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You searched for subject:(immunology). Showing records 1 – 30 of 8538 total matches.

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The George Washington University

1. Marjanovic, Sophia Y. The Role of the Wiskott-Aldrich Syndrome Protein in Regulating T Cell Programmed Cell Death Mechanisms and Implications for Autoimmunity in the Wiskott-Aldrich Syndrome.

Degree: 2016, The George Washington University

  The development of autoimmunity in the setting of immunodeficiency has been a paradoxical observation. Many primary immunodeficiency diseases (PIDDs) are associated with autoimmunity. Wiskott-Aldrich… (more)

Subjects/Keywords: Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Marjanovic, S. Y. (2016). The Role of the Wiskott-Aldrich Syndrome Protein in Regulating T Cell Programmed Cell Death Mechanisms and Implications for Autoimmunity in the Wiskott-Aldrich Syndrome. (Thesis). The George Washington University. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=10006518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marjanovic, Sophia Y. “The Role of the Wiskott-Aldrich Syndrome Protein in Regulating T Cell Programmed Cell Death Mechanisms and Implications for Autoimmunity in the Wiskott-Aldrich Syndrome.” 2016. Thesis, The George Washington University. Accessed March 02, 2021. http://pqdtopen.proquest.com/#viewpdf?dispub=10006518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marjanovic, Sophia Y. “The Role of the Wiskott-Aldrich Syndrome Protein in Regulating T Cell Programmed Cell Death Mechanisms and Implications for Autoimmunity in the Wiskott-Aldrich Syndrome.” 2016. Web. 02 Mar 2021.

Vancouver:

Marjanovic SY. The Role of the Wiskott-Aldrich Syndrome Protein in Regulating T Cell Programmed Cell Death Mechanisms and Implications for Autoimmunity in the Wiskott-Aldrich Syndrome. [Internet] [Thesis]. The George Washington University; 2016. [cited 2021 Mar 02]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10006518.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marjanovic SY. The Role of the Wiskott-Aldrich Syndrome Protein in Regulating T Cell Programmed Cell Death Mechanisms and Implications for Autoimmunity in the Wiskott-Aldrich Syndrome. [Thesis]. The George Washington University; 2016. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10006518

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Yale University

2. Gray, Simon Matthew. PRC2-Mediated H3K27me3 Repression Promotes Effector CD8 + T Cell Terminal Differentiation and Loss of Multipotency.

Degree: 2017, Yale University

  Elucidating how multipotent memory precursor (MP) cells maintain developmental plasticity and longevity to provide long-term immunity while most effector cells develop into terminally differentiated… (more)

Subjects/Keywords: Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gray, S. M. (2017). PRC2-Mediated H3K27me3 Repression Promotes Effector CD8 + T Cell Terminal Differentiation and Loss of Multipotency. (Thesis). Yale University. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=10584947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gray, Simon Matthew. “PRC2-Mediated H3K27me3 Repression Promotes Effector CD8 + T Cell Terminal Differentiation and Loss of Multipotency.” 2017. Thesis, Yale University. Accessed March 02, 2021. http://pqdtopen.proquest.com/#viewpdf?dispub=10584947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gray, Simon Matthew. “PRC2-Mediated H3K27me3 Repression Promotes Effector CD8 + T Cell Terminal Differentiation and Loss of Multipotency.” 2017. Web. 02 Mar 2021.

Vancouver:

Gray SM. PRC2-Mediated H3K27me3 Repression Promotes Effector CD8 + T Cell Terminal Differentiation and Loss of Multipotency. [Internet] [Thesis]. Yale University; 2017. [cited 2021 Mar 02]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10584947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gray SM. PRC2-Mediated H3K27me3 Repression Promotes Effector CD8 + T Cell Terminal Differentiation and Loss of Multipotency. [Thesis]. Yale University; 2017. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10584947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

3. Barbalat, Roman. Non-nucleic acid based viral recognition.

Degree: Molecular & Cell Biology, 2011, University of California – Berkeley

 Most of our understanding about the cellular response to microbes by innate immune cells is shaped by our work with macrophages in culture. Because there… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Barbalat, R. (2011). Non-nucleic acid based viral recognition. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/6nc0h9f7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barbalat, Roman. “Non-nucleic acid based viral recognition.” 2011. Thesis, University of California – Berkeley. Accessed March 02, 2021. http://www.escholarship.org/uc/item/6nc0h9f7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barbalat, Roman. “Non-nucleic acid based viral recognition.” 2011. Web. 02 Mar 2021.

