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You searched for subject:(immune evasion). Showing records 1 – 30 of 125 total matches.

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University of Illinois – Urbana-Champaign

1. Randall, Crystal. Characterization of the anti-inflammatory effects of the Molluscum Contagiosum MC159 protein.

Degree: PhD, 0322, 2013, University of Illinois – Urbana-Champaign

 The Molluscum Contagiosum Virus (MCV) is a dermotropic poxvirus that strictly infects humans. MCV infections produce umbilicated lesions that can persist for six to nine… (more)

Subjects/Keywords: Poxviruses; Immune evasion

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Randall, C. (2013). Characterization of the anti-inflammatory effects of the Molluscum Contagiosum MC159 protein. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/44818

Chicago Manual of Style (16th Edition):

Randall, Crystal. “Characterization of the anti-inflammatory effects of the Molluscum Contagiosum MC159 protein.” 2013. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed August 13, 2020. http://hdl.handle.net/2142/44818.

MLA Handbook (7th Edition):

Randall, Crystal. “Characterization of the anti-inflammatory effects of the Molluscum Contagiosum MC159 protein.” 2013. Web. 13 Aug 2020.

Vancouver:

Randall C. Characterization of the anti-inflammatory effects of the Molluscum Contagiosum MC159 protein. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2013. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/2142/44818.

Council of Science Editors:

Randall C. Characterization of the anti-inflammatory effects of the Molluscum Contagiosum MC159 protein. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2013. Available from: http://hdl.handle.net/2142/44818


University of Alberta

2. Oladiran, Ayoola. Studies on the mechanisms of immune evasion in Trypanosoma carassii infections of the goldfish (Carassius auratus).

Degree: PhD, Department of Biological Sciences, 2012, University of Alberta

 Parasites possess variety of mechanisms to modulate or evade host defence systems to maintain chronic infection and ensure their transmission. The protozoan parasite Trypanosoma carassii… (more)

Subjects/Keywords: Trypanosoma carassii; Goldfish; Immune evasion

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APA (6th Edition):

Oladiran, A. (2012). Studies on the mechanisms of immune evasion in Trypanosoma carassii infections of the goldfish (Carassius auratus). (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/3197xm80c

Chicago Manual of Style (16th Edition):

Oladiran, Ayoola. “Studies on the mechanisms of immune evasion in Trypanosoma carassii infections of the goldfish (Carassius auratus).” 2012. Doctoral Dissertation, University of Alberta. Accessed August 13, 2020. https://era.library.ualberta.ca/files/3197xm80c.

MLA Handbook (7th Edition):

Oladiran, Ayoola. “Studies on the mechanisms of immune evasion in Trypanosoma carassii infections of the goldfish (Carassius auratus).” 2012. Web. 13 Aug 2020.

Vancouver:

Oladiran A. Studies on the mechanisms of immune evasion in Trypanosoma carassii infections of the goldfish (Carassius auratus). [Internet] [Doctoral dissertation]. University of Alberta; 2012. [cited 2020 Aug 13]. Available from: https://era.library.ualberta.ca/files/3197xm80c.

Council of Science Editors:

Oladiran A. Studies on the mechanisms of immune evasion in Trypanosoma carassii infections of the goldfish (Carassius auratus). [Doctoral Dissertation]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/3197xm80c


Universiteit Utrecht

3. Ravesloot, M.M. The coagulation system and its role in bacterial infections.

Degree: 2012, Universiteit Utrecht

 The coagulation system and its inhibitors maintain the hemostatic balance during injury and can modulate the inflammatory response. In addition, the coagulation system is important… (more)

Subjects/Keywords: Coagulation; Immune evasion; Bacteria, Complement

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APA (6th Edition):

Ravesloot, M. M. (2012). The coagulation system and its role in bacterial infections. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/256848

Chicago Manual of Style (16th Edition):

Ravesloot, M M. “The coagulation system and its role in bacterial infections.” 2012. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/256848.

MLA Handbook (7th Edition):

Ravesloot, M M. “The coagulation system and its role in bacterial infections.” 2012. Web. 13 Aug 2020.

Vancouver:

Ravesloot MM. The coagulation system and its role in bacterial infections. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/256848.

Council of Science Editors:

Ravesloot MM. The coagulation system and its role in bacterial infections. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/256848


Universiteit Utrecht

4. Jong, O.G. de. Immune evasion in Staphylococcus aureus.

Degree: 2010, Universiteit Utrecht

 Staphylococcus aureus expresses many proteins that are involved in evasion or downregulation of the human immune system. Proteins involved in evading the complement system and… (more)

Subjects/Keywords: Geneeskunde; Immune evasion Staphylococcus aureus

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APA (6th Edition):

Jong, O. G. d. (2010). Immune evasion in Staphylococcus aureus. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/44321

Chicago Manual of Style (16th Edition):

Jong, O G de. “Immune evasion in Staphylococcus aureus.” 2010. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/44321.

MLA Handbook (7th Edition):

Jong, O G de. “Immune evasion in Staphylococcus aureus.” 2010. Web. 13 Aug 2020.

Vancouver:

Jong OGd. Immune evasion in Staphylococcus aureus. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/44321.

