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You searched for subject:(interleukin 22). Showing records 1 – 19 of 19 total matches.

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Johannes Gutenberg Universität Mainz

1. Schneeweiß, Kristin. Die Rolle von Interleukin-22 bei Asthma bronchiale.

Degree: 2011, Johannes Gutenberg Universität Mainz

Interleukin (IL)-22 ist ein Effektorzytokin, das von Zellen des Immunsystems produziert wird und auf Epithelzellen wirkt. Es nimmt eine duale Rolle ein, indem es abhängig… (more)

Subjects/Keywords: Interleukin-22; Funktion; Asthma bronchiale; Interleukin-22; functional role; allergic asthma; Life sciences

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APA (6th Edition):

Schneeweiß, K. (2011). Die Rolle von Interleukin-22 bei Asthma bronchiale. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2011/2859/

Chicago Manual of Style (16th Edition):

Schneeweiß, Kristin. “Die Rolle von Interleukin-22 bei Asthma bronchiale.” 2011. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed January 25, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2011/2859/.

MLA Handbook (7th Edition):

Schneeweiß, Kristin. “Die Rolle von Interleukin-22 bei Asthma bronchiale.” 2011. Web. 25 Jan 2021.

Vancouver:

Schneeweiß K. Die Rolle von Interleukin-22 bei Asthma bronchiale. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2011. [cited 2021 Jan 25]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2011/2859/.

Council of Science Editors:

Schneeweiß K. Die Rolle von Interleukin-22 bei Asthma bronchiale. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2011. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2011/2859/


Georgia State University

2. Zhang, Zhan. Exploring Alternative Antiviral Therapeutic Strategies Through Immunomodulation and Learning Novel Innate Immune Responses Against Rotavirus Infection.

Degree: PhD, Biology, 2019, Georgia State University

  Rotavirus infections are associated with outbreaks of acute gastroenteritis. Non-dividing highly differentiated enterocytes, located at the tips of small intestinal villi, are the primary… (more)

Subjects/Keywords: Rotavirus; Toll-like receptor 5 (TLR5); NOD-like receptor C4 (NLRC4); Flagellin; Interleukin 22 (IL-22); Interleukin 18 (IL-18)

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APA (6th Edition):

Zhang, Z. (2019). Exploring Alternative Antiviral Therapeutic Strategies Through Immunomodulation and Learning Novel Innate Immune Responses Against Rotavirus Infection. (Doctoral Dissertation). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_diss/214

Chicago Manual of Style (16th Edition):

Zhang, Zhan. “Exploring Alternative Antiviral Therapeutic Strategies Through Immunomodulation and Learning Novel Innate Immune Responses Against Rotavirus Infection.” 2019. Doctoral Dissertation, Georgia State University. Accessed January 25, 2021. https://scholarworks.gsu.edu/biology_diss/214.

MLA Handbook (7th Edition):

Zhang, Zhan. “Exploring Alternative Antiviral Therapeutic Strategies Through Immunomodulation and Learning Novel Innate Immune Responses Against Rotavirus Infection.” 2019. Web. 25 Jan 2021.

Vancouver:

Zhang Z. Exploring Alternative Antiviral Therapeutic Strategies Through Immunomodulation and Learning Novel Innate Immune Responses Against Rotavirus Infection. [Internet] [Doctoral dissertation]. Georgia State University; 2019. [cited 2021 Jan 25]. Available from: https://scholarworks.gsu.edu/biology_diss/214.

Council of Science Editors:

Zhang Z. Exploring Alternative Antiviral Therapeutic Strategies Through Immunomodulation and Learning Novel Innate Immune Responses Against Rotavirus Infection. [Doctoral Dissertation]. Georgia State University; 2019. Available from: https://scholarworks.gsu.edu/biology_diss/214


University of Washington

3. Lim, Chrissie. Alternative splicing takes control of cytokine signaling.

Degree: PhD, 2016, University of Washington

 Immune responses must be tightly controlled for dose, location, strength and duration using genetic, epigenetic or biochemical regulation. Among these, the generation of alternatively-spliced transcripts… (more)

Subjects/Keywords: Cytokine; Infection; Interferon; Interleukin-22; Receptor; Splicing; Immunology; immunology

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APA (6th Edition):

Lim, C. (2016). Alternative splicing takes control of cytokine signaling. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/37154

Chicago Manual of Style (16th Edition):

Lim, Chrissie. “Alternative splicing takes control of cytokine signaling.” 2016. Doctoral Dissertation, University of Washington. Accessed January 25, 2021. http://hdl.handle.net/1773/37154.

