: a novel model for the study of Shiga toxicosis. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2021 May 10]. Available from: http://hdl.handle.net/2144/33001.

Council of Science Editors:

Hall G. Dextran sulfate sodium colitis facilitates murine colonization by Shiga toxin-producing E. coli: a novel model for the study of Shiga toxicosis. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/33001

17. Paus, Roelof Anne de. Impaired type I immunity to mycobacterial infections.

Degree: 2013, Department of Infectious Diseases, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University

 Type I immune responses play an essential role in the control of mycobacterial infections. Mutations in the genes involved in the type I cytokine pathway… (more)

Subjects/Keywords: Immunedeficiency; Cytokine; Interferon; Immunity; Mycobacterium; Tuberculosis; Interleukin-12; Interleukin-23; Immunedeficiency; Cytokine; Interferon; Immunity; Mycobacterium; Tuberculosis; Interleukin-12; Interleukin-23

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APA (6th Edition):

Paus, R. A. d. (2013). Impaired type I immunity to mycobacterial infections. (Doctoral Dissertation). Department of Infectious Diseases, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University. Retrieved from http://hdl.handle.net/1887/20635

Chicago Manual of Style (16th Edition):

Paus, Roelof Anne de. “Impaired type I immunity to mycobacterial infections.” 2013. Doctoral Dissertation, Department of Infectious Diseases, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University. Accessed May 10, 2021. http://hdl.handle.net/1887/20635.

MLA Handbook (7th Edition):

Paus, Roelof Anne de. “Impaired type I immunity to mycobacterial infections.” 2013. Web. 10 May 2021.

Vancouver:

Paus RAd. Impaired type I immunity to mycobacterial infections. [Internet] [Doctoral dissertation]. Department of Infectious Diseases, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University; 2013. [cited 2021 May 10]. Available from: http://hdl.handle.net/1887/20635.

Council of Science Editors:

Paus RAd. Impaired type I immunity to mycobacterial infections. [Doctoral Dissertation]. Department of Infectious Diseases, Faculty of Medicine, Leiden University Medical Center (LUMC), Leiden University; 2013. Available from: http://hdl.handle.net/1887/20635

18. Backes, Fabiane Neiva. Biomarcadores séricos e prognóstico no acidente vascular cerebral isquêmico.

Degree: 2019, Brazil

 Fundamentação: O acidente vascular cerebral (AVC) isquêmico apresenta alta morbidade, sendo responsável por sequelas motoras, sensoriais e cognitivas. A predição de desfechos após o evento… (more)

Subjects/Keywords: Biomarcadores; Acidente vascular cerebral; Prognóstico; Interleucina-17; Interleucina-23; Células mielóides; Sangue; Cerebral ischemic stroke; Blood biomarkers; Outcome; Triggering receptor expressed on myeloid cells type 1; TREM-1; Triggering receptor expressed on myeloid cells type 2; TREM-2; Interleukin 23 (IL-23); Interleukin 17 (IL-17)

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APA (6th Edition):

Backes, F. N. (2019). Biomarcadores séricos e prognóstico no acidente vascular cerebral isquêmico. (Doctoral Dissertation). Brazil. Retrieved from http://hdl.handle.net/10183/204302

Chicago Manual of Style (16th Edition):

Backes, Fabiane Neiva. “Biomarcadores séricos e prognóstico no acidente vascular cerebral isquêmico.” 2019. Doctoral Dissertation, Brazil. Accessed May 10, 2021. http://hdl.handle.net/10183/204302.

MLA Handbook (7th Edition):

Backes, Fabiane Neiva. “Biomarcadores séricos e prognóstico no acidente vascular cerebral isquêmico.” 2019. Web. 10 May 2021.

Vancouver:

Backes FN. Biomarcadores séricos e prognóstico no acidente vascular cerebral isquêmico. [Internet] [Doctoral dissertation]. Brazil; 2019. [cited 2021 May 10]. Available from: http://hdl.handle.net/10183/204302.