Council of Science Editors:

Jong OGd. Immune evasion in Staphylococcus aureus. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/44321


Universiteit Utrecht

5. Altena, S.E.C. van. Tumor surveillance and immune evasion: the interplay between the immune system and tumor cells.

Degree: 2011, Universiteit Utrecht

 This thesis encloses the interplay between the immune system and tumor cells. The immune system is, next to defending the body against pathogens, specialized in… (more)

Subjects/Keywords: tumor surveillance; immune evasion

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APA (6th Edition):

Altena, S. E. C. v. (2011). Tumor surveillance and immune evasion: the interplay between the immune system and tumor cells. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/193270

Chicago Manual of Style (16th Edition):

Altena, S E C van. “Tumor surveillance and immune evasion: the interplay between the immune system and tumor cells.” 2011. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/193270.

MLA Handbook (7th Edition):

Altena, S E C van. “Tumor surveillance and immune evasion: the interplay between the immune system and tumor cells.” 2011. Web. 13 Aug 2020.

Vancouver:

Altena SECv. Tumor surveillance and immune evasion: the interplay between the immune system and tumor cells. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/193270.

Council of Science Editors:

Altena SECv. Tumor surveillance and immune evasion: the interplay between the immune system and tumor cells. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/193270


University of Cape Town

6. Ozturk, Mumin. Tuberculosis transcriptomics: host protection and immune evasion mechanisms.

Degree: Image, Institute of Infectious Disease and Molecular Medicine, 2017, University of Cape Town

 Mycobacterium tuberculosis (Mtb) is the leading cause of death from an infectious disease. The success of the pathogen lies in its ability to subvert hostile… (more)

Subjects/Keywords: Mycobacterium tuberculosis; immune evasion genes

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APA (6th Edition):

Ozturk, M. (2017). Tuberculosis transcriptomics: host protection and immune evasion mechanisms. (Thesis). University of Cape Town. Retrieved from http://hdl.handle.net/11427/26863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ozturk, Mumin. “Tuberculosis transcriptomics: host protection and immune evasion mechanisms.” 2017. Thesis, University of Cape Town. Accessed August 13, 2020. http://hdl.handle.net/11427/26863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ozturk, Mumin. “Tuberculosis transcriptomics: host protection and immune evasion mechanisms.” 2017. Web. 13 Aug 2020.

Vancouver:

Ozturk M. Tuberculosis transcriptomics: host protection and immune evasion mechanisms. [Internet] [Thesis]. University of Cape Town; 2017. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/11427/26863.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ozturk M. Tuberculosis transcriptomics: host protection and immune evasion mechanisms. [Thesis]. University of Cape Town; 2017. Available from: http://hdl.handle.net/11427/26863

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

7. Karssemeijer, R.A. Bacterial immune evasion proteins as laboratory tools.

Degree: 2010, Universiteit Utrecht

Bacteria use several strategies to evade the host immune system. Interestingly, many of these proteins are nowadays efficiently used as laboratory tools. This thesis will describe these bacterial proteins and how they are used in laboratory settings. Advisors/Committee Members: van Strijp, JAG.

Subjects/Keywords: Immune evasion; Bacterial proteins; Laboratory tools

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APA (6th Edition):

Karssemeijer, R. A. (2010). Bacterial immune evasion proteins as laboratory tools. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/187100

Chicago Manual of Style (16th Edition):

Karssemeijer, R A. “Bacterial immune evasion proteins as laboratory tools.” 2010. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/187100.

MLA Handbook (7th Edition):

Karssemeijer, R A. “Bacterial immune evasion proteins as laboratory tools.” 2010. Web. 13 Aug 2020.

Vancouver:

Karssemeijer RA. Bacterial immune evasion proteins as laboratory tools. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/187100.

Council of Science Editors:

Karssemeijer RA. Bacterial immune evasion proteins as laboratory tools. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/187100


University of New South Wales

8. Voli, Florida. Tumour copper levels regulate PD-L1 driven immune evasion.

Degree: Children's Cancer Institute Australia for Medical Research, 2019, University of New South Wales

 Cancer immune evasion is recognised as a central hallmark of tumour development. One mechanism that cancer cells use to protect themselves from anti-tumour immune responses… (more)

Subjects/Keywords: PD-L1; Copper; Immune evasion; Cancer

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APA (6th Edition):

Voli, F. (2019). Tumour copper levels regulate PD-L1 driven immune evasion. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/64905

Chicago Manual of Style (16th Edition):

Voli, Florida. “Tumour copper levels regulate PD-L1 driven immune evasion.” 2019. Doctoral Dissertation, University of New South Wales. Accessed August 13, 2020. http://handle.unsw.edu.au/1959.4/64905.

MLA Handbook (7th Edition):

Voli, Florida. “Tumour copper levels regulate PD-L1 driven immune evasion.” 2019. Web. 13 Aug 2020.

Vancouver:

Voli F. Tumour copper levels regulate PD-L1 driven immune evasion. [Internet] [Doctoral dissertation]. University of New South Wales; 2019. [cited 2020 Aug 13]. Available from: http://handle.unsw.edu.au/1959.4/64905.

Council of Science Editors:

Voli F. Tumour copper levels regulate PD-L1 driven immune evasion. [Doctoral Dissertation]. University of New South Wales; 2019. Available from: http://handle.unsw.edu.au/1959.4/64905


Oklahoma State University

9. Key, Charlotte Elise. Extrusion as a Mechanism of Host Immune Response Evasion in a Chlamydia trachomatis Murine Infection Model.