MLA Handbook (7th Edition):

Lim, Chrissie. “Alternative splicing takes control of cytokine signaling.” 2016. Web. 25 Jan 2021.

Vancouver:

Lim C. Alternative splicing takes control of cytokine signaling. [Internet] [Doctoral dissertation]. University of Washington; 2016. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/1773/37154.

Council of Science Editors:

Lim C. Alternative splicing takes control of cytokine signaling. [Doctoral Dissertation]. University of Washington; 2016. Available from: http://hdl.handle.net/1773/37154

4. Oliveira, Katharina Morant Holanda de. Ausência da interleucina-22 interfere na microbiota bucal e na progressão de lesões periapicais induzidas em dentes de camundongos.

Degree: Mestrado, Odontopediatria, 2013, University of São Paulo

 Introdução: O objetivo deste trabalho foi caracterizar a composição da microbiota bucal e a formação e progressão de lesões periapicais induzidas experimentalmente em dentes de… (more)

Subjects/Keywords: camundongos knockout; immunohistochemistry; imunohistoquímica; inflamação; inflammation; Interleucina-22; Interleukin-22; knockout mice; lesão periapical; micro-organismos; microorganisms; osteoclastos; osteoclasts; periapical lesion

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APA (6th Edition):

Oliveira, K. M. H. d. (2013). Ausência da interleucina-22 interfere na microbiota bucal e na progressão de lesões periapicais induzidas em dentes de camundongos. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/58/58135/tde-17052013-164100/ ;

Chicago Manual of Style (16th Edition):

Oliveira, Katharina Morant Holanda de. “Ausência da interleucina-22 interfere na microbiota bucal e na progressão de lesões periapicais induzidas em dentes de camundongos.” 2013. Masters Thesis, University of São Paulo. Accessed January 25, 2021. http://www.teses.usp.br/teses/disponiveis/58/58135/tde-17052013-164100/ ;.

MLA Handbook (7th Edition):

Oliveira, Katharina Morant Holanda de. “Ausência da interleucina-22 interfere na microbiota bucal e na progressão de lesões periapicais induzidas em dentes de camundongos.” 2013. Web. 25 Jan 2021.

Vancouver:

Oliveira KMHd. Ausência da interleucina-22 interfere na microbiota bucal e na progressão de lesões periapicais induzidas em dentes de camundongos. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2021 Jan 25]. Available from: http://www.teses.usp.br/teses/disponiveis/58/58135/tde-17052013-164100/ ;.

Council of Science Editors:

Oliveira KMHd. Ausência da interleucina-22 interfere na microbiota bucal e na progressão de lesões periapicais induzidas em dentes de camundongos. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/58/58135/tde-17052013-164100/ ;

5. Guillon, Antoine. Altération de la réponse immunitaire dépendante de l'interleukine-22 lors de pathologies respiratoires : Alteration of the interleukin-22 pathway in respiratory diseases.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2014, Université François-Rabelais de Tours

La voie de signalisation impliquant l’interleukine (IL)-22 a un rôle majeur dans le maintien des fonctions de barrière des surfaces exposées du corps humain. Elle… (more)

Subjects/Keywords: Interleukine-22; Immunité antimicrobienne épithéliale; Pathologies respiratoires; P. aeruginosa; Interleukin-22; Innate mucosal immunity; Respiratory pathologies; P. aeruginosa

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APA (6th Edition):

Guillon, A. (2014). Altération de la réponse immunitaire dépendante de l'interleukine-22 lors de pathologies respiratoires : Alteration of the interleukin-22 pathway in respiratory diseases. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2014TOUR3316

Chicago Manual of Style (16th Edition):

Guillon, Antoine. “Altération de la réponse immunitaire dépendante de l'interleukine-22 lors de pathologies respiratoires : Alteration of the interleukin-22 pathway in respiratory diseases.” 2014. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed January 25, 2021. http://www.theses.fr/2014TOUR3316.

MLA Handbook (7th Edition):

Guillon, Antoine. “Altération de la réponse immunitaire dépendante de l'interleukine-22 lors de pathologies respiratoires : Alteration of the interleukin-22 pathway in respiratory diseases.” 2014. Web. 25 Jan 2021.

Vancouver:

Guillon A. Altération de la réponse immunitaire dépendante de l'interleukine-22 lors de pathologies respiratoires : Alteration of the interleukin-22 pathway in respiratory diseases. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2014. [cited 2021 Jan 25]. Available from: http://www.theses.fr/2014TOUR3316.