Council of Science Editors:

Backes FN. Biomarcadores séricos e prognóstico no acidente vascular cerebral isquêmico. [Doctoral Dissertation]. Brazil; 2019. Available from: http://hdl.handle.net/10183/204302

19. Fernanda Manente Milanez. O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante.

Degree: 2017, University of São Paulo

Introdução: Apesar dos recentes avanços no entendimento da fisiopatologia e no tratamento da espondilite anquilosante (EA), pouco se sabe acerca da influência das medicações anti-fator… (more)

Subjects/Keywords: Dinoprostona; Espondilite anquilosante; Fator de necrose tumoral alfa; Inflamação; Interleucina-17; Interleucina-23; Medicações anti-TNF; Anti-TNFdrugs; Dinoprostone; Inflammation; Interleukin-23; Interlukin-17; Spondylitis ankylosing; Tumor necrosis factor-alpha

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APA (6th Edition):

Milanez, F. M. (2017). O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5164/tde-23082017-111419/

Chicago Manual of Style (16th Edition):

Milanez, Fernanda Manente. “O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante.” 2017. Doctoral Dissertation, University of São Paulo. Accessed May 10, 2021. http://www.teses.usp.br/teses/disponiveis/5/5164/tde-23082017-111419/.

MLA Handbook (7th Edition):

Milanez, Fernanda Manente. “O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante.” 2017. Web. 10 May 2021.

Vancouver:

Milanez FM. O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante. [Internet] [Doctoral dissertation]. University of São Paulo; 2017. [cited 2021 May 10]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5164/tde-23082017-111419/.

Council of Science Editors:

Milanez FM. O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante. [Doctoral Dissertation]. University of São Paulo; 2017. Available from: http://www.teses.usp.br/teses/disponiveis/5/5164/tde-23082017-111419/


Freie Universität Berlin

20. Müller, Gerrit. Der Einfluss von entzündlichen Konditionen auf die immunologische Reaktion von kutanen dendritischen Zellen.

Degree: 2019, Freie Universität Berlin

 Epidermale dendritische Zellen, genannt Langerhans-Zellen (LC), stellen zusammen mit dermalen dendritischen Zellen (DDC) die ersten immunologischen Wächter der menschlichen Haut dar. Als spezifische kutane Antigen-präsentierende… (more)

Subjects/Keywords: Dendritic cells; Langerhans cells; Autophagy; Lysosomotropism; Interleukin-23; Inflammation; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA (6th Edition):

Müller, G. (2019). Der Einfluss von entzündlichen Konditionen auf die immunologische Reaktion von kutanen dendritischen Zellen. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-1837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Müller, Gerrit. “Der Einfluss von entzündlichen Konditionen auf die immunologische Reaktion von kutanen dendritischen Zellen.” 2019. Thesis, Freie Universität Berlin. Accessed May 10, 2021. http://dx.doi.org/10.17169/refubium-1837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Müller, Gerrit. “Der Einfluss von entzündlichen Konditionen auf die immunologische Reaktion von kutanen dendritischen Zellen.” 2019. Web. 10 May 2021.

Vancouver:

Müller G. Der Einfluss von entzündlichen Konditionen auf die immunologische Reaktion von kutanen dendritischen Zellen. [Internet] [Thesis]. Freie Universität Berlin; 2019. [cited 2021 May 10]. Available from: http://dx.doi.org/10.17169/refubium-1837.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Müller G. Der Einfluss von entzündlichen Konditionen auf die immunologische Reaktion von kutanen dendritischen Zellen. [Thesis]. Freie Universität Berlin; 2019. Available from: http://dx.doi.org/10.17169/refubium-1837

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Henningsson, Louise. Interleukin 15 and 17 in Staphylococcus aureus arthritis.

Degree: 2011, University of Gothenburg / Göteborgs Universitet

 Staphylococcus aureus–induced arthritis leads to severe joint destruction and high mortality despite antibiotic treatment. Thus, there is a need to identify new treatment targets in… (more)

Subjects/Keywords: Staphylococcus aureus; Interleukin-17A; Interleukin-15; arthritis; osteoclasts; IL-23

…expressed by other cells in addition to these haematopoietic cells. Interleukin-23 IL-23 is a… …downregulation by Interleukin-17A on the upstream cytokine Interleukin-23 under both Gram-positive and… …GM-CSF HSC ICAM IFN ILIL-(17, 15, 23 )R LPS M-CSF MCP MHC II MIP MMP MØ MPO MRSA… …Interferon Interleukin Interleukin receptor Lipopolysaccharide Macrophage-colony stimulating factor… …cytokines such as TNF-α, interleukin (IL)-6 and IL-1 [17]. The bacterium… 

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APA (6th Edition):

Henningsson, L. (2011). Interleukin 15 and 17 in Staphylococcus aureus arthritis. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/26268

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Henningsson, Louise. “Interleukin 15 and 17 in Staphylococcus aureus arthritis.” 2011. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed May 10, 2021. http://hdl.handle.net/2077/26268.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Henningsson, Louise. “Interleukin 15 and 17 in Staphylococcus aureus arthritis.” 2011. Web. 10 May 2021.