Degree: Integrative Biology, 2019, Oklahoma State University

 Chlamydia trachomatis is an obligate intracellular organism that is the leading cause of preventable blindness and sexually transmitted bacterial infections. C. trachomatis exhibits a biphasic… (more)

Subjects/Keywords: chlamydia; evasion; extrusion; histopathology; immune response; murine

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APA (6th Edition):

Key, C. E. (2019). Extrusion as a Mechanism of Host Immune Response Evasion in a Chlamydia trachomatis Murine Infection Model. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/323407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Key, Charlotte Elise. “Extrusion as a Mechanism of Host Immune Response Evasion in a Chlamydia trachomatis Murine Infection Model.” 2019. Thesis, Oklahoma State University. Accessed August 13, 2020. http://hdl.handle.net/11244/323407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Key, Charlotte Elise. “Extrusion as a Mechanism of Host Immune Response Evasion in a Chlamydia trachomatis Murine Infection Model.” 2019. Web. 13 Aug 2020.

Vancouver:

Key CE. Extrusion as a Mechanism of Host Immune Response Evasion in a Chlamydia trachomatis Murine Infection Model. [Internet] [Thesis]. Oklahoma State University; 2019. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/11244/323407.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Key CE. Extrusion as a Mechanism of Host Immune Response Evasion in a Chlamydia trachomatis Murine Infection Model. [Thesis]. Oklahoma State University; 2019. Available from: http://hdl.handle.net/11244/323407

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

10. Movius, Carly Anne. Investigating immune escape mechanisms between high & low risk mucosal human papillomavirus genotypes and cutaneous human papillomavirus genotypes.

Degree: MS, Molecular Microbiology & Immunology, 2012, University of Southern California

 Cervical cancer is associated with high risk HPV genotypes 93 % of the time. HPV infections can take months and even up to a year… (more)

Subjects/Keywords: HPV; immune evasion mechanisms; Langerhans Cells

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APA (6th Edition):

Movius, C. A. (2012). Investigating immune escape mechanisms between high & low risk mucosal human papillomavirus genotypes and cutaneous human papillomavirus genotypes. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/464808/rec/3625

Chicago Manual of Style (16th Edition):

Movius, Carly Anne. “Investigating immune escape mechanisms between high & low risk mucosal human papillomavirus genotypes and cutaneous human papillomavirus genotypes.” 2012. Masters Thesis, University of Southern California. Accessed August 13, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/464808/rec/3625.

MLA Handbook (7th Edition):

Movius, Carly Anne. “Investigating immune escape mechanisms between high & low risk mucosal human papillomavirus genotypes and cutaneous human papillomavirus genotypes.” 2012. Web. 13 Aug 2020.

Vancouver:

Movius CA. Investigating immune escape mechanisms between high & low risk mucosal human papillomavirus genotypes and cutaneous human papillomavirus genotypes. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Aug 13]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/464808/rec/3625.

Council of Science Editors:

Movius CA. Investigating immune escape mechanisms between high & low risk mucosal human papillomavirus genotypes and cutaneous human papillomavirus genotypes. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/464808/rec/3625


University of Edinburgh

11. Murray, Janice. Structure and function of the VAL family in Brugia malayi and Heligmosomoides polygyrus.

Degree: PhD, 2015, University of Edinburgh

Evasion of an immune response mounted by a host is fundamental to the survival of a parasite. Immune evasion can be mediated in many ways… (more)

Subjects/Keywords: 616.9; Heligmosomoides polygyrus; helminth; immune evasion

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APA (6th Edition):

Murray, J. (2015). Structure and function of the VAL family in Brugia malayi and Heligmosomoides polygyrus. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/10539

Chicago Manual of Style (16th Edition):

Murray, Janice. “Structure and function of the VAL family in Brugia malayi and Heligmosomoides polygyrus.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed August 13, 2020. http://hdl.handle.net/1842/10539.

MLA Handbook (7th Edition):

Murray, Janice. “Structure and function of the VAL family in Brugia malayi and Heligmosomoides polygyrus.” 2015. Web. 13 Aug 2020.

Vancouver:

Murray J. Structure and function of the VAL family in Brugia malayi and Heligmosomoides polygyrus. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/1842/10539.

Council of Science Editors:

Murray J. Structure and function of the VAL family in Brugia malayi and Heligmosomoides polygyrus. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/10539

12. Barel, M.T. Downregulation of MHC class I molecules by human cytomegalovirus-encoded US2 and US11.

Degree: 2005, Leiden University, Leiden University Medical Center, Dept. of Experimental Microbiology

Subjects/Keywords: Immune evasion; HCMV; Immune evasion; HCMV

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APA (6th Edition):

Barel, M. T. (2005). Downregulation of MHC class I molecules by human cytomegalovirus-encoded US2 and US11. (Doctoral Dissertation). Leiden University, Leiden University Medical Center, Dept. of Experimental Microbiology. Retrieved from http://hdl.handle.net/1887/4294

Chicago Manual of Style (16th Edition):

Barel, M T. “Downregulation of MHC class I molecules by human cytomegalovirus-encoded US2 and US11.” 2005. Doctoral Dissertation, Leiden University, Leiden University Medical Center, Dept. of Experimental Microbiology. Accessed August 13, 2020. http://hdl.handle.net/1887/4294.

MLA Handbook (7th Edition):

Barel, M T. “Downregulation of MHC class I molecules by human cytomegalovirus-encoded US2 and US11.” 2005. Web. 13 Aug 2020.

