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You searched for subject:(interleukin 1 beta IL 1beta ). Showing records 1 – 30 of 35133 total matches.

[1] [2] [3] [4] [5] … [1172]

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Queens University

1. Simmatis, Leif. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .

Degree: Neuroscience Studies, 2015, Queens University

 The pannexin-1 (panx1) hemichannel is associated with the Nod-Like Receptor Protein-1 (NLRP1) inflammasome following MCAO in a mouse model of focal ischaemic stroke. We investigated… (more)

Subjects/Keywords: Hemichannel; IL-1beta; neuroinflammation; NLRP1; stroke; ischemia; pannexin-1; interleukin-1beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Simmatis, L. (2015). The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Thesis, Queens University. Accessed October 14, 2019. http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Simmatis, Leif. “The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia .” 2015. Web. 14 Oct 2019.

Vancouver:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Internet] [Thesis]. Queens University; 2015. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/1974/13868.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Simmatis L. The Pannexin-1 hemichannel and neuroinflammation in acute focal cerebral ischemia . [Thesis]. Queens University; 2015. Available from: http://hdl.handle.net/1974/13868

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

2. Zhang, Ning. Transcriptional regulation of PDGF receptor alpha in fibroblasts.

Degree: Centre for Vascular Research, 2011, University of New South Wales

 Wound healing is a process of stimulating cell growth and proliferation to achieve the result of repair. This process is controlled by cytokines and growth… (more)

Subjects/Keywords: PDGF receptor alpha; fibroblasts; interleukin 1 beta (IL-1beta); tumour necrosis factor alpha (TNF-alpha); receptor alpha (PDGF-R-alpha); wound healing

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APA (6th Edition):

Zhang, N. (2011). Transcriptional regulation of PDGF receptor alpha in fibroblasts. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Doctoral Dissertation, University of New South Wales. Accessed October 14, 2019. http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

MLA Handbook (7th Edition):

Zhang, Ning. “Transcriptional regulation of PDGF receptor alpha in fibroblasts.” 2011. Web. 14 Oct 2019.

Vancouver:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2019 Oct 14]. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true.

Council of Science Editors:

Zhang N. Transcriptional regulation of PDGF receptor alpha in fibroblasts. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51647 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10314/SOURCE02?view=true

3. Dumont, Adélie. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.

Degree: Docteur es, Physiologie, 2018, Bourgogne Franche-Comté

Selon une étude précédente, une limitation à l'efficacité anticancéreuse du 5-Fluorouracile (5-FU) repose sur la sécrétion d'IL-1β par des cellules myéloïdes immunosuppressives (MDSC). La libération… (more)

Subjects/Keywords: Mdsc; 5 fluorouracil; Dha; IL-1 beta; Jnk; Mdsc; 5-Fluorouracil; Dha; IL-1beta; Jnk; 612

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APA (6th Edition):

Dumont, A. (2018). L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. (Doctoral Dissertation). Bourgogne Franche-Comté. Retrieved from http://www.theses.fr/2018UBFCI013

Chicago Manual of Style (16th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Doctoral Dissertation, Bourgogne Franche-Comté. Accessed October 14, 2019. http://www.theses.fr/2018UBFCI013.

MLA Handbook (7th Edition):

Dumont, Adélie. “L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta.” 2018. Web. 14 Oct 2019.

Vancouver:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Internet] [Doctoral dissertation]. Bourgogne Franche-Comté; 2018. [cited 2019 Oct 14]. Available from: http://www.theses.fr/2018UBFCI013.

Council of Science Editors:

Dumont A. L'action ambivalente de l'agent anti-cancéreux 5-Fluorouracile sur les cellules myéloïdes immunosuppressives sous contrôle de l'acide docosahexaénoïque : Rôle de l'inflammasome NLRP3 et de la voie JNK dans la sécrétion de l'IL-1beta : The ambivalent action of the anti-cancer agent 5-Fluorouracil on myeloid derived suppressor cells under control of docosahexaenoic acid : Role of NLRP3 inflammasome and the JNK pathway in the secretion of IL-1beta. [Doctoral Dissertation]. Bourgogne Franche-Comté; 2018. Available from: http://www.theses.fr/2018UBFCI013


University of Adelaide

4. Liu, Liang. CNS immune signalling and drug addiction: the role of interleukin-1 beta.

Degree: 2011, University of Adelaide

 Opioid and alcohol dependencies are significant public health problems worldwide. The causes of drug dependence are complex, comprising both genetic and environmental factors. Recent evidence… (more)

Subjects/Keywords: alcohol dependence; CNS immune; Glia; interleukin-1 beta; IL-1B; IL-1Ra; IL-1RN; pharmacogenetics; proinflammatory cytokines; opioid dependence

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APA (6th Edition):

Liu, L. (2011). CNS immune signalling and drug addiction: the role of interleukin-1 beta. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/95622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Liang. “CNS immune signalling and drug addiction: the role of interleukin-1 beta.” 2011. Thesis, University of Adelaide. Accessed October 14, 2019. http://hdl.handle.net/2440/95622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Liang. “CNS immune signalling and drug addiction: the role of interleukin-1 beta.” 2011. Web. 14 Oct 2019.

