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You searched for subject:(inflammation). Showing records 1 – 30 of 1393 total matches.

[1] [2] [3] [4] [5] … [47]

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1. Millet, Patrick. INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES.

Degree: 2015, Wake Forest University

 Sepsis is serious medical condition which kills millions of people worldwide each year. In the United States, severe sepsis has a mortality rate of 20-30%,… (more)

Subjects/Keywords: Inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Millet, P. (2015). INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/57421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Millet, Patrick. “INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES.” 2015. Thesis, Wake Forest University. Accessed April 04, 2020. http://hdl.handle.net/10339/57421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Millet, Patrick. “INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES.” 2015. Web. 04 Apr 2020.

Vancouver:

Millet P. INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES. [Internet] [Thesis]. Wake Forest University; 2015. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10339/57421.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Millet P. INTERDEPENDENT REGULATION OF METABOLISM AND INFLAMMATION IN HUMAN MONOCYTES. [Thesis]. Wake Forest University; 2015. Available from: http://hdl.handle.net/10339/57421

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

2. Killi, Ravikumar. Assessment of the ability of two differently constructed equine viral vaccines to induce systemic inflammation.

Degree: MS, Infectious Diseases, 2008, University of Georgia

 Equine vaccines generally induce good serum antibody titers, and help control infectious diseases. Vaccine must induce an inflammatory response to initiate the adaptive immune response.… (more)

Subjects/Keywords: Systemic inflammation

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APA (6th Edition):

Killi, R. (2008). Assessment of the ability of two differently constructed equine viral vaccines to induce systemic inflammation. (Masters Thesis). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/killi_ravikumar_200812_ms

Chicago Manual of Style (16th Edition):

Killi, Ravikumar. “Assessment of the ability of two differently constructed equine viral vaccines to induce systemic inflammation.” 2008. Masters Thesis, University of Georgia. Accessed April 04, 2020. http://purl.galileo.usg.edu/uga_etd/killi_ravikumar_200812_ms.

MLA Handbook (7th Edition):

Killi, Ravikumar. “Assessment of the ability of two differently constructed equine viral vaccines to induce systemic inflammation.” 2008. Web. 04 Apr 2020.

Vancouver:

Killi R. Assessment of the ability of two differently constructed equine viral vaccines to induce systemic inflammation. [Internet] [Masters thesis]. University of Georgia; 2008. [cited 2020 Apr 04]. Available from: http://purl.galileo.usg.edu/uga_etd/killi_ravikumar_200812_ms.

Council of Science Editors:

Killi R. Assessment of the ability of two differently constructed equine viral vaccines to induce systemic inflammation. [Masters Thesis]. University of Georgia; 2008. Available from: http://purl.galileo.usg.edu/uga_etd/killi_ravikumar_200812_ms


University of Rochester

3. Saperstein, Sara C. Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors.

Degree: PhD, 2009, University of Rochester

 Tumor necrosis factor-α (TNF-α) and interleukin-1β (IL-1β) are key mediators of numerous lung inflammatory diseases. Induced by similar stimuli, in many cell types IL-1β modifies… (more)

Subjects/Keywords: Inflammation; Receptors

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APA (6th Edition):

Saperstein, S. C. (2009). Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8631

Chicago Manual of Style (16th Edition):

Saperstein, Sara C. “Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors.” 2009. Doctoral Dissertation, University of Rochester. Accessed April 04, 2020. http://hdl.handle.net/1802/8631.

MLA Handbook (7th Edition):

Saperstein, Sara C. “Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors.” 2009. Web. 04 Apr 2020.

Vancouver:

Saperstein SC. Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1802/8631.

Council of Science Editors:

Saperstein SC. Modulation of TNF-α-mediated acute lung inflammation : a role for IL-1β and soluble TNF receptors. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8631

4. Schnegg, Caroline Isabel. RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO.

Degree: 2012, Wake Forest University

 As a result of improvements in cancer therapy and health care, the population of long-term cancer survivors is growing. For these approximately 12 million long-term… (more)

Subjects/Keywords: Inflammation

…and Inflammation Classically, radiation-induced late normal tissue injury was thought to be… …which contribute to increased oxidative stress and inflammation [36-38]. This model… …act as second messengers to induce inflammation [43]. Although it was once… …believed that the brain was not susceptible to inflammation, it is now recognized that… …and/or inflammation, therefore, appears to be one of the mechanisms underlying radiation… 

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APA (6th Edition):

Schnegg, C. I. (2012). RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/37442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schnegg, Caroline Isabel. “RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO.” 2012. Thesis, Wake Forest University. Accessed April 04, 2020. http://hdl.handle.net/10339/37442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schnegg, Caroline Isabel. “RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO.” 2012. Web. 04 Apr 2020.