Vancouver:

Henningsson L. Interleukin 15 and 17 in Staphylococcus aureus arthritis. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. [cited 2021 May 10]. Available from: http://hdl.handle.net/2077/26268.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Henningsson L. Interleukin 15 and 17 in Staphylococcus aureus arthritis. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2011. Available from: http://hdl.handle.net/2077/26268

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Awoniyi, Muyiwa. The roles of IL-12 and IL-23 in the Innate and Adaptive Immune Responses against Salmonella.

Degree: PhD, 2013, University of Washington

 →Non-typhoidal Salmonella is a major health concern worldwide typically causing gastroenteritis in people of all ages and severe invasive disease in immune-compromised persons in particular… (more)

Subjects/Keywords: IL-12; IL-23; Interferon gamma response; Interleukin-12; Interleukin-23; Salmonella immunity; Immunology; Microbiology; molecular and cellular biology

23 act in concert to establish host immunity to Salmonella infection. In this thesis, I… …will define the role of IL-12 and IL-23 in host immune responses to Salmonella infection… …implicated as key mediators of mucosal immunity. IL-23 is important for protection of the gut… …killer (NK) cells. In this thesis, I will explore the role of IL-12 and IL-23 in… …maculopapular rash 23. In absence of antibiotic therapy 10-15% of cases are fatal and in some regions… 

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APA (6th Edition):

Awoniyi, M. (2013). The roles of IL-12 and IL-23 in the Innate and Adaptive Immune Responses against Salmonella. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/22583

Chicago Manual of Style (16th Edition):

Awoniyi, Muyiwa. “The roles of IL-12 and IL-23 in the Innate and Adaptive Immune Responses against Salmonella.” 2013. Doctoral Dissertation, University of Washington. Accessed May 10, 2021. http://hdl.handle.net/1773/22583.

MLA Handbook (7th Edition):

Awoniyi, Muyiwa. “The roles of IL-12 and IL-23 in the Innate and Adaptive Immune Responses against Salmonella.” 2013. Web. 10 May 2021.

Vancouver:

Awoniyi M. The roles of IL-12 and IL-23 in the Innate and Adaptive Immune Responses against Salmonella. [Internet] [Doctoral dissertation]. University of Washington; 2013. [cited 2021 May 10]. Available from: http://hdl.handle.net/1773/22583.

Council of Science Editors:

Awoniyi M. The roles of IL-12 and IL-23 in the Innate and Adaptive Immune Responses against Salmonella. [Doctoral Dissertation]. University of Washington; 2013. Available from: http://hdl.handle.net/1773/22583

23. Villegas Vázquez, José Adolfo. Targeting the IL-23/Th17 pathway to treat Myasthenia Gravis : Cibler la voie IL-23 / Th17 pour traiter la myasthénie grave.

Degree: Docteur es, Immunologie, 2019, Sorbonne université

La myasthénie grave (MG) est une maladie auto-immune neuromusculaire due à des autoanticorps dirigés contre les récepteurs à l’acétylcholine. Le thymus MG est inflammatoire. Il… (more)

Subjects/Keywords: Myasthénie grave; Interleukine-23; Cellules th17; Modèles murins; Thérapie; Anticorps monoclonal; Centres germinatifs ectopiques; Récepteur à l'acetylcholine; Myasthenia gravis; Interleukin 23; Th17 cells; Murine models; Therapy; Monoclonal antibody; Ectopic germinal centers; Acetylcholine receptors; 571.96; 616.7442

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APA (6th Edition):

Villegas Vázquez, J. A. (2019). Targeting the IL-23/Th17 pathway to treat Myasthenia Gravis : Cibler la voie IL-23 / Th17 pour traiter la myasthénie grave. (Doctoral Dissertation). Sorbonne université. Retrieved from http://www.theses.fr/2019SORUS393

Chicago Manual of Style (16th Edition):

Villegas Vázquez, José Adolfo. “Targeting the IL-23/Th17 pathway to treat Myasthenia Gravis : Cibler la voie IL-23 / Th17 pour traiter la myasthénie grave.” 2019. Doctoral Dissertation, Sorbonne université. Accessed May 10, 2021. http://www.theses.fr/2019SORUS393.