Vancouver:

Liu L. CNS immune signalling and drug addiction: the role of interleukin-1 beta. [Internet] [Thesis]. University of Adelaide; 2011. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/2440/95622.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu L. CNS immune signalling and drug addiction: the role of interleukin-1 beta. [Thesis]. University of Adelaide; 2011. Available from: http://hdl.handle.net/2440/95622

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

5. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: PhD, 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: 573.8; IL-1 receptors, IL-1RAcPb, IL-1alpha, IL-1beta, cytokines, inflammation, IL-6, MAP kinase, Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 14, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 14 Oct 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 14]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-role-of-interleukin1-receptors-in-brain-cell-signalling(78c927ce-a654-4eac-b2d7-2ca4ca8c0fd4).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.529239


University of Manchester

6. Nguyen, Loan. The role of interleukin-1 receptors in brain cell signalling.

Degree: 2010, University of Manchester

IL-1α and IL-1β are two IL-1 agonists which signals at the same receptor complex composed of IL-1R1/IL-1RAcP. However, IL-1α and IL-1β exert differential actions. A… (more)

Subjects/Keywords: IL-1 receptors; IL-1RAcPb; IL-1alpha; IL-1beta; cytokines; inflammation; IL-6; MAP kinase; Src kinase

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APA (6th Edition):

Nguyen, L. (2010). The role of interleukin-1 receptors in brain cell signalling. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038

Chicago Manual of Style (16th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Doctoral Dissertation, University of Manchester. Accessed October 14, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

MLA Handbook (7th Edition):

Nguyen, Loan. “The role of interleukin-1 receptors in brain cell signalling.” 2010. Web. 14 Oct 2019.

Vancouver:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Internet] [Doctoral dissertation]. University of Manchester; 2010. [cited 2019 Oct 14]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038.

Council of Science Editors:

Nguyen L. The role of interleukin-1 receptors in brain cell signalling. [Doctoral Dissertation]. University of Manchester; 2010. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:90038


University of South Florida

7. Sampayo-Escobar, Viviana. osteopontin plays a pivotal role in in increasing severity of respiratory syncytial virus infection.

Degree: 2017, University of South Florida

 The molecular mechanisms underlying susceptibility to severe respiratory syncytial virus (RSV) infection remain poorly understood. Herein, we report on the role of osteopontin (OPN) in… (more)

Subjects/Keywords: Respiratory Syncytial Virus (RSV); Interleukin 1 beta (IL-1β); osteopontin (OPN); CD44; Antiviral response; innate immunity; Biology

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APA (6th Edition):

Sampayo-Escobar, V. (2017). osteopontin plays a pivotal role in in increasing severity of respiratory syncytial virus infection. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/6947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sampayo-Escobar, Viviana. “osteopontin plays a pivotal role in in increasing severity of respiratory syncytial virus infection.” 2017. Thesis, University of South Florida. Accessed October 14, 2019. https://scholarcommons.usf.edu/etd/6947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sampayo-Escobar, Viviana. “osteopontin plays a pivotal role in in increasing severity of respiratory syncytial virus infection.” 2017. Web. 14 Oct 2019.

Vancouver:

Sampayo-Escobar V. osteopontin plays a pivotal role in in increasing severity of respiratory syncytial virus infection. [Internet] [Thesis]. University of South Florida; 2017. [cited 2019 Oct 14]. Available from: https://scholarcommons.usf.edu/etd/6947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sampayo-Escobar V. osteopontin plays a pivotal role in in increasing severity of respiratory syncytial virus infection. [Thesis]. University of South Florida; 2017. Available from: https://scholarcommons.usf.edu/etd/6947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

8. Li, Chunsheng. MyD88: central relay station of Interleukin 1 signaling pathway.

Degree: PhD, Microbiology and Immunology, 2005, Vanderbilt University

 As inflammation is a major mechanism of disease, we investigated the signal transduction processes induced by the key inflammatory cytokine Interleukin (IL) 1 beta as… (more)

Subjects/Keywords: IL-1beta signaling pathway; MyD88; gene expression profiling; inflammation; nuclear import; IL-1 receptor accessary protein; Interleukin-1; Cellular signal transduction

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APA (6th Edition):

Li, C. (2005). MyD88: central relay station of Interleukin 1 signaling pathway. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;

Chicago Manual of Style (16th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Doctoral Dissertation, Vanderbilt University. Accessed October 14, 2019. http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

MLA Handbook (7th Edition):

Li, Chunsheng. “MyD88: central relay station of Interleukin 1 signaling pathway.” 2005. Web. 14 Oct 2019.

Vancouver:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Internet] [Doctoral dissertation]. Vanderbilt University; 2005. [cited 2019 Oct 14]. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;.

Council of Science Editors:

Li C. MyD88: central relay station of Interleukin 1 signaling pathway. [Doctoral Dissertation]. Vanderbilt University; 2005. Available from: http://etd.library.vanderbilt.edu/available/etd-12132005-122759/ ;


University of Vienna

9. Quemada Garrido, Lucia. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.

Degree: 2017, University of Vienna

Rasmussen Enzephalitis (RE) ist eine seltene progressive Form der Epilepsie mit unbekannter Ätiologie. Die Hauptcharakteristika sind unilaterale hemisphärische Entzündung, arzneimittelresistente fokale Epilepsie und progressive neurologischer… (more)

Subjects/Keywords: 44.45 Immunologie; 42.13 Molekularbiologie; 44.90 Neurologie; Rasmussen Enzephalitis / IL-1beta / IL-1beta Rezeptor / Caspase-1 / Epilepsie / Krampfanfall / Neuroinflammation / Mikroglia; Rasmussen’s encephalitis / IL-1beta / IL-1beta receptor / Caspase-1 / epilepsy / epileptic seizure / neuroinflammation / microglia

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APA (6th Edition):

Quemada Garrido, L. (2017). Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Thesis, University of Vienna. Accessed October 14, 2019. http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Quemada Garrido, Lucia. “Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis.” 2017. Web. 14 Oct 2019.