Vancouver:

Schnegg CI. RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO. [Internet] [Thesis]. Wake Forest University; 2012. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10339/37442.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schnegg CI. RADIATION-INDUCED INFLAMMATORY MARKERS OF BRAIN INJURY ARE MODULATED BY PPARä ACTIVATION IN VITRO AND IN VIVO. [Thesis]. Wake Forest University; 2012. Available from: http://hdl.handle.net/10339/37442

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

5. Boyanapalli, Sarandeep S S. Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects.

Degree: PhD, Pharmaceutical Science, 2016, Rutgers University

Chronic inflammation is considered to play an important factor in neoplastic progression via the induction of reactive oxygen, reactive nitrogen species and induction of growth… (more)

Subjects/Keywords: Phytochemicals; Inflammation

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APA (6th Edition):

Boyanapalli, S. S. S. (2016). Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/49917/

Chicago Manual of Style (16th Edition):

Boyanapalli, Sarandeep S S. “Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects.” 2016. Doctoral Dissertation, Rutgers University. Accessed April 04, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/49917/.

MLA Handbook (7th Edition):

Boyanapalli, Sarandeep S S. “Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects.” 2016. Web. 04 Apr 2020.

Vancouver:

Boyanapalli SSS. Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects. [Internet] [Doctoral dissertation]. Rutgers University; 2016. [cited 2020 Apr 04]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/49917/.

Council of Science Editors:

Boyanapalli SSS. Anti-inflammatory, epigenetic regulatory role of phytochemicals and pkpd modeling of pharmacological effects. [Doctoral Dissertation]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/49917/


University of Houston

6. Zambrano Cobo, Amarayca. Inflammation in Zebrafish Models.

Degree: MS, Biomedical Engineering, 2015, University of Houston

 The zebrafish is an outstanding model for in-vivo imaging of inflammation due to its optical translucency and the ability of transgenic lines to express fluorescent… (more)

Subjects/Keywords: Zebrafish; Inflammation

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APA (6th Edition):

Zambrano Cobo, A. (2015). Inflammation in Zebrafish Models. (Masters Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/4848

Chicago Manual of Style (16th Edition):

Zambrano Cobo, Amarayca. “Inflammation in Zebrafish Models.” 2015. Masters Thesis, University of Houston. Accessed April 04, 2020. http://hdl.handle.net/10657/4848.

MLA Handbook (7th Edition):

Zambrano Cobo, Amarayca. “Inflammation in Zebrafish Models.” 2015. Web. 04 Apr 2020.

Vancouver:

Zambrano Cobo A. Inflammation in Zebrafish Models. [Internet] [Masters thesis]. University of Houston; 2015. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10657/4848.

Council of Science Editors:

Zambrano Cobo A. Inflammation in Zebrafish Models. [Masters Thesis]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/4848


University of Utah

7. Osborne, Amber Valeria. The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation.

Degree: PhD, Pathology;, 2010, University of Utah

 This dissertation examines the relationship between inflammatory cytokines and nicotinic acetylcholine receptors (nAChR). WT and nAChRa7 null mice were exposed to 4.8kJ/m2 of UVB on… (more)

Subjects/Keywords: Nicotine Receptors; Inflammation

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APA (6th Edition):

Osborne, A. V. (2010). The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195

Chicago Manual of Style (16th Edition):

Osborne, Amber Valeria. “The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation.” 2010. Doctoral Dissertation, University of Utah. Accessed April 04, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195.

MLA Handbook (7th Edition):

Osborne, Amber Valeria. “The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation.” 2010. Web. 04 Apr 2020.

Vancouver:

Osborne AV. The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Apr 04]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195.

Council of Science Editors:

Osborne AV. The Modulatory Role of Neuronal Nicotinic Receptors on Peripheral Inflammation. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1089/rec/1195


Texas A&M University

8. Laible, Allyson Marie. Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles.

Degree: 2009, Texas A&M University

 Cellular response to inflammatory cytokines involves concerted changes in gene expression. Cytokines, such as IL-6 and TNF-?, regulate gene expression through multiple intracellular signaling pathways.… (more)

Subjects/Keywords: Inflammation; NF-kappaB

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APA (6th Edition):

Laible, A. M. (2009). Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1443

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Laible, Allyson Marie. “Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles.” 2009. Thesis, Texas A&M University. Accessed April 04, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-1443.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Laible, Allyson Marie. “Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles.” 2009. Web. 04 Apr 2020.

Vancouver:

Laible AM. Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles. [Internet] [Thesis]. Texas A&M University; 2009. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1443.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Laible AM. Modeling green fluorescent protein transcription, translation and modification as a method to obtain NF-kappaB activation profiles. [Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1443

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

9. Gao, Lujia. COMPOSITION OF THE ENDOTHELIAL GLYCOCALYX AND ITS EFFECT ON TRANSVASCULAR EXCHANGE OF SMALL SOLUTES.

Degree: PhD, Bioengineering, 2010, Penn State University

 The endothelial cell (EC) of the circulatory system is coated with a layer of macromolecules, composed of proteins and polysaccharides. It has been suggested that… (more)

Subjects/Keywords: Glycocalyx; Inflammation; Glycosaminoglycan

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APA (6th Edition):

Gao, L. (2010). COMPOSITION OF THE ENDOTHELIAL GLYCOCALYX AND ITS EFFECT ON TRANSVASCULAR EXCHANGE OF SMALL SOLUTES. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11005

Chicago Manual of Style (16th Edition):

Gao, Lujia. “COMPOSITION OF THE ENDOTHELIAL GLYCOCALYX AND ITS EFFECT ON TRANSVASCULAR EXCHANGE OF SMALL SOLUTES.” 2010. Doctoral Dissertation, Penn State University. Accessed April 04, 2020. https://etda.libraries.psu.edu/catalog/11005.