Council of Science Editors:

Guillon A. Altération de la réponse immunitaire dépendante de l'interleukine-22 lors de pathologies respiratoires : Alteration of the interleukin-22 pathway in respiratory diseases. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2014. Available from: http://www.theses.fr/2014TOUR3316


University of Iowa

6. Ness, Kristin Jennifer. Cytokine requirements for the differentiation and expansion of Il-17a- and Il-22-producing human Vγ2vδ2 T cells.

Degree: PhD, Immunology, 2011, University of Iowa

  Human γδ T cells expressing the Vγ2Vδ2 T cell antigen receptor play important roles in immune responses to microbial pathogens by monitoring prenyl pyrophosphate… (more)

Subjects/Keywords: Cell Differentiation; Humans; Interleukin-17A; Interleukin-22; Receptors; Antigen; T-Cell; gamma-delta; T-Lymphocyte Subsets; Immunology of Infectious Disease

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APA (6th Edition):

Ness, K. J. (2011). Cytokine requirements for the differentiation and expansion of Il-17a- and Il-22-producing human Vγ2vδ2 T cells. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/2751

Chicago Manual of Style (16th Edition):

Ness, Kristin Jennifer. “Cytokine requirements for the differentiation and expansion of Il-17a- and Il-22-producing human Vγ2vδ2 T cells.” 2011. Doctoral Dissertation, University of Iowa. Accessed January 25, 2021. https://ir.uiowa.edu/etd/2751.

MLA Handbook (7th Edition):

Ness, Kristin Jennifer. “Cytokine requirements for the differentiation and expansion of Il-17a- and Il-22-producing human Vγ2vδ2 T cells.” 2011. Web. 25 Jan 2021.

Vancouver:

Ness KJ. Cytokine requirements for the differentiation and expansion of Il-17a- and Il-22-producing human Vγ2vδ2 T cells. [Internet] [Doctoral dissertation]. University of Iowa; 2011. [cited 2021 Jan 25]. Available from: https://ir.uiowa.edu/etd/2751.

Council of Science Editors:

Ness KJ. Cytokine requirements for the differentiation and expansion of Il-17a- and Il-22-producing human Vγ2vδ2 T cells. [Doctoral Dissertation]. University of Iowa; 2011. Available from: https://ir.uiowa.edu/etd/2751

7. Le Rouzic, Olivier. Implication de la voie IL-17 / IL-22 dans la susceptibilité aux infections associée à la broncho-pneumopathie chronique obstructive (BPCO) : IL-17 / IL-22 pathway involvement in infectious chronic obstructive pulmonary disease (COPD) exacerbation susceptibility.

Degree: Docteur es, Immunologie, 2016, Université Lille II – Droit et Santé

 La broncho-pneumopathie chronique obstructive (BPCO) est une maladie inflammatoire chronique des voies aériennes dont le facteur de risque principal est l’exposition chronique à la fumée… (more)

Subjects/Keywords: Broncho-pneumopathie chronique obstructive; Exacerbation; Fumée de cigarette; Interleukine 17; Interleukine 22; Streptococcus pneumoniae; Chronic obstructive pulmonary disease; Exacerbation; Cigarette smoke; Interleukin 17; Interleukin 22; Streptococcus pneumoniae

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APA (6th Edition):

Le Rouzic, O. (2016). Implication de la voie IL-17 / IL-22 dans la susceptibilité aux infections associée à la broncho-pneumopathie chronique obstructive (BPCO) : IL-17 / IL-22 pathway involvement in infectious chronic obstructive pulmonary disease (COPD) exacerbation susceptibility. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2016LIL2S020

Chicago Manual of Style (16th Edition):

Le Rouzic, Olivier. “Implication de la voie IL-17 / IL-22 dans la susceptibilité aux infections associée à la broncho-pneumopathie chronique obstructive (BPCO) : IL-17 / IL-22 pathway involvement in infectious chronic obstructive pulmonary disease (COPD) exacerbation susceptibility.” 2016. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed January 25, 2021. http://www.theses.fr/2016LIL2S020.

MLA Handbook (7th Edition):

Le Rouzic, Olivier. “Implication de la voie IL-17 / IL-22 dans la susceptibilité aux infections associée à la broncho-pneumopathie chronique obstructive (BPCO) : IL-17 / IL-22 pathway involvement in infectious chronic obstructive pulmonary disease (COPD) exacerbation susceptibility.” 2016. Web. 25 Jan 2021.

Vancouver:

Le Rouzic O. Implication de la voie IL-17 / IL-22 dans la susceptibilité aux infections associée à la broncho-pneumopathie chronique obstructive (BPCO) : IL-17 / IL-22 pathway involvement in infectious chronic obstructive pulmonary disease (COPD) exacerbation susceptibility. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2016. [cited 2021 Jan 25]. Available from: http://www.theses.fr/2016LIL2S020.