Vancouver:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Internet] [Thesis]. University of Vienna; 2017. [cited 2019 Oct 14]. Available from: http://othes.univie.ac.at/46783/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Quemada Garrido L. Analysis of Interleukin-1β, Interleukin-1β Receptor and Caspase-1 in Rasmussen’s Encephalitis. [Thesis]. University of Vienna; 2017. Available from: http://othes.univie.ac.at/46783/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Kansas State University

10. Liu, Ziyan. Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta.

Degree: MS, Department of Anatomy and Physiology, 2011, Kansas State University

 Transforming growth factor beta (TGF-β) and interleukin-1β (IL-1β) are both up-regulated in high grade gliomas and their elevated activities have been associated with prognosis in… (more)

Subjects/Keywords: stem; interleukin 1 beta; Biochemistry (0487)

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APA (6th Edition):

Liu, Z. (2011). Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta. (Masters Thesis). Kansas State University. Retrieved from http://hdl.handle.net/2097/8572

Chicago Manual of Style (16th Edition):

Liu, Ziyan. “Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta.” 2011. Masters Thesis, Kansas State University. Accessed October 14, 2019. http://hdl.handle.net/2097/8572.

MLA Handbook (7th Edition):

Liu, Ziyan. “Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta.” 2011. Web. 14 Oct 2019.

Vancouver:

Liu Z. Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta. [Internet] [Masters thesis]. Kansas State University; 2011. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/2097/8572.

Council of Science Editors:

Liu Z. Induction of glioma stem cells by interleukin-1beta and transforming growth factor-beta. [Masters Thesis]. Kansas State University; 2011. Available from: http://hdl.handle.net/2097/8572


ETH Zürich

11. Yilmaz, Vural. IL-1 In Dendritic Cell Activation and Effector T cell Differentiation.

Degree: 2018, ETH Zürich

Subjects/Keywords: IL-1 beta

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APA (6th Edition):

Yilmaz, V. (2018). IL-1 In Dendritic Cell Activation and Effector T cell Differentiation. (Doctoral Dissertation). ETH Zürich. Retrieved from http://hdl.handle.net/20.500.11850/251176

Chicago Manual of Style (16th Edition):

Yilmaz, Vural. “IL-1 In Dendritic Cell Activation and Effector T cell Differentiation.” 2018. Doctoral Dissertation, ETH Zürich. Accessed October 14, 2019. http://hdl.handle.net/20.500.11850/251176.

MLA Handbook (7th Edition):

Yilmaz, Vural. “IL-1 In Dendritic Cell Activation and Effector T cell Differentiation.” 2018. Web. 14 Oct 2019.

Vancouver:

Yilmaz V. IL-1 In Dendritic Cell Activation and Effector T cell Differentiation. [Internet] [Doctoral dissertation]. ETH Zürich; 2018. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/20.500.11850/251176.

Council of Science Editors:

Yilmaz V. IL-1 In Dendritic Cell Activation and Effector T cell Differentiation. [Doctoral Dissertation]. ETH Zürich; 2018. Available from: http://hdl.handle.net/20.500.11850/251176


University of Manchester

12. Summersgill, Holly. Understanding the role of zinc in IL-1β production.

Degree: PhD, 2013, University of Manchester

 Zinc is an essential biological trace element required for proper immune functioning. Zinc deficient individuals have been reported to suffer compromised immune responses and increased… (more)

Subjects/Keywords: 616; zinc; Interleukin-1beta

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APA (6th Edition):

Summersgill, H. (2013). Understanding the role of zinc in IL-1β production. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/understanding-the-role-of-zinc-in-il1-production(7505e99d-41c3-492b-9ffc-223411f8f1e6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570292

Chicago Manual of Style (16th Edition):

Summersgill, Holly. “Understanding the role of zinc in IL-1β production.” 2013. Doctoral Dissertation, University of Manchester. Accessed October 14, 2019. https://www.research.manchester.ac.uk/portal/en/theses/understanding-the-role-of-zinc-in-il1-production(7505e99d-41c3-492b-9ffc-223411f8f1e6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570292.

MLA Handbook (7th Edition):

Summersgill, Holly. “Understanding the role of zinc in IL-1β production.” 2013. Web. 14 Oct 2019.

Vancouver:

Summersgill H. Understanding the role of zinc in IL-1β production. [Internet] [Doctoral dissertation]. University of Manchester; 2013. [cited 2019 Oct 14]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/understanding-the-role-of-zinc-in-il1-production(7505e99d-41c3-492b-9ffc-223411f8f1e6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570292.

Council of Science Editors:

Summersgill H. Understanding the role of zinc in IL-1β production. [Doctoral Dissertation]. University of Manchester; 2013. Available from: https://www.research.manchester.ac.uk/portal/en/theses/understanding-the-role-of-zinc-in-il1-production(7505e99d-41c3-492b-9ffc-223411f8f1e6).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.570292

13. Αγγελετοπούλου, Ιωάννα. Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1.