Vancouver:

Barbalat R. Non-nucleic acid based viral recognition. [Internet] [Thesis]. University of California – Berkeley; 2011. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/6nc0h9f7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barbalat R. Non-nucleic acid based viral recognition. [Thesis]. University of California – Berkeley; 2011. Available from: http://www.escholarship.org/uc/item/6nc0h9f7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

4. Ushach, Irina. Functional characterization of IL-39, a novel cytokine produced by macrophages and barrier tissues.

Degree: Biological Sciences, 2015, University of California – Irvine

 Cytokines are fundamental components of the immune system and understanding their biology is a necessary step in understanding immune functions in both health and disease.… (more)

Subjects/Keywords: Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ushach, I. (2015). Functional characterization of IL-39, a novel cytokine produced by macrophages and barrier tissues. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/1775x7fp

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ushach, Irina. “Functional characterization of IL-39, a novel cytokine produced by macrophages and barrier tissues.” 2015. Thesis, University of California – Irvine. Accessed March 02, 2021. http://www.escholarship.org/uc/item/1775x7fp.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ushach, Irina. “Functional characterization of IL-39, a novel cytokine produced by macrophages and barrier tissues.” 2015. Web. 02 Mar 2021.

Vancouver:

Ushach I. Functional characterization of IL-39, a novel cytokine produced by macrophages and barrier tissues. [Internet] [Thesis]. University of California – Irvine; 2015. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/1775x7fp.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ushach I. Functional characterization of IL-39, a novel cytokine produced by macrophages and barrier tissues. [Thesis]. University of California – Irvine; 2015. Available from: http://www.escholarship.org/uc/item/1775x7fp

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

5. Yamada, Douglas. Suppression of Fcγ-Receptor-Mediated Antibody Effector Function during Persistent Viral Infection.

Degree: Microbiology, Immunology, & Molecular Genetics, 2015, UCLA

 Understanding how viruses subvert host immunity and persist is essential for developing strategies to eliminate infection. T cell exhaustion during chronic viral infection is well… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Yamada, D. (2015). Suppression of Fcγ-Receptor-Mediated Antibody Effector Function during Persistent Viral Infection. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/1b2623wg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamada, Douglas. “Suppression of Fcγ-Receptor-Mediated Antibody Effector Function during Persistent Viral Infection.” 2015. Thesis, UCLA. Accessed March 02, 2021. http://www.escholarship.org/uc/item/1b2623wg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamada, Douglas. “Suppression of Fcγ-Receptor-Mediated Antibody Effector Function during Persistent Viral Infection.” 2015. Web. 02 Mar 2021.

Vancouver:

Yamada D. Suppression of Fcγ-Receptor-Mediated Antibody Effector Function during Persistent Viral Infection. [Internet] [Thesis]. UCLA; 2015. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/1b2623wg.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamada D. Suppression of Fcγ-Receptor-Mediated Antibody Effector Function during Persistent Viral Infection. [Thesis]. UCLA; 2015. Available from: http://www.escholarship.org/uc/item/1b2623wg

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

6. Vazquez, Monica Ivonne. Functional Characterization of IL-40 a novel B cell cytokine and Physiological implications of CCL28 ablation.

Degree: Biomedical Sciences, 2015, University of California – Irvine

 Immune responses are regulated by an intricate balance between cytokine production and actions of various immune cell types. Cytokines are key regulators of the immune… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Vazquez, M. I. (2015). Functional Characterization of IL-40 a novel B cell cytokine and Physiological implications of CCL28 ablation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/2m7018vx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vazquez, Monica Ivonne. “Functional Characterization of IL-40 a novel B cell cytokine and Physiological implications of CCL28 ablation.” 2015. Thesis, University of California – Irvine. Accessed March 02, 2021. http://www.escholarship.org/uc/item/2m7018vx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vazquez, Monica Ivonne. “Functional Characterization of IL-40 a novel B cell cytokine and Physiological implications of CCL28 ablation.” 2015. Web. 02 Mar 2021.