Vancouver:

Barel MT. Downregulation of MHC class I molecules by human cytomegalovirus-encoded US2 and US11. [Internet] [Doctoral dissertation]. Leiden University, Leiden University Medical Center, Dept. of Experimental Microbiology; 2005. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/1887/4294.

Council of Science Editors:

Barel MT. Downregulation of MHC class I molecules by human cytomegalovirus-encoded US2 and US11. [Doctoral Dissertation]. Leiden University, Leiden University Medical Center, Dept. of Experimental Microbiology; 2005. Available from: http://hdl.handle.net/1887/4294


Universiteit Utrecht

13. Budding, K. Immune Evasion Strategies of Hepatitis C virus.

Degree: 2011, Universiteit Utrecht

 In this thesis I will discuss the “tug of war” between hepatitis C virus (HCV) and our innate and adaptive immune system. A chronic HCV… (more)

Subjects/Keywords: HCV; immune evasion; adaptive immune system; innate immune system; viral infection; hepatitis c virus

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APA (6th Edition):

Budding, K. (2011). Immune Evasion Strategies of Hepatitis C virus. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/194130

Chicago Manual of Style (16th Edition):

Budding, K. “Immune Evasion Strategies of Hepatitis C virus.” 2011. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/194130.

MLA Handbook (7th Edition):

Budding, K. “Immune Evasion Strategies of Hepatitis C virus.” 2011. Web. 13 Aug 2020.

Vancouver:

Budding K. Immune Evasion Strategies of Hepatitis C virus. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/194130.

Council of Science Editors:

Budding K. Immune Evasion Strategies of Hepatitis C virus. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/194130


University of California – San Diego

14. McGowan, Matthew Allen. How the Streptococcal M87 Protein Binds Human C4b-Binding Protein.

Degree: Chemistry, 2017, University of California – San Diego

 ABSTRACT OF THE THESISHow the Streptococcal M87 Protein Binds Human C4b-Binding ProteinbyMatthew Allen McGowanMaster of ScienceUniversity of California, San Diego, 2017Professor Partho Ghosh, ChairThe pathogenic… (more)

Subjects/Keywords: Biochemistry; bacterial immune evasion; bacterial pathogenesis; Streptococcus pyogenes; X-ray crystallography

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APA (6th Edition):

McGowan, M. A. (2017). How the Streptococcal M87 Protein Binds Human C4b-Binding Protein. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/8hg1b4v4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McGowan, Matthew Allen. “How the Streptococcal M87 Protein Binds Human C4b-Binding Protein.” 2017. Thesis, University of California – San Diego. Accessed August 13, 2020. http://www.escholarship.org/uc/item/8hg1b4v4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McGowan, Matthew Allen. “How the Streptococcal M87 Protein Binds Human C4b-Binding Protein.” 2017. Web. 13 Aug 2020.

Vancouver:

McGowan MA. How the Streptococcal M87 Protein Binds Human C4b-Binding Protein. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2020 Aug 13]. Available from: http://www.escholarship.org/uc/item/8hg1b4v4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McGowan MA. How the Streptococcal M87 Protein Binds Human C4b-Binding Protein. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/8hg1b4v4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Silva, Ludmila Bezerra da. Interação da proteína de superfície LcpA de Leptospira com Fator H, principal regulador solúvel da via alternativa do sistema complemento humano.

Degree: Mestrado, Epidemiologia Experimental Aplicada às Zoonoses, 2013, University of São Paulo

A leptospirose é uma zoonose de distribuição mundial, com maior incidência nas regiões tropicais. As bactérias que causam a doença pertencem ao gênero Leptospira, família… (more)

Subjects/Keywords: Leptospira; Leptospira; Complement System; Evasão imune; Immune evasion; Sistema Complemento

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APA (6th Edition):

Silva, L. B. d. (2013). Interação da proteína de superfície LcpA de Leptospira com Fator H, principal regulador solúvel da via alternativa do sistema complemento humano. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10134/tde-26112013-143254/ ;

Chicago Manual of Style (16th Edition):

Silva, Ludmila Bezerra da. “Interação da proteína de superfície LcpA de Leptospira com Fator H, principal regulador solúvel da via alternativa do sistema complemento humano.” 2013. Masters Thesis, University of São Paulo. Accessed August 13, 2020. http://www.teses.usp.br/teses/disponiveis/10/10134/tde-26112013-143254/ ;.

MLA Handbook (7th Edition):

Silva, Ludmila Bezerra da. “Interação da proteína de superfície LcpA de Leptospira com Fator H, principal regulador solúvel da via alternativa do sistema complemento humano.” 2013. Web. 13 Aug 2020.

Vancouver:

Silva LBd. Interação da proteína de superfície LcpA de Leptospira com Fator H, principal regulador solúvel da via alternativa do sistema complemento humano. [Internet] [Masters thesis]. University of São Paulo; 2013. [cited 2020 Aug 13]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10134/tde-26112013-143254/ ;.

Council of Science Editors:

Silva LBd. Interação da proteína de superfície LcpA de Leptospira com Fator H, principal regulador solúvel da via alternativa do sistema complemento humano. [Masters Thesis]. University of São Paulo; 2013. Available from: http://www.teses.usp.br/teses/disponiveis/10/10134/tde-26112013-143254/ ;


McMaster University

16. Lai, Frances W. The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response.

Degree: PhD, 2013, McMaster University

Coronaviruses are the largest known RNA viruses and infect a wide range of hosts. Human coronaviruses traditionally have been known to be the cause… (more)

Subjects/Keywords: interferon; NF-kappaB; microRNA; immune evasion; Virology; Virology

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APA (6th Edition):

Lai, F. W. (2013). The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/15288

Chicago Manual of Style (16th Edition):

Lai, Frances W. “The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response.” 2013. Doctoral Dissertation, McMaster University. Accessed August 13, 2020. http://hdl.handle.net/11375/15288.