Council of Science Editors:

Le Rouzic O. Implication de la voie IL-17 / IL-22 dans la susceptibilité aux infections associée à la broncho-pneumopathie chronique obstructive (BPCO) : IL-17 / IL-22 pathway involvement in infectious chronic obstructive pulmonary disease (COPD) exacerbation susceptibility. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2016. Available from: http://www.theses.fr/2016LIL2S020


University of California – San Francisco

8. Lowe, Margaret. Identification of Cinnabarinic Acid as a Novel Endogenous Aryl Hydrocarbon Receptor Ligand That Drives Th22 Differentiation.

Degree: Pharmaceutical Sciences and Pharmacogenomics, 2013, University of California – San Francisco

 The aryl hydrocarbon receptor (AHR) is a cytosolic transcription factor that recognizes and induces metabolic enzymes in response to a wide variety of xenobiotics. However,… (more)

Subjects/Keywords: Immunology; aryl hydrocarbon receptor; cinnabarinic acid; cytokine response; interleukin 22; tryptophan metabolite

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APA (6th Edition):

Lowe, M. (2013). Identification of Cinnabarinic Acid as a Novel Endogenous Aryl Hydrocarbon Receptor Ligand That Drives Th22 Differentiation. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/472365t0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lowe, Margaret. “Identification of Cinnabarinic Acid as a Novel Endogenous Aryl Hydrocarbon Receptor Ligand That Drives Th22 Differentiation.” 2013. Thesis, University of California – San Francisco. Accessed January 25, 2021. http://www.escholarship.org/uc/item/472365t0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lowe, Margaret. “Identification of Cinnabarinic Acid as a Novel Endogenous Aryl Hydrocarbon Receptor Ligand That Drives Th22 Differentiation.” 2013. Web. 25 Jan 2021.

Vancouver:

Lowe M. Identification of Cinnabarinic Acid as a Novel Endogenous Aryl Hydrocarbon Receptor Ligand That Drives Th22 Differentiation. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2021 Jan 25]. Available from: http://www.escholarship.org/uc/item/472365t0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lowe M. Identification of Cinnabarinic Acid as a Novel Endogenous Aryl Hydrocarbon Receptor Ligand That Drives Th22 Differentiation. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/472365t0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Besnard, Anne-Gaëlle. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2010, Université d'Orléans

L’asthme est une maladie inflammatoire chronique des voies aériennes. Chez les individus sensibles, l’inhalation d’allergènes entraine une inflammation pulmonaire se traduisant par des épisodes récurrents… (more)

Subjects/Keywords: Interleukine-22; Interleukine-17; Interleukine-33; Interleukine-1; NLRP3 inflammasome; Interleukin-22; Interleukin-17; Interleukin-33; Interleukin-1; NLRP3 inflammasome

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APA (6th Edition):

Besnard, A. (2010). Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. (Doctoral Dissertation). Université d'Orléans. Retrieved from http://www.theses.fr/2010ORLE2044

Chicago Manual of Style (16th Edition):

Besnard, Anne-Gaëlle. “Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.” 2010. Doctoral Dissertation, Université d'Orléans. Accessed January 25, 2021. http://www.theses.fr/2010ORLE2044.

MLA Handbook (7th Edition):

Besnard, Anne-Gaëlle. “Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation.” 2010. Web. 25 Jan 2021.

Vancouver:

Besnard A. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. [Internet] [Doctoral dissertation]. Université d'Orléans; 2010. [cited 2021 Jan 25]. Available from: http://www.theses.fr/2010ORLE2044.

Council of Science Editors:

Besnard A. Etude des mécanismes immunitaires dans un modèle d'inflammation pulmonaire allergique chez la souris : rôles de l'interleukine-22 : Roles of interleukin-22 in a mouse model of allergic airways inflammation. [Doctoral Dissertation]. Université d'Orléans; 2010. Available from: http://www.theses.fr/2010ORLE2044

10. Bleicher, Lucas. Implementação da análise de acoplamentos estatísticos e sua aplicação à família de proteínas tirosina fosfatases.

Degree: PhD, Física Aplicada, 2009, University of São Paulo

A Análise de Acoplamentos Estatísticos é uma técnica computacional capaz de identificar resíduos importantes para a estrutura e função de proteínas em uma família por… (more)

Subjects/Keywords: Análise de Acoplamentos Estatísticos; Arsenate reductases. Protein crystallography; Arsenato Redutases; Cristalografia de Proteínas; Interleucina-22; Interleukin-22; Laminarinase; Laminarinase; Protein tyrosine phosphatases; Proteínas tirosina fosfatases; Statistical coupling analysis

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APA (6th Edition):

Bleicher, L. (2009). Implementação da análise de acoplamentos estatísticos e sua aplicação à família de proteínas tirosina fosfatases. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/76/76132/tde-26032009-130135/ ;

Chicago Manual of Style (16th Edition):

Bleicher, Lucas. “Implementação da análise de acoplamentos estatísticos e sua aplicação à família de proteínas tirosina fosfatases.” 2009. Doctoral Dissertation, University of São Paulo. Accessed January 25, 2021. http://www.teses.usp.br/teses/disponiveis/76/76132/tde-26032009-130135/ ;.