Vancouver:

Vazquez MI. Functional Characterization of IL-40 a novel B cell cytokine and Physiological implications of CCL28 ablation. [Internet] [Thesis]. University of California – Irvine; 2015. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/2m7018vx.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vazquez MI. Functional Characterization of IL-40 a novel B cell cytokine and Physiological implications of CCL28 ablation. [Thesis]. University of California – Irvine; 2015. Available from: http://www.escholarship.org/uc/item/2m7018vx

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

7. Dang, Angeline Tilly. Antimicrobial mechanisms in response to Mycobaterium leprae infection.

Degree: Microbiology, Immunology, & Molecular Genetics, 2016, UCLA

 The human body is constantly exposed to a myriad of bacterial organisms, and inmost cases, colonization of the host by these commensal bacteria is harmless… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Dang, A. T. (2016). Antimicrobial mechanisms in response to Mycobaterium leprae infection. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/16d8q45t

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dang, Angeline Tilly. “Antimicrobial mechanisms in response to Mycobaterium leprae infection.” 2016. Thesis, UCLA. Accessed March 02, 2021. http://www.escholarship.org/uc/item/16d8q45t.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dang, Angeline Tilly. “Antimicrobial mechanisms in response to Mycobaterium leprae infection.” 2016. Web. 02 Mar 2021.

Vancouver:

Dang AT. Antimicrobial mechanisms in response to Mycobaterium leprae infection. [Internet] [Thesis]. UCLA; 2016. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/16d8q45t.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dang AT. Antimicrobial mechanisms in response to Mycobaterium leprae infection. [Thesis]. UCLA; 2016. Available from: http://www.escholarship.org/uc/item/16d8q45t

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

8. Lee, Peishan. B cell-intrinsic functions of the MALT1 paracaspase.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 During a T cell-dependent immune response, formation of the germinal center (GC) is essential for the generation of high-affinity plasma cells and memory B cells.… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Lee, P. (2016). B cell-intrinsic functions of the MALT1 paracaspase. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/2s12g343

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Peishan. “B cell-intrinsic functions of the MALT1 paracaspase.” 2016. Thesis, University of California – San Diego. Accessed March 02, 2021. http://www.escholarship.org/uc/item/2s12g343.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Peishan. “B cell-intrinsic functions of the MALT1 paracaspase.” 2016. Web. 02 Mar 2021.

Vancouver:

Lee P. B cell-intrinsic functions of the MALT1 paracaspase. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/2s12g343.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee P. B cell-intrinsic functions of the MALT1 paracaspase. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/2s12g343

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

9. CHEN, Shuwen. New roles for Id3 in B and T cell development.

Degree: Biology, 2016, University of California – San Diego

 E proteins have been shown to play an important role during various stages of B and T cell development. E proteins execute this regulatory function… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

CHEN, S. (2016). New roles for Id3 in B and T cell development. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/50g0w8t6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CHEN, Shuwen. “New roles for Id3 in B and T cell development.” 2016. Thesis, University of California – San Diego. Accessed March 02, 2021. http://www.escholarship.org/uc/item/50g0w8t6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CHEN, Shuwen. “New roles for Id3 in B and T cell development.” 2016. Web. 02 Mar 2021.

Vancouver:

CHEN S. New roles for Id3 in B and T cell development. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/50g0w8t6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHEN S. New roles for Id3 in B and T cell development. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/50g0w8t6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

10. Shifrin, Nataliya. Responsiveness and Tolerance of Natural Killer Cells.

Degree: Molecular & Cell Biology, 2013, University of California – Berkeley

 A major role of natural killer cells is distinguishing between “self” and “non–self”. This is accomplished through their ability to recognize MHC class I molecules… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Shifrin, N. (2013). Responsiveness and Tolerance of Natural Killer Cells. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/3sz7c3fd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shifrin, Nataliya. “Responsiveness and Tolerance of Natural Killer Cells.” 2013. Thesis, University of California – Berkeley. Accessed March 02, 2021. http://www.escholarship.org/uc/item/3sz7c3fd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shifrin, Nataliya. “Responsiveness and Tolerance of Natural Killer Cells.” 2013. Web. 02 Mar 2021.

Vancouver:

Shifrin N. Responsiveness and Tolerance of Natural Killer Cells. [Internet] [Thesis]. University of California – Berkeley; 2013. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/3sz7c3fd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shifrin N. Responsiveness and Tolerance of Natural Killer Cells. [Thesis]. University of California – Berkeley; 2013. Available from: http://www.escholarship.org/uc/item/3sz7c3fd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

11. Lu, Jennifer. Regulation of Necroptosis and Autophagy in T cell Homeostasis and Function.

Degree: Biological Sciences, 2014, University of California – Irvine

 Development of functional adaptive and innate immune responses requires strict regulation of programmed cell death signaling pathways. These signaling pathways are essential for shaping and… (more)

Subjects/Keywords: Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lu, J. (2014). Regulation of Necroptosis and Autophagy in T cell Homeostasis and Function. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/8kf7c50q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lu, Jennifer. “Regulation of Necroptosis and Autophagy in T cell Homeostasis and Function.” 2014. Thesis, University of California – Irvine. Accessed March 02, 2021. http://www.escholarship.org/uc/item/8kf7c50q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lu, Jennifer. “Regulation of Necroptosis and Autophagy in T cell Homeostasis and Function.” 2014. Web. 02 Mar 2021.