MLA Handbook (7th Edition):

Lai, Frances W. “The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response.” 2013. Web. 13 Aug 2020.

Vancouver:

Lai FW. The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response. [Internet] [Doctoral dissertation]. McMaster University; 2013. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/11375/15288.

Council of Science Editors:

Lai FW. The Human Coronavirus Nucleocapsid Protein and Its Effects on the Innate Immune Response. [Doctoral Dissertation]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15288

17. Grevelink, T. Evaluation of research techniques to study fungal evasion.

Degree: 2014, Universiteit Utrecht

 The incidence of patients with fungal infections is increased due to increased numbers of immunosuppresed individuals. Opportunistic fungi are a major cause of death under… (more)

Subjects/Keywords: Fungi; Immune Evasion; Techniques

evasion of immune recognition, complement attack, phagocytotic trafficking, oxidative bursts and… …evade from complement attacks48. Once a protein or gene involved in immune evasion has been… …interference. To understand the molecular mechanisms of immune evasion pathways that involve DNA… …neoformans52. Another valuable tool to study immune evasion is to genetically manipulate genes that… …developed immune evasion mechanisms. These mechanisms involve escape from recognition by PRRs… 

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APA (6th Edition):

Grevelink, T. (2014). Evaluation of research techniques to study fungal evasion. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/296006

Chicago Manual of Style (16th Edition):

Grevelink, T. “Evaluation of research techniques to study fungal evasion.” 2014. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/296006.

MLA Handbook (7th Edition):

Grevelink, T. “Evaluation of research techniques to study fungal evasion.” 2014. Web. 13 Aug 2020.

Vancouver:

Grevelink T. Evaluation of research techniques to study fungal evasion. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/296006.

Council of Science Editors:

Grevelink T. Evaluation of research techniques to study fungal evasion. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/296006

18. Dinther, D. van. Possible TAP inhibitors in HHV-6 and HHV-7 based on known characteristics of TAP inhibitors and their evolutionary development in herpesviruses.

Degree: 2013, Universiteit Utrecht

 Herpesviruses are well-known because of their latency and immune evasion strategies. The MHC class I antigen presentation pathway is an important target, especially MHC class… (more)

Subjects/Keywords: herpesvirus; immune evasion; evolution

…As immune evasion proteins have co-evolved with their host, these are relatively new… …of the genomes. HHV-6 and HHV-7 immune evasion The fact that so many different TAP… …immune system. The Roseoloviruses will therefore still need to express immune evasion proteins… …helper T cells are still unknown. Some other immune evasion strategies of HHV-6 and 7 have been… …For example; HCMV is encoding at least four immune evasion proteins, while EBV encodes three… 

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APA (6th Edition):

Dinther, D. v. (2013). Possible TAP inhibitors in HHV-6 and HHV-7 based on known characteristics of TAP inhibitors and their evolutionary development in herpesviruses. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/281587

Chicago Manual of Style (16th Edition):

Dinther, D van. “Possible TAP inhibitors in HHV-6 and HHV-7 based on known characteristics of TAP inhibitors and their evolutionary development in herpesviruses.” 2013. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/281587.

MLA Handbook (7th Edition):

Dinther, D van. “Possible TAP inhibitors in HHV-6 and HHV-7 based on known characteristics of TAP inhibitors and their evolutionary development in herpesviruses.” 2013. Web. 13 Aug 2020.

Vancouver:

Dinther Dv. Possible TAP inhibitors in HHV-6 and HHV-7 based on known characteristics of TAP inhibitors and their evolutionary development in herpesviruses. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/281587.

Council of Science Editors:

Dinther Dv. Possible TAP inhibitors in HHV-6 and HHV-7 based on known characteristics of TAP inhibitors and their evolutionary development in herpesviruses. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/281587


Universiteit Utrecht

19. Driel, B.J. van. evasion of interferon responses by herpes viruses.

Degree: 2010, Universiteit Utrecht

 Mammalian hosts have developed a highly effective anti-viral strategy that uses interferons (signaling molecules). The recognition of a virus by the wide arsenal of detection… (more)

Subjects/Keywords: herpes; virus; innate; immune; evasion; pattern recognition receptor; interferon type 1

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APA (6th Edition):

Driel, B. J. v. (2010). evasion of interferon responses by herpes viruses. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/188849

Chicago Manual of Style (16th Edition):

Driel, B J van. “evasion of interferon responses by herpes viruses.” 2010. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/188849.

MLA Handbook (7th Edition):

Driel, B J van. “evasion of interferon responses by herpes viruses.” 2010. Web. 13 Aug 2020.

Vancouver:

Driel BJv. evasion of interferon responses by herpes viruses. [Internet] [Masters thesis]. Universiteit Utrecht; 2010. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/188849.