MLA Handbook (7th Edition):

Gao, Lujia. “COMPOSITION OF THE ENDOTHELIAL GLYCOCALYX AND ITS EFFECT ON TRANSVASCULAR EXCHANGE OF SMALL SOLUTES.” 2010. Web. 04 Apr 2020.

Vancouver:

Gao L. COMPOSITION OF THE ENDOTHELIAL GLYCOCALYX AND ITS EFFECT ON TRANSVASCULAR EXCHANGE OF SMALL SOLUTES. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2020 Apr 04]. Available from: https://etda.libraries.psu.edu/catalog/11005.

Council of Science Editors:

Gao L. COMPOSITION OF THE ENDOTHELIAL GLYCOCALYX AND ITS EFFECT ON TRANSVASCULAR EXCHANGE OF SMALL SOLUTES. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11005


University of California – Merced

10. Al-Kuhlani, Mufadhal Mohammed. Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection.

Degree: Quantitative and Systems Biology, 2013, University of California – Merced

 Chlamydia trachomatis is the most common bacterial sexually-transmitted disease (STD) around the world. The response of the immune system to eliminate C. trachomatis infection through… (more)

Subjects/Keywords: Immunology; Chlamydia; Inflammation

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APA (6th Edition):

Al-Kuhlani, M. M. (2013). Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection. (Thesis). University of California – Merced. Retrieved from http://www.escholarship.org/uc/item/1m5036rd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Kuhlani, Mufadhal Mohammed. “Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection.” 2013. Thesis, University of California – Merced. Accessed April 04, 2020. http://www.escholarship.org/uc/item/1m5036rd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Kuhlani, Mufadhal Mohammed. “Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection.” 2013. Web. 04 Apr 2020.

Vancouver:

Al-Kuhlani MM. Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection. [Internet] [Thesis]. University of California – Merced; 2013. [cited 2020 Apr 04]. Available from: http://www.escholarship.org/uc/item/1m5036rd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Kuhlani MM. Negative Regulators of Toll-Like Receptors During Chlamydia trachomatis Infection. [Thesis]. University of California – Merced; 2013. Available from: http://www.escholarship.org/uc/item/1m5036rd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Tech

11. Wendelsdorf, Katherine Veronica. Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis.

Degree: PhD, Genetics, Bioinformatics, and Computational Biology, 2011, Virginia Tech

 The scientific method requires the creation of a unifying hypothesis that reconciles an observed health outcome of infection with experimental data gathered about the disease… (more)

Subjects/Keywords: inflammation; simulation; immunopathology

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APA (6th Edition):

Wendelsdorf, K. V. (2011). Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis. (Doctoral Dissertation). Virginia Tech. Retrieved from http://hdl.handle.net/10919/29717

Chicago Manual of Style (16th Edition):

Wendelsdorf, Katherine Veronica. “Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis.” 2011. Doctoral Dissertation, Virginia Tech. Accessed April 04, 2020. http://hdl.handle.net/10919/29717.

MLA Handbook (7th Edition):

Wendelsdorf, Katherine Veronica. “Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis.” 2011. Web. 04 Apr 2020.

Vancouver:

Wendelsdorf KV. Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis. [Internet] [Doctoral dissertation]. Virginia Tech; 2011. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10919/29717.

Council of Science Editors:

Wendelsdorf KV. Models of the Mucosal Inflammatory and Regulatory Immune Pathways: The Role of Host Response in Microbial Persistence and Pathogenesis. [Doctoral Dissertation]. Virginia Tech; 2011. Available from: http://hdl.handle.net/10919/29717


Drexel University

12. Shenoda, Botros. A Role for XIST in the Regulation of Inflammatory Response by Female Cells.

Degree: 2017, Drexel University

Biological sex influences inflammatory response [1]. There is a greater incidence of acute inflammation in men, and chronic inflammation in women. X-inactive specific transcript (XIST)… (more)

Subjects/Keywords: Pharmacology; Physiology; Inflammation

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APA (6th Edition):

Shenoda, B. (2017). A Role for XIST in the Regulation of Inflammatory Response by Female Cells. (Thesis). Drexel University. Retrieved from https://idea.library.drexel.edu/islandora/object/idea%3A9409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shenoda, Botros. “A Role for XIST in the Regulation of Inflammatory Response by Female Cells.” 2017. Thesis, Drexel University. Accessed April 04, 2020. https://idea.library.drexel.edu/islandora/object/idea%3A9409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shenoda, Botros. “A Role for XIST in the Regulation of Inflammatory Response by Female Cells.” 2017. Web. 04 Apr 2020.