MLA Handbook (7th Edition):

Bleicher, Lucas. “Implementação da análise de acoplamentos estatísticos e sua aplicação à família de proteínas tirosina fosfatases.” 2009. Web. 25 Jan 2021.

Vancouver:

Bleicher L. Implementação da análise de acoplamentos estatísticos e sua aplicação à família de proteínas tirosina fosfatases. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2021 Jan 25]. Available from: http://www.teses.usp.br/teses/disponiveis/76/76132/tde-26032009-130135/ ;.

Council of Science Editors:

Bleicher L. Implementação da análise de acoplamentos estatísticos e sua aplicação à família de proteínas tirosina fosfatases. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/76/76132/tde-26032009-130135/ ;


Universidade de Lisboa

11. Almeida, Luís Miguel Ferreira de. Role of neurotrophic factor receptors in Innate Lymphoid Cell immunity.

Degree: 2015, Universidade de Lisboa

Tese de mestrado, Bioquímica (Bioquímica Médica), Universidade de Lisboa, Faculdade de Ciências, 2015

O intestino humano é considerado o maior compartimento do sistema imunitário, tendo… (more)

Subjects/Keywords: RET; Innate lymphoid cells; Interleukin-22; Epithelial reactivity; Intestinal organoids; Teses de mestrado - 2015; Departamento de Química e Bioquímica

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APA (6th Edition):

Almeida, L. M. F. d. (2015). Role of neurotrophic factor receptors in Innate Lymphoid Cell immunity. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/22290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Almeida, Luís Miguel Ferreira de. “Role of neurotrophic factor receptors in Innate Lymphoid Cell immunity.” 2015. Thesis, Universidade de Lisboa. Accessed January 25, 2021. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/22290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Almeida, Luís Miguel Ferreira de. “Role of neurotrophic factor receptors in Innate Lymphoid Cell immunity.” 2015. Web. 25 Jan 2021.

Vancouver:

Almeida LMFd. Role of neurotrophic factor receptors in Innate Lymphoid Cell immunity. [Internet] [Thesis]. Universidade de Lisboa; 2015. [cited 2021 Jan 25]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/22290.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Almeida LMFd. Role of neurotrophic factor receptors in Innate Lymphoid Cell immunity. [Thesis]. Universidade de Lisboa; 2015. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/22290

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Bradford

12. Alase, Adewonuola Adelodi. The role of interleukin-10 family members in inflammatory skin diseases : understanding the mechanism of action of interferon lambda and interleukin-22 on human primary keratinocytes and dermal fibroblasts with a focus on healing responses in inflammatory skin diseases.

Degree: PhD, 2015, University of Bradford

 Cutaneous lupus erythematosus (CLE) is an autoimmune disease that resolves with or without permanent scars depending on the subtype. Interferons (IFNs), including the skin specific… (more)

Subjects/Keywords: 616.5; Interferon lambda; Interleukin-22; Interferon stimulated genes; Keratinocytes; Inflammatory skin diseases; Fibroblasts; Inflammation; SOCS; MAPKs

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APA (6th Edition):

Alase, A. A. (2015). The role of interleukin-10 family members in inflammatory skin diseases : understanding the mechanism of action of interferon lambda and interleukin-22 on human primary keratinocytes and dermal fibroblasts with a focus on healing responses in inflammatory skin diseases. (Doctoral Dissertation). University of Bradford. Retrieved from http://hdl.handle.net/10454/14303

Chicago Manual of Style (16th Edition):

Alase, Adewonuola Adelodi. “The role of interleukin-10 family members in inflammatory skin diseases : understanding the mechanism of action of interferon lambda and interleukin-22 on human primary keratinocytes and dermal fibroblasts with a focus on healing responses in inflammatory skin diseases.” 2015. Doctoral Dissertation, University of Bradford. Accessed January 25, 2021. http://hdl.handle.net/10454/14303.

MLA Handbook (7th Edition):

Alase, Adewonuola Adelodi. “The role of interleukin-10 family members in inflammatory skin diseases : understanding the mechanism of action of interferon lambda and interleukin-22 on human primary keratinocytes and dermal fibroblasts with a focus on healing responses in inflammatory skin diseases.” 2015. Web. 25 Jan 2021.

