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You searched for subject:(insulin biosynthesis). Showing records 1 – 7 of 7 total matches.

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Penn State University

1. Wang, Rong. PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells.

Degree: 2014, Penn State University

Insulin synthesis and secretion, as well as cell proliferation are under tight regulation in pancreatic β-cells to maintain glucose homeostasis. Dysfunction in any of these… (more)

Subjects/Keywords: PERK eIF2alpha kinase; Ca2+ dynamics; insulin secretion; insulin biosynthesis; diabetes; ER stress; beta cell proliferation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, R. (2014). PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/21417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Rong. “PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells.” 2014. Thesis, Penn State University. Accessed April 14, 2021. https://submit-etda.libraries.psu.edu/catalog/21417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Rong. “PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells.” 2014. Web. 14 Apr 2021.

Vancouver:

Wang R. PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Apr 14]. Available from: https://submit-etda.libraries.psu.edu/catalog/21417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang R. PERK eIF2alpha kinase regulates cell proliferation, insulin synthesis and secretion in pancreatic beta cells. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/21417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

2. Govender, Kamini. Sub/supercritical fluid chromatography purification of biologics.

Degree: 2020, University of KwaZulu-Natal

 Peptide and protein drugs are highly versatile with numerous therapeutic properties such as anti-cancer, anti-diabetic, anti-hypertensive, and anti-microbial; which are therefore ideal candidates for the… (more)

Subjects/Keywords: Biosynthesis of human insulin - drug development.; Supercritical fluid chromatography.; Biologics.; Diabetes.

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APA (6th Edition):

Govender, K. (2020). Sub/supercritical fluid chromatography purification of biologics. (Thesis). University of KwaZulu-Natal. Retrieved from https://researchspace.ukzn.ac.za/handle/10413/19002

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Govender, Kamini. “Sub/supercritical fluid chromatography purification of biologics.” 2020. Thesis, University of KwaZulu-Natal. Accessed April 14, 2021. https://researchspace.ukzn.ac.za/handle/10413/19002.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Govender, Kamini. “Sub/supercritical fluid chromatography purification of biologics.” 2020. Web. 14 Apr 2021.

Vancouver:

Govender K. Sub/supercritical fluid chromatography purification of biologics. [Internet] [Thesis]. University of KwaZulu-Natal; 2020. [cited 2021 Apr 14]. Available from: https://researchspace.ukzn.ac.za/handle/10413/19002.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Govender K. Sub/supercritical fluid chromatography purification of biologics. [Thesis]. University of KwaZulu-Natal; 2020. Available from: https://researchspace.ukzn.ac.za/handle/10413/19002

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Technology, Sydney

3. Kasz, Robert. Investigating DHCR24 as a protector against cellular stress : more than just a cholesterol-synthesising enzyme.

Degree: 2017, University of Technology, Sydney

 Atherosclerosis and insulin resistance are globally prevalent metabolic diseases, primarily driven by endothelial and hepatic inflammation, respectively. High density lipoprotein (HDL) reduces the inflammation in… (more)

Subjects/Keywords: Atherosclerosis.; Insulin resistance.; Cholesterol biosynthesis enzyme DHCR24; High density lipoprotein (HDL); HCAECs and HuH7 cells.

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APA (6th Edition):

Kasz, R. (2017). Investigating DHCR24 as a protector against cellular stress : more than just a cholesterol-synthesising enzyme. (Thesis). University of Technology, Sydney. Retrieved from http://hdl.handle.net/10453/90267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kasz, Robert. “Investigating DHCR24 as a protector against cellular stress : more than just a cholesterol-synthesising enzyme.” 2017. Thesis, University of Technology, Sydney. Accessed April 14, 2021. http://hdl.handle.net/10453/90267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kasz, Robert. “Investigating DHCR24 as a protector against cellular stress : more than just a cholesterol-synthesising enzyme.” 2017. Web. 14 Apr 2021.