Vancouver:

Shenoda B. A Role for XIST in the Regulation of Inflammatory Response by Female Cells. [Internet] [Thesis]. Drexel University; 2017. [cited 2020 Apr 04]. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A9409.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shenoda B. A Role for XIST in the Regulation of Inflammatory Response by Female Cells. [Thesis]. Drexel University; 2017. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A9409

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

13. Sinha, Siddhartha. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages.

Degree: 2014, Cornell University

 Atherosclerosis is a decades-long process whose patients often remain asymptomatic until after a heart attack or stroke. Novel therapeutic approaches focus on tuning the body's… (more)

Subjects/Keywords: Atherosclerosis; Inflammation; Macrophage

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APA (6th Edition):

Sinha, S. (2014). Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/37128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. ” 2014. Thesis, Cornell University. Accessed April 04, 2020. http://hdl.handle.net/1813/37128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sinha, Siddhartha. “Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. ” 2014. Web. 04 Apr 2020.

Vancouver:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Internet] [Thesis]. Cornell University; 2014. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1813/37128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sinha S. Role Of Substrate Stiffness In Driving Atherogenic Behavior In Macrophages. [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/37128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

14. Mitchell, Michael. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation .

Degree: 2014, Cornell University

 The application of fluid shear stress on leukocytes and tumor cells in the circulation plays a critical role in the progression of inflammation and cancer… (more)

Subjects/Keywords: Inflammation; Metastasis; Nanomedicine

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APA (6th Edition):

Mitchell, M. (2014). Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/39384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mitchell, Michael. “Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation .” 2014. Thesis, Cornell University. Accessed April 04, 2020. http://hdl.handle.net/1813/39384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mitchell, Michael. “Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation .” 2014. Web. 04 Apr 2020.

Vancouver:

Mitchell M. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation . [Internet] [Thesis]. Cornell University; 2014. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1813/39384.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mitchell M. Mechanotransduction And Therapeutic Targeting Of Cells In The Circulation . [Thesis]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/39384

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Brandeis University

15. Kaye, Karen. Acute Psychosocial Stress Induces Changes in Norepinephrine Sensitivity of Stimulated TNF-Alpha Production.

Degree: 2015, Brandeis University

 Background and Rationale: It is well established that psychosocial stress induces changes in glucocorticoid (GC) sensitivity, but little is known about how repeated psychosocial stress… (more)

Subjects/Keywords: Norepinephrine; Stress; Inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kaye, K. (2015). Acute Psychosocial Stress Induces Changes in Norepinephrine Sensitivity of Stimulated TNF-Alpha Production. (Thesis). Brandeis University. Retrieved from http://hdl.handle.net/10192/31110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kaye, Karen. “Acute Psychosocial Stress Induces Changes in Norepinephrine Sensitivity of Stimulated TNF-Alpha Production.” 2015. Thesis, Brandeis University. Accessed April 04, 2020. http://hdl.handle.net/10192/31110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kaye, Karen. “Acute Psychosocial Stress Induces Changes in Norepinephrine Sensitivity of Stimulated TNF-Alpha Production.” 2015. Web. 04 Apr 2020.

Vancouver:

Kaye K. Acute Psychosocial Stress Induces Changes in Norepinephrine Sensitivity of Stimulated TNF-Alpha Production. [Internet] [Thesis]. Brandeis University; 2015. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10192/31110.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kaye K. Acute Psychosocial Stress Induces Changes in Norepinephrine Sensitivity of Stimulated TNF-Alpha Production. [Thesis]. Brandeis University; 2015. Available from: http://hdl.handle.net/10192/31110

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

16. Pang, Jingbo. Galectin-3 in Obesity and Endotoxin-Induced Inflammation.

Degree: 2016, University of Illinois – Chicago

 Obesity has become a worldwide epidemic that causes both health and economic burdens. The mild but persistent elevation of the bacterial product lipopolysaccharide (LPS) in… (more)

Subjects/Keywords: Obesity; Inflammation; Endotoxin

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APA (6th Edition):

Pang, J. (2016). Galectin-3 in Obesity and Endotoxin-Induced Inflammation. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pang, Jingbo. “Galectin-3 in Obesity and Endotoxin-Induced Inflammation.” 2016. Thesis, University of Illinois – Chicago. Accessed April 04, 2020. http://hdl.handle.net/10027/21593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pang, Jingbo. “Galectin-3 in Obesity and Endotoxin-Induced Inflammation.” 2016. Web. 04 Apr 2020.