Vancouver:

Lu J. Regulation of Necroptosis and Autophagy in T cell Homeostasis and Function. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/8kf7c50q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lu J. Regulation of Necroptosis and Autophagy in T cell Homeostasis and Function. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/8kf7c50q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

12. Mouchess, Maria Luz. Self/Non-self Discrimination by Toll-like Receptor 9.

Degree: Molecular & Cell Biology, 2011, University of California – Berkeley

 Recognition of nucleic acids as a signature of infection by Toll-like receptors (TLRs) 7 and 9 exposes the host to potential self-recognition and autoimmunity. It… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Mouchess, M. L. (2011). Self/Non-self Discrimination by Toll-like Receptor 9. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/1s96k38c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mouchess, Maria Luz. “Self/Non-self Discrimination by Toll-like Receptor 9.” 2011. Thesis, University of California – Berkeley. Accessed March 02, 2021. http://www.escholarship.org/uc/item/1s96k38c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mouchess, Maria Luz. “Self/Non-self Discrimination by Toll-like Receptor 9.” 2011. Web. 02 Mar 2021.

Vancouver:

Mouchess ML. Self/Non-self Discrimination by Toll-like Receptor 9. [Internet] [Thesis]. University of California – Berkeley; 2011. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/1s96k38c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mouchess ML. Self/Non-self Discrimination by Toll-like Receptor 9. [Thesis]. University of California – Berkeley; 2011. Available from: http://www.escholarship.org/uc/item/1s96k38c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

13. Mok, Ka Ho Stephen. Combination Treatment for Melanoma.

Degree: Molec & Med Pharmacology, 2014, UCLA

 Targeted therapies like vemurafenib and dabrafenib that block oncogenic BRAF result in high response rates and improved overall survival in patients with melanoma. However, the… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Mok, K. H. S. (2014). Combination Treatment for Melanoma. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/55g4z78w

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mok, Ka Ho Stephen. “Combination Treatment for Melanoma.” 2014. Thesis, UCLA. Accessed March 02, 2021. http://www.escholarship.org/uc/item/55g4z78w.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mok, Ka Ho Stephen. “Combination Treatment for Melanoma.” 2014. Web. 02 Mar 2021.

Vancouver:

Mok KHS. Combination Treatment for Melanoma. [Internet] [Thesis]. UCLA; 2014. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/55g4z78w.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mok KHS. Combination Treatment for Melanoma. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/55g4z78w

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

14. Kibbie, Jon J. Exploration of Endothelial Cell Directed Innate Immunity in Leprosy.

Degree: Microbiology, Immunology, & Molecular Genetics, 2013, UCLA

 As an initial point of contact with circulating innate immune precursor cells, the endothelium is poised to deliver instructive signals that influence innate immune cell… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Kibbie, J. J. (2013). Exploration of Endothelial Cell Directed Innate Immunity in Leprosy. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/8f76t9d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kibbie, Jon J. “Exploration of Endothelial Cell Directed Innate Immunity in Leprosy.” 2013. Thesis, UCLA. Accessed March 02, 2021. http://www.escholarship.org/uc/item/8f76t9d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kibbie, Jon J. “Exploration of Endothelial Cell Directed Innate Immunity in Leprosy.” 2013. Web. 02 Mar 2021.

Vancouver:

Kibbie JJ. Exploration of Endothelial Cell Directed Innate Immunity in Leprosy. [Internet] [Thesis]. UCLA; 2013. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/8f76t9d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kibbie JJ. Exploration of Endothelial Cell Directed Innate Immunity in Leprosy. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/8f76t9d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

15. Chiu, Honyin. The PI3K/mTOR/eIF4E Signaling Network in B Cell Differentiation.

Degree: Biological Sciences, 2018, University of California – Irvine

 As humans, we have evolved to build a complex humoral immune response against infection. The process of differentiating B cells into antibody secreting cells is… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Chiu, H. (2018). The PI3K/mTOR/eIF4E Signaling Network in B Cell Differentiation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/6x49d7g3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chiu, Honyin. “The PI3K/mTOR/eIF4E Signaling Network in B Cell Differentiation.” 2018. Thesis, University of California – Irvine. Accessed March 02, 2021. http://www.escholarship.org/uc/item/6x49d7g3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chiu, Honyin. “The PI3K/mTOR/eIF4E Signaling Network in B Cell Differentiation.” 2018. Web. 02 Mar 2021.