MLA Handbook (7th Edition):

Villegas Vázquez, José Adolfo. “Targeting the IL-23/Th17 pathway to treat Myasthenia Gravis : Cibler la voie IL-23 / Th17 pour traiter la myasthénie grave.” 2019. Web. 10 May 2021.

Vancouver:

Villegas Vázquez JA. Targeting the IL-23/Th17 pathway to treat Myasthenia Gravis : Cibler la voie IL-23 / Th17 pour traiter la myasthénie grave. [Internet] [Doctoral dissertation]. Sorbonne université; 2019. [cited 2021 May 10]. Available from: http://www.theses.fr/2019SORUS393.

Council of Science Editors:

Villegas Vázquez JA. Targeting the IL-23/Th17 pathway to treat Myasthenia Gravis : Cibler la voie IL-23 / Th17 pour traiter la myasthénie grave. [Doctoral Dissertation]. Sorbonne université; 2019. Available from: http://www.theses.fr/2019SORUS393


The Ohio State University

24. Smith, Alan Jay. IL-23 Receptor Expression and Effects of Signaling on T Cell Encephalitogenicity.

Degree: PhD, Integrated Biomedical Science Graduate Program, 2011, The Ohio State University

 Multiple Sclerosis (MS) is an immune mediated disease of the central nervous systemcharacterized by the destruction of myelin sheaths surrounding nerve axons, and it is… (more)

Subjects/Keywords: Immunology; Interleukin-23; IL-23; experimental autoimmune encephalomyelitis; EAE; Citrobacter rodentium; CFA; Multiple Sclerosis; MS; Th17; IL-17; adjuvant

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APA (6th Edition):

Smith, A. J. (2011). IL-23 Receptor Expression and Effects of Signaling on T Cell Encephalitogenicity. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1309464694

Chicago Manual of Style (16th Edition):

Smith, Alan Jay. “IL-23 Receptor Expression and Effects of Signaling on T Cell Encephalitogenicity.” 2011. Doctoral Dissertation, The Ohio State University. Accessed May 10, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1309464694.

MLA Handbook (7th Edition):

Smith, Alan Jay. “IL-23 Receptor Expression and Effects of Signaling on T Cell Encephalitogenicity.” 2011. Web. 10 May 2021.

Vancouver:

Smith AJ. IL-23 Receptor Expression and Effects of Signaling on T Cell Encephalitogenicity. [Internet] [Doctoral dissertation]. The Ohio State University; 2011. [cited 2021 May 10]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1309464694.

Council of Science Editors:

Smith AJ. IL-23 Receptor Expression and Effects of Signaling on T Cell Encephalitogenicity. [Doctoral Dissertation]. The Ohio State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1309464694

25. Dodd, Christopher H. Toll-like receptor stimulation can lead to differential production of IL-23 and IL-12.

Degree: PhD, 2008, University of Alabama – Birmingham

Historically, multiple sclerosis, rheumatoid arthritis, and inflammatory bowel disease have been associated with Th1 cells, characterized by production of Interferon gamma (IFN-[gamma]), and IL-12, a… (more)

Subjects/Keywords: Dendritic Cells<; br>; Interleukin-12 Subunit p40  – analysis<; br>; Interleukin-23 Subunit p19<; br>; analysis<; br>; Th1 Cells  – immunology<; br>; Toll-Like Receptors  – metabolism

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APA (6th Edition):

Dodd, C. H. (2008). Toll-like receptor stimulation can lead to differential production of IL-23 and IL-12. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,440

Chicago Manual of Style (16th Edition):

Dodd, Christopher H. “Toll-like receptor stimulation can lead to differential production of IL-23 and IL-12.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed May 10, 2021. http://contentdm.mhsl.uab.edu/u?/etd,440.

MLA Handbook (7th Edition):

Dodd, Christopher H. “Toll-like receptor stimulation can lead to differential production of IL-23 and IL-12.” 2008. Web. 10 May 2021.

Vancouver:

Dodd CH. Toll-like receptor stimulation can lead to differential production of IL-23 and IL-12. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 May 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,440.

Council of Science Editors:

Dodd CH. Toll-like receptor stimulation can lead to differential production of IL-23 and IL-12. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,440


University of Queensland

26. Lee, James. The role of skin macrophages under conditions of stress and injury: ultraviolet irradiation and wound healing.