Vancouver:

Pang J. Galectin-3 in Obesity and Endotoxin-Induced Inflammation. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10027/21593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pang J. Galectin-3 in Obesity and Endotoxin-Induced Inflammation. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

17. Liu, Menglin. Sox17 Mediated Regeneration of the Lung Endothelium Following Acute Lung Injury.

Degree: 2019, University of Illinois – Chicago

 Repair of the endothelial cell barrier after inflammatory injury is essential for tissue fluid homeostasis and normalizing leukocyte transmigration. However, the mechanisms of endothelial regeneration… (more)

Subjects/Keywords: Endothelial regeneration; Inflammation

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APA (6th Edition):

Liu, M. (2019). Sox17 Mediated Regeneration of the Lung Endothelium Following Acute Lung Injury. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Menglin. “Sox17 Mediated Regeneration of the Lung Endothelium Following Acute Lung Injury.” 2019. Thesis, University of Illinois – Chicago. Accessed April 04, 2020. http://hdl.handle.net/10027/23725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Menglin. “Sox17 Mediated Regeneration of the Lung Endothelium Following Acute Lung Injury.” 2019. Web. 04 Apr 2020.

Vancouver:

Liu M. Sox17 Mediated Regeneration of the Lung Endothelium Following Acute Lung Injury. [Internet] [Thesis]. University of Illinois – Chicago; 2019. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10027/23725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu M. Sox17 Mediated Regeneration of the Lung Endothelium Following Acute Lung Injury. [Thesis]. University of Illinois – Chicago; 2019. Available from: http://hdl.handle.net/10027/23725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

18. Botelho, Danielle Joy. Effects of nanoparticle functionalization, shape & size on lung function & inflammation.

Degree: PhD, Toxicology, 2015, Rutgers University

With the rise of nanoparticle use in commercial products, industry, and medicine, there is a need to better understand the consequences of nanoparticle exposure. Due… (more)

Subjects/Keywords: Nanoparticles; Inflammation; Lungs

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APA (6th Edition):

Botelho, D. J. (2015). Effects of nanoparticle functionalization, shape & size on lung function & inflammation. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/48405/

Chicago Manual of Style (16th Edition):

Botelho, Danielle Joy. “Effects of nanoparticle functionalization, shape & size on lung function & inflammation.” 2015. Doctoral Dissertation, Rutgers University. Accessed April 04, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/48405/.

MLA Handbook (7th Edition):

Botelho, Danielle Joy. “Effects of nanoparticle functionalization, shape & size on lung function & inflammation.” 2015. Web. 04 Apr 2020.

Vancouver:

Botelho DJ. Effects of nanoparticle functionalization, shape & size on lung function & inflammation. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2020 Apr 04]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48405/.

Council of Science Editors:

Botelho DJ. Effects of nanoparticle functionalization, shape & size on lung function & inflammation. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48405/


University of Southern California

19. Ha, Helen Nen. Identification of small-molecules targeting CXCR2 function and signaling.

Degree: PhD, Pharmaceutical Sciences, 2013, University of Southern California

 Chemokine receptor, CXCR2, and its ligands are an essential component of the immune system that mediates the trafficking of neutrophils to sites of infection. It… (more)

Subjects/Keywords: CXCR2; CXCL8; inflammation

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APA (6th Edition):

Ha, H. N. (2013). Identification of small-molecules targeting CXCR2 function and signaling. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/308809/rec/3324

Chicago Manual of Style (16th Edition):

Ha, Helen Nen. “Identification of small-molecules targeting CXCR2 function and signaling.” 2013. Doctoral Dissertation, University of Southern California. Accessed April 04, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/308809/rec/3324.

MLA Handbook (7th Edition):

Ha, Helen Nen. “Identification of small-molecules targeting CXCR2 function and signaling.” 2013. Web. 04 Apr 2020.

Vancouver:

Ha HN. Identification of small-molecules targeting CXCR2 function and signaling. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2020 Apr 04]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/308809/rec/3324.

Council of Science Editors:

Ha HN. Identification of small-molecules targeting CXCR2 function and signaling. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/308809/rec/3324


Texas A&M University

20. Lucia, Jessica Lauren. Influence of an Intra-articular Lipopolysaccharide Challenge on Markers of Inflammation and Cartilage Metabolism and the Ability of Oral Glucosamine to Mitigate these Alterations in Young Horses.

Degree: 2013, Texas A&M University

 This project established an in vivo method to identify and manipulate expression of markers of osteoarthritis (OA). Specifically, strategies that predictably induce joint inflammation to… (more)

Subjects/Keywords: lipopolysaccharide; cartilage; inflammation; horse

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APA (6th Edition):

Lucia, J. L. (2013). Influence of an Intra-articular Lipopolysaccharide Challenge on Markers of Inflammation and Cartilage Metabolism and the Ability of Oral Glucosamine to Mitigate these Alterations in Young Horses. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lucia, Jessica Lauren. “Influence of an Intra-articular Lipopolysaccharide Challenge on Markers of Inflammation and Cartilage Metabolism and the Ability of Oral Glucosamine to Mitigate these Alterations in Young Horses.” 2013. Thesis, Texas A&M University. Accessed April 04, 2020. http://hdl.handle.net/1969.1/149284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lucia, Jessica Lauren. “Influence of an Intra-articular Lipopolysaccharide Challenge on Markers of Inflammation and Cartilage Metabolism and the Ability of Oral Glucosamine to Mitigate these Alterations in Young Horses.” 2013. Web. 04 Apr 2020.