Vancouver:

Chiu H. The PI3K/mTOR/eIF4E Signaling Network in B Cell Differentiation. [Internet] [Thesis]. University of California – Irvine; 2018. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/6x49d7g3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chiu H. The PI3K/mTOR/eIF4E Signaling Network in B Cell Differentiation. [Thesis]. University of California – Irvine; 2018. Available from: http://www.escholarship.org/uc/item/6x49d7g3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

16. Marcovecchio, Paola. Insights into the mechanism and activation of nonclassical monocyte patrolling in murine models of atherosclerosis and metastatic melanoma.

Degree: Biomedical Sciences, 2018, University of California – San Diego

 Patrolling is a unique surveillance phenotype carried out by a subset of monocytes (nonclassical; Ly6C- mouse or CD16+ human) on the endothelium of blood vessels… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Marcovecchio, P. (2018). Insights into the mechanism and activation of nonclassical monocyte patrolling in murine models of atherosclerosis and metastatic melanoma. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/89f4w5vc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marcovecchio, Paola. “Insights into the mechanism and activation of nonclassical monocyte patrolling in murine models of atherosclerosis and metastatic melanoma.” 2018. Thesis, University of California – San Diego. Accessed March 02, 2021. http://www.escholarship.org/uc/item/89f4w5vc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marcovecchio, Paola. “Insights into the mechanism and activation of nonclassical monocyte patrolling in murine models of atherosclerosis and metastatic melanoma.” 2018. Web. 02 Mar 2021.

Vancouver:

Marcovecchio P. Insights into the mechanism and activation of nonclassical monocyte patrolling in murine models of atherosclerosis and metastatic melanoma. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/89f4w5vc.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marcovecchio P. Insights into the mechanism and activation of nonclassical monocyte patrolling in murine models of atherosclerosis and metastatic melanoma. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/89f4w5vc

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

17. Kong, Weimin. THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS.

Degree: PhD, 2010, Temple University

Physiology

Dendritic cells (DC) are professional antigen presenting cells which link innate and adaptive immunity through recognition and processing of pathogens, migration to secondary lymph… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Kong, W. (2010). THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,119523

Chicago Manual of Style (16th Edition):

Kong, Weimin. “THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS.” 2010. Doctoral Dissertation, Temple University. Accessed March 02, 2021. http://digital.library.temple.edu/u?/p245801coll10,119523.

MLA Handbook (7th Edition):

Kong, Weimin. “THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS.” 2010. Web. 02 Mar 2021.

Vancouver:

Kong W. THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2021 Mar 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,119523.

Council of Science Editors:

Kong W. THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,119523


Temple University

18. Robinson, Rebecca Hartzell. Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Cannabinoids are known to have anti-inflammatory and immunomodulatory properties. Cannabinoid receptor 2 (CB2) is expressed mainly on leukocytes and is the receptor… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Robinson, R. H. (2014). Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,246094

Chicago Manual of Style (16th Edition):

Robinson, Rebecca Hartzell. “Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection.” 2014. Doctoral Dissertation, Temple University. Accessed March 02, 2021. http://digital.library.temple.edu/u?/p245801coll10,246094.

MLA Handbook (7th Edition):

Robinson, Rebecca Hartzell. “Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection.” 2014. Web. 02 Mar 2021.

Vancouver:

Robinson RH. Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2021 Mar 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,246094.

Council of Science Editors:

Robinson RH. Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,246094


University of California – San Diego

19. Venkatesh, Hrishi. Type-I interferon regulates resistance to carboplatin in the human ovarian cancer cell line CAOV3.

Degree: Biology, 2018, University of California – San Diego

 Interferons are a group of signaling proteins produced by host cells in response to infection by a variety of pathogens, as well as during cancer.… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Venkatesh, H. (2018). Type-I interferon regulates resistance to carboplatin in the human ovarian cancer cell line CAOV3. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/25r67440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Venkatesh, Hrishi. “Type-I interferon regulates resistance to carboplatin in the human ovarian cancer cell line CAOV3.” 2018. Thesis, University of California – San Diego. Accessed March 02, 2021. http://www.escholarship.org/uc/item/25r67440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Venkatesh, Hrishi. “Type-I interferon regulates resistance to carboplatin in the human ovarian cancer cell line CAOV3.” 2018. Web. 02 Mar 2021.