Degree: 2015, University of Patras

 Η πρωτεΐνη Tip60 (Tat-interactive protein, 60 kDa) είναι μέλος της οικογένειας πρωτεϊνών MYST και αρχικά προσδιορίστηκε ως αλληλεπιδρώσα πρωτεΐνη με την HIV-1 ΤΑΤ πρωτεΐνη. Πολλά… (more)

Subjects/Keywords: Ιντερλευκίνη 2; 572.645; Interleukin-2 (IL-2); HIV-1; Tip60

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APA (6th Edition):

Αγγελετοπούλου, . (2015). Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/8699

Chicago Manual of Style (16th Edition):

Αγγελετοπούλου, Ιωάννα. “Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1.” 2015. Masters Thesis, University of Patras. Accessed October 14, 2019. http://hdl.handle.net/10889/8699.

MLA Handbook (7th Edition):

Αγγελετοπούλου, Ιωάννα. “Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1.” 2015. Web. 14 Oct 2019.

Vancouver:

Αγγελετοπούλου . Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1. [Internet] [Masters thesis]. University of Patras; 2015. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/10889/8699.

Council of Science Editors:

Αγγελετοπούλου . Η έκφραση της ακετυλοτρανσφεράσης Tip60 σε υποπληθυσμούς Τ λεμφοκυττάρων και ο ρόλος της στη μεταγραφή του γονιδίου της ιντερλευκίνης-2 και του ιού HIV-1. [Masters Thesis]. University of Patras; 2015. Available from: http://hdl.handle.net/10889/8699


University of Rochester

14. Ghosh, Simantini; O'Banion, M. Kerry. The Effect of Sustained Overexpression of Interleukin-1β on Pathology in Murine Models of Alzheimer’s disease and Tauopathy.

Degree: PhD, 2014, University of Rochester

 Alzheimer’s disease (AD) is the most common form of dementia in the elderly and is marked by extraneuronal beta Amyloid (Aβ) plaques and intraneuronal tangles… (more)

Subjects/Keywords: 3xTgAD; IL-1β; Interleukin; Microglia; Tau; JNPL3; COX-1; SC560; Cyclooxygenase

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APA (6th Edition):

Ghosh, Simantini; O'Banion, M. K. (2014). The Effect of Sustained Overexpression of Interleukin-1β on Pathology in Murine Models of Alzheimer’s disease and Tauopathy. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/28434

Chicago Manual of Style (16th Edition):

Ghosh, Simantini; O'Banion, M Kerry. “The Effect of Sustained Overexpression of Interleukin-1β on Pathology in Murine Models of Alzheimer’s disease and Tauopathy.” 2014. Doctoral Dissertation, University of Rochester. Accessed October 14, 2019. http://hdl.handle.net/1802/28434.

MLA Handbook (7th Edition):

Ghosh, Simantini; O'Banion, M Kerry. “The Effect of Sustained Overexpression of Interleukin-1β on Pathology in Murine Models of Alzheimer’s disease and Tauopathy.” 2014. Web. 14 Oct 2019.

Vancouver:

Ghosh, Simantini; O'Banion MK. The Effect of Sustained Overexpression of Interleukin-1β on Pathology in Murine Models of Alzheimer’s disease and Tauopathy. [Internet] [Doctoral dissertation]. University of Rochester; 2014. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/1802/28434.

Council of Science Editors:

Ghosh, Simantini; O'Banion MK. The Effect of Sustained Overexpression of Interleukin-1β on Pathology in Murine Models of Alzheimer’s disease and Tauopathy. [Doctoral Dissertation]. University of Rochester; 2014. Available from: http://hdl.handle.net/1802/28434


Kyoto University

15. Kanamori, Yohei. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .

Degree: 2018, Kyoto University

Subjects/Keywords: hepcidin; AP-1; activin B; IL-1beta; inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kanamori, Y. (2018). Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Thesis, Kyoto University. Accessed October 14, 2019. http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanamori, Yohei. “Transcriptional regulation of hepcidin by molecules mediating inflammatory responses .” 2018. Web. 14 Oct 2019.

Vancouver:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Internet] [Thesis]. Kyoto University; 2018. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/2433/232336.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanamori Y. Transcriptional regulation of hepcidin by molecules mediating inflammatory responses . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232336

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

16. Ward, Christie. Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis.

Degree: PhD, Veterinary Medicine, 2009, University of Minnesota

 Bovine pneumonic mannheimiosis (BPM), an acute fibrinonecrotic pleuropneumonia caused by Mannheimia (Pasteurella) haemolytica, remains a leading source of economic losses to North American beef and… (more)

Subjects/Keywords: Cattle; Interleukin-1 beta; Interleukin-8; Mannheimia Haemolytica; Pneumonia; Tumor Necrosis Factor-alpha; Veterinary Medicine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ward, C. (2009). Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/53837

Chicago Manual of Style (16th Edition):

Ward, Christie. “Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis.” 2009. Doctoral Dissertation, University of Minnesota. Accessed October 14, 2019. http://purl.umn.edu/53837.

MLA Handbook (7th Edition):

Ward, Christie. “Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis.” 2009. Web. 14 Oct 2019.

Vancouver:

Ward C. Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis. [Internet] [Doctoral dissertation]. University of Minnesota; 2009. [cited 2019 Oct 14]. Available from: http://purl.umn.edu/53837.