Vancouver:

Lucia JL. Influence of an Intra-articular Lipopolysaccharide Challenge on Markers of Inflammation and Cartilage Metabolism and the Ability of Oral Glucosamine to Mitigate these Alterations in Young Horses. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1969.1/149284.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lucia JL. Influence of an Intra-articular Lipopolysaccharide Challenge on Markers of Inflammation and Cartilage Metabolism and the Ability of Oral Glucosamine to Mitigate these Alterations in Young Horses. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149284

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

21. Arbizu Berrocal, Shirley. Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro.

Degree: 2013, Texas A&M University

 Breast cancer is the leading cause of cancer death among women worldwide and polyphenols are under investigation as an alternative to conventional treatment approaches of… (more)

Subjects/Keywords: mango; polyphenols; inflammation; breast cancer

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APA (6th Edition):

Arbizu Berrocal, S. (2013). Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arbizu Berrocal, Shirley. “Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro.” 2013. Thesis, Texas A&M University. Accessed April 04, 2020. http://hdl.handle.net/1969.1/151359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arbizu Berrocal, Shirley. “Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro.” 2013. Web. 04 Apr 2020.

Vancouver:

Arbizu Berrocal S. Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1969.1/151359.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arbizu Berrocal S. Anti-inflammatory and Cytotoxic Activities of Mango (Mangifera indica L. var Keitt) Polyphenols in Cancer and Non-cancer Breast Fibroblasts in Vitro. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151359

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

22. Kahn, Meredith K. Age-Related Effects on Markers of Inflammation and Cartilage Metabolism in Response to an Intra-Articular Lipopolysaccharide Challenge.

Degree: 2014, Texas A&M University

 Eighteen Quarter Horses were utilized in a randomized complete design for a 28 d experiment to evaluate age-related effects on inflammation and cartilage turnover after… (more)

Subjects/Keywords: Age; Horse; Lipopolysaccharide; Inflammation; Cartilage

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APA (6th Edition):

Kahn, M. K. (2014). Age-Related Effects on Markers of Inflammation and Cartilage Metabolism in Response to an Intra-Articular Lipopolysaccharide Challenge. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/154113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kahn, Meredith K. “Age-Related Effects on Markers of Inflammation and Cartilage Metabolism in Response to an Intra-Articular Lipopolysaccharide Challenge.” 2014. Thesis, Texas A&M University. Accessed April 04, 2020. http://hdl.handle.net/1969.1/154113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kahn, Meredith K. “Age-Related Effects on Markers of Inflammation and Cartilage Metabolism in Response to an Intra-Articular Lipopolysaccharide Challenge.” 2014. Web. 04 Apr 2020.

Vancouver:

Kahn MK. Age-Related Effects on Markers of Inflammation and Cartilage Metabolism in Response to an Intra-Articular Lipopolysaccharide Challenge. [Internet] [Thesis]. Texas A&M University; 2014. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1969.1/154113.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kahn MK. Age-Related Effects on Markers of Inflammation and Cartilage Metabolism in Response to an Intra-Articular Lipopolysaccharide Challenge. [Thesis]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/154113

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Barffour, Maxwell Adusei. INTERACTIONS BETWEEN MALARIA AND IRON OR VITAMIN A STATUS IN RURAL ZAMBIAN CHILDREN.

Degree: 2014, Johns Hopkins University

 Background and objectives Evidence regarding the bidirectional interactions between iron or vitamin A status and malaria remains inconclusive. We assessed the longitudinal associations between iron… (more)

Subjects/Keywords: Malaria; Inflammation; Iron; Vitamin A

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APA (6th Edition):

Barffour, M. A. (2014). INTERACTIONS BETWEEN MALARIA AND IRON OR VITAMIN A STATUS IN RURAL ZAMBIAN CHILDREN. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/38008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barffour, Maxwell Adusei. “INTERACTIONS BETWEEN MALARIA AND IRON OR VITAMIN A STATUS IN RURAL ZAMBIAN CHILDREN.” 2014. Thesis, Johns Hopkins University. Accessed April 04, 2020. http://jhir.library.jhu.edu/handle/1774.2/38008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barffour, Maxwell Adusei. “INTERACTIONS BETWEEN MALARIA AND IRON OR VITAMIN A STATUS IN RURAL ZAMBIAN CHILDREN.” 2014. Web. 04 Apr 2020.

Vancouver:

Barffour MA. INTERACTIONS BETWEEN MALARIA AND IRON OR VITAMIN A STATUS IN RURAL ZAMBIAN CHILDREN. [Internet] [Thesis]. Johns Hopkins University; 2014. [cited 2020 Apr 04]. Available from: http://jhir.library.jhu.edu/handle/1774.2/38008.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barffour MA. INTERACTIONS BETWEEN MALARIA AND IRON OR VITAMIN A STATUS IN RURAL ZAMBIAN CHILDREN. [Thesis]. Johns Hopkins University; 2014. Available from: http://jhir.library.jhu.edu/handle/1774.2/38008

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hawaii – Manoa

24. Rose, Aaron Hugh. Inflammation associated cancers and potential therapeutics.

Degree: 2015, University of Hawaii – Manoa

Ph.D. University of Hawaii at Manoa 2014.