Vancouver:

Venkatesh H. Type-I interferon regulates resistance to carboplatin in the human ovarian cancer cell line CAOV3. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/25r67440.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Venkatesh H. Type-I interferon regulates resistance to carboplatin in the human ovarian cancer cell line CAOV3. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/25r67440

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

20. Yu, Bingfei. Multi-layered epigenetic control of T cell fate decisions.

Degree: Biology, 2018, University of California – San Diego

 CD8 + T cells are a central component of the adaptive immune system. Upon infection, a naive CD8 + T cell will differentiate into a… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Yu, B. (2018). Multi-layered epigenetic control of T cell fate decisions. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/8rs7c7b3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yu, Bingfei. “Multi-layered epigenetic control of T cell fate decisions.” 2018. Thesis, University of California – San Diego. Accessed March 02, 2021. http://www.escholarship.org/uc/item/8rs7c7b3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yu, Bingfei. “Multi-layered epigenetic control of T cell fate decisions.” 2018. Web. 02 Mar 2021.

Vancouver:

Yu B. Multi-layered epigenetic control of T cell fate decisions. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/8rs7c7b3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yu B. Multi-layered epigenetic control of T cell fate decisions. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/8rs7c7b3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

21. Wang, Jie. MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES.

Degree: PhD, Immunology and Infectious Disease, 2017, Cornell University

 Neonatal infection is a major cause of morbidity and mortality worldwide. While adults generate robust immunity to most intracellular pathogens, neonates have an impaired ability… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Wang, J. (2017). MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/59007

Chicago Manual of Style (16th Edition):

Wang, Jie. “MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES.” 2017. Doctoral Dissertation, Cornell University. Accessed March 02, 2021. http://hdl.handle.net/1813/59007.

MLA Handbook (7th Edition):

Wang, Jie. “MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES.” 2017. Web. 02 Mar 2021.

Vancouver:

Wang J. MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Mar 02]. Available from: http://hdl.handle.net/1813/59007.

Council of Science Editors:

Wang J. MECHANISMS UNDERLYING NEONATAL CD8+ T CELL DEVELOPMENT AND RESPONSES. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/59007

22. Xu, Yun. Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections.

Degree: Pathobiology, 2018, Brown University

 Abstract of “Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections” by Yun Xu, Ph.D., Brown University, May 2018 Abstract… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Xu, Y. (2018). Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections. (Thesis). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:792768/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xu, Yun. “Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections.” 2018. Thesis, Brown University. Accessed March 02, 2021. https://repository.library.brown.edu/studio/item/bdr:792768/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xu, Yun. “Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections.” 2018. Web. 02 Mar 2021.

Vancouver:

Xu Y. Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections. [Internet] [Thesis]. Brown University; 2018. [cited 2021 Mar 02]. Available from: https://repository.library.brown.edu/studio/item/bdr:792768/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xu Y. Modeling Innate Immune Triage: The Early Responses to Concurrent Dermal Wounds and Pulmonary Infections. [Thesis]. Brown University; 2018. Available from: https://repository.library.brown.edu/studio/item/bdr:792768/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Erick, Timothy. Group 1 Innate Lymphoid Cells of the Submandibular Salivary Gland and Lacrimal Gland.

Degree: Department of Molecular Biology, Cell Biology and Biochemistry, 2017, Brown University

 Innate lymphoid cells (ILCs) are diverse innate lymphocytes that contribute to the immune response against pathogens and cancer cells, and play a variety of roles… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Erick, T. (2017). Group 1 Innate Lymphoid Cells of the Submandibular Salivary Gland and Lacrimal Gland. (Thesis). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:733321/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Erick, Timothy. “Group 1 Innate Lymphoid Cells of the Submandibular Salivary Gland and Lacrimal Gland.” 2017. Thesis, Brown University. Accessed March 02, 2021. https://repository.library.brown.edu/studio/item/bdr:733321/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Erick, Timothy. “Group 1 Innate Lymphoid Cells of the Submandibular Salivary Gland and Lacrimal Gland.” 2017. Web. 02 Mar 2021.

Vancouver:

Erick T. Group 1 Innate Lymphoid Cells of the Submandibular Salivary Gland and Lacrimal Gland. [Internet] [Thesis]. Brown University; 2017. [cited 2021 Mar 02]. Available from: https://repository.library.brown.edu/studio/item/bdr:733321/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Erick T. Group 1 Innate Lymphoid Cells of the Submandibular Salivary Gland and Lacrimal Gland. [Thesis]. Brown University; 2017. Available from: https://repository.library.brown.edu/studio/item/bdr:733321/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Andrews, Christina. A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment.