Vancouver:

Alase AA. The role of interleukin-10 family members in inflammatory skin diseases : understanding the mechanism of action of interferon lambda and interleukin-22 on human primary keratinocytes and dermal fibroblasts with a focus on healing responses in inflammatory skin diseases. [Internet] [Doctoral dissertation]. University of Bradford; 2015. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/10454/14303.

Council of Science Editors:

Alase AA. The role of interleukin-10 family members in inflammatory skin diseases : understanding the mechanism of action of interferon lambda and interleukin-22 on human primary keratinocytes and dermal fibroblasts with a focus on healing responses in inflammatory skin diseases. [Doctoral Dissertation]. University of Bradford; 2015. Available from: http://hdl.handle.net/10454/14303

13. Huang, Shu-Mei. Effects of high glucose on keratinocyte functions: focusing on the impaired diabetic wound healing.

Degree: PhD, Biological Sciences, 2015, NSYSU

 Diabetes mellitus is characterized by elevated plasma glucose and increased rate of skin infection. Altered immune responses have been suggested to contribute to this prevalent… (more)

Subjects/Keywords: hBD3; interleukin-8; keratinocyte; hBD2; high glucose; interleukin-22

…immunohistochemistry IL-1β: interleukin-1β IL-8: interleukin-8 IL-22: interleukin-22 IL-22 BP: interleukin-22… …口周圍皮膚 BD3 mRNA 表現 . …..22 圖 2-4 不同糖濃度培養下角質細胞存活率 .23 圖 2-5 不同糖濃度… …邊血單 球經高糖培養後之 IL-22 mRNA 表現 ...96 圖 5-8 周邊血單 球細胞培養液經 IL-22 結合蛋白處理後對角質細胞 MMP-3 表現… …x 的影響 ….. ...97 圖 5-9 周邊血單 球細胞培養液經 IL-22 結合蛋白處理後對角質細胞移動性的影 響… …98 圖 5-10 角質細胞經 MMP-3 基 靜默後對移動性的影響 ..99 圖 5-11 糖尿病大 皮膚的 IL-22 mRNA 表現情形… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, S. (2015). Effects of high glucose on keratinocyte functions: focusing on the impaired diabetic wound healing. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823115-001730

Chicago Manual of Style (16th Edition):

Huang, Shu-Mei. “Effects of high glucose on keratinocyte functions: focusing on the impaired diabetic wound healing.” 2015. Doctoral Dissertation, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823115-001730.

MLA Handbook (7th Edition):

Huang, Shu-Mei. “Effects of high glucose on keratinocyte functions: focusing on the impaired diabetic wound healing.” 2015. Web. 25 Jan 2021.

Vancouver:

Huang S. Effects of high glucose on keratinocyte functions: focusing on the impaired diabetic wound healing. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823115-001730.

Council of Science Editors:

Huang S. Effects of high glucose on keratinocyte functions: focusing on the impaired diabetic wound healing. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823115-001730


Kyoto University / 京都大学

14. Zhang, Guangyuan. Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy : インビボ遺伝子治療を目的とした肝臓吸引に基づくプラスミド導入システムの開発.

Degree: 博士(薬学), 2014, Kyoto University / 京都大学

新制・課程博士

甲第18551号

薬博第813号

Subjects/Keywords: naked plasmid DNA; liver suction-mediated transfection; transgene expression; interleukin-22

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, G. (2014). Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy : インビボ遺伝子治療を目的とした肝臓吸引に基づくプラスミド導入システムの開発. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/192151 ; http://dx.doi.org/10.14989/doctor.k18551

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Guangyuan. “Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy : インビボ遺伝子治療を目的とした肝臓吸引に基づくプラスミド導入システムの開発.” 2014. Thesis, Kyoto University / 京都大学. Accessed January 25, 2021. http://hdl.handle.net/2433/192151 ; http://dx.doi.org/10.14989/doctor.k18551.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Guangyuan. “Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy : インビボ遺伝子治療を目的とした肝臓吸引に基づくプラスミド導入システムの開発.” 2014. Web. 25 Jan 2021.