Inflammation is a major contributing factor in approximately 20% of cancers, and two types of cancer in which… (more)

Subjects/Keywords: Inflammation; mesothelioma; colorectal carcinoma

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APA (6th Edition):

Rose, A. H. (2015). Inflammation associated cancers and potential therapeutics. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/100474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rose, Aaron Hugh. “Inflammation associated cancers and potential therapeutics.” 2015. Thesis, University of Hawaii – Manoa. Accessed April 04, 2020. http://hdl.handle.net/10125/100474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rose, Aaron Hugh. “Inflammation associated cancers and potential therapeutics.” 2015. Web. 04 Apr 2020.

Vancouver:

Rose AH. Inflammation associated cancers and potential therapeutics. [Internet] [Thesis]. University of Hawaii – Manoa; 2015. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/10125/100474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rose AH. Inflammation associated cancers and potential therapeutics. [Thesis]. University of Hawaii – Manoa; 2015. Available from: http://hdl.handle.net/10125/100474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

25. Su, Xiaomin; Federoff, Howard. a-Synuclein and microglial activation in Parkinson's disease.

Degree: PhD, 2009, University of Rochester

 Parkinson’s disease (PD) is an age-related slowly progressive neurodegenerative disorder with preferential loss of nigrostriatal dopamine neurons. The pathological hallmark of PD is the presence… (more)

Subjects/Keywords: Inflammation; Synuclein; Microglia; Parkinson's disease

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APA (6th Edition):

Su, Xiaomin; Federoff, H. (2009). a-Synuclein and microglial activation in Parkinson's disease. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6533

Chicago Manual of Style (16th Edition):

Su, Xiaomin; Federoff, Howard. “a-Synuclein and microglial activation in Parkinson's disease.” 2009. Doctoral Dissertation, University of Rochester. Accessed April 04, 2020. http://hdl.handle.net/1802/6533.

MLA Handbook (7th Edition):

Su, Xiaomin; Federoff, Howard. “a-Synuclein and microglial activation in Parkinson's disease.” 2009. Web. 04 Apr 2020.

Vancouver:

Su, Xiaomin; Federoff H. a-Synuclein and microglial activation in Parkinson's disease. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1802/6533.

Council of Science Editors:

Su, Xiaomin; Federoff H. a-Synuclein and microglial activation in Parkinson's disease. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6533


University of Rochester

26. Aggrey, Angela Akosua. A Novel Protective Role for Platelet-Mediated Immune Responses in Murine Experimental Cerebral Malaria.

Degree: PhD, 2013, University of Rochester

 Cerebral malaria is caused by complications from Plasmodium falciparum infection and claims hundreds of thousands of lives of young children in endemic countries annually. Platelets… (more)

Subjects/Keywords: Platelets; Innate Immunity, Malaria; Inflammation

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APA (6th Edition):

Aggrey, A. A. (2013). A Novel Protective Role for Platelet-Mediated Immune Responses in Murine Experimental Cerebral Malaria. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/27239

Chicago Manual of Style (16th Edition):

Aggrey, Angela Akosua. “A Novel Protective Role for Platelet-Mediated Immune Responses in Murine Experimental Cerebral Malaria.” 2013. Doctoral Dissertation, University of Rochester. Accessed April 04, 2020. http://hdl.handle.net/1802/27239.

MLA Handbook (7th Edition):

Aggrey, Angela Akosua. “A Novel Protective Role for Platelet-Mediated Immune Responses in Murine Experimental Cerebral Malaria.” 2013. Web. 04 Apr 2020.

Vancouver:

Aggrey AA. A Novel Protective Role for Platelet-Mediated Immune Responses in Murine Experimental Cerebral Malaria. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/1802/27239.

Council of Science Editors:

Aggrey AA. A Novel Protective Role for Platelet-Mediated Immune Responses in Murine Experimental Cerebral Malaria. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/27239


North Carolina State University

27. Summers, Caroline Rueda. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.

Degree: MS, Nutrition, 2010, North Carolina State University

 Recently, the World Health Organization estimated that chronic diseases are responsible for 46% of disease occurrence and 59% of all deaths worldwide. Growing evidence suggests… (more)

Subjects/Keywords: LPS; tea; mouse macrophage; inflammation

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APA (6th Edition):

Summers, C. R. (2010). Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/6332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Summers, Caroline Rueda. “Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.” 2010. Thesis, North Carolina State University. Accessed April 04, 2020. http://www.lib.ncsu.edu/resolver/1840.16/6332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Summers, Caroline Rueda. “Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells.” 2010. Web. 04 Apr 2020.

Vancouver:

Summers CR. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. [Internet] [Thesis]. North Carolina State University; 2010. [cited 2020 Apr 04]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6332.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Summers CR. Modulation of Inflammatory Responses by Green and Black Tea in LPS-induced RAW 264.7 Cells. [Thesis]. North Carolina State University; 2010. Available from: http://www.lib.ncsu.edu/resolver/1840.16/6332

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

28. Xu, Changjie. The Effects and Mechanisms of Consuming Sulfur-Containing Dietary Supplements on Chronic Inflammation in Adults with Obesity.