Council of Science Editors:

Ward C. Pulmonary expression of inflammatory cytokines in experimental Bovine pneumonic mannheimiosis. [Doctoral Dissertation]. University of Minnesota; 2009. Available from: http://purl.umn.edu/53837


Universidade Federal de Santa Maria

17. Maria Fernanda Manica Rizzi Cattani. ASSOCIAÇÃO ENTRE O POLIMORFISMO +3954 DO GENE DA INTERLEUCINA-1 BETA, OBESIDADE, LDL-OXIDADO E SEU POTENCIAL EFEITO LIPOTÓXICO.

Degree: 2012, Universidade Federal de Santa Maria

A obesidade é um inflamação sistêmica de baixo grau associado a elevada liberação de moléculas pró-inflamatórias pelo tecido adiposo. O aumento da adiposidade resulta em… (more)

Subjects/Keywords: LDLoxidado; Interleucina - 1beta; obesidade; CIENCIAS BIOLOGICAS; obesity; Interleukin-1beta; Oxidized-LDL

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cattani, M. F. M. R. (2012). ASSOCIAÇÃO ENTRE O POLIMORFISMO +3954 DO GENE DA INTERLEUCINA-1 BETA, OBESIDADE, LDL-OXIDADO E SEU POTENCIAL EFEITO LIPOTÓXICO. (Thesis). Universidade Federal de Santa Maria. Retrieved from http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cattani, Maria Fernanda Manica Rizzi. “ASSOCIAÇÃO ENTRE O POLIMORFISMO +3954 DO GENE DA INTERLEUCINA-1 BETA, OBESIDADE, LDL-OXIDADO E SEU POTENCIAL EFEITO LIPOTÓXICO.” 2012. Thesis, Universidade Federal de Santa Maria. Accessed October 14, 2019. http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cattani, Maria Fernanda Manica Rizzi. “ASSOCIAÇÃO ENTRE O POLIMORFISMO +3954 DO GENE DA INTERLEUCINA-1 BETA, OBESIDADE, LDL-OXIDADO E SEU POTENCIAL EFEITO LIPOTÓXICO.” 2012. Web. 14 Oct 2019.

Vancouver:

Cattani MFMR. ASSOCIAÇÃO ENTRE O POLIMORFISMO +3954 DO GENE DA INTERLEUCINA-1 BETA, OBESIDADE, LDL-OXIDADO E SEU POTENCIAL EFEITO LIPOTÓXICO. [Internet] [Thesis]. Universidade Federal de Santa Maria; 2012. [cited 2019 Oct 14]. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cattani MFMR. ASSOCIAÇÃO ENTRE O POLIMORFISMO +3954 DO GENE DA INTERLEUCINA-1 BETA, OBESIDADE, LDL-OXIDADO E SEU POTENCIAL EFEITO LIPOTÓXICO. [Thesis]. Universidade Federal de Santa Maria; 2012. Available from: http://coralx.ufsm.br/tede/tde_busca/arquivo.php?codArquivo=4461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

18. Koh, Gavin Christian Kia Wee. The effect of glibenclamide on the pathogenesis of melioidosis.

Degree: PhD, 2012, University of Cambridge

 Melioidosis is an important cause of community-acquired sepsis, endemic to Southeast Asia and Northern Australia. Melioidosis is caused by the soil saprophyte, Burkholderia pseudomallei, a… (more)

Subjects/Keywords: 610; Glibenclamide; Melioidosis; Burkholderia pseudomallei; Diabetes mellitus; Interleukin-1 beta

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Koh, G. C. K. W. (2012). The effect of glibenclamide on the pathogenesis of melioidosis. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/242187 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555209

Chicago Manual of Style (16th Edition):

Koh, Gavin Christian Kia Wee. “The effect of glibenclamide on the pathogenesis of melioidosis.” 2012. Doctoral Dissertation, University of Cambridge. Accessed October 14, 2019. https://www.repository.cam.ac.uk/handle/1810/242187 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555209.

MLA Handbook (7th Edition):

Koh, Gavin Christian Kia Wee. “The effect of glibenclamide on the pathogenesis of melioidosis.” 2012. Web. 14 Oct 2019.

Vancouver:

Koh GCKW. The effect of glibenclamide on the pathogenesis of melioidosis. [Internet] [Doctoral dissertation]. University of Cambridge; 2012. [cited 2019 Oct 14]. Available from: https://www.repository.cam.ac.uk/handle/1810/242187 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555209.

Council of Science Editors:

Koh GCKW. The effect of glibenclamide on the pathogenesis of melioidosis. [Doctoral Dissertation]. University of Cambridge; 2012. Available from: https://www.repository.cam.ac.uk/handle/1810/242187 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.555209

19. Li, Yijing. Interleukin-1 beta promotes epithelial-mesenchymal transition and a stem cell phenotype of colon cancer cells via Zeb1/2.

Degree: MS, Department of Anatomy and Physiology, 2012, Kansas State University

Interleukin-1 beta (IL-1β) is an important mediator of inflammatory response, and the elevated expression of IL-1β is correlated with tumor growth and metastasis. Epithelial-mesenchymal transition… (more)

Subjects/Keywords: Interleukin-1 beta; colon cancer; Biology (0306); Veterinary Medicine (0778)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, Y. (2012). Interleukin-1 beta promotes epithelial-mesenchymal transition and a stem cell phenotype of colon cancer cells via Zeb1/2. (Masters Thesis). Kansas State University. Retrieved from http://hdl.handle.net/2097/13730

Chicago Manual of Style (16th Edition):

Li, Yijing. “Interleukin-1 beta promotes epithelial-mesenchymal transition and a stem cell phenotype of colon cancer cells via Zeb1/2.” 2012. Masters Thesis, Kansas State University. Accessed October 14, 2019. http://hdl.handle.net/2097/13730.