Vancouver:

Zhang G. Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy : インビボ遺伝子治療を目的とした肝臓吸引に基づくプラスミド導入システムの開発. [Internet] [Thesis]. Kyoto University / 京都大学; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/2433/192151 ; http://dx.doi.org/10.14989/doctor.k18551.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang G. Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy : インビボ遺伝子治療を目的とした肝臓吸引に基づくプラスミド導入システムの開発. [Thesis]. Kyoto University / 京都大学; 2014. Available from: http://hdl.handle.net/2433/192151 ; http://dx.doi.org/10.14989/doctor.k18551

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lund

15. Hsiung, Sabrina. Tissue Repair Responses and Integrity of the Atherosclerotic Plaque.

Degree: 2020, University of Lund

Subjects/Keywords: Basic Medicine; atherosclerosis; diabetes; tissue repair; plaque; interleukin-22; cardiovascular disease; apolipoprotein B-100; shear stress

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APA (6th Edition):

Hsiung, S. (2020). Tissue Repair Responses and Integrity of the Atherosclerotic Plaque. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/f450f6e7-fab7-4021-8177-345036c91ed4 ; https://portal.research.lu.se/ws/files/77105874/Sabrina_Hsiung_web.pdf

Chicago Manual of Style (16th Edition):

Hsiung, Sabrina. “Tissue Repair Responses and Integrity of the Atherosclerotic Plaque.” 2020. Doctoral Dissertation, University of Lund. Accessed January 25, 2021. https://lup.lub.lu.se/record/f450f6e7-fab7-4021-8177-345036c91ed4 ; https://portal.research.lu.se/ws/files/77105874/Sabrina_Hsiung_web.pdf.

MLA Handbook (7th Edition):

Hsiung, Sabrina. “Tissue Repair Responses and Integrity of the Atherosclerotic Plaque.” 2020. Web. 25 Jan 2021.

Vancouver:

Hsiung S. Tissue Repair Responses and Integrity of the Atherosclerotic Plaque. [Internet] [Doctoral dissertation]. University of Lund; 2020. [cited 2021 Jan 25]. Available from: https://lup.lub.lu.se/record/f450f6e7-fab7-4021-8177-345036c91ed4 ; https://portal.research.lu.se/ws/files/77105874/Sabrina_Hsiung_web.pdf.

Council of Science Editors:

Hsiung S. Tissue Repair Responses and Integrity of the Atherosclerotic Plaque. [Doctoral Dissertation]. University of Lund; 2020. Available from: https://lup.lub.lu.se/record/f450f6e7-fab7-4021-8177-345036c91ed4 ; https://portal.research.lu.se/ws/files/77105874/Sabrina_Hsiung_web.pdf

16. Zhang, Guangyuan. Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy .

Degree: 2014, Kyoto University

Subjects/Keywords: naked plasmid DNA; liver suction-mediated transfection; transgene expression; interleukin-22

…reporter luciferase and IL-22 protein was achieved by suction treatment. Both of these transgene… …therapeutic IL-22 protein against Con A-induced acute hepatitis mice model. - 17 - - 18… …x28;Basel) 5(12) (2012) 1372-1392. [22] D.J. Wells, Gene… …delivery systems for gene delivery. - 22 - Adv Biomed Res 1 (2012) 27. [36]… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, G. (2014). Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/192151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Guangyuan. “Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy .” 2014. Thesis, Kyoto University. Accessed January 25, 2021. http://hdl.handle.net/2433/192151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Guangyuan. “Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy .” 2014. Web. 25 Jan 2021.

Vancouver:

Zhang G. Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy . [Internet] [Thesis]. Kyoto University; 2014. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/2433/192151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang G. Development of liver suction-mediated naked plasmid DNA delivery system for in vivo gene therapy . [Thesis]. Kyoto University; 2014. Available from: http://hdl.handle.net/2433/192151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

17. Kebir, Hania. Rôle des lymphocytes TH17 dans la fragilisation de la barrière hémo-encéphalique et la formation des lésions de sclérose en plaques.

Degree: 2020, Université de Montréal

Subjects/Keywords: Barrière hémo-encéphalique (BHE); Interféron gamma (IFN-γ); Interleukine-17 (IL-17); Interleukine-22 (IL-22),; Lymphocytes TH17; Migration leucocytaire; Sclérose en plaques (SEP); Blood-brain barrier (BBB); Interferon gamma (IFN-γ); Interleukin 17 (IL-17); Interleukin 22 (IL-22); Leukocyte transmigration; Multiple sclerosis (MS); TH17 lymphocytes; Health Sciences - Immunology / Sciences de la santé - Immunologie (UMI : 0982)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kebir, H. (2020). Rôle des lymphocytes TH17 dans la fragilisation de la barrière hémo-encéphalique et la formation des lésions de sclérose en plaques. (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/23531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kebir, Hania. “Rôle des lymphocytes TH17 dans la fragilisation de la barrière hémo-encéphalique et la formation des lésions de sclérose en plaques.” 2020. Thesis, Université de Montréal. Accessed January 25, 2021. http://hdl.handle.net/1866/23531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kebir, Hania. “Rôle des lymphocytes TH17 dans la fragilisation de la barrière hémo-encéphalique et la formation des lésions de sclérose en plaques.” 2020. Web. 25 Jan 2021.