Degree: Biomedical Engineering, 2018, Brown University

 Neutrophils are the most abundant circulating white blood cell in the human body, and play a crucial role in the innate immune response to infection… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Andrews, C. (2018). A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment. (Thesis). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:792823/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andrews, Christina. “A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment.” 2018. Thesis, Brown University. Accessed March 02, 2021. https://repository.library.brown.edu/studio/item/bdr:792823/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andrews, Christina. “A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment.” 2018. Web. 02 Mar 2021.

Vancouver:

Andrews C. A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment. [Internet] [Thesis]. Brown University; 2018. [cited 2021 Mar 02]. Available from: https://repository.library.brown.edu/studio/item/bdr:792823/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andrews C. A Biophysical Analysis of Neutrophil Force Generation in a Biochemical Environment. [Thesis]. Brown University; 2018. Available from: https://repository.library.brown.edu/studio/item/bdr:792823/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

25. Omid, Shaida. The role of glycoprotein 130 in homing.

Degree: MS, Medical Sciences, 2015, Boston University

 Coordinated lymphocyte adhesion and migration is a hallmark of the adaptive immune response in both physiological and pathological conditions. Therefore, understanding the mechanisms underlying lymphocyte… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Omid, S. (2015). The role of glycoprotein 130 in homing. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16246

Chicago Manual of Style (16th Edition):

Omid, Shaida. “The role of glycoprotein 130 in homing.” 2015. Masters Thesis, Boston University. Accessed March 02, 2021. http://hdl.handle.net/2144/16246.

MLA Handbook (7th Edition):

Omid, Shaida. “The role of glycoprotein 130 in homing.” 2015. Web. 02 Mar 2021.

Vancouver:

Omid S. The role of glycoprotein 130 in homing. [Internet] [Masters thesis]. Boston University; 2015. [cited 2021 Mar 02]. Available from: http://hdl.handle.net/2144/16246.

Council of Science Editors:

Omid S. The role of glycoprotein 130 in homing. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16246


Boston University

26. Babwah, Amaara. Immune response to BK virus reactivation in renal transplant recipients.

Degree: MS, Medical Sciences, 2016, Boston University

 BK virus, a virus of the polyomaviridae family, is a latent infection in up to 70% of the general population. However, it has been an… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Babwah, A. (2016). Immune response to BK virus reactivation in renal transplant recipients. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16803

Chicago Manual of Style (16th Edition):

Babwah, Amaara. “Immune response to BK virus reactivation in renal transplant recipients.” 2016. Masters Thesis, Boston University. Accessed March 02, 2021. http://hdl.handle.net/2144/16803.

MLA Handbook (7th Edition):

Babwah, Amaara. “Immune response to BK virus reactivation in renal transplant recipients.” 2016. Web. 02 Mar 2021.

Vancouver:

Babwah A. Immune response to BK virus reactivation in renal transplant recipients. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Mar 02]. Available from: http://hdl.handle.net/2144/16803.

Council of Science Editors:

Babwah A. Immune response to BK virus reactivation in renal transplant recipients. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16803


UCLA

27. Kim, Elliot. Host-Pathogen interactions of the innate immune system.

Degree: Molecular Biology, 2018, UCLA

 Understanding host-pathogen interactions between microbes and the innate immune system will provide insight into the host defense pathways and microbial virulence factors. The macrophage (MΦ)… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Kim, E. (2018). Host-Pathogen interactions of the innate immune system. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/4ws8q910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Elliot. “Host-Pathogen interactions of the innate immune system.” 2018. Thesis, UCLA. Accessed March 02, 2021. http://www.escholarship.org/uc/item/4ws8q910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Elliot. “Host-Pathogen interactions of the innate immune system.” 2018. Web. 02 Mar 2021.

Vancouver:

Kim E. Host-Pathogen interactions of the innate immune system. [Internet] [Thesis]. UCLA; 2018. [cited 2021 Mar 02]. Available from: http://www.escholarship.org/uc/item/4ws8q910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim E. Host-Pathogen interactions of the innate immune system. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/4ws8q910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

28. Sage, Peter The. Mechanisms of CD4 T Cell Antigen Recognition and Effector Cell Differentiation and Function.

Degree: PhD, Immunology, 2013, Harvard University

 The ability for CD4 T cells to efficiently search for and subsequently respond to microbial pathogens is essential for protective immunity, but mechanisms controlling these… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Sage, P. T. (2013). Mechanisms of CD4 T Cell Antigen Recognition and Effector Cell Differentiation and Function. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:10416137

Chicago Manual of Style (16th Edition):

Sage, Peter The. “Mechanisms of CD4 T Cell Antigen Recognition and Effector Cell Differentiation and Function.” 2013. Doctoral Dissertation, Harvard University. Accessed March 02, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:10416137.