Degree: Faculty of Medicine, 2018, University of Queensland

Subjects/Keywords: UV irradiation; Immune Suppression; Macrophage; Wound Healing; Chronic Wounds; Treg; Clodronate Liposomes; Interleukin-17; Interleukin-23; 0606 Physiology; 1107 Immunology

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APA (6th Edition):

Lee, J. (2018). The role of skin macrophages under conditions of stress and injury: ultraviolet irradiation and wound healing. (Thesis). University of Queensland. Retrieved from https://espace.library.uq.edu.au/view/UQ:ccafa7c/s4209759_PhD_Thesis_Final.pdf ; https://espace.library.uq.edu.au/view/UQ:ccafa7c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, James. “The role of skin macrophages under conditions of stress and injury: ultraviolet irradiation and wound healing.” 2018. Thesis, University of Queensland. Accessed May 10, 2021. https://espace.library.uq.edu.au/view/UQ:ccafa7c/s4209759_PhD_Thesis_Final.pdf ; https://espace.library.uq.edu.au/view/UQ:ccafa7c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, James. “The role of skin macrophages under conditions of stress and injury: ultraviolet irradiation and wound healing.” 2018. Web. 10 May 2021.

Vancouver:

Lee J. The role of skin macrophages under conditions of stress and injury: ultraviolet irradiation and wound healing. [Internet] [Thesis]. University of Queensland; 2018. [cited 2021 May 10]. Available from: https://espace.library.uq.edu.au/view/UQ:ccafa7c/s4209759_PhD_Thesis_Final.pdf ; https://espace.library.uq.edu.au/view/UQ:ccafa7c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee J. The role of skin macrophages under conditions of stress and injury: ultraviolet irradiation and wound healing. [Thesis]. University of Queensland; 2018. Available from: https://espace.library.uq.edu.au/view/UQ:ccafa7c/s4209759_PhD_Thesis_Final.pdf ; https://espace.library.uq.edu.au/view/UQ:ccafa7c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

27. Ivanov, Stefan. Interleukin-17 in models of neutrophilic lung disease.

Degree: 2006, University of Gothenburg / Göteborgs Universitet

 Several acute or chronic lung disorders like adult respiratory distress syndrome, acute severe asthma, chronic obstructive pulmonary disease, chronic lung allograft rejection and cystic fibrosis,… (more)

Subjects/Keywords: Interleukin-17; Interleukin-23; Matrix metalloproteinase-9; neutrophils; macrophages; CD4 cells; innate response; lungs

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APA (6th Edition):

Ivanov, S. (2006). Interleukin-17 in models of neutrophilic lung disease. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ivanov, Stefan. “Interleukin-17 in models of neutrophilic lung disease.” 2006. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed May 10, 2021. http://hdl.handle.net/2077/791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ivanov, Stefan. “Interleukin-17 in models of neutrophilic lung disease.” 2006. Web. 10 May 2021.

Vancouver:

Ivanov S. Interleukin-17 in models of neutrophilic lung disease. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. [cited 2021 May 10]. Available from: http://hdl.handle.net/2077/791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ivanov S. Interleukin-17 in models of neutrophilic lung disease. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. Available from: http://hdl.handle.net/2077/791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

28. Mathews, Amber Lynn. Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia.

Degree: MS, 2010, Louisiana State University

 Microsporidia are ubiquitous, obligate intracellular eukaryotes that cause chronic diarrhea and disseminated diseases in humans, especially in immunocompromised individuals. Macrophages, cellular components of the innate… (more)

Subjects/Keywords: interleukin 23; p38 gamma; innate immunity; Encephalitozoon; microsporidia; macrophage; dendritic cells; interleukin 12; immune response; p38 MAPK; p38 gamma; p38 delta

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APA (6th Edition):

Mathews, A. L. (2010). Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia. (Masters Thesis). Louisiana State University. Retrieved from etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366

Chicago Manual of Style (16th Edition):

Mathews, Amber Lynn. “Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia.” 2010. Masters Thesis, Louisiana State University. Accessed May 10, 2021. etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366.

MLA Handbook (7th Edition):

Mathews, Amber Lynn. “Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia.” 2010. Web. 10 May 2021.

Vancouver:

Mathews AL. Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia. [Internet] [Masters thesis]. Louisiana State University; 2010. [cited 2021 May 10]. Available from: etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366.