Vancouver:

Kebir H. Rôle des lymphocytes TH17 dans la fragilisation de la barrière hémo-encéphalique et la formation des lésions de sclérose en plaques. [Internet] [Thesis]. Université de Montréal; 2020. [cited 2021 Jan 25]. Available from: http://hdl.handle.net/1866/23531.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kebir H. Rôle des lymphocytes TH17 dans la fragilisation de la barrière hémo-encéphalique et la formation des lésions de sclérose en plaques. [Thesis]. Université de Montréal; 2020. Available from: http://hdl.handle.net/1866/23531

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Bellemore, Stacey M. The role of IL-22 produced by Th17 cells in Type 1 Diabetes.

Degree: 2013, University of Western Ontario

Interleukin-22 (IL-22) is produced by T helper 17 (Th17) cells. Th17 cells have been shown to be pathogenic in autoimmune diseases, however their role in… (more)

Subjects/Keywords: Type 1 Diabetes; Interleukin-22; Non-obese Diabetic mice; Th17 cells; Animal Diseases; Endocrine System Diseases; Immune System Diseases; Medical Immunology

…MS patients’ serum and spinal fluid (76). 1.3 Interleukin 22 IL-22 was first… …45 Figure 3.2 IL-22 production is optimal after 4 days of culture in IL-6 + IL-23 polarized… …and IL-22 production in BDC2.5 cells activated by antigen… …54 Figure 3.5 Addition of anti-IFN-γ to IL-6 plus IL-23 in culture induces maximal IL-22… …The effects of neutralizing anti-TGF-β and anti-IFN-γ antibodies on IL-22 production from… 

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APA (6th Edition):

Bellemore, S. M. (2013). The role of IL-22 produced by Th17 cells in Type 1 Diabetes. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/1598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bellemore, Stacey M. “The role of IL-22 produced by Th17 cells in Type 1 Diabetes.” 2013. Thesis, University of Western Ontario. Accessed January 25, 2021. https://ir.lib.uwo.ca/etd/1598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bellemore, Stacey M. “The role of IL-22 produced by Th17 cells in Type 1 Diabetes.” 2013. Web. 25 Jan 2021.

Vancouver:

Bellemore SM. The role of IL-22 produced by Th17 cells in Type 1 Diabetes. [Internet] [Thesis]. University of Western Ontario; 2013. [cited 2021 Jan 25]. Available from: https://ir.lib.uwo.ca/etd/1598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bellemore SM. The role of IL-22 produced by Th17 cells in Type 1 Diabetes. [Thesis]. University of Western Ontario; 2013. Available from: https://ir.lib.uwo.ca/etd/1598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

19. LAVOIE,TEGAN N. Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models.

Degree: MS, Medical Sciences - Medicine, 2011, University of Florida

 Sj?gren's syndrome (SjS) is a complex autoimmune disease which targets the exocrine glands resulting in xerostomia/ keratoconjunctivitis sicca. Presently, we examined the presence, source and… (more)

Subjects/Keywords: Cells; Cytokines; Diseases; Mice; Natural killer cells; Receptors; Saliva; Salivary glands; Sjogrens syndrome; Spleen; 22  – AEC1R1AEC2  – APOPTOSIS  – C57BL6  – HSG  – IL  – INTERLEUKIN  – LTI  – LYMPHOID  – MODEL  – MOUSE  – NK  – NKP46  – PROLIFERATION  – ROR  – RORGAMMAT  – SJOGREN  – SJOGRENS  – SYNDROME

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

N, L. (2011). Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models. (Masters Thesis). University of Florida. Retrieved from https://ufdc.ufl.edu/UFE0042955

Chicago Manual of Style (16th Edition):

N, LAVOIE,TEGAN. “Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models.” 2011. Masters Thesis, University of Florida. Accessed January 25, 2021. https://ufdc.ufl.edu/UFE0042955.

MLA Handbook (7th Edition):

N, LAVOIE,TEGAN. “Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models.” 2011. Web. 25 Jan 2021.

Vancouver:

N L. Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models. [Internet] [Masters thesis]. University of Florida; 2011. [cited 2021 Jan 25]. Available from: https://ufdc.ufl.edu/UFE0042955.

Council of Science Editors:

N L. Expression and Effects of Interleukin-22 in Human Sjogren's Syndrome and Mouse Models. [Masters Thesis]. University of Florida; 2011. Available from: https://ufdc.ufl.edu/UFE0042955

.