Vancouver:

Kasz R. Investigating DHCR24 as a protector against cellular stress : more than just a cholesterol-synthesising enzyme. [Internet] [Thesis]. University of Technology, Sydney; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10453/90267.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kasz R. Investigating DHCR24 as a protector against cellular stress : more than just a cholesterol-synthesising enzyme. [Thesis]. University of Technology, Sydney; 2017. Available from: http://hdl.handle.net/10453/90267

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Indian Institute of Science

4. Tripathy, Sudeshna. Elucidation of 17β-Estradiol (E2) Role in the Regulation of Corpus Luteum Function in Mammals : Analysis of IGFBP5 Expression during Ea-mediated Actions.

Degree: PhD, Faculty of Science, 2017, Indian Institute of Science

 Corpus luteum is a transient endocrine structure formed from the ruptured ovarian follicle. Its main function is to secrete P4, a pro-gestational hormone, essential for… (more)

Subjects/Keywords: Corpus Luteum; Estradiol (E2) Signalling; Luteal Steroidogenesis; Estradiol (E2) Biosynthesis; Insulin-Like Growth Factor Binding Protein (IGFBP); Luteolysis; Luteal Transcriptome; Prostaglandin F2-alpha Treatment; 17β-estradiol (E2); IGFBP5; Endocrinology

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APA (6th Edition):

Tripathy, S. (2017). Elucidation of 17β-Estradiol (E2) Role in the Regulation of Corpus Luteum Function in Mammals : Analysis of IGFBP5 Expression during Ea-mediated Actions. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/2904

Chicago Manual of Style (16th Edition):

Tripathy, Sudeshna. “Elucidation of 17β-Estradiol (E2) Role in the Regulation of Corpus Luteum Function in Mammals : Analysis of IGFBP5 Expression during Ea-mediated Actions.” 2017. Doctoral Dissertation, Indian Institute of Science. Accessed April 14, 2021. http://etd.iisc.ac.in/handle/2005/2904.

MLA Handbook (7th Edition):

Tripathy, Sudeshna. “Elucidation of 17β-Estradiol (E2) Role in the Regulation of Corpus Luteum Function in Mammals : Analysis of IGFBP5 Expression during Ea-mediated Actions.” 2017. Web. 14 Apr 2021.

Vancouver:

Tripathy S. Elucidation of 17β-Estradiol (E2) Role in the Regulation of Corpus Luteum Function in Mammals : Analysis of IGFBP5 Expression during Ea-mediated Actions. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2017. [cited 2021 Apr 14]. Available from: http://etd.iisc.ac.in/handle/2005/2904.

Council of Science Editors:

Tripathy S. Elucidation of 17β-Estradiol (E2) Role in the Regulation of Corpus Luteum Function in Mammals : Analysis of IGFBP5 Expression during Ea-mediated Actions. [Doctoral Dissertation]. Indian Institute of Science; 2017. Available from: http://etd.iisc.ac.in/handle/2005/2904

5. Μπάρτζης, Μιχαήλ. Μοριακή μελέτη του συνδρόμου των πολυκυστικών ωοθηκών.

Degree: 2001, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Subjects/Keywords: Σύνδρομο πολυκυστικών ωοθηκών; Βιοσύνθεση ανδρογόνων; Ινσουλίνη, Αντίσταση στην; Ένζυμο P450c17a; Γονίδιο CYP17; Ένζυμο P450scc; Γονίδιο CYP11α; Μικροδορυφορικός πολυμορφισμός; Polycystic ovary syndrome (PCOS); Androgen biosynthesis; Insulin resistance; Enzyme P450c17a; Gene CYP17; Enzyme P450scc; Gene CYP11a; Microsatellite polymorphism

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APA (6th Edition):

Μπάρτζης, . . (2001). Μοριακή μελέτη του συνδρόμου των πολυκυστικών ωοθηκών. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/22871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Μπάρτζης, Μιχαήλ. “Μοριακή μελέτη του συνδρόμου των πολυκυστικών ωοθηκών.” 2001. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 14, 2021. http://hdl.handle.net/10442/hedi/22871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Μπάρτζης, Μιχαήλ. “Μοριακή μελέτη του συνδρόμου των πολυκυστικών ωοθηκών.” 2001. Web. 14 Apr 2021.