Council of Science Editors:

Driel BJv. evasion of interferon responses by herpes viruses. [Masters Thesis]. Universiteit Utrecht; 2010. Available from: http://dspace.library.uu.nl:8080/handle/1874/188849


Universiteit Utrecht

20. Hulswit, R.J.G. HIV adaptation to HLA: Loss of Protection?.

Degree: 2014, Universiteit Utrecht

 HLA-B-restricted CD8+ T cell responses exert strong pressure on HIV replication and adaptation in HIV-infected individuals and are associated with delayed progression to AIDS. Development… (more)

Subjects/Keywords: HIV; HLA; viral fitness; HLA-B; immune evasion

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APA (6th Edition):

Hulswit, R. J. G. (2014). HIV adaptation to HLA: Loss of Protection?. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/292015

Chicago Manual of Style (16th Edition):

Hulswit, R J G. “HIV adaptation to HLA: Loss of Protection?.” 2014. Masters Thesis, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/292015.

MLA Handbook (7th Edition):

Hulswit, R J G. “HIV adaptation to HLA: Loss of Protection?.” 2014. Web. 13 Aug 2020.

Vancouver:

Hulswit RJG. HIV adaptation to HLA: Loss of Protection?. [Internet] [Masters thesis]. Universiteit Utrecht; 2014. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/292015.

Council of Science Editors:

Hulswit RJG. HIV adaptation to HLA: Loss of Protection?. [Masters Thesis]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/292015


Universiteit Utrecht

21. Kuipers, A. Mechanisms to suppress or enhance phagocytosis of staphylococci.

Degree: 2016, Universiteit Utrecht

 Staphylococcus aureus (S. aureus) is a major human pathogen responsible for many community- and hospital-acquired infections. In humans, host clearance of S. aureus critically depends… (more)

Subjects/Keywords: Staphylococcus aureus; immune evasion molecules; Efb; antibodies; phagocytosis; neutrophils

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APA (6th Edition):

Kuipers, A. (2016). Mechanisms to suppress or enhance phagocytosis of staphylococci. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/334326

Chicago Manual of Style (16th Edition):

Kuipers, A. “Mechanisms to suppress or enhance phagocytosis of staphylococci.” 2016. Doctoral Dissertation, Universiteit Utrecht. Accessed August 13, 2020. http://dspace.library.uu.nl:8080/handle/1874/334326.

MLA Handbook (7th Edition):

Kuipers, A. “Mechanisms to suppress or enhance phagocytosis of staphylococci.” 2016. Web. 13 Aug 2020.

Vancouver:

Kuipers A. Mechanisms to suppress or enhance phagocytosis of staphylococci. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2016. [cited 2020 Aug 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/334326.

Council of Science Editors:

Kuipers A. Mechanisms to suppress or enhance phagocytosis of staphylococci. [Doctoral Dissertation]. Universiteit Utrecht; 2016. Available from: http://dspace.library.uu.nl:8080/handle/1874/334326


Washington State University

22. [No author]. DECIPHERING MECHANISMS OF VLSE-MEDIATED IMMUNE AVOIDANCE BY BORRELIA BURGDORFERI .

Degree: 2014, Washington State University

The current work has focused on antigenic variation of the VlsE surface lipoprotein, a key mechanism for immune evasion and persistent infection by the Lyme disease spirochete Advisors/Committee Members: Bankhead, Troy (advisor).

Subjects/Keywords: Microbiology; antigenic variation; Borrelia burgdorferi; immune evasion; reinfection; superinfection; VlsE

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APA (6th Edition):

author], [. (2014). DECIPHERING MECHANISMS OF VLSE-MEDIATED IMMUNE AVOIDANCE BY BORRELIA BURGDORFERI . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/5402

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “DECIPHERING MECHANISMS OF VLSE-MEDIATED IMMUNE AVOIDANCE BY BORRELIA BURGDORFERI .” 2014. Thesis, Washington State University. Accessed August 13, 2020. http://hdl.handle.net/2376/5402.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “DECIPHERING MECHANISMS OF VLSE-MEDIATED IMMUNE AVOIDANCE BY BORRELIA BURGDORFERI .” 2014. Web. 13 Aug 2020.

Vancouver:

author] [. DECIPHERING MECHANISMS OF VLSE-MEDIATED IMMUNE AVOIDANCE BY BORRELIA BURGDORFERI . [Internet] [Thesis]. Washington State University; 2014. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/2376/5402.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. DECIPHERING MECHANISMS OF VLSE-MEDIATED IMMUNE AVOIDANCE BY BORRELIA BURGDORFERI . [Thesis]. Washington State University; 2014. Available from: http://hdl.handle.net/2376/5402

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

23. Nagy, Kristina. Acceptance of Allogeneic Cell Transplants without Systemic Immune Suppression.

Degree: PhD, 2020, University of Toronto

 Cell-based therapies hold great promise for the future of modern medicine. A large number of injuries and diseases that are currently not possible to cure… (more)

Subjects/Keywords: allograft tolerance; immune evasion; stem cells; tolerance; transplantation; 0379

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APA (6th Edition):

Nagy, K. (2020). Acceptance of Allogeneic Cell Transplants without Systemic Immune Suppression. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/100950

Chicago Manual of Style (16th Edition):

Nagy, Kristina. “Acceptance of Allogeneic Cell Transplants without Systemic Immune Suppression.” 2020. Doctoral Dissertation, University of Toronto. Accessed August 13, 2020. http://hdl.handle.net/1807/100950.

MLA Handbook (7th Edition):

Nagy, Kristina. “Acceptance of Allogeneic Cell Transplants without Systemic Immune Suppression.” 2020. Web. 13 Aug 2020.