MLA Handbook (7th Edition):

Li, Yijing. “Interleukin-1 beta promotes epithelial-mesenchymal transition and a stem cell phenotype of colon cancer cells via Zeb1/2.” 2012. Web. 14 Oct 2019.

Vancouver:

Li Y. Interleukin-1 beta promotes epithelial-mesenchymal transition and a stem cell phenotype of colon cancer cells via Zeb1/2. [Internet] [Masters thesis]. Kansas State University; 2012. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/2097/13730.

Council of Science Editors:

Li Y. Interleukin-1 beta promotes epithelial-mesenchymal transition and a stem cell phenotype of colon cancer cells via Zeb1/2. [Masters Thesis]. Kansas State University; 2012. Available from: http://hdl.handle.net/2097/13730


University of Toronto

20. Rajkumar, Andrew. The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells.

Degree: 2018, University of Toronto

Intimal dendritic-like cells (DCs) reside in regions of the aorta predisposed to atherosclerosis. LPS and interleukin-1 beta (IL-1β) injections into C57BL/6 mice result in decreased… (more)

Subjects/Keywords: Dendritic Cell; Interleukin-1 Beta; Intima; Lipopolysaccharide; Reverse Transendothelial Migration; 0982

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rajkumar, A. (2018). The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91394

Chicago Manual of Style (16th Edition):

Rajkumar, Andrew. “The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells.” 2018. Masters Thesis, University of Toronto. Accessed October 14, 2019. http://hdl.handle.net/1807/91394.

MLA Handbook (7th Edition):

Rajkumar, Andrew. “The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells.” 2018. Web. 14 Oct 2019.

Vancouver:

Rajkumar A. The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/1807/91394.

Council of Science Editors:

Rajkumar A. The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91394


Georgia State University

21. Chesnokov, Anton P, Mr. Modulation of NALP3 Inflammasome Genes by Estrogen.

Degree: MS, Biology, 2011, Georgia State University

  Immunity is known to be sexually dimorphic. This dimorphism may be attributed to the action of different hormones. Aluminum is a component of several… (more)

Subjects/Keywords: Nalp3 inflammasome; Estrogen; IL-1beta; Aluminum hydroxide

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chesnokov, Anton P, M. (2011). Modulation of NALP3 Inflammasome Genes by Estrogen. (Thesis). Georgia State University. Retrieved from https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Thesis, Georgia State University. Accessed October 14, 2019. https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chesnokov, Anton P, Mr. “Modulation of NALP3 Inflammasome Genes by Estrogen.” 2011. Web. 14 Oct 2019.

Vancouver:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Internet] [Thesis]. Georgia State University; 2011. [cited 2019 Oct 14]. Available from: https://scholarworks.gsu.edu/biology_theses/33.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chesnokov, Anton P M. Modulation of NALP3 Inflammasome Genes by Estrogen. [Thesis]. Georgia State University; 2011. Available from: https://scholarworks.gsu.edu/biology_theses/33

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

22. Kuiper, J.J.W. Reactive Oxygen Species in Inflammasome Activation.

Degree: 2009, Universiteit Utrecht

 The fundamental role of the innate immune system is to form the first barrier in bacterial and viral defence. It is based upon the recognition… (more)

Subjects/Keywords: Geneeskunde; NOD-like receptor, P2x7-receptor, NLRP3, Reactive Oxygen Species, ROS, Caspase-1, IL-1beta, IL-18, Inflammasome, ERK1/2, Cryopyrinopathies, CAPS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kuiper, J. J. W. (2009). Reactive Oxygen Species in Inflammasome Activation. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/35211

Chicago Manual of Style (16th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Masters Thesis, Universiteit Utrecht. Accessed October 14, 2019. http://dspace.library.uu.nl:8080/handle/1874/35211.

MLA Handbook (7th Edition):

Kuiper, J J W. “Reactive Oxygen Species in Inflammasome Activation.” 2009. Web. 14 Oct 2019.

Vancouver:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Internet] [Masters thesis]. Universiteit Utrecht; 2009. [cited 2019 Oct 14]. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211.

Council of Science Editors:

Kuiper JJW. Reactive Oxygen Species in Inflammasome Activation. [Masters Thesis]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/35211


University of Toronto

23. Cho, Lindsay Kristin. The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis.

Degree: 2016, University of Toronto

Previous studies have demonstrated mitochondrial DNA (mtDNA) activates Toll-like receptor-9 signaling, and may activate inflammasomes leading to the release of pro-inflammatory cytokine IL-1b. Chronic inflammation… (more)

Subjects/Keywords: IL-1 beta; Juvenile Idiopathic Arthritis; macrophages; mitochondrial DNA; 0982

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cho, L. K. (2016). The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/74567

Chicago Manual of Style (16th Edition):

Cho, Lindsay Kristin. “The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis.” 2016. Masters Thesis, University of Toronto. Accessed October 14, 2019. http://hdl.handle.net/1807/74567.