MLA Handbook (7th Edition):

Sage, Peter The. “Mechanisms of CD4 T Cell Antigen Recognition and Effector Cell Differentiation and Function.” 2013. Web. 02 Mar 2021.

Vancouver:

Sage PT. Mechanisms of CD4 T Cell Antigen Recognition and Effector Cell Differentiation and Function. [Internet] [Doctoral dissertation]. Harvard University; 2013. [cited 2021 Mar 02]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10416137.

Council of Science Editors:

Sage PT. Mechanisms of CD4 T Cell Antigen Recognition and Effector Cell Differentiation and Function. [Doctoral Dissertation]. Harvard University; 2013. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10416137


Harvard University

29. Sitrin, Jonathan Ryan. Regulation and Heterogeneity of Pancreatic Natural Killer Cells During Type 1 Diabetes.

Degree: PhD, Biology: Medical Sciences, Division of, 2014, Harvard University

 The vertebrate immune system contains a diverse inventory of genetic, epigenetic, molecular and cellular mechanisms dedicated to distinguishing and to determining appropriate responsiveness to "self"… (more)

Subjects/Keywords: Immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sitrin, J. R. (2014). Regulation and Heterogeneity of Pancreatic Natural Killer Cells During Type 1 Diabetes. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070062

Chicago Manual of Style (16th Edition):

Sitrin, Jonathan Ryan. “Regulation and Heterogeneity of Pancreatic Natural Killer Cells During Type 1 Diabetes.” 2014. Doctoral Dissertation, Harvard University. Accessed March 02, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070062.

MLA Handbook (7th Edition):

Sitrin, Jonathan Ryan. “Regulation and Heterogeneity of Pancreatic Natural Killer Cells During Type 1 Diabetes.” 2014. Web. 02 Mar 2021.

Vancouver:

Sitrin JR. Regulation and Heterogeneity of Pancreatic Natural Killer Cells During Type 1 Diabetes. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2021 Mar 02]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070062.

Council of Science Editors:

Sitrin JR. Regulation and Heterogeneity of Pancreatic Natural Killer Cells During Type 1 Diabetes. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070062


Wake Forest University

30. Tang, Shuai. BOTH EFFECTOR AND INHIBITORY IMMUNE MECHANISMS ARE AMPLIFIED IN CASTRATION-RESISTANT PROSTATE CANCER.

Degree: 2012, Wake Forest University

 Immune function is modulated by androgen ablation therapy for prostate cancer. Androgen ablation leads to apoptosis of primary prostate tumor and transient potentiation of immune… (more)

Subjects/Keywords: immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tang, S. (2012). BOTH EFFECTOR AND INHIBITORY IMMUNE MECHANISMS ARE AMPLIFIED IN CASTRATION-RESISTANT PROSTATE CANCER. (Thesis). Wake Forest University. Retrieved from http://hdl.handle.net/10339/37656

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tang, Shuai. “BOTH EFFECTOR AND INHIBITORY IMMUNE MECHANISMS ARE AMPLIFIED IN CASTRATION-RESISTANT PROSTATE CANCER.” 2012. Thesis, Wake Forest University. Accessed March 02, 2021. http://hdl.handle.net/10339/37656.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tang, Shuai. “BOTH EFFECTOR AND INHIBITORY IMMUNE MECHANISMS ARE AMPLIFIED IN CASTRATION-RESISTANT PROSTATE CANCER.” 2012. Web. 02 Mar 2021.

Vancouver:

Tang S. BOTH EFFECTOR AND INHIBITORY IMMUNE MECHANISMS ARE AMPLIFIED IN CASTRATION-RESISTANT PROSTATE CANCER. [Internet] [Thesis]. Wake Forest University; 2012. [cited 2021 Mar 02]. Available from: http://hdl.handle.net/10339/37656.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tang S. BOTH EFFECTOR AND INHIBITORY IMMUNE MECHANISMS ARE AMPLIFIED IN CASTRATION-RESISTANT PROSTATE CANCER. [Thesis]. Wake Forest University; 2012. Available from: http://hdl.handle.net/10339/37656

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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