Vancouver:

Nagy K. Acceptance of Allogeneic Cell Transplants without Systemic Immune Suppression. [Internet] [Doctoral dissertation]. University of Toronto; 2020. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/1807/100950.

Council of Science Editors:

Nagy K. Acceptance of Allogeneic Cell Transplants without Systemic Immune Suppression. [Doctoral Dissertation]. University of Toronto; 2020. Available from: http://hdl.handle.net/1807/100950


Duke University

24. Vazquez, Christine. Novel mechanisms of antiviral innate immune regulation by the hepatitis C virus NS3-NS4A protease .

Degree: 2019, Duke University

  Hepatitis C virus (HCV) evasion of the host immune system is largely mediated by the actions of the HCV NS3-NS4A protease complex, which consists… (more)

Subjects/Keywords: Virology; Immunology; HCV; Immune evasion; Innate Immunity; MAVS; RIG-I; Riplet

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APA (6th Edition):

Vazquez, C. (2019). Novel mechanisms of antiviral innate immune regulation by the hepatitis C virus NS3-NS4A protease . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/20109

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vazquez, Christine. “Novel mechanisms of antiviral innate immune regulation by the hepatitis C virus NS3-NS4A protease .” 2019. Thesis, Duke University. Accessed August 13, 2020. http://hdl.handle.net/10161/20109.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vazquez, Christine. “Novel mechanisms of antiviral innate immune regulation by the hepatitis C virus NS3-NS4A protease .” 2019. Web. 13 Aug 2020.

Vancouver:

Vazquez C. Novel mechanisms of antiviral innate immune regulation by the hepatitis C virus NS3-NS4A protease . [Internet] [Thesis]. Duke University; 2019. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/10161/20109.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vazquez C. Novel mechanisms of antiviral innate immune regulation by the hepatitis C virus NS3-NS4A protease . [Thesis]. Duke University; 2019. Available from: http://hdl.handle.net/10161/20109

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

25. Palladinetti, Patricia. The role of viral chemokine receptor US28 in the pathogenesis of HCMV disease.

Degree: Medical Sciences, 2012, University of New South Wales

 This thesis is the first to report in vivo expression of the US28 gene and provides strong evidence of a potential role in HCMV disease.… (more)

Subjects/Keywords: Cytomegalovirus; Viral Chemokine Receptor US28; Viral immune evasion

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APA (6th Edition):

Palladinetti, P. (2012). The role of viral chemokine receptor US28 in the pathogenesis of HCMV disease. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53568 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12265/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Palladinetti, Patricia. “The role of viral chemokine receptor US28 in the pathogenesis of HCMV disease.” 2012. Doctoral Dissertation, University of New South Wales. Accessed August 13, 2020. http://handle.unsw.edu.au/1959.4/53568 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12265/SOURCE02?view=true.

MLA Handbook (7th Edition):

Palladinetti, Patricia. “The role of viral chemokine receptor US28 in the pathogenesis of HCMV disease.” 2012. Web. 13 Aug 2020.

Vancouver:

Palladinetti P. The role of viral chemokine receptor US28 in the pathogenesis of HCMV disease. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Aug 13]. Available from: http://handle.unsw.edu.au/1959.4/53568 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12265/SOURCE02?view=true.

Council of Science Editors:

Palladinetti P. The role of viral chemokine receptor US28 in the pathogenesis of HCMV disease. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/53568 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12265/SOURCE02?view=true


University of Oxford

26. Gwela, Agnes A. Analysis of the immune evasion mechanisms of varicella zoster virus.

Degree: PhD, 2013, University of Oxford

 Varicella zoster virus (VZV) is an alpha herpes virus that causes primary infection with varicella (chicken pox), establishes latency in ganglia and may later reactivate… (more)

Subjects/Keywords: 614.5; Medical Sciences; Immunology; Viruses; human immunology; herpesviruses; immune evasion

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APA (6th Edition):

Gwela, A. A. (2013). Analysis of the immune evasion mechanisms of varicella zoster virus. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:79814bbd-ed0a-47b4-9894-96710892eefa ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.629494

Chicago Manual of Style (16th Edition):

Gwela, Agnes A. “Analysis of the immune evasion mechanisms of varicella zoster virus.” 2013. Doctoral Dissertation, University of Oxford. Accessed August 13, 2020. http://ora.ox.ac.uk/objects/uuid:79814bbd-ed0a-47b4-9894-96710892eefa ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.629494.

MLA Handbook (7th Edition):

Gwela, Agnes A. “Analysis of the immune evasion mechanisms of varicella zoster virus.” 2013. Web. 13 Aug 2020.

Vancouver:

Gwela AA. Analysis of the immune evasion mechanisms of varicella zoster virus. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Aug 13]. Available from: http://ora.ox.ac.uk/objects/uuid:79814bbd-ed0a-47b4-9894-96710892eefa ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.629494.

Council of Science Editors:

Gwela AA. Analysis of the immune evasion mechanisms of varicella zoster virus. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:79814bbd-ed0a-47b4-9894-96710892eefa ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.629494


University of Pennsylvania

27. Munoz, Vanessa Lynne. Surface Polysaccharides Promote Innate Immune Evasion By The Pediatric Pathogen Kingella Kingae.