MLA Handbook (7th Edition):

Cho, Lindsay Kristin. “The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis.” 2016. Web. 14 Oct 2019.

Vancouver:

Cho LK. The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/1807/74567.

Council of Science Editors:

Cho LK. The Role of Circulating Mitochondrial DNA in Juvenile Idiopathic Arthritis. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/74567

24. Gómez Ochoa, Pablo. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.

Degree: 2011, Universidad de Zaragoza

 El diseño de este trabajo intenta establecer las condiciones de cultivo y la metodología para la evaluación del grupo de las Stem Cells adultas y… (more)

Subjects/Keywords: Campos electromagnéticos Pulsados; Stem Cells; TNF-alfa; IL-1beta; IL-10; PEMF; Stem Cells; TNF-alfa; IL-1beta; IL-10

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APA (6th Edition):

Gómez Ochoa, P. (2011). Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. (Thesis). Universidad de Zaragoza. Retrieved from http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Thesis, Universidad de Zaragoza. Accessed October 14, 2019. http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gómez Ochoa, Pablo. “Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes.” 2011. Web. 14 Oct 2019.

Vancouver:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Internet] [Thesis]. Universidad de Zaragoza; 2011. [cited 2019 Oct 14]. Available from: http://zaguan.unizar.es/record/4497.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gómez Ochoa P. Efectos de los campos electromagnéticos pulsados (PEMF) en el cultivo de las stem cells circulantes. [Thesis]. Universidad de Zaragoza; 2011. Available from: http://zaguan.unizar.es/record/4497

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

25. Lavin, Desiree N. Immunological and behavioral consequences of high-fat diet feeding in mice.

Degree: MS, 0191, 2010, University of Illinois – Urbana-Champaign

 Obesity is a harmful and costly condition which continues to increase in prevalence. Co-morbidities accompanying this disease include brain-based disorders that impact cognition and mood.… (more)

Subjects/Keywords: mice; high-fat diet; saccharin preference; leptin; Interleukin 1 receptor antagonist (IL-1RA)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lavin, D. N. (2010). Immunological and behavioral consequences of high-fat diet feeding in mice. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/15982

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lavin, Desiree N. “Immunological and behavioral consequences of high-fat diet feeding in mice.” 2010. Thesis, University of Illinois – Urbana-Champaign. Accessed October 14, 2019. http://hdl.handle.net/2142/15982.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lavin, Desiree N. “Immunological and behavioral consequences of high-fat diet feeding in mice.” 2010. Web. 14 Oct 2019.

Vancouver:

Lavin DN. Immunological and behavioral consequences of high-fat diet feeding in mice. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2010. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/2142/15982.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lavin DN. Immunological and behavioral consequences of high-fat diet feeding in mice. [Thesis]. University of Illinois – Urbana-Champaign; 2010. Available from: http://hdl.handle.net/2142/15982

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Connecticut

26. Kwarko, Trisha K. Serum Amyloid A3 (Saa3) Regulates IL-1β Release from Pre-Osteoclasts.

Degree: MS, Biomedical Science, 2016, University of Connecticut

Subjects/Keywords: Bone remodeling; osteoclast; RANKL; serum amyloid A (SAA); interleukin-1 beta (IL-1β); Nlrp3 inflammasome; cyclooxygenase 2 (Cox2); prostaglandin E2 (PGE2)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kwarko, T. K. (2016). Serum Amyloid A3 (Saa3) Regulates IL-1β Release from Pre-Osteoclasts. (Masters Thesis). University of Connecticut. Retrieved from https://opencommons.uconn.edu/gs_theses/897

Chicago Manual of Style (16th Edition):

Kwarko, Trisha K. “Serum Amyloid A3 (Saa3) Regulates IL-1β Release from Pre-Osteoclasts.” 2016. Masters Thesis, University of Connecticut. Accessed October 14, 2019. https://opencommons.uconn.edu/gs_theses/897.

MLA Handbook (7th Edition):

Kwarko, Trisha K. “Serum Amyloid A3 (Saa3) Regulates IL-1β Release from Pre-Osteoclasts.” 2016. Web. 14 Oct 2019.

Vancouver:

Kwarko TK. Serum Amyloid A3 (Saa3) Regulates IL-1β Release from Pre-Osteoclasts. [Internet] [Masters thesis]. University of Connecticut; 2016. [cited 2019 Oct 14]. Available from: https://opencommons.uconn.edu/gs_theses/897.

Council of Science Editors:

Kwarko TK. Serum Amyloid A3 (Saa3) Regulates IL-1β Release from Pre-Osteoclasts. [Masters Thesis]. University of Connecticut; 2016. Available from: https://opencommons.uconn.edu/gs_theses/897


Case Western Reserve University

27. Trueblood, Katherine Eileen. Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy.

Degree: PhD, Physiology and Biophysics, 2011, Case Western Reserve University

 Diabetic retinopathy, a common complication of diabetes, is the leading cause of blindness in adults in the United States. It is known that high blood… (more)

Subjects/Keywords: Physiology; diabetic retinopathy; inflammation; caspase-1; interleukin-1-beta; NLRP3; purinergic signaling

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APA (6th Edition):

Trueblood, K. E. (2011). Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450

Chicago Manual of Style (16th Edition):

Trueblood, Katherine Eileen. “Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy.” 2011. Doctoral Dissertation, Case Western Reserve University. Accessed October 14, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450.