Vancouver:

Μπάρτζης . Μοριακή μελέτη του συνδρόμου των πολυκυστικών ωοθηκών. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2001. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10442/hedi/22871.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Μπάρτζης . Μοριακή μελέτη του συνδρόμου των πολυκυστικών ωοθηκών. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2001. Available from: http://hdl.handle.net/10442/hedi/22871

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

6. Lai, Elida Wing Shan. Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis.

Degree: 2008, University of Toronto

Chronic hyperlipidemia (lipotoxicity) and hyperglycemia (glucotoxicity) have recently been shown to induce Endoplasmic Reticulum (ER) stress, which may contribute to pancreatic beta-cell dysfunction in type… (more)

Subjects/Keywords: pancreatic beta-cells; type 2 diabetes; ER stress; apoptosis; pancreatic beta-cell dysfunction; free fatty acids; lipotoxicity; palmitate; high glucose; hyperglycemia; glucotoxicity; insulin biosynthesis; unfolded protein response; chaperone; GRP78; PDI; INS-1; MIN6; human islets; stable cell line; 0379

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APA (6th Edition):

Lai, E. W. S. (2008). Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/11149

Chicago Manual of Style (16th Edition):

Lai, Elida Wing Shan. “Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis.” 2008. Masters Thesis, University of Toronto. Accessed April 14, 2021. http://hdl.handle.net/1807/11149.

MLA Handbook (7th Edition):

Lai, Elida Wing Shan. “Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis.” 2008. Web. 14 Apr 2021.

Vancouver:

Lai EWS. Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis. [Internet] [Masters thesis]. University of Toronto; 2008. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1807/11149.

Council of Science Editors:

Lai EWS. Diabetes and Endoplasmic Reticulum Stress in Pancreatic beta-cells: Effects on Insulin Biosynthesis and beta-cell Apoptosis. [Masters Thesis]. University of Toronto; 2008. Available from: http://hdl.handle.net/1807/11149

7. Penque, Brent A. Mechanisms of hexosamine-induced cholesterol accumulation and therapeutic actions of chromium.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Excess caloric intake and/or obesity currently remain the largest predisposing risk factors for the development of type 2 diabetes. Discerning… (more)

Subjects/Keywords: Chromium  – Therapeutic use  – Research; Hexosamines  – Synthesis; Amino sugars; Diabetes  – Pathophysiology; Obesity; Insulin resistance  – Physiological effect; Biosynthesis; Adipose tissues  – Pathophysiology; Glucosamine; Cholesterol  – Metabolism; Actin; Cardiovascular system  – Diseases; Lipid membranes  – Metabolism  – Research

…discussion. I.B.3. Hexosamine Biosynthesis and Insulin Resistance Dating to the 1980’s, it was… …1 I.A. Insulin-Mediated Glucose Regulation… …4 I.B. Mechanisms of Insulin Resistance… …and Insulin Resistance via Transcriptional Activation of Sp1 ..... 45 II.B. Chromium… …Accumulation and Insulin Resistance ............................................................. 78… 

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APA (6th Edition):

Penque, B. A. (2014). Mechanisms of hexosamine-induced cholesterol accumulation and therapeutic actions of chromium. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/3805

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Penque, Brent A. “Mechanisms of hexosamine-induced cholesterol accumulation and therapeutic actions of chromium.” 2014. Thesis, IUPUI. Accessed April 14, 2021. http://hdl.handle.net/1805/3805.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Penque, Brent A. “Mechanisms of hexosamine-induced cholesterol accumulation and therapeutic actions of chromium.” 2014. Web. 14 Apr 2021.

Vancouver:

Penque BA. Mechanisms of hexosamine-induced cholesterol accumulation and therapeutic actions of chromium. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1805/3805.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Penque BA. Mechanisms of hexosamine-induced cholesterol accumulation and therapeutic actions of chromium. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/3805

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.