Degree: PhD, Nutritional Sciences, 2017, University of Florida

 Obesity is global health issue. In the United States, more than one-third of adults are obese. Obesity induces chronic systemic inflammation and increasing obesity positively… (more)

Subjects/Keywords: garlic  – glutathione  – inflammation  – obesity

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APA (6th Edition):

Xu, C. (2017). The Effects and Mechanisms of Consuming Sulfur-Containing Dietary Supplements on Chronic Inflammation in Adults with Obesity. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0051738

Chicago Manual of Style (16th Edition):

Xu, Changjie. “The Effects and Mechanisms of Consuming Sulfur-Containing Dietary Supplements on Chronic Inflammation in Adults with Obesity.” 2017. Doctoral Dissertation, University of Florida. Accessed April 04, 2020. http://ufdc.ufl.edu/UFE0051738.

MLA Handbook (7th Edition):

Xu, Changjie. “The Effects and Mechanisms of Consuming Sulfur-Containing Dietary Supplements on Chronic Inflammation in Adults with Obesity.” 2017. Web. 04 Apr 2020.

Vancouver:

Xu C. The Effects and Mechanisms of Consuming Sulfur-Containing Dietary Supplements on Chronic Inflammation in Adults with Obesity. [Internet] [Doctoral dissertation]. University of Florida; 2017. [cited 2020 Apr 04]. Available from: http://ufdc.ufl.edu/UFE0051738.

Council of Science Editors:

Xu C. The Effects and Mechanisms of Consuming Sulfur-Containing Dietary Supplements on Chronic Inflammation in Adults with Obesity. [Doctoral Dissertation]. University of Florida; 2017. Available from: http://ufdc.ufl.edu/UFE0051738


Boston University

29. Delio, Melissa Caitlin. The protective role of fat specific protein 27 (FSP27) against interleukin-1 beta and interleukin-6 induced lipolysis and insulin resistance in human adipocytes.

Degree: MS, Medical Sciences, 2015, Boston University

 The global rise in the prevalence of obesity has been labeled a pandemic. The increasing rates of overweight and obese persons across the world is… (more)

Subjects/Keywords: Medicine; Inflammation; Obesity; T2DM

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Delio, M. C. (2015). The protective role of fat specific protein 27 (FSP27) against interleukin-1 beta and interleukin-6 induced lipolysis and insulin resistance in human adipocytes. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16269

Chicago Manual of Style (16th Edition):

Delio, Melissa Caitlin. “The protective role of fat specific protein 27 (FSP27) against interleukin-1 beta and interleukin-6 induced lipolysis and insulin resistance in human adipocytes.” 2015. Masters Thesis, Boston University. Accessed April 04, 2020. http://hdl.handle.net/2144/16269.

MLA Handbook (7th Edition):

Delio, Melissa Caitlin. “The protective role of fat specific protein 27 (FSP27) against interleukin-1 beta and interleukin-6 induced lipolysis and insulin resistance in human adipocytes.” 2015. Web. 04 Apr 2020.

Vancouver:

Delio MC. The protective role of fat specific protein 27 (FSP27) against interleukin-1 beta and interleukin-6 induced lipolysis and insulin resistance in human adipocytes. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Apr 04]. Available from: http://hdl.handle.net/2144/16269.

Council of Science Editors:

Delio MC. The protective role of fat specific protein 27 (FSP27) against interleukin-1 beta and interleukin-6 induced lipolysis and insulin resistance in human adipocytes. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16269


The Ohio State University

30. Miller, Brandon Andrew. The effects of excitotoxicity and microglial activation on oligodendrocyte survival.

Degree: PhD, Neuroscience, 2007, The Ohio State University

 The nervous system transmits information over long distances by action potentials carried by neurons. In both the central and peripheral nervous system, neurons are insulated… (more)

Subjects/Keywords: oligodendrocyte; microglia; excitotoxicity; inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Miller, B. A. (2007). The effects of excitotoxicity and microglial activation on oligodendrocyte survival. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1177537595

Chicago Manual of Style (16th Edition):

Miller, Brandon Andrew. “The effects of excitotoxicity and microglial activation on oligodendrocyte survival.” 2007. Doctoral Dissertation, The Ohio State University. Accessed April 04, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1177537595.

MLA Handbook (7th Edition):

Miller, Brandon Andrew. “The effects of excitotoxicity and microglial activation on oligodendrocyte survival.” 2007. Web. 04 Apr 2020.

Vancouver:

Miller BA. The effects of excitotoxicity and microglial activation on oligodendrocyte survival. [Internet] [Doctoral dissertation]. The Ohio State University; 2007. [cited 2020 Apr 04]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1177537595.

Council of Science Editors:

Miller BA. The effects of excitotoxicity and microglial activation on oligodendrocyte survival. [Doctoral Dissertation]. The Ohio State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1177537595

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