Degree: 2019, University of Pennsylvania

 The gram-negative coccobacillus Kingella kingae is an emerging pediatric pathogen and is increasingly recognized as a common etiological agent of osteoarticular infections and bacteremia in… (more)

Subjects/Keywords: Immune evasion; Innate immunity; Kingella kingae; Surface polysaccharides; Microbiology

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APA (6th Edition):

Munoz, V. L. (2019). Surface Polysaccharides Promote Innate Immune Evasion By The Pediatric Pathogen Kingella Kingae. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/3308

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Munoz, Vanessa Lynne. “Surface Polysaccharides Promote Innate Immune Evasion By The Pediatric Pathogen Kingella Kingae.” 2019. Thesis, University of Pennsylvania. Accessed August 13, 2020. https://repository.upenn.edu/edissertations/3308.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Munoz, Vanessa Lynne. “Surface Polysaccharides Promote Innate Immune Evasion By The Pediatric Pathogen Kingella Kingae.” 2019. Web. 13 Aug 2020.

Vancouver:

Munoz VL. Surface Polysaccharides Promote Innate Immune Evasion By The Pediatric Pathogen Kingella Kingae. [Internet] [Thesis]. University of Pennsylvania; 2019. [cited 2020 Aug 13]. Available from: https://repository.upenn.edu/edissertations/3308.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Munoz VL. Surface Polysaccharides Promote Innate Immune Evasion By The Pediatric Pathogen Kingella Kingae. [Thesis]. University of Pennsylvania; 2019. Available from: https://repository.upenn.edu/edissertations/3308

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Miami University

28. Feng, Monica. Properties and development of Mycoplasma pneumoniae biofilms in relation to persistence and cytotoxicity.

Degree: PhD, Microbiology, 2019, Miami University

 Mycoplasma pneumoniae is a bacterial pathogen that primarily infects the humanrespiratory tract but is also capable of causing extrapulmonary disease. M. pneumoniaeinfections are often underdiagnosed… (more)

Subjects/Keywords: Microbiology; Mycoplasma pneumoniae, biofilm, antibiotic resistance, immune evasion

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APA (6th Edition):

Feng, M. (2019). Properties and development of Mycoplasma pneumoniae biofilms in relation to persistence and cytotoxicity. (Doctoral Dissertation). Miami University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=miami1565869297815629

Chicago Manual of Style (16th Edition):

Feng, Monica. “Properties and development of Mycoplasma pneumoniae biofilms in relation to persistence and cytotoxicity.” 2019. Doctoral Dissertation, Miami University. Accessed August 13, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=miami1565869297815629.

MLA Handbook (7th Edition):

Feng, Monica. “Properties and development of Mycoplasma pneumoniae biofilms in relation to persistence and cytotoxicity.” 2019. Web. 13 Aug 2020.

Vancouver:

Feng M. Properties and development of Mycoplasma pneumoniae biofilms in relation to persistence and cytotoxicity. [Internet] [Doctoral dissertation]. Miami University; 2019. [cited 2020 Aug 13]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1565869297815629.

Council of Science Editors:

Feng M. Properties and development of Mycoplasma pneumoniae biofilms in relation to persistence and cytotoxicity. [Doctoral Dissertation]. Miami University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1565869297815629


Iowa State University

29. Ramanan, Parameshwaran. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.

Degree: 2012, Iowa State University

 Filoviruses are among the most deadly pathogens that cause acute disease in humans. Ebolavirus (EBOV) and marburgvirus (MARV) are the two members of this family,… (more)

Subjects/Keywords: dsRNA binding; immune evasion; Marburg virus; type I interferon; VP35; Biochemistry

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APA (6th Edition):

Ramanan, P. (2012). Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/12889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramanan, Parameshwaran. “Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.” 2012. Thesis, Iowa State University. Accessed August 13, 2020. https://lib.dr.iastate.edu/etd/12889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramanan, Parameshwaran. “Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion.” 2012. Web. 13 Aug 2020.

Vancouver:

Ramanan P. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. [Internet] [Thesis]. Iowa State University; 2012. [cited 2020 Aug 13]. Available from: https://lib.dr.iastate.edu/etd/12889.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramanan P. Structural and biochemical characterization of marburgvirus VP35 and its role in immune evasion. [Thesis]. Iowa State University; 2012. Available from: https://lib.dr.iastate.edu/etd/12889

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Melbourne

30. Nivarthi, Usha Kiranmayee. T cell receptor repertoire recognition of hypervariable Hepatitis C virus.

Degree: 2012, University of Melbourne

 Infection with Hepatitis C virus (HCV) is a major public health problem, affecting more than 200 million people worldwide. 70% of the infected individuals progress… (more)

Subjects/Keywords: T cell response; Hepatitis C virus; immune evasion; vaccines

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nivarthi, U. K. (2012). T cell receptor repertoire recognition of hypervariable Hepatitis C virus. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37324

Chicago Manual of Style (16th Edition):

Nivarthi, Usha Kiranmayee. “T cell receptor repertoire recognition of hypervariable Hepatitis C virus.” 2012. Doctoral Dissertation, University of Melbourne. Accessed August 13, 2020. http://hdl.handle.net/11343/37324.

MLA Handbook (7th Edition):

Nivarthi, Usha Kiranmayee. “T cell receptor repertoire recognition of hypervariable Hepatitis C virus.” 2012. Web. 13 Aug 2020.

Vancouver:

Nivarthi UK. T cell receptor repertoire recognition of hypervariable Hepatitis C virus. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2020 Aug 13]. Available from: http://hdl.handle.net/11343/37324.

Council of Science Editors:

Nivarthi UK. T cell receptor repertoire recognition of hypervariable Hepatitis C virus. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37324

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