Council of Science Editors:

Mathews AL. Comparative studies of innate host defense mechanisms against virulent and avirulent species of microsporidia. [Masters Thesis]. Louisiana State University; 2010. Available from: etd-07072010-171808 ; https://digitalcommons.lsu.edu/gradschool_theses/2366


University of Cincinnati

29. Klenk, Erin Ingersoll. Endoplasmic Reticulum Stress and the Unfolded Protein Response Result in Synergistic Upregulation of Interleukin-23 and Interleukin-12 by LPS.

Degree: MS, Medicine : Immunobiology, 2009, University of Cincinnati

  The unfolded protein response (UPR) is a conserved mechanism by which cells cope with endogenous threats result in endoplasmic reticulum (ER) stress. Our laboratory… (more)

Subjects/Keywords: Biomedical Research; Cellular Biology; Immunology; Interleukin-23; Interleukin-12; HLA-B27; Unfolded Protein Response; UPR; ER stress

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APA (6th Edition):

Klenk, E. I. (2009). Endoplasmic Reticulum Stress and the Unfolded Protein Response Result in Synergistic Upregulation of Interleukin-23 and Interleukin-12 by LPS. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250092843

Chicago Manual of Style (16th Edition):

Klenk, Erin Ingersoll. “Endoplasmic Reticulum Stress and the Unfolded Protein Response Result in Synergistic Upregulation of Interleukin-23 and Interleukin-12 by LPS.” 2009. Masters Thesis, University of Cincinnati. Accessed May 10, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250092843.

MLA Handbook (7th Edition):

Klenk, Erin Ingersoll. “Endoplasmic Reticulum Stress and the Unfolded Protein Response Result in Synergistic Upregulation of Interleukin-23 and Interleukin-12 by LPS.” 2009. Web. 10 May 2021.

Vancouver:

Klenk EI. Endoplasmic Reticulum Stress and the Unfolded Protein Response Result in Synergistic Upregulation of Interleukin-23 and Interleukin-12 by LPS. [Internet] [Masters thesis]. University of Cincinnati; 2009. [cited 2021 May 10]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250092843.

Council of Science Editors:

Klenk EI. Endoplasmic Reticulum Stress and the Unfolded Protein Response Result in Synergistic Upregulation of Interleukin-23 and Interleukin-12 by LPS. [Masters Thesis]. University of Cincinnati; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1250092843


Freie Universität Berlin

30. Struck, Daniela. The impact of IL-12 and IL-23 on immune response to Toxoplasma gondii.

Degree: 2009, Freie Universität Berlin

 Following infection with Toxoplasma gondii a protective Th1-immune response is induced, characterized by IL-12 associated IFN--production. Following high- dose infection, suszeptible mice develop a Th1-induced… (more)

Subjects/Keywords: Toxoplasma gondii; mice; mice; knockout (MeSH); experimental animal; immune response; interleukin 12; interleukin 18, interleukin 23; interleukin-12 subunit p35 (MeSH); interleukin-12 subunit p40 (MeSH); bone marrow; radiation chimera (MeSH); bone marrow transplantation (MeSH); bone marrow cells; immunopathology; t lymphocytes; th1 cells (MeSH); inflammatory bowel diseases; 600 Technik, Medizin, angewandte Wissenschaften::630 Landwirtschaft

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APA (6th Edition):

Struck, D. (2009). The impact of IL-12 and IL-23 on immune response to Toxoplasma gondii. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-13395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Struck, Daniela. “The impact of IL-12 and IL-23 on immune response to Toxoplasma gondii.” 2009. Thesis, Freie Universität Berlin. Accessed May 10, 2021. http://dx.doi.org/10.17169/refubium-13395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Struck, Daniela. “The impact of IL-12 and IL-23 on immune response to Toxoplasma gondii.” 2009. Web. 10 May 2021.

Vancouver:

Struck D. The impact of IL-12 and IL-23 on immune response to Toxoplasma gondii. [Internet] [Thesis]. Freie Universität Berlin; 2009. [cited 2021 May 10]. Available from: http://dx.doi.org/10.17169/refubium-13395.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Struck D. The impact of IL-12 and IL-23 on immune response to Toxoplasma gondii. [Thesis]. Freie Universität Berlin; 2009. Available from: http://dx.doi.org/10.17169/refubium-13395

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

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