MLA Handbook (7th Edition):

Trueblood, Katherine Eileen. “Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy.” 2011. Web. 14 Oct 2019.

Vancouver:

Trueblood KE. Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2011. [cited 2019 Oct 14]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450.

Council of Science Editors:

Trueblood KE. Caspase-1-Dependent Inflammatory Signaling in Retinal Müller Cells During the Development of Diabetic Retinopathy. [Doctoral Dissertation]. Case Western Reserve University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1322483450


Universitat Autònoma de Barcelona

28. Bothe González, Carolina. Papel de la IL-1 en el riesgo de aparción de metástasis a distancia en pacientes con carcinoma escamoso de cabeza y cuello.

Degree: Departament de Cirurgia, 2016, Universitat Autònoma de Barcelona

 The development of distant metastasis in patients with head and neck squamous cell carcinoma (HNSSC) that have achieved loco-regional control, is one of the main… (more)

Subjects/Keywords: IL-1; Interleuquina-1; Interleukin-1; Metástasis; Metàstasis; Metastasis; Carcinoma escamoso; Carcinoma escatós; Squamous cell carcinoma; Ciències de la Salut; 616.2

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APA (6th Edition):

Bothe González, C. (2016). Papel de la IL-1 en el riesgo de aparción de metástasis a distancia en pacientes con carcinoma escamoso de cabeza y cuello. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/384855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bothe González, Carolina. “Papel de la IL-1 en el riesgo de aparción de metástasis a distancia en pacientes con carcinoma escamoso de cabeza y cuello.” 2016. Thesis, Universitat Autònoma de Barcelona. Accessed October 14, 2019. http://hdl.handle.net/10803/384855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bothe González, Carolina. “Papel de la IL-1 en el riesgo de aparción de metástasis a distancia en pacientes con carcinoma escamoso de cabeza y cuello.” 2016. Web. 14 Oct 2019.

Vancouver:

Bothe González C. Papel de la IL-1 en el riesgo de aparción de metástasis a distancia en pacientes con carcinoma escamoso de cabeza y cuello. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2016. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/10803/384855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bothe González C. Papel de la IL-1 en el riesgo de aparción de metástasis a distancia en pacientes con carcinoma escamoso de cabeza y cuello. [Thesis]. Universitat Autònoma de Barcelona; 2016. Available from: http://hdl.handle.net/10803/384855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

29. Che Mat, Nor Fazila. Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection .

Degree: Microbiology and Immunology, 2011, Queens University

 Interluekin-23 (IL-23) is a pro-inflammatory cytokine critical to the regulation of innate and adaptive immune responses. The main role for this cytokine is in the… (more)

Subjects/Keywords: Virus; Interleukin-23 (IL-23)

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APA (6th Edition):

Che Mat, N. F. (2011). Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6666

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Che Mat, Nor Fazila. “Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection .” 2011. Thesis, Queens University. Accessed October 14, 2019. http://hdl.handle.net/1974/6666.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Che Mat, Nor Fazila. “Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection .” 2011. Web. 14 Oct 2019.

Vancouver:

Che Mat NF. Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection . [Internet] [Thesis]. Queens University; 2011. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/1974/6666.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Che Mat NF. Impact of RNA Viruses on the Regulation of IL-23 in Mouse and Human Models of Infection . [Thesis]. Queens University; 2011. Available from: http://hdl.handle.net/1974/6666

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

30. Joesting, Jennifer J. The effect of fasting on anti-inflammatory mediators, IL-1RA and IL-1R2, and their role in resistance to sickness behaviors in mice.

Degree: MS, 0002, 2013, University of Illinois – Urbana-Champaign

 Dietary regimens involving fasting have long been linked to beneficial health outcomes including; reduction in heart disease, diabetes, improved mood and cognition, and resistance to… (more)

Subjects/Keywords: Fasting; Dietary Restriction; Anti-inflammation; Interleukin 1 Receptor Type 2 (IL-1R2); Interleukin 1 Receptor Antagonist (IL-1RA); sickness behavior; IL-1 decoy receptor; Free Fatty Acids (FFA); Non-Esterified Fatty Acids (NEFA)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Joesting, J. J. (2013). The effect of fasting on anti-inflammatory mediators, IL-1RA and IL-1R2, and their role in resistance to sickness behaviors in mice. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/44463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Joesting, Jennifer J. “The effect of fasting on anti-inflammatory mediators, IL-1RA and IL-1R2, and their role in resistance to sickness behaviors in mice.” 2013. Thesis, University of Illinois – Urbana-Champaign. Accessed October 14, 2019. http://hdl.handle.net/2142/44463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Joesting, Jennifer J. “The effect of fasting on anti-inflammatory mediators, IL-1RA and IL-1R2, and their role in resistance to sickness behaviors in mice.” 2013. Web. 14 Oct 2019.

Vancouver:

Joesting JJ. The effect of fasting on anti-inflammatory mediators, IL-1RA and IL-1R2, and their role in resistance to sickness behaviors in mice. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2013. [cited 2019 Oct 14]. Available from: http://hdl.handle.net/2142/44463.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Joesting JJ. The effect of fasting on anti-inflammatory mediators, IL-1RA and IL-1R2, and their role in resistance to sickness behaviors in mice. [Thesis]. University of Illinois – Urbana-Champaign; 2013. Available from: http://hdl.handle.net/2142/44463

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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